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1
benign neoplasm of kidney disorder 2005:2010[pubdate] *count=100
61161 results
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benign neoplasm of kidney disorder
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Items 1 to 100 of about 61161
1.
Hohenstein B, Kuo MC, Addabbo F, Yasuda K, Ratliff B, Schwarzenberger C, Eckardt KU, Hugo CP, Goligorsky MS:
Enhanced progenitor cell recruitment and endothelial repair after selective endothelial injury of the mouse kidney.
Am J Physiol Renal Physiol
; 2010 Jun;298(6):F1504-14
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[Title]
Enhanced progenitor cell recruitment and endothelial repair after selective endothelial injury of the mouse
kidney
.
Primary and/or secondary injury of
the renal
microvascular endothelium is a common finding in various
renal
diseases.
However, these mechanisms have so far not been studied after selective microvascular injury in the
kidney
.
The present study investigated the time course of EPC and HSC stimulation and homing following induction of selective EC injury in the mouse
kidney
along with various angiogenic factors potentially involved in EC repair and progenitor cell stimulation.
Progenitor cells could be first detected in the circulation and the spleen before they selectively homed to the diseased
kidney
.
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consumer health - Stem Cells
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(PMID = 20237239.001).
[ISSN]
1522-1466
[Journal-full-title]
American journal of physiology. Renal physiology
[ISO-abbreviation]
Am. J. Physiol. Renal Physiol.
[Language]
ENG
[Grant]
United States / NIDDK NIH HHS / DK / R01 DK045462; United States / NIDDK NIH HHS / DK / DK-052783; United States / NIDDK NIH HHS / DK / DK-054602; United States / NIDDK NIH HHS / DK / DK-45462
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Angiogenic Proteins; 11096-26-7 / Erythropoietin; 147336-22-9 / Green Fluorescent Proteins; EC 2.7.10.1 / Receptor, TIE-2
[Other-IDs]
NLM/ PMC2886821
2.
Orr A, Willis S, Holmes M, Britton P, Orr D:
Living with a kidney transplant: a qualitative investigation of quality of life.
J Health Psychol
; 2007 Jul;12(4):653-62
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[Title]
Living with a
kidney
transplant: a qualitative investigation of quality of life.
Research has shown that transplantation improves quality of life for patients with end stage
renal
disease (ESRD), although it does not return to pre-
kidney
failure levels.
This study used focus groups to explore the experience of living with a transplanted
kidney
.
These may necessitate constant vigilance, reduced spontaneity and preoccupation with self-care to maintain the
kidney
's health; being treated differently from others; pressure not to let themselves and others down; and the urge to increase their knowledge about their condition.
[MeSH-major]
Kidney
Transplantation. Quality of Life. Survivors / psychology
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
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(PMID = 17584816.001).
[ISSN]
1359-1053
[Journal-full-title]
Journal of health psychology
[ISO-abbreviation]
J Health Psychol
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
England
3.
Jovanovic V, Giacomelli L, Sivozhelezov V, Degauque N, Lair D, Soulillou JP, Pechkova E, Nicolini C, Brouard S:
AKT1 leader gene and downstream targets are involved in a rat model of kidney allograft tolerance.
J Cell Biochem
; 2010 Oct 15;111(3):709-19
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[Title]
AKT1 leader gene and downstream targets are involved in a rat model
of kidney
allograft tolerance.
We previously showed that administration and anti-donor anti-class II serum on the day of transplantation induce tolerance to a
kidney
allograft in the LEW-1W to LEW-1A strain combination.
In this study, we used DNA microarrays to compare gene patterns involved in anti-donor anti-class II tolerated or untreated syngeneic
kidney
transplants in this strain combination.
Through this model, we showed that AKT1 gene, WNT pathway and NO synthesis are strictly connected to each other and may play an important role in
kidney
tolerance and rejection processes, with AKT1 gene being the center of this complex network of interactions.
[MeSH-major]
Gene Expression Regulation.
Kidney
Transplantation / immunology. Proto-Oncogene Proteins c-akt / genetics. Transplantation Tolerance
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
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[Copyright]
© 2010 Wiley-Liss, Inc.
(PMID = 20607729.001).
[ISSN]
1097-4644
[Journal-full-title]
Journal of cellular biochemistry
[ISO-abbreviation]
J. Cell. Biochem.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Histocompatibility Antigens Class II; 0 / Wnt Proteins; 31C4KY9ESH / Nitric Oxide; EC 2.7.11.1 / Akt1 protein, rat; EC 2.7.11.1 / Proto-Oncogene Proteins c-akt
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4.
Wu YL, Wei C, Wang HT:
[Effect of tongshenluo capsule on the components of extracellular matrix and their metabolism in kidney of rats with diabetic nephropathy].
Zhongguo Zhong Xi Yi Jie He Za Zhi
; 2007 Apr;27(4):326-30
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[Title]
[Effect of tongshenluo capsule on the components of extracellular matrix and their metabolism in
kidney of
rats with diabetic nephropathy].
OBJECTIVE: To investigate the effect of Tongshenluo Capsule (TSL) on the components of extracellular matrix (ECM) and their metabolism in
kidney of
rats with diabetic nephropathy (DN), and to explore its mechanism
of kidney
protecting.
Levels of fasting blood glucose (FBG) and 24-h urinary micro-content of albumin (24 h mAlb) were determined dynamically; the serum glycosyl hemoglobin (HbA1c)was determined after the last medication; the ultrastructural changes
of kidney
were observed by transmission electron microscope; the expressions of collagen IV (IV-C), fibronctin (FN), laminin(
LN
), and the ECM metabolism influencing factors, including MMP-2, tissue inhibitor of metalloproteinase (TIMP-2), transfer growth factor-beta1 (TGF-beta1) in
renal
tissue were detected by immunohistological chemistry and image collecting analytical system.
CONCLUSION: TSL plays a role
of kidney
protection by decreasing the ECM components expression and regulate ECM metabolism.
MedlinePlus Health Information.
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(PMID = 17526172.001).
[ISSN]
1003-5370
[Journal-full-title]
Zhongguo Zhong xi yi jie he za zhi Zhongguo Zhongxiyi jiehe zazhi = Chinese journal of integrated traditional and Western medicine
[ISO-abbreviation]
Zhongguo Zhong Xi Yi Jie He Za Zhi
[Language]
CHI
[Publication-type]
English Abstract; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
China
[Chemical-registry-number]
0 / Capsules; 0 / Collagen Type IV; 0 / Drugs, Chinese Herbal; 0 / Fibronectins; 0 / Laminin
5.
Su W, Fang C, Yang HC, Gu Y, Hao CM:
[Expression of nestin in human kidney and its clinical significance].
Zhonghua Bing Li Xue Za Zhi
; 2008 May;37(5):309-12
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[Title]
[Expression of nestin in human
kidney
and its clinical significance].
