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1. Lee SH, Cheon JH, Kim JH, Park JP, Lee SK, Lee YC: [Non-healing iatrogenic gastric ulcers after endoscopic mucosal resection for gastric epithelial neoplasia: report of two cases]. Korean J Gastroenterol; 2008 Feb;51(2):127-31
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  • [Title] [Non-healing iatrogenic gastric ulcers after endoscopic mucosal resection for gastric epithelial neoplasia: report of two cases].
  • Endoscopic mucosal resection (EMR) is widely accepted as a standard treatment for early gastric cancer or gastric adenoma.
  • Although the characteristics of EMR-induced ulceration are not fully understood, this type of ulcer is thought to heal faster and to recur less often than non-iatrogenic gastric ulcer.
  • Herein, we report two cases of non-healing persistent gastric ulcers after EMR.
  • One is a case of gastric carcinoma which developed at the same site of previous EMR site for the low grade dysplasia.
  • The other is a case in which persistent EMR-induced ulcer was healed in the long run after Helicobacter pylori eradication therapy.
  • [MeSH-major] Gastric Mucosa / surgery. Stomach Neoplasms / surgery. Stomach Ulcer / etiology
  • [MeSH-minor] Aged. Endoscopy, Gastrointestinal. Helicobacter Infections / complications. Helicobacter Infections / drug therapy. Helicobacter pylori. Humans. Iatrogenic Disease. Male. Middle Aged

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  • (PMID = 18349575.001).
  • [ISSN] 1598-9992
  • [Journal-full-title] The Korean journal of gastroenterology = Taehan Sohwagi Hakhoe chi
  • [ISO-abbreviation] Korean J Gastroenterol
  • [Language] kor
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Korea (South)
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2. Hunt RH, Sumanac K, Huang JQ: Review article: should we kill or should we save Helicobacter pylori? Aliment Pharmacol Ther; 2001 Jun;15 Suppl 1:51-9
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  • Results from epidemiological studies and therapeutic clinical trials have shown that Helicobacter pylori infection causes acute and chronic active gastritis and is the initiating factor for the majority of peptic ulcer disease.
  • Eradication of the infection with antibiotics resolves gastritis and restores normal gastric physiology, accelerates healing of peptic ulcer disease, and virtually eliminates recurrence of duodenal ulcer disease.
  • The infection also plays an important role in the initiation and/or progression of gastric atrophy and intestinal metaplasia, which may eventually lead to the development of distal gastric cancer.
  • Furthermore, almost all patients with gastric MALT lymphoma are infected with H. pylori and cure of the infection leads to histological regression of the tumor and maintains the regression in over 80% of patients during long-term follow-up.
  • Preliminary uncontrolled data from Japan show that eradication of the infection significantly reduced metachronous intestinal-type gastric cancer following initial endoscopic resection of early gastric cancer and might also prevent the progression of gastric adenoma to gastric dysplasia or gastric cancer.
  • Eradication of H. pylori may remove some beneficial bacterial strains and may provoke esophageal disease or gastric cancer at the cardia.
  • [MeSH-major] Helicobacter Infections / complications. Helicobacter Infections / drug therapy. Helicobacter pylori
  • [MeSH-minor] Gastritis / etiology. Gastritis / pathology. Humans. Lymphoma, B-Cell, Marginal Zone / etiology. Lymphoma, B-Cell, Marginal Zone / pathology. Stomach Neoplasms / etiology. Stomach Neoplasms / pathology

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  • (PMID = 11488662.001).
  • [ISSN] 0269-2813
  • [Journal-full-title] Alimentary pharmacology & therapeutics
  • [ISO-abbreviation] Aliment. Pharmacol. Ther.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Number-of-references] 76
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3. Satoh K, Yamamoto H, Kawata H, Osawa H, Hanatsuka K, Kita H, Sunada K, Hirasawa T, Yoshizawa M, Ajibe H, Satoh Y, Sunada F, Sugano K: Comparison of hemostatic effects by route of H2 receptor antagonist administration following endoscopic mucosal resection in patients with neoplastic gastric lesions. Aliment Pharmacol Ther; 2005 Jun;21 Suppl 2:105-10
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  • [Title] Comparison of hemostatic effects by route of H2 receptor antagonist administration following endoscopic mucosal resection in patients with neoplastic gastric lesions.
  • BACKGROUND: To date, there has not been an in-depth investigation to identify differences in the effects of bleeding prevention among different routes of administration of H2 receptor antagonists to treat gastric ulcers following endoscopic mucosal resection (EMR).
  • METHODS: Fifty-three patients with neoplastic gastric lesions (33 carcinoma and 20 adenoma) treated by EMR were included.
  • Patients with odd ID numbers were assigned to IV therapy (30 cases) while even numbers were given PO therapy (23 cases).
  • No significant difference was seen in the endoscopic findings of bleeding and therapy, either, with respective IV and PO findings at 23 and 26%.
  • CONCLUSIONS: No significant difference was observed in frequency of bleeding within 2 days after gastric EMR between IV and oral administrations of famotidine.
  • [MeSH-major] Adenocarcinoma / drug therapy. Famotidine / administration & dosage. Gastrointestinal Hemorrhage / prevention & control. Histamine H2 Antagonists / administration & dosage. Postoperative Hemorrhage / prevention & control. Stomach Neoplasms / drug therapy

