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3. Umehara F, Hagiwara T, Yoshimura M, Higashi K, Arimura K: Enlarged, multifocal upper limb neuropathy with HTLV-I associated myelopathy in a patient with chronic adult T-cell leukemia. J Neurol Sci; 2008 Mar 15;266(1-2):167-70
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  • [Title] Enlarged, multifocal upper limb neuropathy with HTLV-I associated myelopathy in a patient with chronic adult T-cell leukemia.
  • The authors herein describe a case of multifocal peripheral neuropathy with HTLV-I-associated myelopathy (HAM) in a patient with chronic adult T-cell leukemia (ATL).
  • The clinical features included subacute progressive sensory-motor neuropathy in the bilateral upper limbs, and bilateral pyramidal tract involvement with bladder dysfunction.
  • An MRI with (67)gadolinium enhancement revealed enlargement of the affected peripheral nerves. (8)FDG positron emission tomography (PET) disclosed increased uptake in the affected nerves, suggesting neurolymphomatosis or inflammation.
  • Chemotherapy for ATL resulted in marked improvement of motor functions in the upper limbs.
  • [MeSH-major] Leukemia, Prolymphocytic, T-Cell / complications. Paraparesis, Tropical Spastic / complications. Peripheral Nervous System Diseases / etiology. Peripheral Nervous System Diseases / pathology. Upper Extremity / pathology
  • [MeSH-minor] Blood Cell Count. Contrast Media. Female. Fluorodeoxyglucose F18. Gadolinium. Humans. Magnetic Resonance Imaging. Middle Aged. Neural Conduction / physiology. Neurologic Examination. Peripheral Nerves / pathology. Peripheral Nerves / physiopathology. Positron-Emission Tomography

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  • (PMID = 18096188.001).
  • [ISSN] 0022-510X
  • [Journal-full-title] Journal of the neurological sciences
  • [ISO-abbreviation] J. Neurol. Sci.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Contrast Media; 0Z5B2CJX4D / Fluorodeoxyglucose F18; AU0V1LM3JT / Gadolinium
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4. Marshall R, Gupta ND, Palacios E, Neitzschman HR: Progressive paresthesia and weakness after intrathecal chemotherapy. J La State Med Soc; 2008 Mar-Apr;160(2):92-4
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  • [Title] Progressive paresthesia and weakness after intrathecal chemotherapy.
  • A 53-year-old woman was diagnosed with acute lymphoblastic leukemia (ALL) confirmed by bone marrow biopsy to be pre-B ALL.
  • [MeSH-major] Antineoplastic Combined Chemotherapy Protocols / adverse effects. Muscle Weakness / etiology. Paresthesia / etiology. Precursor Cell Lymphoblastic Leukemia-Lymphoma / drug therapy. Subacute Combined Degeneration / chemically induced
  • [MeSH-minor] Cyclophosphamide / administration & dosage. Cyclophosphamide / adverse effects. Dexamethasone / administration & dosage. Dexamethasone / adverse effects. Diagnosis, Differential. Doxorubicin / administration & dosage. Doxorubicin / adverse effects. Female. Follow-Up Studies. Humans. Injections, Spinal. Magnetic Resonance Imaging. Middle Aged. Thoracic Vertebrae. Time Factors. Vincristine / administration & dosage. Vincristine / adverse effects

