[X] Close
You are about to erase all the values you have customized, search history, page format, etc.
Click here to RESET all values       Click here to GO BACK without resetting any value
Items 1 to 100 of about 13179
1. Huang ES, Turner BG, Fernandez-Del-Castillo C, Brugge WR, Hur C: Pancreatic cystic lesions: clinical predictors of malignancy in patients undergoing surgery. Aliment Pharmacol Ther; 2010 Jan 15;31(2):285-94
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Pancreatic cystic lesions: clinical predictors of malignancy in patients undergoing surgery.
  • BACKGROUND: Despite advances in cross-sectional imaging and the use of molecular markers, distinguishing between benign and malignant cysts remains a clinical challenge.
  • METHODS: A retrospective analysis was performed on consecutive patients with pancreatic cysts who underwent endoscopic ultrasound (EUS) and surgical resection from May 1996 to December 2007 at a tertiary centre.
  • Clinical history, EUS characteristics, cytology, tumour markers and surgical histology were collected.
  • RESULTS: A total of 153 patients underwent a EUS and subsequent surgical intervention.Of the 153 patients, 57 (37%) had a histological diagnosis of malignancy.
  • History of pancreatitis (P = 0.008) and endoscopist impression of pseudocyst (P = 0.001) were found to be associated with benign cysts.

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • ClinicalTrials.gov. clinical trials - ClinicalTrials.gov .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Ann Surg. 2007 Oct;246(4):644-51; discussion 651-4 [17893501.001]
  • [Cites] Clin Gastroenterol Hepatol. 2008 Nov;6(11):1194-7 [18848907.001]
  • [Cites] Ann Surg Oncol. 2008 Jan;15(1):199-205 [17909912.001]
  • [Cites] J Gastrointest Surg. 2008 Feb;12(2):234-42 [18040749.001]
  • [Cites] Ann Oncol. 2008 May;19 Suppl 2:ii104-5 [18456741.001]
  • [Cites] Am J Surg. 2008 Jul;196(1):100-13 [18466869.001]
  • [Cites] Ann Surg. 2004 Jun;239(6):788-97; discussion 797-9 [15166958.001]
  • [Cites] N Engl J Med. 2004 Sep 16;351(12):1218-26 [15371579.001]
  • [Cites] Acta Cytol. 1991 Jul-Aug;35(4):456-63 [1718115.001]
  • [Cites] Gastroenterology. 1995 Apr;108(4):1230-5 [7535275.001]
  • [Cites] Gastrointest Endosc. 1997 Mar;45(3):268-76 [9087833.001]
  • [Cites] Gut. 2005 Jun;54 Suppl 4:iv1-16 [15888809.001]
  • [Cites] Dig Liver Dis. 2006 Jan;38(1):39-44 [16314152.001]
  • [Cites] Ann Surg. 2006 Oct;244(4):572-82 [16998366.001]
  • [Cites] Clin Gastroenterol Hepatol. 2007 Apr;5(4):489-95 [17350894.001]
  • [Cites] Gastroenterology. 2007 Jul;133(1):72-9; quiz 309-10 [17631133.001]
  • [Cites] J Comput Assist Tomogr. 1999 Nov-Dec;23(6):906-12 [10589565.001]
  • [Cites] Ann Surg. 2000 Feb;231(2):205-12 [10674612.001]
  • [Cites] AJR Am J Roentgenol. 2000 Jul;175(1):99-103 [10882255.001]
  • [Cites] Am J Surg Pathol. 2000 Oct;24(10):1372-7 [11023098.001]
  • [Cites] Gastrointest Endosc. 2002 Oct;56(4):543-7 [12297771.001]
  • [Cites] Arch Surg. 2002 Nov;137(11):1274-8 [12413317.001]
  • [Cites] J Clin Gastroenterol. 2003 Mar;36(3):261-5 [12590239.001]
  • [Cites] Arch Surg. 2003 Jun;138(6):610-7; discussion 617-8 [12799331.001]
  • [Cites] Gastrointest Endosc. 2003 Jul;58(1):59-64 [12838222.001]
  • [Cites] Am J Gastroenterol. 2003 Jul;98(7):1516-24 [12873573.001]
  • [Cites] Br J Surg. 2003 Oct;90(10):1244-9 [14515294.001]
  • [Cites] Gastrointest Endosc. 2003 Nov;58(5):701-6 [14595305.001]
  • [Cites] Ann Surg. 2004 May;239(5):651-7; discussion 657-9 [15082969.001]
  • [Cites] Ann Surg. 2004 May;239(5):678-85; discussion 685-7 [15082972.001]
  • [Cites] Pancreas. 2004 Apr;28(3):241-6 [15084964.001]
  • [Cites] Gastroenterology. 2004 May;126(5):1330-6 [15131794.001]
  • [Cites] AJR Am J Roentgenol. 2007 Sep;189(3):648-56 [17715113.001]
  • [Cites] J Comput Assist Tomogr. 2007 Nov-Dec;31(6):860-7 [18043347.001]
  • (PMID = 19845568.001).
  • [ISSN] 1365-2036
  • [Journal-full-title] Alimentary pharmacology & therapeutics
  • [ISO-abbreviation] Aliment. Pharmacol. Ther.
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / CA107060-05; United States / NCI NIH HHS / CA / K07CA107060; United States / NIDDK NIH HHS / DK / T32 DK007191; United States / NCI NIH HHS / CA / K07 CA107060-05; United States / NIDDK NIH HHS / DK / T32DK007191; United States / NCI NIH HHS / CA / K07 CA107060
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] England
  • [Other-IDs] NLM/ NIHMS153996; NLM/ PMC2807915
  •  go-up   go-down


2. Kang CM, Lee JW: Spleen preserving laparoscopic distal pancreatectomy with segmental resection of splenic artery in a solid pseudo papillary tumor of the pancreas. Hepatogastroenterology; 2009 Jul-Aug;56(93):1207-10
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Spleen preserving laparoscopic distal pancreatectomy with segmental resection of splenic artery in a solid pseudo papillary tumor of the pancreas.
  • Laparoscopic distal pancreatectomy is suitable for benign and premalignant neoplasms located in the body and tail of the pancreas.
  • In addition, spleen-preservation following distal pancreatectomy for benign or low-grade malignant disease is known to be safe.
  • We present a case of 32-year-old female patient with a solid pseudopapillary tumor of the pancreas treated by spleen-preserving laparoscopic distal pancreatectomy with segmental resection of the splenic artery and splenic vein intact.
  • [MeSH-major] Adenocarcinoma, Papillary / surgery. Laparoscopy. Pancreatectomy / methods. Pancreatic Neoplasms / surgery. Splenic Artery / surgery

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 19760971.001).
  • [ISSN] 0172-6390
  • [Journal-full-title] Hepato-gastroenterology
  • [ISO-abbreviation] Hepatogastroenterology
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Greece
  •  go-up   go-down


3. Kobayashi N, Kubota K, Kato S, Watanabe S, Shimamura T, Kirikoshi H, Saito S, Ueda M, Endo I, Inayama Y, Maeda S, Nakajima A: FOXP3+ regulatory T cells and tumoral indoleamine 2,3-dioxygenase expression predicts the carcinogenesis of intraductal papillary mucinous neoplasms of the pancreas. Pancreatology; 2010;10(5):631-40
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] FOXP3+ regulatory T cells and tumoral indoleamine 2,3-dioxygenase expression predicts the carcinogenesis of intraductal papillary mucinous neoplasms of the pancreas.
  • A recent study revealed that indoleamine 2,3-dioxygenase (IDO)-mediated tryptophan depletion was able to affect local tumor-infiltrating lymphocytes.
  • The aim of this study was to investigate the clinical significance of the tumor-infiltrating Tregs and tumoral IDO expression during the progression of intraductal papillary mucinous neoplasms (IPMNs) of the pancreas.
  • IDO expression in the tumor was observed in 5 cases with IPMNs (IPMC, n = 1; I-IPMC, n = 4).
  • IDO expression in the tumor was positively correlated with the prevalence of Tregs in IPMNs.
  • IDO expression in the tumor is one of the late-stage phenomena of multistage carcinogenesis of IPMNs.
  • [MeSH-major] Adenocarcinoma, Mucinous / pathology. Carcinoma, Pancreatic Ductal / pathology. Cell Transformation, Neoplastic / pathology. Forkhead Transcription Factors / metabolism. Indoleamine-Pyrrole 2,3,-Dioxygenase / metabolism. Pancreatic Neoplasms / pathology

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] Copyright © 2010 S. Karger AG, Basel.
  • (PMID = 21051918.001).
  • [ISSN] 1424-3911
  • [Journal-full-title] Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.]
  • [ISO-abbreviation] Pancreatology
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 0 / FOXP3 protein, human; 0 / Forkhead Transcription Factors; 0 / Indoleamine-Pyrrole 2,3,-Dioxygenase
  •  go-up   go-down


Advertisement
4. Anhalt H, Bohannon NJ: Insulin patch pumps: their development and future in closed-loop systems. Diabetes Technol Ther; 2010 Jun;12 Suppl 1:S51-8
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Steady progress is being made toward the development of a so-called "artificial pancreas," which may ultimately be a fully automated, closed-loop, glucose control system comprising a continuous glucose monitor, an insulin pump, and a controller.
  • A major factor propelling artificial pancreas development is the substantial incidence of-and attendant patient, parental, and physician concerns about-hypoglycemia and extreme hyperglycemia associated with current means of insulin delivery for type 1 diabetes mellitus (T1DM).
  • A successful fully automated artificial pancreas would likely reduce the frequency of and anxiety about hypoglycemia and marked hyperglycemia.
  • Patch-pump systems ("patch pumps") are likely to be used increasingly in the control of T1DM and may be incorporated into the artificial pancreas systems of tomorrow.
  • The use of advanced algorithms in the clinical development of closed-loop systems is reviewed along with projected next steps in artificial pancreas development.

  • MedlinePlus Health Information. consumer health - Diabetes Type 1.
  • The Lens. Cited by Patents in .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Diabetes Care. 2003 Apr;26(4):1176-80 [12663593.001]
  • [Cites] J Diabetes Sci Technol. 2009 Jan;3(1):44-55 [19444330.001]
  • [Cites] Diabetes. 1997 Feb;46(2):271-86 [9000705.001]
  • [Cites] Diabet Med. 1999 Jun;16(6):466-71 [10391393.001]
  • [Cites] Diabetes Metab Res Rev. 2004 Nov-Dec;20(6):479-86 [15386817.001]
  • [Cites] Diabetes Technol Ther. 2005 Feb;7(1):28-47 [15738702.001]
  • [Cites] N Engl J Med. 2005 Dec 22;353(25):2643-53 [16371630.001]
  • [Cites] Diabetes. 2006 Dec;55(12):3344-50 [17130478.001]
  • [Cites] IEEE Trans Biomed Eng. 2006 Dec;53(12 Pt 1):2472-8 [17153204.001]
  • [Cites] IEEE Trans Biomed Eng. 2007 Oct;54(10):1740-9 [17926672.001]
  • [Cites] Pediatr Diabetes. 2007 Dec;8(6):362-8 [18036061.001]
  • [Cites] Curr Opin Endocrinol Diabetes Obes. 2008 Apr;15(2):118-22 [18316945.001]
  • [Cites] IEEE Trans Biomed Eng. 2008 Mar;55(3):857-65 [18334377.001]
  • [Cites] Diabetes Care. 2008 May;31(5):934-9 [18252903.001]
  • [Cites] Diabet Med. 2008 Jul;25(7):765-74 [18644063.001]
  • [Cites] Curr Diabetes Rev. 2008 Aug;4(3):269-79 [18690909.001]
  • [Cites] Diabetes Technol Ther. 2008 Dec;10(6):441-4 [19049372.001]
  • [Cites] J Clin Endocrinol Metab. 2009 Feb;94(2):410-5 [19066300.001]
  • [Cites] Diabetes Technol Ther. 2009 Apr;11(4):207-10 [19344194.001]
  • [Cites] JAMA. 2009 Apr 15;301(15):1525-7 [19366764.001]
  • [Cites] Diabetes Technol Ther. 2009 Mar;11(3):187-94 [19191486.001]
  • [Cites] Diabetes Educ. 2009 May-Jun;35(3):465-75 [19321802.001]
  • [Cites] Expert Rev Med Devices. 2009 Jul;6(4):401-10 [19572795.001]
  • [Cites] Diabetes Technol Ther. 2009 Jun;11 Suppl 1:S113-9 [19621478.001]
  • [Cites] J Diabetes Sci Technol. 2009 Sep;3(5):1014-21 [20144414.001]
  • [Cites] Chronic Illn. 2009 Dec;5(4):250-6 [19933244.001]
  • [Cites] Physiol Meas. 2004 Aug;25(4):905-20 [15382830.001]
  • (PMID = 20515308.001).
  • [ISSN] 1557-8593
  • [Journal-full-title] Diabetes technology & therapeutics
  • [ISO-abbreviation] Diabetes Technol. Ther.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Hypoglycemic Agents; 0 / Insulin
  • [Other-IDs] NLM/ PMC2924780
  •  go-up   go-down


5. Yang L, Liu YF, Liu SR, Wu G, Zhang JL, Meng YM, Shong SW, Li GC: Prevention and treatment of rejection after simultaneous pancreas-kidney transplantation. Chin Med Sci J; 2005 Sep;20(3):210-3
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Prevention and treatment of rejection after simultaneous pancreas-kidney transplantation.
  • OBJECTIVE: To explore methods of preventing and reversing rejection after simultaneous pancreas-kidney (SPK) transplantation.
  • RESULTS: No primary non-functionality of either kidney or pancreas occurred in this series of transplantations.
  • One patient experienced the accelerated rejection, resulting in the resection of the pancreas and kidney grafts because of the failure of conservative therapy.
  • Only the kidney was involved in 35.3% (6/17); and both the pancreas and kidney were involved in 11.8% (2/17).
  • OKT3 (0.5 mg x d(-1)) was administered for 7-10 days in two patients with both renal and pancreas rejection.
  • Monitoring renal function and pancreas exocrine secretion, and reasonable application of immunosuppressants play important roles in the diagnosis and treatment of rejection.

  • Genetic Alliance. consumer health - Transplantation.
  • MedlinePlus Health Information. consumer health - Kidney Transplantation.
  • MedlinePlus Health Information. consumer health - Pancreas Transplantation.
  • Hazardous Substances Data Bank. PREDNISONE .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16261897.001).
  • [ISSN] 1001-9294
  • [Journal-full-title] Chinese medical sciences journal = Chung-kuo i hsueh k'o hsueh tsa chih
  • [ISO-abbreviation] Chin. Med. Sci. J.
  • [Language] ENG
  • [Publication-type] Journal Article
  • [Publication-country] China
  • [Chemical-registry-number] 0 / Antibodies, Monoclonal; 0 / Antibodies, Monoclonal, Humanized; 0 / Immunoglobulin G; 0 / Immunosuppressive Agents; 0 / Muromonab-CD3; 67U96J8P35 / meprednisone; AYI8EX34EU / Creatinine; CUJ2MVI71Y / daclizumab; VB0R961HZT / Prednisone
  •  go-up   go-down


6. Frystyk J, Ritzel RA, Maubach J, Büsing M, Lück R, Klempnauer J, Schmiegel W, Nauck MA: Comparison of pancreas-transplanted type 1 diabetic patients with portal-venous versus systemic-venous graft drainage: impact on glucose regulatory hormones and the growth hormone/insulin-like growth factor-I axis. J Clin Endocrinol Metab; 2008 May;93(5):1758-66
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Comparison of pancreas-transplanted type 1 diabetic patients with portal-venous versus systemic-venous graft drainage: impact on glucose regulatory hormones and the growth hormone/insulin-like growth factor-I axis.
  • CONTEXT: Pancreas grafts can be drained through the iliac vein (systemic drainage) or the portal vein.
  • OBJECTIVE: We hypothesized that normalization of portal insulin in patients with portal pancreas graft drainage stimulates the GH/IGF-I axis and thereby contributes to glucose control.
  • METHODS: We compared patients after combined kidney and pancreas transplantation with portal drainage (n = 7) to patients with systemic drainage of the pancreas graft (n = 8) and nondiabetic controls (n = 8).
  • CONCLUSION: Portal drainage of pancreatic endocrine secretion in pancreas graft recipients raises IGF-I and lowers GH secretion.
  • [MeSH-major] Diabetes Mellitus, Type 1 / surgery. Human Growth Hormone / physiology. Insulin-Like Growth Factor I / physiology. Pancreas Transplantation

  • MedlinePlus Health Information. consumer health - Diabetes Type 1.
  • MedlinePlus Health Information. consumer health - Pancreas Transplantation.
  • COS Scholar Universe. author profiles.
  • Hazardous Substances Data Bank. GLUCAGON .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 18319317.001).
  • [ISSN] 0021-972X
  • [Journal-full-title] The Journal of clinical endocrinology and metabolism
  • [ISO-abbreviation] J. Clin. Endocrinol. Metab.
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / C-Peptide; 0 / Insulin; 0 / Insulin-Like Growth Factor Binding Protein 3; 12629-01-5 / Human Growth Hormone; 67763-96-6 / Insulin-Like Growth Factor I; 9007-92-5 / Glucagon
  •  go-up   go-down


7. Smale E, Davison AM, Smith M, Pritchard C: Fatal intra-abdominal haemorrhage following percutaneous endoscopic gastrostomy. BMJ Case Rep; 2009;2009
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • A postmortem examination revealed 2.5 litres of blood and bloodstained fluid within the abdominal cavity and a haemorrhagic pancreas.
  • Microscopy of the pancreas showed a defect in a small to medium-sized artery, likely to be a branch of the splenic artery.
  • The cause of death (as per section 1 of the death certificate) was (1a) intra-abdominal haemorrhage, (1b) pancreatic trauma at PEG feeding tube insertion and (1c) dysphagia due to cerebrovascular disease.

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] BMJ. 1998 Aug 22;317(7157):524-5 [9712605.001]
  • [Cites] Gut. 2003 Dec;52 Suppl 7:vii1-vii12 [14612488.001]
  • [Cites] Ir Med J. 2003 Oct;96(9):265-7 [14753579.001]
  • [Cites] Arch Surg. 1998 Oct;133(10):1076-83 [9790204.001]
  • (PMID = 21853009.001).
  • [ISSN] 1757-790X
  • [Journal-full-title] BMJ case reports
  • [ISO-abbreviation] BMJ Case Rep
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC3027889
  •  go-up   go-down


8. Onishi T, Igarashi T, Ichikawa T: [Case of retroperitoneal fibrosis after surgical treatment of autoimmune pancreatitis]. Hinyokika Kiyo; 2009 Sep;55(9):551-4
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • During the routine follow-up for colon cancer, a swollen pancreas tail was detected on enhanced CT.
  • He received distal pancreatectomy under the diagnosis of pancreas cancer on 4th October, 2007.
  • Pathological diagnosis revealed the autoimmune pancreatitis.
  • He was treated with predonisolone aiming at the diagnosis and/or therapy.
  • Now, it is thought that autoimmune pancreatitis is a systemic sclerosing disease accompanied with extra-pancreatic pathologic changes such as RPF.
  • [MeSH-major] Autoimmune Diseases / complications. Pancreatitis / complications. Retroperitoneal Fibrosis / diagnosis. Retroperitoneal Fibrosis / etiology


9. Li Z, Ruan L, Lin S, Gittes GK: Clock controls timing of mouse pancreatic differentiation through regulation of Wnt- and Notch-based and cell division components. Biochem Biophys Res Commun; 2007 Aug 3;359(3):491-6
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Clock controls timing of mouse pancreatic differentiation through regulation of Wnt- and Notch-based and cell division components.
  • Here we first show that before embryonic circadian rhythms occur, the oscillation (nucleocytoplasmic shuttling) of core circadian gene Clock is tissue-specific and correlated with the state of differentiation during both early development and later pancreas organogenesis.
  • Disruption of Clock as well as Timeless in the embryonic pancreas does not block pancreatic differentiation but alters the balance and maturity of endocrine and exocrine cells.
  • [MeSH-major] Cell Differentiation. Circadian Rhythm / physiology. Pancreas / cytology. Pancreas / metabolism. Receptors, Notch / metabolism. Trans-Activators / metabolism. Wnt Proteins / metabolism

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17559809.001).
  • [ISSN] 0006-291X
  • [Journal-full-title] Biochemical and biophysical research communications
  • [ISO-abbreviation] Biochem. Biophys. Res. Commun.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Oligonucleotides, Antisense; 0 / Receptors, Notch; 0 / Trans-Activators; 0 / Transcription Factors; 0 / Wnt Proteins; EC 2.3.1.48 / CLOCK Proteins; EC 2.3.1.48 / Clock protein, mouse
  •  go-up   go-down


10. Roda O, Chiva C, Espuña G, Gabius HJ, Real FX, Navarro P, Andreu D: A proteomic approach to the identification of new tPA receptors in pancreatic cancer cells. Proteomics; 2006 Apr;6 Suppl 1:S36-41
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] A proteomic approach to the identification of new tPA receptors in pancreatic cancer cells.
  • We have developed a strategy to identify putative tissue-type plasminogen activator (tPA)receptors present in pancreatic cancer cells by affinity capture with tPA-Sepharose followed by 2-DE and MALDI-MS PMF.
  • Proteins pulled down from either total lysates or raft membrane fractions were characterized and compared with those from a total lysate of an endothelial cell line (HUVEC) to identify pancreas-restricted tPA receptors.
  • A total of 31 proteins were found by this approach, including annexin A2, already described as a tPA receptor in pancreas and endothelial cells, other proteins acting as tPA receptors (i.e., enolase, cytokeratins 8 and 18) in other tissues, and additional proteins not previously identified as candidate tPA receptors.
  • [MeSH-major] Pancreatic Neoplasms / metabolism. Proteomics. Receptors, Cell Surface / metabolism. Tissue Plasminogen Activator / metabolism

  • Genetic Alliance. consumer health - Pancreatic cancer.
  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16544279.001).
  • [ISSN] 1615-9861
  • [Journal-full-title] Proteomics
  • [ISO-abbreviation] Proteomics
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / Annexin A2; 0 / Receptors, Cell Surface; EC 3.4.21.68 / Tissue Plasminogen Activator
  • [Number-of-references] 36
  •  go-up   go-down


11. Burgos FJ, Gómez V, Pascual J, Marcen R, Villafruela JJ, Correa C, Cuevas B, Mampaso F, García-Gonzalez R: Pancreas islet transplantation in the genitourinary tract associated with renal transplantation: an experimental study. Transplant Proc; 2006 Oct;38(8):2585-7
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Pancreas islet transplantation in the genitourinary tract associated with renal transplantation: an experimental study.
  • Conceptually, pancreas islet transplantation (PIT) associated with renal transplantation (RT) should resolve not only chronic renal failure but also diabetes.
  • The mean weight of the pancreatic specimens was 27.8 g (13 to 46).
  • For the first time, the feasibility of the bladder submucosa as an implantation site for pancreas islets was demonstrated.

  • MedlinePlus Health Information. consumer health - Islet Cell Transplantation.
  • MedlinePlus Health Information. consumer health - Kidney Transplantation.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17098010.001).
  • [ISSN] 0041-1345
  • [Journal-full-title] Transplantation proceedings
  • [ISO-abbreviation] Transplant. Proc.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  •  go-up   go-down


12. Cohen ND, Shaw JE: Diabetes: advances in treatment. Intern Med J; 2007 Jun;37(6):383-8
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Pancreas and islet cell transplantation are the subject of ongoing research, but currently require immunosuppressive treatment regimes.
  • The main limitation is lack of availability of donor pancreases.

  • Genetic Alliance. consumer health - Diabetes.
  • MedlinePlus Health Information. consumer health - Diabetes Type 1.
  • MedlinePlus Health Information. consumer health - Diabetes Type 2.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17535382.001).
  • [ISSN] 1445-5994
  • [Journal-full-title] Internal medicine journal
  • [ISO-abbreviation] Intern Med J
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Australia
  • [Number-of-references] 35
  •  go-up   go-down


13. Yamaguchi K, Nakamura M, Shirahane K, Kawamoto M, Konomi H, Ohta M, Tanaka M: Pancreatic juice cytology in IPMN of the pancreas. Pancreatology; 2005;5(4-5):416-21; discussion 421
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Pancreatic juice cytology in IPMN of the pancreas.
  • BACKGROUND: Intraductal papillary-mucinous neoplasm (IPMN) of the pancreas is a disease ranging from adenoma to borderline (with moderate dysplasia) and further to carcinoma (noninvasive and invasive) and surgical strategy is different by the grades of dysplasia.
  • METHODS: Preoperative pancreatic juice cytology in IPMN was reviewed in 71 patients with IPMN who underwent surgical resection.
  • The sensitivity of pancreatic juice cytology in malignant IPMN was 40% (4/10).
  • In 4 patients with the 48 IPM adenomas, diagnosis of pancreatic juice cytology was class IV or V.
  • Sensitivity of pancreatic juice cytology was higher in IPMN of the main duct type with mucin hypersecretion and with mural nodules.
  • CONCLUSIONS: These findings suggest that pancreatic juice cytology in IPMN is useful especially in the main duct type with mucin hypersecretion and mural nodules.
  • When the diagnosis of pancreatic juice cytology is malignant in otherwise benign-looking IPMNs, coexistence of pancreatic carcinoma should be suspected.
  • [MeSH-major] Adenocarcinoma, Mucinous / diagnosis. Carcinoma, Pancreatic Ductal / diagnosis. Carcinoma, Papillary / diagnosis. Cytodiagnosis / methods. Pancreatic Juice / cytology. Pancreatic Neoplasms / diagnosis
  • [MeSH-minor] Adenoma / classification. Adenoma / diagnosis. Aged. Female. Humans. Male. Neoplasm Staging. Retrospective Studies. Sensitivity and Specificity

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] Copyright 2005 S. Karger AG, Basel and IAP.
  • (PMID = 15985766.001).
  • [ISSN] 1424-3903
  • [Journal-full-title] Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.]
  • [ISO-abbreviation] Pancreatology
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Switzerland
  •  go-up   go-down


14. Demir MK, Kilicoğlu G, Akinci O: Alveolar hydatid disease of the liver: brief review and spectrum of adjacent organ invasion. Australas Radiol; 2007 Aug;51(4):346-50
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Local invasion to right kidney and adrenal, right hemidiaphragm and lung were detected in one patient, right adrenal in another patient and gall bladder, duodenum, gastric wall and pancreas invasion in the other.
  • AHD may rarely extend to the gall bladder, stomach, duodenum, pancreas, right adrenal and kidney, diaphragm, pleura and lung.

