BioMedLib Search Engine
[ goto HOMEPAGE ]
Find more relevant answers, faster!
Skip to content
Advanced Search
Search History
MeSH Query
Page Format
Query is expanded
Login
Skip to content
Export Citations
Search Results
RSS
Email
Articles' Details
Start new query
Reset All
Refine your query
(more in Advanced-Search):
Search all of MEDLINE
Focus on the recent 5 years
Focus on the current year
Focus on the last 30 days
More choices ...
Focus on articles with free fulltexts
More choices ...
Do simple 'keyword' search (no query expansion)
[X] Close
You are about to erase all the values you have customized, search history, page format, etc.
Click
here to
RESET
all values
Click
here to
GO BACK
without resetting any value
Advanced Search
Submit one or more of the following items, and they will be searched along with your query in the search box above.
Any submit button will submit all of the items you have changed.
+
Publication-Date
Published in the last:
30 days
60 days
90 days
6 months
12 months
this year
2 years
3 years
5 years
10 years
Or published in the following date range: From (yyyy/mm/dd - month and day are optional)
to ('to' is optional)
+
Full Text
Retrieve articles with hyperlinks to:
full text (either free or subscription)
free full text
subscription full text
no full text link
+
Sort-Order
Sort the retrieved articles by:
relevance
publication date
+
Language
And with languages:
English
French
German
Italian
Japanese
Russian
Spanish
More languages:
Afrikaans
Albanian
Amharic
Arabic
Armenian
Azerbaijani
Bengali
Bosnian
Bulgarian
Catalan
Chinese
Czech
Danish
Dutch
Esperanto
Estonian
Finnish
Georgian
Scottish Gaelic
Greek, Modern
Hebrew
Hindi
Hungarian
Icelandic
Indonesian
Kinyarwanda
Korean
Latin
Latvian
Lithuanian
Macedonian
Malayalam
Maori
Malay
Multiple languages
Norwegian
Persian
Polish
Portuguese
Pushto
Romanian
Sanskrit
Serbian
Croatian
Slovak
Slovenian
Swedish
Thai
Turkish
Ukrainian
Undetermined
Urdu
Vietnamese
Welsh
+
Publication-Type
And with publication types:
Clinical Trial
Editorial
Letter
Meta-Analysis
Practice Guideline
Randomized Controlled Trial
Review
More publication types:
Addresses
Bibliography
Biography
Case Reports
Classical Article
Clinical Conference
Clinical Trial, Phase I
Clinical Trial, Phase II
Clinical Trial, Phase III
Clinical Trial, Phase IV
Comment
Comparative Study
Congresses
Consensus Development Conference
Consensus Development Conference, NIH
Controlled Clinical Trial
Corrected and Republished Article
Dictionary
Directory
Duplicate Publication
English Abstract
Evaluation Studies
Festschrift
Government Publications
Guideline
Historical Article
Interactive Tutorial
Interview
Introductory Journal Article
In Vitro
Journal Article
Lectures
Legal Cases
Legislation
Multicenter Study
News
Newspaper Article
Overall
Patient Education Handout
Periodical Index
Portraits
Published Erratum
Retracted Publication
Research Support, N.I.H., Extramural
Research Support, N.I.H., Intramural
Research Support, Non-U.S. Gov't
Research Support, U.S. Gov't, Non-P.H.S.
Research Support, U.S. Gov't, P.H.S.
Retraction of Publication
Scientific Integrity Review
Technical Report
Twin Study
Validation Studies
+
Species
And for:
Humans
Animals
+
Gender
And for:
Male
Female
+
Age
And for these age groups:
Newborn: birth to 1 month
Infant: 1 to 23 months
Preschool child: 2 to 5 years
Child: 6 to 12 years
Adolescent: 13 to 18 years
Adult: 19 to 44 years
Middle aged: 45 to 64 years
Aged: 65+ years
80 and over: 80+ years
+
Title
And for this query matching the titles:
+
Transliterated-Title
And for this query matching the title in original language:
+
Abstract
And for this query matching the abstratcs:
+
Major-Mesh
And for this query matching the MeSH-Major terms:
+
Mesh
And for this query matching any MeSH terms:
+
Journal
And for one or more of these journal abbreviated names:
OR
OR
(see
title abbreviations
)
+
Volume
And with journal volume number:
+
Issue
And with journal issue number:
+
Page
And with page number:
+
ISSN
And with ISSN:
+
Publication-Place
And with journal's country of publication:
+
Author
And for...
all these author names:
AND
AND
(see
help
)
one or more of these author names:
OR
OR
but not having any of these unwanted author names:
NOT
NOT
+
Affiliation
And with affiliation to:
+
Has-Abstract
Find MEDLINE records with the abstract status:
has abstract
does not have abstract
include both record types
include both record types but rank higher the records having abstract (the default BML behavior)
+
PMID
Show me only articles for these PMIDs (PubMed IDs):
+
Semantic-Type
And with semantic types:
A. Entity
A1. Physical Object
A1.1. Organism
A1.1.1. Archaeon
A1.1.2. Bacterium
A1.1.3. Eukaryote
A1.1.3.1. Animal
A1.1.3.1.1. Vertebrate
A1.1.3.1.1.1. Amphibian
A1.1.3.1.1.2. Bird
A1.1.3.1.1.3. Fish
A1.1.3.1.1.4. Mammal
A1.1.3.1.1.4.1. Human
A1.1.3.1.1.5. Reptile
A1.1.3.2. Fungus
A1.1.3.3. Plant
A1.1.4. Virus
A1.2. Anatomical Structure
A1.2.1. Embryonic Structure
A1.2.2. Anatomical Abnormality
A1.2.2.1. Congenital Abnormality
A1.2.2.2. Acquired Abnormality
A1.2.3. Fully Formed Anatomical Structure
A1.2.3.1. Body Part, Organ, or Organ Component
A1.2.3.2. Tissue
A1.2.3.3. Cell
A1.2.3.4. Cell Component
A1.2.3.5. Gene or Genome
A1.3. Manufactured Object
A1.3.1. Medical Device
A1.3.1.1. Drug Delivery Device
A1.3.2. Research Device
A1.3.3. Clinical Drug
A1.4. Substance
A1.4.1. Chemical
A1.4.1.1. Chemical Viewed Functionally
A1.4.1.1.1. Pharmacologic Substance
A1.4.1.1.1.1. Antibiotic
A1.4.1.1.2. Biomedical or Dental Material
A1.4.1.1.3. Biologically Active Substance
A1.4.1.1.3.1. Neuroreactive Substance or Biogenic Amine
A1.4.1.1.3.2. Hormone
A1.4.1.1.3.3. Enzyme
A1.4.1.1.3.4. Vitamin
A1.4.1.1.3.5. Immunologic Factor
A1.4.1.1.3.6. Receptor
A1.4.1.1.4. Indicator, Reagent, or Diagnostic Aid
A1.4.1.1.5. Hazardous or Poisonous Substance
A1.4.1.2. Chemical Viewed Structurally
A1.4.1.2.1. Organic Chemical
A1.4.1.2.1.5. Nucleic Acid, Nucleoside, or Nucleotide
A1.4.1.2.1.6. Organophosphorus Compound
A1.4.1.2.1.7. Amino Acid, Peptide, or Protein
A1.4.1.2.1.8. Carbohydrate
A1.4.1.2.1.9. Lipid
A1.4.1.2.1.9.1. Steroid
A1.4.1.2.1.9.2. Eicosanoid
A1.4.1.2.2. Inorganic Chemical
A1.4.1.2.3. Element, Ion, or Isotope
A1.4.2. Body Substance
A1.4.3. Food
A2. Conceptual Entity
A2.1. Idea or Concept
A2.1.1. Temporal Concept
A2.1.2. Qualitative Concept
A2.1.3. Quantitative Concept
A2.1.4. Functional Concept
A2.1.4.1. Body System
A2.1.5. Spatial Concept
A2.1.5.1. Body Space or Junction
A2.1.5.2. Body Location or Region
A2.1.5.3. Molecular Sequence
A2.1.5.3.1. Nucleotide Sequence
A2.1.5.3.2. Amino Acid Sequence
A2.1.5.3.3. Carbohydrate Sequence
A2.1.5.4. Geographic Area
A2.2. Finding
A2.2.1. Laboratory or Test Result
A2.2.2. Sign or Symptom
A2.3. Organism Attribute
A2.3.1. Clinical Attribute
A2.4. Intellectual Product
A2.4.1. Classification
A2.4.2. Regulation or Law
A2.5. Language
A2.6. Occupation or Discipline
A2.6.1. Biomedical Occupation or Discipline
A2.7. Organization
A2.7.1. Health Care Related Organization
A2.7.2. Professional Society
A2.7.3. Self-help or Relief Organization
A2.8. Group Attribute
A2.9. Group
A2.9.1. Professional or Occupational Group
A2.9.2. Population Group
A2.9.3. Family Group
A2.9.4. Age Group
A2.9.5. Patient or Disabled Group
B. Event
B1. Activity
B1.1. Behavior
B1.1.1. Social Behavior
B1.1.2. Individual Behavior
B1.2. Daily or Recreational Activity
B1.3. Occupational Activity
B1.3.1. Health Care Activity
B1.3.1.1. Laboratory Procedure
B1.3.1.2. Diagnostic Procedure
B1.3.1.3. Therapeutic or Preventive Procedure
B1.3.2. Research Activity
B1.3.2.1. Molecular Biology Research Technique
B1.3.3. Governmental or Regulatory Activity
B1.3.4. Educational Activity
B1.4. Machine Activity
B2. Phenomenon or Process
B2.1. Human-caused Phenomenon or Process
B2.1.1. Environmental Effect of Humans
B2.2. Natural Phenomenon or Process
B2.2.1. Biologic Function
B2.2.1.1. Physiologic Function
B2.2.1.1.1. Organism Function
B2.2.1.1.1.1. Mental Process
B2.2.1.1.2. Organ or Tissue Function
B2.2.1.1.3. Cell Function
B2.2.1.1.4. Molecular Function
B2.2.1.1.4.1. Genetic Function
B2.2.1.2. Pathologic Function
B2.2.1.2.1. Disease or Syndrome
B2.2.1.2.1.1. Mental or Behavioral Dysfunction
B2.2.1.2.1.2. Neoplastic Process
B2.2.1.2.2. Cell or Molecular Dysfunction
B2.2.1.2.3. Experimental Model of Disease
B2.3. Injury or Poisoning
Page Format
Any submit button will submit all of the items you have changed.
[theme]
Use this page design theme:
original
twenty ten
[shown]
Results per page:
5
10
20
50
100
200
500
[expand/collapse]
show these sections expanded by default:
Advanced search
MeSH query
Search history
Page format
Query expansion
Articles details
Export citations
Email
[text size]
use this font size for text:
25%
50%
75%
100%
125%
150%
200%
or enter your choice of font size:
[page width]
use this page width (relative to the default initial value):
25%
50%
75%
100%
125%
150%
200%
or enter your choice of page width:
[highlight color]
use this color to highlight query words in the articles:
red
green
blue
black
purple
yellow
orange
navy
olive
maroon
none
[query history]
maximum number of queries shown in the history section:
[annotate]
Annotate these parts of each article:
title
abstract
both
none
Reset all values
Find best MeSH terms for
Search History
1
benign renal neoplasm 2005:2010[pubdate] *count=100
61321 results
Searchbox
Export
PDF
RSS
Email
Delete
Email this search result to the following email address:
[X] Close
Expand the query
'
benign renal neoplasm
' expanded to all its synonyms;
details
Email the results to the following email address:
Export the checked citations in RIS format (RIS format is used by RefWorks, Endnote, among others).
Items 1 to 100 of about 61321
1.
Verrey F, Singer D, Ramadan T, Vuille-dit-Bille RN, Mariotta L, Camargo SM:
Kidney amino acid transport.
Pflugers Arch
; 2009 May;458(1):53-60
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Kidney
amino acid transport.
Near complete reabsorption of filtered amino acids is a main specialized transport function of the
kidney
proximal tubule.
A number of other amino acid transporters expressed in the basolateral membrane of proximal
kidney
tubule cells subserve either specialized metabolic functions, such as the production of ammonium, or are part of the cellular housekeeping equipment.
Some mutations
of B
(0)AT1 differentially interact with these accessory subunits, providing an explanation for differential intestinal phenotypes among Hartnup patients.
The basolateral efflux of numerous amino acids from
kidney
tubular cells is mediated by heteromeric amino acid transporters that function as obligatory exchangers.
[MeSH-major]
Amino Acid Transport Systems / metabolism. Amino Acids / metabolism.
Kidney
/ metabolism
[MeSH-minor]
Amino Acid Transport Systems, Neutral / metabolism. Amino Acid Transport Systems, Neutral / physiology. Animals. Anion Transport Proteins / physiology. Antiporters / physiology. Biological Transport. Humans.
Kidney
Tubules, Proximal / physiology
COS Scholar Universe.
author profiles
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Nat Genet. 1999 Mar;21(3):293-6
[
10080182.001
]
[Cites]
Nature. 1996 Oct 17;383(6601):634-7
[
8857541.001
]
[Cites]
J Pharmacol Exp Ther. 2004 Aug;310(2):695-702
[
15051798.001
]
[Cites]
Pflugers Arch. 2005 Nov;451(2):338-48
[
16133263.001
]
[Cites]
Curr Opin Clin Nutr Metab Care. 2000 Jan;3(1):51-7
[
10642084.001
]
[Cites]
Gene. 2002 Jun 12;292(1-2):81-90
[
12119102.001
]
[Cites]
J Biol Chem. 1999 Dec 3;274(49):34948-54
[
10574970.001
]
[Cites]
Am J Clin Nutr. 2004 Feb;79(2):185-97
[
14749222.001
]
[Cites]
Pflugers Arch. 2003 Feb;445(5):529-33
[
12634921.001
]
[Cites]
Nat Genet. 2004 Sep;36(9):999-1002
[
15286787.001
]
[Cites]
J Am Soc Nephrol. 2003 Apr;14(4):837-47
[
12660317.001
]
[Cites]
Physiol Rev. 1998 Oct;78(4):969-1054
[
9790568.001
]
[Cites]
J Biol Chem. 2002 Jun 7;277(23):21017-26
[
11907033.001
]
[Cites]
Physiol Rev. 2008 Jan;88(1):249-86
[
18195088.001
]
[Cites]
J Clin Invest. 2008 Dec;118(12):3881-92
[
19033659.001
]
[Cites]
Nature. 2006 Dec 21;444(7122):1088-91
[
17167413.001
]
[Cites]
EMBO J. 1999 Jan 4;18(1):49-57
[
9878049.001
]
[Cites]
Biochem J. 2005 Aug 1;389(Pt 3):745-51
[
15804236.001
]
[Cites]
Nature. 2004 Oct 14;431(7010):811-8
[
15483603.001
]
[Cites]
Pflugers Arch. 2004 Feb;447(5):465-8
[
14624363.001
]
[Cites]
J Biol Chem. 1996 Jun 21;271(25):14883-90
[
8662767.001
]
[Cites]
Tohoku J Exp Med. 2006 Jan;208(1):25-31
[
16340170.001
]
[Cites]
Biochem Biophys Res Commun. 2003 May 16;304(4):747-54
[
12727219.001
]
[Cites]
Nature. 1992 Dec 3;360(6403):467-71
[
1280334.001
]
[Cites]
Gastroenterology. 2004 Nov;127(5):1410-22
[
15521011.001
]
[Cites]
Am J Physiol Renal Physiol. 2007 Feb;292(2):F555-66
[
17003226.001
]
[Cites]
Am J Physiol Renal Physiol. 2006 Feb;290(2):F376-83
[
16174864.001
]
[Cites]
J Biol Chem. 1998 Dec 4;273(49):32437-45
[
9829974.001
]
[Cites]
Pflugers Arch. 2004 Feb;447(5):610-8
[
12905028.001
]
[Cites]
J Biol Chem. 2004 Jun 4;279(23):24467-76
[
15044460.001
]
[Cites]
J Inherit Metab Dis. 2007 Oct;30(5):716-21
[
17588131.001
]
[Cites]
Am J Physiol. 1997 Dec;273(6 Pt 2):F1023-9
[
9435692.001
]
[Cites]
Am J Physiol Renal Physiol. 2007 Feb;292(2):F533-44
[
16985211.001
]
[Cites]
Annu Rev Nutr. 1995;15:133-59
[
8527215.001
]
[Cites]
FASEB J. 2008 Aug;22(8):2880-7
[
18424768.001
]
[Cites]
Pflugers Arch. 2004 Feb;447(5):776-9
[
12748860.001
]
[Cites]
Am J Physiol Endocrinol Metab. 2002 Feb;282(2):E428-34
[
11788376.001
]
[Cites]
Mol Cell Biol. 2004 May;24(10 ):4166-73
[
15121838.001
]
[Cites]
Biochem J. 2005 Mar 15;386(Pt 3):417-22
[
15689184.001
]
[Cites]
Nature. 2005 Sep 8;437(7056):215-23
[
16041361.001
]
[Cites]
J Membr Biol. 2001 Apr 1;180(3):213-20
[
11337893.001
]
[Cites]
Proc Natl Acad Sci U S A. 2001 Jun 19;98(13):7206-11
[
11390972.001
]
[Cites]
J Membr Biol. 1999 Dec 1;172(3):181-92
[
10568788.001
]
[Cites]
Am J Physiol Cell Physiol. 2001 Sep;281(3):C963-71
[
11502573.001
]
[Cites]
Am J Physiol Renal Physiol. 2001 Jan;280(1):F10-8
[
11133510.001
]
[Cites]
J Membr Biol. 1982;66(3):213-25
[
6808139.001
]
[Cites]
J Biol Chem. 2005 Mar 11;280(10):8974-84
[
15632147.001
]
[Cites]
Gastroenterology. 2009 Mar;136(3):872-82
[
19185582.001
]
[Cites]
Pflugers Arch. 2007 Jun;454(3):507-16
[
17273864.001
]
[Cites]
Curr Probl Clin Biochem. 1976;6:173-89
[
11964.001
]
[Cites]
Physiology (Bethesda). 2006 Apr;21:93-102
[
16565475.001
]
[Cites]
J Cell Physiol. 2006 Mar;206(3):771-9
[
16245314.001
]
[Cites]
Annu Rev Physiol. 2005;67:557-72
[
15709970.001
]
[Cites]
Physiol Rev. 1990 Jan;70(1):43-77
[
2404290.001
]
[Cites]
J Biol Chem. 2003 Jan 10;278(2):1316-22
[
12417581.001
]
[Cites]
Nat Genet. 1999 Mar;21(3):297-301
[
10080183.001
]
[Cites]
J Am Soc Nephrol. 2005 Apr;16(4):869-77
[
15716335.001
]
[Cites]
Am J Physiol Renal Physiol. 2003 May;284(5):F885-92
[
12676733.001
]
[Cites]
J Biol Chem. 2006 Sep 8;281(36):26552-61
[
16825196.001
]
[Cites]
Nat Genet. 2004 Sep;36(9):1003-7
[
15286788.001
]
(PMID = 19184091.001).
[ISSN]
1432-2013
[Journal-full-title]
Pflugers Archiv : European journal of physiology
[ISO-abbreviation]
Pflugers Arch.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
Germany
[Chemical-registry-number]
0 / Amino Acid Transport Systems; 0 / Amino Acid Transport Systems, Neutral; 0 / Amino Acids; 0 / Anion Transport Proteins; 0 / Antiporters; 0 / SLC26A8 protein, human; 0 / SLC6A19 protein, human
[Number-of-references]
60
2.
Zhou Y, Vaidya VS, Brown RP, Zhang J, Rosenzweig BA, Thompson KL, Miller TJ, Bonventre JV, Goering PL:
Comparison of kidney injury molecule-1 and other nephrotoxicity biomarkers in urine and kidney following acute exposure to gentamicin, mercury, and chromium.
Toxicol Sci
; 2008 Jan;101(1):159-70
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Comparison
of kidney
injury molecule-1 and other nephrotoxicity biomarkers in urine and
kidney
following acute exposure to gentamicin, mercury, and chromium.
Sensitive biomarkers are needed to detect
kidney
injury at the earliest stages.
The objective of this study was to determine whether the appearance
of kidney
injury molecule-1 (Kim-1) protein ectodomain in urine and
kidney
injury molecule-1/hepatitis A viral cellular receptor-1 (Kim-1/Havcr1) gene expression in
kidney
tissue may be more predictive of
renal
injury after exposure to nephrotoxicants when compared to traditionally used biomarkers.
The results showed that increases in urinary Kim-1 and
kidney
Kim-1/Havcr1 gene expression paralleled the degree of severity of
renal
histopathology and were detected at lower doses of nephrotoxicants when compared to blood urea nitrogen (BUN), serum creatinine, and urinary N-acetyl-beta-D-glucosaminidase (NAG).
Urinary Kim-1 and
kidney
Kim-1/Havcr1 expression appear to be sensitive and tissue-specific biomarkers that will improve detection of early acute
kidney
injury following exposure to nephrotoxic chemicals and drugs.
MedlinePlus Health Information.
consumer health - Kidney Diseases
.
MedlinePlus Health Information.
consumer health - Mercury
.
COS Scholar Universe.
author profiles
.
Hazardous Substances Data Bank.
MERCURY, ELEMENTAL
.
Hazardous Substances Data Bank.
CHROMIUM, ELEMENTAL
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Acta Vitaminol Enzymol. 1984;6(2):103-7
[
6496253.001
]
[Cites]
Am J Physiol Renal Physiol. 2007 Jan;292(1):F313-20
[
16896183.001
]
[Cites]
Environ Health Perspect. 1991 May;92:71-4
[
1935854.001
]
[Cites]
Pharmacol Toxicol. 1991 May;68(5):317-21
[
1946176.001
]
[Cites]
Gen Pharmacol. 1995 Nov;26(7):1477-87
[
8690234.001
]
[Cites]
EMBO J. 1996 Aug 15;15(16):4282-96
[
8861957.001
]
[Cites]
Drug Saf. 1997 Mar;16(3):205-31
[
9098657.001
]
[Cites]
J Biol Chem. 1998 Feb 13;273(7):4135-42
[
9461608.001
]
[Cites]
Toxicol Pathol. 1998 Jan-Feb;26(1):92-103
[
9502391.001
]
[Cites]
J Virol. 1998 Aug;72(8):6621-8
[
9658108.001
]
[Cites]
J Am Soc Nephrol. 2005 Apr;16(4):1126-34
[
15744000.001
]
[Cites]
BMC Nephrol. 2005;6:4
[
15854231.001
]
[Cites]
J Am Soc Nephrol. 2005 Nov;16(11):3365-70
[
16177006.001
]
[Cites]
Am J Physiol Renal Physiol. 2006 Feb;290(2):F517-29
[
16174863.001
]
[Cites]
J Am Soc Nephrol. 2007 Mar;18(3):904-12
[
17267747.001
]
[Cites]
Am J Surg Pathol. 2007 Mar;31(3):371-81
[
17325478.001
]
[Cites]
Ren Fail. 2002 Nov;24(6):687-90
[
12472192.001
]
[Cites]
Free Radic Biol Med. 2003 Jun 1;34(11):1390-8
[
12757849.001
]
[Cites]
Toxicol Sci. 2003 Sep;75(1):208-22
[
12832660.001
]
[Cites]
Am J Physiol Renal Physiol. 2004 Mar;286(3):F552-63
[
14600030.001
]
[Cites]
Am J Med. 1968 May;44(5):664-705
[
5646427.001
]
[Cites]
Toxicol Appl Pharmacol. 1980 Jul;54(3):443-53
[
6446781.001
]
[Cites]
J Toxicol Environ Health. 1982 Jan;9(1):119-26
[
6460873.001
]
[Cites]
Biochem Pharmacol. 1982 Oct 1;31(19):3093-100
[
6216890.001
]
[Cites]
Pharmacol Rev. 2000 Mar;52(1):113-43
[
10699157.001
]
[Cites]
Therapie. 2000 Jan-Feb;55(1):91-6
[
10860006.001
]
[Cites]
J Environ Sci Health B. 2001 Sep;36(5):687-97
[
11599730.001
]
[Cites]
Kidney Int. 2002 Jul;62(1):237-44
[
12081583.001
]
[Cites]
Cleve Clin J Med. 2002 Jul;69(7):569-74
[
12109642.001
]
[Cites]
J Biol Chem. 2002 Oct 18;277(42):39739-48
[
12138159.001
]
[Cites]
Kidney Int. 1991 Apr;39(4):639-46
[
1711136.001
]
[Cites]
Am J Physiol Renal Physiol. 2006 Aug;291(2):F456-64
[
16467126.001
]
[Cites]
Am J Physiol Renal Physiol. 2007 Jan;292(1):F131-9
[
16835406.001
]
[Cites]
Am J Physiol Renal Physiol. 2002 Dec;283(6):F1326-36
[
12388382.001
]
(PMID = 17934191.001).
[ISSN]
1096-6080
[Journal-full-title]
Toxicological sciences : an official journal of the Society of Toxicology
[ISO-abbreviation]
Toxicol. Sci.
[Language]
ENG
[Grant]
United States / NIDDK NIH HHS / DK / DK 072831; United States / NIEHS NIH HHS / ES / ES016723-02; United States / NIDDK NIH HHS / DK / R37 DK039773; United States / NIDDK NIH HHS / DK / DK 074099; United States / NIDDK NIH HHS / DK / R01 DK072381-04; United States / NIDDK NIH HHS / DK / R01 DK072381; United States / NIDDK NIH HHS / DK / R33 DK074099; United States / NIEHS NIH HHS / ES / R00 ES016723-02; United States / NIDDK NIH HHS / DK / DK 039773; United States / NIDDK NIH HHS / DK / R21 DK074099; United States / NIEHS NIH HHS / ES / R00 ES016723; United States / NIDDK NIH HHS / DK / R01 DK039773; None / None / / R01 DK072381-04
[Publication-type]
Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
[Publication-country]
United States
[Chemical-registry-number]
0 / Biomarkers; 0 / Cell Adhesion Molecules; 0 / Gentamicins; 0 / Havcr1protein, rat; 0 / Membrane Proteins; 0 / Protein Synthesis Inhibitors; 0R0008Q3JB / Chromium; 63231-63-0 / RNA; 7535-00-4 / Galactosamine; FXS1BY2PGL / Mercury; V956696549 / Acetylglucosamine
[Other-IDs]
NLM/ NIHMS110357; NLM/ PMC2744478
3.
Resende L, Guerra J, Santana A, Mil-Homens C, Abreu F, da Costa AG:
Influence of dialysis duration and modality on kidney transplant outcomes.
Transplant Proc
; 2009 Apr;41(3):837-9
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Influence of dialysis duration and modality on
kidney
transplant outcomes.
BACKGROUND: The influence of pretransplantation dialysis on
kidney
transplant outcomes has been the subject of longstanding interest.
Although increased time on dialysis prior to
kidney
transplantation is associated with decreased graft and patient survivals, analyses of the impact of dialysis modality on
kidney
allograft outcome have produced conflicting results.
OBJECTIVE: The objective of this study was to evaluate the influence of dialysis duration and modality on the function and survival of
renal
allografts.
PATIENTS: We retrospectively reviewed the clinical data of 421 adults who received first
kidney
transplantations from cadaveric heart-beating donors performed in our unit from May 1989 to May 2007.
Three hundred seventy-four patients (88.8%) were on hemodialysis (HD) prior to
kidney
transplantation, including 247 patients (58.7%) on treatment for at least 24 months.
Renal
function at 3, 12, 60, and 96 months was similar between the 2 groups.
No differences were observed in
renal
function or graft and patient survivals comparing HD or peritoneal dialysis (PD).
[MeSH-major]
Kidney
Failure, Chronic / surgery.
Kidney
Transplantation / physiology.
Renal
Dialysis
[MeSH-minor]
Adult. Age Factors. Female. Graft Survival. Humans.
Kidney
Function Tests. Living Donors. Male. Middle Aged. Postoperative Complications / epidemiology. Retrospective Studies. Sex Characteristics. Survival Analysis. Survivors. Time Factors. Tissue Donors / supply & distribution. Treatment Outcome. Young Adult
MedlinePlus Health Information.
consumer health - Dialysis
.
MedlinePlus Health Information.
consumer health - Kidney Failure
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19376365.001).
[ISSN]
0041-1345
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
Advertisement
4.
Casas-Aparicio G, Castillo-Martínez L, Orea-Tejeda A, Abasta-Jiménez M, Keirns-Davies C, Rebollar-González V:
The effect of successful kidney transplantation on ventricular dysfunction and pulmonary hypertension.
Transplant Proc
; 2010 Nov;42(9):3524-8
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
The effect of successful
kidney
transplantation on ventricular dysfunction and pulmonary hypertension.
BACKGROUND: Heart disease is a frequent complication of chronic
kidney
disease and the major cause of death in patients on
renal
replacement therapy.
The purpose of this study was to evaluate the impact of successful
kidney
transplantation on systolic and diastolic ventricular dysfunction and pulmonary arterial hypertension in patients with chronic
kidney
disease (CKD).
METHODS: The study included 35 patients >18 years of age with CKD who had successful
kidney
transplantations.
The LVEF of the entire group increased from 52% to 64% (P < .001) by 12 months after
kidney
transplant.
CONCLUSIONS: Because
kidney
transplantation led to considerable improvement in left ventricular systolic and diastolic function as well as pulmonary arterial pressure of patients with CKD, optimal treatment for dysfunction and transplant as soon as possible is recommended.
[MeSH-major]
Hypertension, Pulmonary / etiology.
Kidney
Failure, Chronic / surgery.
Kidney
Transplantation. Ventricular Dysfunction, Left / etiology
Genetic Alliance.
consumer health - Pulmonary Hypertension
.
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Kidney Failure
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Pulmonary Hypertension
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Copyright]
Copyright © 2010 Elsevier Inc. All rights reserved.
(PMID = 21094809.001).
[ISSN]
1873-2623
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
5.
Canales MT, Kasiske BL, Rosenberg ME:
Transplant tourism: Outcomes of United States residents who undergo kidney transplantation overseas.
Transplantation
; 2006 Dec 27;82(12):1658-61
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Transplant tourism: Outcomes of United States residents who undergo
kidney
transplantation overseas.
BACKGROUND: Although international commerce in
kidney
transplantation is a reality, little is known about U.S. residents who travel abroad for
kidney
transplantation.
METHODS: We retrospectively reviewed the clinical outcomes of patients who were evaluated at the University of Minnesota Medical Center or Hennepin County Medical Center, but then surreptitiously underwent
kidney
transplantation overseas.
RESULTS: We identified 10 patients who underwent
kidney
transplantation outside the United States between September 16, 2002 and June 30, 2006 and then returned for care in our programs.
CONCLUSIONS:
Kidney
function and graft survival were generally good after surreptitious overseas
kidney
transplantation.
[MeSH-major]
Graft Survival.
Kidney
Transplantation. Travel
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Traveler's Health
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17198255.001).
[ISSN]
0041-1337
[Journal-full-title]
Transplantation
[ISO-abbreviation]
Transplantation
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
6.
Mosconi G, Panicali L, Persici E, Conte D, Cappuccilli ML, Cuna V, Capelli I, Todeschini P, D'Arcangelo GL, Stefoni S:
Native kidney function after renal transplantation combined with other solid organs in preemptive patients.
Transplant Proc
; 2010 May;42(4):1017-20
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Native
kidney
function after
renal
transplantation combined with other solid organs in preemptive patients.
Kidney
transplantations combined with other solid organs are progressively increasing in number.
There are no guidelines regarding the nephrologic indications for combined transplantations, namely liver-
kidney
(LKT), or heart-
kidney
(HKT), in preemptive patients with chronic
kidney
failure who are not on regular dialysis therapy.
The objective of this study was to assess the functional contribution of the native
kidneys
after preemptive
kidney
transplantation combined with other solid organs.
From 2004, 9 patients (aged 50.3 +/- 8.5 years) with chronic
kidney
failure (creatinine 2.5 +/- 1.0 mg/dL) caused by polycystic
kidney
disease (n = 4), vascular nephropathy (n = 2), interstitial nephropathy (n = 1), glomerulonephritis (n = 1), or end-stage
kidney
disease (n = 1), underwent combined transplantations (8 LKT, 1 HKT).
A scintigraphic functional study (Tc-99DMSA or Tc-99mMAG3), was performed at 4 +/- 3 months after transplantation to evaluate the functional contribution of both the native
kidneys
and the graft.
At the time of scintigraphy,
renal
function in all patients was 1.3 +/- 0.3 mg/dL.
The functional contribution of the transplanted
kidneys
was on average 77 +/- 18%.
At follow-up after 36 months, patient and
kidney
survivals were 100%.
The study confirmed a high risk of loss of native
kidney
function in the presence of organic nephropathy.
[MeSH-major]
Kidney
Transplantation / physiology. Organ Transplantation / physiology
[MeSH-minor]
Adult. Creatinine / blood. Female. Glomerular Filtration Rate. Heart Transplantation / physiology. Humans.
Kidney
Diseases / classification.
Kidney
Diseases / surgery.
Kidney
Failure, Chronic / surgery.
Kidney
Function Tests. Liver Transplantation / physiology. Male. Middle Aged. Polycystic
Kidney
Diseases / surgery
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Organ Transplantation
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Copyright]
Copyright (c) 2010. Published by Elsevier Inc.
(PMID = 20534213.001).
[ISSN]
1873-2623
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
AYI8EX34EU / Creatinine
7.
Jiang JC, Li CG, Liang QQ, Bian Q, Zhou Q, Cui XJ, Huang M, Liu QG, Lu S, Zhou CJ, Shi Q, Wang YJ:
[Establishment of a rat model of cervical syndrome with kidney deficiency].
Zhong Xi Yi Jie He Xue Bao
; 2008 Oct;6(10):1034-9
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
[Establishment of a rat model of cervical syndrome with
kidney
deficiency].
OBJECTIVE: To establish a rat model of cervical syndrome (CS) with
kidney
deficiency.
METHODS: A group of 30 three-month-old female Sprague-Dawley rats were randomly divided into normal control group, CS group and CS with
kidney
deficiency group (combined group), with 10 rats in each group.