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  • (PMID = 18681351.001).
  • [ISSN] 0024-6921
  • [Journal-full-title] The Journal of the Louisiana State Medical Society : official organ of the Louisiana State Medical Society
  • [ISO-abbreviation] J La State Med Soc
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 5J49Q6B70F / Vincristine; 7S5I7G3JQL / Dexamethasone; 80168379AG / Doxorubicin; 8N3DW7272P / Cyclophosphamide; CVAD protocol
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5. Turtureanu-Hanganu E: Emergency care: the tumor lysis syndrome. Rev Med Chir Soc Med Nat Iasi; 2002 Oct-Dec;106(4):705-11
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  • The Tumor Lysis Syndrome (TLS) is a constellation of metabolic disturbances due to the rapid tumoral cell destruction, either spontaneous or induced by cytoreduction therapy, which manifests as an acute, subacute or chronic renal insufficiency.
  • The therapy consists in management of hydro-electrolytic and metabolic disturbances, treatment of hyperuricemia (Allopurinol and a new therapeutical agent, Uricase), hemodialysis.
  • If promptly supported during the TLS, a number of these patients have an excellent probability of long-term remission.
  • [MeSH-major] Tumor Lysis Syndrome / therapy
  • [MeSH-minor] Adult. Allopurinol / therapeutic use. Antimetabolites / therapeutic use. Antineoplastic Combined Chemotherapy Protocols / adverse effects. Antineoplastic Combined Chemotherapy Protocols / therapeutic use. Child. Child, Preschool. Emergencies. Humans. Hyperuricemia / etiology. Leukemia / drug therapy. Lymphoma / drug therapy. Renal Dialysis. Treatment Outcome

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  • (PMID = 14974215.001).
  • [ISSN] 0048-7848
  • [Journal-full-title] Revista medico-chirurgicală̆ a Societă̆ţ̜ii de Medici ş̧i Naturaliş̧ti din Iaş̧i
  • [ISO-abbreviation] Rev Med Chir Soc Med Nat Iasi
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Romania
  • [Chemical-registry-number] 0 / Antimetabolites; 63CZ7GJN5I / Allopurinol
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6. Tan HK, Lim JS, Tan CK, Ng HS, Chow P, Lui HF, Wong GC, Tan PH, Raghuram J, Ng HN, Choong LH, Wong KS, Woo KT: MARS therapy in critically ill patients with advanced malignancy: a clinical and technical report. Liver Int; 2003;23 Suppl 3:52-60
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  • [Title] MARS therapy in critically ill patients with advanced malignancy: a clinical and technical report.
  • The postoperative course following second surgery was complicated by severe methicillin-resistant Staphylococcus aureus (MRSA) sepsis, mild azotaemia and subacute cholestatic liver failure.
  • Case 2 was a female patient with advanced acute lymphoblastic leukaemia (ALL) with post bone marrow transplantation (BMT) acute haemolytic-uraemic syndrome (HUS) secondary to cyclosporin A (Cy A), cytomegalovirus (CMV) infection, severe nosocomial pneumonia, acute renal failure (ARF) treated with continuous haemofiltration and acute veno-occlusive disease resulting in Budd-Chiari syndrome.
  • Case 3 was a male patient with advanced, refractory Hodgkin's disease previously treated with multiple courses of chemotherapy.
  • ALF developed secondary to acute viral hepatitis B flare.
  • RESULTS: Mean MARS intradialytic systemic pressures were as follows: systolic pressure range was 95 +/- 17 to 128 +/- 17 mmHg and diastolic pressure range was 51 +/- 5 to 67 +/- 7 mmHg.
  • Ultrafiltration (UF) was 633 +/- 622 mL over mean treatment duration of 6.3 +/- 0.9 h with a total heparin dose of 1583 +/- 817 IU.
  • MARS had a significant de-uraemization effect (pre- and post-MARS serum creatinine and urea: P=0.046 and 0.028, respectively) but did not significantly attenuate blood lactate, ammonia or total bilirubin levels.
  • Although MARS had a significant de-uraemization effect, this appeared to be limited by the duration of MARS operation.
  • This would affect the optimal duration of MARS therapy.
  • [MeSH-major] Carcinoma, Hepatocellular / therapy. Liver Failure, Acute / therapy. Liver Neoplasms / therapy. Renal Dialysis. Sorption Detoxification
  • [MeSH-minor] Adolescent. Adult. Critical Illness. Fatal Outcome. Female. Hemolytic-Uremic Syndrome / etiology. Hemolytic-Uremic Syndrome / therapy. Hodgkin Disease / complications. Humans. Male. Middle Aged. Precursor Cell Lymphoblastic Leukemia-Lymphoma / complications