OBJECTIVE: To study the expression and significance of nestin (a type of cytoskeletal protein) in normal and diseased human
kidneys
.
METHODS: Diseased
kidney
tissues were obtained from needle biopsies in 32 patients with glomerulonephritis (including 8 cases of membranous glomerulopathy, 3 cases of focal segmental glomerulosclerosis, 17 cases of IgA nephropathy with proteinuria and 4 cases of IgA nephropathy without proteinuria).
Control
kidney
tissues were obtained from nephrectomy specimens for
renal tumors
.
The expression of nestin in the control
kidney
tissues was studied using immunoelectronic microscopy and immunohistochemistry.
The expression of nestin in the diseased
kidney
tissues was detected by immunohistochemistry and real-time reverse transcription-polymerase chain reaction.
RESULTS: In normal
kidney
tissues, nestin was detected at the periphery of glomerular capillary loops.
However, the glomerular nestin expression levels in cases of IgA nephropathy with proteinuria, membranous glomerulopathy and focal segmental glomerulosclerosis were significantly lower than those in the normal
kidneys
and IgA nephropathy without proteinuria.
CONCLUSION: Nestin may play an important role in maintaining the normal function of podocytes in human
kidney
.
[MeSH-minor]
Adult. Gene Expression Regulation. Humans. Immunohistochemistry.
Kidney
.
Kidney
Diseases / metabolism.
Kidney
Diseases / pathology.
Kidney
Glomerulus / metabolism.
Kidney
Glomerulus / pathology. Middle Aged. Nestin. Proteinuria / metabolism
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(PMID = 18956648.001).
[ISSN]
0529-5807
[Journal-full-title]
Zhonghua bing li xue za zhi = Chinese journal of pathology
[ISO-abbreviation]
Zhonghua Bing Li Xue Za Zhi
[Language]
chi
[Publication-type]
English Abstract; Journal Article
[Publication-country]
China
[Chemical-registry-number]
0 / Intermediate Filament Proteins; 0 / NES protein, human; 0 / Nerve Tissue Proteins; 0 / Nestin
6.
Simmons WN, Cocks FH, Zhong P, Preminger G:
A composite kidney stone phantom with mechanical properties controllable over the range of human kidney stones.
J Mech Behav Biomed Mater
; 2010 Jan;3(1):130-3
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[Title]
A composite
kidney
stone phantom with mechanical properties controllable over the range of human
kidney
stones.
A novel composite
kidney
stone phantom has been developed.
This stone phantom is producible with mechanical properties mimicking the range of tensile fracture strength and acoustic properties of human
kidney
stones and is an inorganic/organic composite material, as are natural
kidney
stones.
Diametral compression testing was used to measure tensile fracture strength, which determines the acoustic comminution behavior
of kidney
stones.
Both the tensile fracture strength (controllable from 1 to approximately 5 MPa) and acoustic properties (C(L) = 2700-4400 m/s and C(T)=1600-2300m/s) of these composite phantom stones match those of a wide variety of human
kidney
stones.
[MeSH-major]
Kidney
Calculi / physiopathology. Phantoms, Imaging
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[Cites]
J Acoust Soc Am. 2002 Oct;112(4):1265-8
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J Urol. 1986 Dec;136(6):1367-72
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J Biomed Mater Res. 1989 May;23(5):507-21
[
2715163.001
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(PMID = 19878912.001).
[ISSN]
1878-0180
[Journal-full-title]
Journal of the mechanical behavior of biomedical materials
[ISO-abbreviation]
J Mech Behav Biomed Mater
[Language]
eng
[Grant]
United States / NIDDK NIH HHS / DK / R01 DK052985; United States / NIDDK NIH HHS / DK / R01 DK052985; United States / NIDDK NIH HHS / DK / R37 DK052985
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
Netherlands
[Other-IDs]
NLM/ NIHMS498637; NLM/ PMC3756310
7.
Nezami N, Tarzamni MK, Argani H, Nourifar M:
Doppler ultrasonographic indexes in kidney transplant recipients: its relationship with kidney function.
Iran J Kidney Dis
; 2007 Oct;1(2):82-7
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[Title]
Doppler ultrasonographic indexes in
kidney
transplant recipients: its relationship with
kidney
function.
INTRODUCTION: Doppler ultrasonography (
DU
) is mostly used for assessment of both graft and native
kidneys
' vascular status.
In this study, correlation between
the DU
indexes and
kidney
allograft function was evaluated.
MATERIALS AND METHODS: Hospital records of 273
kidney
transplant patients (154 men and 119 women) were reviewed.
In all cases,
DU
had been performed 1 month after
kidney
transplantation.
We evaluated the data on the resistive index (
RI
) and pulsatility index (PI) in the interlobar arteries and
renal
artery stenosis (RAS), and
renal
vessels thrombosis were determined.
Concurrent serum creatinine and cyclosporine values were assessed in relation to
the DU
findings.
RESULTS:
The RI
and PI had significant linear correlations with serum creatinine (P = .03 and P = .002, respectively).
Also, there were direct linear correlations between the age of the patients and
the RI
and PI values.
Despite this finding,
RI
and PI were significantly lower in patients with RAS than in the patients with patent renovascular tributary (0.59 +/- 0.15 versus 0.65 +/- 0.11; P = .03 and 1.02 +/- 0.40 versus 1.18 +/- 0.46; P = .049, respectively).
There were no associations between serum cyclosporine level or panel reactive antibodies and
the RI
or PI.
CONCLUSIONS:
The RI
and PI are valuable
DU
markers for determining the
kidney
allograft function and the related vascular complications.
[MeSH-major]
Kidney
Transplantation / ultrasonography.
Renal
Artery Obstruction / ultrasonography. Thrombosis / ultrasonography
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(PMID = 19363282.001).
[ISSN]
1735-8582
[Journal-full-title]
Iranian journal of kidney diseases
[ISO-abbreviation]
Iran J Kidney Dis
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Iran
[Chemical-registry-number]
0 / Biomarkers; AYI8EX34EU / Creatinine
8.
Engholm G, Hakulinen T, Gislum M, Tryggvadóttir L, Klint A, Bray F, Storm HH:
Trends in the survival of patients diagnosed with kidney or urinary bladder cancer in the Nordic countries 1964-2003 followed up to the end of 2006.
Acta Oncol
; 2010 Jun;49(5):655-64
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[Title]
Trends in the survival of patients diagnosed with
kidney
or urinary bladder cancer in the Nordic countries 1964-2003 followed up to the end of 2006.
BACKGROUND: Previous studies have shown systematic differences between the Nordic Countries in population-based relative survival following a
kidney
or urinary bladder cancer
diagnosis
.
RESULTS: The survival following bladder cancer was higher than for
kidney
cancer and highest for men.