Rats in the normal control group received no treatment, rats in the CS group underwent only resection of cervical muscles and ligaments as unbalanced dynamic and static animal model, and rats in combined group underwent resection of both cervical muscles and ovaries as
kidney
deficiency model.
Kidney
deficiency was determined by observing the morphologic changes of uterus and appendages, detecting the weight of uterus and appendages and the content of serum estradiol (E(2)).
CONCLUSION: The rat model of CS with
kidney
deficiency is established.
Kidney
deficiency can aggravate cervical intervertebral disc degeneration.
[MeSH-minor]
Animals. Female.
Kidney
Diseases. Ovariectomy. Random Allocation. Rats. Rats, Sprague-Dawley
MedlinePlus Health Information.
consumer health - Neck Injuries and Disorders
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18847538.001).
[ISSN]
1672-1977
[Journal-full-title]
Zhong xi yi jie he xue bao = Journal of Chinese integrative medicine
[ISO-abbreviation]
Zhong Xi Yi Jie He Xue Bao
[Language]
chi
[Publication-type]
English Abstract; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
China
8.
El-Agroudy AE, Sabry AA, Wafa EW, Neamatalla AH, Ismail AM, Mohsen T, Khalil AA, Shokeir AA, Ghoneim MA:
Long-term follow-up of living kidney donors: a longitudinal study.
BJU Int
; 2007 Dec;100(6):1351-5
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Long-term follow-up of living
kidney
donors: a longitudinal study.
OBJECTIVE: To analyse retrospectively the general health status and
renal
and cardiovascular consequences of living-related
kidney
donation, as the long-term effects of unilateral nephrectomy for
kidney
donation are of particular interest with the currently increasing practice of living-donor transplantation.
PATIENTS AND METHODS: Living-related
kidney
donors (1400) who had donated their
kidneys
between 1976 and 2002 were asked to attend a dedicated donor follow-up clinic starting in 2004.
We attempted to contact all donors to determine the long-term outcome of their remaining
kidney
.
All
kidney
donors who responded had a detailed assessment, and were questioned about rehabilitation and their feelings on donating a
kidney
.
[MeSH-major]
Health Status.
Kidney
. Living Donors. Nephrectomy / adverse effects. Tissue and Organ Harvesting / adverse effects
MedlinePlus Health Information.
consumer health - Organ Donation
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17941927.001).
[ISSN]
1464-410X
[Journal-full-title]
BJU international
[ISO-abbreviation]
BJU Int.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
England
9.
Klatte T, Patard JJ, de Martino M, Bensalah K, Verhoest G, de la Taille A, Abbou CC, Allhoff EP, Carrieri G, Riggs SB, Kabbinavar FF, Belldegrun AS, Pantuck AJ:
Tumor size does not predict risk of metastatic disease or prognosis of small renal cell carcinomas.
J Urol
; 2008 May;179(5):1719-26
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Tumor
size does not predict risk of metastatic disease or prognosis of small
renal
cell carcinomas.
PURPOSE: We characterized the clinicopathological features and the prognosis of small solid
renal
tumors
defined as
tumors
4 cm or smaller.
MATERIALS AND METHODS: We identified 1,208 patients who were treated with nephrectomy at 5 international academic centers for small solid
renal
tumors
.
RESULTS: Of the
tumors
88% were
renal
cell carcinoma and 12% were
benign
.
Of those with
renal
cell carcinoma 995 (93%) were localized (N0M0) and 72 (7%) presented with metastatic disease.
Tumor
size did not predict synchronous metastatic disease.
The incidence of metastatic disease in
the tumor
size ranges 0.1 to 1.0, 1.1 to 2.0, 2.1 to 3.0 and 3.1 to 4.0 cm was 7%, 6%, 5% and 8%, respectively (p = 0.322).
The majority of patients who died of
renal
cell carcinoma (54%) presented with synchronous metastatic disease, but 3% of patients with localized disease also died of
renal
cell carcinoma.
Progression-free survival (28 months) was better than for patients with metastatic disease having a primary
tumor
greater than 4 cm (8 months).
Tumor
size was not retained as an independent prognostic factor of survival in multivariate analyses.
The University of California Integrated Staging System and the Karakiewicz nomogram were the best predictors of cancer specific survival for all
renal
cell carcinoma stages (c-index 0.87).
CONCLUSIONS: More than 85% of small solid
renal
tumors
are
renal
cell carcinoma.
The majority of localized small
renal
tumors
can be cured with existing surgical approaches.
Tumor
size alone is not sufficient to distinguish
renal
cell carcinoma with
benign
behavior from aggressive small
renal
cell carcinoma.
Survival of patients with small metastatic
renal
cell carcinoma is better then expected.
The biology of these unique
tumors
should be further studied.
[MeSH-major]
Carcinoma,
Renal
Cell / pathology.
Kidney Neoplasms
/ pathology
[MeSH-minor]
Female. Humans. Male. Middle Aged.
Neoplasm
Recurrence, Local. Nephrectomy. Prognosis. Survival Rate
MedlinePlus Health Information.
consumer health - Kidney Cancer
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[CommentIn]
Eur Urol. 2009 Mar;55(3):751-2
[
19650211.001
]
[CommentIn]
J Urol. 2008 May;179(5):1657
[
18343448.001
]
(PMID = 18343437.001).
[ISSN]
1527-3792
[Journal-full-title]
The Journal of urology
[ISO-abbreviation]
J. Urol.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
10.
Kestenbaum B, Rudser KD, de Boer IH, Peralta CA, Fried LF, Shlipak MG, Palmas W, Stehman-Breen C, Siscovick DS:
Differences in kidney function and incident hypertension: the multi-ethnic study of atherosclerosis.
Ann Intern Med
; 2008 Apr 1;148(7):501-8
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Differences in
kidney
function and incident hypertension: the multi-ethnic study of atherosclerosis.
BACKGROUND:
Kidney
disease and hypertension commonly coexist, yet the direction of their association is still debated.
OBJECTIVE: To evaluate whether early
kidney
dysfunction, measured by serum cystatin C levels and urinary albumin excretion, predates hypertension in adults without clinically recognized
kidney
or cardiovascular disease.
PARTICIPANTS: 2767 MESA participants without prevalent hypertension, cardiovascular disease, or clinically recognized
kidney
disease (an estimated glomerular filtration rate <60 mL/min per 1.73 m2 or microalbuminuria).
LIMITATIONS: Unmeasured characteristics may have confounded observed associations
of kidney
markers with hypertension.
CONCLUSION: Differences in
kidney
function, indicated by cystatin C levels, are associated with incident hypertension among individuals without clinical
kidney
or cardiovascular disease.
These population-based findings complement experimental work implicating early
kidney
damage in the pathogenesis of essential hypertension.
MedlinePlus Health Information.
consumer health - High Blood Pressure
.
COS Scholar Universe.
author profiles
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Am J Kidney Dis. 1996 Dec;28(6):811-21
[
8957032.001
]
[Cites]
J Hum Hypertens. 1996 Oct;10(10):691-4
[
9004096.001
]
[Cites]
Diabetes Care. 1998 Jul;21(7):1076-9
[
9653598.001
]
[Cites]
Ann Intern Med. 1999 Mar 16;130(6):461-70
[
10075613.001
]
[Cites]
Scand J Clin Lab Invest. 1999 Feb;59(1):1-8
[
10206092.001
]
[Cites]
Lancet. 1963 Mar 30;1(7283):677-82
[
14014100.001
]
[Cites]
Circulation. 2005 Mar 22;111(11):1370-6
[
15738353.001
]
[Cites]
Am J Transplant. 2001 Sep;1(3):222-7
[
12102255.001
]
[Cites]
Lancet. 2002 Aug 31;360(9334):659-65
[
12241871.001
]
[Cites]
Arch Intern Med. 2005 Apr 25;165(8):923-8
[
15851645.001
]
[Cites]
Kidney Int. 2005 Jun;67(6):2089-100
[
15882252.001
]
[Cites]
N Engl J Med. 2005 May 19;352(20):2049-60
[
15901858.001
]
[Cites]
Kidney Int Suppl. 2005 Aug;(97):S68-77
[
16014104.001
]
[Cites]
Mayo Clin Proc. 2005 Oct;80(10):1270-7
[
16212138.001
]
[Cites]
J Am Soc Nephrol. 2006 Feb;17(2):331-5
[
16434504.001
]
[Cites]
Ann Intern Med. 2006 Aug 15;145(4):247-54
[
16908915.001
]
[Cites]
Am J Kidney Dis. 2000 Jul;36(1):29-34
[
10873868.001
]
[Cites]
N Engl J Med. 2002 Mar 21;346(12):913-23
[
11907292.001
]
[Cites]
J Am Soc Nephrol. 2002 Apr;13(4):1034-9
[
11912263.001
]
[Cites]
Am J Epidemiol. 2002 Nov 1;156(9):871-81
[
12397006.001
]
[Cites]
Am J Kidney Dis. 2003 Jan;41(1):1-12
[
12500213.001
]
[Cites]
N Engl J Med. 2003 Jan 9;348(2):101-8
[
12519920.001
]
[Cites]
Diabetes Care. 2003 Nov;26(11):3160-7
[
14578255.001
]
[Cites]
Hypertension. 2003 Dec;42(6):1206-52
[
14656957.001
]
[Cites]
Kidney Int. 2004 Apr;65(4):1416-21
[
15086483.001
]
[Cites]
Am J Med. 1972 May;52(5):584-94
[
4337474.001
]
[Cites]
Circ Res. 1975 Jun;36(6):692-6
[
1093748.001
]
[Cites]
J Clin Invest. 1986 Jun;77(6):1993-2000
[
3011863.001
]
[Cites]
Arch Intern Med. 1987 May;147(5):943-4
[
3555378.001
]
[Cites]
J Clin Epidemiol. 1992 Jun;45(6):683-92
[
1607909.001
]
[Cites]
N Engl J Med. 1992 Jul 16;327(3):151-6
[
1608406.001
]
[Cites]
Lancet. 1992 Dec 12;340(8833):1435-6
[
1360561.001
]
[Cites]
BMJ. 1993 Jan 2;306(6869):24-7
[
8435572.001
]
[Cites]
Hypertension. 1994 Sep;24(3):301-8
[
8082936.001
]
[Cites]
Kidney Int. 1995 Jan;47(1):312-8
[
7731163.001
]
[Cites]
Clin Exp Hypertens. 1996 Apr-May;18(3-4):305-21
[
8743023.001
]
[Cites]
Circulation. 1996 Sep 15;94(6):1310-5
[
8822985.001
]
[CommentIn]
Ann Intern Med. 2008 Aug 19;149(4):284; author reply 284
[
18711167.001
]
(PMID = 18378946.001).
[ISSN]
1539-3704
[Journal-full-title]
Annals of internal medicine
[ISO-abbreviation]
Ann. Intern. Med.
[Language]
ENG
[Grant]
United States / NHLBI NIH HHS / HC / N01-HC-95162; United States / NHLBI NIH HHS / HC / N01-HC-95163; United States / NHLBI NIH HHS / HL / N01HC95169; United States / NHLBI NIH HHS / HC / N01-HC-95159; United States / NCRR NIH HHS / RR / KL2 RR025015-01; United States / NHLBI NIH HHS / HC / N01-HC-95165; United States / NCATS NIH HHS / TR / TL1 TR000422; United States / NHLBI NIH HHS / HC / N01-HC-95169; United States / NHLBI NIH HHS / HC / N01-HC-95164; United States / NHLBI NIH HHS / HL / N01HC95165; United States / NHLBI NIH HHS / HL / N01HC95159; United States / NHLBI NIH HHS / HC / N01-HC-95160; United States / NIDDK NIH HHS / DK / K23 DK63274-01; United States / NCRR NIH HHS / RR / KL2 RR025015; United States / NHLBI NIH HHS / HC / N01-HC-95161; United States / NCATS NIH HHS / TR / UL1 TR000423; United States / NIDDK NIH HHS / DK / K23 DK063274
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
United States
[Chemical-registry-number]
0 / CST3 protein, human; 0 / Cystatin C; 0 / Cystatins; AYI8EX34EU / Creatinine
[Other-IDs]
NLM/ NIHMS267500; NLM/ PMC3044648
11.
Ranganathan N, Ranganathan P, Friedman EA, Joseph A, Delano B, Goldfarb DS, Tam P, Rao AV, Anteyi E, Musso CG:
Pilot study of probiotic dietary supplementation for promoting healthy kidney function in patients with chronic kidney disease.
Adv Ther
; 2010 Sep;27(9):634-47
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Pilot study of probiotic dietary supplementation for promoting healthy
kidney
function in patients with chronic
kidney
disease.
INTRODUCTION: Uremic syndrome consists of nitrogenous waste retention, deficiency in
kidney
-derived hormones, and reduced acid excretion, and, if untreated, may progress to coma and eventual death.
Previous experience suggests that oral administration of a probiotic formulation of selected microbial strains may extend renoprotection via intraintestinal extraction of toxic waste solutes in patients with chronic
kidney
disease (CKD)stages 3 and 4.
[MeSH-major]
Probiotics.
Renal
Insufficiency, Chronic / therapy. Uremia / prevention & control
[MeSH-minor]
Adult. Aged. Argentina. Canada. Creatinine / analysis. Dietary Supplements / standards. Disease Progression. Double-Blind Method. Female. Humans.
Kidney
Function Tests. Male. Middle Aged. Nigeria. Pilot Projects. Protective Agents / pharmacokinetics. Quality of Life. Self Report. Treatment Outcome. United States. Uric Acid / analysis. Young Adult
Genetic Alliance.
consumer health - Kidney Disease
.
MedlinePlus Health Information.
consumer health - Chronic Kidney Disease
.
COS Scholar Universe.
author profiles
.
ClinicalTrials.gov.
clinical trials - ClinicalTrials.gov
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 20721651.001).
[ISSN]
1865-8652
[Journal-full-title]
Advances in therapy
[ISO-abbreviation]
Adv Ther
[Language]
eng
[Publication-type]
Journal Article; Multicenter Study; Randomized Controlled Trial
[Publication-country]
United States
[Chemical-registry-number]
0 / Protective Agents; 268B43MJ25 / Uric Acid; AYI8EX34EU / Creatinine
12.
Veroux M, Corona D, Giuffrida G, Gagliano M, Vizcarra D, Tallarita T, Zerbo D, Giaquinta A, Sorbello M, Macarone M, Veroux P:
Sirolimus-based immunosuppression in kidney transplantation for type 2 diabetic nephropathy.
Urol Int
; 2010;84(3):301-4
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Sirolimus-based immunosuppression in
kidney
transplantation for type 2 diabetic nephropathy.
INTRODUCTION:
Kidney
transplantation is the best replacement therapy of type 2 diabetic patients and recently similar graft and patient survival between diabetic and nondiabetic recipients has been reported.
We present our experience with sirolimus-based immunosuppression in a population of 24 type 2 diabetic patients who underwent a
kidney
transplantation.
PATIENTS AND METHODS: From January 2001 to December 2006, 396
kidney
transplantations were performed.
Twenty-four patients had type 2 diabetes mellitus as a cause of end-stage
renal
disease.
A slightly better
kidney
functionality was observed in group B patients.
CONCLUSIONS: Sirolimus-based immunosuppression is safe and efficacious in type 2 diabetic patients who underwent a
kidney
transplantation, allowing a better glucose metabolism control.
[MeSH-major]
Diabetes Mellitus, Type 2 / complications. Diabetic Nephropathies / etiology. Diabetic Nephropathies / surgery. Immunosuppression. Immunosuppressive Agents / therapeutic use.
Kidney
Transplantation. Sirolimus / therapeutic use
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Diabetes Type 2
.
MedlinePlus Health Information.
consumer health - Diabetic Kidney Problems
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
Hazardous Substances Data Bank.
SIROLIMUS
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Copyright]
Copyright 2010 S. Karger AG, Basel.
(PMID = 20389159.001).
[ISSN]
1423-0399
[Journal-full-title]
Urologia internationalis
[ISO-abbreviation]
Urol. Int.
[Language]
eng
[Publication-type]
Journal Article; Randomized Controlled Trial
[Publication-country]
Switzerland
[Chemical-registry-number]
0 / Immunosuppressive Agents; W36ZG6FT64 / Sirolimus
13.
Liu SK, Yan C, Wu LL, Pan Y:
[Study strategies for neurobiology mechanism of "kidney storing will and responding to fear"].
Zhong Xi Yi Jie He Xue Bao
; 2010 Feb;8(2):106-10
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
[Study strategies for neurobiology mechanism of "
kidney
storing will and responding to fear"].
The theory of traditional Chinese medicine (TCM) deems that
kidney
essence is the material basis of formation, development and function of the brain and it maintains human being's psychomotility and behavior.
Moreover,
kidney
essence is the material basis of emotional activity.
The emotion theory in TCM deems that
kidney
stores will and responds to fear.
Combining the TCM theory with the modern psychological theory on stress, and from the points of views of the emotion regulation, the formation, consolidation, extraction and extinction of fear memory, and the plasticity of amygdala-hippocampus-prefrontal cortex nervous pathway, as well as the results of pharmacological studies of prescriptions and herbs for invigorating
the kidney
, the authors explained in this article the pathological mechanisms
of kidney
deficiency due to attack of fear, and revealed the material basis, the action modes, and the neurobiological mechanisms of the
kidney
in controlling and regulating emotional activity.
[MeSH-major]
Fear / physiology.
Kidney
/ physiology. Medicine, Chinese Traditional. Stress, Psychological
MedlinePlus Health Information.
consumer health - Stress
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 20141730.001).
[ISSN]
1672-1977
[Journal-full-title]
Zhong xi yi jie he xue bao = Journal of Chinese integrative medicine
[ISO-abbreviation]
Zhong Xi Yi Jie He Xue Bao
[Language]
chi
[Publication-type]
English Abstract; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
China
14.
Temmar M, Liabeuf S, Renard C, Czernichow S, Esper NE, Shahapuni I, Presne C, Makdassi R, Andrejak M, Tribouilloy C, Galan P, Safar ME, Choukroun G, Massy Z:
Pulse wave velocity and vascular calcification at different stages of chronic kidney disease.
J Hypertens
; 2010 Jan;28(1):163-9
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Pulse wave velocity and vascular calcification at different stages of chronic
kidney
disease.
BACKGROUND: Increased arterial stiffness and vascular calcification have been recognized as important predictors of cardiovascular mortality in patients with chronic
kidney
disease.
METHOD: In order to examine the precise temporal link between aortic stiffness and cardiovascular risk at the earliest stages of chronic
kidney
disease, we studied a cohort of 150 patients with chronic
kidney
disease (52 stage 2/3 patients, 51 stage 4/5 patients and 47 stage 5D patients).
RESULTS: Regardless of the disease stage, patients with chronic
kidney
disease had higher adjusted pulse wave velocity than controls with preserved
renal
function (14.6 +/- 3.8 vs. 10.7 +/- 1.7 m/s, respectively; P < 0.0001).
Regarding aortic calcification, there was a gradual but significant rise in later chronic
kidney
disease stages.
CONCLUSION: We found that both vascular stiffness and vascular calcification appear early in patients with chronic
kidney
disease, but only vascular calcification worsens as the disease progresses.
The increase of vascular stiffness in adult patients with chronic
kidney
disease seems to be more related to age, systolic blood pressure, diabetes and vascular calcification than to uremic toxicity.
[MeSH-major]
Aorta, Abdominal / pathology. Aorta, Thoracic / pathology. Blood Circulation / physiology. Blood Pressure / physiology. Calcinosis / pathology.
Kidney
Failure, Chronic / pathology
Genetic Alliance.
consumer health - Kidney Disease
.
MedlinePlus Health Information.
consumer health - Kidney Failure
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19927012.001).
[ISSN]
1473-5598
[Journal-full-title]
Journal of hypertension
[ISO-abbreviation]
J. Hypertens.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
15.
Andratschke N, Schnaiter A, Weber WA, Cai L, Schill S, Wiedenmann N, Schwaiger M, Molls M, Nieder C:
Preclinical evaluation of erythropoietin administration in a model of radiation-induced kidney dysfunction.
Int J Radiat Oncol Biol Phys
; 2006 Apr 1;64(5):1513-8
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Preclinical evaluation of erythropoietin administration in a model of radiation-induced
kidney
dysfunction.
PURPOSE: To test whether the clinically available growth factor erythropoietin (EPO) influences radiation-induced normal-tissue damage in a model
of kidney
dysfunction.
METHODS: Animal experiments were conducted to test the role of EPO administration in a C3H mouse model of unilateral
kidney
irradiation with 6, 8, and 10 Gy and to assess the effects of 2 different dose levels of EPO.
The kidney
function was assessed before radiotherapy, as well as 19, 25, 31, and 37 weeks thereafter by means of (99m)Tc-dimercaptosuccinat scans (static scintigraphy).
RESULTS: Concomitant EPO administration significantly increased the degree of radiation-induced
kidney
dysfunction.
CONCLUSION: Administration of growth factors concomitant to radiotherapy might modify the development
of kidney
dysfunction.
Although insulin-like growth factor-1 has previously been shown to protect
the kidney
, such an effect could not be demonstrated for EPO.
[MeSH-major]
Erythropoietin / adverse effects.
Kidney
/ radiation effects. Radiation Injuries, Experimental / prevention & control. Radiation Tolerance / drug effects
Hazardous Substances Data Bank.
EPOETIN ALFA
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[ErratumIn]
Int J Radiat Oncol Biol Phys. 2006 Jul 1;65(3):960. Schnaitera, Andrea [corrected to Schnaiter, Andrea]; Caia, Lu [corrected to Cai, Lu]
(PMID = 16580501.001).
[ISSN]
0360-3016
[Journal-full-title]
International journal of radiation oncology, biology, physics
[ISO-abbreviation]
Int. J. Radiat. Oncol. Biol. Phys.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Radiopharmaceuticals; 0 / Recombinant Proteins; 11096-26-7 / Erythropoietin; 494JNQ8L28 / Technetium Tc 99m Dimercaptosuccinic Acid; 64FS3BFH5W / Epoetin Alfa
16.
Feltes CM, Van Eyk J, Rabb H:
Distant-organ changes after acute kidney injury.
Nephron Physiol
; 2008;109(4):p80-4
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Distant-organ changes after acute
kidney
injury.
Acute
kidney
injury (AKI) contributes significantly to morbidity and mortality in both adults and children.
Leukocyte trafficking, cytokine expression, cell adhesion-molecule expression and membrane ion and water-channel expression in distant organs are deranged after
kidney
injury.
The responses to oxidative stress after AKI are also altered, suggesting complex mechanisms of crosstalk between the injured
kidney
and distant organs.
[MeSH-major]
Acute
Kidney
Injury / complications. Acute
Kidney
Injury / immunology. Cytokines / immunology. Models, Immunological. Multiple Organ Failure / complications. Multiple Organ Failure / immunology. Neutrophil Activation / immunology
Faculty of 1000.
commentaries/discussion - See the articles recommended by F1000Prime's Faculty of more than 8,000 leading experts in Biology and Medicine.
(subscription/membership/fee required).
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Copyright]
Copyright 2008 S. Karger AG, Basel.
(PMID = 18802379.001).
[ISSN]
1660-2137
[Journal-full-title]
Nephron. Physiology
[ISO-abbreviation]
Nephron Physiol
[Language]
eng
[Grant]
United States / NHLBI NIH HHS / HV / N01 HV028180
[Publication-type]
Journal Article; Review
[Publication-country]
Switzerland
[Chemical-registry-number]
0 / Cytokines
[Number-of-references]
26
17.
Alie TM, Vrljicak PJ, Myburgh DB, Gupta IR:
Microinjection and electroporation of embryonic kidney explants: an improved method.
Kidney Int
; 2007 Jul;72(1):121-5
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Microinjection and electroporation of embryonic
kidney
explants: an improved method.
Embryonic
kidney
explants are routinely used to study the molecular regulation
of kidney
development.
In this study, we show that a high voltage with a short pulse time is preferable for mouse
kidney
explants.
We suggest that our described conditions will make microinjection and electroporation a more effective method to study gene function in the developing mouse
kidney
.
[MeSH-major]
Electroporation / methods.
Kidney
/ embryology. Microinjections / methods
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17495853.001).
[ISSN]
0085-2538
[Journal-full-title]
Kidney international
[ISO-abbreviation]
Kidney Int.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
147336-22-9 / Green Fluorescent Proteins; EC 3.2.1.23 / beta-Galactosidase
18.
Ashley RA, Reinberg YE:
Familial multilocular cystic nephroma: a variant of a unique renal neoplasm.
Urology
; 2007 Jul;70(1):179.e9-10
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Familial multilocular cystic nephroma: a variant of a unique
renal neoplasm
.
Multilocular cystic nephroma (MCN) is a
benign renal
lesion believed to be unilateral and nonfamilial.
[MeSH-major]
Kidney Neoplasms
/ genetics
[MeSH-minor]
Child, Preschool. Humans. Infant.
Kidney
Diseases, Cystic / pathology. Male
MedlinePlus Health Information.
consumer health - Kidney Cancer
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17656243.001).
[ISSN]
1527-9995
[Journal-full-title]
Urology
[ISO-abbreviation]
Urology
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
19.
Costa MG, Garcia VD, Leirias MM, Santos SR, Oliveira DM:
Urgency priority in kidney transplantation in Rio Grande do Sul.
Transplant Proc
; 2007 Mar;39(2):381-2
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Urgency priority in
kidney
transplantation in Rio Grande do Sul.
In 2002, it was established a system of urgency priority for
kidney
transplantations in cases with no vascular or peritoneal access for dialysis.
We reviewed cases of urgency priority request for
kidney
transplantation addressed to the CNCDO from May 2002 to August 2005.
Within this period the CNCDO received 35 urgency priority requests for
kidney
transplantation (mean, 1 every 1.2 months).
Among the 31 accepted, 26 (83%) had the transplantation performed in an average time of 19.6 days (range, 1-90), representing only 3.2% of all cadaveric
kidney
transplantations during that period.
[MeSH-major]
Kidney
Transplantation / statistics & numerical data. Tissue and Organ Procurement / statistics & numerical data. Waiting Lists
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17362736.001).
[ISSN]
0041-1345
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
20.
Ahmadnia H, Shamsa A, Yarmohammadi A, Darabi M, Asl Zare M:
Kidney transplantation in older adults: does age affect graft survival?
Urol J
; 2005;2(2):93-6
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Kidney
transplantation in older adults: does age affect graft survival?
INTRODUCTION: There is a paucity of data on long-term patient and graft survival in the older
kidney
recipients.
Our aim was to evaluate the long-term outcomes
of kidney
transplantation in patients aged 50 years and older and compare them with outcomes in younger recipients.
Patients who had received a cadaveric
kidney
allograft were excluded from the study.
CONCLUSION:
Kidney
transplantation should be considered in patients older than 50 years, since the graft survival rate is acceptable in this population, and early mortality and complications in this group are not different than those of younger recipients.
Although older patients have a shorter life expectancy, they benefit from
renal
transplantation in ways similar to younger
kidney
transplant recipients.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17629878.001).
[ISSN]
1735-1308
[Journal-full-title]
Urology journal
[ISO-abbreviation]
Urol J
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Iran
21.
Posadas MA, Yang V, Ho B, Omer M, Batlle D:
Acute renal failure and severe hypertension from a page kidney post-transplant biopsy.
ScientificWorldJournal
; 2010;10:1539-42
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Acute
renal
failure and severe hypertension from a page
kidney
post-transplant biopsy.
Page
kidney
refers to a clinical picture characterized by acute onset of hypertension due to external compression of the
kidneys
from hematoma,
tumor
, lymphocele, or urinoma.
Hypertension is believed to result from renin-angiotensin-aldosterone activation triggered by
renal
hypoperfusion and microvascular ischemia.
Renal
failure, in addition to hypertension, may occur in the setting of a single functional
kidney
or a diseased contralateral
kidney
.
We report a case of a patient who had a transplant
kidney
biopsy complicated by a subcapsular perinephric hematoma.
The patient presented with an acute increase in blood pressure and a rapid rise in serum creatinine following a transplant
kidney
routine biopsy.
Early recognition and rapid intervention are needed in order to correct hypertension and reverse acute
renal
failure in Page
kidney
occurring in
renal
transplant recipients.
[MeSH-major]
Acute
Kidney
Injury / physiopathology. Hematoma / physiopathology. Hypertension / physiopathology.
Kidney
Transplantation / adverse effects
MedlinePlus Health Information.
consumer health - High Blood Pressure
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 20694451.001).
[ISSN]
1537-744X
[Journal-full-title]
TheScientificWorldJournal
[ISO-abbreviation]
ScientificWorldJournal
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
England
22.
Kinsey GR, Sharma R, Huang L, Li L, Vergis AL, Ye H, Ju ST, Okusa MD:
Regulatory T cells suppress innate immunity in kidney ischemia-reperfusion injury.
J Am Soc Nephrol
; 2009 Aug;20(8):1744-53
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Regulatory T cells suppress innate immunity in
kidney
ischemia-reperfusion injury.
Both innate and adaptive mechanisms participate in the pathogenesis
of kidney
ischemia-reperfusion injury (IRI), but the role of regulatory immune mechanisms is unknown.
We hypothesized that the anti-inflammatory effects of CD4(+)CD25(+)FoxP3(+) regulatory T cells (Tregs) protect against
renal
IRI.
Partial depletion of Tregs with an anti-CD25 mAb potentiated
kidney
damage induced by IRI.
Reducing the number of Tregs resulted in more neutrophils, macrophages, and innate cytokine transcription in
the kidney
after IRI but did not affect CD4(+) T cells or B cells.
FoxP3(+) Treg-deficient mice accumulated a greater number of inflammatory leukocytes after
renal
IRI than mice containing Tregs.
To confirm that a lack of Tregs potentiated
renal
injury, we co-transferred isolated Tregs and Scurfy lymph node cells; Treg repletion significantly attenuated IRI-induced
renal
injury and leukocyte accumulation.
Furthermore, although adoptive transfer of wild-type Tregs into RAG-1 knockout mice was sufficient to prevent
kidney
IRI, transfer of IL-10-deficient Tregs was not.
Taken together, these results demonstrate that Tregs modulate injury after
kidney
IRI through IL-10-mediated suppression of the innate immune system.
MedlinePlus Health Information.
consumer health - Kidney Diseases
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
J Immunol. 2005 Aug 1;175(3):1965-73
[
16034141.001
]
[Cites]
Hum Immunol. 2005 Mar;66(3):222-30
[
15784460.001
]
[Cites]
J Am Soc Nephrol. 2005 Nov;16(11):3315-25
[
16192425.001
]
[Cites]
Trends Immunol. 2005 Dec;26(12):632-6
[
16243583.001
]
[Cites]
J Immunol. 2006 Mar 1;176(5):3108-14
[
16493070.001
]
[Cites]
Kidney Int. 2006 May;69(9):1601-8
[
16572108.001
]
[Cites]
Am J Physiol Renal Physiol. 2006 Jun;290(6):F1516-24
[
16403835.001
]
[Cites]
Nature. 2006 May 11;441(7090):235-8
[
16648838.001
]
[Cites]
J Immunol. 2006 Sep 1;177(5):3380-7
[
16920979.001
]
[Cites]
J Am Soc Nephrol. 2006 Oct;17(10):2731-41
[
16988067.001
]
[Cites]
J Immunol. 2006 Nov 15;177(10):6780-6
[
17082591.001
]
[Cites]
J Immunol. 2006 Nov 15;177(10):7155-63
[
17082633.001
]
[Cites]
J Leukoc Biol. 2007 Jan;81(1):144-53
[
16959895.001
]
[Cites]
Cancer Sci. 2007 Mar;98(3):416-23
[
17270031.001
]
[Cites]
J Autoimmun. 2006 Dec;27(4):289-96
[
17207605.001
]
[Cites]
J Immunol. 2007 Apr 1;178(7):4136-46
[
17371969.001
]
[Cites]
J Immunol. 2007 May 1;178(9):5899-911
[
17442974.001
]
[Cites]
J Autoimmun. 2007 Aug;29(1):10-9
[
17521882.001
]
[Cites]
Blood. 2007 Aug 15;110(4):1225-32
[
17449799.001
]
[Cites]
Nat Med. 2007 Oct;13(10):1248-52
[
17891146.001
]
[Cites]
J Immunol. 2008 Apr 1;180(7):4366-70
[
18354156.001
]
[Cites]
Eur J Immunol. 2008 Jun;38(6):1643-53
[
18493984.001
]
[Cites]
J Immunol. 2008 Sep 1;181(5):3524-34
[
18714025.001
]
[Cites]
J Immunol. 2008 Nov 15;181(10):6934-41
[
18981113.001
]
[Cites]
Infect Immun. 2008 Dec;76(12):5834-42
[
18824539.001
]
[Cites]
Kidney Int. 2008 Dec;74(12):1526-37
[
18843253.001
]
[Cites]
J Immunol. 2009 Jan 1;182(1):259-73
[
19109157.001
]
[Cites]
Kidney Int. 2009 Jan;75(2):167-75
[
18971925.001
]
[Cites]
Nat Med. 2009 Feb;15(2):192-9
[
19169263.001
]
[Cites]
J Leukoc Biol. 2008 Dec;84(6):1400-9
[
18765477.001
]
[Cites]
Annu Rev Immunol. 2001;19:683-765
[
11244051.001
]
[Cites]
J Clin Invest. 2001 Nov;108(9):1283-90
[
11696572.001
]
[Cites]
Kidney Int. 2001 Dec;60(6):2118-28
[
11737586.001
]
[Cites]
J Exp Med. 2002 Sep 16;196(6):851-7
[
12235217.001
]
[Cites]
J Immunol. 2002 Nov 1;169(9):4850-60
[
12391195.001
]
[Cites]
J Exp Med. 2003 Jan 6;197(1):111-9
[
12515818.001
]
[Cites]
Nat Immunol. 2003 Apr;4(4):330-6
[
12612578.001
]
[Cites]
Blood. 2003 Jul 1;102(1):328-35
[
12623845.001
]
[Cites]
Eur J Immunol. 2003 Aug;33(8):2090-7
[
12884282.001
]
[Cites]
J Immunol. 2003 Sep 15;171(6):3210-5
[
12960350.001
]
[Cites]
Arthritis Res Ther. 2004;6(5):215-22
[
15380036.001
]
[Cites]
J Clin Invest. 1996 Feb 15;97(4):1056-63
[
8613529.001
]
[Cites]
Am J Physiol. 1996 Mar;270(3 Pt 2):F500-9
[
8780254.001
]
[Cites]
J Immunol. 2005 Mar 1;174(5):2957-63
[
15728508.001
]
[Cites]
Am J Physiol Renal Physiol. 2005 Apr;288(4):F722-31
[
15561971.001
]
[Cites]
Nat Immunol. 2005 Nov;6(11):1142-51
[
16227984.001
]
(PMID = 19497969.001).