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  • (PMID = 12950962.001).
  • [ISSN] 1478-3223
  • [Journal-full-title] Liver international : official journal of the International Association for the Study of the Liver
  • [ISO-abbreviation] Liver Int.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
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7. Pöhlmann C, Hochauf K, Röllig C, Schetelig J, Wunderlich O, Bandt D, Ehninger G, Jacobs E, Rohayem J: Chlorpromazine combined with cidofovir for treatment of a patient suffering from progressive multifocal leukoencephalopathy. Intervirology; 2007;50(6):412-7
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  • [Title] Chlorpromazine combined with cidofovir for treatment of a patient suffering from progressive multifocal leukoencephalopathy.
  • We report on a stem cell-transplanted patient with B cell chronic lymphatic leukemia who presented with a subacute onset of focal neurological deficits, gait abnormalities, emotional lability and dementia.
  • Progressive multifocal leukoencephalopathy was diagnosed by magnetic resonance imaging (MRI) of the brain and detection of JC virus genome in the cerebrospinal fluid.
  • A follow-up MRI of the brain 2 weeks after initiation of the antiviral therapy displayed progress of the demyelination, and the patient died 3 months after onset of the neurological symptoms.
  • This report highlights the need for the development of novel and potent strategies for treatment of progressive multifocal leukoencephalopathy.
  • [MeSH-major] AIDS-Related Opportunistic Infections / drug therapy. Antiviral Agents / administration & dosage. Brain / drug effects. Chlorpromazine / administration & dosage. Cytosine / analogs & derivatives. JC Virus. Leukoencephalopathy, Progressive Multifocal / drug therapy. Organophosphonates / administration & dosage
  • [MeSH-minor] DNA, Viral / cerebrospinal fluid. Drug Therapy, Combination. Fatal Outcome. Humans. Magnetic Resonance Imaging. Male. Middle Aged

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  • [Copyright] (c) 2008 S. Karger AG, Basel
  • (PMID = 18182774.001).
  • [ISSN] 1423-0100
  • [Journal-full-title] Intervirology
  • [ISO-abbreviation] Intervirology
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 0 / Antiviral Agents; 0 / DNA, Viral; 0 / Organophosphonates; 8J337D1HZY / Cytosine; JIL713Q00N / cidofovir; U42B7VYA4P / Chlorpromazine
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8. Haykin ME, Gorman M, van Hoff J, Fulbright RK, Baehring JM: Diffusion-weighted MRI correlates of subacute methotrexate-related neurotoxicity. J Neurooncol; 2006 Jan;76(2):153-7
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  • [Title] Diffusion-weighted MRI correlates of subacute methotrexate-related neurotoxicity.
  • OBJECTIVES: A delayed stroke-like leukoencephalopathy has been observed in patients receiving methotrexate (MTX) for childhood leukemia.
  • Diffusion-weighted MRI (DWI) may help to distinguish between ischemic stroke and chemotherapy-related leukoencephalopathy.
  • Our objective is to present a retrospective analysis of the DWI findings in four patients who suffered subacute neurotoxicity after intrathecal MTX.
  • DESIGN: We reviewed the medical records of four patients, who were seen by us between July 2000 and February 2004 for sudden onset of a central neurological syndrome within days of intrathecal MTX.
  • [MeSH-minor] Adolescent. Diffusion Magnetic Resonance Imaging. Edema / chemically induced. Edema / pathology. Female. Humans. Leukemia, B-Cell / drug therapy. Leukemia, B-Cell / pathology. Leukemia, Lymphoid / complications. Magnetic Resonance Imaging. Retrospective Studies