Survival increased over the years in all countries, more for
kidney
cancer than bladder cancer.
For Danish
kidney
cancer patients, the rate of increase over all the years has been lower than in the other countries, especially among women, resulting in a survival in Denmark some 10-20% points lower than elsewhere in 1999-2003.
The differences were mainly found in the first year following
diagnosis
, where a higher excess mortality in Denmark was observed.
Survival decreased with higher age at
diagnosis
.
CONCLUSION: The increasing 5-year relative survival in all the Nordic countries for both
kidney
and bladder cancer are promising, but for
kidney
cancer a higher percentage detected coincidentally during an imaging investigation for other diseases could play a role.
Denmark had the lowest survival, despite their known practice of including
benign
conditions with invasive bladder cancers.
The lower Danish survival after
kidney
and bladder cancer in the first year after
diagnosis
could be due to later
diagnosis
on average, a higher co-morbidity from smoking-related diseases, and perhaps, less adequate cancer treatment and management in Denmark.
[MeSH-major]
Kidney
Neoplasms
/ mortality. Urinary Bladder
Neoplasms
/ mortality
[MeSH-minor]
Adult. Age Distribution. Aged. Aged, 80 and over. Denmark / epidemiology. Female. Finland / epidemiology. Follow-Up Studies. Humans. Iceland / epidemiology. Incidence. Male. Mass Screening. Middle Aged. Mortality / trends.
Neoplasm
Staging. Norway / epidemiology. Registries. Risk Factors. Survival Analysis. Survival Rate / trends. Sweden / epidemiology
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(PMID = 20156116.001).
[ISSN]
1651-226X
[Journal-full-title]
Acta oncologica (Stockholm, Sweden)
[ISO-abbreviation]
Acta Oncol
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
9.
Weiss AS, Smits G, Wiseman AC:
Standard criteria donor pancreas donation status is associated with improved kidney transplant outcomes.
Clin Transplant
; 2009 Sep-Oct;23(5):732-9
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[Title]
Standard criteria donor pancreas donation status is associated with improved
kidney
transplant outcomes.
BACKGROUND: Organ donor characteristics can be used to predict outcomes in
kidney
transplantation.
We hypothesized that pancreas donation status could reflect organ quality and be predictive
of kidney
graft outcomes following Standard Criteria Donor (SCD)
kidney
transplantation.
METHODS: We performed a retrospective analysis of deceased donor
kidney
alone (DD KA) transplants reported to SRTR from 1992 to 2005.
Group 1 =
kidney
alone recipients from pancreas donors (KA, P+) and Group 2 =
kidney
alone recipients from non-pancreas donors (KA, P-).
CONCLUSION: Donor pancreas donation status is an independent predictor of improved outcomes of SCD
kidney
recipients.
Further study of the pancreas organ donor pre-procurement is warranted to optimize not only pancreas utilization but also
kidney
graft outcomes.
[MeSH-major]
Graft Rejection / prevention & control. Graft Survival / physiology.
Kidney
Transplantation. Pancreas Transplantation. Tissue Donors. Tissue and Organ Procurement / standards
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(PMID = 19732097.001).
[ISSN]
1399-0012
[Journal-full-title]
Clinical transplantation
[ISO-abbreviation]
Clin Transplant
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Denmark
10.
Duffield JS, Park KM, Hsiao LL, Kelley VR, Scadden DT, Ichimura T, Bonventre JV:
Restoration of tubular epithelial cells during repair of the postischemic kidney occurs independently of bone marrow-derived stem cells.
J Clin Invest
; 2005 Jul;115(7):1743-55
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[Title]
Restoration of tubular epithelial cells during repair of the postischemic
kidney
occurs independently of bone marrow-derived stem cells.
Ischemia causes
kidney
tubular cell damage and abnormal
renal
function.
The
kidney
is capable of morphological restoration of tubules and recovery of function.
We studied
kidney
repair in chimeric mice expressing GFP or bacterial beta-gal or harboring the male Y chromosome exclusively in bone marrow-derived cells.
Upon i.v. injection of bone marrow mesenchymal stromal cells, postischemic functional
renal
impairment was reduced, but there was no evidence of differentiation of these cells into tubular cells of the
kidney
.
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[ISSN]
0021-9738
[Journal-full-title]
The Journal of clinical investigation
[ISO-abbreviation]
J. Clin. Invest.
[Language]
ENG
[Grant]
United States / NIDDK NIH HHS / DK / R37 DK039773; United States / NIDDK NIH HHS / DK / DK 39773; United States / NIDDK NIH HHS / DK / DK 38452; United States / NIDDK NIH HHS / DK / P01 DK038452; United States / NIDDK NIH HHS / DK / R01 DK039773
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
[Publication-country]
United States
[Chemical-registry-number]
0 / Recombinant Proteins; 0 / enhanced green fluorescent protein; 147336-22-9 / Green Fluorescent Proteins
[Other-IDs]
NLM/ PMC1159124
11.
Friedman AL, Peters TG, Jones KW, Boulware LE, Ratner LE:
Fatal and nonfatal hemorrhagic complications of living kidney donation.
Ann Surg
; 2006 Jan;243(1):126-30
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[Title]
Fatal and nonfatal hemorrhagic complications of living
kidney
donation.
OBJECTIVE: After anecdotal reports of severe hemorrhage from failure of surgical clips to sustain closure of
renal
artery stumps in live donor nephrectomies were received, this study was designed to identify specific surgical techniques that are associated with an increased risk of failure to control bleeding and might represent opportunities to improve patient safety.
BACKGROUND: Preventing complications for living
kidney
donors must be paramount in addressing end-stage
renal
failure through living
kidney
donation.
Open and laparoscopic approaches to living
kidney
donation use several vascular control methods, some of which may be more prone to failure and life-endangering hemorrhage than others.
METHODS: To define hemorrhagic complications of living
kidney
donation, a survey was sent to all 893 surgeon-members of the American Society of Transplant Surgeons.
Descriptive and bivariate analyses were used to ascertain study participant characteristics, most frequently used vascular control techniques, and incidence of events (death, transfusion, reexploration or conversion to open nephrectomy, or contralateral [remaining
kidney
]
renal
failure).
Among arterial control problems, 2 resulted in donor death and 2 resulted in
renal
failure; 19 episodes required transfusion.
Locking and standard clips applied to
the renal
artery were associated with the greatest risks.
CONCLUSIONS: Significant hemorrhagic complications occur with living
kidney
donation in both open and laparoscopic approaches.
[MeSH-major]
Hemorrhage / etiology.