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  • (PMID = 15943856.001).
  • [ISSN] 0269-2813
  • [Journal-full-title] Alimentary pharmacology & therapeutics
  • [ISO-abbreviation] Aliment. Pharmacol. Ther.
  • [Language] eng
  • [Publication-type] Clinical Trial; Comparative Study; Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Adjuvants, Immunologic; 0 / Histamine H2 Antagonists; 5QZO15J2Z8 / Famotidine; 9004-61-9 / Hyaluronic Acid
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4. Ito M, Tanaka S, Takata S, Oka S, Imagawa S, Ueda H, Egi Y, Kitadai Y, Yasui W, Yoshihara M, Haruma K, Chayama K: Morphological changes in human gastric tumours after eradication therapy of Helicobacter pylori in a short-term follow-up. Aliment Pharmacol Ther; 2005 Mar 1;21(5):559-66
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  • [Title] Morphological changes in human gastric tumours after eradication therapy of Helicobacter pylori in a short-term follow-up.
  • BACKGROUND: It is controversial as to whether the development of gastric cancer is influenced by Helicobacter pylori eradication.
  • AIM: To investigate the morphological changes in the gastric neoplasm after H. pylori eradication.
  • METHODS: We studied 37 patients with eradication therapy.
  • All lesions were of the superficial-elevated type and the height of the lesions decreased.
  • CONCLUSIONS: The morphology of the gastric neoplasm change after eradication in the short-term.
  • [MeSH-major] Adenocarcinoma / pathology. Adenoma / pathology. Helicobacter Infections / drug therapy. Helicobacter pylori. Stomach Neoplasms / pathology

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  • (PMID = 15740539.001).
  • [ISSN] 0269-2813
  • [Journal-full-title] Alimentary pharmacology & therapeutics
  • [ISO-abbreviation] Aliment. Pharmacol. Ther.
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Gastrins; 0 / Ki-67 Antigen; 9001-10-9 / Pepsinogen A
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5. Miehlke S, Kirsch C, Dragosics B, Gschwantler M, Oberhuber G, Antos D, Dite P, Läuter J, Labenz J, Leodolter A, Malfertheiner P, Neubauer A, Ehninger G, Stolte M, Bayerdörffer E: Helicobacter pylori and gastric cancer:current status of the Austrain Czech German gastric cancer prevention trial (PRISMA Study). World J Gastroenterol; 2001 Apr;7(2):243-7
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  • [Title] Helicobacter pylori and gastric cancer:current status of the Austrain Czech German gastric cancer prevention trial (PRISMA Study).
  • AIM: To test the hypothesis that Helicobacter pylori eradication alone can reduce the incidence of gastric cancer in a subgroup of individuals with an increased risk for this fatal disease.
  • Men between 55 and 65 years of age with a gastric cancer phenotype of Helicobacter pylori gastritis are randomized to receive a 7 day course of omeprazole 2 X 20mg, clarithromycin 2 X 500mg, and amoxicillin 2 X 1g for 7 days, or omeprazole 2 X 20mg plus placebo.
  • Follow-up endoscopy is scheduled 3 months after therapy, and thereafter in one-year intervals.
  • Predefined study endpoints are gastric cancer, precancerous lesions (dysplasia, adenoma), other cancers, and death.
  • RESULTS: Since March 1998, 1524 target patients have been screened, 279 patients (18.3%) had a corpus dominant type of H. pylori gastritis, and 167 of those were randomized (58.8%).
  • In the active treatment group (r = 86), H. pylori infection infection was cured in 88.9% of patients.
  • Currently, the cumulative follow-up time is 3046 months (253.38 patient years, median follow up 16 months).
  • So far, none of the patients developed gastric cancer or any precancerous lesion.
  • Three (1.8%) patients reached study endpoints other than gastric cancer.
  • CONCLUSION: Among men between 55 and 65 years of age, the gastric cancer phenotype of H. pylori gastritis appears to be more common than expected.
  • [MeSH-major] Amoxicillin / therapeutic use. Anti-Bacterial Agents / therapeutic use. Clarithromycin / therapeutic use. Gastritis / drug therapy. Gastrointestinal Agents / therapeutic use. Helicobacter Infections / drug therapy. Helicobacter pylori. Omeprazole / therapeutic use. Stomach Neoplasms / microbiology. Stomach Neoplasms / prevention & control
  • [MeSH-minor] Drug Therapy, Combination. Humans. Male. Middle Aged. Prospective Studies