  • Genetic Alliance. consumer health - Liver Disease.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17635471.001).
  • [ISSN] 0004-8461
  • [Journal-full-title] Australasian radiology
  • [ISO-abbreviation] Australas Radiol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Australia
  •  go-up   go-down


15. Brandhorst H, Alt A, Huettler S, Raemsch-Guenther N, Kurfuerst M, Bretzel RG, Brandhorst D: The ratio between class II and class I collagenase determines the amount of neutral protease activity required for efficient islet release from the rat pancreas. Transplant Proc; 2005 Jan-Feb;37(1):215-6
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The ratio between class II and class I collagenase determines the amount of neutral protease activity required for efficient islet release from the rat pancreas.
  • Previous investigations clearly showed that the successful release of islets from the pancreas is mediated by both neutral protease (NP) and collagenase, consisting of subclasses I and II showing different capacities to cleave islets from the pancreas.
  • METHODS: Following intraductal pancreas collagenase distension, rat islets were isolated utilizing 20 PZ-U Serva collagenase NB 1 and 1.0 or 0.4 DMC-U NP.
  • [MeSH-major] Collagenases. Islets of Langerhans / cytology. Pancreas / cytology. Peptide Hydrolases / metabolism

  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 15808598.001).
  • [ISSN] 0041-1345
  • [Journal-full-title] Transplantation proceedings
  • [ISO-abbreviation] Transplant. Proc.
  • [Language] eng
  • [Grant] United States / NIDDK NIH HHS / DK / DK 56962-2
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] EC 3.4.- / Peptide Hydrolases; EC 3.4.24.- / Collagenases
  •  go-up   go-down


16. Rupnik M: The physiology of rodent beta-cells in pancreas slices. Acta Physiol (Oxf); 2009 Jan;195(1):123-38
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The physiology of rodent beta-cells in pancreas slices.
  • Beta-cells in pancreatic islets form complex syncytia.
  • Pancreas slice is a novel method of choice to study the physiology of beta-cells still embedded in their normal cellulo-social context.
  • I present major advantages, list drawbacks and provide an overview on recent advances in our understanding of the physiology of beta-cells using the pancreas slice approach.
  • [MeSH-major] Insulin-Secreting Cells / physiology. Pancreas / physiology

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 18983446.001).
  • [ISSN] 1748-1716
  • [Journal-full-title] Acta physiologica (Oxford, England)
  • [ISO-abbreviation] Acta Physiol (Oxf)
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] England
  • [Number-of-references] 200
  •  go-up   go-down


17. Hayashibe A, Sakamoto K, Shinbo M, Makimoto S, Nakamoto T: A resected case of multiple intraductal papillary mucinous tumors of the pancreas with US-guided ductal branch-oriented partial pancreatectomy. Pancreatology; 2005;5(4-5):462-5
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] A resected case of multiple intraductal papillary mucinous tumors of the pancreas with US-guided ductal branch-oriented partial pancreatectomy.
  • The cystic tumor of pancreas head had a diameter of 2 cm, and the mural nodule of the cystic tumor measured only 3 mm.
  • In the pancreas body the cystic tumor was measured at 1.5 cm with the mural nodule of the cystic tumor measuring 3 mm.
  • It was believed that the tumors were benign.
  • The reduction operation for preservation of pancreatic parenchyma should be selected for these circumstances.
  • Ductal branch-oriented partial pancreatectomy was performed on September 6, 2002 with intraoperative ultrasonography and a Cavitron Ultrasonic Aspirator, preserving the main pancreatic duct and normal pancreatic parenchyma.
  • The operation was successful, and the histopathological diagnosis of the tumors was intraductal papillary adenoma of the pancreas.
  • [MeSH-major] Adenoma / surgery. Digestive System Surgical Procedures. Pancreatectomy / methods. Pancreatic Ducts / surgery. Pancreatic Neoplasms / surgery

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] Copyright 2005 S. Karger AG, Basel and IAP.
  • (PMID = 15985773.001).
  • [ISSN] 1424-3903
  • [Journal-full-title] Pancreatology : official journal of the International Association of Pancreatology (IAP) ... [et al.]
  • [ISO-abbreviation] Pancreatology
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 0 / Mucins
  •  go-up   go-down


18. Plate JM, Plate AE, Shott S, Bograd S, Harris JE: Effect of gemcitabine on immune cells in subjects with adenocarcinoma of the pancreas. Cancer Immunol Immunother; 2005 Sep;54(9):915-25
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Effect of gemcitabine on immune cells in subjects with adenocarcinoma of the pancreas.
  • Effects of gemcitabine (Gemzar) on immune cells were examined in pancreas cancer patients to determine whether it was immunosuppressive, or potentially could be combined with vaccines or other immunotherapy to enhance patient's responses to their tumors.
  • Functional activity was measured by intracellular staining for cytokines before and after T-cell activation, and by interferon gamma production in EliSpot responses to tumor presentation.
  • Immune T-cells were functional in pancreas cancer patients treated with gemcitabine.
  • [MeSH-major] Antimetabolites, Antineoplastic / therapeutic use. Deoxycytidine / analogs & derivatives. Lymphocyte Activation / drug effects. Pancreatic Neoplasms / drug therapy. Pancreatic Neoplasms / immunology. T-Lymphocytes

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • COS Scholar Universe. author profiles.
  • The Lens. Cited by Patents in .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 15782312.001).
  • [ISSN] 0340-7004
  • [Journal-full-title] Cancer immunology, immunotherapy : CII
  • [ISO-abbreviation] Cancer Immunol. Immunother.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / Antimetabolites, Antineoplastic; 0W860991D6 / Deoxycytidine; B76N6SBZ8R / gemcitabine; EC 1.17.4.- / Ribonucleotide Reductases
  •  go-up   go-down


19. Kristoffersen AB, Viljugrein H, Kongtorp RT, Brun E, Jansen PA: Risk factors for pancreas disease (PD) outbreaks in farmed Atlantic salmon and rainbow trout in Norway during 2003-2007. Prev Vet Med; 2009 Jul 1;90(1-2):127-36
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Risk factors for pancreas disease (PD) outbreaks in farmed Atlantic salmon and rainbow trout in Norway during 2003-2007.
  • Pancreas disease (PD) is an emerging infectious disease in farmed Atlantic salmon (Salmo salar L.) and rainbow trout (Oncorhynchus mykiss Walbaum) caused by salmonid alphavirus (SAV).
  • Records of clinical diagnosis of PD on marine farm sites were used to identify PD case cohorts.
  • In PD case cohorts, PD-outbreaks were defined to start the month the diagnosis was recorded and last until the cohort was terminated.
  • [MeSH-major] Alphavirus Infections / veterinary. Disease Outbreaks / veterinary. Fish Diseases / epidemiology. Oncorhynchus mykiss. Pancreatic Diseases / veterinary. Salmo salar

  • MedlinePlus Health Information. consumer health - Pancreatic Diseases.
  • COS Scholar Universe. author profiles.
  • The Lens. Cited by Patents in .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 19419787.001).
  • [ISSN] 1873-1716
  • [Journal-full-title] Preventive veterinary medicine
  • [ISO-abbreviation] Prev. Vet. Med.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  •  go-up   go-down


20. Li X, Han D, Kin Ting Kam R, Guo X, Chen M, Yang Y, Zhao H, Chen Y: Developmental expression of sideroflexin family genes in Xenopus embryos. Dev Dyn; 2010 Oct;239(10):2742-7
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Whole-mount in situ hybridization analysis reveals that while sideroflexin2 is expressed in the pancreas, sideroflexin1 and 3 display a complex expression in the central nervous system, somites, pronephros, liver, and pancreas.
  • [MeSH-minor] Amino Acid Sequence. Animals. Brain / embryology. Brain / metabolism. Cation Transport Proteins / chemistry. Cation Transport Proteins / classification. Cation Transport Proteins / genetics. Cation Transport Proteins / metabolism. Gene Expression Regulation, Developmental / genetics. Gene Expression Regulation, Developmental / physiology. Liver / embryology. Liver / metabolism. Molecular Sequence Data. Pancreas / embryology. Pancreas / metabolism. Phylogeny. Sequence Homology, Amino Acid. Xenopus

  • Xenbase. Xenbase .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 20737508.001).
  • [ISSN] 1097-0177
  • [Journal-full-title] Developmental dynamics : an official publication of the American Association of Anatomists
  • [ISO-abbreviation] Dev. Dyn.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Cation Transport Proteins; 0 / Xenopus Proteins
  •  go-up   go-down


21. Nakazawa N, Okazaki T, Miyano T: Prenatal detection of isolated gastric duplication cyst. Pediatr Surg Int; 2005 Oct;21(10):831-4
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • A routine prenatal ultrasonography (US) showed an abdominal cyst with peristalsis and a provisional diagnosis of enteric duplication was made.
  • A healthy male infant was born at 39 weeks gestation and postnatal US identified a cyst, 5x3x2 cm in size, adjacent to the pancreas.
  • At laparotomy, a cyst was found located in the lesser sac, but completely separated from the stomach, and partially adhered to the body of the pancreas and the crura of the diaphragm.

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Eur J Pediatr Surg. 1991 Oct;1(5):309-11 [1747365.001]
  • [Cites] Am J Perinatol. 2002 Oct;19(7):361-6 [12442225.001]
  • [Cites] Arch Dis Child. 1960 Feb;35:76-86 [13799033.001]
  • [Cites] Gastroenterology. 1980 Aug;79(2):344-7 [7399240.001]
  • [Cites] J Pediatr Surg. 1999 Aug;34(8):1284-6 [10466615.001]
  • [Cites] Prenat Diagn. 2000 Feb;20(2):163-7 [10694692.001]
  • [Cites] Radiographics. 1993 Sep;13(5):1063-80 [8210590.001]
  • [Cites] J Ultrasound Med. 1986 Oct;5(10):589-91 [3534293.001]
  • [Cites] Abdom Imaging. 2003 Jan-Feb;28(1):12-4 [12483377.001]
  • [Cites] J Pediatr Surg. 2004 Aug;39(8):e5-7 [15300555.001]
  • [Cites] J Ultrasound Med. 1997 Mar;16(3):219-22 [9166822.001]
  • [Cites] Dig Surg. 2000;17(6):634-636 [11155012.001]
  • (PMID = 16200403.001).
  • [ISSN] 0179-0358
  • [Journal-full-title] Pediatric surgery international
  • [ISO-abbreviation] Pediatr. Surg. Int.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Germany
  •  go-up   go-down


22. Krejci V, Hiltebrand LB, Sigurdsson GH: Effects of epinephrine, norepinephrine, and phenylephrine on microcirculatory blood flow in the gastrointestinal tract in sepsis. Crit Care Med; 2006 May;34(5):1456-63
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Microcirculatory flow was measured in gastric, jejunal, and colon mucosa; jejunal muscularis; and pancreas, liver, and kidney using multiple-channel laser Doppler flowmetry.
  • Both agents caused a significant reduction in superior mesenteric artery flow (11 +/- 4%, p < .05, and 26 +/- 6%, p < .01, respectively) and in microcirculatory blood flow in the jejunal mucosa (21 +/- 5%, p < .01, and 23 +/- 3%, p < .01, respectively) and in the pancreas (16 +/- 3%, p < .05, and 8 +/- 3%, not significant, respectively).
  • In fact, norepinephrine and epinephrine appeared to divert blood flow away from the mesenteric circulation and decrease microcirculatory blood flow in the jejunal mucosa and pancreas.
  • [MeSH-minor] Analysis of Variance. Animals. Blood Pressure / drug effects. Cross-Over Studies. Hemodynamics / drug effects. Intestines / blood supply. Liver / blood supply. Microcirculation / drug effects. Pancreas / blood supply. Random Allocation. Swine

  • Hazardous Substances Data Bank. Norepinephrine .
  • Hazardous Substances Data Bank. EPINEPHRINE .
  • Hazardous Substances Data Bank. PHENYLEPHRINE .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [CommentIn] Crit Care Med. 2006 May;34(5):1565-6 [16633263.001]
  • (PMID = 16557162.001).
  • [ISSN] 0090-3493
  • [Journal-full-title] Critical care medicine
  • [ISO-abbreviation] Crit. Care Med.
  • [Language] eng
  • [Publication-type] Comparative Study; Evaluation Studies; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Vasoconstrictor Agents; 1WS297W6MV / Phenylephrine; X4W3ENH1CV / Norepinephrine; YKH834O4BH / Epinephrine
  •  go-up   go-down


23. Doley RP, Yadav TD, Kang M, Dalal A, Jayant M, Sharma R, Wig JD: Traumatic Transection of Pancreas at the Neck: Feasibility of Parenchymal Preserving Strategy. Gastroenterology Res; 2010 Apr;3(2):79-85
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Traumatic Transection of Pancreas at the Neck: Feasibility of Parenchymal Preserving Strategy.
  • BACKGROUND: To assess the feasibility and safety of a pancreas preserving operative technique in the management of isolated complete pancreatic neck transection following blunt abdominal trauma.
  • METHODS: Two patients with isolated blunt fracture of the pancreatic neck underwent pancreas preserving procedure comprising of oversewing of the proximal pancreas and Roux-en-Y pancreatico jejunostomy to the distal remnant.
  • Both had pancreatic transection at neck in the line of superior mesenteric vessels.
  • These patients had pancreas parenchyma preserving surgery - internal drainage of the left remnant in a Roux-en-Y jejunal loop.
  • CONCLUSIONS: Pancreas preserving strategy - suture of head side of pancreas and an internal drainage of left remnant with a Roux-en-Y jejunal loop is feasible and safe and should be considered in selected cases.
  • Substantial amount of normal pancreatic parenchyma is preserved.

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 27956990.001).
  • [ISSN] 1918-2805
  • [Journal-full-title] Gastroenterology research
  • [ISO-abbreviation] Gastroenterology Res
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Canada
  • [Keywords] NOTNLM ; Abdominal injuries / Organ preservation / Pancreas / Pancreatic anastomosis / Pancreatic injuries
  •  go-up   go-down


24. DiNorcia J, Ahmed L, Lee MK, Reavey PL, Yakaitis EA, Lee JA, Schrope BA, Chabot JA, Allendorf JD: Better preservation of endocrine function after central versus distal pancreatectomy for mid-gland lesions. Surgery; 2010 Dec;148(6):1247-54; discussion 1254-6
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • BACKGROUND: Traditional resections for benign and low-grade malignant neoplasms of the mid pancreas result in loss of normal parenchyma that can cause pancreatic endocrine and exocrine insufficiency.
  • Overall morbidity was 41.1% with pancreatic fistula in 20.5%.
  • CONCLUSION: CP is safe and effective for select neoplasms of the mid pancreas.
  • [MeSH-major] Pancreatectomy / methods. Pancreatic Diseases / surgery. Pancreatic Fistula / surgery
  • [MeSH-minor] Continental Population Groups. Cysts / surgery. Diabetes Mellitus / epidemiology. Diabetes Mellitus / etiology. Disease Progression. Drainage / methods. Female. Humans. Male. Middle Aged. Neoplasm Invasiveness. Pancreatic Neoplasms / pathology. Pancreatic Neoplasms / surgery. Postoperative Complications / classification. Postoperative Complications / therapy. Retrospective Studies. Safety. Sepsis / epidemiology. Treatment Outcome

  • MedlinePlus Health Information. consumer health - Pancreatic Diseases.
  • COS Scholar Universe. author profiles.
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] Copyright © 2010 Mosby, Inc. All rights reserved.
  • (PMID = 21134558.001).
  • [ISSN] 1532-7361
  • [Journal-full-title] Surgery
  • [ISO-abbreviation] Surgery
  • [Language] eng
  • [Grant] United States / NHLBI NIH HHS / HL / T32 HL-007854-14
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  •  go-up   go-down


26. Huguier M, Barrier A, Gouillat C, Suc B, Jaeck D, Launois B: [Pancreaticoduodenectomy for cancer of the head of the pancreas]. J Chir (Paris); 2008 Jan-Feb;145(1):9-15
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Pancreaticoduodenectomy for cancer of the head of the pancreas].
  • Preoperative evidence of mesenteric or portal vein involvement does not contraindicate pancreatic resection and survival rates are similar to those of patients with no venous involvement.
  • Three trials and one meta-analysis of pancreatico-gastric anastomosis have failed to demonstrate a decrease in the risk of pancreatic fistula.
  • Two trials suggest that the risk of fistula formation is decreased by implantation of the pancreatic remnant into the jejunum or by trans-jejunal stenting of the pancreatico-jejunal anastomosis with external drainage; but these findings are not supported by a third trial.
  • The use of fibrin glue to occlude the pancreatic duct or seal the cut surface of the pancreas did not decrease the rate of intra-abdominal complications.
  • In conclusion, the pancreaticoduodenal resection described by Whipple may still be considered the gold standard for resection of pancreatic cancer.
  • [MeSH-major] Pancreatic Neoplasms / surgery. Pancreaticoduodenectomy / methods

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [CommentIn] J Chir (Paris). 2008 Jan-Feb;145(1):6-8 [18438275.001]
  • (PMID = 18438276.001).
  • [ISSN] 0021-7697
  • [Journal-full-title] Journal de chirurgie
  • [ISO-abbreviation] J Chir (Paris)
  • [Language] fre
  • [Publication-type] English Abstract; Journal Article; Review
  • [Publication-country] France
  • [Number-of-references] 76
  •  go-up   go-down


27. Zhao HL, Sui Y, Guan J, Lai FM, Gu XM, He L, Zhu X, Rowlands DK, Xu G, Tong PC, Chan JC: Topographical associations between islet endocrine cells and duct epithelial cells in the adult human pancreas. Clin Endocrinol (Oxf); 2008 Sep;69(3):400-6
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Topographical associations between islet endocrine cells and duct epithelial cells in the adult human pancreas.
  • OBJECTIVES: The pancreatic ducts, endocrine islets and exocrine acini are three functionally related components.
  • The objective of this study is to investigate the topographical associations between the islet endocrine cells and duct epithelial cells in the adult human pancreas.
  • MATERIALS AND METHODS: Panels of immunomarkers for the exocrine acinar cells (amylase), duct cells [cytokeratin 19 (CK19)], endocrine cells (chromogranin A, neurone specific enolase, synaptophysin) and islet hormones (glucagon, insulin, somatostatin, pancreatic polypeptide) were applied to sequential pancreatic tissue sections obtained from autopsy specimens of 10-nondiabetic human adults.
  • CONCLUSIONS: Topographical associations between islet endocrine cells and pancreatic duct cells are frequent in adult human pancreas.
  • The islet-duct association suggests possible functional interactions between the two interrelated pancreatic compartments.
  • [MeSH-major] Epithelial Cells / cytology. Islets of Langerhans / cytology. Pancreatic Ducts / cytology
  • [MeSH-minor] Aged. Aged, 80 and over. Biomarkers / metabolism. Endocrine Cells / cytology. Endocrine Cells / immunology. Endocrine Cells / metabolism. Female. Glucagon / metabolism. Humans. Insulin / metabolism. Male. Middle Aged. Pancreas / cytology. Pancreas / immunology. Pancreas / metabolism

  • Hazardous Substances Data Bank. GLUCAGON .
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 18221396.001).
  • [ISSN] 1365-2265
  • [Journal-full-title] Clinical endocrinology
  • [ISO-abbreviation] Clin. Endocrinol. (Oxf)
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Biomarkers; 0 / Insulin; 9007-92-5 / Glucagon
  •  go-up   go-down


28. Sakuramoto S, Kikuchi S, Futawatari N, Katada N, Moriya H, Hirai K, Yamashita K, Watanabe M: Laparoscopy-assisted pancreas- and spleen-preserving total gastrectomy for gastric cancer as compared with open total gastrectomy. Surg Endosc; 2009 Nov;23(11):2416-23
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Laparoscopy-assisted pancreas- and spleen-preserving total gastrectomy for gastric cancer as compared with open total gastrectomy.
  • METHODS: From July 2004 to July 2007, we performed pancreas- and spleen-preserving total gastrectomy with D1 + beta or D2 lymph-node dissection and Roux-en-Y reconstruction in 74 patients with cancer located in the upper or middle third of the stomach.
  • However, major complications such as anastomotic leakage, abdominal abscess, and pancreatic leakage occurred in six patients (13.6%) in the OTG group, but in none of the patients in the LATG group.
  • [MeSH-major] Gastrectomy / methods. Gastroscopy / methods. Lymph Nodes / pathology. Neoplasm Recurrence, Local / epidemiology. Stomach Neoplasms / surgery
  • [MeSH-minor] Anastomosis, Surgical. Chi-Square Distribution. Cohort Studies. Female. Follow-Up Studies. Humans. Laparotomy / adverse effects. Laparotomy / methods. Lymph Node Excision / methods. Male. Middle Aged. Neoplasm Invasiveness / pathology. Neoplasm Staging. Pancreas. Postoperative Complications / mortality. Postoperative Complications / physiopathology. Retrospective Studies. Risk Assessment. Spleen. Survival Analysis. Treatment Outcome

  • MedlinePlus Health Information. consumer health - Stomach Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [CommentIn] Surg Endosc. 2010 Sep;24(9):2355-7 [20177935.001]
  • [Cites] J Surg Oncol. 2008 Feb 1;97(2):169-72 [18095269.001]
  • [Cites] Am J Surg. 2007 Dec;194(6):839-44; discussion 844 [18005781.001]
  • [Cites] Gastric Cancer. 2007;10(3):176-80 [17922096.001]
  • [Cites] Surg Endosc. 2008 Sep;22(9):1997-2002 [18594925.001]
  • [Cites] J Am Coll Surg. 2004 Sep;199(3):508-15 [15325625.001]
  • [Cites] Br J Surg. 2007 Feb;94(2):204-7 [17058319.001]
  • [Cites] Surg Endosc. 2008 Apr;22(4):980-4 [17690934.001]
  • [Cites] Surg Endosc. 2006 Sep;20(9):1497-500 [16755350.001]
  • [Cites] Surgery. 2002 Jan;131(1 Suppl):S306-11 [11821829.001]
  • [Cites] Gastric Cancer. 2000 Dec;3(4):219-225 [11984739.001]
  • [Cites] Ann Surg. 2007 Jan;245(1):68-72 [17197967.001]
  • [Cites] Gastric Cancer. 1999 Dec;2(4):230-234 [11957104.001]
  • [Cites] Ann Surg. 1999 Jan;229(1):49-54 [9923799.001]
  • [Cites] World J Surg. 2002 Sep;26(9):1145-9 [12209244.001]
  • [Cites] Gastric Cancer. 1998 Dec;1(1):10-24 [11957040.001]
  • [Cites] Surg Laparosc Endosc. 1994 Apr;4(2):146-8 [8180768.001]
  • [Cites] Surg Endosc. 2006 Jan;20(1):55-60 [16283580.001]
  • [Cites] Surg Endosc. 2008 May;22(5):1161-4 [18322744.001]
  • [Cites] Surg Laparosc Endosc Percutan Tech. 2005 Dec;15(6):309-14 [16340559.001]
  • [Cites] Gastrointest Endosc. 2002 Nov;56(5):708-13 [12397280.001]
  • [Cites] Br J Surg. 2001 Jan;88(1):128-32 [11136325.001]
  • (PMID = 19266232.001).
  • [ISSN] 1432-2218
  • [Journal-full-title] Surgical endoscopy
  • [ISO-abbreviation] Surg Endosc
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article
  • [Publication-country] Germany
  •  go-up   go-down


