BioMedLib Search Engine
[ goto HOMEPAGE ]
Find more relevant answers, faster!
Skip to content
Advanced Search
Search History
MeSH Query
Page Format
Query is expanded
Login
Skip to content
Export Citations
Search Results
RSS
Email
Articles' Details
Start new query
Reset All
Refine your query
(more in Advanced-Search):
Search all of MEDLINE
Focus on the recent 5 years
Focus on the current year
Focus on the last 30 days
More choices ...
Focus on articles with free fulltexts
More choices ...
Do simple 'keyword' search (no query expansion)
[X] Close
You are about to erase all the values you have customized, search history, page format, etc.
Click
here to
RESET
all values
Click
here to
GO BACK
without resetting any value
Advanced Search
Submit one or more of the following items, and they will be searched along with your query in the search box above.
Any submit button will submit all of the items you have changed.
+
Publication-Date
Published in the last:
30 days
60 days
90 days
6 months
12 months
this year
2 years
3 years
5 years
10 years
Or published in the following date range: From (yyyy/mm/dd - month and day are optional)
to ('to' is optional)
+
Full Text
Retrieve articles with hyperlinks to:
full text (either free or subscription)
free full text
subscription full text
no full text link
+
Sort-Order
Sort the retrieved articles by:
relevance
publication date
+
Language
And with languages:
English
French
German
Italian
Japanese
Russian
Spanish
More languages:
Afrikaans
Albanian
Amharic
Arabic
Armenian
Azerbaijani
Bengali
Bosnian
Bulgarian
Catalan
Chinese
Czech
Danish
Dutch
Esperanto
Estonian
Finnish
Georgian
Scottish Gaelic
Greek, Modern
Hebrew
Hindi
Hungarian
Icelandic
Indonesian
Kinyarwanda
Korean
Latin
Latvian
Lithuanian
Macedonian
Malayalam
Maori
Malay
Multiple languages
Norwegian
Persian
Polish
Portuguese
Pushto
Romanian
Sanskrit
Serbian
Croatian
Slovak
Slovenian
Swedish
Thai
Turkish
Ukrainian
Undetermined
Urdu
Vietnamese
Welsh
+
Publication-Type
And with publication types:
Clinical Trial
Editorial
Letter
Meta-Analysis
Practice Guideline
Randomized Controlled Trial
Review
More publication types:
Addresses
Bibliography
Biography
Case Reports
Classical Article
Clinical Conference
Clinical Trial, Phase I
Clinical Trial, Phase II
Clinical Trial, Phase III
Clinical Trial, Phase IV
Comment
Comparative Study
Congresses
Consensus Development Conference
Consensus Development Conference, NIH
Controlled Clinical Trial
Corrected and Republished Article
Dictionary
Directory
Duplicate Publication
English Abstract
Evaluation Studies
Festschrift
Government Publications
Guideline
Historical Article
Interactive Tutorial
Interview
Introductory Journal Article
In Vitro
Journal Article
Lectures
Legal Cases
Legislation
Multicenter Study
News
Newspaper Article
Overall
Patient Education Handout
Periodical Index
Portraits
Published Erratum
Retracted Publication
Research Support, N.I.H., Extramural
Research Support, N.I.H., Intramural
Research Support, Non-U.S. Gov't
Research Support, U.S. Gov't, Non-P.H.S.
Research Support, U.S. Gov't, P.H.S.
Retraction of Publication
Scientific Integrity Review
Technical Report
Twin Study
Validation Studies
+
Species
And for:
Humans
Animals
+
Gender
And for:
Male
Female
+
Age
And for these age groups:
Newborn: birth to 1 month
Infant: 1 to 23 months
Preschool child: 2 to 5 years
Child: 6 to 12 years
Adolescent: 13 to 18 years
Adult: 19 to 44 years
Middle aged: 45 to 64 years
Aged: 65+ years
80 and over: 80+ years
+
Title
And for this query matching the titles:
+
Transliterated-Title
And for this query matching the title in original language:
+
Abstract
And for this query matching the abstratcs:
+
Major-Mesh
And for this query matching the MeSH-Major terms:
+
Mesh
And for this query matching any MeSH terms:
+
Journal
And for one or more of these journal abbreviated names:
OR
OR
(see
title abbreviations
)
+
Volume
And with journal volume number:
+
Issue
And with journal issue number:
+
Page
And with page number:
+
ISSN
And with ISSN:
+
Publication-Place
And with journal's country of publication:
+
Author
And for...
all these author names:
AND
AND
(see
help
)
one or more of these author names:
OR
OR
but not having any of these unwanted author names:
NOT
NOT
+
Affiliation
And with affiliation to:
+
Has-Abstract
Find MEDLINE records with the abstract status:
has abstract
does not have abstract
include both record types
include both record types but rank higher the records having abstract (the default BML behavior)
+
PMID
Show me only articles for these PMIDs (PubMed IDs):
+
Semantic-Type
And with semantic types:
A. Entity
A1. Physical Object
A1.1. Organism
A1.1.1. Archaeon
A1.1.2. Bacterium
A1.1.3. Eukaryote
A1.1.3.1. Animal
A1.1.3.1.1. Vertebrate
A1.1.3.1.1.1. Amphibian
A1.1.3.1.1.2. Bird
A1.1.3.1.1.3. Fish
A1.1.3.1.1.4. Mammal
A1.1.3.1.1.4.1. Human
A1.1.3.1.1.5. Reptile
A1.1.3.2. Fungus
A1.1.3.3. Plant
A1.1.4. Virus
A1.2. Anatomical Structure
A1.2.1. Embryonic Structure
A1.2.2. Anatomical Abnormality
A1.2.2.1. Congenital Abnormality
A1.2.2.2. Acquired Abnormality
A1.2.3. Fully Formed Anatomical Structure
A1.2.3.1. Body Part, Organ, or Organ Component
A1.2.3.2. Tissue
A1.2.3.3. Cell
A1.2.3.4. Cell Component
A1.2.3.5. Gene or Genome
A1.3. Manufactured Object
A1.3.1. Medical Device
A1.3.1.1. Drug Delivery Device
A1.3.2. Research Device
A1.3.3. Clinical Drug
A1.4. Substance
A1.4.1. Chemical
A1.4.1.1. Chemical Viewed Functionally
A1.4.1.1.1. Pharmacologic Substance
A1.4.1.1.1.1. Antibiotic
A1.4.1.1.2. Biomedical or Dental Material
A1.4.1.1.3. Biologically Active Substance
A1.4.1.1.3.1. Neuroreactive Substance or Biogenic Amine
A1.4.1.1.3.2. Hormone
A1.4.1.1.3.3. Enzyme
A1.4.1.1.3.4. Vitamin
A1.4.1.1.3.5. Immunologic Factor
A1.4.1.1.3.6. Receptor
A1.4.1.1.4. Indicator, Reagent, or Diagnostic Aid
A1.4.1.1.5. Hazardous or Poisonous Substance
A1.4.1.2. Chemical Viewed Structurally
A1.4.1.2.1. Organic Chemical
A1.4.1.2.1.5. Nucleic Acid, Nucleoside, or Nucleotide
A1.4.1.2.1.6. Organophosphorus Compound
A1.4.1.2.1.7. Amino Acid, Peptide, or Protein
A1.4.1.2.1.8. Carbohydrate
A1.4.1.2.1.9. Lipid
A1.4.1.2.1.9.1. Steroid
A1.4.1.2.1.9.2. Eicosanoid
A1.4.1.2.2. Inorganic Chemical
A1.4.1.2.3. Element, Ion, or Isotope
A1.4.2. Body Substance
A1.4.3. Food
A2. Conceptual Entity
A2.1. Idea or Concept
A2.1.1. Temporal Concept
A2.1.2. Qualitative Concept
A2.1.3. Quantitative Concept
A2.1.4. Functional Concept
A2.1.4.1. Body System
A2.1.5. Spatial Concept
A2.1.5.1. Body Space or Junction
A2.1.5.2. Body Location or Region
A2.1.5.3. Molecular Sequence
A2.1.5.3.1. Nucleotide Sequence
A2.1.5.3.2. Amino Acid Sequence
A2.1.5.3.3. Carbohydrate Sequence
A2.1.5.4. Geographic Area
A2.2. Finding
A2.2.1. Laboratory or Test Result
A2.2.2. Sign or Symptom
A2.3. Organism Attribute
A2.3.1. Clinical Attribute
A2.4. Intellectual Product
A2.4.1. Classification
A2.4.2. Regulation or Law
A2.5. Language
A2.6. Occupation or Discipline
A2.6.1. Biomedical Occupation or Discipline
A2.7. Organization
A2.7.1. Health Care Related Organization
A2.7.2. Professional Society
A2.7.3. Self-help or Relief Organization
A2.8. Group Attribute
A2.9. Group
A2.9.1. Professional or Occupational Group
A2.9.2. Population Group
A2.9.3. Family Group
A2.9.4. Age Group
A2.9.5. Patient or Disabled Group
B. Event
B1. Activity
B1.1. Behavior
B1.1.1. Social Behavior
B1.1.2. Individual Behavior
B1.2. Daily or Recreational Activity
B1.3. Occupational Activity
B1.3.1. Health Care Activity
B1.3.1.1. Laboratory Procedure
B1.3.1.2. Diagnostic Procedure
B1.3.1.3. Therapeutic or Preventive Procedure
B1.3.2. Research Activity
B1.3.2.1. Molecular Biology Research Technique
B1.3.3. Governmental or Regulatory Activity
B1.3.4. Educational Activity
B1.4. Machine Activity
B2. Phenomenon or Process
B2.1. Human-caused Phenomenon or Process
B2.1.1. Environmental Effect of Humans
B2.2. Natural Phenomenon or Process
B2.2.1. Biologic Function
B2.2.1.1. Physiologic Function
B2.2.1.1.1. Organism Function
B2.2.1.1.1.1. Mental Process
B2.2.1.1.2. Organ or Tissue Function
B2.2.1.1.3. Cell Function
B2.2.1.1.4. Molecular Function
B2.2.1.1.4.1. Genetic Function
B2.2.1.2. Pathologic Function
B2.2.1.2.1. Disease or Syndrome
B2.2.1.2.1.1. Mental or Behavioral Dysfunction
B2.2.1.2.1.2. Neoplastic Process
B2.2.1.2.2. Cell or Molecular Dysfunction
B2.2.1.2.3. Experimental Model of Disease
B2.3. Injury or Poisoning
Page Format
Any submit button will submit all of the items you have changed.
[theme]
Use this page design theme:
original
twenty ten
[shown]
Results per page:
5
10
20
50
100
200
500
[expand/collapse]
show these sections expanded by default:
Advanced search
MeSH query
Search history
Page format
Query expansion
Articles details
Export citations
Email
[text size]
use this font size for text:
25%
50%
75%
100%
125%
150%
200%
or enter your choice of font size:
[page width]
use this page width (relative to the default initial value):
25%
50%
75%
100%
125%
150%
200%
or enter your choice of page width:
[highlight color]
use this color to highlight query words in the articles:
red
green
blue
black
purple
yellow
orange
navy
olive
maroon
none
[query history]
maximum number of queries shown in the history section:
[annotate]
Annotate these parts of each article:
title
abstract
both
none
Reset all values
Find best MeSH terms for
Search History
1
benign pituitary neoplasm 2005:2010[pubdate] *count=100
1458 results
Searchbox
Export
PDF
RSS
Email
Delete
Email this search result to the following email address:
[X] Close
Expand the query
'
benign pituitary neoplasm
' expanded to all its synonyms;
details
Email the results to the following email address:
Export the checked citations in RIS format (RIS format is used by RefWorks, Endnote, among others).
Items 1 to 100 of about 1458
1.
Sheehan MT, Islam R:
Silent thyroiditis, isolated corticotropin deficiency, and alopecia universalis in a patient with ulcerative colitis and elevated levels of plasma factor VIII: an unusual case of autoimmune polyglandular syndrome type 3.
Endocr Pract
; 2009 Mar;15(2):138-42
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
His ACTH level was low, 21-hydroxylase antibodies were not present, and further testing demonstrated otherwise intact
pituitary
function.
Magnetic resonance imaging of his
pituitary
gland
showed normal findings.
[MeSH-major]
Adrenal Insufficiency /
diagnosis
. Alopecia Areata /
diagnosis
. Colitis, Ulcerative /
diagnosis
. Factor VIII / metabolism. Polyendocrinopathies, Autoimmune / complications. Polyendocrinopathies, Autoimmune / pathology. Thyroiditis / pathology
Genetic Alliance.
consumer health - Ulcerative Colitis
.
Genetic Alliance.
consumer health - Alopecia universalis
.
Genetic Alliance.
consumer health - Autoimmune Polyglandular Syndrome Type 3
.
MedlinePlus Health Information.
consumer health - Ulcerative Colitis
.
The Weizmann Institute of Science GeneCards and MalaCards databases.
gene/protein/disease-specific - MalaCards for alopecia universalis
.
The Weizmann Institute of Science GeneCards and MalaCards databases.
gene/protein/disease-specific - MalaCards for autoimmune polyglandular syndrome type 3
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19289325.001).
[ISSN]
1934-2403
[Journal-full-title]
Endocrine practice : official journal of the American College of Endocrinology and the American Association of Clinical Endocrinologists
[ISO-abbreviation]
Endocr Pract
[Language]
eng
[Publication-type]
Case Reports; Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
United States
[Chemical-registry-number]
9001-27-8 / Factor VIII
[Number-of-references]
22
2.
Kouadjo KE, Nishida Y, Cadrin-Girard JF, Yoshioka M, St-Amand J:
Housekeeping and tissue-specific genes in mouse tissues.
BMC Genomics
; 2007;8:127
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
The results reveal several tissue-specific genes highly expressed in testis and
pituitary
gland
.
SciCrunch.
ArrayExpress: Data: Microarray
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Annu Rev Biochem. 1981;50:465-95
[
6267989.001
]
[Cites]
Nat Genet. 1999 Dec;23(4):387-8
[
10581018.001
]
[Cites]
Genomics. 1999 Dec 15;62(3):537-9
[
10644455.001
]
[Cites]
Biochemistry. 2000 Jul 18;39(28):8291-7
[
10889038.001
]
[Cites]
Genome Res. 2000 Jul;10(7):1051-60
[
10899154.001
]
[Cites]
Physiol Genomics. 2000 Apr 27;2(3):143-7
[
11015593.001
]
[Cites]
FASEB J. 2001 Mar;15(3):684-92
[
11259386.001
]
[Cites]
Mol Reprod Dev. 2004 Jun;68(2):142-8
[
15095334.001
]
[Cites]
Science. 2004 Jun 18;304(5678):1815-9
[
15118123.001
]
[Cites]
J Biol Chem. 2004 Jul 9;279(28):29761-6
[
15131127.001
]
[Cites]
J Biol Chem. 2004 Aug 27;279(35):37079-86
[
15226296.001
]
[Cites]
J Exp Med. 1971 Oct 1;134(4):907-34
[
4106490.001
]
[Cites]
Nucleic Acids Res. 2005;33(3):e26
[
15716308.001
]
[Cites]
Biotechniques. 2005 Feb;38(2):287-93
[
15727135.001
]
[Cites]
EMBO J. 1987 Dec 1;6(12):3711-7
[
3428272.001
]
[Cites]
Genes Dev. 1987 Dec;1(10):1075-84
[
3123313.001
]
[Cites]
Dev Biol. 1990 Apr;138(2):443-53
[
1690676.001
]
[Cites]
Biochem Biophys Res Commun. 1991 Jan 31;174(2):417-23
[
1704220.001
]
[Cites]
J Biol Chem. 1991 Sep 5;266(25):16903-10
[
1840592.001
]
[Cites]
Proc Natl Acad Sci U S A. 1992 Sep 1;89(17):8215-9
[
1518849.001
]
[Cites]
J Biol Chem. 1992 Oct 15;267(29):21193-9
[
1400430.001
]
[Cites]
Endocr Rev. 1992 Aug;13(3):476-98
[
1425484.001
]
[Cites]
Biochem J. 1992 Dec 1;288 ( Pt 2):545-51
[
1463458.001
]
[Cites]
Biochem J. 1993 Dec 15;296 ( Pt 3):571-6
[
8280054.001
]
[Cites]
J Neurosci. 1994 Sep;14(9):5223-35
[
8083732.001
]
[Cites]
Mol Biol Rep. 1994 May;19(3):161-70
[
7969104.001
]
[Cites]
J Neurosci. 1995 Mar;15(3 Pt 2):2471-81
[
7891182.001
]
[Cites]
Science. 1995 Oct 20;270(5235):484-7
[
7570003.001
]
[Cites]
Eur J Biochem. 1995 Sep 15;232(3):789-97
[
7588717.001
]
[Cites]
Gene. 1996 Mar 9;169(2):241-5
[
8647455.001
]
[Cites]
Am J Obstet Gynecol. 1997 Feb;176(2):452-6
[
9065197.001
]
[Cites]
Arch Biochem Biophys. 1997 Sep 1;345(1):171-4
[
9281325.001
]
[Cites]
Prostate. 1998 Apr 1;35(1):18-26
[
9537595.001
]
[Cites]
Am J Respir Cell Mol Biol. 1998 Aug;19(2):177-201
[
9698590.001
]
[Cites]
Genome Res. 1999 May;9(5):506-13
[
10330131.001
]
[Cites]
J Mol Endocrinol. 2004 Oct;33(2):429-44
[
15525599.001
]
[Cites]
Proc Natl Acad Sci U S A. 2004 Nov 23;101(47):16501-6
[
15546993.001
]
[Cites]
Haematologica. 2004 Dec;89(12):1428-33
[
15590391.001
]
[Cites]
FEBS Lett. 2005 Jan 17;579(2):295-301
[
15642335.001
]
[Cites]
BMC Mol Biol. 2005;6:4
[
15720708.001
]
[Cites]
Proc Natl Acad Sci U S A. 2005 Mar 15;102(11):4057-62
[
15753291.001
]
[Cites]
Genomics. 2005 Jun;85(6):679-87
[
15885495.001
]
[Cites]
Biotechniques. 2005 May;38(5):739-45
[
15948292.001
]
[Cites]
J Biol Chem. 2001 Aug 24;276(34):31567-74
[
11399755.001
]
[Cites]
Physiol Genomics. 2001 Dec 21;7(2):95-6
[
11773595.001
]
[Cites]
Physiol Genomics. 2001 Dec 21;7(2):97-104
[
11773596.001
]
[Cites]
J Clin Invest. 2002 Jan;109(1):51-8
[
11781350.001
]
[Cites]
Dev Dyn. 2002 Jan;223(1):59-69
[
11803570.001
]
[Cites]
Biotechniques. 2002 Apr;32(4):776-8, 780, 782
[
11962599.001
]
[Cites]
Hum Mol Genet. 2002 Apr 15;11(8):937-43
[
11971875.001
]
[Cites]
Genomics. 2002 Jun;79(6):799-808
[
12036294.001
]
[Cites]
Mech Dev. 2002 Sep;117(1-2):293-8
[
12204273.001
]
[Cites]
Biol Reprod. 2002 Dec;67(6):1708-18
[
12444044.001
]
[Cites]
Am J Respir Crit Care Med. 2002 Dec 1;166(11):1498-509
[
12406855.001
]
[Cites]
Science. 2002 Dec 20;298(5602):2388-90
[
12493914.001
]
[Cites]
Life Sci. 2003 Feb 28;72(15):1695-704
[
12559391.001
]
[Cites]
Nat Genet. 2003 Mar;33 Suppl:245-54
[
12610534.001
]
[Cites]
J Cell Biochem. 2003 Apr 1;88(5):999-1011
[
12616537.001
]
[Cites]
Arch Biochem Biophys. 2003 Jun 1;414(1):91-100
[
12745259.001
]
[Cites]
FASEB J. 2003 Jul;17(10):1313-5
[
12738806.001
]
[Cites]
Cell Biol Int. 2003;27(8):611-24
[
12867153.001
]
[Cites]
Mol Endocrinol. 2003 Oct;17(10):1910-20
[
12869589.001
]
[Cites]
Dev Biol. 2004 Mar 1;267(1):203-15
[
14975727.001
]
[Cites]
Mol Biol Evol. 2004 Feb;21(2):236-9
[
14595094.001
]
[Cites]
Obes Res. 2005 Jun;13(6):1024-30
[
15976145.001
]
[Cites]
BMC Bioinformatics. 2005;6:126
[
15918906.001
]
[Cites]
Proc Natl Acad Sci U S A. 2005 Dec 27;102(52):19168-73
[
16357203.001
]
[Cites]
Clin Cancer Res. 2006 Mar 1;12(5):1420-30
[
16533764.001
]
[Cites]
Physiol Genomics. 2006 Mar 13;25(1):96-104
[
16368873.001
]
[Cites]
J Mol Endocrinol. 2006 Apr;36(2):247-59
[
16595697.001
]
[Cites]
Biol Reprod. 2006 Sep;75(3):462-8
[
16707773.001
]
(PMID = 17519037.001).
[ISSN]
1471-2164
[Journal-full-title]
BMC genomics
[ISO-abbreviation]
BMC Genomics
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Other-IDs]
NLM/ PMC1888706
3.
Ghirardello S, Garrè ML, Rossi A, Maghnie M:
The diagnosis of children with central diabetes insipidus.
J Pediatr Endocrinol Metab
; 2007 Mar;20(3):359-75
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
The diagnosis
of children with central diabetes insipidus.
Proper etiological
diagnosis
can be achieved via a series of steps that start with clinical observations and then progress, as needed, to more sophisticated methods.
Indeed, magnetic resonance imaging (MRI) represents the examination method of choice for evaluating hypothalamic-
pituitary
-related endocrine diseases due to its ability to provide strongly-contrasted high-resolution multi-planar and spatial images.
Specifically, MRI allows a detailed and precise anatomical study of the
pituitary
gland
by differentiating between the anterior and posterior
pituitary
lobes.
MRI identification of
pituitary
hyperintensity in the posterior part of the sella, now considered to be a clear marker of neurohypophyseal functional integrity, together with careful analysis of
pituitary
stalk shape and size, have provided the most striking recent findings contributing to
the diagnosis
and understanding of some forms of 'idiopathic' central diabetes insipidus.
[MeSH-major]
Brain
Neoplasms
/ pathology. Craniopharyngioma / pathology. Diabetes Insipidus, Neurogenic / pathology. Germinoma / pathology. Magnetic Resonance Imaging
Genetic Alliance.
consumer health - Diabetes
.
Genetic Alliance.
consumer health - Diabetes Insipidus
.
Genetics Home Reference.
consumer health - neurohypophyseal diabetes insipidus
.
MedlinePlus Health Information.
consumer health - Brain Tumors
.
MedlinePlus Health Information.
consumer health - Childhood Brain Tumors
.
MedlinePlus Health Information.
consumer health - MRI Scans
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17451074.001).
[ISSN]
0334-018X
[Journal-full-title]
Journal of pediatric endocrinology & metabolism : JPEM
[ISO-abbreviation]
J. Pediatr. Endocrinol. Metab.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
England
[Number-of-references]
97
Advertisement
4.
Davies E, Omer S, Buckingham JC, Morris JF, Christian HC:
Expression and externalization of annexin 1 in the adrenal gland: structure and function of the adrenal gland in annexin 1-null mutant mice.
Endocrinology
; 2007 Mar;148(3):1030-8
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Expression and externalization of annexin 1 in the adrenal
gland
: structure and function of the adrenal
gland
in annexin 1-null mutant mice.
Annexin 1 (ANXA1) is a member of the annexin family of phospholipid- and calcium-binding proteins with a well demonstrated role in early delayed (30 min to 3 h) inhibitory feedback of glucocorticoids in the hypothalamus and
pituitary
gland
.
This study used adrenal
gland
tissue from ANXA1-null transgenic mice, in which a beta-galactosidase (beta-Gal) reporter gene was controlled by the ANXA1 promoter, and wild-type control mice to explore the potential role of ANXA1 in adrenal function.
RT-PCR and Western blotting revealed strong expression of ANXA1 mRNA and protein in the adrenal
gland
.
The N
-terminal peptide ANXA1(Ac2-26) inhibited corticosterone release.
COS Scholar Universe.
author profiles
.
Hazardous Substances Data Bank.
Corticotropin
.
KOMP Repository.
gene/protein/disease-specific - KOMP Repository
(subscription/membership/fee required).
Mouse Genome Informatics (MGI).
Mouse Genome Informatics (MGI)
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17158208.001).
[ISSN]
0013-7227
[Journal-full-title]
Endocrinology
[ISO-abbreviation]
Endocrinology
[Language]
eng
[Grant]
United Kingdom / Biotechnology and Biological Sciences Research Council / / BB/E52708X/1; United Kingdom / Wellcome Trust / /
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Annexin A1; 0 / Antigens, Surface; 9002-60-2 / Adrenocorticotropic Hormone; W980KJ009P / Corticosterone
5.
Cherrington BD, Morency E, Struble AM, Coonrod SA, Wakshlag JJ:
Potential role for peptidylarginine deiminase 2 (PAD2) in citrullination of canine mammary epithelial cell histones.
PLoS One
; 2010;5(7):e11768
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Previous reports have documented that PAD2 expression and activity varies across the estrous cycle in the rodent uterus and
pituitary
gland
, however, the expression and function of PAD2 in mammary tissue has not been previously reported.
Surprisingly, stimulation of canine mammary
tumor
cells (CMT25) shows that EGF, but not estrogen or progesterone, upregulates PAD2 transcription and translation suggesting EGF regulation of PAD2 and possibly citrullination in vivo.
Use of site-specific anti-citrullinated histone antibodies found that
the N
-terminus of histone H3, but not H4, appears to be the primary target of PAD activity in mammary epithelium.
[MeSH-minor]
Animals. Blotting, Western. Cell Line,
Tumor
. Cell Nucleus / metabolism. Cytoplasm / metabolism. Dogs. Epidermal Growth Factor / pharmacology. Estrous Cycle / genetics. Estrous Cycle / physiology. Female. Fluorescent Antibody Technique. Gene Expression / drug effects. Immunohistochemistry. Mammary
Neoplasms
, Animal / metabolism. Polymerase Chain Reaction
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Microsc Res Tech. 2001 Jan 15;52(2):224-30
[
11169869.001
]
[Cites]
BMC Genomics. 2009;10:135
[
19327144.001
]
[Cites]
Arthritis Rheum. 2003 Sep;48(9):2489-500
[
13130468.001
]
[Cites]
Bioessays. 2003 Nov;25(11):1106-18
[
14579251.001
]
[Cites]
Ann Rheum Dis. 2004 Apr;63(4):373-81
[
15020330.001
]
[Cites]
Gene. 2004 Apr 14;330:19-27
[
15087120.001
]
[Cites]
J Cell Sci. 2004 Sep 1;117(Pt 19):4449-59
[
15316069.001
]
[Cites]
J Natl Cancer Inst. 1986 Sep;77(3):783-92
[
3462415.001
]
[Cites]
Endocrinology. 1989 Jun;124(6):2666-70
[
2721440.001
]
[Cites]
J Biol Chem. 1989 Aug 5;264(22):13361-8
[
2753915.001
]
[Cites]
J Biol Chem. 1992 Jan 5;267(1):520-5
[
1730614.001
]
[Cites]
Anal Biochem. 1992 May 15;203(1):94-100
[
1524220.001
]
[Cites]
Vet Res Commun. 1995;19(2):101-13
[
7645193.001
]
[Cites]
Am J Vet Res. 1996 May;57(5):693-6
[
8723884.001
]
[Cites]
J Neurosci Res. 2005 Apr 1;80(1):120-8
[
15704193.001
]
[Cites]
Mol Carcinog. 2006 Mar;45(3):183-96
[
16355400.001
]
[Cites]
Int J Biochem Cell Biol. 2006;38(10):1662-77
[
16730216.001
]
[Cites]
Anal Biochem. 2006 Sep 1;356(1):36-43
[
16844072.001
]
[Cites]
Birth Defects Res B Dev Reprod Toxicol. 2007 Jun;80(3):233-45
[
17570128.001
]
[Cites]
Breast Dis. 2007;28:7-21
[
18057539.001
]
[Cites]
Lab Invest. 2008 Apr;88(4):354-64
[
18227806.001
]
[Cites]
Dis Model Mech. 2008 Nov-Dec;1(4-5):229-40
[
19093029.001
]
[Cites]
Vet Pathol. 2003 Jul;40(4):412-20
[
12824513.001
]
(PMID = 20668670.001).
[ISSN]
1932-6203
[Journal-full-title]
PloS one
[ISO-abbreviation]
PLoS ONE
[Language]
eng
[Publication-type]
Journal Article; Research Support, U.S. Gov't, Non-P.H.S.
[Publication-country]
United States
[Chemical-registry-number]
0 / Histones; 29VT07BGDA / Citrulline; 62229-50-9 / Epidermal Growth Factor; EC 3.- / Hydrolases; EC 3.5.3.15 / protein-arginine deiminase
[Other-IDs]
NLM/ PMC2909897
6.
Tsuchida K:
Myostatin inhibition by a follistatin-derived peptide ameliorates the pathophysiology of muscular dystrophy model mice.
Acta Myol
; 2008 Jul;27:14-8
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Unlike myostatin, activins regulate the growth and differentiation of nearly all cell types, including cells of the gonads,
pituitary
gland
and skeletal muscle.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
J Clin Invest. 2006 Nov;116(11):2924-34
[
17039257.001
]
[Cites]
Am J Pathol. 2006 Jun;168(6):1975-85
[
16723712.001
]
[Cites]
Proc Natl Acad Sci U S A. 2007 Feb 6;104(6):1835-40
[
17267614.001
]
[Cites]
Gene Ther. 2007 May;14(9):733-40
[
17330087.001
]
[Cites]
Neuromuscul Disord. 2007 May;17(5):423-8
[
17433676.001
]
[Cites]
PLoS One. 2007;2(8):e789
[
17726519.001
]
[Cites]
FASEB J. 2008 Feb;22(2):477-87
[
17893249.001
]
[Cites]
Curr Opin Neurol. 2002 Oct;15(5):539-44
[
12351997.001
]
[Cites]
J Biol Chem. 2002 Oct 25;277(43):40735-41
[
12194980.001
]
[Cites]
Ann Neurol. 2002 Dec;52(6):832-6
[
12447939.001
]
[Cites]
Nature. 2002 Nov 28;420(6914):418-21
[
12459784.001
]
[Cites]
J Cell Biol. 2003 Sep 15;162(6):1135-47
[
12963705.001
]
[Cites]
Mol Cell Biol. 2003 Oct;23(20):7230-42
[
14517293.001
]
[Cites]
Proc Natl Acad Sci U S A. 2003 Dec 23;100(26):15842-6
[
14671324.001
]
[Cites]
Annu Rev Cell Dev Biol. 2004;20:61-86
[
15473835.001
]
[Cites]
Am J Pathol. 2005 Feb;166(2):491-7
[
15681832.001
]
[Cites]
FASEB J. 2005 Apr;19(6):543-9
[
15791004.001
]
[Cites]
Dev Cell. 2005 Oct;9(4):535-43
[
16198295.001
]
[Cites]
Biochem Soc Trans. 2005 Dec;33(Pt 6):1513-7
[
16246158.001
]
[Cites]
Proc Natl Acad Sci U S A. 2005 Dec 13;102(50):18117-22
[
16330774.001
]
[Cites]
Expert Opin Biol Ther. 2006 Feb;6(2):147-54
[
16436040.001
]
[Cites]
Cancer Res. 2006 Feb 1;66(3):1320-6
[
16452185.001
]
[Cites]
EMBO J. 2006 Mar 8;25(5):1035-45
[
16482217.001
]
[Cites]
Mini Rev Med Chem. 2006 Nov;6(11):1255-61
[
17100637.001
]
(PMID = 19108572.001).
[ISSN]
1128-2460
[Journal-full-title]
Acta myologica : myopathies and cardiomyopathies : official journal of the Mediterranean Society of Myology
[ISO-abbreviation]
Acta Myol
[Language]
ENG
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Italy
[Chemical-registry-number]
0 / FS I-I protein; 0 / Follistatin; 0 / Mstn protein, mouse; 0 / Myostatin; 0 / Recombinant Fusion Proteins; 0 / Transforming Growth Factor beta; EC 2.7.11.30 / Activin Receptors, Type I; EC 2.7.11.30 / Acvr1 protein, mouse
[Other-IDs]
NLM/ PMC2859604
7.