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  • (PMID = 11819768.001).
  • [ISSN] 1007-9327
  • [Journal-full-title] World journal of gastroenterology
  • [ISO-abbreviation] World J. Gastroenterol.
  • [Language] eng
  • [Publication-type] Clinical Trial; Journal Article; Multicenter Study; Randomized Controlled Trial; Research Support, Non-U.S. Gov't
  • [Publication-country] China
  • [Chemical-registry-number] 0 / Anti-Bacterial Agents; 0 / Gastrointestinal Agents; 804826J2HU / Amoxicillin; H1250JIK0A / Clarithromycin; KG60484QX9 / Omeprazole
  • [Other-IDs] NLM/ PMC4723530
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6. Ramser KL, Sprabery LR, Hamann GL, George CM, Will A: Results of an intervention in an academic Internal Medicine Clinic to continue, step-down, or discontinue proton pump inhibitor therapy related to a tennessee medicaid formulary change. J Manag Care Pharm; 2009 May;15(4):344-50
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  • [Title] Results of an intervention in an academic Internal Medicine Clinic to continue, step-down, or discontinue proton pump inhibitor therapy related to a tennessee medicaid formulary change.
  • In order to obtain an approved PA for a PPI, the patient was required to have either (a) a diagnosis of erosive esophagitis, Barrett's esophagus, Schatzki's ring, a pathological hypersecretory condition (e.g., Zollinger-Ellison syndrome, multiple endocrine adenoma), grade III-IV gastroesophageal reflux disease (GERD), non-steroidal anti-inflammatory drug gastropathy, significant gastrointestinal bleed; or (b) another indication for acid suppression therapy (e.g., GERD, hyperacidity in cystic fibrosis, gastric or duodenal ulcer, gastroparesis) with a history of failure of prior therapy with a histamine-2 receptor antagonist (H2-blocker).
  • OBJECTIVE: To (a) describe the process used by an internal medicine clinic to ensure that patients requiring acid suppression therapy received appropriate treatment according to revised TennCare formulary criteria without unnecessary interruption of therapy, and (b) assess self-reported symptom control 8 months after intervention in the patients who either discontinued therapy or stepped-down to H2-blocker therapy.
  • A clinical pharmacist and an internal medicine physician collaborated to develop a protocol for adjusting acid suppression therapy.
  • A clinical pharmacist reviewed medical records for all patients identified to verify indications for acid suppression therapy and review medication history.
  • Patient telephone interviews were also conducted for patients whose indication or medication history could not be determined by chart review.
  • Patients who met TennCare criteria for PPI therapy were continued on PPI therapy after a PA was obtained (PA group).
  • Patients who had a documented indication for acid suppression therapy but did not meet the PA criteria for PPI therapy were changed to H2-blocker therapy (step-down group).
  • Patients without a documented indication for acid suppression therapy were discontinued from acid suppression therapy (discontinue therapy group).
  • A follow-up chart review and patient telephone interview were conducted 8 months after the intervention for patients in the step-down and discontinue therapy groups.
  • The purpose of this follow-up review was to determine (a) the proportion of patients who were taking acid suppression therapy, (b) the type of acid suppression therapy (PPI or H2-blocker), and (c) self-report of adequate control of symptoms (defined as symptoms once weekly or less).
  • RESULTS: Of 135 TennCare beneficiaries who were active patients of the internal medicine clinic and received a prescription from the outpatient pharmacy for PPI therapy (pantoprazole) between April 20 and June 20, 2005, 6 patients were excluded because they reported stopping PPI therapy on their own.
  • Of the remaining 129 patients, 18 (14.0%) did not have an indication for PPI therapy and acid suppression therapy was discontinued (discontinue therapy group), 40 (31.0%) met the TennCare PA criteria for continuation of PPI therapy (PA group), and 71 (55.0%) did not meet the TennCare PA criteria and were stepped down to a H2-blocker (step-down group).
  • At the 8-month follow-up, acid suppression therapy was assessed in 68 patients (21 patients were lost to follow-up): 13 patients (19.1%) had resumed PPI therapy; 38 (55.9%) were using an H2-blocker; and 17 (25.0%) were not using acid suppression therapy.
  • Telephone interviews were completed for 45 of the 75 patients in the step-down and discontinue therapy groups who did not receive an escalation in acid suppression therapy after the initial intervention (i.e., who did not make a change from H2-blocker therapy to PPI therapy or from no acid suppression therapy to H2-blocker or PPI therapy).
  • CONCLUSIONS: After a proactive collaboration between physicians and clinical pharmacists in response to changes in TennCare formulary criteria for PPIs, more than one-half of patients were stepped down to H2-blocker therapy, and 14% were discontinued from acid suppression therapy.
  • Among the step-down or therapy discontinuation patients for whom data were available at the 8-month follow-up, 81% were still using either an H2-blocker or no acid suppressing therapy at all, and 19% had resumed PPI use.
  • [MeSH-major] Formularies, Hospital. Gastrointestinal Diseases / drug therapy. Health Plan Implementation / statistics & numerical data. Histamine H2 Antagonists / therapeutic use. Medicaid. Practice Patterns, Physicians' / statistics & numerical data. Proton Pump Inhibitors / therapeutic use. Withholding Treatment

