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1. Hohenstein B, Kuo MC, Addabbo F, Yasuda K, Ratliff B, Schwarzenberger C, Eckardt KU, Hugo CP, Goligorsky MS: Enhanced progenitor cell recruitment and endothelial repair after selective endothelial injury of the mouse kidney. Am J Physiol Renal Physiol; 2010 Jun;298(6):F1504-14
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  • [Title] Enhanced progenitor cell recruitment and endothelial repair after selective endothelial injury of the mouse kidney.
  • Primary and/or secondary injury of the renal microvascular endothelium is a common finding in various renal diseases.
  • However, these mechanisms have so far not been studied after selective microvascular injury in the kidney.
  • The present study investigated the time course of EPC and HSC stimulation and homing following induction of selective EC injury in the mouse kidney along with various angiogenic factors potentially involved in EC repair and progenitor cell stimulation.
  • Progenitor cells could be first detected in the circulation and the spleen before they selectively homed to the diseased kidney.

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  • (PMID = 20237239.001).
  • [ISSN] 1522-1466
  • [Journal-full-title] American journal of physiology. Renal physiology
  • [ISO-abbreviation] Am. J. Physiol. Renal Physiol.
  • [Language] ENG
  • [Grant] United States / NIDDK NIH HHS / DK / R01 DK045462; United States / NIDDK NIH HHS / DK / DK-052783; United States / NIDDK NIH HHS / DK / DK-054602; United States / NIDDK NIH HHS / DK / DK-45462
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Angiogenic Proteins; 11096-26-7 / Erythropoietin; 147336-22-9 / Green Fluorescent Proteins; EC 2.7.10.1 / Receptor, TIE-2
  • [Other-IDs] NLM/ PMC2886821
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2. Orr A, Willis S, Holmes M, Britton P, Orr D: Living with a kidney transplant: a qualitative investigation of quality of life. J Health Psychol; 2007 Jul;12(4):653-62
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  • [Title] Living with a kidney transplant: a qualitative investigation of quality of life.
  • Research has shown that transplantation improves quality of life for patients with end stage renal disease (ESRD), although it does not return to pre-kidney failure levels.
  • This study used focus groups to explore the experience of living with a transplanted kidney.
  • These may necessitate constant vigilance, reduced spontaneity and preoccupation with self-care to maintain the kidney's health; being treated differently from others; pressure not to let themselves and others down; and the urge to increase their knowledge about their condition.
  • [MeSH-major] Kidney Transplantation. Quality of Life. Survivors / psychology

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  • (PMID = 17584816.001).
  • [ISSN] 1359-1053
  • [Journal-full-title] Journal of health psychology
  • [ISO-abbreviation] J Health Psychol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
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3. Davidson T, Goitein O, Avigdor A, Zwas ST, Goshen E: 18F- FDG-PET/CT for the diagnosis of tumor thrombosis. Isr Med Assoc J; 2009 Feb;11(2):69-73
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  • [Title] 18F- FDG-PET/CT for the diagnosis of tumor thrombosis.
  • BACKGROUND: Venous thromboembolism is a well-recognized and relatively frequent complication of malignancy, whereas tumor thrombosis is a rare complication of solid cancers.
  • The correct diagnosis of tumor thrombosis and its differentiation from VTE can alter patient management and prevent unnecessary long-term anticoagulation treatment.
  • OBJECTIVES: To evaluate the contribution of 18F-fluorodeoxyglucose positron emission tomography/computed tomography to the diagnosis of tumor thrombosis and its differentiation from VTE.
  • METHODS: PET/CT scans from 11 patients with suspected tumor thrombosis were retrospectively evaluated.
  • RESULTS: Eight occult tumor thromboses were identified by PET/CT-positive scans.
  • Underlying pathologies included pancreatic, colorectal, renal cell, and head-neck squamous cell carcinoma, as well as lymphoma (4 patients).
  • Accuracy of PET/CT to differentiate between tumor thrombosis and benign VTE was 100% in this small study.
  • It appears that PET/CT may be helpful in the diagnosis of occult tumor thrombosis and its differentiation from VTE.
  • [MeSH-major] Neoplastic Cells, Circulating. Positron-Emission Tomography. Thromboembolism / diagnosis. Tomography, X-Ray Computed
  • [MeSH-minor] Adult. Aged. Carcinoma / radiography. Carcinoma / radionuclide imaging. Cohort Studies. Diagnosis, Differential. Digestive System Neoplasms / radiography. Digestive System Neoplasms / radionuclide imaging. Female. Fluorodeoxyglucose F18. Humans. Kidney Neoplasms / radiography. Kidney Neoplasms / radionuclide imaging. Lymphoma / radiography. Lymphoma / radionuclide imaging. Male. Middle Aged. Predictive Value of Tests. Radiopharmaceuticals. Retrospective Studies

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  • (PMID = 19432032.001).
  • [ISSN] 1565-1088
  • [Journal-full-title] The Israel Medical Association journal : IMAJ
  • [ISO-abbreviation] Isr. Med. Assoc. J.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Israel
  • [Chemical-registry-number] 0 / Radiopharmaceuticals; 0Z5B2CJX4D / Fluorodeoxyglucose F18
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4. Ohlmann CH, Ozgür E, Schrader AJ, Konrad L, Hofmann R, Engelmann U, Heidenreich A: Detection of circulating tumor cells in patients with renal cell carcinoma by reverse transcriptase polymerase chain reaction for G250/MNCA-9: results of a prospective trial. Urol Oncol; 2006 Jul-Aug;24(4):287-93
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Detection of circulating tumor cells in patients with renal cell carcinoma by reverse transcriptase polymerase chain reaction for G250/MNCA-9: results of a prospective trial.
  • OBJECTIVES: Radical nephrectomy displays the standard procedure for patients with localized renal cell carcinoma.
  • The transperitoneal approach is often favored compared to the retroperitoneal approach because of the early ligation of the renal vessels, thereby tumor cell shedding by manipulation of the tumor is thought to be avoided.
  • The aim of our study was to investigate the influence of the surgical technique on intraoperative tumor cell shedding.
  • METHODS: A total of 55 consecutive patients with renal tumors suspicious for malignancies were evaluated for this study.
  • Peripheral blood samples were obtained from 44 patients at admission, intraoperatively (before and after kidney removal), and before discharge.
  • Evaluation of MNCA-9 RT-PCR revealed no difference according to operative technique, tumor-nodes-metastasis, or clinical tumor stage.
  • Despite this result, we found positive RT-PCR signals for MNCA-9 in patients with transitional cell cancer of the renal pelvis and benign renal lesions.
  • Furthermore, the retroperitoneal approach does not bear the risk of intraoperative tumor cell shedding by the handling of the tumor.
  • [MeSH-major] Antigens, Neoplasm / genetics. Carbonic Anhydrases / genetics. Carcinoma, Renal Cell / pathology. Kidney Neoplasms / pathology. Neoplastic Cells, Circulating. Reverse Transcriptase Polymerase Chain Reaction / methods

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  • (PMID = 16818180.001).
  • [ISSN] 1078-1439
  • [Journal-full-title] Urologic oncology
  • [ISO-abbreviation] Urol. Oncol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antigens, Neoplasm; EC 4.2.1.1 / CA9 protein, human; EC 4.2.1.1 / Carbonic Anhydrases
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5. Su W, Fang C, Yang HC, Gu Y, Hao CM: [Expression of nestin in human kidney and its clinical significance]. Zhonghua Bing Li Xue Za Zhi; 2008 May;37(5):309-12
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  • [Title] [Expression of nestin in human kidney and its clinical significance].
  • OBJECTIVE: To study the expression and significance of nestin (a type of cytoskeletal protein) in normal and diseased human kidneys.
  • METHODS: Diseased kidney tissues were obtained from needle biopsies in 32 patients with glomerulonephritis (including 8 cases of membranous glomerulopathy, 3 cases of focal segmental glomerulosclerosis, 17 cases of IgA nephropathy with proteinuria and 4 cases of IgA nephropathy without proteinuria).
  • Control kidney tissues were obtained from nephrectomy specimens for renal tumors.
  • The expression of nestin in the control kidney tissues was studied using immunoelectronic microscopy and immunohistochemistry.
  • The expression of nestin in the diseased kidney tissues was detected by immunohistochemistry and real-time reverse transcription-polymerase chain reaction.
  • RESULTS: In normal kidney tissues, nestin was detected at the periphery of glomerular capillary loops.
  • However, the glomerular nestin expression levels in cases of IgA nephropathy with proteinuria, membranous glomerulopathy and focal segmental glomerulosclerosis were significantly lower than those in the normal kidneys and IgA nephropathy without proteinuria.
  • CONCLUSION: Nestin may play an important role in maintaining the normal function of podocytes in human kidney.
  • [MeSH-minor] Adult. Gene Expression Regulation. Humans. Immunohistochemistry. Kidney. Kidney Diseases / metabolism. Kidney Diseases / pathology. Kidney Glomerulus / metabolism. Kidney Glomerulus / pathology. Middle Aged. Nestin. Proteinuria / metabolism

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  • (PMID = 18956648.001).
  • [ISSN] 0529-5807
  • [Journal-full-title] Zhonghua bing li xue za zhi = Chinese journal of pathology
  • [ISO-abbreviation] Zhonghua Bing Li Xue Za Zhi
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] China
  • [Chemical-registry-number] 0 / Intermediate Filament Proteins; 0 / NES protein, human; 0 / Nerve Tissue Proteins; 0 / Nestin
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6. Simmons WN, Cocks FH, Zhong P, Preminger G: A composite kidney stone phantom with mechanical properties controllable over the range of human kidney stones. J Mech Behav Biomed Mater; 2010 Jan;3(1):130-3
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  • [Title] A composite kidney stone phantom with mechanical properties controllable over the range of human kidney stones.
  • A novel composite kidney stone phantom has been developed.
  • This stone phantom is producible with mechanical properties mimicking the range of tensile fracture strength and acoustic properties of human kidney stones and is an inorganic/organic composite material, as are natural kidney stones.
  • Diametral compression testing was used to measure tensile fracture strength, which determines the acoustic comminution behavior of kidney stones.
  • Both the tensile fracture strength (controllable from 1 to approximately 5 MPa) and acoustic properties (C(L) = 2700-4400 m/s and C(T)=1600-2300m/s) of these composite phantom stones match those of a wide variety of human kidney stones.
  • [MeSH-major] Kidney Calculi / physiopathology. Phantoms, Imaging

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  • (PMID = 19878912.001).
  • [ISSN] 1878-0180
  • [Journal-full-title] Journal of the mechanical behavior of biomedical materials
  • [ISO-abbreviation] J Mech Behav Biomed Mater
  • [Language] eng
  • [Grant] United States / NIDDK NIH HHS / DK / R01 DK052985; United States / NIDDK NIH HHS / DK / R01 DK052985; United States / NIDDK NIH HHS / DK / R37 DK052985
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] Netherlands
  • [Other-IDs] NLM/ NIHMS498637; NLM/ PMC3756310
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7. Nezami N, Tarzamni MK, Argani H, Nourifar M: Doppler ultrasonographic indexes in kidney transplant recipients: its relationship with kidney function. Iran J Kidney Dis; 2007 Oct;1(2):82-7
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  • [Title] Doppler ultrasonographic indexes in kidney transplant recipients: its relationship with kidney function.
  • INTRODUCTION: Doppler ultrasonography (DU) is mostly used for assessment of both graft and native kidneys' vascular status.
  • In this study, correlation between the DU indexes and kidney allograft function was evaluated.
  • MATERIALS AND METHODS: Hospital records of 273 kidney transplant patients (154 men and 119 women) were reviewed.
  • In all cases, DU had been performed 1 month after kidney transplantation.
  • We evaluated the data on the resistive index (RI) and pulsatility index (PI) in the interlobar arteries and renal artery stenosis (RAS), and renal vessels thrombosis were determined.
  • Concurrent serum creatinine and cyclosporine values were assessed in relation to the DU findings.
  • RESULTS: The RI and PI had significant linear correlations with serum creatinine (P = .03 and P = .002, respectively).
  • Also, there were direct linear correlations between the age of the patients and the RI and PI values.
  • Despite this finding, RI and PI were significantly lower in patients with RAS than in the patients with patent renovascular tributary (0.59 +/- 0.15 versus 0.65 +/- 0.11; P = .03 and 1.02 +/- 0.40 versus 1.18 +/- 0.46; P = .049, respectively).
  • There were no associations between serum cyclosporine level or panel reactive antibodies and the RI or PI.
  • CONCLUSIONS: The RI and PI are valuable DU markers for determining the kidney allograft function and the related vascular complications.
  • [MeSH-major] Kidney Transplantation / ultrasonography. Renal Artery Obstruction / ultrasonography. Thrombosis / ultrasonography


8. Weiss AS, Smits G, Wiseman AC: Standard criteria donor pancreas donation status is associated with improved kidney transplant outcomes. Clin Transplant; 2009 Sep-Oct;23(5):732-9
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  • [Title] Standard criteria donor pancreas donation status is associated with improved kidney transplant outcomes.
  • BACKGROUND: Organ donor characteristics can be used to predict outcomes in kidney transplantation.
  • We hypothesized that pancreas donation status could reflect organ quality and be predictive of kidney graft outcomes following Standard Criteria Donor (SCD) kidney transplantation.
  • METHODS: We performed a retrospective analysis of deceased donor kidney alone (DD KA) transplants reported to SRTR from 1992 to 2005.
  • Group 1 = kidney alone recipients from pancreas donors (KA, P+) and Group 2 = kidney alone recipients from non-pancreas donors (KA, P-).
  • CONCLUSION: Donor pancreas donation status is an independent predictor of improved outcomes of SCD kidney recipients.
  • Further study of the pancreas organ donor pre-procurement is warranted to optimize not only pancreas utilization but also kidney graft outcomes.
  • [MeSH-major] Graft Rejection / prevention & control. Graft Survival / physiology. Kidney Transplantation. Pancreas Transplantation. Tissue Donors. Tissue and Organ Procurement / standards


9. Friedman AL, Peters TG, Jones KW, Boulware LE, Ratner LE: Fatal and nonfatal hemorrhagic complications of living kidney donation. Ann Surg; 2006 Jan;243(1):126-30
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  • [Title] Fatal and nonfatal hemorrhagic complications of living kidney donation.
  • OBJECTIVE: After anecdotal reports of severe hemorrhage from failure of surgical clips to sustain closure of renal artery stumps in live donor nephrectomies were received, this study was designed to identify specific surgical techniques that are associated with an increased risk of failure to control bleeding and might represent opportunities to improve patient safety.
  • BACKGROUND: Preventing complications for living kidney donors must be paramount in addressing end-stage renal failure through living kidney donation.
  • Open and laparoscopic approaches to living kidney donation use several vascular control methods, some of which may be more prone to failure and life-endangering hemorrhage than others.
  • METHODS: To define hemorrhagic complications of living kidney donation, a survey was sent to all 893 surgeon-members of the American Society of Transplant Surgeons.
  • Descriptive and bivariate analyses were used to ascertain study participant characteristics, most frequently used vascular control techniques, and incidence of events (death, transfusion, reexploration or conversion to open nephrectomy, or contralateral [remaining kidney] renal failure).
  • Among arterial control problems, 2 resulted in donor death and 2 resulted in renal failure; 19 episodes required transfusion.
  • Locking and standard clips applied to the renal artery were associated with the greatest risks.
  • CONCLUSIONS: Significant hemorrhagic complications occur with living kidney donation in both open and laparoscopic approaches.
  • [MeSH-major] Hemorrhage / etiology. Kidney Transplantation / adverse effects. Living Donors. Nephrectomy / adverse effects. Surgical Instruments / adverse effects

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  • (PMID = 16371747.001).
  • [ISSN] 0003-4932
  • [Journal-full-title] Annals of surgery
  • [ISO-abbreviation] Ann. Surg.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC1449959
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10. Horváth HC, Lakatos P, Kósa JP, Bácsi K, Borka K, Bises G, Nittke T, Hershberger PA, Speer G, Kállay E: The candidate oncogene CYP24A1: A potential biomarker for colorectal tumorigenesis. J Histochem Cytochem; 2010 Mar;58(3):277-85
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  • Synthesis and degradation of the secosteroid occurs not only in the kidney but also in normal tissue or malignant extrarenal tissues such as the colon.
  • Although 76% of the normal and benign colonic tissue was either completely devoid of or expressed very low levels of CYP24A1, in the majority of the adenocarcinomas (69%), the enzyme was present at high concentrations.
  • [MeSH-major] Adenocarcinoma / enzymology. Adenoma / enzymology. Biomarkers, Tumor / biosynthesis. Colorectal Neoplasms / enzymology. Steroid Hydroxylases / biosynthesis

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  • (PMID = 19901270.001).
  • [ISSN] 1551-5044
  • [Journal-full-title] The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society
  • [ISO-abbreviation] J. Histochem. Cytochem.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Biomarkers, Tumor; 0 / Ki-67 Antigen; 0 / RNA, Messenger; 0 / Receptors, Calcitriol; EC 1.14.- / Steroid Hydroxylases; EC 1.14.13.126 / CYP24A1 protein, human; EC 1.14.13.126 / Vitamin D3 24-Hydroxylase
  • [Other-IDs] NLM/ PMC2825493
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11. Shoji T, Ishimura E, Nishizawa Y: Body fat measurement in chronic kidney disease: implications in research and clinical practice. Curr Opin Nephrol Hypertens; 2007 Nov;16(6):572-6
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  • [Title] Body fat measurement in chronic kidney disease: implications in research and clinical practice.
  • PURPOSE OF REVIEW: The paradoxical and inverse association between body mass index and mortality risk in patients with end-stage renal disease has raised a question of whether an increased fat mass is good or bad for patients with chronic kidney disease.
  • The purpose of this review is to update the concept on body fat in patients with chronic kidney disease.
  • Following the initiation of dialysis, chronic kidney disease patients gain body weight due mainly to increased fat mass.
  • In predialysis chronic kidney disease, there is also an inverse association between body mass index and mortality risk.
  • The metabolic syndrome and a high body mass index are independent predictors for development of chronic kidney disease and end-stage renal disease, respectively.
  • In diabetic patients with chronic kidney disease, however, a high initial body mass index is associated with a slower decline in glomerular filtration rate.
  • SUMMARY: The impacts of fat mass on survival and renal function appear to vary depending upon the absence or presence, and stages of chronic kidney disease.
  • Further research is required for optimal nutritional management and improved outcomes of patients with chronic kidney disease.
  • [MeSH-major] Adipose Tissue. Kidney Failure, Chronic / pathology
  • [MeSH-minor] Anthropometry. Humans. Kidney Function Tests. Predictive Value of Tests. Survival Rate

