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1
benign pancreatic neoplasm nos 2005:2010[pubdate] *count=100
14428 results
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Items 1 to 100 of about 14428
1.
Chen XL, Ma Y, Wan Y, Duan LG:
Experimental study of the safety of pancreas cryosurgery: the comparison of 2 different techniques of cryosurgery.
Pancreas
; 2010 Jan;39(1):92-6
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[Title]
Experimental study of the safety
of pancreas
cryosurgery: the comparison of 2 different techniques of cryosurgery.
OBJECTIVES: To test the feasibility of cryosurgery for
pancreatic
carcinoma and to observe the consequence of cryosurgery by 2 different techniques.
METHODS: Twelve healthy pigs underwent laparotomy, during which, chop amputation of common bile duct and duodenum were performed, meanwhile other intra-abdominal organs with
the pancreas
were isolated.
Two different techniques of cryosurgery were performed on
the pancreas
.
All animals in group B survived during the observation, in which only a transient increment and a gradual correction of
pancreatic
amylase level were recorded.
Small
pancreatic
pseudocyst occurred in 1 case.
CONCLUSIONS: Mild hypothermic cryosurgery with liquid nitrogen superficial refrigeration might lead to
pancreatic
injury and induce acute pancreatitis, yet deep hypothermic cryosurgery with adequate time showed a promising effect in destroying
pancreatic
tissue and preventing acute pancreatitis.
[MeSH-major]
Cryosurgery / methods.
Pancreas
/ surgery
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
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(PMID = 19952969.001).
[ISSN]
1536-4828
[Journal-full-title]
Pancreas
[ISO-abbreviation]
Pancreas
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
2.
Ryan GA, Wang CJ, Chamberlain JL, Attridge K, Schmidt EM, Kenefeck R, Clough LE, Dunussi-Joannopoulos K, Toellner KM, Walker LS:
B1 cells promote pancreas infiltration by autoreactive T cells.
J Immunol
; 2010 Sep 01;185(5):2800-7
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[Title]
B1 cells promote
pancreas
infiltration by autoreactive T cells.
The entry of autoreactive T cells into
the pancreas
is a critical checkpoint in the development of autoimmune diabetes.
In transgenic mice with islet-specific T cells, but no B cells, T cells are primed in the
pancreatic
lymph node but fail to enter
the pancreas
.
Reconstitution of the B1 cell population by adoptive transfer permits extensive T cell
pancreas
infiltration.
Reconstituted B1 cells traffic to
the pancreas
and modify expression of adhesion molecules on
pancreatic
vasculature, notably VCAM-1.
Despite substantial
pancreas
infiltration, islet destruction is minimal unless regulatory T cells are depleted.
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(PMID = 20675587.001).
[ISSN]
1550-6606
[Journal-full-title]
Journal of immunology (Baltimore, Md. : 1950)
[ISO-abbreviation]
J. Immunol.
[Language]
eng
[Grant]
United Kingdom / Wellcome Trust / / 085896; United Kingdom / Medical Research Council / / G120/854; United Kingdom / Medical Research Council / / G0802382; United Kingdom / Medical Research Council / / ; United Kingdom / Wellcome Trust / /
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Vascular Cell Adhesion Molecule-1; 9006-59-1 / Ovalbumin
[Other-IDs]
NLM/ EMS58025; NLM/ PMC3983558
3.
Wente MN, Schmied BM, Schmidt J, Büchler MW:
[Differentiated therapy for intraductal papillary mucinous neoplasms].
Chirurg
; 2009 Jan;80(1):7-13
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Intraductal papillary mucinous neoplasms (IPMN) of the
pancreas
are of increasing interest in the field of
pancreatic
surgery ever since their first description as an individual
pancreatic
tumor
entity in 1982.
Invasive IPMN forms (carcinoma in situ and invasive carcinoma) and in particular noninvasive IPMNs (adenoma and borderline tumors) reveal significantly better survival rates than ductal adenocarcinoma of the
pancreas
.
[MeSH-major]
Adenocarcinoma, Mucinous / surgery. Carcinoma,
Pancreatic
Ductal / surgery. Carcinoma, Papillary / surgery. Pancreatectomy / methods.
Pancreatic
Neoplasms / surgery
[MeSH-minor]
Adenocarcinoma /
diagnosis
. Adenocarcinoma / mortality. Adenocarcinoma / pathology. Adenocarcinoma / surgery. Carcinoma in Situ /
diagnosis
. Carcinoma in Situ / mortality. Carcinoma in Situ / pathology. Carcinoma in Situ / surgery. Humans. Magnetic Resonance Imaging.
Neoplasm
Invasiveness.
Neoplasm
Staging.
Pancreas
/ pathology. Prognosis. Radiography, Dual-Energy Scanned Projection. Survival Rate. Tomography, Spiral Computed
MedlinePlus Health Information.
consumer health - Pancreatic Cancer
.
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]
(PMID = 19082569.001).
[ISSN]
1433-0385
[Journal-full-title]
Der Chirurg; Zeitschrift fur alle Gebiete der operativen Medizen
[ISO-abbreviation]
Chirurg
[Language]
ger
[Publication-type]
Comparative Study; English Abstract; Journal Article; Review
[Publication-country]
Germany
[Number-of-references]
50
Advertisement
4.
Farnell MB:
Surgical management of intraductal papillary mucinous neoplasm (IPMN) of the pancreas.
J Gastrointest Surg
; 2008 Mar;12(3):414-6
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[Title]
Surgical management of intraductal papillary mucinous
neoplasm
(IPMN) of the
pancreas
.
Intraductal papillary mucinous
neoplasm
(IPMN) of the
pancreas
is characterized by papillary growths within the
pancreatic
ductal system that are at risk for undergoing malignant transformation.
When operation is indicated, targeted
pancreatic
resection with frozen-section analysis of margins is recommended.
Survival following
pancreatic
resection for noninvasive IPMN is excellent.
The risk of recurrence following
pancreatic
resection for invasive IPMN is significant.
Surveillance is warranted both for patients subjected to
pancreatic
resection and for those under observation with side branch IPMN.
Much is yet to be learned regarding this
neoplasm
, and surgical management remains in evolution.
[MeSH-major]
Adenoma / surgery. Carcinoma in Situ / surgery. Carcinoma,
Pancreatic
Ductal / surgery. Pancreatectomy.
Pancreatic
Neoplasms / surgery. Precancerous Conditions / surgery
[MeSH-minor]
Carcinoma, Papillary / surgery. Diagnostic Imaging. Dilatation, Pathologic. Humans.
Pancreatic
Ducts / pathology. Pancreaticoduodenectomy
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consumer health - Pancreatic Cancer
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[ISSN]
1091-255X
[Journal-full-title]
Journal of gastrointestinal surgery : official journal of the Society for Surgery of the Alimentary Tract
[ISO-abbreviation]
J. Gastrointest. Surg.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Number-of-references]
24
5.
Chu K, Tsai MJ:
Neuronatin, a downstream target of BETA2/NeuroD1 in the pancreas, is involved in glucose-mediated insulin secretion.
Diabetes
; 2005 Apr;54(4):1064-73
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[Title]
Neuronatin, a downstream target of BETA2/NeuroD1 in
the pancreas
, is involved in glucose-mediated insulin secretion.
BETA2 plays an important role in the development of the
pancreas
and the nervous system.
Using microarray technology, we identified neuronatin (Nnat) as differentially expressed between wild-type (WT) and knockout (KO)
pancreatic
RNA from embryonic day 14 (e14.5).
Northern blot and in situ hybridization analysis of WT and KO samples confirmed the downregulation of Nnat in
pancreas of
mutant BETA2 embryos.
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.
Gene Ontology.
gene/protein/disease-specific - Gene Ontology annotations from this paper
.
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(registration required).
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.
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(subscription/membership/fee required).
Mouse Genome Informatics (MGI).
Mouse Genome Informatics (MGI)
.
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(PMID = 15793245.001).
[ISSN]
0012-1797
[Journal-full-title]
Diabetes
[ISO-abbreviation]
Diabetes
[Language]
ENG
[Grant]
United States / NICHD NIH HHS / HD / R01 HD017379; United States / NICHD NIH HHS / HD / R37 HD017379; United States / NICHD NIH HHS / HD / HD-17379
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, P.H.S.
[Publication-country]
United States
[Chemical-registry-number]
0 / Basic Helix-Loop-Helix Transcription Factors; 0 / DNA-Binding Proteins; 0 / Insulin; 0 / Membrane Proteins; 0 / Nerve Tissue Proteins; 0 / Neurod1 protein, mouse; 0 / Nnat protein, mouse; 0 / RNA, Small Interfering; 0 / Trans-Activators; IY9XDZ35W2 / Glucose
[Other-IDs]
NLM/ NIHMS2527; NLM/ PMC1197706
6.
Schima W, Ba-Ssalamah A, Plank C, Kulinna-Cosentini C, Püspök A:
[Pancreas. Part I: congenital changes, acute and chronic pancreatitis].
Radiologe
; 2006 Apr;46(4):321-35; quiz 336
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[Title]
[
Pancreas
. Part I: congenital changes, acute and chronic pancreatitis].
[Transliterated title]
Pankreas
. Teil I: Angeborene Veränderungen, akute und chronische Pankreatitis.
The pancreas
develops from ventral and the dorsal buds, which undergo fusion.
Failure to fuse results in
pancreas
divisum, which is defined by separate
pancreatic
ductal systems draining into the duodenum.
Risk of developing pancreatitis is increased in
pancreas
divisum because of insufficient drainage.
MR cholangiopancreatography (MRCP) is the technique of choice for detecting
pancreas
divisum non-invasively.
Annular
pancreas
is the result of incomplete rotation of the
pancreatic
bud around the duodenum with the persistence of parenchyma or a fibrous band encircling (and sometimes stenosing) the duodenum.
Chronic pancreatitis results in relentless and irreversible loss of exocrine (and sometimes endocrine) function of the
pancreas
.
Inflammatory pseudotumor in chronic pancreatitis and groove pancreatitis are difficult to differentiate from
pancreatic
cancer.
In these cases, multiple imaging methods such as MDCT, MRI and endosonography including biopsy may be used to make a
diagnosis
.
[MeSH-major]
Cholangiopancreatography, Magnetic Resonance / methods. Image Enhancement / methods. Pancreatitis / congenital. Pancreatitis /
diagnosis
. Tomography, X-Ray Computed / methods
Genetic Alliance.
consumer health - Pancreatitis
.
MedlinePlus Health Information.
consumer health - CT Scans
.
MedlinePlus Health Information.
consumer health - Pancreatitis
.
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[ISSN]
0033-832X
[Journal-full-title]
Der Radiologe
[ISO-abbreviation]
Radiologe
[Language]
ger
[Publication-type]
English Abstract; Journal Article; Review
[Publication-country]
Germany
[Chemical-registry-number]
0 / Contrast Media
[Number-of-references]
33
7.
Skowronek J, Sowier A, Skrzywanek P:
Trans-hepatic technique and intraluminal Pulsed Dose Rate (PDR-BT) brachytherapy in treatment of locally advanced bile duct and pancreas cancer.
J Contemp Brachytherapy
; 2009 Jun;1(2):97-104
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[Title]
Trans-hepatic technique and intraluminal Pulsed Dose Rate (PDR-BT) brachytherapy in treatment of locally advanced bile duct and
pancreas
cancer.
PURPOSE: To assess the feasibility of intraluminal palliative Pulsed Dose Rate (PDR-BT) brachytherapy in the treatment of locally advanced bile duct and
pancreas
cancer.
MATERIAL AND METHODS: Forty-eight patients with advanced bile duct or
pancreas
cancer, disqualified from surgery or radical external beam radiation therapy (EBRT), were treated with trans-hepatic technique and intraluminal PDR-BT: 29 patients with bile duct cancer and 19 -
pancreas
cancer.
Target volume encompassed
tumor
visualized at cholangiography with one or two cm margin measured proximally and distally.
In 19 out of 29 (65.5%) of bile duct cancer cases and in 10 out of 19 (52.6%)
of pancreas
cancer patients clinical improvement (decrease of jaundice) was noted in first control after 4 weeks.
Median overall survival time (OS) for bile ducts cancer patients was 11.2 months and for
pancreas
cancer patients - 5.2 months.
2. In most cases a satisfied palliative effect was achieved, however it was more apparent in bile duct cancer patients then in
pancreas
cancer patients.
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(PMID = 27795719.001).
[ISSN]
1689-832X
[Journal-full-title]
Journal of contemporary brachytherapy
[ISO-abbreviation]
J Contemp Brachytherapy
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Poland
[Keywords]
NOTNLM ; PDR brachytherapy / bile duct cancer / pancreas cancer
8.
Rangel EB, Melaragno CS, Neves MD, Dib SA, Gonzalez AM, Linhares MM, Pacheco-Silva A, Sá JR:
Family history of diabetes as a new determinant of insulin sensitivity and secretion in patients who have undergone a simultaneous pancreas-kidney transplant.
Exp Clin Transplant
; 2010 Mar;8(1):29-37
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[Title]
Family history of diabetes as a new determinant of insulin sensitivity and secretion in patients who have undergone a simultaneous
pancreas
-kidney transplant.
OBJECTIVES: We used homeostasis model assessment to investigate insulin sensitivity and secretion after a simultaneous
pancreas
-kidney transplant or kidney transplant alone.
MATERIALS AND METHODS: Factors (eg, age, sex, race, delayed kidney allograft function) were correlated with homeostasis model assessment of beta-cell function and homeostasis model assessment of insulin sensitivity values after simultaneous
pancreas
-kidney transplant (n=89) or kidney transplant alone (n=68), and the results were compared with those in healthy subjects (n=49).
RESULTS: Homeostasis model assessment of beta-cell function values were similar in patients who underwent kidney transplant alone or a simultaneous
pancreas
-kidney transplant, and were higher than homeostasis model assessment of beta cell function values in healthy subjects.
The homeostasis model assessment of insulin sensitivity showed intermediate values for patients who underwent a simultaneous
pancreas
-kidney transplant and correlated with prednisone dosages (in those who underwent kidney transplant alone) and tacrolimus levels (in patients who underwent a simultaneous
pancreas
-kidney transplant).
Homeostasis model assessment of beta-cell function values correlated with prednisone dosages in both groups and with tacrolimus levels in only those who underwent a simultaneous
pancreas
-kidney transplant.
A family history of diabetes in subjects who underwent a simultaneous
pancreas
-kidney transplant correlated with homeostasis model assessment of beta-cell function results and homeostasis model assessment of insulin sensitivity results.
A family history of diabetes was linked with higher values of insulin secretion and lower insulin sensitivity in patients who underwent a simultaneous
pancreas
-kidney transplant.
[MeSH-major]
Diabetes Mellitus / genetics. Insulin / secretion. Insulin Resistance / physiology. Kidney Transplantation / physiology.
Pancreas
Transplantation / physiology. Pedigree
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(PMID = 20199368.001).
[ISSN]
2146-8427
[Journal-full-title]
Experimental and clinical transplantation : official journal of the Middle East Society for Organ Transplantation
[ISO-abbreviation]
Exp Clin Transplant
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Turkey
[Chemical-registry-number]
0 / Blood Glucose; 0 / C-Peptide; 0 / Immunosuppressive Agents; 0 / Insulin; VB0R961HZT / Prednisone; WM0HAQ4WNM / Tacrolimus
9.
Hering R, Bolten JC, Kreyer S, Berg A, Wrigge H, Zinserling J, Putensen C:
Spontaneous breathing during airway pressure release ventilation in experimental lung injury: effects on hepatic blood flow.
Intensive Care Med
; 2008 Mar;34(3):523-7
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During APRV with SB blood flow (ml g(-1) min(-1)) was 0.91+/-0.26 (hepatic arterial), 0.29+/-0.05 (stomach), 0.64+/-0.08 (duodenum), 0.62+/-0.10 (jejunum), 0.53+/-0.07 (ileum), 0.53+/-0.07 (colon), 0.46+/-0.09 (
pancreas
) and 3.59+/-0.55 (spleen).
During APRV without SB applying high P(aw) it decreased to 0.13+/-0.01 (stomach), 0.37+/-0.03 (duodenum), 0.29+/-0.03 (jejunum), 0.31+/-0.05 (ileum), 0.32+/-0.03 (colon) and 0.23+/-0.04 (
pancreas
) p<0.01, respectively.
During APRV without SB applying same Paw limits it decreased to 0.18+/-0.03 (stomach, p<0.01), 0.47+/-0.06 (duodenum, p<0.05), 0.38+/-0.05 (jejunum, p<0.01), 0.36+/-0.03 (ileum, p<0.05), 0.39+/-0.05 (colon, p<0.05), and 0.27+/-0.04 (
pancreas
, p<0.01).
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[CommentIn]
Intensive Care Med. 2008 Mar;34(3):397-9
[
18087690.001
]
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Arch Surg. 1976 Aug;111(8):881-3
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(PMID = 18087691.001).
[ISSN]
0342-4642
[Journal-full-title]
Intensive care medicine
[ISO-abbreviation]
Intensive Care Med
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
2UMI9U37CP / Oleic Acid; S88TT14065 / Oxygen
10.
Nishino T, Toki F, Oyama H, Shimizu K, Shiratori K:
Long-term outcome of autoimmune pancreatitis after oral prednisolone therapy.
Intern Med
; 2006;45(8):497-501
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OBJECTIVE: We investigated the long-term outcome of autoimmune pancreatitis (AIP) including morphological changes in
the pancreas
,
pancreatic
duct, biliary tract,
pancreatic
function, and changes in the clinical manifestations after oral prednisolone (PSL) therapy.
The morphological findings consisted of
pancreatic
enlargement (n=12), an irregularly narrowed main
pancreatic
duct (n=12), and bile duct stricture (n=10), and salivary gland swelling was observed in six patients.
The enlargement of the
pancreas
and the irregularly narrowed main
pancreatic
duct improved to almost normal.
Pancreatic
atrophy developed in four of them (4/12, 33%), but no
pancreatic
calcification was observed in any of the patients.
There was no recurrence of enlargement of the
pancreas
or irregularly narrowed main
pancreatic
duct after PSL therapy, but the bile duct stricture recurred in one case, and in three cases there was a relapse of salivary gland swelling that required a temporary increase in PSL dose during tapering.
No deterioration of
pancreatic
exocrine function was detected in any of the patients.
A malignant
tumor
was diagnosed in two patients during PSL therapy: early gastric cancer in one and rectal cancer in the other.
CONCLUSIONS: AIP treated with PSL has a favorable long-term outcome based on the morphological findings and assessments of
pancreatic
function.
[MeSH-minor]
Administration, Oral. Aged. Biliary Tract / pathology. Cholangiopancreatography, Endoscopic Retrograde. Female. Humans. Immunoglobulin G / blood. Male. Middle Aged.
Pancreatic
Ducts / pathology.
Pancreatic
Function Tests. Salivary Glands / pathology. Sjogren's Syndrome /
diagnosis
. Tomography, X-Ray Computed
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(PMID = 16702740.001).
[ISSN]
1349-7235
[Journal-full-title]
Internal medicine (Tokyo, Japan)
[ISO-abbreviation]
Intern. Med.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Japan
[Chemical-registry-number]
0 / Glucocorticoids; 0 / Immunoglobulin G; 9PHQ9Y1OLM / Prednisolone
11.
Jia D, Taguchi M, Otsuki M:
Preventive and therapeutic effects of the protease inhibitor camostat on pancreatic fibrosis and atrophy in CCK-1 receptor-deficient rats.