Zhao LF, Iwasaki Y, Oki Y, Tsugita M, Taguchi T, Nishiyama M, Takao T, Kambayashi M, Hashimoto K:
Purinergic receptor ligands stimulate pro-opiomelanocortin gene expression in AtT-20 pituitary corticotroph cells.
J Neuroendocrinol
; 2006 Apr;18(4):273-8
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Purinergic receptor ligands stimulate pro-opiomelanocortin gene expression in AtT-20
pituitary
corticotroph cells.
Although recent studies have suggested that purinergic receptors are expressed in the anterior
pituitary
gland
, their involvement in the regulation of
pituitary
hormone gene expression is not completely understood.
Because adenosine and ATP are known to be produced within the
pituitary
gland
, it is possible they may be acting in an autocrine/paracrine fashion.
[MeSH-major]
Adenosine / metabolism. Corticotropin-Releasing Hormone / metabolism. Gene Expression Regulation / physiology.
Pituitary
Gland
/ metabolism. Pro-Opiomelanocortin / metabolism. Receptors, Purinergic / metabolism
Hazardous Substances Data Bank.
Adenosine
.
Hazardous Substances Data Bank.
Corticotropin
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16503922.001).
[ISSN]
0953-8194
[Journal-full-title]
Journal of neuroendocrinology
[ISO-abbreviation]
J. Neuroendocrinol.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
England
[Chemical-registry-number]
0 / Ligands; 0 / RNA, Messenger; 0 / Receptors, Purinergic; 66796-54-1 / Pro-Opiomelanocortin; 8L70Q75FXE / Adenosine Triphosphate; 9002-60-2 / Adrenocorticotropic Hormone; 9015-71-8 / Corticotropin-Releasing Hormone; K72T3FS567 / Adenosine
8.
Burri E, Nüesch R, Zulewski H:
[Hyperprolactinemia in a man's world].
Praxis (Bern 1994)
; 2008 Dec 3;97(24):1295-9
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Ultrasonography of the mamillary
gland
was not definite for gynecomastia but repeated serum prolactin concentrations were elevated 5-fold the upper limit of normal.
Magnetic resonance imaging (MRI) of the
pituitary
gland
could not identify a tumoral mass.
[MeSH-minor]
Algorithms.
Diagnosis
, Differential. Dopamine Agonists / therapeutic use. Ergolines / administration & dosage. Ergolines / therapeutic use. Follow-Up Studies. Gynecomastia /
diagnosis
. Gynecomastia / ultrasonography. Humans. Hypogonadism /
diagnosis
. Male. Middle Aged. Prolactin / blood. Testosterone / blood. Time Factors. Ultrasonography, Mammary
Genetic Alliance.
consumer health - Galactorrhoea-Hyperprolactinaemia
.
Hazardous Substances Data Bank.
TESTOSTERONE
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19048508.001).
[ISSN]
1661-8157
[Journal-full-title]
Praxis
[ISO-abbreviation]
Praxis (Bern 1994)
[Language]
ger
[Publication-type]
Case Reports; English Abstract; Journal Article
[Publication-country]
Switzerland
[Chemical-registry-number]
0 / Dopamine Agonists; 0 / Ergolines; 3XMK78S47O / Testosterone; 9002-62-4 / Prolactin; LL60K9J05T / cabergoline
9.
Chesnokova V, Melmed S:
Pituitary senescence: the evolving role of Pttg.
Mol Cell Endocrinol
; 2010 Sep 15;326(1-2):55-9
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Pituitary
senescence: the evolving role of Pttg.
Despite the high prevalence of
pituitary
adenomas they are invariably
benign
, indicative of unique intrinsic mechanisms controlling
pituitary
cell proliferation.
Cellular senescence is characterized by a largely irreversible cell cycle arrest and constitutes a strong anti-proliferative response, which can be triggered by DNA damage, chromosomal instability and aneuploidy, loss
of tumor
suppressive signaling or oncogene activation.
Here we discuss prospective mechanisms underlying senescence-associated molecular pathways activated in
benign pituitary
adenomas.
Both deletion and over-expression of
pituitary
tumor
transforming gene (Pttg) promote chromosomal instability and aneuploidy.
Pttg deletion abrogates
tumor
development by activating p53/p21-dependent senescence pathways.
Abundant PTTG in GH-secreting
pituitary
adenomas also triggers p21-dependent senescence.
Pituitary
p21 may therefore safeguard against further chromosomal instability by constraining
pituitary
tumor
growth.
These observations point to senescence as a target for effective therapy for both
tumor
silencing and growth restraint towards development of
pituitary
malignancy.
MedlinePlus Health Information.
consumer health - Pituitary Tumors
.
KOMP Repository.
gene/protein/disease-specific - KOMP Repository
(subscription/membership/fee required).
Mouse Genome Informatics (MGI).
Mouse Genome Informatics (MGI)
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Copyright]
2010 Elsevier Ireland Ltd. All rights reserved.
[Cites]
Mol Endocrinol. 2001 Nov;15(11):1870-9
[
11682618.001
]
[Cites]
Trends Cell Biol. 2001 Nov;11(11):S27-31
[
11684439.001
]
[Cites]
J Clin Invest. 2001 Dec;108(12):1729-33
[
11748253.001
]
[Cites]
Oncogene. 2002 Jan 21;21(4):503-11
[
11850775.001
]
[Cites]
Cell. 2002 May 3;109(3):335-46
[
12015983.001
]
[Cites]
Nat Genet. 2003 Jan;33(1):49-54
[
12469122.001
]
[Cites]
Proc Natl Acad Sci U S A. 2003 Mar 18;100(6):3428-32
[
12626748.001
]
[Cites]
Front Neuroendocrinol. 2003 Apr;24(2):94-127
[
12763000.001
]
[Cites]
Endocr Relat Cancer. 2003 Jun;10(2):323-30
[
12790793.001
]
[Cites]
Cell. 2003 Jun 13;113(6):703-16
[
12809602.001
]
[Cites]
Endocrinology. 2003 Nov;144(11):4991-8
[
12960092.001
]
[Cites]
J Clin Invest. 2003 Dec;112(11):1603-18
[
14660734.001
]
[Cites]
J Clin Invest. 2004 Jan;113(2):160-8
[
14722605.001
]
[Cites]
Nature. 1989 Aug 31;340(6236):692-6
[
2549426.001
]
[Cites]
Nature. 1990 May 31;345(6274):458-60
[
2342578.001
]
[Cites]
Proc Natl Acad Sci U S A. 1995 Sep 26;92(20):9363-7
[
7568133.001
]
[Cites]
Cell. 1997 Mar 7;88(5):593-602
[
9054499.001
]
[Cites]
Mol Endocrinol. 1997 Apr;11(4):433-41
[
9092795.001
]
[Cites]
Mol Endocrinol. 1999 Jan;13(1):156-66
[
9892021.001
]
[Cites]
Prog Mol Subcell Biol. 1998;20:43-71
[
9928526.001
]
[Cites]
Proc Natl Acad Sci U S A. 1999 Feb 2;96(3):1002-7
[
9927683.001
]
[Cites]
Nat Med. 1999 Nov;5(11):1317-21
[
10546001.001
]
[Cites]
J Clin Endocrinol Metab. 1999 Feb;84(2):761-7
[
10022450.001
]
[Cites]
Science. 1999 Jul 16;285(5426):418-22
[
10411507.001
]
[Cites]
Exp Cell Res. 1961 Dec;25:585-621
[
13905658.001
]
[Cites]
Ann Diagn Pathol. 2005 Feb;9(1):6-10
[
15692944.001
]
[Cites]
Mol Endocrinol. 2005 May;19(5):1383-91
[
15677710.001
]
[Cites]
Oncogene. 2005 Jul 14;24(30):4861-6
[
15897900.001
]
[Cites]
Nature. 2005 Aug 4;436(7051):642
[
16079833.001
]
[Cites]
Nature. 2005 Aug 4;436(7051):660-5
[
16079837.001
]
[Cites]
Nature. 2005 Aug 4;436(7051):720-4
[
16079850.001
]
[Cites]
Science. 2005 Aug 5;309(5736):886-7
[
16081723.001
]
[Cites]
Mol Endocrinol. 2005 Sep;19(9):2371-9
[
15919720.001
]
[Cites]
Cancer Res. 2005 Oct 1;65(19):8747-53
[
16204044.001
]
[Cites]
Anal Quant Cytol Histol. 2005 Oct;27(5):241-52
[
16447816.001
]
[Cites]
Endocr Relat Cancer. 2006 Sep;13(3):707-16
[
16954426.001
]
[Cites]
N Engl J Med. 2006 Sep 7;355(10):1037-46
[
16957149.001
]
[Cites]
Nature. 2006 Nov 30;444(7119):633-7
[
17136093.001
]
[Cites]
Am J Physiol Cell Physiol. 2007 Sep;293(3):C1082-92
[
17626243.001
]
[Cites]
Cancer Res. 2007 Nov 1;67(21):10564-72
[
17975001.001
]
[Cites]
Endocrinology. 2007 Dec;148(12):6019-25
[
17872367.001
]
[Cites]
Cell Death Differ. 2008 Jan;15(1):202-12
[
17962814.001
]
[Cites]
Trends Genet. 2008 Feb;24(2):77-85
[
18192065.001
]
[Cites]
Science. 2008 Mar 7;319(5868):1352-5
[
18323444.001
]
[Cites]
Cell. 2008 Jun 13;133(6):958-61
[
18555773.001
]
[Cites]
Proc Natl Acad Sci U S A. 2008 Nov 11;105(45):17498-503
[
18981426.001
]
[Cites]
Pituitary. 2009;12(1):40-50
[
18270844.001
]
[Cites]
Nat Rev Cancer. 2009 Feb;9(2):81-94
[
19132009.001
]
[Cites]
Cell Cycle. 2009 Mar 1;8(5):677-8
[
19223763.001
]
[Cites]
Clin Endocrinol (Oxf). 2010 Mar;72(3):377-82
[
19650784.001
]
[Cites]
Nat Clin Pract Endocrinol Metab. 2006 Dec;2(12):681-93
[
17143315.001
]
[Cites]
Nature. 2006 Dec 21;444(7122):1038-43
[
17183314.001
]
[Cites]
Proc Natl Acad Sci U S A. 2006 Dec 26;103(52):19842-7
[
17170138.001
]
[Cites]
Carcinogenesis. 2007 Mar;28(3):749-59
[
17071631.001
]
[Cites]
Endocr Rev. 2007 Apr;28(2):165-86
[
17325339.001
]
[Cites]
Cell. 2007 Jul 27;130(2):223-33
[
17662938.001
]
[Cites]
J Biol Chem. 2000 Nov 24;275(47):36502-5
[
11013229.001
]
[Cites]
J Clin Endocrinol Metab. 2001 Feb;86(2):867-74
[
11158059.001
]
(PMID = 20153804.001).
[ISSN]
1872-8057
[Journal-full-title]
Molecular and cellular endocrinology
[ISO-abbreviation]
Mol. Cell. Endocrinol.
[Language]
ENG
[Grant]
United States / NCI NIH HHS / CA / CA075979-11A1; United States / NCI NIH HHS / CA / R01 CA075979; United States / NCI NIH HHS / CA / R01 CA075979-11A1
[Publication-type]
Journal Article; Review
[Publication-country]
Ireland
[Chemical-registry-number]
0 / Neoplasm Proteins; 0 / Securin; 0 / pituitary tumor-transforming protein 1, human
[Other-IDs]
NLM/ NIHMS185500; NLM/ PMC2906651
10.
Brym P, Malewski T, Starzyński R, Flisikowski K, Wójcik E, Ruść A, Zwierzchowski L, Kamiński S:
Effect of new SNP within bovine prolactin gene enhancer region on expression in the pituitary gland.
Biochem Genet
; 2007 Oct;45(9-10):743-54
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Effect of new SNP within bovine prolactin gene enhancer region on expression in the
pituitary
gland
.
The application of real-time PCR revealed that the prolactin gene expression level in the
pituitary
was higher in cattle with the AA genotype than in those with the GG genotype.
[MeSH-major]
Cattle / genetics.
Pituitary
Gland
/ metabolism. Polymorphism, Single Nucleotide. Prolactin / genetics
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17929163.001).
[ISSN]
0006-2928
[Journal-full-title]
Biochemical genetics
[ISO-abbreviation]
Biochem. Genet.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / DNA Primers; 0 / RNA, Messenger; 9002-62-4 / Prolactin; 9007-49-2 / DNA
11.
Korenkov AI, Imhof HG, Brandner S, Taub E, Huguenin PU, Gaab MR, Yonekawa Y:
Growth retardation and bilateral cataracts followed by anaplastic meningioma 23 years after high-dose cranial and whole-body irradiation for acute lymphoblastic leukemia: case report and review of the literature.
J Neurooncol
; 2005 Sep;74(2):195-9
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Radiotherapy in this case also caused early radiation injury to the lenses and the
pituitary
gland
, with growth retardation and mineralizing angiopathy.
Survivors of childhood ALL treated with high-dose cranial irradiation are at risk both for early radiation injury in radiosensitive organs, such as the lens and
pituitary
gland
, and for the later development of a radiation-induced meningioma.
[MeSH-major]
Cataract / etiology. Cranial Irradiation / adverse effects. Growth Disorders / etiology. Meningeal
Neoplasms
/ etiology. Meningioma / etiology.
Neoplasms
, Radiation-Induced / etiology. Precursor Cell Lymphoblastic Leukemia-Lymphoma / radiotherapy
[MeSH-minor]
Adult. Humans. Lens, Crystalline / radiation effects. Magnetic Resonance Imaging. Male.
Pituitary
Gland
/ radiation effects. Time Factors. Tomography, X-Ray Computed. Whole-Body Irradiation
Genetic Alliance.
consumer health - Cataracts
.
Genetic Alliance.
consumer health - Acute Lymphoblastic Leukemia
.
Genetic Alliance.
consumer health - Meningioma
.
MedlinePlus Health Information.
consumer health - Cataract
.
MedlinePlus Health Information.
consumer health - Growth Disorders
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
J Neurosurg. 1994 Nov;81(5):666-75
[
7931612.001
]
[Cites]
Surg Neurol. 1985 Jul;24(1):35-9
[
4012556.001
]
[Cites]
Lancet. 1988 Feb 27;1(8583):460-2
[
2893877.001
]
[Cites]
Mol Biol Med. 1988 Feb;5(1):15-22
[
2897611.001
]
[Cites]
Br J Haematol. 2000 Mar;108(4):665
[
10792267.001
]
[Cites]
Eur J Cancer. 1997 Apr;33(4):526-30
[
9274430.001
]
[Cites]
Intern Med. 1992 May;31(5):629-32
[
1504425.001
]
[Cites]
J Neurosurg. 1995 Mar;82(3):487-8
[
7861229.001
]
[Cites]
Med Pediatr Oncol. 2000 Nov;35(5):456-61
[
11070477.001
]
[Cites]
Int J Radiat Oncol Biol Phys. 2000 Aug 1;48(1):65-73
[
10924973.001
]
[Cites]
Cancer. 2002 Dec 15;95(12):2562-70
[
12467071.001
]
[Cites]
Childs Nerv Syst. 1996 May;12(5):266-9
[
8737803.001
]
[Cites]
Blood. 1971 Mar;37(3):272-81
[
4322483.001
]
[Cites]
Growth. 1967 Jun;31(2):139-48
[
6034725.001
]
[Cites]
Med Pediatr Oncol. 1995 Apr;24(4):265-8
[
7700173.001
]
[Cites]
Bull Cancer. 2002 Feb;89(2):181-96
[
11888858.001
]
[Cites]
Acta Neurol (Napoli). 1986 Apr;8(2):145-9
[
3716900.001
]
[Cites]
J Natl Cancer Inst. 1983 May;70(5):863-6
[
6573530.001
]
[Cites]
J Neurosurg. 1983 Dec;59(6):1048-53
[
6631499.001
]
[Cites]
J Clin Oncol. 1998 Aug;16(8):2848-53
[
9704738.001
]
[Cites]
J Neurosurg. 1984 Nov;61(5):966-71
[
6593438.001
]
[Cites]
J Clin Oncol. 1991 Mar;9(3):400-5
[
1999710.001
]
[Cites]
Br J Ophthalmol. 1970 Apr;54(4):237-47
[
5310660.001
]
[Cites]
Cancer. 1998 Jan 1;82(1):8-34
[
9428476.001
]
[Cites]
Radiother Oncol. 1997 Jun;43(3):285-8
[
9215789.001
]
[Cites]
J Neurosurg. 1998 Jan;88(1):111-5
[
9420081.001
]
[Cites]
JAMA. 1960 Sep 10;174:166-71
[
13801171.001
]
[Cites]
Br J Radiol. 1995 Oct;68(814):1123-5
[
7496718.001
]
[Cites]
Leukemia. 2001 Jan;15(1):41-5
[
11243397.001
]
[Cites]
Br J Ophthalmol. 1953 Dec;37(12):758-62
[
13115610.001
]
[Cites]
Horm Res. 1988;30(2-3):53-61
[
3074030.001
]
[Cites]
Radiology. 1987 Jan;162(1 Pt 1):119-24
[
3538143.001
]
[Cites]
Int J Radiat Oncol Biol Phys. 1990 Feb;18(2):399-406
[
2137438.001
]
[Cites]
J Neurosurg. 2002 Nov;97(5):1078-82
[
12450029.001
]
[Cites]
J Neurosurg. 1991 Oct;75(4):564-74
[
1885974.001
]
[Cites]
Childs Nerv Syst. 1995 Nov;11(11):661-3
[
8608584.001
]
[Cites]
J Child Neurol. 1991 Apr;6(2):128-33
[
2045628.001
]
[Cites]
Neuroradiology. 1994 Nov;36(8):652-5
[
7862289.001
]
[Cites]
Neurosurgery. 1991 Mar;28(3):482
[
2011241.001
]
[Cites]
Lancet. 1974 Feb 23;1(7852):277-9
[
4130470.001
]
[Cites]
Am J Pediatr Hematol Oncol. 1992 Aug;14(3):236-40
[
1510194.001
]
[Cites]
Vopr Neirokhir. 1978 Jan-Feb;(1):51-3
[
636403.001
]
[Cites]
Lancet. 1987 Jan 24;1(8526):190-3
[
2880018.001
]
[Cites]
AJNR Am J Neuroradiol. 1994 Mar;15(3):537-41
[
8197954.001
]
[Cites]
Cancer. 1978 Aug;42(2):717-28
[
277284.001
]
[Cites]
Ophthalmology. 1988 Feb;95(2):151-5
[
3173990.001
]
[Cites]
J Neurosurg. 1981 Aug;55(2):282-6
[
7252552.001
]
[Cites]
N Engl J Med. 1978 Apr 13;298(15):815-8
[
273143.001
]
[Cites]
Neurosurgery. 1988 Apr;22(4):758-61
[
3163779.001
]
[Cites]
J Neurosurg. 1993 Jul;79(1):28-31
[
8315464.001
]
[Cites]
Surg Neurol. 1992 Oct;38(4):261-4
[
1440212.001
]
[Cites]
J Natl Cancer Inst. 1980 Jul;65(1):67-73
[
6930521.001
]
[Cites]
Br J Ophthalmol. 1985 Jun;69(6):459-61
[
3859324.001
]
(PMID = 16193392.001).
[ISSN]
0167-594X
[Journal-full-title]
Journal of neuro-oncology
[ISO-abbreviation]
J. Neurooncol.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
United States
12.
Beyea JA, Olson DM, Vandergriend RA, Harvey S:
Expression of growth hormone and its receptor in the lungs of embryonic chicks.
Cell Tissue Res
; 2005 Dec;322(3):379-92
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Pituitary
GH is therefore probably involved in normal lung growth or development, although perinatal lung development occurs prior to the differentiation of
pituitary
somatotrophs and the ontogeny of
pituitary
GH secretion.
A 690-bp cDNA, identical in size and nucleotide sequence to the full-length
pituitary
GH transcript, was amplified by reverse transcription/polymerase chain reaction from total RNA extracted from the lungs of embryos at 11, 13, 15, and 18 days of the 21-day incubation period.
Lung GH immunoreactivity was primarily associated with a 15-kDa protein, rather than the 26-kDa protein in the
pituitary
gland
.
After the onset of
pituitary
GH secretion (at ED17), GH mRNA was barely detectable in the lungs of ED20 embryos, at the start of lung breathing.
Lung GH may thus have autocrine or paracrine roles in lung development or in pulmonary function prior to the ontogeny of the
pituitary
gland
and the appearance of GH in peripheral plasma.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16047159.001).
[ISSN]
0302-766X
[Journal-full-title]
Cell and tissue research
[ISO-abbreviation]
Cell Tissue Res.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Germany
[Chemical-registry-number]
0 / RNA, Messenger; 0 / Receptors, Somatotropin; 9002-72-6 / Growth Hormone
13.
Flik G, Klaren PH, Van den Burg EH, Metz JR, Huising MO:
CRF and stress in fish.
Gen Comp Endocrinol
; 2006 Mar;146(1):36-44
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Key players in the continuous adaptation process are corticotropin-releasing factor (CRF) from the hypothalamic nucleus preopticus (NPO),
pituitary
adrenocorticotropic hormone (ACTH) and cortisol produced by the interrenal cells in the headkidney (adrenal equivalent of fish).
Pro-opiomelanocortin is produced and processed to ACTH and endorphin in the hypothalamic NPO and
pituitary
pars distalis ACTH-cells, to MSH and acetylated endorphins in the
pituitary
pars intermedia MSH-cells.
Interesting observations were made on the CRF control of
pituitary
cells.
Endorphin, produced in the NPO and transported via axons to the
pituitary
gland
in vivo, reverses the stimulatory CRF action on MSH-cells to a differential inhibition
of N
-acetyl beta-endorphin release in vitro (MSH release is not affected).
In carp (and other fish), the endocrine stress axis is already operational in very early life stages, viz., around hatching and comprises hypothalamic,
pituitary
, and interrenal signaling to adjust the physiology of the hatchling to its dynamically changing environment.
COS Scholar Universe.
author profiles
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16403502.001).
[ISSN]
0016-6480
[Journal-full-title]
General and comparative endocrinology
[ISO-abbreviation]
Gen. Comp. Endocrinol.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Chemical-registry-number]
0 / Endorphins; 66796-54-1 / Pro-Opiomelanocortin; 9002-79-3 / Melanocyte-Stimulating Hormones; 9015-71-8 / Corticotropin-Releasing Hormone
[Number-of-references]
37
14.
Roche JR, Blache D, Kay JK, Miller DR, Sheahan AJ, Miller DW:
Neuroendocrine and physiological regulation of intake with particular reference to domesticated ruminant animals.
Nutr Res Rev
; 2008 Dec;21(2):207-34
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Information regarding metabolic state can be transmitted to the appetite control centres of the brain by a diverse array of signals, such as stimulation of the vagus nerve, or metabolic 'feedback' factors derived from the
pituitary
gland
, adipose tissue, stomach/abomasum, intestine, pancreas and/or muscle.
COS Scholar Universe.
author profiles
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19087372.001).
[ISSN]
1475-2700
[Journal-full-title]
Nutrition research reviews
[ISO-abbreviation]
Nutr Res Rev
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
England
[Chemical-registry-number]
0 / Cannabinoids; 0 / Hormones
[Number-of-references]
388
15.
Ono H, Hoshino Y, Yasuo S, Watanabe M, Nakane Y, Murai A, Ebihara S, Korf HW, Yoshimura T:
Involvement of thyrotropin in photoperiodic signal transduction in mice.
Proc Natl Acad Sci U S A
; 2008 Nov 25;105(47):18238-42
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Recent functional genomics analysis in birds has shown that long days induce thyroid-stimulating hormone production in the pars tuberalis (PT) of the
pituitary
gland
, which triggers DIO2 expression in the ependymal cells (EC) of the MBH.
COS Scholar Universe.
author profiles
.
Hazardous Substances Data Bank.
MELATONIN
.
KOMP Repository.
gene/protein/disease-specific - KOMP Repository
(subscription/membership/fee required).
Mouse Genome Informatics (MGI).
Mouse Genome Informatics (MGI)
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Am J Physiol Regul Integr Comp Physiol. 2007 Mar;292(3):R1315-9
[
17110533.001
]
[Cites]
Endocrinology. 2006 Jan;147(1):432-40
[
16195409.001
]
[Cites]
Endocrinology. 2007 Aug;148(8):3608-17
[
17478556.001
]
[Cites]
Endocrinology. 2007 Sep;148(9):4385-92
[
17540726.001
]
[Cites]
Nature. 2008 Mar 20;452(7185):317-22
[
18354476.001
]
[Cites]
Reproduction. 2008 Jul;136(1):1-8
[
18515309.001
]
[Cites]
Curr Biol. 2008 Aug 5;18(15):1147-52
[
18674911.001
]
[Cites]
Curr Biol. 2008 Sep 9;18(17):R795-R804
[
18786385.001
]
[Cites]
Lab Anim. 1977 Jul;11(3):159-62
[
886825.001
]
[Cites]
Science. 1979 Jul 27;205(4404):366-72
[
221983.001
]
[Cites]
Endocr Rev. 1980 Spring;1(2):109-31
[
6263600.001
]
[Cites]
Endocrinology. 1983 Jul;113(1):329-36
[
6861705.001
]
[Cites]
Science. 1986 Jan 31;231(4737):491-3
[
3941912.001
]
[Cites]
Cell Tissue Res. 1988 Jan;251(1):183-7
[
3342436.001
]
[Cites]
J Endocrinol. 1988 Oct;119(1):R1-3
[
2848087.001
]
[Cites]
J Pineal Res. 1989;7(2):195-204
[
2769571.001
]
[Cites]
Eur J Pharmacol. 1990 May 16;180(2-3):387-90
[
2365011.001
]
[Cites]
Neuron. 1994 Nov;13(5):1177-85
[
7946354.001
]
[Cites]
Endocrinology. 1996 May;137(5):1804-13
[
8612518.001
]
[Cites]
Endocrinology. 1996 Aug;137(8):3469-77
[
8754776.001
]
[Cites]
Endocrinology. 1997 Mar;138(3):1019-28
[
9048604.001
]
[Cites]
Int Rev Cytol. 1999;185:157-94
[
9750267.001
]
[Cites]
J Neuroendocrinol. 2000 Mar;12(3):207-16
[
10718916.001
]
[Cites]
Brain Res Mol Brain Res. 2000 May 31;78(1-2):207-15
[
10891604.001
]
[Cites]
Am J Physiol Regul Integr Comp Physiol. 2001 Aug;281(2):R666-72
[
11448873.001
]
[Cites]
Proc Natl Acad Sci U S A. 2002 Nov 26;99(24):15776-81
[
12432094.001
]
[Cites]
J Histochem Cytochem. 2002 Dec;50(12):1647-57
[
12486087.001
]
[Cites]
Nature. 2003 Nov 13;426(6963):178-81
[
14614506.001
]
[Cites]
Endocrinology. 2004 Apr;145(4):1546-9
[
14726436.001
]
[Cites]
Endocrinology. 2006 Oct;147(10):4680-7
[
16873538.001
]
[Cites]
Am J Physiol Regul Integr Comp Physiol. 2007 Jan;292(1):R568-72
[
17197645.001
]
[Cites]
Brain Res Mol Brain Res. 1998 Dec 10;63(1):189-97
[
9838107.001
]
[Cites]
Science. 1965 Jun 18;148(3677):1609-11
[
14287606.001
]
[Cites]
Endocrinology. 2005 Jun;146(6):2551-4
[
15746251.001
]
[Cites]
Am J Physiol Regul Integr Comp Physiol. 2007 Jun;292(6):R2368-72
[
17272662.001
]
(PMID = 19015516.001).
[ISSN]
1091-6490
[Journal-full-title]
Proceedings of the National Academy of Sciences of the United States of America
[ISO-abbreviation]
Proc. Natl. Acad. Sci. U.S.A.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Receptors, Thyrotropin; 9002-71-5 / Thyrotropin; EC 1.11.1.- / iodothyronine deiodinase type II; EC 1.11.1.8 / Iodide Peroxidase; JL5DK93RCL / Melatonin
[Other-IDs]
NLM/ PMC2587639
16.
Maddineni SR, Ocón-Grove OM, Krzysik-Walker SM, Hendricks GL 3rd, Ramachandran R:
Gonadotropin-inhibitory hormone (GnIH) receptor gene is expressed in the chicken ovary: potential role of GnIH in follicular maturation.
Reproduction
; 2008 Feb;135(2):267-74
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Gonadotropin-inhibitory hormone (GnIH), an RFamide peptide, has been found to inhibit
pituitary
LH secretion in avian and mammalian species.
The gene encoding a putative receptor for GnIH (GnIHR) was recently identified in the chicken and Japanese quail brain and
pituitary
gland
.
COS Scholar Universe.
author profiles
.
Hazardous Substances Data Bank.
ESTRADIOL
.
Hazardous Substances Data Bank.
PROGESTERONE
.
Hazardous Substances Data Bank.
MENOTROPINS
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18239054.001).
[ISSN]
1470-1626
[Journal-full-title]
Reproduction (Cambridge, England)
[ISO-abbreviation]
Reproduction
[Language]
eng
[Publication-type]
Journal Article; Research Support, U.S. Gov't, Non-P.H.S.
[Publication-country]
England
[Chemical-registry-number]
0 / Avian Proteins; 0 / Hypothalamic Hormones; 0 / RNA, Messenger; 4G7DS2Q64Y / Progesterone; 4TI98Z838E / Estradiol; 9002-68-0 / Follicle Stimulating Hormone
17.
Górski K, Gajewska A, Romanowicz K, Misztal T:
Genistein-induced pituitary prolactin gene expression and prolactin release in ovariectomized ewes following a series of intracerebroventricular infusions.
Reprod Biol
; 2007 Nov;7(3):233-46
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Genistein-induced
pituitary
prolactin gene expression and prolactin release in ovariectomized ewes following a series of intracerebroventricular infusions.
The aim of the study was to evaluate whether genistein, a phytoestrogen commonly present in feed plants, affects prolactin release and its gene expression in the
pituitary
gland
.
Northern blot analysis revealed that
pituitary
prolactin mRNA content increased significantly in response to genistein, compared to the vehicle-infused ewes (p<0.05).
[MeSH-major]
Genistein / pharmacology.
Pituitary
Gland
/ drug effects. Prolactin / biosynthesis
Hazardous Substances Data Bank.
GENISTEIN
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18059975.001).
[ISSN]
2300-732X
[Journal-full-title]
Reproductive biology
[ISO-abbreviation]
Reprod Biol
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Poland
[Chemical-registry-number]
0 / RNA, Messenger; 9002-62-4 / Prolactin; DH2M523P0H / Genistein
18.
Schaaf L, Pickel J, Zinner K, Hering U, Höfler M, Goretzki PE, Spelsberg F, Raue F, von zur Mühlen A, Gerl H, Hensen J, Bartsch DK, Rothmund M, Schneyer U, Dralle H, Engelbach M, Karges W, Stalla GK, Höppner W:
Developing effective screening strategies in multiple endocrine neoplasia type 1 (MEN 1) on the basis of clinical and sequencing data of German patients with MEN 1.
Exp Clin Endocrinol Diabetes
; 2007 Sep;115(8):509-17
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Developing effective screening strategies in multiple endocrine
neoplasia
type 1 (MEN 1) on the basis of clinical and sequencing data of German patients with MEN 1.
BACKGROUND: Multiple-endocrine-
neoplasia
-type-1 (MEN1) is an autosomal-dominant inherited
disorder
characterized by the combined occurrence of primary hyperparathyroidism (pHPT), gastroenteropancreatic neuroendocrine
tumors
(GEP), adenomas of the
pituitary
gland
(APA), adrenal cortical
tumors
(ADR) and other
tumors
.
As
the tumors
appear in an unpredictable schedule, uncertainty about screening programs is persisting.
RESULTS: A total of 683
tumors
occurred consisting of 273 pHPT, 138 APA, 166 GEP, 57 ADR, 24 thymic- and bronchial-carcinoids as well as 25
neoplasms of
other tissues.
CONCLUSION: In view of the morbidity and frequency in familial cases an effective screening programme should aim at an early
diagnosis of
GEP particularly when truncating, especially nonsense mutations are found.
[MeSH-major]
Mass Screening / methods. Multiple Endocrine
Neoplasia
Type 1 / epidemiology
Genetic Alliance.
consumer health - Multiple Endocrine Neoplasia
.
Genetic Alliance.
consumer health - Multiple endocrine neoplasia type 1
.
COS Scholar Universe.
author profiles
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17853334.001).