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  • (PMID = 18089973.001).
  • [ISSN] 1062-4821
  • [Journal-full-title] Current opinion in nephrology and hypertension
  • [ISO-abbreviation] Curr. Opin. Nephrol. Hypertens.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Number-of-references] 25
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12. de Klerk M, Zuidema WC, Ijzermans JN, Weimar W: On chain lengths, domino-paired and unbalanced altruistic kidney donations. Clin Transpl; 2009;:247-52
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  • [Title] On chain lengths, domino-paired and unbalanced altruistic kidney donations.
  • Kidney transplantations with living related and unrelated donors are the optimal option for patients with end-stage renal disease.
  • For patients with a willing--but blood-type or HLA incompatible donor--a living-donor kidney exchange program could be an opportunity.
  • In Asia, the United States and Europe, kidney exchange programs were developed under different conditions, with different exchange algorithms, and with different match results.
  • The easiest way to organize a living-donor kidney exchange program is to enlist national or regional cooperation, initiated by an independent organization that is already responsible for the allocation of deceased donor organs.
  • For incompatible donor-recipient pairs who have been unsuccessful in finding suitable matches in an exchange program, domino-paired kidney transplantations triggered by Good Samaritan donors is the next alternative.
  • If no Good Samaritan donors are available, an unbalanced kidney paired-exchange program with compatible and incompatible pairs is another strategy that merits future development.
  • [MeSH-major] Altruism. Kidney Transplantation / statistics & numerical data. Living Donors / psychology

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  • (PMID = 20524290.001).
  • [ISSN] 0890-9016
  • [Journal-full-title] Clinical transplants
  • [ISO-abbreviation] Clin Transpl
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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13. Gao S, Manns BJ, Culleton BF, Tonelli M, Quan H, Crowshoe L, Ghali WA, Svenson LW, Hemmelgarn BR, Alberta Kidney Disease Network: Prevalence of chronic kidney disease and survival among aboriginal people. J Am Soc Nephrol; 2007 Nov;18(11):2953-9
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  • [Title] Prevalence of chronic kidney disease and survival among aboriginal people.
  • Globally, it is known that the incidence of end-stage renal disease is higher among Aboriginals, but it is unknown whether this is due to an increased prevalence of chronic kidney disease or other unidentified factors.
  • We studied 658,664 people of non-First Nations and 14,989 people of First Nations and found that the age- and sex-adjusted prevalence of chronic kidney disease was significantly higher among those of non-First Nations compared to those of First Nations (67.5 versus 59.5 per 1000 population; P < 0.0001).
  • However, severe chronic kidney disease (estimated glomerular filtration rate <30 ml/min per 1.73 m2) was almost two-fold higher among people of First Nations (P < 0.0001).
  • Cox proportional hazards models suggested that compared to people of non-First Nations, those of First Nations with chronic kidney disease had a 77% increased risk of death after adjusting for age, gender, diabetes and baseline eGFR.
  • In conclusion, whether the higher incidence of end-stage renal disease among people of First Nations is due to suboptimal management of chronic kidney disease and its associated comorbidities, more rapid loss of kidney function, or other unidentified factors remains to be determined.
  • [MeSH-major] Indians, North American / statistics & numerical data. Kidney Diseases / ethnology. Kidney Diseases / mortality


14. Fearnley C, Wakeley PR, Gallego-Beltran J, Dalley C, Williamson S, Gaudie C, Woodward MJ: The development of a real-time PCR to detect pathogenic Leptospira species in kidney tissue. Res Vet Sci; 2008 Aug;85(1):8-16
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  • [Title] The development of a real-time PCR to detect pathogenic Leptospira species in kidney tissue.
  • To assess the suitability of these PCR methods for diagnosis, a trial was performed on kidneys taken from adult pigs with evidence of leptospiral infection, primarily a history of reproductive disease and serological evidence of exposure to pathogenic leptospires (n=180) and aborted pig foetuses (n=24).
  • In a subsidiary experiment, the 'pathogenic' PCR was used to analyse kidney samples from rodents (n=7) collected as part of vermin control in a zoo, with show animals with high microagglutination titres to Leptospira species, and five were positive.
  • Fifteen PCR amplicons from 1 mouse, 2 rat and 14 pig kidney samples, were selected at random from positive PCRs (n=30) and sequenced.
  • The only successful culture was from this mouse kidney and the isolate was confirmed to be L. inadai by classical serology.
  • [MeSH-major] Kidney / microbiology. Leptospira / isolation & purification. Leptospirosis / veterinary. Polymerase Chain Reaction / veterinary. Swine Diseases / microbiology
  • [MeSH-minor] Animals. Kidney Diseases / microbiology. Kidney Diseases / veterinary. Swine. Temperature

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  • (PMID = 17961617.001).
  • [ISSN] 0034-5288
  • [Journal-full-title] Research in veterinary science
  • [ISO-abbreviation] Res. Vet. Sci.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
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15. Reisaeter AV, Røislien J, Henriksen T, Irgens LM, Hartmann A: Pregnancy and birth after kidney donation: the Norwegian experience. Am J Transplant; 2009 Apr;9(4):820-4
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  • [Title] Pregnancy and birth after kidney donation: the Norwegian experience.
  • Reports on pregnancies in kidney donors are scarce.
  • Linkage with the Norwegian Renal Registry provided data on pregnancies of kidney donors 1967-2002.
  • A random sample from the Medical Birth Registry was control group, as was pregnancies in kidney donors prior to donation.
  • No differences were observed in the occurrence of adverse pregnancy outcome in kidney donors and in the general population in unadjusted analysis.
  • Our finding of more frequent preeclampsia in pregnancies after kidney donation in the secondary analysis must be interpreted with caution, as the number of events was low.


16. Girgin C, Sezer A, Sahin O, Oder M, Dinçel C: Giant renal calculus in a solitary functioning kidney. Urol Int; 2007;78(1):91-2
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  • [Title] Giant renal calculus in a solitary functioning kidney.
  • We report a case of giant renal calculus 230 x 140 mm in size and weighing 1,350 g in a solitary functioning kidney treated by nephrolithotomy.
  • A giant renal calculus in his right kidney and atrophic nonfunctioning left kidney was diagnosed by ultrasonography, IVP and CT scan.
  • This case is the largest and the heaviest stone reported in the literature in a solitary functioning kidney.
  • [MeSH-major] Kidney Calculi
  • [MeSH-minor] Diagnosis, Differential. Follow-Up Studies. Humans. Male. Middle Aged. Nephrostomy, Percutaneous. Tomography, X-Ray Computed. Urodynamics

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  • (PMID = 17192742.001).
  • [ISSN] 0042-1138
  • [Journal-full-title] Urologia internationalis
  • [ISO-abbreviation] Urol. Int.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Switzerland
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17. Go AS, Lo JC: Epidemiology of non-dialysis-requiring chronic kidney disease and cardiovascular disease. Curr Opin Nephrol Hypertens; 2006 May;15(3):296-302
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  • [Title] Epidemiology of non-dialysis-requiring chronic kidney disease and cardiovascular disease.
  • PURPOSE OF REVIEW: To review recent literature about the relationship between non-dialysis-requiring chronic kidney disease and cardiovascular disease as well as possible explanatory factors.
  • This risk is not linearly associated with level of kidney function.
  • Chronic kidney disease is associated with a larger burden of traditional vascular risk factors but is also linked to abnormalities in a variety of nontraditional pathways such as dysregulation of mineral metabolism and arterial calcification, vessel stiffness and endothelial dysfunction, insulin resistance, inflammation, malnutrition, and anemia, among others.
  • Other novel kidney-specific proteins (e.g. renalase) may play direct mediating roles.
  • The relative contribution of these factors to excess cardiovascular disease in chronic kidney disease remains unclear.
  • SUMMARY: Recent evidence demonstrates the importance of non-dialysis-requiring chronic kidney disease as a potent predictor of cardiovascular disease and its complications.
  • Randomized trials should be performed to determine whether modification of traditional and nontraditional risk factors can reduce incident cardiovascular disease as well as which interventions can optimize treatment outcomes in persons with chronic kidney disease and cardiovascular disease.
  • [MeSH-major] Cardiovascular Diseases / epidemiology. Kidney Diseases / epidemiology
  • [MeSH-minor] Chronic Disease. Glomerular Filtration Rate. Humans. Renal Dialysis. Risk Factors

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  • (PMID = 16609298.001).
  • [ISSN] 1062-4821
  • [Journal-full-title] Current opinion in nephrology and hypertension
  • [ISO-abbreviation] Curr. Opin. Nephrol. Hypertens.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Number-of-references] 67
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18. Hansberry MR, Whittier WL, Krause MW: The elderly patient with chronic kidney disease. Adv Chronic Kidney Dis; 2005 Jan;12(1):71-7
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  • [Title] The elderly patient with chronic kidney disease.
  • The elderly are a fast growing population in the United States, and they have a high prevalence of chronic kidney disease.
  • The elderly are particularly susceptible to kidney damage from age-related declines in glomerular filtration as well as kidney damage from chronic disease states such as diabetes mellitus, hypertension, glomerular, and tubulointerstitial disorders.
  • A significant number of elderly individuals are reaching end-stage renal disease that require renal replacement therapy.
  • This expanding population provides a challenge for health-care providers because the elderly are often referred late to a nephrologist, have a shortened survival on renal replacement therapy as compared with younger individuals, and suffer from more comorbidities such as cardiovascular disease, malnutrition, and hearing and visual disabilities.
  • The elderly also have difficulties with dialysis vascular access and often are not candidates for renal transplantation.
  • Despite these obstacles, age alone is not a justification for withholding diagnostic or therapeutic interventions, because many elderly individuals have an improvement in their quality of life and social support once their kidney disease is identified and treated.
  • [MeSH-major] Kidney Failure, Chronic / epidemiology
  • [MeSH-minor] Age Factors. Aged. Disease Susceptibility / epidemiology. Humans. Prevalence. Renal Replacement Therapy / utilization. Risk Factors. United States / epidemiology

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  • (PMID = 15719336.001).
  • [ISSN] 1548-5595
  • [Journal-full-title] Advances in chronic kidney disease
  • [ISO-abbreviation] Adv Chronic Kidney Dis
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 54
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19. Ruppar TM, Russell CL: Medication adherence in successful kidney transplant recipients. Prog Transplant; 2009 Jun;19(2):167-72
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  • [Title] Medication adherence in successful kidney transplant recipients.
  • OBJECTIVE: To explore the medication-taking behavior of successful kidney transplant recipients and determine what behaviors were common among this group.
  • METHODS: Open-ended interviews were conducted by telephone with 19 individuals who had successfully maintained a transplanted kidney for 25 years or more.
  • RESULTS: Four themes emerged as participants described the behaviors they developed to adhere successfully to the immunosuppressive medication required for maintaining their transplanted kidneys.
  • Kidney transplant recipients identified the importance of developing and maintaining medication-taking skills and routines on medication adherence.
  • CONCLUSIONS: Interventions focusing on medication-taking skills, habit formation, and resources for problem solving may improve immunosuppressive medication adherence and clinical outcomes in kidney transplant recipients.

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  • (PMID = 19588667.001).
  • [ISSN] 1526-9248
  • [Journal-full-title] Progress in transplantation (Aliso Viejo, Calif.)
  • [ISO-abbreviation] Prog Transplant
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / U24 CA092656; United States / NCI NIH HHS / CA / R24 CA092656; United States / NINR NIH HHS / NR / P30 NR003979; United States / NINR NIH HHS / NR / P30 NR003979-15; None / None / / P30 NR003979-15
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Immunosuppressive Agents
  • [Other-IDs] NLM/ NIHMS266536; NLM/ PMC3071038
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20. Lorenz M, Regele H, Schillinger M, Kletzmayr J, Haidbauer B, Derfler K, Druml W, Böhmig GA: Peritransplant immunoadsorption: a strategy enabling transplantation in highly sensitized crossmatch-positive cadaveric kidney allograft recipients. Transplantation; 2005 Mar 27;79(6):696-701
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  • [Title] Peritransplant immunoadsorption: a strategy enabling transplantation in highly sensitized crossmatch-positive cadaveric kidney allograft recipients.
  • BACKGROUND: Kidney transplant recipients with a current positive complement-dependent cytotoxicity crossmatch (CDCXM) are at high risk for hyperacute rejection and graft loss.
  • In this analysis, we evaluated effectiveness of peritransplant IA as an anti-humoral strategy to overcome a current positive CDCXM in presensitized renal allograft recipients.
  • METHODS: Between 1999 and 2003, 40 high risk cadaveric kidney allograft recipients (median CDC panel reactive antibody [PRA] level, 77%; number of retransplants, n = 38) were subjected to peritransplant IA with protein A (one pretransplant IA session followed by a course of repeat posttransplant IA sessions) in addition to preemptive antilymphocyte antibody therapy.
  • CONCLUSION: Our results demonstrate that peritransplant IA enables successful cadaveric kidney transplantation in the context of a positive CDCXM.
  • [MeSH-major] Graft Survival / immunology. Histocompatibility Testing. Immunosorbents / immunology. Kidney Transplantation / immunology


21. Schukfeh N, Kuebler JF, Schirg E, Petersen C, Ure BM, Glüer S: Dysplastic kidney and not renal agenesis is the commonly associated anomaly in infants with seminal vesicle cyst. BJU Int; 2009 Mar;103(6):816-9
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  • [Title] Dysplastic kidney and not renal agenesis is the commonly associated anomaly in infants with seminal vesicle cyst.
  • OBJECTIVE: To determine whether the association of seminal vesicle cyst (SVC) and renal anomaly in young children correlates with previously reported cases of SVCs in adolescent and adult patients, as congenital SVCs, although rare, are frequently described in association with ipsilateral renal agenesis, mainly in adolescent and adult patients, whereas reports on SVCs in younger children are sparse.
  • PATIENTS AND METHODS: We report on nine infants (median age 4 months) with congenital SVCs, all of them associated with ipsilateral dysplastic kidneys.
  • All patients had ultrasonography of the renal system and voiding cysto-urethrography.
  • RESULTS: The SVCs were found incidentally during ultrasonography for the renal anomaly.
  • Three patients had dysplastic and six had multicystic dysplastic kidneys.
  • In previous reported adult cases of SVCs the most common associated renal anomaly was agenesis of the ipsilateral kidney (25 of 44 cases), whereas only one case of dysplastic kidney was reported.
  • CONCLUSION: As the appearance of renal agenesis might result from a former congenital dysplastic kidney, our findings indicate that cases of ipsilateral renal agenesis in adult patients with congenital SVCs might represent former dysplastic or multicystic dysplastic kidney.
  • [MeSH-major] Cysts / etiology. Genital Diseases, Male / etiology. Kidney / abnormalities. Multicystic Dysplastic Kidney / complications. Seminal Vesicles

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  • (PMID = 19040535.001).
  • [ISSN] 1464-410X
  • [Journal-full-title] BJU international
  • [ISO-abbreviation] BJU Int.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
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22. Langer R, Tóth A, Máthé Z, Remport A, Járay J, Kahan BD: [Lymphocele and kidney transplantation]. Orv Hetil; 2007 Aug 5;148(31):1475-80
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  • [Title] [Lymphocele and kidney transplantation].
  • INTRODUCTION: Lymphocele is a special complication following kidney transplantation.
  • The authors examined the factors associated with an increased occurrence of clinically significant perinephric fluid collections and/or lymphoceles among sirolimus-treated renal transplant recipients.
  • In both subgroups the serum creatinine levels were elevated at the time of diagnosis from a nadir of 179.5 +/- 141.7 to 359.9 +/- 259.6 mmol/l (Group III, sirolimus treated) and from 222.6 +/- 205.9 to 383.7 +/- 255.2 mmol/l (Group IV, sirolimus free).
  • Despite an Influenza A + Chlamydia pneumonia and acute rejection which was followed by a GI bleeding and stomach resection he fully recovered and is doing well with an excellent kidney function a year after.
  • [MeSH-major] Immunosuppressive Agents / adverse effects. Kidney Transplantation / adverse effects. Lymphocele / etiology

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  • (PMID = 17656338.001).
  • [ISSN] 0030-6002
  • [Journal-full-title] Orvosi hetilap
  • [ISO-abbreviation] Orv Hetil
  • [Language] hun
  • [Publication-type] Case Reports; Comparative Study; English Abstract; Journal Article
  • [Publication-country] Hungary
  • [Chemical-registry-number] 0 / Immunosuppressive Agents; 83HN0GTJ6D / Cyclosporine; MRK240IY2L / Azathioprine; VB0R961HZT / Prednisone; W36ZG6FT64 / Sirolimus
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23. Yaqoob MM: Acidosis and progression of chronic kidney disease. Curr Opin Nephrol Hypertens; 2010 Sep;19(5):489-92
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  • [Title] Acidosis and progression of chronic kidney disease.
  • PURPOSE OF REVIEW: Chronic kidney disease progressively impairs the ability of kidneys to excrete hydrogen ions owing to the reduced capacity of the kidney to synthesize ammonia resulting in metabolic acidosis.
  • There is good experimental evidence that metabolic acidosis contributes to protein energy wasting disorder and progression of chronic kidney disease (CKD).
  • RECENT FINDINGS: Three recent publications have confirmed the experimental evidence and the only randomized controlled study of its kind has suggested that the correction of acidosis by sodium bicarbonate in patients with advanced CKD is associated with attenuation of the rate of decline of renal function, reduction in the incidence of end stage renal disease and improvement of nutritional parameters.
  • SUMMARY: In light of these recent studies, it appears that this cheap and simple strategy, which is in line with current renal recommendations, has the potential of translating into significant economic, quality of life and clinical outcome benefits in an expanding pool of patients with CKD.
  • [MeSH-major] Acidosis / complications. Kidney Diseases / complications
  • [MeSH-minor] Animals. Disease Progression. Glomerular Filtration Rate. Humans. Kidney / physiopathology

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  • (PMID = 20539229.001).
  • [ISSN] 1473-6543
  • [Journal-full-title] Current opinion in nephrology and hypertension
  • [ISO-abbreviation] Curr. Opin. Nephrol. Hypertens.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
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24. Ishibe S, Cantley LG: Epithelial-mesenchymal-epithelial cycling in kidney repair. Curr Opin Nephrol Hypertens; 2008 Jul;17(4):379-85
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  • [Title] Epithelial-mesenchymal-epithelial cycling in kidney repair.
  • PURPOSE OF REVIEW: Tubule repair following acute kidney injury involves epithelial de-differentiation followed by cell migration and proliferation and eventual re-differentiation.
  • RECENT FINDINGS: Epithelial de-differentiation after kidney injury or in epithelial culture systems is controlled by secreted factors such as transforming growth factor beta and hepatocyte growth factor as well as cell-cell and cell-matrix interactions.
  • SUMMARY: This review focuses on the underlying molecular mechanism of epithelial de-differentiation and re-differentiation in tubule repair in vivo and formation in vitro, thus giving insight into possible strategies for improving recovery following acute kidney injury and preventing progression to chronic kidney disease.
  • [MeSH-major] Epithelium / physiology. Kidney / physiology. Mesoderm / physiology
  • [MeSH-minor] Animals. Cell Differentiation / physiology. Humans. Kidney Diseases / pathology. Kidney Diseases / physiopathology. Kidney Tubules / growth & development. Kidney Tubules / physiology. Wnt Proteins / metabolism

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  • (PMID = 18660674.001).
  • [ISSN] 1062-4821
  • [Journal-full-title] Current opinion in nephrology and hypertension
  • [ISO-abbreviation] Curr. Opin. Nephrol. Hypertens.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Wnt Proteins
  • [Number-of-references] 58
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25. Yen M, Huang JJ, Teng HL: Education for patients with chronic kidney disease in Taiwan: a prospective repeated measures study. J Clin Nurs; 2008 Nov;17(21):2927-34
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  • [Title] Education for patients with chronic kidney disease in Taiwan: a prospective repeated measures study.
  • AIM: To investigate the physical, knowledge and quality of life outcomes of an educational intervention for patients with early stage chronic kidney disease.
  • BACKGROUND: A comprehensive predialysis education care team can be effective in slowing the progression of chronic kidney disease.
  • A predialysis, team-delivered educational intervention covering renal function health care, dietary management of renal function and the effects of Chinese herb medication on renal function was designed and implemented.
  • Study outcomes included physical indicators, knowledge (renal function protection, use of Chinese herbs and renal function and diet) and quality of life.
  • The primary indicator of renal function, glomerular filtration rate, remained stable throughout the 12 months of follow-up, despite the relatively older mean age of study participants.
  • CONCLUSION: A predialysis education care team can provide effective disease-specific knowledge and may help retard deterioration of renal function in persons with early-stage chronic kidney disease.
  • [MeSH-major] Kidney Diseases / physiopathology. Patient Education as Topic