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  • (PMID = 19422274.001).
  • [ISSN] 1083-4087
  • [Journal-full-title] Journal of managed care pharmacy : JMCP
  • [ISO-abbreviation] J Manag Care Pharm
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Histamine H2 Antagonists; 0 / Proton Pump Inhibitors
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7. Saito K, Arai K, Mori M, Kobayashi R, Ohki I: Effect of Helicobacter pylori eradication on malignant transformation of gastric adenoma. Gastrointest Endosc; 2000 Jul;52(1):27-32
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  • [Title] Effect of Helicobacter pylori eradication on malignant transformation of gastric adenoma.
  • BACKGROUND: A nonrandomized trial of Helicobacter pylori eradication was conducted in patients with endoscopically diagnosed gastric adenoma to determine the long-term effect of antimicrobial treatment on progression of the adenoma.
  • METHODS: Of 64 patients with an endoscopically diagnosed gastric adenoma and H pylori infection, 32 were treated with omeprazole and antibiotics to eradicate the infection, and 32 were not.
  • RESULTS: During 2 years of follow-up, 4 (12.5%) of the 32 patients in the untreated group developed an early stage, intestinal-type gastric cancer, whereas no gastric cancer was found in the 32 patients in the treated group.
  • CONCLUSION: H Pylori eradication may inhibit progression of gastric adenoma to carcinoma.
  • [MeSH-major] Adenoma / pathology. Amoxicillin / administration & dosage. Cell Transformation, Neoplastic / pathology. Clarithromycin / administration & dosage. Helicobacter Infections / drug therapy. Helicobacter pylori / drug effects. Omeprazole / administration & dosage. Stomach Neoplasms / pathology
  • [MeSH-minor] Aged. Aged, 80 and over. Carcinoma / diagnosis. Carcinoma / epidemiology. Carcinoma / microbiology. Carcinoma / pathology. Drug Therapy, Combination. Female. Gastric Mucosa / pathology. Gastroscopy. Humans. Incidence. Male. Probability. Reference Values. Statistics, Nonparametric. Treatment Outcome

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  • (PMID = 10882958.001).
  • [ISSN] 0016-5107
  • [Journal-full-title] Gastrointestinal endoscopy
  • [ISO-abbreviation] Gastrointest. Endosc.
  • [Language] eng
  • [Publication-type] Clinical Trial; Controlled Clinical Trial; Journal Article
  • [Publication-country] UNITED STATES
  • [Chemical-registry-number] 804826J2HU / Amoxicillin; H1250JIK0A / Clarithromycin; KG60484QX9 / Omeprazole
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8. Yumiba T, Ito T, Ikushima H, Taniguchi E, Inoue Y, Nishida T, Kitagawa T, Nishikawa K, Ohashi S, Matsuda H: Effect of mucosal suture on the healing of mucosal defect in laparoscopic intragastric surgery. Gastric Cancer; 2003;6(2):96-9

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • BACKGROUND: The efficacy of mucosal suturing for the healing of a mucosal defect in laparoscopic intragastric surgery (LIGS) for gastric lesions is not yet known.
  • METHODS: We prospectively studied ten patients who underwent mucosal resection by LIGS for gastric tumors: four patients with early gastric cancer and six with gastric adenoma.
  • The medication cost (H2-blocker) in group I was significantly lower than that in group II.
  • CONCLUSION: Mucosal defect suturing after mucosal resection by LIGS promotes more rapid healing of mucosal defects and reduces drug costs for patients.
  • [MeSH-major] Gastric Mucosa / surgery. Laparoscopy. Suture Techniques. Wound Healing / physiology
  • [MeSH-minor] Adenoma / physiopathology. Adenoma / surgery. Aged. Anti-Ulcer Agents / therapeutic use. Endoscopy, Gastrointestinal. Female. Histamine H2 Antagonists / therapeutic use. Humans. Incidence. Male. Middle Aged. Neoplasm Staging. Postoperative Complications / diagnosis. Postoperative Complications / drug therapy. Postoperative Complications / physiopathology. Prospective Studies. Randomized Controlled Trials as Topic. Stomach Neoplasms / physiopathology. Stomach Neoplasms / surgery. Stomach Ulcer / diagnosis. Stomach Ulcer / drug therapy. Stomach Ulcer / physiopathology. Time Factors. Treatment Outcome