Pancreas
; 2005 Jan;30(1):54-61
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[Title]
Preventive and therapeutic effects of the protease inhibitor camostat on
pancreatic
fibrosis and atrophy in CCK-1 receptor-deficient rats.
OBJECTIVES: Recent studies have demonstrated that synthetic protease inhibitors could ameliorate the progression of
pancreatic
fibrosis in some animal models.
Since oral administration of protease inhibitors increases the plasma cholecystokinin (CCK) levels and causes hypertrophy of the
pancreas
in rats, there is a possibility that the protease inhibitor inhibits fibrosis in
the pancreas
via endogenous CCK release.
We examined the effects of camostat, a synthetic protease inhibitor, on histopathologic changes in Otsuka Long-Evans Tokushima Fatty (OLETF) rat that has genetically no expression of CCK-1 receptor and displays inflammation and degeneration of the
pancreas
.
RESULTS:
Pancreatic
wet weight and
pancreatic
contents of protein, DNA, amylase, lipase, and trypsin in camostat-treated rats were significantly higher than those in the untreated control rats.
Immunohistochemical studies of the
pancreas
showed that expressions of interleukin-1beta, interleukin-6,
tumor
necrosis factor-alpha, and alpha-smooth muscle actin in camostat-treated rats were greatly suppressed compared with those in the untreated control rats.
Atrophy and fibrosis in
the pancreas
observed in the untreated control rats were not found in camostat-fed rats.
CONCLUSION: The results of the present study suggest that camostat greatly inhibits
pancreatic
inflammation and prevents and reverses fibrosis and atrophy of the
pancreas
in the genetically obese and CCK-1 receptor-deficient OLETF rats.
[MeSH-minor]
Actins / metabolism. Amylases / metabolism. Animals. Atrophy. Eating. Fibrosis. Interleukin-6 / metabolism. Lipase / metabolism. Male. Obesity / complications. Organ Size.
Pancreas
/ enzymology.
Pancreas
/ pathology. Rats. Rats, Inbred OLETF. Transforming Growth Factor beta / metabolism. Trypsin / metabolism.
Tumor
Necrosis Factor-alpha / metabolism
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(PMID = 15632700.001).
[ISSN]
1536-4828
[Journal-full-title]
Pancreas
[ISO-abbreviation]
Pancreas
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Actins; 0 / Interleukin-6; 0 / Protease Inhibitors; 0 / Receptor, Cholecystokinin A; 0 / Transforming Growth Factor beta; 0 / Tumor Necrosis Factor-alpha; 0FD207WKDU / camostat; 4V7M9137X9 / Gabexate; EC 3.1.1.3 / Lipase; EC 3.2.1.- / Amylases; EC 3.4.21.4 / Trypsin
12.
Cross SE, Hughes SJ, Partridge CJ, Clark A, Gray DW, Johnson PR:
Collagenase penetrates human pancreatic islets following standard intraductal administration.
Transplantation
; 2008 Oct 15;86(7):907-11
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[Title]
Collagenase penetrates human
pancreatic
islets following standard intraductal administration.
The aim of this study was to characterize collagenase distribution in relation to islets in infused human
pancreases
.
METHODS: : Human
pancreases
were retrieved from multiorgan donors with appropriate consent.
RESULTS: : Collagenase labeling was widespread throughout
the pancreas
, associated with collagen VI, and adjacent to CK19-labeled ducts.
Intraislet collagenase was observed in 70%+/-3% of islets in the
pancreatic
tail, compared with 58%+/-2% and 53%+/-2% of islets in the body and neck, respectively (P<0.05 tail vs. neck), and was more prevalent in islets with diameters more than 150 microm (98%+/-1% of islets >150 microm vs. 52%+/-2% of islets <150 microm, P<0.05).
There was no difference in intraislet collagenase labeling between perfused and syringe-loaded
pancreases
.
[MeSH-minor]
Drug Administration Routes. Humans. Organ Preservation Solutions.
Pancreas
/ anatomy & histology.
Pancreas
/ enzymology.
Pancreas
, Exocrine / enzymology.
Pancreatic
Ducts / metabolism
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(PMID = 18852654.001).
[ISSN]
1534-6080
[Journal-full-title]
Transplantation
[ISO-abbreviation]
Transplantation
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Organ Preservation Solutions; EC 3.4.24.- / Collagenases
13.
Atias S, Mizrahi SS, Shaco-Levy R, Yussim A:
Preservation of pancreatic tissue morphology, viability and energy metabolism during extended cold storage in two-layer oxygenated University of Wisconsin/perfluorocarbon solution.
Isr Med Assoc J
; 2008 Apr;10(4):273-6
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[Title]
Preservation of
pancreatic
tissue morphology, viability and energy metabolism during extended cold storage in two-layer oxygenated University of Wisconsin/perfluorocarbon solution.
BACKGROUND: In contrast to the relative scarcity of donor kidneys and hearts, the potential supply of deceased donor
pancreata
is exceeding the demand.
However, this organ surplus is not being fully realized because, in current transplantation practice, the duration
of pancreas
storage before transplantation is limited to 8-10 hours due to the extreme vulnerability of
pancreatic
tissue to anaerobic damage caused by preservation.
OBJECTIVES: To reduce cold ischemic injury in order to increase the utilization of donor
pancreases
in Israel for whole-organ and cell transplantation.
METHODS: We evaluated a novel two-layer preservation oxygenated cold storage method that uses perfluorocarbon to continuously supply oxygen to
the pancreas
during preservation in conventional University of Wisconsin solution.
RESULTS:
Pancreatic
tissue morphology, viability and adenosine-triphosphate content were serially examined during preservation of the pig
pancreas
for 24 hours either by a two-layer or by conventional simple cold storage.
CONCLUSIONS: The UW/PFC two-layer preservation method allowed tissue ATP synthesis and amelioration of cold ischemic tissue damage during extended 24 hour
pancreas
preservation.
This method could be implemented in clinical practice to maximize utilization
of pancreata
for whole-organ and islet transplantation as well as for
pancreas
sharing with remote centers.
[MeSH-major]
Cryopreservation / methods. Fluorocarbons. Organ Preservation / methods.
Pancreas
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(PMID = 18548980.001).
[ISSN]
1565-1088
[Journal-full-title]
The Israel Medical Association journal : IMAJ
[ISO-abbreviation]
Isr. Med. Assoc. J.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Israel
[Chemical-registry-number]
0 / Fluorocarbons; 0 / Organ Preservation Solutions; 8L70Q75FXE / Adenosine Triphosphate
14.
Malaise J, Secchi A, Caldara R, Tydén G, Sandberg J, Van Ophem D, Squifflet JP, EUROSPK Study Group:
Metabolic assessment after simultaneous pancreas-kidney transplantation.
Transplant Proc
; 2005 Jul-Aug;37(6):2851-2
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[Title]
Metabolic assessment after simultaneous
pancreas
-kidney transplantation.
Simultaneous
pancreas
-kidney (SPK) transplantation has become a standard therapy for patients with type 1 diabetes and end-stage renal disease.
PATIENTS AND METHODS: We analyzed 205 patients enrolled in the Euro-SPK001 study for fasting blood glucose, fasting C peptide, glycated hemoglobin (HbA(1c)), blood lipids (total cholesterol and triglycerides), and
pancreatic
enzymes at regular intervals during the study.
[MeSH-major]
Hemoglobin A, Glycosylated / analysis. Kidney Transplantation / physiology.
Pancreas
Transplantation / physiology
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(PMID = 16182831.001).
[ISSN]
0041-1345
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Multicenter Study; Randomized Controlled Trial; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Blood Glucose; 0 / C-Peptide; 0 / Hemoglobin A, Glycosylated; 0 / Immunosuppressive Agents; 0 / Lipids; EC 3.2.1.- / Amylases
15.
Ye R, Mareninova OA, Barron E, Wang M, Hinton DR, Pandol SJ, Lee AS:
Grp78 heterozygosity regulates chaperone balance in exocrine pancreas with differential response to cerulein-induced acute pancreatitis.
Am J Pathol
; 2010 Dec;177(6):2827-36
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[Title]
Grp78 heterozygosity regulates chaperone balance in exocrine
pancreas
with differential response to cerulein-induced acute pancreatitis.
The endoplasmic reticulum (ER) is abundant in the acinar cells of the exocrine
pancreas
.
Exocrine
pancreata of
RD-fed Grp78(+/-) mice in an outbred C57BL/6 × 129/sv genetic background exhibited ER lumen dilation, a reduction in chaperones calnexin (CNX) and calreticulin (CRT), and exacerbated pancreatitis associated with high CHOP induction.
Thus, in exocrine
pancreata
, Grp78 heterozygosity regulates ER chaperone balance, in dietary- and genetic background-dependent manners, and improved ER protein folding capacity might be protective against pancreatitis.
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[ISSN]
1525-2191
[Journal-full-title]
The American journal of pathology
[ISO-abbreviation]
Am. J. Pathol.
[Language]
ENG
[Grant]
United States / NIAAA NIH HHS / AA / AA16010; United States / NIAAA NIH HHS / AA / AA11999; United States / NIDDK NIH HHS / DK / DK070582; United States / NIAAA NIH HHS / AA / R21 AA016010; United States / NCI NIH HHS / CA / R01 CA027607; United States / NIDDK NIH HHS / DK / R21 DK070582; United States / NIDDK NIH HHS / DK / P30 DK048522; United States / NCI NIH HHS / CA / CA027607; United States / NIAAA NIH HHS / AA / P50 AA011999; United States / NIDDK NIH HHS / DK / DK048522
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
[Publication-country]
United States
[Chemical-registry-number]
0 / Dietary Fats; 0 / Gastrointestinal Agents; 0 / Heat-Shock Proteins; 0 / Molecular Chaperones; 0 / molecular chaperone GRP78; 888Y08971B / Ceruletide
[Other-IDs]
NLM/ PMC2993313
16.
Seki M, Ninomiya E, Takano K, Fujita R, Aruga A, Yamada K, Tanaka H, Matsueda K, Mikami K, Hiki N, Saiura A, Yamamoto J, Yamaguchi T, Yanagisawa A, Ikeda M, Sasaki K, Kato Y:
Pancreatogram findings for carcinoma in situ (CIS) of the pancreas seen on endoscopic retrograde cholangiopancreatography and postoperative pancreatography of resected specimens: can CIS be diagnosed preoperatively?
Pancreatology
; 2008;8(2):142-52
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[Title]
Pancreatogram findings for carcinoma in situ (CIS) of the
pancreas
seen on endoscopic retrograde cholangiopancreatography and postoperative pancreatography of resected specimens: can CIS be diagnosed preoperatively?
In addition, 7 of 79 invasive ductal carcinomata (IDC) of the
pancreas
were accompanied by CIS > or =2 cm long.
A total of 11 patients were reviewed here for pancreatographic findings for CIS of the
pancreas
.
METHODS: All resected pancreatobiliary materials were sliced serially at 5- to 8-mm intervals in a plane at right angles to the main
pancreatic
duct, referring to POP images.
CONCLUSIONS: I, N, G, and D are most important pancreatographic findings in ERCP and highly suggestive of CIS of the
pancreas
, so that whenever they are encountered, cytological and/or pathological examination of the
pancreatic
duct should be actively performed.
[MeSH-major]
Carcinoma in Situ / radiography. Cholangiopancreatography, Endoscopic Retrograde.
Pancreas
/ radiography.
Pancreatic
Neoplasms / radiography
[MeSH-minor]
Aged. Aged, 80 and over. Female. Humans. Incidental Findings. Male. Middle Aged.
Pancreatic
Ducts / radiography. Postoperative Care. Preoperative Care
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[Copyright]
Copyright 2008 S. Karger AG, Basel.
(PMID = 18382100.001).
[ISSN]
1424-3911
[Journal-full-title]
Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.]
[ISO-abbreviation]
Pancreatology
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Switzerland
17.
Vinkers MT, Rahmel AO, Slot MC, Smits JM, Schareck WD:
How to recognize a suitable pancreas donor: a Eurotransplant study of preprocurement factors.
Transplant Proc
; 2008 Jun;40(5):1275-8
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[Title]
How to recognize a suitable
pancreas
donor: a Eurotransplant study of preprocurement factors.
INTRODUCTION: Because of the increasing demand for
pancreas
transplantation, more marginal donors are offered to Eurotransplant.
The aim of this study was to validate a donor quality score that would facilitate recognition of a suitable
pancreas
donor among all reported donors.
MATERIALS AND METHODS: We analyzed all 3180 consecutively reported
pancreas
donors for the period between January 1, 2002 and June 30, 2005 and determined the influence of the preprocurement
pancreas
suitability score (P-PASS) on the acceptance of a
pancreas
.
RESULTS: Multiple regression analysis using
pancreas
acceptance as an outcome variable identified P-PASS > or = 17 as a significant cutoff point (P < .001).
Pancreata
from donors with P-PASS > or = 17 were three times more likely to be refused.
CONCLUSION: The donor score can help in screening for potential
pancreas
donors, where an ideal donor has a P-PASS < 17.
Our data demonstrate that consideration of a combination of preprocurement factors can help identify a suitable
pancreas
donor.
Therefore, we recommend that a
pancreas
donor score be calculated for each potential
pancreas
donor, and all donors with a P-PASS < 17 should be considered for
pancreas
donation.
[MeSH-major]
Pancreas
.
Pancreas
Transplantation / methods. Tissue Donors / statistics & numerical data. Tissue and Organ Procurement / methods
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consumer health - Organ Donation
.
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.
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(PMID = 18589086.001).
[ISSN]
0041-1345
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / ABO Blood-Group System
18.
Ulicna M, Danisovic L, Danihel L, Vojtassak J:
Diabetes--adult stem cells as an future alternative therapy?
Bratisl Lek Listy
; 2009;110(12):773-6
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One way to achieve a constant normoglycemic state without hypoglycemic episodes is either whole
pancreas
transplantation, or transplantation of isolated islets of Langerhans.
Another approach to correct the beta-cell deficit is the stimulation of beta-cells in
pancreas
to regeneration.
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.
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(PMID = 20196471.001).
[ISSN]
0006-9248
[Journal-full-title]
Bratislavské lekárske listy
[ISO-abbreviation]
Bratisl Lek Listy
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
Slovakia
[Number-of-references]
47
19.
Kawamoto S, Lawler LP, Horton KM, Eng J, Hruban RH, Fishman EK:
MDCT of intraductal papillary mucinous neoplasm of the pancreas: evaluation of features predictive of invasive carcinoma.
AJR Am J Roentgenol
; 2006 Mar;186(3):687-95
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[Title]
MDCT of intraductal papillary mucinous
neoplasm
of the
pancreas
: evaluation of features predictive of invasive carcinoma.
OBJECTIVE: The purpose of our study was to evaluate factors predictive of the presence of invasive carcinoma associated with intraductal papillary mucinous
neoplasm
(IPMN) of the
pancreas
on MDCT.
MATERIALS AND METHODS: Preoperative MDCT of 36 consecutive patients (23 men, 13 women; mean age, 66.6 years) who had undergone surgical resection and had a pathologic
diagnosis of
IPMN were retrospectively assessed.
Type of ductal involvement, location,
tumor
size in branch duct type and combined type lesions, caliber of the main
pancreatic
duct, caliber of the common bile duct or common hepatic duct, and solid appearance of the lesion were assessed on CT and correlated with pathologic findings for invasive carcinoma.
With invasive carcinoma, the size of the
tumor
in branch duct type and combined type, and the caliber of the main
pancreatic
duct were significantly larger compared with the lesions without invasive carcinoma (4.7 +/- 1.7 cm vs 2.6 +/- 1.4 cm [p = 0.0007] and 9.3 +/- 5.5 mm vs 4.6 +/- 4.1 mm [p = 0.006], respectively).
[MeSH-major]
Adenocarcinoma, Mucinous / radiography. Carcinoma,
Pancreatic
Ductal / radiography.
Pancreatic
Neoplasms / radiography. Tomography, X-Ray Computed / methods
[MeSH-minor]
Adult. Aged. Aged, 80 and over. Contrast Media. Female. Humans. Male. Middle Aged.
Neoplasm
Invasiveness. Predictive Value of Tests. Retrospective Studies. Sensitivity and Specificity
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.
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(PMID = 16498096.001).
[ISSN]
0361-803X
[Journal-full-title]
AJR. American journal of roentgenology
[ISO-abbreviation]
AJR Am J Roentgenol
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Contrast Media
20.
Mancuso F, Calvitti M, Luca G, Nastruzzi C, Baroni T, Mazzitelli S, Becchetti E, Arato I, Boselli C, Ngo Nselel MD, Calafiore R:
Acceleration of functional maturation and differentiation of neonatal porcine islet cell monolayers shortly in vitro cocultured with microencapsulated sertoli cells.
Stem Cells Int
; 2010;2010:587213
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The limited availability of cadaveric human donor
pancreata as
well as the incomplete success of the Edmonton protocol for human islet allografts fasten search for new sources of insulin the producing cells for substitution cell therapy of insulin-dependent diabetes mellitus (T1DM).
Starting from isolated neonatal porcine
pancreatic
islets (NPIs), we have obtained cell monolayers that were exposed to microencapsulated monolayered Sertoli cells (ESCs) for different time periods (7, 14, 21 days).
The insulin/c-kit positive cells might represent a new, still unknown functionally immature β-cell like element in the porcine
pancreas
.
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(PMID = 21048849.001).
[ISSN]
1687-9678
[Journal-full-title]
Stem cells international
[ISO-abbreviation]
Stem Cells Int
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
England
[Other-IDs]
NLM/ PMC2956457
21.
Siddique K, Ali Q, Mirza S, Jamil A, Ehsan A, Latif S, Malik AZ:
Evaluation of the aetiological spectrum of obstructive jaundice.
J Ayub Med Coll Abbottabad
; 2008 Oct-Dec;20(4):62-6
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The surgical jaundice can be caused by the obstruction of the bile duct as with gall stones, strictures, malignancy, such as cholangiocarcinoma (in which the jaundice is persistent and progressive), periampullary carcinoma, carcinoma gall bladder and carcinoma head
of pancreas
.
Malignant obstructive jaundice was seen in 34 (56.66%) patients while 26 (43.33%) had
benign
etiology.
Amongst the commonest symptom; clay coloured stools (75%) was more frequent in patients with malignant disease whereas abdominal pain (51.66%) was most common in
benign
conditions.
Commonest malignancy was Carcinoma (Ca) of the head
of pancreas
18/60 (30%) followed by Ca gall bladder 8/60 (13.33%), cholangiocarcinoma 7/60 (11.66%), and periampullary carcinoma 1/60 (1.66%).
Choledocholithiasis 21/60 (35%) was the commonest
benign
cause followed by stricture of common bile duct 3/60 (5%) and acute pancreatitis 2/60 (3.33%).
Ca head
of pancreas
is the commonest malignancy while Choledocholithiasis is the commonest
benign
cause.
[MeSH-major]
Jaundice, Obstructive /
diagnosis
. Jaundice, Obstructive / etiology
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(PMID = 19999207.001).
[ISSN]
1025-9589
[Journal-full-title]
Journal of Ayub Medical College, Abbottabad : JAMC
[ISO-abbreviation]
J Ayub Med Coll Abbottabad
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Pakistan
22.
Leslie MA, Moran ET Jr, Bedford MR:
The effect of phytase and glucanase on the ileal digestible energy of corn and soybean meal fed to broilers.
