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1. Yuca K, Cankaya H, Bayram I, Ozbek H, Kiris M: Local irritant effects of topical oral sprays on oral mucosa in mice. Adv Ther; 2006 Jan-Feb;23(1):98-106
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Local irritant effects of topical oral sprays on oral mucosa in mice.
  • Topical oral sprays are frequently used to prevent and manage oropharyngeal inflammation and lesions.
  • This study investigated the histopathologic changes noted in the oral mucosa of mice after topical application of 3 widely prescribed antibacterial products.
  • The 25 animals were divided into 5 groups and treated for 10 days with 2 sprays daily, as follows: group 1-chlorhexidine gluconate 0.12% + benzydamine hydrochloride 0.15%; group 2-benzydamine 0.27 mg/0.18 mL x 30 mL; group 3-chlorhexidine 0.2%; group 4-fusafungine 1%; and group 5 (cohort)-physiologic serum.
  • On day 10 after drug administration, biopsy specimens were taken from the oropharyngeal mucosa of the tongue, the cheek mucosa, and the tongue base; these were examined under a light microscope and were classified as normal or pathologic.
  • All topical oral sprays produced some degree of histopathologic change, such as hyperplasia, fibrosis, low-grade dysplasia, congestion, or edema.
  • The local irritant effects of topical oral sprays should be considered when treatment is selected for patients with oropharyngeal disorder.
  • [MeSH-major] Anti-Infective Agents, Local / adverse effects. Mouth Mucosa / drug effects
  • [MeSH-minor] Administration, Oral. Administration, Topical. Aerosols. Animals. Edema / chemically induced. Fibrosis. Hyperplasia. Male. Mice

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  • (PMID = 16644611.001).
  • [ISSN] 0741-238X
  • [Journal-full-title] Advances in therapy
  • [ISO-abbreviation] Adv Ther
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Aerosols; 0 / Anti-Infective Agents, Local
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2. Borrie F, Musthyala R, Macintyre D: Ectopic geographic tongue--a case report. Dent Update; 2007 Mar;34(2):121-2
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  • This report describes a case of 'ectopic' geographic tongue where lesions, clinically and histologically similar to those normally confined to the tongue, presented on the floor of the mouth and cheek mucosa.
  • The patient, a type II diabetic, had a history of thrombocytopaenia and, more recently, autoimmune haemolytic anaemia.
  • CLINICAL RELEVANCE: Geographic tongue is a common oral medicine condition and the purpose of this paper is to highlight the fact that it can present ectopically on the oral mucosa.
  • [MeSH-minor] Diagnosis, Differential. Female. Humans. Middle Aged. Mouth Mucosa / pathology

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  • (PMID = 17432777.001).
  • [ISSN] 0305-5000
  • [Journal-full-title] Dental update
  • [ISO-abbreviation] Dent Update
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] England
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3. Kupfer DM, White VL, Jenkins MC, Burian D: Examining smoking-induced differential gene expression changes in buccal mucosa. BMC Med Genomics; 2010;3:24
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  • [Title] Examining smoking-induced differential gene expression changes in buccal mucosa.
  • BACKGROUND: Gene expression changes resulting from conditions such as disease, environmental stimuli, and drug use, can be monitored in the blood.
  • Buccal mucosa cells are easily collected and may be an alternative sample material for biomarker testing.
  • A limited number of studies, primarily in the smoker/oral cancer literature, address this tissue's efficacy as an RNA source for expression analysis.
  • The current study was undertaken to determine if total RNA isolated from buccal mucosa could be used as an alternative tissue source to assay relative gene expression.
  • The amplified cDNA was used in RT-qPCR and microarray analyses to evaluate gene expression in buccal cells.
  • Initially, RT-qPCR was used to assess relative transcript levels of four genes from whole blood and buccal cells collected from the same seven individuals, concurrently.
  • Second, buccal cell RNA was used for microarray-based differential gene expression studies by comparing gene expression between a group of female smokers and nonsmokers.
  • RESULTS: An amplification protocol allowed use of less buccal cell total RNA (50 ng) than had been reported previously with human microarrays.
  • Total RNA isolated from buccal cells was degraded but was of sufficient quality to be used with RT-qPCR to detect expression of specific genes.
  • We report here the finding of a small number of statistically significant differentially expressed genes between smokers and nonsmokers, using buccal cells as starting material.
  • CONCLUSIONS: Our results suggest that despite a high degree of degradation, RNA from buccal cells from cheek mucosa could be used to detect differential gene expression between smokers and nonsmokers.
  • However the RNA degradation, increase in sample variability and microarray failure rate show that buccal samples should be used with caution as source material in expression studies.
  • [MeSH-major] Gene Expression Profiling. Mouth Mucosa / metabolism. Smoking