29. Akimoto Y, Hart GW, Wells L, Vosseller K, Yamamoto K, Munetomo E, Ohara-Imaizumi M, Nishiwaki C, Nagamatsu S, Hirano H, Kawakami H: Elevation of the post-translational modification of proteins by O-linked N-acetylglucosamine leads to deterioration of the glucose-stimulated insulin secretion in the pancreas of diabetic Goto-Kakizaki rats. Glycobiology; 2007 Feb;17(2):127-40
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Elevation of the post-translational modification of proteins by O-linked N-acetylglucosamine leads to deterioration of the glucose-stimulated insulin secretion in the pancreas of diabetic Goto-Kakizaki rats.
  • By immunoblot and immunohistochemical analyses, the expression of O-GlcNAc transferase protein and O-GlcNAc-modified proteins in whole pancreas and islets of Langerhans of 15-week-old diabetic GK rats and nondiabetic Wistar rats was examined.
  • The expression of O-GlcNAc transferase at the protein level and O-GlcNAc transferase activity were increased significantly in the diabetic pancreas and islets.
  • The diabetic pancreas and islets also showed an increase in total cellular O-GlcNAc-modified proteins.
  • These results indicate that elevation of the O-GlcNAcylation of proteins leads to deterioration of insulin secretion in the pancreas of diabetic GK rats, further providing evidence for the role of O-GlcNAc in the insulin secretion.
  • [MeSH-minor] Animals. Glucose / pharmacology. Islets of Langerhans / chemistry. Islets of Langerhans / drug effects. Islets of Langerhans / secretion. N-Acetylglucosaminyltransferases / analysis. N-Acetylglucosaminyltransferases / antagonists & inhibitors. N-Acetylglucosaminyltransferases / metabolism. Pancreas / chemistry. Pancreas / drug effects. Pancreas / secretion. RNA Interference. Rats. Rats, Wistar

  • MedlinePlus Health Information. consumer health - Diabetes.
  • MedlinePlus Health Information. consumer health - Diabetes Medicines.
  • COS Scholar Universe. author profiles.
  • Gene Ontology. gene/protein/disease-specific - Gene Ontology annotations from this paper .
  • Hazardous Substances Data Bank. GLUCOSE .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17095531.001).
  • [ISSN] 0959-6658
  • [Journal-full-title] Glycobiology
  • [ISO-abbreviation] Glycobiology
  • [Language] eng
  • [Grant] United States / NIDDK NIH HHS / DK / DK61671
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Homeodomain Proteins; 0 / Insulin; 0 / Trans-Activators; 0 / pancreatic and duodenal homeobox 1 protein; EC 2.4.1.- / N-Acetylglucosaminyltransferases; EC 2.4.1.- / UDP-N-acetylglucosamine-peptide beta-N-acetylglucosaminyltransferase; IY9XDZ35W2 / Glucose; V956696549 / Acetylglucosamine
  •  go-up   go-down


30. Stringer MD, Davison SM, McClean P, Rajwal S, Puntis JW, Sheridan M, Ramsden W, Woodley H: Multidisciplinary management of surgical disorders of the pancreas in childhood. J Pediatr Gastroenterol Nutr; 2005 Mar;40(3):363-7
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Multidisciplinary management of surgical disorders of the pancreas in childhood.
  • OBJECTIVES: To describe the frequency and range of pancreatic disorders in children requiring surgical intervention and to highlight the importance of multidisciplinary management.
  • METHODS: An audit of all children under 17 years of age referred with surgical disorders of the pancreas or pancreatitis to a regional pediatric gastroenterology unit in the United Kingdom during a 10-year period.
  • RESULTS: Surgical intervention was required for the following pancreatic disorders: persistent hyperinsulinemic hypoglycemia of infancy (n = 4), pancreatic tumors (n = 5), pancreaticobiliary malunion (n = 12), pancreatic trauma (n = 6) and pancreatitis (n = 10).
  • The indications for surgery in acute pancreatitis were a persistent pseudocyst (n = 1) and treatment of an underlying cause of pancreatitis (n = 4); in chronic pancreatitis, surgery was used to treat symptomatic pancreatic duct strictures (n = 4).
  • All 33 children with acute pancreatitis, including four with pancreatic necrosis, survived.
  • CONCLUSIONS: Surgery for pancreatic disorders in children is rarely required but may be necessary a) for definitive management of primary pancreatic pathology, b) to treat sequelae of acute or chronic pancreatitis and c) to treat an underlying cause of pancreatitis.
  • [MeSH-major] Pancreatic Diseases / surgery. Pancreatitis / surgery
  • [MeSH-minor] Acute Disease. Adolescent. Child. Child, Preschool. Congenital Hyperinsulinism / pathology. Congenital Hyperinsulinism / surgery. Disease-Free Survival. Female. Great Britain. Humans. Infant. Male. Pancreas / injuries. Pancreas / pathology. Pancreas / surgery. Pancreatectomy. Pancreatic Cyst / pathology. Pancreatic Cyst / surgery. Pancreatic Neoplasms / pathology. Pancreatic Neoplasms / surgery. Retrospective Studies. Treatment Outcome

  • MedlinePlus Health Information. consumer health - Pancreatic Diseases.
  • MedlinePlus Health Information. consumer health - Pancreatitis.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 15735494.001).
  • [ISSN] 0277-2116
  • [Journal-full-title] Journal of pediatric gastroenterology and nutrition
  • [ISO-abbreviation] J. Pediatr. Gastroenterol. Nutr.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  •  go-up   go-down


31. Qian ZY, Miao Y, Dai CC, Xu ZK, Liu XL: [Combined multiple organ resection in 16 patients with adenocarcinoma of the body or tail of the pancreas]. Zhongguo Yi Xue Ke Xue Yuan Xue Bao; 2005 Oct;27(5):572-4
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Combined multiple organ resection in 16 patients with adenocarcinoma of the body or tail of the pancreas].
  • OBJECTIVE: To investigate the feasibility and therapeutic results of multiple organ resection in patients with tumor of the body and tail of pancreas.
  • METHODS: The clinical and pathological data were analysed in 16 consecutive patients with neoplasm of the body and tail of pancreas from 1999 to 2004 retrospectively.
  • RESULTS: Multiple organ resection was performed in 6 cases of primary pancreatic adenocarcinoma of the body and tail (3 cases of pancreatic cancer, 2 cases of malignant glucagonoma, and 1 case of well-differentiated pancreatic stromal sarcoma) and 10 cases of extrapancreatic malignancy (4 cases of gastric cancer, 2 cases of gastric leiomyosarcoma, 1 case of duodenal cancer, and 3 cases of colon cancer of hepatic flexure).
  • Patients with primary pancreatic cancer or pancreatic stromal sarcoma died within 1 year.
  • [MeSH-major] Adenocarcinoma / surgery. Pancreatectomy / methods. Pancreatic Neoplasms / surgery

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16274034.001).
  • [ISSN] 1000-503X
  • [Journal-full-title] Zhongguo yi xue ke xue yuan xue bao. Acta Academiae Medicinae Sinicae
  • [ISO-abbreviation] Zhongguo Yi Xue Ke Xue Yuan Xue Bao
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] China
  •  go-up   go-down


32. Shen L, Liu X, Hou W, Yang G, Wu Y, Zhang R, Li X, Che H, Lu Z, Zhang Y, Liu X, Yao L: NDRG2 is highly expressed in pancreatic beta cells and involved in protection against lipotoxicity. Cell Mol Life Sci; 2010 Apr;67(8):1371-81
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] NDRG2 is highly expressed in pancreatic beta cells and involved in protection against lipotoxicity.
  • The N-myc downstream-regulated gene 2 (NDRG2) is involved in cell differentiation and apoptosis, but its function in the pancreas remains to be established.
  • Herein we examine the expression and function of NDRG2 in the endocrine pancreas.
  • NDRG2 immunoreactivity was localized mainly in the cytoplasm of pancreatic beta cells.
  • Collectively, these data indicate that NDRG2 acts as a key molecule in pancreatic beta cells and is involved in the Akt-mediated protection of beta cells against lipotoxicity.
  • [MeSH-major] Fatty Acids, Nonesterified / pharmacology. Insulin-Secreting Cells / metabolism. Proteins / metabolism. Tumor Suppressor Proteins / metabolism

  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • antibodies-online. View related products from antibodies-online.com (subscription/membership/fee required).
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Neuroreport. 2008 Jun 11;19(9):927-31 [18520995.001]
  • [Cites] Lancet. 2007 May 26;369(9575):1823-31 [17531891.001]
  • [Cites] J Biol Chem. 2004 Apr 30;279(18):18623-32 [14985363.001]
  • [Cites] Endocrinology. 2004 Nov;145(11):5087-96 [15297438.001]
  • [Cites] Diabetologia. 2007 Apr;50(4):752-63 [17268797.001]
  • [Cites] Diabetologia. 2004 May;47(5):806-15 [15095038.001]
  • [Cites] J Mol Endocrinol. 2007 Jun;38(6):651-61 [17556534.001]
  • [Cites] J Biol Chem. 2007 Sep 21;282(38):28264-73 [17652085.001]
  • [Cites] Nucleic Acids Res. 2008 Sep;36(16):5335-49 [18689861.001]
  • [Cites] Histochem Cell Biol. 2002 Nov;118(5):399-408 [12432451.001]
  • [Cites] Diabetes. 2005 Apr;54(4):968-75 [15793234.001]
  • [Cites] J Physiol. 2009 Apr 1;587(Pt 7):1619-34 [19204049.001]
  • [Cites] J Exp Clin Cancer Res. 2007 Jun;26(2):261-8 [17725107.001]
  • [Cites] Neurosci Lett. 2005 Nov 18;388(3):157-62 [16039777.001]
  • [Cites] Exp Mol Med. 2007 Dec 31;39(6):705-14 [18160841.001]
  • [Cites] Endocrinology. 2002 Feb;143(2):339-42 [11796484.001]
  • [Cites] J Biol Chem. 2009 Oct 30;284(44):30039-48 [19734538.001]
  • [Cites] Int J Cancer. 2003 Sep 1;106(3):342-7 [12845671.001]
  • [Cites] Brain Res Dev Brain Res. 2002 Apr 30;135(1-2):55-63 [11978393.001]
  • [Cites] Neurobiol Dis. 2004 Jun;16(1):48-58 [15207261.001]
  • [Cites] Diabetologia. 2006 Dec;49(12):2900-9 [17053882.001]
  • [Cites] Am J Physiol Endocrinol Metab. 2007 May;292(5):E1447-55 [17264229.001]
  • [Cites] Diabetologia. 2006 Mar;49(3):506-18 [16440210.001]
  • [Cites] Diabetes. 2006 Aug;55(8):2301-10 [16873694.001]
  • [Cites] J Cell Sci. 2005 Sep 1;118(Pt 17):3905-15 [16091421.001]
  • [Cites] J Biol Chem. 2006 Aug 4;281(31):21942-53 [16731532.001]
  • [Cites] Regul Pept. 2005 Nov;131(1-3):82-8 [16102855.001]
  • [Cites] Cell Physiol Biochem. 2008;21(1-3):239-50 [18209490.001]
  • [Cites] Eur J Biochem. 2000 Sep;267(17):5421-6 [10951200.001]
  • [Cites] Proc Natl Acad Sci U S A. 2008 Jun 17;105(24):8452-7 [18550819.001]
  • [Cites] J Biol Chem. 2006 Feb 3;281(5):2764-72 [16314423.001]
  • [Cites] Appl Biochem Biotechnol. 2009 Feb;152(2):306-15 [18758694.001]
  • [Cites] Cell Tissue Res. 2006 Jul;325(1):67-76 [16520977.001]
  • [Cites] Diabetologia. 2002 Mar;45(3):309-26 [11914736.001]
  • [Cites] J Biol Chem. 2003 Apr 18;278(16):13829-37 [12574167.001]
  • [Cites] Sheng Li Xue Bao. 2006 Aug 25;58(4):331-6 [16906333.001]
  • [Cites] Acta Biochim Biophys Sin (Shanghai). 2008 Jul;40(7):625-35 [18604454.001]
  • (PMID = 20127388.001).
  • [ISSN] 1420-9071
  • [Journal-full-title] Cellular and molecular life sciences : CMLS
  • [ISO-abbreviation] Cell. Mol. Life Sci.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 0 / Fatty Acids, Nonesterified; 0 / NDRG2 protein, human; 0 / Ndr2 protein, mouse; 0 / Proteins; 0 / RNA, Messenger; 0 / RNA, Small Interfering; 0 / Tumor Suppressor Proteins
  •  go-up   go-down


33. Golson ML, Loomes KM, Oakey R, Kaestner KH: Ductal malformation and pancreatitis in mice caused by conditional Jag1 deletion. Gastroenterology; 2009 May;136(5):1761-71.e1
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • BACKGROUND & AIMS: Alagille syndrome is an autosomal dominant disorder caused by mutations in Notch signaling pathway genes, usually JAGGED1.
  • Up to 40% of Alagille syndrome patients also display exocrine pancreatic insufficiency, the pathobiology of which is unknown.
  • METHODS: We conditionally deleted both alleles of Jagged1 in the murine pancreas using Cre-loxP technology and analyzed histologic and morphologic features in postnatal and adult pancreas such as duct structure, acinar mass, and T-lymphocyte infiltration, as well as markers of pancreatic function, including fecal fat.
  • RESULTS: Jagged1-deficient mice displayed malformed pancreatic ducts with resulting acinar cell death, fatty infiltration of the parenchyma, fibrosis, pancreatitis, and pancreatic insufficiency.
  • CONCLUSIONS: Pancreatic ductal malformation and acinar cell loss may be responsible for pancreatic insufficiency in Jagged1-deficient mice and, by corollary, in Alagille syndrome patients.
  • [MeSH-major] Calcium-Binding Proteins / genetics. Gene Deletion. Intercellular Signaling Peptides and Proteins / genetics. Membrane Proteins / genetics. Pancreatic Ducts / abnormalities. Pancreatitis / genetics
  • [MeSH-minor] Adipocytes / pathology. Alagille Syndrome / genetics. Animals. Glucose / metabolism. Homeostasis. Mice. Mice, Knockout. Mice, Mutant Strains. Pancreas / metabolism. Pancreas / pathology

  • MedlinePlus Health Information. consumer health - Pancreatitis.
  • COS Scholar Universe. author profiles.
  • Hazardous Substances Data Bank. GLUCOSE .
  • KOMP Repository. gene/protein/disease-specific - KOMP Repository (subscription/membership/fee required).
  • Mouse Genome Informatics (MGI). Mouse Genome Informatics (MGI) .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 19208348.001).
  • [ISSN] 1528-0012
  • [Journal-full-title] Gastroenterology
  • [ISO-abbreviation] Gastroenterology
  • [Language] eng
  • [Grant] United States / NIDDK NIH HHS / DK / DK055342; United States / NIDDK NIH HHS / DK / DK071; United States / NIDDK NIH HHS / DK / P30DK19525; United States / NIDDK NIH HHS / DK / P30DK50306
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Calcium-Binding Proteins; 0 / Intercellular Signaling Peptides and Proteins; 0 / Membrane Proteins; 134324-36-0 / Serrate proteins; IY9XDZ35W2 / Glucose
  •  go-up   go-down


34. Carrara S, Arcidiacono PG, Albarello L, Addis A, Enderle MD, Boemo C, Campagnol M, Ambrosi A, Doglioni C, Testoni PA: Endoscopic ultrasound-guided application of a new hybrid cryotherm probe in porcine pancreas: a preliminary study. Endoscopy; 2008 Apr;40(4):321-6
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Endoscopic ultrasound-guided application of a new hybrid cryotherm probe in porcine pancreas: a preliminary study.
  • BACKGROUND AND STUDY AIMS: Open, laparoscopic, or percutaneous radiofrequency (RF) ablation of the pancreas is still dangerous, whereas endoscopic ultrasound (EUS)-guided ablation might reduce risk because it is less invasive and provides real-time monitoring.
  • CONCLUSIONS: Selective transluminal RF ablation of the pancreas under EUS control in a living pig model is feasible.
  • [MeSH-major] Catheter Ablation / instrumentation. Cryosurgery / instrumentation. Pancreas / surgery. Pancreas / ultrasonography

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 18389449.001).
  • [ISSN] 1438-8812
  • [Journal-full-title] Endoscopy
  • [ISO-abbreviation] Endoscopy
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Germany
  •  go-up   go-down


35. Hubert T, Arnalsteen L, Jany T, Prieur E, Triponez F, Nunes B, Vantyghem MC, Gmyr V, Kerr-Conte J, Proye C, Pattou F: [Technique of pancreatic procurement for pancreatic islet isolation]. Ann Chir; 2005 Jul-Aug;130(6-7):384-90
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Technique of pancreatic procurement for pancreatic islet isolation].
  • [Transliterated title] Technique du prélèvement pancréatique pour l'isolement des îlots de Langerhans.
  • AIM OF THE STUDY: The allograft of pancreatic islets represents a potential alternative to insulin therapy in patients suffering from the most severe forms of Type 1 diabetes.
  • Here we report our experience of pancreatic procurement for isolation and islet allograft.
  • MATERIALS AND METHODS: Pancreata were procured in brain-dead donors.
  • RESULTS: Twenty-nine pancreata were procured and 14 preparations were grafted to 7 patients.
  • CONCLUSION: These preliminary clinical results confirmed that the isolation technique of human islets and the technique of pancreas procurement are mastered by our team.
  • [MeSH-major] Islets of Langerhans Transplantation / methods. Pancreas Transplantation / methods. Tissue and Organ Procurement / methods

  • MedlinePlus Health Information. consumer health - Islet Cell Transplantation.
  • MedlinePlus Health Information. consumer health - Pancreas Transplantation.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16023459.001).
  • [ISSN] 0003-3944
  • [Journal-full-title] Annales de chirurgie
  • [ISO-abbreviation] Ann Chir
  • [Language] fre
  • [Publication-type] English Abstract; Journal Article; Review
  • [Publication-country] France
  • [Chemical-registry-number] 0 / Immunosuppressive Agents
  • [Number-of-references] 16
  •  go-up   go-down


36. Miro JM, Ricart MJ, Trullas JC, Cofan F, Cervera C, Brunet M, Tuset M, Manzardo C, Oppenheimer F, Moreno A: Simultaneous pancreas-kidney transplantation in HIV-infected patients: a case report and literature review. Transplant Proc; 2010 Nov;42(9):3887-91
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Simultaneous pancreas-kidney transplantation in HIV-infected patients: a case report and literature review.
  • In contrast with liver and kidney transplantation, only three simultaneous pancreas-kidney transplants (SPKT) have been reported among HIV-infected patients.
  • The pancreas graft failed at 2 weeks and the patient died at 9 months because of a Pseudomonas aeruginosa infection.
  • The three recipients reported in the literature lived, despite the failure of both the pancreas and kidney grafts in one subject.
  • [MeSH-major] Anti-Retroviral Agents / therapeutic use. HIV Infections / drug therapy. Immunosuppressive Agents / therapeutic use. Kidney Transplantation. Pancreas Transplantation


37. Tracey JY, Moossa AR: Unusual tumours of the pancreas. Surgeon; 2009 Aug;7(4):216-23
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Unusual tumours of the pancreas.
  • All pancreatic masses are not necessarily the dismal pancreatic ductal adenocaricoma (PDA) and do not necessarily deserve a gloomy prognosis or a nihilistic attitude.
  • We review a rarer group of pancreatic lesions and discuss their pathogenesis, diagnosis and treatment.
  • A tumour specific selective surgical approach is recommended.
  • The outcome is dependent on the tumour histology and the biological behaviour.
  • The degree of malignancy is variable and ranges across benign, borderline and malignant entities.
  • BACKGROUND: The advent of more sophisticated and ever widely employed imaging modalities has identified the presence of many unsuspected unusual masses in the pancreas.
  • These lesions display natural histories and biological behaviours distinct from adenocarcinoma of the pancreas (PDA).
  • Though the list is long they include neuroendocrine tumours, cystic tumours, primary pancreatic lymphoma, solid pseudopapillary tumours, connective tissue tumours, metastatic lesions to the pancreas and many others.
  • [MeSH-major] Pancreatic Neoplasms / pathology. Pancreatic Neoplasms / surgery

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 19736888.001).
  • [ISSN] 1479-666X
  • [Journal-full-title] The surgeon : journal of the Royal Colleges of Surgeons of Edinburgh and Ireland
  • [ISO-abbreviation] Surgeon
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Scotland
  • [Number-of-references] 50
  •  go-up   go-down


38. Gaudez F, Meria P, Desgrandchamps F, Roussin F, Roussin O, Teillac P: [Total pancreas sampling and transplantation]. Ann Urol (Paris); 2006 Feb;40(1):39-49
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Total pancreas sampling and transplantation].
  • [Transliterated title] Prélévement et transplantation de pancréas total.
  • Pancreas removal and transplantation consists in three main steps: sampling, preparation of the transplant with reconstruction of vessels and finally transplantation.
  • Sampling requires good anatomical knowledge and perfect synchronization between hepatic surgeons so as to ensure adequate dissection of liver and pancreas vessels: portal vein, splenic, upper mesenteric and hepatic arteries.
  • The preparation of the pancreatic graft consists in reconstructing the unique arterial axis using an iliac arterial fork sutured with upper mesenteric and splenic arteries.
  • It must be done carefully to avoid any risk of pancreas fistula.
  • [MeSH-major] Pancreas / surgery. Pancreas Transplantation / methods. Tissue and Organ Harvesting / methods

  • Genetic Alliance. consumer health - Transplantation.
  • MedlinePlus Health Information. consumer health - Pancreas Transplantation.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16552905.001).
  • [ISSN] 0003-4401
  • [Journal-full-title] Annales d'urologie
  • [ISO-abbreviation] Ann Urol (Paris)
  • [Language] fre
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] France
  •  go-up   go-down


39. Chiang CP, Wu CW, Lee SP, Chung CC, Wang CW, Lee SL, Nieh S, Yin SJ: Expression pattern, ethanol-metabolizing activities, and cellular localization of alcohol and aldehyde dehydrogenases in human pancreas: implications for pathogenesis of alcohol-induced pancreatic injury. Alcohol Clin Exp Res; 2009 Jun;33(6):1059-68
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Expression pattern, ethanol-metabolizing activities, and cellular localization of alcohol and aldehyde dehydrogenases in human pancreas: implications for pathogenesis of alcohol-induced pancreatic injury.
  • The metabolic effect and metabolites contribute to pathogenesis of pancreatic injury.
  • The goal of this study was to determine the functional expressions and cellular localization of ADH and ALDH families in human pancreas.
  • METHODS: Fifty five surgical specimens of normal pancreas as well as 15 samples each for chronic pancreatitis and pancreatic cancer from archival formalin-fixed paraffin-embedded tissue specimens were investigated.
  • The expression of ADH1C appeared to be increased in the activated pancreatic stellate cells in chronic pancreatitis and pancreatic cancer.
  • CONCLUSIONS: Alcohol dehydrogenase and ALDH family members are differentially expressed in the various cell types of pancreas.
  • ADH1C may play an important role in modulation of activation of pancreatic stellate cells.
  • [MeSH-major] Alcohol Dehydrogenase / metabolism. Aldehyde Dehydrogenase / metabolism. Central Nervous System Depressants / metabolism. Ethanol / metabolism. Pancreas / enzymology. Pancreatic Neoplasms / enzymology. Pancreatitis / enzymology

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • MedlinePlus Health Information. consumer health - Pancreatitis.
  • Hazardous Substances Data Bank. ETHANOL .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 19382905.001).
  • [ISSN] 1530-0277
  • [Journal-full-title] Alcoholism, clinical and experimental research
  • [ISO-abbreviation] Alcohol. Clin. Exp. Res.
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Central Nervous System Depressants; 0 / Isoenzymes; 3K9958V90M / Ethanol; EC 1.1.1.1 / ADH1B protein, human; EC 1.1.1.1 / ADH1C protein, human; EC 1.1.1.1 / Alcohol Dehydrogenase; EC 1.2.1.3 / ALDH2 protein, human; EC 1.2.1.3 / Aldehyde Dehydrogenase
  •  go-up   go-down


40. Barreda Bolaños F, Landeo Aliaga I, Pando Huarcaya S, Bayro Peñaloza F: [Pancreatic adenocarcinoma in young patient diagnosed by endoscopic ultrasonography]. Rev Gastroenterol Peru; 2008 Apr-Jun;28(2):162-6
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Pancreatic adenocarcinoma in young patient diagnosed by endoscopic ultrasonography].
  • [Transliterated title] Adenocarcinoma de páncreas en paciente joven diagnosticado por ultrasonografía endoscópica.
  • The Pancreatic adenocarcinoma appears generally in patients with more than 60 years old.
  • The tomografic image showed a mass located in the head of the pancreas with hepatic and ganglionar metastases.
  • It was evaluated by means of endoscopic ultrasonography (USE) and performed a directed fine needle puncture aspiration (PAAF), that obtained a bad differentiated adenocarcinoma from the pancreas.
  • We present the case by the unusual occurrence in the related age group and by the importance of the puncture guided by endoscopic ultrasonography in the diagnosis and handling of this pathology [corrected]
  • [MeSH-major] Adenocarcinoma / ultrasonography. Endosonography. Pancreatic Neoplasms / ultrasonography

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [ErratumIn] Rev Gastroenterol Peru. 2008 Jul-Sep;28(3):293
  • (PMID = 18641779.001).
  • [ISSN] 1022-5129
  • [Journal-full-title] Revista de gastroenterología del Perú : órgano oficial de la Sociedad de Gastroenterología del Perú
  • [ISO-abbreviation] Rev Gastroenterol Peru
  • [Language] spa
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Peru
  •  go-up   go-down


41. Yoshizumi T, Shimada M, Soejima Y, Terashi T, Taketomi A, Maehara Y: Successful pylorus-preserving pancreaticoduodenectomy for a patient with carcinoma of the papilla Vater two years after living donor liver transplantation. Hepatogastroenterology; 2007 Apr-May;54(75):941-3
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • For this reason, recurrent primary disease and/or de novo malignancy regarded as chronic immunosuppressant have been paid a great deal of attention.
  • Even pancreas cancer after liver transplantation is extremely rare and has never been successfully treated.
  • Furthermore, cancer of the papilla Vater, which is less frequent than pancreas cancer after liver transplantation has not been reported as yet.