Kidney
Transplantation / adverse effects. Living Donors. Nephrectomy / adverse effects. Surgical Instruments / adverse effects
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[Cites]
Clin Transplant. 1999 Feb;13(1 Pt 2):108-12
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Clin Transplant. 2000 Aug;14(4 Pt 2):433-8
[
10946784.001
]
[Cites]
Ann Surg. 2001 Aug;234(2):149-64
[
11505060.001
]
[Cites]
Clin Transplant. 2002;16 Suppl 7:62-8
[
12372047.001
]
[Cites]
Am J Transplant. 2002 Nov;2(10):959-64
[
12482149.001
]
[Cites]
Transplant Proc. 2003 Mar;35(2):835-7
[
12644157.001
]
[Cites]
Am J Transplant. 2003 Jul;3(7):775-85
[
12814469.001
]
[Cites]
Am Surg. 1999 Mar;65(3):197-204
[
10075290.001
]
[Cites]
J Urol. 2004 Jan;171(1):47-51
[
14665841.001
]
[Cites]
JAMA. 2004 Jan 21;291(3):325-34
[
14734595.001
]
[Cites]
Ann Surg. 2004 Aug;240(2):205-13
[
15273542.001
]
[Cites]
Ann Surg. 2004 Aug;240(2):358-63
[
15273562.001
]
[Cites]
J Am Coll Surg. 2004 Sep;199(3):374-81
[
15325606.001
]
[Cites]
Surgery. 2004 Oct;136(4):881-90
[
15467675.001
]
[Cites]
N Engl J Med. 1995 Aug 10;333(6):333-6
[
7609748.001
]
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Transplantation. 1995 Nov 15;60(9):1047-9
[
7491680.001
]
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J Am Soc Nephrol. 1996 Nov;7(11):2288-313
[
8959619.001
]
[CommentIn]
Ann Surg. 2006 Oct;244(4):629; author reply 629-30
[
16998373.001
]
(PMID = 16371747.001).
[ISSN]
0003-4932
[Journal-full-title]
Annals of surgery
[ISO-abbreviation]
Ann. Surg.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Other-IDs]
NLM/ PMC1449959
12.
Shoji T, Ishimura E, Nishizawa Y:
Body fat measurement in chronic kidney disease: implications in research and clinical practice.
Curr Opin Nephrol Hypertens
; 2007 Nov;16(6):572-6
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[Title]
Body fat measurement in chronic
kidney
disease: implications in research and clinical practice.
PURPOSE OF REVIEW: The paradoxical and inverse association between body mass index and mortality risk in patients with end-stage
renal
disease has raised a question of whether an increased fat mass is good or bad for patients with chronic
kidney
disease.
The purpose of this review is to update the concept on body fat in patients with chronic
kidney
disease.
Following the initiation of dialysis, chronic
kidney
disease patients gain body weight due mainly to increased fat mass.
In predialysis chronic
kidney
disease, there is also an inverse association between body mass index and mortality risk.
The metabolic syndrome and a high body mass index are independent predictors for development of chronic
kidney
disease and end-stage
renal
disease, respectively.
In diabetic patients with chronic
kidney
disease, however, a high initial body mass index is associated with a slower decline in glomerular filtration rate.
SUMMARY: The impacts of fat mass on survival and
renal
function appear to vary depending upon the absence or presence, and stages of chronic
kidney
disease.
Further research is required for optimal nutritional management and improved outcomes of patients with chronic
kidney
disease.
[MeSH-major]
Adipose Tissue.
Kidney
Failure, Chronic / pathology
[MeSH-minor]
Anthropometry. Humans.
Kidney
Function Tests. Predictive Value of Tests. Survival Rate
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(PMID = 18089973.001).
[ISSN]
1062-4821
[Journal-full-title]
Current opinion in nephrology and hypertension
[ISO-abbreviation]
Curr. Opin. Nephrol. Hypertens.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
England
[Number-of-references]
25
13.
de Klerk M, Zuidema WC, Ijzermans JN, Weimar W:
On chain lengths, domino-paired and unbalanced altruistic kidney donations.
Clin Transpl
; 2009;:247-52
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[Title]
On chain lengths, domino-paired and unbalanced altruistic
kidney
donations.
Kidney
transplantations with living related and unrelated donors are the optimal option for patients with end-stage
renal
disease.
For patients with a willing--but blood-type or HLA incompatible donor--a living-donor
kidney
exchange program could be an opportunity.
In Asia, the United States and Europe,
kidney
exchange programs were developed under different conditions, with different exchange algorithms, and with different match results.
The easiest way to organize a living-donor
kidney
exchange program is to enlist national or regional cooperation, initiated by an independent organization that is already responsible for the allocation of deceased donor organs.
For incompatible donor-recipient pairs who have been unsuccessful in finding suitable matches in an exchange program, domino-paired
kidney
transplantations triggered by Good Samaritan donors is the next alternative.
If no Good Samaritan donors are available, an unbalanced
kidney
paired-exchange program with compatible and incompatible pairs is another strategy that merits future development.
[MeSH-major]
Altruism.
Kidney
Transplantation / statistics & numerical data. Living Donors / psychology
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.
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.
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(PMID = 20524290.001).
[ISSN]
0890-9016
[Journal-full-title]
Clinical transplants
[ISO-abbreviation]
Clin Transpl
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
14.
Gao S, Manns BJ, Culleton BF, Tonelli M, Quan H, Crowshoe L, Ghali WA, Svenson LW, Hemmelgarn BR, Alberta Kidney Disease Network:
Prevalence of chronic kidney disease and survival among aboriginal people.
J Am Soc Nephrol
; 2007 Nov;18(11):2953-9
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[Title]
Prevalence of chronic
kidney
disease and survival among aboriginal people.
Globally, it is known that the incidence of end-stage
renal
disease is higher among Aboriginals, but it is unknown whether this is due to an increased prevalence of chronic
kidney
disease or other unidentified factors.
We studied 658,664 people of non-First Nations and 14,989 people of First Nations and found that the age- and sex-adjusted prevalence of chronic
kidney
disease was significantly higher among those of non-First Nations compared to those of First Nations (67.5 versus 59.5 per 1000 population; P < 0.0001).
However, severe chronic
kidney
disease (estimated glomerular filtration rate <30 ml/min per 1.73 m2) was almost two-fold higher among people of First Nations (P < 0.0001).
Cox proportional hazards models suggested that compared to people of non-First Nations, those of First Nations with chronic
kidney
disease had a 77% increased risk of death after adjusting for age, gender, diabetes and baseline eGFR.
In conclusion, whether the higher incidence of end-stage
renal
disease among people of First Nations is due to suboptimal management of chronic
kidney
disease and its associated comorbidities, more rapid loss
of kidney
function, or other unidentified factors remains to be determined.
[MeSH-major]
Indians, North American / statistics & numerical data.
Kidney
Diseases / ethnology.
Kidney
Diseases / mortality
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.
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(PMID = 17942955.001).