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  • (PMID = 20576139.001).
  • [ISSN] 1755-8794
  • [Journal-full-title] BMC medical genomics
  • [ISO-abbreviation] BMC Med Genomics
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 63231-63-0 / RNA
  • [Other-IDs] NLM/ PMC3161387
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4. Martins RA, Gomes GA, Aguiar O Jr, Ribeiro DA: Biomonitoring of oral epithelial cells in petrol station attendants: comparison between buccal mucosa and lateral border of the tongue. Environ Int; 2009 Oct;35(7):1062-5
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  • [Title] Biomonitoring of oral epithelial cells in petrol station attendants: comparison between buccal mucosa and lateral border of the tongue.
  • The aim of the present study was to comparatively evaluate DNA damage (micronucleus) and cellular death (pyknosis, karyolysis and karyorrhexis) in exfoliated oral mucosa cells from gas petrol attendants using two different anatomic buccal sites: cheek mucosa and lateral border of the tongue.
  • Individuals had epithelial cells from cheek and lateral border of the tongue mechanically exfoliated, placed in fixative and dropped in clean slides which were checked for the above nuclear phenotypes.
  • The results pointed out significant statistical differences (p<0.05) of micronucleated oral mucosa cells from gas petrol attendants for both oral sites evaluated.
  • [MeSH-major] Carcinogens, Environmental / toxicity. Gasoline / toxicity. Mouth Mucosa / drug effects. Occupational Exposure. Tongue / drug effects
  • [MeSH-minor] Adult. Cell Death. DNA Damage. Environmental Monitoring. Epithelial Cells / drug effects. Female. Humans. Male. Micronuclei, Chromosome-Defective. Micronucleus Tests. Middle Aged

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  • (PMID = 19559482.001).
  • [ISSN] 1873-6750
  • [Journal-full-title] Environment international
  • [ISO-abbreviation] Environ Int
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Carcinogens, Environmental; 0 / Gasoline
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5. Onishi H, Sakata O, Masuda K, Machida Y: Novel mucoadhesive oral patch containing diazepam. Drug Dev Ind Pharm; 2005 Aug;31(7):607-13
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  • [Title] Novel mucoadhesive oral patch containing diazepam.
  • The oral patch of diazepam (DZ) was developed to achieve a rapid absorption of DZ for the emergency treatment of epileptic seizure or anxiety disorder.
  • The patch was composed of the outer mucoadhesive Carbopol 934 region, central drug region, and Tegaderm backing film.
  • DZ (3 mg) was dissolved in propylene glycol (PG) alone or PG containing oleic acid (OA) at 5.6% (w/w), and used as the drug region.
  • The patches with and without OA were attached to the mucosa of cheek in rats.
  • To the contrary, in the in vitro drug permeation using a cellulose membrane, the patch without OA showed a three times faster permeation rate than the patch with OA, suggesting that the direct action of OA to mucosa might be associated with absorption enhancement.
  • It was demonstrated that the patch with OA showed a good adhesion to oral mucosa and worked efficiently for rapid absorption of DZ.
  • [MeSH-major] Diazepam / administration & dosage. Drug Delivery Systems. Mouth Mucosa / metabolism. Oleic Acid / pharmacology
  • [MeSH-minor] Absorption / drug effects. Acrylates / chemistry. Adhesiveness. Animals. Anti-Anxiety Agents / administration & dosage. Anti-Anxiety Agents / blood. Anti-Anxiety Agents / pharmacokinetics. Anticonvulsants / administration & dosage. Anticonvulsants / blood. Anticonvulsants / pharmacokinetics. Cellulose. Cheek. In Vitro Techniques. Male. Membranes, Artificial. Propylene Glycol / chemistry. Rats. Rats, Wistar