[ISSN]
0947-7349
[Journal-full-title]
Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association
[ISO-abbreviation]
Exp. Clin. Endocrinol. Diabetes
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Germany
[Chemical-registry-number]
9007-49-2 / DNA
19.
Lepore DA, Thomas GP, Knight KR, Hussey AJ, Callahan T, Wagner J, Morrison WA, Thomas PQ:
Survival and differentiation of pituitary colony-forming cells in vivo.
Stem Cells
; 2007 Jul;25(7):1730-6
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Survival and differentiation of
pituitary
colony-forming cells in vivo.
Recently, our laboratory identified a cell type within the adult
pituitary
gland
with stem cell-like properties, which we have termed
pituitary
colony-forming cells (PCFCs).
[MeSH-major]
Cell Differentiation.
Pituitary
Gland
/ cytology. Stem Cells / cytology
MedlinePlus Health Information.
consumer health - Stem Cells
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17395770.001).
[ISSN]
1066-5099
[Journal-full-title]
Stem cells (Dayton, Ohio)
[ISO-abbreviation]
Stem Cells
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
9002-72-6 / Growth Hormone
20.
Sciara AA, Rubiolo JA, Somoza GM, Arranz SE:
Molecular cloning, expression and immunological characterization of pejerrey (Odontesthes bonariensis) growth hormone.
Comp Biochem Physiol C Toxicol Pharmacol
; 2006 Mar-Apr;142(3-4):284-92
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
The transcript was detected not only in the
pituitary
gland
but also in the testis.
[MeSH-minor]
Amino Acid Sequence. Animals. Base Sequence. Cloning, Molecular. Escherichia coli / genetics. Female. Gene Expression. Immunohistochemistry / methods. Male. Molecular Sequence Data. Phylogeny.
Pituitary
Gland
/ metabolism. RNA, Messenger / metabolism. Recombinant Proteins / immunology. Sequence Alignment. Testis / metabolism
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16326143.001).
[ISSN]
1532-0456
[Journal-full-title]
Comparative biochemistry and physiology. Toxicology & pharmacology : CBP
[ISO-abbreviation]
Comp. Biochem. Physiol. C Toxicol. Pharmacol.
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / RNA, Messenger; 0 / Recombinant Proteins; 9002-72-6 / Growth Hormone
21.
Mullis PE:
Genetics of growth hormone deficiency.
Endocrinol Metab Clin North Am
; 2007 Mar;36(1):17-36
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
This article focuses on the GH gene, the various gene alterations, and their possible impact on the
pituitary
gland
.
Transcription factors regulating
pituitary
gland
development may cause multiple
pituitary
hormone deficiency but may present initially as GH deficiency.
[MeSH-major]
Dwarfism,
Pituitary
/ genetics
[MeSH-minor]
Animals. Growth Hormone / genetics. Humans. Mice. Mice, Transgenic. Mutation.
Pituitary
Gland
/ growth & development. RNA Splice Sites / genetics. Secretory Vesicles / pathology. Transcription Factors / genetics. Transcription Factors / physiology
Genetic Alliance.
consumer health - Growth Hormone Deficiency
.
COS Scholar Universe.
author profiles
.
SciCrunch.
KEGG: Data: Disease Annotation
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17336732.001).
[ISSN]
0889-8529
[Journal-full-title]
Endocrinology and metabolism clinics of North America
[ISO-abbreviation]
Endocrinol. Metab. Clin. North Am.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
United States
[Chemical-registry-number]
0 / RNA Splice Sites; 0 / Transcription Factors; 9002-72-6 / Growth Hormone
[Number-of-references]
82
22.
Xu J, Zhang S, You C, Wang X, Zhou Q:
Microvascular density and vascular endothelial growth factor have little correlation with prognosis of craniopharyngioma.
Surg Neurol
; 2006;66 Suppl 1:S30-4
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
BACKGROUND: Craniopharyngioma is histologically a
benign
epithelial
tumor
located in the supersellar cistern that often presents aggressive growth and repeated recurrence.
METHODS: The cohorts consisted of 32 patients with AE and 31 patients with SP
tumor
.
CONCLUSIONS: Microvascular density and VEGF in craniopharyngioma tissue have no correlation with prognosis of the
tumor
, which may be explained by the minimal blood circulation in the craniopharyngioma.
[MeSH-major]
Craniopharyngioma / blood supply. Craniopharyngioma / metabolism.
Neoplasm
Recurrence, Local / etiology.
Pituitary
Neoplasms
/ blood supply.
Pituitary
Neoplasms
/ metabolism. Vascular Endothelial Growth Factor A / metabolism
Genetic Alliance.
consumer health - Craniopharyngioma
.
MedlinePlus Health Information.
consumer health - Pituitary Tumors
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16904996.001).
[ISSN]
0090-3019
[Journal-full-title]
Surgical neurology
[ISO-abbreviation]
Surg Neurol
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Vascular Endothelial Growth Factor A
23.
Marsh JC, Garg S, Wendt JA, Gielda BT, Turian JV, Herskovic AM:
Intracranial metastatic disease rarely involves the pituitary: retrospective analysis of 935 metastases in 155 patients and review of the literature.
Pituitary
; 2010 Sep;13(3):260-5
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Intracranial metastatic disease rarely involves the
pituitary
: retrospective analysis of 935 metastases in 155 patients and review of the literature.
We present a case report of a patient recently treated at our institution for an isolated non-small cell lung cancer metastatic lesion to the sella, report the lack of involvement of the
pituitary
gland
in a large single-institution series of treated intracranial parenchymal metastases, and review the pertinent literature.
Special attention was paid to the skull base to document the presence of any metastatic disease involving the
pituitary
gland
, infundibular stalk, sella turcica (including anterior and posterior clinoids), or diaphragm sellae.
We found no other involvement of the
pituitary
gland
or other sellar structures by metastatic disease in this series.
Intracranial metastatic disease rarely involves the
pituitary
gland
and infundibular stalk parenchyma, suggesting that this structure may be safely omitted from the treatment field during WBRT and prophylactic cranial irradiation (PCI).
[MeSH-major]
Brain
Neoplasms
/
diagnosis
. Brain
Neoplasms
/ secondary.
Pituitary
Gland
/ pathology
MedlinePlus Health Information.
consumer health - Brain Tumors
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
J Clin Endocrinol Metab. 2005 Dec;90(12):6355-60
[
16144946.001
]
[Cites]
Expert Rev Anticancer Ther. 2008 Dec;8(12):1931-8
[
19046113.001
]
[Cites]
Urology. 2004 Sep;64(3):589-90
[
15351603.001
]
[Cites]
Cancer. 1989 Jun 15;63(12):2404-8
[
2720586.001
]
[Cites]
Int J Radiat Oncol Biol Phys. 2007 Jul 15;68(4):978-85
[
17467925.001
]
[Cites]
Nat Clin Pract Endocrinol Metab. 2009 Feb;5(2):88-99
[
19165221.001
]
[Cites]
Pituitary. 2009;12(1):40-50
[
18270844.001
]
[Cites]
J Child Neurol. 2009 Nov;24(11):1418-30
[
19841429.001
]
[Cites]
Can J Neurol Sci. 2007 Aug;34(3):322-7
[
17803030.001
]
[Cites]
Urology. 2003 Aug;62(2):352
[
12893361.001
]
[Cites]
Recenti Prog Med. 2007 Feb;98(2):87-9
[
17439068.001
]
[Cites]
Rev Med Interne. 2009 May;30(5):425-9
[
19231038.001
]
[Cites]
J Thorac Cardiovasc Surg. 2001 Sep;122(3):548-53
[
11547308.001
]
[Cites]
Nihon Kokyuki Gakkai Zasshi. 2003 Jan;41(1):48-53
[
12693006.001
]
[Cites]
Endocr Dev. 2010;17:185-96
[
19955767.001
]
[Cites]
Arch Neurol. 1989 Apr;46(4):449-55
[
2650664.001
]
[Cites]
Lung Cancer. 1999 May;24(2):99-106
[
10444060.001
]
[Cites]
Int J Radiat Oncol Biol Phys. 1999 Oct 1;45(3):693-8
[
10524424.001
]
[Cites]
Endocr Dev. 2009;15:1-24
[
19293601.001
]
[Cites]
Ann Endocrinol (Paris). 2005 Apr;66(2 Pt 1):117-20
[
15959412.001
]
[Cites]
J Endocrinol Invest. 1992 Oct;15(9):677-81
[
1479150.001
]
[Cites]
Interact Cardiovasc Thorac Surg. 2009 Apr;8(4):467-73
[
19155223.001
]
[Cites]
Clin Endocrinol (Oxf). 2002 Dec;57(6):713-7
[
12460319.001
]
[Cites]
N Engl J Med. 1999 Aug 12;341(7):476-84
[
10441603.001
]
[Cites]
Tumori. 2008 Sep-Oct;94(5):765-8
[
19112958.001
]
[Cites]
Cancer. 1978 Jan;41(1):130-7
[
626923.001
]
[Cites]
Am J Ophthalmol. 1995 Jun;119(6):779-85
[
7785694.001
]
[Cites]
J Clin Endocrinol Metab. 2007 May;92(5):1666-72
[
17284618.001
]
[Cites]
Arch Neurol. 2009 Aug;66(8):1036-7
[
19667229.001
]
[Cites]
Intern Med. 1994 Dec;33(12):795-8
[
7718964.001
]
[Cites]
Neurol Med Chir (Tokyo). 2005 Aug;45(8):418-22
[
16127262.001
]
[Cites]
J Neurosurg. 2010 Nov;113(5):1059-71
[
19929198.001
]
[Cites]
Growth Horm IGF Res. 2004 Jun;14 Suppl A:S118-24
[
15135792.001
]
[Cites]
ANZ J Surg. 2005 Nov;75(11):963-6
[
16336388.001
]
[Cites]
J Clin Oncol. 2004 Dec 1;22(23):4851-3
[
15570089.001
]
[Cites]
Stroke. 1995 Jan;26(1):131-6
[
7839383.001
]
[Cites]
Int J Radiat Oncol Biol Phys. 2010 Nov 1;78(3):946-54
[
20472348.001
]
[Cites]
Int J Radiat Oncol Biol Phys. 2007 Oct 1;69(2):589-97
[
17869672.001
]
[Cites]
Neurol Med Chir (Tokyo). 1991 Jun;31(6):336-41
[
1724298.001
]
[Cites]
Int J Radiat Oncol Biol Phys. 2010 Apr;76(5):1480-5
[
19625140.001
]
[Cites]
Neuropediatrics. 2006 Dec;37(6):364-6
[
17357039.001
]
[Cites]
J Neurosurg. 1989 Jul;71(1):138-40
[
2661740.001
]
[Cites]
Pituitary. 2005;8(3-4):203-11
[
16508716.001
]
[Cites]
Radiat Oncol. 2009 Oct 14;4:42
[
19828022.001
]
[Cites]
Skull Base. 2009 Mar;19(2):133-40
[
19721769.001
]
[Cites]
Int J Radiat Oncol Biol Phys. 1997 Mar 1;37(4):745-51
[
9128946.001
]
[Cites]
Horm Res. 2008;69(2):65-74
[
18059086.001
]
[Cites]
Intern Med. 2002 Oct;41(10):834-8
[
12413005.001
]
[Cites]
Neurology. 1967 Dec;17(12):1190-2
[
6070020.001
]
[Cites]
J Child Neurol. 2009 Nov;24(11):1397-408
[
19841428.001
]
[Cites]
Surg Neurol. 1986 Jan;25(1):49-54
[
3941968.001
]
[Cites]
Int J Radiat Oncol Biol Phys. 2007 Jul 15;68(4):971-7
[
17446005.001
]
[Cites]
Eur J Cardiothorac Surg. 2004 Jun;25(6):1107-13
[
15145017.001
]
[Cites]
Neurosurg Focus. 2008;24(5):E2
[
18447741.001
]
[Cites]
Pediatr Neurosurg. 2003 Nov;39(5):264-9
[
14512691.001
]
[Cites]
Int J Radiat Oncol Biol Phys. 2010 Feb 1;76(2):504-12
[
20117288.001
]
[Cites]
Stroke. 1992 Jun;23 (6):908-11
[
1595114.001
]
[Cites]
J Child Neurol. 2009 Nov;24(11):1431-8
[
19841430.001
]
(PMID = 20405323.001).
[ISSN]
1573-7403
[Journal-full-title]
Pituitary
[ISO-abbreviation]
Pituitary
[Language]
eng
[Publication-type]
Case Reports; Journal Article; Review
[Publication-country]
United States
24.
Haap M, Gallwitz B, Meyermann R, Mittelbronn M:
Cushing's disease associated with both pituitary microadenoma and corticotroph hyperplasia.
Exp Clin Endocrinol Diabetes
; 2009 Jun;117(6):289-93
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Cushing's disease associated with both
pituitary
microadenoma and corticotroph hyperplasia.
MRI imaging revealed a possible
pituitary
microadenoma.
To confirm
the diagnosis
a bilateral inferior petrosal sinus sampling was performed presenting higher ACTH levels on the right side.
Histological examination of the
tumor
revealed a microadenoma.
Neuropathological autopsy revealed nodular proliferations of corticotropic cells in the
pituitary
gland
that are assumed to be morphological entities between diffuse hyperplasias and adenomas, termed as tumorlets.
In single reports, multiple
pituitary
lesions in patients with Cushing's disease have been demonstrated, but to our knowledge none of these cases presented the combination of an ACTH-producing microadenoma and corticotroph cell hyperplasia in the same patient.
Therefore, even after resection of a
pituitary
microadenoma one should be aware of the possibility of continuously elevated ACTH level being due to multifocal nodular corticotroph hyperplasia which is invisible by neuroradiological examination.
[MeSH-major]
ACTH-Secreting
Pituitary
Adenoma / pathology.
Pituitary
ACTH Hypersecretion / pathology.
Pituitary
Neoplasms
/ pathology
MedlinePlus Health Information.
consumer health - Pituitary Tumors
.
Hazardous Substances Data Bank.
HYDROCORTISONE
.
Hazardous Substances Data Bank.
Corticotropin
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[CommentIn]
Exp Clin Endocrinol Diabetes. 2010 Jan;118(1):68
[
20127571.001
]
(PMID = 19085700.001).
[ISSN]
1439-3646
[Journal-full-title]
Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association
[ISO-abbreviation]
Exp. Clin. Endocrinol. Diabetes
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Germany
[Chemical-registry-number]
9002-60-2 / Adrenocorticotropic Hormone; WI4X0X7BPJ / Hydrocortisone
25.
Hurty CA, Flatland B:
Feline acromegaly: a review of the syndrome.
J Am Anim Hosp Assoc
; 2005 Sep-Oct;41(5):292-7
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Acromegaly is characterized by chronic excessive growth hormone (GH) secretion by the
pituitary
gland
.
Feline acromegaly is most commonly caused by a functional
pituitary
tumor
.
Definitive
diagnosis
can be difficult because of the gradual disease onset, subtle clinical signs, unavailability of relevant laboratory tests, and client financial investment.
Diagnosis
is currently based upon brain imaging and measurement of serum GH and/or insulin-like growth factor-1 concentrations.
[MeSH-major]
Acromegaly / veterinary. Cat Diseases /
diagnosis
. Growth Hormone / secretion. Insulin-Like Growth Factor I / analysis
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16141180.001).
[ISSN]
1547-3317
[Journal-full-title]
Journal of the American Animal Hospital Association
[ISO-abbreviation]
J Am Anim Hosp Assoc
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Chemical-registry-number]
67763-96-6 / Insulin-Like Growth Factor I; 9002-72-6 / Growth Hormone
[Number-of-references]
48
26.
Voutetakis A, Sertedaki A, Livadas S, Xekouki P, Bossis I, Dacou-Voutetakis C, Argyropoulou MI:
Pituitary size fluctuation in long-term MR studies of PROP1 deficient patients: A persistent pathophysiological mechanism?
J Endocrinol Invest
; 2006 May;29(5):462-6
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Pituitary
size fluctuation in long-term MR studies of PROP1 deficient patients: A persistent pathophysiological mechanism?
Inactivating PROP1 gene alterations are responsible for over 50% of familial combined
pituitary
hormone deficiency cases.
Pituitary
enlargement followed by regression and subnormal
pituitary
size has been documented in a number of PROP1 deficient patients.
Nevertheless, long-term magnetic resonance imaging (MRI) findings in two PROP1 deficient patients suggest the evolution of
pituitary
pathology as more complex and persistent than previously described.
Patient A had enlarged
pituitary
gland
(
pituitary
height: 9-10 mm), demonstrated by serial MRI carried out from age 5 to 8.5 yr, small
pituitary
gland
(4 mm) at age 10 yr and
pituitary
enlargement (11 mm) at age 19 yr.
Patient B had a
pituitary
gland of
normal size at age 7 yr (5 mm), whereas at age 14.3 and 16.3 yr, an enlarged
pituitary
gland
was disclosed (10 and 11 mm, respectively).
Both series of events are suggestive of a persistent pathophysiological mechanism in the
pituitary
gland of
patients with PROP1 gene defects.
Therefore, long-term
pituitary
follow-up by MRI in such patients may be necessary even in the case of a small or normal
pituitary
gland
.
It must be noted that current data from the Ames dwarf mouse cannot fully explain the observed
pituitary
size fluctuation.
[MeSH-major]
Homeodomain Proteins / genetics.
Pituitary
Diseases / physiopathology.
Pituitary
Gland
/ pathology.
Pituitary
Hormones / deficiency
MedlinePlus Health Information.
consumer health - Pituitary Disorders
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Endocr Rev. 2002 Aug;23(4):431-42
[
12202459.001
]
[Cites]
Pediatr Radiol. 1991;21(4):247-9
[
1870916.001
]
[Cites]
Development. 2002 Sep;129(18):4229-39
[
12183375.001
]
[Cites]
AJR Am J Roentgenol. 2000 Feb;174(2):555-9
[
10658742.001
]
[Cites]
Clin Endocrinol (Oxf). 2002 Aug;57(2):283-91
[
12153609.001
]
[Cites]
Genes Dev. 2001 Dec 1;15(23 ):3193-207
[
11731482.001
]
[Cites]
J Clin Endocrinol Metab. 2001 Sep;86(9):4353-7
[
11549674.001
]
[Cites]
J Clin Endocrinol Metab. 1999 Dec;84(12 ):4362-70
[
10599689.001
]
[Cites]
Mol Endocrinol. 2005 Mar;19(3):698-710
[
15591534.001
]
[Cites]
Clin Endocrinol (Oxf). 2005 Jul;63(1):10-8
[
15963055.001
]
[Cites]
Nat Genet. 1998 Feb;18(2):147-9
[
9462743.001
]
[Cites]
J Clin Endocrinol Metab. 1998 Oct;83(10):3727-34
[
9768691.001
]
[Cites]
J Clin Endocrinol Metab. 2004 May;89(5):2200-6
[
15126542.001
]
[Cites]
J Clin Endocrinol Metab. 2004 Oct;89(10):5256-65
[
15472232.001
]
[Cites]
J Clin Endocrinol Metab. 1999 Mar;84(3):942-5
[
10084575.001
]
[Cites]
Science. 2002 Mar 22;295(5563):2231-5
[
11910101.001
]
[Cites]
Nature. 1996 Nov 28;384(6607):327-33
[
8934515.001
]
[Cites]
N Engl J Med. 1978 Mar 30;298(13):698-702
[
628396.001
]
(PMID = 16794371.001).
[ISSN]
0391-4097
[Journal-full-title]
Journal of endocrinological investigation
[ISO-abbreviation]
J. Endocrinol. Invest.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Italy
[Chemical-registry-number]
0 / Homeodomain Proteins; 0 / Pituitary Hormones; 0 / Prophet of Pit-1 protein
27.
Kondrat'ev BV, Vinogradov VM, Shalek RA, Ialynych NN, Kopaneva MV:
[Proton irradiation of the pituitary gland for alleviating pain in patients with disseminated prostate cancer].
Vopr Onkol
; 2006;52(1):92-4
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
[Proton irradiation of the
pituitary
gland
for alleviating pain in patients with disseminated prostate cancer].
Central Research Institute of Roentgeno-Radiology; Medical Academy for Further Education, St. Petersburg Stereotactic ablation of the frontal lobe of the
pituitary
with narrow beams of 1,000 MeV protons was performed in 80 patients to alleviate pain caused by bone metastases.
[MeSH-major]
Analgesics / administration & dosage. Bone
Neoplasms
/ complications. Pain / radiotherapy.
Pituitary
Gland
, Anterior / radiation effects. Prostatic
Neoplasms
/ pathology. Protons / therapeutic use
Genetic Alliance.
consumer health - Pituitary cancer
.
Genetic Alliance.
consumer health - Prostate cancer
.
MedlinePlus Health Information.
consumer health - Bone Cancer
.
MedlinePlus Health Information.
consumer health - Pain
.
MedlinePlus Health Information.
consumer health - Prostate Cancer
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16715713.001).
[ISSN]
0507-3758
[Journal-full-title]
Voprosy onkologii
[ISO-abbreviation]
Vopr Onkol
[Language]
rus
[Publication-type]
English Abstract; Journal Article
[Publication-country]
Russia (Federation)
[Chemical-registry-number]
0 / Analgesics; 0 / Analgesics, Opioid; 0 / Protons
28.
Chambery A, Parente A, Topo E, Garcia-Fernàndez J, D'Aniello S:
Characterization and putative role of a type I gonadotropin-releasing hormone in the cephalochordate amphioxus.
Endocrinology
; 2009 Feb;150(2):812-20
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Employing reverse-phase chromatography, we purified a peptide of relative molecular mass of 1182.60
Da
from the cephalochordate amphioxus Branchiostoma lanceolatum.
Furthermore, the biological activity of amphioxus GnRH was investigated by examining its capability to elicit LH release from the rodent
pituitary
gland
.
The seasonal variations of amphioxus GnRH also suggest an ancient role of this peptide in the control of reproduction in chordates, even before the evolution of a proper
pituitary
gland
.
[MeSH-minor]
Animals. Luteinizing Hormone / metabolism. Phylogeny.
Pituitary
Gland
/ metabolism. Rats. Rats, Wistar. Seasons
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18927217.001).
[ISSN]
1945-7170
[Journal-full-title]
Endocrinology
[ISO-abbreviation]
Endocrinology
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
33515-09-2 / Gonadotropin-Releasing Hormone; 9002-67-9 / Luteinizing Hormone
29.
Maddineni S, Ocón-Grove OM, Krzysik-Walker SM, Hendricks GL 3rd, Proudman JA, Ramachandran R:
Gonadotrophin-inhibitory hormone receptor expression in the chicken pituitary gland: potential influence of sexual maturation and ovarian steroids.
J Neuroendocrinol
; 2008 Sep;20(9):1078-88
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Gonadotrophin-inhibitory hormone receptor expression in the chicken
pituitary
gland
: potential influence of sexual maturation and ovarian steroids.
Gonadotrophin-inhibitory hormone (GnIH), a hypothalamic RFamide, has been found to inhibit gonadotrophin secretion from the anterior
pituitary
gland
originally in birds and, subsequently, in mammalian species.
The gene encoding a transmembrane receptor for GnIH (GnIHR) was recently identified in the brain,
pituitary
gland
and gonads of song bird, chicken and Japanese quail.
The objectives of the present study are to characterise the expression of GnIHR mRNA and protein in the chicken
pituitary
gland
, and to determine whether sexual maturation and gonadal steroids influence
pituitary
GnIHR mRNA abundance.
GnIHR mRNA quantity was found to be significantly higher in diencephalon compared to either anterior
pituitary
gland
or ovaries.
Oestradiol or a combination of oestradiol and progesterone treatment caused a significant decrease in
pituitary
GnIHR mRNA quantity relative to vehicle controls.
GnIHR-immunoreactive (ir) cells were identified in the chicken
pituitary
gland
cephalic and caudal lobes.
GnIH treatment significantly decreased LH release from anterior
pituitary
gland
slices collected from sexually immature, but not from sexually mature chickens.
[MeSH-major]
Avian Proteins / genetics. Chickens / genetics. Gonadal Steroid Hormones / pharmacology.
Pituitary
Gland
/ metabolism. Receptors, Cell Surface / genetics. Sexual Maturation / physiology
COS Scholar Universe.
author profiles
.
Hazardous Substances Data Bank.
ESTRADIOL
.
Hazardous Substances Data Bank.
PROGESTERONE
.
Hazardous Substances Data Bank.
MENOTROPINS
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18638025.001).
[ISSN]
1365-2826
[Journal-full-title]
Journal of neuroendocrinology
[ISO-abbreviation]
J. Neuroendocrinol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
[Publication-country]
England
[Chemical-registry-number]
0 / Avian Proteins; 0 / Gonadal Steroid Hormones; 0 / RNA, Messenger; 0 / Receptors, Cell Surface; 4G7DS2Q64Y / Progesterone; 4TI98Z838E / Estradiol; 9002-67-9 / Luteinizing Hormone; 9002-68-0 / Follicle Stimulating Hormone
30.
Reith W:
[Tumors in the region of the sella turcica].
Radiologe
; 2009 Jul;49(7):624-31
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
[
Tumors
in the region of the sella turcica].
Tumors of
the
pituitary
gland
can lead to limitation
of hypophysis
function (
hypophysis
insufficiency) or hypersecretion of different hormones (acromegaly, Cushing's syndrome, prolactinoma, TSH-secreting adenoma).
The optic chiasma lies in close proximity to the
pituitary
gland
and can be compressed by
tumors
leading to visual disturbances (bilateral hemianopsia).
Tumors
can be separated into hormone secreting and hormone inactive
tumors
, as well as into microadenoma with a diameter <10 mm and macroadenomas >10 mm.
A rare group
of tumors
of the
hypophysis
region are craniopharyngiomas, meningiomas, germinomas, gliomas, metastases and granulomotous inflammations, such as sarcoidosis and tuberculosis.
[MeSH-major]
Magnetic Resonance Imaging / methods.
Pituitary
Neoplasms
/
diagnosis
. Sella Turcica / diagnostic imaging. Sella Turcica / pathology. Skull
Neoplasms
/
diagnosis
. Tomography, X-Ray Computed / methods
MedlinePlus Health Information.
consumer health - CT Scans
.
MedlinePlus Health Information.
consumer health - MRI Scans
.
MedlinePlus Health Information.
consumer health - Pituitary Tumors
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Clin Radiol. 2007 May;62(5):453-62
[
17398271.001
]
[Cites]
Eur Radiol. 1999;9(5):918-23
[
10369991.001
]
[Cites]
J Neurosurg. 2008 Dec;109(6):1180-2; author reply 1182-3
[
19035739.001
]
[Cites]
J Pediatr Endocrinol Metab. 2002 Feb;15(2):157-62
[
11874180.001
]
[Cites]
AJR Am J Roentgenol. 2003 Aug;181(2):577-82
[
12876051.001
]
[Cites]
Indian J Pathol Microbiol. 2008 Apr-Jun;51(2):269-70
[
18603706.001
]
[Cites]
Neuroradiology. 2007 Apr;49(4):327-33
[
17200863.001
]
[Cites]
Eur J Clin Invest. 2007 Jul;37(7):552-7
[
17576206.001
]
[Cites]
Surg Neurol. 2007 Mar;67(3):251-7; discussion 257
[
17320630.001
]
[Cites]
Neurosurg Focus. 1996 Jul 15;1(1):e7
[
15096000.001
]
[Cites]
J Clin Neurosci. 2009 Mar;16(3):385-9
[
19147363.001
]
[Cites]
Acta Neurochir (Wien). 2007 Aug;149(8):759-69; discussion 769
[
17594050.001
]
[Cites]
Acta Neurochir (Wien). 2008 Nov;150(11):1193-6; discussion 1196
[
18958393.001
]
[Cites]
Rev Endocr Metab Disord. 2008 Mar;9(1):13-9
[
18236162.001
]
(PMID = 19568729.001).
[ISSN]
1432-2102
[Journal-full-title]
Der Radiologe
[ISO-abbreviation]
Radiologe
[Language]
ger
[Publication-type]
Journal Article; Review
[Publication-country]
Germany
[Number-of-references]
17
31.
Herrick J, Shecterle LM, St Cyr JA:
D-ribose--an additive with caffeine.
Med Hypotheses
; 2009 May;72(5):499-500
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Researchers have proposed mechanisms responsible for caffeine's interactions, which include its blocking capacity of adenosine receptors, its role with the
pituitary
gland
, increasing levels of dopamine, and its role with the intracellular release of calcium from the sarcoplasmic reticulum, which is dependent on intracellular adenosine triphosphate levels.
MedlinePlus Health Information.
consumer health - Caffeine
.
Hazardous Substances Data Bank.
CAFFEINE
.
The Lens.
Cited by Patents in
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19223125.001).
[ISSN]
1532-2777
[Journal-full-title]
Medical hypotheses
[ISO-abbreviation]
Med. Hypotheses
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Scotland
[Chemical-registry-number]
3G6A5W338E / Caffeine; 681HV46001 / Ribose
32.
de Lange TE, Simsek S, Kramer MH, Nanayakkara PW:
A case of cocaine-induced panhypopituitarism with human neutrophil elastase-specific anti-neutrophil cytoplasmic antibodies.
Eur J Endocrinol
; 2009 Mar;160(3):499-502
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Magnetic resonance imaging and computed tomography scan showed a normal-sized
pituitary
gland
entirely embedded in a dense, oedematous, contrast-enhancing mass, and a total destruction of the nasal septum with the absence of conchae and severely impaired sinus walls.
Genetic Alliance.
consumer health - Pituitary hormone deficiency, combined 2
.
MedlinePlus Health Information.
consumer health - Cocaine
.
Hazardous Substances Data Bank.
COCAINE
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19114541.001).
[ISSN]
1479-683X
[Journal-full-title]
European journal of endocrinology
[ISO-abbreviation]
Eur. J. Endocrinol.
[Language]
ENG
[Publication-type]
Case Reports; Journal Article
[Publication-country]
England
[Chemical-registry-number]
0 / Antibodies, Antineutrophil Cytoplasmic; 0 / Vasoconstrictor Agents; EC 3.4.21.37 / Leukocyte Elastase; I5Y540LHVR / Cocaine
33.
Assié G, Bahurel H, Coste J, Silvera S, Kujas M, Dugué MA, Karray F, Dousset B, Bertherat J, Legmann P, Bertagna X:
Corticotroph tumor progression after adrenalectomy in Cushing's Disease: A reappraisal of Nelson's Syndrome.
J Clin Endocrinol Metab
; 2007 Jan;92(1):172-9
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Corticotroph
tumor
progression after adrenalectomy in Cushing's Disease: A reappraisal of Nelson's Syndrome.
However, it may lead to Nelson's syndrome, originally defined by the association of a
pituitary
macroadenoma and high plasma ACTH concentrations, a much feared complication.
OBJECTIVE: The objective of the study was to reconsider Nelson's syndrome by investigating corticotroph
tumor
progression based on
pituitary
magnetic resonance imaging scan and search for predictive factors.
PATIENTS: Patients included 53 Cushing's disease patients treated by adrenalectomy between 1991 and 2002, without previous
pituitary
irradiation.
MEASUREMENTS: Clinical data,
pituitary
magnetic resonance imaging data, and plasma ACTH concentrations for all patients and
pituitary
gland
pathology data for 25 patients were recorded.
Corticotroph
tumor
progression-free survival was studied by Kaplan-Meier, and the influence of recorded parameters was studied by Cox regression.
RESULTS: Corticotroph
tumor
progression ultimately occurred in half the patients, generally within 3 yr after adrenalectomy.
A shorter duration of Cushing's disease (adjusted hazard ratio: 0.884/yr), and a high plasma ACTH concentration in the year after adrenalectomy [adjusted hazard ratio per 100 pg/ml (22 pmol/liter): 1.069] were predictive of corticotroph
tumor
progression.
In one case, corticotroph
tumor
progression was complicated by transitory oculomotor nerve palsy.
During follow-up, corticotroph
tumor
progression was associated with the increase of corresponding ACTH concentrations (odds ratio per 100 pg/ml of ACTH variation: 1.055).
CONCLUSION: After adrenalectomy in Cushing's disease, one should no longer wait for the occurrence of Nelson's syndrome: modern imaging allows early detection and management of corticotroph
tumor
progression.
[MeSH-major]
Adrenalectomy / adverse effects. Nelson Syndrome / etiology.
Pituitary
ACTH Hypersecretion / surgery
Genetic Alliance.
consumer health - Cushing's Syndrome
.
Faculty of 1000.
commentaries/discussion - See the articles recommended by F1000Prime's Faculty of more than 8,000 leading experts in Biology and Medicine.