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  • (PMID = 16411025.001).
  • [ISSN] 0167-594X
  • [Journal-full-title] Journal of neuro-oncology
  • [ISO-abbreviation] J. Neurooncol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antimetabolites, Antineoplastic; YL5FZ2Y5U1 / Methotrexate
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9. Lichtman MA, Rowe JM: The relationship of patient age to the pathobiology of the clonal myeloid diseases. Semin Oncol; 2004 Apr;31(2):185-97
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The incidence of the major clonal myeloid diseases, clonal cytopenias, acute, subacute (oligoblastic), and chronic myelogenous leukemia, polycythemia vera, thrombocythemia, and idiopathic myelofibrosis increases in a log-linear manner from young adulthood through advanced age.
  • In older patients, diseases requiring cytotoxic treatment are more difficult and less successful to manage because comorbid conditions and poor performance status are more prevalent, decreasing the tolerance to therapy and increasing the frequency of side effects.
  • This age effect is highlighted by the dramatically less favorable outcome in older than younger patients with acute myeloid leukemia with similar "favorable" cytogenetic changes.
  • In addition, in acute and subacute myeloid leukemia in older patients, the disease is intrinsically more resistant to therapy.
  • Overexpression of drug resistance genes and unfavorable genetic mutations are more prevalent in older patients and provide evidence that acute myeloid leukemia is often qualitatively different in these patients.
  • Although improved drug schedules have led to significant improvements in event-free survival in younger patients, these improvements have been far less evident in older patients.
  • New approaches, especially the development of drugs aimed at new targets, will be required to obtain a high frequency of long-term remissions in older patients.
  • Agents that reverse inherent cellular drug resistance, farnesyltransferase inhibitors, BCL-2 inhibitors, and FLT3 inhibitors are early examples of such approaches.
  • [MeSH-major] Leukemia, Myeloid

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  • (PMID = 15112149.001).
  • [ISSN] 0093-7754
  • [Journal-full-title] Seminars in oncology
  • [ISO-abbreviation] Semin. Oncol.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 95
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10. Takuma T, Okada K, Uchida Y, Yamagata A, Sawae Y: Invasive pulmonary aspergillosis resulting in respiratory failure during neutrophil recovery from postchemotherapy neutropenia in three patients with acute leukaemia. J Intern Med; 2002 Aug;252(2):173-7
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  • [Title] Invasive pulmonary aspergillosis resulting in respiratory failure during neutrophil recovery from postchemotherapy neutropenia in three patients with acute leukaemia.
  • We herein describe three cases of subacute respiratory failure that occurred during the recovery phase of neutropenia following induction chemotherapy for acute leukaemia with IPA.
  • [MeSH-major] Antineoplastic Combined Chemotherapy Protocols / adverse effects. Aspergillosis / complications. Leukemia / drug therapy. Lung Diseases, Fungal / complications. Neutropenia / complications. Respiratory Insufficiency / microbiology


11. Iarussi D, Indolfi P, Galderisi M, Bossone E: Cardiac toxicity after anthracycline chemotherapy in childhood. Herz; 2000 Nov;25(7):676-88
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  • [Title] Cardiac toxicity after anthracycline chemotherapy in childhood.
  • Overt heart failure occurs in 4.5% to 7% of patients treated with anthracyclines and the incidence of cardiac function abnormalities increases with the time.
  • This cardiotoxicity can be divided, on the base of when it started, into acute, subacute and progressive late, chronic form.
  • Various invasive and non-invasive methods have been used to measure the extent of cardiac damage done.
  • [MeSH-major] Antibiotics, Antineoplastic / adverse effects. Cardiomyopathies / chemically induced. Heart Failure / chemically induced. Leukemia / drug therapy. Neoplasms / drug therapy
  • [MeSH-minor] Age Factors. Child. Dose-Response Relationship, Drug. Female. Humans. Male. Risk Factors. Sex Factors


12. Eichler AF, Batchelor TT, Henson JW: Diffusion and perfusion imaging in subacute neurotoxicity following high-dose intravenous methotrexate. Neuro Oncol; 2007 Jul;9(3):373-7
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  • [Title] Diffusion and perfusion imaging in subacute neurotoxicity following high-dose intravenous methotrexate.
  • Methotrexate (MTX) is a widely used chemotherapeutic agent that can cause acute, subacute, and chronic neurological complications.
  • Subacute MTX neurotoxicity is manifest by abrupt onset of focal cerebral dysfunction occurring days to weeks after MTX administration, usually in children.
  • We describe the neuroimaging features of an adult patient with primary CNS lymphoma who presented with transient aphasia and right hemiparesis 12 days after receiving intravenous high-dose MTX (8 g/m2) chemotherapy.
  • The absence of vascular or perfusion abnormalities suggests that transient cytotoxic edema in white matter may explain the syndrome of subacute MTX neurotoxicity.
  • [MeSH-minor] Adult. Brain Neoplasms / drug therapy. Humans. Lymphoma / drug therapy. Magnetic Resonance Imaging. Male. Tomography, X-Ray Computed