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  • (PMID = 16207607.001).
  • [ISSN] 0363-9045
  • [Journal-full-title] Drug development and industrial pharmacy
  • [ISO-abbreviation] Drug Dev Ind Pharm
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Acrylates; 0 / Anti-Anxiety Agents; 0 / Anticonvulsants; 0 / Membranes, Artificial; 0 / carbopol 934P; 2UMI9U37CP / Oleic Acid; 6DC9Q167V3 / Propylene Glycol; 9004-34-6 / Cellulose; Q3JTX2Q7TU / Diazepam
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6. Liu S, Liao C, Wang D: [The clinical application and evaluation of combined chemotherapy in comprehensive treatment for oral squamous cell carcinoma]. Hua Xi Kou Qiang Yi Xue Za Zhi; 2003 Apr 20;21(2):109-11
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  • [Title] [The clinical application and evaluation of combined chemotherapy in comprehensive treatment for oral squamous cell carcinoma].
  • OBJECTIVE: To study and evaluate the clinical effects of combined preoperative chemotherapy and their relations with multi drug resistance (MDR).
  • METHODS: 102 cases with oral squamous cell carcinoma(OSCC) were included in the study (63 males and 39 females, aged 22 to 67 years).
  • Among the subjects there were 57 cases with cancer of tongue and 45 cases with cancer of buccal mucosa.
  • 27 cases in the group were classified as stage II, 55 as stage III and 20 cases as stage IV according to TNM standard.
  • The diagnosis of all cases were proved as OSCC by biopsy.

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  • (PMID = 12838692.001).
  • [ISSN] 1000-1182
  • [Journal-full-title] Hua xi kou qiang yi xue za zhi = Huaxi kouqiang yixue zazhi = West China journal of stomatology
  • [ISO-abbreviation] Hua Xi Kou Qiang Yi Xue Za Zhi
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] China
  • [Chemical-registry-number] 11056-06-7 / Bleomycin; 77108-50-0 / zhengguangmycin; Q20Q21Q62J / Cisplatin; U3P01618RT / Fluorouracil
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7. Meyskens FL: Development of Bowman-Birk inhibitor for chemoprevention of oral head and neck cancer. Ann N Y Acad Sci; 2001 Dec;952:116-23
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  • [Title] Development of Bowman-Birk inhibitor for chemoprevention of oral head and neck cancer.
  • Leukoplakia in the oral cavity has been used as a putative surrogate marker of head and neck cancer development.
  • Bowman-Birk inhibitor (BBI) is a protease inhibitor derived from soybeans that has demonstrated chemoprevention activity in many in vitro and animal systems, including the hamster cheek pouch model.
  • Pilot, Phase I and Phase IIa studies of Bowman-Birk Inhibitor in patients with oral leukoplakia have demonstrated no detectable side effects.
  • In the Phase IIa trial, changes in the protease activity in oral mucosal cells after BBI Concentratec (BBIC) treatment correlated with the changes in neu protein levels.
  • Additionally, evidence for a dose-related treatment effect of BBIC on oral leukoplakia was demonstrated.
  • [MeSH-major] Anticarcinogenic Agents / therapeutic use. Leukoplakia, Oral / drug therapy. Mouth Neoplasms / prevention & control. Trypsin Inhibitor, Bowman-Birk Soybean / therapeutic use
  • [MeSH-minor] Adult. Amino Acid Sequence. Biomarkers, Tumor. Clinical Trials, Phase II as Topic. Female. Forecasting. Genes, erbB-2. Humans. Male. Molecular Sequence Data. Mouth Mucosa / drug effects. Mouth Mucosa / enzymology. Neoplasm Proteins / analysis. Pregnancy. Receptor, ErbB-2 / analysis. Smoking / metabolism. Treatment Outcome

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  • (PMID = 11795430.001).
  • [ISSN] 0077-8923
  • [Journal-full-title] Annals of the New York Academy of Sciences
  • [ISO-abbreviation] Ann. N. Y. Acad. Sci.
  • [Language] eng
  • [Grant] United States / NCI NIH HHS / CA / CA62203; United States / NCI NIH HHS / CA / CA72294
  • [Publication-type] Journal Article; Research Support, U.S. Gov't, P.H.S.; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Anticarcinogenic Agents; 0 / Biomarkers, Tumor; 0 / Neoplasm Proteins; 0 / Trypsin Inhibitor, Bowman-Birk Soybean; EC 2.7.10.1 / Receptor, ErbB-2
  • [Number-of-references] 11
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8. Casto BC, Kresty LA, Kraly CL, Pearl DK, Knobloch TJ, Schut HA, Stoner GD, Mallery SR, Weghorst CM: Chemoprevention of oral cancer by black raspberries. Anticancer Res; 2002 Nov-Dec;22(6C):4005-15
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  • [Title] Chemoprevention of oral cancer by black raspberries.
  • Oral cavity cancers represent 2.5% of the cancers that occur in the United States and are ranked sixth worldwide.
  • In the study reported herein, the hamster cheek pouch (HCP) was used to evaluate the ability of black raspberries to inhibit oral cavity tumors.
  • The animals were sacrificed 12-13 weeks from the beginning of DMBA treatment and the number and volume of tumors (mm3) determined.
  • There was a significant difference (p = 0.02) in the number of tumors between the 5% LBR and control groups (27 tumors/14 animals and 48 tumors/15 animals, respectively) and an intermediate number of tumors in the 10% berry-treated animals (39 tumors/15 animals).
  • These experiments support previous studies from our laboratories showing the chemopreventive activity of black raspberries and show, for the first time, that dietary black raspberries will inhibit tumor formation in the oral cavity.