[ISSN]
1533-3450
[Journal-full-title]
Journal of the American Society of Nephrology : JASN
[ISO-abbreviation]
J. Am. Soc. Nephrol.
[Language]
ENG
[Grant]
United States / NIDDK NIH HHS / DK / DK62324; United States / NIAMS NIH HHS / AR / AR-051203; United States / NIDDK NIH HHS / DK / DK56223; United States / NIAMS NIH HHS / AR / R01 AR051203; United States / NIDCR NIH HHS / DE / DE-017579; United States / NIDDK NIH HHS / DK / R01 DK062324; United States / NIDDK NIH HHS / DK / R01 DK056223; United States / NIDDK NIH HHS / DK / T32 DK072922; United States / NIDDK NIH HHS / DK / DK58413; United States / NIDDK NIH HHS / DK / R44 DK058413; United States / NHLBI NIH HHS / HL / P01 HL073361; United States / NIDDK NIH HHS / DK / R41 DK058413
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
United States
[Chemical-registry-number]
0 / Interleukin-2 Receptor alpha Subunit; 130068-27-8 / Interleukin-10
[Other-IDs]
NLM/ PMC2723989
23.
Karner CM, Chirumamilla R, Aoki S, Igarashi P, Wallingford JB, Carroll TJ:
Wnt9b signaling regulates planar cell polarity and kidney tubule morphogenesis.
Nat Genet
; 2009 Jul;41(7):793-9
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Wnt9b signaling regulates planar cell polarity and
kidney
tubule morphogenesis.
Although many vertebrate organs, such as
kidneys
, lungs and liver, are composed of epithelial tubules, little is known of the mechanisms that establish the length or diameter of these tubules.
In
the kidney
, defects in the establishment or maintenance of tubule diameter are associated with one of the most common inherited human disorders, polycystic
kidney
disease.
Here we show that attenuation of Wnt9b signaling during
kidney
morphogenesis affects the planar cell polarity of the epithelium and leads to tubules with significantly increased diameter.
Although previous studies showed that polarized cell divisions maintain the diameter of postnatal
kidney
tubules, we find that cell divisions are randomly oriented during embryonic development.
COS Scholar Universe.
author profiles
.
Gene Ontology.
gene/protein/disease-specific - Gene Ontology annotations from this paper
.
Faculty of 1000.
commentaries/discussion - See the articles recommended by F1000Prime's Faculty of more than 8,000 leading experts in Biology and Medicine.
(subscription/membership/fee required).
KOMP Repository.
gene/protein/disease-specific - KOMP Repository
(subscription/membership/fee required).
Mouse Genome Informatics (MGI).
Mouse Genome Informatics (MGI)
.
The Lens.
Cited by Patents in
.
Xenbase.
Xenbase
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Lancet. 2007 Apr 14;369(9569):1287-301
[
17434405.001
]
[Cites]
J Am Soc Nephrol. 2007 May;18(5):1389-98
[
17429050.001
]
[Cites]
Development. 2007 Jul;134(13):2533-9
[
17537789.001
]
[Cites]
Dev Cell. 2007 Aug;13(2):214-25
[
17681133.001
]
[Cites]
Development. 2007 Sep;134(17):3049-54
[
17652351.001
]
[Cites]
Nat Rev Genet. 2007 Oct;8(10):791-802
[
17878895.001
]
[Cites]
Pediatr Nephrol. 2007 Nov;22(11):1825-38
[
17554566.001
]
[Cites]
Cell. 1998 Jul 10;94(1):109-18
[
9674432.001
]
[Cites]
Am J Physiol. 1999 Jul;277(1 Pt 2):F146-56
[
10409308.001
]
[Cites]
J Biol Chem. 2004 Dec 10;279(50):52703-13
[
15456783.001
]
[Cites]
Curr Biol. 2005 Apr 26;15(8):787-93
[
15854914.001
]
[Cites]
Nat Genet. 2005 May;37(5):537-43
[
15852005.001
]
[Cites]
Dev Cell. 2005 Aug;9(2):283-92
[
16054034.001
]
[Cites]
Nat Genet. 2005 Sep;37(9):980-5
[
16116426.001
]
[Cites]
EMBO J. 1999 Nov 1;18(21):5931-42
[
10545105.001
]
[Cites]
Development. 2000 May;127(10):2227-38
[
10769246.001
]
[Cites]
Nature. 2000 May 4;405(6782):76-81
[
10811221.001
]
[Cites]
Nature. 2000 May 4;405(6782):81-5
[
10811222.001
]
[Cites]
Development. 2000 Jul;127(14):3091-100
[
10862746.001
]
[Cites]
Nature. 2000 Sep 28;407(6803):527-30
[
11029006.001
]
[Cites]
Int J Dev Biol. 2001;45(1):225-7
[
11291850.001
]
[Cites]
Oncogene. 2001 Sep 20;20(42):5972-81
[
11593404.001
]
[Cites]
Am J Physiol Renal Physiol. 2002 Mar;282(3):F541-52
[
11832437.001
]
[Cites]
J Clin Invest. 2002 Feb;109(4):533-40
[
11854326.001
]
[Cites]
Dev Biol. 2002 Apr 15;244(2):305-18
[
11944939.001
]
[Cites]
Curr Biol. 2002 Jun 4;12(11):876-84
[
12062050.001
]
[Cites]
Curr Biol. 2002 Jun 4;12(11):R378-80
[
12062067.001
]
[Cites]
Dev Biol. 2002 Jul 1;247(1):165-81
[
12074560.001
]
[Cites]
J Am Soc Nephrol. 2002 Jul;13(7):1837-46
[
12089379.001
]
[Cites]
J Am Soc Nephrol. 2002 Oct;13(10):2508-16
[
12239239.001
]
[Cites]
Development. 2003 Jan;130(2):249-58
[
12466193.001
]
[Cites]
Nat Genet. 2007 Nov;39(11):1350-60
[
17906624.001
]
[Cites]
Development. 2007 Dec;134(23):4273-82
[
17978004.001
]
[Cites]
Nat Cell Biol. 2008 Jan;10(1):70-6
[
18084282.001
]
[Cites]
Dev Biol. 2008 Feb 1;314(1):112-26
[
18177851.001
]
[Cites]
PLoS One. 2008;3(4):e1964
[
18398480.001
]
[Cites]
Dev Biol. 2008 May 1;317(1):225-33
[
18384766.001
]
[Cites]
Hum Mol Genet. 2008 Jun 1;17(11):1578-90
[
18263895.001
]
[Cites]
Nat Genet. 2008 Aug;40(8):1010-5
[
18604206.001
]
[Cites]
J Cell Biol. 2008 Nov 3;183(3):377-84
[
18981227.001
]
[Cites]
Development. 2009 Jan;136(1):161-71
[
19060336.001
]
[Cites]
Nat Genet. 2003 Feb;33(2):129-37
[
12514735.001
]
[Cites]
Development. 2003 May;130(9):1725-34
[
12642479.001
]
[Cites]
Proc Natl Acad Sci U S A. 2003 Apr 29;100(9):5286-91
[
12672950.001
]
[Cites]
Nat Genet. 2003 Aug;34(4):413-20
[
12872123.001
]
[Cites]
J Biol Chem. 2003 Sep 5;278(36):34211-8
[
12821668.001
]
[Cites]
Hum Mol Genet. 2003 Oct 15;12(20):2703-10
[
12925574.001
]
[Cites]
Development. 2004 Aug;131(16):4085-93
[
15269167.001
]
[Cites]
Nature. 2004 Aug 5;430(7000):689-93
[
15254551.001
]
[Cites]
Curr Opin Cell Biol. 2004 Oct;16(5):493-9
[
15363798.001
]
[Cites]
Kidney Int. 2004 Oct;66(4):1345-55
[
15458427.001
]
[Cites]
Nature. 1989 Mar 16;338(6212):263-4
[
2493583.001
]
[Cites]
Development. 1992 Dec;116(4):901-14
[
1295743.001
]
[Cites]
Genes Dev. 1994 Jan;8(1):118-30
[
8288125.001
]
[Cites]
Nature. 1997 May 15;387(6630):292-5
[
9153394.001
]
[Cites]
Nat Genet. 1997 Oct;17(2):179-81
[
9326937.001
]
[Cites]
Development. 1998 Mar;125(6):983-94
[
9463345.001
]
[Cites]
Cell. 2003 Jan 10;112(1):19-28
[
12526790.001
]
[Cites]
Genes Dev. 2003 Jan 15;17(2):295-309
[
12533515.001
]
[Cites]
Kidney Int. 2005 Nov;68(5):2010-8
[
16221201.001
]
[Cites]
Dev Biol. 2005 Dec 1;288(1):179-93
[
16277981.001
]
[Cites]
Nat Genet. 2006 Jan;38(1):21-3
[
16341222.001
]
[Cites]
Curr Biol. 2006 Jan 24;16(2):180-5
[
16431370.001
]
[Cites]
Curr Biol. 2006 Jan 24;16(2):186-94
[
16431371.001
]
[Cites]
Dev Cell. 2006 Mar;10(3):391-6
[
16516841.001
]
[Cites]
Curr Biol. 2006 Jun 20;16(12):1224-31
[
16782014.001
]
[Cites]
Semin Cell Dev Biol. 2006 Apr;17(2):194-203
[
16839790.001
]
[Cites]
Development. 2007 Jan;134(1):147-55
[
17164420.001
]
[Cites]
Differentiation. 2006 Dec;74(9-10):638-47
[
17177859.001
]
[Cites]
Blood. 2007 Feb 15;109(4):1345-52
[
17068148.001
]
[Cites]
Cell Cycle. 2007 Apr 1;6(7):776-9
[
17377490.001
]
(PMID = 19543268.001).
[ISSN]
1546-1718
[Journal-full-title]
Nature genetics
[ISO-abbreviation]
Nat. Genet.
[Language]
ENG
[Grant]
United States / NIDDK NIH HHS / DK / 5 P30 DK07403802; United States / NIDDK NIH HHS / DK / P30 DK074038; United States / NIDDK NIH HHS / DK / 1 R01 DK080004; United States / NIDDK NIH HHS / DK / R37 DK042921; United States / NIDDK NIH HHS / DK / P30 DK079328; United States / NIDDK NIH HHS / DK / DK074038-01; United States / NIDDK NIH HHS / DK / R01 DK080004; United States / NIDDK NIH HHS / DK / P30 DK074038-01
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Wnt Proteins; 0 / Wnt9b protein, mouse
[Other-IDs]
NLM/ NIHMS119253; NLM/ PMC2761080
24.
Arany I:
When less is more: apoptosis during acute kidney injury.
Kidney Int
; 2008 Aug;74(3):261-2
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
When less is more: apoptosis during acute
kidney
injury.
The paper by Ma and Devarajan suggests that the subtle apoptotic events that occur in the distal nephron after acute
kidney
injury might have a greater than expected impact on the adjacent proximal tubules and
kidney
function.
Understanding these events might facilitate development of therapeutic means to ameliorate acute
kidney
injury.
[MeSH-major]
Acute
Kidney
Injury / pathology. Apoptosis / physiology. Reperfusion Injury / pathology
[MeSH-minor]
Animals. Humans.
Kidney
/ physiopathology.
Kidney
Tubules, Proximal / physiopathology. Nephrons / physiopathology
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[CommentOn]
Kidney Int. 2008 Aug;74(3):310-8
[
18480747.001
]
(PMID = 18626494.001).
[ISSN]
1523-1755
[Journal-full-title]
Kidney international
[ISO-abbreviation]
Kidney Int.
[Language]
eng
[Publication-type]
Comment; Journal Article
[Publication-country]
United States
25.
Ciancio G, Burke GW 3rd:
Alemtuzumab (Campath-1H) in kidney transplantation.
Am J Transplant
; 2008 Jan;8(1):15-20
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Alemtuzumab (Campath-1H) in
kidney
transplantation.
Kidney
transplantation has become the treatment of choice for both the quality of life and survival in patients with end-stage
renal
disease (ESRD).
However, the immunosuppressive regimen which allows optimal
kidney
transplant outcome remains elusive.
One of the more promising induction agents, Alemtuzumab, was introduced to
kidney
transplantation by Calne in the late 1990s with low dose cyclosporine A monotherapy, with the hope of establishing 'prope' or near tolerance.
[MeSH-major]
Antibodies, Monoclonal / therapeutic use. Antibodies,
Neoplasm
/ therapeutic use. Graft Rejection / prevention & control. Immunosuppressive Agents / therapeutic use.
Kidney
Transplantation
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18093269.001).
[ISSN]
1600-6143
[Journal-full-title]
American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons
[ISO-abbreviation]
Am. J. Transplant.
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Review
[Publication-country]
Denmark
[Chemical-registry-number]
0 / Antibodies, Monoclonal; 0 / Antibodies, Monoclonal, Humanized; 0 / Antibodies, Neoplasm; 0 / Immunosuppressive Agents; 3A189DH42V / alemtuzumab
[Number-of-references]
41
26.
Gómez-Ferrer Lozano A, Navarro Antón JA, Mola Arizo MJ, Polo i Peris AC:
[Erythrocytosis related with hydronephrosis in a horseshoe kidney].
Actas Urol Esp
; 2005 Apr;29(4):414-5
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
[Erythrocytosis related with hydronephrosis in a horseshoe
kidney
].
We report a case of a man with erythrocytosis secondary to hydronephrosis in a right horseshoe
kidney
.
Following nephrectomy of the right
kidney
erythrocyte count returned to normal values.
Secondary polyglobulia may be due to increased erythropoietine values and has been related in some patients with
renal
diseases such as hydronephrosis, but very rarely in association with hydronephrotic horseshoe
kidney
.
[MeSH-major]
Hydronephrosis / complications.
Kidney
/ abnormalities. Polycythemia / etiology
Genetic Alliance.
consumer health - Horseshoe kidney
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 15981431.001).
[ISSN]
0210-4806
[Journal-full-title]
Actas urologicas espanolas
[ISO-abbreviation]
Actas Urol Esp
[Language]
spa
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Spain
27.
Rao PS, Ojo A:
The alphabet soup of kidney transplantation: SCD, DCD, ECD--fundamentals for the practicing nephrologist.
Clin J Am Soc Nephrol
; 2009 Nov;4(11):1827-31
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
The alphabet soup
of kidney
transplantation: SCD, DCD, ECD--fundamentals for the practicing nephrologist.
There is significant variability in the quality of deceased-donor
kidneys
that are used for transplantation.
The quality of the donor
kidney
has a direct effect on important clinical outcomes such as acute rejection, delayed graft function, and patient and allograft survival.
Expanded-criteria donors (ECDs) refer to older
kidney
donors (> or =60 yr) or donors who are aged 50 to 59 yr and have two of the following three features: Hypertension, terminal serum creatinine >1.5 mg/dl, or death from cerebrovascular accident.
By definition, ECD
kidneys
have a 70% greater likelihood of failure compared with one from a 35-yr-old male donor who died from a motor vehicle accident.
An ECD
kidney
transplant recipient has a projected average added-life-years of 5.1 yr compared with 10 yr for a
kidney
recipient from a standard-criteria donor.
Kidney
transplantation from DCD seems to have similar allograft and patient survival compared with
kidney
from donation after brain death; however DCD transplantation has a 42 to 51% risk for delayed graft function (need for at least one dialysis treatment during the first week after transplantation) compared with 24% in an standard-criteria donor
kidney
transplant.
Familiarity with the comprehensive allocation rules governing different categories of deceased-donor
kidneys
by the nephrologists and dialysis team providers is essential to maximizing patient autonomy and to improve the outcomes
of kidney
transplantation.
[MeSH-major]
Kidney
Transplantation. Nephrology. Tissue Donors. Tissue and Organ Procurement
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Organ Donation
.
COS Scholar Universe.
author profiles
.
ClinicalTrials.gov.
clinical trials - ClinicalTrials.gov
.
The Lens.
Cited by Patents in
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19808229.001).
[ISSN]
1555-905X
[Journal-full-title]
Clinical journal of the American Society of Nephrology : CJASN
[ISO-abbreviation]
Clin J Am Soc Nephrol
[Language]
eng
[Grant]
United States / NIDDK NIH HHS / DK / K24 DK062234
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Number-of-references]
24
28.
Tahvanainen E, Tahvanainen P, Kääriäinen H, Höckerstedt K:
Polycystic liver and kidney diseases.
Ann Med
; 2005;37(8):546-55
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Polycystic liver and
kidney
diseases.
There have been remarkable advances in research on polycystic liver and
kidney
diseases recently, covering cloning of new genes, refining disease classifications, and advances in understanding more about the molecular pathology of these diseases.
Autosomal dominant polycystic
kidney
disease (ADPKD) is the most common hereditary disease affecting
kidneys
.
It affects 1/400 to 1/1000 live births and accounts for 5% of the end stage
renal
disease in the United States and Europe, and is caused by gene defects in the PKD1 or PKD2 genes.
Autosomal recessive polycystic
kidney
disease (ARPKD) belongs to a group of congenital hepatorenal fibrocystic syndromes.
[MeSH-major]
Cysts / genetics. Liver Diseases / genetics. Polycystic
Kidney
Diseases / genetics
[MeSH-minor]
Diagnosis
, Differential. Genetic Markers. Humans. Membrane Proteins / genetics. Proteins / genetics. TRPP Cation Channels
Genetics Home Reference.
consumer health - polycystic kidney disease
.
MedlinePlus Health Information.
consumer health - Liver Diseases
.
COS Scholar Universe.
author profiles
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16338757.001).
[ISSN]
0785-3890
[Journal-full-title]
Annals of medicine
[ISO-abbreviation]
Ann. Med.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
Sweden
[Chemical-registry-number]
0 / Genetic Markers; 0 / Membrane Proteins; 0 / Proteins; 0 / SEC63 protein, human; 0 / TRPP Cation Channels; 0 / polycystic kidney disease 1 protein; 0 / polycystic kidney disease 2 protein
[Number-of-references]
95
29.
Cohen EP, Pais P, Moulder JE:
Chronic kidney disease after hematopoietic stem cell transplantation.
Semin Nephrol
; 2010 Nov;30(6):627-34
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Chronic
kidney
disease after hematopoietic stem cell transplantation.
Acute and chronic
kidney
diseases occur after hematopoietic stem cell transplantation.
Recent estimates show that near 15% of subjects undergoing hematopoietic stem cell transplantation will develop chronic
kidney
disease, which is a complication rate that can affect outcome and reduce survival.
Investigation of the causes of chronic
kidney
disease is needed, as are ways to prevent, mitigate, and treat it.
Genetic Alliance.
consumer health - Kidney Disease
.
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Chronic Kidney Disease
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Copyright]
Published by Elsevier Inc.
[Cites]
J Pathol. 2000 Mar;190(4):484-8
[
10699999.001
]
[Cites]
Curr Drug Targets. 2010 Nov;11(11):1423-9
[
20583975.001
]
[Cites]
Radiat Res. 2002 Apr;157(4):393-401
[
11893241.001
]
[Cites]
J Lab Clin Med. 2002 Apr;139(4):251-7
[
12024113.001
]
[Cites]
Am J Transplant. 2003 Mar;3(3):301-5
[
12614285.001
]
[Cites]
Blood. 2003 Oct 1;102(7):2695; author reply 2695-6
[
14504068.001
]
[Cites]
Pediatr Transplant. 2004 Oct;8(5):507-12
[
15367289.001
]
[Cites]
N Engl J Med. 1975 Apr 17;292(16):832-43
[
234595.001
]
[Cites]
Int J Radiat Oncol Biol Phys. 1989 Jun;16(6):1501-9
[
2656600.001
]
[Cites]
Nephron. 1989;51(4):555-6
[
2662038.001
]
[Cites]
Kidney Int. 1989 Jun;35(6):1336-44
[
2671466.001
]
[Cites]
N Engl J Med. 1990 Feb 22;322(8):485-94
[
2300120.001
]
[Cites]
Ann Intern Med. 1991 Jan 15;114(2):113-8
[
1984385.001
]
[Cites]
Ann Intern Med. 1991 Dec 15;115(12):925-30
[
1952488.001
]
[Cites]
Nephron. 1993;64(4):626-35
[
8366991.001
]
[Cites]
N Engl J Med. 1994 Mar 24;330(12):827-38
[
8114836.001
]
[Cites]
Lab Invest. 1996 Sep;75(3):349-60
[
8804358.001
]
[Cites]
Lancet. 1998 Jan 17;351(9097):178-81
[
9449873.001
]
[Cites]
Bone Marrow Transplant. 1997 Dec;20(12):1069-74
[
9466280.001
]
[Cites]
Nephron. 1998 Aug;79(4):408-12
[
9689155.001
]
[Cites]
Int J Radiat Biol. 1999 Apr;75(4):473-9
[
10331853.001
]
[Cites]
Clin Transplant. 1999 Aug;13(4):330-5
[
10485375.001
]
[Cites]
Kidney Int Suppl. 2005 Aug;(97):S68-77
[
16014104.001
]
[Cites]
Br J Haematol. 2005 Oct;131(1):74-9
[
16173966.001
]
[Cites]
Am J Transplant. 2006 Jan;6(1):89-94
[
16433761.001
]
[Cites]
Bone Marrow Transplant. 2006 Sep;38(5):351-7
[
16862167.001
]
[Cites]
Semin Radiat Oncol. 2007 Apr;17(2):141-8
[
17395044.001
]
[Cites]
Bone Marrow Transplant. 2007 May;39(9):571-2
[
17351643.001
]
[Cites]
Nephrol Dial Transplant. 2007 May;22(5):1369-76
[
17255123.001
]
[Cites]
Bone Marrow Transplant. 2007 Jun;39(11):717-23
[
17401393.001
]
[Cites]
Medicine (Baltimore). 2007 Jul;86(4):215-24
[
17632263.001
]
[Cites]
Clin J Am Soc Nephrol. 2007 Sep;2(5):1014-23
[
17702721.001
]
[Cites]
Biol Blood Marrow Transplant. 2008 Apr;14(4):403-8
[
18342782.001
]
[Cites]
Int J Radiat Oncol Biol Phys. 2008 Apr 1;70(5):1546-51
[
18029109.001
]
[Cites]
Nephrology (Carlton). 2008 Jun;13(4):322-30
[
18221254.001
]
[Cites]
Biol Blood Marrow Transplant. 2008 Jun;14(6):658-63
[
18489991.001
]
[Cites]
Nephrol Dial Transplant. 2008 Aug;23(8):2700-1; author reply 2701
[
18385388.001
]
[Cites]
Cancer. 2008 Oct 1;113(7):1580-7
[
18704986.001
]
[Cites]
Am J Transplant. 2008 Nov;8(11):2378-90
[
18925905.001
]
[Cites]
Radiat Res. 2009 Feb;171(2):164-72
[
19267541.001
]
[Cites]
Radiat Res. 2009 Aug;172(2):260-4
[
19630531.001
]
[Cites]
Biol Blood Marrow Transplant. 2009 Oct;15(10):1251-7
[
19747632.001
]
[Cites]
Transplant Proc. 2009 Sep;41(7):2895-7
[
19765466.001
]
[Cites]
Nephrol Dial Transplant. 2010 Jan;25(1):278-82
[
19762604.001
]
[Cites]
Kidney Int. 2010 Feb;77(3):219-24
[
19940841.001
]
[Cites]
Bone Marrow Transplant. 2010 Feb;45(2):219-34
[
19584824.001
]
[Cites]
Exp Hematol. 2010 Apr;38(4):270-81
[
20116413.001
]
[Cites]
Clin J Am Soc Nephrol. 2010 Jun;5(6):1107-13
[
20299374.001
]
[Cites]
Clin Transplant. 2010 Jul-Aug;24(4):E94-102
[
19919608.001
]
[Cites]
Radiat Res. 2001 Mar;155(3):474-80
[
11182799.001
]
(PMID = 21146127.001).
[ISSN]
1558-4488
[Journal-full-title]
Seminars in nephrology
[ISO-abbreviation]
Semin. Nephrol.
[Language]
ENG
[Grant]
United States / NIAID NIH HHS / AI / U19 AI067734; United States / NIAID NIH HHS / AI / 1U19AI067734
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Review
[Publication-country]
United States
[Other-IDs]
NLM/ NIHMS249843; NLM/ PMC3005300
30.
Kim SS, Park HJ, Han J, Gwak SJ, Park MH, Song KW, Rhee YH, Min Chung H, Kim BS:
Improvement of kidney failure with fetal kidney precursor cell transplantation.
Transplantation
; 2007 May 15;83(9):1249-58
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Improvement
of kidney
failure with fetal
kidney
precursor cell transplantation.
BACKGROUND: Current therapies for end-stage
renal
disease have severe limitations.
Dialysis is only a temporary treatment and does not restore
kidney
function.
Here, we show that the transplantation of fetal
kidney
precursor cells reconstitutes
kidney
tissues, reduces uremic symptoms, and provides life-saving metabolic support in
kidney
failure animal models.
METHODS:
Kidney
failure was surgically induced by resecting
kidneys
, leaving approximately 1/6 of the total
kidney
mass (5/6 nephrectomy).
Fetal
kidney
precursor cells were isolated from metanephroi of E17.5 rat fetuses using collagenase/dispase digestion.
Five weeks after the nephrectomy procedure, isolated fetal
kidney
precursor cells were transplanted under
the kidney
capsule of rats using fibrin gel matrix.
The cell transplantation into
the kidneys of kidney
failure-induced rats resulted in
kidney
tissue reconstitution and the transplanted cells were observed in the reconstitution region of the
kidneys
as evidenced by the presence of fluorescently labeled cells.
In addition, biochemical parameters from serum and urine samples showed improved
kidney
functions compared with non-treated group without severe immune response after ten weeks.
CONCLUSION: Transplanting fetal
kidney
precursor cells showed the potential for the partial augmentation
of kidney
structure and function in the treatment
of kidney
failure.
[MeSH-major]
Embryonic Stem Cells / transplantation.
Renal
Insufficiency / physiopathology.
Renal
Insufficiency / surgery
[MeSH-minor]
Animals. Cell Membrane. Disease Models, Animal. Disease Progression. Disease Susceptibility. Glomerulosclerosis, Focal Segmental / pathology. Glomerulosclerosis, Focal Segmental / physiopathology. Graft Rejection / etiology. Hypertrophy.
Kidney
/ pathology.
Kidney
/ physiopathology.
Kidney
Glomerulus / pathology.
Kidney
Glomerulus / physiopathology. Nephrectomy. Phenotype. Rats. Rats, Sprague-Dawley. Regeneration. Survival Analysis. Uremia / physiopathology
The Lens.
Cited by Patents in
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17496543.001).
[ISSN]
0041-1337
[Journal-full-title]
Transplantation
[ISO-abbreviation]
Transplantation
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
31.
Schaeffner ES, Födinger M, Kramar R, Frei U, Hörl WH, Sunder-Plassmann G, Winkelmayer WC:
Prognostic associations between lipid markers and outcomes in kidney transplant recipients.
Am J Kidney Dis
; 2006 Mar;47(3):509-17
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Prognostic associations between lipid markers and outcomes in
kidney
transplant recipients.
BACKGROUND: Hyperlipidemia is highly prevalent in
kidney
transplant recipients, but the prognostic significance for mortality and allograft survival in these patients has not been established sufficiently.
METHODS: We prospectively enrolled 733
kidney
transplant recipients between 1996 and 1998.
Using multivariate proportional hazard regression, independent relations of fasting plasma triglyceride (TG), total cholesterol (TC), high-density lipoprotein cholesterol, and low-density lipoprotein cholesterol levels to risk for death from any cause and risk for
kidney
allograft loss were examined.
RESULTS: During a median follow-up of 6.1 years, 154 patients died and 260
kidney
allografts were lost.
Further analysis of associations between high-density lipoprotein cholesterol or low-density lipoprotein cholesterol categories and patient mortality or
kidney
allograft loss did not show associations.
CONCLUSION: Elevated levels of TC or its subfractions and elevated TG levels are not associated with increased risk for patient mortality or allograft loss in these
kidney
transplant recipients.
[MeSH-major]
Cholesterol / blood. Graft Survival.
Kidney
Transplantation
MedlinePlus Health Information.
consumer health - Cholesterol
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
COS Scholar Universe.
author profiles
.
Hazardous Substances Data Bank.
CHOLESTEROL
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16490631.001).
[ISSN]
1523-6838
[Journal-full-title]
American journal of kidney diseases : the official journal of the National Kidney Foundation
[ISO-abbreviation]
Am. J. Kidney Dis.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Biomarkers; 0 / Triglycerides; 97C5T2UQ7J / Cholesterol
32.
Paleologo G, Abdelkawy H, Barsotti M, Basha A, Bernabini G, Bianchi A, Caprio F, Emad A, Grassi G, Nerucci B, Tregnaghi C, Rizzo G, Donadio C:
Kidney dimensions at sonography are correlated with glomerular filtration rate in renal transplant recipients and in kidney donors.
Transplant Proc
; 2007 Jul-Aug;39(6):1779-81
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Kidney
dimensions at sonography are correlated with glomerular filtration rate in
renal
transplant recipients and in
kidney
donors.
The gold standard to assess
renal
function is the measurement of glomerular filtration rate (GFR).
For practical reasons,
renal
function is often evaluated from serum creatinine (S Cr) or cystatin C (S Cys), and GFR is predicted from SCr.
Ultrasound scanning of the
kidneys
is used only to evaluate
renal
morphology.
The aim of this study was to evaluate the relationship between sonographic
renal
dimensions and GFR in
renal
transplant recipients and in
kidney
donors.
GFR was also predicted using the Cockcroft and Gault (CG) formula and with the simplified Modification of Diet in
Renal
Disease (MDRD) formula.
Length, width, and depth
of kidneys
and
renal
sinus were measured using
renal
sonography.
Among sonographic measurements,
kidney
length showed the best correlation with GFR.
In either case, the correlation
of kidney
length with GFR was greater than that of S Cr or S Cys, and similar to that of CG or MDRD GFR.
Accuracy
of kidney
length as an indicator of GFR impairment was not statistically different from laboratory tests.
In conclusion,
renal
dimensions at sonography closely correlated with GFR.
Thus,
renal
sonography can give information also on the function of the
renal
graft and of the remaining
kidney of
living donors.
[MeSH-major]
Glomerular Filtration Rate.
Kidney
/ anatomy & histology.
Kidney
/ physiology.
Kidney
Transplantation / physiology. Tissue Donors
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Organ Donation
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17692610.001).
[ISSN]
0041-1345
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Biomarkers; 0 / CST3 protein, human; 0 / Cystatin C; 0 / Cystatins; AYI8EX34EU / Creatinine
33.
Zhuang WL, Wu YM, Ge SY, Mei CL:
[Detection of differentially expressed genes in human autosomal dominant polycystic kidney tissue].
Zhonghua Yi Xue Yi Chuan Xue Za Zhi
; 2005 Dec;22(6):705-8
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
[Detection of differentially expressed genes in human autosomal dominant polycystic
kidney
tissue].
OBJECTIVE: To detect the differentially expressed genes in human polycystic
kidney
by cDNA microarray.
Both mRNAs isolated from polycystic
kidney
tissue and normal
kidney
tissue were reversely transcribed to cDNAs with the incorporation of fluorescent dUTP (Cy5-dUTP and Cy3-dUTP) for preparing the hybridization probes.
RESULTS: The result indicated that the expressions of 263 genes were up regulated while the expressions of 94 genes were down regulated in the polycystic
kidney
tissue among the 8398 target genes.
Three hundred and fifty-seven differentially expressed genes with different functions were revealed in the polycystic
kidney
tissue, which may play some roles in the progression of polycystic
kidney
.
[MeSH-major]
Gene Expression Profiling. Oligonucleotide Array Sequence Analysis / methods. Polycystic
Kidney
, Autosomal Dominant / genetics
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16331579.001).
[ISSN]
1003-9406
[Journal-full-title]
Zhonghua yi xue yi chuan xue za zhi = Zhonghua yixue yichuanxue zazhi = Chinese journal of medical genetics
[ISO-abbreviation]
Zhonghua Yi Xue Yi Chuan Xue Za Zhi
[Language]
chi
[Publication-type]
English Abstract; Journal Article
[Publication-country]
China
[Chemical-registry-number]
0 / Carbocyanines; 0 / DNA, Complementary; 0 / Fluorescent Dyes; 0 / cyanine dye 3; 0 / cyanine dye 5; 67763-96-6 / Insulin-Like Growth Factor I
34.
Otsuka M, Ambiru S, Uryuhara K, Herman P, Talpe S, Dehoux JP, Jamar F, Gianello P:
Early biological and immune response to semi-identical liver or kidney allograft in miniature swine.
Transpl Int
; 2005 Jan;18(1):78-88
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Early biological and immune response to semi-identical liver or
kidney
allograft in miniature swine.
In inbred miniature swine, semi-identical liver allograft recipients survive up to 3 months without immunosuppression, whereas similarly mismatched
kidney
allografts are uniformly rejected within 2 weeks.
SLA(d/d) pigs (MGH, Harvard Medical School, Boston, MA, USA) received liver or
kidney
allograft from heterozygous SLA(c/d) miniature swine.
Kidney
allografts were uniformly rejected within 2 weeks, whereas liver allografts survived for up to 87 days.
After both liver and
kidney
transplantation, the peripheral lymphocyte count decreased during the first week concomitantly to a significant elevation of plasma cortisol level.
Early decrease of peripheral platelet count was observed after liver but not
renal
transplantation.
Up-regulation of transforming growth factor beta1 (TGF-beta1) and interferon-gamma (IFN-gamma) was observed during the first postoperative week in semi-identical liver allografts and IFN-gamma as well as IL-10 in
kidney
allografts.