  • Genetic Alliance. consumer health - Transplantation.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17591098.001).
  • [ISSN] 0172-6390
  • [Journal-full-title] Hepato-gastroenterology
  • [ISO-abbreviation] Hepatogastroenterology
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Greece
  •  go-up   go-down


42. Kampf C, Jansson L: Mast cells accumulate in the renal capsule adjacent to transplanted pancreatic islets in rats. Cell Biol Int; 2006 Dec;30(12):1054-6
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Mast cells accumulate in the renal capsule adjacent to transplanted pancreatic islets in rats.
  • Mast cells are important mediators of normal angiogenesis, and participate in normal would healing, i.e. processes involved in pancreatic islet engraftment.
  • The animals were killed 1 month later, and the kidneys and endogenous pancreas were removed, fixed and embedded in paraffin.
  • Mast cells interspersed between graft endocrine cells were as rare as in the endogenous pancreas.

  • MedlinePlus Health Information. consumer health - Islet Cell Transplantation.
  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17074512.001).
  • [ISSN] 1065-6995
  • [Journal-full-title] Cell biology international
  • [ISO-abbreviation] Cell Biol. Int.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  •  go-up   go-down


43. Demirjian AN, Vollmer CM, McDermott DF, Mullen JT, Atkins MB, Callery MP: Refining indications for contemporary surgical treatment of renal cell carcinoma metastatic to the pancreas. HPB (Oxford); 2009 Mar;11(2):150-3
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Refining indications for contemporary surgical treatment of renal cell carcinoma metastatic to the pancreas.
  • BACKGROUND: The pancreas is a rare location for metastatic disease, with only 2-11% of all pancreatic tumours being of non-primary origin.
  • It is also uncommon for renal cell carcinoma (RCC) to metastasize to the pancreas (1-3% of cases) and, when it does, it typically occurs substantially after index nephrectomy.
  • It is not known whether all pancreatic metastases need be resected because today's chemo- and biological therapies are increasingly effective in controlling advanced disease.
  • CONCLUSIONS: Renal cell carcinoma metastases to the pancreas typically occur long after index nephrectomy.

  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Jpn J Clin Oncol. 2004 Nov;34(11):696-9 [15613561.001]
  • [Cites] Mayo Clin Proc. 2000 Jun;75(6):581-5 [10852418.001]
  • [Cites] Br J Surg. 2003 May;90(5):555-9 [12734861.001]
  • [Cites] Ann Surg Oncol. 2008 Apr;15(4):1161-8 [18196343.001]
  • [Cites] Am Surg. 2007 Nov;73(11):1158-60 [18092654.001]
  • [Cites] Br J Cancer. 2006 Mar 13;94(5):614-9 [16465192.001]
  • [Cites] Ann Surg Oncol. 2006 Jan;13(1):75-85 [16372157.001]
  • [Cites] Int J Urol. 2005 Nov;12(11):994-5 [16351657.001]
  • [Cites] J Clin Oncol. 2006 Jan 1;24(1):16-24 [16330672.001]
  • [Cites] N Engl J Med. 2005 Dec 8;353(23):2477-90 [16339096.001]
  • [Cites] Cancer Treat Rev. 2005 Nov;31(7):536-45 [16236454.001]
  • [Cites] J Clin Oncol. 2005 Nov 1;23(31):7889-96 [16204015.001]
  • [Cites] J Gastrointest Surg. 2005 Sep-Oct;9(7):934-7 [16137587.001]
  • [Cites] JOP. 2005 Jul;6(4):339-43 [16006684.001]
  • [Cites] JOP. 2005 Jan;6(1):26-8 [15650281.001]
  • [Cites] Ann Surg Oncol. 2002 Aug;9(7):675-9 [12167582.001]
  • [Cites] Urology. 2000 Aug 1;56(2):211-5 [10925080.001]
  • [Cites] Ann Surg Oncol. 2003 Oct;10(8):922-6 [14527912.001]
  • (PMID = 19590640.001).
  • [ISSN] 1365-182X
  • [Journal-full-title] HPB : the official journal of the International Hepato Pancreato Biliary Association
  • [ISO-abbreviation] HPB (Oxford)
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC2697876
  • [Keywords] NOTNLM ; operative palliatively / pancreatic metastases / renal cell carcinoma
  •  go-up   go-down


44. Lai HW, Su CH, Li AF, Wu LH, Shyr YM, Chen TH, Wu CW, Lui WY: Malignant solid and pseudopapillary tumor of the pancreas--clinicohistological, immunohistochemical, and flow cytometric evaluation. Hepatogastroenterology; 2006 Mar-Apr;53(68):291-5
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Malignant solid and pseudopapillary tumor of the pancreas--clinicohistological, immunohistochemical, and flow cytometric evaluation.
  • BACKGROUND/AIMS: Solid and pseudopapillary tumor of the pancreas is a benign and low malignant potential tumor.
  • We compared benign and malignant cases with clinicopathological, immunohistochemical and DNA flow cytometric studies.
  • METHODOLOGY: From January 1991 to July 2004, seven patients were found to have solid and pseudopapillary tumor of the pancreas at Taipei Veterans General Hospital.
  • RESULTS: It included 6 benign and one malignant case.
  • In the malignant patient, the tumor in the first operation showed diploid result, but tumors in second and third operations showed aneuploidy.
  • CONCLUSIONS: Solid and pseudopapillary tumor of the pancreas should be considered as a potentially malignant disease in all patients and regular follow-up is mandatory.
  • [MeSH-major] Cystadenoma, Papillary. Pancreatic Neoplasms
  • [MeSH-minor] Adolescent. Adult. Biomarkers, Tumor / metabolism. Child. Female. Humans. Male. Phosphopyruvate Hydratase / metabolism. Ploidies. Receptors, Steroid / metabolism. Tumor Suppressor Protein p53 / metabolism. Vimentin / metabolism. alpha 1-Antitrypsin / metabolism

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • COS Scholar Universe. author profiles.
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16608042.001).
  • [ISSN] 0172-6390
  • [Journal-full-title] Hepato-gastroenterology
  • [ISO-abbreviation] Hepatogastroenterology
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Greece
  • [Chemical-registry-number] 0 / Biomarkers, Tumor; 0 / Receptors, Steroid; 0 / SERPINA1 protein, human; 0 / Tumor Suppressor Protein p53; 0 / Vimentin; 0 / alpha 1-Antitrypsin; EC 4.2.1.11 / Phosphopyruvate Hydratase
  •  go-up   go-down


45. Harries LW, Ellard S, Stride A, Morgan NG, Hattersley AT: Isomers of the TCF1 gene encoding hepatocyte nuclear factor-1 alpha show differential expression in the pancreas and define the relationship between mutation position and clinical phenotype in monogenic diabetes. Hum Mol Genet; 2006 Jul 15;15(14):2216-24
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Isomers of the TCF1 gene encoding hepatocyte nuclear factor-1 alpha show differential expression in the pancreas and define the relationship between mutation position and clinical phenotype in monogenic diabetes.
  • Heterozygous mutations in the transcription factor hepatocyte nuclear factor-1 alpha (HNF1A or TCF1 gene) result in early-onset diabetes as a result of pancreatic beta-cell dysfunction.
  • Real-time PCR demonstrated variation in tissue expression of HNF1A isomers: HNF1A(A), with the lowest transactivation activity compared with the truncated isoforms HNF1A(B) and HNF1A(C), is the major isomer in liver (54%) and kidney (67%) but not in adult pancreas (24%) and islets (26%).
  • However, in fetal pancreas HNF1A(A) is the major transcript (84%), which supports developmental regulation of isomer expression.
  • [MeSH-major] Diabetes Mellitus, Type 2 / genetics. Diabetes Mellitus, Type 2 / metabolism. Hepatocyte Nuclear Factor 1-alpha / genetics. Mutation. Pancreas / metabolism

  • Genetic Alliance. consumer health - Diabetes.
  • Genetic Alliance. consumer health - Monogenic diabetes.
  • MedlinePlus Health Information. consumer health - Diabetes Type 2.
  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16760222.001).
  • [ISSN] 0964-6906
  • [Journal-full-title] Human molecular genetics
  • [ISO-abbreviation] Hum. Mol. Genet.
  • [Language] eng
  • [Grant] United Kingdom / Wellcome Trust / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / HNF1A protein, human; 0 / Hepatocyte Nuclear Factor 1-alpha; 0 / Protein Isoforms; 0 / RNA, Messenger
  •  go-up   go-down


46. Porrett PM, Yeh H, Frank A, Deng S, Kim JI, Barker CF, Markmann JF: Availability of suitable islet donors in the United States. Transplantation; 2007 Jul 27;84(2):280-2
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • RESULTS: Of 27,552 potential donors during this period, 6,140 donor pancreata were used for whole organ transplant.

  • MedlinePlus Health Information. consumer health - Islet Cell Transplantation.
  • MedlinePlus Health Information. consumer health - Organ Donation.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17667824.001).
  • [ISSN] 0041-1337
  • [Journal-full-title] Transplantation
  • [ISO-abbreviation] Transplantation
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  •  go-up   go-down


47. Tuveson DA, Zhu L, Gopinathan A, Willis NA, Kachatrian L, Grochow R, Pin CL, Mitin NY, Taparowsky EJ, Gimotty PA, Hruban RH, Jacks T, Konieczny SF: Mist1-KrasG12D knock-in mice develop mixed differentiation metastatic exocrine pancreatic carcinoma and hepatocellular carcinoma. Cancer Res; 2006 Jan 1;66(1):242-7
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Mist1-KrasG12D knock-in mice develop mixed differentiation metastatic exocrine pancreatic carcinoma and hepatocellular carcinoma.
  • Despite the prevalence of oncogenic Kras mutations in the earliest stages of pancreatic ductal adenocarcinoma, the cellular compartment in which oncogenic Kras initiates tumorigenesis remains unknown.
  • To address this, we have gene targeted KrasG12D into the open reading frame of Mist1, a basic helix-loop-helix transcription factor that is expressed during pancreatic development and required for proper pancreatic acinar organization.
  • Although the pancreata of Mist1(KrasG12D/+) mutant mice predictably exhibited acinar metaplasia and dysplasia, the frequent death of these mice from invasive and metastatic pancreatic cancer with mixed histologic characteristics, including acinar, cystic, and ductal features, was unexpected and in contrast to previously described mutant mice that ectopically expressed the Kras oncogene in either acinar or ductal compartments.
  • Interestingly, many of the mutant mice developed hepatocellular carcinoma, implicating Mist1(KrasG12D/+) cells in both pancreatic and hepatic neoplasia.
  • These findings suggest that Mist1-expressing cells represent a permissive compartment for transformation by oncogenic Kras in pancreatic tumorigenesis.


48. Saber AA, Helbling B, Khaghany K, Nirmit G, Pimental R, McLeod MK: Safety zone for splenic hilar control during splenectomy: a computed tomography scan mapping of the tail of the pancreas in relation to the splenic hilum. Am Surg; 2007 Sep;73(9):890-4
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Safety zone for splenic hilar control during splenectomy: a computed tomography scan mapping of the tail of the pancreas in relation to the splenic hilum.
  • Inadvertent injury to the tail of the pancreas is a potentially serious but preventable complication that can occur during laparoscopic splenectomy.
  • The aim of this study was to determine the feasibility of using computed tomography to map the location of the tail of the pancreas relative to the spleen to locate a possible safe zone for splenic hilar dissection and/or hemostasis.
  • The distance from the tail of the pancreas to the hilum of the spleen was determined for each patient.
  • Computed tomography was successful in mapping the distance from the tail of the pancreas to the splenic hilum in 148 patients.
  • The average distance from the tail of the pancreas to the splenic hilum was 3.42 cm +/- 1.54 cm (95% confidence interval, 3.17-3.67).
  • During splenic vascular control, it is important to stay within 1 cm from the splenic hilum to minimize the risk of injury to the tail of the pancreas during splenectomy.
  • [MeSH-major] Laparoscopy. Pancreas / radiography. Spleen / radiography. Splenectomy / methods. Tomography, X-Ray Computed

  • MedlinePlus Health Information. consumer health - CT Scans.
  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17939420.001).
  • [ISSN] 0003-1348
  • [Journal-full-title] The American surgeon
  • [ISO-abbreviation] Am Surg
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Contrast Media
  •  go-up   go-down


49. Ku SK, Lee HS: Distribution and frequency of endocrine cells in the pancreas of the ddY mouse: an immunohistochemical study. Eur J Histochem; 2005 Apr-Jun;49(2):125-30
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Distribution and frequency of endocrine cells in the pancreas of the ddY mouse: an immunohistochemical study.
  • The regional distribution and frequency of pancreatic endocrine cells in ddY mice were studied by an immunohistochemical (peroxidase anti-peroxidase;.
  • PAP) method using four types of specific antisera against insulin, glucagon, somatostatin and human pancreatic polypeptide (hPP).
  • In the pancreatic islets, most of insulin-immunoreactive (IR) cells were located in the central portion.
  • In the exocrine pancreas, insulin-, glucagon-, somatostatin- and hPP-IR cells were detected; they occurred mainly among the exocrine parenchyma as solitary cells.
  • Cell clusters consisted of only insulin- or only glucagon-IR cells and were distributed in the pancreas parenchyma as small islets.
  • In addition, insulin- and glucagon-IR cells were also demonstrated in the pancreatic duct regions.
  • In conclusion, some strain-dependent characteristic distributional patterns of pancreatic endocrine cells were found in the ddY mouse.
  • [MeSH-major] Endocrine System / cytology. Pancreas / cytology

  • Hazardous Substances Data Bank. GLUCAGON .
  • SciCrunch. The Antibody Registry: Reagent: Antibodies .
  • antibodies-online. View related products from antibodies-online.com (subscription/membership/fee required).
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 15967740.001).
  • [ISSN] 1121-760X
  • [Journal-full-title] European journal of histochemistry : EJH
  • [ISO-abbreviation] Eur J Histochem
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Italy
  • [Chemical-registry-number] 0 / Biomarkers; 0 / Insulin; 9007-92-5 / Glucagon
  •  go-up   go-down


50. Lee KY, Ahn HC, Kim C, Kim SH, Kim DK, Park HS: Pancreatic exocrine response to long-term high-fat diets in rats. JOP; 2006;7(4):397-404
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Pancreatic exocrine response to long-term high-fat diets in rats.
  • CONTEXT: Although the synthesis and secretion of pancreatic enzymes are dependent on the composition of diet, little is known about the long-term adaptation of the exocrine pancreas to the chronic intake of high-fat, low-carbohydrate diets.
  • OBJECTIVE: The effects of long-term (48-weeks) high-fat, low-carbohydrate feeding on the intracellular activity as well as on the secretory activities of the pancreas regarding the secretion of amylase and lipase in isolated pancreatic lobules of rats were investigated.
  • MAIN OUTCOME MEASURES: The intracellular activity of alpha-amylase and lipase were defined as enzyme activity per 1 mg protein in the pancreatic homogenate.
  • The secretory activity of the pancreatic enzymes was defined as enzyme activity per 1 mg protein or as the mean percentage of released enzyme activity in the medium to that initially retained in the lobules over the entire 90 minute incubation period.
  • RESULTS: The intracellular activity as well as the secretory activities of pancreatic amylase in the basal and in the cholecystokinin (10 pmol/L)-stimulated states of high-fat, low-carbohydrate diet fed rats were significantly lower than that in the control rats.
  • CONCLUSIONS: The reduction of the intracellular amylase activity and the secretory amylase activity may be a long-term adaptive response of the pancreas to the reduced carbohydrate composition of the diets.
  • However, we failed to observe a long-term adaptive response of pancreatic lipase to increased fat composition of diets.
  • [MeSH-major] Amylases / metabolism. Dietary Fats / pharmacology. Pancreas, Exocrine / drug effects. Pancreas, Exocrine / enzymology

  • MedlinePlus Health Information. consumer health - Dietary Fats.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16832137.001).
  • [ISSN] 1590-8577
  • [Journal-full-title] JOP : Journal of the pancreas
  • [ISO-abbreviation] JOP
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Italy
  • [Chemical-registry-number] 0 / Dietary Fats; 9011-97-6 / Cholecystokinin; EC 3.1.1.3 / Lipase; EC 3.2.1.- / Amylases
  •  go-up   go-down


51. Dib MJ, Ho KJ, Hanto DW, Karp SJ, Johnson SR: Roux limb volvulus after pancreas transplantation: an unusual cause of pancreatic graft loss. Clin Transplant; 2009 Nov-Dec;23(6):807-11
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Roux limb volvulus after pancreas transplantation: an unusual cause of pancreatic graft loss.
  • Pancreas transplantation with enteric drainage avoids the long-term urological complications of bladder drainage.
  • This report describes a patient five yr after left-sided pancreas transplant with Roux-en-Y enteric drainage, presenting with abdominal pain, leukocytosis and radiological evidence of bowel obstruction.
  • Exploration revealed a volvulus of the Roux limb as it passed through the mesocolon, with necrosis of the allograft duodenum and marked congestion of the pancreas.
  • This is the first report of pancreas graft loss due to this entity, which should be recognized as an unusual cause of abdominal pain after pancreas transplantation.
  • Potential bowel complications related to the sigmoid mesentery in left-sided pancreas transplantation are additional reasons for right-sided placement of the pancreas allograft.
  • [MeSH-major] Graft Rejection / etiology. Intestinal Volvulus / complications. Pancreas Transplantation / adverse effects

  • Genetic Alliance. consumer health - Transplantation.
  • MedlinePlus Health Information. consumer health - Pancreas Transplantation.
  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 19191816.001).
  • [ISSN] 1399-0012
  • [Journal-full-title] Clinical transplantation
  • [ISO-abbreviation] Clin Transplant
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Denmark
  •  go-up   go-down


52. Kodama S, Davis M, Faustman DL: Diabetes and stem cell researchers turn to the lowly spleen. Sci Aging Knowledge Environ; 2005 Jan 19;2005(3):pe2
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Long seen as superfluous, the adult spleen of the mouse has recently been shown to hold stem cells that, in diabetic mice or genetically altered mice that lack a pancreas, effectively regenerate insulin-producing islet cells of the pancreas.
  • [MeSH-minor] Aging. Animals. Cell Lineage. Homeodomain Proteins / metabolism. Islets of Langerhans / cytology. Islets of Langerhans Transplantation. Mice. Pancreas / cytology. Pancreas / embryology. RNA, Messenger / metabolism. Stem Cells / cytology. Stem Cells / metabolism

  • MedlinePlus Health Information. consumer health - Diabetes.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 15659719.001).
  • [ISSN] 1539-6150
  • [Journal-full-title] Science of aging knowledge environment : SAGE KE
  • [ISO-abbreviation] Sci Aging Knowledge Environ
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Homeodomain Proteins; 0 / RNA, Messenger; 0 / Tlx1 protein, mouse
  • [Number-of-references] 61
  •  go-up   go-down


53. Oikarinen M, Tauriainen S, Honkanen T, Vuori K, Karhunen P, Vasama-Nolvi C, Oikarinen S, Verbeke C, Blair GE, Rantala I, Ilonen J, Simell O, Knip M, Hyöty H: Analysis of pancreas tissue in a child positive for islet cell antibodies. Diabetologia; 2008 Oct;51(10):1796-802
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Analysis of pancreas tissue in a child positive for islet cell antibodies.
  • AIMS/HYPOTHESIS: Type 1 diabetes is caused by an immune-mediated process, reflected by the appearance of autoantibodies against pancreatic islets in the peripheral circulation.
  • The present study assesses whether positivity for a single autoantibody correlates with pathological changes in the pancreas.
  • METHODS: We studied post mortem pancreatic tissue of a child who repeatedly tested positive for islet cell antibodies (ICA) in serial measurements.
  • RESULTS: The structure of the pancreas did not differ from normal.
  • Beta-2-microglobulin and CAR were strongly produced in the islets, but not in the exocrine pancreas.
  • However, it is most likely that the pancreatic islets were infected by an enterovirus in this child.
  • [MeSH-major] Autoantibodies / immunology. Islets of Langerhans / immunology. Pancreas / immunology

  • COS Scholar Universe. author profiles.
  • Hazardous Substances Data Bank. GLUCAGON .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • The Lens. Cited by Patents in .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Lancet. 1974 Nov 30;2(7892):1279-83 [4139522.001]
  • [Cites] Ann Med. 2002;34(3):138-47 [12173683.001]
  • [Cites] Diabetes. 2007 Sep;56(9):2400-4 [17563060.001]
  • [Cites] Acta Paediatr. 2003 Dec;92(12):1403-10 [14971790.001]
  • [Cites] Diabetologia. 1991 Jul;34(7):534-5 [1916061.001]
  • [Cites] APMIS. 1996 Mar;104(3):161-7 [8611188.001]
  • [Cites] Diabetologia. 1990 May;33(5):290-8 [2376300.001]
  • [Cites] Diabetologia. 1997 Jan;40(1):53-61 [9028718.001]
  • [Cites] J Med Virol. 1998 Mar;54(3):204-9 [9515770.001]
  • [Cites] Mol Cell Probes. 1991 Feb;5(1):11-20 [1850115.001]
  • [Cites] Br J Haematol. 2002 Jul;118(1):195-201 [12100148.001]
  • [Cites] J Clin Endocrinol Metab. 2005 May;90(5):2712-7 [15872335.001]
  • [Cites] Virology. 2000 Jun 5;271(2):276-88 [10860882.001]
  • [Cites] Diabetologia. 2005 Aug;48(8):1560-4 [15991022.001]
  • [Cites] Proc Natl Acad Sci U S A. 2007 Mar 20;104(12):5115-20 [17360338.001]
  • [Cites] Diabetes. 2008 Jun;57(6):1584-94 [18334605.001]
  • [Cites] J Infect Dis. 1997 Mar;175(3):693-6 [9041346.001]
  • [Cites] J Med Virol. 2000 Nov;62(3):383-91 [11055249.001]
  • [Cites] Diabetes. 2003 Jan;52(1):102-10 [12502499.001]
  • [Cites] J Med Virol. 1999 Nov;59(3):378-84 [10502272.001]
  • [Cites] Diabetologia. 2004 Feb;47(2):225-39 [14727023.001]
  • [Cites] Gene Ther. 1999 Sep;6(9):1512-9 [10490760.001]
  • [Cites] N Engl J Med. 1979 May 24;300(21):1173-9 [219345.001]
  • [Cites] Diabetes Metab Rev. 1998 Mar;14(1):31-67 [9605629.001]
  • [Cites] N Engl J Med. 1994 Nov 24;331(21):1428-36 [7969282.001]
  • [Cites] Clin Exp Immunol. 2008 Jan;151(1):71-5 [17991291.001]
  • [Cites] Intervirology. 1987;28(3):163-70 [2836335.001]
  • (PMID = 18696046.001).
  • [ISSN] 0012-186X
  • [Journal-full-title] Diabetologia
  • [ISO-abbreviation] Diabetologia
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / Antibodies, Viral; 0 / Antigens, CD; 0 / Antigens, CD3; 0 / Antigens, Differentiation, Myelomonocytic; 0 / Autoantibodies; 0 / CD68 antigen, human; 0 / Insulin; 0 / Interferon-alpha; 0 / Receptors, Virus; 0 / adenovirus receptor; 0 / islet cell antibody; 51110-01-1 / Somatostatin; 9007-92-5 / Glucagon; EC 1.14.99.1 / Cyclooxygenase 2
  •  go-up   go-down