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  • (PMID = 12553025.001).
  • [ISSN] 0250-7005
  • [Journal-full-title] Anticancer research
  • [ISO-abbreviation] Anticancer Res.
  • [Language] ENG
  • [Grant] United States / NIDCR NIH HHS / DE / P01 DE012704; United States / NIDCR NIH HHS / DE / P01 DE12704; United States / NCI NIH HHS / CA / P30 5CA16058; United States / NIDCR NIH HHS / DE / R01 DE 11943
  • [Publication-type] Journal Article; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] Greece
  • [Chemical-registry-number] 0 / Carcinogens; 0 / DNA Adducts; 0 / Nitrosamines; 3417WMA06D / Benzo(a)pyrene; 57-97-6 / 9,10-Dimethyl-1,2-benzanthracene; 64091-91-4 / 4-(N-methyl-N-nitrosamino)-1-(3-pyridyl)-1-butanone
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9. Zorzetto DL, Garcia PJ, Toledo Filho JL, Zorzetto NL: Ultrastructural study of the cheek oral mucosa of rats submitted to experimental chronic alcoholism. J Submicrosc Cytol Pathol; 2002 Jul;34(3):345-53
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  • [Title] Ultrastructural study of the cheek oral mucosa of rats submitted to experimental chronic alcoholism.
  • Ultrastructural features of the cheek oral mucosa of rats (Rattus norvegicus) submitted to experimental chronic alcoholism were studied by transmission electron microscopy.
  • The animals of the two groups were weighed and sacrificed after 60, 120, and 180 days of treatment.
  • Samples of the oral mucosa from the cheek region were dissected and processed for ultrastructural analysis.
  • The most frequent alterations observed were an increased intercellular space, the presence of lipid droplets in the cytoplasm, and irregular nuclei with a pyknotic aspect.
  • [MeSH-major] Alcoholism / pathology. Mouth Mucosa / drug effects. Mouth Mucosa / ultrastructure
  • [MeSH-minor] Animals. Cell Nucleus / drug effects. Cell Nucleus / ultrastructure. Cheek / pathology. Cytoplasm / drug effects. Cytoplasm / ultrastructure. Desmosomes / drug effects. Desmosomes / ultrastructure. Disease Models, Animal. Lipids. Male. Microscopy, Electron. Rats

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  • (PMID = 12408369.001).
  • [ISSN] 1122-9497
  • [Journal-full-title] Journal of submicroscopic cytology and pathology
  • [ISO-abbreviation] J. Submicrosc. Cytol. Pathol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Italy
  • [Chemical-registry-number] 0 / Lipids
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10. Mahalingam R, Ravivarapu H, Redkar S, Li X, Jasti BR: Transbuccal delivery of 5-aza-2 -deoxycytidine: effects of drug concentration, buffer solution, and bile salts on permeation. AAPS PharmSciTech; 2007;8(3):E55
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  • [Title] Transbuccal delivery of 5-aza-2 -deoxycytidine: effects of drug concentration, buffer solution, and bile salts on permeation.
  • Delivery of 5-aza-2 -deoxycytidine (decitabine) across porcine buccal mucosa was evaluated as an alternative to the complex intravenous infusion regimen currently used to administer the drug.
  • A reproducible high-performance liquid chromatography method was developed and optimized for the quantitative determination of this drug.
  • Decitabine showed a concentration-dependent passive diffusion process across porcine buccal mucosa.
  • [MeSH-major] Azacitidine / analogs & derivatives. Bile Acids and Salts / pharmacology. Mouth Mucosa / metabolism
  • [MeSH-minor] Animals. Buffers. Cheek. Chromatography, High Pressure Liquid. Osmolar Concentration. Permeability. Swine