Poult Sci
; 2007 Nov;86(11):2350-7
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At the end of each experimental period, the broilers were euthanized and the contents of the ileum, duodenum and jejunum (pooled), and
pancreas
were removed for analysis.
The pancreas
and duodenal-jejunal samples were analyzed for proteolytic and amylase activity to determine the influence of practical levels of phytate on enzyme activity.
Results showed that neither phytase nor glucanase influenced enzyme activity in the digesta or
pancreas
, suggesting that practical levels of phytate did not influence the activity of proteolytic enzymes or amylase.
The IDE and DM digestibility of corn and the digesta and
pancreatic
enzyme activities increased with age, whereas the IDE of SBM was similar among age groups.
[MeSH-minor]
Aging. Animal Nutritional Physiological Phenomena. Animals. Diet / veterinary. Dietary Supplements. Energy Metabolism / drug effects. Male.
Pancreas
. Soybeans / metabolism. Zea mays / metabolism
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(PMID = 17954585.001).
[ISSN]
0032-5791
[Journal-full-title]
Poultry science
[ISO-abbreviation]
Poult. Sci.
[Language]
eng
[Publication-type]
Controlled Clinical Trial; Journal Article
[Publication-country]
United States
[Chemical-registry-number]
EC 3.1.3.26 / 6-Phytase; EC 3.2.1.- / Glycoside Hydrolases
23.
Andrades P, Asiedu C, Ray P, Rodriguez C, Goodwin J, McCarn J, Thomas JM:
Islet yield after different methods of pancreatic Liberase delivery.
Transplant Proc
; 2007 Jan-Feb;39(1):183-4
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[Title]
Islet yield after different methods of
pancreatic
Liberase delivery.
OBJECTIVE: Enzymatic digestion of the
pancreas
is a fundamental step in islet isolation and there are many ways to administer the enzyme during procurement.
The aim of this study was to evaluate the influence of different methods of Liberase delivery during
pancreas
harvest on the quality and quantity of islets.
After injection,
the pancreata
were harvested, digested in Liberase solution, mechanically disrupted, and purified using discontinuous gradient centrifugation.
CONCLUSION: Intraductal administration is the best enzyme delivery method for
pancreatic
islet isolation.
The
pancreatic
ducts are the most anatomic and physiological way to transport the enzyme uniformly inside
the pancreas
, determining an adequate digestion and better islet quantity and quality when compared with other delivery methods.
[MeSH-major]
Collagenases. Islets of Langerhans / cytology.
Pancreas
/ cytology. Thermolysin
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(PMID = 17275501.001).
[ISSN]
0041-1345
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Grant]
United States / NIDDK NIH HHS / DK / R03 DK58965-01
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
United States
[Chemical-registry-number]
EC 3.4.24.- / Collagenases; EC 3.4.24.- / Liberase; EC 3.4.24.27 / Thermolysin
24.
Miao G, Ito T, Uchikoshi F, Tanemura M, Kawamoto K, Shimada K, Nozawa M, Matsuda H:
Development of islet-like cell clusters after pancreas transplantation in the spontaneously diabetic Torri rat.
Am J Transplant
; 2005 Oct;5(10):2360-7
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[Title]
Development of islet-like cell clusters after
pancreas
transplantation in the spontaneously diabetic Torri rat.
Pancreas
transplantation (PTx) has evolved as a clinical therapy to achieve sustained euglycemia.
However, it remains unclear if naive diseased islets of the
pancreas
benefit from the avoidance of glucose toxicity by PTx.
Moreover, we found that the beta-cell mass was significantly increased in the naive
pancreases of
40-week-old PTx recipients (PTx40-naive).
Interestingly, islet-like cell clusters of varying size were found close to ductal structures of PTx40-naive
pancreases
, suggesting that these cells are derived from ductal cells.
Furthermore,
pancreatic
and duodenal homeobox factor-1 (PDX-1) was more clearly expressed in the nuclei of PTx40-naive
pancreatic
islet-like cell clusters.
Our results demonstrate the development of duct-derived beta cells in
the pancreas
of type 2 diabetic recipients after PTx.
[MeSH-major]
Diabetes Mellitus, Experimental / pathology. Islets of Langerhans / cytology.
Pancreas
Transplantation / methods.
Pancreas
Transplantation / pathology
[MeSH-minor]
Age of Onset. Animals. Blotting, Western. Cell Nucleus / metabolism. Cell Proliferation. Disease Models, Animal. Glucagon / chemistry. Glucose / pharmacology. Glucose / toxicity. Glucose Tolerance Test. Homeodomain Proteins / biosynthesis. Immunohistochemistry. Insulin / chemistry. Insulin / metabolism. Insulin / pharmacology. Insulin-Secreting Cells / cytology. Islets of Langerhans Transplantation / methods. Ki-67 Antigen / biosynthesis. Male.
Pancreas
/ metabolism.
Pancreas
/ pathology. Rats. Time Factors. Trans-Activators / biosynthesis
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.
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GLUCAGON
.
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(PMID = 16162183.001).
[ISSN]
1600-6135
[Journal-full-title]
American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons
[ISO-abbreviation]
Am. J. Transplant.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Denmark
[Chemical-registry-number]
0 / Homeodomain Proteins; 0 / Insulin; 0 / Ki-67 Antigen; 0 / Trans-Activators; 0 / pancreatic and duodenal homeobox 1 protein; 9007-92-5 / Glucagon; IY9XDZ35W2 / Glucose
25.
Liao Z, Gao R, Wang W, Ye Z, Lai XW, Wang XT, Hu LH, Li ZS:
A systematic review on endoscopic detection rate, endotherapy, and surgery for pancreas divisum.
Endoscopy
; 2009 May;41(5):439-44
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[Title]
A systematic review on endoscopic detection rate, endotherapy, and surgery for
pancreas
divisum.
BACKGROUND AND STUDY AIMS: The rates for endoscopic detection
of pancreas
divisum at routine endoscopic retrograde cholangiopancreatography (ERCP) vary worldwide, and the sample sizes in the reported studies on endoscopy and surgery for
pancreas
divisum are very small and variable.
The aim of this study was to systematically analyze the pooled data and determine endoscopic detection rates for
pancreas
divisum and pain relief rates in patients with
pancreas
divisum after endotherapy or surgery.
MATERIALS AND METHODS: A search for published data was performed by using the Medline database (1950 to 1st May 2008) with "
pancreas
divisum" as the keyword.
Publications, mainly on endoscopic detection rate, endotherapy, or surgery for
pancreas
divisum, were deemed relevant, and were further fully reviewed and analyzed.
The overall endoscopic detection rate for
pancreas
divisum was 2.9% (899/31,413), with the rate being significantly higher in the United States (5.8%) and Europe (6.0%) than in Asia (1.5%) (both P < 0.001).
In addition, there were significant differences in the combined response rates (for endotherapy and for surgery) between patients with
pancreas
divisum of acute recurrent pancreatitis (ARP)-type (81.2 %) compared with chronic pancreatitis-type (68.8%), and between ARP-type and pain-type (53.1%) (both P < 0.05).
CONCLUSIONS: The endoscopic detection rate for
pancreas
divisum is much higher in western countries than in Asian countries.
The pooled response rates of patients with
pancreas
divisum to endotherapy and surgery are similar in the reported series.
Patients with ARP-type
pancreas
divisum respond better to endotherapy or surgery than those with chronic pancreatitis-type and pain-type.
[MeSH-major]
Cholangiopancreatography, Endoscopic Retrograde.
Pancreas
/ abnormalities. Pancreatitis, Acute Necrotizing /
diagnosis
. Pancreatitis, Chronic /
diagnosis
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(PMID = 19337962.001).
[ISSN]
1438-8812
[Journal-full-title]
Endoscopy
[ISO-abbreviation]
Endoscopy
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
Germany
[Chemical-registry-number]
EC 3.1.1.3 / Lipase; EC 3.2.1.- / Amylases
[Number-of-references]
51
26.
Keenan DB, Cartaya R, Mastrototaro JJ:
The pathway to the closed-loop artificial pancreas: research and commercial perspectives.
Pediatr Endocrinol Rev
; 2010 Aug;7 Suppl 3:445-51
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[Title]
The pathway to the closed-loop artificial
pancreas
: research and commercial perspectives.
In addition, artificial
pancreas
(AP) research has progressed to clinical studies, using combinations of commercially available devices.
[MeSH-major]
Diabetes Mellitus / drug therapy. Diabetes Mellitus / metabolism. Insulin Infusion Systems.
Pancreas
, Artificial
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(PMID = 20877259.001).
[ISSN]
1565-4753
[Journal-full-title]
Pediatric endocrinology reviews : PER
[ISO-abbreviation]
Pediatr Endocrinol Rev
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
Israel
27.
Kartalis N, Lindholm TL, Aspelin P, Permert J, Albiin N:
Diffusion-weighted magnetic resonance imaging of pancreas tumours.
Eur Radiol
; 2009 Aug;19(8):1981-90
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[Title]
Diffusion-weighted magnetic resonance imaging
of pancreas
tumours.
The purpose of this study was to evaluate the accuracy of diffusion-weighted imaging (DWI) in
diagnosis of pancreas
cancer, to compare DWI with a conventional comprehensive MRI (MRI-c) and to analyse apparent diffusion coefficient (ADC) values of lesions.
Thirty-six patients with
pancreatic
lesions (12 malignant and 24
benign
) and 39 patients without lesions were included.
Mean ADC values of malignant lesions were significantly lower than those of
benign
lesions.
DWI has a similar accuracy to MRI-c in
diagnosis of pancreas
cancer.
[MeSH-major]
Diffusion Magnetic Resonance Imaging / methods.
Pancreatic
Neoplasms /
diagnosis
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[CommentIn]
Eur Radiol. 2010 Jul;20(7):1768-9; author reply 1770-1
[
20204646.001
]
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(PMID = 19308414.001).
[ISSN]
1432-1084
[Journal-full-title]
European radiology
[ISO-abbreviation]
Eur Radiol
[Language]
eng
[Publication-type]
Comparative Study; Evaluation Studies; Journal Article
[Publication-country]
Germany
28.
Su C, Chen X, Zhang ZY, Gu WQ, Zhang MJ, Zhou GW, Li XY, Ning G, Li HW:
[Protective effect of heme oxygenase-1 induction in vivo to pancreas islet xenograft].
Zhonghua Wai Ke Za Zhi
; 2009 Aug 15;47(16):1249-52
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[Title]
[Protective effect of heme oxygenase-1 induction in vivo to
pancreas
islet xenograft].
OBJECTIVE: To study the protective effect of islet xenograft and its possible mechanism of high expression of heme oxygenase-1 (HO-1) in donor
pancreas
islet induced by cobalt protoporphyrin (CoPP).
[MeSH-major]
Heme Oxygenase-1 / metabolism.
Pancreas
Transplantation
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(PMID = 19781175.001).
[ISSN]
0529-5815
[Journal-full-title]
Zhonghua wai ke za zhi [Chinese journal of surgery]
[ISO-abbreviation]
Zhonghua Wai Ke Za Zhi
[Language]
chi
[Publication-type]
English Abstract; Journal Article
[Publication-country]
China
[Chemical-registry-number]
0 / Protoporphyrins; 130068-27-8 / Interleukin-10; 63AAN3JDZE / cobaltiprotoporphyrin; EC 1.14.99.3 / Heme Oxygenase-1
29.
Grützmann R, Niedergethmann M, Pilarsky C, Klöppel G, Saeger HD:
Intraductal papillary mucinous tumors of the pancreas: biology, diagnosis, and treatment.
Oncologist
; 2010;15(12):1294-309
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[Title]
Intraductal papillary mucinous tumors of the
pancreas
: biology,
diagnosis
, and treatment.
Pancreatic
intraductal papillary mucinous neoplasms (IPMNs) rank among the most common cystic tumors of the
pancreas
.
[MeSH-major]
Pancreatic
Neoplasms /
diagnosis
.
Pancreatic
Neoplasms / therapy
[MeSH-minor]
Adenocarcinoma, Mucinous /
diagnosis
. Adenocarcinoma, Mucinous / metabolism. Adenocarcinoma, Mucinous / therapy. Carcinoma,
Pancreatic
Ductal /
diagnosis
. Carcinoma,
Pancreatic
Ductal / metabolism. Carcinoma,
Pancreatic
Ductal / therapy. Carcinoma, Papillary /
diagnosis
. Carcinoma, Papillary / metabolism. Carcinoma, Papillary / therapy. Humans. Prognosis
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(PMID = 21147870.001).
[ISSN]
1549-490X
[Journal-full-title]
The oncologist
[ISO-abbreviation]
Oncologist
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Other-IDs]
NLM/ PMC3227924
30.
Li WC:
In vitro transdifferentiation of human hepatoma cells into pancreatic-like cells.
Methods Mol Biol
; 2009;560:99-110
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[Title]
In vitro transdifferentiation of human hepatoma cells into
pancreatic
-like cells.
Transdifferentiation from different cellular origins into
pancreatic
-like beta-cells is of clinical significance since this approach may offer a potential cure for diabetes.
In order to achieve this goal, the liver is considered as a suitable candidate due to its close developmental relationship to
the pancreas
, its large size and a well-documented regenerative capacity that could provide enough original tissues to initiate the transdifferentiation procedure.
In this chapter, we describe a protocol to overexpress Pdx1, a master regulator essential for
pancreas
development in the cultured human liver cell line, HepG2.
[MeSH-major]
Carcinoma, Hepatocellular / pathology. Cell Differentiation. Cytological Techniques. Liver Neoplasms / pathology.
Pancreas
/ cytology
[MeSH-minor]
Cell Line,
Tumor
. Cell Transdifferentiation. Humans. Insulin-Secreting Cells / cytology
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(PMID = 19504247.001).
[ISSN]
1940-6029
[Journal-full-title]
Methods in molecular biology (Clifton, N.J.)
[ISO-abbreviation]
Methods Mol. Biol.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
31.
Kawakami T, Saeki K, Takeyama N, Wu G, Sakudo A, Matsumoto Y, Hayashi T, Onodera T:
Detection of proteolytic cleavages of diabetes-associated protein IA-2 beta in the pancreas and the brain using novel anti-IA-2 beta monoclonal antibodies.
Int J Mol Med
; 2007 Aug;20(2):177-85
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[Title]
Detection of proteolytic cleavages of diabetes-associated protein IA-2 beta in
the pancreas
and the brain using novel anti-IA-2 beta monoclonal antibodies.
IA-2 beta exists mainly in a 60-kDa form, and is frequently located in the dense-core secretory vesicles of
pancreatic
beta cells.
In the present study, we characterized the major forms of IA-2 beta in the brain and
pancreas of
normal and non-obese diabetic (NOD) mice.
On the contrary, only the 60-kDa isoform of IA-2 beta was expressed in the mouse
pancreas
and in the mouse
pancreatic
beta cell line, MIN6.
Furthermore, Western blotting and immunohistochemistry demonstrated that NOD mice expressed similar isoforms present in the brains and
pancreatic
islets of C57BL/6J, BALC/CA and ICR mice, accordingly.
[MeSH-major]
Antibodies, Monoclonal / pharmacology. Autoantigens / analysis. Autoantigens / immunology. Autoantigens / metabolism. Brain / metabolism. Membrane Proteins / analysis. Membrane Proteins / immunology. Membrane Proteins / metabolism.
Pancreas
/ metabolism. Protein Tyrosine Phosphatases / analysis. Protein Tyrosine Phosphatases / immunology. Protein Tyrosine Phosphatases / metabolism
[MeSH-minor]
Amino Acid Sequence. Animals. Diabetes Mellitus, Type 1 / enzymology. Isoenzymes / metabolism. Mice. Mice, Inbred BALB C. Mice, Inbred C57BL. Mice, Inbred ICR. Mice, Inbred NOD. Mice, Nude. Mice, Transgenic. Molecular Sequence Data. Peptide Fragments / analysis. Protein Processing, Post-Translational. Protein Tyrosine Phosphatase, Non-Receptor Type 1. Receptor-Like Protein Tyrosine Phosphatases, Class 8.
Tumor
Cells, Cultured
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(PMID = 17611635.001).
[ISSN]
1107-3756
[Journal-full-title]
International journal of molecular medicine
[ISO-abbreviation]
Int. J. Mol. Med.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Greece
[Chemical-registry-number]
0 / Antibodies, Monoclonal; 0 / Autoantigens; 0 / Isoenzymes; 0 / Membrane Proteins; 0 / Peptide Fragments; EC 3.1.3.48 / Protein Tyrosine Phosphatase, Non-Receptor Type 1; EC 3.1.3.48 / Protein Tyrosine Phosphatases; EC 3.1.3.48 / Ptprn protein, mouse; EC 3.1.3.48 / Receptor-Like Protein Tyrosine Phosphatases, Class 8
32.
Adams AL, Siegal GP, Jhala NC:
Solid pseudopapillary tumor of the pancreas: a review of salient clinical and pathologic features.
Adv Anat Pathol
; 2008 Jan;15(1):39-45
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[Title]
Solid pseudopapillary
tumor of the pancreas
: a review of salient clinical and pathologic features.
Solid pseudopapillary
tumor
(SPT) of the
pancreas
is a rare
tumor of
uncertain histogenesis characterized, as the name suggests, by a cystic and solid pattern of growth with formation of pseudopapillae.
Accounting for only a small percentage of
pancreatic
neoplasms, SPT occurs primarily in young women, although cases in older patients and men have been reported.
The tumor
is thought to have low-grade malignant potential, as the majority of the cases are cured by simple but complete surgical resection.
Knowledge of the unique morphologic and demographic characteristics of this
neoplasm
is essential for accurate
diagnosis
.
Herein, we review the clinical and pathologic features, which can help separate SPTs from other primary
pancreatic
tumors.
[MeSH-major]
Carcinoma, Papillary / pathology.
Pancreatic
Neoplasms / pathology
[MeSH-minor]
Diagnosis
, Differential. Humans. Immunophenotyping.
Pancreatic
Cyst /
diagnosis
.
Pancreatic
Cyst / immunology.
Pancreatic
Cyst / pathology. Prognosis
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(PMID = 18156811.001).
[ISSN]
1072-4109
[Journal-full-title]
Advances in anatomic pathology
[ISO-abbreviation]
Adv Anat Pathol
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Number-of-references]
51
33.
Kung JW, Brown A, Kruskal JB, Goldsmith JD, Pedrosa I:
Heterotopic pancreas: typical and atypical imaging findings.
Clin Radiol
; 2010 May;65(5):403-7
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[Title]
Heterotopic
pancreas
: typical and atypical imaging findings.
Heterotopic
pancreas
is a common condition often encountered during laporotomy or autopsy.
Prospective radiographic
diagnosis
is challenging because of the variable imaging appearances.
The purpose of this review is to present the typical and atypical appearances of heterotopic
pancreas
on imaging studies.
[MeSH-major]
Choristoma /
diagnosis
. Gastrointestinal Diseases /
diagnosis
.
Pancreas
[MeSH-minor]
Adolescent. Adult. Biomarkers / blood.
Diagnosis
, Differential. Endosonography. Female. Humans. Magnetic Resonance Imaging. Male. Middle Aged. Tomography, X-Ray Computed
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(PMID = 20380941.001).
[ISSN]
1365-229X
[Journal-full-title]
Clinical radiology
[ISO-abbreviation]
Clin Radiol
[Language]
eng
[Publication-type]
Case Reports; Journal Article; Review
[Publication-country]
England
[Chemical-registry-number]
0 / Biomarkers
[Number-of-references]
25
34.