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  • (PMID = 19012761.001).
  • [ISSN] 1365-2702
  • [Journal-full-title] Journal of clinical nursing
  • [ISO-abbreviation] J Clin Nurs
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
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26. Caixeta A, Mehran R: Evidence-based management of patients undergoing PCI: contrast-induced acute kidney injury. Catheter Cardiovasc Interv; 2010 Mar 1;75 Suppl 1:S15-20
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Evidence-based management of patients undergoing PCI: contrast-induced acute kidney injury.
  • Contrast-induced acute kidney injury (CI-AKI) is one of the leading causes of hospital-acquired acute kidney injury.
  • CI-AKI is highly prevalent in patients with well-known risk factors, including older age, chronic renal insufficiency, congestive heart failure, and diabetes.
  • [MeSH-major] Angioplasty, Balloon, Coronary. Contrast Media / adverse effects. Kidney Diseases / prevention & control. Radiography, Interventional / adverse effects

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  • [Copyright] (c) 2010 Wiley-Liss, Inc.
  • (PMID = 20333702.001).
  • [ISSN] 1522-726X
  • [Journal-full-title] Catheterization and cardiovascular interventions : official journal of the Society for Cardiac Angiography & Interventions
  • [ISO-abbreviation] Catheter Cardiovasc Interv
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Contrast Media; 0 / Protective Agents; 8MDF5V39QO / Sodium Bicarbonate; INU8H2KAWG / Fenoldopam; VTD58H1Z2X / Dopamine; WYQ7N0BPYC / Acetylcysteine
  • [Number-of-references] 47
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27. Danovitch GM, Delmonico FL: The prohibition of kidney sales and organ markets should remain. Curr Opin Organ Transplant; 2008 Aug;13(4):386-94
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  • [Title] The prohibition of kidney sales and organ markets should remain.
  • RECENT FINDINGS: In 'natural experiments' performed in developing countries the outcome for kidney vendors, in terms of both their medical and psychosocial health, has been shown to be poor.
  • SUMMARY: Commercialization of living kidney donation does not serve the interests of the donors, endangers the health of recipients, and undermines the healthy development of the international transplant endeavor.
  • [MeSH-major] Financing, Organized / legislation & jurisprudence. Government Regulation. Health Policy / economics. Kidney Transplantation / economics. Living Donors / legislation & jurisprudence. Tissue and Organ Procurement / economics
  • [MeSH-minor] Altruism. Cost-Benefit Analysis. Crime / prevention & control. Gift Giving. Health Care Costs. Health Services Accessibility / economics. Health Services Accessibility / legislation & jurisprudence. Health Services Needs and Demand / economics. Health Services Needs and Demand / legislation & jurisprudence. Humans. Motivation. Organizational Objectives. Public Opinion. Renal Dialysis / economics. Risk Assessment. United States. Waiting Lists

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  • (PMID = 18685334.001).
  • [ISSN] 1531-7013
  • [Journal-full-title] Current opinion in organ transplantation
  • [ISO-abbreviation] Curr Opin Organ Transplant
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 37
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28. Bang H, Vupputuri S, Shoham DA, Klemmer PJ, Falk RJ, Mazumdar M, Gipson D, Colindres RE, Kshirsagar AV: SCreening for Occult REnal Disease (SCORED): a simple prediction model for chronic kidney disease. Arch Intern Med; 2007 Feb 26;167(4):374-81
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] SCreening for Occult REnal Disease (SCORED): a simple prediction model for chronic kidney disease.
  • BACKGROUND: Despite the wide availability and low cost of serum creatinine measurement, at-risk populations are not routinely tested for chronic kidney disease (CKD).
  • METHODS: We used a cross-sectional analysis of a nationally representative, population-based survey to develop a system, SCORED (SCreening for Occult REnal Disease), that uses routinely available demographic and medical information to identify individuals with an increased likelihood of CKD.
  • Chronic kidney disease was defined as a glomerular filtration rate less than 60 mL/min per 1.73 m(2).
  • CONCLUSION: This scoring system, weighted toward common variables associated with CKD, may be a useful tool to identify individuals with a high likelihood of occult kidney disease.
  • [MeSH-major] Creatinine / blood. Kidney Failure, Chronic / diagnosis. Mass Screening / methods. Models, Biological. Population Surveillance


29. Barbari AG, Masri MA, Stephan AG, El Ghoul B, Rizk S, Mourad N, Kamel GS, Kilani HE, Karam AS: Cyclosporine lymphocyte maximum level monitoring in de novo kidney transplant patients: a prospective study. Exp Clin Transplant; 2006 Jun;4(1):400-5
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  • [Title] Cyclosporine lymphocyte maximum level monitoring in de novo kidney transplant patients: a prospective study.
  • OBJECTIVES: To determine prospectively the temporal variations of cyclosporine-A lymphocyte maximum level, whole blood maximum concentration, and total lymphocyte count in patients with de novo kidney transplantation.
  • MATERIALS AND METHODS: Lymphocyte maximum level, whole blood maximum concentration, and total lymphocyte count were prospectively measured in 35 patients at 1, 2, and 3 months after kidney transplantation.
  • Cyclosporine-A lymphocyte maximum level seems to offer a more reliable alternative than does whole blood maximum concentration for cyclosporine-A monitoring in patients with kidney transplantation.
  • [MeSH-major] Cyclosporine / blood. Immunosuppressive Agents / blood. Kidney Transplantation. Lymphocytes / metabolism

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  • (PMID = 16827634.001).
  • [ISSN] 1304-0855
  • [Journal-full-title] Experimental and clinical transplantation : official journal of the Middle East Society for Organ Transplantation
  • [ISO-abbreviation] Exp Clin Transplant
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Turkey
  • [Chemical-registry-number] 0 / Immunosuppressive Agents; 83HN0GTJ6D / Cyclosporine
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30. van der Mei SF, van Sonderen EL, van Son WJ, de Jong PE, Groothoff JW, van den Heuvel WJ: Social participation after successful kidney transplantation. Disabil Rehabil; 2007 Mar 30;29(6):473-83
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  • [Title] Social participation after successful kidney transplantation.
  • PURPOSE: To explore and describe the degree of social participation after kidney transplantation and to examine associated factors.
  • METHOD: A cross-sectional study on 239 adult patients 1-7.3 years after kidney transplantation was performed via in-home interviews on participation in obligatory activities (i.e., employment, education, household tasks) and leisure activities (volunteer work, assisting others, recreation, sports, clubs/associations, socializing, going out).
  • RESULTS: Kidney transplantation patients had a lower educational level, spent less time on obligatory activities, had part-time jobs more often, and participated less in sports compared to a control group from the general population.
  • CONCLUSIONS: Although kidney transplantation patients participate less in employment and sports, they do participate in household tasks, volunteer work, going out, socializing and other leisure activities.
  • [MeSH-major] Activities of Daily Living. Employment. Interpersonal Relations. Kidney Transplantation. Leisure Activities

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  • (PMID = 17364802.001).
  • [ISSN] 0963-8288
  • [Journal-full-title] Disability and rehabilitation
  • [ISO-abbreviation] Disabil Rehabil
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
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31. Hahn H: Genetics of kidney development: pathogenesis of renal anomalies. Korean J Pediatr; 2010 Jul;53(7):729-34
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  • [Title] Genetics of kidney development: pathogenesis of renal anomalies.
  • Congenital anomalies of the kidney and urinary tract (CAKUT) account for more than 50% of abdominal masses found in neonates and involve about 0.5% of all pregnancies.
  • CAKUT has a major role in renal failure, and increasing evidence suggests that certain abnormalities predispose to the development of hypertension and cardiovascular disease in adulthood.
  • To understand the pathogenesis of human renal anomalies, understanding the development of kidney is important.
  • Diverse anomalies of the kidney corresponding to defects at a particular stage of development have been documented recently; however, more research is required to understand the molecular networks underlying kidney development, and such an investigation will provide a clue to the therapeutic intervention for CAKUT.

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  • (PMID = 21189947.001).
  • [ISSN] 2092-7258
  • [Journal-full-title] Korean journal of pediatrics
  • [ISO-abbreviation] Korean J Pediatr
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Korea (South)
  • [Other-IDs] NLM/ PMC3004483
  • [Keywords] NOTNLM ; Congenital anomalies / Development / Kidney / Urinary tract
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32. Colović R, Colović N, Grubor N, Radak V, Micev M, Stojković M: [Bilateral angiomyolipoma of the kidney in patient with tuberous sclerosis]. Srp Arh Celok Lek; 2005 Sep-Oct;133(9-10):433-7
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  • [Title] [Bilateral angiomyolipoma of the kidney in patient with tuberous sclerosis].
  • Angiomyolipomas are relatively frequent tumours of the kidney.
  • It is believed that about 10 million people worldwide have such a tumour.
  • Due to the benign nature of angiomyolipomas, surgical treatment and embolisation of the tumours are generally not recommended, unless renal function is endangered, the symptoms are severe, or the kidney in question becomes completely dysfunctional.
  • We present a 24-year-old woman with tuberous sclerosis in whom bilateral kidney tumours were diagnosed 7 years earlier and in whom we carried out a left nephrectomy of a 5300 gram angiomyolipoma, which caused pain and complete loss of function.
  • Although tumourous, the right kidney was functional, so it was left untouched.
  • After an uneventful recovery, a close follow-up was recommended, as well as HLA typing, as it is highly probable that the right kidney will gradually become inadequate or completely dysfunctional, so that haemodialysis and/or kidney transplantation along with nephrectomy will become necessary.
  • [MeSH-major] Angiomyolipoma / complications. Kidney Neoplasms / complications. Tuberous Sclerosis / complications

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  • (PMID = 16640189.001).
  • [ISSN] 0370-8179
  • [Journal-full-title] Srpski arhiv za celokupno lekarstvo
  • [ISO-abbreviation] Srp Arh Celok Lek
  • [Language] srp
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Serbia and Montenegro
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33. Rouquie D, Eggenspieler P, Algayres JP, Béchade D, Camparo P, Baranger B: [Malignant-like angiomyolipoma of the liver: report of one case and review of the literature]. Ann Chir; 2006 May;131(5):338-41
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  • Frequently found in kidney, angiomyolipoma is a rare mesenchymal tumor when diagnosed in the liver and usually benign composed of proliferative blood vessels, fatty tissue and smooth muscle.
  • We report the case of a 67-year-old woman who underwent a left hepatectomy for a 4th segment tumor unidentified after imaging and fine needle biopsy.
  • A review of the literature found only two cases of malignant hepatic angiomyolipoma with fatal issue, however, their incidence must be underrated because of their scarcity and the difficulty of their diagnosis, which needs immunohistochemical confirmation with HMB 45 in particularly.
  • The latter which are important for a correct diagnosis, in order to obtain reliable data about frequency, possible malignant behaviour and therefore consensus management for hepatic angiomyolipoma.
  • [MeSH-major] Angiomyolipoma / surgery. Liver Neoplasms / surgery

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  • (PMID = 16386232.001).
  • [ISSN] 0003-3944
  • [Journal-full-title] Annales de chirurgie
  • [ISO-abbreviation] Ann Chir
  • [Language] fre
  • [Publication-type] Case Reports; English Abstract; Journal Article; Review
  • [Publication-country] France
  • [Number-of-references] 16
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34. Shah SV, Rajapurkar MM: The role of labile iron in kidney disease and treatment with chelation. Hemoglobin; 2009;33(5):378-85
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  • [Title] The role of labile iron in kidney disease and treatment with chelation.
  • There are two major forms of kidney disease: acute renal failure [also referred to as acute kidney injury (AKI)] and chronic kidney disease (CKD).
  • Acute renal failure is an abrupt loss of kidney function within 48 h, whereas CKD is a loss of kidney function greater than 3 months.
  • There is a large amount of experimental evidence for an increase of labile iron in a wide variety of models of kidney disease.
  • These observations suggest that iron chelators may provide a new modality of prevention and treatment of kidney disease.
  • [MeSH-major] Acute Kidney Injury / drug therapy. Acute Kidney Injury / metabolism. Iron / metabolism. Iron Chelating Agents / therapeutic use. Kidney Diseases / drug therapy. Kidney Diseases / metabolism

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  • (PMID = 19821781.001).
  • [ISSN] 1532-432X
  • [Journal-full-title] Hemoglobin
  • [ISO-abbreviation] Hemoglobin
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Iron Chelating Agents; E1UOL152H7 / Iron
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35. Palmer BF: Hypertension management in patients with chronic kidney disease. Curr Hypertens Rep; 2008 Oct;10(5):367-73
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  • [Title] Hypertension management in patients with chronic kidney disease.
  • Hypertension is one of the major risk factors for the development and progression of chronic kidney disease.
  • The loss of renal function leads to impaired renal autoregulation and renders the kidney vulnerable to the damaging effects of uncontrolled hypertension.
  • Mounting evidence indicates that angiotensin-converting enzyme inhibitors and angiotensin receptor blockers slow the progression of chronic kidney disease through effects beyond lowering blood pressure.
  • Studies are needed to determine whether high doses of the single agent or combination therapy is most effective in providing renal protection.
  • Urinary protein excretion is a useful tool for monitoring and titrating therapy to maximize renal protection.
  • Changes in the serum creatinine concentration and hyperkalemia are complications of antihypertensive therapy in patients with chronic kidney disease that can be successfully managed to allow continued use of renin-angiotensin blockade.
  • [MeSH-major] Hypertension / prevention & control. Renal Insufficiency, Chronic / etiology. Renal Insufficiency, Chronic / prevention & control

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  • (PMID = 18775113.001).
  • [ISSN] 1534-3111
  • [Journal-full-title] Current hypertension reports
  • [ISO-abbreviation] Curr. Hypertens. Rep.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antihypertensive Agents; EC 3.4.23.15 / Renin
  • [Number-of-references] 40
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36. Wołyniec W, Zdrojewski Z, Rutkowski B: [Metabolic acidosis after kidney transplantation]. Przegl Lek; 2005;62(1):68-71
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  • [Title] [Metabolic acidosis after kidney transplantation].
  • Metabolic acidosis is a common complication in patients after kidney transplantation.
  • It is caused by inability of transplanted kidney to regenerate bicarbonate buffer.
  • Although every type of acidosis may be present in these patients, the most frequent are renal tubular acidosis and uremic acidosis.
  • Ketoacidosis due to a post-transplant diabetes mellitus and bicarbonate loss in patients after simultaneous pancreas/kidney transplantation are rarely observed.
  • The proper diagnosis and appropriate treatment with oral supplements are essential.
  • [MeSH-major] Acidosis, Renal Tubular / metabolism. Kidney Transplantation / adverse effects

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  • (PMID = 16053226.001).
  • [ISSN] 0033-2240
  • [Journal-full-title] Przegla̧d lekarski
  • [ISO-abbreviation] Prz. Lek.
  • [Language] pol
  • [Publication-type] English Abstract; Journal Article; Review
  • [Publication-country] Poland
  • [Chemical-registry-number] 0 / Bicarbonates
  • [Number-of-references] 28
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37. el-Assmy A, el-Nahas AR, Hekal IA, Badran M, Youssef RF, Sheir KZ: Long-term effects of extracorporeal shock wave lithotripsy on renal function: our experience with 156 patients with solitary kidney. J Urol; 2008 Jun;179(6):2229-32
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  • [Title] Long-term effects of extracorporeal shock wave lithotripsy on renal function: our experience with 156 patients with solitary kidney.
  • PURPOSE: We studied the long-term impact of shock wave lithotripsy on renal function, stone recurrence and hypertension in patients with a solitary kidney.
  • Patients with a solitary kidney provide a unique opportunity to evaluate any clinically significant change in renal function.
  • MATERIALS AND METHODS: We retrospectively reviewed the records of 156 patients with stones in a solitary kidney treated with shock wave lithotripsy monotherapy.
  • Serum creatinine, systolic and diastolic blood pressure, new onset hypertension, calculated glomerular filtration rate, and kidney morphology were determined before and after treatment, and compared by chi-square, paired and unpaired t tests.
  • Renal obstruction caused by steinstrasse after shock wave lithotripsy occurred in 14 (8.9%) patients.
  • CONCLUSIONS: The demonstrated effectiveness, small number of complications at short-term followup, insignificant effect on renal function, blood pressure and relatively small number of recurrences at the long-term followup confirm that shock wave lithotripsy is not only effective but is also safe in the long run.
  • [MeSH-major] Kidney / abnormalities. Kidney / physiopathology. Kidney Calculi / physiopathology. Kidney Calculi / therapy. Kidney Failure, Chronic

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  • (PMID = 18423733.001).
  • [ISSN] 1527-3792
  • [Journal-full-title] The Journal of urology
  • [ISO-abbreviation] J. Urol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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38. Aydin H, Magi-Galluzzi C, Lane BR, Sercia L, Lopez JI, Rini BI, Zhou M: Renal angiomyolipoma: clinicopathologic study of 194 cases with emphasis on the epithelioid histology and tuberous sclerosis association. Am J Surg Pathol; 2009 Feb;33(2):289-97
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  • [Title] Renal angiomyolipoma: clinicopathologic study of 194 cases with emphasis on the epithelioid histology and tuberous sclerosis association.
  • The majority of renal angiomyolipoma (AML) is sporadic and occasionally it occurs as part of tuberous sclerosis complex (TSC).
  • Histologically, the epithelioid tumor cells were categorized into small, intermediate, and large cell type based on the cell size.
  • Worrisome histologic features were seen in many EAMLs, including coagulative tumor necrosis in 27% (4/15), nuclear atypia in 93% (14/15), mitosis in 47% (7/15), and atypical mitosis in 1 case.
  • In summary, all 15 cases of EAMLs in our study had benign clinical outcomes despite adverse pathologic features.
  • [MeSH-major] Angiomyolipoma / pathology. Epithelioid Cells / pathology. Kidney Neoplasms / pathology. Tuberous Sclerosis / pathology


39. Brown CT, Kooiman G, Sharma DM, Poulsen J, Grange P: Scarless single-port laparoscopic pelvic kidney nephrectomy. J Laparoendosc Adv Surg Tech A; 2010 Nov;20(9):743-6
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  • [Title] Scarless single-port laparoscopic pelvic kidney nephrectomy.
  • INTRODUCTION: We report the first pelvic kidney removal through the umbilicus using a scarless pure single-port technique in a young woman.
  • PATIENTS AND METHODS: A 27-year-old woman presented with uro-sepsis and acute renal failure secondary to a dilated, chronically infected, nonfunctioning left-sided pelvic kidney with ureteropelvic obstruction causing an obstruction to the right kidney.
  • Definitive treatment involved removal of the diseased pelvic kidney through the umbilicus via a single-port access device (TriPor™; Olympus).
  • [MeSH-major] Acute Kidney Injury / surgery. Kidney Pelvis / surgery. Laparoscopy / methods. Nephrectomy / methods. Ureteral Obstruction / surgery

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  • (PMID = 20874248.001).
  • [ISSN] 1557-9034
  • [Journal-full-title] Journal of laparoendoscopic & advanced surgical techniques. Part A
  • [ISO-abbreviation] J Laparoendosc Adv Surg Tech A
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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40. Cassuto JR, Reese PP, Sonnad S, Bloom RD, Levine MH, Olthoff KM, Shaked A, Naji A, Abt P: Wait list death and survival benefit of kidney transplantation among nonrenal transplant recipients. Am J Transplant; 2010 Nov;10(11):2502-11
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  • [Title] Wait list death and survival benefit of kidney transplantation among nonrenal transplant recipients.
  • The disparity between the number of patients waiting for kidney transplantation and the limited supply of kidney allografts has renewed interest in the benefit from kidney transplantation experienced by different groups.
  • This study evaluated kidney transplant survival benefit in prior nonrenal transplant recipients (kidney after liver, KALi; lung, KALu; heart, KAH) compared to primary isolated (KA1) or repeat isolated kidney (KA2) transplant.
  • Following kidney transplant, patient survival was greatest for KA1, similar among KA2, KALi, KAH, and inferior for KALu.
  • Compared to the entire wait list, renal transplantation was associated with a survival benefit among all groups except KALu (p = 0.017; HR = 1.61; CI = 1.09-2.38), where posttransplant survival was inferior to the wait list population.
  • Recipients of KA1 kidney transplantation have the greatest posttransplant survival and compared to the overall kidney wait list, the greatest survival benefit.