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  • (PMID = 17915805.001).
  • [ISSN] 1530-9932
  • [Journal-full-title] AAPS PharmSciTech
  • [ISO-abbreviation] AAPS PharmSciTech
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Bile Acids and Salts; 0 / Buffers; 776B62CQ27 / decitabine; M801H13NRU / Azacitidine
  • [Other-IDs] NLM/ PMC2750551
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11. Wang Z, Polavaram R, Fuentes CF, Shapshay SM: Topical chemoprevention of oral cancer with tretinoin "biofilm". Arch Otolaryngol Head Neck Surg; 2003 Aug;129(8):869-73
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  • [Title] Topical chemoprevention of oral cancer with tretinoin "biofilm".
  • BACKGROUND: Oral cancer is a common malignancy.
  • Topical application of chemopreventive agents is an attractive alternative that reduces toxic effects.
  • This study is based on the hypothesis that topical application of mucosal adhesive film (MAF), as a means to deliver tretinoin, is effective and safe for oral cancer chemoprevention.
  • SETTING: Randomized animal study conducted at the Boston University School of Medicine.
  • DESIGN: This study uses the hamster cheek-pouch model to test efficacy and safety of the MAF/tretinoin patch for oral cancer prevention.
  • The oral mucosa of 36 hamsters was painted with dimethylbenzanthracene to produce premalignant lesions.
  • MAIN OUTCOME MEASURES: Tumor growth and burden were measured over time.
  • The duration of MAF patch retention on mucosa and local tissue reaction to the treatment were also evaluated.
  • RESULTS: The patch stayed on the mucosa for at least 5 hours with no evidence of inflammatory or other adverse reactions from the treated tissue.
  • There was a significant difference in the tumor growth measurement between the control and systemic tretinoin groups (P<.001), and between the control and MAF patch groups (P<.001).
  • CONCLUSIONS: This is the first study, to our knowledge, to use a polymer MAF technique for oral cancer prevention.
  • [MeSH-major] Antineoplastic Agents / administration & dosage. Mouth Neoplasms / prevention & control. Tretinoin / administration & dosage
  • [MeSH-minor] Administration, Topical. Animals. Cricetinae. Male. Mouth Mucosa / drug effects

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  • (PMID = 12925347.001).
  • [ISSN] 0886-4470
  • [Journal-full-title] Archives of otolaryngology--head & neck surgery
  • [ISO-abbreviation] Arch. Otolaryngol. Head Neck Surg.
  • [Language] eng
  • [Grant] United States / NCI NIH HHS / CA / CA 91324; United States / NCI NIH HHS / CA / CA 96509
  • [Publication-type] Journal Article; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antineoplastic Agents; 5688UTC01R / Tretinoin
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12. Ciolino LA, McCauley HA, Fraser DB, Wolnik KA: The relative buffering capacities of saliva and moist snuff: implications for nicotine absorption. J Anal Toxicol; 2001 Jan-Feb;25(1):15-25
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  • Commercial moist snuff products are used by placing a portion of tobacco inside the mouth between the inner cheek or lip and gum.
  • Nicotine is absorbed into the blood stream via transfer across various oral membranes including the buccal mucosa (cheek lining).
  • The resulting salivary pH when a given moist snuff product is placed in the mouth is an important factor for nicotine absorption because it will affect the proportion of free base nicotine that is readily available for absorption.
  • The resulting salivary pH for a given moist snuff product will be determined in part by the relative acid-base buffering capacities of the saliva and moist snuff, as well as the pHs of the saliva and moist snuff prior to coming in contact with one another.
  • In the current study, the acid-base buffering capacities (mu eq/g) of a series of commercial moist snuff products were determined and compared to the acid-base buffering capacity for unstimulated, whole human saliva.
  • The buffering capacities of the moist snuff products were determined to be 10-20 times higher than the buffering capacity of human saliva.
  • The resulting salivary pH ranges after contact between an artifical saliva and the various moist snuff products were also determined; the results were used to predict the proportion of free base nicotine that can be expected to occur in the mouth during the first few minutes of product use.
  • These studies provide a basis for examining and understanding the effects that moist snuff product pHs and buffering capacities may be expected to have on nicotine absorption.