In liver recipients, labelled autologous lymphocytes accumulated in the liver graft and native spleen, whereas after
renal
allograft, lymphocytes accumulated in the native spleen and liver but never in
the kidney
allograft.
Specific cellular anti-donor unresponsiveness was observed from the first post-transplant day in both liver and
kidney
recipients, while the humoral anti-donor response remained intact.
In semi-identical liver allograft, recipient rejection is milder and slower than in similarly matched
kidney
allograft.
The rapid, nonspecific accumulation of recipient lymphocytes in the liver allograft but not in
kidney
allograft might also play a role in the different survival time in this model.
[MeSH-major]
Kidney
Transplantation / physiology. Liver Transplantation / physiology
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Liver Transplantation
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 15612988.001).
[ISSN]
0934-0874
[Journal-full-title]
Transplant international : official journal of the European Society for Organ Transplantation
[ISO-abbreviation]
Transpl. Int.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Germany
[Chemical-registry-number]
0 / Cytokines; 0 / DNA Primers; 0 / RNA, Messenger
35.
Lin F:
Stem cells in kidney regeneration following acute renal injury.
Pediatr Res
; 2006 Apr;59(4 Pt 2):74R-8R
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Stem cells in
kidney
regeneration following acute
renal
injury.
Acute
renal
failure has 50-80% mortality.
Stem cells offer an exciting potential for
kidney
regeneration.
This review discusses pathogenesis of acute
renal
failure resulting from ischemia-reperfusion injury and the role of stem cells in reversing or mitigating this
disorder
.
Specifically, the issues of differentiation
of kidney
cells from embryonic stem cells and bone marrow stem cells, and whether adult
kidney
stem/progenitor cells exist in the postnatal
kidney
are discussed.
Evidence to support the conclusion that intra-
renal
cells, including surviving tubular epithelial cells and potential
renal
stem/progenitor cells, are the main source for
renal
regeneration is provided.
Future research in selecting the type(s) of stem cells and optimizing the dose, frequency and route of administration of the cells will be fundamental in successful cell replacement therapy in acute
renal
failure.
Methods for enhancing endogenous
renal
cell proliferation and differentiation for
renal
repair continue to be important research directions.
[MeSH-major]
Kidney
/ physiopathology. Regeneration. Stem Cells / cytology
[MeSH-minor]
Bone Marrow Cells / cytology. Cell Differentiation. Humans.
Renal
Insufficiency / physiopathology.
Renal
Insufficiency / therapy
MedlinePlus Health Information.
consumer health - Stem Cells
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16549552.001).
[ISSN]
0031-3998
[Journal-full-title]
Pediatric research
[ISO-abbreviation]
Pediatr. Res.
[Language]
eng
[Grant]
United States / NIDDK NIH HHS / DK / K08 DK062839; United States / NIDDK NIH HHS / DK / R01 DK66535
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review
[Publication-country]
United States
[Number-of-references]
48
36.
Masoumi A, Reed-Gitomer B, Kelleher C, Schrier RW:
Potential pharmacological interventions in polycystic kidney disease.
Drugs
; 2007;67(17):2495-510
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Potential pharmacological interventions in polycystic
kidney
disease.
Polycystic
kidney
diseases (autosomal dominant and autosomal recessive) are progressive
renal
tubular cystic diseases, which are characterised by cyst expansion and loss of normal
kidney
structure and function.
Autosomal dominant polycystic
kidney
disease (ADPKD) is the most common life- threatening, hereditary disease.
Early
diagnosis
and treatment of hypertension with inhibitors of the renin-angiotensin-aldosterone system (RAAS) and its potential protective effect on left ventricular hypertrophy has been one of the major therapeutic goals to decrease cardiac complications and contribute to improved prognosis of the disease.
Because of activation of the mammalian target of rapamycin (mTOR) pathway, the use of sirolimus (rapamycin) an mTOR inhibitor, markedly reduced cyst formation and decreased polycystic
kidney
size in several animal models.
Caspase inhibitors have been shown to decrease cytogenesis and
renal
failure in rats with cystic disease.
Cystic fluid secretion results in cyst enlargement and somatostatin analogues have been shown to decrease
renal
cyst progression in patients with ADPKD.
[MeSH-major]
Polycystic
Kidney
Diseases / drug therapy
Genetic Alliance.
consumer health - Kidney Disease
.
Genetic Alliance.
consumer health - Polycystic Kidney Disease
.
COS Scholar Universe.
author profiles
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
J Am Soc Nephrol. 2006 Aug;17(8):2220-7
[
16807403.001
]
[Cites]
Kidney Int. 2002 Apr;61(4):1220-30
[
11918728.001
]
[Cites]
Adv Cancer Res. 2000;77:25-79
[
10549355.001
]
[Cites]
Proc Natl Acad Sci U S A. 2001 Jun 19;98(13):7037-44
[
11416184.001
]
[Cites]
Proc Natl Acad Sci U S A. 2002 Jul 9;99(14):9260-5
[
12082174.001
]
[Cites]
Proc Natl Acad Sci U S A. 2002 Oct 1;99(20):13254-9
[
12239346.001
]
[Cites]
Mamm Genome. 2003 Apr;14(4):242-9
[
12682776.001
]
[Cites]
J Am Soc Nephrol. 1997 Aug;8(8):1292-7
[
9259356.001
]
[Cites]
Science. 1998 Apr 3;280(5360):32-4
[
9556450.001
]
[Cites]
Mol Cell Biol. 2002 Jul;22(14):4984-96
[
12077328.001
]
[Cites]
Kidney Int. 1991 Jan;39(1):57-62
[
2002633.001
]
[Cites]
Cell Signal. 2003 May;15(5):463-9
[
12639709.001
]
[Cites]
Biochem Biophys Res Commun. 2001 Mar 23;282(1):341-50
[
11264013.001
]
[Cites]
Am J Physiol. 1991 Dec;261(6 Pt 2):F951-6
[
1661085.001
]
[Cites]
Cell Signal. 1999 Sep;11(9):651-63
[
10530873.001
]
[Cites]
J Am Soc Nephrol. 2001 Feb;12(2):379-84
[
11158230.001
]
[Cites]
Kidney Int. 1996 Oct;50(4):1327-36
[
8887295.001
]
[Cites]
Am J Kidney Dis. 1994 Oct;24(4):561-8
[
7942810.001
]
[Cites]
Am J Physiol. 1995 Aug;269(2 Pt 1):C487-95
[
7653531.001
]
[Cites]
J Clin Invest. 1997 Mar 15;99(6):1380-9
[
9077548.001
]
[Cites]
J Biol Chem. 2001 Jun 8;276(23):20451-7
[
11278652.001
]
[Cites]
J Pharmacol Exp Ther. 1995 Feb;272(2):546-51
[
7853167.001
]
[Cites]
Biochem Biophys Res Commun. 2004 Oct 1;322(4):1374-83
[
15336986.001
]
[Cites]
Kidney Int. 2004 Sep;66(3):964-73
[
15327388.001
]
[Cites]
Dev Cell. 2003 May;4(5):753-9
[
12737809.001
]
[Cites]
Kidney Int. 1989 Feb;35(2):675-80
[
2709672.001
]
[Cites]
Adv Nephrol Necker Hosp. 1998;28:439-78
[
9890004.001
]
[Cites]
Proc Natl Acad Sci U S A. 1998 Feb 17;95(4):1432-7
[
9465032.001
]
[Cites]
Kidney Int. 2005 Mar;67(3):909-19
[
15698430.001
]
[Cites]
J Am Soc Nephrol. 2002 Sep;13(9):2384-98
[
12191984.001
]
[Cites]
FASEB J. 2001 Nov;15(13):2536-8
[
11641256.001
]
[Cites]
J Cell Biochem. 2002;86(1):21-8
[
12112012.001
]
[Cites]
Curr Biol. 2002 Jun 4;12(11):R378-80
[
12062067.001
]
[Cites]
Crit Rev Oncol Hematol. 2000 Jan;33(1):7-23
[
10714959.001
]
[Cites]
Methods. 2003 Jul;30(3):191-7
[
12798133.001
]
[Cites]
Kidney Int. 1999 Jan;55(1):29-62
[
9893113.001
]
[Cites]
J Urol. 1998 Jan;159(1):291-6
[
9400497.001
]
[Cites]
J Am Soc Nephrol. 2003 Feb;14 (2):367-76
[
12538737.001
]
[Cites]
Am J Physiol Renal Physiol. 2003 Nov;285(5):F998-F1012
[
12837680.001
]
[Cites]
J Am Soc Nephrol. 2000 Jul;11(7):1179-87
[
10864573.001
]
[Cites]
Clin Cancer Res. 1998 Apr;4(4):821-8
[
9563874.001
]
[Cites]
Kidney Int. 2000 Apr;57(4):1460-71
[
10760082.001
]
[Cites]
Pediatrics. 2003 May;111(5 Pt 1):1072-80
[
12728091.001
]
[Cites]
Biochem Biophys Res Commun. 1993 Dec 30;197(3):1083-93
[
8280123.001
]
[Cites]
J Am Soc Nephrol. 1993 Jan;3(7):1378-86
[
8094982.001
]
[Cites]
J Membr Biol. 2001 Nov 1;184(1):71-9
[
11687880.001
]
[Cites]
J Biol Chem. 2004 Jul 9;279(28):29728-39
[
15123714.001
]
[Cites]
Nat Genet. 2005 May;37(5):537-43
[
15852005.001
]
[Cites]
Circulation. 2001 May 15;103(19):2387-94
[
11352889.001
]
[Cites]
Kidney Int. 2005 Jul;68(1):206-16
[
15954910.001
]
[Cites]
Kidney Int. 1988 Nov;34(5):683-90
[
2974094.001
]
[Cites]
Am J Kidney Dis. 1996 Dec;28(6):788-803
[
8957030.001
]
[Cites]
Am J Kidney Dis. 1997 Nov;30(5):703-9
[
9370187.001
]
[Cites]
Pol Arch Med Wewn. 1996 Oct;96(4):329-43
[
9082344.001
]
[Cites]
Physiol Rev. 1998 Oct;78(4):1165-91
[
9790573.001
]
[Cites]
Cell. 2004 Jun 11;117(6):693-7
[
15186771.001
]
[Cites]
J Clin Invest. 2000 Jan;105(1):9-13
[
10619855.001
]
[Cites]
Am J Physiol Renal Physiol. 2004 Oct;287(4):F775-88
[
15187005.001
]
[Cites]
J Pediatr. 1987 May;110(5):729-34
[
2883274.001
]
[Cites]
Kidney Int. 1992 Jan;41(1):206-10
[
1317477.001
]
[Cites]
J Clin Invest. 1998 Mar 1;101(5):935-9
[
9486961.001
]
[Cites]
Kidney Int. 2005 Apr;67(4):1234-47
[
15780076.001
]
[Cites]
J Cell Sci. 2003 Apr 15;116(Pt 8):1519-25
[
12640036.001
]
[Cites]
J Am Soc Nephrol. 2002 Oct;13(10):2508-16
[
12239239.001
]
[Cites]
Kidney Int. 1992 Aug;42(2):364-73
[
1405319.001
]
[Cites]
Proc Natl Acad Sci U S A. 1998 Jan 20;95(2):554-9
[
9435230.001
]
[Cites]
J Clin Invest. 2002 Oct;110(8):1083-91
[
12393844.001
]
[Cites]
Semin Nephrol. 1991 Nov;11(6):653-60
[
1767138.001
]
[Cites]
Nat Genet. 2003 Feb;33(2):129-37
[
12514735.001
]
[Cites]
J Am Soc Nephrol. 1993 Oct;4(4):1064-72
[
7904485.001
]
[Cites]
Kidney Int. 1988 Jun;33(6):1084-90
[
3043076.001
]
[Cites]
Proc Natl Acad Sci U S A. 2003 Apr 29;100(9):5286-91
[
12672950.001
]
[Cites]
J Biol Chem. 2001 Mar 9;276(10):7320-6
[
11058599.001
]
[Cites]
Am J Physiol Renal Physiol. 2002 Dec;283(6):F1313-25
[
12388409.001
]
[Cites]
Trends Cell Biol. 2004 Feb;14 (2):78-85
[
15102439.001
]
[Cites]
Oncogene. 1999 Nov 11;18(47):6505-12
[
10597253.001
]
[Cites]
J Am Soc Nephrol. 2005 Jan;16(1):46-51
[
15563559.001
]
[Cites]
Am J Physiol. 1998 Sep;275(3 Pt 2):F387-94
[
9729511.001
]
[Cites]
Hum Mol Genet. 2003 Aug 1;12(15):1875-80
[
12874107.001
]
[Cites]
Nat Genet. 1995 Jun;10(2):151-60
[
7663510.001
]
[Cites]
Nat Med. 2004 Apr;10(4):363-4
[
14991049.001
]
[Cites]
Circ Res. 2005 Apr 29;96(8):873-80
[
15790956.001
]
[Cites]
FASEB J. 2004 May;18(7):884-6
[
15001556.001
]
[Cites]
Curr Biol. 2001 May 1;11(9):R356-60
[
11369247.001
]
[Cites]
Mayo Clin Proc. 1991 Oct;66(10):1010-7
[
1921483.001
]
[Cites]
J Clin Invest. 2004 Mar;113(6):814-25
[
15067314.001
]
[Cites]
Oncogene. 1998 Sep 17;17(11 Reviews):1395-413
[
9779987.001
]
[Cites]
J Anat. 2001 Oct;199(Pt 4):393-405
[
11693300.001
]
[Cites]
J Am Soc Nephrol. 2005 Apr;16(4):846-51
[
15728778.001
]
[Cites]
N Engl J Med. 2004 Jan 8;350(2):151-64
[
14711914.001
]
[Cites]
J Am Soc Nephrol. 2002 Nov;13(11):2723-9
[
12397042.001
]
[Cites]
J Biol Chem. 1997 Jul 18;272(29):17907-11
[
9218414.001
]
[Cites]
J Biol Chem. 2000 Oct 13;275(41):31921-9
[
10931830.001
]
[Cites]
Mol Cell Biol. 1999 May;19(5):3423-34
[
10207066.001
]
[Cites]
Kidney Int. 2003 Nov;64(5):1792-9
[
14531813.001
]
[Cites]
Am J Kidney Dis. 1995 Sep;26(3):501-7
[
7645559.001
]
[Cites]
Proc Natl Acad Sci U S A. 1999 May 25;96(11):6241-8
[
10339572.001
]
[Cites]
Trends Biochem Sci. 2004 Jan;29(1):32-8
[
14729330.001
]
[Cites]
Lab Invest. 1974 Oct;31(4):303-13
[
4412080.001
]
[Cites]
Hum Mol Genet. 2003 Oct 15;12(20):2703-10
[
12925574.001
]
[Cites]
Dev Biol. 1990 Mar;138(1):225-30
[
1968405.001
]
[Cites]
Am J Nephrol. 1985;5(3):176-81
[
3893129.001
]
[Cites]
J Am Soc Nephrol. 1994 Dec;5(6):1349-54
[
7894001.001
]
[Cites]
Nephron. 1979;24(4):198-204
[
40149.001
]
[Cites]
Cancer Res. 1995 Jan 15;55(2):354-9
[
7812968.001
]
[Cites]
J Am Soc Nephrol. 1998 May;9(5):903-16
[
9596091.001
]
[Cites]
Genes Dev. 2002 Jun 15;16(12 ):1472-87
[
12080086.001
]
[Cites]
Am J Kidney Dis. 2000 Mar;35(3):427-32
[
10692268.001
]
[Cites]
Am J Physiol. 1999 Jun;276(6 Pt 2):F837-46
[
10362772.001
]
[Cites]
EMBO J. 2004 Apr 7;23(7):1657-68
[
15029248.001
]
[Cites]
N Engl J Med. 1990 Oct 18;323(16):1091-6
[
2215576.001
]
[Cites]
J Clin Endocrinol Metab. 1987 May;64(5):975-9
[
2881944.001
]
[Cites]
N Engl J Med. 1983 Dec 15;309(24):1495-501
[
6139753.001
]
[Cites]
Exp Toxicol Pathol. 2001 Jun;53(2-3):123-8
[
11484829.001
]
[Cites]
Proc Natl Acad Sci U S A. 2000 Oct 24;97(22):11960-5
[
11035810.001
]
[Cites]
Br Med J (Clin Res Ed). 1986 Jun 28;292(6537):1701-2
[
2873863.001
]
[Cites]
Kidney Int. 1995 Feb;47(2):490-9
[
7723235.001
]
[Cites]
Proc Natl Acad Sci U S A. 1994 Apr 26;91(9):3700-4
[
8170972.001
]
[Cites]
Mol Cell. 2001 Apr;7(4):823-32
[
11336705.001
]
[Cites]
Nat Genet. 2005 Jan;37(1):19-24
[
15624019.001
]
[Cites]
J Biol Chem. 2003 Jan 31;278(5):2807-18
[
12429740.001
]
[Cites]
Cell. 2000 Jul 7;102(1):1-4
[
10929706.001
]
[Cites]
Nat Med. 1997 Oct;3(10):1089-95
[
9334719.001
]
[Cites]
J Biol Chem. 2004 Jan 23;279(4):2975-83
[
14593099.001
]
[Cites]
J Clin Invest. 2001 May;107(9):1145-52
[
11342578.001
]
[Cites]
Physiol Rev. 1997 Jan;77(1):75-197
[
9016301.001
]
[Cites]
J Am Soc Nephrol. 1998 Jul;9(7):1169-77
[
9644626.001
]
[Cites]
Kidney Int. 2000 Jan;57(1):33-40
[
10620185.001
]
[Cites]
Physiol Res. 2004;53(6):629-34
[
15588131.001
]
[Cites]
Nature. 1995 Dec 21-28;378(6559):785-9
[
8524413.001
]
[Cites]
J Hypertens. 1997 Sep;15(9):925-33
[
9321739.001
]
[Cites]
Cell Calcium. 2005 Jun;37(6):593-601
[
15862350.001
]
[Cites]
Science. 2002 Aug 23;297(5585):1352-4
[
12193789.001
]
[Cites]
Oncogene. 1996 Sep 19;13(6):1153-60
[
8808689.001
]
[Cites]
Proc Natl Acad Sci U S A. 2001 Aug 14;98(17):9666-70
[
11493700.001
]
[Cites]
Kidney Int. 1992 May;41(5):1222-36
[
1319521.001
]
[Cites]
Clin Nephrol. 1975;3(3):99-105
[
1139805.001
]
[Cites]
Biochim Biophys Acta. 1999 May 31;1423(3):C19-30
[
10382537.001
]
[Cites]
Contrib Nephrol. 1995;115:118-21
[
8585897.001
]
[Cites]
Kidney Int. 1972 Aug;2(2):107-13
[
4671535.001
]
[Cites]
Transplantation. 2002 Oct 27;74(8):1070-6
[
12438948.001
]
[Cites]
Cell. 2000 Oct 13;103(2):253-62
[
11057898.001
]
[Cites]
Proc Natl Acad Sci U S A. 1999 Mar 30;96(7):3934-9
[
10097141.001
]
[Cites]
Proc Natl Acad Sci U S A. 2005 May 10;102(19):6954-9
[
15863619.001
]
[Cites]
Am J Nephrol. 1998;18(5):391-8
[
9730562.001
]
[Cites]
Mol Cell. 2000 Nov;6(5):1267-73
[
11106764.001
]
[Cites]
Biol Cell. 2001 Sep;93(1-2):53-62
[
11730323.001
]
[Cites]
J Am Soc Nephrol. 2000 Aug;11(8):1505-11
[
10906164.001
]
[Cites]
J Urol. 1969 Aug;102(2):156-61
[
5796888.001
]
[Cites]
J Natl Cancer Inst. 1998 Jul 15;90(14):1087-94
[
9672257.001
]
[Cites]
J Am Soc Nephrol. 1995 Jun;5(12):2048-56
[
7579053.001
]
[Cites]
Kidney Int. 2003 Jun;63(6):1983-94
[
12753285.001
]
[Cites]
Pediatr Nephrol. 1988 Jul;2(3):296-302
[
3153029.001
]
[Cites]
Proc Natl Acad Sci U S A. 2001 Jan 30;98(3):1182-7
[
11252306.001
]
[Cites]
J Clin Endocrinol Metab. 1993 Nov;77(5):1323-8
[
7915721.001
]
[Cites]
Cell. 2003 Jul 11;114(1):61-73
[
12859898.001
]
[Cites]
J Am Soc Nephrol. 2002 Jul;13(7):1733-9
[
12089368.001
]
[Cites]
Nat Cell Biol. 2002 Mar;4(3):191-7
[
11854751.001
]
[Cites]
Nat Med. 2003 Oct;9(10):1323-6
[
14502283.001
]
[Cites]
Kidney Blood Press Res. 2006;29(3):182-8
[
16943682.001
]
[Cites]
Am J Pathol. 1993 Apr;142(4):1051-60
[
8097368.001
]
[Cites]
Kidney Int. 2003 Nov;64(5):1573-9
[
14531789.001
]
[Cites]
Mol Cell Biol. 1998 Nov;18(11):6666-78
[
9774681.001
]
[Cites]
J Urol. 1995 Mar;153(3 Pt 1):578-83
[
7861486.001
]
[Cites]
Kidney Int. 2004 Nov;66(5):1766-73
[
15496147.001
]
[Cites]
Am J Nephrol. 2001 Mar-Apr;21(2):98-103
[
11359016.001
]
[Cites]
JAMA. 1991 Feb 20;265(7):888-92
[
1992187.001
]
[Cites]
J Biol Chem. 1995 Jan 20;270(3):1149-55
[
7836373.001
]
[Cites]
Nephrol Dial Transplant. 1999 May;14(5):1113-6
[
10344347.001
]
[Cites]
Am J Pathol. 2004 Nov;165(5):1719-30
[
15509540.001
]
[Cites]
Proc Natl Acad Sci U S A. 2006 Apr 4;103(14):5466-71
[
16567633.001
]
[Cites]
Acta Med Scand. 1986;219(4):399-405
[
3716882.001
]
[Cites]
Arch Dermatol Res. 2004 Jul;296(2):74-82
[
15278365.001
]
[Cites]
Curr Opin Pharmacol. 2003 Aug;3(4):371-7
[
12901945.001
]
[Cites]
Cancer Res. 2002 Oct 15;62(20):5645-50
[
12384518.001
]
[Cites]
N Engl J Med. 1995 Jul 6;333(1):18-25
[
7776989.001
]
[Cites]
Kidney Int. 1996 Jan;49(1):135-46
[
8770959.001
]
(PMID = 18034588.001).
[ISSN]
0012-6667
[Journal-full-title]
Drugs
[ISO-abbreviation]
Drugs
[Language]
eng
[Grant]
United States / NIDDK NIH HHS / DK / U01 DK624
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review
[Publication-country]
New Zealand
[Number-of-references]
190
37.
Longenecker CT, Scherzer R, Bacchetti P, Lewis CE, Grunfeld C, Shlipak MG:
HIV viremia and changes in kidney function.
AIDS
; 2009 Jun 1;23(9):1089-96
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
HIV viremia and changes in
kidney
function.
OBJECTIVE: To evaluate the effect of HIV infection on longitudinal changes in
kidney
function and to identify independent predictors
of kidney
function changes in HIV-infected individuals.
CONCLUSION: Compared with controls, HIV-infected persons were more likely both to have clinical decline and clinical improvement in
kidney
function during 5 years of follow-up.
The extent of viremic control had a strong association with longitudinal changes in
kidney
function.
Genetic Alliance.
consumer health - HIV
.
MedlinePlus Health Information.
consumer health - HIV/AIDS
.
MedlinePlus Health Information.
consumer health - HIV/AIDS in Women
.
MedlinePlus Health Information.
consumer health - Kidney Diseases
.
COS Scholar Universe.
author profiles
.
Faculty of 1000.
commentaries/discussion - See the articles recommended by F1000Prime's Faculty of more than 8,000 leading experts in Biology and Medicine.
(subscription/membership/fee required).
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
J Am Soc Nephrol. 2006 Jan;17(1):254-61
[
16267155.001
]
[Cites]
J Am Soc Nephrol. 2005 May;16(5):1404-12
[
15788478.001
]
[Cites]
Antivir Ther. 2006;11(5):641-5
[
16964834.001
]
[Cites]
N Engl J Med. 2006 Nov 30;355(22):2283-96
[
17135583.001
]
[Cites]
AIDS. 2007 May 11;21(8):1003-9
[
17457094.001
]
[Cites]
AIDS. 2007 May 31;21(9):1119-27
[
17502722.001
]
[Cites]
Clin J Am Soc Nephrol. 2006 Jan;1(1):117-29
[
17699198.001
]
[Cites]
Nephrol Dial Transplant. 2007 Nov;22(11):3186-90
[
17575315.001
]
[Cites]
Arch Intern Med. 2007 Nov 12;167(20):2213-9
[
17998494.001
]
[Cites]
Kidney Int. 2007 Dec;72(11):1380-7
[
17805235.001
]
[Cites]
AIDS. 2007 Nov 30;21(18):2435-43
[
18025880.001
]
[Cites]
J Clin Endocrinol Metab. 2008 Jan;93(1):216-24
[
17940113.001
]
[Cites]
Am J Med Sci. 2008 Feb;335(2):89-94
[
18277114.001
]
[Cites]
Am J Kidney Dis. 2008 Mar;51(3):395-406
[
18295055.001
]
[Cites]
AIDS. 2008 Feb 19;22(4):481-7
[
18301060.001
]
[Cites]
Clin Infect Dis. 2008 Apr 15;46(8):1271-81
[
18444867.001
]
[Cites]
Am J Kidney Dis. 2008 Jun;51(6):914-24
[
18455851.001
]
[Cites]
Arch Intern Med. 2008 Nov 10;168(20):2212-8
[
19001197.001
]
[Cites]
AIDS. 2009 Jan 2;23(1):71-82
[
19050388.001
]
[Cites]
Kidney Int. 2009 Feb;75(4):428-34
[
19052538.001
]
[Cites]
Am J Nephrol. 2009;30(3):171-8
[
19349699.001
]
[Cites]
Biometrics. 2000 Sep;56(3):779-88
[
10985216.001
]
[Cites]
Am J Kidney Dis. 2002 Aug;40(2):221-6
[
12148093.001
]
[Cites]
J Am Soc Nephrol. 2002 Dec;13(12):2997-3004
[
12444220.001
]
[Cites]
AIDS. 2004 Feb 20;18(3):541-6
[
15090808.001
]
[Cites]
Kidney Int. 2004 Sep;66(3):1145-52
[
15327410.001
]
[Cites]
J Clin Epidemiol. 1988;41(11):1105-16
[
3204420.001
]
[Cites]
Am J Nephrol. 1995;15(3):217-21
[
7618646.001
]
[Cites]
Kidney Int. 1995 Aug;48(2):311-20
[
7564098.001
]
[Cites]
Scand J Clin Lab Invest. 1999 Feb;59(1):1-8
[
10206092.001
]
[Cites]
Stat Methods Med Res. 1999 Mar;8(1):3-15
[
10347857.001
]
[Cites]
J Am Soc Nephrol. 2004 Dec;15(12):3184-91
[
15579522.001
]
[Cites]
J Am Soc Nephrol. 2005 Feb;16(2):459-66
[
15615823.001
]
[Cites]
Am J Epidemiol. 2006 May 1;163(9):860-9
[
16524955.001
]
(PMID = 19352136.001).
[ISSN]
1473-5571
[Journal-full-title]
AIDS (London, England)
[ISO-abbreviation]
AIDS
[Language]
ENG
[Grant]
United States / NHLBI NIH HHS / HL / R01-HL-74814; United States / NIDDK NIH HHS / DK / R01 DK057508-01S1; United States / NCRR NIH HHS / RR / M01 RR000083-38S30468; United States / NIDDK NIH HHS / DK / R01 DK057508; United States / NCRR NIH HHS / RR / M01-RR00083; United States / NHLBI NIH HHS / HL / R01 HL074814-07; United States / NIDDK NIH HHS / DK / R01 DK057508-01; United States / NCRR NIH HHS / RR / M01 RR000083-380468; United States / NCRR NIH HHS / RR / M01 RR000083-40S10468; United States / NCRR NIH HHS / RR / M01 RR000083-430512; United States / NCRR NIH HHS / RR / M01 RR000865; United States / NCRR NIH HHS / RR / M01 RR000036; United States / NHLBI NIH HHS / HL / R01 HL074814-04; United States / NCRR NIH HHS / RR / M01 RR000083-38S20468; United States / NCRR NIH HHS / RR / M01 RR000083-420512; United States / NHLBI NIH HHS / HL / R01 HL074814-06; United States / NIDDK NIH HHS / DK / R01 DK057508-03S1; United States / NCRR NIH HHS / RR / M01 RR000054; United States / NCRR NIH HHS / RR / M01-RR00052; United States / NIDDK NIH HHS / DK / R01 DK057508-03S2; United States / NCRR NIH HHS / RR / M01 RR000052; United States / NHLBI NIH HHS / HL / R01 HL074814-05; United States / NCRR NIH HHS / RR / M01 RR000051; United States / NCRR NIH HHS / RR / M01-RR00051; United States / NCRR NIH HHS / RR / M01-RR00036; United States / NHLBI NIH HHS / HL / R01 HL074814; United States / NIDDK NIH HHS / DK / R01 DK057508-02; United States / NCRR NIH HHS / RR / M01 RR000083-400468; United States / NHLBI NIH HHS / HL / R01-HL-53359; United States / NCRR NIH HHS / RR / M01-RR00636; United States / NCRR NIH HHS / RR / M01 RR000083-390468; United States / NCRR NIH HHS / RR / M01-RR00054; United States / NCRR NIH HHS / RR / M01 RR000083; United States / NIDDK NIH HHS / DK / R01 DK057508-03; United States / NIDDK NIH HHS / DK / R01-DK-57508
[Publication-type]
Journal Article; Multicenter Study; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / Cystatin C
[Other-IDs]
NLM/ NIHMS492632; NLM/ PMC3725756
38.
Goldstein SL, Devarajan P:
Progression from acute kidney injury to chronic kidney disease: a pediatric perspective.
Adv Chronic Kidney Dis
; 2008 Jul;15(3):278-83
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Progression from acute
kidney
injury to chronic
kidney
disease: a pediatric perspective.
Although emerging evidence indicates that the incidence of both acute
kidney
injury (AKI) and chronic
kidney
disease (CKD) in children is rising and the etiologies are dramatically changing, relatively little is currently known regarding the potential for transition from AKI to CKD.
These include neutrophil gelatinase-associated lipocalin, liver-type fatty acid-binding protein, and
kidney
injury molecule-1.
Genetic Alliance.
consumer health - Kidney Disease
.
MedlinePlus Health Information.
consumer health - Chronic Kidney Disease
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Nephrol Dial Transplant. 2008 Jan;23(1):414-6
[
17893105.001
]
[Cites]
Transplantation. 2007 Dec 27;84(12):1625-30
[
18165774.001
]
[Cites]
Kidney Int. 2008 Feb;73(4):465-72
[
18094680.001
]
[Cites]
Curr Opin Nephrol Hypertens. 2008 Mar;17(2):127-32
[
18277143.001
]
[Cites]
Crit Care. 2007;11(6):R127
[
18070344.001
]
[Cites]
Clin J Am Soc Nephrol. 2008 May;3(3):665-73
[
18337554.001
]
[Cites]
Pediatr Crit Care Med. 2008 May;9(3):279-84
[
18446113.001
]
[Cites]
Adv Chronic Kidney Dis. 2008 Jul;15(3):222-34
[
18565474.001
]
[Cites]
Pediatr Nephrol. 2000 Nov;15(1-2):11-3
[
11095002.001
]
[Cites]
Curr Opin Pediatr. 2002 Apr;14(2):183-8
[
11981288.001
]
[Cites]
Kidney Int. 2002 Jul;62(1):237-44
[
12081583.001
]
[Cites]
Crit Care Med. 2002 Sep;30(9):2156-7
[
12352064.001
]
[Cites]
Pediatr Nephrol. 2002 Dec;17(12):1032-7
[
12478353.001
]
[Cites]
Kidney Int. 2003 May;63(5):1714-24
[
12675847.001
]
[Cites]
Pediatr Nephrol. 2003 Aug;18(8):796-804
[
12811650.001
]
[Cites]
J Am Soc Nephrol. 2003 Oct;14(10):2534-43
[
14514731.001
]
[Cites]
Ann Thorac Surg. 2003 Nov;76(5):1443-9
[
14602265.001
]
[Cites]
Pediatr Nephrol. 2004 Jan;19(1):91-5
[
14634863.001
]
[Cites]
Am J Nephrol. 2004 May-Jun;24(3):307-15
[
15148457.001
]
[Cites]
Crit Care. 2004 Aug;8(4):R204-12
[
15312219.001
]
[Cites]
Am J Kidney Dis. 1995 Jan;25(1):113-8
[
7810517.001
]
[Cites]
Am J Kidney Dis. 2005 Jan;45(1):96-101
[
15696448.001
]
[Cites]
Lancet. 2005 Apr 2-8;365(9466):1231-8
[
15811456.001
]
[Cites]
J Am Soc Nephrol. 2005 Nov;16(11):3365-70
[
16177006.001
]
[Cites]
Am J Kidney Dis. 2005 Dec;46(6):1038-48
[
16310569.001
]
[Cites]
J Am Soc Nephrol. 2005 Dec;16(12):3736-41
[
16267160.001
]
[Cites]
Kidney Int. 2006 Jan;69(1):184-9
[
16374442.001
]
[Cites]
Ann Thorac Surg. 2006 Feb;81(2):542-6
[
16427848.001
]
[Cites]
Mol Cell Biochem. 2006 Mar;284(1-2):175-82
[
16532260.001
]
[Cites]
Pediatr Nephrol. 2006 Jun;21(6):856-63
[
16528543.001
]
[Cites]
Pediatr Nephrol. 2006 Jul;21(7):989-94
[
16773412.001
]
[Cites]
Crit Care Med. 2006 Jul;34(7):1913-7
[
16715038.001
]
[Cites]
Am J Transplant. 2006 Jul;6(7):1639-45
[
16827865.001
]
[Cites]
Crit Care. 2006;10(3):R73
[
16696865.001
]
[Cites]
Arthritis Rheum. 2006 Aug;54(8):2577-84
[
16868980.001
]
[Cites]
Pediatrics. 2006 Sep;118(3):e786-91
[
16894011.001
]
[Cites]
Pediatr Nephrol. 2007 Jan;22(1):101-8
[
17072653.001
]
[Cites]
Pediatr Crit Care Med. 2007 Jan;8(1):29-35
[
17251879.001
]
[Cites]
Clin Immunol. 2007 May;123(2):227-34
[
17360238.001
]
[Cites]
Kidney Int. 2007 May;71(10):1028-35
[
17396113.001
]
[Cites]
J Pathol. 2007 Jun;212(2):209-17
[
17471468.001
]
[Cites]
Am J Nephrol. 2007;27(4):373-8
[
17570904.001
]
[Cites]
Kidney Int. 2007 Aug;72(3):348-58
[
17495861.001
]
[Cites]
Am J Kidney Dis. 2007 Aug;50(2):169-80
[
17660017.001
]
[Cites]
Clin J Am Soc Nephrol. 2006 Sep;1(5):1006-15
[
17699320.001
]
[Cites]
J Am Soc Nephrol. 2007 Oct;18(10):2704-14
[
17898236.001
]
[Cites]
J Am Soc Nephrol. 2007 Nov;18(11):2894-902
[
17942962.001
]
[Cites]
Pediatr Nephrol. 2007 Dec;22(12):2089-95
[
17874137.001
]
[Cites]
Semin Nephrol. 2007 Nov;27(6):637-51
[
18061846.001
]
[Cites]
Crit Care. 2007;11(4):R84
[
17678545.001
]
(PMID = 18565478.001).