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  • [Copyright] ©2010 The Authors Journal compilation©2010 The American Society of Transplantation and the American Society of Transplant Surgeons.
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  • (PMID = 20977641.001).
  • [ISSN] 1600-6143
  • [Journal-full-title] American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons
  • [ISO-abbreviation] Am. J. Transplant.
  • [Language] ENG
  • [Grant] United States / NIDDK NIH HHS / DK / K23 DK078688; United States / NIDDK NIH HHS / DK / K23 DK078688-01
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Other-IDs] NLM/ NIHMS235283; NLM/ PMC2966021
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46. Yusenko MV, Kuiper RP, Boethe T, Ljungberg B, van Kessel AG, Kovacs G: High-resolution DNA copy number and gene expression analyses distinguish chromophobe renal cell carcinomas and renal oncocytomas. BMC Cancer; 2009;9:152
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  • [Title] High-resolution DNA copy number and gene expression analyses distinguish chromophobe renal cell carcinomas and renal oncocytomas.
  • BACKGROUND: The diagnosis of benign renal oncocytomas (RO) and chromophobe renal cell carcinomas (RCC) based on their morphology remains uncertain in several cases.
  • CONCLUSION: The SNP-array analysis failed to detect recurrent small deletions, which may mark loci of genes involved in the tumor development.
  • Therefore, detection of these chromosomal changes can be used for the accurate diagnosis in routine histology.
  • [MeSH-major] Adenoma, Oxyphilic / genetics. Carcinoma, Renal Cell / genetics. Gene Dosage. Gene Expression Profiling. Kidney Neoplasms / genetics
  • [MeSH-minor] Chromosomes, Human / genetics. Humans. Oligonucleotide Array Sequence Analysis. Tumor Cells, Cultured

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  • (PMID = 19445733.001).
  • [ISSN] 1471-2407
  • [Journal-full-title] BMC cancer
  • [ISO-abbreviation] BMC Cancer
  • [Language] eng
  • [Databank-accession-numbers] GEO/ GSE11151
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC2686725
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47. Ix JH, De Boer IH, Peralta CA, Adeney KL, Duprez DA, Jenny NS, Siscovick DS, Kestenbaum BR: Serum phosphorus concentrations and arterial stiffness among individuals with normal kidney function to moderate kidney disease in MESA. Clin J Am Soc Nephrol; 2009 Mar;4(3):609-15
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  • [Title] Serum phosphorus concentrations and arterial stiffness among individuals with normal kidney function to moderate kidney disease in MESA.
  • DESIGN, SETTING, PARTICIPANTS, & MEASUREMENTS: This study examined the cross-sectional associations of serum phosphorus with ankle brachial index (ABI), pulse pressure, and large and small artery elasticity by radial artery waveform analysis among 1370 individuals (440 with moderate chronic kidney disease) who did not have clinical CVD and participated in the Multi-Ethnic Study of Atherosclerosis.
  • Participants with phosphorus levels >4 mg/dl had greater than four-fold risk for high ABI compared with participants with phosphate levels <3 mg/dl (relative risk 4.6; 95% confidence interval 1.6 to 13.2; P = 0.01) after adjustment for demographics, traditional CVD risk factors, and kidney function.

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  • (PMID = 19211667.001).
  • [ISSN] 1555-905X
  • [Journal-full-title] Clinical journal of the American Society of Nephrology : CJASN
  • [ISO-abbreviation] Clin J Am Soc Nephrol
  • [Language] ENG
  • [Grant] United States / NHLBI NIH HHS / HL / N01HC95169; United States / NHLBI NIH HHS / HC / N01-HC-95165; United States / NHLBI NIH HHS / HC / N01-HC-95169; United States / NHLBI NIH HHS / HL / N01HC95165; United States / NHLBI NIH HHS / HL / N01HC95159
  • [Publication-type] Journal Article; Multicenter Study; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 27YLU75U4W / Phosphorus
  • [Other-IDs] NLM/ PMC2653665
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48. Berger A, Preiser W, Kachel HG, Stürmer M, Doerr HW: HBV reactivation after kidney transplantation. J Clin Virol; 2005 Feb;32(2):162-5
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  • [Title] HBV reactivation after kidney transplantation.
  • In this study, we reviewed the virologic laboratory records from kidney recipients.
  • Out of 1512 patients, 228 had been diagnosed with resolved HBV infection (anti-HBc positive, HBsAg negative) but normal liver enzyme levels prior to kidney transplantation.
  • Reappearance of HBsAg after kidney transplantation was observed in two (0.9%) of those patients, which may be attributed to reactivation of a latent infection or to a new HBV infection.
  • Periodic follow-up of HBV serology for early diagnosis of reactivation is highly recommended in transplant recipients.
  • [MeSH-major] Hepatitis B / virology. Hepatitis B Surface Antigens / blood. Hepatitis B virus / physiology. Kidney Transplantation / adverse effects. Virus Activation

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  • (PMID = 15653420.001).
  • [ISSN] 1386-6532
  • [Journal-full-title] Journal of clinical virology : the official publication of the Pan American Society for Clinical Virology
  • [ISO-abbreviation] J. Clin. Virol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Hepatitis B Surface Antigens
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49. Genberg H, Hansson A, Wernerson A, Wennberg L, Tydén G: Pharmacodynamics of rituximab in kidney allotransplantation. Am J Transplant; 2006 Oct;6(10):2418-28
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  • [Title] Pharmacodynamics of rituximab in kidney allotransplantation.
  • The anti-CD20 antibody rituximab has recently gained interest as a B-cell depleting agent in renal transplantation.
  • However, little is known about the pharmacodynamics of rituximab in renal transplant recipients.
  • A total of 49 renal transplant recipients received single-dose rituximab, as induction therapy (n = 36) or as anti-rejection therapy (n = 13).
  • We counted B cells in peripheral blood and performed immunohistochemical staining on lymph nodes and kidney transplant tissue samples to assess the prevalence of B cells.
  • The immunohistochemical staining showed a complete elimination of B cells in kidney tissue and a reduction of B cells in lymph nodes.
  • In conclusion, single-dose rituximab in kidney transplant recipients evokes a long-term elimination of B cells in peripheral blood as well as within the kidney transplant.
  • [MeSH-major] Antibodies, Monoclonal / pharmacokinetics. Graft Rejection / drug therapy. Immunologic Factors / pharmacokinetics. Kidney Transplantation / pathology

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  • (PMID = 16925569.001).
  • [ISSN] 1600-6135
  • [Journal-full-title] American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons
  • [ISO-abbreviation] Am. J. Transplant.
  • [Language] eng
  • [Databank-accession-numbers] ClinicalTrials.gov/ NCT00255593
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Denmark
  • [Chemical-registry-number] 0 / Antibodies, Monoclonal; 0 / Antibodies, Monoclonal, Murine-Derived; 0 / Antigens, CD19; 0 / Antigens, CD20; 0 / Immunologic Factors; 4F4X42SYQ6 / Rituximab
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50. Montagner J, Michelon T, Fontanelle B, Oliveira A, Silveira J, Schroeder R, Neumann J, Keitel E, Alexandre CO: BKV-infection in kidney graft dysfunction. Braz J Infect Dis; 2010 Mar-Apr;14(2):170-4
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  • [Title] BKV-infection in kidney graft dysfunction.
  • INTRODUCTION: BKV nephropathy (BKN) causes kidney graft loss, whose specific diagnosis is invasive and might be predicted by the early detection of active viral infection.
  • OBJECTIVE: Determine the BKV-infection prevalence in late kidney graft dysfunction by urinary decoy cell (DC) and viral DNA detection in urine (viruria) and blood (viremia; active infection).
  • METHODS: Kidney recipients with >1 month follow-up and creatinine >1.5 mg/dL and/or recent increasing >20% (n = 120) had their urine and blood tested for BKV by semi-nested PCR, DC searching, and graft biopsy.
  • Diagnosis efficacy of DC and viruria were compared to viremia.
  • Intense viruria was the single predictive test for active infection (3+; LR = 2.8).1,6-4,9 CONCLUSION: DC, BKV-viruria and -viremia are commun findings under late kidney graft dysfunction.
  • [MeSH-major] BK Virus / isolation & purification. Kidney Transplantation / adverse effects. Polyomavirus Infections / diagnosis. Primary Graft Dysfunction / virology. Tumor Virus Infections / diagnosis

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  • (PMID = 20563444.001).
  • [ISSN] 1678-4391
  • [Journal-full-title] The Brazilian journal of infectious diseases : an official publication of the Brazilian Society of Infectious Diseases
  • [ISO-abbreviation] Braz J Infect Dis
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Brazil
  • [Chemical-registry-number] 0 / DNA, Viral
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51. Szeto CC, Kwan BC, Lai KB, Lai FM, Chow KM, Wang G, Luk CC, Li PK: Urinary expression of kidney injury markers in renal transplant recipients. Clin J Am Soc Nephrol; 2010 Dec;5(12):2329-37
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  • [Title] Urinary expression of kidney injury markers in renal transplant recipients.
  • BACKGROUND AND OBJECTIVES: The outcome of renal transplantation after an episode of acute rejection is difficult to predict, even with an allograft biopsy.
  • We examined whether urinary expression of specific biomarker mRNA could be used as a noninvasive prognostic marker in kidney transplant recipients.
  • DESIGN, SETTING, PARTICIPANTS, & MEASUREMENTS: We studied 63 kidney transplant recipients who require graft biopsy because of progressive worsening of kidney function.
  • The mRNA of neutrophil gelatinase-associated lipocalin, kidney injury molecule-1 (KIM-1), IL-18, surfactant protein-C, and S100 calcium-binding proteins A8 and A9 in urinary sediment were quantified.
  • After followed for an average of 39.7 ± 21.1 months, the rate of renal function decline significantly correlated with urinary KIM-1 expression (r = -0.434, P = 0.0004) but not other target genes.
  • CONCLUSIONS: In kidney allograft recipients, urinary KIM-1 expression provides prognostic information in relation to the rate of renal function decline, irrespective of the kidney pathology.
  • [MeSH-major] Acute Kidney Injury / urine. Biomarkers / urine. Kidney Transplantation

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  • (PMID = 20671224.001).
  • [ISSN] 1555-905X
  • [Journal-full-title] Clinical journal of the American Society of Nephrology : CJASN
  • [ISO-abbreviation] Clin J Am Soc Nephrol
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Acute-Phase Proteins; 0 / Biomarkers; 0 / Calgranulin A; 0 / HAVCR1 protein, human; 0 / Interleukin-18; 0 / LCN2 protein, human; 0 / Lipocalins; 0 / Membrane Glycoproteins; 0 / Proto-Oncogene Proteins; 0 / Pulmonary Surfactant-Associated Protein C; 0 / RNA, Messenger; 0 / Receptors, Virus; 0 / SFTPC protein, human
  • [Other-IDs] NLM/ PMC2994096
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52. Moudouni SM, Lakmichi A, Tligui M, Rafii A, Tchala K, Haab F, Gattegno B, Thibault P, Doublet JD: Renal cell carcinoma of native kidney in renal transplant recipients. BJU Int; 2006 Aug;98(2):298-302
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  • [Title] Renal cell carcinoma of native kidney in renal transplant recipients.
  • OBJECTIVE: To evaluate the prevalence, prognosis and possible risk factors of renal cell carcinoma (RCC) of the native kidney in renal transplant recipients.
  • PATIENTS AND METHODS: We retrospectively re-examined the follow-up data of 373 consecutive renal transplant recipients at our institution between August 1993 and September 2004.
  • We collected the data of all de novo RCC of the native kidney in the current analysis.
  • RESULTS: Of the 373 patients examined, 12 tumours of the native kidney were diagnosed in 10 individuals.
  • The mean ages at transplantation and diagnosis were 33 and 45.8 years, respectively.
  • Among the renal ultrasonograms there were two false-negative results.
  • The mean tumour size was 21 mm.
  • Among the 12 kidney malignancies, there were five conventional RCCs and seven papillary RCCs.
  • One of the 10 patients died, from progression of metastases 6 years after diagnosis.
  • One patient had a local recurrence 2 years after diagnosis.
  • CONCLUSIONS: There appears to be a greater risk of RCC of the native kidney in patients with end-stage renal disease.
  • The present results suggest that an annual examination of the native kidney before and after renal transplantation is essential.
  • [MeSH-major] Carcinoma, Renal Cell / etiology. Kidney Failure, Chronic / surgery. Kidney Neoplasms / etiology. Kidney Transplantation. Postoperative Complications / etiology


53. Chang MY, Ong AC: Autosomal dominant polycystic kidney disease: recent advances in pathogenesis and treatment. Nephron Physiol; 2008;108(1):p1-7
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  • [Title] Autosomal dominant polycystic kidney disease: recent advances in pathogenesis and treatment.
  • Autosomal dominant polycystic kidney disease (ADPKD) is a common genetic disorder affecting 1 in 1,000 people in the general population and accounts for up to 10% of all patients on renal replacement therapy.
  • Numerous fluid-filled epithelial cysts arise from different nephron segments as spherical dilatations or small out-pouchings, enlarge progressively and eventually become disconnected from the rest of the renal tubule.
  • The development of cysts is accompanied by destruction of the renal parenchyma, interstitial fibrosis, cellular infiltration and loss of functional nephrons.
  • ADPKD is not only a kidney disease but also a systemic disorder associated with intracranial arterial aneurysms, cardiac valvular defects, colonic diverticulosis and cyst formation in other organs such as the liver, spleen and pancreas.
  • The identification of PKD1 and PKD2 together with the drive to elucidate the functions of their encoded proteins, polycystin-1 (PC1) and polycystin-2 (PC2), has led to an explosion of clinical and scientific interest in this common disorder.
  • [MeSH-major] Polycystic Kidney, Autosomal Dominant / etiology. Polycystic Kidney, Autosomal Dominant / therapy


54. Stasević Z, Gorgieva GS, Vasić S, Ristić S, Djukanović L, Lezaić V: High prevalence of kidney disease in two rural communities in Kosovo and Metohia. Ren Fail; 2010 Jun;32(5):541-6
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  • [Title] High prevalence of kidney disease in two rural communities in Kosovo and Metohia.
  • A systematic survey was carried out in an enclave in Kosovo and Metohia, with the aim of assessing the prevalence of kidney diseases.
  • Persons with any detected disorder indicating kidney disease were invited for additional examination of kidney function.
  • Glomerular filtration rate (Modification of Diet in Renal Disease (MDRD) formula) below 60 mL/min/1.73 m(2) was found in 5.2% of subjects.
  • Kidney ultrasound examination detected reduced length of right and left kidneys in 38 and 24 persons, respectively.
  • Cysts were also a frequent finding, but polycystic kidney, hydronephrosis, and kidney stones were found in about 2% each.
  • The analysis of data obtained by the present examination and available medical documentation revealed kidney and urinary tract diseases in 98 persons: 52 patients with already known disease and 46 patients detected in the survey.
  • Out of them in 22 patients diagnosis of kidney disease could not be established during the survey but laboratory analyses indicated that they might suffer from tubulointerstitial disease: 14 had tubular dysfunctions, 8 of them low-grade proteinuria, and 12 had a positive family history for kidney disease.
  • In the enclave of Velika Hoca and Orahovac the prevalence of kidney disease was 7.0% indicating that these communities might be placed among those with a high prevalence of kidney disease in Serbia.
  • [MeSH-major] Kidney Diseases / epidemiology

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  • (PMID = 20486835.001).
  • [ISSN] 1525-6049
  • [Journal-full-title] Renal failure
  • [ISO-abbreviation] Ren Fail
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
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55. Kruck S, Bedke J, Hennenlotter J, Ohneseit PA, Kuehs U, Senger E, Sievert KD, Stenzl A: Activation of mTOR in renal cell carcinoma is due to increased phosphorylation rather than protein overexpression. Oncol Rep; 2010 Jan;23(1):159-63
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  • [Title] Activation of mTOR in renal cell carcinoma is due to increased phosphorylation rather than protein overexpression.
  • The aim of the present pilot study was to determine mTOR expression in clear cell renal cell carcinoma (RCC) and to evaluate mTOR activation and phosphorylation at Ser2448.
  • Tissue microarray immunohistochemistry and Western blot analysis of tumor and benign tissue from 10 patients subjected to tumor nephrectomy were investigated.
  • Ratios of tumor versus benign tissue were calculated and compared by the Wilcoxon/Kruskal-Wallis test.
  • Immunohistochemical expressions of mTOR and p-mTOR were 49 and 40% in benign renal parenchyma, whereas it was 20 and 42% in tumor tissue.
  • Ratios of tumor versus benign tissue revealed a reduction to 0.44 for mTOR and corresponding elevation to 1.29 for p-mTOR (p<0.05).
  • The rate of p-mTOR to mTOR was 1.19 in benign, whereas it was 5.30 in tumor tissue.
  • Western blot densitometry detected lower expressions of mTOR in tumor compared to benign tissues.
  • Ratio of p-mTOR to mTOR were significantly different in benign versus tumor tissue (0.86 vs. 1.37; p<0.04).
  • The observation that RCC specimens exhibit higher levels of p-mTOR in RCC compared to benign renal parenchyma indicate the role of mTOR phosphorylation in RCC tumor development and progression.
  • [MeSH-major] Carcinoma, Renal Cell / metabolism. Gene Expression Regulation, Neoplastic. Kidney Neoplasms / metabolism. Protein-Serine-Threonine Kinases / biosynthesis

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  • (PMID = 19956876.001).
  • [ISSN] 1791-2431
  • [Journal-full-title] Oncology reports
  • [ISO-abbreviation] Oncol. Rep.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Greece
  • [Chemical-registry-number] 0 / Intracellular Signaling Peptides and Proteins; EC 2.7.1.1 / MTOR protein, human; EC 2.7.1.1 / TOR Serine-Threonine Kinases; EC 2.7.11.1 / Protein-Serine-Threonine Kinases
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56. Sulejmanagić H, Sulejmanagić N, Prohić S, Secić S, Miseljić S: Dental treatment of patients with kidney diseases-review. Bosn J Basic Med Sci; 2005 Feb;5(1):52-6
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  • [Title] Dental treatment of patients with kidney diseases-review.
  • In their practice every dentist is brought into a situation to treat patients with grossly impaired kidney function.
  • Kidney diseases, whether acute or acquired, imply a number of body dysfunctions such as prolonged bleeding, high blood pressure, infection tendency etc. which, in turn, pose a threat involving serious complications in cases of dental interventions in these patients.
  • The aim of this article is to provide a review of current dental practice in patients with kidney disease.
  • This implies dental intervention and preparations of patients with chronic renal disease, nephritic syndrome, patients on dialysis, and patients with kidney transplants.
  • Certainly, cooperation between the dentist and nephrologist is an imperative for the appropriate dental treatment of patients with grossly impaired renal function.