(subscription/membership/fee required).
Hazardous Substances Data Bank.
Corticotropin
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17062771.001).
[ISSN]
0021-972X
[Journal-full-title]
The Journal of clinical endocrinology and metabolism
[ISO-abbreviation]
J. Clin. Endocrinol. Metab.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
9002-60-2 / Adrenocorticotropic Hormone
34.
Aleem M, Choudhari J, Padwal V, Balasinor N, Parte P, Gill-Sharma MK:
Hyperprolactinemia affects spermiogenesis in adult male rats.
J Endocrinol Invest
; 2005 Jan;28(1):39-48
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
In our earlier study in adult male rats, we reported that fluphenazine at a dose of 3 mg/kg/day suppressed serum FSH but not testosterone (T) through increasing dopamine (
DA
) metabolism in the
pituitary
gland
, within 60 days.
Hazardous Substances Data Bank.
Fluphenazine
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Biol Reprod. 1989 May;40(5):1037-45
[
2765609.001
]
[Cites]
Andrologia. 1996 Jul-Aug;28(4):197-202
[
8844112.001
]
[Cites]
Nature. 1970 Aug 15;227(5259):680-5
[
5432063.001
]
[Cites]
Clin Endocrinol (Oxf). 1988 Jul;29(1):77-112
[
3073881.001
]
[Cites]
Biol Reprod. 2000 May;62(5):1146-59
[
10775161.001
]
[Cites]
Nature. 1992 Jan 2;355(6355):80-4
[
1370576.001
]
[Cites]
Mol Cell Endocrinol. 1982 Jan;25(1):25-33
[
6802692.001
]
[Cites]
Biol Reprod. 2004 Jul;71(1):117-29
[
14998910.001
]
[Cites]
Int J Fertil. 1991 Jul-Aug;36(4):243-51
[
1680827.001
]
[Cites]
Endocrinology. 1992 Sep;131(3):1343-9
[
1324158.001
]
[Cites]
Mol Cell Endocrinol. 2002 Dec 30;198(1-2):131-41
[
12573823.001
]
[Cites]
J Clin Endocrinol Metab. 2004 Feb;89(2):621-5
[
14764772.001
]
[Cites]
J Reprod Fertil Suppl. 1979;(26):175-81
[
293408.001
]
[Cites]
Biol Reprod. 1998 Aug;59(2):379-87
[
9687311.001
]
[Cites]
Endocrinology. 2000 Mar;141(3):1168-77
[
10698194.001
]
[Cites]
Proc Natl Acad Sci U S A. 2000 Apr 25;97(9):4683-8
[
10781074.001
]
[Cites]
Proc Natl Acad Sci U S A. 1995 Dec 19;92(26):12451-5
[
8618919.001
]
[Cites]
Exp Cell Res. 1996 Jun 15;225(2):374-81
[
8660926.001
]
[Cites]
J Steroid Biochem Mol Biol. 1995 Jun;53(1-6):561-5
[
7626510.001
]
[Cites]
J Clin Endocrinol Metab. 1998 Oct;83(10):3722-6
[
9768690.001
]
[Cites]
Biol Reprod. 2001 Oct;65(4):1201-7
[
11566744.001
]
[Cites]
Nature. 1993 Mar 18;362(6417):264-7
[
7681549.001
]
[Cites]
J Reprod Fertil Suppl. 1979;(26):147-63
[
118251.001
]
[Cites]
Endocrinology. 1984 Apr;114(4):1419-25
[
6538478.001
]
[Cites]
J Androl. 1985 May-Jun;6(3):179-89
[
4039718.001
]
[Cites]
Mol Reprod Dev. 2001 Apr;58(4):357-8
[
11241770.001
]
[Cites]
Nature. 1996 Mar 14;380(6570):159-62
[
8600390.001
]
[Cites]
Mol Reprod Dev. 2001 Apr;58(4):437-43
[
11241781.001
]
[Cites]
J Androl. 2002 Sep-Oct;23(5):598-609
[
12185088.001
]
[Cites]
Folia Histochem Cytobiol. 2002;40(2):163-4
[
12056626.001
]
[Cites]
Proc Natl Acad Sci U S A. 1998 Nov 10;95(23):13612-7
[
9811848.001
]
[Cites]
Mol Endocrinol. 1997 Sep;11(10):1415-24
[
9280057.001
]
[Cites]
Fed Proc. 1978 Sep;37(11):2522-5
[
357187.001
]
[Cites]
Mol Reprod Dev. 2004 Jul;68(3):269-78
[
15112319.001
]
[Cites]
Endocrinology. 1984 Apr;114(4):1413-8
[
6538477.001
]
[Cites]
Nature. 1996 Mar 14;380(6570):162-5
[
8600391.001
]
[Cites]
J Biol Chem. 1951 Nov;193(1):265-75
[
14907713.001
]
[Cites]
J Endocrinol Invest. 2003 Apr;26(4):316-26
[
12841539.001
]
[Cites]
Vitam Horm. 1994;49:197-280
[
7810071.001
]
[Cites]
Biol Chem Hoppe Seyler. 1989 Apr;370(4):293-301
[
2757789.001
]
[Cites]
Mol Cell Endocrinol. 1993 Oct;96(1-2):69-73
[
8276140.001
]
[Cites]
J Clin Endocrinol Metab. 1990 Aug;71(2):398-404
[
2166070.001
]
[Cites]
J Endocrinol Invest. 2001 Sep;24(8):598-607
[
11686542.001
]
[Cites]
Cell Tissue Res. 1985;239(2):443-5
[
2983897.001
]
(PMID = 15816370.001).
[ISSN]
0391-4097
[Journal-full-title]
Journal of endocrinological investigation
[ISO-abbreviation]
J. Endocrinol. Invest.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Italy
[Chemical-registry-number]
0 / Antipsychotic Agents; 0 / Chromosomal Proteins, Non-Histone; 0 / DNA-Binding Proteins; 0 / Protamines; 0 / Sulfhydryl Compounds; 0 / Transcription Factors; 0 / spermatid transition proteins; 135844-64-3 / Cyclic AMP Response Element Modulator; E0399OZS9N / Cyclic AMP; S79426A41Z / Fluphenazine
35.
Mai PL, Korde L, Kramer J, Peters J, Mueller CM, Pfeiffer S, Stratakis CA, Pinto PA, Bratslavsky G, Merino M, Choyke P, Linehan WM, Greene MH:
A possible new syndrome with growth-hormone secreting pituitary adenoma, colonic polyposis, lipomatosis, lentigines and renal carcinoma in association with familial testicular germ cell malignancy: A case report.
J Med Case Rep
; 2007 Mar 28;1:9
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
A possible new syndrome with growth-hormone secreting
pituitary
adenoma, colonic polyposis, lipomatosis, lentigines and renal carcinoma in association with familial testicular germ cell malignancy: A case report.
BACKGROUND: Germ-cell testicular cancer has not been definitively linked to any known hereditary cancer susceptibility
disorder
.
His evaluation as part of an etiologic study of familial testicular cancer revealed multiple colon polyps (adenomatous, hyperplastic, and hamartomatous) first found in his 50 s, multiple lipomas, multiple hyperpigmented skin lesions, left kidney cancer diagnosed at age 64, and a growth-hormone producing
pituitary
adenoma with associated acromegaly diagnosed at age 64.
The patient underwent genetic testing for Cowden syndrome (PTEN gene), Carney complex (PRKAR1A gene), and multiple endocrine
neoplasia
syndrome type 1 (MEN1 gene); no deleterious mutations were identified.
DISCUSSION: The constellation of
benign
and malignant
neoplasms
in the context of this patient's familial testicular cancer raised the possibility that these might be manifestations of a known hereditary susceptibility cancer syndrome; however, genetic testing for the three syndromes that were most likely to explain these findings did not show any mutation.
Alternatively, this family's phenotype might represent a novel
neoplasm
susceptibility
disorder
.
COS Scholar Universe.
author profiles
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Br J Neurosurg. 2004 Dec;18(6):629-31
[
15799199.001
]
[Cites]
Horm Res. 2004;62 Suppl 1:108-15
[
15761242.001
]
[Cites]
Am J Gastroenterol. 2005 Feb;100(2):476-90
[
15667510.001
]
[Cites]
Dig Dis Sci. 2004 Apr;49(4):662-6
[
15185875.001
]
[Cites]
Lancet Oncol. 2004 Jun;5(6):363-71
[
15172357.001
]
[Cites]
Cancer. 2003 Feb 15;97(4):984-92
[
12569597.001
]
[Cites]
J Clin Endocrinol Metab. 2001 Dec;86(12):5658-71
[
11739416.001
]
[Cites]
J Clin Endocrinol Metab. 2001 Sep;86(9):4041-6
[
11549623.001
]
[Cites]
J Neurosurg. 2000 Mar;92(3):413-8
[
10701527.001
]
[Cites]
J Natl Cancer Inst. 2005 Sep 21;97(18):1354-65
[
16174857.001
]
[Cites]
Science. 2006 May 26;312(5777):1228-30
[
16728643.001
]
[Cites]
Hum Mol Genet. 2006 Feb 1;15(3):443-51
[
16407372.001
]
(PMID = 17411461.001).
[Journal-full-title]
Journal of medical case reports
[ISO-abbreviation]
J Med Case Rep
[Language]
ENG
[Grant]
United States / NCI NIH HHS / CP / N02CP11019
[Publication-type]
Journal Article
[Publication-country]
England
[Other-IDs]
NLM/ PMC1847830
36.
Nagai Y, Aso H, Ogasawara H, Tanaka S, Taketa Y, Watanabe K, Ohwada S, Rose MT, Kitazawa H, Yamaguchi T:
Anterior pituitary progenitor cells express costimulatory molecule 4Ig-B7-H3.
J Immunol
; 2008 Nov 1;181(9):6073-81
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Anterior
pituitary
progenitor cells express costimulatory molecule 4Ig-B7-H3.
Stem/Progenitor cells in the postnatal
pituitary
gland
are embedded in a marginal cell layer around Rathke's pouch.
However, the nature and behavior of anterior
pituitary
progenitor cells remain unclear.
We established bovine anterior
pituitary
progenitor cell line (BAPC)-1 from the anterior
pituitary
gland
, which expressed stem/progenitor cell-related genes and several inflammatory cytokines.
To characterize and localize these
pituitary
progenitor cells, we produced a mAb (12B mAb) against BAPC-1.
The 12B-immunoreactive cells in the bovine anterior
pituitary
gland
were localized around Rathke's pouch and expressed IL-18 and MHC class II.
In addition, the 12B-immunoreactive cells were observed around the pars tuberalis closely bordering the median eminence and in the blood vessels of the primary portal plexus in the anterior
pituitary
gland
.
[MeSH-major]
Antigens, CD / genetics.
Pituitary
Gland
, Anterior / immunology.
Pituitary
Gland
, Anterior / metabolism. Receptors, Immunologic / genetics. Stem Cells / immunology. Stem Cells / metabolism
MedlinePlus Health Information.
consumer health - Stem Cells
.
COS Scholar Universe.
author profiles
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18941196.001).
[ISSN]
1550-6606
[Journal-full-title]
Journal of immunology (Baltimore, Md. : 1950)
[ISO-abbreviation]
J. Immunol.
[Language]
eng
[Databank-accession-numbers]
GENBANK/ AB271019; RefSeq/ NP/ 001019907
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Antigens, CD; 0 / B7 Antigens; 0 / CD276 protein, human; 0 / RNA, Messenger; 0 / Receptors, Immunologic
37.
Katoh M, Katoh M:
Comparative integromics on Eph family.
Int J Oncol
; 2006 May;28(5):1243-7
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Human EPHA7 mRNA was expressed in embryonic stem (ES) cells, neural tissues, duodenal cancer and parathyroid
tumors
, while mouse Epha7 mRNA was expressed in fertilized egg, Rathke's pouche, visual cortex,
pituitary
gland
, other neural tissues, pancreas, lung
tumors
and mammary
tumors
.
Deletion and/or promoter CpG hypermethylation could explain the EPHA7 down-regulation in human
tumors
.
Mouse Genome Informatics (MGI).
Mouse Genome Informatics (MGI)
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16596241.001).
[ISSN]
1019-6439
[Journal-full-title]
International journal of oncology
[ISO-abbreviation]
Int. J. Oncol.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Greece
[Chemical-registry-number]
0 / Ligands; EC 2.7.10.1 / Receptors, Eph Family
38.
Kaur A, Agrawal A, Mittal M:
Presumed pituitary abscess without infectious source treated successfully with antibiotics alone.
J Neuroophthalmol
; 2005 Sep;25(3):185-8
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Presumed
pituitary
abscess without infectious source treated successfully with antibiotics alone.
A presumptive
diagnosis of
pituitary
abscess was made on the basis of suggestive magnetic resonance imaging findings.
These consisted of a large non-enhancing area within the
pituitary
gland
and thin irregular glandular rim enhancement.
This case highlights the need for a high index of suspicion for
pituitary
abscess based on unusual imaging findings even when there is no source of infection.
[MeSH-major]
Anti-Bacterial Agents / therapeutic use. Brain Abscess / drug therapy.
Pituitary
Diseases / drug therapy
MedlinePlus Health Information.
consumer health - Antibiotics
.
MedlinePlus Health Information.
consumer health - Pituitary Disorders
.
Hazardous Substances Data Bank.
CEFAZOLIN
.
Hazardous Substances Data Bank.
AMIKACIN
.
Hazardous Substances Data Bank.
CHLORAMPHENICOL
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16148624.001).
[ISSN]
1070-8022
[Journal-full-title]
Journal of neuro-ophthalmology : the official journal of the North American Neuro-Ophthalmology Society
[ISO-abbreviation]
J Neuroophthalmol
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Anti-Bacterial Agents; 280111G160 / Cefadroxil; 66974FR9Q1 / Chloramphenicol; 84319SGC3C / Amikacin; IHS69L0Y4T / Cefazolin
39.
Jethwa PH, Ebling FJ:
Role of VGF-derived peptides in the control of food intake, body weight and reproduction.
Neuroendocrinology
; 2008;88(2):80-7
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
The VGF gene is expressed abundantly in the brain, and in peripheral endocrine tissues including the
pituitary
gland
, the adrenal glands and the pancreas but also in the gastrointestinal tract in both the myenteric plexus and in endocrine cells.
MedlinePlus Health Information.
consumer health - Body Weight
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Copyright]
(c) 2008 S. Karger AG, Basel.
(PMID = 18408361.001).
[ISSN]
1423-0194
[Journal-full-title]
Neuroendocrinology
[ISO-abbreviation]
Neuroendocrinology
[Language]
eng
[Grant]
United Kingdom / Biotechnology and Biological Sciences Research Council / / BB/D525064/1; United Kingdom / Biotechnology and Biological Sciences Research Council / / S17106; United Kingdom / Biotechnology and Biological Sciences Research Council / / BBS/B/10765
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
Switzerland
[Chemical-registry-number]
0 / Neuropeptides; 0 / VGF peptide; 0 / Vgf protein, mouse
[Number-of-references]
41
40.
Bakoto N, Strivay M:
[Germinoma responsible for central diabetes insipidus].
Rev Med Liege
; 2009 Jul-Aug;64(7-8):386-9
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
A
diagnosis of
diabetes insipidus was confirmed and the MRI showed a
pituitary
stalk enlargement.
The MRI showed an intrasellar mass with an enlargement of the
pituitary
gland
.
The differential
diagnosis
will be reviewed.
[MeSH-major]
Diabetes Insipidus / etiology. Germinoma / complications.
Pituitary
Neoplasms
/ complications
[MeSH-minor]
Adult. Anti-Inflammatory Agents / therapeutic use. Antidiuretic Agents / therapeutic use. Chemotherapy, Adjuvant. Deamino Arginine Vasopressin / therapeutic use.
Diagnosis
, Differential. Drug Therapy, Combination. Female. Humans. Hydrocortisone / therapeutic use. Hypopituitarism / etiology. Polyuria / etiology. Radiotherapy, Adjuvant. Thyroxine / therapeutic use. Treatment Outcome
Genetic Alliance.
consumer health - Diabetes
.
Genetic Alliance.
consumer health - Diabetes Insipidus
.
Genetic Alliance.
consumer health - Germinoma
.
MedlinePlus Health Information.
consumer health - Diabetes Insipidus
.
MedlinePlus Health Information.
consumer health - Pituitary Tumors
.
Hazardous Substances Data Bank.
HYDROCORTISONE
.
Hazardous Substances Data Bank.
LEVOTHYROXINE
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19777917.001).
[ISSN]
0370-629X
[Journal-full-title]
Revue médicale de Liège
[ISO-abbreviation]
Rev Med Liege
[Language]
fre
[Publication-type]
Case Reports; English Abstract; Journal Article
[Publication-country]
Belgium
[Chemical-registry-number]
0 / Anti-Inflammatory Agents; 0 / Antidiuretic Agents; ENR1LLB0FP / Deamino Arginine Vasopressin; Q51BO43MG4 / Thyroxine; WI4X0X7BPJ / Hydrocortisone
41.
Schaeffer M, Hodson DJ, Lafont C, Mollard P:
Functional importance of blood flow dynamics and partial oxygen pressure in the anterior pituitary.
Eur J Neurosci
; 2010 Dec;32(12):2087-95
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Functional importance of blood flow dynamics and partial oxygen pressure in the anterior
pituitary
.
With a particular focus on the
pituitary
gland
as a model system, we review here the importance of the interplay between blood flow regulation and oxygen tensions in the functioning of endocrine systems, and the known regulatory signals involved in the modification of flow patterns under both normal physiological and pathological conditions.
[MeSH-major]
Oxygen / blood.
Pituitary
Gland
, Anterior / blood supply. Regional Blood Flow / physiology
[MeSH-minor]
Animals. Endocrine System / blood supply. Endocrine System / metabolism. Humans. Oxygen Consumption. Partial Pressure.
Pituitary
Hormones, Anterior / secretion
Hazardous Substances Data Bank.
OXYGEN
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Copyright]
© 2010 The Authors. European Journal of Neuroscience © 2010 Federation of European Neuroscience Societies and Blackwell Publishing Ltd.
(PMID = 21143663.001).
[ISSN]
1460-9568
[Journal-full-title]
The European journal of neuroscience
[ISO-abbreviation]
Eur. J. Neurosci.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
France
[Chemical-registry-number]
0 / Pituitary Hormones, Anterior; S88TT14065 / Oxygen
42.
So G, Takeshita T, Morofuji Y, Iseki M, Hayashi T, Matsuo T, Suyama K, Nagata I:
[Nonfunctioning suprasellar ectopic pituitary adenoma. A case report].
No Shinkei Geka
; 2008 Dec;36(12):1121-5
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
[Nonfunctioning suprasellar ectopic
pituitary
adenoma. A case report].
A case of nonfunctioning suprasellar ectopic
pituitary
adenoma in a 49-year-old man is reported.
MR imagings revealed an enhancing suprasellar
tumor
and a normal
pituitary
gland
in the sella turcica.
The patient underwent a craniotomy and
the tumor
was partially removed, although it was firmly attached to the
pituitary
stalk.
The tumor
was diagnosed histologically as a nonfunctioning
pituitary
adenoma.
Cases of nonfunctioning suprasellar ectopic
pituitary
adenoma have rarely been reported.
The pathophysiology of such
tumors
is discussed.
[MeSH-major]
Adenoma /
diagnosis
. Choristoma /
diagnosis
.
Pituitary
Neoplasms
/
diagnosis
. Sella Turcica. Skull
Neoplasms
/
diagnosis
[MeSH-minor]
Craniotomy. Humans. Magnetic Resonance Imaging. Male. Middle Aged.
Pituitary
Gland
MedlinePlus Health Information.
consumer health - Pituitary Tumors
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19086443.001).
[ISSN]
0301-2603
[Journal-full-title]
No shinkei geka. Neurological surgery
[ISO-abbreviation]
No Shinkei Geka
[Language]
jpn
[Publication-type]
Case Reports; English Abstract; Journal Article; Review
[Publication-country]
Japan
[Number-of-references]
22
43.
Skinner DC:
Rethinking the stalk effect: a new hypothesis explaining suprasellar tumor-induced hyperprolactinemia.
Med Hypotheses
; 2009 Mar;72(3):309-10
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Rethinking the stalk effect: a new hypothesis explaining suprasellar
tumor
-induced hyperprolactinemia.
The pars tuberalis is a distinct subdivision of the
pituitary
gland
but its function remains poorly understood.
Suprasellar
tumors
in this pars tuberalis region are frequently accompanied by hyperprolactinemia.
As these
tumors
do not immunoreact for any of the established
pituitary
hormones, they are classified as nonsecretory.
It has been postulated that these suprasellar
tumors
induce hyperprolactinemia by compressing the
pituitary
stalk, resulting in impaired dopamine delivery to the
pituitary
and, consequently, disinhibition of the lactotropes.
An alternative hypothesis proposed is that suprasellar
tumors
secrete a specific pars tuberalis factor that stimulates prolactin secretion.
Genetic Alliance.
consumer health - Galactorrhoea-Hyperprolactinaemia
.
MedlinePlus Health Information.
consumer health - Pituitary Tumors
.
COS Scholar Universe.
author profiles
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Fertil Steril. 1987 May;47(5):785-91
[
3569555.001
]
[Cites]
Br J Neurosurg. 1995;9(4):453-7
[
7576271.001
]
[Cites]
J Neuroendocrinol. 2000 Apr;12(4):287-95
[
10718925.001
]
[Cites]
Endocr Rev. 2001 Dec;22(6):724-63
[
11739329.001
]
[Cites]
Peptides. 2006 Nov;27(11):3007-19
[
16930771.001
]
[Cites]
J Clin Endocrinol Metab. 1992 Sep;75(3):692-7
[
1517356.001
]
[Cites]
J Reprod Fertil. 1995 Jul;104(2):243-50
[
7473415.001
]
[Cites]
Biol Mass Spectrom. 1993 Jan;22(1):89-97
[
8381675.001
]
(PMID = 19028420.001).
[ISSN]
0306-9877
[Journal-full-title]
Medical hypotheses
[ISO-abbreviation]
Med. Hypotheses
[Language]
ENG
[Grant]
United States / NCRR NIH HHS / RR / P20 RR015640; United States / NCRR NIH HHS / RR / RR015640-076871; United States / NCRR NIH HHS / RR / RR015640-086599; United States / NCRR NIH HHS / RR / P20 RR015640-086599; United States / NCRR NIH HHS / RR / P20 RR015640-076871; United States / NCRR NIH HHS / RR / P20RR015640
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
[Publication-country]
United States
[Other-IDs]
NLM/ NIHMS98103; NLM/ PMC2668659
44.
Ganesh HK, George J, Vimal MV, Bandgar T, Menon PS, Shah NS:
An unusual variant of Cushing syndrome.
Endocr Pract
; 2008 Sep;14(6):717-20
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Findings on magnetic resonance imaging of the
pituitary
gland
and abdomen were within normal limits.
[MeSH-major]
Adrenal Cortex Diseases /
diagnosis
. Cushing Syndrome / complications. Cushing Syndrome / pathology
[MeSH-minor]
Adrenocorticotropic Hormone / metabolism. Child, Preschool. Humans. Magnetic Resonance Imaging. Male.
Pituitary
Gland
/ pathology
Genetic Alliance.
consumer health - Cushing's Syndrome
.
MedlinePlus Health Information.
consumer health - Cushing's Syndrome
.
Hazardous Substances Data Bank.
Corticotropin
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18996791.001).
[ISSN]
1934-2403
[Journal-full-title]
Endocrine practice : official journal of the American College of Endocrinology and the American Association of Clinical Endocrinologists
[ISO-abbreviation]
Endocr Pract
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
United States
[Chemical-registry-number]
9002-60-2 / Adrenocorticotropic Hormone
45.
Zhu X, Wang J, Ju BG, Rosenfeld MG:
Signaling and epigenetic regulation of pituitary development.
Curr Opin Cell Biol
; 2007 Dec;19(6):605-11
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Signaling and epigenetic regulation of
pituitary
development.
The developing
pituitary
gland
provides an instructive model system for elucidating the molecular mechanisms by which distinct cell types arise from a common progenitor lineage accompanied by changes in the chromatin status in response to multiple extrinsic and intrinsic signals.
Investigation of the in vivo function of the histone modifying enzyme LSD1 has revealed a new layer of regulatory mechanism in
pituitary
organogenesis.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Science. 1999 Apr 30;284(5415):770-6
[
10221902.001
]
[Cites]
Mol Endocrinol. 1996 Dec;10(12):1570-81
[
8961267.001
]
[Cites]
Nature. 2005 Sep 15;437(7057):432-5
[
16079794.001
]
[Cites]
Nature. 2005 Sep 15;437(7057):436-9
[
16079795.001
]
[Cites]
Mol Cell. 2005 Sep 16;19(6):857-64
[
16140033.001
]
[Cites]
Mol Cell Biol. 2005 Dec;25(23):10379-90
[
16287852.001
]
[Cites]
Mol Cell Biol. 2005 Dec;25(23):10433-41
[
16287856.001
]
[Cites]
Development. 2006 Mar;133(5):913-23
[
16452096.001
]
[Cites]
Development. 2006 Apr;133(7):1367-78
[
16510501.001
]
[Cites]
Cell. 2006 Mar 10;124(5):973-83
[
16530044.001
]
[Cites]
Cell. 2006 Mar 10;124(5):985-96
[
16530045.001
]
[Cites]
Cell. 2006 May 5;125(3):593-605
[
16678101.001
]
[Cites]
Nature. 2006 May 18;441(7091):349-53
[
16625203.001
]
[Cites]
Mol Cell. 2006 Aug 4;23(3):365-75
[
16885026.001
]
[Cites]
Mol Cell. 2006 Aug 4;23(3):377-87
[
16885027.001
]
[Cites]
J Biol Chem. 2006 Aug 11;281(32):22429-33
[
16793760.001
]
[Cites]
Nat Rev Mol Cell Biol. 2006 Sep;7(9):678-89
[
16921404.001
]
[Cites]
Genes Dev. 2006 Oct 1;20(19):2739-53
[
17015435.001
]
[Cites]
Mol Cell. 2002 Feb;9(2):291-302
[
11864603.001
]
[Cites]
Science. 2002 Mar 22;295(5563):2231-5
[
11910101.001
]
[Cites]
EMBO J. 2002 Oct 15;21(20):5417-26
[
12374742.001
]
[Cites]
Mol Cell Biol. 2002 Nov;22(22):7812-9
[
12391150.001
]
[Cites]
J Cell Physiol. 2003 Mar;194(3):237-55
[
12548545.001
]
[Cites]
Genes Dev. 2003 Mar 15;17(6):711-6
[
12651888.001
]
[Cites]
Genes Dev. 2003 Mar 15;17(6):738-47
[
12651892.001
]
[Cites]
Mol Endocrinol. 2003 Nov;17(11):2152-61
[
12907761.001
]
[Cites]
Trends Endocrinol Metab. 2004 Jan-Feb;15(1):40-5
[
14693425.001
]
[Cites]
Development. 2004 Mar;131(5):965-73
[
14973298.001
]
[Cites]
Mech Dev. 2004 Feb;121(2):183-94
[
15037319.001
]
[Cites]
Nature. 1990 Oct 11;347(6293):528-33
[
1977085.001
]
[Cites]
Genomics. 1990 Nov;8(3):586-90
[
1981057.001
]
[Cites]
Mol Endocrinol. 2006 Nov;20(11):2898-908
[
16840533.001
]
[Cites]
Cell. 2006 Nov 3;127(3):469-80
[
17081971.001
]
[Cites]
Cell. 2007 Feb 9;128(3):505-18
[
17289570.001
]
[Cites]
Science. 2007 Feb 16;315(5814):988-92
[
17303754.001
]
[Cites]
Cell. 2007 Feb 23;128(4):635-8
[
17320500.001
]
[Cites]
Cell. 2007 Feb 23;128(4):693-705
[
17320507.001
]
[Cites]
Cell. 2007 Feb 23;128(4):747-62
[
17320511.001
]
[Cites]
Neuron. 2007 Mar 15;53(6):813-27
[
17359917.001
]
[Cites]
Proc Natl Acad Sci U S A. 2007 Mar 20;104(12):4852-7
[
17360330.001
]
[Cites]
Dev Biol. 2007 Apr 15;304(2):455-66
[
17367776.001
]
[Cites]
Mol Cell. 2007 Apr 13;26(1):89-101
[
17434129.001
]
[Cites]
Nature. 2007 Apr 19;446(7138):882-7
[
17392792.001
]
[Cites]
Endocrinology. 2007 May;148(5):1946-53
[
17289844.001
]
[Cites]
Cell. 2007 May 18;129(4):823-37
[
17512414.001
]
[Cites]
Mol Endocrinol. 2007 Jun;21(6):1458-66
[
17426285.001
]
[Cites]
Genes Dev. 2007 Jun 1;21(11):1322-7
[
17545467.001
]
[Cites]
Physiol Rev. 2007 Jul;87(3):933-63
[
17615393.001
]
[Cites]
Science. 2007 Jul 13;317(5835):248-51
[
17626886.001
]
[Cites]
Nature. 2007 Aug 2;448(7153):553-60
[
17603471.001
]
[Cites]
Mamm Genome. 2007 Jul;18(6-7):521-37
[
17557180.001
]
[Cites]
Curr Biol. 2006 Jan 24;16(2):119-29
[
16431364.001
]
[Cites]
Science. 2000 Nov 10;290(5494):1127-31
[
11073444.001
]
[Cites]
Development. 2001 Jan;128(2):147-54
[
11124111.001
]
[Cites]
Cell. 2001 Mar 23;104(6):849-59
[
11290323.001
]
[Cites]
Mamm Genome. 2001 Jul;12(7):485-94
[
11420609.001
]
[Cites]
Proc Natl Acad Sci U S A. 2001 Jul 17;98(15):8674-9
[
11447259.001
]
[Cites]
Genes Dev. 1998 Jun 1;12(11):1691-704
[
9620855.001
]
[Cites]
Genes Dev. 1998 Aug 1;12(15):2269-77
[
9694793.001
]
[Cites]
Nature. 1996 Nov 28;384(6607):327-33
[
8934515.001
]
[Cites]
Mol Cell. 2004 Nov 19;16(4):509-20
[
15546612.001
]
(PMID = 17988851.001).
[ISSN]
0955-0674
[Journal-full-title]
Current opinion in cell biology
[ISO-abbreviation]
Curr. Opin. Cell Biol.
[Language]
ENG
[Grant]
United States / NIDDK NIH HHS / DK / DK018477-34; United States / NIDDK NIH HHS / DK / R01 DK018477; United States / NIDDK NIH HHS / DK / R01 DK018477-34
[Publication-type]
Journal Article; Review
[Publication-country]
England
[Number-of-references]
61
[Other-IDs]
NLM/ NIHMS36359; NLM/ PMC2796608
46.
Fatović-Ferencić S, Gnjidić Z:
Centenary of the first trans-sphenoidal surgery of the hypophysis (Hermann Schloffer 1907) and its echoes within Croatian neurosurgical practice.
Wien Med Wochenschr
; 2007;157(23-24):618-24
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Centenary of the first trans-sphenoidal surgery of the
hypophysis
(Hermann Schloffer 1907) and its echoes within Croatian neurosurgical practice.
In this paper we aim to reminisce the role of the Austrian surgeon Hermann Schloffer (1868-1937) as the pioneer of a trans-sphenoidal approach to the
pituitary
gland
.
On the 16th of March 1907 he operated a patient with
pituitary
tumor
and published his report in the Wiener Klinische Wochenschrift on 23rd May 1907.
Schloffer's method was spread and modified worldwide, Croatia included, a country in which the interest in trans-sphenoidal approach to
pituitary
tumors
has not diminished or been lost, but slowly modified.
Today, almost a whole century after its introduction, it is still used to operate about 95% of sellar region
tumors
.
MedlinePlus Health Information.
consumer health - Endoscopy
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Neurosurgery. 2002 Mar;50(3):607-11; discussion 611-2
[
11841730.001
]
[Cites]
Laryngoscope. 1984 Aug;94(8):1066-74
[
6379349.001
]
[Cites]
Surgery. 1986 Dec;100(6):1185-90
[
3538463.001
]
[Cites]
Neurosurg Clin N Am. 2003 Jan;14(1):1-10
[
12690975.001
]
[Cites]
Croat Med J. 2006 Apr;47(2):310-7
[
16625698.001
]
[Cites]
J Neurosurg. 2001 Dec;95(6):1097-103
[
11765831.001
]
[Cites]
Neurosurgery. 1998 Apr;42(4):909-11; discussion 911-2
[
9574656.001
]
[Cites]
Lijec Vjesn. 2004 Jan-Feb;126(1-2):26-31
[
15526749.001
]
[Cites]
J Neurosurg. 2001 Dec;95(6):1083-96
[
11765830.001
]
(PMID = 18204963.001).