54. Grubbs EG, Abdel-Wahab Z, Tyler DS, Pruitt SK: Utilizing quantitative polymerase chain reaction to evaluate prostate stem cell antigen as a tumor marker in pancreatic cancer. Ann Surg Oncol; 2006 Dec;13(12):1645-54
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Utilizing quantitative polymerase chain reaction to evaluate prostate stem cell antigen as a tumor marker in pancreatic cancer.
  • BACKGROUND: Real-time quantitative polymerase chain reaction (qPCR) may prove to be a sensitive technique by which to evaluate potential tumor markers in pancreatic cancer.
  • METHODS: The prostate stem cell antigen (PSCA) gene was identified as a marker highly expressed in pancreatic adenocarcinoma and not normal pancreas.
  • RNA from pancreatic and nonpancreatic cancer cell lines as well as tissue and blood from pancreatic cancer and control patients was reverse-transcribed and PSCA quantified by qPCR.
  • Five of six pancreatic cell lines had PSCA/actin ratios 10-fold greater than nonpancreatic cancer lines.
  • Mean PSCA expression in pancreatic tumor tissue was significantly higher (P < 0.05, Student's t-test) than in the tissue of benign pancreatic processes.
  • The close correlation of PSCA/actin copy number with number of tumor cells in the blood was demonstrated by regression analysis (r = 0.768, P = 0.0001).
  • PSCA copy number was significantly higher in the blood of patients with metastatic pancreatic cancer than in that of normal patients (P < 0.05, Student's t-test).
  • CONCLUSIONS: Such trends suggest that PSCA may prove to be a valuable pancreatic cancer tumor marker.
  • [MeSH-major] Adenocarcinoma / genetics. Biomarkers, Tumor / genetics. Membrane Glycoproteins / genetics. Neoplasm Proteins / genetics. Pancreatic Neoplasms / genetics
  • [MeSH-minor] Antigens, Neoplasm. Carcinoma, Pancreatic Ductal / genetics. Carcinoma, Pancreatic Ductal / metabolism. Carcinoma, Pancreatic Ductal / pathology. Cell Line, Tumor. GPI-Linked Proteins. Gene Dosage. Humans. Pancreas / metabolism. Pancreas / pathology. Pancreatic Cyst / genetics. Pancreatic Cyst / metabolism. Prognosis. RNA, Messenger / genetics. RNA, Messenger / metabolism. Reverse Transcriptase Polymerase Chain Reaction. Sensitivity and Specificity

  • Genetic Alliance. consumer health - Pancreatic cancer.
  • Genetic Alliance. consumer health - Prostate cancer.
  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16957968.001).
  • [ISSN] 1068-9265
  • [Journal-full-title] Annals of surgical oncology
  • [ISO-abbreviation] Ann. Surg. Oncol.
  • [Language] eng
  • [Grant] United States / NCI NIH HHS / CA / F32 CA 94639; United States / NCI NIH HHS / CA / K08 CA74241
  • [Publication-type] Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antigens, Neoplasm; 0 / Biomarkers, Tumor; 0 / GPI-Linked Proteins; 0 / Membrane Glycoproteins; 0 / Neoplasm Proteins; 0 / PSCA protein, human; 0 / RNA, Messenger
  •  go-up   go-down


55. Cosentini V, Angione V, Lo Cicero A, Ferraro-Mortellaro R, Barillà A, Floccari F: [Recurrent multifocal cancer lesions in a patient on hemodialysis after kidney pancreas transplant failure]. G Ital Nefrol; 2010 May-Jun;27(3):301-5
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Recurrent multifocal cancer lesions in a patient on hemodialysis after kidney pancreas transplant failure].
  • [Transliterated title] Lesioni neoplastiche multifocali e recidivanti in un paziente in emodialisi dopo fallimento di trapianto combinato rene-pancreas.
  • A 59-year-old man, ex-smoker, who had been on hemodialysis for seven months because of uremic degeneration of diabetic nephropathy, underwent a combined kidney-pancreas transplant in 1991, complicated by slow-resolution CMV infection.
  • In 1993, after kidney graft failure due to chronic rejection, hemodialysis treatment was restarted with good pancreatic function.
  • [MeSH-major] Carcinoma, Squamous Cell. Kidney Transplantation. Mouth Neoplasms. Neoplasm Recurrence, Local. Pancreas Transplantation. Renal Dialysis. Skin Neoplasms


56. Oida T, Mimastu K, Kawasaki A, Kuboi Y, Kano H, Amano S: Toward zero pancreatic leakage after pancreaticoduodenectomy for soft pancreas in low-volume pancreatic surgery centers. Hepatogastroenterology; 2009 May-Jun;56(91-92):886-90
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Toward zero pancreatic leakage after pancreaticoduodenectomy for soft pancreas in low-volume pancreatic surgery centers.
  • Pancreatic leakage continues to occur after pancreaticoduodenectomy (PD) and it is a serious problem.
  • It has been proposed that a normal-sized, soft, friable, and functionally normal pancreas with a thin-walled main pancreatic duct is at a greater risk of anastomotic leakage.
  • However, despite the evaluation of various improvised procedures, pancreatic leakage persists as a complication.
  • Here, we describe our PD technique that facilitates the prevention of pancreatic leakage.
  • Our operative procedures includes 3 stages: pancreaticogastrostomy (PG), external and continuous suction drainage of pancreatic juice, and wrapping of the pancreatic anastomosis by using round ligament and applying fibrin glue sealant to the surface of the pancreatic anastomosis.
  • Twenty-six patients with periampullary malignancy with soft pancreas underwent PG without any pancreatic leakage or fistula.
  • The combined procedures, including PG, total external drainage with continuous suction of the pancreatic juice, and wrapping of the pancreatic anastomotic site by using round ligament is a useful method to prevent pancreatic leakage for cases of soft pancreas in pancreatic centers that do not encounter many such cases.
  • [MeSH-major] Ampulla of Vater. Bile Duct Neoplasms / surgery. Pancreatic Neoplasms / surgery. Pancreaticoduodenectomy / methods

  • MedlinePlus Health Information. consumer health - Bile Duct Cancer.
  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 19621723.001).
  • [ISSN] 0172-6390
  • [Journal-full-title] Hepato-gastroenterology
  • [ISO-abbreviation] Hepatogastroenterology
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Greece
  •  go-up   go-down


57. Mori Y, Ohtsuka T, Tsutsumi K, Yasui T, Sadakari Y, Ueda J, Takahata S, Nakamura M, Tanaka M: Multifocal pancreatic ductal adenocarcinomas concomitant with intraductal papillary mucinous neoplasms of the pancreas detected by intraoperative pancreatic juice cytology. A case report. JOP; 2010;11(4):389-92
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Multifocal pancreatic ductal adenocarcinomas concomitant with intraductal papillary mucinous neoplasms of the pancreas detected by intraoperative pancreatic juice cytology. A case report.
  • CONTEXT: Intraductal papillary mucinous neoplasms (IPMNs) of the pancreas have been detected with increasing frequency as a result of the progression of diagnostic modalities.
  • Recently, invasive ductal carcinoma of the pancreas concomitant with IPMNs has been the focus of attention.
  • CASE REPORT: We report the case of a 57-year-old man with multifocal ductal carcinomas of the pancreas concomitant with IPMNs detected by intraoperative cytology.
  • During a follow-up for branch duct IPMNs, a stenotic lesion of the main duct in the pancreatic body was found by ERCP, and brush cytology of the stenosis revealed an adenocarcinoma.
  • A distal pancreatectomy was proposed; however, intraoperative pancreatic juice cytology from the pancreatic head also revealed adenocarcinoma, and a total pancreatectomy was finally carried out.
  • Pathological examination of the resected specimen showed multifocal ductal carcinomas and IPMNs in the distal pancreas, and invasive ductal carcinoma in the pancreatic head which had not been detected by preoperative imaging studies.
  • Intraoperative pancreatic juice cytology should always be performed in order to confirm the absence of carcinoma in the pancreas to be left in place after planned resection.
  • [MeSH-major] Adenocarcinoma, Mucinous / diagnosis. Carcinoma, Pancreatic Ductal / diagnosis. Carcinoma, Papillary / diagnosis. Neoplasms, Multiple Primary / diagnosis. Pancreatic Juice / cytology. Pancreatic Neoplasms / diagnosis

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 20601817.001).
  • [ISSN] 1590-8577
  • [Journal-full-title] JOP : Journal of the pancreas
  • [ISO-abbreviation] JOP
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Italy
  •  go-up   go-down


58. Davenport C, Hamid N, O'Sullivan EP, Daly P, Mohan P, Little D, Thompson CJ, Agha A, Hickey D, Smith D: The impact of pancreas and kidney transplant on cardiovascular risk factors (analyzed by mode of immunosuppression and exocrine drainage). Clin Transplant; 2009 Sep-Oct;23(5):616-20
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The impact of pancreas and kidney transplant on cardiovascular risk factors (analyzed by mode of immunosuppression and exocrine drainage).
  • INTRODUCTION: The aim of this study was to determine the cardiovascular (CV) risk factor response in Irish patients with type 1 diabetes following simultaneous pancreas and kidney transplantation (SPK), analyzing response based on mode of immunosuppression and surgical drainage in a uniquely homogenous population.
  • METHODS: A retrospective review of SPKs carried out between 1993 and 2005 in the National Renal and Pancreatic Centre of Ireland was performed.
  • [MeSH-major] Cardiovascular Diseases / etiology. Diabetes Mellitus, Type 1 / surgery. Graft Rejection / prevention & control. Kidney Failure, Chronic / surgery. Kidney Transplantation. Pancreas Transplantation. Postoperative Complications


59. Komar G, Kauhanen S, Liukko K, Seppänen M, Kajander S, Ovaska J, Nuutila P, Minn H: Decreased blood flow with increased metabolic activity: a novel sign of pancreatic tumor aggressiveness. Clin Cancer Res; 2009 Sep 1;15(17):5511-7
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Decreased blood flow with increased metabolic activity: a novel sign of pancreatic tumor aggressiveness.
  • PURPOSE: To study blood flow (BF) and metabolism in normal pancreas and in different pancreatic lesions.
  • We then determined the effect of these biomarkers on outcome in patients with pancreatic cancer.
  • EXPERIMENTAL DESIGN: Oxygen-15-labeled water and fluorodeoxyglucose positron emission tomography/computed tomography scans were used in 26 patients with a suspicion of pancreatic cancer to measure pancreatic BF and metabolism.
  • Patients were divided into three groups: patients with a finding of normal pancreas (n = 7), benign lesions (n = 8), and malignant tumors (n = 11).
  • RESULTS: Patients with benign and malignant pancreatic tumors had decreased BF of the lesion by 48% and 60%, respectively, compared with patients with normal pancreatic tissue.
  • SUV(max) was 3-fold higher in malignant tumors compared with both benign lesions and normal pancreas (P < 0.05).
  • In contrast, the SUV(max) of patients with benign lesions and normal pancreas did not differ.
  • The SUV/BF ratio was significantly higher in malignant lesions than in benign lesions or in patients with normal pancreas (P < 0.05).
  • In patients with cancer, high SUV/BF ratio was a stronger predictor of poor survival compared with high metabolism or lower-than-normal pancreatic BF.
  • CONCLUSIONS: BF in pancreatic cancer is significantly reduced compared with the normal pancreas, which may in part explain the poor success of both radiotherapy and chemotherapy.
  • We suggest that the composite measurement of BF and metabolism in pancreatic cancer could serve as a novel tool in the planning of treatments targeting vasculature.
  • [MeSH-major] Biomarkers, Tumor / metabolism. Glucose / metabolism. Pancreatic Neoplasms / pathology. Regional Blood Flow
  • [MeSH-minor] Aged. Female. Fluorodeoxyglucose F18 / metabolism. Humans. Male. Middle Aged. Neoplasm Invasiveness. Pancreas / blood supply. Pancreas / metabolism. Pancreas / pathology. Positron-Emission Tomography. Prognosis

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • Hazardous Substances Data Bank. GLUCOSE .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [CommentIn] Clin Cancer Res. 2010 Jan 1;16(1):367; author reply 567 [20028758.001]
  • [CommentIn] Clin Cancer Res. 2009 Sep 1;15(17):5294-6 [19706819.001]
  • [CommentIn] Future Oncol. 2010 Jan;6(1):13-5 [20021204.001]
  • (PMID = 19706808.001).
  • [ISSN] 1078-0432
  • [Journal-full-title] Clinical cancer research : an official journal of the American Association for Cancer Research
  • [ISO-abbreviation] Clin. Cancer Res.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Biomarkers, Tumor; 0Z5B2CJX4D / Fluorodeoxyglucose F18; IY9XDZ35W2 / Glucose
  •  go-up   go-down


60. Monstein HJ, Grahn N, Ohlsson B: Proenkephalin-A mRNA is widely expressed in tissues of the human gastrointestinal tract. Eur Surg Res; 2006;38(5):464-8
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • RESULTS: The present study demonstrates proenkephalin-A mRNA expression in the human esophagus, gastrointestinal tract, pancreas, and gallbladder.
  • CONCLUSION: The present study demonstrates proenkephalin-A mRNA expression in regions of the human esophagus, gastrointestinal tract, pancreas, and gallbladder tissues which provides information for the future mapping of proenkephalin-A mRNA and protein expression/co-expression at the cellular level.
  • [MeSH-minor] Aged. Aged, 80 and over. Female. Gallbladder / metabolism. Gene Expression. Humans. Male. Middle Aged. Pancreas / metabolism. RNA, Messenger / metabolism. Reverse Transcriptase Polymerase Chain Reaction

  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] Copyright (c) 2006 S. Karger AG, Basel.
  • (PMID = 16940731.001).
  • [ISSN] 0014-312X
  • [Journal-full-title] European surgical research. Europäische chirurgische Forschung. Recherches chirurgicales européennes
  • [ISO-abbreviation] Eur Surg Res
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 0 / Enkephalins; 0 / Protein Precursors; 0 / RNA, Messenger; 0 / proenkephalin
  •  go-up   go-down


61. Magno L, Sivero L, Napolitano V, Ruggiero S, Fontanarosa G, Massa S: [The role of endoscopy in gastroenteropancreatic neuroendocrine tumors]. G Chir; 2010 Jun-Jul;31(6-7):341-3
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Massa I tumori neuroendocrini (NET) gastro-entero-pancreatici (GEP) sono neoplasie rare che originano dalle cellule neuroendocrine del tubo digerente e del pancreas.
  • Massa Gastroenteropancreatic (GEP) neuroendocrine tumors (NET) are rare neoplasia arisen from neuroendocrine cells present in the gut mucosa and pancreas.
  • Digestive endoscopy and endoscopic ultrasonography play a relevant role in NET diagnosis, stadiation and surveillance.
  • [MeSH-major] Endoscopy, Gastrointestinal. Endosonography. Gastrointestinal Neoplasms / diagnosis. Neuroendocrine Tumors / diagnosis. Pancreatic Neoplasms / diagnosis
  • [MeSH-minor] Duodenoscopy / methods. Humans. Neoplasm Staging. Predictive Value of Tests. Sensitivity and Specificity. Treatment Outcome

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 20646389.001).
  • [ISSN] 0391-9005
  • [Journal-full-title] Il Giornale di chirurgia
  • [ISO-abbreviation] G Chir
  • [Language] ita
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] Italy
  •  go-up   go-down


62. Matrone A, Russo M, Mollica C, Lombardi D, Lombardi G, Maurea S, Imbriaco M: Lymphoepithelial pancreatic cyst: an atypical benign pancreatic mass presenting with a "cheerios-like" appearance. JOP; 2010;11(2):170-2
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Lymphoepithelial pancreatic cyst: an atypical benign pancreatic mass presenting with a "cheerios-like" appearance.
  • CONTEXT: Lymphoepithelial cyst of the pancreas is a rare benign lesion which often presents as an incidental radiological finding, but may cause symptoms, such as abdominal pain and nausea.
  • It can occur at any location in the pancreas.
  • The differential diagnosis includes primary splenic cysts, pseudocysts, mucinous cystic neoplasms of the pancreas, left adrenal cysts, duplication cysts and retroperitoneal cysts.
  • Some of these diagnoses can be excluded by MRI; however, for the final diagnosis, fine needle aspiration or surgical resection may be necessary.
  • CASE REPORT: We report a rare case of a pancreatic lymphoepithelial cyst in an asymptomatic 63-year-old man, discovered in a clinical follow-up for colon cancer.
  • CONCLUSION: This particular finding could help in the differential diagnosis of this benign lesion from other pancreatic masses.
  • [MeSH-major] Pancreatic Cyst / diagnosis. Pancreatic Cyst / pathology
  • [MeSH-minor] Diagnosis, Differential. Edible Grain. Epidermal Cyst / pathology. Humans. Lymphocele / pathology. Male. Middle Aged. Organ Size. Pancreas / pathology. Pancreatic Neoplasms / diagnosis. Pancreatic Neoplasms / pathology

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 20208329.001).
  • [ISSN] 1590-8577
  • [Journal-full-title] JOP : Journal of the pancreas
  • [ISO-abbreviation] JOP
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Italy
  •  go-up   go-down


63. Takada H, Nakazawa T, Ohara H, Ando T, Hayashi K, Naito I, Okumura F, Tanaka H, Yamada T, Takahashi S, Joh T: Role of osteopontin in calcification in autoimmune pancreatitis. Dig Dis Sci; 2009 Apr;54(4):793-801
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • OBJECTIVES: The aim of the present study was to determine the potential for pancreatic calcification in autoimmune pancreatitis by investigating osteopontin and CD44 expression.
  • METHODS: Human pancreatic tissues in normal pancreas, chronic pancreatitis, and autoimmune pancreatitis were obtained from the surgical specimens of 42 patients.
  • Pancreatic tissues from male Wistar Bonn/Kobori rats were also used as an animal autoimmune pancreatitis model.
  • RESULTS: The incidences of osteopontin expression in centroacinar cells in chronic pancreatitis with calcification and in autoimmune pancreatitis were significantly greater than that in normal pancreas (P < 0.05).
  • CONCLUSIONS: These results suggest that autoimmune pancreatitis has the potential for pancreatic calcification over a long-term clinical course.
  • [MeSH-major] Antigens, CD44 / metabolism. Calcinosis / metabolism. Osteopontin / metabolism. Pancreas / metabolism. Pancreatitis, Chronic / metabolism

  • Genetic Alliance. consumer health - Pancreatitis.
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Gastroenterology. 1988 Oct;95(4):1018-28 [3410215.001]
  • [Cites] Dig Dis Sci. 1995 Jul;40(7):1561-8 [7628283.001]
  • [Cites] Circ Res. 1999 Feb 5;84(2):166-78 [9933248.001]
  • [Cites] Pancreas. 2002 Aug;25(2):182-7 [12142743.001]
  • [Cites] Biochem Biophys Res Commun. 1987 Nov 13;148(3):1129-36 [3500718.001]
  • [Cites] Gastroenterology. 2003 Apr;124(4):1010-9 [12671898.001]
  • [Cites] Biochem Biophys Res Commun. 1999 Mar 24;256(3):527-31 [10080931.001]
  • [Cites] Am J Physiol Gastrointest Liver Physiol. 2004 Jul;287(1):G264-73 [15044174.001]
  • [Cites] N Engl J Med. 2001 Mar 8;344(10):732-8 [11236777.001]
  • [Cites] J Biochem. 1993 Nov;114(5):702-7 [8113224.001]
  • [Cites] Proc Natl Acad Sci U S A. 1986 Dec;83(23):8819-23 [3024151.001]
  • [Cites] Virchows Arch. 1999 Jan;434(1):83-9 [10071240.001]
  • [Cites] Science. 1996 Jan 26;271(5248):509-12 [8560266.001]
  • [Cites] Virchows Arch A Pathol Anat Histol. 1977 Feb 18;373(1):37-54 [139023.001]
  • [Cites] J Neuroimmunol. 2004 Jun;151(1-2):78-84 [15145606.001]
  • [Cites] Arterioscler Thromb Vasc Biol. 1999 Aug;19(8):1852-61 [10446063.001]
  • [Cites] Toxicol Appl Pharmacol. 2005 Aug 22;207(1):25-38 [15885730.001]
  • [Cites] Biochem Biophys Res Commun. 1992 Apr 30;184(2):859-64 [1575754.001]
  • [Cites] Am J Physiol Gastrointest Liver Physiol. 2001 Dec;281(6):G1397-404 [11705744.001]
  • [Cites] Immunol Today. 1989 Sep;10(9):289-91 [2686676.001]
  • [Cites] Biochem J. 1985 Dec 15;232(3):715-24 [4091817.001]
  • [Cites] Am J Gastroenterol. 2004 May;99(5):932-7 [15128363.001]
  • [Cites] Pancreas. 2005 Apr;30(3):e64-70 [15782091.001]
  • [Cites] J Biol Chem. 2000 Jun 30;275(26):20197-203 [10766759.001]
  • [Cites] J Cell Biochem Suppl. 1998;30-31:92-102 [9893260.001]
  • [Cites] Am J Dig Dis. 1961 Jul;6:688-98 [13746542.001]
  • [Cites] J Clin Invest. 1998 Apr 1;101(7):1468-78 [9525990.001]
  • [Cites] Clin Exp Immunol. 2008 Apr;152(1):1-12 [18279444.001]
  • [Cites] J Pathol. 2006 Mar;208(4):473-85 [16440289.001]
  • [Cites] J Cell Biol. 1988 Feb;106(2):441-50 [3339096.001]
  • [Cites] Am J Pathol. 1995 Jan;146(1):95-100 [7856741.001]
  • [Cites] J Biol Chem. 1992 Dec 5;267(34):24871-8 [1447223.001]
  • [Cites] J Gastroenterol. 2006 Jul;41(7):626-31 [16932998.001]
  • [Cites] Pancreas. 2005 Jan;30(1):20-5 [15632695.001]
  • [Cites] J Endocrinol. 2005 Nov;187(2):237-47 [16293771.001]
  • [Cites] Int J Pancreatol. 1990 Jun;6(4):231-47 [1698893.001]
  • [Cites] Bone Miner. 1993 Aug;22(2):147-59 [8251766.001]
  • [Cites] Pancreas. 2005 Oct;31(3):232-7 [16163054.001]
  • [Cites] Acta Diabetol Lat. 1985 Oct-Dec;22(4):335-42 [3914158.001]
  • [Cites] Nephron. 2001 Dec;89(4):416-21 [11721159.001]
  • [Cites] J Biol Chem. 1993 Jul 15;268(20):15180-4 [8325891.001]
  • [Cites] Mol Biol Cell. 1992 Oct;3(10):1169-80 [1421573.001]
  • [Cites] J Vet Med Sci. 2006 Apr;68(4):379-82 [16679731.001]
  • [Cites] Pancreas. 2000 Oct;21(3):296-304 [11039475.001]
  • [Cites] Pancreas. 1990 Jul;5(4):452-9 [1696384.001]
  • (PMID = 18651220.001).
  • [ISSN] 1573-2568
  • [Journal-full-title] Digestive diseases and sciences
  • [ISO-abbreviation] Dig. Dis. Sci.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antigens, CD44; 106441-73-0 / Osteopontin
  •  go-up   go-down


64. Takahashi H, Delacruz V, Sarwar S, Selvaggi G, Moon J, Nishida S, Weppler D, Levi D, Kato T, Tzakis A, Ruiz P: Contemporaneous chronic rejection of multiple allografts with principal pancreatic involvement in modified multivisceral transplantation. Pediatr Transplant; 2007 Jun;11(4):448-52
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Contemporaneous chronic rejection of multiple allografts with principal pancreatic involvement in modified multivisceral transplantation.
  • The patient was a 10 yr-old-male with short gut syndrome secondary to Hirschsprung's disease, who underwent a modified (no liver) multivisceral transplant (stomach, pancreas, small and large intestine).
  • Re-transplantation of a multivisceral transplant (stomach, pancreas, liver, small and large intestine) was performed.
  • Microscopic examinations of the explanted allograft organ block revealed varying degrees of chronic rejection in many of the organs but with the pancreatic allograft being affected most severely.
  • The malabsorption symptom following the first transplant may have been caused by the early onset of chronic pancreatic allograft dysfunction.
  • Our case indicates varying severity of chronic rejection among multiple allografts where the pancreatic allograft appeared most susceptible to chronic rejection.
  • [MeSH-major] Graft Rejection / complications. Organ Transplantation / methods. Pancreatic Diseases / etiology
  • [MeSH-minor] Child. Chronic Disease. Duodenum / transplantation. Follow-Up Studies. Hirschsprung Disease / surgery. Humans. Intestine, Large / transplantation. Intestine, Small / transplantation. Male. Pancreas Transplantation / methods. Reoperation. Severity of Illness Index


65. Soga T, Ohishi T, Matsui T, Saito T, Matsumoto M, Takasaki J, Matsumoto S, Kamohara M, Hiyama H, Yoshida S, Momose K, Ueda Y, Matsushime H, Kobori M, Furuichi K: Lysophosphatidylcholine enhances glucose-dependent insulin secretion via an orphan G-protein-coupled receptor. Biochem Biophys Res Commun; 2005 Jan 28;326(4):744-51
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • LPC has been reported to induce insulin secretion from pancreatic beta-cells, however, the precise mechanism has remained elusive to date.
  • LPC potently enhances insulin secretion in response to high concentrations of glucose in the perfused rat pancreas via stimulation of adenylate cyclase, and dose-dependently induces intracellular cAMP accumulation and insulin secretion in a mouse pancreatic beta-cell line, NIT-1 cells.
  • The Gs-protein-coupled receptor for LPC was identified as GPR119, which is predominantly expressed in the pancreas.
  • [MeSH-minor] Animals. Cell Line. Dose-Response Relationship, Drug. Hormones / metabolism. Humans. In Vitro Techniques. Male. Organ Specificity. Pancreas. Rats. Rats, Wistar. Tissue Distribution


66. Kobinger GP, Deng S, Louboutin JP, Vatamaniuk M, Rivera VM, Lian MM, Markmann JF, Clackson T, Raper SE, Matschinsky F, Wilson JM: Pharmacologically regulated regeneration of functional human pancreatic islets. Mol Ther; 2005 Jan;11(1):105-11
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Pharmacologically regulated regeneration of functional human pancreatic islets.
  • Limited availability of donor pancreas tissues restricts the application of this therapeutic modality to a subset of eligible recipients.
  • In an attempt to expand the utility of available donor human pancreas tissue, we developed a method to stimulate the proliferation of insulin-secreting beta-cells within human islets.