[ISSN]
1533-3450
[Journal-full-title]
Journal of the American Society of Nephrology : JASN
[ISO-abbreviation]
J. Am. Soc. Nephrol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
AYI8EX34EU / Creatinine
15.
Fearnley C, Wakeley PR, Gallego-Beltran J, Dalley C, Williamson S, Gaudie C, Woodward MJ:
The development of a real-time PCR to detect pathogenic Leptospira species in kidney tissue.
Res Vet Sci
; 2008 Aug;85(1):8-16
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[Title]
The development of a real-time PCR to detect pathogenic Leptospira species in
kidney
tissue.
To assess the suitability of these PCR methods for
diagnosis
, a trial was performed on
kidneys
taken from adult pigs with evidence of leptospiral infection, primarily a history of reproductive disease and serological evidence of exposure to pathogenic leptospires (n=180) and aborted pig foetuses (n=24).
In a subsidiary experiment, the 'pathogenic' PCR was used to analyse
kidney
samples from rodents (n=7) collected as part of vermin control in a zoo, with show animals with high microagglutination titres to Leptospira species, and five were positive.
Fifteen PCR amplicons from 1 mouse, 2 rat and 14 pig
kidney
samples, were selected at random from positive PCRs (n=30) and sequenced.
The only successful culture was from this mouse
kidney
and the isolate was confirmed to be L. inadai by classical serology.
[MeSH-major]
Kidney
/ microbiology. Leptospira / isolation & purification. Leptospirosis / veterinary. Polymerase Chain Reaction / veterinary. Swine Diseases / microbiology
[MeSH-minor]
Animals.
Kidney
Diseases / microbiology.
Kidney
Diseases / veterinary. Swine. Temperature
The Lens.
Cited by Patents in
.
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(PMID = 17961617.001).
[ISSN]
0034-5288
[Journal-full-title]
Research in veterinary science
[ISO-abbreviation]
Res. Vet. Sci.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
England
16.
Harirforoosh S, Jamali F:
Effect of inflammation on kidney function and pharmacokinetics of COX-2 selective nonsteroidal anti-inflammatory drugs rofecoxib and meloxicam.
J Appl Toxicol
; 2008 Oct;28(7):829-38
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[Title]
Effect of inflammation on
kidney
function and pharmacokinetics of COX-2 selective nonsteroidal anti-inflammatory drugs rofecoxib and meloxicam.
We studied (a) the effects of inflammation on
the renal
function and pharmacokinetics of rofecoxib and meloxicam;.
(b) whether inflammation could exacerbate reduced electrolytes excretion changes observed with NSAIDs; and (c) the influence of inflammation on distribution of these drugs into the
kidney
.
The ratios of the
kidney
:plasma concentrations were not significantly altered by inflammation following either drug.
Inflammation altered
kidney
function, demonstrated by increases in BUN and plasma creatinine.
Since we have observed similar patterns of the effect of NSAIDs on
kidney
under healthy and inflammatory conditions, one may conclude that inflammation does not exacerbate the adverse effect.
[MeSH-major]
Arthritis, Experimental / physiopathology. Arthritis, Rheumatoid / physiopathology. Cyclooxygenase 2 Inhibitors / pharmacokinetics.
Kidney
/ metabolism.
Kidney
/ physiopathology. Lactones / pharmacokinetics. Sulfones / pharmacokinetics. Thiazines / pharmacokinetics. Thiazoles / pharmacokinetics
[MeSH-minor]
Administration, Oral. Animals. Area Under Curve.
Kidney
Function Tests. Male. Rats. Rats, Sprague-Dawley
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Meloxicam
.
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(PMID = 18344196.001).
[ISSN]
0260-437X
[Journal-full-title]
Journal of applied toxicology : JAT
[ISO-abbreviation]
J Appl Toxicol
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / Cyclooxygenase 2 Inhibitors; 0 / Lactones; 0 / Sulfones; 0 / Thiazines; 0 / Thiazoles; 0QTW8Z7MCR / rofecoxib; 71125-38-7 / meloxicam
17.
Reisaeter AV, Røislien J, Henriksen T, Irgens LM, Hartmann A:
Pregnancy and birth after kidney donation: the Norwegian experience.
Am J Transplant
; 2009 Apr;9(4):820-4
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[Title]
Pregnancy and birth after
kidney
donation: the Norwegian experience.
Reports on pregnancies in
kidney
donors are scarce.
Linkage with the Norwegian
Renal
Registry provided data on pregnancies
of kidney
donors 1967-2002.
A random sample from the Medical Birth Registry was control group, as was pregnancies in
kidney
donors prior to donation.
No differences were observed in the occurrence of adverse pregnancy outcome in
kidney
donors and in the general population in unadjusted analysis.
Our finding of more frequent preeclampsia in pregnancies after
kidney
donation in the secondary analysis must be interpreted with caution, as the number of events was low.
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[CommentIn]
Am J Transplant. 2009 Apr;9(4):661-8
[
19344459.001
]
(PMID = 18853953.001).
[ISSN]
1600-6143
[Journal-full-title]
American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons
[ISO-abbreviation]
Am. J. Transplant.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Denmark
18.
Girgin C, Sezer A, Sahin O, Oder M, Dinçel C:
Giant renal calculus in a solitary functioning kidney.
Urol Int
; 2007;78(1):91-2
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[Title]
Giant
renal
calculus in a solitary functioning
kidney
.
We report a case of giant
renal
calculus 230 x 140 mm in size and weighing 1,350 g in a solitary functioning
kidney
treated by nephrolithotomy.
A giant
renal
calculus in his right
kidney
and atrophic nonfunctioning left
kidney
was diagnosed by ultrasonography, IVP and CT scan.
This case is the largest and the heaviest stone reported in the literature in a solitary functioning
kidney
.
[MeSH-major]
Kidney
Calculi
[MeSH-minor]
Diagnosis
, Differential. Follow-Up Studies. Humans. Male. Middle Aged. Nephrostomy, Percutaneous. Tomography, X-Ray Computed. Urodynamics
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(PMID = 17192742.001).
[ISSN]
0042-1138
[Journal-full-title]
Urologia internationalis
[ISO-abbreviation]
Urol. Int.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Switzerland
19.
Go AS, Lo JC:
Epidemiology of non-dialysis-requiring chronic kidney disease and cardiovascular disease.
Curr Opin Nephrol Hypertens
; 2006 May;15(3):296-302
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[Title]
Epidemiology of non-dialysis-requiring chronic
kidney
disease and cardiovascular disease.
PURPOSE OF REVIEW: To review recent literature about the relationship between non-dialysis-requiring chronic
kidney
disease and cardiovascular disease as well as possible explanatory factors.
This risk is not linearly associated with level
of kidney
function.
Chronic
kidney
disease is associated with a larger burden of traditional vascular risk factors but is also linked to abnormalities in a variety of nontraditional pathways such as dysregulation of mineral metabolism and arterial calcification, vessel stiffness and endothelial dysfunction, insulin resistance, inflammation, malnutrition, and anemia, among others.