[ISSN]
0043-5341
[Journal-full-title]
Wiener medizinische Wochenschrift (1946)
[ISO-abbreviation]
Wien Med Wochenschr
[Language]
eng
[Publication-type]
Biography; Historical Article; Journal Article; Portraits
[Publication-country]
Austria
[Personal-name-as-subject]
Schloffer H
47.
Slavotinek A, Parisi M, Heike C, Hing A, Huang E:
Craniofacial defects of blastogenesis: duplication of pituitary with cleft palate and orophgaryngeal tumors.
Am J Med Genet A
; 2005 May 15;135(1):13-20
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Craniofacial defects of blastogenesis: duplication of
pituitary
with cleft palate and orophgaryngeal
tumors
.
To further elucidate the phenotypes associated with organ duplications, we present three infants with duplication of the
pituitary
gland
(DPG).
A review of previously reported cases with DPG showed that the commonest additional findings were hypothalamic enlargement, a broad or duplicated sella, cleft palate, hypertelorism, oropharyngeal
tumors
, agenesis or hypoplasia of the corpus callosum, and abnormalities of vertebrae.
[MeSH-major]
Abnormalities, Multiple / pathology. Cleft Palate / pathology. Craniofacial Abnormalities / pathology. Oropharyngeal
Neoplasms
/ pathology.
Pituitary
Gland
/ abnormalities
Genetic Alliance.
consumer health - Cleft Palate
.
MedlinePlus Health Information.
consumer health - Cleft Lip and Palate
.
MedlinePlus Health Information.
consumer health - Craniofacial Abnormalities
.
MedlinePlus Health Information.
consumer health - Facial Injuries and Disorders
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Copyright]
(c) 2005 Wiley-Liss, Inc.
(PMID = 15810008.001).
[ISSN]
1552-4825
[Journal-full-title]
American journal of medical genetics. Part A
[ISO-abbreviation]
Am. J. Med. Genet. A
[Language]
eng
[Publication-type]
Case Reports; Journal Article; Review
[Publication-country]
United States
[Number-of-references]
34
48.
Riemenschneider MJ, Beseoglu K, Hänggi D, Reifenberger G:
Prostate adenocarcinoma metastasis in the pituitary gland.
Arch Neurol
; 2009 Aug;66(8):1036-7
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Prostate adenocarcinoma metastasis in the
pituitary
gland
.
[MeSH-major]
Adenocarcinoma / secondary.
Pituitary
Neoplasms
/ secondary. Prostatic
Neoplasms
/
diagnosis
[MeSH-minor]
Abducens Nerve Diseases / etiology.
Diagnosis
, Differential. Diplopia / etiology. Endoscopy. Humans. Magnetic Resonance Imaging. Male. Middle Aged.
Pituitary
Gland
/ pathology
MedlinePlus Health Information.
consumer health - Pituitary Tumors
.
MedlinePlus Health Information.
consumer health - Prostate Cancer
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19667229.001).
[ISSN]
1538-3687
[Journal-full-title]
Archives of neurology
[ISO-abbreviation]
Arch. Neurol.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
United States
49.
Schmalisch K, Beschorner R, Psaras T, Honegger J:
Postoperative intracranial seeding of craniopharyngiomas--report of three cases and review of the literature.
Acta Neurochir (Wien)
; 2010 Feb;152(2):313-9; discussion 319
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Postoperative MRI showed no evidence of residual
tumor
.
Two years later, MRI revealed a local recurrence and in addition a separated cystic
tumor
on the right side adjacent to the middle cerebral artery consistent with seeding along the surgical route.
Both
tumors
were removed by re-operation.
On histopathological examination, both, the local recurrent
tumor
and the distant deposit turned out to be adamantinomatous craniopharyngiomas.
This deposit in the operative pathway was found to be an adamantinomatous craniopharyngioma, as was the initial
tumor
.
The recent control MRT revealed a right parietal intracranial
tumor
with peripheral contrast enhancement, which was located distant to the former craniotomy.
The tumor
was removed and histopathological examination revealed an adamantinomatous craniopharyngioma in accordance with the initial
tumor
.
CONCLUSION: Although craniopharyngiomas exhibit a
benign
histopathological pattern, cerebrospinal fluid seeding along the surgical route or along the CSF pathways has been observed.
Ectopic recurrence of craniopharyngioma suggests that meticulous protection of the whole surgical field and careful handling of the
tumor
during the operation are required.
[MeSH-major]
Brain
Neoplasms
/ secondary. Craniopharyngioma / secondary.
Neoplasm
Metastasis / pathology.
Neoplasm
Seeding.
Pituitary
Neoplasms
/ pathology
[MeSH-minor]
Adolescent. Adult. Craniotomy. Female. Frontal Bone / pathology. Frontal Bone / surgery. Humans. Magnetic Resonance Imaging. Male.
Neoplasm
Recurrence, Local / pathology.
Neoplasm
Recurrence, Local / surgery. Parietal Bone / pathology. Parietal Bone / surgery. Reoperation. Treatment Outcome
MedlinePlus Health Information.
consumer health - Brain Tumors
.
MedlinePlus Health Information.
consumer health - Pituitary Tumors
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19859655.001).
[ISSN]
0942-0940
[Journal-full-title]
Acta neurochirurgica
[ISO-abbreviation]
Acta Neurochir (Wien)
[Language]
eng
[Publication-type]
Case Reports; Journal Article; Review
[Publication-country]
Austria
[Number-of-references]
31
50.
Grebe SO, Borrmann M, Altenburg A, Wesselman U, Hein D, Haage P:
Chronic inflammation and accelerated atherosclerosis as important cofactors in nephrogenic systemic fibrosis following intravenous gadolinium exposure.
Clin Exp Nephrol
; 2008 Oct;12(5):403-6
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Nephrogenic systemic fibrosis (NSF) is a rare
disorder
in patients with chronic kidney disease characterized by an increased tissue deposition of collagen.
Her past medical history revealed a secondary HPT, multiple vascular complications, a seronegative rheumatoid arthritis, and a
pituitary
gland
adenoma.
Genetic Alliance.
consumer health - Nephrogenic Systemic Fibrosis
.
MedlinePlus Health Information.
consumer health - Atherosclerosis
.
MedlinePlus Health Information.
consumer health - Kidney Diseases
.
Hazardous Substances Data Bank.
GADOLINIUM, ELEMENTAL
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Curr Rheumatol Rep. 2006 Apr;8(2):151-7
[
16569375.001
]
[Cites]
Nephrol Dial Transplant. 2006 Apr;21(4):1104-8
[
16431890.001
]
[Cites]
J Am Soc Nephrol. 2006 Sep;17(9):2359-62
[
16885403.001
]
[Cites]
Blood Purif. 2004;22(1):28-37
[
14732809.001
]
[Cites]
J Am Soc Nephrol. 2007 Oct;18(10):2636-43
[
17855637.001
]
[Cites]
Eur Radiol. 2006 Dec;16(12):2619-21
[
17061066.001
]
[Cites]
Ann Intern Med. 2006 Aug 1;145(3):234-5
[
16880475.001
]
[Cites]
J Am Acad Dermatol. 2007 Jan;56(1):27-30
[
17109993.001
]
(PMID = 18551245.001).
[ISSN]
1342-1751
[Journal-full-title]
Clinical and experimental nephrology
[ISO-abbreviation]
Clin. Exp. Nephrol.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Japan
[Chemical-registry-number]
0 / Contrast Media; AU0V1LM3JT / Gadolinium
51.
Gustafsson L, Oreland S, Hoffmann P, Nylander I:
The impact of postnatal environment on opioid peptides in young and adult male Wistar rats.
Neuropeptides
; 2008 Apr;42(2):177-91
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Measurements of immunoreactive (ir) Met-enkephalin-Arg6Phe7 (MEAP) and dynorphin B (DYNB) peptide levels in the
pituitary
gland
and in a number of brain areas, were performed at three and 10 weeks of age, respectively.
[MeSH-major]
Environment. Maternal Deprivation. Opioid Peptides / metabolism.
Pituitary
Gland
/ metabolism. Stress, Psychological / metabolism
MedlinePlus Health Information.
consumer health - Stress
.
COS Scholar Universe.
author profiles
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18082882.001).
[ISSN]
0143-4179
[Journal-full-title]
Neuropeptides
[ISO-abbreviation]
Neuropeptides
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Scotland
[Chemical-registry-number]
0 / Endorphins; 0 / Opioid Peptides; 58569-55-4 / Enkephalin, Methionine; 73024-95-0 / enkephalin-Met, Arg(6)-Phe(7)-; 74913-18-1 / Dynorphins; 83335-41-5 / rimorphin
52.
Dorman DC, Struve MF, Marshall MW, Parkinson CU, James RA, Wong BA:
Tissue manganese concentrations in young male rhesus monkeys following subchronic manganese sulfate inhalation.
Toxicol Sci
; 2006 Jul;92(1):201-10
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
The olfactory epithelium, olfactory bulb, globus pallidus, caudate, putamen,
pituitary
gland
, and bile developed the greatest relative increase in manganese concentration following MnSO(4) exposure.
COS Scholar Universe.
author profiles
.
Hazardous Substances Data Bank.
MANGANESE, ELEMENTAL
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16624849.001).
[ISSN]
1096-6080
[Journal-full-title]
Toxicological sciences : an official journal of the Society of Toxicology
[ISO-abbreviation]
Toxicol. Sci.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
42Z2K6ZL8P / Manganese
53.
Takagi H, Nagashima K, Inoue M, Sakata I, Sakai T:
Detailed analysis of formation of chicken pituitary primordium in early embryonic development.
Cell Tissue Res
; 2008 Sep;333(3):417-26
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Detailed analysis of formation of chicken
pituitary
primordium in early embryonic development.
However, in the early phase of
pituitary
development, the detailed process by which the oral ectoderm develops into the adenohypophysis remains largely unknown.
Thus, the primordium of the
pituitary
gland
corresponding to the Lhx3-expressing region is surrounded by the Shh-expressing region, which appears in two steps, and the mass growth and invagination of RP of chicken result from the coordination of the dorsal extension of the anterior region and the ventral extension of the posterior region of RP.
[MeSH-major]
Ectoderm / embryology.
Pituitary
Gland
/ embryology
Hazardous Substances Data Bank.
BROMODEOXYURIDINE
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18584208.001).
[ISSN]
0302-766X
[Journal-full-title]
Cell and tissue research
[ISO-abbreviation]
Cell Tissue Res.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Germany
[Chemical-registry-number]
0 / Glycoprotein Hormones, alpha Subunit; 0 / Hedgehog Proteins; 0 / Homeodomain Proteins; 0 / LIM-Homeodomain Proteins; 0 / Lhx3 protein; 0 / RNA, Messenger; 0 / Transcription Factors; G34N38R2N1 / Bromodeoxyuridine
54.
Zoeller RT, Bansal R, Parris C:
Bisphenol-A, an environmental contaminant that acts as a thyroid hormone receptor antagonist in vitro, increases serum thyroxine, and alters RC3/neurogranin expression in the developing rat brain.
Endocrinology
; 2005 Feb;146(2):607-12
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
These findings suggest that BPA acts as a TH antagonist on the beta-TR, which mediates the negative feedback effect of TH on the
pituitary
gland
, but that BPA is less effective at antagonizing TH on the alpha-TR, leaving TRalpha-mediated events to respond to elevated T4.
COS Scholar Universe.
author profiles
.
Hazardous Substances Data Bank.
BISPHENOL A DISODIUM SALT
.
Hazardous Substances Data Bank.
LEVOTHYROXINE
.
Hazardous Substances Data Bank.
BISPHENOL A
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 15498886.001).
[ISSN]
0013-7227
[Journal-full-title]
Endocrinology
[ISO-abbreviation]
Endocrinology
[Language]
eng
[Grant]
United States / NIEHS NIH HHS / ES / ES10026
[Publication-type]
Journal Article; Research Support, U.S. Gov't, P.H.S.
[Publication-country]
United States
[Chemical-registry-number]
0 / Air Pollutants, Occupational; 0 / Benzhydryl Compounds; 0 / Calmodulin-Binding Proteins; 0 / Nerve Tissue Proteins; 0 / Nrgn protein, rat; 0 / Phenols; 0 / Receptors, Thyroid Hormone; 132654-77-4 / Neurogranin; MLT3645I99 / bisphenol A; Q51BO43MG4 / Thyroxine
55.
Bauer-Dantoin AC, Hanke CJ:
Using a classic paper by I. E. Lawton and N. B. Schwartz to consider the array of factors that control luteinizing hormone production.
Adv Physiol Educ
; 2007 Dec;31(4):318-22
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
The primary objective of the study is to determine whether tonic (pulsatile) secretion of luteinizing hormone (LH) from the
pituitary
gland
exhibits a circadian rhythm.
A review of the historical context in which the study was conducted, and a series of discovery learning questions are included to facilitate classroom discussions and to help deepen students' understanding of the complex nature of
pituitary
hormone regulation.
[MeSH-major]
Circadian Rhythm. Endocrinology / education. Luteinizing Hormone / secretion.
Pituitary
Gland
/ metabolism. Students
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18057402.001).
[ISSN]
1522-1229
[Journal-full-title]
Advances in physiology education
[ISO-abbreviation]
Adv Physiol Educ
[Language]
eng
[Publication-type]
Historical Article; Journal Article; Review
[Publication-country]
United States
[Chemical-registry-number]
9002-67-9 / Luteinizing Hormone
[Number-of-references]
26
56.
Correa-de-Santana E, Fröhlich B, Labeur M, Páez-Pereda M, Theodoropoulou M, Monteserin JL, Renner U, Stalla GK:
NOD2 receptors in adenopituitary folliculostellate cells: expression and function.
J Endocrinol
; 2009 Oct;203(1):111-22
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Folliculostellate cells (FS cells) are non-endocrine cells from the
pituitary
gland
that respond to bacterial endotoxins by producing cytokines.
Herein, we describe for the first time the expression and function of NOD receptors in human
pituitary
and FS TtT/GF cell line.
In conclusion, the present study demonstrates that NOD molecules play a modulatory role in the
pituitary
by regulating the function and activation of FS cells in response to bacterial components.
[MeSH-major]
Interleukin-6 / metabolism. NF-kappa B / metabolism. Nod2 Signaling Adaptor Protein / metabolism.
Pituitary
Gland
/ metabolism. Toll-Like Receptor 4 / metabolism
COS Scholar Universe.
author profiles
.
Gene Ontology.
gene/protein/disease-specific - Gene Ontology annotations from this paper
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19608614.001).
[ISSN]
1479-6805
[Journal-full-title]
The Journal of endocrinology
[ISO-abbreviation]
J. Endocrinol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / Interleukin-6; 0 / Lipopolysaccharides; 0 / NF-kappa B; 0 / NOD2 protein, human; 0 / Nod1 Signaling Adaptor Protein; 0 / Nod2 Signaling Adaptor Protein; 0 / STAT3 Transcription Factor; 0 / Toll-Like Receptor 4; 53678-77-6 / Acetylmuramyl-Alanyl-Isoglutamine
57.
Scheithauer BW, Silva AI, Parisi JE, Kovacs K, Horvath E:
Ganglioglioma of the neurohypophysis.
Endocr Pathol
; 2008;19(2):112-6
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
The finding of neurons within the neurohypophysis is exceedingly rare, as are ganglion cell
tumors
at this site.
In this paper, we report a ganglion cell
tumor of
the neurohypophysis found incidentally at autopsy.
The morphologic and immunohistochemical features of the
tumor
are presented, cytogenetic considerations are discussed, and literature regarding neuronal lesions of the
pituitary
gland
is reviewed.
[MeSH-major]
Ganglioglioma / pathology.
Pituitary
Gland
, Posterior / pathology.
Pituitary
Neoplasms
/ pathology
[MeSH-minor]
Aged, 80 and over. Alzheimer Disease / pathology. Brain / pathology. Female. Humans. Immunohistochemistry. Inappropriate ADH Syndrome /
diagnosis
. Inappropriate ADH Syndrome / pathology.
Pituitary
Gland
/ pathology. Sodium / blood
Genetic Alliance.
consumer health - Ganglioglioma
.
MedlinePlus Health Information.
consumer health - Pituitary Tumors
.
Hazardous Substances Data Bank.
SODIUM
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Microsc Res Tech. 1992 Jan 15;20(2):177-86
[
1547358.001
]
[Cites]
J Neurosurg. 1996 Nov;85(5):953-60
[
8893739.001
]
[Cites]
Am J Pathol. 1997 Sep;151(3):769-84
[
9284826.001
]
[Cites]
Pituitary. 2008;11(1):85-7
[
17440820.001
]
[Cites]
J Clin Endocrinol Metab. 1984 May;58(5):796-803
[
6423659.001
]
[Cites]
Clin Endocrinol (Oxf). 1989 Mar;30(3):213-24
[
2512034.001
]
[Cites]
J Neuropathol Exp Neurol. 1983 Nov;42(6):648-63
[
6631456.001
]
[Cites]
Ultrastruct Pathol. 1994 Nov-Dec;18(6):565-74
[
7855931.001
]
[Cites]
Ann Intern Med. 1984 Dec;101(6):789-93
[
6333843.001
]
[Cites]
Neuropathol Appl Neurobiol. 2002 Jun;28(3):252-5
[
12060349.001
]
[Cites]
Neuropathol Appl Neurobiol. 2001 Jun;27(3):197-205
[
11489139.001
]
[Cites]
Exp Clin Endocrinol Diabetes. 1998;106(5):425-30
[
9831310.001
]
[Cites]
J Neurosurg. 2001 Jul;95(1):167-8
[
11453393.001
]
[Cites]
Neuropathol Appl Neurobiol. 2008 Feb;34(1):118-23
[
17961139.001
]
[Cites]
Brain Tumor Pathol. 2002;19(2):63-7
[
12622135.001
]
[Cites]
Acta Neuropathol. 1989;77(3):320-8
[
2922994.001
]
[Cites]
Brain Pathol. 2002 Jan;12(1):137-9
[
11770898.001
]
[Cites]
Am J Surg Pathol. 2000 Apr;24(4):607-13
[
10757410.001
]
[Cites]
Exp Clin Endocrinol Diabetes. 1995;103(3):129-49
[
7584515.001
]
[Cites]
Am J Pathol. 1936 Mar;12(2):205-216.1
[
19970261.001
]
[Cites]
J Vet Med Sci. 1997 Sep;59(9):833-6
[
9342712.001
]
[Cites]
Virchows Arch. 1994;425(1):93-9
[
7921420.001
]
[Cites]
Acta Neuropathol. 2000 Jul;100(1):106-10
[
10912928.001
]
(PMID = 18496772.001).
[ISSN]
1046-3976
[Journal-full-title]
Endocrine pathology
[ISO-abbreviation]
Endocr. Pathol.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
United States
[Chemical-registry-number]
9NEZ333N27 / Sodium
58.
Torigian DA, Li G, Alavi A:
The Role of CT, MR Imaging, and Ultrasonography in Endocrinology.
PET Clin
; 2007 Jul;2(3):395-408
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
In particular, we focus our review on imaging evaluation of the
pituitary
gland
, the thyroid
gland
, the parathyroid glands, and the adrenal
gland
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Copyright]
Copyright © 2008 Elsevier Inc. All rights reserved.
(PMID = 27158019.001).
[ISSN]
1556-8598
[Journal-full-title]
PET clinics
[ISO-abbreviation]
PET Clin
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Keywords]
NOTNLM ; Adrenal / Computed tomography (CT) / Endocrine / Endocrinology / Magnetic resonance (MR) imaging / Parathyroid / Pituitary / Thyroid / Ultrasonography (US)
59.
Vankelecom H:
Non-hormonal cell types in the pituitary candidating for stem cell.
Semin Cell Dev Biol
; 2007 Aug;18(4):559-70
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Non-hormonal cell types in the
pituitary
candidating for stem cell.
Hormone balances in the body are primarily governed by the hypothalamus-
pituitary
system.
For its pivotal role, the
pituitary
gland
relies on an assortment of different hormone-producing cell types, the proportions of which dynamically change in response to fluctuating endocrine demands.
Mechanisms of
pituitary
cellular plasticity are at present far from understood, and may include proliferation and transdifferentiation of hormonal cells.
Here, I will review these data by focusing on the non-hormonal cell types that have been advanced as candidates for the
pituitary
stem cell position.
[MeSH-major]
Hypothalamus / embryology.
Pituitary
Gland
/ embryology. Stem Cells / physiology
MedlinePlus Health Information.
consumer health - Stem Cells
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17509912.001).
[ISSN]
1084-9521
[Journal-full-title]
Seminars in cell & developmental biology
[ISO-abbreviation]
Semin. Cell Dev. Biol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
England
[Number-of-references]
127
60.
Pepe GJ, Lynch TJ, Davies WA, Albrecht ED:
Regulation of baboon fetal pituitary prolactin expression by estrogen.
Biol Reprod
; 2009 Jun;80(6):1189-95
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Regulation of baboon fetal
pituitary
prolactin expression by estrogen.
However, because prolactin can modulate fetal adrenal and adult
pituitary
/ovarian function, the current study determined whether estrogen action involved estradiol-regulated changes in fetal prolactin/luteinizing hormone (LH) expression.
Fetal prolactin levels and the number of prolactin-positive fetal
pituitary
cells (per 0.37 mm(2)) were increased (P < 0.01) between mid (6 +/- 1 ng/ml; 15.8 +/- 2.4) and late (257 +/- 28 ng/ml; 57.3 +/- 5.1) gestation, reduced (P < 0.01) in late-gestation fetuses in which estradiol was suppressed (>95%) by letrozole (61 +/- 11 ng/ml; 19.3 +/- 2.0), and minimally but not significantly increased by letrozole and estradiol (99 +/- 11 ng/ml; 32.7 +/- 5.2).
In contrast, the number of LH-positive fetal
pituitary
cells decreased (P < 0.01) between mid (48.8 +/- 9.5) and late (17.4 +/- 3.2) gestation, remained elevated (P < 0.01) in estrogen-suppressed animals (56.6 +/- 5.1), and was partially but not significantly decreased by letrozole-estradiol (28.8 +/- 5.2).
We conclude that estrogen regulates fetal
pituitary
prolactin and LH expression and fetal prolactin levels.
However, because prolactin and LH expressions in estrogen-suppressed fetuses were inversely related to previously demonstrated changes in adrenal/ovarian development, we propose that estrogen regulates the fetal ovary and adrenal
gland
directly and not via action on the fetal
pituitary
gland
.
COS Scholar Universe.
author profiles
.
Hazardous Substances Data Bank.
ESTRADIOL
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Endocrinology. 1987 Dec;121(6):2011-7
[
2960516.001
]
[Cites]
Am J Obstet Gynecol. 1987 May;156(5):1275-8
[
3034061.001
]
[Cites]
Endocrinology. 1988 Feb;122(2):546-51
[
2828002.001
]
[Cites]
Endocrinology. 1988 Feb;122(2):646-50
[
2828008.001
]
[Cites]
Prog Clin Biol Res. 1990;322:159-69
[
2406729.001
]
[Cites]
Endocr Rev. 1990 Feb;11(1):124-50
[
2180685.001
]
[Cites]
Endocrinology. 1999 Dec;140(12):5953-61
[
10579363.001
]
[Cites]
J Clin Endocrinol Metab. 2000 Jan;85(1):270-4
[
10634398.001
]
[Cites]
Am J Obstet Gynecol. 2000 Feb;182(2):432-8
[
10694348.001
]
[Cites]
J Neuroendocrinol. 2001 Feb;13(2):175-81
[
11168843.001
]
[Cites]
Biochem Soc Trans. 2001 May;29(Pt 2):38-41
[
11356123.001
]
[Cites]
Exp Biol Med (Maywood). 2001 Feb;226(2):140-3
[
11446438.001
]
[Cites]
Endocrinology. 2001 Oct;142(10):4496-503
[
11564715.001
]
[Cites]
Biol Reprod. 2002 Apr;66(4):1054-60
[
11906925.001
]
[Cites]
Biol Reprod. 2002 Oct;67(4):1148-56
[
12297530.001
]
[Cites]
Biol Reprod. 2003 May;68(5):1911-7
[
12606356.001
]
[Cites]
Am J Obstet Gynecol. 1977 Jan 15;127(2):187-90
[
831500.001
]
[Cites]
Endocr Rev. 1990 Feb;11(1):151-76
[
2180686.001
]
[Cites]
Endocrinology. 1990 Jun;126(6):3083-8
[
2161746.001
]
[Cites]
J Steroid Biochem Mol Biol. 1992 Feb;41(2):171-8
[
1543685.001
]
[Cites]
Endocrinology. 1994 Dec;135(6):2581-7
[
7988446.001
]
[Cites]
Endocrinology. 1995 Sep;136(9):3892-900
[
7649097.001
]
[Cites]
J Clin Endocrinol Metab. 1995 Nov;80(11):3201-8
[
7593427.001
]
[Cites]
Endocr Rev. 1995 Oct;16(5):608-48
[
8529574.001
]
[Cites]
J Neuroendocrinol. 1996 Dec;8(12):929-33
[
8953471.001
]
[Cites]
Genes Dev. 1997 Jan 15;11(2):167-78
[
9009200.001
]
[Cites]
Biol Reprod. 1997 Mar;56(3):597-601
[
9047002.001
]
[Cites]
Eur J Endocrinol. 1998 Sep;139(3):337-42
[
9758446.001
]
[Cites]
Hum Reprod Update. 1998 Jul-Aug;4(4):406-19
[
9825855.001
]
[Cites]
Placenta. 1999 Mar-Apr;20(2-3):129-39
[
10195732.001
]
[Cites]
J Clin Endocrinol Metab. 1999 Sep;84(9):3344-50
[
10487709.001
]
[Cites]
Endocrinology. 2005 Apr;146(4):1737-44
[
15618362.001
]
[Cites]
Mol Cell Endocrinol. 2006 Mar 9;247(1-2):41-6
[
16420971.001
]
[Cites]
Reproduction. 2007 Feb;133(2):361-9
[
17307904.001
]
[Cites]
Endocrine. 2008 Jun;33(3):254-60
[
18484193.001
]
[Cites]
Endocrinology. 1969 Nov;85(5):916-23
[
5818077.001
]
[Cites]
J Clin Endocrinol Metab. 1975 Sep;41(3):626-9
[
1159067.001
]
[Cites]
J Pediatr. 1978 Dec;93(6):1011-4
[
152807.001
]
[Cites]
Endocrinology. 1979 May;104(5):1243-6
[
108091.001
]
[Cites]
Am J Obstet Gynecol. 1980 Mar 1;136(5):569-74
[
6766669.001
]
[Cites]
J Clin Endocrinol Metab. 1982 Jul;55(1):166-9
[
7076803.001
]
[Cites]
Endocrinology. 1988 Jan;122(1):78-83
[
3335215.001
]
(PMID = 19176882.001).
[ISSN]
0006-3363
[Journal-full-title]
Biology of reproduction
[ISO-abbreviation]
Biol. Reprod.
[Language]
ENG
[Grant]
United States / NICHD NIH HHS / HD / R01 HD013294; United States / NICHD NIH HHS / HD / U54 HD036207; United States / NICHD NIH HHS / HD / R01 HD-13294; United States / NICHD NIH HHS / HD / U54 HD 36207
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
United States
[Chemical-registry-number]
0 / Estrogens; 4TI98Z838E / Estradiol; 9002-62-4 / Prolactin; 9002-67-9 / Luteinizing Hormone
[Other-IDs]
NLM/ PMC2804803
61.
Esposito F, Kelly DF, Vinters HV, DeSalles AA, Sercarz J, Gorgulhos AA:
Primary sphenoid sinus neoplasms: a report of four cases with common clinical presentation treated with transsphenoidal surgery and adjuvant therapies.
J Neurooncol
; 2006 Feb;76(3):299-306
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Primary sphenoid sinus
neoplasms
: a report of four cases with common clinical presentation treated with transsphenoidal surgery and adjuvant therapies.
BACKGROUND: Primary
neoplasms of
the sphenoid sinus are a rare occurrence, accounting for approximately 1-2% of all paranasal sinus
tumors
.
METHODS: Four patients with sphenoid sinus
neoplasms
were identified (1%), all treated during the year 2003.
MRIs in all patients demonstrated large sphenoid sinus masses with partial clival and sellar bone erosion but with clear visualization of the
pituitary
gland
above the mass.
Cavernous sinus invasion was present in all four cases, including one patient with
tumor
in the ethmoid sinus and intra-tumoral hemorrhage.
All patients underwent subtotal
tumor
removal via an endonasal transsphenoidal route.
Tumor
histology included neuroendocrine carcinoma, sinonasal undifferentiated carcinoma, mucoepidermoid carcinoma, and giant cell
tumor
.
One patient required a second endonasal
tumor
debulking 15 months after the first for new visual loss that then resolved.
CONCLUSIONS: Intra-sphenoidal
tumors
are locally invasive
tumors
that include a wide pathological spectrum.
Recognizing their distinctive clinical presentation and MRI features is helpful in differentiating them from primary sellar
tumors
.
[MeSH-major]
Neurosurgical Procedures. Paranasal Sinus
Neoplasms
/ pathology. Paranasal Sinus
Neoplasms
/ therapy. Sphenoid Sinus / pathology
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Neurosurgery. 2002 Sep;51(3):699-705; discussion 705-7
[
12188948.001
]
[Cites]
Head Neck Surg. 1984 Jan-Feb;6(3):761-76
[
6319335.001
]
[Cites]
Am J Surg Pathol. 2002 Mar;26(3):371-6
[
11859210.001
]
[Cites]
Laryngoscope. 1989 Jul;99(7 Pt 1):716-20
[
2747395.001
]
[Cites]
Laryngoscope. 1963 May;73:537-46
[
14011951.001
]
[Cites]
Cephalalgia. 1988 Dec;8(4):229-36
[
3219724.001
]
[Cites]
Laryngoscope. 1997 Dec;107(12 Pt 1):1590-5
[
9396670.001
]
[Cites]
Int J Radiat Oncol Biol Phys. 1988 Jan;14 (1):11-22
[
3335447.001
]
[Cites]
Yonsei Med J. 1988;29(3):209-18
[
3057747.001
]
[Cites]
Head Neck. 1996 Mar-Apr;18(2):160-5; discussion 166
[
8647682.001
]
[Cites]
Neurosurgery. 1993 Oct;33(4):602-8; discussion 608-9
[
8232799.001
]
[Cites]
J Neurosurg. 1981 Aug;55(2):187-93
[
7252541.001
]
[Cites]
Cancer. 2003 Sep 15;98(6):1179-87
[
12973841.001
]
[Cites]
Laryngoscope. 1973 Aug;83(8):1252-65
[
4758128.001
]
[Cites]
Pituitary. 2002;5(4):261-5
[
14558675.001
]
[Cites]
World J Surg. 2003 Jul;27(7):849-55
[
14509518.001
]
[Cites]
Minim Invasive Neurosurg. 1998 Jun;41(2):66-73
[
9651913.001
]
[Cites]
J Neuropathol Exp Neurol. 2002 Aug;61(8):663-72
[
12152781.001
]
[Cites]
AJR Am J Roentgenol. 1992 Sep;159(3):581-9
[
1503031.001
]
[Cites]
J Otolaryngol. 1978 Oct;7(5):379-88
[
105151.001
]
[Cites]
Arch Otolaryngol. 1978 Oct;104(10):585-7
[
697636.001
]
[Cites]
Arch Otolaryngol Head Neck Surg. 1996 Jul;122(7):765-8
[
8663951.001
]
[Cites]
Neurosurgery. 2000 May;46(5):1084-91; discussion 1091-2
[
10807240.001
]
[Cites]
Otolaryngol Head Neck Surg. 1990 Jun;102(6):709-16
[
2115658.001
]
[Cites]
Eur J Pediatr. 1996 Aug;155(8):717-9
[
8839732.001
]
[Cites]
J Otolaryngol. 1990 Apr;19(2):122-9
[
2348505.001
]
[Cites]
Cancer. 2001 Dec 15;92(12):3012-29
[
11753979.001
]
[Cites]
Head Neck. 1991 May-Jun;13(3):208-12
[
2037472.001
]
[Cites]
Med J Aust. 2000 Nov 20;173(10):548-9
[
11194741.001
]
[Cites]
Neuroradiology. 1998 Oct;40(10 ):651-5
[
9833894.001
]
[Cites]
Brain. 1984 Sep;107 ( Pt 3):855-70
[
6478180.001
]
[Cites]
J Craniofac Surg. 1995 Jan;6(1):15-23
[
8601000.001
]
[Cites]
J Neurosurg Sci. 1999 Mar;43(1):25-36
[
10494663.001
]
[Cites]
Head Neck. 2002 Sep;24(9):821-9
[
12211046.001
]
[Cites]
Br J Neurosurg. 1994;8(1):51-5
[
8011194.001
]
[Cites]
Eur Arch Otorhinolaryngol. 2002 May;259(5):266-8
[
12107531.001
]
[Cites]
Am J Otolaryngol. 1995 Mar-Apr;16(2):109-14
[
7793504.001
]
[Cites]
J Neurosurg. 2003 Feb;98(2):350-8
[
12593622.001
]
[Cites]
Pathology (Phila). 1996;3(2):513-34
[
8795833.001
]
[Cites]
Arch Otolaryngol Head Neck Surg. 1994 Jan;120(1):19-25
[
8274251.001
]
[Cites]
Am J Emerg Med. 2001 Jan;19(1):88-90
[
11146033.001
]
[Cites]
Neurosurgery. 1998 Apr;42(4):913-5; discussion 915-6
[
9574657.001
]
[Cites]
Ann Oncol. 2003 Mar;14(3):367-72
[
12598339.001
]
[Cites]
Neurosurgery. 2003 Nov;53(5):1126-35; discussion 1135-7
[
14580279.001
]
[Cites]
Cancer. 1977 Dec;40(6):3038-41
[
412586.001
]
[Cites]
Laryngoscope. 2002 Nov;112(11):1964-9
[
12439163.001
]
[Cites]
J Laryngol Otol. 1995 Oct;109(10):951-5
[
7499947.001
]
(PMID = 16163447.001).