  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 15585411.001).
  • [ISSN] 1525-0016
  • [Journal-full-title] Molecular therapy : the journal of the American Society of Gene Therapy
  • [ISO-abbreviation] Mol. Ther.
  • [Language] eng
  • [Grant] United States / NIDDK NIH HHS / DK / P30 DK47757
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Blood Glucose
  •  go-up   go-down


67. Zhang QF, Bai YQ: [Dynamic change in microcirculation of pancreas after experimental high-voltage electric burn]. Zhonghua Shao Shang Za Zhi; 2009 Oct;25(5):368-71
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Dynamic change in microcirculation of pancreas after experimental high-voltage electric burn].
  • OBJECTIVE: To observe the changes in surface microcirculation of pancreas after high-voltage electric burn (HEB).
  • The morphology of the pancreas microvessels and its surrounding tissues, and the dynamic changes in microvascular blood flow were observed with WX-9 microscope and its image analytical system.
  • Arteriole, venule and capillary network on the surface of pancreatic lobules of rabbits in both groups were clearly seen and well-distributed in the natural way before HEB.
  • There was no exudation and bleeding around the microvessels on the pancreas surface of rabbits in both groups before HEB.
  • CONCLUSIONS: HEB produces microcirculation disturbance and functional disturbance of pancreas.
  • [MeSH-major] Burns, Electric / pathology. Microcirculation. Pancreas / blood supply

  • MedlinePlus Health Information. consumer health - Electrical Injuries.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 19951561.001).
  • [ISSN] 1009-2587
  • [Journal-full-title] Zhonghua shao shang za zhi = Zhonghua shaoshang zazhi = Chinese journal of burns
  • [ISO-abbreviation] Zhonghua Shao Shang Za Zhi
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] China
  •  go-up   go-down


68. Chen S, Li C, Yuan G, Xie F: Anatomical and histological observation on the pancreas in adult zebrafish. Pancreas; 2007 Jan;34(1):120-5
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Anatomical and histological observation on the pancreas in adult zebrafish.
  • OBJECTIVES: To investigate the anatomic structure of the pancreas and the distribution of the islets in adult zebrafish.
  • RESULTS: The pancreas along the intestine included 4 relatively independent and concentrated lobes, in which 4 kinds of islets-principal islets, Brockmann bodies, diffusely existing islets, and single beta-cell-were observed.
  • The different islets were differently distributed in the 4 pancreatic lobes with statistical significance (P < 0.05).
  • CONCLUSIONS: According to the distinct distribution, concentration of the pancreas, and different contents of the islets within the pancreas, 4 lobes of the pancreas along the intestine-the gallbladder-spleen lobe, the middle lobe, the left lobe, and the ventral lobe-were identified in adult zebrafish.
  • [MeSH-major] Glucagon-Secreting Cells / cytology. Insulin-Secreting Cells / cytology. Pancreas, Exocrine / anatomy & histology. Zebrafish / anatomy & histology

  • Hazardous Substances Data Bank. GLUCAGON .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17198193.001).
  • [ISSN] 1536-4828
  • [Journal-full-title] Pancreas
  • [ISO-abbreviation] Pancreas
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Insulin; 9007-92-5 / Glucagon
  •  go-up   go-down


69. Dixon E, Pasieka JL: Functioning and nonfunctioning neuroendocrine tumors of the pancreas. Curr Opin Oncol; 2007 Jan;19(1):30-5
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Functioning and nonfunctioning neuroendocrine tumors of the pancreas.
  • PURPOSE OF REVIEW: Neuroendocrine tumors of the pancreas are a small subgroup of tumors characterized by a variety of biological behaviors.
  • With recent advances in diagnosis and staging, the treatment options for all neuroendocrine tumors have evolved.
  • Presented here is a review of the current-day knowledge for neuroendocrine tumors of the pancreas.
  • RECENT FINDINGS: A consensus by leading experts in the neuroendocrine tumors field has proposed an algorithm for the diagnosis, treatment and follow-up of these rare tumors.
  • Contrary to adenocarcinoma of the pancreas, hormonal and biotherapy offer unique treatment strategies for these rare tumors.
  • SUMMARY: Presented here is a summary of the recent literature on the diagnosis and treatment of neuroendocrine tumors of the pancreas.
  • [MeSH-major] Neuroendocrine Tumors / physiopathology. Pancreatic Neoplasms / physiopathology

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17133109.001).
  • [ISSN] 1040-8746
  • [Journal-full-title] Current opinion in oncology
  • [ISO-abbreviation] Curr Opin Oncol
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 46
  •  go-up   go-down


70. Basturk O, Coban I, Adsay NV: Pancreatic cysts: pathologic classification, differential diagnosis, and clinical implications. Arch Pathol Lab Med; 2009 Mar;133(3):423-38
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Pancreatic cysts: pathologic classification, differential diagnosis, and clinical implications.
  • CONTEXT: Cystic lesions of the pancreas are being recognized with increasing frequency and have become a more common finding in clinical practice because of the widespread use of advanced imaging modalities and the sharp drop in the mortality rate of pancreatic surgery.
  • Consequently, in the past 2 decades, the nature of many cystic tumors in this organ has been better characterized, and significant developments have taken place in the classification and in our understanding of pancreatic cystic lesions.
  • OBJECTIVE: To provide an overview of the current concepts in classification, differential diagnosis, and clinical/biologic behavior of pancreatic cystic tumors.
  • CONCLUSIONS: In contrast to solid tumors, most of which are invasive ductal adenocarcinomas with dismal prognosis, cystic lesions of the pancreas are often either benign or low-grade indolent neoplasia.
  • Those that are nonmucinous such as serous tumors, congenital cysts, lymphoepithelial cysts, and squamoid cyst of pancreatic ducts have no malignant potential.
  • Only rare nonmucinous cystic tumors that occur as a result of degenerative/necrotic changes in otherwise solid neoplasia, such as cystic ductal adenocarcinomas, cystic pancreatic endocrine neoplasia, and solid-pseudopapillary neoplasm, are also malignant and have variable degrees of aggressiveness.
  • [MeSH-major] Pancreatic Cyst / diagnosis. Pancreatic Cyst / pathology
  • [MeSH-minor] Diagnosis, Differential. Humans. Pancreatic Neoplasms / diagnosis. Pancreatic Neoplasms / pathology. Pancreatic Pseudocyst / diagnosis. Pancreatic Pseudocyst / pathology

  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 19260748.001).
  • [ISSN] 1543-2165
  • [Journal-full-title] Archives of pathology & laboratory medicine
  • [ISO-abbreviation] Arch. Pathol. Lab. Med.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 204
  •  go-up   go-down


71. Huszarik K, Wright B, Keller C, Nikoopour E, Krougly O, Lee-Chan E, Qin HY, Cameron MJ, Gurr WK, Hill DJ, Sherwin RS, Kelvin DJ, Singh B: Adjuvant immunotherapy increases beta cell regenerative factor Reg2 in the pancreas of diabetic mice. J Immunol; 2010 Nov 1;185(9):5120-9
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Adjuvant immunotherapy increases beta cell regenerative factor Reg2 in the pancreas of diabetic mice.
  • Insulin-producing β cells can partially regenerate in adult pancreatic tissues, both in human and animal models of type 1 diabetes (T1D).
  • In this study, we investigated factors involved in the regeneration of β cells in the pancreas of NOD mice during diabetes development and after treatment with adjuvants.
  • Reg2 expression was found to be upregulated in pancreatic islets both during diabetes development and as a result of adjuvant treatment in diabetic NOD mice and in C57BL/6 mice made diabetic by streptozotocin treatment.
  • We also observed upregulation of Reg2 in the pancreas of diabetic mice undergoing β cell regenerative therapy with exendin-4 or with islet neogenesis-associated protein.
  • Reg2 expression following adjuvant treatment correlated with a reduction in insulitis, an increase in insulin secretion, and an increase in the number of small islets in the pancreas of diabetic NOD mice and with improved glucose tolerance tests in streptozotocin-treated diabetic C57BL/6 mice.
  • [MeSH-major] Adjuvants, Immunologic / pharmacology. Diabetes Mellitus, Type 1 / metabolism. Immunotherapy / methods. Insulin-Secreting Cells / metabolism. Pancreas / physiology. Proteins / metabolism

  • MedlinePlus Health Information. consumer health - Diabetes Type 1.
  • KOMP Repository. gene/protein/disease-specific - KOMP Repository (subscription/membership/fee required).
  • Mouse Genome Informatics (MGI). Mouse Genome Informatics (MGI) .
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 20876350.001).
  • [ISSN] 1550-6606
  • [Journal-full-title] Journal of immunology (Baltimore, Md. : 1950)
  • [ISO-abbreviation] J. Immunol.
  • [Language] eng
  • [Grant] Canada / Canadian Institutes of Health Research / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Adjuvants, Immunologic; 0 / Pap protein, mouse; 0 / Proteins; 9007-81-2 / Freund's Adjuvant
  •  go-up   go-down


72. Reddy S, Wolfgang CL: Solid pseudopapillary neoplasms of the pancreas. Adv Surg; 2009;43:269-82
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Solid pseudopapillary neoplasms of the pancreas.
  • SPN is a rare neoplasm that affects mainly young women.
  • The genetic mechanism behind the development of SPN is distinct from the more lethal ductal carcinoma of the pancreas.

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 19845185.001).
  • [ISSN] 0065-3411
  • [Journal-full-title] Advances in surgery
  • [ISO-abbreviation] Adv Surg
  • [Language] ENG
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 52
  •  go-up   go-down


73. Maekawa H, Yoneyama H, Komatsu Y, Orita H, Sakurada M, Sato K, Yamano M: Pancreatic lymphoepithelial cyst with an intracystic papillary projection. Report of a case. JOP; 2009;10(6):694-6
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Pancreatic lymphoepithelial cyst with an intracystic papillary projection. Report of a case.
  • CONTEXT: Lymphoepithelial cysts of the pancreas show various presentations on imaging studies often making a differential diagnosis difficult.
  • Abdominal US was carried out and demonstrated a cystic lesion 5 cm in diameter which included an intracystic projection into the pancreatic body.
  • Since we could not exclude a pancreatic cystic neoplasm, a distal pancreatectomy was performed.
  • Postoperative pathological examination demonstrated that the lesion was a lymphoepithelial cyst of the pancreas.
  • CONCLUSION: Resection is inevitable when a true pancreatic neoplasm cannot be excluded.
  • [MeSH-major] Cell Movement. Epithelial Cells / pathology. Lymphocytes / pathology. Pancreatic Cyst / diagnosis. Pancreatic Cyst / pathology
  • [MeSH-minor] Carcinoma, Papillary / pathology. Carcinoma, Papillary / surgery. Humans. Male. Middle Aged. Neoplasm Invasiveness. Pancreatectomy. Pancreatic Neoplasms / pathology. Pancreatic Neoplasms / surgery

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 19890197.001).
  • [ISSN] 1590-8577
  • [Journal-full-title] JOP : Journal of the pancreas
  • [ISO-abbreviation] JOP
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Italy
  •  go-up   go-down


74. Okuyama H, Kubota A, Kawahara H, Shimizu Y, Watanabe T, Yamanaka H, Tani G, Takeyama Y: Frey procedure for chronic pancreatitis in a child with duodenal atresia and complex pancreaticobiliary disorders. Eur J Pediatr Surg; 2010 Jan;20(1):45-7
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • A boy aged 2 years and 7 months who had undergone duodeno-duodenostomy for duodenal atresia and annular pancreas in the neonatal period presented with recurrent pancreatitis.
  • ERCP showed an incomplete pancreas divisum associated with pancreaticobiliary maljunction.
  • The main pancreatic duct was opened in the body and the head was cored out anteriorly.
  • [MeSH-major] Bile Ducts, Extrahepatic / abnormalities. Duodenum / abnormalities. Intestinal Atresia / complications. Pancreas / abnormalities. Pancreatitis, Chronic / surgery

  • Genetic Alliance. consumer health - Duodenal Atresia.
  • Genetic Alliance. consumer health - Pancreatitis.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] (c) Georg Thieme Verlag KG Stuttgart-New York.
  • (PMID = 19347806.001).
  • [ISSN] 1439-359X
  • [Journal-full-title] European journal of pediatric surgery : official journal of Austrian Association of Pediatric Surgery ... [et al] = Zeitschrift für Kinderchirurgie
  • [ISO-abbreviation] Eur J Pediatr Surg
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Germany
  •  go-up   go-down


75. Oberholzer J, Tzvetanov I, Mele A, Benedetti E: Laparoscopic and robotic donor pancreatectomy for living donor pancreas and pancreas-kidney transplantation. J Hepatobiliary Pancreat Sci; 2010 Mar;17(2):97-100
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Laparoscopic and robotic donor pancreatectomy for living donor pancreas and pancreas-kidney transplantation.
  • Pancreas transplantation is a widely accepted procedure that can efficiently restore euglycemia and prevent progression of complications.
  • Living donor pancreas transplant was introduced at the University of Minnesota in 1979.
  • The only case reported worldwide of robotic distal pancreatectomy and nephrectomy for living donor pancreas-kidney transplantation was successfully performed by our team in 2006 at the University of Illinois at Chicago and proved as a promising technique.


76. Azzi J, Geara AS, El-Sayegh S, Abdi R: Immunological aspects of pancreatic islet cell transplantation. Expert Rev Clin Immunol; 2010 Jan;6(1):111-24
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Immunological aspects of pancreatic islet cell transplantation.
  • Whole-pancreas and islet cell transplantation seem to provide a potential solution by restoring the normal physiology of glucose-insulin homeostasis.
  • Although islet transplantation is less invasive than whole-pancreas transplantation, the insulin-free state after islet transplantation remained low (10%) at 5 years after surgery.

  • Genetic Alliance. consumer health - Transplantation.
  • MedlinePlus Health Information. consumer health - Diabetes Type 1.
  • MedlinePlus Health Information. consumer health - Islet Cell Transplantation.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 20383895.001).
  • [ISSN] 1744-666X
  • [Journal-full-title] Expert review of clinical immunology
  • [ISO-abbreviation] Expert Rev Clin Immunol
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Immunosuppressive Agents; 0 / Inflammation Mediators
  • [Number-of-references] 167
  •  go-up   go-down


77. Kutsuna N, Yamazaki S, Itoh Y, Wakabayashi K, Iwama A, Watanabe Y, Haraguchi Y, Ueda T, Takayama T: Arterial stimulation and venous sampling (ASVS) is useful for recurrent lesions of insulinoma: a case report. Surg Laparosc Endosc Percutan Tech; 2009 Aug;19(4):e138-9
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • We encountered a recurrent case of benign solitary insulinoma in the pancreatic tail, which may have been caused by an inadequate surgical margin in the use of an ultrasonic dissector.
  • A 45-year-old man was referred with hypoglycemia and diagnosed solitary insulinoma in the pancreas.
  • Laparoscopic pancreatic enucleation was performed using an ultrasonic dissector.
  • The tumor was extracted and the surgical margins were microscopically negative.
  • He underwent resection of all visible lesions with omentum and wide excision of the soft tissue surrounding the pancreas after preoperative arterial stimulation and venous sampling test.
  • [MeSH-major] Calcium Gluconate / administration & dosage. Gastrointestinal Agents / administration & dosage. Insulin / blood. Insulinoma / blood. Pancreatic Neoplasms / blood

  • Genetic Alliance. consumer health - Insulinoma.
  • MedlinePlus Health Information. consumer health - Diabetes Medicines.
  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • Hazardous Substances Data Bank. CALCIUM GLUCONATE .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 19692865.001).
  • [ISSN] 1534-4908
  • [Journal-full-title] Surgical laparoscopy, endoscopy & percutaneous techniques
  • [ISO-abbreviation] Surg Laparosc Endosc Percutan Tech
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Gastrointestinal Agents; 0 / Insulin; SQE6VB453K / Calcium Gluconate
  •  go-up   go-down


78. Huang H, Dong X, Gao SL, Wu YL: Conservative resection for benign tumors of the proximal pancreas. World J Gastroenterol; 2009 Aug 28;15(32):4044-8
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Conservative resection for benign tumors of the proximal pancreas.
  • AIM: To evaluate the safety and long-term prognosis of conservative resection (CR) for benign or borderline tumor of the proximal pancreas.
  • Modified continuous closed lavage (MCCL) was performed for patients with pancreatic fistula.
  • Overall, pancreatic fistula was present in 25% of patients.
  • CONCLUSION: CR is a safe and effective procedure for patients with benign tumors in the proximal pancreas, with careful CCI-PJ and postoperative MCCL.
  • [MeSH-major] Pancreas / surgery. Pancreatectomy / instrumentation. Pancreatectomy / methods. Pancreatic Neoplasms / surgery. Pancreaticojejunostomy / instrumentation. Pancreaticojejunostomy / methods
  • [MeSH-minor] Adult. Aged. Cystadenoma, Serous / surgery. Female. Humans. Insulinoma / surgery. Islets of Langerhans / pathology. Islets of Langerhans / surgery. Male. Middle Aged. Pancreatic Fistula / surgery. Retrospective Studies. Surgical Procedures, Operative / methods. Treatment Outcome

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Ann Surg. 1995 Oct;222(4):580-8; discussion 588-92 [7574936.001]
  • [Cites] Am J Surg. 1995 Jan;169(1):179-85 [7817989.001]
  • [Cites] Diabet Med. 1998 Jul;15(7):539-53 [9686693.001]
  • [Cites] J Gastrointest Surg. 1998 Nov-Dec;2(6):509-16; discussion 516-7 [10457309.001]
  • [Cites] Arch Surg. 2006 Mar;141(3):293-9 [16549696.001]
  • [Cites] J Gastrointest Surg. 2006 Jun;10(6):804-12 [16769536.001]
  • [Cites] Br J Surg. 2007 Oct;94(10):1254-9 [17583892.001]
  • [Cites] J Gastrointest Surg. 2007 Dec;11(12):1704-11 [17929105.001]
  • [Cites] Br J Surg. 2008 Jan;95(1):85-91 [18041022.001]
  • [Cites] Pancreas. 2008 Jan;36(1):44-9 [18192880.001]
  • [Cites] Br J Surg. 2000 Apr;87(4):434-8 [10759738.001]
  • [Cites] Ann Surg. 2000 Jun;231(6):890-8 [10816633.001]
  • [Cites] J Am Coll Surg. 2000 Jun;190(6):711-6 [10873007.001]
  • [Cites] Br J Surg. 2000 Jul;87(7):883-9 [10931023.001]
  • [Cites] J Hepatobiliary Pancreat Surg. 2000;7(5):466-72 [11180872.001]
  • [Cites] Surgery. 2002 Nov;132(5):836-43 [12464868.001]
  • [Cites] Dig Surg. 2003;20(6):506-10 [14506331.001]
  • [Cites] J Gastrointest Surg. 2004 Jul-Aug;8(5):532-8 [15239986.001]
  • [Cites] Ann Surg. 1976 Oct;184(4):403-13 [1015887.001]
  • [Cites] Surgery. 1984 Oct;96(4):608-16 [6435270.001]
  • [Cites] Surgery. 1993 May;113(5):532-5 [8488471.001]
  • [Cites] Ann Surg. 1993 Sep;218(3):229-37; discussion 237-8 [8103982.001]
  • [Cites] J Am Coll Surg. 1994 Nov;179(5):545-52 [7952456.001]
  • [Cites] Arch Surg. 1998 Mar;133(3):327-31 [9517749.001]
  • (PMID = 19705501.001).
  • [ISSN] 2219-2840
  • [Journal-full-title] World journal of gastroenterology
  • [ISO-abbreviation] World J. Gastroenterol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] China
  • [Other-IDs] NLM/ PMC2731956
  •  go-up   go-down


79. Wolf A, Bernhardt J, Patrzyk M, Heidecke CD: The value of endoscopic diagnosis and the treatment of pancreas injuries following blunt abdominal trauma. Surg Endosc; 2005 May;19(5):665-9
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The value of endoscopic diagnosis and the treatment of pancreas injuries following blunt abdominal trauma.
  • BACKGROUND: Injuries to the pancreas following blunt abdominal trauma are rare due to its protected retroperitoneal position.
  • Many pancreatic lesions remain unnoticed at first and only become apparent when complications arise or during treatment of other injuries.
  • METHODS: Using medical records over the past 5 years, we investigated when and in what circumstances endoscopic retrograde cholangiopancreaticography (ERCP) was used in the diagnosis and treatment of pancreas injuries after blunt abdominal trauma.
  • RESULTS: An ERCP was performed on a total of five patients with suspected injuries to the pancreas after blunt abdominal trauma.
  • No duct participation could be determined in three of the patients with a first degree pancreatic lesion.
  • Because of the nature of her injuries, pancreatic left resection with splenectomy was necessary.
  • After the operation, a pancreatic fistula diagnosed.
  • CONCLUSION: The major advantage of the prompt retrograde discription of the pancreatobiliary system after an accident in which pancreas involvement is suspected is the more precise assessment of the extent of the injuries.
  • [MeSH-major] Abdominal Injuries / diagnosis. Cholangiopancreatography, Endoscopic Retrograde. Pancreas / injuries. Wounds, Nonpenetrating / diagnosis
  • [MeSH-minor] Accidents, Traffic. Adult. Algorithms. Ampulla of Vater / injuries. Ampulla of Vater / surgery. Amylases / blood. Animals. Case Management. Contusions / diagnosis. Contusions / etiology. Drainage. Horses. Humans. Lipase / blood. Male. Middle Aged. Pancreatic Ducts / injuries. Pancreatic Ducts / surgery. Pancreatic Fistula / diagnosis. Pancreatic Fistula / etiology. Pancreatic Fistula / surgery. Pancreatitis / diagnosis. Pancreatitis / etiology. Pancreatitis / surgery. Retrospective Studies. Stents. Tomography, X-Ray Computed. Trauma Severity Indices. Ultrasonography

  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Ann Surg. 1997 Jul;226(1):70-6 [9242340.001]
  • [Cites] Chirurg. 1999 Nov;70(11):1255-68 [10591762.001]
  • [Cites] Unfallchirurg. 1999 Apr;102(4):298-304 [10355346.001]
  • [Cites] Am Surg. 2001 Mar;67(3):227-30; discussion 230-1 [11270879.001]
  • [Cites] Chirurg. 1997 Jun;68(6):624-9 [9324443.001]
  • [Cites] J Pediatr Surg. 2001 Feb;36(2):345-8 [11172431.001]
  • [Cites] Dig Surg. 2002;19(4):291-7; discussion 297-9 [12207073.001]
  • [Cites] Langenbecks Arch Chir. 1993;378(2):102-5 [8474291.001]
  • [Cites] Surg Clin North Am. 1977 Feb;57(1):49-65 [854854.001]
  • [Cites] J Trauma. 2000 Apr;48(4):745-51; discussion 751-2 [10780612.001]
  • [Cites] Chirurg. 1999 Nov;70(11):1269-77 [10591763.001]
  • [Cites] Gastrointest Endosc. 2002 Jul;56(1):18-24 [12085030.001]
  • [Cites] Eur Radiol. 1999;9(2):244-9 [10101645.001]
  • [Cites] Br J Surg. 1996 Jul;83(7):934-7 [8813778.001]
  • [Cites] Surg Today. 2002;32(7):659-62 [12111529.001]
  • [Cites] Surg Endosc. 1998 May;12 (5):400-4 [9569357.001]
  • [Cites] Surg Clin North Am. 1995 Apr;75(2):293-303 [7899999.001]
  • [Cites] Chirurg. 1999 Nov;70(11):1246-54 [10591761.001]
  • [Cites] Int J Legal Med. 2001 Oct;115(2):72-5 [11724433.001]
  • [Cites] Int J Legal Med. 2001 Dec;115(3):162-4 [11775019.001]
  • [Cites] Gastrointest Endosc. 2001 Jul;54(1):49-55 [11427841.001]
  • (PMID = 15759197.001).
  • [ISSN] 1432-2218
  • [Journal-full-title] Surgical endoscopy
  • [ISO-abbreviation] Surg Endosc
  • [Language] eng
  • [Publication-type] Case Reports; Evaluation Studies; Journal Article
  • [Publication-country] Germany
  • [Chemical-registry-number] EC 3.1.1.3 / Lipase; EC 3.2.1.- / Amylases
  •  go-up   go-down


80. Ait-Ali A, Sall I, El-Kaoui H, Bouchentouf SM, El-Hjouji A, Rouibaa F, Benkirane A, Bounaim A, Zentar A, Sair K: Medial pancreatectomy for a neuroendocrine tumor invading the splenic artery and vein. JOP; 2010;11(1):75-7
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Medial pancreatectomy for a neuroendocrine tumor invading the splenic artery and vein.
  • CONTEXT: Pancreatic tumors in the midportion have traditionally been treated by an extended right or left pancreatectomy.
  • A medial or central pancreatectomy is an alternative technique for benign or low-grade malignant neoplasms located to the left of the gastroduodenal artery and close to the splenomesenteric confluence.
  • A computed tomography scan showed a 4 cm heterogeneous lesion within the pancreatic body.
  • This tumor invaded the splenic artery and vein.
  • The patient continues to be well after a 10-month follow-up without pancreatic insufficiency or local recurrence, and CT has demonstrated splenic perfusion by the collateral vessels.
  • CONCLUSION: We believe that a medial or central pancreatectomy may be a safe procedure where there is involvement of the large splenic vessels by a low grade malignant pancreatic tumor and that a systematic splenectomy is not justified.
  • [MeSH-major] Neuroendocrine Tumors / surgery. Pancreatectomy / methods. Pancreatic Neoplasms / surgery. Splenic Artery / surgery. Splenic Vein / surgery
  • [MeSH-minor] Adult. Female. Humans. Neoplasm Invasiveness. Splenic Neoplasms / prevention & control. Splenic Neoplasms / secondary