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  • (PMID = 12861400.001).
  • [ISSN] 1436-3291
  • [Journal-full-title] Gastric cancer : official journal of the International Gastric Cancer Association and the Japanese Gastric Cancer Association
  • [ISO-abbreviation] Gastric Cancer
  • [Language] eng
  • [Publication-type] Clinical Trial; Comparative Study; Journal Article; Randomized Controlled Trial
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / Anti-Ulcer Agents; 0 / Histamine H2 Antagonists
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9. Gheorghe C, Iacob R, Dumbrava M, Becheanu G, Ionescu M: Confocal laser endomicroscopy and ultrasound endoscopy during the same endoscopic session for diagnosis and staging of gastric neoplastic lesions. Chirurgia (Bucur); 2009 Jan-Feb;104(1):17-24
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  • [Title] Confocal laser endomicroscopy and ultrasound endoscopy during the same endoscopic session for diagnosis and staging of gastric neoplastic lesions.
  • INTRODUCTION: Confocal LASER endomicroscopy (CLE) is a newly developed endoscopic technique which allows subsurface in vivo histological assessment during ongoing endoscopy and targeted biopsies.
  • We describe for the first time the use of both techniques during the same endoscopic session, in a pilot study, in order to increase the diagnostic yield of histological assessment and provide the staging of the gastric neoplastic lesions thus decreasing the time to therapeutic decision.
  • The indication of CLE/EUS exploration was the presence of a gastric polypoid lesion in 37% of cases, atypical gastric ulcer in 27% of patients, gastric lymphoma 18%, suspicion of gastric cancer recurrence after resection 9% and infiltrating type gastric cancer 9%.
  • Histological assessment after targeted biopsy has established the diagnosis of gastric adenocarcinoma in 55% of cases, gastric lymphoma in 18% of cases, gastric adenoma, gastric GIST and gastric foveolar hyperplasia in 9% of cases respectively.
  • In 2 patients - one case with suspected recurrent gastric cancer after surgery and one case of gastric lymphoma, CLE has indicated normal gastric mucosa.
  • The EUS evaluation showed in one gastric lymphoma patient a lesion interesting the mucosa and submucosa with regional adenopathy and a submucosal lesion with regional adenopathy in the other gastric lymphoma case.
  • The therapeutic decision was surgery in 73% of cases, chemotherapy and follow-up in 18% of cases and follow-up in 9% of cases.
  • CONCLUSION: CLE and EUS can be successfully associated during the same endoscopic session, for upper GI neoplastic lesions allowing targeted biopsies for histological assessment and disease staging for optimal therapeutic decision.
  • [MeSH-major] Endosonography. Microscopy, Confocal. Stomach Neoplasms / pathology. Stomach Neoplasms / ultrasonography
  • [MeSH-minor] Adult. Aged. Aged, 80 and over. Biopsy. Diagnosis, Differential. Female. Gastric Mucosa / pathology. Gastric Mucosa / ultrasonography. Humans. Male. Middle Aged. Neoplasm Staging. Pilot Projects. Reproducibility of Results. Sensitivity and Specificity

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  • (PMID = 19388564.001).
  • [ISSN] 1221-9118
  • [Journal-full-title] Chirurgia (Bucharest, Romania : 1990)
  • [ISO-abbreviation] Chirurgia (Bucur)
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Romania
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10. Abela JE, Wright DM, Hennessy A, Roberts F, Anderson JR: Chemotherapy induced carcinoma-adenoma regression in the caecum. J Clin Pathol; 2009 Mar;62(3):282-3

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  • [Title] Chemotherapy induced carcinoma-adenoma regression in the caecum.
  • The case of a male patient with synchronous oesophago-gastric junction (OGJ) and caecal adenocarcinomas is reported.
  • His management is described, particularly the administration of neoadjuvant chemotherapy targeting his upper gastrointestinal cancer.
  • Histopathological examination of the surgical specimen confirmed the OGJ cancer but only identified a caecal adenoma.
  • This is believed to be the first description of chemotherapy induced reversal of the adenoma-carcinoma sequence.
  • [MeSH-major] Adenocarcinoma / drug therapy. Adenoma / pathology. Cecal Neoplasms / drug therapy. Neoplasms, Multiple Primary / drug therapy
  • [MeSH-minor] Aged. Chemotherapy, Adjuvant. Esophagogastric Junction. Humans. Male. Neoadjuvant Therapy. Remission Induction. Stomach Neoplasms / drug therapy. Stomach Neoplasms / pathology. Stomach Neoplasms / surgery

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  • (PMID = 19251957.001).
  • [ISSN] 1472-4146
  • [Journal-full-title] Journal of clinical pathology
  • [ISO-abbreviation] J. Clin. Pathol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] England
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11. Chang HJ, Oh SN, Park MY, Rha SE, Choi BG: Fraudulent retouching of digital radiographic images--a potential risk. Clin Radiol; 2010 Dec;65(12):967-73

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • MATERIALS AND METHODS: Ten representative key images were selected of aortic dissection, hepatocellular carcinoma, renal cell carcinoma, colon cancer, liver metastasis, hepatic cyst, gallbladder stones, splenic artery aneurysm, adrenal adenoma, and stomach cancer from abdominal computed tomography (CT) imaging performed in 2008.
  • The time to complete retouching was recorded for each image.
  • Radiologists were requested to make a diagnosis for the 10 images, and were then asked to identify possible retouched images.
  • The time taken to reach a decision in each case was recorded.
  • RESULTS: The time to complete retouching was 15.2±3.15 min.
  • None of the reviewers recognized that some images were retouched during diagnosis.
  • The rate of correct diagnosis was 90% (range 71.7-100%).
  • After reviewers were informed of possible image retouching, the detection rate of retouched images was 50% (40-58.3%).
  • There was no significant difference between residents and attending radiologists in the detection rate of retouched images (p=0.786).
  • The time to diagnosis and the time to detection of the retouched images were 15 (14-17) and 6 (5-7) min, respectively.
  • [MeSH-minor] Deception. Fraud. Humans. Insurance Claim Reporting / legislation & jurisprudence. Internship and Residency. Radiology. Republic of Korea. Software. Time Factors. Tomography, X-Ray Computed