Other novel
kidney
-specific proteins (e.g. renalase) may play direct mediating roles.
The relative contribution of these factors to excess cardiovascular disease in chronic
kidney
disease remains unclear.
SUMMARY: Recent evidence demonstrates the importance of non-dialysis-requiring chronic
kidney
disease as a potent predictor of cardiovascular disease and its complications.
Randomized trials should be performed to determine whether modification of traditional and nontraditional risk factors can reduce incident cardiovascular disease as well as which interventions can optimize treatment outcomes in persons with chronic
kidney
disease and cardiovascular disease.
[MeSH-major]
Cardiovascular Diseases / epidemiology.
Kidney
Diseases / epidemiology
[MeSH-minor]
Chronic Disease. Glomerular Filtration Rate. Humans.
Renal
Dialysis. Risk Factors
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(PMID = 16609298.001).
[ISSN]
1062-4821
[Journal-full-title]
Current opinion in nephrology and hypertension
[ISO-abbreviation]
Curr. Opin. Nephrol. Hypertens.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
England
[Number-of-references]
67
20.
Mashiach R, Davidovits M, Eisenstein B, Kidron D, Kovo M, Shalev J, Merlob P, Vardimon D, Efrat Z, Meizner I:
Fetal hyperechogenic kidney with normal amniotic fluid volume: a diagnostic dilemma.
Prenat Diagn
; 2005 Jul;25(7):553-8
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[Title]
Fetal hyperechogenic
kidney
with normal amniotic fluid volume: a diagnostic dilemma.
OBJECTIVE: To determine the prognostic value of sonographically detected fetal hyperechogenic
kidneys
with normal amniotic fluid volume.
METHODS: Seven cases of hyperechogenic fetal
kidneys
were identified by sonography over a 7-year period (1996--2002).
Increased
renal
echogenicity was diagnosed when
the renal
parenchyma was of greater echogenicity than adjacent liver tissue.
RESULTS: Three of the live-born infants had autosomal dominant polycystic
kidney
disease and one had autosomal recessive polycystic
kidney
.
In the remainder, autopsy study revealed multifocal
renal
dysplasia in two cases and normal
kidneys
in one.
CONCLUSIONS: Increased
renal
echogenicity with normal amniotic fluid volume in a fetus without other anomalies is a difficult diagnostic dilemma.
Although it is usually indicative of
renal
parenchymal disease with possible
renal
failure after birth or in early childhood, in some cases, it represents a normal variant. .
[MeSH-major]
Polycystic
Kidney
Diseases / ultrasonography. Ultrasonography, Prenatal
[MeSH-minor]
Amniotic Fluid / ultrasonography.
Diagnosis
, Differential. Female. Humans. Infant, Newborn. Predictive Value of Tests. Pregnancy. Pregnancy Trimester, Second. Pregnancy Trimester, Third
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[Copyright]
Copyright (c) 2005 John Wiley & Sons, Ltd.
[CommentIn]
Prenat Diagn. 2006 Feb;26(2):190-1
[
16470574.001
]
(PMID = 16032764.001).
[ISSN]
0197-3851
[Journal-full-title]
Prenatal diagnosis
[ISO-abbreviation]
Prenat. Diagn.
[Language]
eng
[Publication-type]
Case Reports; Evaluation Studies; Journal Article
[Publication-country]
England
21.
Hansberry MR, Whittier WL, Krause MW:
The elderly patient with chronic kidney disease.
Adv Chronic Kidney Dis
; 2005 Jan;12(1):71-7
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[Title]
The elderly patient with chronic
kidney
disease.
The elderly are a fast growing population in the United States, and they have a high prevalence of chronic
kidney
disease.
The elderly are particularly susceptible to
kidney
damage from age-related declines in glomerular filtration as well as
kidney
damage from chronic disease states such as diabetes mellitus, hypertension, glomerular, and tubulointerstitial disorders.
A significant number of elderly individuals are reaching end-stage
renal
disease that require
renal
replacement therapy.
This expanding population provides a challenge for health-care providers because the elderly are often referred late to a nephrologist, have a shortened survival on
renal
replacement therapy as compared with younger individuals, and suffer from more comorbidities such as cardiovascular disease, malnutrition, and hearing and visual disabilities.
The elderly also have difficulties with dialysis vascular access and often are not candidates for
renal
transplantation.
Despite these obstacles, age alone is not a justification for withholding diagnostic or therapeutic interventions, because many elderly individuals have an improvement in their quality of life and social support once their
kidney
disease is identified and treated.
[MeSH-major]
Kidney
Failure, Chronic / epidemiology
[MeSH-minor]
Age Factors. Aged. Disease Susceptibility / epidemiology. Humans. Prevalence.
Renal
Replacement Therapy / utilization. Risk Factors. United States / epidemiology
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(PMID = 15719336.001).
[ISSN]
1548-5595
[Journal-full-title]
Advances in chronic kidney disease
[ISO-abbreviation]
Adv Chronic Kidney Dis
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Number-of-references]
54
22.
Ruppar TM, Russell CL:
Medication adherence in successful kidney transplant recipients.
Prog Transplant
; 2009 Jun;19(2):167-72
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[Title]
Medication adherence in successful
kidney
transplant recipients.
OBJECTIVE: To explore the medication-taking behavior of successful
kidney
transplant recipients and determine what behaviors were common among this group.
METHODS: Open-ended interviews were conducted by telephone with 19 individuals who had successfully maintained a transplanted
kidney
for 25 years or more.
RESULTS: Four themes emerged as participants described the behaviors they developed to adhere successfully to the immunosuppressive medication required for maintaining their transplanted
kidneys
.
Kidney
transplant recipients identified the importance of developing and maintaining medication-taking skills and routines on medication adherence.
CONCLUSIONS: Interventions focusing on medication-taking skills, habit formation, and resources for problem solving may improve immunosuppressive medication adherence and clinical outcomes in
kidney
transplant recipients.
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Acta Gastroenterol Belg. 2005 Jul-Sep;68(3):347-52
[
16268422.001
]
[Cites]
Kidney Int. 2001 Oct;60(4):1565-70
[
11576374.001
]
(PMID = 19588667.001).
[ISSN]
1526-9248
[Journal-full-title]
Progress in transplantation (Aliso Viejo, Calif.)
[ISO-abbreviation]
Prog Transplant
[Language]
ENG
[Grant]
United States / NCI NIH HHS / CA / U24 CA092656; United States / NCI NIH HHS / CA / R24 CA092656; United States / NINR NIH HHS / NR / P30 NR003979; United States / NINR NIH HHS / NR / P30 NR003979-15; None / None / / P30 NR003979-15
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Immunosuppressive Agents
[Other-IDs]
NLM/ NIHMS266536; NLM/ PMC3071038
23.