[ISSN]
1548-5609
[Journal-full-title]
Advances in chronic kidney disease
[ISO-abbreviation]
Adv Chronic Kidney Dis
[Language]
ENG
[Grant]
United States / NIDDK NIH HHS / DK / R01 DK069749-04; United States / NIDDK NIH HHS / DK / DK069749-04; United States / NIDDK NIH HHS / DK / R21 DK070163-02; United States / NIDDK NIH HHS / DK / R01 DK069749; United States / NIDDK NIH HHS / DK / R21 DK070163
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Chemical-registry-number]
0 / Biomarkers
[Number-of-references]
54
[Other-IDs]
NLM/ NIHMS57871; NLM/ PMC2481383
39.
Doctor RB, Serkova NJ, Hasebroock KM, Zafar I, Edelstein CL:
Distinct patterns of kidney and liver cyst growth in pkd2(WS25/-) mice.
Nephrol Dial Transplant
; 2010 Nov;25(11):3496-504
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Distinct patterns
of kidney
and liver cyst growth in pkd2(WS25/-) mice.
BACKGROUND: Autosomal dominant polycystic
kidney
disease (ADPKD) is a common genetic disease that results in the development of cystic
kidneys
and liver.
Providing the foundation for future long-term studies, the present work documents distinct patterns of long-term cyst growth in
the kidneys
and liver of male and female pkd2(WS25/-) mice.
METHODS: Gravimetric measurements documented the progression
of kidney
and liver growth in male and female pkd2(WS25/-) mice over 12 months.
A fast imaging with steady-state precision-magnetic resonance imaging (FISP-MRI) technique to measure
kidney
and liver organ and cyst volumes was optimized and validated.
RESULTS: Male and female pkd2(WS25/-) mice had significant increases in
kidney
weights after 4 months of age.
The progression
of kidney
growth was minimal after 4 months of age.
CONCLUSIONS: Pkd2(WS25/-) mice are a genetic mouse model that recapitulates the early phenotypic characteristics of human ADPKD
kidney
cystogenesis.
MedlinePlus Health Information.
consumer health - Liver Diseases
.
COS Scholar Universe.
author profiles
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Hepatology. 1988 Nov-Dec;8(6):1627-34
[
3192176.001
]
[Cites]
Kidney Int. 2004 Apr;65(4):1511-6
[
15086495.001
]
[Cites]
Magn Reson Imaging. 1991;9(3):429-34
[
1881263.001
]
[Cites]
Hepatology. 1997 Nov;26(5):1282-6
[
9362373.001
]
[Cites]
Cell. 1998 Apr 17;93(2):177-88
[
9568711.001
]
[Cites]
N Engl J Med. 2006 May 18;354(20):2122-30
[
16707749.001
]
[Cites]
Nephron Clin Pract. 2006;103(4):c173-80
[
16636585.001
]
[Cites]
J Magn Reson Imaging. 2006 Sep;24(3):646-56
[
16878308.001
]
[Cites]
N Engl J Med. 2007 Apr 12;356(15):1560
[
17429087.001
]
[Cites]
Clin J Am Soc Nephrol. 2006 Jan;1(1):64-9
[
17699192.001
]
[Cites]
Clin Cancer Res. 2007 Nov 1;13(21):6450-8
[
17975157.001
]
[Cites]
Kidney Int. 2008 Mar;73(6):778-81
[
18185504.001
]
[Cites]
Am J Physiol Lung Cell Mol Physiol. 2008 Jul;295(1):L152-61
[
18441091.001
]
[Cites]
AJR Am J Roentgenol. 2008 Aug;191(2):352-8
[
18647901.001
]
[Cites]
N Engl J Med. 2008 Oct 2;359(14):1477-85
[
18832246.001
]
[Cites]
Acad Radiol. 2009 Jan;16(1):88-95
[
19064216.001
]
[Cites]
Kidney Int. 2009 Jan;75(2):235-41
[
18971924.001
]
[Cites]
J Am Soc Nephrol. 2009 Jan;20(1):6-8
[
19073819.001
]
[Cites]
Recent Pat Anticancer Drug Discov. 2009 Jan;4(1):36-53
[
19149687.001
]
[Cites]
Annu Rev Med. 2009;60:321-37
[
18947299.001
]
[Cites]
Nat Genet. 2000 Jan;24(1):75-8
[
10615132.001
]
[Cites]
J Am Soc Nephrol. 2000 Aug;11(8):1505-11
[
10906164.001
]
[Cites]
Kidney Int. 2000 Dec;58(6):2492-501
[
11115083.001
]
[Cites]
Comp Med. 2002 Oct;52(5):433-8
[
12405636.001
]
[Cites]
J Magn Reson Imaging. 2003 Feb;17(2):190-6
[
12541226.001
]
[Cites]
Kidney Int. 2003 Sep;64(3):1035-45
[
12911554.001
]
[Cites]
Hepatology. 1990 Jun;11(6):1033-7
[
2365280.001
]
(PMID = 20388629.001).
[ISSN]
1460-2385
[Journal-full-title]
Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association
[ISO-abbreviation]
Nephrol. Dial. Transplant.
[Language]
ENG
[Grant]
United States / NIDDK NIH HHS / DK / DK-074835; United States / NCRR NIH HHS / RR / UL1 RR025780; United States / NIDDK NIH HHS / DK / R01 DK074835; United States / NCI NIH HHS / CA / P30 CA046934; United States / NIDDK NIH HHS / DK / DK-056851; United States / NIDDK NIH HHS / DK / R01 DK056851
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / TRPP Cation Channels; 0 / polycystic kidney disease 2 protein
[Other-IDs]
NLM/ PMC2980992
40.
Collan Y, Hirsimäki P, Aho H, Wuorela M, Sundström J, Tertti R, Metsärinne K:
Value of electron microscopy in kidney biopsy diagnosis.
Ultrastruct Pathol
; 2005 Nov-Dec;29(6):461-8
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Value of electron microscopy in
kidney
biopsy
diagnosis
.
Kidney
biopsy reports given during 2003 were collected from the authors' pathology database.
Five
tumor
samples were not studied with electron microscopy (EM).
EM was not performed in 10/24 transplant biopsies, or in 11/82 cases of suspected primary
kidney
disease.
(1) EM was essential for
diagnosis
, (2) EM contributed to the interpretation and cleared uncertainties, and (3) EM had no influence on the diagnostic process.
In transplant biopsies EM influenced the final
diagnosis
in 86% of cases (category 2).
In biopsies performed for primary
kidney
disease EM was essential for
diagnosis
in 18.3% clearly contributed in 53.5%, and had no influence on the final
diagnosis
in 28.2% of cases.
Because only about 25% of the EM reports did not have any influence on the diagnostic process, it is recommended that
kidney
biopsy protocols should include EM in all biopsy cases, or at least tissue should be reserved for EM studies of all cases.
[MeSH-major]
Kidney
/ ultrastructure.
Kidney
Diseases /
diagnosis
[MeSH-minor]
Adult. Aged. Biopsy.
Diagnosis
, Differential. Humans.
Kidney
Transplantation / pathology. Male. Microscopy, Electron, Transmission
MedlinePlus Health Information.
consumer health - Kidney Diseases
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16316946.001).
[ISSN]
0191-3123
[Journal-full-title]
Ultrastructural pathology
[ISO-abbreviation]
Ultrastruct Pathol
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
41.
Rodrigo E, Arias M:
A practical approach to immune monitoring in kidney transplantation.
Minerva Urol Nefrol
; 2007 Sep;59(3):337-52
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
A practical approach to immune monitoring in
kidney
transplantation.
In the last years, there has been a growing interest in the development of new assays available to monitor
kidney
transplantation.
These assays will permit strategies for the minimization of immunosuppression, for induction of operational tolerance, for an early and noninvasive
diagnosis of
acute rejection, for prevention of adverse effects of immunosuppressive drugs and for measurement of the net immunosuppressive state.
Today, there is not one test that helps us monitor the outcome
of kidney
recipients safely.
Further studies and further developed assays are needed to obtain an adequate immune monitoring strategy in
kidney
transplantation.
Transvivo DTH assay, tetramer staining, quantification of cell proliferation by CFSE labelling, human leukocyte antigen-G determination, phenotyping of recipient immune cells, detection of tolerogeneic dendritic cell precursors, T-cell receptor landscaping and quantification of gene and protein expression need more studies to know their exact role as monitoring markers in
kidney
transplant patients.
[MeSH-major]
Kidney
Transplantation / immunology. Monitoring, Immunologic
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17912229.001).
[ISSN]
0393-2249
[Journal-full-title]
Minerva urologica e nefrologica = The Italian journal of urology and nephrology
[ISO-abbreviation]
Minerva Urol Nefrol
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
Italy
[Number-of-references]
96
42.
Feyssa E, Jones-Burton C, Ellison G, Philosophe B, Howell C:
Racial/ethnic disparity in kidney transplantation outcomes: influence of donor and recipient characteristics.
J Natl Med Assoc
; 2009 Feb;101(2):111-5
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Racial/ethnic disparity in
kidney
transplantation outcomes: influence of donor and recipient characteristics.
OBJECTIVE: The purpose of this study was to evaluate the basis for the racial/ethnic disparity in
kidney
allograft survival.
METHODS: We conducted a retrospective study of 2130 patients who underwent
kidney
transplantation between January 1995 and December 2003.
Additionally, black recipients were more likely to have a prior
kidney
transplant (16.7% vs 11.0%) and to have a cadaver
kidney
donor (74% vs 56.5%).
Previous
kidney
transplantation, cadaveric donor, donor age, recipient employment status, and recipient tobacco use were associated with allograft survival in a Cox proportional hazard model.
CONCLUSIONS: Graft survival rate in black
kidney
transplant recipients is significantly lower than whites, and this disparity can be partially explained by the low rate of live donors and a higher previous transplantation rate in blacks.
[MeSH-major]
Graft Survival. Healthcare Disparities / statistics & numerical data.
Kidney
Transplantation / ethnology.
Kidney
Transplantation / immunology
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Health Disparities
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19378626.001).
[ISSN]
1943-4693
[Journal-full-title]
Journal of the National Medical Association
[ISO-abbreviation]
J Natl Med Assoc
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
43.
Hirsch S:
Prerenal success in chronic kidney disease.
Am J Med
; 2007 Sep;120(9):754-9
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Prerenal success in chronic
kidney
disease.
Renin-angiotensin system inhibitors and diuretics are commonly prescribed to patients with chronic
kidney
disease to reduce systemic blood pressure.
Several reasons for this reluctance are discussed, including the failure to distinguish between hemodynamic- and parenchymal-mediated changes in
kidney
function.
Finally, recent literature describing harmful effects of increases in serum creatinine in other cohorts is reviewed; these cohorts are sufficiently different from the stable chronic
kidney
disease patient that the results ought not to be extrapolated.
[MeSH-major]
Renal
Insufficiency, Chronic / physiopathology
Genetic Alliance.
consumer health - Kidney Disease
.
MedlinePlus Health Information.
consumer health - Chronic Kidney Disease
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17765040.001).
[ISSN]
1555-7162
[Journal-full-title]
The American journal of medicine
[ISO-abbreviation]
Am. J. Med.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Chemical-registry-number]
AYI8EX34EU / Creatinine
[Number-of-references]
36
44.
Gheith OA, El-Saadany SA, Abuo Donia SA, Salem YM:
Compliance of kidney transplant patients to the recommended lifestyle behaviours: single centre experience.
Int J Nurs Pract
; 2008 Oct;14(5):398-407
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Compliance
of kidney
transplant patients to the recommended lifestyle behaviours: single centre experience.
Successful management
of kidney
transplant patients requires lifelong therapeutic regimen.
The aim of the study was to identify compliance
of kidney
transplant patients to the recommended lifestyle behaviours.
One hundred adult
kidney
transplant patients of 6 months duration and more participated in this study regardless of age or sex.
A structured questionnaire was developed which included socio-demographic characteristics, the recommended lifestyle behaviours of the
kidney
transplant patients and
the kidney
transplant patient's health condition and his results from the laboratory tests.
The nurse must provide
the kidney
transplant patients with the necessary knowledge of the recommended lifestyle behaviours.
[MeSH-major]
Kidney
Transplantation. Life Style. Patient Compliance
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18808541.001).
[ISSN]
1440-172X
[Journal-full-title]
International journal of nursing practice
[ISO-abbreviation]
Int J Nurs Pract
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Australia
[Chemical-registry-number]
0 / Immunosuppressive Agents
45.
Briet M, Schiffrin EL:
Aldosterone: effects on the kidney and cardiovascular system.
Nat Rev Nephrol
; 2010 May;6(5):261-73
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Aldosterone: effects on
the kidney
and cardiovascular system.
Aldosterone, a steroid hormone with mineralocorticoid activity, is mainly recognized for its action on sodium reabsorption in the distal nephron of the
kidney
, which is mediated by the epithelial sodium channel (ENaC).
Beyond this well-known action, however, aldosterone exerts other effects on
the kidney
, blood vessels and the heart, which can have pathophysiological consequences, particularly in the presence of a high salt intake.
Aldosterone is implicated in
renal
inflammatory and fibrotic processes, as well as in podocyte injury and mesangial cell proliferation.
Small interventional studies in patients with chronic
kidney
disease have shown promising results, with a significant reduction of proteinuria associated with aldosterone antagonism, but large interventional trials that test the efficacy and safety of mineralocorticoid-receptor antagonists in chronic
kidney
disease are needed.
[MeSH-major]
Aldosterone / adverse effects. Aldosterone / pharmacology. Angiotensin II Type 1 Receptor Blockers / pharmacology. Cardiovascular Diseases / chemically induced. Cardiovascular Diseases / drug therapy. Cardiovascular System / drug effects.
Kidney
/ drug effects. Mineralocorticoid Receptor Antagonists / pharmacology
ClinicalTrials.gov.
clinical trials - ClinicalTrials.gov
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 20234356.001).
[ISSN]
1759-507X
[Journal-full-title]
Nature reviews. Nephrology
[ISO-abbreviation]
Nat Rev Nephrol
[Language]
eng
[Grant]
Canada / Canadian Institutes of Health Research / / 37917; Canada / Canadian Institutes of Health Research / / 82790
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
England
[Chemical-registry-number]
0 / Angiotensin II Type 1 Receptor Blockers; 0 / Angiotensin-Converting Enzyme Inhibitors; 0 / Mineralocorticoid Receptor Antagonists; 0 / Sodium Channels; 0 / Sodium, Dietary; 4964P6T9RB / Aldosterone
[Number-of-references]
141
46.
Ashrith G, Elayda MA, Wilson JM:
Revascularization options in patients with chronic kidney disease.
Tex Heart Inst J
; 2010;37(1):9-18
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Revascularization options in patients with chronic
kidney
disease.
Cardiovascular disease is the leading cause of death in patients who have chronic
kidney
disease or end-stage
renal
disease and are undergoing hemodialysis.
Chronic
kidney
disease is a recognized risk factor for premature atherosclerosis.
Unfortunately, most major randomized clinical trials that form the basis for evidence-based use of revascularization procedures exclude patients who have
renal
insufficiency.
Retrospective, observational studies suggest that patients with end-stage
renal
disease and severe coronary occlusive disease have a lower risk of death if they undergo coronary revascularization rather than medical therapy alone.
This difference appears to result from poor long-term results of percutaneous coronary intervention in patients who have chronic
kidney
disease or end-stage
renal
disease.Because randomized trials comparing percutaneous coronary intervention and coronary artery bypass grafting have included patients undergoing balloon angioplasty and placement of bare-metal stents, their conclusions are suspect in the era of drug-eluting stents.
In this review, we discuss different revascularization options for patients with chronic
kidney
disease, the outcomes of revascularization procedures, and the risk factors for adverse outcomes.
Genetic Alliance.
consumer health - Kidney Disease
.
MedlinePlus Health Information.
consumer health - Coronary Artery Disease
.
MedlinePlus Health Information.
consumer health - Dialysis
.
MedlinePlus Health Information.
consumer health - Kidney Failure
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Chest. 2005 Aug;128(2):855-62
[
16100178.001
]
[Cites]
J Thorac Cardiovasc Surg. 2005 Sep;130(3):746-52
[
16153923.001
]
[Cites]
Circulation. 2005 Aug 30;112(9 Suppl):I270-5
[
16159830.001
]
[Cites]
J Interv Cardiol. 2005 Oct;18(5):331-7
[
16202107.001
]
[Cites]
Ann Thorac Surg. 2005 Dec;80(6):2148-53
[
16305860.001
]
[Cites]
Am Heart J. 2005 Dec;150(6):1163-70
[
16338253.001
]
[Cites]
Ann Thorac Surg. 2006 Feb;81(2):591-8; discussion 598
[
16427858.001
]
[Cites]
Circulation. 2006 Feb 28;113(8):1063-70
[
16490821.001
]
[Cites]
Kidney Blood Press Res. 2005;28(5-6):270-4
[
16534220.001
]
[Cites]
Eur J Cardiothorac Surg. 2006 Apr;29(4):461-5
[
16483789.001
]
[Cites]
J Am Soc Nephrol. 2006 Apr;17(4):1175-80
[
16481413.001
]
[Cites]
J Thorac Cardiovasc Surg. 2006 Jun;131(6):1261-6
[
16733155.001
]
[Cites]
J Invasive Cardiol. 2006 Jun;18(6):273-7
[
16751681.001
]
[Cites]
Kidney Int. 2006 Aug;70(4):757-64
[
16788687.001
]
[Cites]
J Invasive Cardiol. 2006 Sep;18(9):405-8
[
16954577.001
]
[Cites]
J Invasive Cardiol. 2006 Sep;18(9):409-10
[
16954578.001
]
[Cites]
Ann Thorac Cardiovasc Surg. 2006 Aug;12(4):257-64
[
16977295.001
]
[Cites]
J Invasive Cardiol. 2006 Dec;18(12):577-83
[
17197706.001
]
[Cites]
Circulation. 2007 Jul 17;116(3):298-304
[
17606842.001
]
[Cites]
J Invasive Cardiol. 2007 Aug;19(8):331-7
[
17712200.001
]
[Cites]
Clin Transplant. 2007 Sep-Oct;21(5):609-14
[
17845634.001
]
[Cites]
JAMA. 2007 Nov 7;298(17):2038-47
[
17986697.001
]
[Cites]
Am J Kidney Dis. 2008 Jan;51(1 Suppl 1):S1-320
[
18086389.001
]
[Cites]
J Am Coll Cardiol. 2008 May 27;51(21):2017-24
[
18498954.001
]
[Cites]
Ann Thorac Surg. 1999 Oct;68(4):1257-61
[
10543489.001
]
[Cites]
Kidney Int. 2000 Jan;57(1):307-13
[
10620213.001
]
[Cites]
Cardiol Rev. 2008 Jul-Aug;16(4):213-8
[
18562812.001
]
[Cites]
Circulation. 2008 Oct 28;118(18):1817-27
[
18852368.001
]
[Cites]
Circulation. 2008 Nov 18;118(21):2130-8
[
18981306.001
]
[Cites]
Artif Organs. 2001 Apr;25(4):239-47
[
11318749.001
]
[Cites]
Artif Organs. 2001 Apr;25(4):268-72
[
11318755.001
]
[Cites]
Ital Heart J. 2001 May;2(5):379-83
[
11392643.001
]
[Cites]
Kidney Int. 2001 Jul;60(1):292-9
[
11422764.001
]
[Cites]
Jpn J Thorac Cardiovasc Surg. 2001 Nov;49(11):660-5
[
11757338.001
]
[Cites]
Circulation. 2004 Jun 15;109(23):2866-71
[
15159290.001
]
[Cites]
Eur J Cardiothorac Surg. 1999 Jan;15(1):51-4
[
10077373.001
]
[Cites]
Artif Organs. 2003 Feb;27(2):174-80
[
12580775.001
]
[Cites]
Am J Cardiol. 2000 Jun 1;85(11):1365-8
[
10831956.001
]
[Cites]
Am J Cardiol. 2000 Aug 15;86(4):395-9
[
10946031.001
]
[Cites]
Am J Cardiol. 2000 Sep 1;86(5):485-9
[
11009262.001
]
[Cites]
Ann Thorac Surg. 2000 Sep;70(3):813-8; discussion 819
[
11016315.001
]
[Cites]
Circulation. 2000 Dec 12;102(24):2973-7
[
11113048.001
]
[Cites]
Ann Thorac Surg. 2001 Feb;71(2):543-8
[
11235703.001
]
[Cites]
Heart. 2001 May;85(5):556-60
[
11303010.001
]
[Cites]
Am J Nephrol. 1999;19(1):38-44
[
10085448.001
]
[Cites]
Eur J Cardiothorac Surg. 1999 May;15(5):691-6
[
10386419.001
]
[Cites]
Kidney Int. 1999 Jul;56(1):324-32
[
10411709.001
]
[Cites]
Ann Thorac Surg. 2004 Dec;78(6):2044-9
[
15561032.001
]
[Cites]
Am J Cardiol. 2005 Jan 15;95(2):167-72
[
15642546.001
]
[Cites]
Scand J Urol Nephrol. 2004;38(5):405-16
[
15764253.001
]
[Cites]
Ann Thorac Surg. 2005 May;79(5):1577-83
[
15854936.001
]
[Cites]
Circulation. 2005 May 31;111(21):2858-64
[
15927994.001
]
[Cites]
Eur Heart J. 2005 Aug;26(15):1488-93
[
15860519.001
]
[Cites]
Jpn J Thorac Cardiovasc Surg. 2001 Dec;49(12):693-9
[
11808090.001
]
[Cites]
J Am Coll Cardiol. 2002 Apr 3;39(7):1113-9
[
11923033.001
]
[Cites]
Int Urol Nephrol. 2001;32(4):717-23
[
11989572.001
]
[Cites]
Circulation. 2002 May 14;105(19):2253-8
[
12010906.001
]
[Cites]
Circulation. 2002 Oct 22;106(17):2207-11
[
12390949.001
]
[Cites]
Ann Thorac Surg. 2003 Feb;75(2):496-500
[
12607660.001
]
[Cites]
Kidney Int. 2003 Mar;63(3):1121-9
[
12631096.001
]
[Cites]
Circulation. 2003 Jun 3;107(21):2653-5
[
12756161.001
]
[Cites]
J Am Soc Nephrol. 2003 Sep;14(9):2373-80
[
12937316.001
]
[Cites]
Am J Cardiol. 2003 Sep 15;92(6):721-5
[
12972118.001
]
[Cites]
J Am Soc Nephrol. 2003 Oct;14(10):2556-72
[
14514733.001
]
[Cites]
J Am Soc Nephrol. 2004 Mar;15(3):517-23
[
14978153.001
]
[Cites]
Kidney Int. 2004 Jul;66(1):441-7
[
15200454.001
]
[Cites]
Am J Cardiol. 2004 Jul 1;94(1):30-4
[
15219504.001
]
[Cites]
Am J Kidney Dis. 2004 Oct;44(4):627-35
[
15384013.001
]
[Cites]
J Card Surg. 2004 Sep-Oct;19(5):449-52
[
15383059.001
]
[Cites]
Am Heart J. 2004 Sep;148(3):430-8
[
15389229.001
]
[Cites]
Mayo Clin Proc. 1991 Jan;66(1):45-53
[
1988758.001
]
[Cites]
Am J Kidney Dis. 1995 Feb;25(2):281-90
[
7847356.001
]
[Cites]
Ann Thorac Surg. 1996 Jun;61(6):1793-6
[
8651786.001
]
[Cites]
N Engl J Med. 1996 Jul 25;335(4):217-25
[
8657237.001
]
[Cites]
Heart. 1997 Oct;78(4):337-42
[
9404246.001
]
[Cites]
Cardiovasc Surg. 1998 Oct;6(5):500-5
[
9794271.001
]
(PMID = 20200622.001).
[ISSN]
1526-6702
[Journal-full-title]
Texas Heart Institute journal
[ISO-abbreviation]
Tex Heart Inst J
[Language]
ENG
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Number-of-references]
76
[Other-IDs]
NLM/ PMC2829816
[Keywords]
NOTNLM ; Angioplasty, transluminal, percutanous coronary / coronary artery bypass / coronary artery bypass, off-pump / creatinine/blood/metabolism / drug-eluting stents / extracorporeal circulation / glomerular filtration rate / kidney failure, chronic / renal dialysis / stents / treatment outcome
47.
Parzanese I, Maccarone D, Caniglia L, Pisani F, Mazzotta C, Rizza V, Famulari A:
Risk factors that can influence kidney transplant outcome.
Transplant Proc
; 2006 May;38(4):1022-3
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Risk factors that can influence
kidney
transplant outcome.
The survival and function of a
kidney
transplant are influenced by numerous immunological and nonimmunological factors.
The aim of this study was to evaluate the role of a number of cadaveric donor parameters on transplanted
kidney
function, and in particular on the occurrence of delayed graft function (DGF) since DGF is one of the most important factors in long-term organ survival.
This study looked at 143 patients who underwent
kidney
transplant of whom 32 displayed DGF.
We observed that a donor history of hypertension (46.8% DGF vs 23.27% no DGF; P = .01) and high levels of donor creatinine prior to organ removal (1.9 +/- 1.2 mg/dL DGF vs 1.2 +/- 0.9 mg/dL no DGF; P = .007) were significant risk factors for DGF among
kidney
recipients.
[MeSH-major]
Delayed Graft Function / physiopathology. Graft Survival / physiology.
Kidney
Transplantation / adverse effects.
Kidney
Transplantation / physiology
[MeSH-minor]
Cadaver. Creatinine / blood. Follow-Up Studies. Humans.
Kidney
Function Tests. Retrospective Studies. Risk Factors. Time Factors. Tissue Donors / statistics & numerical data. Treatment Outcome
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16757251.001).
[ISSN]
0041-1345
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
AYI8EX34EU / Creatinine
48.
Campese VM, Hadaya B, Chiu J:
HMG-CoA reductase inhibitors and the kidney.
Curr Hypertens Rep
; 2005 Oct;7(5):337-42
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
HMG-CoA reductase inhibitors and
the kidney
.
It has not been well established whether statins confer similar benefits to
the kidney
.
In this review, we critically consider the available data whereby dyslipidemia mediates
renal
dysfunction by modulating the inflammatory response to diverse cytokines.
We also review the emerging database suggesting that statins may modulate
renal
dysfunction by altering the response of the
kidney
to dyslipidemia, particularly in patients with end-stage
renal
disease (ESRD) and post-
kidney
transplant.
[MeSH-major]
Cardiovascular Diseases / prevention & control. Hydroxymethylglutaryl-CoA Reductase Inhibitors / therapeutic use.
Kidney
Diseases / therapy
[MeSH-minor]
Dyslipidemias / complications. Dyslipidemias / drug therapy. Humans. Inflammation / drug therapy.
Kidney
Failure, Chronic / complications.
Kidney
Failure, Chronic / therapy.
Kidney
Transplantation
MedlinePlus Health Information.
consumer health - Kidney Diseases
.
MedlinePlus Health Information.
consumer health - Statins
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Transplantation. 1996 May 27;61(10):1469-74
[
8633373.001
]
[Cites]
Arterioscler Thromb Vasc Biol. 2003 Jan 1;23 (1):58-63
[
12524225.001
]
[Cites]
Arterioscler Thromb Vasc Biol. 2002 Sep 1;22(9):1452-8
[
12231565.001
]
[Cites]
Arterioscler Thromb Vasc Biol. 2003 May 1;23(5):729-36
[
12615672.001
]
[Cites]
Circulation. 1997 Mar 4;95(5):1126-31
[
9054840.001
]
[Cites]
J Clin Invest. 1998 Jun 15;101(12):2711-9
[
9637705.001
]
[Cites]
Transplantation. 1994 Jan;57(1):68-72
[
8291116.001
]
[Cites]
Atherosclerosis. 2002 Jan;160(1):141-6
[
11755931.001
]
[Cites]
J Hypertens. 2002 Dec;20(12):2465-73
[
12473872.001
]
[Cites]
BMC Cardiovasc Disord. 2003 Jul 19;3:6
[
12871602.001
]
[Cites]
JAMA. 2004 Mar 3;291(9):1071-80
[
14996776.001
]
[Cites]
Ann Intern Med. 2003 Oct 21;139(8):670-82
[
14568856.001
]
[Cites]
Kidney Int Suppl. 1999 Jul;71:S10-3
[
10412727.001
]
[Cites]
N Engl J Med. 1997 Aug 7;337(6):408-16
[
9241131.001
]
[Cites]
Kidney Int. 1993 Apr;43(4):918-27
[
8479130.001
]
[Cites]
Hypertension. 1995 Oct;26(4):670-5
[
7558229.001
]
[Cites]
N Engl J Med. 2001 Nov 8;345(19):1419-21
[
11794177.001
]
[Cites]
Nephron. 2002 Apr;90(4):373-8
[
11961394.001
]
[Cites]
Kidney Int Suppl. 1999 Jul;71:S84-7
[
10412745.001
]
[Cites]
J Nephrol. 2003 Mar-Apr;16(2):238-44
[
12768071.001
]
[Cites]
J Am Soc Nephrol. 1993 Aug;4(2):187-94
[
8400082.001
]
[Cites]
J Clin Pathol. 2004 Jul;57(7):728-34
[
15220366.001
]
[Cites]
N Engl J Med. 2004 Sep 23;351(13):1285-95
[
15385655.001
]
[Cites]
Transplantation. 2001 Jul 27;72(2):223-7
[
11477342.001
]
[Cites]
Kidney Int. 1993 Jan;43(1):218-25
[
8433562.001
]
[Cites]
Circulation. 1998 Sep 1;98(9):839-44
[
9738637.001
]
[Cites]
Pharmacol Res. 2002 Feb;45(2):147-54
[
11846628.001
]
[Cites]
Circulation. 1998 Mar 31;97(12):1129-35
[
9537338.001
]
[Cites]
J Am Soc Nephrol. 2003 Aug;14(8):2084-91
[
12874462.001
]
[Cites]
Am J Kidney Dis. 2003 Apr;41(4 Suppl 3):I-IV, S1-91
[
12671933.001
]
[Cites]
Free Radic Biol Med. 2002 Oct 15;33(8):1026-36
[
12374614.001
]
[Cites]
Am J Kidney Dis. 2002 Jun;39(6):1213-7
[
12046033.001
]
[Cites]
Kidney Int. 2002 Jan;61(1):297-304
[
11786112.001
]
[Cites]
Am J Kidney Dis. 2003 Mar;41(3):565-70
[
12612979.001
]
[Cites]
Kidney Int. 1997 Oct;52(4):1016-27
[
9328940.001
]
[Cites]
J Biol Chem. 1991 Jul 5;266(19):12216-22
[
1712015.001
]
[Cites]
Kidney Int. 2002 Nov;62(5):1524-38
[
12371953.001
]
[Cites]
N Engl J Med. 2004 Sep 23;351(13):1296-305
[
15385656.001
]
[Cites]
Nephrol Dial Transplant. 2002 Mar;17(3):347-8
[
11865072.001
]
[Cites]
Am J Kidney Dis. 1998 Jan;31(1):190-4
[
9428473.001
]
[Cites]
Circulation. 2001 Apr 17;103(15):1933-5
[
11306519.001
]
[Cites]
Circulation. 1999 Jul 20;100(3):230-5
[
10411845.001
]
[Cites]
J Am Soc Nephrol. 2003 Jun;14(6):1605-13
[
12761262.001
]
[Cites]
Clin Sci (Lond). 2003 Sep;105(3):251-66
[
12793855.001
]
[Cites]
N Engl J Med. 1995 Sep 7;333(10 ):621-7
[
7637722.001
]
[Cites]
Arterioscler Thromb Vasc Biol. 2001 Jul;21(7):1165-71
[
11451746.001
]
[Cites]
Atherosclerosis. 1999 Dec;147(2):253-61
[
10559511.001
]
[Cites]
Kidney Int. 2001 Jan;59(1):260-9
[
11135079.001
]
(PMID = 16157074.001).
[ISSN]
1522-6417
[Journal-full-title]
Current hypertension reports
[ISO-abbreviation]
Curr. Hypertens. Rep.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Chemical-registry-number]
0 / Hydroxymethylglutaryl-CoA Reductase Inhibitors
[Number-of-references]
50
49.
Aregger F, Pilop C, Uehlinger DE, Brunisholz R, Carrel TP, Frey FJ, Frey BM:
Urinary proteomics before and after extracorporeal circulation in patients with and without acute kidney injury.