Dubova EA, Shchegolev AI:
[Clinical and morphological characteristics of intraductal papillary mucinous tumors of the pancreas].
Arkh Patol
; 2009 Mar-Apr;71(2):9-12
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[Title]
[Clinical and morphological characteristics of intraductal papillary mucinous tumors of the
pancreas
].
The paper presents the results of a comprehensive morphological study of the surgical material from 5 patients (males aged 49 to 69 years) with intraductal papillary mucinous tumors (IDPMT) of the
pancreas
.
The pancreatobiliary type of IDPMT was established in 4 cases (one adenoma from the peripheral branches of the
pancreatic
duct, one IDPMT with the borderline malignancy potential from the major
pancreatic
duct, and two intraductal papillary mucinous carcinomas from the major
pancreatic
duct).
One patient was diagnosed as having an enteric type of a
tumor
(IDPMT with the borderline malignancy from the major
pancreatic
duct), which was characterized by the expression in the MUC2 and MUC5AC cells and by that of cytokeratin 20.
[MeSH-major]
Adenocarcinoma, Mucinous / metabolism. Adenocarcinoma, Mucinous / pathology. Adenoma / metabolism. Adenoma / pathology. Carcinoma,
Pancreatic
Ductal / metabolism. Carcinoma,
Pancreatic
Ductal / pathology
[MeSH-minor]
Aged. Gene Expression Regulation, Neoplastic. Humans. Keratins / biosynthesis. Male. Middle Aged. Mucin 5AC / biosynthesis. Mucin-1 / biosynthesis.
Neoplasm
Proteins / biosynthesis
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(PMID = 19507570.001).
[ISSN]
0004-1955
[Journal-full-title]
Arkhiv patologii
[ISO-abbreviation]
Arkh. Patol.
[Language]
rus
[Publication-type]
English Abstract; Journal Article
[Publication-country]
Russia (Federation)
[Chemical-registry-number]
0 / MUC1 protein, human; 0 / MUC5AC protein, human; 0 / Mucin 5AC; 0 / Mucin-1; 0 / Neoplasm Proteins; 68238-35-7 / Keratins
35.
Xu J, Liang Z, Hao S, Zhu L, Ashish M, Jin C, Fu D, Ni Q:
Pancreatic adenocarcinoma: dynamic 64-slice helical CT with perfusion imaging.
Abdom Imaging
; 2009 Nov;34(6):759-66
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[Title]
Pancreatic
adenocarcinoma: dynamic 64-slice helical CT with perfusion imaging.
Thus, in lesions of the tissues of the
pancreas
, this offers to increase the accuracy of CT
diagnosis
.
In this study, our aim was to explore the perfusion characteristics of normal
pancreas
and
pancreatic
adenocarcinoma.
METHODS: Dynamic 64-slice helical CT was conducted in 36 patients with non-
pancreatic
disease and in 40 patients with histopathologically proven
pancreatic
adenocarcinoma.
RESULTS: There was no significant difference noted between the distribution of BF, BV, and PS values in different regions of the
pancreas
, namely the head, neck, body, and tail (P > 0.05).
The BF, BV, and PS of normal
pancreas
were recorded as 135.24 +/- 48.36 ml min(-1) 100 g(-1), 200.55 +/- 54.96 ml 100 g(-1), and 49.75 +/- 24.27 ml min(-1) 100 g(-1), respectively.
BF, BV, and PS values of the
tumor
tissue of
pancreatic
adenocarcinoma decreased significantly compared to normal
pancreas
(P < 0.05).
CONCLUSIONS: Normal
pancreas
appears homogenous on perfusion CT.
A significant decrease of BF, BV, and PS was observed in
pancreatic
adenocarcinoma.
Dynamic 64-slice helical CT with perfusion imaging should be considered a potential modality to increase the accuracy of CT
diagnosis
for
pancreatic
adenocarcinoma.
[MeSH-major]
Adenocarcinoma / radiography.
Pancreatic
Neoplasms / radiography. Tomography, Spiral Computed / methods
[MeSH-minor]
Case-Control Studies. Contrast Media. Female. Humans. Iohexol. Male. Middle Aged.
Pancreas
/ blood supply.
Pancreas
/ radiography. Pancreaticoduodenectomy. Prospective Studies
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(PMID = 19672566.001).
[ISSN]
1432-0509
[Journal-full-title]
Abdominal imaging
[ISO-abbreviation]
Abdom Imaging
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Contrast Media; 4419T9MX03 / Iohexol
36.
Puglisi MA, Giuliani L, Fierabracci A:
Identification and characterization of a novel expandable adult stem/progenitor cell population in the human exocrine pancreas.
J Endocrinol Invest
; 2008 Jun;31(6):563-72
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[Title]
Identification and characterization of a novel expandable adult stem/progenitor cell population in the human exocrine
pancreas
.
In particular, the existence of
pancreatic
stem cells remains elusive because specific markers for their identification are not available.
We established a method for the isolation of a population of stem/progenitor cells from the human exocrine
pancreas
, and propose it as a model for other human compact organs.
We identified a novel predominant functional type of stem/progenitor cell within the human exocrine
pancreas
, able to generate insulin-producing cells and potentially non-
pancreatic
cells.
[MeSH-major]
Cell Proliferation.
Pancreas
, Exocrine / cytology. Stem Cells / cytology
[MeSH-minor]
Adult. Cell Differentiation / physiology. Cell Line,
Tumor
. Cells, Cultured. Coculture Techniques / methods. Humans
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[ISSN]
1720-8386
[Journal-full-title]
Journal of endocrinological investigation
[ISO-abbreviation]
J. Endocrinol. Invest.
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Italy
37.
Tajima Y, Kuroki T, Tsutsumi R, Fukuda K, Kitasato A, Adachi T, Mishima T, Kanematsu T:
Risk factors for pancreatic anastomotic leakage: the significance of preoperative dynamic magnetic resonance imaging of the pancreas as a predictor of leakage.
J Am Coll Surg
; 2006 May;202(5):723-31
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[Title]
Risk factors for
pancreatic
anastomotic leakage: the significance of preoperative dynamic magnetic resonance imaging of the
pancreas as
a predictor of leakage.
BACKGROUND: The histologic degree of
pancreatic
fibrosis can be assessed preoperatively by using the time-signal intensity curve (TIC) of the
pancreas
obtained from dynamic magnetic resonance imaging.
STUDY DESIGN: To identify risk factors for postoperative
pancreatic
anastomotic leakage and to assess the impact of
pancreatic
TIC on this complication, 89 patients who underwent a
pancreatic
head resection with an end-to-side pancreaticojejunostomy between December 1998 and August 2005 were retrospectively reviewed.
The
pancreatic
TIC profiles were classified into 3 types: type I, indicating a normal
pancreas
without fibrosis; and types II and III indicating fibrotic
pancreas
.
In a univariate analysis,
pancreatic
texture (hard, 3% versus intermediate, 20% versus soft, 23%; p = 0.046),
pancreatic
duct size (> 3 mm, 8% versus <or= 3 mm, 25%; p = 0.037), and
pancreatic
TIC (types II, III, 3% versus type I, 25%; p = 0.006) were notably associated with
pancreatic
anastomotic leakage.
In a multivariable analysis,
pancreatic
TIC (odds ratio [OR], 9.58; 95% CI, 1.1 to 91.7) was the only marked independent predictor of postoperative
pancreatic
leakage.
A subanalysis of 52 patients with type I
pancreatic
TIC demonstrated hemoglobin A1c (odds ratio, 9.81; 95% CI, 1.2 to 127.9) to be a notable predictor of leakage and
pancreatic
leakage developed in diabetic patients with a high hemoglobin A1c concentration (> 6.0%) than in those with a normal hemoglobin A1c level.
CONCLUSIONS:
Pancreatic
TIC from dynamic MRI provides reliable information for predicting risk of
pancreatic
anastomotic leakage after
pancreatic
head resection.
Especially in patients with type I
pancreatic
TIC, the presence of uncontrolled diabetes is considered a primary risk factor for postoperative
pancreatic
leakage.
[MeSH-major]
Anastomosis, Surgical / adverse effects. Magnetic Resonance Imaging.
Pancreatic
Diseases / surgery. Pancreaticojejunostomy. Postoperative Complications /
diagnosis
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(PMID = 16648011.001).
[ISSN]
1072-7515
[Journal-full-title]
Journal of the American College of Surgeons
[ISO-abbreviation]
J. Am. Coll. Surg.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Contrast Media
38.
Hoffman BG, Zavaglia B, Witzsche J, Ruiz de Algara T, Beach M, Hoodless PA, Jones SJ, Marra MA, Helgason CD:
Identification of transcripts with enriched expression in the developing and adult pancreas.
Genome Biol
; 2008;9(6):R99
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[Title]
Identification of transcripts with enriched expression in the developing and adult
pancreas
.
BACKGROUND: Despite recent advances, the transcriptional hierarchy driving
pancreas
organogenesis remains largely unknown, in part due to the paucity of comprehensive analyses.
To address this deficit we generated ten SAGE libraries from the developing murine
pancreas
spanning Theiler stages 17-26, making use of available Pdx1 enhanced green fluorescent protein (EGFP) and Neurog3 EGFP reporter strains, as well as tissue from adult islets and ducts.
RESULTS: We used a specificity metric to identify 2,536 tags with
pancreas
-enriched expression compared to 195 other mouse SAGE libraries.
We subsequently grouped co-expressed transcripts with differential expression during
pancreas
development using K-means clustering.
We validated the clusters first using quantitative real time PCR and then by analyzing the Theiler stage 22
pancreas
in situ hybridization staining patterns of over 600 of the identified genes using the GenePaint database.
These were then categorized into one of the five expression domains within the developing
pancreas
.
Based on these results we identified a cascade of transcriptional regulators expressed in the endocrine
pancreas
lineage and, from this, we developed a predictive regulatory network describing beta-cell development.
CONCLUSION: Taken together, this work provides evidence that the SAGE libraries generated here are a valuable resource for continuing to elucidate the molecular mechanisms regulating
pancreas
development.
Furthermore, our studies provide a comprehensive analysis
of pancreas
development, and insights into the regulatory networks driving this process are revealed.
[MeSH-major]
Gene Expression Profiling.
Pancreas
/ embryology
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]
[Cites]
Nucleic Acids Res. 2006 Jan 1;34(Database issue):D68-73
[
16381958.001
]
[Cites]
Endocrinology. 2006 Apr;147(4):1950-8
[
16373422.001
]
[Cites]
BMC Bioinformatics. 2006;7:116
[
16524483.001
]
[Cites]
Development. 2006 May;133(10):1955-66
[
16651540.001
]
[Cites]
Genomics. 2006 Aug;88(2):133-42
[
16698233.001
]
[Cites]
Genome Res. 2007 Jan;17(1):108-16
[
17135571.001
]
[Cites]
Genome Biol. 2007;8(1):R6
[
17210078.001
]
(PMID = 18554416.001).
[ISSN]
1474-760X
[Journal-full-title]
Genome biology
[ISO-abbreviation]
Genome Biol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / enhanced green fluorescent protein; 147336-22-9 / Green Fluorescent Proteins
[Other-IDs]
NLM/ PMC2481431
39.
Saif MW, Oettle H, Vervenne WL, Thomas JP, Spitzer G, Visseren-Grul C, Enas N, Richards DA:
Randomized double-blind phase II trial comparing gemcitabine plus LY293111 versus gemcitabine plus placebo in advanced adenocarcinoma of the pancreas.
Cancer J
; 2009 Jul-Aug;15(4):339-43
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[Title]
Randomized double-blind phase II trial comparing gemcitabine plus LY293111 versus gemcitabine plus placebo in advanced adenocarcinoma of the
pancreas
.
BACKGROUND: LY293111 (LY) is a novel oral anticancer agent with leukotriene B4 receptor antagonist and peroxisome proliferator-activated receptor gamma agonist properties, producing promising results alone and in combination with gemcitabine in
pancreatic
cancer xenograft models.
PATIENTS AND METHODS: Chemotherapy-naive patients with histologically confirmed locally advanced or metastatic adenocarcinoma of the
pancreas
were randomly assigned to gemcitabine 1000 mg/m on days 1, 8, and 15 of a 28-day cycle and continuously administered LY 600 mg twice daily or gemcitabine 1000 mg/m on days 1, 8, and 15 of a 28-day cycle and daily oral placebo.
CONCLUSIONS: These results do not demonstrate any benefit to adding LY to gemcitabine in unpretreated patients with advanced
pancreatic
carcinoma.
[MeSH-major]
Adenocarcinoma / drug therapy. Antimetabolites, Antineoplastic / therapeutic use. Benzoates / therapeutic use. Deoxycytidine / analogs & derivatives.
Pancreatic
Neoplasms / drug therapy
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consumer health - Pancreatic Cancer
.
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(PMID = 19672152.001).
[ISSN]
1528-9117
[Journal-full-title]
Cancer journal (Sudbury, Mass.)
[ISO-abbreviation]
Cancer J
[Language]
eng
[Publication-type]
Clinical Trial, Phase II; Journal Article; Randomized Controlled Trial; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Antimetabolites, Antineoplastic; 0 / Benzoates; 0 / LY 293111; 0 / Receptors, Leukotriene B4; 0W860991D6 / Deoxycytidine; B76N6SBZ8R / gemcitabine
40.
Kayacetin E, Yol S, Kayacetin S:
Giant aneurysm of the splenic artery adherent to the pancreas with splenic infarct: report of a case.
Acta Chir Belg
; 2006 May-Jun;106(3):348-50
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[Title]
Giant aneurysm of the splenic artery adherent to
the pancreas
with splenic infarct: report of a case.
We present a case of a 9 cm giant splenic artery aneurysm tightly adherent to
the pancreas
which was treated surgically.
[MeSH-major]
Aneurysm /
diagnosis
. Splenic Artery / surgery. Splenic Infarction /
diagnosis
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consumer health - Aneurysms
.
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(PMID = 16910011.001).
[ISSN]
0001-5458
[Journal-full-title]
Acta chirurgica Belgica
[ISO-abbreviation]
Acta Chir. Belg.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Belgium
41.
Patel VG, Eltayeb OM, Zakaria M, Fortson JK, Weaver WL:
Spontaneous subcapsular splenic hematoma: a rare complication of pancreatitis.
Am Surg
; 2005 Dec;71(12):1066-9
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Subcapsular hematoma of the spleen is a rare complication of pancreatitis despite its close proximity to
the pancreas
.
Pancreatic
pseudocyst involving the tail of the
pancreas
may erode into the splenic hilum causing hilar vessel bleeding with subcapsular dissection and hematoma formation.
[MeSH-major]
Gastrointestinal Hemorrhage /
diagnosis
. Hematoma /
diagnosis
.
Pancreatic
Pseudocyst /
diagnosis
. Pancreatitis /
diagnosis
. Splenic Rupture /
diagnosis
[MeSH-minor]
Abdominal Pain /
diagnosis
. Abdominal Pain / etiology. Adult. Follow-Up Studies. Hemodynamics / physiology. Humans. Magnetic Resonance Imaging. Male. Remission, Spontaneous. Risk Assessment. Tomography, X-Ray Computed. Ultrasonography, Doppler
Genetic Alliance.
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.
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consumer health - Gastrointestinal Bleeding
.
MedlinePlus Health Information.
consumer health - Pancreatitis
.
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(PMID = 16447482.001).
[ISSN]
0003-1348
[Journal-full-title]
The American surgeon
[ISO-abbreviation]
Am Surg
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
United States
42.
Meuth-Metzinger VL, Philouze-Rome V, Metzinger L, Gespach C, Guilloteau P:
Differential activation of adenylate cyclase by secretin and VIP receptors in the calf pancreas.
Pancreas
; 2005 Aug;31(2):174-81
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[Title]
Differential activation of adenylate cyclase by secretin and VIP receptors in the calf
pancreas
.
OBJECTIVE: Secretin is a key regulator of
pancreatic
secretion, but the molecular basis of its action is not well understood, especially in the calf
pancreas
.
Our study investigated the expression and functional competence of secretin receptors (SEC-R) in calf
pancreatic
membranes.
RESULTS: We successfully amplified, by reverse transcriptase-polymerase chain reaction, a fragment of the SEC-R gene from 119-day-old calf
pancreas
.
Accordingly, secretin stimulates AC activity in calf
pancreatic
membranes isolated from 28- and 119-day-old animals with a potency (Ka) of 1.9 to 2.7 nmol/L.
CONCLUSION: Our data indicate that secretin exerts a direct action on
pancreatic
secretion through specific SEC-R coupled to the AC system.
Calf
pancreatic
SEC-Rs are coexpressed with VIP-2 receptors that we previously identified by ligand binding and cross-linking experiments.
[MeSH-major]
Adenylyl Cyclases / metabolism.
Pancreas
/ metabolism. Receptors, G-Protein-Coupled / genetics. Receptors, Gastrointestinal Hormone / genetics. Receptors, Vasoactive Intestinal Peptide, Type II / metabolism. Receptors, Vasoactive Intestinal Polypeptide, Type I / metabolism. Secretin / metabolism
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(PMID = 16025005.001).
[ISSN]
1536-4828
[Journal-full-title]
Pancreas
[ISO-abbreviation]
Pancreas
[Language]
eng
[Databank-accession-numbers]
GENBANK/ AF118556
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Pituitary Adenylate Cyclase-Activating Polypeptide; 0 / Receptors, G-Protein-Coupled; 0 / Receptors, Gastrointestinal Hormone; 0 / Receptors, Vasoactive Intestinal Peptide, Type II; 0 / Receptors, Vasoactive Intestinal Polypeptide, Type I; 0 / secretin receptor; 1393-25-5 / Secretin; 37221-79-7 / Vasoactive Intestinal Peptide; EC 4.6.1.1 / Adenylyl Cyclases
43.
Newman SJ, Steiner JM, Woosley K, Williams DA, Barton L:
Histologic assessment and grading of the exocrine pancreas in the dog.
J Vet Diagn Invest
; 2006 Jan;18(1):115-8
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[Title]
Histologic assessment and grading of the exocrine
pancreas
in the dog.
Histologic grading schemes for canine inflammatory conditions are sparse, and in the case of the canine
pancreas
, have not been previously described.
In a previous study, we determined that histologic lesions of the exocrine
pancreas
occurred much more frequently than gross lesions.
The intention of the current study was to develop a histologic grading scheme for nonneoplastic lesions following extensive assessment of the exocrine
pancreas
from dogs presented for necropsy examination.
The parameters of the proposed scheme include neutrophilic inflammation, lymphocytic inflammation,
pancreatic
necrosis,
pancreatic
fat necrosis, edema, fibrosis, atrophy, and hyperplastic nodules.
In this case series, the most common lesion was
pancreatic
hyperplastic nodules (80.2%), followed by lymphocytic inflammation (52.5%), fibrosis (49.5%), atrophy (46.5%), neutrophilic inflammation (31.7%),
pancreatic
fat necrosis (25.7%),
pancreatic
necrosis (16.8%), and edema (9.9%).
Neutrophilic inflammation, when present, was often associated with necrosis (
pancreatic
necrosis,
pancreatic
fat necrosis, or both) and occasionally with hyperplastic nodules.
The utilization of a grading scheme for exocrine
pancreatic
lesions will be useful in advancing the classification of exocrine
pancreatic
disease in the dog, which may lead to multicenter studies of exocrine
pancreatic
disorders in the dog and in other species.
[MeSH-major]
Dog Diseases / pathology.
Pancreas
, Exocrine / pathology. Pancreatitis / veterinary
MedlinePlus Health Information.
consumer health - Pancreatitis
.