  • Genetic Alliance. consumer health - Pancreatic islet cell tumors.
  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 20065560.001).
  • [ISSN] 1590-8577
  • [Journal-full-title] JOP : Journal of the pancreas
  • [ISO-abbreviation] JOP
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Italy
  •  go-up   go-down


81. Nakajima Y, Kakizaki S, Kanda D, Shimada Y, Sohara N, Sato K, Takagi H, Mori M, Watanabe H: Pancreatic and gastric metastases of leiomyosarcoma arising in the left leg. Int J Clin Oncol; 2005 Oct;10(5):342-7
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Pancreatic and gastric metastases of leiomyosarcoma arising in the left leg.
  • Pancreatic or gastric metastases from other primary malignancies are rare, especially from leiomyosarcoma.
  • We report a case of leiomyosarcoma in the left lower leg with metastases to the pancreas and stomach.
  • On admission, endoscopic examination and computed tomography were performed for a routine checkup to exclude esophageal varices and liver tumor.
  • Although the patient had no specific symptoms, multiple gastric and pancreatic metastases were identified by endoscopy and computed tomography, respectively.
  • In general, metastases to the pancreas and stomach are rare.
  • We discuss the clinical and diagnostic findings of pancreatic and gastric metastases by reviewing previously reported cases.
  • [MeSH-major] Leg. Leiomyosarcoma / secondary. Muscle Neoplasms / pathology. Pancreatic Neoplasms / secondary. Stomach Neoplasms / secondary

  • Genetic Alliance. consumer health - Leiomyosarcoma.
  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • MedlinePlus Health Information. consumer health - Stomach Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Virchows Arch. 2004 Jun;444(6):527-35 [15057558.001]
  • [Cites] AJR Am J Roentgenol. 1993 Jan;160(1):49-52 [8416644.001]
  • [Cites] Hepatogastroenterology. 2003 Sep-Oct;50(53):1675-7 [14571814.001]
  • [Cites] Hepatogastroenterology. 2000 Mar-Apr;47(32):540-4 [10791233.001]
  • [Cites] Radiographics. 1998 Mar-Apr;18(2):369-78 [9536484.001]
  • [Cites] Acta Radiol. 1989 Nov-Dec;30(6):615-9 [2631949.001]
  • [Cites] J Comput Assist Tomogr. 1994 Nov-Dec;18(6):905-10 [7962797.001]
  • [Cites] Ann Surg Oncol. 2002 Aug;9(7):675-9 [12167582.001]
  • [Cites] Radiology. 1986 Aug;160(2):399-402 [3523591.001]
  • [Cites] J Surg Oncol. 2003 Jul;83(3):161-6; discussion 166 [12827684.001]
  • [Cites] Pathol Int. 2001 Sep;51(9):686-90 [11696171.001]
  • [Cites] Eur Radiol. 1999;9(4):711-4 [10354890.001]
  • [Cites] Pathol Int. 2000 Feb;50(2):162-5 [10792777.001]
  • [Cites] J Orthop Sci. 2000;5(5):463-9 [11180903.001]
  • [Cites] Gan To Kagaku Ryoho. 2004 Sep;31(9):1324-30 [15446551.001]
  • [Cites] Endoscopy. 2001 Jun;33(6):507-10 [11437044.001]
  • [Cites] Surg Gynecol Obstet. 1989 Apr;168(4):345-7 [2928909.001]
  • (PMID = 16247662.001).
  • [ISSN] 1341-9625
  • [Journal-full-title] International journal of clinical oncology
  • [ISO-abbreviation] Int. J. Clin. Oncol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Japan
  •  go-up   go-down


82. Scott WE 3rd, Weegman BP, Ferrer-Fabrega J, Stein SA, Anazawa T, Kirchner VA, Rizzari MD, Stone J, Matsumoto S, Hammer BE, Balamurugan AN, Kidder LS, Suszynski TM, Avgoustiniatos ES, Stone SG, Tempelman LA, Sutherland DE, Hering BJ, Papas KK: Pancreas oxygen persufflation increases ATP levels as shown by nuclear magnetic resonance. Transplant Proc; 2010 Jul-Aug;42(6):2011-5
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Pancreas oxygen persufflation increases ATP levels as shown by nuclear magnetic resonance.
  • Due to a shortage of suitable human pancreata, high cost, and the large dose of islets presently required for long-term diabetes reversal; it is important to maximize viable islet yield.
  • Traditional methods of pancreas preservation have been identified as suboptimal due to insufficient oxygenation.
  • METHODS: Human pancreata were obtained from brain-dead donors.
  • Porcine pancreata were procured by en bloc viscerectomy from heparinized donation after cardiac death donors and were either preserved by either two-layer method (TLM) or PSF.
  • RESULTS: Human and porcine pancreata can be effectively preserved by PSF.
  • MRI showed that pancreatic tissue was homogeneously filled with gas.
  • TLM can effectively raise ATP:P(i) levels in rat pancreata but not in larger porcine pancreata.
  • When human or porcine organs were preserved by PSF, ATP:P(i) was elevated to levels similar to those observed in rat pancreata.
  • CONCLUSION: The methods developed for human and porcine pancreas PSF homogeneously deliver oxygen throughout the organ.

  • MedlinePlus Health Information. consumer health - Pancreas Transplantation.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] Copyright 2010 Elsevier Inc. All rights reserved.
  • [Cites] Annu Rev Biomed Eng. 2005;7:287-326 [16004573.001]
  • [Cites] Liver Int. 2005 Apr;25(2):273-81 [15780050.001]
  • [Cites] Methods Mol Med. 2006;124:225-48 [16506424.001]
  • [Cites] Transplantation. 2006 May 27;81(10):1471-2; author reply 1472 [16732188.001]
  • [Cites] Nephrol Dial Transplant. 2006 Jul;21(7):1803-8 [16556630.001]
  • [Cites] J Invest Surg. 2006 Jul-Aug;19(4):219-27 [16835136.001]
  • [Cites] Transplantation. 2006 Nov 27;82(10):1286-90 [17130776.001]
  • [Cites] Transplantation. 2007 Oct 15;84(7):864-9 [17984839.001]
  • [Cites] Transplant Proc. 2008 Mar;40(2):403-6 [18374082.001]
  • [Cites] Transplantation. 2008 Dec 15;86(11):1503-10 [19077881.001]
  • [Cites] J Hepatol. 2000 Jan;32(1):105-11 [10673074.001]
  • [Cites] Eur J Cardiothorac Surg. 2000 Jan;17(1):71-6 [10735415.001]
  • [Cites] N Engl J Med. 2000 Jul 27;343(4):230-8 [10911004.001]
  • [Cites] Transplantation. 2001 Feb 27;71(4):498-502 [11258427.001]
  • [Cites] Transplant Proc. 2002 Nov;34(7):2688-9 [12431575.001]
  • [Cites] Transplantation. 2002 Dec 27;74(12):1804-9 [12499904.001]
  • [Cites] Transplantation. 2003 Mar 27;75(6):750-6 [12660496.001]
  • [Cites] Transplantation. 2003 May 15;75(9):1524-7 [12792508.001]
  • [Cites] Curr Opin Clin Nutr Metab Care. 2003 Sep;6(5):501-9 [12913667.001]
  • [Cites] Lancet. 2003 Oct 11;362(9391):1242 [14568760.001]
  • [Cites] Transplant Proc. 2003 Nov;35(7):2461-2 [14611986.001]
  • [Cites] Dis Markers. 2003-2004;19(2-3):49-68 [15096705.001]
  • [Cites] Transplant Proc. 2004 May;36(4):1037-9 [15194360.001]
  • [Cites] Transplantation. 2004 Jul 15;78(1):78-82 [15257042.001]
  • [Cites] Transplantation. 2004 Jul 15;78(1):96-100 [15257045.001]
  • [Cites] Transplantation. 1984 Aug;38(2):102-6 [6380036.001]
  • [Cites] Transplantation. 1988 Sep;46(3):457-60 [3047936.001]
  • [Cites] Transplantation. 1989 Aug;48(2):339-42 [2667212.001]
  • [Cites] Magn Reson Imaging. 1995;13(3):463-9 [7791556.001]
  • [Cites] Liver Transpl Surg. 1997 Sep;3(5):481-93 [9346790.001]
  • [Cites] Transplant Proc. 1997 Nov;29(7):2994-6 [9365641.001]
  • [Cites] Scand J Urol Nephrol. 1997 Oct;31(5):487-92 [9406313.001]
  • [Cites] Transplantation. 1998 Dec 15;66(11):1450-9 [9869085.001]
  • [Cites] Transplant Proc. 2005 Oct;37(8):3501-4 [16298642.001]
  • (PMID = 20692395.001).
  • [ISSN] 1873-2623
  • [Journal-full-title] Transplantation proceedings
  • [ISO-abbreviation] Transplant. Proc.
  • [Language] ENG
  • [Grant] United States / NIDDK NIH HHS / DK / R43 DK070400; United States / NIDDK NIH HHS / DK / R44 DK070400; United States / NIDDK NIH HHS / DK / R43 DK070400-02; United States / NCRR NIH HHS / RR / U42 RR016598; United States / NIDDK NIH HHS / DK / DK070400-03A1; United States / NIDDK NIH HHS / DK / R44 DK070400-03A1; United States / NIDDK NIH HHS / DK / DK070400-02S1; United States / NIDDK NIH HHS / DK / R43 DK070400-02S1; United States / NIDDK NIH HHS / DK / DK070400-02; United States / NIDDK NIH HHS / DK / R43 DK070400-01A1; United States / NIDDK NIH HHS / DK / DK070400-01A1
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Organ Preservation Solutions
  • [Other-IDs] NLM/ NIHMS223789; NLM/ PMC2947552
  •  go-up   go-down


83. Zhou J, Zhou S, Tang J, Zhang K, Guang L, Huang Y, Xu Y, Ying Y, Zhang L, Li D: Protective effect of berberine on beta cells in streptozotocin- and high-carbohydrate/high-fat diet-induced diabetic rats. Eur J Pharmacol; 2009 Mar 15;606(1-3):262-8
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The present study was designed to evaluate the protective effects of berberine against beta cell damage and antioxidant of pancreas in diabetic rats.
  • After 16 weeks treatment, serum insulin level, insulin expression in pancreas, and malonaldehyde content, superoxide dismutase activity in pancreatic homogenate were assayed.
  • Pancreas was examined by hematoxylin/eosin staining and transmission electron microscope.
  • Pancreas to body weight ratio, insulin level, insulin sensitivity index, malonaldehyde content and superoxide dismutase activity were altered in diabetic rats, and were near control levels treated with 150, 300 mg/kg berberine.
  • The pancreatic islet area atrophied and secretory granules of beta cells decreased in diabetic rats.
  • Slight pathological changes existed in beta cells of 150, 300 mg/kg berberine-treated diabetic pancreas.

  • MedlinePlus Health Information. consumer health - Carbohydrates.
  • MedlinePlus Health Information. consumer health - Dietary Fats.
  • Hazardous Substances Data Bank. STREPTOZOTOCIN .
  • Hazardous Substances Data Bank. HEMATOXYLIN .
  • Hazardous Substances Data Bank. MALONALDEHYDE .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 19374872.001).
  • [ISSN] 1879-0712
  • [Journal-full-title] European journal of pharmacology
  • [ISO-abbreviation] Eur. J. Pharmacol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Dietary Carbohydrates; 0 / Dietary Fats; 0 / Insulin; 0I8Y3P32UF / Berberine; 4Y8F71G49Q / Malondialdehyde; 5W494URQ81 / Streptozocin; EC 1.15.1.1 / Superoxide Dismutase; TDQ283MPCW / Eosine Yellowish-(YS); YKM8PY2Z55 / Hematoxylin
  •  go-up   go-down


84. Fridell JA, Wozniak TC, Powelson JA, Reynolds JM: Simultaneous bilateral lung and pancreas transplantation in recipient with cystic fibrosis. Transplant Proc; 2008 Mar;40(2):494-7
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Simultaneous bilateral lung and pancreas transplantation in recipient with cystic fibrosis.
  • INTRODUCTION: Cystic fibrosis (CF) is an inherited disorder that presents in childhood as a multisystem disease.
  • Pulmonary failure and pancreatic insufficiency, including CF-related diabetes (CFRD) and exocrine insufficiency, are significant causes of morbidity and mortality in these patients.
  • In this report we have reviewed our experience with a simultaneous lung and pancreas transplantation in a patient with CF.
  • METHODS: The recipient was a 25-year-old man with CF complicated by bronchiectasis with recurrent episodes of pneumonia, pancreatic exocrine insufficiency, and CFRD.
  • SURGICAL TECHNIQUE: The lung and pancreas allografts were procured from a single cadaveric donor.
  • The pancreas transplantation was performed through a midline incision with systemic venous drainage and proximal enteric exocrine drainage.
  • The pancreas allograft functioned well with normal blood glucose independent of insulin.
  • As a result of the enteric drainage of the pancreas allograft, the patient no longer required supplemental pancreatic enzymes.
  • At 1-year posttransplantation he remains free of supplemental oxygen, insulin, and pancreatic enzyme replacement.
  • CONCLUSION: Simultaneous lung and pancreas transplantation in a patient with CF was performed safely, providing the advantages of normalization of glucose and improved nutrition for a patient requiring lung transplantation.
  • [MeSH-major] Cystic Fibrosis / surgery. Lung Transplantation. Pancreas Transplantation


85. Zacharias T, Jaeck D, Oussoultzoglou E, Neuville A, Bachellier P: Impact of lymph node involvement on long-term survival after R0 pancreaticoduodenectomy for ductal adenocarcinoma of the pancreas. J Gastrointest Surg; 2007 Mar;11(3):350-6
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Impact of lymph node involvement on long-term survival after R0 pancreaticoduodenectomy for ductal adenocarcinoma of the pancreas.
  • Pancreaticoduodenectomy remains the only potentially curative treatment for adenocarcinoma of the pancreas.
  • The aim of this study was to analyze prognostic factors impacting survival after R0 pancreaticoduodenectomy for adenocarcinoma in the head of the pancreas.
  • Between 1995 and 2002, a potentially curative (R0) pancreaticoduodenectomy with pancreatogastrostomy for ductal adenocarcinoma in the head of the pancreas was performed in 81 patients (42 women and 39 men) with a mean age of 64 years (range 35-84).
  • Factors associated with poor survival in multivariate analysis were (1) two or more positive lymph nodes, (2) tumor diameter greater than 30 mm, and (3) age greater than 70 years.
  • The main risk factor associated with poor survival after an R0 pancreaticoduodenectomy for adenocarcinoma in the head of pancreas was lymph node status: The presence of two or more positive lymph nodes was associated with decreased survival.
  • [MeSH-major] Adenocarcinoma / surgery. Carcinoma, Pancreatic Ductal / surgery. Pancreatic Neoplasms / surgery. Pancreaticoduodenectomy

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Am J Surg. 2001 Aug;182(2):120-9 [11574081.001]
  • [Cites] Ann Surg. 1998 Oct;228(4):508-17 [9790340.001]
  • [Cites] Surgery. 1995 Jan;117(1):50-5 [7809836.001]
  • [Cites] Ann Surg. 2002 Sep;236(3):355-66; discussion 366-8 [12192322.001]
  • [Cites] Arch Surg. 2007 Feb;142(2):172-9; discussion 180 [17309969.001]
  • [Cites] Br J Surg. 1993 Dec;80(12):1575-8 [7507785.001]
  • [Cites] Ann Surg. 2003 Jan;237(1):74-85 [12496533.001]
  • [Cites] World J Surg. 1999 Sep;23(9):926-9 [10449822.001]
  • [Cites] Ann Surg. 2002 Aug;236(2):137-48 [12170018.001]
  • [Cites] Hepatogastroenterology. 2002 Jan-Feb;49(43):258-62 [11941970.001]
  • [Cites] Br J Surg. 2005 Sep;92(9):1150-4 [16035134.001]
  • [Cites] Ann Surg. 1990 Apr;211(4):447-58 [2322039.001]
  • [Cites] Am J Surg. 2006 Jun;191(6):726-32; discussion 733-4 [16720139.001]
  • [Cites] Br J Surg. 1996 May;83(5):625-31 [8689203.001]
  • [Cites] J Gastrointest Surg. 2000 Nov-Dec;4(6):567-79 [11307091.001]
  • [Cites] Eur J Cancer. 2004 Mar;40(4):549-58 [14962722.001]
  • [Cites] Dig Surg. 2004;21(3):202-9 [15218236.001]
  • [Cites] Am J Gastroenterol. 2001 Sep;96(9):2609-15 [11569683.001]
  • [Cites] Arch Surg. 2004 Mar;139(3):327-35 [15006893.001]
  • [Cites] Ann Surg. 1999 May;229(5):613-22; discussion 622-4 [10235519.001]
  • [Cites] Br J Surg. 2003 Sep;90(9):1131-6 [12945082.001]
  • [Cites] Arch Pathol Lab Med. 2002 Oct;126(10):1169-73 [12296752.001]
  • [Cites] Ann Surg. 1997 Sep;226(3):248-57; discussion 257-60 [9339931.001]
  • [Cites] J Gastrointest Surg. 2006 Jun;10(6):813-22 [16769537.001]
  • [Cites] Surgery. 1997 Sep;122(3):553-66 [9308613.001]
  • [Cites] World J Surg. 1995 May-Jun;19(3):410-4; discussion 414-5 [7638998.001]
  • [Cites] Hepatogastroenterology. 1998 May-Jun;45(21):855-66 [9684147.001]
  • [Cites] Pancreas. 2003 Apr;26(3):243-9 [12657950.001]
  • [Cites] Ann Surg. 1996 Jun;223(6):718-25; discussion 725-8 [8645045.001]
  • (PMID = 17458610.001).
  • [ISSN] 1091-255X
  • [Journal-full-title] Journal of gastrointestinal surgery : official journal of the Society for Surgery of the Alimentary Tract
  • [ISO-abbreviation] J. Gastrointest. Surg.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  •  go-up   go-down


86. Wayne M, Neragi-Miandoab S, Kasmin F, Brown W, Pahuja A, Cooperman AM: Central pancreatectomy without anastomosis. World J Surg Oncol; 2009;7:67
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • BACKGROUND: Central pancreatectomy has a unique application for lesions in the neck of the pancreas.
  • It preserves the distal pancreas and its endocrine functions.
  • RESULTS: All 10 lesions were in the neck of the pancreas and included: 2 branch intraductal papillary mucinous neoplasms (IPMNs), a mucinous cyst, a lymphoid cyst, 5 neuroendocrine tumors, and a clear cell adenoma.
  • CONCLUSION: Central pancreatectomy without pancreatico-enteric anastomosis for lesions in the neck and proximal pancreas is a safe and effective procedure.
  • [MeSH-major] Adenocarcinoma, Mucinous / surgery. Carcinoma, Pancreatic Ductal / surgery. Carcinoma, Papillary / surgery. Pancreatectomy. Pancreatic Neoplasms / surgery
  • [MeSH-minor] Adult. Aged. Anastomosis, Surgical. Female. Humans. Male. Middle Aged. Neoplasm Invasiveness. Neoplasm Recurrence, Local. Neoplasm Staging. Prognosis. Retrospective Studies. Survival Rate. Treatment Outcome

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] World J Surg. 2001 Apr;25(4):452-60 [11344398.001]
  • [Cites] Ann Surg Oncol. 2008 Aug;15(8):2096-103 [18521682.001]
  • [Cites] Am J Surg. 2002 Mar;183(3):237-41 [11943118.001]
  • [Cites] Hepatogastroenterology. 2002 Nov-Dec;49(48):1713-5 [12397773.001]
  • [Cites] Surgery. 2002 Nov;132(5):836-43 [12464868.001]
  • [Cites] World J Surg. 2003 May;27(5):595-8 [12715230.001]
  • [Cites] Dig Surg. 2004;21(1):48-53 [14707393.001]
  • [Cites] J Am Coll Surg. 2004 Jun;198(6):871-6 [15194067.001]
  • [Cites] J Gastrointest Surg. 2004 Jul-Aug;8(5):532-8 [15239986.001]
  • [Cites] Pancreas. 1989;4(4):496-503 [2668933.001]
  • [Cites] Ann Surg. 1992 Dec;216(6):656-62 [1466619.001]
  • [Cites] Arch Surg. 1996 Mar;131(3):247-52 [8611088.001]
  • [Cites] Arch Surg. 1998 Mar;133(3):327-31 [9517749.001]
  • [Cites] J Clin Gastroenterol. 1999 Jan;28(1):3-10 [9916657.001]
  • [Cites] Mem Acad Chir (Paris). 1957 Nov 13-20;83(27-28):869-71 [13503655.001]
  • [Cites] Rev Gastroenterol Peru. 2005 Oct-Dec;25(4):349-55 [16333390.001]
  • [Cites] Arch Surg. 2006 Mar;141(3):293-9 [16549696.001]
  • [Cites] J Gastrointest Surg. 2006 Jun;10(6):804-12 [16769536.001]
  • [Cites] ANZ J Surg. 2006 Nov;76(11):987-95 [17054548.001]
  • [Cites] World J Surg. 2007 Jan;31(1):164-8; discussion 169-70 [17171499.001]
  • [Cites] Curr Gastroenterol Rep. 2007 Apr;9(2):116-22 [17418056.001]
  • [Cites] Arch Surg. 2008 Feb;143(2):175-80; discussion 180-1 [18283143.001]
  • [Cites] Dig Surg. 2008;25(1):46-51 [18292661.001]
  • [Cites] J Surg Oncol. 2001 Jun;77(2):132-5 [11398167.001]
  • (PMID = 19719851.001).
  • [ISSN] 1477-7819
  • [Journal-full-title] World journal of surgical oncology
  • [ISO-abbreviation] World J Surg Oncol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC2743692
  •  go-up   go-down


87. Yuan Z, Chen J, Zheng Q, Huang XY, Yang Z, Tang J: Heterotopic pancreas in the gastrointestinal tract. World J Gastroenterol; 2009 Aug 7;15(29):3701-3
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Heterotopic pancreas in the gastrointestinal tract.
  • Heterotopic pancreas is defined as pancreatic tissue found outside the usual anatomical location of the pancreas.
  • In this report, a 60-year-old man with carcinoid syndrome caused by heterotopic pancreatic tissue in the duodenum is described, along with a 62-year-old man with abdominal pain caused by heterotopic pancreatic tissue in the gastric antrum.
  • The difficulty of making an accurate diagnosis is highlighted.
  • Although heterotopic pancreas is rare, it should be considered in the differential diagnosis of gastrointestinal stromal tumor.
  • [MeSH-major] Choristoma / diagnosis. Duodenal Diseases / diagnosis. Pancreas. Stomach Diseases / diagnosis

  • MedlinePlus Health Information. consumer health - Small Intestine Disorders.
  • MedlinePlus Health Information. consumer health - Stomach Disorders.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Hepatobiliary Pancreat Dis Int. 2002 May;1(2):299-301 [14612289.001]
  • [Cites] Int J Surg. 2008 Dec;6(6):e52-4 [17400528.001]
  • [Cites] Gastrointest Radiol. 1980 Feb 1;5(1):37-42 [6965644.001]
  • [Cites] G Chir. 1994 Apr;15(4):162-6 [8086304.001]
  • [Cites] Dig Dis Sci. 1996 Jun;41(6):1238-40 [8654158.001]
  • [Cites] Gastrointest Endosc. 1997 Jun;45(6):468-73 [9199902.001]
  • [Cites] JOP. 2004 Nov;5(6):498-501 [15536290.001]
  • [Cites] Med J Malaysia. 2004 Oct;59(4):541-3 [15779591.001]
  • [Cites] Med Sci Monit. 2006 Jun;12(6):CS49-52 [16733487.001]
  • [Cites] World J Surg. 2006 Sep;30(9):1682-9 [16902740.001]
  • [Cites] Diagn Interv Radiol. 2006 Dec;12(4):180-2 [17160801.001]
  • [Cites] JOP. 2007;8(5):588-91 [17873464.001]
  • [Cites] JOP. 2008;9(6):725-32 [18981555.001]
  • [Cites] World J Gastroenterol. 2008 Nov 21;14(43):6757-9 [19034986.001]
  • [Cites] Arch Pathol Lab Med. 2004 Jan;128(1):111-2 [14692822.001]
  • (PMID = 19653355.001).
  • [ISSN] 2219-2840
  • [Journal-full-title] World journal of gastroenterology
  • [ISO-abbreviation] World J. Gastroenterol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] China
  • [Other-IDs] NLM/ PMC2721251
  •  go-up   go-down


88. Tashiro H, Iwata H, Warnock GL, Tanigawa M, Ototani Y, Tsuji T: Functional assessment of microencapsulated porcine islets with agarose polystyrene sulfonic acid in vitro and in xenotransplantation. Transplant Proc; 2005 Oct;37(8):3512-3
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Islets were isolated from the pancreata of CSK miniature swine by manual collagenase digestion and Ficoll purification.