J Thorac Cardiovasc Surg
; 2010 Mar;139(3):692-700
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Urinary proteomics before and after extracorporeal circulation in patients with and without acute
kidney
injury.
OBJECTIVE: Acute
kidney
injury is a well-known complication with high morbidity and mortality after cardiopulmonary bypass.
Cardiopulmonary bypass-associated acute
kidney
injury is still poorly understood.
Acute
kidney
injury was defined according to the RIFLE classification.
To identify differentially regulated proteins in acute
kidney
injury, we next compared the urinary proteome obtained on the first postoperative day between patients with and without acute
kidney
injury.
Acute
kidney
injury developed in 6 of 36 patients.
A modified urinary albumin was increased, and zinc-alpha-2-glycoprotein and a fragment of adrenomedullin-binding protein were decreased in patients with acute
kidney
injury.
Decreased excretion of zinc-alpha-2-glycoprotein in patients with acute
kidney
injury was confirmed by Western blot and enzyme-linked immunosorbent assay in an independent cohort of 22 patients with and 46 patients without acute
kidney
injury.
Zinc-alpha-2-glycoprotein is a potentially useful predictive marker for acute
kidney
injury after cardiopulmonary bypass surgery.
[MeSH-major]
Cardiopulmonary Bypass / adverse effects.
Kidney
Diseases / etiology.
Kidney
Diseases / urine. Proteomics
MedlinePlus Health Information.
consumer health - Kidney Diseases
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Copyright]
Copyright 2010 The American Association for Thoracic Surgery. Published by Mosby, Inc. All rights reserved.
(PMID = 20176211.001).
[ISSN]
1097-685X
[Journal-full-title]
The Journal of thoracic and cardiovascular surgery
[ISO-abbreviation]
J. Thorac. Cardiovasc. Surg.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
50.
Can B, Kulaksiz Erkmen G, Dalmizrak O, Ogus IH, Ozer N:
Purification and characterisation of rat kidney glutathione reductase.
Protein J
; 2010 May;29(4):250-6
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Purification and characterisation of rat
kidney
glutathione reductase.
Kidney
and liver tissues were considered as a rich source of GR.
In this study, rat
kidney
GR was purified and some of its properties were investigated.
NADH was used as a coenzyme by rat
kidney
GR but with a lower efficiency (32.7%) than NADPH.
An optimum pH of 6.5 and optimum temperature of 65 degrees C were found for rat
kidney
GR.
[MeSH-major]
Glutathione Reductase / chemistry. Glutathione Reductase / metabolism.
Kidney
/ enzymology
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Mol Cell Biochem. 2007 Mar;297(1-2):139-49
[
17075686.001
]
[Cites]
Anal Biochem. 1981 Sep 15;116(2):531-6
[
7316181.001
]
[Cites]
J Bacteriol. 1984 Apr;158(1):317-24
[
6425264.001
]
[Cites]
J Nutr. 2004 Mar;134(3):489-92
[
14988435.001
]
[Cites]
Nature. 1970 Aug 15;227(5259):680-5
[
5432063.001
]
[Cites]
Biochem J. 1987 Aug 1;245(3):875-80
[
3311037.001
]
[Cites]
Biochemistry. 1990 Jun 19;29(24):5790-6
[
2200516.001
]
[Cites]
Science. 1983 Apr 29;220(4596):472-7
[
6836290.001
]
[Cites]
J Biol Chem. 1986 Feb 5;261(4):1629-35
[
3003077.001
]
[Cites]
Eur J Biochem. 1996 Jan 15;235(1-2):345-50
[
8631352.001
]
[Cites]
Biochemistry. 1982 Dec 21;21(26):6628-33
[
7159551.001
]
[Cites]
J Exp Bot. 2002 May;53(372):1283-304
[
11997376.001
]
[Cites]
FEBS Lett. 1989 Jun 19;250(1):72-4
[
2737302.001
]
[Cites]
Blood. 1963 May;21:573-85
[
13967896.001
]
[Cites]
Protist. 2010 Jan;161(1):91-101
[
19664954.001
]
[Cites]
Eur J Biochem. 1979 Aug 1;98(2):487-99
[
39757.001
]
[Cites]
J Biol Chem. 1975 Jul 25;250(14):5475-80
[
237922.001
]
[Cites]
J Biol Chem. 1968 Feb 25;243(4):809-14
[
5638597.001
]
[Cites]
J Biol Chem. 1952 Jan;194(1):119-30
[
14927599.001
]
[Cites]
Protein Pept Lett. 2010 May;17(5):667-74
[
19702563.001
]
[Cites]
Methods Enzymol. 1985;113:484-90
[
3003504.001
]
[Cites]
EMBO Rep. 2006 Mar;7(3):271-5
[
16607396.001
]
[Cites]
Biochem J. 1987 Mar 1;242(2):511-5
[
3109393.001
]
[Cites]
Enzyme. 1991;45(3):121-4
[
1815946.001
]
[Cites]
Anal Biochem. 1976 May 7;72:248-54
[
942051.001
]
[Cites]
Pathol Biol (Paris). 1996 Jan;44(1):77-85
[
8734304.001
]
[Cites]
Brain Res. 1995 May 22;680(1-2):16-22
[
7663973.001
]
[Cites]
Protein Expr Purif. 1998 Jun;13(1):41-4
[
9631513.001
]
[Cites]
Biochem Med Metab Biol. 1991 Feb;45(1):65-73
[
2015111.001
]
[Cites]
Protein J. 2004 Jul;23(5):317-24
[
15328887.001
]
[Cites]
Plant Physiol. 1992 Sep;100(1):138-45
[
16652936.001
]
(PMID = 20490902.001).
[ISSN]
1875-8355
[Journal-full-title]
The protein journal
[ISO-abbreviation]
Protein J.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Netherlands
[Chemical-registry-number]
53-59-8 / NADP; EC 1.8.1.7 / Glutathione Reductase; ULW86O013H / Glutathione Disulfide
51.
Leclerc E, Baudoin R, Corlu A, Griscom L, Luc Duval J, Legallais C:
Selective control of liver and kidney cells migration during organotypic cocultures inside fibronectin-coated rectangular silicone microchannels.
Biomaterials
; 2007 Apr;28(10):1820-9
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Selective control of liver and
kidney
cells migration during organotypic cocultures inside fibronectin-coated rectangular silicone microchannels.
In this work, the behaviors of embryonic liver and
kidney
explants were studied inside rectangular polydimethylsiloxane (PDMS) microchannels.
In
kidney
monocultures, with and without fibronectin, we did not observe any migration inside the microchannels.
Contrary to liver cells,
kidney
cell migration was triggered when both fibronectin coating and coculture with liver or another
kidney
explant were used.
The migration was more largely observed in coculture with liver when compared to
kidney
-
kidney
cocultures.
In the case of liver-
kidney
coculture with fibronectin, the progression of the
kidney
cells inside the microchannels appears as a displacement of the entire
kidney
explant in the direction of the liver.
After contact, we observed a complete merging of both liver and
kidney
explants.
In contrast, for liver-
kidney
cocultures without fibronectin, only the liver moved toward
the kidney
.
[MeSH-major]
Fibronectins / pharmacology.
Kidney
/ cytology. Liver / cytology. Liver / growth & development. Liver, Artificial. Organ Culture Techniques / instrumentation. Tissue Engineering / instrumentation
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17178157.001).
[ISSN]
0142-9612
[Journal-full-title]
Biomaterials
[ISO-abbreviation]
Biomaterials
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / Coated Materials, Biocompatible; 0 / Fibronectins; 0 / Silicones
52.
Anchi T, Tamura K, Inoue K, Ashida S, Yamasaki I, Takeuchi T, Shuin T:
[A case of carcinoid tumor arising from a horseshoe kidney].
Hinyokika Kiyo
; 2009 Jun;55(6):327-30
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
[A case of carcinoid
tumor
arising from a horseshoe
kidney
].
Carcinoid
tumor of the kidney
is an extremely rare
neoplasm
.
We report a case of primary carcinoid
tumor
arising from a horseshoe
kidney
in a 31-year-old man.
From the results of computed tomography, magnetic resonance imaging and angiography, we suspected
renal
tumor
arising from a horseshoe
kidney
and performed left partial nephrectomy with isthmectomy.
Pathological findings of hematoxylin eosin staining revealed
tumor
cells proliferating in a cord-like and ribbon-like structure and
the tumor
cells stained strongly for chromogranin A, grimelius and neuron specific enolase.
According to these findings, we diagnosed carcinoid
tumor
arising from a horseshoe
kidney
.
[MeSH-major]
Carcinoid
Tumor
/ etiology.
Kidney
/ abnormalities.
Kidney Neoplasms
/ etiology
Genetic Alliance.
consumer health - Carcinoid Tumor
.
Genetic Alliance.
consumer health - Horseshoe kidney
.
MedlinePlus Health Information.
consumer health - Carcinoid Tumors
.
MedlinePlus Health Information.
consumer health - Kidney Cancer
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19588864.001).
[ISSN]
0018-1994
[Journal-full-title]
Hinyokika kiyo. Acta urologica Japonica
[ISO-abbreviation]
Hinyokika Kiyo
[Language]
jpn
[Publication-type]
Case Reports; English Abstract; Journal Article
[Publication-country]
Japan
53.
Crane HM, Kestenbaum B, Harrington RD, Kitahata MM:
Amprenavir and didanosine are associated with declining kidney function among patients receiving tenofovir.
AIDS
; 2007 Jul 11;21(11):1431-9
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Amprenavir and didanosine are associated with declining
kidney
function among patients receiving tenofovir.
OBJECTIVE: To examine the effect of antiretroviral agents and clinical factors on the development of tenofovir-associated
kidney
dysfunction.
Patients'
kidney
function prior to initiating and while receiving tenofovir was evaluated in relation to other antiretroviral medications and demographic and clinical characteristics.
Decline in
kidney
function was assessed by the glomerular filtration rate (GFR) as estimated by the Cockcroft-Gault (CG) equation, which incorporates weight.
Secondary analyses used the simplified Modification of Diet in
Renal
Disease (MDRD) equation.
RESULTS: Among the 445 patients initiating tenofovir, 51 (11%) developed a decline in
kidney
function.
In multivariate analysis, there was a significant association between decline in
kidney
function and concurrent use of amprenavir [odds ratio (OR) 3.6; P = 0.045] and didanosine (OR, 3.1; P = 0.006), age over 50 years (OR, 4.4; P = 0.03), and lower baseline weight (OR, 0.95/kg; P < 0.001).
Patients identified with
kidney
dysfunction by the MDRD equation did not fully overlap with those identified by the CG equation.
CONCLUSIONS: Didanosine and amprenavir use, increased age, and lower baseline weight were significantly associated with risk for
kidney
dysfunction among patients receiving tenofovir.
[MeSH-minor]
Adult. Age Factors. Analysis of Variance. Antiretroviral Therapy, Highly Active. Body Weight. Carbamates / adverse effects. Carbamates / therapeutic use. Creatinine / urine. Didanosine / adverse effects. Didanosine / therapeutic use. Drug Interactions. Female. Glomerular Filtration Rate / drug effects. Humans.
Kidney
/ drug effects.
Kidney
/ metabolism. Linear Models. Male. Middle Aged. Sulfonamides / adverse effects. Sulfonamides / therapeutic use. Tenofovir. Time Factors
MedlinePlus Health Information.
consumer health - HIV/AIDS
.
MedlinePlus Health Information.
consumer health - HIV/AIDS in Women
.
MedlinePlus Health Information.
consumer health - HIV/AIDS Medicines
.
COS Scholar Universe.
author profiles
.
HIV InSite.
treatment guidelines - Renal Manifestations of HIV
.
Hazardous Substances Data Bank.
AMPRENAVIR
.
Hazardous Substances Data Bank.
DIDEOXYINOSINE
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[CommentIn]
AIDS. 2007 Nov 30;21(18):2566
[
18025906.001
]
(PMID = 17589189.001).
[ISSN]
0269-9370
[Journal-full-title]
AIDS (London, England)
[ISO-abbreviation]
AIDS
[Language]
eng
[Grant]
United States / NIAID NIH HHS / AI / AI-060464; United States / NIAID NIH HHS / AI / AI-27757
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
England
[Chemical-registry-number]
0 / Anti-HIV Agents; 0 / Carbamates; 0 / Organophosphonates; 0 / Sulfonamides; 5S0W860XNR / amprenavir; 99YXE507IL / Tenofovir; AYI8EX34EU / Creatinine; JAC85A2161 / Adenine; K3GDH6OH08 / Didanosine
54.
Montgomery RA, Zachary AA, Ratner LE, Segev DL, Hiller JM, Houp J, Cooper M, Kavoussi L, Jarrett T, Burdick J, Maley WR, Melancon JK, Kozlowski T, Simpkins CE, Phillips M, Desai A, Collins V, Reeb B, Kraus E, Rabb H, Leffell MS, Warren DS:
Clinical results from transplanting incompatible live kidney donor/recipient pairs using kidney paired donation.
JAMA
; 2005 Oct 5;294(13):1655-63
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Clinical results from transplanting incompatible live
kidney
donor/recipient pairs using
kidney
paired donation.
CONTEXT: First proposed 2 decades ago, live
kidney
paired donation (KPD) was considered a promising new approach to addressing the shortage of organs for transplantation.
DESIGN, SETTING, AND PATIENTS: Prospective series of paired donations matched and transplanted from a pool of blood type or crossmatch incompatible donors and recipients with end-stage
renal
disease (6 conventional and 4 unconventional KPD transplants) at a US tertiary referral center (between June 2001 and November 2004) with expertise in performing transplants in patients with high immunologic risk.
INTERVENTION:
Kidney
paired donation and live donor
renal
transplantation.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Organ Donation
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[CommentIn]
JAMA. 2005 Oct 5;294(13):1691-3
[
16204670.001
]
(PMID = 16204665.001).
[ISSN]
1538-3598
[Journal-full-title]
JAMA
[ISO-abbreviation]
JAMA
[Language]
ENG
[Publication-type]
Journal Article
[Publication-country]
United States
55.
Nakamura Y, Urashima M, Nishihara R, Matsuura A, Bekku K, Iguchi H, Uesugi T, Saegusa M, Aramaki K:
Inflammatory pseudotumor of the kidney with renal artery penetration.
Radiat Med
; 2007 Dec;25(10):541-7
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Inflammatory pseudotumor of the
kidney
with
renal
artery penetration.
Inflammatory pseudotumor (IPT) is a quasineoplastic lesion that most commonly involves the lung and the orbit;
kidney
involvement is rare.
We report a case of inflammatory pseudotumor of the
kidney
.
Nonenhanced computed tomography (CT) demonstrated an ill-defined, isodensity mass measuring 3.5 cm in the lower portion of the left
kidney
.
Contrast-enhanced CT showed that branches of the
renal
artery without encasement penetrated
the tumor
; there was a little enhancement in the mass on the arterial phase and homogeneous enhancement on the venous phase.
Most IPTs of the
kidney
appear as an ill-defined, hypovascular, homogeneous
tumor
on CT images, with variable signal intensity on MRI T1WIs and low signal intensity on T2WIs.
Our case had the same imaging findings, with branches of the
renal
artery penetrating
the tumor
.
If the
renal
tumor
has these radiological findings,
the tumor
may be IPT.
[MeSH-major]
Granuloma, Plasma Cell / pathology.
Kidney
Diseases / pathology.
Renal
Artery / pathology
[MeSH-minor]
Diagnosis
, Differential. Humans. Magnetic Resonance Imaging. Male. Middle Aged.
Neoplasm
Invasiveness. Tomography, X-Ray Computed
MedlinePlus Health Information.
consumer health - Kidney Diseases
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Arch Esp Urol. 2002 Jan-Feb;55(1):85-7
[
11957761.001
]
[Cites]
Acta Cytol. 2000 May-Jun;44(3):478-80
[
10834015.001
]
[Cites]
Int J Urol. 2004 May;11(5):337-9
[
15147553.001
]
[Cites]
Actas Urol Esp. 2003 Oct;27(9):739-41
[
14626687.001
]
[Cites]
Int Urol Nephrol. 2004;36(2):141-3
[
15368680.001
]
[Cites]
Prog Urol. 2003 Feb;13(1):135-9
[
12703371.001
]
[Cites]
Arch Esp Urol. 2003 May;56(4):428-30
[
12830617.001
]
[Cites]
Am J Nephrol. 2000 May-Jun;20(3):217-21
[
10878405.001
]
[Cites]
AJR Am J Roentgenol. 1996 May;166(5):1151-5
[
8615260.001
]
[Cites]
Am J Surg Pathol. 2003 May;27(5):658-66
[
12717250.001
]
[Cites]
Int J Urol. 1999 Feb;6(2):107-10
[
10226817.001
]
[Cites]
Am J Kidney Dis. 1998 Jun;31(6):E5
[
10074585.001
]
[Cites]
J Urol. 1972 Jun;107(6):938-9
[
5033977.001
]
[Cites]
J Thorac Surg. 1954 Jul;28(1):55-63
[
13175281.001
]
[Cites]
Hinyokika Kiyo. 2004 Sep;50(9):629-31
[
15518129.001
]
[Cites]
Hinyokika Kiyo. 2006 Jan;52(1):31-3
[
16479987.001
]
[Cites]
South Med J. 1998 Nov;91(11):1050-3
[
9824189.001
]
[Cites]
Br J Urol. 1990 May;65(5):548-9
[
2354326.001
]
[Cites]
Verh Dtsch Ges Pathol. 1998;82:75-82
[
10095420.001
]
[Cites]
AJR Am J Roentgenol. 1976 Jun;126(6):1197-1202
[
179381.001
]
[Cites]
Bone Marrow Transplant. 2004 Nov;34(9):831-2
[
15322569.001
]
[Cites]
Radiographics. 2003 May-Jun;23(3):719-29
[
12740472.001
]
[Cites]
Rev Med Interne. 2000 Oct;21(10):889-92
[
11075397.001
]
[Cites]
Ann Urol (Paris). 2001 Jan;35(1):30-3
[
11233317.001
]
[Cites]
Radiology. 2005 Aug;236(2):441-50
[
16040900.001
]
[Cites]
J Urol. 1982 Jun;127(6):1177-8
[
7087029.001
]
[Cites]
Hinyokika Kiyo. 2000 Jan;46(1):23-6
[
10723660.001
]
[Cites]
AJR Am J Roentgenol. 1990 Oct;155(4):713-22
[
2119098.001
]
[Cites]
Nihon Hinyokika Gakkai Zasshi. 2001 Jul;92(5):589-92
[
11517572.001
]
[Cites]
Actas Urol Esp. 2005 Sep;29(8):794-6
[
16304914.001
]
[Cites]
Urology. 1976 Jul;8(1):89-91
[
941370.001
]
[Cites]
Surg Today. 2006;36(8):710-3
[
16865515.001
]
[Cites]
Int Urol Nephrol. 2004;36(2):137-40
[
15368679.001
]
[Cites]
Korean J Radiol. 2000 Oct-Dec;1(4):219-22
[
11752959.001
]
[Cites]
Ann Urol (Paris). 2003 Aug;37(4):147-9
[
12951701.001
]
[Cites]
Arch Pathol Lab Med. 2000 Aug;124(8):1209-12
[
10923085.001
]
[Cites]
Pediatr Pathol. 1992 Jul-Aug;12 (4):557-61
[
1409154.001
]
(PMID = 18085406.001).
[ISSN]
0288-2043
[Journal-full-title]
Radiation medicine
[ISO-abbreviation]
Radiat Med
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Japan
56.
Arija I, Centeno C, Viveros A, Brenes A, Marzo F, Illera JC, Silvan G:
Nutritional evaluation of raw and extruded kidney bean (Phaseolus vulgaris L. var. pinto) in chicken diets.
Poult Sci
; 2006 Apr;85(4):635-44
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Nutritional evaluation of raw and extruded
kidney
bean (Phaseolus vulgaris L. var. pinto) in chicken diets.
An experiment was conducted to study the effect of inclusion of different concentrations (0, 100, 200, and 300 g/kg) of raw
kidney
bean and extruded
kidney
bean in broiler chick (0 to 21 d of age) diets on performance, digestive organ sizes, protein and amino acid digestibilities, intestinal viscosity, cecal pH, and blood parameters.
Data were analyzed as a 3 x 2 factorial arrangement with 3 levels
of kidney
bean with and without extrusion.
Positive control without
kidney
bean was used.
Increasing
the kidney
bean content in the diet reduced weight gain and consumption, and increased the feed-to-gain ratio.
Relative pancreas, liver, and jejunum weights, and intestinal viscosity were increased in response to increasing
kidney
bean concentration in the diet.
The inclusion of different concentrations
of kidney
bean did not affect the apparent ileal digestibility of essential and nonessential amino acids, except for Met, Phe, and Cys, which were increased.
Increasing
kidney
bean in the diet did not affect blood parameters, except for total protein, which was increased, and for androstenedione and testosterone, which were reduced.
We concluded that the inclusion
of kidney
bean in chicken diets cause a negative effect on performance and CP and amino acid digestibilities, and modified digestive organ sizes, intestinal viscosity, cecal pH, and some blood parameters.
These effects were counteracted by the extrusion
of kidney
bean.
However, the inclusion of extruded
kidney
bean in a chick diet resulted in poorer performance compared with that obtained with a corn-soybean diet.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16615347.001).
[ISSN]
0032-5791
[Journal-full-title]
Poultry science
[ISO-abbreviation]
Poult. Sci.
[Language]
eng
[Publication-type]
Journal Article; Randomized Controlled Trial; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Plant Preparations
57.
Al-Bazzaz PH:
Kidney transplantation in Erbil, Iraq: a single-center experience.
Saudi J Kidney Dis Transpl
; 2010 Mar;21(2):359-62
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Kidney
transplantation in Erbil, Iraq: a single-center experience.
Kidney
transplantation is associated with improved quality of life and better survival among patients with end-stage
renal
disease.
The aim of this study is to assess the experience
of kidney
transplant program in a single center in Erbil, Iraq.
The records of 83 pairs, donors and recipients, treated with
kidney
transplantation at the Zheen Hospital in Erbil, over a two-year period were collected and analyzed.
In conclusion, even though experience related to
kidney
transplantation in Erbil is limited, the reported results are encouraging for a promising future.
[MeSH-major]
Kidney
Failure, Chronic / surgery.
Kidney
Transplantation. Tissue Donors / supply & distribution
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Kidney Failure
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Organ Donation
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 20228533.001).
[ISSN]
1319-2442
[Journal-full-title]
Saudi journal of kidney diseases and transplantation : an official publication of the Saudi Center for Organ Transplantation, Saudi Arabia
[ISO-abbreviation]
Saudi J Kidney Dis Transpl
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Saudi Arabia
58.
Musquera Felip M, Peri Cusí L, Alcaraz Asensio A:
[Surgical aspects of living-donor kidney transplantation].
Nefrologia
; 2010;30 Suppl 2:71-9
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
[Surgical aspects of living-donor
kidney
transplantation].
For these reasons, we can accept laparoscopic
kidney
living donor nephrectomy as the gold standard surgical technique in these patients.
In order to decide which
kidney
is better to extract, it is mandatory to maintain the best
kidney
in the donor.
[MeSH-major]
Kidney
Transplantation. Living Donors. Nephrectomy / methods
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Organ Donation
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 21183965.001).
[ISSN]
0211-6995
[Journal-full-title]
Nefrología : publicación oficial de la Sociedad Española Nefrologia
[ISO-abbreviation]
Nefrologia
[Language]
spa
[Publication-type]
English Abstract; Journal Article; Randomized Controlled Trial
[Publication-country]
Spain
59.
Petrovic V, Jovanovic I, Pesic I, Stefanovic V:
Role of stem cells in kidney repair.
Ren Fail
; 2010;32(10):1237-44
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Role of stem cells in
kidney
repair.
End-stage
renal
disease and acute
renal
failure are the most important issues of practical and clinical nephrology, bearing in mind their high mortality rate, solely symptomatic treatment, and overall economic impact on society.
The advances in stem cell biology opened the door for the new era in treatment of many disorders, including
renal
, offering new therapeutical solutions.
Findings suggesting that the adult
kidney
contains stem cells and that stem cells from bone marrow have potential to differentiate into
renal
cells focused research on the possible application of these cells in therapy
of kidney
disorders.
The other promising candidates for stem cell therapy for
the kidney
are embryonic stem cells and amniotic fluid-derived stem cells.
[MeSH-major]
Kidney
Failure, Chronic / surgery
MedlinePlus Health Information.
consumer health - Kidney Failure
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 20954989.001).
[ISSN]
1525-6049
[Journal-full-title]
Renal failure
[ISO-abbreviation]
Ren Fail
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
England
60.
Qiao H, Bai J, Chen Y, Tian J:
Kidney modelling for FDG excretion with PET.
Int J Biomed Imaging
; 2007;2007:63234
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Kidney
modelling for FDG excretion with PET.
The filtration process in
kidney
can be seen in the sequential images of each study.
Variational distribution of FDG in
kidney
can be detected in dynamic data.
According to the structure and function,
kidney
is divided into parenchyma and pelvis.
A unidirectional three-compartment model is proposed to describe the
renal
function in FDG excretion.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
J Nucl Med. 1999 Aug;40(8):1352-7
[
10450688.001
]
[Cites]
J Nucl Med. 1999 Aug;40(8):1358-66
[
10450689.001
]
[Cites]
J Nucl Med. 2002 Oct;43(10):1331-8
[
12368371.001
]
[Cites]
J Nucl Med. 2003 Jul;44(7):1113-47
[
12843230.001
]
[Cites]
Am J Physiol. 1980 Jan;238(1):E69-82
[
6965568.001
]
(PMID = 17728841.001).
[ISSN]
1687-4188
[Journal-full-title]
International journal of biomedical imaging
[ISO-abbreviation]
Int J Biomed Imaging
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Other-IDs]
NLM/ PMC1950229
61.
Nemati E, Pourfarziani V, Jafari AM, Assari S, Moghani-Lankarani M, Khedmat H, Bagheri N, Saadat SH:
Prediction of inpatient survival and graft loss in rehospitalized kidney recipients.
Transplant Proc
; 2007 May;39(4):974-7
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Prediction of inpatient survival and graft loss in rehospitalized
kidney
recipients.
INTRODUCTION: Despite a sizeable amount of research conducted hitherto into predictors of
renal
transplantation outcomes, there are scarce, data on predictors of in-hospital outcomes of post-
kidney
transplant rehospitalization.
This study sought to provide a user-friendly prediction model for inpatient mortality and graft loss among rehospitalized
kidney
recipients.
METHOD: This retrospective review of 424 consecutive
kidney
recipients rehospitalized after
kidney
transplantation between the years 2000 and 2005 used multiple logistic regression analysis to evaluate predictors of hospitalization outcomes.
RESULTS: Multivariate analysis showed that age at admission, diabetes mellitus as the cause of end-stage
renal
disease (ESRD), admission due to cerebrovascular accident (CVA), surgical complications were predictors of in-hospital death; age at transplantation, surgical complications, and rejection were predictors of graft loss.
CONCLUSIONS: Our prediction equations, using simple demographic and clinical variables, estimated the probability of inpatient mortality and graft loss among re-hospitalized
kidney
recipients.
[MeSH-major]
Graft Survival / physiology. Hospital Mortality. Inpatients / statistics & numerical data.
Kidney
Transplantation / physiology. Patient Readmission / statistics & numerical data
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17524866.001).
[ISSN]
0041-1345
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
62.
Hilhorst MT, Kranenburg LW, Busschbach JJ:
Should health care professionals encourage living kidney donation?
Med Health Care Philos
; 2007 Mar;10(1):81-90
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Should health care professionals encourage living
kidney
donation?
Living
kidney
donation provides a promising opportunity in situations where the scarcity of cadaveric
kidneys
is widely acknowledged.
While many patients and their relatives are willing to accept its benefits, others are concerned about living
kidney
programs; they appear to feel pressured into accepting living
kidney
transplantations as the only proper option for them.
Evidence suggests that both patients and their relatives have attitudes towards living
kidney
donation that are often open to change and, accordingly, can be influenced.
[MeSH-major]
Counseling / ethics.
Kidney
Transplantation / psychology. Living Donors / psychology. Physician-Patient Relations / ethics. Professional-Family Relations / ethics
MedlinePlus Health Information.
consumer health - Choosing a Doctor or Health Care Service
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Organ Donation
.
MedlinePlus Health Information.
consumer health - Talking With Your Doctor
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Health Policy. 1998 Jun;44(3):215-32
[
10182294.001
]
[Cites]
Transpl Int. 2005 May;18(5):519-23
[
15819799.001
]
[Cites]
JAMA. 2005 Apr 20;293(15):1883-90
[
15840863.001
]
[Cites]
J Med Ethics. 2005 Jun;31(6):362-5
[
15923488.001
]
[Cites]
Transplantation. 2005 Jun 15;79(11):1470-4
[
15940033.001
]
[Cites]
Ethical Theory Moral Pract. 2005 Apr;8(1-2):197-215
[
16459404.001
]
[Cites]
Transplantation. 2004 Jul 27;78(2):194-7
[
15280677.001
]
[Cites]
Med Health Care Philos. 2002;5(1):53-63
[
11954994.001
]
[Cites]
Med Health Care Philos. 2002;5(2):199-204
[
12168995.001
]
[Cites]
JAMA. 2002 Oct 2;288(13):1589-93
[
12350189.001
]
[Cites]
J Med Ethics. 2003 Jun;29(3):142-6
[
12796432.001
]
[Cites]
Transplantation. 2003 Nov 27;76(10):1437-44
[
14657682.001
]
[Cites]
Nephrol Dial Transplant. 2004 Jun;19(6):1600-5
[
15004261.001
]
[Cites]
Kennedy Inst Ethics J. 2001 Sep;11(3):285-303
[
11700684.001
]
(PMID = 16847727.001).
[ISSN]
1386-7423
[Journal-full-title]
Medicine, health care, and philosophy
[ISO-abbreviation]
Med Health Care Philos
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Netherlands
[Other-IDs]
NLM/ PMC2778634
63.
Kageyama S, Tsuru T, Okamoto K, Narita M, Okada Y:
Primary synovial sarcoma arising from a crossed ectopic kidney with fusion.
Int J Urol
; 2010 Jan;17(1):96-8
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Primary synovial sarcoma arising from a crossed ectopic
kidney
with fusion.
Crossed
renal
ectopia with fusion is a rare anomaly of the
kidney
.
The present case report describes a 67-year-old man with
renal
tumor
who had been diagnosed as having a crossed ectopic
kidney
with fusion for 25 years.
The pathological
diagnosis of
the primary
tumor
specimen was Wilms'
tumor
with favorable histology.
Upon
tumor
recurrence, molecular detection of SYT-SSX2 fusion transcripts lead to
the diagnosis
of synovial sarcoma of the
kidney
.
To our knowledge, this is the first case of primary synovial sarcoma arising from a crossed ectopic
kidney
with fusion.
[MeSH-major]
Kidney
/ abnormalities.
Kidney Neoplasms
/ complications. Sarcoma, Synovial / complications
Genetic Alliance.
consumer health - Synovial sarcoma
.
MedlinePlus Health Information.
consumer health - Kidney Cancer
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 20377831.001).
[ISSN]
1442-2042
[Journal-full-title]
International journal of urology : official journal of the Japanese Urological Association
[ISO-abbreviation]
Int. J. Urol.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Australia
64.
Misra S, Gordon JD, Fu AA, Glockner JF, Chade AR, Mandrekar J, Lerman L, Mukhopadhyay D:
The porcine remnant kidney model of chronic renal insufficiency.
J Surg Res
; 2006 Oct;135(2):370-9
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
The porcine remnant
kidney
model of chronic
renal
insufficiency.
BACKGROUND: The purpose of the present study was to develop and characterize a porcine model of chronic
renal
insufficiency created by
renal
artery embolization.
In the pilot group, the embolization procedure was optimized with respect to the size of polyvinyl acrylide (PVA) particles, coils, and amount
of kidney
embolized.
The animals were followed serially for 4 weeks after the embolization procedure to determine the
renal
function and hypertensive response.
RESULTS:
The kidney
function after the embolization was characterized by acute deterioration in
renal
function, followed by improvement, and "stable" chronic
renal
insufficiency with statistically significant elevation in creatinine and BUN being observed until day 42.
The remnant
kidney
developed fibrosis in the tublointerstitial compartment as it hypertrophied and increased its weight which remained significantly elevated after embolization.
CONCLUSIONS: A reproducible remnant
kidney
model of chronic
renal
insufficiency in pigs was developed.
In this model, stable
renal
insufficiency develops by 4 weeks that lasts until 12 weeks.
[MeSH-major]
Disease Models, Animal.
Kidney
/ pathology.
Renal
Insufficiency, Chronic / pathology
[MeSH-minor]
Angiography. Animals. Blood Pressure. Blood Urea Nitrogen. Creatinine / metabolism. Male. Organ Size.
Renal
Artery Obstruction / surgery. Sus scrofa. Time Factors
MedlinePlus Health Information.
consumer health - Chronic Kidney Disease
.
The Lens.
Cited by Patents in
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16815448.001).
[ISSN]
0022-4804
[Journal-full-title]
The Journal of surgical research
[ISO-abbreviation]
J. Surg. Res.
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
AYI8EX34EU / Creatinine
65.
Yang B, Sonawane ND, Zhao D, Somlo S, Verkman AS:
Small-molecule CFTR inhibitors slow cyst growth in polycystic kidney disease.
J Am Soc Nephrol
; 2008 Jul;19(7):1300-10
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Small-molecule CFTR inhibitors slow cyst growth in polycystic
kidney
disease.
Cyst expansion in polycystic
kidney
disease (PKD) involves progressive fluid accumulation, which is believed to require chloride transport by the cystic fibrosis transmembrane conductance regulator (CFTR) protein.
These compounds also inhibited cyst number and growth by >80% in an embryonic
kidney
cyst model involving 4-d organ culture of embryonic day 13.5 mouse
kidneys
in 8-Br-cAMP-containing medium.
Subcutaneous delivery of tetrazolo-CFTR(inh)-172 and Ph-GlyH-101 to neonatal,
kidney
-specific PKD1 knockout mice produced stable, therapeutic inhibitor concentrations of >3 microM in urine and
kidney
tissue.