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  • [Copyright] Copyright © 2010 The Royal College of Radiologists. Published by Elsevier Ltd. All rights reserved.
  • (PMID = 21070899.001).
  • [ISSN] 1365-229X
  • [Journal-full-title] Clinical radiology
  • [ISO-abbreviation] Clin Radiol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
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12. Cavallaro G, Albanese V, Taranto F, Pustorino S, Baldari S: Brunner's adenoma, esophageal reflux and gastric ulcer. A case report. Chir Ital; 2000 Nov-Dec;52(6):703-6
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  • [Title] Brunner's adenoma, esophageal reflux and gastric ulcer. A case report.
  • In this work the authors report a clinical case of Brunner's adenoma, which was responsible for the onset of other pathologies in the upper gastrointestinal tract, such as gastroesophageal reflux (GER), esophagitis and ulcerations of the antral mucosa.
  • The preoperative diagnostic procedure (endoscopy, esophageal manometry, gastric emptying) and the follow-up at 3, 6 and 12 months from the surgery confirmed the relationship between the Brunner's adenoma and the alterations of the lower esophageal sphyncter (LES) tone and the gastric emptying.
  • [MeSH-minor] Aged. Female. Gastroesophageal Reflux / complications. Gastroesophageal Reflux / drug therapy. Humans. Stomach Ulcer / complications

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  • (PMID = 11200007.001).
  • [ISSN] 0009-4773
  • [Journal-full-title] Chirurgia italiana
  • [ISO-abbreviation] Chir Ital
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Italy
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13. Watanabe Y, Kato N, Maehata T, Okamoto M, Tsuda T, Hattori S, Yamauchi S, Fujita K, Baba S, Nakaya S, Inaba H, Kitajima S, Suzuki M, Niwa H, Itoh F: Safer endoscopic gastric mucosal resection: preoperative proton pump inhibitor administration. J Gastroenterol Hepatol; 2006 Nov;21(11):1675-80
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  • [Title] Safer endoscopic gastric mucosal resection: preoperative proton pump inhibitor administration.
  • Proton pump inhibitors (PPI) are potent suppressors of gastric acid; however, PPI need to be continuously administered orally for several days, and thus initial effects may be insufficient if PPI is only administered immediately after EMR.
  • [MeSH-major] Adenoma / surgery. Anti-Ulcer Agents / administration & dosage. Gastric Mucosa / surgery. Gastroscopy. Hemostasis, Surgical / methods. Preoperative Care. Proton Pump Inhibitors. Stomach Neoplasms / surgery
  • [MeSH-minor] Administration, Oral. Aged. Blood Loss, Surgical / prevention & control. Breath Tests. Female. Humans. Hydrogen-Ion Concentration. Male. Postoperative Hemorrhage / prevention & control. Stomach Ulcer / drug therapy. Treatment Outcome

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  • (PMID = 16984588.001).
  • [ISSN] 0815-9319
  • [Journal-full-title] Journal of gastroenterology and hepatology
  • [ISO-abbreviation] J. Gastroenterol. Hepatol.
  • [Language] eng
  • [Publication-type] Journal Article; Randomized Controlled Trial
  • [Publication-country] Australia
  • [Chemical-registry-number] 0 / Anti-Ulcer Agents; 0 / Proton Pump Inhibitors
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14. Takhar AS, Eremin O, Watson SA: The role of gastrin in colorectal carcinogenesis. Surgeon; 2004 Oct;2(5):251-7
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  • Currently, treatment options for this debilitating disease are limited and surgical resection is the only curative treatment available.
  • Despite rapid advances in surgery, as well as in adjuvant therapies such as radiotherapy and chemotherapy, there has been only a relatively modest improvement in mortality.
  • The majority of colorectal cancers are epithelial-derived adenocarcinomas and arise from benign adenomas through the gain of mutations in key genes.
  • Gastrin, an important polypeptide hormone, responsible for gastric acid secretion has been found to be involved in tumourigenesis in the gastrointestinal tract.
  • In colonic tumourigenesis, gene expression of both gastrin and the gastrin/CCK-2 receptor is activated within epithelial cells at an early stage of the adenoma-carcinoma sequence.
  • This review details the role played by gastrin in the adenoma-carcinoma sequence of colorectal carcinogenesis.