COS Scholar Universe.
author profiles
.
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(PMID = 16566269.001).
[ISSN]
1040-6387
[Journal-full-title]
Journal of veterinary diagnostic investigation : official publication of the American Association of Veterinary Laboratory Diagnosticians, Inc
[ISO-abbreviation]
J. Vet. Diagn. Invest.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
44.
Bindi ML, Biancofiore G, Meacci L, Bellissima G, Nardi S, Pieri M, Vistoli F, Boggi U, Sansevero A, Mosca F:
Early morbidity after pancreas transplantation.
Transpl Int
; 2005 Dec;18(12):1356-60
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[Title]
Early morbidity after
pancreas
transplantation.
This study aims to evaluate and compare the early outcome of both
pancreas
-alone transplantation (PTA) and simultaneous kidney-
pancreas
transplantation (SPKT) focusing on the complications affecting the first month after the procedures.
[MeSH-major]
Kidney Transplantation / adverse effects. Kidney Transplantation / methods.
Pancreas
Transplantation / adverse effects.
Pancreas
Transplantation / methods
Genetic Alliance.
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.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Pancreas Transplantation
.
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(PMID = 16297054.001).
[ISSN]
0934-0874
[Journal-full-title]
Transplant international : official journal of the European Society for Organ Transplantation
[ISO-abbreviation]
Transpl. Int.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Germany
[Chemical-registry-number]
0 / Blood Glucose; 0 / Immunosuppressive Agents; 0 / Insulin
45.
Pekary AE, Stevens SA, Sattin A:
Valproate and copper accelerate TRH-like peptide synthesis in male rat pancreas and reproductive tissues.
Peptides
; 2006 Nov;27(11):2901-11
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[Title]
Valproate and copper accelerate TRH-like peptide synthesis in male rat
pancreas
and reproductive tissues.
Because high levels of TRH-like peptides occur in
the pancreas
and pGlu-Glu-Pro-NH(2) (Glu-TRH) has been shown to be a fertilization promoting peptide, we hypothesized that these peptides mediate some of the metabolic and reproductive side effects of Valp.
AC, CHR and WD treatments significantly altered TRH and/or TRH-like peptide levels in
pancreas
and reproductive tissues.
Phe-TRH, the most abundant TRH-like peptide in
the pancreas
, increased 4-fold with AC Valp.
Copper (500 microM) increased the in vitro C-terminal amidation of TRH-like peptides by 8- and 4-fold during 24 degrees C incubation of homogenates
of pancreas
and testis, respectively.
Valp (7 microM) accelerated 3-fold the processing of TRH and TRH-like peptide precursors in
pancreatic
LDCV's incubated at 24 degrees C.
[MeSH-major]
Copper / pharmacology. Genitalia, Male / drug effects.
Pancreas
/ drug effects. Peptides / metabolism. Thyrotropin-Releasing Hormone / metabolism. Valproic Acid / pharmacology
Hazardous Substances Data Bank.
COPPER, ELEMENTAL
.
Hazardous Substances Data Bank.
VALPROIC ACID
.
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(PMID = 16945452.001).
[ISSN]
0196-9781
[Journal-full-title]
Peptides
[ISO-abbreviation]
Peptides
[Language]
eng
[Publication-type]
Journal Article; Research Support, U.S. Gov't, Non-P.H.S.
[Publication-country]
United States
[Chemical-registry-number]
0 / Blood Glucose; 0 / Peptides; 5Y5F15120W / Thyrotropin-Releasing Hormone; 614OI1Z5WI / Valproic Acid; 789U1901C5 / Copper
46.
Ablorsu E, Ghazanfar A, Mehra S, Campbell B, Riad H, Pararajasingam R, Parrott N, Picton M, Augustine T, Tavakoli A:
Outcome of pancreas transplantation in recipients older than 50 years: a single-centre experience.
Transplantation
; 2008 Dec 15;86(11):1511-4
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[Title]
Outcome
of pancreas
transplantation in recipients older than 50 years: a single-centre experience.
BACKGROUND:
Pancreas
transplantation (PT) remains the only treatment that can restore insulin independence among insulin-dependent diabetics.
So, as the incidence of other surgical complication in the more than or equal to 50 group compared with less than 50 (graft thrombosis 13% vs. 11.5%; bleeding 19% vs. 6.7%; abdominal abscess 23% vs. 19%;
pancreatic
leak 13% vs. 9.6%).
One-year patient survival was 88% in more than or equal to 50 vs. 92% in less than 50 group, P=0.399; and
pancreas
graft survival rate was similar (79% in the >or=50 and 74% in <50, P=0.399).
[MeSH-major]
Pancreas
Transplantation / methods
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.
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consumer health - Pancreas Transplantation
.
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(PMID = 19077882.001).
[ISSN]
1534-6080
[Journal-full-title]
Transplantation
[ISO-abbreviation]
Transplantation
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
47.
Dixit R, Sharma S, Dave L:
Massive haemothorax in asymptomatic pseudocyst pancreas.
Lung India
; 2008 Jul;25(3):126-8
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[Title]
Massive haemothorax in asymptomatic pseudocyst
pancreas
.
The case of a 35-year old man who presented with massive left sided haemothorax as a complication of an asymtomatic
pancreatic
pseudocyst is descibed.
The diagnosis
was confirmed by very high amylase content of the pleural fluid.
The complications of pancreatitis and
pancreatic
pseudocyst are also briefly discussed.
Haemothorax represents an unusual pulmonary complication of pseudocyst
pancreas
and should be considered in the differential
diagnosis of
pleural fluid collection in pancreatitis.
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[Cites]
Thorax. 1979 Feb;34(1):106-12
[
441988.001
]
[Cites]
Thorax. 1989 Oct;44(10):824-5
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2595625.001
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Am J Surg. 1972 Nov;124(5):600-6
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5079792.001
]
(PMID = 20165665.001).
[ISSN]
0974-598X
[Journal-full-title]
Lung India : official organ of Indian Chest Society
[ISO-abbreviation]
Lung India
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
India
[Other-IDs]
NLM/ PMC2822336
[Keywords]
NOTNLM ; Haemothorax / Pseudocyst pancreas
48.
Fridell JA, Rogers J, Stratta RJ:
The pancreas allograft donor: current status, controversies, and challenges for the future.
Clin Transplant
; 2010 Jul-Aug;24(4):433-49
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[Title]
The pancreas
allograft donor: current status, controversies, and challenges for the future.
The pancreas
allograft is a scarce resource that is currently underutilized.
The selection of appropriate deceased donors for
pancreas
procurement is of paramount importance for minimizing technical failure and optimizing long-term outcomes in
pancreas
transplantation.
Despite the increasing demand for
pancreas
transplantation, increases in overall organ donation rates and the evolution of criteria that constitute an "acceptable"
pancreas
donor, the number of deceased donor
pancreas
transplants being performed in the United States has actually declined in recent years.
Although there are many factors that must be considered during evaluation of the potential
pancreas
allograft donor to minimize morbidity and graft loss, it is evident that there are transplantable organs that are not used.
In this review, deceased donor
pancreas
identification, management, selection, allocation, assessment, preservation, and the problem
of pancreas
underutilization will be discussed.
[MeSH-major]
Pancreas
Transplantation / trends. Tissue Donors
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.
MedlinePlus Health Information.
consumer health - Pancreas Transplantation
.
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[Copyright]
© 2010 John Wiley & Sons A/S.
(PMID = 20384731.001).
[ISSN]
1399-0012
[Journal-full-title]
Clinical transplantation
[ISO-abbreviation]
Clin Transplant
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
Denmark
49.
Bradley SP, Kowalik TF, Rastellini C, da Costa MA, Bloomenthal AB, Cicalese L, Basadonna GP, Uknis ME:
Successful incorporation of short-interfering RNA into islet cells by in situ perfusion.
Transplant Proc
; 2005 Jan-Feb;37(1):233-6
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We describe the first successful introduction of siRNA directly into
pancreatic
islet cells both during in situ perfusion and from intravenous tail vein injection (in vivo).
METHODS: siRNA was targeted to the
pancreatic
islets of BALB/c mice by retrograde portal vein perfusion or tail vein injection.
After delivery
pancreata
were placed in cold storage at 4 degrees C in UW solution for 24 hours, followed by processing for immunofluorescent staining for insulin.
RESULTS: In situ delivery of siRNA was demonstrated by fluorescent imaging composites of (red) siRNA in and along (green) insulin stained islets from
pancreas
sections as compared with untreated control sections.
The siRNA was detected mainly in and along venous structures throughout the
pancreatic
tissue.
CONCLUSIONS: We have described the successful delivery of siRNA to
pancreatic
islets via a novel in situ
pancreas
perfusion technique and in vivo delivery via tail vein injection.
MedlinePlus Health Information.
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.
Hazardous Substances Data Bank.
Allopurinol
.
Hazardous Substances Data Bank.
Adenosine
.
The Lens.
Cited by Patents in
.
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(PMID = 15808605.001).
[ISSN]
0041-1345
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Insulin; 0 / Organ Preservation Solutions; 0 / RNA, Small Interfering; 0 / University of Wisconsin-lactobionate solution; 63CZ7GJN5I / Allopurinol; GAN16C9B8O / Glutathione; K72T3FS567 / Adenosine; N5O3QU595M / Raffinose
50.
Dalla Valle R, Capocasale E, Mazzoni MP, Busi N, Piazza P, Benozzi L, Sianesi M:
Embolization of a ruptured pseudoaneurysm with massive hemorrhage following pancreas transplantation: a case report.
Transplant Proc
; 2005 Jun;37(5):2275-7
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[Title]
Embolization of a ruptured pseudoaneurysm with massive hemorrhage following
pancreas
transplantation: a case report.
We report a case of an arterioduodenal fistula related to a ruptured pseudoaneurysm after simultaneous
pancreas
-kidney transplantation (SPK) with massive gastrointestinal hemorrhage treated by embolization of the Y graft.
No rejection episodes were documented; the patient was discharged with normal
pancreatic
and renal function.
Few reports exist in the literature regarding the development of a pseudoaneurysm after
pancreas
transplantation.
[MeSH-major]
Aneurysm, False / etiology. Aneurysm, Ruptured / etiology. Diabetes Mellitus, Type 1 / surgery. Embolization, Therapeutic. Gastrointestinal Hemorrhage / etiology.
Pancreas
Transplantation / adverse effects
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Diabetes Type 1
.
MedlinePlus Health Information.
consumer health - Gastrointestinal Bleeding
.
MedlinePlus Health Information.
consumer health - Pancreas Transplantation
.
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(PMID = 15964398.001).
[ISSN]
0041-1345
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Case Reports; Journal Article; Review
[Publication-country]
United States
[Number-of-references]
11
51.
Hayata T, Blitz IL, Iwata N, Cho KW:
Identification of embryonic pancreatic genes using Xenopus DNA microarrays.
Dev Dyn
; 2009 Jun;238(6):1455-66
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[Title]
Identification of embryonic
pancreatic
genes using Xenopus DNA microarrays.
The pancreas
is both an exocrine and endocrine endodermal organ involved in digestion and glucose homeostasis.
During embryogenesis, the anlagen of the
pancreas
arise from dorsal and ventral evaginations of the foregut that later fuse to form a single organ.
To better understand the molecular genetics of early
pancreas
development, we sought to isolate markers that are uniquely expressed in this tissue.
Microarray analysis was performed comparing dissected
pancreatic
buds, liver buds, and the stomach region of tadpole stage Xenopus embryos.
K-means clustering analysis predicted 120 of these genes to be specifically enriched in
the pancreas
.
Our analyses implicate the involvement of previously unsuspected signaling pathways during early
pancreas
development.
[MeSH-major]
Oligonucleotide Array Sequence Analysis.
Pancreas
. Xenopus laevis
Xenbase.
Xenbase
.
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(PMID = 19191222.001).
[ISSN]
1058-8388
[Journal-full-title]
Developmental dynamics : an official publication of the American Association of Anatomists
[ISO-abbreviation]
Dev. Dyn.
[Language]
eng
[Grant]
United States / NICHD NIH HHS / HD / R01 HD029507; United States / NICHD NIH HHS / HD / R01 HD056219
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
United States
[Chemical-registry-number]
0 / Biomarkers; 0 / Frizzled Receptors; 0 / Hedgehog Proteins; 0 / Immunoglobulin J Recombination Signal Sequence-Binding Protein; 0 / Receptors, G-Protein-Coupled; 0 / Wnt Proteins; 0 / Xenopus Proteins; 0 / fzd10a protein, Xenopus; 62031-54-3 / Fibroblast Growth Factors
[Other-IDs]
NLM/ NIHMS283451; NLM/ PMC4203858
52.
Kato M, Kubota K, Kita J, Shimoda M, Rokkaku K, Inaba N, Fukasawa I, Honma K:
Huge mucinous cystadenoma of the pancreas developing during pregnancy: a case report.
Pancreas
; 2005 Mar;30(2):186-8
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[Title]
Huge mucinous cystadenoma of the
pancreas
developing during pregnancy: a case report.
A tentative
diagnosis of
ovarian or
pancreatic
mucinous cystadenoma was made.
The cyst was found to originate from
the pancreas
, and distal pancreatectomy with splenectomy was performed.
Histological
diagnosis
was a
benign
mucinous cystadenoma.
Up to the present, there have been three reported cases of
pancreatic
mucinous cystadenoma, including our case, and two cases of
pancreatic
mucinous cystadenocarcinoma, in association with pregnancy.
Our case is the third reported of successful resection of the
tumor
during pregnancy resulting in a healthy infant.
[MeSH-major]
Cystadenoma, Mucinous / pathology.
Pancreatic
Neoplasms / pathology. Pregnancy Complications, Neoplastic
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(PMID = 15714143.001).
[ISSN]
1536-4828
[Journal-full-title]
Pancreas
[ISO-abbreviation]
Pancreas
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
United States
53.
Pogue BW, Samkoe KS, Hextrum S, O'Hara JA, Jermyn M, Srinivasan S, Hasan T:
Imaging targeted-agent binding in vivo with two probes.
J Biomed Opt
; 2010 May-Jun;15(3):030513
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Images of a binding rate constant are created that reveal lower than expected uptake of epidermal growth factor in an orthotopic xenograft
pancreas tumor
(2.3 x 10(-5) s(-1)), as compared to the normal
pancreas
(3.4 x 10(-5) s(-1)).
This approach allows noninvasive assessment
of tumor
receptor targeting in vivo to determine the expected contrast, spatial localization, and efficacy in therapeutic agent delivery.
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[Cites]
Am J Surg. 2003 Nov;186(5):431-6
[
14599602.001
]
[Cites]
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]
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Cancer Biomark. 2005;1(2-3):157-75
[
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]
(PMID = 20614996.001).
[ISSN]
1560-2281
[Journal-full-title]
Journal of biomedical optics
[ISO-abbreviation]
J Biomed Opt
[Language]
ENG
[Grant]
United States / NCI NIH HHS / CA / P01 CA084203; United States / NCI NIH HHS / CA / R01 CA109558; United States / NCI NIH HHS / CA / P01CA84203; United States / NCI NIH HHS / CA / R01CA109558
[Publication-type]
Letter; Research Support, N.I.H., Extramural
[Publication-country]
United States
[Chemical-registry-number]
0 / Fluorescent Dyes; 62229-50-9 / Epidermal Growth Factor
[Other-IDs]
NLM/ PMC2909298
54.
Novellas S, Chevallier P, Saint Paul MC, Gugenheim J, Bruneton JN:
MRI features of a pancreatic schwannoma.
Clin Imaging
; 2005 Nov-Dec;29(6):434-6
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[Title]
MRI features of a
pancreatic
schwannoma.
A case of a
pancreatic
schwannoma is presented.
The patient, a previously healthy woman, is hospitalized with
the diagnosis
of purulent pleuritis.
Ultrasonography (US) of the abdomen shows a 3-cm mass in the head of the
pancreas
.
A duodenopancreatotomy is performed, and the pathologic specimen demonstrates a schwannoma of the
pancreas
with Antoni A pattern.
[MeSH-major]
Neurilemmoma /
diagnosis
.
Pancreatic
Neoplasms /
diagnosis
[MeSH-minor]
Contrast Media.
Diagnosis
, Differential. Female. Gadolinium DTPA. Humans. Magnetic Resonance Imaging. Middle Aged
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(PMID = 16274899.001).
[ISSN]
0899-7071
[Journal-full-title]
Clinical imaging
[ISO-abbreviation]
Clin Imaging
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Contrast Media; K2I13DR72L / Gadolinium DTPA
55.
Ibis C, Albayrak D, Altan A:
Primary hydatid disease of pancreas mimicking cystic neoplasm.
South Med J
; 2009 May;102(5):529-30
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[Title]
Primary hydatid disease
of pancreas
mimicking cystic
neoplasm
.
Primary hydatid disease of the
pancreas
is very rare.
We report a 33-year-old female who was admitted to the hospital with abdominal discomfort due to the
pancreatic
mass.
A
diagnosis of
a
pancreatic
cystic mass was established through abdominal ultrasonography and computed tomography scan.
Hydatid disease as well as a cystic
neoplasm
of the
pancreas
were both thought in the differential
diagnosis
.
The histopathologic evaluation of the specimen revealed a hydatid cyst affecting the tail of the
pancreas
.
Hydatid disease should be considered in the differential
diagnosis of
all cystic masses of the
pancreas
, especially in endemic regions.
[MeSH-major]
Echinococcosis /
diagnosis
.
Pancreatic
Diseases /
diagnosis
[MeSH-minor]
Adult.
Diagnosis
, Differential. Female. Humans. Tomography, X-Ray Computed
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(PMID = 19373169.001).
[ISSN]
1541-8243
[Journal-full-title]
Southern medical journal
[ISO-abbreviation]
South. Med. J.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
United States
56.
Szmidt J, Gałazka Z, Frunze S, Grochowiecki T, Nazarewski S, Durlik M, Jakimowicz T, Wojtaszek M, Grygiel K, Paczek L:
Secondary kidney transplantation in a patient 16 years after simultaneous pancreas and kidney transplantation--a case report.
Ann Transplant
; 2006;11(1):40-2
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[Title]
Secondary kidney transplantation in a patient 16 years after simultaneous
pancreas
and kidney transplantation--a case report.
Simultaneous
pancreas
and kidney transplantation (spktx) is currently the most effective method of treatment of type 1 diabetes complicated by renal insufficiency.
[MeSH-major]
Kidney Transplantation / physiology.
Pancreas
Transplantation / physiology. Reoperation
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(PMID = 17025029.001).
[ISSN]
1425-9524
[Journal-full-title]
Annals of transplantation
[ISO-abbreviation]
Ann. Transplant.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Poland
57.
Mirarchi M, De Raffele E, Lega S, Calculli L, Vaccari S, Cola B:
[Synchronous adenocarcinoma of the sigmoid colon and multifocal intraductal papillary mucinous neoplasm of the pancreas in an elderly patient].
Chir Ital
; 2009 May-Jun;61(3):357-67
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[Title]
[Synchronous adenocarcinoma of the sigmoid colon and multifocal intraductal papillary mucinous
neoplasm
of the
pancreas
in an elderly patient].
[Transliterated title]
Adenocarcinoma
del
colon e
neoplasia
papillare intraduttale mucinosa multifocale sincrona
del pancreas
in un paziente anziano: caso clinico e revisione della letteratura.
Intraductal papillary mucinous neoplasms are a well-recognized pathologic entity of the
pancreas
that is being reported with increasing frequency.