[ISSN]
0167-594X
[Journal-full-title]
Journal of neuro-oncology
[ISO-abbreviation]
J. Neurooncol.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
United States
62.
Yilmazlar S, Kocaeli H, Aydiner F, Korfali E:
Medial portion of the cavernous sinus: quantitative analysis of the medial wall.
Clin Anat
; 2005 Sep;18(6):416-22
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Pituitary
tumors
invade the cavernous sinus via the medial wall.
Researchers have speculated that this wall is composed of dura and that substances secreted by
tumors
might damage this barrier.
Each of these eight specimens was divided into three approximately equal-sized pieces, with cuts made in the coronal plane from posterior to anterior starting at the anterior level of the
pituitary
stalk.
The investigations showed that the MWCS is a distinct dural layer that forms a barrier between the medial venous space of the cavernous sinus and the
pituitary
gland
.
The thinness of the posterior MWCS suggests that this is the most likely path for extension of
pituitary
tumors
into the cavernous sinus.
[MeSH-minor]
Dura Mater / anatomy & histology. Humans.
Pituitary
Gland
/ anatomy & histology. Sella Turcica / anatomy & histology
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Copyright]
Copyright 2005 Wiley-Liss, Inc.
(PMID = 16015624.001).
[ISSN]
0897-3806
[Journal-full-title]
Clinical anatomy (New York, N.Y.)
[ISO-abbreviation]
Clin Anat
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
63.
Lee JH, Silva AC, Merkle H, Koretsky AP:
Manganese-enhanced magnetic resonance imaging of mouse brain after systemic administration of MnCl2: dose-dependent and temporal evolution of T1 contrast.
Magn Reson Med
; 2005 Mar;53(3):640-8
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
The largest decreases occurred in the
pituitary
gland
, where post-MnCl(2) T(1) ranged from 231 +/- 23 ms following the lowest dose to 143 +/- 43 ms after the highest dose, while the smallest decreases were observed in cortex (post-MnCl(2) T(1) = 1060 +/- 5 ms for low dose and 637 +/- 5 ms for high dose).
MedlinePlus Health Information.
consumer health - MRI Scans
.
COS Scholar Universe.
author profiles
.
Hazardous Substances Data Bank.
MANGANESE COMPOUNDS
.
Hazardous Substances Data Bank.
MANGANESE CHLORIDE
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Copyright]
(c) 2005 Wiley-Liss, Inc.
(PMID = 15723400.001).
[ISSN]
0740-3194
[Journal-full-title]
Magnetic resonance in medicine
[ISO-abbreviation]
Magn Reson Med
[Language]
eng
[Grant]
United States / Intramural NIH HHS / / Z01 NS002989-08
[Publication-type]
Journal Article; Research Support, U.S. Gov't, P.H.S.
[Publication-country]
United States
[Chemical-registry-number]
0 / Chlorides; 0 / Contrast Media; 0 / Manganese Compounds; QQE170PANO / manganese chloride
64.
MacMaster FP, El-Sheikh R, Upadhyaya AR, Nutche J, Rosenberg DR, Keshavan M:
Effect of antipsychotics on pituitary gland volume in treatment-naïve first-episode schizophrenia: a pilot study.
Schizophr Res
; 2007 May;92(1-3):207-10
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Effect of antipsychotics on
pituitary
gland
volume in treatment-naïve first-episode schizophrenia: a pilot study.
The objective of this study was to examine the effect of antipsychotics on
pituitary
volume in schizophrenic subjects.
Pituitary
volumes were measured in 16 patients with schizophrenia at baseline and 12 months after treatment with an antipsychotic medication using magnetic resonance imaging (MRI).
Pituitary
volume significantly increased in the schizophrenic subjects after treatment (12% increase).
In controls,
pituitary
volume did not change significantly (3% decrease).
Pituitary
volume may be a useful biomarker for treatments that affect neuroendocrine function.
Genetic Alliance.
consumer health - Schizophrenia
.
MedlinePlus Health Information.
consumer health - Schizophrenia
.
COS Scholar Universe.
author profiles
.
Hazardous Substances Data Bank.
HYDROCORTISONE
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17337162.001).
[ISSN]
0920-9964
[Journal-full-title]
Schizophrenia research
[ISO-abbreviation]
Schizophr. Res.
[Language]
ENG
[Grant]
United States / NCATS NIH HHS / TR / UL1 TR000005; United States / NIMH NIH HHS / MH / MH43156
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
Netherlands
[Chemical-registry-number]
0 / Antipsychotic Agents; 0 / Biomarkers; WI4X0X7BPJ / Hydrocortisone
65.
Hermle T, Goestemeyer AK, Sweny P, Burns A:
Successful therapeutic use of rituximab in refractory Wegener's granulomatosis after renal transplantation.
Clin Nephrol
; 2007 Nov;68(5):322-6
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Predominantly affected organs were kidneys and
pituitary
gland
.
Genetic Alliance.
consumer health - Transplantation
.
Genetic Alliance.
consumer health - Wegener's granulomatosis
.
MedlinePlus Health Information.
consumer health - Granulomatosis with Polyangiitis
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
Hazardous Substances Data Bank.
RITUXIMAB
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18044265.001).
[ISSN]
0301-0430
[Journal-full-title]
Clinical nephrology
[ISO-abbreviation]
Clin. Nephrol.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Germany
[Chemical-registry-number]
0 / Antibodies, Antineutrophil Cytoplasmic; 0 / Antibodies, Monoclonal; 0 / Antibodies, Monoclonal, Murine-Derived; 0 / Antigens, CD19; 4F4X42SYQ6 / Rituximab
66.
Adams TE:
Using gonadotropin-releasing hormone (GnRH) and GnRH analogs to modulate testis function and enhance the productivity of domestic animals.
Anim Reprod Sci
; 2005 Aug;88(1-2):127-39
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Gonadotropin releasing hormone (GnRH) controls the activity of the gonadotrope cells of the
pituitary
gland
and, as a consequence, is a critical component of the endocrine cascade that determines the growth, development, and functional activity of testicular tissue.
MedlinePlus Health Information.
consumer health - Pet Health
.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 15970407.001).
[ISSN]
0378-4320
[Journal-full-title]
Animal reproduction science
[ISO-abbreviation]
Anim. Reprod. Sci.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
Netherlands
[Chemical-registry-number]
33515-09-2 / Gonadotropin-Releasing Hormone
[Number-of-references]
82
67.
Bur IM, Zouaoui S, Fontanaud P, Coutry N, Molino F, Martin AO, Mollard P, Bonnefont X:
The comparison between circadian oscillators in mouse liver and pituitary gland reveals different integration of feeding and light schedules.
PLoS One
; 2010;5(12):e15316
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
The comparison between circadian oscillators in mouse liver and
pituitary
gland
reveals different integration of feeding and light schedules.
In this study, we used real-time quantitative PCR to assess circadian clock gene expression in the liver and
pituitary
gland
from mice raised under various photoperiods, or under a temporal restricted feeding protocol.
Whereas the liver oscillator always tracked meal time, the
pituitary
circadian clockwork showed an intermediate response, in between entrainment by the light regimen and the feeding-fasting rhythm.
The same composite response was also observed in the
pituitary
gland
from adrenalectomized mice under daytime restricted feeding, suggesting that circulating glucocorticoids do not inhibit full entrainment of the
pituitary
clockwork by meal time.
Altogether our results reveal further aspects in the complexity of phase entrainment in the circadian system, and suggest that the
pituitary
may host oscillators able to integrate multiple time cues.
[MeSH-major]
Liver / metabolism.
Pituitary
Gland
/ metabolism
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Proc Natl Acad Sci U S A. 2004 Aug 3;101(31):11227-32
[
15273285.001
]
[Cites]
Proc Natl Acad Sci U S A. 2004 Apr 13;101(15):5339-46
[
14963227.001
]
[Cites]
Nature. 2004 Oct 14;431(7010):869-73
[
15483616.001
]
[Cites]
J Theor Biol. 1978 Feb 6;70(3):297-313
[
633922.001
]
[Cites]
Proc Natl Acad Sci U S A. 1995 Aug 15;92(17):7754-8
[
7644490.001
]
[Cites]
Nat Neurosci. 1998 Dec;1(8):708-13
[
10196587.001
]
[Cites]
Proc Natl Acad Sci U S A. 2005 Feb 22;102(8):3111-6
[
15710878.001
]
[Cites]
Cell Metab. 2005 Nov;2(5):297-307
[
16271530.001
]
[Cites]
J Biol Rhythms. 2005 Dec;20(6):500-12
[
16275769.001
]
[Cites]
Eur J Neurosci. 2005 Dec;22(11):2845-54
[
16324119.001
]
[Cites]
J Neurosci. 2006 Jun 14;26(24):6406-12
[
16775127.001
]
[Cites]
Hum Mol Genet. 2006 Oct 15;15 Spec No 2:R271-7
[
16987893.001
]
[Cites]
PLoS Biol. 2007 Feb;5(2):e34
[
17298173.001
]
[Cites]
Proc Natl Acad Sci U S A. 2007 May 1;104(18):7664-9
[
17463091.001
]
[Cites]
J Neuroendocrinol. 2008 Mar;20(3):323-9
[
18208549.001
]
[Cites]
Cold Spring Harb Symp Quant Biol. 2007;72:381-6
[
18419295.001
]
[Cites]
Proc Natl Acad Sci U S A. 2008 Sep 30;105(39):15172-7
[
18779586.001
]
[Cites]
J Biol Chem. 2009 Apr 3;284(14):9066-73
[
19211562.001
]
[Cites]
Proc Natl Acad Sci U S A. 2009 Jun 16;106(24):9890-5
[
19487679.001
]
[Cites]
J Endocrinol. 2009 Aug;202(2):279-85
[
19474059.001
]
[Cites]
Mol Brain. 2009;2:28
[
19712475.001
]
[Cites]
Annu Rev Physiol. 2010;72:551-77
[
20148688.001
]
[Cites]
Neurobiol Aging. 2010 Apr;31(4):696-705
[
18614257.001
]
[Cites]
Proc Natl Acad Sci U S A. 2010 Apr 6;107(14):6412-7
[
20308563.001
]
[Cites]
J Neuroendocrinol. 2010 Mar;22(3):209-16
[
20070481.001
]
[Cites]
Genes Dev. 2010 Jun 15;24(12):1317-28
[
20551177.001
]
[Cites]
J Clin Invest. 2010 Jul;120(7):2600-9
[
20577050.001
]
[Cites]
Nature. 2010 Jul 29;466(7306):627-31
[
20562852.001
]
[Cites]
Cell. 2007 Aug 24;130(4):730-41
[
17719549.001
]
[Cites]
Endocrinology. 2007 Dec;148(12):5635-9
[
17901232.001
]
[Cites]
Am J Physiol Heart Circ Physiol. 2008 Feb;294(2):H1036-47
[
18156197.001
]
[Cites]
Am J Physiol. 1999 Nov;277(5 Pt 2):R1376-84
[
10564210.001
]
[Cites]
Nat Neurosci. 2000 Apr;3(4):372-6
[
10725927.001
]
[Cites]
J Neurosci. 1999 Jun 15;19(12):RC11
[
10366649.001
]
[Cites]
Science. 2000 Sep 29;289(5488):2344-7
[
11009419.001
]
[Cites]
Genes Dev. 2000 Dec 1;14(23):2950-61
[
11114885.001
]
[Cites]
Science. 2001 Jan 19;291(5503):490-3
[
11161204.001
]
[Cites]
Genes Cells. 2001 Mar;6(3):269-78
[
11260270.001
]
[Cites]
EMBO J. 2001 Dec 17;20(24):7128-36
[
11742989.001
]
[Cites]
J Neurosci. 2002 Jan 1;22(1):350-6
[
11756518.001
]
[Cites]
Nature. 2002 May 2;417(6884):78-83
[
11967526.001
]
[Cites]
Cell. 2002 May 3;109(3):307-20
[
12015981.001
]
[Cites]
Curr Biol. 2002 Jul 9;12(13):1130-3
[
12121621.001
]
[Cites]
Neuroscience. 2003;118(3):831-43
[
12710990.001
]
[Cites]
Biol Chem. 2003 May;384(5):711-9
[
12817467.001
]
[Cites]
FEBS Lett. 2003 Jul 31;548(1-3):49-52
[
12885406.001
]
[Cites]
Nat Rev Neurosci. 2003 Aug;4(8):649-61
[
12894240.001
]
[Cites]
Curr Biol. 2003 Sep 2;13(17):1543-8
[
12956958.001
]
[Cites]
Nature. 2004 Oct 14;431(7010):862-8
[
15483615.001
]
(PMID = 21179516.001).
[ISSN]
1932-6203
[Journal-full-title]
PloS one
[ISO-abbreviation]
PLoS ONE
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Other-IDs]
NLM/ PMC3002272
68.
Teshima T, Hara Y, Shigihara K, Takekoshi S, Nezu Y, Harada Y, Yogo T, Teramoto A, Osamura RY, Tagawa M:
Coexistence of corticotroph adenoma and thyrotroph hyperplasia in a dog.
J Vet Med Sci
; 2009 Jan;71(1):93-8
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Pituitary
thyrotroph hyperplasia results from prolonged primary hypothyroidism in humans, mice and rats.
On histological examination, the resected
pituitary
gland
contained both a corticotroph adenoma and thyrotroph hyperplasia.
In the present case, the
pituitary
thyrotroph hyperplasia was probably caused by primary hypothyroidism.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19194082.001).
[ISSN]
0916-7250
[Journal-full-title]
The Journal of veterinary medical science
[ISO-abbreviation]
J. Vet. Med. Sci.
[Language]
ENG
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Japan
69.
Kidambi S, Raff H, Findling JW:
Limitations of nocturnal salivary cortisol and urine free cortisol in the diagnosis of mild Cushing's syndrome.
Eur J Endocrinol
; 2007 Dec;157(6):725-31
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Limitations of nocturnal salivary cortisol and urine free cortisol in
the diagnosis
of mild Cushing's syndrome.
We present 11 cases of CS with normal or mildly elevated UFC in whom NSC was helpful in making a
diagnosis
.
Imaging studies included magnetic resonance imaging (MRI) of
pituitary
or computer tomography scan of abdomen.
Out of eleven patients, six had an abnormality in the
pituitary
gland
found by MRI and two out of eleven had adrenal masses.
The remaining three had normal
pituitary
MRI but had inferior petrosal sinus (IPS) sampling indicating Cushing's disease.
All patients had appropriate surgery, and histopathology of all except one was suggestive of either a cortisol-producing adrenal adenoma or an ACTH-secreting
pituitary
adenoma.
Multiple samples (urine/saliva) and DST are needed to make
the diagnosis
of mild CS.
Genetic Alliance.
consumer health - Cushing's Syndrome
.
MedlinePlus Health Information.
consumer health - Cushing's Syndrome
.
COS Scholar Universe.
author profiles
.
Hazardous Substances Data Bank.
DEXAMETHASONE
.
Hazardous Substances Data Bank.
HYDROCORTISONE
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18057379.001).
[ISSN]
1479-683X
[Journal-full-title]
European journal of endocrinology
[ISO-abbreviation]
Eur. J. Endocrinol.
[Language]
ENG
[Publication-type]
Journal Article
[Publication-country]
England
[Chemical-registry-number]
7S5I7G3JQL / Dexamethasone; WI4X0X7BPJ / Hydrocortisone
70.
Macgregor DJ, Lincoln GA:
A physiological model of a circannual oscillator.
J Biol Rhythms
; 2008 Jun;23(3):252-64
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Recent evidence based on studies in hypothalamo-
pituitary
disconnected Soay sheep suggests that the generation of circannual rhythms may be local to specific tissues or physiological systems.
Now, the authors present a physiological model of a circannual rhythm generator centered in the
pituitary
gland
based on the interaction between melatonin-responsive cells in the pars tuberalis that act to decode photoperiod, and lactotroph cells of the adjacent pars distalis that secrete prolactin.
The authors highlight specific features of the
pituitary
dynamics as a guide to future research on circannual rhythms.
[MeSH-minor]
Animals.
Pituitary
Gland
, Anterior / physiology.
Pituitary
Gland
, Anterior / secretion. Prolactin / secretion
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 18487417.001).
[ISSN]
0748-7304
[Journal-full-title]
Journal of biological rhythms
[ISO-abbreviation]
J. Biol. Rhythms
[Language]
eng
[Grant]
United Kingdom / Medical Research Council / / G0600678
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
9002-62-4 / Prolactin
71.
Harvey S:
Extrapituitary growth hormone.
Endocrine
; 2010 Dec;38(3):335-59
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Pituitary
somatotrophs secrete growth hormone (GH) into the bloodstream, to act as a hormone at receptor sites in most, if not all, tissues.
These endocrine actions of circulating GH are abolished after
pituitary
ablation or hypophysectomy, indicating its
pituitary
source.
GH gene expression is, however, not confined to the
pituitary
gland
, as it occurs in neural, immune, reproductive, alimentary, and respiratory tissues and in the integumentary, muscular, skeletal, and cardiovascular systems, in which GH may act locally rather than as an endocrine.
These actions are likely to be involved in the proliferation and differentiation of cells and tissues prior to the ontogeny of the
pituitary
gland
.
They are also likely to complement the endocrine actions of GH and are likely to maintain them after
pituitary
senescence and the somatopause.
[MeSH-minor]
Animals. Bone and Bones / metabolism. Cardiovascular System / metabolism. Gastrointestinal Tract / metabolism. Genitalia / metabolism. Humans. Immune System / metabolism. Muscles / metabolism.
Neoplasms
/ metabolism. Nervous System / metabolism.
Pituitary
Gland
/ metabolism. Respiratory System / metabolism. Tissue Distribution
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Gen Comp Endocrinol. 2008 May 1;156(3):544-51
[
18395204.001
]
[Cites]
J Mol Neurosci. 2003 Feb;20(1):1-14
[
12663929.001
]
[Cites]
Semin Arthritis Rheum. 2005 Aug;35(1):24-34
[
16084221.001
]
[Cites]
Endocrinology. 1997 Nov;138(11):4543-51
[
9348176.001
]
[Cites]
J Endocrinol. 1999 Feb;160(2):217-22
[
9924190.001
]
[Cites]
Trends Endocrinol Metab. 2008 Mar;19(2):74-81
[
18054496.001
]
[Cites]
Mol Reprod Dev. 1996 Nov;45(3):372-7
[
8916049.001
]
[Cites]
Nihon Sanka Fujinka Gakkai Zasshi. 1995 Aug;47(8):763-74
[
7594885.001
]
[Cites]
Zygote. 2003 Nov;11(4):293-7
[
15085728.001
]
[Cites]
Endocrinology. 2004 Sep;145(9):4162-75
[
15142985.001
]
[Cites]
Mol Cell Endocrinol. 1997 Jun 20;130(1-2):53-60
[
9220021.001
]
[Cites]
Growth Horm IGF Res. 2006 Oct-Dec;16(5-6):277-89
[
17101287.001
]
[Cites]
Anat Embryol (Berl). 2004 Nov;209(1):1-9
[
15480774.001
]
[Cites]
J Cutan Pathol. 2000 Jul;27(6):276-82
[
10885403.001
]
[Cites]
Adv Exp Med Biol. 2008;617:493-500
[
18497074.001
]
[Cites]
Trends Endocrinol Metab. 2002 Aug;13(6):245-50
[
12128285.001
]
[Cites]
Ann N Y Acad Sci. 2009 Apr;1163:414-6
[
19456374.001
]
[Cites]
Endocrinology. 2003 Mar;144(3):929-36
[
12586770.001
]
[Cites]
J Soc Gynecol Investig. 1994 Oct-Dec;1(4):285-9
[
9419785.001
]
[Cites]
Endocrine. 2002 Oct;19(1):73-9
[
12583604.001
]
[Cites]
Clin Endocrinol (Oxf). 2008 Jul;69(1):153-8
[
18034778.001
]
[Cites]
Endocr Rev. 2003 Dec;24(6):782-801
[
14671005.001
]
[Cites]
Biol Reprod. 2001 Jun;64(6):1826-34
[
11369615.001
]
[Cites]
J Steroid Biochem Mol Biol. 2000 Dec 31;75(4-5):219-28
[
11282275.001
]
[Cites]
Endocrinology. 1990 Apr;126(4):2214-21
[
2156686.001
]
[Cites]
Endocrinology. 1989 Sep;125(3):1556-64
[
2569392.001
]
[Cites]
Endocrinology. 1998 Sep;139(9):3837-42
[
9724037.001
]
[Cites]
J Mol Neurosci. 2002 Feb-Apr;18(1-2):89-95
[
11931354.001
]
[Cites]
J Mammary Gland Biol Neoplasia. 2008 Mar;13(1):131-45
[
18253708.001
]
[Cites]
Neuroimmunomodulation. 1995 Mar-Apr;2(2):108-14
[
8521139.001
]
[Cites]
Theriogenology. 2003 Mar;59(5-6):1393-402
[
12527085.001
]
[Cites]
Cell Tissue Res. 2003 Jul;313(1):81-91
[
12827495.001
]
[Cites]
Mol Cell Endocrinol. 1995 Sep 22;113(2):225-34
[
8674830.001
]
[Cites]
Mol Cell Endocrinol. 1995 Mar;109(1):47-61
[
7789615.001
]
[Cites]
Growth Horm IGF Res. 2002 Apr;12(2):126-36
[
12175650.001
]
[Cites]
Mol Cell Endocrinol. 2001 Dec 20;185(1-2):161-71
[
11738806.001
]
[Cites]
Horm Res. 2005;64 Suppl 3:66-72
[
16439847.001
]
[Cites]
Mol Cell Endocrinol. 1994 Dec;106(1-2):207-12
[
7895909.001
]
[Cites]
Endocrinol Metab Clin North Am. 1996 Sep;25(3):649-63
[
8879991.001
]
[Cites]
J Clin Endocrinol Metab. 1996 Jul;81(7):2663-9
[
8675594.001
]
[Cites]
Proc Natl Acad Sci U S A. 2004 Oct 19;101(42):15166-71
[
15353581.001
]
[Cites]
J Endocrinol. 2000 Sep;166(3):489-502
[
10974643.001
]
[Cites]
Endocrine. 2002 Dec;19(3):239-48
[
12624423.001
]
[Cites]
Gen Comp Endocrinol. 2007 Aug-Sep;153(1-3):124-31
[
17303134.001
]
[Cites]
Brain Res Mol Brain Res. 1999 May 7;68(1-2):88-100
[
10320786.001
]
[Cites]
J Bone Miner Res. 2001 Jun;16(6):1068-76
[
11393784.001
]
[Cites]
Mol Cell Endocrinol. 1997 Aug 8;131(2):127-36
[
9296371.001
]
[Cites]
J Clin Endocrinol Metab. 2005 Jun;90(6):3614-21
[
15784701.001
]
[Cites]
Pediatr Res. 2008 Jun;63(6):662-6
[
18520331.001
]
[Cites]
Endocrinology. 1991 Oct;129(4):1727-34
[
1717238.001
]
[Cites]
Endocrinology. 1988 Apr;122(4):1515-20
[
3345724.001
]
[Cites]
Cancer Res. 2005 Jul 1;65(13):5620-7
[
15994934.001
]
[Cites]
Exp Cell Res. 1999 Jul 10;250(1):35-50
[
10388519.001
]
[Cites]
Mol Cell Endocrinol. 2008 May 14;286(1-2):230-7
[
18359151.001
]
[Cites]
Neuroimmunomodulation. 2004;11(3):149-59
[
15067206.001
]
[Cites]
Biol Reprod. 2003 Sep;69(3):1013-22
[
12773434.001
]
[Cites]
Comp Biochem Physiol B Biochem Mol Biol. 2005 Apr;140(4):615-28
[
15763517.001
]
[Cites]
J Reprod Fertil Suppl. 1997;51:363-7
[
9404307.001
]
[Cites]
Endocrinology. 2008 May;149(5):2219-29
[
18276758.001
]
[Cites]
Mol Biol Cell. 1996 Jul;7(7):1003-14
[
8862516.001
]
[Cites]
Neurosci Lett. 2000 Mar 10;281(2-3):147-50
[
10704764.001
]
[Cites]
Endocrinology. 1993 Oct;133(4):1744-52
[
8404617.001
]
[Cites]
Endocrinology. 1997 Feb;138(2):580-7
[
9002989.001
]
[Cites]
Cell Mol Life Sci. 1998 Oct;54(10):1095-101
[
9817988.001
]
[Cites]
Endocrinology. 2001 Feb;142(2):767-77
[
11159849.001
]
[Cites]
Anim Reprod Sci. 2004 Jul;82-83:551-66
[
15271479.001
]
[Cites]
Arch Oral Biol. 1995 Sep;40(9):789-99
[
8651883.001
]
[Cites]
Endocrine. 1997 Dec;7(3):267-79
[
9657062.001
]
[Cites]
J Assist Reprod Genet. 2006 Jan;23(1):47-9
[
16447100.001
]
[Cites]
Proc Natl Acad Sci U S A. 2006 Apr 11;103(15):6031-6
[
16574776.001
]
[Cites]
Growth Horm IGF Res. 1998 Feb;8 Suppl A:47-54
[
10993591.001
]
[Cites]
J Clin Endocrinol Metab. 1998 Aug;83(8):2878-85
[
9709963.001
]
[Cites]
Gen Comp Endocrinol. 2008 May 1;156(3):613-21
[
18358475.001
]
[Cites]
Growth Horm IGF Res. 2004 Aug;14(4):309-18
[
15231300.001
]
[Cites]
Cell Tissue Res. 2005 Dec;322(3):379-92
[
16047159.001
]
[Cites]
J Mol Neurosci. 2006;28(3):257-64
[
16691013.001
]
[Cites]
J Endocrinol. 2002 Nov;175(2):307-18
[
12429029.001
]
[Cites]
Can J Physiol Pharmacol. 2003 Apr;81(4):371-84
[
12769229.001
]
[Cites]
Ann N Y Acad Sci. 2000;917:534-40
[
11268381.001
]
[Cites]
Prostate. 2001 Oct 1;49(2):116-21
[
11582590.001
]
[Cites]
Biol Reprod. 2002 Oct;67(4):1115-24
[
12297526.001
]
[Cites]
Mol Reprod Dev. 1997 Jun;47(2):175-80
[
9136119.001
]
[Cites]
Eur J Endocrinol. 2003 Nov;149(5):377-92
[
14585082.001
]
[Cites]
Immunopharmacol Immunotoxicol. 2003 May;25(2):159-77
[
12784910.001
]
[Cites]
J Endocrinol. 1999 Feb;160(2):205-16
[
9924189.001
]
[Cites]
Neurobiol Aging. 2005 Jun;26(6):929-37
[
15718052.001
]
[Cites]
Int J Biochem Cell Biol. 2008;40(10):1984-9
[
17888716.001
]
[Cites]
Comp Biochem Physiol C Pharmacol Toxicol Endocrinol. 1998 Jun;119(3):305-15
[
9827003.001
]
[Cites]
Cell Tissue Res. 2000 Mar;299(3):371-83
[
10772251.001
]
[Cites]
Pediatr Endocrinol Rev. 2007 Sep;5(1):510-5
[
17925792.001
]
[Cites]
J Mol Endocrinol. 1998 Aug;21(1):1-5
[
9723858.001
]
[Cites]
Mol Reprod Dev. 1998 Apr;49(4):444-53
[
9508096.001
]
[Cites]
World J Gastroenterol. 2000 Dec;6(6):842-847
[
11819706.001
]
[Cites]
Minerva Endocrinol. 2005 Mar;30(1):1-13
[
15877009.001
]
[Cites]
Endocrinology. 1999 Dec;140(12):5587-97
[
10579322.001
]
[Cites]
Science. 1982 Jun 11;216(4551):1237-9
[
7079756.001
]
[Cites]
Gen Comp Endocrinol. 2010 Jan 1;165(1):111-9
[
19539627.001
]
[Cites]
Vet Res Commun. 2008 Sep;32 Suppl 1:S131-4
[
18685999.001
]
[Cites]
Biol Reprod. 2003 May;68(5):1584-9
[
12606495.001
]
[Cites]
Endocrinology. 2001 Dec;142(12):5158-66
[
11713210.001
]
[Cites]
J Endocrinol. 2002 Jan;172(1):1-19
[
11786370.001
]
[Cites]
Endocr J. 1999 Mar;46 Suppl:S85-8
[
12054128.001
]
[Cites]
Adv Exp Med Biol. 2000;480:71-6
[
10959411.001
]
[Cites]
Int J Oncol. 2008 Mar;32(3):593-601
[
18292936.001
]
[Cites]
Acta Paediatr Suppl. 1997 Nov;423:5-11
[
9401531.001
]
[Cites]
Mol Reprod Dev. 2006 May;73(5):600-6
[
16489623.001
]
[Cites]
J Endocrinol. 2003 May;177(2):223-34
[
12740010.001
]
[Cites]
Cytokine Growth Factor Rev. 1999 Mar;10(1):5-14
[
10379908.001
]
[Cites]
Endocrinology. 2009 Mar;150(3):1341-52
[
18974274.001
]
[Cites]
Placenta. 2008 Mar;29 Suppl A:S36-41
[
17981323.001
]
[Cites]
Eur J Endocrinol. 2005 Aug;153(2):335-44
[
16061841.001
]
[Cites]
Mol Cell Endocrinol. 2002 Nov 29;197(1-2):173-8
[
12431810.001
]
[Cites]
Trends Mol Med. 2001 Oct;7(10):447-54
[
11597519.001
]
[Cites]
Mol Cell Endocrinol. 2002 Nov 29;197(1-2):179-85
[
12431811.001
]
[Cites]
Mol Endocrinol. 2004 Jul;18(7):1658-69
[
15056730.001
]
[Cites]
J Mol Neurosci. 2004;22(1-2):139-45
[
14742918.001
]
[Cites]
J Endocrinol. 2010 Jul;206(1):1-11
[
20371569.001
]
[Cites]
Mol Cell Endocrinol. 2002 Nov 29;197(1-2):187-95
[
12431812.001
]
[Cites]
Hum Reprod. 2002 Apr;17(4):1017-22
[
11925399.001
]
[Cites]
Domest Anim Endocrinol. 1997 Nov;14(6):399-407
[
9437576.001
]
[Cites]
Gen Comp Endocrinol. 2004 May 15;137(1):37-49
[
15094334.001
]
[Cites]
Eur J Endocrinol. 2005 Mar;152(3):327-32
[
15757846.001
]
[Cites]
Proteomics. 2008 Jan;8(2):389-401
[
18203262.001
]
[Cites]
Rheumatol Int. 2003 Jan;23(1):11-4
[
12548436.001
]
[Cites]
J Endocrinol. 2004 Apr;181(1):65-76
[
15072567.001
]
[Cites]
Endocrinology. 2003 Dec;144(12):5459-68
[
12960021.001
]
[Cites]
Oncogene. 2008 Apr 17;27(18):2602-12
[
17998942.001
]
[Cites]
Neuroscience. 2001;104(3):677-87
[
11440801.001
]
[Cites]
J Musculoskelet Neuronal Interact. 2001 Sep;2(1):49-58
[
15758476.001
]
[Cites]
Pediatr Nephrol. 1991 Jul;5(4):451-3
[
1911121.001
]
[Cites]
Minerva Endocrinol. 2007 Jun;32(2):103-13
[
17557036.001
]
[Cites]