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  • (PMID = 11215994.001).
  • [ISSN] 0146-4760
  • [Journal-full-title] Journal of analytical toxicology
  • [ISO-abbreviation] J Anal Toxicol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Buffers; 6M3C89ZY6R / Nicotine
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13. Clarke J, Butler R, Howarth G, Read L, Regester G: Exposure of oral mucosa to bioactive milk factors reduces severity of chemotherapy-induced mucositis in the hamster. Oral Oncol; 2002 Jul;38(5):478-85
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  • [Title] Exposure of oral mucosa to bioactive milk factors reduces severity of chemotherapy-induced mucositis in the hamster.
  • A biologically active extract containing bovine whey proteins, whey growth factor extract-A (WGFE-A) was administered topically to the oral mucosa of hamsters and its ability to prevent and treat chemotherapy-induced oral mucositis investigated.
  • Oral mucositis was induced in Syrian golden hamsters through a combination treatment of the antimetabolite chemotherapy drug 5-fluorouracil (5-FU), and mild abrasion of the cheek pouch.
  • WGFE-A administered to the oral mucosa via hydrogel and liquid treatments, pre and concurrent to 5-FU therapy, resulted in significantly reduced mucosal ulceration.
  • In a separate study, cell cycle staining indicated that cheek pouch mucosal epithelial cells pre-exposed to WGFE-A in-vivo showed a reduced rate of proliferation, measured as a 21% reduction in the bromodeoxyuridine (BrdU) cell labelling index (P<0.04).
  • Several WGFE-A constituents are likely to confer protective effects on the cheek mucosa, including anti-proliferative, anti-apoptotic and anti-microbial factors.
  • WGFE-A provides a potentially valuable source of topically delivered proteins for clinical application in preventing severe oral mucositis caused by chemotherapy.
  • [MeSH-major] Antimetabolites, Antineoplastic / adverse effects. Fluorouracil / adverse effects. Milk Proteins / therapeutic use. Stomatitis / prevention & control
  • [MeSH-minor] Animals. Cell Division / drug effects. Cheek. Cricetinae. Dose-Response Relationship, Drug. Epithelial Cells / drug effects. Mesocricetus. Mouth Mucosa / cytology. Mouth Mucosa / drug effects. Whey Proteins

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  • (PMID = 12110343.001).
  • [ISSN] 1368-8375
  • [Journal-full-title] Oral oncology
  • [ISO-abbreviation] Oral Oncol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Antimetabolites, Antineoplastic; 0 / Milk Proteins; 0 / Whey Proteins; U3P01618RT / Fluorouracil
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14. Kreimann EL, Itoiz ME, Dagrosa A, Garavaglia R, Farías S, Batistoni D, Schwint AE: The hamster cheek pouch as a model of oral cancer for boron neutron capture therapy studies: selective delivery of boron by boronophenylalanine. Cancer Res; 2001 Dec 15;61(24):8775-81
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  • [Title] The hamster cheek pouch as a model of oral cancer for boron neutron capture therapy studies: selective delivery of boron by boronophenylalanine.
  • Herein we propose and validate the hamster cheek pouch model of oral cancer for boron neutron capture therapy (BNCT) studies.
  • This model serves to explore new applications of the technique, study the biology and radiobiology of BNCT, and assess the uptake of boron compounds and response of tumor, precancerous tissue, and clinically relevant normal tissues.
  • The success of BNCT is dependent on the absolute amount of boron in the tumor, and the tumor:blood and tumor:normal tissue boron concentration ratios.
  • Tumors were induced in the hamsters with a carcinogenesis protocol that uses dimethyl-1,2-benzanthracene and mimics spontaneous tumor development in human oral mucosa.
  • The animals were then used for biodistribution and pharmacokinetic studies of boronophenylalanine (BPA).
  • Blood, tumor, precancerous pouch tissue surrounding tumor, normal pouch tissue, tongue, skin, cheek mucosa, palate mucosa, liver, and spleen, were sampled at 0-12 h after administration of 300 mg BPA/kg.
  • The data reveal selective uptake of BPA by tumor tissue and, to a lesser degree, by precancerous tissue.
  • Mean tumor boron concentration was 36.9 +/- 17.5 ppm at 3.5 h and the mean boron ratios were 2.4:1 for tumor:normal pouch tissue and 3.2:1 for tumor:blood.
  • Higher doses of BPA (600 and 1200 mg BPA/kg) increased tumor uptake.
  • Potentially therapeutic absolute boron concentrations, and tumor:normal tissue and tumor:blood ratios can be achieved in the hamster oral cancer model using BPA as the delivery agent.
  • [MeSH-major] Boron Compounds / administration & dosage. Boron Neutron Capture Therapy / methods. Mouth Neoplasms / metabolism. Mouth Neoplasms / radiotherapy. Phenylalanine / administration & dosage. Phenylalanine / analogs & derivatives. Radiation-Sensitizing Agents / administration & dosage
  • [MeSH-minor] 9,10-Dimethyl-1,2-benzanthracene. Animals. Boron / blood. Boron / pharmacokinetics. Carcinogens. Cheek. Cricetinae. Disease Models, Animal. Dose-Response Relationship, Drug. Injections, Intraperitoneal. Injections, Intravenous. Injections, Subcutaneous. Mesocricetus. Tissue Distribution