A 78-year-old man presented with rectal bleeding which led to
the diagnosis
of a stenosing adenocarcinoma of the sigmoid colon.
No metastatic lesions were present but a 30 mm intraductal papillary mucinous
neoplasm
with mural nodules was detected in the uncinate process of the
pancreas
.
Histology showed a combined-type intraductal papillary mucinous
neoplasm
with foci of non-invasive carcinoma.
The co-existence of a potentially malignant
pancreatic
tumour
with an extra-
pancreatic
overt malignancy in elderly patients poses difficulties in the attempt to cure the patient with minimal morbidity.
In the present case we considered a staged surgical procedure with the aim of reducing the perioperative risk, since the excision of the
pancreatic neoplasm
required a pancreaticoduodenectomy in an elderly patient.
[MeSH-major]
Adenocarcinoma / pathology. Adenocarcinoma, Mucinous / pathology. Adenocarcinoma, Papillary / pathology. Carcinoma,
Pancreatic
Ductal / pathology. Neoplasms, Multiple Primary / pathology.
Pancreatic
Neoplasms / pathology. Sigmoid Neoplasms / pathology
[MeSH-minor]
Aged. Colectomy / methods. Humans. Male.
Neoplasm
Staging. Pancreaticoduodenectomy / methods. Treatment Outcome
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(PMID = 19694240.001).
[ISSN]
0009-4773
[Journal-full-title]
Chirurgia italiana
[ISO-abbreviation]
Chir Ital
[Language]
ita
[Publication-type]
Case Reports; English Abstract; Journal Article
[Publication-country]
Italy
58.
Kanno A, Satoh K, Hirota M, Hamada S, Umino J, Itoh H, Masamune A, Asakura T, Shimosegawa T:
Prediction of invasive carcinoma in branch type intraductal papillary mucinous neoplasms of the pancreas.
J Gastroenterol
; 2010 Sep;45(9):952-9
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[Title]
Prediction of invasive carcinoma in branch type intraductal papillary mucinous neoplasms of the
pancreas
.
BACKGROUND: Patients with branch duct type intraductal papillary mucinous
neoplasm
(BD-IPMN) without invasion usually show favorable prognosis.
In addition, recent studies have revealed that BD-IPMN is frequently complicated by common type
pancreatic
ductal carcinoma.
METHODS: Invasive
pancreatic
carcinoma associating with BD-IPMN was classified as invasive IPMN group (invasive carcinoma derived directly from IPMN lesions) and concomitant group (common type of invasive carcinoma concomitant with BD-IPMN).
Diameter of dilated branch (P < 0.001) or main
pancreatic
duct (MPD) (P = 0.001), size of mural nodule (P < 0.001), serum CEA level (P < 0.001) and serum CA19-9 level (P < 0.001) were factors associated significantly with invasive IPMN by univariate analysis.
CONCLUSIONS: Our results suggested that careful imaging study of the entire
pancreas
in addition to
tumor
lesions and measurement of serum CEA and CA19-9 would be required to find out the development of the two types of invasive carcinoma in BD-IPMN.
[MeSH-major]
Adenocarcinoma, Mucinous / pathology. Adenocarcinoma, Papillary / pathology. Carcinoma,
Pancreatic
Ductal / pathology.
Pancreatic
Neoplasms / pathology
[MeSH-minor]
Aged. Aged, 80 and over. CA-19-9 Antigen / blood. Carcinoembryonic Antigen / blood. Disease Progression. Female. Follow-Up Studies. Humans. Male. Middle Aged.
Neoplasm
Invasiveness. Prognosis. Retrospective Studies. Risk Factors
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[Cites]
Radiology. 2008 Sep;248(3):876-86
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18632526.001
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[ISSN]
1435-5922
[Journal-full-title]
Journal of gastroenterology
[ISO-abbreviation]
J. Gastroenterol.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Japan
[Chemical-registry-number]
0 / CA-19-9 Antigen; 0 / Carcinoembryonic Antigen
59.
Flamez D, Roland I, Berton A, Kutlu B, Dufrane D, Beckers MC, De Waele E, Rooman I, Bouwens L, Clark A, Lonneux M, Jamar JF, Goldman S, Maréchal D, Goodman N, Gianello P, Van Huffel C, Salmon I, Eizirik DL:
A genomic-based approach identifies FXYD domain containing ion transport regulator 2 (FXYD2)gammaa as a pancreatic beta cell-specific biomarker.
Diabetologia
; 2010 Jul;53(7):1372-83
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[Title]
A genomic-based approach identifies FXYD domain containing ion transport regulator 2 (FXYD2)gammaa as a
pancreatic
beta cell-specific biomarker.
AIMS/HYPOTHESIS: Non-invasive imaging of the
pancreatic
beta cell mass (BCM) requires the identification of novel and specific beta cell biomarkers.
For four of the proteins we selected or produced antibodies targeting specifically the human proteins and their splice variants; all four candidates were confirmed as islet-specific in human
pancreas
.
Two splice variants of FXYD domain containing ion transport regulator 2 (FXYD2), a regulating subunit of the Na(+)-K(+)-ATPase, were identified as preferentially present in human
pancreatic
islets.
The presence of FXYD2gammaa was restricted to
pancreatic
islets and selectively detected in
pancreatic
beta cells.
Analysis of human fetal
pancreas
samples showed the presence of FXYD2gammaa at an early stage (15 weeks).
Histological examination of
pancreatic
sections from individuals with type 1 diabetes or sections from
pancreases of
streptozotocin-treated Macacus cynomolgus monkeys indicated a close correlation between loss of FXYD2gammaa and loss of insulin-positive cells.
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[CommentIn]
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BMC Med Genomics. 2009 Jan 15;2:3
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19146692.001
]
(PMID = 20379810.001).
[ISSN]
1432-0428
[Journal-full-title]
Diabetologia
[ISO-abbreviation]
Diabetologia
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Germany
[Chemical-registry-number]
0 / Biomarkers; EC 3.6.1.- / FXYD2 protein, human; EC 3.6.3.9 / Sodium-Potassium-Exchanging ATPase
60.
Ikemoto T, Noguchi H, Shimoda M, Naziruddin B, Jackson A, Tamura Y, Fujita Y, Onaca N, Levy MF, Matsumoto S:
Islet cell transplantation for the treatment of type 1 diabetes in the USA.
J Hepatobiliary Pancreat Surg
; 2009;16(2):118-23
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ICTx requires complex procedures, including
pancreas
procurement and preservation;
pancreas
digestion; islet purification; and transplantation.
A new preservation strategy for
pancreata
and
pancreatic
ducts using ET-Kyoto solution as well as a new islet purification method using iodixanol has substantially improved islet yields.
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(PMID = 19110650.001).
[ISSN]
1436-0691
[Journal-full-title]
Journal of hepato-biliary-pancreatic surgery
[ISO-abbreviation]
J Hepatobiliary Pancreat Surg
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Japan
61.
Dotta F, Galleri L, Sebastiani G, Vendrame F:
Virus infections: lessons from pancreas histology.
Curr Diab Rep
; 2010 Oct;10(5):357-61
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[Title]
Virus infections: lessons from
pancreas
histology.
Type 1 diabetes mellitus is a chronic autoimmune disease resulting from the progressive immune-mediated destruction of
pancreatic
beta cells in genetically susceptible individuals, with the likely contribution of environmental factors, among which viruses have been extensively studied.
We review the recent literature on the viral contribution to beta-cell destruction and dysfunction in type 1 diabetes, with particular reference to the pathology of the
pancreatic
islet in humans and in animal models of the disease.
[MeSH-major]
Pancreas
/ pathology.
Pancreas
/ virology. Virus Diseases / pathology
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J Autoimmun. 2001 May;16(3):211-7
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(PMID = 20665131.001).
[ISSN]
1539-0829
[Journal-full-title]
Current diabetes reports
[ISO-abbreviation]
Curr. Diab. Rep.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
United States
62.
Meier R, Ockenga J, Pertkiewicz M, Pap A, Milinic N, Macfie J, DGEM (German Society for Nutritional Medicine), Löser C, Keim V, ESPEN (European Society for Parenteral and Enteral Nutrition):
ESPEN Guidelines on Enteral Nutrition: Pancreas.
Clin Nutr
; 2006 Apr;25(2):275-84
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[Title]
ESPEN Guidelines on Enteral Nutrition:
Pancreas
.
The two major forms of inflammatory
pancreatic
diseases, acute and chronic pancreatitis, require different approaches in nutritional management, which are presented in the present guideline.
In chronic pancreatitis more than 80% of patients can be treated adequately with normal food supplemented by
pancreatic
enzymes.
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(PMID = 16678943.001).
[ISSN]
0261-5614
[Journal-full-title]
Clinical nutrition (Edinburgh, Scotland)
[ISO-abbreviation]
Clin Nutr
[Language]
eng
[Publication-type]
Consensus Development Conference; Journal Article; Practice Guideline
[Publication-country]
England
[Number-of-references]
78
63.
Kraemer A, Lewin M, Balladur P, Mourra N, Tiret E, Paye F:
[Autoimmune pancreatitis mimicking an intra-ductal papillary mucinous neoplasm of the pancreas: an original case].
Gastroenterol Clin Biol
; 2008 Jun-Jul;32(6-7):635-9
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[Title]
[Autoimmune pancreatitis mimicking an intra-ductal papillary mucinous
neoplasm
of the
pancreas
: an original case].
[Transliterated title]
Pancréatite auto-immune mimant une
tumeur
intracanalaire papillaire et mucineuse : une observation originale et trompeuse.
It can be associated with diabetes mellitus and other systemic autoimmune diseases, or with bile ducts lesions, which are also responsive to steroid therapy as
pancreatic
lesions.
We report the case of a 34-year-old man with a history of a first acute pancreatitis, attributed to an intraductal papillary-mucinous
neoplasm
of the
pancreas
(IPMN) with segmental involvement of the main
pancreatic
duct.
To our knowledge, this is the first reported case of AIP mimicking IPMN of the main
pancreatic
duct.
[MeSH-major]
Adenocarcinoma, Mucinous /
diagnosis
. Autoimmune Diseases /
diagnosis
. Carcinoma, Papillary /
diagnosis
.
Pancreatic
Neoplasms /
diagnosis
. Pancreatitis /
diagnosis
[MeSH-minor]
Adult.
Diagnosis
, Differential. Humans. Male
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(PMID = 18355996.001).
[ISSN]
0399-8320
[Journal-full-title]
Gastroentérologie clinique et biologique
[ISO-abbreviation]
Gastroenterol. Clin. Biol.
[Language]
fre
[Publication-type]
Case Reports; English Abstract; Journal Article
[Publication-country]
France
64.
Tian JY, Li G, Gu YY, Zhang HL, Zhou WZ, Wang X, Zhu HD, Luo TH, Luo M:
Role and mechanism of rosiglitazone on the impairment of insulin secretion induced by free fatty acids on isolated rat islets.
Chin Med J (Engl)
; 2006 Apr 5;119(7):574-80
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BACKGROUND: Prolonged exposure of
pancreatic
beta-cells to fatty acids increases basal insulin secretion but inhibits glucose-stimulated insulin secretion.
METHODS:
Pancreatic
islets were isolated from
the pancreata
of male Sprague-Dawley rats by the collagenase digestion and by the dextran gradient centrifugation method.
CONCLUSION: The protective effects of rosiglitazone on insulin secretion of isolated
pancreatic
islets under chronic exposure to palmitate might be mediated through the downregulation of UCP-2 expression.
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.
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(PMID = 16620699.001).
[ISSN]
0366-6999
[Journal-full-title]
Chinese medical journal
[ISO-abbreviation]
Chin. Med. J.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
China
[Chemical-registry-number]
0 / Fatty Acids, Nonesterified; 0 / Hypoglycemic Agents; 0 / Insulin; 0 / Ion Channels; 0 / Membrane Transport Proteins; 0 / Mitochondrial Proteins; 0 / PPAR gamma; 0 / RNA, Messenger; 0 / Thiazolidinediones; 0 / mitochondrial uncoupling protein 2; 05V02F2KDG / rosiglitazone
65.
Melcher ML, Olson JL, Baxter-Lowe LA, Stock PG, Posselt AM:
Antibody-mediated rejection of a pancreas allograft.
Am J Transplant
; 2006 Feb;6(2):423-8
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[Title]
Antibody-mediated rejection of a
pancreas
allograft.
The role of antibody-mediated rejection (AMR) in
pancreas
transplantation is poorly understood.
Here, we report on a patient who developed AMR of his
pancreas
allograft after receiving a simultaneous
pancreas
-kidney transplant.
A
pancreatic
biopsy at this time showed no cellular infiltrate but strong immunofluorescent staining for C4d in the interacinar capillaries.
He received intravenous immunoglobulin (IVIg), rituximab and plasmapheresis, and his
pancreatic
function normalized.
We conclude that clinically significant AMR can develop in a
pancreas
allograft and recommend that
pancreatic
biopsies be assessed for C4d deposition if the patient has risk factors for AMR and/or the pathologic evidence for cell-mediated rejection is underwhelming.
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(PMID = 16426331.001).
[ISSN]
1600-6135
[Journal-full-title]
American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons
[ISO-abbreviation]
Am. J. Transplant.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Denmark
[Chemical-registry-number]
0 / Isoantibodies
66.
Colović R, Grubor N, Misev M, Jovanović M, Radak V:
[Fibromyxoid sarcoma of the pancreas].
Srp Arh Celok Lek
; 2008 Mar-Apr;136(3-4):158-61
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[Title]
[Fibromyxoid sarcoma of the
pancreas
].
INTRODUCTION: Fibromyxoid sarcoma is a rare mesenchymal
neoplasm
, usually appearing in the soft tissue of the extremities, less frequently in the groin, trunk, neck, and upper extremities.
Within the abdomen,
the tumour
is usually localised within the retroperitoneum.
CASE OUTLINE: We present a 56-year-old woman in whom, during the routinely performed investigation for atacks of choking with lots of bronchial secretion, and arterial hypertension, an ultrasonographer found a
tumour
within the head of the
pancreas
6 x 6 cm in diameter.
At operation, a dark pink, lobulated soft
tumour
, surrounded by a tiny capsule, clearly different from the completely normal
pancreatic
tissue of the posterior side of the head of the
pancreas
, was easily and ideally excised.
The histology of the 80 g weighing
tumour
showed a circumscribed fibromyxoid sarcoma of low malignancy.
CONCLUSION: Primary sarcomas of the
pancreas
are very rare, but should be considered in differential
diagnosis of
pancreatic
neoplasms.
To the best of our knowledge, there has been no previously described fibromyxoid sarcoma of the
pancreas
.
[MeSH-major]
Fibrosarcoma /
diagnosis
.
Pancreatic
Neoplasms /
diagnosis
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(PMID = 18720751.001).
[ISSN]
0370-8179
[Journal-full-title]
Srpski arhiv za celokupno lekarstvo
[ISO-abbreviation]
Srp Arh Celok Lek
[Language]
srp
[Publication-type]
Case Reports; English Abstract; Journal Article
[Publication-country]
Serbia
67.
Kongkam P, Wagner DL, Sherman S, Fogel EL, Whittaker SC, Watkins JL, McHenry L, Lehman GA:
Intrathecal narcotic infusion pumps for intractable pain of chronic pancreatitis: a pilot series.
Am J Gastroenterol
; 2009 May;104(5):1249-55
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ITNP offers the advantages of reversibility, lower total narcotic dose, and
the pancreas
remaining intact.
Each patient had multiple other failed treatment modalities, including partial
pancreatic
resection (n = 6).
Etiologies of CP included idiopathy (n = 3), cystic fibrosis (n = 2), alcohol (n = 2), and
pancreas
divisum (n = 6).
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(PMID = 19367269.001).
[ISSN]
1572-0241
[Journal-full-title]
The American journal of gastroenterology
[ISO-abbreviation]
Am. J. Gastroenterol.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Narcotics
68.
Payne RL, Bidner TD, Fakler TM, Southern LL:
Growth and intestinal morphology of pigs from sows fed two zinc sources during gestation and lactation.
J Anim Sci
; 2006 Aug;84(8):2141-9
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At weaning and at the end of phase III, 1 gilt per replicate was killed, and the left front foot, liver,
pancreas
, and entire small intestine were removed.
Pancreas
Zn was increased (P < 0.05) in pigs fed ZnSO4 compared with those fed the control diet.
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ZINC, ELEMENTAL
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(PMID = 16864875.001).
[ISSN]
1525-3163
[Journal-full-title]
Journal of animal science
[ISO-abbreviation]
J. Anim. Sci.
[Language]
eng
[Publication-type]
Journal Article; Randomized Controlled Trial
[Publication-country]
United States
[Chemical-registry-number]
J41CSQ7QDS / Zinc
69.
Li Z, Korzh V, Gong Z:
DTA-mediated targeted ablation revealed differential interdependence of endocrine cell lineages in early development of zebrafish pancreas.
Differentiation
; 2009 Nov;78(4):241-52
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[Title]
DTA-mediated targeted ablation revealed differential interdependence of endocrine cell lineages in early development of zebrafish
pancreas
.
In order to study the zebrafish endocrine
pancreas
cell lineage, transgenic expression of diphtheria toxin gene A chain (DTA) under two cell type-specific promoters derived from the insulin (ins) and somatostatin2 (sst2) genes was used to ablate the two types of endocrine cells: insulin-producing beta-cells and somatostatin-producing delta-cells, respectively.
[MeSH-major]
Glucagon-Secreting Cells / metabolism. Islets of Langerhans / cytology.
Pancreas
/ cytology. Somatostatin-Secreting Cells / metabolism. Zebrafish / genetics
ZFIN.
ZFIN
.
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(PMID = 19553000.001).
[ISSN]
1432-0436
[Journal-full-title]
Differentiation; research in biological diversity
[ISO-abbreviation]
Differentiation
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / Diphtheria Toxin; 0 / Insulin; 0 / Peptide Fragments; 0 / diphtheria toxin fragment A; 147336-22-9 / Green Fluorescent Proteins; 51110-01-1 / Somatostatin
70.
Junior RF, Kubrusly MS, Bellodi-Privato M, Molan NA, Machado MC, D'Albuquerque LA:
Beneficial effects of N-acetyl cysteine on pancreas and kidney following experimental pancreatic ischemia-reperfusion in rats.
Clinics (Sao Paulo)
; 2010 Mar;65(3):311-6
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[Title]
Beneficial effects
of N
-acetyl cysteine on
pancreas
and kidney following experimental
pancreatic
ischemia-reperfusion in rats.
OBJECTIVE: To evaluate the protective effects
of N
-acetyl cysteine on
the pancreas
and kidney after
pancreatic
ischemia reperfusion injury in a rat model.
METHODS AND MATERIALS:
Pancreatic
ischemia reperfusion was performed in Wistar rats for 1 hour.
A total of 24 animals were divided into four groups: Group 1: sham; Group 2:
pancreatic
ischemia reperfusion without treatment; Group 3:
pancreatic
ischemia reperfusion plus N-acetyl cysteine intravenously; and Group 4:
pancreatic
ischemia reperfusion plus N-acetyl cysteine per os.