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  • (PMID = 15771603.001).
  • [ISSN] 1512-8601
  • [Journal-full-title] Bosnian journal of basic medical sciences
  • [ISO-abbreviation] Bosn J Basic Med Sci
  • [Language] ENG
  • [Publication-type] Journal Article; Review
  • [Publication-country] Bosnia and Herzegovina
  • [Number-of-references] 12
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57. Neuhofer W, Pittrow D: Endothelin receptor selectivity in chronic kidney disease: rationale and review of recent evidence. Eur J Clin Invest; 2009 Jun;39 Suppl 2:50-67
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  • [Title] Endothelin receptor selectivity in chronic kidney disease: rationale and review of recent evidence.
  • In addition to its substantial contribution to normal renal function, a large body of evidence suggests that derangement of the renal ET system is involved in the initiation and progression of chronic kidney disease (CKD) in diabetes, hypertension and glomerulonephritis.
  • Recent literature on the role of the renal ET system in the healthy kidney was reviewed.
  • ET-1 acts primarily as an autocrine or paracrine factor in the kidney.
  • In normal physiology, ET-1 promotes diuresis and natriuresis by local production and action through ET(B) receptors in the renal medulla.
  • [MeSH-major] Endothelin Receptor Antagonists. Endothelin-1 / physiology. Kidney Failure, Chronic / physiopathology

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  • [ErratumIn] Eur J Clin Invest. 2009 Jul;39(7):630
  • (PMID = 19335747.001).
  • [ISSN] 1365-2362
  • [Journal-full-title] European journal of clinical investigation
  • [ISO-abbreviation] Eur. J. Clin. Invest.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Antihypertensive Agents; 0 / Endothelin A Receptor Antagonists; 0 / Endothelin B Receptor Antagonists; 0 / Endothelin Receptor Antagonists; 0 / Endothelin-1; 0 / Receptors, Endothelin
  • [Number-of-references] 125
  • [General-notes] NLM/ Original DateCompleted: 20090407
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58. Kim YW, Park BS, Ryu CH, Park SJ, Kang SW, Kim YH, Song IS, Shon JH: Allopurinol-induced aplastic anemia in a patient with chronic kidney disease. Clin Nephrol; 2009 Feb;71(2):203-6
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  • [Title] Allopurinol-induced aplastic anemia in a patient with chronic kidney disease.
  • We report an unusual case of aplastic anemia associated with allopurinol therapy for hyperuricemia in a patient with chronic kidney disease.
  • A 37-year-old female patient diagnosed with Stage III chronic kidney disease was admitted with pancytopenia.
  • These results suggested a diagnosis of aplastic anemia.
  • [MeSH-major] Allopurinol / adverse effects. Anemia, Aplastic / chemically induced. Enzyme Inhibitors / adverse effects. Kidney Failure, Chronic / complications


59. Peces R, Peces C, Pérez-Dueñas V, Cuesta-López E, Azorín S, Selgas R: Rapamycin reduces kidney volume and delays the loss of renal function in a patient with autosomal-dominant polycystic kidney disease. NDT Plus; 2009 Apr;2(2):133-5
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  • [Title] Rapamycin reduces kidney volume and delays the loss of renal function in a patient with autosomal-dominant polycystic kidney disease.
  • This is the first report of a case of a reduction in kidney volume and preservation of renal function in a patient with autosomal-dominant polycystic kidney disease (ADPKD) receiving rapamycin.
  • A 42-year-old man with ADPKD and a severe persistent bleeding from his solitary left kidney was successfully treated with tranexamic acid (TXA).
  • He also received low-dose rapamycin for 8 months, and this was associated with a 23.5% reduction in kidney volume, improvement and stabilization of renal function, and normalization of haemoglobin levels.
  • When treatment with rapamycin was interrupted, renal function deteriorated within an 8-month period and haemodialysis (HD) became necessary.
  • Kidney volume increased at once, and life-threatening bleeding prompted a nephrectomy 4 months after the onset of HD.
  • These data suggest that the reduction in kidney volume and preservation of renal function with rapamycin could be the result of the antiangiogenic, antiproliferative effects of rapamycin.

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  • (PMID = 25949309.001).
  • [ISSN] 1753-0784
  • [Journal-full-title] NDT plus
  • [ISO-abbreviation] NDT Plus
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC4421355
  • [Keywords] NOTNLM ; ADPKD / mTOR / rapamycin / renal function / volume change
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60. Lee M, Partridge NC: Parathyroid hormone signaling in bone and kidney. Curr Opin Nephrol Hypertens; 2009 Jul;18(4):298-302
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  • [Title] Parathyroid hormone signaling in bone and kidney.
  • PURPOSE OF REVIEW: Parathyroid hormone (PTH) maintains a physiological balance of calcium and phosphate concentrations by binding to its receptor on the plasma membrane of cells in bone and kidney.
  • Here, we will review the recent advancements regarding PTH signaling in bone and kidney.
  • In kidney, sodium-hydrogen exchanger regulatory factor 1, originally known for its role in the retention of NaPi-IIa at the apical membrane, was shown to have multiple roles in PTH signaling, both as a mediator and regulator.
  • SUMMARY: PTH activates a number of different signaling pathways by binding to a single receptor in bone and kidney.
  • [MeSH-major] Bone and Bones / metabolism. Kidney / metabolism. Parathyroid Hormone / physiology. Signal Transduction / physiology

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  • (PMID = 19395963.001).
  • [ISSN] 1473-6543
  • [Journal-full-title] Current opinion in nephrology and hypertension
  • [ISO-abbreviation] Curr. Opin. Nephrol. Hypertens.
  • [Language] eng
  • [Grant] United States / NIDDK NIH HHS / DK / DK47420
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Parathyroid Hormone; 0 / Phosphoproteins; 0 / Receptor, Parathyroid Hormone, Type 1; 0 / Sodium-Hydrogen Antiporter; 0 / sodium-hydrogen exchanger regulatory factor; EC 2.7.11.11 / Cyclic AMP-Dependent Protein Kinases; EC 2.7.11.13 / Protein Kinase C; SY7Q814VUP / Calcium
  • [Number-of-references] 58
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61. Gerosa C, Fanni D, Nemolato S, Locci A, Marinelli V, Cabras T, Messana I, Castagnola M, Monga G, Fanos V, Faa G: Thymosin beta-10 expression in developing human kidney. J Matern Fetal Neonatal Med; 2010 Oct;23 Suppl 3:125-8
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  • [Title] Thymosin beta-10 expression in developing human kidney.
  • The report that the Tβ10 gene is expressed at high levels in embryonic human brain as well in human kidney induced us to study Tβ10 reactivity in the preterm kidney in order to verify, at tissue level, the expression of this peptide during renal embryogenesis.
  • To this end, we analyzed, using immunocytochemistry, the expression of Tβ10 in samples of human kidney obtained, at autopsy, from 8 fetuses, 12 preterm infants, ranging from 25 to 36 weeks of gestation and 3 at term newborns.
  • Tβ10 immunoreactivity was detected in 20 out of 22 kidneys examined, and was mainly localized in proximal and distal tubular structures, in the cytoplasm and occasionally in the nuclei of ductal cells.
  • In 13 kidneys, we also observed immunostaining for Tβ10 inside the "comma-shaped bodies" and the "S-shaped bodies" during active glomerulogenesis.
  • Our data show, for the first time, the expression of Tβ10 in the human kidney during the initial phases of its physiological development, mainly restricted in the proximal and the distal tubuli.
  • Further studies are needed in order to better characterize the role of Tβ10 in kidney embryogenesis.
  • [MeSH-major] Kidney / embryology. Kidney / metabolism. Thymosin / metabolism

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  • (PMID = 20836742.001).
  • [ISSN] 1476-4954
  • [Journal-full-title] The journal of maternal-fetal & neonatal medicine : the official journal of the European Association of Perinatal Medicine, the Federation of Asia and Oceania Perinatal Societies, the International Society of Perinatal Obstetricians
  • [ISO-abbreviation] J. Matern. Fetal. Neonatal. Med.
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 61512-21-8 / Thymosin; 87397-91-9 / thymosin beta(10)
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62. Zukowski M, Bohatyrewicz R, Biernawska J, Kotfis K, Zegan M, Knap R, Janeczek M, Zietek Z: Risk factors for septic complications in kidney transplant recipients. Transplant Proc; 2009 Oct;41(8):3043-5
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  • [Title] Risk factors for septic complications in kidney transplant recipients.
  • INTRODUCTION: Septic complications following kidney transplantation are a leading cause of therapeutic failure.
  • An early diagnosis may protect the recipient from the severe consequences of sepsis.
  • We sought to determine the risk factors influencing the occurrence of septic complications among kidney transplant recipients.
  • Recipient data included age, gender, period of prior hemodialysis, panel reactive antibodies, cold ischemia time, and cause of renal insufficiency.
  • The 232 kidney recipients were examined for occurrence of septic complications including septicemia, pneumonia, peritonitis, or graft infection.
  • RESULTS: Kidney transplants from donors with MAP < 70 mm Hg and SVRI < 1200 dyne x s/cm(5) x m(2) showed a significantly higher occurrence of septic complications in recipients (P < .05) where mortality rate was also significantly greater (P < .01).
  • CONCLUSIONS: MAP < 70 mm Hg and SVRI < 1200 dyne x s/cm(5) x m(2) among organ donors predicted greater occurrence of septic complications and increased mortality among kidney transplant recipients.
  • [MeSH-major] Kidney Transplantation / physiology. Sepsis / epidemiology
  • [MeSH-minor] Adolescent. Adult. Blood Pressure / physiology. Brain Death. Cadaver. Cause of Death. Central Venous Pressure / physiology. Child. Female. Heart Rate. Humans. Male. Middle Aged. Renal Dialysis. Risk Factors. Shock, Septic / epidemiology. Tissue Donors. Vascular Resistance. Young Adult

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  • (PMID = 19857672.001).
  • [ISSN] 1873-2623
  • [Journal-full-title] Transplantation proceedings
  • [ISO-abbreviation] Transplant. Proc.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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63. Li L, Okusa MD: Macrophages, dendritic cells, and kidney ischemia-reperfusion injury. Semin Nephrol; 2010 May;30(3):268-77
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  • [Title] Macrophages, dendritic cells, and kidney ischemia-reperfusion injury.
  • Dendritic cells and macrophages are critical early initiators of innate immunity in the kidney and orchestrate inflammation subsequent to ischemia-reperfusion injury.
  • They are the most abundant leukocytes present in the kidney, and they represent a heterogeneous population of cells that are capable of inducing sterile inflammation after reperfusion directly through the production of proinflammatory cytokines and other soluble inflammatory mediators or indirectly through activation of effector T lymphocytes and natural killer T cells.
  • In addition, recent studies have indicated that kidney and immune cell micro-RNAs control gene expression and have the ability to regulate the initial inflammatory response to injury.
  • Although dendritic cells and macrophages contribute to both innate and adaptive immunity and to injury and repair, this review focuses on the initial innate response to kidney ischemia-reperfusion injury.

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  • (PMID = 20620671.001).
  • [ISSN] 1558-4488
  • [Journal-full-title] Seminars in nephrology
  • [ISO-abbreviation] Semin. Nephrol.
  • [Language] ENG
  • [Grant] United States / NIDDK NIH HHS / DK / DK062324-08; United States / NIDDK NIH HHS / DK / DK062324-07; United States / NIDDK NIH HHS / DK / R01 DK056223-08; United States / NIDDK NIH HHS / DK / R01 DK085259; United States / NIDDK NIH HHS / DK / R01DK56223; United States / NIDDK NIH HHS / DK / R01 DK062324; United States / NIDDK NIH HHS / DK / R01 DK056223; United States / NIDDK NIH HHS / DK / R01DK62324; United States / NIDDK NIH HHS / DK / DK056223-08; United States / NIDDK NIH HHS / DK / R01 DK062324-07; United States / NIDDK NIH HHS / DK / R01 DK062324-08
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] United States
  • [Other-IDs] NLM/ NIHMS188223; NLM/ PMC2904394
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64. Bansal N, Hsu CY: Long-term outcomes of patients with chronic kidney disease. Nat Clin Pract Nephrol; 2008 Oct;4(10):532-3
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Long-term outcomes of patients with chronic kidney disease.
  • This Practice Point commentary discusses a recent paper by Menon et al. that described the natural history of a cohort of nondiabetic patients with stage 2-4 chronic kidney disease (CKD) who had been recruited from nephrology practices and screened for entry into the Modification of Diet in Renal Disease (MDRD) trial.
  • Kidney failure was the most common outcome during long-term follow-up among these patients and there was a low competing risk of death, findings in contrast to observations in other cohorts of patients with CKD.
  • Patients with lower glomerular filtration rate and greater proteinuria at baseline were at increased risk for both kidney failure and death, but kidney failure remained more likely than death in all glomerular filtration rate subgroups.
  • Nephrologists should not rely on CKD staging alone to direct management of or risk-stratify patients with CKD, but should also consider the etiology and rate of progression of kidney disease, patient age and cardiovascular disease risk factors.

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  • [CommentOn] Kidney Int. 2008 Jun;73(11):1310-5 [18337713.001]
  • (PMID = 18695707.001).
  • [ISSN] 1745-8331
  • [Journal-full-title] Nature clinical practice. Nephrology
  • [ISO-abbreviation] Nat Clin Pract Nephrol
  • [Language] eng
  • [Grant] United States / NIDDK NIH HHS / DK / K23 DK088865
  • [Publication-type] Comment; Journal Article
  • [Publication-country] England
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65. Obulareddy SJ, Xin J, Truskinovsky AM, Anderson JK, Franklin MJ, Dudek AZ: Metanephric adenoma of the kidney: an unusual diagnostic challenge. Rare Tumors; 2010;2(2):e38
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  • [Title] Metanephric adenoma of the kidney: an unusual diagnostic challenge.
  • Although metanephric adenoma (MA) is a rare, benign neoplasm of epithelial cells, it is often difficult to distinguish this entity from other malignant neoplasms preoperatively.
  • We report a case of a large renal mass for which preoperative diagnosis was indeterminate, with the differential diagnosis including Wilm's tumor, MA, and papillary renal cell carcinoma (PRCC).
  • Accurate postoperative differentiation of MA from PRCC is critical because adjuvant therapy is considered after surgical resection of PRCC tumors.

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  • (PMID = 21139840.001).
  • [ISSN] 2036-3613
  • [Journal-full-title] Rare tumors
  • [ISO-abbreviation] Rare Tumors
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Italy
  • [Other-IDs] NLM/ PMC2994510
  • [Keywords] NOTNLM ; Wilm’s tumor / differential diagnosis. / metanephric adenoma / papillary renal cell carcinoma
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66. Pecoits-Filho R: Dietary protein intake and kidney disease in Western diet. Contrib Nephrol; 2007;155:102-12
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  • [Title] Dietary protein intake and kidney disease in Western diet.
  • Components of the diet related to changes in eating habits that characterize the modern Western world are important factors in the increasingly high prevalence of chronic disease, including obesity, diabetes, hypertension and as a consequence, chronic kidney disease.
  • Unfortunately, very few clinical studies focused on diet-based strategies of prevention of kidney disorders.
  • Furthermore, this review will propose that the concept that protein restricted diets decrease the risk of developing kidney disease in the general population is not supported by the scientific literature.
  • Indeed, preliminary studies showing a positive effect of relatively high protein diets on risk factors for chronic kidney disease (particularly on obesity, hypertension and diabetes) point to the need for future studies addressing diets that could prevent the increasingly high prevalence of kidney disease in the Western world.
  • On the other hand, there is a potential role for protein restriction in patients with established kidney disease, particularly in patients with significant decrease in glomerular filtration rate.
  • The exact protective action of protein restriction in patients with established renal disease needs further analysis, taking into account the more broad effects of protein restriction (lower phosphate, acidosis, uric acid) and a more current definition of malnutrition.
  • [MeSH-major] Diet / ethnology. Diet, Protein-Restricted. Dietary Proteins. Kidney Diseases / prevention & control

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  • (PMID = 17369718.001).
  • [ISSN] 0302-5144
  • [Journal-full-title] Contributions to nephrology
  • [ISO-abbreviation] Contrib Nephrol
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 0 / Dietary Proteins
  • [Number-of-references] 41
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67. Stratta RJ, Sundberg AK, Rohr MS, Farney AC, Hartmann EL, Roskopf JA, Iskandar SS, Hairston G, Kiger DF, Gautreaux MD, Anderson TK, Adams PL: Optimal use of older donors and recipients in kidney transplantation. Surgery; 2006 Mar;139(3):324-33
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Optimal use of older donors and recipients in kidney transplantation.
  • BACKGROUND: The aging donor and recipient population have led to new challenges in kidney transplantation.
  • The purpose of this study was to review retrospectively our single center experience in deceased-donor kidney transplantation, with respect to donor and recipient age.
  • METHODS: From October 1, 2001, through February 20, 2004, we performed 144 deceased-donor kidney transplantations, which included 37 procedures (26%) in recipients > or =60 years old and 107 procedures (74%) in recipients 19 to 59 years old.
  • ECD kidneys were used by matching estimated renal functional mass to recipient size (body mass index, <25 kg/m(2)), which included the use of dual kidney transplantations (n = 9).
  • ECD kidney recipients were further selected on the basis of age >40 years and low immunologic risk.
  • In recipients > or =60 years old, 23 recipients (62%) received kidney transplants from ECDs compared with 34 kidney transplants from ECDs (32%; P < .001) in recipients who were <60 years old.
  • Kidney graft survival rates were 84% in both recipient groups.
  • CONCLUSION: By the matching of nephron mass with recipient size and avoiding the use of ECD kidneys in recipients with a high immunologic risk, short-term outcomes that are comparable with standard criteria donor kidneys in younger patients can be achieved with either older donors or recipients, regardless of age.
  • [MeSH-major] Kidney Transplantation. Patient Selection. Tissue Donors