Neuropeptides. 2000 Apr;34(2):98-107
[
10985926.001
]
[Cites]
Domest Anim Endocrinol. 1998 Mar;15(2):93-102
[
9532423.001
]
[Cites]
Endocrinology. 1996 Nov;137(11):4886-92
[
8895361.001
]
[Cites]
Mol Reprod Dev. 1999 Jun;53(2):127-34
[
10331450.001
]
[Cites]
Endocr Rev. 1998 Feb;19(1):55-79
[
9494780.001
]
[Cites]
Mol Reprod Dev. 1999 Aug;53(4):398-406
[
10398415.001
]
[Cites]
Mol Vis. 2009;15:920-6
[
19421410.001
]
[Cites]
Endocrinology. 2000 Apr;141(4):1571-84
[
10746665.001
]
[Cites]
Endocrinology. 2009 Jun;150(6):2758-66
[
19213842.001
]
[Cites]
Horm Res. 2005;64(4):157-65
[
16205094.001
]
[Cites]
Mol Cell Biochem. 2009 Jan;321(1-2):197-204
[
18985281.001
]
[Cites]
Proc Natl Acad Sci U S A. 2007 Aug 14;104(33):13331-6
[
17690250.001
]
[Cites]
Theriogenology. 2006 Jul 15;66(2):484-90
[
16442609.001
]
[Cites]
Mol Reprod Dev. 2003 Jan;64(1):32-40
[
12420297.001
]
[Cites]
J Clin Endocrinol Metab. 1992 Nov;75(5):1368-73
[
1430099.001
]
[Cites]
Neurosci Lett. 2009 Aug 21;460(1):87-91
[
19463895.001
]
[Cites]
Cell Signal. 2003 Jun;15(6):615-23
[
12681449.001
]
[Cites]
J Endocrinol. 2001 Jan;168(1):39-48
[
11139768.001
]
[Cites]
ScientificWorldJournal. 2006 Jan 18;6:53-80
[
16432628.001
]
[Cites]
Comp Biochem Physiol B Biochem Mol Biol. 2006 Sep;145(1):27-34
[
16828323.001
]
[Cites]
Gen Comp Endocrinol. 2007 Aug-Sep;153(1-3):302-10
[
17391672.001
]
[Cites]
J Clin Endocrinol Metab. 1997 Oct;82(10):3337-41
[
9329365.001
]
[Cites]
Mol Endocrinol. 2007 Nov;21(11):2832-46
[
17666586.001
]
[Cites]
J Biol Chem. 2005 Jun 24;280(25):23987-4003
[
15845533.001
]
[Cites]
J Mol Histol. 2006 Jan;37(1-2):37-41
[
16807770.001
]
[Cites]
Gen Comp Endocrinol. 2007 Jan 1;150(1):151-63
[
16970945.001
]
[Cites]
Endocrinology. 2007 Jan;148(1):103-15
[
17008400.001
]
[Cites]
Mol Reprod Dev. 2002 Jul;62(3):407-15
[
12112606.001
]
[Cites]
J Endocrinol. 1995 Dec;147(3):413-22
[
8543911.001
]
[Cites]
Int J Clin Pharmacol Res. 1994;14(2):79-85
[
7836029.001
]
[Cites]
Ann Oncol. 2001;12 Suppl 2:S83-7
[
11762358.001
]
[Cites]
J Histochem Cytochem. 2001 Mar;49(3):347-54
[
11181738.001
]
[Cites]
J Biol Chem. 2006 Mar 10;281(10):6471-81
[
16373333.001
]
[Cites]
J Endocrinol. 1991 Sep;130(3):425-33
[
1940716.001
]
[Cites]
J Gerontol A Biol Sci Med Sci. 1999 Apr;54(4):M212-5
[
10219013.001
]
[Cites]
J Biol Chem. 2002 Jul 19;277(29):26662-72
[
11994274.001
]
[Cites]
Nat Neurosci. 2002 Nov;5(11):1155-62
[
12368809.001
]
[Cites]
Transplant Proc. 2004 Jun;36(5):1613-4
[
15251397.001
]
[Cites]
J Dairy Sci. 2008 May;91(5):1802-13
[
18420611.001
]
[Cites]
Am J Pathol. 1997 Mar;150(3):1037-47
[
9060840.001
]
[Cites]
Cell Immunol. 2006 Mar;240(1):22-30
[
16839530.001
]
[Cites]
Endocrinology. 1991 Apr;128(4):2053-7
[
2004616.001
]
[Cites]
Endocrinol Metab Clin North Am. 2001 Sep;30(3):647-69
[
11571935.001
]
[Cites]
Neuroscience. 2007 Aug 10;148(1):151-63
[
17618059.001
]
[Cites]
J Pediatr Endocrinol Metab. 1999 Mar-Apr;12(2):113-24
[
10392357.001
]
[Cites]
Hippocampus. 2002;12(6):821-33
[
12542233.001
]
[Cites]
Gen Comp Endocrinol. 2005 Oct;144(1):28-37
[
15936023.001
]
[Cites]
J Mol Neurosci. 2007;31(3):261-71
[
17726230.001
]
[Cites]
J Pediatr Endocrinol Metab. 2004 Sep;17 Suppl 4:1321-6
[
15506078.001
]
[Cites]
Endocrinology. 1968 Oct;83(4):783-90
[
4879544.001
]
[Cites]
Growth Horm IGF Res. 2004 Dec;14(6):404-17
[
15519248.001
]
[Cites]
Oncogene. 2007 Jun 7;26(27):3998-4008
[
17213808.001
]
[Cites]
Endocrinology. 1996 Sep;137(9):3659-66
[
8756530.001
]
[Cites]
Cell Immunol. 2005 Mar;234(1):54-66
[
15964559.001
]
[Cites]
J Invest Dermatol. 2009 May;129(5):1071-87
[
19110541.001
]
[Cites]
Clin Neurol Neurosurg. 2006 Mar;108(3):255-8
[
16386830.001
]
[Cites]
Mol Cell Endocrinol. 1999 Mar 25;149(1-2):129-39
[
10375025.001
]
[Cites]
Endocrinology. 1994 Jan;134(1):287-92
[
7506206.001
]
[Cites]
Mol Endocrinol. 2000 May;14 (5):650-61
[
10809229.001
]
[Cites]
Trends Endocrinol Metab. 1999 Dec;10(10):383-391
[
10542394.001
]
[Cites]
Endocrinology. 2005 Mar;146(3):1138-44
[
15564324.001
]
[Cites]
Biochem Biophys Res Commun. 1999 Sep 7;262(3):575-8
[
10471365.001
]
[Cites]
Mol Cell Endocrinol. 2002 Nov 29;197(1-2):127-31
[
12431805.001
]
[Cites]
Cell Tissue Res. 2008 May;332(2):317-28
[
18335240.001
]
[Cites]
Invest Ophthalmol Vis Sci. 2006 Feb;47(2):738-44
[
16431975.001
]
[Cites]
J Dent Res. 1992 Nov;71(11):1807-11
[
1401442.001
]
[Cites]
J Lab Clin Med. 2000 Dec;136(6):476-81
[
11128749.001
]
[Cites]
J Histochem Cytochem. 2004 Sep;52(9):1191-7
[
15314086.001
]
[Cites]
J Dairy Sci. 2002 Dec;85(12 ):3260-7
[
12512599.001
]
[Cites]
Proc Natl Acad Sci U S A. 1997 May 13;94(10):5125-30
[
9144201.001
]
[Cites]
Int Rev Immunol. 1989 Jun;4(3):193-211
[
2488726.001
]
[Cites]
J Mammary Gland Biol Neoplasia. 2008 Mar;13(1):3-11
[
18204889.001
]
[Cites]
Cancer Res. 2005 Jan 1;65(1):317-24
[
15665309.001
]
[Cites]
Invest Ophthalmol Vis Sci. 2008 Jul;49(7):3080-9
[
18390640.001
]
[Cites]
J Mammary Gland Biol Neoplasia. 2008 Mar;13(1):105-17
[
18219565.001
]
[Cites]
J Endocrinol Invest. 2006 Feb;29(2):109-14
[
16610235.001
]
[Cites]
Gen Comp Endocrinol. 2007 Jun-Jul;152(2-3):295-303
[
17289045.001
]
[Cites]
Vet Q. 1999 Oct;21(4):111-5
[
10567999.001
]
[Cites]
Histochemistry. 1979 Feb 21;59(4):343-6
[
372158.001
]
[Cites]
J Clin Endocrinol Metab. 1981 Dec;53(6):1138-44
[
7028770.001
]
[Cites]
FASEB J. 1988 Sep;2(12):2812-8
[
3044906.001
]
[Cites]
Endocrinology. 1999 Dec;140(12):5907-14
[
10579357.001
]
[Cites]
Can J Physiol Pharmacol. 2000 Dec;78(12):1013-28
[
11149379.001
]
[Cites]
Acta Histochem. 2006;108(1):13-8
[
16564564.001
]
[Cites]
Endocrinology. 1992 May;130(5):2446-54
[
1572277.001
]
[Cites]
J Endocrinol. 2001 Jan;168(1):1-23
[
11139766.001
]
[Cites]
Gen Comp Endocrinol. 2009 Sep 1;163(1-2):63-9
[
19344664.001
]
[Cites]
Trends Endocrinol Metab. 2004 Apr;15(3):110-5
[
15046739.001
]
[Cites]
Endocrine. 2000 Dec;13(3):243-50
[
11216634.001
]
[Cites]
Theriogenology. 2006 Sep 1;66(4):797-803
[
16497368.001
]
[Cites]
Reprod Domest Anim. 2010 Jun;45(3):530-6
[
19032427.001
]
[Cites]
J Dent Res. 2004 Jan;83(1):35-9
[
14691110.001
]
[Cites]
J Physiol. 2003 Aug 15;551(Pt 1):323-36
[
12813157.001
]
[Cites]
J Clin Endocrinol Metab. 2000 Aug;85(8):2865-71
[
10946895.001
]
[Cites]
Int J Clin Pharmacol Res. 1995;15(1):27-32
[
7490172.001
]
[Cites]
Growth Horm IGF Res. 2006 Apr;16(2):67-85
[
16632396.001
]
[Cites]
Anim Reprod Sci. 2008 Sep;107(3-4):179-96
[
18571346.001
]
[Cites]
Dev Dyn. 2005 Oct;234(2):404-12
[
16127721.001
]
[Cites]
J Biol Chem. 2001 Jun 15;276(24):21464-75
[
11297545.001
]
[Cites]
Endocrinology. 2006 Apr;147(4):1819-29
[
16423870.001
]
[Cites]
Anticancer Res. 2000 Jul-Aug;20(4):2371-6
[
10953298.001
]
[Cites]
J Endocrinol Invest. 1993 Sep;16(8):625-33
[
8258651.001
]
[Cites]
Endocrinology. 1991 Sep;129(3):1628-34
[
1874192.001
]
[Cites]
Proc Natl Acad Sci U S A. 1986 Oct;83(20):7932-4
[
3464007.001
]
[Cites]
Clin Ter. 1997 Mar;148(3):75-81
[
9377843.001
]
[Cites]
J Endocrinol. 2004 Feb;180(2):247-55
[
14765976.001
]
[Cites]
J Dent Res. 2001 Aug;80(8):1742-7
[
11669486.001
]
[Cites]
Oncogene. 2005 May 26;24(23):3774-85
[
15782123.001
]
[Cites]
Exp Eye Res. 2006 Nov;83(5):1205-14
[
16893540.001
]
[Cites]
Horm Metab Res. 1999 Feb-Mar;31(2-3):50-4
[
10226781.001
]
[Cites]
Reprod Domest Anim. 2002 Oct;37(5):305-9
[
12354185.001
]
[Cites]
J Pediatr Endocrinol Metab. 2000;13 Suppl 6:1483-91
[
11202225.001
]
[Cites]
Gen Comp Endocrinol. 2005 Oct;144(1):78-89
[
16055124.001
]
[Cites]
Hormones (Athens). 2005 Jul-Sep;4(3):155-60
[
16613825.001
]
[Cites]
Endocrinology. 2001 Jul;142(7):2968-77
[
11416018.001
]
[Cites]
Neurosci Lett. 2009 May 22;455(3):199-202
[
19429121.001
]
[Cites]
Hum Pathol. 1999 Oct;30(10):1201-6
[
10534168.001
]
[Cites]
Invest Ophthalmol Vis Sci. 2003 Dec;44(12):5404-9
[
14638744.001
]
[Cites]
J Assist Reprod Genet. 2008 Apr;25(4):145-58
[
18278582.001
]
[Cites]
EMBO J. 1996 Aug 1;15(15):3871-9
[
8670892.001
]
[Cites]
J Dent Res. 2007 May;86(5):463-8
[
17452569.001
]
[Cites]
Theriogenology. 2002 Apr 15;57(7):1793-800
[
12041683.001
]
[Cites]
Endocrinology. 2008 Aug;149(8):3909-19
[
18450952.001
]
[Cites]
Comp Biochem Physiol C Toxicol Pharmacol. 2006 Mar-Apr;142(3-4):284-92
[
16326143.001
]
[Cites]
Comp Biochem Physiol A Mol Integr Physiol. 2006 Apr;143(4):514-22
[
16515871.001
]
[Cites]
Reprod Biol Endocrinol. 2003 May 07;1:40
[
12777179.001
]
[Cites]
Invest Ophthalmol Vis Sci. 2006 Jul;47(7):3036-43
[
16799050.001
]
[Cites]
Proteomics. 2006 Jan;6(1):341-8
[
16287172.001
]
[Cites]
Exp Cell Res. 1997 Nov 25;237(1):196-206
[
9417883.001
]
[Cites]
Reproduction. 2003 Mar;125(3):327-36
[
12611596.001
]
[Cites]
Neuroreport. 2006 Nov 6;17(16):1715-8
[
17047459.001
]
[Cites]
J Biol Chem. 2003 Feb 28;278(9):7580-90
[
12482855.001
]
[Cites]
Diabetes. 2005 Jan;54(1):51-62
[
15616010.001
]
[Cites]
Gene. 2005 May 9;350(2):183-92
[
15848116.001
]
[Cites]
Fish Physiol Biochem. 2009 Aug;35(3):501-9
[
19082753.001
]
[Cites]
Gen Comp Endocrinol. 2004 Nov;139(2):158-67
[
15504394.001
]
[Cites]
Rev Reprod. 2000 Sep;5(3):175-82
[
11006167.001
]
[Cites]
Comp Biochem Physiol B Biochem Mol Biol. 2007 Jun;147(2):325-39
[
17341448.001
]
[Cites]
J Endocrinol. 2000 Sep;166(3):503-10
[
10974644.001
]
[Cites]
Endocrine. 1995 Oct;3(10):729-35
[
21153162.001
]
[Cites]
Endocrinology. 2005 Nov;146(11):4898-904
[
16099858.001
]
[Cites]
Endocrinology. 1998 Sep;139(9):3855-62
[
9724040.001
]
[Cites]
Tohoku J Exp Med. 2008 Oct;216(2):165-72
[
18832799.001
]
[Cites]
Growth Factors. 2009 Dec;27(6):438-47
[
19824875.001
]
[Cites]
Growth Factors. 1997;14(2-3):131-43
[
9255605.001
]
[Cites]
Dev Comp Immunol. 2008;32(11):1313-25
[
18539326.001
]
[Cites]
Endocrine. 2000 Apr;12(2):197-201
[
10905380.001
]
[Cites]
Endocrinology. 2005 Feb;146(2):920-30
[
15539551.001
]
[Cites]
Growth Horm IGF Res. 2009 Jun;19(3):187-97
[
19144554.001
]
[Cites]
Anat Embryol (Berl). 2001 Dec;204(6):503-10
[
11876536.001
]
[Cites]
Eur J Neurol. 2006 Dec;13(12):1340-5
[
17116217.001
]
[Cites]
J Neuroimmunol. 2002 Feb;123(1-2):180-7
[
11880162.001
]
[Cites]
Eur J Histochem. 2007 Jul-Sep;51(3):173-80
[
17921112.001
]
[Cites]
Endocrinology. 2008 Mar;149(3):1366-76
[
18079205.001
]
[Cites]
J Clin Endocrinol Metab. 1996 Mar;81(3):1278-82
[
8772612.001
]
[Cites]
Mol Vis. 2007 Aug 29;13:1529-38
[
17893652.001
]
[Cites]
Am J Pathol. 1997 Jul;151(1):55-61
[
9212731.001
]
[Cites]
Gen Comp Endocrinol. 2009 Mar;161(1):123-32
[
19116154.001
]
[Cites]
J Endocrinol. 2001 May;169(2):389-96
[
11312155.001
]
[Cites]
Exp Eye Res. 2005 Nov;81(5):551-60
[
15913606.001
]
[Cites]
Endocrinology. 1998 May;139(5):2545-51
[
9564870.001
]
[Cites]
Cancer Invest. 1986;4(6):543-54
[
3030515.001
]
[Cites]
Transgenic Res. 2005 Feb;14(1):95-104
[
15865052.001
]
[Cites]
Endocrinology. 1997 Dec;138(12):5535-40
[
9389541.001
]
[Cites]
J Invest Dermatol. 1992 Sep;99(3):343-9
[
1324963.001
]
[Cites]
J Endocrinol. 2001 Jun;169(3):487-98
[
11375119.001
]
[Cites]
Comp Biochem Physiol A Mol Integr Physiol. 2005 Apr;140(4):423-9
[
15936701.001
]
[Cites]
Gen Comp Endocrinol. 2010 Jun 1;167(2):297-307
[
20347824.001
]
[Cites]
Mol Reprod Dev. 2000 Nov;57(3):247-55
[
11013432.001
]
[Cites]
Proc Natl Acad Sci U S A. 2008 Dec 23;105(51):20470-5
[
19075233.001
]
[Cites]
Gynecol Endocrinol. 2001 Aug;15(4):251-8
[
11560097.001
]
[Cites]
J Mol Evol. 2006 Nov;63(5):591-601
[
17009125.001
]
[Cites]
Mar Biotechnol (NY). 2003 Jan-Feb;5(1):79-91
[
12925922.001
]
[Cites]
Wien Klin Wochenschr. 1996;108(17):541-6
[
8992787.001
]
[Cites]
Am J Reprod Immunol. 2004 Feb;51(2):112-6
[
14748836.001
]
[Cites]
Osteoarthritis Cartilage. 2004 Jul;12 (7):543-51
[
15219569.001
]
[Cites]
Mol Reprod Dev. 2002 Feb;61(2):180-6
[
11803552.001
]
[Cites]
Mol Cell Endocrinol. 2002 Nov 29;197(1-2):153-65
[
12431808.001
]
[Cites]
Breast Cancer Res Treat. 2006 Aug;98 (3):315-27
[
16541323.001
]
[Cites]
Dev Dyn. 2008 Jun;237(6):1537-52
[
18498096.001
]
(PMID = 20972718.001).
[ISSN]
1559-0100
[Journal-full-title]
Endocrine
[ISO-abbreviation]
Endocrine
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
United States
[Chemical-registry-number]
12629-01-5 / Human Growth Hormone
72.
Azevedo MF, Xekouki P, Keil MF, Lange E, Patronas N, Stratakis CA:
An unusual presentation of pediatric Cushing disease: recurrent corticotropinoma of the posterior pituitary lobe.
J Pediatr Endocrinol Metab
; 2010 Jun;23(6):607-12
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
An unusual presentation of pediatric Cushing disease: recurrent corticotropinoma of the posterior
pituitary
lobe.
Cushing's syndrome (CS) is rare in childhood and adolescence and its
diagnosis
and work up are often challenging.
We report the case of a 15-year-old girl with a recurrent corticotrophin (ACTH)-secreting adenoma, located in the posterior lobe of the
pituitary
gland
.
At the age of 11, she presented with classic CS symptoms; biochemical investigation was compatible with ACTH-dependent Cushing disease, although
pituitary
gland
imaging did not show any
tumor
.
Following transsphenoidal surgery (TSS), histopathological analysis identified an ACTH-secreting
pituitary
microadenoma arising from the posterior
gland
.
The patient went into remission but 4 years later she presented with recurrent CS; this time,
pituitary
gland
imaging showed a microadenoma located in the posterior lobe, which was resected after TSS.
Posterior lobe
pituitary
adenomas are very rare and often hard to diagnose and treat; this is the first case of such a
tumor
causing recurrent Cushing's disease in a child.
[MeSH-major]
ACTH-Secreting
Pituitary
Adenoma / pathology. Adenoma / pathology.
Pituitary
ACTH Hypersecretion /
diagnosis
[MeSH-minor]
Child. Female. Humans. Magnetic Resonance Imaging.
Neoplasm
Recurrence, Local
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 20662335.001).
[ISSN]
0334-018X
[Journal-full-title]
Journal of pediatric endocrinology & metabolism : JPEM
[ISO-abbreviation]
J. Pediatr. Endocrinol. Metab.
[Language]
eng
[Grant]
United States / Intramural NIH HHS / / ZIA HD000642-12
[Publication-type]
Case Reports; Journal Article; Research Support, N.I.H., Intramural
[Publication-country]
England
[Other-IDs]
NLM/ NIHMS752367; NLM/ PMC4727444
73.
Ciric IS, Zhao J:
Transsphenoidal microsurgery: past, present and future.
Expert Rev Anticancer Ther
; 2006 Sep;6 Suppl 9:S75-8
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
We further elaborate on three fundamental surgical anatomy principles of transsphenoidal surgery for
pituitary
adenomas.
First, the
pituitary
gland
and, therefore,
pituitary
adenomas are extra-arachnoid structures, therefore the operation should be executed without penetration into the subarachnoid space.
Second, the
pituitary
gland
and, therefore,
pituitary
adenomas are midline structures, thus, veering off midline can result in potentially serious complications.
Third,
pituitary
adenomas commence inside the
pituitary
gland
, which distends around them as they grow.
Thus,
pituitary
macroadenomas are surrounded on their surface by a layer of attenuated residual normal anterior
pituitary
.
The operative technique for
pituitary
micro- and macroadenomas is described in detail.
Finally, we discuss the likely future treatment methods for
pituitary
adenomas.
[MeSH-minor]
Animals. Forecasting. Humans.
Pituitary
Neoplasms
/ pathology.
Pituitary
Neoplasms
/ surgery
COS Scholar Universe.
author profiles
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17004860.001).
[ISSN]
1744-8328
[Journal-full-title]
Expert review of anticancer therapy
[ISO-abbreviation]
Expert Rev Anticancer Ther
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
England
[Number-of-references]
5
74.
Grommen SV, Geysens S, Darras VM, De Groef B:
Chicken folliculo-stellate cells express thyrotropin receptor mRNA.
Domest Anim Endocrinol
; 2009 Nov;37(4):236-42
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
We investigated the presence of thyrotropin receptor (TSHR) mRNA in chicken
pituitary
and brain, and quantified the changes in its expression during the last week of embryonic development.
We found that in the
pituitary
gland
, TSHR mRNA co-localizes with folliculo-stellate cells but not with thyrotropic cells, suggesting the existence of a paracrine ultra-short thyrotropin feedback loop.
During late embryogenesis, when the activity of the hypothalamo-
pituitary
-thyroidal axis increases markedly, a significant rise in TSHR mRNA expression was observed in
pituitary
, which may signify an important change in
pituitary
ultra-short thyrotropin feedback regulation around the period of hatching.
[MeSH-major]
Brain / metabolism. Chickens / metabolism. Neuroendocrine Cells / metabolism.
Pituitary
Gland
, Anterior / metabolism. RNA, Messenger / metabolism. Receptors, Thyrotropin / metabolism
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19683409.001).
[ISSN]
1879-0054
[Journal-full-title]
Domestic animal endocrinology
[ISO-abbreviation]
Domest. Anim. Endocrinol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / RNA, Messenger; 0 / Receptors, Thyrotropin
75.
Guerrero CA, Krayenbühl N, Husain M, Krisht AF:
Ectopic suprasellar growth hormone-secreting pituitary adenoma: case report.
Neurosurgery
; 2007 Oct;61(4):E879; discussion E879
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Ectopic suprasellar growth hormone-secreting
pituitary
adenoma: case report.
OBJECTIVE: Ectopic
pituitary
adenomas are rare.
We present an unusual case of an ectopic growth hormone-secreting
pituitary
adenoma in the suprasellar space.
Magnetic resonance imaging scans revealed a suprasellar mass not arising from the normal looking
pituitary
gland
.
INTERVENTION: The patient underwent gross total removal of the
tumor
through a pterional approach.
Histological examination showed a growth hormone-secreting
pituitary
adenoma CONCLUSION: Although uncommon, growth hormone-secreting
pituitary
adenomas are encountered in the suprasellar region.
They should be added to the differential
diagnosis of tumors
in this location.
[MeSH-major]
Adenoma / radiography. Choristoma. Growth Hormone-Secreting
Pituitary
Adenoma / radiography
Genetic Alliance.
consumer health - Pituitary adenoma, growth hormone-secreting
.
The Weizmann Institute of Science GeneCards and MalaCards databases.
gene/protein/disease-specific - MalaCards for growth hormone secreting pituitary adenoma
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17986925.001).
[ISSN]
1524-4040
[Journal-full-title]
Neurosurgery
[ISO-abbreviation]
Neurosurgery
[Language]
eng
[Publication-type]
Case Reports; Journal Article; Review
[Publication-country]
United States
[Number-of-references]
23
76.
Galter D, Westerlund M, Belin AC, Olson L:
DJ-1 and UCH-L1 gene activity patterns in the brains of controls, Parkinson and schizophrenia patients and in rodents.
Physiol Behav
; 2007 Sep 10;92(1-2):46-53
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Of note, DJ-1 is expressed in several other tissues such as the liver, gastrointestinal tract, adrenal and
pituitary
gland
and during embryonic development, while UCH-L1 has a strictly neuronal expression also outside the CNS.
We conclude that DJ-1 and UCH-L1, like other genes linked to PD, are not expressed specifically in
DA
neurons, but instead generally in neurons.
Genetic Alliance.
consumer health - Schizophrenia
.
MedlinePlus Health Information.
consumer health - Parkinson's Disease
.
MedlinePlus Health Information.
consumer health - Schizophrenia
.
COS Scholar Universe.
author profiles
.
Hazardous Substances Data Bank.
DOPAMINE
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17599367.001).
[ISSN]
0031-9384
[Journal-full-title]
Physiology & behavior
[ISO-abbreviation]
Physiol. Behav.
[Language]
eng
[Grant]
United States / OMHHE CDC HHS / MN / R24-MN069955
[Publication-type]
Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Intracellular Signaling Peptides and Proteins; 0 / Oncogene Proteins; 0 / PARK7 protein, human; 0 / RNA, Messenger; EC 3.1.2.15 / Ubiquitin Thiolesterase; EC 3.1.2.15 / Ubiquitin carboxyl-Terminal Hydrolase L-1, human; VTD58H1Z2X / Dopamine
77.
Oliveira KJ, Paula GS, Costa-e-Sousa RH, Souza LL, Moraes DC, Curty FH, Pazos-Moura CC:
Peptide YY (PYY)3-36 modulates thyrotropin secretion in rats.
J Endocrinol
; 2006 Nov;191(2):459-63
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
In addition, recently, the ability of the hormone to regulate gonadotropin secretion, acting at
pituitary
and at hypothalamus has been reported.
PYY3-36-incubated rat
pituitary
glands showed a dose-dependent decrease in TSH release, with 44 and 62% reduction at 10(-8) and 10(-6) M (P < 0.05 and P < 0.001 respectively), and no alteration in TSH response to thyrotropin-releasing hormone.
Therefore, in the present paper, we have demonstrated that the gut hormone PYY3-36 acts directly on the
pituitary
gland
to inhibit TSH release, and in the fasting situation, in vivo, when serum PYY3-36 is reduced, the activity of thyroid axis is reduced as well.
In such a situation, systemically injected PYY3-36 was able to acutely activate the thyrotrope axis, suggesting a new role for PYY3-36 as a regulator of the hypothalamic-
pituitary
-thyroid axis.
[MeSH-major]
Fasting / metabolism. Peptide YY / pharmacology.
Pituitary
Gland
, Anterior / secretion. Thyrotropin / secretion
Hazardous Substances Data Bank.
LEVOTHYROXINE
.
Hazardous Substances Data Bank.
LIOTHYRONINE
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17088415.001).
[ISSN]
0022-0795
[Journal-full-title]
The Journal of endocrinology
[ISO-abbreviation]
J. Endocrinol.
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / Leptin; 06LU7C9H1V / Triiodothyronine; 106388-42-5 / Peptide YY; 5Y5F15120W / Thyrotropin-Releasing Hormone; 9002-71-5 / Thyrotropin; Q51BO43MG4 / Thyroxine
78.
Nasr C, Mason A, Mayberg M, Staugaitis SM, Asa SL:
Acromegaly and somatotroph hyperplasia with adenomatous transformation due to pituitary metastasis of a growth hormone-releasing hormone-secreting pulmonary endocrine carcinoma.
J Clin Endocrinol Metab
; 2006 Dec;91(12):4776-80
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Acromegaly and somatotroph hyperplasia with adenomatous transformation due to
pituitary
metastasis of a growth hormone-releasing hormone-secreting pulmonary endocrine carcinoma.
CONTEXT: GHRH excess from extracranial endocrine
tumors
is known to cause somatotroph hyperplasia and acromegaly.
Hypothalamic gangliocytomas producing GHRH are also known to be associated with
pituitary
adenomas causing acromegaly.
OBJECTIVES: The objective of this study was to describe a case of acromegaly due to a pulmonary GHRH-secreting endocrine carcinoma with metastasis to the
pituitary
gland
and to look at the peculiar histological features of this case.
SUBJECT: The patient was a 44-yr-old woman who was diagnosed with a biopsy-proven metastatic pulmonary endocrine
tumor
during pregnancy.
The patient underwent uneventful transsphenoidal resection of the sellar
tumor
.
Histological examination confirmed metastatic endocrine carcinoma to the
pituitary
, and immunohistochemistry localized GHRH to
the tumor
cells.
The adjacent
pituitary
exhibited somatotroph hyperplasia with abundant reactivity for GH and alpha-subunit.
CONCLUSION: This is the first report of a GHRH-producing endocrine
tumor
metastasizing to the
pituitary
and causing local hyperstimulation with somatotroph hyperplasia and adenomatous transformation.
[MeSH-major]
Acromegaly / complications. Acromegaly / etiology. Adenoma / etiology. Carcinoma / complications. Growth Hormone-Releasing Hormone / secretion. Lung
Neoplasms
/ complications. Paraneoplastic Endocrine Syndromes / complications.
Pituitary
Neoplasms
/ secondary. Somatotrophs / pathology
Genetic Alliance.
consumer health - Acromegaly
.
MedlinePlus Health Information.
consumer health - Lung Cancer
.
MedlinePlus Health Information.
consumer health - Pituitary Tumors
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16968791.001).
[ISSN]
0021-972X
[Journal-full-title]
The Journal of clinical endocrinology and metabolism
[ISO-abbreviation]
J. Clin. Endocrinol. Metab.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Hormones, Ectopic; 0 / Indium Radioisotopes; 9034-39-3 / Growth Hormone-Releasing Hormone
79.
Santos-Silva AP, Moura EG, Pinheiro CR, Rios AS, Abreu-Villaça Y, Passos MC, Oliveira E, Lisboa PC:
Neonatal nicotine exposure alters leptin signaling in the hypothalamus-pituitary-thyroid axis in the late postnatal period and adulthood in rats.
Life Sci
; 2010 Jul 31;87(5-6):187-95
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Neonatal nicotine exposure alters leptin signaling in the hypothalamus-
pituitary
-thyroid axis in the late postnatal period and adulthood in rats.
As leptin and thyroid hormones share the ability to increase energy expenditure, we studied the effects of maternal nicotine exposure during lactation on the leptin signaling in the hypothalamus-
pituitary
-thyroid axis of suckling and adult offspring.