RESULTS: There were significant differences in amylase levels between Group 1 (6.11+/-0.55) and Group 2 (10.30+/-0.50) [p=0.0002] as well as between Group 2 (10.30+/-0.50) and Group 4 (7.82+/-0.38) [p=0.003]; creatinine levels between Group 1 (0.52 +/- 0.07) and Group 2 (0.77+/-0.18) [p=0.035] as well as between Group 2 (0.77+/-0.18) and Group 3 (0.48+/-0.13) [p=0.012]; and
pancreatic
tissue thiobarbituric acid reactive substance levels between Group 1 (1.27+/-0.96) and Group 2 (2.60+/-3.01) [p=0.026] as well as between Group 2 (2.60+/-3.01) and Group 4 (0.52+/-0.56) [p=0.002].
A decrease in
pancreatic
tissue GST-alpha3 gene expression was observed in Group 2 in comparison to Group 1 (p =0.006), and an increase was observed in Groups 3 and 4 when compared to Group 2 (p= 0.025 and p=0.010, respectively).
CONCLUSION: This study provides evidence that N-acetyl cysteine has a beneficial effect on
pancreatic
ischemia reperfusion injury and renal function in a rat model.
[MeSH-major]
Acetylcysteine / pharmacology. Kidney / drug effects.
Pancreas
/ drug effects. Reperfusion Injury / drug therapy
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N-ACETYLCYSTEINE
.
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(PMID = 20360923.001).
[ISSN]
1980-5322
[Journal-full-title]
Clinics (São Paulo, Brazil)
[ISO-abbreviation]
Clinics (Sao Paulo)
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Brazil
[Chemical-registry-number]
EC 2.5.1.18 / Glutathione Transferase; WYQ7N0BPYC / Acetylcysteine
[Other-IDs]
NLM/ PMC2845773
[Keywords]
NOTNLM ; Ischemia / N-Acetyl Cysteine / Pancreas / Reperfusion / Transplantation
71.
Rautou PE, Lévy P, Vullierme MP, O'Toole D, Couvelard A, Cazals-Hatem D, Palazzo L, Aubert A, Sauvanet A, Hammel P, Hentic O, Rebours V, Pelletier AL, Maire F, Ruszniewski P:
Morphologic changes in branch duct intraductal papillary mucinous neoplasms of the pancreas: a midterm follow-up study.
Clin Gastroenterol Hepatol
; 2008 Jul;6(7):807-14
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[Title]
Morphologic changes in branch duct intraductal papillary mucinous neoplasms of the
pancreas
: a midterm follow-up study.
BACKGROUND & AIMS: Because there is a low risk of malignancy for intraductal papillary and mucinous neoplasms of the
pancreas
(IPMNs) confined to branch ducts (BD), patient follow-up evaluation without surgery is possible.
METHODS: All consecutive patients seen from 1999 to 2005 with highly suspected IPMNs confined to BD without criteria suggesting a malignant development (mural nodule, cyst wall thickness >2 mm, BD diameter >30 mm, or main
pancreatic
duct involvement) were followed up prospectively using computerized tomography, magnetic resonance cholangiopancreatography, and endoscopic ultrasonography.
[MeSH-major]
Adenocarcinoma, Mucinous / pathology. Carcinoma,
Pancreatic
Ductal / pathology.
Pancreatic
Neoplasms / pathology
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[CommentIn]
Clin Gastroenterol Hepatol. 2008 Jul;6(7):724-5
[
18602034.001
]
(PMID = 18304885.001).
[ISSN]
1542-7714
[Journal-full-title]
Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association
[ISO-abbreviation]
Clin. Gastroenterol. Hepatol.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
72.
Malek SK, Potdar S, Martin JA, Tublin M, Shapiro R, Fung JJ:
Percutaneous ultrasound-guided pancreas allograft biopsy: a single-center experience.
Transplant Proc
; 2005 Dec;37(10):4436-7
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[Title]
Percutaneous ultrasound-guided
pancreas
allograft biopsy: a single-center experience.
Percutaneous ultrasound-guided
pancreas
allograft biopsy is the preferred technique for evaluating
pancreas
allograft rejection.
Thirty-three patients received simultaneous
pancreas
and kidney transplants, 14 received isolated
pancreas
transplants, and 7 received a
pancreas
transplant after kidney transplantation.
Biopsies were performed by
pancreas
transplantation surgeons with the assistance of radiologists under ultrasound guidance using an Acuson XP 128/10 ultrasound machine.
Eighteen (15%) biopsy samples had no
pancreatic
tissue and showed surrounding fat and small bowel.
One (1.8%) patient had a
pancreatic
fistula, which healed with nonoperative management.
Percutaneous ultrasound-guided
pancreas
allograft biopsy is a safe procedure with a low complication rate and a high tissue yield for histopathologic examination.
[MeSH-major]
Pancreas
Transplantation / pathology. Ultrasonography
[MeSH-minor]
Biopsy. Humans. Kidney Transplantation / pathology.
Pancreas
/ pathology.
Pancreas
/ ultrasonography. Retrospective Studies. Transplantation, Homologous / pathology
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(PMID = 16387139.001).
[ISSN]
0041-1345
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
73.
Mainra R, Elder GJ:
Individualized therapy to prevent bone mineral density loss after kidney and kidney-pancreas transplantation.
Clin J Am Soc Nephrol
; 2010 Jan;5(1):117-24
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[Title]
Individualized therapy to prevent bone mineral density loss after kidney and kidney-
pancreas
transplantation.
BACKGROUND AND OBJECTIVES: Most patients who undergo kidney or kidney-
pancreas
transplantation have renal osteodystrophy, and immediately after transplantation bone mineral density (BMD) commonly falls.
DESIGN, SETTING, PARTICIPANTS, & MEASUREMENTS: A total of 153 kidney (61%) and kidney-
pancreas
(39%) transplant recipients were allocated to bisphosphonate (62%) or calcitriol (38%) therapy using an algorithm that incorporated BMD, prevalent vertebral fracture, biomarkers of bone turnover, and risk factor assessment.
[MeSH-major]
Bone Density. Bone Density Conservation Agents / therapeutic use. Calcitriol / therapeutic use. Diphosphonates / therapeutic use. Kidney Transplantation / adverse effects.
Pancreas
Transplantation / adverse effects
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.
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.
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.
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1,25-DIHYDROXYCHOLECALCIFEROL
.
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[ISSN]
1555-905X
[Journal-full-title]
Clinical journal of the American Society of Nephrology : CJASN
[ISO-abbreviation]
Clin J Am Soc Nephrol
[Language]
eng
[Publication-type]
Clinical Trial; Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Bone Density Conservation Agents; 0 / Diphosphonates; FXC9231JVH / Calcitriol
[Other-IDs]
NLM/ PMC2801646
74.
Cetingok M, Winsett RP, Russell CL, Hathaway DK:
Relationships between sex, race, and social class and social support networks in kidney, liver, and pancreas transplant recipients.
Prog Transplant
; 2008 Jun;18(2):80-8
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[Title]
Relationships between sex, race, and social class and social support networks in kidney, liver, and
pancreas
transplant recipients.
A total of 258 kidney, liver, and
pancreas
transplant recipients participated, 61% of whom were less than 50-years-old.
[MeSH-major]
Kidney Transplantation / psychology. Liver Transplantation / psychology.
Pancreas
Transplantation / psychology. Social Class. Social Support
Genetic Alliance.
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.
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(PMID = 18615972.001).
[ISSN]
1526-9248
[Journal-full-title]
Progress in transplantation (Aliso Viejo, Calif.)
[ISO-abbreviation]
Prog Transplant
[Language]
eng
[Grant]
United States / NINR NIH HHS / NR / R01-NR-0468702
[Publication-type]
Comparative Study; Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
United States
75.
Koshinaga T, Hoshino M, Inoue M, Gotoh H, Sugito K, Ikeda T, Hagiwara N, Tomita R:
Pancreatitis complicated with dilated choledochal remnant after congenital choledochal cyst excision.
Pediatr Surg Int
; 2005 Nov;21(11):936-8
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In these three cases, the choledochal remnant in
the pancreas
head was markedly dilated, probably because of an incomplete resection of the cyst at the primary operation, and an increase in intraluminal pressure of the
pancreatic
duct caused by a dynamic obstruction by a protein plug or a
pancreatic
calculus.
Complete cyst excision, including the choledochal wall in
the pancreas
, is therefore strongly recommended.
[MeSH-minor]
Abdominal Pain / etiology. Adolescent. Adult. Dilatation, Pathologic. Female. Humans.
Pancreas
/ diagnostic imaging. Tomography, X-Ray Computed
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0179-0358
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Pediatric surgery international
[ISO-abbreviation]
Pediatr. Surg. Int.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Germany
76.
Frossard JL, Quadri R, Hadengue A, Morel P, Pastor CM:
Endothelial nitric oxide synthase regulation is altered in pancreas from cirrhotic rats.
World J Gastroenterol
; 2006 Jan 14;12(2):228-33
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[Title]
Endothelial nitric oxide synthase regulation is altered in
pancreas
from cirrhotic rats.
AIM: To determine whether biliary cirrhosis could induce
pancreatic
dysfunction such as modifications in endothelial nitric oxide synthase(eNOS) expression and whether the regulation of eNOS could be altered by the regulatory proteins caveolin and heat shock protein 90 (Hsp90), as well as by the modifications of calmodulin binding to eNOS.
METHODS: Immunoprecipitations and Western blotting analysis were performed in
pancreas
isolated from sham and cirrhotic rats.
RESULTS:
Pancreatic
injury was minor in cirrhotic rats but eNOS expression importantly decreased with the length (and the severity) of the disease.
CONCLUSION: In biliary cirrhosis,
pancreatic
injury is minor but the
pancreatic
nitric oxide (NO) production is significantly decreased by two mechanisms: a decreased expression of the enzyme and a decreased binding of calmodulin to eNOS.
[MeSH-major]
Liver Cirrhosis, Biliary / enzymology. Nitric Oxide Synthase Type III / analysis.
Pancreas
/ enzymology
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[ISSN]
1007-9327
[Journal-full-title]
World journal of gastroenterology
[ISO-abbreviation]
World J. Gastroenterol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
China
[Chemical-registry-number]
0 / Cav1 protein, rat; 0 / Caveolin 1; 0 / HSP90 Heat-Shock Proteins; EC 1.14.13.39 / Nitric Oxide Synthase Type III
[Other-IDs]
NLM/ PMC4066031
77.
Al-Masri M, Krishnamurthy M, Li J, Fellows GF, Dong HH, Goodyer CG, Wang R:
Effect of forkhead box O1 (FOXO1) on beta cell development in the human fetal pancreas.
Diabetologia
; 2010 Apr;53(4):699-711
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[Title]
Effect of forkhead box O1 (FOXO1) on beta cell development in the human fetal
pancreas
.
However, the role of FOXO1 during
pancreatic
development remains largely unknown.
The present study aimed to characterise the expression of the FOXO1 transcription factor in the early to mid-gestation human fetal
pancreas
and to understand its role in islet cell development.
METHODS: Human (8-21 week fetal age)
pancreases
were examined using immunohistological, quantitative RT-PCR and western blotting.
RESULTS: Nuclear and cytoplasmic FOXO1 were widely produced during human fetal endocrine
pancreatic
development, co-localising in cells with the transcription factors
pancreatic
and duodenal homeobox 1 (PDX-1) and neurogenin 3 (NGN3) as well as cytokeratin 19 (CK19), insulin and glucagon.
CONCLUSIONS/INTERPRETATION: Our results indicate that FOXO1 may negatively regulate beta cell differentiation in the human fetal
pancreas
by controlling critical transcription factors, including NGN3 and NKX6-1.
[MeSH-major]
Fetal Development / physiology. Forkhead Transcription Factors / genetics. Insulin-Secreting Cells / physiology.
Pancreas
/ embryology
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(PMID = 20033803.001).
[ISSN]
1432-0428
[Journal-full-title]
Diabetologia
[ISO-abbreviation]
Diabetologia
[Language]
eng
[Grant]
Canada / Canadian Institutes of Health Research / /
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Germany
[Chemical-registry-number]
0 / FOXO1 protein, human; 0 / Forkhead Box Protein O1; 0 / Forkhead Transcription Factors; 0 / Insulin; 0 / RNA, Small Interfering; 0 / Transcription Factors; 9007-92-5 / Glucagon
78.
Smith GC, Trauer T, Kerr PG, Chadban SJ:
Prospective quality-of-life monitoring of simultaneous pancreas and kidney transplant recipients using the 36-item short form health survey.
Am J Kidney Dis
; 2010 Apr;55(4):698-707
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[Title]
Prospective quality-of-life monitoring of simultaneous
pancreas
and kidney transplant recipients using the 36-item short form health survey.
BACKGROUND: Few risk factors for quality-of-life outcomes of simultaneous
pancreas
and kidney transplant recipients are known because of a paucity of data from prospective studies.
PREDICTORS: Demographic data; pretransplant Transplant Evaluation Rating Scale scores; current, past 12 months, and prior lifetime psychiatric
disorder
.
RESULTS: 37 simultaneous
pancreas
and kidney transplant recipients were assessed pretransplant and at 4 months posttransplant.
Current psychiatric
disorder
at pretransplant evaluation predicted higher PCS scores at 4 months (P = 0.002; regression coefficient, -15.42 [95% CI, -24.6 to -6.2]; effect size, 0.22) and 1 year (P = 0.002; regression coefficient, -17.3 [95% CI, -27.9 to -6.7]; effect size, 0.29).
Psychiatric
disorder
before the 12 months before the pretransplant evaluation predicted lower PCS scores at 4 months posttransplant (P < 0.001; regression coefficient, 14.98 [95% CI, 7.1-22.8]; effect size, 0.29).
Past psychiatric
disorder
is a risk factor.
[MeSH-major]
Kidney Transplantation.
Pancreas
Transplantation. Quality of Life
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[Copyright]
Copyright 2010 National Kidney Foundation, Inc. Published by Elsevier Inc. All rights reserved.
(PMID = 20176426.001).
[ISSN]
1523-6838
[Journal-full-title]
American journal of kidney diseases : the official journal of the National Kidney Foundation
[ISO-abbreviation]
Am. J. Kidney Dis.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
79.
Harries LW, Locke JM, Shields B, Hanley NA, Hanley KP, Steele A, Njølstad PR, Ellard S, Hattersley AT:
The diabetic phenotype in HNF4A mutation carriers is moderated by the expression of HNF4A isoforms from the P1 promoter during fetal development.
Diabetes
; 2008 Jun;57(6):1745-52
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RESEARCH DESIGN AND METHODS: We measured the expression of HNF4A isoforms in human adult tissues and gestationally staged fetal
pancreas
by isoform-specific real-time PCR.
The correlation between mutation position and age
of diagnosis
or age-related penetrance was assessed in a cohort of 190 patients with HNF4A mutations.
RESULTS: HNF4A was expressed exclusively from the P2 promoter in adult
pancreas
, but from 9 weeks until at least 26 weeks after conception, up to 23% of expression in fetal
pancreas
was of P1 origin.
In whole
pancreas
, HNF4A9 expression was greater than in islets isolated from the endocrine
pancreas
(relative level 22 vs. 7%).
CONCLUSIONS: We conclude that isoforms derived from the HNF4A P1 promoter are expressed in human fetal, but not adult,
pancreas
, and that their presence during
pancreatic
development may moderate the diabetic phenotype in individuals with mutations in the HNF4A gene.
[MeSH-minor]
Adult. Aged. European Continental Ancestry Group. Female. Gene Expression Profiling. Humans. Kidney / physiology. Kidney / physiopathology. Male. Middle Aged.
Pancreas
/ physiology.
Pancreas
/ physiopathology. Polymerase Chain Reaction. Protein Isoforms / genetics
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[CommentIn]
Diabetes. 2008 Jun;57(6):1461-2
[
18511449.001
]
(PMID = 18356407.001).
[ISSN]
1939-327X
[Journal-full-title]
Diabetes
[ISO-abbreviation]
Diabetes
[Language]
eng
[Grant]
United Kingdom / Wellcome Trust / / 081278/Z/06/Z
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / HNF4A protein, human; 0 / Hepatocyte Nuclear Factor 4; 0 / Protein Isoforms
80.
Naugler C, Xu Z:
Pancreatic adenocarcinoma metastatic to the pineal gland.
J Clin Neurosci
; 2008 Nov;15(11):1284-6
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[Title]
Pancreatic
adenocarcinoma metastatic to the pineal gland.
Here we present the case of a 66-year-old female with autosomal dominant polycystic kidney and liver disease who was found at autopsy to have an unrecognized infiltrating ductal adenocarcinoma of the
pancreas
with metastases to the liver, lungs and pineal gland.
As far as we are aware, this is the first report of a metastasis of infiltrating ductal adenocarcinoma of the
pancreas
to the pineal gland.
[MeSH-major]
Adenocarcinoma / pathology. Brain Neoplasms / secondary.
Pancreatic
Neoplasms / pathology. Pineal Gland. Pinealoma / secondary
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(PMID = 18829324.001).
[ISSN]
0967-5868
[Journal-full-title]
Journal of clinical neuroscience : official journal of the Neurosurgical Society of Australasia
[ISO-abbreviation]
J Clin Neurosci
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Scotland
81.
Atlas E, Nimri R, Miller S, Grunberg EA, Phillip M:
MD-logic artificial pancreas system: a pilot study in adults with type 1 diabetes.
Diabetes Care
; 2010 May;33(5):1072-6
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[Title]
MD-logic artificial
pancreas
system: a pilot study in adults with type 1 diabetes.
OBJECTIVE: Current state-of-the-art artificial
pancreas
systems are either based on traditional linear control theory or rely on mathematical models of glucose-insulin dynamics.
The aim of this study was to describe the principles and clinical performance of the novel MD-Logic Artificial
Pancreas
(MDLAP) System.
[MeSH-major]
Diabetes Mellitus, Type 1 / therapy. Fuzzy Logic. Models, Biological.
Pancreas
, Artificial. Prosthesis Design
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.
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[Cites]
Adv Drug Deliv Rev. 2004 Feb 10;56(2):125-44
[
14741112.001
]
[Cites]
Diabetes Technol Ther. 2004 Jun;6(3):307-18
[
15198833.001
]
[Cites]
Physiol Meas. 2004 Aug;25(4):905-20
[
15382830.001
]
[Cites]
Diabetes Technol Ther. 2009 Mar;11(3):187-94
[
19191486.001
]
[Cites]
J Med Eng Technol. 2005 Mar-Apr;29(2):64-9
[
15804854.001
]
[Cites]
Pediatr Diabetes. 2006 Aug;7 Suppl 4:45-9
[
16774618.001
]
[Cites]
Med Eng Phys. 2007 Sep;29(7):824-7
[
17052939.001
]
[Cites]
IEEE Trans Biomed Eng. 1999 Feb;46(2):148-57
[
9932336.001
]
(PMID = 20150292.001).
[ISSN]
1935-5548
[Journal-full-title]
Diabetes care
[ISO-abbreviation]
Diabetes Care
[Language]
eng
[Databank-accession-numbers]
ClinicalTrials.gov/ NCT00541515
[Publication-type]
Clinical Trial; Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Blood Glucose
[Other-IDs]
NLM/ PMC2858178
82.
Ramos O Jr, Leitão OR, Repka JC, Barros SG:
[Experimental acute pancreatitis induced by L-arginine: a histological and biochemical evaluation].
Arq Gastroenterol
; 2005 Jan-Mar;42(1):55-9
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BACKGROUND: Excessive doses of basic amino acids such as L-arginine are able to injure
the pancreas
of rats.
AIM: To describe and evaluate the biochemical and histological characteristics of acute pancreatitis in rats induced by L-arginine during the installation, development and repair stages of the
pancreatic
inflammatory process.