68. Okamoto M, Suzuki T, Fujiki M, Nobori S, Ushigome H, Sakamoto S, Yoshimura N: The consequences for live kidney donors with preexisting glucose intolerance without diabetic complication: analysis at a single Japanese center. Transplantation; 2010 Jun 15;89(11):1391-5
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The consequences for live kidney donors with preexisting glucose intolerance without diabetic complication: analysis at a single Japanese center.
  • BACKGROUND: Because of lack of deceased donors in Japan, there has been a need to expand the eligibility criteria for live kidney donation.
  • To assess the indications for live kidney donation in glucose intolerance (GI), we analyzed perioperative complications associated with donor nephrectomies performed at our institution and followed up the long-term consequences.
  • METHODS: The 444 live kidney donors were divided into two groups based on the results of the 75-g oral glucose tolerance test: a GI group (n=71) who showed a diabetic (n=27) or impaired glucose tolerance (n=44) pattern, and a non-GI group (n=373) who showed a normal oral glucose tolerance test pattern.
  • Perioperative complications, longterm survival rate, and frequencies of hypertension, diabetes, hyperlipidemia, and renal dysfunction in long term were compared in each group.
  • None of the patients in the diabetes mellitus group had developed severe diabetic complications or end-stage renal disease at a mean follow-up point of 88+/-71 (range, 14-225) months.
  • CONCLUSIONS: Our results suggest that individuals who have GI without diabetic complication may be able to donate their kidney safely with little surgical complication and little major morbidity if strict evaluation is performed before transplant.
  • [MeSH-major] Glucose Intolerance / epidemiology. Kidney Transplantation / physiology. Living Donors. Nephrectomy. Tissue and Organ Procurement / statistics & numerical data


69. Kestenbaum B, Belozeroff V: Mineral metabolism disturbances in patients with chronic kidney disease. Eur J Clin Invest; 2007 Aug;37(8):607-22
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Mineral metabolism disturbances in patients with chronic kidney disease.
  • BACKGROUND: Kidney disease, especially chronic kidney disease (CKD), is a worldwide public health problem with serious adverse health consequences for affected individuals.
  • Secondary hyperparathyroidism, a disorder characterized by elevated serum parathyroid hormone levels, and alteration of calcium and phosphorus homeostasis are common metabolic complications of CKD that may impact cardiovascular health.
  • RESULTS: Mineral metabolism disturbances begin early during the course of chronic kidney disease, and are associated with cardiovascular disease and mortality in observational studies.
  • Vascular calcification is one plausible mechanism connecting renal-related mineral metabolism with cardiovascular risk.
  • CONCLUSIONS: There exists a potential to improve outcomes for patients with CKD through increased awareness of the Bone Metabolism and Disease guidelines set forth by the National Kidney Foundation-Kidney Disease Outcomes Quality Initiative.
  • [MeSH-major] Calcium / metabolism. Cardiovascular Diseases / etiology. Hyperparathyroidism, Secondary / etiology. Kidney Failure, Chronic / complications. Parathyroid Hormone / secretion. Phosphorus / metabolism
  • [MeSH-minor] Calcinosis / etiology. Female. Humans. Male. Renal Dialysis. Risk Factors. Vitamin D / analogs & derivatives. Vitamin D / blood. Vitamin D / therapeutic use

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  • (PMID = 17635571.001).
  • [ISSN] 0014-2972
  • [Journal-full-title] European journal of clinical investigation
  • [ISO-abbreviation] Eur. J. Clin. Invest.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Parathyroid Hormone; 1406-16-2 / Vitamin D; 27YLU75U4W / Phosphorus; SY7Q814VUP / Calcium
  • [Number-of-references] 137
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70. Hanratty R, Chonchol M, Miriam Dickinson L, Beaty BL, Estacio RO, Mackenzie TD, Hurley LP, Linas SL, Steiner JF, Havranek EP: Incident chronic kidney disease and the rate of kidney function decline in individuals with hypertension. Nephrol Dial Transplant; 2010 Mar;25(3):801-7
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Incident chronic kidney disease and the rate of kidney function decline in individuals with hypertension.
  • BACKGROUND: Little is known about the decline of kidney function in patients with normal kidney function at baseline.
  • Our objectives were to (i) identify predictors of incident chronic kidney disease (CKD) and (ii) to estimate rate of decline in kidney function.
  • Rates of incident CKD or in decline of kidney function did not differ by race or ethnicity in this cohort.

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  • (PMID = 19889870.001).
  • [ISSN] 1460-2385
  • [Journal-full-title] Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association
  • [ISO-abbreviation] Nephrol. Dial. Transplant.
  • [Language] ENG
  • [Grant] United States / NHLBI NIH HHS / HL / U01 HL079160; United States / NHLBI NIH HHS / HL / U01 HL079208; United States / NHLBI NIH HHS / HL / 1U01 HL079160; United States / NHLBI NIH HHS / HL / 1U01 HL079208
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC2828608
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71. Cunningham J, Cass A, Anderson K, Snelling P, Devitt J, Preece C, Eris J: Australian nephrologists' attitudes towards living kidney donation. Nephrol Dial Transplant; 2006 May;21(5):1178-83
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  • [Title] Australian nephrologists' attitudes towards living kidney donation.
  • BACKGROUND: The demand for deceased donor kidneys far outweighs the supply.
  • The rate of living kidney donation (LKD) has been steadily increasing world-wide and is associated with excellent outcomes for the recipient.
  • The vast majority (95%) of respondents indicated that they would recommend it to a suitable donor or would themselves (97%) donate a kidney to an immediate family member.
  • However, fewer than half (43%) would recommend LKD to a potential donor, where their relative's end-stage kidney disease (ESKD) had been attributed to diabetes and where there was a strong family history of diabetes.
  • Meeting the growing demand for kidney transplantation will require an increased supply of both live and deceased donor kidneys.
  • [MeSH-major] Attitude of Health Personnel. Kidney Transplantation / psychology. Living Donors. Tissue and Organ Procurement
  • [MeSH-minor] Adult. Australia. Diabetic Nephropathies / diagnosis. Diabetic Nephropathies / surgery. Female. Health Care Surveys. Humans. Logistic Models. Male. Middle Aged. Multivariate Analysis. Nephrectomy / methods. Nephrology / standards. Nephrology / trends. Practice Patterns, Physicians'. Risk Assessment. Surveys and Questionnaires

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  • (PMID = 16490747.001).
  • [ISSN] 0931-0509
  • [Journal-full-title] Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association
  • [ISO-abbreviation] Nephrol. Dial. Transplant.
  • [Language] eng
  • [Publication-type] Comparative Study; Editorial; Research Support, Non-U.S. Gov't
  • [Publication-country] England
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72. Thomas MC, Atkins RC: Blood pressure lowering for the prevention and treatment of diabetic kidney disease. Drugs; 2006;66(17):2213-34
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  • [Title] Blood pressure lowering for the prevention and treatment of diabetic kidney disease.
  • The current pandemic of diabetes mellitus will inevitably be followed by an epidemic of chronic kidney disease.
  • It is anticipated that 25-40% of patients with type 1 diabetes and 5-40% of patients with type 2 diabetes will ultimately develop diabetic kidney disease.
  • Diabetic nephropathy may lead to hypertension through direct actions on renal sodium handling, vascular compliance and vasomotor function.
  • Recent clinical trials also support the utility of blood pressure reduction in the prevention of diabetic kidney disease.
  • In patients with overt nephropathy, blood pressure reduction is associated with reduced urinary albumin excretion and, subsequently, a reduced risk of renal impairment or end stage renal disease.
  • Although there is a clear physiological rationale for blockade of the renin angiotensin system, which is strongly supported by clinical studies, to achieve the optimal lowering of blood pressure, particularly in the setting of established diabetic renal disease, a number of different antihypertensive agents will always be needed.
  • Ultimately, no matter how it is achieved, so long as it is achieved, renal risk can be reduced by agents that lower blood pressure and albuminuria.
  • [MeSH-major] Antihypertensive Agents / therapeutic use. Blood Pressure / drug effects. Diabetes Complications / diagnosis. Diabetes Mellitus / therapy. Diabetic Nephropathies / prevention & control. Hypertension / drug therapy


73. Ibrahim HN, Foley R, Tan L, Rogers T, Bailey RF, Guo H, Gross CR, Matas AJ: Long-term consequences of kidney donation. N Engl J Med; 2009 Jan 29;360(5):459-69
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  • [Title] Long-term consequences of kidney donation.
  • BACKGROUND: The long-term renal consequences of kidney donation by a living donor are attracting increased appropriate interest.
  • The overall evidence suggests that living kidney donors have survival similar to that of nondonors and that their risk of end-stage renal disease (ESRD) is not increased.
  • METHODS: We ascertained the vital status and lifetime risk of ESRD in 3698 kidney donors who donated kidneys during the period from 1963 through 2007; from 2003 through 2007, we also measured the glomerular filtration rate (GFR) and urinary albumin excretion and assessed the prevalence of hypertension, general health status, and quality of life in 255 donors.
  • RESULTS: The survival of kidney donors was similar to that of controls who were matched for age, sex, and race or ethnic group.
  • CONCLUSIONS: Survival and the risk of ESRD in carefully screened kidney donors appear to be similar to those in the general population.
  • [MeSH-major] Health Status. Kidney Failure, Chronic / epidemiology. Kidney Transplantation. Living Donors. Quality of Life

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  • [Copyright] 2009 Massachusetts Medical Society
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  • (PMID = 19179315.001).
  • [ISSN] 1533-4406
  • [Journal-full-title] The New England journal of medicine
  • [ISO-abbreviation] N. Engl. J. Med.
  • [Language] eng
  • [Grant] United States / NCRR NIH HHS / RR / M01 RR000400; United States / NIDDK NIH HHS / DK / P01 DK013083; United States / NCRR NIH HHS / RR / M01-RR00400; United States / NIDDK NIH HHS / DK / P01DK13083
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] AYI8EX34EU / Creatinine
  • [Other-IDs] NLM/ NIHMS434270; NLM/ PMC3559132
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74. Hou CP, Chiang YJ, Chu SH, Ng KF, Wang HH: Mixed epithelial and stromal tumor of the kidney--a case report. Chang Gung Med J; 2010 Nov-Dec;33(6):693-8
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  • [Title] Mixed epithelial and stromal tumor of the kidney--a case report.
  • She visited our hospital and a renal echo showed a heterogenous mass on the left kidney.
  • Abdominal computed tomography showed a multicystic tumor, about 7 cm, on the left renal pelvis and the proximal ureter.
  • The tumor was enhanced after contrast injection.
  • Ureteroscopy showed an intraluminal polypoid tumor.
  • Cystic renal cell carcinoma or urothelial carcinoma was suspected preoperatively.
  • The pathology report demonstrated that the tumor was composed of an admixture of stroma and flattened to cuboidal urothelium.
  • The tumor stromal cells expressed both estrogen and progesterone receptors, and no malignant cells were found.
  • There has been no recurrence or deterioration of the patient's renal function since surgery.
  • We suggest keeping in mind the diagnosis of mixed epithelial and stromal tumor of the kidney when encountering perimenopausal women with renal cystic tumors.
  • [MeSH-major] Kidney Neoplasms / pathology. Neoplasms, Complex and Mixed / pathology

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  • (PMID = 21199615.001).
  • [ISSN] 2309-835X
  • [Journal-full-title] Chang Gung medical journal
  • [ISO-abbreviation] Chang Gung Med J
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] China (Republic : 1949- )
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75. Vassalotti JA, Stevens LA, Levey AS: Testing for chronic kidney disease: a position statement from the National Kidney Foundation. Am J Kidney Dis; 2007 Aug;50(2):169-80
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Testing for chronic kidney disease: a position statement from the National Kidney Foundation.
  • Chronic kidney disease (CKD) is common in the United States.
  • Major outcomes of CKD include progression to kidney failure, development of complications of impaired kidney function, and increased risk for cardiovascular disease.
  • CKD is usually silent until its late stages, thus many patients with CKD are detected only shortly before the onset of symptomatic kidney failure, when there are few opportunities to prevent adverse outcomes.
  • [MeSH-major] Foundations / standards. Kidney Diseases / blood. Kidney Diseases / urine. Practice Guidelines as Topic

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  • [CommentIn] Am J Kidney Dis. 2008 Jan;51(1):161-2; author reply 162-3 [18155547.001]
  • [CommentIn] Am J Kidney Dis. 2008 Jan;51(1):162; author reply 162-3 [18155548.001]
  • (PMID = 17660017.001).
  • [ISSN] 1523-6838
  • [Journal-full-title] American journal of kidney diseases : the official journal of the National Kidney Foundation
  • [ISO-abbreviation] Am. J. Kidney Dis.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 42
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76. Tombul ST, Aki FT, Gunay M, Inci K, Hazirolan T, Karcaaltincaba M, Erkan I, Bakkaloglu A, Yasavul U, Bakkaloglu M: Preoperative evaluation of hilar vessel anatomy with 3-D computerized tomography in living kidney donors. Transplant Proc; 2008 Jan-Feb;40(1):47-9
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  • [Title] Preoperative evaluation of hilar vessel anatomy with 3-D computerized tomography in living kidney donors.
  • OBJECTIVES: Digital subtract angiography is the gold standard for anatomic assessment of renal vasculature for living renal donors.
  • However, multidetector-row computerized tomography (MDCT) is less invasive than digital subtract angiography and provides information of kidney stones and other intra-abdominal organs.
  • In this study, preoperative MDCT angiography results were compared with the peroperative findings to evaluate the accuracy of MDCT for the evaluation of renal anatomy.
  • METHODS: From December 2002 to May 2007, all 60 consecutive living kidney donors were evaluated with MDCT angiography preoperatively.
  • We reported the number and origin of renal arteries, presence of early branching arteries, and any intrinsic renal artery disease.
  • Renal venous anatomy was evaluated for the presence of accessory, retroaortic, and circumaortic veins using venous phase axial images.
  • RESULTS: A total of 67 renal arteries were seen peroperatively in 60 renal units.
  • CONCLUSION: MDCT angiography offers a less invasive, rapid, and accurate preoperative investigation modality for vascular anatomy in living kidney donors.
  • [MeSH-major] Kidney. Living Donors. Renal Artery / anatomy & histology. Renal Circulation. Tomography, X-Ray Computed

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  • (PMID = 18261544.001).
  • [ISSN] 0041-1345
  • [Journal-full-title] Transplantation proceedings
  • [ISO-abbreviation] Transplant. Proc.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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77. Tsakiris P, Beemster P, Wijkstra H, de la Rosette J, Laguna P: In vivo factors influencing the freezing cycle during cryoablation of small renal masses. J Endourol; 2009 Mar;23(3):545-9
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  • [Title] In vivo factors influencing the freezing cycle during cryoablation of small renal masses.
  • PURPOSE: The aim of this study is to present a procedural analysis of the cryoablations performed in our department for small renal tumors and to try to identify clinical parameters or factors that influence the freezing rate during the procedure.
  • Based on the intraoperative biopsy result, we grouped the cases in two groups: renal-cell carcinoma (RCC) and benign.
  • RESULTS: A total of 70 cryoablations of small renal tumors in 67 patients were performed during this period.
  • The RCC group consisted of 48 cases (39 RCC and 9 lesions with a nondiagnostic biopsy) while 8 formed the benign group.
  • Preoperative creatinine levels above 120 IU, diabetes mellitus, American Society of Anesthesiologists score 3, and location of the tumor at the lower pole were found to increase the freezing rate.
  • The multivariate analysis showed that the location of the tumor and diabetes mellitus influence more significantly the temperature v time graph.
  • CONCLUSIONS: The freezing rate during cryotherapy of small renal tumors is significantly influenced by various clinical factors, while there are no differences in the freezing rate of those proven small malignant tumors and the small benign lesions.

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  • (PMID = 19216633.001).
  • [ISSN] 1557-900X
  • [Journal-full-title] Journal of endourology
  • [ISO-abbreviation] J. Endourol.
  • [Language] ENG
  • [Publication-type] Journal Article
  • [Publication-country] United States
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78. Vora N, Perrone R, Bianchi DW: Reproductive issues for adults with autosomal dominant polycystic kidney disease. Am J Kidney Dis; 2008 Feb;51(2):307-18
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  • [Title] Reproductive issues for adults with autosomal dominant polycystic kidney disease.
  • Autosomal dominant polycystic kidney disease (ADPKD) is a common disorder.
  • Affected women with ADPKD and normal renal function have a high rate of successful uncomplicated pregnancies.
  • Pregnant women with ADPKD with compromised kidney function should be monitored carefully for the development of hypertension and preeclampsia.
  • The diagnosis of ADPKD should always be considered when prenatal sonographic findings of hyperechogenic enlarged kidneys are found.
  • In this setting, a family history and renal sonogram of both parents is indicated.
  • Sequencing of the PKD1 and PKD2 genes is available and can be used for both prenatal and preimplantation genetic diagnosis.
  • [MeSH-major] Infertility, Male / physiopathology. Polycystic Kidney, Autosomal Dominant / physiopathology. Pregnancy Complications / physiopathology. Reproduction
  • [MeSH-minor] Adult. Age of Onset. Counseling. Cysts / genetics. Female. Fetal Growth Retardation / prevention & control. Genital Diseases, Male / genetics. Humans. Hypertension, Pregnancy-Induced / prevention & control. Kidney / ultrasonography. Male. Oligohydramnios / prevention & control. Placental Circulation. Pre-Eclampsia / prevention & control. Pregnancy. Preimplantation Diagnosis. Seminal Vesicles / pathology. Sequence Analysis, DNA. Spermatozoa / abnormalities. TRPP Cation Channels. Ultrasonography, Prenatal


79. Russo P: Partial nephrectomy achieves local tumor control and prevents chronic kidney disease. Expert Rev Anticancer Ther; 2006 Dec;6(12):1745-51
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  • [Title] Partial nephrectomy achieves local tumor control and prevents chronic kidney disease.
  • Over the last 10 years, a rapidly changing clinical landscape has created an optimal environment to expand the use of partial nephrectomy for the treatment of renal cortical tumors.
  • The main factors responsible for the increasing use of partial nephrectomy relate to the appreciation of the diversity of renal cortical tumor histology, the tumor stage and size migration associated with their incidental detection, and the oncological efficacy of partial nephrectomy for tumors of 7 cm or smaller.
  • Evidence has now emerged that radical nephrectomy performed during the treatment of a small renal mass may further reduce an already impaired baseline renal function and place the patient in the realm of chronic kidney disease.
  • Important new information concerning chronic kidney disease, a condition far more prevalent in the aging US population than previously appreciated, and its associated cardiovascular morbidity and mortality, now places maximal renal functional preservation on a par with local tumor control as surgical plans are formulated for the resection of a small renal cortical tumor.
  • [MeSH-major] Kidney Neoplasms / surgery. Nephrectomy / methods
  • [MeSH-minor] Adenocarcinoma, Clear Cell / pathology. Adenocarcinoma, Clear Cell / surgery. Adenoma, Oxyphilic / pathology. Adenoma, Oxyphilic / surgery. Carcinoma, Papillary / pathology. Carcinoma, Papillary / surgery. Carcinoma, Renal Cell / pathology. Carcinoma, Renal Cell / surgery. Cardiovascular Diseases / etiology. Cardiovascular Diseases / mortality. Chronic Disease. Humans. Kidney Cortex / surgery. Kidney Diseases / etiology. Kidney Diseases / prevention & control. Kidney Function Tests. Kidney Transplantation. Postoperative Complications / etiology. Postoperative Complications / prevention & control. Tissue Donors. Treatment Outcome