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  • (PMID = 17522329.001).
  • [ISSN] 1522-8517
  • [Journal-full-title] Neuro-oncology
  • [ISO-abbreviation] Neuro-oncology
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antineoplastic Agents; YL5FZ2Y5U1 / Methotrexate
  • [Other-IDs] NLM/ PMC1907407
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13. Pascual AM, Coret F, Casanova B, Láinez MJ: Anterior lumbosacral polyradiculopathy after intrathecal administration of methotrexate. J Neurol Sci; 2008 Apr 15;267(1-2):158-61
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Transient paraparesis has been reported with intrathecal chemotherapy agents and the most common cause is an incomplete inflammatory myelopathy.
  • We report a case of a 30-year-old man diagnosed with acute lymphoblastic leukaemia who developed subacute anterior lumbosacral polyradiculopathy following intrathecal methotrexate, an unusual complication of intrathecal chemotherapy in adults.
  • Differential diagnosis included toxic and neoplastic polyradiculopathy, and axonal variant of acute inflammatory demyelinating polyradiculoneuropathy.
  • The authors review possible pathogenic mechanisms and propose several therapeutic and preventive options.
  • [MeSH-major] Lumbosacral Plexus / drug effects. Methotrexate / adverse effects. Paraparesis / chemically induced. Polyradiculopathy / chemically induced. Spinal Nerve Roots / drug effects
  • [MeSH-minor] Adult. Antimetabolites, Antineoplastic / administration & dosage. Antimetabolites, Antineoplastic / adverse effects. Antineoplastic Combined Chemotherapy Protocols / therapeutic use. Drug Interactions / physiology. Fatal Outcome. Humans. Hydrocortisone / administration & dosage. Injections, Spinal / adverse effects. Leg / innervation. Leg / physiopathology. Male. Motor Neurons / pathology. Muscle, Skeletal / innervation. Muscle, Skeletal / physiopathology. Neural Conduction / physiology. Paralysis / chemically induced. Precursor Cell Lymphoblastic Leukemia-Lymphoma / drug therapy. Precursor Cell Lymphoblastic Leukemia-Lymphoma / genetics. Respiratory Tract Infections. Sepsis. Urinary Bladder, Neurogenic / chemically induced

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  • (PMID = 17949753.001).
  • [ISSN] 0022-510X
  • [Journal-full-title] Journal of the neurological sciences
  • [ISO-abbreviation] J. Neurol. Sci.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Antimetabolites, Antineoplastic; WI4X0X7BPJ / Hydrocortisone; YL5FZ2Y5U1 / Methotrexate
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14. Haykin M, Gorman M, van Hoff J, Fulbright R, Baehring J: Diffusion-weighted MRI correlates of subacute methotrexate-related neurotoxicity. J Neurooncol; 2006 Jan;76(2):153-7
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  • [Title] Diffusion-weighted MRI correlates of subacute methotrexate-related neurotoxicity.
  • OBJECTIVES: A delayed stroke-like leukoencephalopathy has been observed in patients receiving methotrexate (MTX) for childhood leukemia.
  • Diffusion-weighted MRI (DWI) may help to distinguish between ischemic stroke and chemotherapy-related leukoencephalopathy.
  • Our objective is to present a retrospective analysis of the DWI findings in four patients who suffered subacute neurotoxicity after intrathecal MTX.
  • DESIGN: We reviewed the medical records of four patients, who were seen by us between July 2000 and February 2004 for sudden onset of a central neurological syndrome within days of intrathecal MTX.
  • [MeSH-minor] Adolescent. Diffusion Magnetic Resonance Imaging. Edema / chemically induced. Edema / pathology. Female. Humans. Leukemia, B-Cell / complications. Leukemia, B-Cell / drug therapy. Leukemia, B-Cell / pathology. Magnetic Resonance Imaging. Retrospective Studies