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  • (PMID = 15570843.001).
  • [ISSN] 1479-666X
  • [Journal-full-title] The surgeon : journal of the Royal Colleges of Surgeons of Edinburgh and Ireland
  • [ISO-abbreviation] Surgeon
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Scotland
  • [Chemical-registry-number] 0 / Gastrins; 9011-97-6 / Cholecystokinin
  • [Number-of-references] 58
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15. Uesato M, Nabeya Y, Akai T, Inoue M, Watanabe Y, Kawahira H, Mamiya T, Ohta Y, Motojima R, Kagaya A, Muto Y, Hayashi H, Matsubara H: Salivary amylase activity is useful for assessing perioperative stress in response to pain in patients undergoing endoscopic submucosal dissection of gastric tumors under deep sedation. Gastric Cancer; 2010 Jun;13(2):84-9
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  • [Title] Salivary amylase activity is useful for assessing perioperative stress in response to pain in patients undergoing endoscopic submucosal dissection of gastric tumors under deep sedation.
  • BACKGROUND: Although endoscopic submucosal dissection (ESD) for patients with gastric tumors under the conditions of unconsciousness is considered to be minimally invasive, no objective assessment of the perioperative stress of ESD has yet been conducted.
  • METHODS: A total of 40 patients with gastric cancers/adenomas removed by ESD under general anesthesia (GA; n = 20) and under deep sedation (DS; n = 20) were enrolled. sAMY was measured using the enzyme analysis equipment, sAMY Monitor (NIPRO, Osaka, Japan) during the perioperative period of the ESD.
  • [MeSH-major] Amylases / metabolism. Saliva / enzymology. Stomach Neoplasms / surgery. Stress, Physiological
  • [MeSH-minor] Adenoma / pathology. Adenoma / surgery. Aged. Analgesics / therapeutic use. Anesthesia, General. Female. Gastric Mucosa / surgery. Gastroscopy / adverse effects. Humans. Male. Middle Aged. Pain / drug therapy. Pain / enzymology. Pain / etiology. Perioperative Care / methods. Surveys and Questionnaires

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  • [Cites] Anesthesiology. 2002 Apr;96(4):1004-17 [11964611.001]
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  • (PMID = 20602194.001).
  • [ISSN] 1436-3305
  • [Journal-full-title] Gastric cancer : official journal of the International Gastric Cancer Association and the Japanese Gastric Cancer Association
  • [ISO-abbreviation] Gastric Cancer
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / Analgesics; EC 3.2.1.- / Amylases
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16. Lee SY, Kim JJ, Lee JH, Kim YH, Rhee PL, Paik SW, Rhee JC: Healing rate of EMR-induced ulcer in relation to the duration of treatment with omeprazole. Gastrointest Endosc; 2004 Aug;60(2):213-7
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  • [Title] Healing rate of EMR-induced ulcer in relation to the duration of treatment with omeprazole.
  • BACKGROUND: Although EMR-induced ulcers heal faster and recur less often than noniatrogenic gastric ulcers, there is no consensus regarding the duration of therapy for these ulcers.
  • This study prospectively evaluated healing of EMR-induced ulcers according to the duration of omeprazole therapy.
  • METHODS: A total of 69 patients were randomly assigned, after EMR, to treatment with omeprazole (20 mg per day) for 7 days (1-week group) or with omeprazole (20 mg per day) for 28 days (4-week group).
  • Each patient kept a daily diary of drugs consumed and ulcer-related symptoms during the 4-week period after EMR.
  • In addition, no difference was observed between the two groups with respect to ulcer-related symptoms or use of additional gastric-coating medication (p=0.48).
  • CONCLUSIONS: For EMR-induced ulcer, treatment with omeprazole for 1 week is equivalent to treatment for 4 weeks.
  • Short-term therapy with omeprazole can be considered for EMR-induced ulcer.
  • [MeSH-major] Anti-Ulcer Agents / administration & dosage. Endoscopy, Gastrointestinal / adverse effects. Omeprazole / administration & dosage. Stomach Ulcer / drug therapy
  • [MeSH-minor] Adenoma / surgery. Aged. Female. Humans. Male. Prospective Studies. Stomach Neoplasms / surgery. Time Factors. Wound Healing / drug effects

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  • (PMID = 15278047.001).
  • [ISSN] 0016-5107
  • [Journal-full-title] Gastrointestinal endoscopy
  • [ISO-abbreviation] Gastrointest. Endosc.
  • [Language] eng
  • [Publication-type] Clinical Trial; Journal Article; Randomized Controlled Trial
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Anti-Ulcer Agents; KG60484QX9 / Omeprazole
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17. Yamagishi M, Natsume M, Osakabe N, Nakamura H, Furukawa F, Imazawa T, Nishikawa A, Hirose M: Effects of cacao liquor proanthocyanidins on PhIP-induced mutagenesis in vitro, and in vivo mammary and pancreatic tumorigenesis in female Sprague-Dawley rats. Cancer Lett; 2002 Nov 28;185(2):123-30
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  • In the Ames assay using Salmonella typhimurium TA98, CLPr showed strong antimutagenic effects against PhIP when assayed in the presence of S-9 mixture.
  • For determination of the influence on initiation and subsequent development of lesions, CLPr (0.025% or 0.25%) were fed during the period of PhIP application (100 mg/kg given to rats via gastric tubes eight times over 4 weeks), or thereafter until the termination at 48 weeks.
  • CLPr treatments did not affect body or organ weights.
  • [MeSH-major] Adenocarcinoma / prevention & control. Adenoma / drug therapy. Anthocyanins / pharmacology. Anticarcinogenic Agents / pharmacology. Antineoplastic Agents, Phytogenic / therapeutic use. Cacao / chemistry. Flavonoids. Mammary Neoplasms, Experimental / prevention & control. Mutagenesis / drug effects. Pancreatic Neoplasms / prevention & control. Phytotherapy. Plant Extracts / pharmacology. Proanthocyanidins
  • [MeSH-minor] Animals. Biotransformation. Carcinogens / toxicity. Catechin / isolation & purification. Catechin / pharmacology. Catechin / therapeutic use. Dose-Response Relationship, Drug. Drug Screening Assays, Antitumor. Female. Imidazoles / toxicity. Microsomes, Liver / metabolism. Mutagenicity Tests. Organ Specificity. Phenols / isolation & purification. Phenols / pharmacology. Phenols / therapeutic use. Polymers / isolation & purification. Polymers / pharmacology. Polymers / therapeutic use. Rats. Rats, Sprague-Dawley. Salmonella typhimurium / drug effects