In the
pituitary
gland
, NIC offspring showed lower JAK-2 content (-52%) at 15 days, but no differences in adulthood.
In the thyroid
gland
, the NIC group presented lower OB-R, JAK-2 and STAT-3 (-44%, -50%, -47%) and higher pSTAT-3 expression (+80%) at 15 days.
[MeSH-minor]
Animals. Animals, Newborn. Animals, Suckling. Blotting, Western. Female. Hypothalamus / drug effects. Hypothalamus / metabolism. Hypothyroidism / chemically induced. Lactation. Male.
Pituitary
Gland
/ drug effects.
Pituitary
Gland
/ metabolism. Rats. Rats, Wistar. Thyroid
Gland
/ drug effects. Thyroid
Gland
/ metabolism. Time Factors
Hazardous Substances Data Bank.
NICOTINE
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Copyright]
Copyright 2010 Elsevier Inc. All rights reserved.
(PMID = 20600149.001).
[ISSN]
1879-0631
[Journal-full-title]
Life sciences
[ISO-abbreviation]
Life Sci.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Netherlands
[Chemical-registry-number]
0 / Leptin; 0 / Nicotinic Agonists; 6M3C89ZY6R / Nicotine
80.
Menetti F, Bartolomei I, Ambrosini-Spaltro A, Salvi F, Agati R, Leonardi M:
Amyloidoma Involving the Orbit, Meckel's Cave and Infratemporal Fossa: 3T MRI Findings.
Neuroradiol J
; 2009 Mar 23;22(1):41-7
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
The few literature reports of intracranial amyloidomas include lesions involving the
pituitary
gland
, orbit, cerebral hemispheres, temporal bone, cerebellopontine angle and jugular foramen.
The lesion closely mimicked a malignant
tumor
with perineural
tumor
infiltration, so we performed fine needle biopsy of the portion of the lesion near the right foramen ovale under fluoroscopic guidance.
Further clinical and blood examinations, serum chemistry, followed by biopsy of the periumbilical fat showed no signs of systemic amyloidosis or an underlying inflammatory or neoplastic
disorder
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 24206952.001).
[ISSN]
1971-4009
[Journal-full-title]
The neuroradiology journal
[ISO-abbreviation]
Neuroradiol J
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
81.
Nonogaki K:
Ghrelin and feedback systems.
Vitam Horm
; 2008;77:149-70
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Ghrelin also stimulates
pituitary
gland
secretion of growth hormone (GH) via the afferent vagus nerve.
The Lens.
Cited by Patents in
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17983856.001).
[ISSN]
0083-6729
[Journal-full-title]
Vitamins and hormones
[ISO-abbreviation]
Vitam. Horm.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
United States
[Chemical-registry-number]
0 / Ghrelin; 9002-72-6 / Growth Hormone
[Number-of-references]
123
82.
Rudnik A, Zawadzki T, Gałuszka-Ignasiak B, Larysz D, Bazowski P, Zdeb M:
Endoscopic transsphenoidal treatment of empty sella turcica syndrome using a silastic coil.
Minim Invasive Neurosurg
; 2006 Dec;49(6):376-9
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
An empty sella turcica is due to the presence of an arachnoid diverticulum with its fluid content in the sella turcica, exerting pressure on the
pituitary
gland
.
Genetic Alliance.
consumer health - Empty Sella Syndrome
.
Hazardous Substances Data Bank.
POLYDIMETHYLSILOXANES
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17323268.001).
[ISSN]
0946-7211
[Journal-full-title]
Minimally invasive neurosurgery : MIN
[ISO-abbreviation]
Minim Invasive Neurosurg
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Germany
[Chemical-registry-number]
0 / Dimethylpolysiloxanes; 0 / Silicones; 0 / Tissue Adhesives; 63148-62-9 / baysilon
83.
Andersson AC, Yun Z, Sperber GO, Larsson E, Blomberg J:
ERV3 and related sequences in humans: structure and RNA expression.
J Virol
; 2005 Jul;79(14):9270-84
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
A search in dbEST revealed ERV3 RNA expression in placenta, skin, carcinoid
tumor
, and adrenal glands.
QPCR results for ERV3 were compatible with previously published results, with a stronger expression in adrenal
gland
and placenta than in 15 other human tissues.
Expression was found in corpus luteum, testis, adrenal
gland
, Hassal's bodies in thymus, brown fat,
pituitary
gland
, and epithelium of the lung.
The Lens.
Cited by Patents in
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
Nucleic Acids Res. 2004 Jan 1;32(Database issue):D50
[
14681356.001
]
[Cites]
Mol Biol Evol. 2004 May;21(5):781-98
[
14739248.001
]
[Cites]
Proc Natl Acad Sci U S A. 1998 Dec 22;95(26):15345-50
[
9860971.001
]
[Cites]
J Gen Virol. 1999 Jan;80 ( Pt 1):255-60
[
9934709.001
]
[Cites]
Placenta. 1999 Jan;20(1):109-18
[
9950152.001
]
[Cites]
J Biol Chem. 1999 Apr 2;274(14):9327-34
[
10092610.001
]
[Cites]
J Virol. 1999 Jun;73(6):5186-90
[
10233986.001
]
[Cites]
Genomics. 1999 May 1;57(3):371-9
[
10329003.001
]
[Cites]
Dis Markers. 1998 Nov;14(3):127-33
[
10427470.001
]
[Cites]
Autoimmunity. 1999;30(2):81-3
[
10435720.001
]
[Cites]
J Virol. 1999 Nov;73(11):9496-507
[
10516058.001
]
[Cites]
Nucleic Acids Res. 1993 Jan 11;21(1):135-43
[
8382789.001
]
[Cites]
Nucleic Acids Res. 1993 May 25;21(10):2493-501
[
8506143.001
]
[Cites]
Virology. 1993 Oct;196(2):905-9
[
8372456.001
]
[Cites]
J Virol. 1993 Nov;67(11):6778-87
[
7692084.001
]
[Cites]
J Virol. 1994 Oct;68(10):6605-18
[
8083996.001
]
[Cites]
Int J Cancer. 1994 Sep 15;58(6):836-40
[
7927876.001
]
[Cites]
Nucleic Acids Res. 1994 Nov 11;22(22):4673-80
[
7984417.001
]
[Cites]
Mol Cell Biol. 1995 Jul;15(7):3759-66
[
7791783.001
]
[Cites]
Genomics. 2004 May;83(5):940-3
[
15081124.001
]
[Cites]
J Virol Methods. 2004 Jun 15;118(2):83-94
[
15081603.001
]
[Cites]
J Virol. 2004 May;78(10):5139-46
[
15113896.001
]
[Cites]
Radiat Res. 2004 May;161(5):597-602
[
15161363.001
]
[Cites]
Nucleic Acids Res. 1981 Dec 11;9(23):6615-26
[
6172779.001
]
[Cites]
Cell. 1981 Dec;27(2 Pt 1):321-30
[
7199388.001
]
[Cites]
Virology. 1984 Oct 30;138(2):225-35
[
6495650.001
]
[Cites]
Proc Natl Acad Sci U S A. 1985 May;82(9):2834-8
[
2859593.001
]
[Cites]
Virology. 1985 Dec;147(2):449-58
[
3840930.001
]
[Cites]
Mol Cell Biol. 1985 Nov;5(11):2959-66
[
3837839.001
]
[Cites]
J Virol. 1987 Jul;61(7):2182-91
[
2884330.001
]
[Cites]
Arch Dermatol. 1987 Nov;123(11):1538a-1541
[
2960273.001
]
[Cites]
Int J Cancer. 1988 Mar 15;41(3):380-5
[
3346101.001
]
[Cites]
J Mol Biol. 1989 Feb 20;205(4):765-9
[
2467007.001
]
[Cites]
Proc Natl Acad Sci U S A. 1989 Jul;86(13):5109-13
[
2740346.001
]
[Cites]
Curr Top Microbiol Immunol. 1989;148:115-32
[
2684548.001
]
[Cites]
Nature. 1990 Jul 19;346(6281):240-4
[
1695712.001
]
[Cites]
J Mol Biol. 1990 Oct 5;215(3):403-10
[
2231712.001
]
[Cites]
J Virol. 1991 Nov;65(11):6343-8
[
1920638.001
]
[Cites]
J Gen Virol. 1992 Sep;73 ( Pt 9):2463-6
[
1402820.001
]
[Cites]
J Virol. 1993 Feb;67(2):1122-6
[
8419641.001
]
[Cites]
Proc Natl Acad Sci U S A. 1997 Jul 8;94(14):7583-8
[
9207135.001
]
[Cites]
Leukemia. 1997 Apr;11 Suppl 3:145-6
[
9209324.001
]
[Cites]
J Gen Virol. 1997 Sep;78 ( Pt 9):2379-88
[
9292028.001
]
[Cites]
J Gen Virol. 1997 Oct;78 ( Pt 10):2575-85
[
9349478.001
]
[Cites]
Int J Oncol. 1998 Feb;12(2):309-13
[
9458354.001
]
[Cites]
J Virol. 1998 Apr;72(4):3442-5
[
9525678.001
]
[Cites]
Bioessays. 1998 Apr;20(4):307-16
[
9619102.001
]
[Cites]
Pathobiology. 1998;66(5):209-15
[
9732235.001
]
[Cites]
J Virol. 1995 Sep;69(9):5455-60
[
7636991.001
]
[Cites]
Virology. 1995 Aug 20;211(2):589-92
[
7645262.001
]
[Cites]
Dev Biol. 1995 Aug;170(2):664-78
[
7649392.001
]
[Cites]
Gene. 1995 Aug 19;161(2):163-70
[
7665072.001
]
[Cites]
J Invest Dermatol. 1996 Jan;106(1):125-8
[
8592062.001
]
[Cites]
Proc Natl Acad Sci U S A. 1996 May 28;93(11):5177-84
[
8643549.001
]
[Cites]
Mol Cell Biol. 1996 Aug;16(8):4495-503
[
8754850.001
]
[Cites]
AIDS Res Hum Retroviruses. 1996 Jun 10;12(9):833-40
[
8738436.001
]
[Cites]
Virology. 1996 Aug 15;222(2):451-6
[
8806530.001
]
[Cites]
Genomics. 1996 Sep 1;36(2):354-8
[
8812465.001
]
[Cites]
Autoimmunity. 1996;23(2):111-7
[
8871766.001
]
[Cites]
Am J Pathol. 1996 Nov;149(5):1727-35
[
8909261.001
]
[Cites]
Proc Natl Acad Sci U S A. 1996 Dec 10;93(25):14759-64
[
8962128.001
]
[Cites]
AIDS Res Hum Retroviruses. 1997 Apr 10;13(6):507-16
[
9100993.001
]
[Cites]
Histochem J. 1997 Feb;29(2):127-33
[
9147069.001
]
[Cites]
Genomics. 1999 Oct 15;61(2):133-44
[
10534399.001
]
[Cites]
Curr Opin Genet Dev. 1999 Dec;9(6):657-63
[
10607616.001
]
[Cites]
Nature. 2000 Feb 17;403(6771):785-9
[
10693809.001
]
[Cites]
Acta Neurol Scand. 2000 Apr;101(4):229-38
[
10770518.001
]
[Cites]
Trends Genet. 2000 Sep;16(9):418-20
[
10973072.001
]
[Cites]
Virology. 2001 Jan 5;279(1):280-91
[
11145909.001
]
[Cites]
Autoimmunity. 2000;33(1):15-21
[
11204249.001
]
[Cites]
J Virol Methods. 2001 Feb;91(2):109-17
[
11164492.001
]
[Cites]
Nat Rev Genet. 2000 Nov;1(2):134-44
[
11253653.001
]
[Cites]
J Virol. 2001 Dec;75(23):11709-19
[
11689652.001
]
[Cites]
Curr Biol. 2001 Nov 13;11(22):R914-6
[
11719237.001
]
[Cites]
J Hum Genet. 2001;46(11):619-25
[
11721880.001
]
[Cites]
J Virol. 2002 Mar;76(6):2789-95
[
11861846.001
]
[Cites]
Virology. 2002 Jun 5;297(2):220-5
[
12083821.001
]
[Cites]
Genome Biol. 2002 Jun 18;3(7):RESEARCH0034
[
12184808.001
]
[Cites]
Eukaryot Cell. 2002 Feb;1(1):44-55
[
12455970.001
]
[Cites]
Mol Biol Cell. 2002 Dec;13(12):4179-94
[
12475944.001
]
[Cites]
J Biol Chem. 2003 May 30;278(22):19723-31
[
12644456.001
]
(PMID = 15994821.001).
[ISSN]
0022-538X
[Journal-full-title]
Journal of virology
[ISO-abbreviation]
J. Virol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / RNA, Viral; 0 / Viral Envelope Proteins
[Other-IDs]
NLM/ PMC1168766
84.
Amiya N, Amano M, Takahashi A, Yamanome T, Yamamori K:
Profiles of alpha-melanocyte-stimulating hormone in the Japanese flounder as revealed by a newly developed time-resolved fluoroimmunoassay and immunohistochemistry.
Gen Comp Endocrinol
; 2007 Mar;151(1):135-41
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
A TR-FIA for alpha-MSH was newly developed, and its levels in the
pituitary
gland
and plasma of Japanese flounder reared in a white or black tank for 5 months were compared.
Displacement curves of serially twofold-diluted hypothalamus extract,
pituitary
gland
extract, and plasma extract of Japanese flounder with the assay buffer were parallel to the alpha-MSH standard curve.
Moreover, displacement curves of serially twofold-diluted hypothalamus and/or
pituitary
gland
extract of masu salmon, goldfish, red seabream, Japanese eel, tiger puffer, and barfin flounder with the assay buffer were also parallel to the alpha-MSH standard.
In Japanese flounder, total immunoreactive (ir)-alpha-MSH levels in the
pituitary
gland
were lower in the black tank, whereas those in the plasma tended to be higher in the black tank, suggesting that the synthesis and release of alpha-MSH are higher in the black tank. alpha-MSH-ir cells were detected in the pars intermedia and a small part of the pars distalis of the
pituitary
gland
. alpha-MSH-ir cell bodies were located in the basal hypothalamus and alpha-MSH-ir fibers were distributed not only in the hypothalamus but also in the telencephalon, midbrain, cerebellum, and medulla oblongata, suggesting that alpha-MSH functions as a neuromodulator in the brain.
[MeSH-minor]
Animals. Brain / metabolism. Hypothalamus / metabolism.
Pituitary
Gland
/ metabolism. Reference Standards. Reproducibility of Results
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17286977.001).
[ISSN]
0016-6480
[Journal-full-title]
General and comparative endocrinology
[ISO-abbreviation]
Gen. Comp. Endocrinol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
581-05-5 / alpha-MSH
85.
Chiu EK, Nichols JW:
Sellar lesions and visual loss: key concepts in neuro-ophthalmology.
Expert Rev Anticancer Ther
; 2006 Sep;6 Suppl 9:S23-8
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
The
pituitary
gland
serves an essential role in the activity and regulation of the endocrine system.
Mass lesions within the
pituitary
gland
account for approximately 10-15% of intracranial
neoplasms
.
Patients with
pituitary
adenomas may present with endocrine dysfunction or neuro-ophthalmic pathology, resulting from compression of surrounding structures, most notably the optic chiasm.
Visual deficits from chiasmal
tumors
may manifest as visual field defects, visual loss, diplopia, nystagmus and visual hallucinations.
The specific visual field defect usually results from the anatomic compression of the
tumor
upon the optic chiasm.
It is important to recognize characteristic visual deficits in
the diagnosis
and treatment of chiasmal
tumors
.
[MeSH-minor]
Animals. Humans.
Pituitary
Neoplasms
/ complications.
Pituitary
Neoplasms
/ pathology. Skull
Neoplasms
/
diagnosis
. Skull
Neoplasms
/ etiology. Skull
Neoplasms
/ pathology
MedlinePlus Health Information.
consumer health - Vision Impairment and Blindness
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 17004853.001).
[ISSN]
1744-8328
[Journal-full-title]
Expert review of anticancer therapy
[ISO-abbreviation]
Expert Rev Anticancer Ther
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
England
[Number-of-references]
9
86.
Elston MS, McDonald KL, Clifton-Bligh RJ, Robinson BG:
Familial pituitary tumor syndromes.
Nat Rev Endocrinol
; 2009 Aug;5(8):453-61
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Familial
pituitary
tumor
syndromes.
The vast majority of
pituitary
tumors
are
benign
and occur sporadically; however, they can still result in significant morbidity and even premature mortality through mass effects and hormone dysfunction.
The etiology of sporadic
tumors
is still poorly understood; by contrast, advances have been made in our understanding of familial
pituitary
adenoma syndromes in the past decade.
Currently, four genes are known to be associated with familial
pituitary
tumor
syndromes: MEN1, CDKN1B, PRKAR1A and AIP.
The first three genes are associated with a variety of extrapituitary pathologies, for example, primary hyperparathyroidism with multiple endocrine
neoplasia
type 1, which might aid identification of these syndromes.
By contrast, AIP mutations seem to occur in the setting of isolated familial
pituitary
adenomas, particularly of the growth-hormone-secreting subtype.
Awareness and identification of familial
pituitary
tumor
syndromes is important because of potential associated pathologies and important implications for family members.
Here, we review the current knowledge of familial
pituitary
tumor
syndromes.
[MeSH-major]
Genetic Predisposition to Disease.
Pituitary
Neoplasms
/ genetics.
Pituitary
Neoplasms
/ pathology
[MeSH-minor]
Cyclic AMP-Dependent Protein Kinase RIalpha Subunit / genetics. Cyclin-Dependent Kinase Inhibitor p27. Humans. Intracellular Signaling Peptides and Proteins / genetics. Multiple Endocrine
Neoplasia
/ genetics. Multiple Endocrine
Neoplasia
/ pathology. Proto-Oncogene Proteins / genetics. Syndrome
MedlinePlus Health Information.
consumer health - Pituitary Tumors
.
COS Scholar Universe.
author profiles
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19564887.001).
[ISSN]
1759-5037
[Journal-full-title]
Nature reviews. Endocrinology
[ISO-abbreviation]
Nat Rev Endocrinol
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
England
[Chemical-registry-number]
0 / CDKN1B protein, human; 0 / Cyclic AMP-Dependent Protein Kinase RIalpha Subunit; 0 / Intracellular Signaling Peptides and Proteins; 0 / MEN1 protein, human; 0 / PRKAR1A protein, human; 0 / Proto-Oncogene Proteins; 0 / aryl hydrocarbon receptor-interacting protein; 147604-94-2 / Cyclin-Dependent Kinase Inhibitor p27
[Number-of-references]
106
87.
Doraiswamy PM, Schott G, Star K, Edwards R, Mueller-Oerlinghausen B:
Atypical antipsychotics and pituitary neoplasms in the WHO database.
Psychopharmacol Bull
; 2007;40(1):74-6
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Atypical antipsychotics and
pituitary
neoplasms
in the WHO database.
Preclinical evidence shows that chronic administration of antipsychotics can cause
pituitary
adenomas in female mice.
To investigate the clinical relevance in this finding, reports of
pituitary
neoplasms
in the WHO adverse drug reaction (ADR) database were reviewed.
Amisulpride and risperidone [corrected] had among the highest Information Component (IC) scores for
benign pituitary neoplasm
; amisulpride (IC = 3.31, IC025 = 1.83, 5 reports) and risperidone (IC = 4.03, IC025 = 3.33, 19 reports), and not otherwise specified (
NOS
)
pituitary neoplasm
: amisulpride (IC = 2.69, IC025 = 0.70, 3 reports) and risperidone (IC = 4.49, IC025 = 3.86, 23 reports).
We conclude that there is a need for prospective studies to confirm causality and suggest that clinicians, until then, would consider a
pituitary
adenoma in patients experiencing severe hyperprolactinemia or associated symptoms when receiving potent D2 antagonists [corrected]
[MeSH-major]
Adenoma / chemically induced. Antipsychotic Agents / adverse effects.
Pituitary
Neoplasms
/ chemically induced. Risperidone / adverse effects. Schizophrenia / drug therapy. Sulpiride / analogs & derivatives
MedlinePlus Health Information.
consumer health - Pituitary Tumors
.
MedlinePlus Health Information.
consumer health - Schizophrenia
.
COS Scholar Universe.
author profiles
.
Hazardous Substances Data Bank.
RISPERIDONE
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[ErratumIn]
Psychopharmacol Bull. 2007;40(2):5
(PMID = 17285098.001).
[ISSN]
0048-5764
[Journal-full-title]
Psychopharmacology bulletin
[ISO-abbreviation]
Psychopharmacol Bull
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Antipsychotic Agents; 7MNE9M8287 / Sulpiride; AA0G3TW31W / sultopride; L6UH7ZF8HC / Risperidone
88.
Ozdemir D, Dagdelen S, Erbas T:
Endocrine involvement in systemic amyloidosis.
Endocr Pract
; 2010 Nov-Dec;16(6):1056-63
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
METHODS: We conducted a review of the medical literature using MEDLINE data sources, including clinical trials, in vitro studies, and case reports on
pituitary
, thyroid, parathyroid, pancreatic, adrenal, and gonadal involvement in systemic amyloidosis.
Amyloid deposition commonly seen in the
pituitary
gland
and the pancreas of patients with Alzheimer disease and type 2 diabetes mellitus, respectively, is generally classified as local amyloidosis and should not be confused with systemic involvement.
Involvement of
pituitary
, parathyroid, and pancreatic sites in systemic amyloidosis still remains to be clarified.
Genetic Alliance.
consumer health - Amyloidosis
.
MedlinePlus Health Information.
consumer health - Amyloidosis
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 20570812.001).
[ISSN]
1934-2403
[Journal-full-title]
Endocrine practice : official journal of the American College of Endocrinology and the American Association of Clinical Endocrinologists
[ISO-abbreviation]
Endocr Pract
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
89.
Boelen A, Kwakkel J, Chassande O, Fliers E:
Thyroid hormone receptor β mediates acute illness-induced alterations in central thyroid hormone metabolism.
J Neuroendocrinol
; 2009 May;21(5):465-72
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
Acute illness in mice profoundly affects thyroid hormone metabolism in the hypothalamus and
pituitary
gland
.
LPS decreased
pituitary
thyroid-stimulating hormone β mRNA expression in WT at 24 h but not in TRβ(-/-) mice.
The peak in
pituitary
D2 expression at t = 4 h in WT was absent in TRβ(-/-) mice.
Our results suggest that TRβ is involved in suppression of the central component of the hypothalamic-
pituitary
-thyroid axis in acute illness.
[MeSH-major]
Acute Disease. Thyroid
Gland
/ metabolism. Thyroid Hormone Receptors beta / metabolism. Thyroid Hormones / metabolism
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19302190.001).
[ISSN]
1365-2826
[Journal-full-title]
Journal of neuroendocrinology
[ISO-abbreviation]
J. Neuroendocrinol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / Lipopolysaccharides; 0 / Thyroid Hormone Receptors beta; 0 / Thyroid Hormones; EC 1.11.1.8 / Iodide Peroxidase
90.
Unlu A, Meco C, Ugur HC, Comert A, Ozdemir M, Elhan A:
Endoscopic anatomy of sphenoid sinus for pituitary surgery.
Clin Anat
; 2008 Oct;21(7):627-32
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Endoscopic anatomy of sphenoid sinus for
pituitary
surgery.
This study aimed to highlight surgical landmarks and their anatomical relationships for
pituitary
surgery through endoscopic perspective.
The width of the
pituitary
, which is the distance between the medial margins of the carotid prominences, was measured as 21 +/- 2.5 mm and the distance between the medial margin of the carotid prominences at the lower margin of the
pituitary
was 18 +/- 3.1 mm.
The width of the
pituitary
, which is the distance between the medial margins of the anterior curvature of the ICA, was measured as 23.2 +/- 3 mm, while the distance between the posterior curvature of the ICA was 19.7 +/- 4.9 mm.
Endoscopic view provided superior detailed visualization of the close relationships between
pituitary
gland
, internal carotid arteries, and optic nerves.
[MeSH-major]
Endoscopy / methods. Neurosurgical Procedures / methods.
Pituitary
Gland
/ surgery. Sphenoid Sinus / anatomy & histology
[MeSH-minor]
Adenoma / surgery. Adult. Aged. Carotid Arteries / anatomy & histology. Humans. Male. Middle Aged. Optic Nerve / anatomy & histology.
Pituitary
Neoplasms
/ surgery
MedlinePlus Health Information.
consumer health - Endoscopy
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Copyright]
(c) 2008 Wiley-Liss, Inc.
(PMID = 18816443.001).
[ISSN]
1098-2353
[Journal-full-title]
Clinical anatomy (New York, N.Y.)
[ISO-abbreviation]
Clin Anat
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
91.
Ramachandran R, Ocón-Grove OM, Metzger SL:
Molecular cloning and tissue expression of chicken AdipoR1 and AdipoR2 complementary deoxyribonucleic acids.
Domest Anim Endocrinol
; 2007 Jul;33(1):19-31
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
We also investigated the effect of feed deprivation on the expression of AdipoR1 or AdipoR2 mRNA in the chicken diencephalon, liver, anterior
pituitary
gland
, and adipose tissue.
By RT-PCR, we detected AdipoR1 and AdipoR2 mRNA in adipose tissue, liver, anterior
pituitary
gland
, diencephalon, skeletal muscle, kidney, spleen, ovary, and blood.
AdipoR1 or AdipoR2 mRNA expression in various tissues was quantified by real-time quantitative PCR, and AdipoR1 mRNA expression was the highest in skeletal muscle, adipose tissue and diencephalon, followed by kidney, ovary, liver, anterior
pituitary
gland
, and spleen.
AdipoR2 mRNA expression was the highest in adipose tissue followed by skeletal muscle, liver, ovary, diencephalon, anterior
pituitary
gland
, kidney, and spleen.
We also found that a 48 h feed deprivation significantly decreased AdipoR1 mRNA quantity in the chicken
pituitary
gland
, while AdipoR2 mRNA quantity was significantly increased in adipose tissue (P<0.05).
We conclude that the AdipoR1 and AdipoR2 genes are ubiquitously expressed in chicken tissues and that their expression is altered by feed deprivation in the anterior
pituitary
gland
and adipose tissue.
COS Scholar Universe.
author profiles
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 16697136.001).
[ISSN]
0739-7240
[Journal-full-title]
Domestic animal endocrinology
[ISO-abbreviation]
Domest. Anim. Endocrinol.
[Language]
eng
[Databank-accession-numbers]
GENBANK/ DQ072275/ DQ072276
[Publication-type]
Journal Article; Research Support, U.S. Gov't, Non-P.H.S.
[Publication-country]
United States
[Chemical-registry-number]
0 / Adiponectin; 0 / DNA, Complementary; 0 / Receptors, Cell Surface; 63231-63-0 / RNA
92.
Walvoord E:
Sex steroid replacement for induction of puberty in multiple pituitary hormone deficiency.
Pediatr Endocrinol Rev
; 2009 Jan;6 Suppl 2:298-305
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Sex steroid replacement for induction of puberty in multiple
pituitary
hormone deficiency.
Hypopituitarism results from the inability of the
pituitary
gland
to make sufficient levels of more than one of the following hormones: adrenocorticotrophic hormone, thyroid-stimulating hormone, luteinizing hormone, follicle-stimulating hormone, prolactin, and growth hormone (GH).
This review addresses the main factors that need to be considered when initiating sex steroid replacement in pubertal age patients with multiple
pituitary
hormone deficiency and offers some insight into newer treatment options.
[MeSH-major]
Estrogens / administration & dosage. Hormone Replacement Therapy / methods. Hypopituitarism / drug therapy.
Pituitary
Hormones / administration & dosage.
Pituitary
Hormones / deficiency. Puberty / drug effects. Testosterone / administration & dosage
MedlinePlus Health Information.
consumer health - Hormone Replacement Therapy
.
MedlinePlus Health Information.
consumer health - Puberty
.
Hazardous Substances Data Bank.
TESTOSTERONE
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
(PMID = 19337185.001).
[ISSN]
1565-4753
[Journal-full-title]
Pediatric endocrinology reviews : PER
[ISO-abbreviation]
Pediatr Endocrinol Rev
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
Israel
[Chemical-registry-number]
0 / Estrogens; 0 / Pituitary Hormones; 3XMK78S47O / Testosterone
[Number-of-references]
27
93.
Kim JP, Park BJ, Kim SB, Lim YJ:
Pituitary Apoplexy due to Pituitary Adenoma Infarction.
J Korean Neurosurg Soc
; 2008 May;43(5):246-9
[Fulltext service]
Download
fulltext PDF
of
this article and others
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Pituitary
Apoplexy due to
Pituitary
Adenoma Infarction.
Cause of
pituitary
apoplexy has been known as hemorrhage, hemorrhagic infarction or infarction of
pituitary
adenoma or adjacent tissues of
pituitary
gland
.
However,
pituitary
apoplexy caused by pure infarction of
pituitary
adenoma has been rarely reported.
Here, we present the two cases
pituitary
apoplexies caused by
pituitary
adenoma infarction that were confirmed by transsphenoidal approach (TSA) and pathologic reports.
Pathologic report of first case revealed total
tumor
infarction of a nonfunctioning
pituitary
macroadenoma and second case partial
tumor
infarction of ACTH secreting
pituitary
macroadenoma.
Patients with
pituitary
apoplexy which was caused by
pituitary
adenoma infarction unrelated to hemorrhage or hemorrhagic infarction showed good response to TSA treatment.
Further study on the predisposing factors of
pituitary
apoplexy and the mechanism of infarction in
pituitary
adenoma is necessary.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
[Email]
Email this result item
Email the results to the following email address:
[X] Close
[Cites]
AJNR Am J Neuroradiol. 2007 Nov-Dec;28(10):2023-9
[
17898201.001
]
[Cites]
J Neurol Neurosurg Psychiatry. 2001 Oct;71(4):542-5
[
11561045.001
]
[Cites]
J Neurosurg. 2006 Jun;104(6):931-7
[
16776337.001
]
[Cites]
J Neurosurg. 2006 Jun;104(6):892-8
[
16776332.001
]
[Cites]
Pituitary. 2005;8(2):81-7
[
16195779.001
]
[Cites]
Medicine (Baltimore). 2005 May;84(3):188-96
[
15879908.001
]
[Cites]
Neurosurgery. 2005;56(1):65-72; discussion 72-3
[
15617587.001
]
[Cites]
J Clin Endocrinol Metab. 2004 Nov;89(11):5649-54
[
15531524.001
]
[Cites]
J Neurosurg. 1950 Sep;7(5):421-39
[
14774761.001
]
[Cites]
Acta Anaesthesiol Scand. 1999 Feb;43(2):236-8
[
10027037.001
]
[Cites]
Endocr J. 1999 Feb;46(1):147-51
[
10426579.001
]
[Cites]
Postgrad Med J. 1996 Mar;72(845):172-3
[
8731710.001
]
[Cites]
Neurosurgery. 1984 Mar;14(3):363-73
[
6369168.001
]
[Cites]
J Neurosurg. 1981 Aug;55(2):187-93
[
7252541.001
]
[Cites]
AJNR Am J Neuroradiol. 2002 Aug;23(7):1240-5
[
12169486.001
]
[Cites]
Endocr J. 2001 Aug;48(4):493-8
[
11603573.001
]
[Cites]
J Clin Neurosci. 2006 Dec;13(10):1057-62
[
17071092.001
]
(PMID = 19096606.001).
[ISSN]
2005-3711
[Journal-full-title]
Journal of Korean Neurosurgical Society
[ISO-abbreviation]
J Korean Neurosurg Soc
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Korea (South)
[Other-IDs]
NLM/ PMC2588219
[Keywords]
NOTNLM ; Pituitary adenoma infarction / Pituitary apoplexy
94.
Bertola G, Giambona S, Balza G, Oriani A, Sironi C, Calabrese G, Colombo E:
[Panhypopituitarism from pituitary metastases of breast cancer].
Recenti Prog Med
; 2007 Feb;98(2):87-9
[Fulltext service]
Get downloadable
fulltext PDFs
of
articles closely matching to this article
, as many as you want.
[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
[Panhypopituitarism from
pituitary
metastases of breast cancer].
[Transliterated title]
Panipopituitarismo
da
metastasi ipofisarie di carcinoma mammario.
Metastases to the
pituitary
gland
are uncommon causes of hypopituitarism, to be particularly considered in patients affected with disseminated cancers, arising in the breast or in the lung.
Differential
diagnos