Lorenz M, Regele H, Schillinger M, Kletzmayr J, Haidbauer B, Derfler K, Druml W, Böhmig GA:
Peritransplant immunoadsorption: a strategy enabling transplantation in highly sensitized crossmatch-positive cadaveric kidney allograft recipients.
Transplantation
; 2005 Mar 27;79(6):696-701
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[Title]
Peritransplant immunoadsorption: a strategy enabling transplantation in highly sensitized crossmatch-positive cadaveric
kidney
allograft recipients.
BACKGROUND:
Kidney
transplant recipients with a current positive complement-dependent cytotoxicity crossmatch (CDCXM) are at high risk for hyperacute rejection and graft loss.
In this analysis, we evaluated effectiveness of peritransplant IA as an anti-humoral strategy to overcome a current positive CDCXM in presensitized
renal
allograft recipients.
METHODS: Between 1999 and 2003, 40 high risk cadaveric
kidney
allograft recipients (median CDC panel reactive antibody [PRA] level, 77%; number of retransplants, n = 38) were subjected to peritransplant IA with protein A (one pretransplant IA session followed by a course of repeat posttransplant IA sessions) in addition to preemptive antilymphocyte antibody therapy.
CONCLUSION: Our results demonstrate that peritransplant IA enables successful cadaveric
kidney
transplantation in the context of a positive CDCXM.
[MeSH-major]
Graft Survival / immunology. Histocompatibility Testing. Immunosorbents / immunology.
Kidney
Transplantation / immunology
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consumer health - Kidney Transplantation
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
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(PMID = 15785376.001).
[ISSN]
0041-1337
[Journal-full-title]
Transplantation
[ISO-abbreviation]
Transplantation
[Language]
eng
[Publication-type]
Clinical Trial; Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Immunosorbents
24.
Schukfeh N, Kuebler JF, Schirg E, Petersen C, Ure BM, Glüer S:
Dysplastic kidney and not renal agenesis is the commonly associated anomaly in infants with seminal vesicle cyst.
BJU Int
; 2009 Mar;103(6):816-9
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[Title]
Dysplastic
kidney
and not
renal
agenesis is the commonly associated anomaly in infants with seminal vesicle cyst.
OBJECTIVE: To determine whether the association of seminal vesicle cyst (SVC) and
renal
anomaly in young children correlates with previously reported cases of SVCs in adolescent and adult patients, as congenital SVCs, although rare, are frequently described in association with ipsilateral
renal
agenesis, mainly in adolescent and adult patients, whereas reports on SVCs in younger children are sparse.
PATIENTS AND METHODS: We report on nine infants (median age 4 months) with congenital SVCs, all of them associated with ipsilateral dysplastic
kidneys
.
All patients had ultrasonography of
the renal
system and voiding cysto-urethrography.
RESULTS: The SVCs were found incidentally during ultrasonography for
the renal
anomaly.
Three patients had dysplastic and six had multicystic dysplastic
kidneys
.
In previous reported adult cases of SVCs the most common associated
renal
anomaly was agenesis of the ipsilateral
kidney
(25 of 44 cases), whereas only one case of dysplastic
kidney
was reported.
CONCLUSION: As the appearance of
renal
agenesis might result from a former congenital dysplastic
kidney
, our findings indicate that cases of ipsilateral
renal
agenesis in adult patients with congenital SVCs might represent former dysplastic or multicystic dysplastic
kidney
.
[MeSH-major]
Cysts / etiology. Genital Diseases, Male / etiology.
Kidney
/ abnormalities. Multicystic Dysplastic
Kidney
/ complications. Seminal Vesicles
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(PMID = 19040535.001).
[ISSN]
1464-410X
[Journal-full-title]
BJU international
[ISO-abbreviation]
BJU Int.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
England
25.
Jeha GS, Tatevian N, Heptulla RA:
Congenital hypothyroidism in association with Caroli's disease and autosomal recessive polycystic kidney disease: patient report.
J Pediatr Endocrinol Metab
; 2005 Mar;18(3):315-8
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[Title]
Congenital hypothyroidism in association with Caroli's disease and autosomal recessive polycystic
kidney
disease: patient report.
Autosomal recessive polycystic
kidney
disease (ARPKD) is an important
renal
disease of childhood.
Congenital hypothyroidism has been associated with glomerulocystic
kidney
disease, but to date no association has been made with ARPKD.
[MeSH-major]
Caroli Disease / complications. Caroli Disease / genetics. Congenital Hypothyroidism. Hypothyroidism / etiology. Polycystic
Kidney
, Autosomal Recessive / complications. Polycystic
Kidney
, Autosomal Recessive / genetics
Genetic Alliance.
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.
Genetic Alliance.
consumer health - Kidney Disease
.
Genetic Alliance.
consumer health - Polycystic Kidney Disease
.
Genetic Alliance.
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.
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.
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(PMID = 15813611.001).
[ISSN]
0334-018X
[Journal-full-title]
Journal of pediatric endocrinology & metabolism : JPEM
[ISO-abbreviation]
J. Pediatr. Endocrinol. Metab.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
England
26.
Häcker A, Chauhan S, Peters K, Hildenbrand R, Marlinghaus E, Alken P, Michel MS:
Multiple high-intensity focused ultrasound probes for kidney-tissue ablation.
J Endourol
; 2005 Oct;19(8):1036-40
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[Title]
Multiple high-intensity focused ultrasound probes for
kidney
-tissue ablation.
BACKGROUND AND PURPOSE: To investigate
kidney
-tissue ablation by high-intensity focused ultrasound (HIFU) using multiple and single probes.
MATERIALS AND METHODS: Ultrasound beams (1.75 MHz) produced by a piezoceramic element (focal distance 80 mm) were focused at the center of
renal
parenchyma.
Lesion dimensions were examined in perfused and unperfused ex vivo porcine
kidneys
at different power levels (40, 60, and 80 W) and treatment times (4, 6, and 8 seconds).
Lesions in perfused
kidneys
were smaller than those in unperfused
kidneys
.
CONCLUSIONS: Ex vivo,
kidney
-tissue ablation by means of multiple HIFU probes offers significant advantages over single HIFU probes in respect of lesion size and formation.
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(PMID = 16253077.001).
[ISSN]
0892-7790
[Journal-full-title]
Journal of endourology
[ISO-abbreviation]
J. Endourol.
[Language]
ENG
[Publication-type]
Journal Article
[Publication-country]
United States
27.
Langer R, Tóth A, Máthé Z, Remport A, Járay J, Kahan BD:
[Lymphocele and kidney transplantation].
Orv Hetil
; 2007 Aug 5;148(31):1475-80
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[Title]
[Lymphocele and
kidney
transplantation].