  • MedlinePlus Health Information. consumer health - Islet Cell Transplantation.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16298645.001).
  • [ISSN] 0041-1345
  • [Journal-full-title] Transplantation proceedings
  • [ISO-abbreviation] Transplant. Proc.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Capsules; 0 / Polystyrenes; 0 / Resins, Synthetic; 70KO0R01RY / polystyrene sulfonic acid; 9012-36-6 / Sepharose
  •  go-up   go-down


89. Iglesias ML, Koll F, Delfante A, Sun Ho H, Grosembacher L, Rodota L: [Nutritional assessment of patients candidates for waiting list simultaneous kidney pancreas transplantation]. Nutr Hosp; 2010 May-Jun;25(3):406-13
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Nutritional assessment of patients candidates for waiting list simultaneous kidney pancreas transplantation].
  • [Transliterated title] Evaluación nutricional de pacientes candidatos a lista de espera de trasplante renopancreático.
  • BACKGROUND: Simultaneous Kidney Pancreas transplantation is currently the treatment of choice for patients with type 1 diabetes ERD advanced.
  • [MeSH-major] Kidney Transplantation. Nutrition Assessment. Nutritional Status. Pancreas Transplantation. Waiting Lists


90. Akerblom B, Annerén C, Welsh M: A role of FRK in regulation of embryonal pancreatic beta cell formation. Mol Cell Endocrinol; 2007 May 30;270(1-2):73-8
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] A role of FRK in regulation of embryonal pancreatic beta cell formation.
  • The role of FRK in pancreatic beta cells has been addressed by studies of knockout or FRK transgenic mice.
  • To elucidate whether FRK affects pancreatic beta cell number during embryogenesis and shortly after birth, pancreata were collected from FRK(-/-) mice at these stages.
  • Histological analysis of insulin stained pancreatic sections showed that the insulin positive cell area in FRK(-/-) mice was reduced at embryonal day 15 and at birth to 31 and 70% of that of wild-type mice, respectively.
  • FRK(-/-) pancreas weight on day 1 neonatally was similar to that of the control, indicating that the obtained results were not due to altered pancreatic growth.

  • KOMP Repository. gene/protein/disease-specific - KOMP Repository (subscription/membership/fee required).
  • Mouse Genome Informatics (MGI). Mouse Genome Informatics (MGI) .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17416457.001).
  • [ISSN] 0303-7207
  • [Journal-full-title] Molecular and cellular endocrinology
  • [ISO-abbreviation] Mol. Cell. Endocrinol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Ireland
  • [Chemical-registry-number] EC 2.7.10.2 / Frk protein, mouse; EC 2.7.10.2 / src-Family Kinases
  •  go-up   go-down


91. Wang DB, Wang QB, Chai WM, Chen KM, Deng XX: Imaging features of solid pseudopapillary tumor of the pancreas on multi-detector row computed tomography. World J Gastroenterol; 2009 Feb 21;15(7):829-35
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Imaging features of solid pseudopapillary tumor of the pancreas on multi-detector row computed tomography.
  • AIM: To retrospectively analyze the imaging features of solid-pseudopapillary tumors (SPTs) of the pancreas on multi-detector row computed tomography (MDCT) and define the imaging findings suggestive of malignant potential.
  • METHODS: A total of 24 consecutive cases with surgically and pathologically confirmed SPTs of the pancreas underwent preoperative abdominal MDCT studies in our hospital.
  • RESULTS: Of the 24 cases of SPTs, 11 cases (45.8%) occurred in the pancreatic head and seven (29.1%) in the tail.
  • Eighteen were pathologically diagnosed as benign and six as malignant.
  • MDCT diagnosis of SPTs was well correlated with the surgical and pathological results (Kappa = 0.6, P < 0.05).
  • When the size of the tumor was greater than 6 cm (including 6 cm), the possibilities of vascular (8 vs 1) and capsular invasion (9 vs 0) increased significantly (P < 0.05).
  • Two pathologically benign cases with vascular invasion and disrupted capsule on MDCT presented with local recurrence and hepatic metastases during follow-up about 1 year after the resection of the primary tumors.
  • CONCLUSION: Vascular and capsular invasion with superimposed spread into the adjacent pancreatic parenchyma and nearby structures in SPTs of the pancreas can be accurately revealed by MDCT preoperatively.
  • These imaging findings are predictive of the malignant potential associated with the aggressive behavior of the tumor, even in the pathologically benign cases.
  • [MeSH-major] Pancreatic Neoplasms / radiography
  • [MeSH-minor] Adolescent. Adult. Child. Female. Humans. Jaundice / etiology. Male. Middle Aged. Neoplasm Invasiveness. Pain / etiology. Retrospective Studies. Tomography, X-Ray Computed. Young Adult

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Pediatr Radiol. 2005 Aug;35(8):819-22 [15864574.001]
  • [Cites] Surgery. 2005 Jun;137(6):591-6 [15933625.001]
  • [Cites] Pancreas. 2006 Apr;32(3):276-80 [16628083.001]
  • [Cites] World J Gastroenterol. 2007 Mar 28;13(12):1811-5 [17465471.001]
  • [Cites] JOP. 2008;9(2):150-9 [18326922.001]
  • [Cites] Semin Diagn Pathol. 2000 Feb;17(1):66-80 [10721808.001]
  • [Cites] Ann Surg Oncol. 2002 Jan-Feb;9(1):35-40 [11833495.001]
  • [Cites] J Pediatr Surg. 2002 Sep;37(9):1370-3 [12194139.001]
  • [Cites] AJR Am J Roentgenol. 2004 Feb;182(2):419-25 [14736675.001]
  • [Cites] Am J Surg Pathol. 1979 Feb;3(1):69-75 [534384.001]
  • [Cites] Radiology. 1984 Jan;150(1):39-40 [6689785.001]
  • [Cites] Cancer. 1984 Oct 1;54(7):1469-74 [6467170.001]
  • [Cites] Virchows Arch A Pathol Anat Histopathol. 1990;416(6):535-8 [2110701.001]
  • [Cites] Radiology. 1996 Jun;199(3):707-11 [8637992.001]
  • [Cites] Am J Surg Pathol. 2005 Apr;29(4):512-9 [15767807.001]
  • [Cites] J Am Coll Surg. 2005 Jun;200(6):965-72 [15922212.001]
  • [Cites] JOP. 2006;7(1):131-6 [16407635.001]
  • (PMID = 19230043.001).
  • [ISSN] 2219-2840
  • [Journal-full-title] World journal of gastroenterology
  • [ISO-abbreviation] World J. Gastroenterol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] China
  • [Other-IDs] NLM/ PMC2653382
  •  go-up   go-down


92. Dall'Aglio C, Pedini V, Scocco P, Boiti C, Ceccarelli P: Immunohistochemical evidence of Orexin-A in the pancreatic beta cells of domestic animals. Res Vet Sci; 2010 Oct;89(2):147-9
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Immunohistochemical evidence of Orexin-A in the pancreatic beta cells of domestic animals.
  • A large body of information proves that Orexin-A is present in the pancreatic endocrine cells of humans and laboratory animals; more detailed studies identify Orexin-A-immunopositive cells as beta cells.
  • Because no data have been reported on the pancreas of domestic animals, we investigated the presence and the distribution of cells containing Orexin-A in the pancreas of cattle, sheep and pigs by means of immunohistochemical techniques.
  • Our results, besides showing the presence of Orexin-A in the endocrine pancreas of domestic animals, together with data present in the literature, could contribute to the understanding of complex mechanisms regulating the functionality of the endocrine pancreas in domestic animals.

  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] 2010 Elsevier Ltd. All rights reserved.
  • (PMID = 20359730.001).
  • [ISSN] 1532-2661
  • [Journal-full-title] Research in veterinary science
  • [ISO-abbreviation] Res. Vet. Sci.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / HCRT protein, human; 0 / Intracellular Signaling Peptides and Proteins; 0 / Neuropeptides; 0 / Orexins
  •  go-up   go-down


93. Weiss FU, Halangk W, Lerch MM: New advances in pancreatic cell physiology and pathophysiology. Best Pract Res Clin Gastroenterol; 2008;22(1):3-15
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] New advances in pancreatic cell physiology and pathophysiology.
  • The mammalian pancreas originates from two developing buds on the dorsal and ventral side of the duodenum which fuse and convert into a single mixed gland, composed of exocrine and endocrine cells.
  • In the adult organism, the exocrine pancreas consists of acinar and ductal cells which are organised in a lobular branched tissue architecture and secrete and transport digestive enzymes into the duodenum.
  • Mature endocrine cells, which represent only 1-2% of the pancreatic organ volume, form aggregates of so called islets of Langerhans within the exocrine pancreatic tissue and control glucose homeostasis by secretion of glucagon, insulin and other hormones into the bloodstream.
  • Pancreatitis is the most common and a potentially lethal disorder of the exocrine pancreas with limited therapeutic options.
  • This review will summarise recent advances in our understanding of the physiological mechanisms involved in the early disease processes of the exocrine pancreas.
  • [MeSH-major] Pancreas, Exocrine / physiology. Pancreas, Exocrine / physiopathology. Pancreatitis / physiopathology

  • MedlinePlus Health Information. consumer health - Pancreatitis.
  • COS Scholar Universe. author profiles.
  • Hazardous Substances Data Bank. ETHANOL .
  • Hazardous Substances Data Bank. CHYMOTRYPSIN .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 18206809.001).
  • [ISSN] 1521-6918
  • [Journal-full-title] Best practice & research. Clinical gastroenterology
  • [ISO-abbreviation] Best Pract Res Clin Gastroenterol
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Carrier Proteins; 0 / Enzyme Precursors; 0 / SPINK1 protein, human; 3K9958V90M / Ethanol; 9011-97-6 / Cholecystokinin; EC 3.4.21.1 / Chymotrypsin; EC 3.4.21.2 / chymotrypsin C; EC 3.4.21.4 / Trypsin
  • [Number-of-references] 60
  •  go-up   go-down


94. Alvarez SS, Jiménez LM, Murillo AZ, Gómez IG, Ligero JM, Gómez-Pineda A, Rollán-Landeras E, Cuevas P, Jara-Albarrán A: A new approach for bone marrow-derived stem cells intrapancreatic autotransplantation in diabetic rats. Microsurgery; 2006;26(7):539-42
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The second day, cells were injected, via aortic celiac trunk, into the pancreas of the same rat.
  • Intrapancreatic homing of the injected cells was suggested with methylene blue injection that stained the pancreas, and proved by labeled cells in pancreas sections.
  • Second, we inject the cells in the pancreas of the donor rat.
  • This approach can be applied to experimental diabetes and other pancreatic processes.
  • [MeSH-major] Bone Marrow Cells. Diabetes Mellitus, Experimental / surgery. Pancreas / surgery. Stem Cell Transplantation / methods

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] (c) 2006 Wiley-Liss, Inc.
  • (PMID = 17006956.001).
  • [ISSN] 0738-1085
  • [Journal-full-title] Microsurgery
  • [ISO-abbreviation] Microsurgery
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  •  go-up   go-down


95. Tanaka A, Inoue A, Takeguchi A, Washizu T, Bonkobara M, Arai T: Comparison of expression of glucokinase gene and activities of enzymes related to glucose metabolism in livers between dog and cat. Vet Res Commun; 2005 Aug;29(6):477-85
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Nucleotide sequences of the conserved region of glucokinase (GK) cDNA that contained ATP- and glucose-binding domains were determined in canine liver and feline pancreas for design of the species-specific oligonucleotide primers for reverse transcription-polymerase chain reaction (RT-PCR) analysis.
  • The partial sequences of canine liver GK and feline pancreas GK cDNA were respectively 88% and 89% identical with the rat liver GK cDNA.
  • Expression of GK gene was observed in canine liver and pancreas and feline pancreas with RT-PCR using species specific primers based on the cDNA sequences.
  • [MeSH-minor] Animals. Base Sequence. Blood Glucose. Gene Expression. Male. Pancreas / enzymology. Sequence Homology, Nucleic Acid

  • Hazardous Substances Data Bank. GLUCOSE .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Proc Natl Acad Sci U S A. 1991 Aug 15;88(16):7294-7 [1871135.001]
  • [Cites] J Comp Pathol. 1986 Jul;96(4):357-69 [2874160.001]
  • [Cites] Comp Biochem Physiol C Pharmacol Toxicol Endocrinol. 1998 Jul;120(1):53-6 [9827016.001]
  • [Cites] J Biol Chem. 2001 Oct 19;276(42):38337-40 [11546755.001]
  • [Cites] J Biol Chem. 1987 May 5;262(13):6032-8 [3553185.001]
  • [Cites] Vet Res Commun. 1998 Apr;22(3):187-92 [9618890.001]
  • [Cites] Comp Biochem Physiol Comp Physiol. 1992 Jun;102(2):285-7 [1354578.001]
  • [Cites] Annu Rev Biochem. 1988;57:755-83 [3052289.001]
  • [Cites] J Biol Chem. 1969 Jun 10;244(11):2891-901 [4306287.001]
  • [Cites] Comp Biochem Physiol. 1968 Jun;25(3):903-12 [4992107.001]
  • [Cites] J Biol Chem. 1989 Jan 5;264(1):363-9 [2909525.001]
  • [Cites] Proc Natl Acad Sci U S A. 2003 Sep 2;100(18):10207-12 [12920182.001]
  • [Cites] Lancet. 1957 Aug 24;273(6991):368-70 [13464070.001]
  • [Cites] Am J Physiol. 1984 Jan;246(1 Pt 1):E1-13 [6364828.001]
  • [Cites] J Biol Chem. 1967 Jul 25;242(14):3360-5 [6029444.001]
  • [Cites] Vet Res Commun. 1989;13(6):421-6 [2561034.001]
  • [Cites] Curr Top Cell Regul. 1976;11:1-50 [187384.001]
  • [Cites] Proc Nutr Soc. 1971 Dec;30(3):259-65 [4949352.001]
  • [Cites] Res Vet Sci. 1999 Oct;67(2):205-6 [10502495.001]
  • [Cites] Anal Biochem. 1976 May 7;72:248-54 [942051.001]
  • [Cites] Am J Vet Res. 1993 Mar;54(3):463-7 [8498753.001]
  • [Cites] J Clin Invest. 1994 Mar;93(3):1120-30 [8132752.001]
  • [Cites] J Biol Chem. 1963 Mar;238:1175-7 [13997409.001]
  • [Cites] Proc Natl Acad Sci U S A. 1993 Mar 1;90(5):1932-6 [8446612.001]
  • (PMID = 16215838.001).
  • [ISSN] 0165-7380
  • [Journal-full-title] Veterinary research communications
  • [ISO-abbreviation] Vet. Res. Commun.
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Blood Glucose; EC 2.7.1.2 / Glucokinase; IY9XDZ35W2 / Glucose
  •  go-up   go-down


96. Cheung KK, Coutinho-Silva R, Chan WY, Burnstock G: Early expression of adenosine 5'-triphosphate-gated P2X7 receptors in the developing rat pancreas. Pancreas; 2007 Aug;35(2):164-8
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Early expression of adenosine 5'-triphosphate-gated P2X7 receptors in the developing rat pancreas.
  • OBJECTIVES: Extracellular adenosine 5'-triphosphate modulates the functions of the adult pancreas via 2 nucleotide receptor families, the P2X and P2Y receptors.
  • Expression of the P2X7 receptor has been demonstrated in islet cells of the pancreas, particularly the mature alpha cells that secrete glucagon.
  • METHODS: In the present study, we have examined the expression of P2X7 receptors in the developing pancreas from embryonic days 10 (E10) to E18.
  • RESULTS: We detected P2X7 receptor-immunoreactive cells in pancreatic islet cells as early as E11' before glucagon expression.
  • [MeSH-major] Pancreas / embryology. Receptors, Purinergic P2 / physiology

  • Hazardous Substances Data Bank. GLUCAGON .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17632323.001).
  • [ISSN] 1536-4828
  • [Journal-full-title] Pancreas
  • [ISO-abbreviation] Pancreas
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Insulin; 0 / P2rx7 protein, rat; 0 / Receptors, Purinergic P2; 0 / Receptors, Purinergic P2X7; 8L70Q75FXE / Adenosine Triphosphate; 9007-92-5 / Glucagon
  •  go-up   go-down


97. Kanazawa A, Tanaka H, Hirohashi K, Shuto T, Takemura S, Tanaka S, Hamuro M, Kinoshita H, Kubo S: Pseudoaneurysm of the dorsal pancreatic artery with obstruction of the celiac axis after pancreatoduodenectomy: report of a case. Surg Today; 2005;35(4):332-5
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Pseudoaneurysm of the dorsal pancreatic artery with obstruction of the celiac axis after pancreatoduodenectomy: report of a case.
  • Pancreatoduodenectomy can be difficult in patients with obstruction of the celiac axis because of the development of collateral arteries around the head of the pancreas.
  • We report a case of a pseudoaneurysm of an enlarged dorsal pancreatic artery, which formed as a result of obstruction of the celiac axis after pancreatoduodenectomy.
  • We assumed that a weakness in the wall of this dorsal pancreatic artery caused by the dissection led to the formation of the aneurysm.
  • This case demonstrates that the dissection of arteries that have developed around the head of the pancreas must be considered in patients with obstruction of the celiac axis.
  • [MeSH-major] Aneurysm, False / etiology. Arterial Occlusive Diseases / etiology. Celiac Artery. Pancreas / blood supply. Pancreaticoduodenectomy / adverse effects

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 15815854.001).
  • [ISSN] 0941-1291
  • [Journal-full-title] Surgery today
  • [ISO-abbreviation] Surg. Today
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Japan
  •  go-up   go-down


98. Oki E, Higashi H, Honboh T, Haraoka S, Okuyama T, Yoshida M: Huge gastric carcinoma showing an exophytic growth pattern: a case report and review of the literature. J Gastrointest Cancer; 2008;39(1-4):42-5
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • A radical resection was judged to be impossible preoperatively since the tumor invasion of the pancreas and liver was demonstrated on computed tomography.
  • A pathological examination demonstrated the tumor to directly invade the pancreas and transverse colon; however, no metastasis was observed in the regional lymph nodes.
  • DISCUSSION: To obtain a better prognosis for huge gastric carcinoma showing an exophytic growth pattern, extended radical surgery is recommended since the size of the exophytic mass sometimes does not indicate the extent of the tumor.

  • MedlinePlus Health Information. consumer health - Stomach Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Rinsho Hoshasen. 1988 Sep;33(9):1027-30 [2461457.001]
  • [Cites] AJR Am J Roentgenol. 1994 Jul;163(1):77-80 [8010252.001]
  • [Cites] J Clin Gastroenterol. 1996 Mar;22(2):150-3 [8742659.001]
  • [Cites] J R Coll Surg Edinb. 1993 Feb;38(1):23-7 [8437148.001]
  • [Cites] Gastrointest Radiol. 1988 Oct;13(4):306-8 [3169477.001]
  • [Cites] Abdom Imaging. 1993;18(3):232-3 [8508081.001]
  • [Cites] Gastrointest Radiol. 1983;8(1):11-7 [6601036.001]
  • [Cites] Arch Surg. 1991 Aug;126(8):985-9; discussion 989-90 [1863217.001]
  • [Cites] G Chir. 2001 Jun-Jul;22(6-7):217-21 [11515456.001]
  • [Cites] World J Surg. 2001 Mar;25(3):290-5 [11343178.001]
  • [Cites] Int Surg. 1998 Oct-Dec;83(4):291-3 [10096744.001]
  • [Cites] J Comput Tomogr. 1986 Apr;10 (2):187-90 [3698637.001]
  • [Cites] Clin Imaging. 1993 Jul-Sep;17 (3):210-2 [8364795.001]
  • [Cites] Abdom Imaging. 2000 Sep-Oct;25(5):545-7 [10931995.001]
  • (PMID = 19130312.001).
  • [ISSN] 1941-6628
  • [Journal-full-title] Journal of gastrointestinal cancer
  • [ISO-abbreviation] J Gastrointest Cancer
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  •  go-up   go-down


99. Niu MJ, Yang JK, Lin SS, Ji XJ, Guo LM: Loss of angiotensin-converting enzyme 2 leads to impaired glucose homeostasis in mice. Endocrine; 2008 Aug-Dec;34(1-3):56-61
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Immunohistochemistry was used to investigate ACE2 expression in the pancreas.
  • We found that ACE2 was positively expressed in the pancreas.

  • Hazardous Substances Data Bank. GLUCOSE .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Kidney Int. 2000 Apr;57(4):1493-501 [10760085.001]
  • [Cites] Diabetologia. 2005 Feb;48(2):282-9 [15654602.001]
  • [Cites] J Am Soc Nephrol. 2000 Jul;11(7):1278-86 [10864584.001]
  • [Cites] Hypertension. 2005 Nov;46(5):1169-74 [16203874.001]
  • [Cites] J Am Soc Nephrol. 2006 Nov;17(11):3067-75 [17021266.001]
  • [Cites] Endocrine. 2008 Feb;33(1):32-9 [18392786.001]
  • [Cites] Curr Opin Investig Drugs. 2002 Apr;3(4):569-77 [12090726.001]
  • [Cites] Lancet. 2004 Jun 19;363(9426):2022-31 [15207952.001]
  • [Cites] Diabetes. 2004 Apr;53(4):989-97 [15047614.001]
  • [Cites] J Am Coll Cardiol. 2005 Sep 6;46(5):821-6 [16139131.001]
  • [Cites] Cell. 2005 Aug 12;122(3):337-49 [16096055.001]
  • [Cites] J Biol Chem. 2002 Apr 26;277(17):14838-43 [11815627.001]
  • [Cites] Hypertension. 2003 Mar;41(3):392-7 [12623933.001]
  • [Cites] Biochem J. 2004 Oct 1;383(Pt 1):45-51 [15283675.001]
  • [Cites] Diabetes. 2003 Jan;52(1):102-10 [12502499.001]
  • [Cites] Circ Res. 2006 Mar 3;98(4):463-71 [16514079.001]
  • [Cites] Biochem Biophys Res Commun. 2004 Mar 26;316(1):114-22 [15003519.001]
  • [Cites] Eur J Endocrinol. 2004 Feb;150(2):97-104 [14763905.001]
  • [Cites] J Biol Chem. 2000 Oct 27;275(43):33238-43 [10924499.001]
  • [Cites] J Clin Invest. 2002 May;109(9):1193-201 [11994408.001]
  • [Cites] Diabetes. 2006 Feb;55(2):367-74 [16443769.001]
  • [Cites] Biochem Biophys Res Commun. 2006 Jul 21;346(1):26-32 [16756954.001]
  • [Cites] JAMA. 2001 Oct 17;286(15):1882-5 [11597291.001]
  • [Cites] Endocrine. 2002 Dec;19(3):287-92 [12624428.001]
  • [Cites] Drugs. 2004;64(22):2537-65 [15516153.001]
  • [Cites] Diabetologia. 2004 Feb;47(2):240-8 [14722647.001]
  • [Cites] Am J Pathol. 1999 Aug;155(2):429-40 [10433936.001]
  • [Cites] Am J Pathol. 2006 Jun;168(6):1808-20 [16723697.001]
  • [Cites] Diabetologia. 1998 Feb;41(2):127-33 [9498644.001]
  • [Cites] Nature. 2002 Jun 20;417(6891):822-8 [12075344.001]
  • [Cites] Circ Res. 2002 Sep 6;91(5):406-13 [12215489.001]
  • (PMID = 18956256.001).
  • [ISSN] 1355-008X
  • [Journal-full-title] Endocrine
  • [ISO-abbreviation] Endocrine
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Blood Glucose; 0 / Insulin; EC 3.4.15.1 / Peptidyl-Dipeptidase A; EC 3.4.17.- / angiotensin converting enzyme 2; IY9XDZ35W2 / Glucose
  •  go-up   go-down


100. Kuroki T, Tajima Y, Tsutsumi R, Adachi T, Kitasato A, Hamasaki K, Kanematsu T: Surgical management for stenosis of the pancreaticojejunostomy. Int Surg; 2008 May-Jun;93(3):155-7
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The management of the stenosis of the pancreaticojejunostomy is dictated by the state of the anastomosis and the residual pancreas, endocrine, and exocrine pancreatic function.
  • Four years ago, she was diagnosed with pancreatic injury with a transection of the body of the pancreas.
  • A computed tomography scan showed a pancreatic laceration, and she underwent a Letton-Wilson surgical procedure.
  • [MeSH-minor] Adult. Anastomosis, Surgical / adverse effects. Cholangiopancreatography, Magnetic Resonance. Female. Humans. Pancreas / injuries. Pancreatitis / diagnosis. Pancreatitis / surgery. Recurrence. Tomography, X-Ray Computed

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 18828270.001).
  • [ISSN] 0020-8868
  • [Journal-full-title] International surgery
  • [ISO-abbreviation] Int Surg
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Italy
  •  go-up   go-down






Advertisement