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  • (PMID = 17181488.001).
  • [ISSN] 1744-8328
  • [Journal-full-title] Expert review of anticancer therapy
  • [ISO-abbreviation] Expert Rev Anticancer Ther
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Number-of-references] 42
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80. Bölling T, Janke K, Wolters HH, Glashörster M, Ernst I, Willich N, Brockmann J, Könemann S: Kidney-autotransplantation before radiotherapy: a case report. Anticancer Res; 2009 Aug;29(8):3397-400
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  • [Title] Kidney-autotransplantation before radiotherapy: a case report.
  • BACKGROUND: A 28-year-old man suffering from a Ewing tumour arising from the 9th-11th ribs with infiltration of neuroforamina without distant metastases was planned to receive radiotherapy following primary intralesional surgery and induction chemotherapy.
  • Due to pleural infiltration and effusion, a hemithorax irradiation with a sequential boost to the primary tumour site had to be administered.
  • Different treatment planning variants failed to provide sufficient radiotherapy planning in view of target volume coverage and avoidance of organs at risk, especially due to high calculated radiation doses potentially compromising the left kidney.
  • MATERIALS AND METHODS: To prevent left kidney organ exposure, an autotransplantation of the left kidney into the right fossa iliaca was performed.
  • An infiltration of the kidney was initially excluded.
  • RESULTS: Postoperatively, a renal scintigraphy showed a normal function of both kidneys allowing sufficient radiotherapy treatment planning.
  • [MeSH-major] Bone Neoplasms / surgery. Kidney Transplantation. Sarcoma, Ewing / surgery
  • [MeSH-minor] Adult. Humans. Kidney / radiation effects. Kidney / surgery. Magnetic Resonance Imaging. Male. Radiotherapy Dosage. Radiotherapy Planning, Computer-Assisted. Radiotherapy, Intensity-Modulated. Transplantation, Autologous

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  • (PMID = 19661363.001).
  • [ISSN] 1791-7530
  • [Journal-full-title] Anticancer research
  • [ISO-abbreviation] Anticancer Res.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Greece
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81. Hao CM, Breyer MD: Roles of lipid mediators in kidney injury. Semin Nephrol; 2007 May;27(3):338-51
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  • [Title] Roles of lipid mediators in kidney injury.
  • In addition to their role in regulating normal kidney function, these lipids also play important roles in the pathogenesis of kidney diseases.
  • P450 arachidonic acid mono-oxygenase-derived 20-hydroxyeicosatetraenoic acid and epoxyeicosatrienoic acids are involved in several forms of kidney injury, including renal injury in metabolic syndrome.
  • Ceramide also has been shown to be an important signaling molecule that is involved in the pathogenesis of acute kidney injury caused by ischemia/reperfusion and toxic insults.
  • Those pathways should provide fruitful targets for intervention in the pharmacologic treatment of renal disease.
  • [MeSH-major] Kidney Diseases / metabolism. Leukotrienes / physiology. Lipoxygenase / physiology. Prostaglandin-Endoperoxide Synthases / metabolism. Prostaglandins / metabolism

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  • (PMID = 17533010.001).
  • [ISSN] 0270-9295
  • [Journal-full-title] Seminars in nephrology
  • [ISO-abbreviation] Semin. Nephrol.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Arachidonic Acids; 0 / Cyclooxygenase Inhibitors; 0 / Leukotrienes; 0 / Prostaglandins; 9035-51-2 / Cytochrome P-450 Enzyme System; EC 1.13.11.12 / Lipoxygenase; EC 1.14.99.1 / Prostaglandin-Endoperoxide Synthases
  • [Number-of-references] 178
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82. Filimonovic D, Gojnic M, Arsenijevic Lj, Popovic N, Dugalic V: Increased AFP as an indicator of ovarian carcinoma and fetal kidney carcinoma--case report. Eur J Gynaecol Oncol; 2007;28(1):57-9
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  • [Title] Increased AFP as an indicator of ovarian carcinoma and fetal kidney carcinoma--case report.
  • Having in mind a mild decrease in ovarian artery resistance index (RI) and suspected findings of fetal kidney, this situation was delicate due to its double pathology which was later confirmed.
  • Wilms' tumor is the most common urogenital tumor in childhood, and it is detectable in the prenatal period by ultrasound examination.
  • In utero kidney biopsy confirms diagnosis and facilitates decisions concerning the course of pregnancy.
  • [MeSH-major] Kidney Neoplasms / diagnosis. Ovarian Neoplasms / diagnosis. Pregnancy Complications, Neoplastic / diagnosis. Wilms Tumor / diagnosis. alpha-Fetoproteins / metabolism


83. Raiteri M, Ferraresso M, Pozzoli E, Beretta C, Pasciucco A, Carini M, Berardinelli L: Value of intraoperative resistive index in kidney transplant. Transplant Proc; 2005 Jul-Aug;37(6):2472-3
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  • [Title] Value of intraoperative resistive index in kidney transplant.
  • The value of the resistive index (RI) obtained by echo color doppler evaluation of the transplanted kidney is still not well established.
  • Many authors consider the RI to be nonspecific sign of rejection, acute tubular necrosis, or urinary tract obstruction, but its specificity remains low.
  • In this paper, we report our experience with RI determinations in 34 consecutive kidney transplants at different times namely: perioperatively, at 24 hours, at 3 days, at 6 and at 9 days posttransplant.
  • In all patients intraoperative RI was normal.
  • RI increased significantly after transplantation in 10 patients who eventually developed a complication: delayed function, acute rejection, and spontaneous kidney ruptures.
  • This increment from the baseline value was already significant at 24 hours after the kidney transplant, indicating a possible posttransplant complication (0.62 +/- 0.07 vs 0.76 +/- 0.04; P = .0004).
  • We conclude that the value of RI in the early posttransplant phase should be considered an important aid for the early diagnosis of posttransplant complications.
  • [MeSH-major] Kidney Transplantation / physiology. Vascular Resistance
  • [MeSH-minor] Adolescent. Adult. Aged. Child. Female. Humans. Male. Middle Aged. Monitoring, Intraoperative. Postoperative Period. Preoperative Care. Renal Circulation / physiology. Tissue Donors / statistics & numerical data

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  • (PMID = 16182713.001).
  • [ISSN] 0041-1345
  • [Journal-full-title] Transplantation proceedings
  • [ISO-abbreviation] Transplant. Proc.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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84. Phisitkul S, Hacker C, Simoni J, Tran RM, Wesson DE: Dietary protein causes a decline in the glomerular filtration rate of the remnant kidney mediated by metabolic acidosis and endothelin receptors. Kidney Int; 2008 Jan;73(2):192-9
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  • [Title] Dietary protein causes a decline in the glomerular filtration rate of the remnant kidney mediated by metabolic acidosis and endothelin receptors.
  • Dietary casein promotes a progressive decline in the glomerular filtration rate (GFR) of remnant kidneys associated with metabolic acidosis and an endothelin-mediated increase in renal acidification.
  • We tested whether diets that affect the acid-base status contributes to the decline of GFR through endothelin receptors in rats with a remnant kidney.
  • Rats on a casein diet had metabolic acidosis at baseline and developed a progressive decline in GFR after renal mass reduction.
  • Dietary soy protein did not induce baseline metabolic acidosis and rats with remnant kidney on a soy diet had no decrease in their GFR.
  • By contrast, rats with a remnant kidney on soy protein given dietary acid developed metabolic acidosis and a decreased GFR.
  • Our study suggests that the casein-induced decline in GFR of the remnant kidney is mediated by metabolic acidosis through endothelin A receptors.


85. Lee S, Garner EI, Welch WR, Berkowitz RS, Mok SC: Over-expression of hypoxia-inducible factor 1 alpha in ovarian clear cell carcinoma. Gynecol Oncol; 2007 Aug;106(2):311-7
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  • Since tumor hypoxia has been shown to be strongly associated with poor prognosis, deregulation of the representative factor of tissue hypoxia; hypoxia-inducible factor 1 alpha (HIF-1alpha) and related protein; Von Hippel-Lindau (VHL) may be associated with poor prognosis of ovarian clear cell carcinoma.
  • METHODS: Immunolocalization of both HIF-1alpha and VHL was performed on 56 cases of paraffin-embedded tissue sections of four different histological types of epithelial ovarian carcinoma and 5 cases of benign ovarian tumors as a control.

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  • (PMID = 17532031.001).
  • [ISSN] 0090-8258
  • [Journal-full-title] Gynecologic oncology
  • [ISO-abbreviation] Gynecol. Oncol.
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / R33 CA103595; United States / NCI NIH HHS / CA / R33 CA103595-04; United States / NCI NIH HHS / CA / CA103595-04; United States / NCI NIH HHS / CA / CA105009-030002; United States / NCI NIH HHS / CA / P50 CA105009; United States / NCI NIH HHS / CA / P50 CA105009-030002; United States / PHS HHS / / P50105009; United States / NCI NIH HHS / CA / R01 CA133057; United States / NCI NIH HHS / CA / R33CA103595
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / HIF1A protein, human; 0 / Hypoxia-Inducible Factor 1, alpha Subunit; 0 / RNA, Messenger; EC 6.3.2.19 / VHL protein, human; EC 6.3.2.19 / Von Hippel-Lindau Tumor Suppressor Protein
  • [Other-IDs] NLM/ NIHMS28316; NLM/ PMC1995602
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86. Hemmelgarn BR, Zhang J, Manns BJ, Tonelli M, Larsen E, Ghali WA, Southern DA, McLaughlin K, Mortis G, Culleton BF: Progression of kidney dysfunction in the community-dwelling elderly. Kidney Int; 2006 Jun;69(12):2155-61
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  • [Title] Progression of kidney dysfunction in the community-dwelling elderly.
  • Despite the high prevalence of chronic kidney disease among the elderly, few studies have described their loss of kidney function.
  • We sought to determine the progression of kidney dysfunction among a community-based cohort of elderly subjects.
  • In conclusion, we found that the majority of elderly subjects have no or minimal progression of kidney disease over 2 years.
  • Strategies aimed at slowing progression of kidney disease should consider underlying risk factors for progression and the negligible loss of kidney function that occurs in the majority of older adults.
  • [MeSH-major] Aging / physiology. Glomerular Filtration Rate / physiology. Kidney / physiopathology. Kidney Failure, Chronic / physiopathology


87. McClellan WM, Casey MT, Hughley J, Freund E: Population-based interventions to reduce socioeconomic disparities in chronic kidney disease. Semin Nephrol; 2010 Jan;30(1):33-41
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  • [Title] Population-based interventions to reduce socioeconomic disparities in chronic kidney disease.
  • Disparities in the occurrence and outcomes of chronic kidney disease (CKD) are associated with individual and community socioeconomic (SES) risk factors.
  • Changes in the US health system have eliminated barriers to access for end-stage renal disease care for most US citizens and reduced disparities in outcomes of care after the onset of renal replacement therapy.
  • In particular, we describe a 10-state pilot project initiated by the Centers for Medicare and Medicaid Services in August of 2008 to reduce disparities and improve the detection and treatment of early diabetic kidney disease.
  • [MeSH-major] Healthcare Disparities. Kidney Diseases / therapy

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  • [Copyright] Copyright (c) 2010 Elsevier Inc. All rights reserved.
  • (PMID = 20116646.001).
  • [ISSN] 1558-4488
  • [Journal-full-title] Seminars in nephrology
  • [ISO-abbreviation] Semin. Nephrol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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88. Laissy JP, Idée JM, Fernandez P, Floquet M, Vrtovsnik F, Schouman-Claeys E: Magnetic resonance imaging in acute and chronic kidney diseases: present status. Nephron Clin Pract; 2006;103(2):c50-7
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  • [Title] Magnetic resonance imaging in acute and chronic kidney diseases: present status.
  • Magnetic resonance imaging (MRI) of normal and diseased kidneys shows great promise because of the combined value of anatomical and functional information provided, as well as of specific contrast patterns that can be observed non-invasively.
  • Multicontrast MRI is able to show infiltrative kidney disorders.
  • Diffusion-weighted imaging can assess alterations in renal function and can suggest obstruction or inflammation when present.
  • Furthermore, contrast agents such as ultrasmall particles of iron oxide, specific of inflammation, should be used in the near future to detect active from quiescent involvement, both in native kidneys and renal allografts.
  • Ongoing studies will obviously demonstrate the value of the combination of these various MRI sequences in the diagnosis of acute renal failure and chronic kidney disease.
  • [MeSH-major] Acute Kidney Injury / diagnosis. Kidney Failure, Chronic / diagnosis. Magnetic Resonance Imaging

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  • [Copyright] Copyright 2006 S. Karger AG, Basel.
  • (PMID = 16543756.001).
  • [ISSN] 1660-2110
  • [Journal-full-title] Nephron. Clinical practice
  • [ISO-abbreviation] Nephron Clin Pract
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 0 / Contrast Media
  • [Number-of-references] 34
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89. O'Hare AM, Kaufman JS, Covinsky KE, Landefeld CS, McFarland LV, Larson EB: Current guidelines for using angiotensin-converting enzyme inhibitors and angiotensin II-receptor antagonists in chronic kidney disease: is the evidence base relevant to older adults? Ann Intern Med; 2009 May 19;150(10):717-24
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  • [Title] Current guidelines for using angiotensin-converting enzyme inhibitors and angiotensin II-receptor antagonists in chronic kidney disease: is the evidence base relevant to older adults?
  • Angiotensin-converting enzyme inhibitors and angiotensin II-receptor antagonists are recommended for patients with chronic kidney disease because these drugs can slow disease progression.
  • Older adults account for a large and growing number of patients with chronic kidney disease.
  • The authors evaluated the relevance to adults older than 70 years of the evidence base for major U.S. practice guidelines for the use of these agents in chronic kidney disease.
  • The authors first examined the representation of older adults in randomized trials that underpin these guidelines, then compared the characteristics of participants in these trials with those of a representative sample of older adults with chronic kidney disease in the general population.
  • The authors found that current guidelines for the use of angiotensin-converting enzyme inhibitors and angiotensin II-receptor antagonists in chronic kidney disease are based on evidence with limited relevance to most persons older than 70 years with this condition.

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  • [CommentIn] Ann Intern Med. 2009 May 19;150(10):731-3 [19451583.001]
  • (PMID = 19451579.001).
  • [ISSN] 1539-3704
  • [Journal-full-title] Annals of internal medicine
  • [ISO-abbreviation] Ann. Intern. Med.
  • [Language] ENG
  • [Grant] United States / NIA NIH HHS / AG / K23 AG028980; United States / NIA NIH HHS / AG / K 1K23AG28980
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Angiotensin II Type 1 Receptor Blockers; 0 / Angiotensin-Converting Enzyme Inhibitors
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90. Surányi A, Nyári T, Keresztúri A, Látó K, Pál A: [Biparietal diameter/kidney length ratio in cases with chronic hypoxic state]. Orv Hetil; 2005 Oct 16;146(42):2163-7
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  • [Title] [Biparietal diameter/kidney length ratio in cases with chronic hypoxic state].
  • AIMS: The object of this study was to investigate the fetal biparietal diameter/kidney length ratio in normal and hyperechogenic kidneys during the third trimester of gestation.
  • Depending on the renal manifestation of intrauterine chronic hypoxia, cases were divided into two study groups.
  • Group I was composed of 21 fetuses with pregnancy-associated hypertension and/or proteinuria and hyperechogenic renal medullae.
  • Group II consisted of 162 fetuses with pregnancy-associated hypertension and/or proteinuria and normal echoic kidney.
  • RESULTS: Fetal renal hyperechogenicity correlated with the pathological growth of fetal kidney.
  • The fetal biparietal diameter/kidney length ratio was significantly lower in cases of hyperechogenicity.
  • CONCLUSIONS: The fetal renal hyperechogenicity is a relevant indicator of diminution of fetal renal perfusion.
  • This can lead to abnormal development of the affected kidney and can result in a pathological reduction of biparietal diameter/kidney length ratio, which may also be an in utero indicator of subsequent intrauterine and neonatal complications.
  • Detailed ultrasound examinations of renal parenchyma and kidney length seem to be a useful method in the prenatal diagnosis of decreased renal perfusion and of intrauterine hypoxia and serve to detect pathological conditions in utero.
  • [MeSH-major] Fetal Hypoxia / pathology. Kidney / pathology. Pregnancy Complications / pathology. Pregnancy Trimester, Third. Ultrasonography, Prenatal

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  • (PMID = 16315998.001).
  • [ISSN] 0030-6002
  • [Journal-full-title] Orvosi hetilap
  • [ISO-abbreviation] Orv Hetil
  • [Language] hun
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] Hungary
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91. Köttgen A, Glazer NL, Dehghan A, Hwang SJ, Katz R, Li M, Yang Q, Gudnason V, Launer LJ, Harris TB, Smith AV, Arking DE, Astor BC, Boerwinkle E, Ehret GB, Ruczinski I, Scharpf RB, Chen YD, de Boer IH, Haritunians T, Lumley T, Sarnak M, Siscovick D, Benjamin EJ, Levy D, Upadhyay A, Aulchenko YS, Hofman A, Rivadeneira F, Uitterlinden AG, van Duijn CM, Chasman DI, Paré G, Ridker PM, Kao WH, Witteman JC, Coresh J, Shlipak MG, Fox CS: Multiple loci associated with indices of renal function and chronic kidney disease. Nat Genet; 2009 Jun;41(6):712-7
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  • [Title] Multiple loci associated with indices of renal function and chronic kidney disease.
  • Chronic kidney disease (CKD) has a heritable component and is an important global public health problem because of its high prevalence and morbidity.
  • UMOD encodes the most common protein in human urine, Tamm-Horsfall protein, and rare mutations in UMOD cause mendelian forms of kidney disease.
  • Our findings provide new insights into CKD pathogenesis and underscore the importance of common genetic variants influencing renal function and disease.