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  • (PMID = 16132495.001).
  • [ISSN] 0167-594X
  • [Journal-full-title] Journal of neuro-oncology
  • [ISO-abbreviation] J. Neurooncol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antimetabolites, Antineoplastic; YL5FZ2Y5U1 / Methotrexate
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16. Shuper A, Stark B, Kornreich L, Cohen IJ, Aviner S, Steinmetz A, Stein J, Goshen Y, Yaniv I: Methotrexate treatment protocols and the central nervous system: significant cure with significant neurotoxicity. J Child Neurol; 2000 Sep;15(9):573-80
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  • [Title] Methotrexate treatment protocols and the central nervous system: significant cure with significant neurotoxicity.
  • These effects can be categorized as immediate, acute to subacute, or chronic neurologic syndromes.
  • The acute to subacute syndrome occurs frequently in acute lymphoblastic leukemia treatment protocols, generally manifesting with focal neurologic signs and changes seen on magnetic resonance imaging and single photon emission computed tomography.
  • While in some patients the neurotoxicity is transient and benign and allows for continuation of chemotherapy, in others it can be quite severe and debilitating, leading to permanent neurologic deficits.
  • The need to modify the treatment protocols when neurotoxicity appears is not fully established.
  • It is also unknown whether the use of sufficient amounts of leucovorin can overcome the toxic effects of the drug.
  • [MeSH-minor] Adolescent. Adult. Central Nervous System / diagnostic imaging. Central Nervous System / pathology. Child. Child, Preschool. Electroencephalography. Female. Humans. Leucovorin / therapeutic use. Magnetic Resonance Imaging. Male. Precursor Cell Lymphoblastic Leukemia-Lymphoma / drug therapy. Tomography, Emission-Computed, Single-Photon

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  • (PMID = 11019787.001).
  • [ISSN] 0883-0738
  • [Journal-full-title] Journal of child neurology
  • [ISO-abbreviation] J. Child Neurol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antimetabolites, Antineoplastic; Q573I9DVLP / Leucovorin; YL5FZ2Y5U1 / Methotrexate
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17. Strunk T, Gottschalk S, Goepel W, Bucsky P, Schultz C: Subacute leukencephalopathy after low-dose intrathecal methotrexate in an adolescent heterozygous for the MTHFR C677T polymorphism. Med Pediatr Oncol; 2003 Jan;40(1):48-50
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  • [Title] Subacute leukencephalopathy after low-dose intrathecal methotrexate in an adolescent heterozygous for the MTHFR C677T polymorphism.
  • [MeSH-minor] Adolescent. Heterozygote. Humans. Injections, Spinal. Male. Methylenetetrahydrofolate Reductase (NADPH2). Precursor Cell Lymphoblastic Leukemia-Lymphoma / drug therapy

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  • (PMID = 12426687.001).
  • [ISSN] 0098-1532
  • [Journal-full-title] Medical and pediatric oncology
  • [ISO-abbreviation] Med. Pediatr. Oncol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antimetabolites, Antineoplastic; EC 1.5.- / Oxidoreductases Acting on CH-NH Group Donors; EC 1.5.1.20 / Methylenetetrahydrofolate Reductase (NADPH2); YL5FZ2Y5U1 / Methotrexate
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18. Schanz J, Weiss B, Henrich D, Bohrer MH, Uppenkamp M: [30 year-old patient with multiple pelvic lesions and fecal incontinence]. Internist (Berl); 2009 Sep;50(9):1155, 1157-60
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  • [Transliterated title] Multiple Raumforderungen bei einem 30-Jährigen mit Stuhlinkontinenz.
  • In a 30 year-old patient with subacute loss of bowel control and perianal anesthesia radiologic examination showed multiple bone lesions.
  • The results of a bone marrow aspiration showed acute myeloid leukemia M2 with translocation t(8,21) associated with granulocytic sarcoma.
  • The patient was treated with high dose chemotherapy and had a complete remission after autologous stem cell transplantation.
  • [MeSH-major] Fecal Incontinence / prevention & control. Leukemia, Myeloid, Acute / diagnosis. Leukemia, Myeloid, Acute / surgery. Pelvic Neoplasms / diagnosis. Pelvic Neoplasms / surgery. Stem Cell Transplantation
  • [MeSH-minor] Adult. Humans. Male. Treatment Outcome