Treatment of mice for up to 7 d remarkably slowed
kidney
enlargement and cyst expansion and preserved
renal
function.
These results implicate CFTR in
renal
cyst growth and suggest that CFTR inhibitors may hold therapeutic potential to reduce cyst growth in PKD.
COS Scholar Universe.
author profiles
.
Faculty of 1000.
commentaries/discussion - See the articles recommended by F1000Prime's Faculty of more than 8,000 leading experts in Biology and Medicine.
(subscription/membership/fee required).
Hazardous Substances Data Bank.
GLYCINE
.
The Lens.
Cited by Patents in
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Kidney Int. 1999 Apr;55(4):1187-97
[
10200981.001
]
[Cites]
Am J Kidney Dis. 1998 Dec;32(6):976-83
[
9856513.001
]
[Cites]
Clin Cancer Res. 2005 Oct 1;11(19 Pt 1):6924-32
[
16203784.001
]
[Cites]
FASEB J. 2006 Jan;20(1):130-2
[
16317066.001
]
[Cites]
J Nephrol. 2006 Jul-Aug;19(4):529-34
[
17048214.001
]
[Cites]
Cell Tissue Res. 2006 Dec;326(3):671-85
[
16767405.001
]
[Cites]
J Am Soc Nephrol. 2006 Dec;17(12):3424-37
[
17108316.001
]
[Cites]
J Am Soc Nephrol. 2007 May;18(5):1399-407
[
17429048.001
]
[Cites]
Gastroenterology. 2007 Apr;132(4):1234-44
[
17408659.001
]
[Cites]
Nat Genet. 2000 Jun;25(2):143-4
[
10835625.001
]
[Cites]
Annu Rev Med. 2001;52:93-123
[
11160770.001
]
[Cites]
J Am Soc Nephrol. 2002 Jul;13(7):1837-46
[
12089379.001
]
[Cites]
J Am Soc Nephrol. 2002 Sep;13(9):2384-98
[
12191984.001
]
[Cites]
J Clin Invest. 2002 Dec;110(11):1651-8
[
12464670.001
]
[Cites]
Kidney Int. 2003 Jan;63(1):365-76
[
12472805.001
]
[Cites]
N Engl J Med. 2004 Jan 8;350(2):151-64
[
14711914.001
]
[Cites]
Mol Genet Metab. 2004 Feb;81(2):75-85
[
14741187.001
]
[Cites]
FEBS Lett. 2004 Jan 30;558(1-3):52-6
[
14759515.001
]
[Cites]
Nat Med. 2004 Apr;10(4):363-4
[
14991049.001
]
[Cites]
J Gen Physiol. 2004 Aug;124(2):125-37
[
15277574.001
]
[Cites]
Kidney Int. 2004 Nov;66(5):1926-38
[
15496164.001
]
[Cites]
Scan Electron Microsc. 1986;(Pt 3):1135-50
[
3798016.001
]
[Cites]
Scanning Microsc. 1988 Sep;2(3):1739-63
[
3059485.001
]
[Cites]
Proc Natl Acad Sci U S A. 1989 Aug;86(15):6007-11
[
2474825.001
]
[Cites]
Trans Assoc Am Physicians. 1989;102:158-62
[
2561639.001
]
[Cites]
N Engl J Med. 1993 Jul 29;329(5):310-3
[
8321258.001
]
[Cites]
N Engl J Med. 1993 Jul 29;329(5):332-42
[
8321262.001
]
[Cites]
J Am Soc Nephrol. 1995 Oct;6(4):1230-41
[
8589291.001
]
[Cites]
Hum Mol Genet. 2008 Jun 1;17(11):1505-16
[
18263604.001
]
[Cites]
FASEB J. 1989 Dec;3(14):2629-32
[
2480260.001
]
[Cites]
Kidney Int. 1996 Jul;50(1):208-18
[
8807590.001
]
[Cites]
Proc Natl Acad Sci U S A. 1996 Sep 17;93(19):10206-11
[
8816777.001
]
[Cites]
Eur J Clin Invest. 1996 Jun;26(6):506-13
[
8817166.001
]
[Cites]
Cell. 1996 Dec 13;87(6):979-87
[
8978603.001
]
[Cites]
Biochem J. 1997 Feb 15;322 ( Pt 1):259-65
[
9078271.001
]
[Cites]
Cell. 1998 Apr 17;93(2):177-88
[
9568711.001
]
[Cites]
Physiol Rev. 1998 Oct;78(4):1165-91
[
9790573.001
]
[Cites]
J Pharm Sci. 2005 Jan;94(1):134-43
[
15761937.001
]
(PMID = 18385427.001).
[ISSN]
1533-3450
[Journal-full-title]
Journal of the American Society of Nephrology : JASN
[ISO-abbreviation]
J. Am. Soc. Nephrol.
[Language]
ENG
[Grant]
United States / NHLBI NIH HHS / HL / HL73856; United States / NIDDK NIH HHS / DK / P50 DK057328; United States / NEI NIH HHS / EY / R01 EY013574; United States / NIBIB NIH HHS / EB / R01 EB000415; United States / NIDDK NIH HHS / DK / R01 DK054053; United States / NIDDK NIH HHS / DK / DK57328; United States / NIDDK NIH HHS / DK / R01 DK035124; United States / NHLBI NIH HHS / HL / R01 HL073856; United States / NIDDK NIH HHS / DK / P30 DK072517; United States / NIDDK NIH HHS / DK / DK54053; United States / NIDDK NIH HHS / DK / DK72517; United States / NHLBI NIH HHS / HL / R01 HL059198; United States / NEI NIH HHS / EY / EY13574; United States / NIDDK NIH HHS / DK / DK35124; United States / NIBIB NIH HHS / EB / EB00415; United States / NHLBI NIH HHS / HL / HL59198; United States / NIDDK NIH HHS / DK / R37 DK035124; United States / NIBIB NIH HHS / EB / R37 EB000415
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Cadherins; 0 / Cdh16 protein, mouse; 0 / TRPP Cation Channels; 0 / Thiazolidines; 0 / polycystic kidney disease 1 protein; 126880-72-6 / Cystic Fibrosis Transmembrane Conductance Regulator; 14379-80-7 / glycine hydrazide; EC 2.7.7.- / Cre recombinase; EC 2.7.7.- / Integrases; TE7660XO1C / Glycine
[Other-IDs]
NLM/ PMC2440296
66.
Wang Y, Wang Z, Wang W, Ren H, Zhang W, Chen N:
Analysis of factors associated with renal function in Chinese adults with congenital solitary kidney.
Intern Med
; 2010;49(20):2203-9
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Analysis of factors associated with
renal
function in Chinese adults with congenital solitary
kidney
.
BACKGROUND: Patients with congenital solitary
kidney
have an increased risk of developing hypertension, proteinuria and
renal
insufficiency.
However, the specific factors associated with the progression of
renal
function in adults with congenital solitary
kidney
remain still unclear.
The purpose of this study was to identify factors that are independently associated with
renal
function progression in patients with congenital solitary
kidney
.
METHODS: Sixty-five Chinese adults with congenital solitary
kidney
(48 patients with unilateral
renal
agenesis and 17 with severe unilateral
renal
dysplasia) were recruited into our study retrospectively.
RESULTS: Of sixty-five patients with congenital solitary
kidney
, the prevalence of hypertension, proteinuria and
renal
insufficiency was 36.9%, 35.4% and 38.5%, respectively.
While there was no statistically significant difference in prevalence of hypertension between patients with and without
renal
insufficiency, the prevalence of proteinuria in patients with
renal
insufficiency was significantly higher than in those without
renal
insufficiency (p<0.05).
Logistic regression analysis revealed that
kidney
length and proteinuria were independently associated with the progression of
renal
function (OR=0.20, 95%CI 0.05-0.79, and OR=8.30, 95%CI 2.30-29.96, respectively).
CONCLUSION: Hypertension, proteinuria or
renal
insufficiency was present in approximately one-third of adults with congenital solitary
kidney
.
Those with a
kidney
length of less than 120 mm or proteinuria had a much higher risk of
renal
insufficiency.
[MeSH-major]
Hypertension,
Renal
/ etiology.
Kidney
/ abnormalities.
Kidney
Failure, Chronic / etiology. Proteinuria / etiology
MedlinePlus Health Information.
consumer health - Kidney Failure
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 20962438.001).
[ISSN]
1349-7235
[Journal-full-title]
Internal medicine (Tokyo, Japan)
[ISO-abbreviation]
Intern. Med.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Japan
[Chemical-registry-number]
0 / Biomarkers; 268B43MJ25 / Uric Acid; AYI8EX34EU / Creatinine
67.
Newsome BB, Warnock DG, Kiefe CI, Weissman NW, Houston TK, Centor RM, Person SD, McClellan WM, Allison JJ:
Delay in time to receipt of thrombolytic medication among Medicare patients with kidney disease.
Am J Kidney Dis
; 2005 Oct;46(4):595-602
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Delay in time to receipt of thrombolytic medication among Medicare patients with
kidney
disease.
BACKGROUND: Patients with
kidney
disease and acute myocardial infarction (AMI) receive standard therapy, including thrombolytic medication, less frequently than patients with normal
kidney
function.
Our goal is to identify potential differences in thrombolytic medication delays and thrombolytic-associated bleeding events by severity
of kidney
disease.
Average time to thrombolytic therapy was longer in patients with worse
kidney
function.
Adjusted hazard ratios for minutes to thrombolytic therapy were 0.83 (95% confidence interval [CI], 0.79 to 0.87) for patients with a serum creatinine level of 1.6 to 2.0 mg/dL (141 to 177 micromol/L) and 0.58 (95% CI, 0.53 to 0.63) for patients with a creatinine level greater than 2.0 mg/dL (>177 micromol/L) or on dialysis therapy compared with those with normal
kidney
function.
Odds ratios for bleeding events in patients administered thrombolytics versus those who were not decreased with worse
kidney
function: adjusted odds ratios, 2.28 (95% CI, 2.16 to 2.42) in patients with normal
kidney
function and 1.84 (95% CI, 1.09 to 3.10) in dialysis patients.
CONCLUSION: Patients with worse
kidney
function experienced treatment delays, but were not at greater risk for thrombolysis-associated excess bleeding events.
[MeSH-major]
Fibrinolytic Agents / administration & dosage.
Kidney
Diseases / complications. Medicare / statistics & numerical data. Myocardial Infarction / drug therapy. Thrombolytic Therapy / statistics & numerical data
Genetic Alliance.
consumer health - Kidney Disease
.
MedlinePlus Health Information.
consumer health - Heart Attack
.
MedlinePlus Health Information.
consumer health - Kidney Diseases
.
MedlinePlus Health Information.
consumer health - Medicare
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16183413.001).
[ISSN]
1523-6838
[Journal-full-title]
American journal of kidney diseases : the official journal of the National Kidney Foundation
[ISO-abbreviation]
Am. J. Kidney Dis.
[Language]
eng
[Grant]
United States / AHRQ HHS / HS / T32 HS013852; United States / NHLBI NIH HHS / HL / R01 HL70786
[Publication-type]
Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
[Publication-country]
United States
[Chemical-registry-number]
0 / Cardiovascular Agents; 0 / Fibrinolytic Agents; AYI8EX34EU / Creatinine
68.
Mazaris EM, Warrens AN, Papalois VE:
Ethical issues in live donor kidney transplant: views of medical and nursing staff.
Exp Clin Transplant
; 2009 Mar;7(1):1-7
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Ethical issues in live donor
kidney
transplant: views of medical and nursing staff.
OBJECTIVES: The ongoing development of live donor
kidney
transplant has generated many ethical dilemmas.
MATERIALS AND METHODS: An anonymous and confidential questionnaire was sent to 236 members of the medical and nursing staff of the West London
Renal
and Transplant Centre, to assess their views on the ethics of the current practice of live donor
kidney
transplant.
Respondents considered live donor
kidney
transplant ethically acceptable between blood relatives (100%), nonblood relatives and friends (92.6%), and strangers (47.2%).
Most respondents were willing to donate a
kidney
to a blood relative (92.6%) or a nonblood relative or friend (81.5%), and 12.0% were willing to donate to a stranger.
Considering themselves as potential recipients if they had end-stage
renal
disease, most would accept a
kidney
from a blood relative (91.7%) or nonblood relative or friend (85.2%),while 44.5% would accept a
kidney
from a stranger.
CONCLUSIONS: Live related and unrelated
kidney
donation are considered ethically acceptable procedures, and nondirected donation is gaining support among transplant professionals.
[MeSH-major]
Attitude of Health Personnel. Directed Tissue Donation / ethics. Health Knowledge, Attitudes, Practice.
Kidney
Transplantation / ethics. Living Donors / ethics. Tissue and Organ Procurement / ethics
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Organ Donation
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19364304.001).
[ISSN]
1304-0855
[Journal-full-title]
Experimental and clinical transplantation : official journal of the Middle East Society for Organ Transplantation
[ISO-abbreviation]
Exp Clin Transplant
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Turkey
69.
Mozer P, Leroy A, Payan Y, Troccaz J, Chartier-Kastler E, Richard F:
Computer-assisted access to the kidney.
Int J Med Robot
; 2005 Dec;1(4):58-66
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Computer-assisted access to
the kidney
.
OBJECTIVES: The aim of this paper is to introduce the principles of computer-assisted access to
the kidney
.
MATERIAL AND METHODS: Both CT and US images of informed normal volunteer were obtained to perform calculation on the accuracy of registration and punctures were carried out on a
kidney
phantom to measure the precision of the whole of the system.
RESULTS: We carried out millimetric registrations on real data and guidance experiments on a
kidney
phantom showed encouraging results of 4.7 mm between planned and reached targets.
[MeSH-major]
Kidney
/ surgery. Surgery, Computer-Assisted
HAL archives ouvertes.
Full text from
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Copyright]
Copyright 2005 John Wiley & Sons, Ltd.
(PMID = 17518406.001).
[ISSN]
1478-596X
[Journal-full-title]
The international journal of medical robotics + computer assisted surgery : MRCAS
[ISO-abbreviation]
Int J Med Robot
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
England
70.
Geel A, Zuidema W, van Gelder T, van Doornum G, Weimar W:
Successful vaccination against varicella zoster virus prior to kidney transplantation.
Transplant Proc
; 2005 Mar;37(2):952-3
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Successful vaccination against varicella zoster virus prior to
kidney
transplantation.
BACKGROUND: In recent years we have observed a number of severe complications of primary varicella-zoster virus (VZV) infections after adult
kidney
transplantation.
We questioned how many patients on the waiting list for
kidney
transplantation had not been protected against VZV and whether patients with
renal
insufficiency would be able to develop a specific immune response upon VZV vaccination.
METHODS: We examined the VZV antibody titer of 280 patients on
the kidney
transplant waiting list.
Seronegative
kidney
transplant candidates were vaccinated twice with a live attenuated varicella vaccine at an interval of 6 weeks.
[MeSH-major]
Antibodies, Viral / blood. Chickenpox / immunology. Chickenpox Vaccine / therapeutic use.
Kidney
Transplantation / immunology
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Chickenpox
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 15848586.001).
[ISSN]
0041-1345
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Antibodies, Viral; 0 / Chickenpox Vaccine
71.
Bader AA, Tamussino KF, Winter R:
Ectopic (pelvic) kidney mimicking bulky lymph nodes at pelvic lymphadenectomy.
Gynecol Oncol
; 2005 Mar;96(3):873-5
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Ectopic (pelvic)
kidney
mimicking bulky lymph nodes at pelvic lymphadenectomy.
BACKGROUND: Ectopic (pelvic)
kidney
is the most common congenital
renal
anomaly with an incidence of 1 in 500 to 1 in 2000.
A pelvic
kidney
can be encountered at pelvic or paraaortic lymphadenectomy.
CASE REPORTS: In two patients undergoing pelvic lymphadenectomy, lobulated
tumors
near the pelvic brim were initially interpreted as bulky lymph node conglomerates.
Further dissection showed the ureter to originate from the masses, leading to a
diagnosis of
pelvic
kidney
.
CONCLUSION: Pelvic
kidneys
mistaken for bulky lymph nodes are a potential intraoperative pitfall in patients with gynecologic malignancies.
Keys to recognition include an index of suspicion, identifying the course of the ureter and origin of the
renal
vessels, and confirming absence of a
kidney
at the normal location.
[MeSH-major]
Genital
Neoplasms
, Female / surgery.
Kidney
/ abnormalities. Lymph Nodes / surgery
[MeSH-minor]
Aged. Aged, 80 and over.
Diagnosis
, Differential. Female. Humans. Lymph Node Excision. Lymphatic Metastasis. Middle Aged
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 15721442.001).
[ISSN]
0090-8258
[Journal-full-title]
Gynecologic oncology
[ISO-abbreviation]
Gynecol. Oncol.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
United States
72.
Mekeel KL, Mazur MJ, Reddy KS, Mulligan DC, Heilman RL, Chakkera HA, Andrews PE, Moss AA:
Diffuse parenchymal urine leak after kidney transplantation following degloving injury during donor nephrectomy.
Am J Transplant
; 2007 Aug;7(8):2039-41
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Diffuse parenchymal urine leak after
kidney
transplantation following degloving injury during donor nephrectomy.
Laparoscopic donor nephrectomy can result in trauma to
the kidney
which may affect recipient graft function.
In this case,
the kidney
sustained a complete degloving of the capsule during extraction.
The kidney
was transplanted and had immediate, good
renal
function, but postoperative course was complicated by a large urinoma that drained through the wound.
Exploration was negative for a defined urine leak, but the surface of the denuded
kidney
was leaking a significant amount of unconcentrated urine.
The patient was successfully treated with tissue glue treatment to
the kidney
surface and peritoneal window.
[MeSH-major]
Intraoperative Complications.
Kidney
/ injuries.
Kidney
Transplantation / adverse effects. Laparoscopy / adverse effects. Nephrectomy / adverse effects. Tissue Donors. Tissue and Organ Procurement / methods
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Organ Donation
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17578504.001).
[ISSN]
1600-6135
[Journal-full-title]
American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons
[ISO-abbreviation]
Am. J. Transplant.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Denmark
73.
Krajisnik T, Olauson H, Mirza MA, Hellman P, Akerström G, Westin G, Larsson TE, Björklund P:
Parathyroid Klotho and FGF-receptor 1 expression decline with renal function in hyperparathyroid patients with chronic kidney disease and kidney transplant recipients.
Kidney Int
; 2010 Nov;78(10):1024-32
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Parathyroid Klotho and FGF-receptor 1 expression decline with
renal
function in hyperparathyroid patients with chronic
kidney
disease and
kidney
transplant recipients.
However, patients with chronic
kidney
disease (CKD) develop secondary hyperparathyroidism despite high levels of serum FGF23, indicating a parathyroid FGF23 'resistance'.
Here we analyzed the expression of the FGF23 receptors Klotho and FGF receptor 1 (FGFR1) in 88 hyperplastic parathyroid glands from 31 patients with CKD (including 21
renal
allograft recipients), and their regulation in isolated bovine and human hyperplastic parathyroid cells.
Thus parathyroid Klotho and FGFR1 decrease with declining
renal
function, possibly because of alterations in mineral metabolism related to the failing
kidney
.
[MeSH-major]
Glucuronidase / metabolism. Hyperparathyroidism / metabolism.
Kidney
/ physiopathology.
Kidney
Diseases / metabolism.
Kidney
Transplantation / physiology. Parathyroid Glands / metabolism. Receptors, Fibroblast Growth Factor / metabolism
Genetic Alliance.
consumer health - Kidney Disease
.
MedlinePlus Health Information.
consumer health - Kidney Diseases
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
COS Scholar Universe.
author profiles
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[CommentIn]
Kidney Int. 2010 Nov;78(10):953-5
[
21030971.001
]
(PMID = 20686451.001).
[ISSN]
1523-1755
[Journal-full-title]
Kidney international
[ISO-abbreviation]
Kidney Int.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Minerals; 0 / Parathyroid Hormone; 0 / Receptors, Calcitriol; 0 / Receptors, Fibroblast Growth Factor; 0 / fibroblast growth factor 23; 62031-54-3 / Fibroblast Growth Factors; EC 3.2.1.31 / Glucuronidase; EC 3.2.1.31 / klotho protein
74.
Bouman CS, Oudemans-van Straaten HM:
Timing of renal replacement therapy in critically ill patients with acute kidney injury.
Curr Opin Crit Care
; 2007 Dec;13(6):656-61
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Timing of
renal
replacement therapy in critically ill patients with acute
kidney
injury.
PURPOSE OF REVIEW: Timing of
renal
replacement therapy in critically ill patients with acute
kidney
injury is highly subjective, and may influence outcome.
We discuss
renal
and nonrenal criteria for timing considering the recent literature.
All but one randomized controlled trial indicated better outcome with early
renal
replacement therapy but had poor methodological quality.
SUMMARY: In the absence of large randomized controlled trials we can make no firm recommendations for timing of
renal
replacement therapy in acute
kidney
injury.
Since rapid recovery of
renal
function is unlikely when other organ failure persists and the consequences of acute
kidney
injury may be more severe in critically ill patients, we suggest other organ failure is also considered.
Patients with acute
kidney
injury, persisting shock and poorly recovering functions of other organs may benefit from early therapy.
For future studies, we recommend describing
renal
replacement therapy timing according to the 'RIFLE' classification, as modified by the Acute
Kidney
Injury Network, and quantifying the severity of other organ failure.
Biomarkers may refine acute
kidney
injury and timing definitions in the future.
[MeSH-major]
Acute
Kidney
Injury / therapy. Critical Illness.
Kidney
/ injuries.
Renal
Replacement Therapy / methods. Treatment Outcome
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17975386.001).
[ISSN]
1070-5295
[Journal-full-title]
Current opinion in critical care
[ISO-abbreviation]
Curr Opin Crit Care
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Chemical-registry-number]
0 / Biomarkers
[Number-of-references]
51
75.
Stojanovic VD, Doronjski AR, Spasojevic SD, Pavlovic VS, Nikolic MM, Kovacevic BB:
Chronic inflammatory demyelinating polyradiculoneuropathy, in an 8-year-old girl, complicated by deafness and kidney fibrosis.
J Child Neurol
; 2009 Aug;24(8):997-1000
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Chronic inflammatory demyelinating polyradiculoneuropathy, in an 8-year-old girl, complicated by deafness and
kidney
fibrosis.
The disease was complicated by deafness and
kidney
fibrosis.
Because the pathogenesis of chronic inflammatory demyelinating polyradiculoneuropathy has still not been clear, and on the grounds of several cases of chronic inflammatory demyelinating polyradiculoneuropathy conjoined with
the kidney
disease described in literature (glomerulopathy, interstitial nephritis), every patient with chronic inflammatory demyelinating polyradiculoneuropathy needs to undergo the urinalyses.
[MeSH-major]
Deafness / complications. Deafness / drug therapy.
Kidney
Diseases / complications.
Kidney
Diseases / drug therapy. Polyradiculoneuropathy, Chronic Inflammatory Demyelinating / complications. Polyradiculoneuropathy, Chronic Inflammatory Demyelinating / drug therapy
Genetic Alliance.
consumer health - Deafness
.
MedlinePlus Health Information.
consumer health - Hearing Disorders and Deafness
.
MedlinePlus Health Information.
consumer health - Kidney Diseases
.
Hazardous Substances Data Bank.
MYCOPHENOLATE MOFETIL
.
Hazardous Substances Data Bank.
METHYLPREDNISOLONE
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19240045.001).
[ISSN]
1708-8283
[Journal-full-title]
Journal of child neurology
[ISO-abbreviation]
J. Child Neurol.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Anti-Inflammatory Agents, Non-Steroidal; 0 / Immunoglobulins, Intravenous; 0 / Immunologic Factors; 0 / Neuroprotective Agents; 9242ECW6R0 / mycophenolate mofetil; HU9DX48N0T / Mycophenolic Acid; X4W7ZR7023 / Methylprednisolone
76.
Sudarshan S, Linehan WM:
Genetic basis of cancer of the kidney.
Semin Oncol
; 2006 Oct;33(5):544-51
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Genetic basis of cancer of the
kidney
.
Kidney
cancer is not a single disease.
It is made up of a number of different types of cancer that occur in
the kidney
, each with a different histologic type, having a different clinical course, responding differently to therapy and caused by a different gene.
The identification of families with a predisposition to the development of
renal
neoplasms
, including von Hippel-Lindau (VHL), hereditary papillary
renal
carcinoma (HPRC), Birt-Hogg-Dubé (BHD), and hereditary leiomyomatosis and
renal
cell cancer (HLRCC), has made possible the identification of the different genes for these cancers.
The elucidation of molecular pathogenesis in these familial forms
of kidney
cancer should provide the opportunity to determine successful approaches for novel therapeutic agents.
[MeSH-major]
Carcinoma, Papillary / genetics. Carcinoma,
Renal
Cell / genetics. Genetic Predisposition to Disease.
Kidney Neoplasms
/ genetics. von Hippel-Lindau Disease / genetics
Genetic Alliance.
consumer health - Kidney cancer
.
MedlinePlus Health Information.
consumer health - Kidney Cancer
.
MedlinePlus Health Information.
consumer health - Von Hippel-Lindau Disease
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17045083.001).
[ISSN]
0093-7754
[Journal-full-title]
Seminars in oncology
[ISO-abbreviation]
Semin. Oncol.
[Language]
eng
[Grant]
United States / Intramural NIH HHS / /
[Publication-type]
Journal Article; Research Support, N.I.H., Intramural; Review
[Publication-country]
United States
[Number-of-references]
87
77.
Kalb B, Martin DR, Salman K, Sharma P, Votaw J, Larsen C:
Kidney transplantation: structural and functional evaluation using MR Nephro-Urography.
J Magn Reson Imaging
; 2008 Oct;28(4):805-22
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Kidney
transplantation: structural and functional evaluation using MR Nephro-Urography.
End-stage-
renal
disease (ESRD) is a major health issue in the United States, and the Medicare costs of ESRD totaled nearly USD 21 billion in 2005.
Renal
transplantation has emerged as the treatment of choice in this patient population, providing improved quality of life and lower healthcare costs compared with other treatment options.
Imaging evaluation of a graft
kidney
plays a critical role in the postoperative care of the
renal
transplant patient.
In the past, diagnostic evaluation of the transplant
kidney
has depended upon a combination of ultrasonography, computed tomography, MRI, and biopsy, used in conjunction with the patient's clinical presentation.
However, new and developing advances in MR technology has lead to the development of MR Nephro-Urography (MRNU), which provides both anatomic and functional evaluation of the
kidney
in a single examination.
It is expected that the increasing use of MRNU will have a significant impact on the management of
renal
transplant patients.
This review describes MRNU methodology, examines known posttransplant complications, and highlights the utility of MRNU as a comprehensive imaging examination to diagnose both surgical and medical complications of the transplant
kidney
.
[MeSH-major]
Kidney
/ blood supply.
Kidney
Failure, Chronic / surgery.
Kidney
Transplantation. Magnetic Resonance Imaging / methods. Postoperative Complications /
diagnosis
[MeSH-minor]
Graft Rejection. Graft Survival. Humans. Recovery of Function.
Renal
Dialysis
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - After Surgery
.
MedlinePlus Health Information.
consumer health - Kidney Failure
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - MRI Scans
.
COS Scholar Universe.
author profiles
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Copyright]
(c) 2008 Wiley-Liss, Inc.
(PMID = 18821623.001).
[ISSN]
1053-1807
[Journal-full-title]
Journal of magnetic resonance imaging : JMRI
[ISO-abbreviation]
J Magn Reson Imaging
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Number-of-references]
64
78.
Nunes AC, Milani V, Porsch DB, Rossato LB, Mattos CB, Roisenberg I, Barros EJ:
Frequency and clinical profile of patients with polycystic kidney disease in southern Brazil.
Ren Fail
; 2008;30(2):169-73
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Frequency and clinical profile of patients with polycystic
kidney
disease in southern Brazil.
BACKGROUND: Autosomal dominant polycystic
kidney
disease (ADPKD) is one of the most common genetic nephropathies, affecting one in every 800-1000 individuals in the worldwide general population and 5-10% of hemodialysis patients.
RESULTS: Of 1326 patients on hemodialysis in the south of Brazil that composed this study, 99 (7.5%) had polycystic
kidney
as primary cause for chronic
renal
failure.
[MeSH-major]
Kidney
Failure, Chronic / epidemiology.
Kidney
Failure, Chronic / therapy. Polycystic
Kidney
, Autosomal Dominant / epidemiology. Polycystic
Kidney
, Autosomal Dominant / therapy
[MeSH-minor]
Adult. Age Distribution. Analysis of Variance. Brazil / epidemiology. Cross-Sectional Studies. Female. Humans. Incidence.
Kidney
Function Tests. Male. Middle Aged. Multicenter Studies as Topic. Probability. Prognosis.
Renal
Dialysis / statistics & numerical data. Risk Assessment. Severity of Illness Index. Sex Distribution. Statistics, Nonparametric. Survival Analysis. Treatment Outcome
Genetic Alliance.
consumer health - Kidney Disease
.
Genetic Alliance.
consumer health - Polycystic Kidney Disease
.
MedlinePlus Health Information.
consumer health - Kidney Failure
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18300116.001).
[ISSN]
1525-6049
[Journal-full-title]
Renal failure
[ISO-abbreviation]
Ren Fail
[Language]
eng
[Publication-type]
Comparative Study; Journal Article
[Publication-country]
United States
79.
Olson GE, Winfrey VP, Hill KE, Burk RF:
Megalin mediates selenoprotein P uptake by kidney proximal tubule epithelial cells.
J Biol Chem
; 2008 Mar 14;283(11):6854-60
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Megalin mediates selenoprotein P uptake by
kidney
proximal tubule epithelial cells.
Selenoprotein P (Sepp1) contains most of the selenium in blood plasma, and it is utilized by
the kidney
, brain, and testis as a selenium source for selenoprotein synthesis.
We recently demonstrated that apolipoprotein E receptor-2 (ApoER2) is required for Sepp1 uptake by the testis and that deletion of ApoER2 reduces testis and brain, but not
kidney
, selenium levels.
This study examined
the kidney
Sepp1 uptake pathway.
Tissue ligand binding assays using cryosections of Sepp1-/-
kidneys
revealed that the proximal tubule epithelium contained Sepp1-binding sites that were blocked by the receptor-associated protein, RAP, an inhibitor of lipoprotein receptor-ligand interactions.
Ligand blotting assays
of kidney
membrane preparations fractionated by SDS-PAGE revealed that Sepp1 binds megalin, a lipoprotein receptor localized to the proximal tubule epithelium.
Immunolocalization analyses confirmed the in vivo co-localization of Sepp1 and megalin in wild type
kidneys
and demonstrated the absence of proximal tubule Sepp1 uptake in megalin null mice.
These results demonstrate that
kidney
selenium homeostasis is mediated by a megalin-dependent Sepp1 uptake pathway in the proximal tubule.
[MeSH-major]
Epithelial Cells / metabolism. Gene Expression Regulation.
Kidney
Tubules / cytology. Low Density Lipoprotein Receptor-Related Protein-2 / physiology. Selenoprotein P / metabolism
COS Scholar Universe.
author profiles
.
KOMP Repository.
gene/protein/disease-specific - KOMP Repository
(subscription/membership/fee required).
Mouse Genome Informatics (MGI).
Mouse Genome Informatics (MGI)
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18174160.001).
[ISSN]
0021-9258
[Journal-full-title]
The Journal of biological chemistry
[ISO-abbreviation]
J. Biol. Chem.
[Language]
eng
[Grant]
United States / NIEHS NIH HHS / ES / ES02497; United States / NICHD NIH HHS / HD / HD044863
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
United States
[Chemical-registry-number]
0 / LDL-Receptor Related Proteins; 0 / Low Density Lipoprotein Receptor-Related Protein-2; 0 / Receptors, Lipoprotein; 0 / Selenoprotein P; 0 / low density lipoprotein receptor-related protein 8
80.
Barroso LV, Miranda EP, Cruz NI, Medeiros MA, Araújo AC, Mota Filho FH, Medeiros FC:
Analysis of sexual function in kidney transplanted men.
Transplant Proc
; 2008 Dec;40(10):3489-91
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Analysis of sexual function in
kidney
transplanted men.
INTRODUCTION: Sexual dysfunction among
renal
failure and
kidney
transplant patients remains controversial.
The aim of this study was to evaluate sexual functions of men on hemodialysis compared with patients undergoing
kidney
transplantation.
MATERIALS AND METHODS: Our study was based on 36 end-stage
renal
disease (ESRD) patients undergoing hemodialysis versus 32
kidney
transplanted patients.
CONCLUSION: It was possible to conclude from our study that
kidney
transplants do improve sexual function of patients with ESRD on hemodialysis.
[MeSH-major]
Kidney
Transplantation / physiology. Libido.
Renal
Dialysis
[MeSH-minor]
Adolescent. Adult. Coitus / psychology. Humans.
Kidney
Failure, Chronic / physiopathology.
Kidney
Failure, Chronic / psychology. Male. Middle Aged. Orgasm. Penile Erection / physiology. Personal Satisfaction. Reference Values. Sexual Behavior. Young Adult
MedlinePlus Health Information.
consumer health - Dialysis
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19100420.001).
[ISSN]
0041-1345
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
81.
Sokolova IA, Hes O, Michal M, Matsko DE:
[Small-cell variant of renal oncocytoma].
Arkh Patol
; 2007 Sep-Oct;69(5):34-6
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
[Small-cell variant of
renal
oncocytoma].
Renal
oncocytoma is a distinct
benign
tumor
accounting for approximately 3-5% of all
renal
tumors
.
This
neoplasm
is easily recognizable in its classic form: there are polygonal cells with abundant granular eosinophilic cytoplasm filled with mitochondria.
The tumor
cells are arranged in nests and tubular pattern.
Here, 1 case of
renal
oncocytoma with a domination of small cells is reported (the so-called "oncoblasts") arising in elderly woman.
The term "small-cell variant of
renal
oncocytoma" was proposed for these cases.
The unusual extensive small-cell component of the
tumor
may represent a potential diagnostic pitfall for primary or metastatic malignant small cell
tumors
.