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  • (PMID = 12169385.001).
  • [ISSN] 0304-3835
  • [Journal-full-title] Cancer letters
  • [ISO-abbreviation] Cancer Lett.
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article
  • [Publication-country] Ireland
  • [Chemical-registry-number] 0 / Anthocyanins; 0 / Anticarcinogenic Agents; 0 / Antineoplastic Agents, Phytogenic; 0 / Carcinogens; 0 / Flavonoids; 0 / Imidazoles; 0 / Phenols; 0 / Plant Extracts; 0 / Polymers; 0 / Proanthocyanidins; 18206-61-6 / proanthocyanidin; 8R1V1STN48 / Catechin; 909C6UN66T / 2-amino-1-methyl-6-phenylimidazo(4,5-b)pyridine
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18. Reichart M, Busch OR, Bruno MJ, Van Lanschot JJ: Black esophagus: a view in the dark. Dis Esophagus; 2000;13(4):311-3
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  • A 73-year-old man had a low anterior resection for a villous adenoma in the rectosigmoid.
  • On the 4th day after surgery, he suddenly developed severe interscapular pain.
  • With conservative treatment, including proton pump inhibition, he recovered completely.
  • We hypothesize that a transient gastric outlet obstruction and massive gastroesophageal reflux played a significant role in the etiology of this rare and alarming, but, in this case, completely reversible, syndrome.

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  • (PMID = 11284980.001).
  • [ISSN] 1120-8694
  • [Journal-full-title] Diseases of the esophagus : official journal of the International Society for Diseases of the Esophagus
  • [ISO-abbreviation] Dis. Esophagus
  • [Language] ENG
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Anti-Ulcer Agents; 0 / Proton Pump Inhibitors; 54182-58-0 / Sucralfate; KG60484QX9 / Omeprazole
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19. Oh TH, Jung HY, Choi KD, Lee GH, Song HJ, Choi KS, Chung JW, Byeon JS, Myung SJ, Yang SK, Kim JH: Degree of healing and healing-associated factors of endoscopic submucosal dissection-induced ulcers after pantoprazole therapy for 4 weeks. Dig Dis Sci; 2009 Jul;54(7):1494-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Degree of healing and healing-associated factors of endoscopic submucosal dissection-induced ulcers after pantoprazole therapy for 4 weeks.
  • BACKGROUND: There is no consensus regarding the degree of healing of endoscopic submucosal dissection (ESD)-induced ulcers and the optimal duration of proton pump inhibitor (PPI) treatment.
  • AIM: To evaluate the degree of healing and the factors associated with healing of ESD-induced ulcers after PPI therapy for 4 weeks.
  • METHODS: Fifty-six patients who underwent complete ESD for adenoma or early gastric cancer were enrolled.
  • All patients underwent follow-up endoscopy to evaluate the degree of ulcer healing after pantoprazole therapy (40 mg per day) for 4 weeks.
  • Comorbidity, procedure time, and initial specimen size were significantly associated with healing rate, but multivariate analysis showed that initial ulcer size was the only significant parameter (P < 0.015).
  • CONCLUSIONS: Healing degree of ESD-induced ulcers at 4 weeks is dependent on initial ulcer size, indicating that duration of treatment with PPI should be dependent on initial ulcer size.
  • [MeSH-major] 2-Pyridinylmethylsulfinylbenzimidazoles / therapeutic use. Adenoma / surgery. Anti-Ulcer Agents / therapeutic use. Endoscopy, Gastrointestinal / adverse effects. Proton Pump Inhibitors / therapeutic use. Stomach Neoplasms / surgery. Stomach Ulcer / drug therapy. Wound Healing / drug effects

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  • (PMID = 19005762.001).
  • [ISSN] 1573-2568
  • [Journal-full-title] Digestive diseases and sciences
  • [ISO-abbreviation] Dig. Dis. Sci.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / 2-Pyridinylmethylsulfinylbenzimidazoles; 0 / Anti-Ulcer Agents; 0 / Proton Pump Inhibitors; D8TST4O562 / pantoprazole
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