During those times, blood samples were collected for laboratory testing and samples from
the pancreas
were collected for an optical microscopy analysis.
In optical microscopy, after the injection of L-arginine, a
pancreatic
architecture histologically preserved was observed after 6 hours, evidencing an important interstitial edema in 24 hours.
The
pancreatic
structural reconstruction could be observed after 14 days.
Pancreatic
histological changes were not found in the control group.
CONCLUSION: - Experimental acute pancreatitis induced by L-arginine leads to
pancreatic
necrosis showing self-limited evolution with
pancreas
regeneration in 2 weeks.
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(PMID = 15976912.001).
[ISSN]
0004-2803
[Journal-full-title]
Arquivos de gastroenterologia
[ISO-abbreviation]
Arq Gastroenterol
[Language]
por
[Publication-type]
English Abstract; Journal Article
[Publication-country]
Brazil
[Chemical-registry-number]
94ZLA3W45F / Arginine; EC 3.2.1.- / Amylases
83.
Choi CW, Kim GH, Kang DH, Kim HW, Kim DU, Heo J, Song GA, Park DY, Kim S:
Associated factors for a hyperechogenic pancreas on endoscopic ultrasound.
World J Gastroenterol
; 2010 Sep 14;16(34):4329-34
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[Title]
Associated factors for a hyperechogenic
pancreas
on endoscopic ultrasound.
AIM: To identify the associated risk factors for hyperechogenic
pancreas
(HP) which may be observed on endoscopic ultrasound (EUS) and to assess the relationship between HP and obesity.
Patients with a history of
pancreatic
disease or with hepatobiliary or advanced gastrointestinal cancer were excluded.
[MeSH-major]
Endosonography.
Pancreas
/ ultrasonography
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[CommentIn]
World J Gastroenterol. 2011 Apr 21;17(15):2061-2
[
21528089.001
]
(PMID = 20818817.001).
[ISSN]
2219-2840
[Journal-full-title]
World journal of gastroenterology
[ISO-abbreviation]
World J. Gastroenterol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
China
[Other-IDs]
NLM/ PMC2937114
84.
Noguchi H:
Pancreatic islet transplantation.
World J Gastrointest Surg
; 2009 Nov 30;1(1):16-20
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[Title]
Pancreatic
islet transplantation.
Type 1 diabetes mellitus is an autoimmune disease, which results in the permanent destruction of β-cells of the
pancreatic
islets of Langerhans.
Clinical studies have shown that transplantation
of pancreas
or purified
pancreatic
islets can support glucose homeostasis in type 1 diabetic patients.
Islet transplantation carries the special advantages of being less invasive and resulting in fewer complications compared with the traditional
pancreas
or
pancreas
-kidney transplantation.
However, islet transplantation efforts have limitations including the short supply of donor
pancreata
, the paucity of experienced islet isolation teams, side effects of immunosuppressants and poor long-term results.
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(PMID = 21160790.001).
[ISSN]
1948-9366
[Journal-full-title]
World journal of gastrointestinal surgery
[ISO-abbreviation]
World J Gastrointest Surg
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
China
[Other-IDs]
NLM/ PMC2999120
[Keywords]
NOTNLM ; Islet isolation / Islet regeneration / Pancreatic islet transplantation / Pancreatic β-cells
85.
Komar G, Kauhanen S, Liukko K, Seppänen M, Kajander S, Ovaska J, Nuutila P, Minn H:
Decreased blood flow with increased metabolic activity: a novel sign of pancreatic tumor aggressiveness.
Clin Cancer Res
; 2009 Sep 1;15(17):5511-7
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[Title]
Decreased blood flow with increased metabolic activity: a novel sign of
pancreatic
tumor
aggressiveness.
PURPOSE: To study blood flow (BF) and metabolism in normal
pancreas
and in different
pancreatic
lesions.
We then determined the effect of these biomarkers on outcome in patients with
pancreatic
cancer.
EXPERIMENTAL DESIGN: Oxygen-15-labeled water and fluorodeoxyglucose positron emission tomography/computed tomography scans were used in 26 patients with a suspicion of
pancreatic
cancer to measure
pancreatic
BF and metabolism.
Patients were divided into three groups: patients with a finding of normal
pancreas
(n = 7),
benign
lesions (n = 8), and malignant tumors (n = 11).
RESULTS: Patients with
benign
and malignant
pancreatic
tumors had decreased BF of the lesion by 48% and 60%, respectively, compared with patients with normal
pancreatic
tissue.
SUV(max) was 3-fold higher in malignant tumors compared with both
benign
lesions and normal
pancreas
(P < 0.05).
In contrast, the SUV(max) of patients with
benign
lesions and normal
pancreas
did not differ.
The SUV/BF ratio was significantly higher in malignant lesions than in
benign
lesions or in patients with normal
pancreas
(P < 0.05).
In patients with cancer, high SUV/BF ratio was a stronger predictor of poor survival compared with high metabolism or lower-than-normal
pancreatic
BF.
CONCLUSIONS: BF in
pancreatic
cancer is significantly reduced compared with the normal
pancreas
, which may in part explain the poor success of both radiotherapy and chemotherapy.
We suggest that the composite measurement of BF and metabolism in
pancreatic
cancer could serve as a novel tool in the planning of treatments targeting vasculature.
[MeSH-major]
Biomarkers,
Tumor
/ metabolism. Glucose / metabolism.
Pancreatic
Neoplasms / pathology. Regional Blood Flow
[MeSH-minor]
Aged. Female. Fluorodeoxyglucose F18 / metabolism. Humans. Male. Middle Aged.
Neoplasm
Invasiveness.
Pancreas
/ blood supply.
Pancreas
/ metabolism.
Pancreas
/ pathology. Positron-Emission Tomography. Prognosis
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[CommentIn]
Clin Cancer Res. 2010 Jan 1;16(1):367; author reply 567
[
20028758.001
]
[CommentIn]
Clin Cancer Res. 2009 Sep 1;15(17):5294-6
[
19706819.001
]
[CommentIn]
Future Oncol. 2010 Jan;6(1):13-5
[
20021204.001
]
(PMID = 19706808.001).
[ISSN]
1078-0432
[Journal-full-title]
Clinical cancer research : an official journal of the American Association for Cancer Research
[ISO-abbreviation]
Clin. Cancer Res.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Biomarkers, Tumor; 0Z5B2CJX4D / Fluorodeoxyglucose F18; IY9XDZ35W2 / Glucose
86.
Brandhorst D, Iken M, Bretzel RG, Brandhorst H:
Pancreas storage in oxygenated perfluorodecalin does not restore post-transplant function of isolated pig islets pre-damaged by warm ischemia.
Xenotransplantation
; 2006 Sep;13(5):465-70
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[Title]
Pancreas
storage in oxygenated perfluorodecalin does not restore post-transplant function of isolated pig islets pre-damaged by warm ischemia.
BACKGROUND: Cold storage in oxygenated perfluorodecalin (PFD) restores transplant function of ischemically damaged dog
pancreata
and reduces the impact of cold ischemia on recovery of isolated human islets.
Whether PFD storage can improve islet isolation from
pancreata
exposed to significant warm ischemia (WI) is unclear yet.
Resected
pancreata
were intraductally flushed with cold University of Wisconsin solution.
Subsequently,
pancreata
were processed immediately by digestion-filtration (group I: 0 min WI, n=6; II: 30 min WI, n=6) or first stored for 3 h in oxygenated PFD (III: 0 min WI+PFD, n=5; IV: 30 min WI+PFD, n=6).
RESULTS:
Pancreata
subjected to 30 min of WI yielded significantly less islets compared with the corresponding non-ischemic organs (I vs. II, P<0.01; III vs. IV, P<0.05).
PFD storage of ischemic organs partially restored ATP content (217+/-23 ng/1000 IEQ, II vs. IV, P<0.05) and glucose SI (1.60+/-0.09, II vs. IV, P<0.05) to a significant extent that reached the level of corresponding PFD-stored, non-ischemic
pancreata
(III vs. IV, NS).
The significantly reduced graft function of ischemic islets (I vs. II, III vs. IV, P<0.001) was not increased by
pancreatic
oxygenation (II vs. IV, NS).
CONCLUSIONS: The present study suggests that
pancreas
short-term storage in oxygenated PFD improves in vitro but not the in vivo function of ischemically damaged pig islets.
[MeSH-minor]
Adenosine Triphosphate / metabolism. Animals. Cold Temperature. Hot Temperature. Insulin / metabolism.
Pancreas
/ blood supply. Swine
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(PMID = 16925671.001).
[ISSN]
0908-665X
[Journal-full-title]
Xenotransplantation
[ISO-abbreviation]
Xenotransplantation
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Denmark
[Chemical-registry-number]
0 / Fluorocarbons; 0 / Insulin; 8L70Q75FXE / Adenosine Triphosphate
87.
Argyropoulou MI, Kiortsis DN, Astrakas L, Metafratzi Z, Chalissos N, Efremidis SC:
Liver, bone marrow, pancreas and pituitary gland iron overload in young and adult thalassemic patients: a T2 relaxometry study.
Eur Radiol
; 2007 Dec;17(12):3025-30
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[Title]
Liver, bone marrow,
pancreas
and pituitary gland iron overload in young and adult thalassemic patients: a T2 relaxometry study.
T2 relaxation time (T2) of the liver, bone marrow,
pancreas
and pituitary gland was measured in a 1.5-Tesla magnetic resonance (MR) imager, using a multiecho spin-echo sequence (TR/TE 2,000/20, 40, 60, 80, 100, 120, 140, 160 ms).
The T2 of the
pancreas
was lower in adolescents (43.6 +/- 10.3 ms) than in adults (54.4 +/- 10.4 ms).
There was no significant correlation of the T2 among the liver,
pancreas
, pituitary gland and bone marrow.
There was no significant correlation between serum ferritin and T2 of the liver,
pancreas
and bone marrow.
After adolescence, age-related T2 changes are predominantly associated with pituitary siderosis and fatty degeneration of the
pancreas
.
[MeSH-major]
Iron Overload /
diagnosis
. beta-Thalassemia / pathology
[MeSH-minor]
Adolescent. Adult. Bone Marrow / metabolism. Bone Marrow / pathology. Female. Humans. Liver / metabolism. Liver / pathology. Magnetic Resonance Imaging. Male.
Pancreas
/ metabolism.
Pancreas
/ pathology. Pituitary Gland / metabolism. Pituitary Gland / pathology. Statistics, Nonparametric
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[ISSN]
0938-7994
[Journal-full-title]
European radiology
[ISO-abbreviation]
Eur Radiol
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Germany
88.
Wente MN, Kleeff J, Esposito I, Hartel M, Müller MW, Fröhlich BE, Büchler MW, Friess H:
Renal cancer cell metastasis into the pancreas: a single-center experience and overview of the literature.
Pancreas
; 2005 Apr;30(3):218-22
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[Title]
Renal cancer cell metastasis into
the pancreas
: a single-center experience and overview of the literature.
OBJECTIVES:
The pancreas
is a rare target for metastasis from other primary cancers, but
pancreatic
metastasis play a role in the diagnostic workup of patients with
pancreatic
tumors, especially in patients with a history of renal cell carcinoma (RCC).
METHODS: Between October 2001 and June 2004 data from 601 patients undergoing
pancreatic
resection were entered prospectively in a database and were analyzed for metastasis into
the pancreas
from RCC.
RESULTS: Fifteen patients with metastasis to
the pancreas
from RCC were identified.
One patient showed metastatic disease at time of primary
diagnosis
.
In 8 patients,
the pancreas
was the only site of metastasis, whereas in 7 patients, other organs, such as the thyroid gland, the lung, or the liver, were targets of metastasis, either metachronous or simultaneous at the time of
pancreatic
metastasis.
The median interval between primary treatment of RCC and occurrence of
pancreatic
metastasis was 86 months (range, 0-258).
Most patients were asymptomatic and diagnosed during standard
tumor
follow-up.
CONCLUSION:
Pancreatic
metastasis from RCC is rare but can occur even more than 20 years after primary
tumor
manifestation.
Our results show that
pancreatic
resections for metastasis can be performed safely with a low rate of complications.
Patients with a history of RCC should undergo a long-term follow-up to detect and evaluate for
pancreatic
metastases as well for metastasis to other organ sites.
[MeSH-major]
Carcinoma, Renal Cell / secondary. Databases, Factual. Kidney Neoplasms / pathology.
Pancreatic
Neoplasms / secondary
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(PMID = 15782097.001).
[ISSN]
1536-4828
[Journal-full-title]
Pancreas
[ISO-abbreviation]
Pancreas
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Number-of-references]
26
89.
Capocasale E, Busi N, Mazzoni MP, Valle RD, Maggiore U, Bignardi L, Buzio C, Sianesi M:
Simultaneous kidney-pancreas transplantation: the Parma Center experience.
Acta Biomed
; 2007 Aug;78(2):123-7
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[Title]
Simultaneous kidney-
pancreas
transplantation: the Parma Center experience.
Currently, it is demonstrated that simultaneous
pancreas
-kidney transplantation (SPK) shows beneficial effects on patient survival, on some diabetic degenerative complications and on the quality of life.
Aim of the work is to report our experience in
pancreas
transplantation.
Average
pancreas
cold ischemic time was 716 minutes (range 320-968).
No primary or delayed graft function was observed both for
pancreas
and kidney.
Pancreas
and kidney graft survival rate was 76.5% and 94.1%, respectively.
Our experience, as reported in literature, confirm that a successful
pancreas
transplantation not only brings the recipient back to normal glycemic levels, but it also improves the patient's quality of life by stabilizing some of the secondary complications of diabetes.
[MeSH-major]
Diabetes Mellitus, Type 1 / surgery. Kidney Transplantation.
Pancreas
Transplantation
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(PMID = 17936944.001).
[ISSN]
0392-4203
[Journal-full-title]
Acta bio-medica : Atenei Parmensis
[ISO-abbreviation]
Acta Biomed
[Language]
eng
[Publication-type]
Comparative Study; Journal Article
[Publication-country]
Italy
90.
Haider HH, Illanes H, Ciancio G, Miller J, Burke GW:
Bezoar-related pancreatitis in enterically drained pancreas transplant.
Transplant Proc
; 2007 Jan-Feb;39(1):196-8
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[Title]
Bezoar-related pancreatitis in enterically drained
pancreas
transplant.
Simultaneous kidney and
pancreas
transplantation is currently the treatment of choice for type 1 diabetes mellitus with end-stage renal disease.
Causes of pancreatitis after
pancreas
transplantation with enteric drainage are not well documented in the literature.
We report a case of allograft pancreatitis from
pancreatic
duct outflow obstruction due to formation of a bezoar in a diverticulized transplant duodeno-jejunal anastomosis.
[MeSH-major]
Bezoars. Diabetes Mellitus, Type 1 / surgery.
Pancreas
Transplantation / adverse effects. Pancreatitis / etiology
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(PMID = 17275505.001).
[ISSN]
0041-1345
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
United States
91.
Gupta V, Singh V, Kalra N, Vaiphei K:
Pancreas sparing resection for giant hamartoma of Brunner's glands.
JOP
; 2009;10(2):196-9
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[Title]
Pancreas
sparing resection for giant hamartoma of Brunner's glands.
CONTEXT:
Benign
proliferative changes of the Brunner's glands account for about 10% of neoplasias of the duodenal bulb.
A
pancreas
-sparing duodenal resection was performed.
The diagnosis
was established on histopathology.
A
pancreas
-sparing duodenal resection is one of the modalities of treating such lesions.
[MeSH-minor]
Adult.
Diagnosis
, Differential. Duodenum / pathology. Duodenum / surgery. Female. Humans. Pancreaticoduodenectomy / methods. Treatment Outcome
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(PMID = 19287118.001).
[ISSN]
1590-8577
[Journal-full-title]
JOP : Journal of the pancreas
[ISO-abbreviation]
JOP
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Italy
92.
Kilbourn MR:
Rat pancreas uptake of [11C]dihydrotetrabenazine stereoisomers.
Nucl Med Biol
; 2010 Nov;37(8):869-71
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[Title]
Rat
pancreas
uptake of [11C]dihydrotetrabenazine stereoisomers.
(+)-α-[(11)C]Dihydrotetrabenazine ((+)-[(11)C]DTBZ), a radioligand for the vesicular monoamine transporter type 2 (VMAT2), has been previously proposed as an in vivo marker of beta-cell degeneration in
the pancreas
.
The stereospecificity of uptake of [(11)C]DTBZ into rat
pancreas
was examined here using radiolabeled forms of the (+)- and (-)-isomers.
Pancreas
localization of (+)-[(11)C]DTBZ could be partially blocked by prior administration of unlabeled (+)-DTBZ.
Pancreatic
uptake of the (-)-isomer was unexpectedly high and could not be blocked by pretreatment with (+)-DTBZ, but could be significantly reduced by treatment with racemic tetrabenazine, an in vivo source of (-)-DTBZ.
These studies indicate that the inactive isomer of DTBZ does not provide a mechanism for defining the nonspecific binding of (+)-DTBZ in rat
pancreas
.
[MeSH-major]
Pancreas
/ metabolism. Tetrabenazine / analogs & derivatives
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[Copyright]
Copyright © 2010 Elsevier Inc. All rights reserved.
(PMID = 21055616.001).
[ISSN]
1872-9614
[Journal-full-title]
Nuclear medicine and biology
[ISO-abbreviation]
Nucl. Med. Biol.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
England
[Chemical-registry-number]
0 / Carbon Radioisotopes; 3466-75-9 / dihydrotetrabenazine; Z9O08YRN8O / Tetrabenazine
93.
Mohammed A, Janakiram NB, Li Q, Madka V, Ely M, Lightfoot S, Crawford H, Steele VE, Rao CV:
The epidermal growth factor receptor inhibitor gefitinib prevents the progression of pancreatic lesions to carcinoma in a conditional LSL-KrasG12D/+ transgenic mouse model.
Cancer Prev Res (Phila)
; 2010 Nov;3(11):1417-26
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[Title]
The epidermal growth factor receptor inhibitor gefitinib prevents the progression of
pancreatic
lesions to carcinoma in a conditional LSL-KrasG12D/+ transgenic mouse model.
Pancreatic
ductal adenocarcinoma (PDAC) is the most common
pancreatic
malignancy with a dismal prognosis.
Developing novel strategies to prevent or delay
pancreatic
cancer is currently of intense interest.
The chemopreventive efficacy of gefitinib, an epidermal growth factor receptor (EGFR) inhibitor, was evaluated against the progression of
pancreatic
intraepithelial neoplasms (PanIN) to PDAC in conditional LSL-Kras(G12D/+) transgenic mice.
At termination,
pancreases
were evaluated histopathologically for PanINs and PDAC, and various biomarkers were measured by immunohistochemistry, immunofluorescence, immunoblotting, and/or reverse transcription-PCR.
Furthermore, mice treated with 100 and 200 ppm of gefitinib exhibited 67.6% to 77.3% of the
pancreas
to be free from ductal lesions.
Also, gefitinib reduced EGFR, proliferating cell nuclear antigen, cyclin D1, C(2)GNT, RhoA, β-catenin, p38, phospho-extracellular signal-regulated kinase, caveolin-1, and mucin and increased cyclin B1 in the
pancreatic
lesions/PDAC.
In summary, these results show that gefitinib can prevent the progression of
pancreatic
cancer precursor lesions to PDAC in a preclinical model.
The present study highlights the promise of chemoprevention and the potential usefulness of EGFR inhibitors in individuals at high risk for
pancreatic
cancer.
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