[MeSH-major]
Adenoma, Oxyphilic / pathology. Carcinoma, Small Cell / pathology. Cytoplasm / pathology.
Kidney Neoplasms
/ pathology. Mitochondria / pathology
[MeSH-minor]
Aged. Female. Humans.
Neoplasm
Metastasis
Genetic Alliance.
consumer health - Oncocytoma renal
.
MedlinePlus Health Information.
consumer health - Kidney Cancer
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18074818.001).
[ISSN]
0004-1955
[Journal-full-title]
Arkhiv patologii
[ISO-abbreviation]
Arkh. Patol.
[Language]
rus
[Publication-type]
Case Reports; English Abstract; Journal Article
[Publication-country]
Russia (Federation)
82.
Garg AX, Muirhead N, Knoll G, Yang RC, Prasad GV, Thiessen-Philbrook H, Rosas-Arellano MP, Housawi A, Boudville N, Donor Nephrectomy Outcomes Research (DONOR) Network:
Proteinuria and reduced kidney function in living kidney donors: A systematic review, meta-analysis, and meta-regression.
Kidney Int
; 2006 Nov;70(10):1801-10
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Proteinuria and reduced
kidney
function in living
kidney
donors: A systematic review, meta-analysis, and meta-regression.
We reviewed any study where 10 or more healthy adults donated a
kidney
, and proteinuria, or glomerular filtration rate (GFR) was assessed at least 1 year later.
Kidney
donation results in small increases in urinary protein.
[MeSH-major]
Kidney
Transplantation / adverse effects. Living Donors. Proteinuria / etiology.
Renal
Insufficiency / etiology
[MeSH-minor]
Adolescent. Adult. Aged. Aged, 80 and over. Glomerular Filtration Rate / physiology. Humans. Incidence.
Kidney
/ physiopathology. Middle Aged. Nephrectomy. Outcome Assessment (Health Care). Risk Factors
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Organ Donation
.
COS Scholar Universe.
author profiles
.
ClinicalTrials.gov.
clinical trials - ClinicalTrials.gov
.
PubMed Health.
DARE review
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[CommentIn]
Kidney Int. 2007 May;71(10):1077; author reply 1077
[
17495941.001
]
(PMID = 17003822.001).
[ISSN]
0085-2538
[Journal-full-title]
Kidney international
[ISO-abbreviation]
Kidney Int.
[Language]
eng
[Publication-type]
Journal Article; Meta-Analysis; Research Support, Non-U.S. Gov't; Review
[Publication-country]
United States
[Number-of-references]
86
83.
Ríos A, Conesa C, Ramírez P, Galindo PJ, Fernández OM, Rodríguez MM, Parrilla P:
Attitude survey of hospital workers in the surgical services toward living kidney donation.
Transplant Proc
; 2005 Nov;37(9):3621-5
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Attitude survey of hospital workers in the surgical services toward living
kidney
donation.
INTRODUCTION: A living donor
kidney
is currently the most accepted kind of solid organ donation, given the low level of morbidity and mortality and the good results in the recipient.
Our objective was to analyze the attitudes toward living
kidney
donation in a surgical department.
Attitudes toward living
kidney
donation were evaluated using a questionnaire on donation and transplantation, which evaluated various psychosocial variables.
RESULTS: Two hundred sixty-three respondents of mean age 40 +/- 10 years were analysed for attitudes toward living
kidney
donation.
The attitude toward cadaveric organ donation was not reflective of that toward living
kidney
donation (P = .241).
CONCLUSIONS: A favorable attitude toward living
kidney
donation was high among hospital staff of the surgical department.
Those for whom it was not favorable were influenced by personal factors such as partner's attitude and the possibility of needing a
kidney
in the future.
[MeSH-major]
Kidney
. Living Donors. Personnel, Hospital / psychology. Surgery Department, Hospital
MedlinePlus Health Information.
consumer health - Organ Donation
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16386486.001).
[ISSN]
0041-1345
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
84.
Ghanei E, Miladipour A, Nasrollahi A, Homayuni M:
Brucellosis with kidney failure.
Iran J Kidney Dis
; 2009 Apr;3(2):109-11
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Brucellosis with
kidney
failure.
While involvement of the bones, joints, and liver is not rare, brucellosis may rarely involve
the kidney
.
We present a case of brucellosis with hepatitis, pancytopenia, peripheral arthritis, and
kidney
failure.
[MeSH-major]
Acute
Kidney
Injury / microbiology. Arthritis, Infectious / microbiology. Brucellosis / complications. Hepatitis / microbiology. Pancytopenia / microbiology
Genetic Alliance.
consumer health - Brucellosis
.
MedlinePlus Health Information.
consumer health - Hepatitis
.
MedlinePlus Health Information.
consumer health - Infectious Arthritis
.
Hazardous Substances Data Bank.
DOXYCYCLINE
.
Hazardous Substances Data Bank.
STREPTOMYCIN
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19395788.001).
[ISSN]
1735-8582
[Journal-full-title]
Iranian journal of kidney diseases
[ISO-abbreviation]
Iran J Kidney Dis
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Iran
[Chemical-registry-number]
0 / Anti-Bacterial Agents; N12000U13O / Doxycycline; Y45QSO73OB / Streptomycin
85.
Boutet A, De Frutos CA, Maxwell PH, Mayol MJ, Romero J, Nieto MA:
Snail activation disrupts tissue homeostasis and induces fibrosis in the adult kidney.
EMBO J
; 2006 Nov 29;25(23):5603-13
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Snail activation disrupts tissue homeostasis and induces fibrosis in the adult
kidney
.
During embryonic development,
the kidney
epithelium originates from cells that undergo a mesenchymal to epithelial transition (MET).
The reverse process, epithelium to mesenchyme transition (EMT), has been implicated in epithelial
tumor
progression and in the fibrosis that leads to end-stage
kidney
failure.
Snail transcription factors induce both natural and pathological EMT, but their implication in
renal
development and disease is still unclear.
We show that Snail genes are downregulated during the MET that occurs during
renal
development and that this is correlated with Cadherin-16 expression.
Snail suppresses Cadherin-16 via the direct repression of the
kidney
differentiation factor HNF-1beta, a novel route by which Snail disrupts epithelial homeostasis.
Indeed, Snail activation is sufficient to induce EMT and
kidney
fibrosis in adult transgenic mice.
Significantly, Snail is also activated in patients with
renal
fibrosis.
Thus, Snail expression is suppressed during
renal
development and it must remain silent in the mature
kidney
where its aberrant activation leads to fibrosis.
[MeSH-major]
Gene Expression Regulation, Developmental.
Kidney
/ embryology.
Kidney
/ pathology.
Kidney
Diseases / genetics. Transcription Factors / agonists
MedlinePlus Health Information.
consumer health - Kidney Diseases
.
COS Scholar Universe.
author profiles
.
Gene Ontology.
gene/protein/disease-specific - Gene Ontology annotations from this paper
.
Faculty of 1000.
commentaries/discussion - See the articles recommended by F1000Prime's Faculty of more than 8,000 leading experts in Biology and Medicine.
(subscription/membership/fee required).
KOMP Repository.
gene/protein/disease-specific - KOMP Repository
(subscription/membership/fee required).
Mouse Genome Informatics (MGI).
Mouse Genome Informatics (MGI)
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
The Lens.
Cited by Patents in
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
J Biol Chem. 1995 Jul 21;270(29):17594-601
[
7615566.001
]
[Cites]
Cancer Res. 2004 Aug 15;64(16):5527-34
[
15313885.001
]
[Cites]
Proc Natl Acad Sci U S A. 1996 Oct 1;93(20):10887-90
[
8855277.001
]
[Cites]
Development. 1998 Aug;125(16):3111-21
[
9671584.001
]
[Cites]
Mech Dev. 1999 Jun;84(1-2):185-8
[
10473139.001
]
[Cites]
Am J Physiol. 1999 Oct;277(4 Pt 2):F587-98
[
10516284.001
]
[Cites]
Med Hypotheses. 2005;64(1):201-8
[
15533642.001
]
[Cites]
Curr Opin Rheumatol. 2004 Nov;16(6):733-8
[
15577612.001
]
[Cites]
Biol Reprod. 2005 Apr;72(4):916-21
[
15601922.001
]
[Cites]
Nat Med. 2005 Apr;11(4):387-93
[
15793581.001
]
[Cites]
Am J Transplant. 2005 Jun;5(6):1367-74
[
15888043.001
]
[Cites]
Development. 2005 Jul;132(14):3151-61
[
15983400.001
]
[Cites]
Am J Pathol. 2005 Aug;167(2):395-407
[
16049326.001
]
[Cites]
Curr Opin Cell Biol. 2005 Oct;17(5):548-58
[
16098727.001
]
[Cites]
Nephrol Dial Transplant. 2005 Oct;20(10):2215-25
[
16030052.001
]
[Cites]
Kidney Int. 2005 Dec;68(6):2667-79
[
16316342.001
]
[Cites]
J Med Genet. 2006 Jan;43(1):84-90
[
15930087.001
]
[Cites]
Kidney Int. 2006 Jan;69(2):213-7
[
16408108.001
]
[Cites]
Nephron Exp Nephrol. 2006;102(3-4):e71-5
[
16286786.001
]
[Cites]
Nat Rev Mol Cell Biol. 2006 Feb;7(2):131-42
[
16493418.001
]
[Cites]
Nat Cell Biol. 2000 Feb;2(2):76-83
[
10655586.001
]
[Cites]
Nat Cell Biol. 2000 Feb;2(2):84-9
[
10655587.001
]
[Cites]
Oncogene. 2001 Jun 28;20(29):3814-23
[
11439345.001
]
[Cites]
Am J Kidney Dis. 2001 Oct;38(4):761-9
[
11576879.001
]
[Cites]
Development. 2002 Apr;129(7):1583-93
[
11923196.001
]
[Cites]
Nat Rev Mol Cell Biol. 2002 Mar;3(3):155-66
[
11994736.001
]
[Cites]
Kidney Int. 2002 Jul;62(1):137-46
[
12081572.001
]
[Cites]
Oncogene. 2002 May 9;21(20):3241-6
[
12082640.001
]
[Cites]
J Am Soc Nephrol. 2002 Jul;13(7):1824-36
[
12089378.001
]
[Cites]
J Clin Invest. 2002 Aug;110(3):341-50
[
12163453.001
]
[Cites]
Am J Physiol Renal Physiol. 2002 Oct;283(4):F839-51
[
12217876.001
]
[Cites]
Development. 2003 Feb;130(3):483-94
[
12490555.001
]
[Cites]
Mol Cancer Res. 2002 Nov;1(1):68-78
[
12496370.001
]
[Cites]
J Cell Sci. 2003 Feb 1;116(Pt 3):499-511
[
12508111.001
]
[Cites]
N Engl J Med. 2003 Jan 30;348(5):403-13
[
12556543.001
]
[Cites]
Am J Pathol. 2003 May;162(5):1495-502
[
12707032.001
]
[Cites]
Nat Med. 2003 Jul;9(7):964-8
[
12808448.001
]
[Cites]
J Clin Invest. 2003 Aug;112(4):503-16
[
12925691.001
]
[Cites]
J Clin Invest. 2003 Nov;112(10):1486-94
[
14617750.001
]
[Cites]
J Clin Invest. 2003 Dec;112(12):1776-84
[
14679171.001
]
[Cites]
J Am Soc Nephrol. 2004 Jan;15(1):1-12
[
14694152.001
]
[Cites]
J Mol Med (Berl). 2004 Mar;82(3):175-81
[
14752606.001
]
[Cites]
J Clin Invest. 2004 Mar;113(6):814-25
[
15067314.001
]
[Cites]
EMBO J. 2004 Apr 7;23(7):1657-68
[
15029248.001
]
[Cites]
J Cell Biol. 1995 Jul;130(2):393-405
[
7615639.001
]
(PMID = 17093497.001).
[ISSN]
0261-4189
[Journal-full-title]
The EMBO journal
[ISO-abbreviation]
EMBO J.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / Cadherins; 0 / Cdh16 protein, mouse; 0 / RNA, Messenger; 0 / Transcription Factors; 0 / snail family transcription factors; 138674-15-4 / Hepatocyte Nuclear Factor 1-beta
[Other-IDs]
NLM/ PMC1679761
86.
Gorodetskaya I, Zenios S, McCulloch CE, Bostrom A, Hsu CY, Bindman AB, Go AS, Chertow GM:
Health-related quality of life and estimates of utility in chronic kidney disease.
Kidney Int
; 2005 Dec;68(6):2801-8
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Health-related quality of life and estimates of utility in chronic
kidney
disease.
BACKGROUND: Health-related quality of life and estimates of utility have been carefully evaluated in persons with end-stage
renal
disease.
Fewer studies have examined these parameters in persons with chronic
kidney
disease (CKD).
METHODS: To determine the relations among
kidney
function, health-related quality of life, and estimates of utility, we administered
the Kidney
Disease Quality of Life Short Form 36 (KDQOL-36), Health Utilities Index (HUI)-3, and Time Trade-off (TTO) questionnaires to 205 persons with CKD.
On cross-sectional analysis, lower levels
of kidney
function were associated with significantly lower scores on the SF-12 Physical Health Composite (P= 0.002), the Burden
of Kidney
Disease subscale (P < 0.0001), and the Effects
of Kidney
Disease subscale (P < 0.0001) of the KDQOL-36trade mark.
Kidney
function was significantly associated with the TTO (P= 0.008) and global HUI-3 utility (P= 0.016) although these associations were attenuated after adjustment for diabetes.
A decline in eGFR was associated with a significant increase in the reported Burden
of Kidney
Disease (5.0 point change per year per mL/min/1.73 m2 decline in eGFR) and with marginally significant changes in the Dexterity and Pain attributes of the HUI-3.
[MeSH-major]
Cost of Illness. Health Status. Quality of Life.
Renal
Insufficiency, Chronic / psychology
[MeSH-minor]
Adult. Aged. Aged, 80 and over. Creatinine / blood. Cross-Sectional Studies. Female. Glomerular Filtration Rate. Humans.
Kidney
Failure, Chronic / psychology.
Kidney
Failure, Chronic / therapy. Longitudinal Studies. Male. Middle Aged.
Renal
Dialysis. Surveys and Questionnaires
Genetic Alliance.
consumer health - Kidney Disease
.
MedlinePlus Health Information.
consumer health - Chronic Kidney Disease
.
COS Scholar Universe.
author profiles
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16316356.001).
[ISSN]
0085-2538
[Journal-full-title]
Kidney international
[ISO-abbreviation]
Kidney Int.
[Language]
eng
[Grant]
United States / NIDDK NIH HHS / DK / DK58411; United States / NCRR NIH HHS / RR / M01 RR-00079
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
United States
[Chemical-registry-number]
AYI8EX34EU / Creatinine
87.
Khoury JA, Brennan DC:
Infectious complications in kidney transplant recipients: review of the literature.
Saudi J Kidney Dis Transpl
; 2005 Oct-Dec;16(4):453-97
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Infectious complications in
kidney
transplant recipients: review of the literature.
Since the initial successful
kidney
transplantation in humans, the field of
renal
transplantation has made significant progress.
Enhanced surgical techniques also improved the overall survival
of kidney
recipients.
In this article, we provide an overview of infections in
kidney
transplant recipients, a detailed illustration of specific infectious agents with a focus on cytomegalovirus, and finally we lay some general principles for limiting the burden of infectious complications in
kidney
transplants through proper infection control measures.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18202503.001).
[ISSN]
1319-2442
[Journal-full-title]
Saudi journal of kidney diseases and transplantation : an official publication of the Saudi Center for Organ Transplantation, Saudi Arabia
[ISO-abbreviation]
Saudi J Kidney Dis Transpl
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Saudi Arabia
88.
Dear JW, Yuen PS:
Setting the stage for acute-on-chronic kidney injury.
Kidney Int
; 2008 Jul;74(1):7-9
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Setting the stage for acute-on-chronic
kidney
injury.
Acute-on-chronic
kidney
disease will be familiar to many nephrologists.
Hsu et al. quantify the risk of acute-on-chronic disease across the stages of preexisting chronic
kidney
disease.
Their study demonstrates the valuable insights that large epidemiological studies can bring to the field of acute
kidney
injury.
[MeSH-major]
Acute
Kidney
Injury / epidemiology.
Kidney
Failure, Chronic / epidemiology
MedlinePlus Health Information.
consumer health - Kidney Failure
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Clin J Am Soc Nephrol. 2008 May;3(3):887-94
[
18216348.001
]
[Cites]
Circulation. 2007 Jul 3;116(1):85-97
[
17606856.001
]
[Cites]
JAMA. 2007 Nov 7;298(17):2038-47
[
17986697.001
]
[Cites]
Crit Care. 2007;11(2):R31
[
17331245.001
]
[Cites]
Curr Opin Nephrol Hypertens. 2007 May;16(3):227-36
[
17420666.001
]
[Cites]
J Am Soc Nephrol. 2004 Jul;15(7):1677-89
[
15213255.001
]
[Cites]
Kidney Int. 1998 Dec;54(6):1817-31
[
9853246.001
]
[Cites]
JAMA. 2006 Sep 20;296(11):1377-84
[
16985230.001
]
[CommentOn]
Kidney Int. 2008 Jul;74(1):101-7
[
18385668.001
]
(PMID = 18560361.001).
[ISSN]
1523-1755
[Journal-full-title]
Kidney international
[ISO-abbreviation]
Kidney Int.
[Language]
eng
[Grant]
United States / Intramural NIH HHS / / Z99 DK999999
[Publication-type]
Comment; Journal Article; Review
[Publication-country]
United States
[Number-of-references]
10
[Other-IDs]
NLM/ NIHMS295237; NLM/ PMC3113484
89.
Merkle M, Rupprecht HD:
[Lymphoproliferative disease following kidney transplantation].
Dtsch Med Wochenschr
; 2005 Jul 15;130(28-29):1691-4
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
[Lymphoproliferative disease following
kidney
transplantation].
HISTORY AND CLINICAL FINDINGS: A 31-year-old male patient was referred because of a worsening graft function 56 months after an allogenic
kidney
transplantation for interstitial nephritis.
Correction of volume status did not result in an improvement
of kidney
function.
INVESTIGATIONS:
Kidney
biopsy showed a low degree nephrosclerosis and some interstitial fibrosis, but no signs of rejection.
DIAGNOSIS
: EBV-negative post-transplant lymphoproliferative disease (PTLD).
[MeSH-major]
Antineoplastic Combined Chemotherapy Protocols / therapeutic use. Cyclophosphamide / therapeutic use. Doxorubicin / therapeutic use.
Kidney
Transplantation / adverse effects. Lymphoma, B-Cell / etiology. Precursor Cell Lymphoblastic Leukemia-Lymphoma / etiology. Prednisone / therapeutic use. Vincristine / therapeutic use
[MeSH-minor]
Abdominal Pain. Adult.
Diagnosis
, Differential. Diarrhea. Humans. Immunosuppressive Agents / administration & dosage. Immunosuppressive Agents / adverse effects. Male. Remission Induction. Risk Factors
Genetic Alliance.
consumer health - Kidney Disease
.
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Steroids
.
Hazardous Substances Data Bank.
DOXORUBICIN
.
Hazardous Substances Data Bank.
CYCLOPHOSPHAMIDE
.
Hazardous Substances Data Bank.
PREDNISONE
.
Hazardous Substances Data Bank.
VINCRISTINE
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16003604.001).
[ISSN]
0012-0472
[Journal-full-title]
Deutsche medizinische Wochenschrift (1946)
[ISO-abbreviation]
Dtsch. Med. Wochenschr.
[Language]
ger
[Publication-type]
Case Reports; English Abstract; Journal Article
[Publication-country]
Germany
[Chemical-registry-number]
0 / Immunosuppressive Agents; 5J49Q6B70F / Vincristine; 80168379AG / Doxorubicin; 8N3DW7272P / Cyclophosphamide; VB0R961HZT / Prednisone; CHOP protocol
90.
Smith KD:
Toll-like receptors in kidney disease.
Curr Opin Nephrol Hypertens
; 2009 May;18(3):189-96
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Toll-like receptors in
kidney
disease.
This review focuses on the Toll-like receptors (TLRs) of the innate immune system and their role in recognizing infection and injury, and regulating inflammatory responses in
the kidney
.
Tonic interactions between TLRs and environmental agonists derived from commensal microbes and endogenous sources may also influence autoimmune disease and inflammatory disorders affecting
the kidney
.
CONCLUSION: Future studies to decipher the contribution of TLRs and other innate immune receptors in the regulation of inflammation, immune responses, and injury in
the kidney
will pave the way for novel therapeutic interventions.
Genetic Alliance.
consumer health - Kidney Disease
.
MedlinePlus Health Information.
consumer health - Kidney Diseases
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Science. 2000 Sep 29;289(5488):2350-4
[
11009421.001
]
[Cites]
Nature. 2000 Dec 7;408(6813):740-5
[
11130078.001
]
[Cites]
Nature. 2001 Apr 26;410(6832):1099-103
[
11323673.001
]
[Cites]
Int Immunol. 2001 Jul;13(7):933-40
[
11431423.001
]
[Cites]
Nature. 2001 Oct 18;413(6857):732-8
[
11607032.001
]
[Cites]
J Immunol. 2002 Jul 1;169(1):10-4
[
12077222.001
]
[Cites]
J Am Soc Nephrol. 2003 Feb;14(2):317-26
[
12538732.001
]
[Cites]
J Biol Chem. 2004 Mar 26;279(13):12542-50
[
14729660.001
]
[Cites]
Kidney Int. 2004 Aug;66(2):480-5
[
15253693.001
]
[Cites]
Cell. 2004 Jul 23;118(2):229-41
[
15260992.001
]
[Cites]
J Leukoc Biol. 2004 Sep;76(3):514-9
[
15178705.001
]
[Cites]
Kidney Int Suppl. 2004 Oct;(91):S56-61
[
15461705.001
]
[Cites]
Science. 1998 Dec 11;282(5396):2085-8
[
9851930.001
]
[Cites]
J Exp Med. 2005 Jan 3;201(1):19-25
[
15630134.001
]
[Cites]
J Clin Invest. 2005 Feb;115(2):468-75
[
15650774.001
]
[Cites]
Science. 2005 Jun 10;308(5728):1626-9
[
15860593.001
]
[Cites]
Proc Natl Acad Sci U S A. 2005 Jul 5;102(27):9577-82
[
15976025.001
]
[Cites]
J Clin Invest. 2005 Aug;115(8):2223-33
[
16025156.001
]
[Cites]
J Clin Invest. 2005 Oct;115(10):2894-903
[
16167081.001
]
[Cites]
J Immunol. 2006 Jan 1;176(1):632-9
[
16365459.001
]
[Cites]
Am J Pathol. 2006 Feb;168(2):370-85
[
16436653.001
]
[Cites]
Am J Physiol Cell Physiol. 2006 Mar;290(3):C917-24
[
16267105.001
]
[Cites]
Nature. 2006 Mar 9;440(7081):237-41
[
16407889.001
]
[Cites]
Nature. 2006 Mar 9;440(7081):228-32
[
16407890.001
]
[Cites]
Immunity. 2006 Sep;25(3):417-28
[
16973389.001
]
[Cites]
J Immunol. 2006 Nov 15;177(10):6880-8
[
17082602.001
]
[Cites]
Rev Med Virol. 2007 Jan-Feb;17(1):35-43
[
17146842.001
]
[Cites]
J Immunol. 2007 Apr 15;178(8):5124-31
[
17404295.001
]
[Cites]
Semin Immunol. 2007 Feb;19(1):3-10
[
17275324.001
]
[Cites]
Nat Immunol. 2007 May;8(5):487-96
[
17417641.001
]
[Cites]
J Immunol. 2007 May 15;178(10):6252-8
[
17475853.001
]
[Cites]
J Endotoxin Res. 2007;13(1):6-14
[
17621541.001
]
[Cites]
J Leukoc Biol. 2007 Sep;82(3):479-87
[
17595379.001
]
[Cites]
Cell. 2007 Sep 7;130(5):906-17
[
17803912.001
]
[Cites]
Cell. 2007 Sep 21;130(6):1071-82
[
17889651.001
]
[Cites]
J Clin Invest. 2007 Oct;117(10):2847-59
[
17853945.001
]
[Cites]
J Exp Med. 2007 Oct 1;204(10):2407-22
[
17893200.001
]
[Cites]
Immunol Rev. 2007 Dec;220:47-59
[
17979839.001
]
[Cites]
Immunity. 2007 Nov;27(5):801-10
[
17997333.001
]
[Cites]
J Viral Hepat. 2008 Jan;15(1):71-8
[
18088248.001
]
[Cites]
J Exp Med. 2008 Feb 18;205(2):451-64
[
18268035.001
]
[Cites]
Curr Opin Immunol. 2008 Feb;20(1):17-22
[
18272355.001
]
[Cites]
J Immunol. 2008 Apr 1;180(7):5067-74
[
18354232.001
]
[Cites]
Curr Opin Microbiol. 2008 Feb;11(1):66-73
[
18243043.001
]
[Cites]
Nat Rev Immunol. 2008 Apr;8(4):279-89
[
18340345.001
]
[Cites]
Nat Med. 2008 Apr;14(4):399-406
[
18327267.001
]
[Cites]
Immunobiology. 2008;213(3-4):205-24
[
18406368.001
]
[Cites]
Science. 2008 Apr 18;320(5874):379-81
[
18420935.001
]
[Cites]
Arthritis Rheum. 2008 Apr;58(4):1107-15
[
18383384.001
]
[Cites]
J Am Soc Nephrol. 2008 May;19(5):923-32
[
18256356.001
]
[Cites]
Eur J Immunol. 2008 Jul;38(7):1971-8
[
18521959.001
]
[Cites]
Science. 2008 Aug 1;321(5889):691-6
[
18669862.001
]
[Cites]
J Exp Med. 2008 Aug 4;205(8):1879-88
[
18644972.001
]
[Cites]
Curr Opin Rheumatol. 2008 Sep;20(5):538-44
[
18698174.001
]
[Cites]
Immunity. 2008 Aug 15;29(2):272-82
[
18656388.001
]
[Cites]
Immunity. 2008 Aug 15;29(2):249-60
[
18691914.001
]
[Cites]
Cell Host Microbe. 2008 Sep 11;4(3):198-208
[
18779046.001
]
[Cites]
Nature. 2008 Oct 23;455(7216):1109-13
[
18806780.001
]
[Cites]
PLoS One. 2008;3(10):e3596
[
18974879.001
]
[Cites]
Immunity. 2008 Nov 14;29(5):746-57
[
19006693.001
]
[Cites]
J Exp Med. 2008 Dec 22;205(13):2995-3006
[
19047436.001
]
[Cites]
Nat Genet. 2008 Sep;40(9):1062-4
[
19165919.001
]
[Cites]
J Immunol. 2000 Jan 15;164(2):554-7
[
10623793.001
]
(PMID = 19352178.001).
[ISSN]
1473-6543
[Journal-full-title]
Current opinion in nephrology and hypertension
[ISO-abbreviation]
Curr. Opin. Nephrol. Hypertens.
[Language]
ENG
[Grant]
United States / NIAID NIH HHS / AI / AI062859-04; United States / NIAID NIH HHS / AI / R01 AI062859; United States / NIAID NIH HHS / AI / R01 AI062859-04
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Review
[Publication-country]
England
[Chemical-registry-number]
0 / Toll-Like Receptors
[Number-of-references]
63
[Other-IDs]
NLM/ NIHMS202846; NLM/ PMC2896868
91.
Fried LF, Biggs ML, Shlipak MG, Seliger S, Kestenbaum B, Stehman-Breen C, Sarnak M, Siscovick D, Harris T, Cauley J, Newman AB, Robbins J:
Association of kidney function with incident hip fracture in older adults.
J Am Soc Nephrol
; 2007 Jan;18(1):282-6
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Association
of kidney
function with incident hip fracture in older adults.
Kidney
dysfunction is associated with bone loss, and patients with ESRD have an increased risk for hip fracture.
However, the association of mild to moderate
kidney
disease with hip fracture has not been studied previously.
The association
of kidney
function with incident hip fracture was examined among participants in the Cardiovascular Health Study, a community-based cohort of older individuals.
The primary measure
of kidney
function was serum cystatin C, a measure that does not depend on lean mass.
The association
of kidney
function with hip fracture was analyzed with Cox proportional hazards models.
Kidney
dysfunction, as assessed by cystatin C, is associated with an increased risk for hip fracture.
[MeSH-major]
Hip Fractures / physiopathology.
Kidney
/ physiopathology
[MeSH-minor]
Aged. Aged, 80 and over. Cystatin C. Cystatins / blood. Female. Humans.
Kidney
Failure, Chronic / complications.
Kidney
Failure, Chronic / physiopathology. Longitudinal Studies. Male. Osteoporosis / complications. Osteoporosis / physiopathology. Prospective Studies. Risk Factors. Sex Factors
MedlinePlus Health Information.
consumer health - Hip Injuries and Disorders
.
COS Scholar Universe.
author profiles
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17167115.001).
[ISSN]
1046-6673
[Journal-full-title]
Journal of the American Society of Nephrology : JASN
[ISO-abbreviation]
J. Am. Soc. Nephrol.
[Language]
eng
[Grant]
United States / NHLBI NIH HHS / HC / N01-HC-15103; United States / NHLBI NIH HHS / HC / N01-HC-35129; United States / NHLBI NIH HHS / HC / N01-HC-85079; United States / NHLBI NIH HHS / HC / N01-HC-85086
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
[Publication-country]
United States
[Chemical-registry-number]
0 / CST3 protein, human; 0 / Cystatin C; 0 / Cystatins
92.
Rubera I, Hummler E, Beermann F:
Transgenic mice and their impact on kidney research.
Pflugers Arch
; 2009 May;458(1):211-22
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Transgenic mice and their impact on
kidney
research.
The kidney
is a key organ in the maintenance of ion and fluid homeostasis and specific transport systems localized along the nephron guarantee this function.
This review introduces the transgenic technology developed over the past decades, and then focuses on recent strategies for generating
kidney
-specific gene targeting, over-expression, and gene ablation in mice, that will help to understand the physiological role of proteins implicated in salt and water balance in
the kidney
.
[MeSH-major]
Kidney
/ physiology. Mice, Transgenic
[MeSH-minor]
Animals. Aquaporin 2 / genetics. Disease Models, Animal. Embryonic Stem Cells / transplantation. Epithelial Sodium Channels / genetics. Genetic Engineering. Genetic Vectors / physiology. Humans. Integrases / metabolism.
Kidney
Diseases / genetics.
Kidney
Diseases / physiopathology. Lentivirus / genetics. Mice. Mice, Knockout. Transgenes / physiology
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Nat Med. 2005 Apr;11(4):423-8
[
15768028.001
]
[Cites]
Proc Natl Acad Sci U S A. 1999 Feb 16;96(4):1732-7
[
9990093.001
]
[Cites]
Proc Natl Acad Sci U S A. 2006 Apr 11;103(15):6037-42
[
16581908.001
]
[Cites]
Nat Genet. 1999 Jan;21(1):70-1
[
9916792.001
]
[Cites]
J Am Soc Nephrol. 2002 Mar;13(3):788-93
[
11856786.001
]
[Cites]
Am J Physiol Renal Physiol. 2003 Aug;285(2):F310-8
[
12684224.001
]
[Cites]
J Am Soc Nephrol. 2000 Nov;11 Suppl 16:S129-34
[
11065344.001
]
[Cites]
Genesis. 2000 Feb;26(2):99-109
[
10686599.001
]
[Cites]
Am J Physiol Renal Physiol. 2004 May;286(5):F965-71
[
15075192.001
]
[Cites]
Transgenic Res. 2004 Dec;13(6):513-22
[
15672832.001
]
[Cites]
Proc Natl Acad Sci U S A. 1986 Dec;83(23):9065-9
[
3024164.001
]
[Cites]
Proc Natl Acad Sci U S A. 2005 Jun 28;102(26):9406-11
[
15956187.001
]
[Cites]
J Clin Invest. 2003 Aug;112(4):554-65
[
12925696.001
]
[Cites]
J Am Soc Nephrol. 2003 Sep;14(9):2219-28
[
12937297.001
]
[Cites]
Cell. 1981 Nov;27(1 Pt 2):223-31
[
6276022.001
]
[Cites]
Genesis. 2004 Jul;39(3):206-11
[
15282747.001
]
[Cites]
J Biol Chem. 2004 Nov 12;279(46):48048-54
[
15347663.001
]
[Cites]
J Cell Sci. 1986 Mar;81:143-62
[
3733894.001
]
[Cites]
Nat Genet. 1996 Oct;14(2):121-3
[
8841174.001
]
[Cites]
J Clin Invest. 2007 Dec;117(12 ):3810-20
[
18037992.001
]
[Cites]
Genesis. 2002 Feb;32(2):169-72
[
11857811.001
]
[Cites]
PLoS Genet. 2007 Dec;3(12):e232
[
18159948.001
]
[Cites]
Adv Genet. 1987;24:285-322
[
3324700.001
]
[Cites]
J Virol. 2008 Jul;82(14):7111-9
[
18463153.001
]
[Cites]
Nephron Exp Nephrol. 2007;106(1):e11-20
[
17356303.001
]
[Cites]
Am J Physiol. 1998 Jul;275(1 Pt 1):C216-26
[
9688853.001
]
[Cites]
Proc Natl Acad Sci U S A. 1991 Mar 15;88(6):2065-9
[
1848692.001
]
[Cites]
J Am Soc Nephrol. 2007 Jun;18(6):1679-87
[
17475815.001
]
[Cites]
Biochem J. 2002 Jul 1;365(Pt 1):7-11
[
11982485.001
]
[Cites]
Proc Natl Acad Sci U S A. 1988 Feb;85(3):836-40
[
3422466.001
]
[Cites]
Genesis. 2007 Oct;45(10):618-24
[
17941042.001
]
[Cites]
Kidney Int. 2006 Mar;69(6):1016-23
[
16528251.001
]
[Cites]
Transgenic Res. 1999 Feb;8(1):1-8
[
10399363.001
]
[Cites]
Pigment Cell Res