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19. Marsh JC, Gielda BT, Herskovic AM, Abrams RA: Cognitive Sparing during the Administration of Whole Brain Radiotherapy and Prophylactic Cranial Irradiation: Current Concepts and Approaches. J Oncol; 2010;2010:198208
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  • Whole brain radiotherapy (WBRT) for the palliation of metastases, or as prophylaxis to prevent intracranial metastases, can be associated with subacute and late decline in memory and other cognitive functions.
  • Moreover, these changes are often increased in both frequency and severity when cranial irradiation is combined with the use of systemic or intrathecal chemotherapy.
  • Approaches to preventing or reducing this toxicity include the use of stereotactic radiosurgery (SRS) instead of WBRT; dose reduction for PCI; exclusion of the limbic circuit, hippocampal formation, and/or neural stem cell regions of the brain during radiotherapy; avoidance of intrathecal and/or systemic chemotherapy during radiotherapy; the use of high-dose, systemic chemotherapy in lieu of WBRT.

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  • (PMID = 20671962.001).
  • [ISSN] 1687-8469
  • [Journal-full-title] Journal of oncology
  • [ISO-abbreviation] J Oncol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Egypt
  • [Other-IDs] NLM/ PMC2910483
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20. Arkenau HT, Gordon C, Murphy F, Cunningham D: Paraneoplastic syndrome: subacute cerebellar degeneration in Hodgkin's disease. Leuk Lymphoma; 2007 Apr;48(4):819-22
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  • [Title] Paraneoplastic syndrome: subacute cerebellar degeneration in Hodgkin's disease.
  • [MeSH-major] Hodgkin Disease / diagnosis. Paraneoplastic Syndromes / diagnosis. Paraneoplastic Syndromes / pathology
  • [MeSH-minor] Antineoplastic Combined Chemotherapy Protocols / therapeutic use. Brain / pathology. Cerebellar Diseases / drug therapy. Diagnosis, Differential. Humans. Magnetic Resonance Imaging. Male. Middle Aged. Spinocerebellar Degenerations / diagnosis. Spinocerebellar Degenerations / pathology. Treatment Outcome


21. Igata A: Clinical studies on rising and re-rising neurological diseases in Japan--a personal contribution. Proc Jpn Acad Ser B Phys Biol Sci; 2010;86(4):366-77
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  • 1) SMON (subacute myelo-optico-neuropathy).
  • Through the chemical analysis of the green urine, characteristic of this disease, it was found that this disease was caused by intoxication of the administered clioquinol, an anti-diarrheal drug.
  • 3) In 1972, we noticed a group of sporadic paraparesis in Kagoshima, which was 20 years later confirmed to be induced by human T lymphotropic virus type-I (HTLV-I).
  • It gave a strong impact that the causative virus of adult T cell leukemia (ATL) can induce entirely different diseases, in terms of both the clinical course and the pathological features.
  • [MeSH-minor] Beriberi / epidemiology. Beriberi / etiology. Beriberi / therapy. Humans. Japan / epidemiology. Optic Nerve Diseases / chemically induced. Optic Nerve Diseases / epidemiology. Optic Nerve Diseases / therapy. Paraparesis, Tropical Spastic / complications. Paraparesis, Tropical Spastic / epidemiology. Paraparesis, Tropical Spastic / etiology. Paraparesis, Tropical Spastic / transmission

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  • (PMID = 20431261.001).
  • [ISSN] 1349-2896
  • [Journal-full-title] Proceedings of the Japan Academy. Series B, Physical and biological sciences
  • [ISO-abbreviation] Proc. Jpn. Acad., Ser. B, Phys. Biol. Sci.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Japan
  • [Number-of-references] 25
  • [Other-IDs] NLM/ PMC3417800
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