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  • (PMID = 11751398.001).
  • [ISSN] 0008-5472
  • [Journal-full-title] Cancer research
  • [ISO-abbreviation] Cancer Res.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Boron Compounds; 0 / Carcinogens; 0 / Radiation-Sensitizing Agents; 47E5O17Y3R / Phenylalanine; 57-97-6 / 9,10-Dimethyl-1,2-benzanthracene; N9E3X5056Q / Boron; UID84303EL / 4-boronophenylalanine
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15. Caldeira EJ, Carvalho CA, Padovani CR, Camilli JA, Garcia PJ, Cagnon VH: Morphological alterations in the epithelium of the oral mucosa of rats (Rattus norvegicus) submitted to long-term systemic nicotine treatment. Arch Oral Biol; 2007 Jan;52(1):83-9
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  • [Title] Morphological alterations in the epithelium of the oral mucosa of rats (Rattus norvegicus) submitted to long-term systemic nicotine treatment.
  • The local action of tobacco on the oral mucosa can cause precancerous and cancerous lesions.
  • Thus, the aim of the present study was to characterize the cellular changes of the cheek mucosa of rats submitted to long-term systemic nicotine treatment.
  • Twenty male rats were divided into two experimental groups: a nicotine group and a control group, each consisting of 10 animals.
  • All animals received a solid diet and water ad libitum.
  • After 90 days of treatment, all animals were weighed and sacrificed.
  • Samples of cheek mucosa were collected for light and transmission electron microscopy.
  • The results revealed oral epithelium containing atypical cells that were characterized by atrophy, cell membrane disorganization and tissue damage.
  • It was concluded that systemic administration of nicotine damaged the cellular integrity of the oral mucosa, impairing tissue function and predisposing the tissue to the action of different pathogenic agents and also to that of other carcinogenic substances present in tobacco.
  • [MeSH-major] Mouth Mucosa / drug effects. Nicotine / administration & dosage
  • [MeSH-minor] Animals. Drug Administration Schedule. Epithelial Cells / drug effects. Epithelial Cells / pathology. Epithelium / drug effects. Epithelium / pathology. Male. Microscopy, Electron / methods. Organelles / drug effects. Organelles / pathology. Rats. Rats, Wistar

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  • (PMID = 17097602.001).
  • [ISSN] 0003-9969
  • [Journal-full-title] Archives of oral biology
  • [ISO-abbreviation] Arch. Oral Biol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 6M3C89ZY6R / Nicotine
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16. Sacono NT, Costa CA, Bagnato VS, Abreu-e-Lima FC: Light-emitting diode therapy in chemotherapy-induced mucositis. Lasers Surg Med; 2008 Nov;40(9):625-33
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  • BACKGROUND AND OBJECTIVE: Mucositis is the most common oral complication of cancer chemotherapy, which causes pain on mastication and swallowing, impairs patients' ability to eat and take oral drugs and may determine interruption of the treatment.
  • STUDY DESIGN/MATERIALS AND METHODS: Animals of both experimental (Group I; n = 32) and positive control (Group II; n = 32) groups received intraperitoneal injections of 5-fluorouracil on days 0 and 2.
  • All animals had their right and left cheek pouch irritated by superficial scratching on days 3 and 4.
  • The oral mucosa was photographed from day 4 to 14 at 2-day intervals.
  • Biopsies of the cheek pouches of 8 animals (Group I and Group II) were surgically obtained on days 5, 9, 13 and 15 and processed for histological examination.
  • CONCLUSION: It may be concluded that the LED therapy protocol established for this in vivo study was effective in reducing the severity of oral mucositis, although the oral lesions were not completely prevented.
  • [MeSH-major] Antineoplastic Agents / adverse effects. Fluorouracil / adverse effects. Phototherapy / methods. Stomatitis / chemically induced. Stomatitis / therapy
  • [MeSH-minor] Animals. Cheek. Cricetinae. Disease Models, Animal. Male. Mesocricetus