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  • (PMID = 19430482.001).
  • [ISSN] 1546-1718
  • [Journal-full-title] Nature genetics
  • [ISO-abbreviation] Nat. Genet.
  • [Language] ENG
  • [Grant] United States / NHLBI NIH HHS / HL / N01HC55020; United States / NCRR NIH HHS / RR / UL1RR025005; United States / NHLBI NIH HHS / HL / N02-HL-6-4278; United States / NHLBI NIH HHS / HC / N01-HC-25195; United States / NHLBI NIH HHS / HC / N01 HC055022; United States / NHLBI NIH HHS / HC / N01 HC085086; United States / NIDDK NIH HHS / DK / K01 DK067207; United States / NIA NIH HHS / AG / R01 AG027002-01; United States / NIA NIH HHS / AG / N01AG12100; United States / NCRR NIH HHS / RR / KL2 RR025015-03; United States / NHLBI NIH HHS / HL / N01HC55018; United States / NHLBI NIH HHS / HC / N01-HC-85085; United States / NHLBI NIH HHS / HL / R01HL59367; United States / NCRR NIH HHS / RR / UL1 RR025005; United States / NHGRI NIH HHS / HG / U01 HG004402-01; United States / NIA NIH HHS / AG / N01 AG012100; United States / NIDDK NIH HHS / DK / DK067207-01A1; United States / NHLBI NIH HHS / HL / U01 HL080295; United States / NHLBI NIH HHS / HL / HL 043851; United States / NIDDK NIH HHS / DK / K01 DK067207-01A1; United States / NHLBI NIH HHS / HC / N01 HC075150; United States / NHLBI NIH HHS / HC / N01-HC-55022; United States / NHLBI NIH HHS / HC / N01-HC-85081; United States / NHLBI NIH HHS / HL / R01 HL043851; United States / NHLBI NIH HHS / HL / R01 HL059367; United States / NHLBI NIH HHS / HC / N01 HC015103; United States / NCRR NIH HHS / RR / M01 RR000069; United States / NHLBI NIH HHS / HC / N01-HC-55016; United States / NHLBI NIH HHS / HC / N01 HC055018; United States / NHLBI NIH HHS / HC / N01 HC025195; United States / NHLBI NIH HHS / HL / R01 HL086694; United States / NHLBI NIH HHS / HL / N02 HL64278; United States / NHLBI NIH HHS / HL / HL087652-01; United States / NHLBI NIH HHS / HL / R01 HL087652; None / None / / KL2 RR025015-03; United States / NHLBI NIH HHS / HL / R01 HL059367-02; United States / PHS HHS / / HHSN268200625226C; United States / NHGRI NIH HHS / HG / U01 HG004402; United States / NHLBI NIH HHS / HL / N01HC55022; United States / NHLBI NIH HHS / HL / N01HC55222; United States / NHLBI NIH HHS / HC / N01-HC-55021; United States / NHLBI NIH HHS / HC / N01-HC-85086; United States / NHLBI NIH HHS / HL / N01HC55015; United States / NHLBI NIH HHS / HL / N01HC85086; United States / NHGRI NIH HHS / HG / U01HG004402; United States / NCI NIH HHS / CA / CA 047988; United States / NIDDK NIH HHS / DK / K01DK067207; United States / NHLBI NIH HHS / HC / N01-HC-85082; United States / NHLBI NIH HHS / HC / N01 HC055019; United States / NIDDK NIH HHS / DK / P30 DK063491; United States / Intramural NIH HHS / / ; United States / NHLBI NIH HHS / HC / N01-HC-35129; United States / NIDDK NIH HHS / DK / P30 DK063491-019004; United States / NHLBI NIH HHS / HC / N01-HC-55019; United States / NIDDK NIH HHS / DK / DK063491-019004; United States / NIDDK NIH HHS / DK / DK063491-05; United States / NHLBI NIH HHS / HL / R01HL087641; United States / NHLBI NIH HHS / HC / N01-HC-55015; United States / NHLBI NIH HHS / HC / N01 HC055222; United States / NHLBI NIH HHS / HL / R01 HL086694-01A1; United States / NHLBI NIH HHS / HC / N01-HC-55222; United States / NCRR NIH HHS / RR / M01RR00069; United States / NHLBI NIH HHS / HC / N01-HC-85083; United States / NHLBI NIH HHS / HC / N01-HC-75150; United States / NHLBI NIH HHS / HC / N01-HC-55020; United States / NHLBI NIH HHS / HC / N01-HC-85080; United States / NHLBI NIH HHS / HC / N01 HC-15103; United States / NHLBI NIH HHS / HL / N01HC55016; United States / NIDDK NIH HHS / DK / R01 DK076770; United States / NHLBI NIH HHS / HL / R01 HL043851-09; United States / NIDDK NIH HHS / DK / P30 DK063491-05; United States / NHLBI NIH HHS / HC / N01 HC055015; United States / NIA NIH HHS / AG / R01 AG027002; United States / NHLBI NIH HHS / HC / N01 HC055021; None / None / / U01 HL080295-01; United States / NCI NIH HHS / CA / R01 CA047988; United States / NCRR NIH HHS / RR / UL1 RR025005-01; United States / NHLBI NIH HHS / HL / N01HC55019; United States / NHLBI NIH HHS / HL / N01HC75150; United States / NIA NIH HHS / AG / N01-AG-12100; United States / NHLBI NIH HHS / HL / HL059367-02; United States / NCRR NIH HHS / RR / KL2 RR025015; United States / NHLBI NIH HHS / HL / N01HC25195; United States / NHLBI NIH HHS / HL / R01 HL087641-01; United States / NIDDK NIH HHS / DK / DK063491; United States / NHLBI NIH HHS / HC / N01-HC-45133; United States / NHLBI NIH HHS / HC / N01-HC-85079; United States / NHLBI NIH HHS / HC / N01 HC055020; United States / NCATS NIH HHS / TR / UL1 TR000423; United States / NHLBI NIH HHS / HL / R01 HL087652-01; United States / NHLBI NIH HHS / HL / N01HC85079; United States / NHLBI NIH HHS / HC / N01 HC085079; United States / NIA NIH HHS / AG / AG012100; United States / NHLBI NIH HHS / HC / N01-HC-55018; United States / NHLBI NIH HHS / HL / N01HC55021; United States / NHLBI NIH HHS / HL / R01 HL087641; United States / NHLBI NIH HHS / HL / HL086694-01A1; United States / NHLBI NIH HHS / HC / N01 HC045133; United States / NHLBI NIH HHS / HC / N01 HC035129; United States / NHLBI NIH HHS / HC / N01 HC055016; United States / NHLBI NIH HHS / HC / N01-HC-85084; United States / NHLBI NIH HHS / HL / R01HL086694; United States / NHLBI NIH HHS / HL / U01 HL080295-01
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Mucoproteins; 0 / UMOD protein, human; 0 / Uromodulin
  • [Other-IDs] NLM/ NIHMS267520; NLM/ PMC3039280
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92. Manuel O, Pascual M, Trendelenburg M, Meylan PR: Association between mannose-binding lectin deficiency and cytomegalovirus infection after kidney transplantation. Transplantation; 2007 Feb 15;83(3):359-62
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Association between mannose-binding lectin deficiency and cytomegalovirus infection after kidney transplantation.
  • We measured plasma MBL levels in 16 kidney transplant recipients with high-risk CMV serostatus (donor-positive/recipient-negative).
  • MBL deficiency may be a significant risk factor for the development of CMV infection in kidney transplant recipients, suggesting a role for innate immunity in the control of CMV infection after organ transplantation.
  • [MeSH-major] Cytomegalovirus Infections / etiology. Kidney Transplantation. Mannose-Binding Lectin / deficiency


93. Hanish SI, Samaniego M, Mezrich JD, Foley DP, Leverson GE, Lorentzen DF, Sollinger HW, Pirsch JD, D'Alessandro AM, Fernandez LA: Outcomes of simultaneous liver/kidney transplants are equivalent to kidney transplant alone: a preliminary report. Transplantation; 2010 Jul 15;90(1):52-60
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Outcomes of simultaneous liver/kidney transplants are equivalent to kidney transplant alone: a preliminary report.
  • BACKGROUND: With adoption of Model for End-Stage Liver Disease, the number of simultaneous liver-kidney transplants (SLK) has greatly increased.
  • A recent registry study questioned the equity of allocating kidney transplants (KTx) simultaneously with liver transplantation due to poor outcomes (Locke et al., Transplantation 2008; 85: 935).
  • METHODS: To investigate outcome of KTx in SLK, all SLK (n=36) performed at our center from January 2000 to December 2007 were reviewed and KTx outcomes compared with those of kidney transplant alone (KTA) performed during that period (n=1283).

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  • [Cites] Am J Transplant. 2009 Mar;9(3):578-85 [19260837.001]
  • [Cites] Am J Transplant. 2003 Mar;3(3):348-56 [12614293.001]
  • [Cites] Transplant Proc. 2008 Dec;40(10):3424-7 [19100404.001]
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  • [Cites] Transplantation. 2008 Apr 15;85(7):935-42 [18408571.001]
  • [Cites] Liver Transpl. 2007 Jun;13(6):838-43 [17539003.001]
  • [Cites] Transplantation. 2006 Nov 27;82(10):1298-303 [17130778.001]
  • [Cites] Am J Transplant. 2006 Sep;6(9):2183-90 [16827789.001]
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  • [Cites] Liver Transpl Surg. 1999 Jul;5(4):261-8 [10388498.001]
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  • [Cites] Hepatology. 2002 May;35(5):1179-85 [11981768.001]
  • [Cites] Transpl Int. 2009 Feb;22(2):242-6 [18954373.001]
  • (PMID = 20626084.001).
  • [ISSN] 1534-6080
  • [Journal-full-title] Transplantation
  • [ISO-abbreviation] Transplantation
  • [Language] ENG
  • [Grant] None / None / / UL1 RR025011-01; United States / NCRR NIH HHS / RR / UL1 RR025011-03; United States / NCRR NIH HHS / RR / UL1 RR025011-01; None / None / / UL1 RR025011-05; United States / NCRR NIH HHS / RR / UL1 RR025011; United States / NCRR NIH HHS / RR / UL1 RR025011-02; United States / NCRR NIH HHS / RR / UL1 RR025011-05; None / None / / UL1 RR025011-03; United States / NCRR NIH HHS / RR / UL1 RR025011-04; None / None / / UL1 RR025011-04; None / None / / UL1 RR025011-02
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Other-IDs] NLM/ NIHMS279256; NLM/ PMC3085017
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94. Mironov V, Drake C, Wen X: Research project: Charleston Bioengineered Kidney Project. Biotechnol J; 2006 Sep;1(9):903-5
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Research project: Charleston Bioengineered Kidney Project.
  • The goal of Charleston Bioengineered Kidney Project is to engineer a functional living human kidney suitable for surgical implantation using principles of directed tissue self-assembly and tissue fusion.
  • The conceptual framework, engineering principles, design, potential cell source as well as the first preliminary data demonstrating the feasibility of the proposed Charleston Bioengineered Kidney Project are outlined.
  • [MeSH-major] Bioartificial Organs. Biotechnology / methods. Kidney / anatomy & histology. Kidney / physiology. Tissue Engineering / methods

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  • (PMID = 16986103.001).
  • [ISSN] 1860-7314
  • [Journal-full-title] Biotechnology journal
  • [ISO-abbreviation] Biotechnol J
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / Biocompatible Materials
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95. Bivol LM, Vågnes OB, Iversen BM: The renal vascular response to ANG II injection is reduced in the nonclipped kidney of two-kidney, one-clip hypertension. Am J Physiol Renal Physiol; 2005 Aug;289(2):F393-400
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The renal vascular response to ANG II injection is reduced in the nonclipped kidney of two-kidney, one-clip hypertension.
  • The ANG II receptor 1 (AT(1)R) level in the nonclipped kidney of two-kidney, one-clip hypertension (2K1C) has shown to be unchanged despite a high circulating angiotensin (ANG) II level.
  • To examine the vasoreactive response to ANG II in this kidney, injections of ANG II into renal artery were performed 6 wk after clipping of the kidney and compared with normotensive controls.
  • The renal blood flow (RBF) response to 2.5 ng ANG II was measured by a Transonic transit-time flowmeter, before and after indomethacin and candesartan treatment, and analyzed by a computer program.
  • The mRNA for AT(1A) and AT(1B) as well as Western blotting for AT(1)R in renal resistance vessels were determined, and plasma renin activity (PRA) was measured.
  • Injection of ANG II reduced RBF with 10 +/- 2% in the nonclipped kidney and 24 +/- 3% in controls (P < 0.001).
  • The doses of candesartan needed to completely inhibit RBF response to ANG II were 30 microg/kg in the nonclipped kidney and 100 microg/kg in controls (P < 0.001).
  • Western blotting and mRNA for AT(1A) and AT(1B) in the nonclipped kidney were similar to the controls.
  • The results indicate that despite no difference in total AT(1)R levels, functional AT(1)R is downregulated in the nonclipped kidney of 2K1C rats.
  • [MeSH-major] Angiotensin II / pharmacology. Hypertension, Renovascular / physiopathology. Renal Circulation / drug effects

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  • (PMID = 15784843.001).
  • [ISSN] 1931-857X
  • [Journal-full-title] American journal of physiology. Renal physiology
  • [ISO-abbreviation] Am. J. Physiol. Renal Physiol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Benzimidazoles; 0 / Cyclooxygenase Inhibitors; 0 / Proteins; 0 / RNA, Messenger; 0 / Receptors, Angiotensin; 0 / Tetrazoles; 0 / Vasoconstrictor Agents; 11128-99-7 / Angiotensin II; 113-79-1 / Arginine Vasopressin; 63231-63-0 / RNA; EC 3.4.23.15 / Renin; S8Q36MD2XX / candesartan; XXE1CET956 / Indomethacin
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96. Pietrzak-Nowacka M, Safranow K, Byra E, Nowosiad M, Marchelek-Myśliwiec M, Ciechanowski K: Glucose metabolism parameters during an oral glucose tolerance test in patients with autosomal dominant polycystic kidney disease. Scand J Clin Lab Invest; 2010 Dec;70(8):561-7
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Glucose metabolism parameters during an oral glucose tolerance test in patients with autosomal dominant polycystic kidney disease.
  • OBJECTIVE: The aim of the study was to estimate the pancreatic beta cell function and insulin resistance indexes in a group of Autosomal Dominant Polycystic Kidney Disease (ADPKD) patients with normal kidney function and no previous diabetes mellitus diagnosis.
  • CONCLUSIONS: Presence of ADPKD in patients with normal kidney function is associated with impaired beta cell function after an oral glucose load, without a significant decrease in insulin sensitivity.
  • [MeSH-major] Glucose / metabolism. Glucose Tolerance Test / methods. Polycystic Kidney, Autosomal Dominant / metabolism


97. Kielstein JT, Zoccali C: Asymmetric dimethylarginine: a novel marker of risk and a potential target for therapy in chronic kidney disease. Curr Opin Nephrol Hypertens; 2008 Nov;17(6):609-15
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Asymmetric dimethylarginine: a novel marker of risk and a potential target for therapy in chronic kidney disease.
  • These preclinical findings are thought to be of major importance as ADMA predicts cardiovascular events and mortality in the general population and in patients with chronic kidney disease.
  • Also, ADMA uniformly predicts the progression of moderate and severe chronic kidney disease.
  • Symmetrical dimethylarginine, the structural isomer of ADMA, which was mistakenly thought to be without biological relevance, indicates the degree of renal impairment.
  • SUMMARY: ADMA also beautifully explains many facets of the pathophysiology of chronic kidney disease.
  • Future preclinical and especially clinical studies are required to prove the importance of ADMA in renal and cardiovascular disease.
  • [MeSH-major] Arginine / analogs & derivatives. Kidney Diseases / therapy

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  • (PMID = 18941355.001).
  • [ISSN] 1062-4821
  • [Journal-full-title] Current opinion in nephrology and hypertension
  • [ISO-abbreviation] Curr. Opin. Nephrol. Hypertens.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Biomarkers; 30315-93-6 / N,N-dimethylarginine; 9002-72-6 / Growth Hormone; 94ZLA3W45F / Arginine; EC 3.5.- / Amidohydrolases; EC 3.5.3.18 / dimethylargininase
  • [Number-of-references] 69
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98. Sankaran D, Bankovic-Calic N, Peng CY, Ogborn MR, Aukema HM: Dietary flax oil during pregnancy and lactation retards disease progression in rat offspring with inherited kidney disease. Pediatr Res; 2006 Dec;60(6):729-33
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Dietary flax oil during pregnancy and lactation retards disease progression in rat offspring with inherited kidney disease.
  • Dietary flax oil (FO) retards disease progression in growing or adult animal models of kidney disease.
  • To determine whether dietary flax oil during the perinatal period would alter renal disease progression in offspring, Han-SPRD-cy rats with inherited cystic kidney disease were given diets with either 7% FO or corn oil (CO), throughout pregnancy and lactation.
  • Rats given FO during the maternal period had 15% less renal cyst growth compared with rats given FO only in the postweaning period.
  • Dietary FO, compared with CO, in the maternal period also resulted in 12% lower cell proliferation and 15% less oxidant injury in diseased kidneys of offspring.
  • Including FO in both the maternal and postweaning period resulted in 29-34% less renal interstitial fibrosis and 22-23% lower glomerular hypertrophy.
  • The potential for dietary FO during pregnancy and lactation to positively modulate adult renal disease has significant implications for the 1 in 1000 individuals with congenital cystic kidney disease.
  • [MeSH-major] Dietary Fats, Unsaturated / pharmacology. Lactation / physiology. Linseed Oil / pharmacology. Polycystic Kidney Diseases / genetics. Polycystic Kidney Diseases / physiopathology. Pregnancy, Animal / physiology
  • [MeSH-minor] Animals. Animals, Newborn. Cell Proliferation. Disease Progression. Female. Fibrosis / pathology. Hypertrophy / pathology. Kidney / metabolism. Kidney / pathology. Male. Pregnancy. Rats. Rats, Mutant Strains

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  • (PMID = 17065582.001).
  • [ISSN] 0031-3998
  • [Journal-full-title] Pediatric research
  • [ISO-abbreviation] Pediatr. Res.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Dietary Fats, Unsaturated; 8001-26-1 / Linseed Oil
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99. Boschiero L, Fior F, Nacchia F: Bimodal distribution of major cardiovascular events in kidney allograft recipients. Transplant Proc; 2009 May;41(4):1183-6
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Bimodal distribution of major cardiovascular events in kidney allograft recipients.
  • Cardiovascular disease (CVD) is the major cause of death after renal transplantation.
  • We have retrospectively analyzed the incidence and the time of appearance of CVD among 870 consecutive cadaveric kidney transplant recipients, including 143 patients (16.5%) who experienced a fatal or nonfatal event after transplantation.
  • This trend of CVD after kidney transplantation may be explained by inadequate evaluation and management of CVD risk factors during waiting list time and, after transplantation, by the cumulative effects of traditional and nontraditional risk factors.
  • [MeSH-major] Cardiovascular Diseases / epidemiology. Kidney Transplantation

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  • (PMID = 19460511.001).
  • [ISSN] 1873-2623
  • [Journal-full-title] Transplantation proceedings
  • [ISO-abbreviation] Transplant. Proc.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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100. Palmer S, McGregor DO, Strippoli GF: Interventions for preventing bone disease in kidney transplant recipients. Cochrane Database Syst Rev; 2005;(2):CD005015
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Interventions for preventing bone disease in kidney transplant recipients.
  • BACKGROUND: Patients with chronic kidney disease have significant abnormalities of bone remodelling and calcium homeostasis and are at increased risk of fracture.
  • The fracture risk for a kidney transplant recipient is four times that of the general population and higher than that for a patient on dialysis.
  • OBJECTIVES: To evaluate the use of interventions for the treatment of bone disease following kidney transplantation.
  • SELECTION CRITERIA: Randomised trials of treatment of bone disease following kidney transplantation were included.
  • Trials of recipients of any transplant other than a kidney transplant including trials of kidney-pancreas transplants were excluded.
  • AUTHORS' CONCLUSIONS: No benefit from any intervention known to reduce risk of fracture from bone disease could be demonstrated to reduce fracture incidence in kidney transplant recipients.
  • [MeSH-major] Fractures, Bone / prevention & control. Kidney Transplantation / adverse effects
  • [MeSH-minor] Bone Diseases / etiology. Bone Diseases / prevention & control. Calcitonin / therapeutic use. Calcium / therapeutic use. Diphosphonates / therapeutic use. Female. Humans. Kidney Diseases / complications. Male. Randomized Controlled Trials as Topic. Vitamin D / therapeutic use

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  • [UpdateIn] Cochrane Database Syst Rev. 2007;(3):CD005015 [17636784.001]
  • (PMID = 15846740.001).
  • [ISSN] 1469-493X
  • [Journal-full-title] The Cochrane database of systematic reviews
  • [ISO-abbreviation] Cochrane Database Syst Rev
  • [Language] eng
  • [Publication-type] Journal Article; Meta-Analysis; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Diphosphonates; 1406-16-2 / Vitamin D; 9007-12-9 / Calcitonin; SY7Q814VUP / Calcium
  • [Number-of-references] 64
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