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  • [Copyright] (c) 2008 Wiley-Liss, Inc.
  • (PMID = 18951429.001).
  • [ISSN] 1096-9101
  • [Journal-full-title] Lasers in surgery and medicine
  • [ISO-abbreviation] Lasers Surg Med
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antineoplastic Agents; U3P01618RT / Fluorouracil
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17. Yamamoto T, Nakane T, Osaki T: The mechanism of mononuclear cell infiltration in oral lichen planus: the role of cytokines released from keratinocytes. J Clin Immunol; 2000 Jul;20(4):294-305
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  • [Title] The mechanism of mononuclear cell infiltration in oral lichen planus: the role of cytokines released from keratinocytes.
  • To clarify the pathogenesis of oral lichen planus (OLP), we investigated the roles of keratinocytes (KC) in mononuclear cell infiltration.
  • When peripheral blood mononuclear cells (PBMC) obtained from healthy donors were cultured in the presence of culture supernatants of KC separated from the noninflamed gingivae (Nor-KC) and cheek mucosae of patients with OLP (OLP-KC), the number of migrated PBMC across monolayered human umbilical vein endothelial cells (HUVEC) were increased to about 1.3-fold and 1.5-fold of the control level, respectively, with increases of the expression of CD11a, CD11b, CD18, and CD49d on PBMC and intracellular adhesion molecule-1, vascular cell adhesion molecule-1, and endothelial-leukocyte adhesion molecule-1 on HUVEC.
  • In agreement with these results, the culture supernatants of OLP-KC up-regulated tyrosine phosphorylation of 62-kDa, 70-kDa, and 102-kDa proteins, phosphatidylinositol-3 kinase, and protein kinase C activities and activated Rho protein level more so than did those of Nor-KC.
  • [MeSH-major] Chemotactic Factors / secretion. Chemotaxis, Leukocyte. Cytokines / secretion. Keratinocytes / secretion. Leukocytes, Mononuclear / physiology. Lichen Planus, Oral / immunology. Neutrophil Infiltration
  • [MeSH-minor] Actin Cytoskeleton / drug effects. Actin Cytoskeleton / ultrastructure. Actins / drug effects. Actins / ultrastructure. Antibodies, Monoclonal / pharmacology. Antigens, CD18 / immunology. Antigens, CD18 / physiology. Cell Adhesion Molecules / biosynthesis. Cell Adhesion Molecules / genetics. Cells, Cultured / drug effects. Culture Media, Conditioned / pharmacology. Culture Media, Serum-Free. Cytoskeleton / ultrastructure. Endothelium, Vascular / cytology. Enzyme Activation / drug effects. Enzyme Inhibitors / pharmacology. Gene Expression Regulation / drug effects. Guanosine Triphosphate / physiology. Humans. Intercellular Adhesion Molecule-1 / immunology. Intercellular Adhesion Molecule-1 / physiology. Lymphocyte Function-Associated Antigen-1 / immunology. Lymphocyte Function-Associated Antigen-1 / physiology. Mouth Mucosa / immunology. Mouth Mucosa / pathology. Phosphorylation / drug effects. Protein Kinase Inhibitors. Protein Kinases / physiology. Protein Processing, Post-Translational / drug effects. Signal Transduction. Umbilical Veins. rho GTP-Binding Proteins / physiology

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  • (PMID = 10939717.001).
  • [ISSN] 0271-9142
  • [Journal-full-title] Journal of clinical immunology
  • [ISO-abbreviation] J. Clin. Immunol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Actins; 0 / Antibodies, Monoclonal; 0 / Antigens, CD18; 0 / Cell Adhesion Molecules; 0 / Chemotactic Factors; 0 / Culture Media, Conditioned; 0 / Culture Media, Serum-Free; 0 / Cytokines; 0 / Enzyme Inhibitors; 0 / Lymphocyte Function-Associated Antigen-1; 0 / Protein Kinase Inhibitors; 126547-89-5 / Intercellular Adhesion Molecule-1; 86-01-1 / Guanosine Triphosphate; EC 2.7.- / Protein Kinases; EC 3.6.5.2 / rho GTP-Binding Proteins
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