INTRODUCTION: Lymphocele is a special complication following
kidney
transplantation.
The authors examined the factors associated with an increased occurrence of clinically significant perinephric fluid collections and/or lymphoceles among sirolimus-treated
renal
transplant recipients.
In both subgroups the serum creatinine levels were elevated at the time of
diagnosis
from a nadir of 179.5 +/- 141.7 to 359.9 +/- 259.6 mmol/l (Group III, sirolimus treated) and from 222.6 +/- 205.9 to 383.7 +/- 255.2 mmol/l (Group IV, sirolimus free).
Despite an Influenza A + Chlamydia pneumonia and acute rejection which was followed by a GI bleeding and stomach resection he fully recovered and is doing well with an excellent
kidney
function a year after.
[MeSH-major]
Immunosuppressive Agents / adverse effects.
Kidney
Transplantation / adverse effects. Lymphocele / etiology
Genetic Alliance.
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.
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consumer health - Kidney Transplantation
.
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.
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PREDNISONE
.
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SIROLIMUS
.
Hazardous Substances Data Bank.
CYCLOSPORIN A
.
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(PMID = 17656338.001).
[ISSN]
0030-6002
[Journal-full-title]
Orvosi hetilap
[ISO-abbreviation]
Orv Hetil
[Language]
hun
[Publication-type]
Case Reports; Comparative Study; English Abstract; Journal Article
[Publication-country]
Hungary
[Chemical-registry-number]
0 / Immunosuppressive Agents; 83HN0GTJ6D / Cyclosporine; MRK240IY2L / Azathioprine; VB0R961HZT / Prednisone; W36ZG6FT64 / Sirolimus
28.
Yaqoob MM:
Acidosis and progression of chronic kidney disease.
Curr Opin Nephrol Hypertens
; 2010 Sep;19(5):489-92
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[Title]
Acidosis and progression of chronic
kidney
disease.
PURPOSE OF REVIEW: Chronic
kidney
disease progressively impairs the ability of
kidneys
to excrete hydrogen ions owing to the reduced capacity of the
kidney
to synthesize ammonia resulting in metabolic acidosis.
There is good experimental evidence that metabolic acidosis contributes to protein energy wasting
disorder
and progression of chronic
kidney
disease (CKD).
RECENT FINDINGS: Three recent publications have confirmed the experimental evidence and the only randomized controlled study of its kind has suggested that the correction of acidosis by sodium bicarbonate in patients with advanced CKD is associated with attenuation of the rate of decline of
renal
function, reduction in the incidence of end stage
renal
disease and improvement of nutritional parameters.
SUMMARY: In light of these recent studies, it appears that this cheap and simple strategy, which is in line with current
renal
recommendations, has the potential of translating into significant economic, quality of life and clinical outcome benefits in an expanding pool of patients with CKD.
[MeSH-major]
Acidosis / complications.
Kidney
Diseases / complications
[MeSH-minor]
Animals. Disease Progression. Glomerular Filtration Rate. Humans.
Kidney
/ physiopathology
Genetic Alliance.
consumer health - Kidney Disease
.
MedlinePlus Health Information.
consumer health - Kidney Diseases
.
The Lens.
Cited by Patents in
.
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(PMID = 20539229.001).
[ISSN]
1473-6543
[Journal-full-title]
Current opinion in nephrology and hypertension
[ISO-abbreviation]
Curr. Opin. Nephrol. Hypertens.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
England
29.
Ishibe S, Cantley LG:
Epithelial-mesenchymal-epithelial cycling in kidney repair.
Curr Opin Nephrol Hypertens
; 2008 Jul;17(4):379-85
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[Title]
Epithelial-mesenchymal-epithelial cycling in
kidney
repair.
PURPOSE OF REVIEW: Tubule repair following acute
kidney
injury involves epithelial de-differentiation followed by cell migration and proliferation and eventual re-differentiation.
RECENT FINDINGS: Epithelial de-differentiation after
kidney
injury or in epithelial culture systems is controlled by secreted factors such as transforming growth factor beta and hepatocyte growth factor as well as cell-cell and cell-matrix interactions.
SUMMARY: This review focuses on the underlying molecular mechanism of epithelial de-differentiation and re-differentiation in tubule repair in vivo and formation in vitro, thus giving insight into possible strategies for improving recovery following acute
kidney
injury and preventing progression to chronic
kidney
disease.
[MeSH-major]
Epithelium / physiology.
Kidney
/ physiology. Mesoderm / physiology
[MeSH-minor]
Animals. Cell Differentiation / physiology. Humans.
Kidney
Diseases / pathology.
Kidney
Diseases / physiopathology.
Kidney
Tubules / growth & development.
Kidney
Tubules / physiology. Wnt Proteins / metabolism
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(PMID = 18660674.001).
[ISSN]
1062-4821
[Journal-full-title]
Current opinion in nephrology and hypertension
[ISO-abbreviation]
Curr. Opin. Nephrol. Hypertens.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
England
[Chemical-registry-number]
0 / Wnt Proteins
[Number-of-references]
58
30.
Yen M, Huang JJ, Teng HL:
Education for patients with chronic kidney disease in Taiwan: a prospective repeated measures study.
J Clin Nurs
; 2008 Nov;17(21):2927-34
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[Title]
Education for patients with chronic
kidney
disease in Taiwan: a prospective repeated measures study.
AIM: To investigate the physical, knowledge and quality of life outcomes of an educational intervention for patients with early stage chronic
kidney
disease.
BACKGROUND: A comprehensive predialysis education care team can be effective in slowing the progression of chronic
kidney
disease.
A predialysis, team-delivered educational intervention covering
renal
function health care, dietary management of
renal
function and the effects of Chinese herb medication on
renal
function was designed and implemented.
Study outcomes included physical indicators, knowledge (
renal
function protection, use of Chinese herbs and
renal
function and diet) and quality of life.
The primary indicator of
renal
function, glomerular filtration rate, remained stable throughout the 12 months of follow-up, despite the relatively older mean age of study participants.
CONCLUSION: A predialysis education care team can provide effective disease-specific knowledge and may help retard deterioration of
renal
function in persons with early-stage chronic
kidney
disease.
[MeSH-major]
Kidney
Diseases / physiopathology. Patient Education as Topic
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consumer health - Kidney Diseases
.
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(PMID = 19012761.001).
[ISSN]
1365-2702
[Journal-full-title]
Journal of clinical nursing
[ISO-abbreviation]
J Clin Nurs
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
31.
Caixeta A, Mehran R:
Evidence-based management of patients undergoing PCI: contrast-induced acute kidney injury.
Catheter Cardiovasc Interv
; 2010 Mar 1;75 Suppl 1:S15-20
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[Title]
Evidence-based management of patients undergoing PCI: contrast-induced acute
kidney
injury.
Contrast-induced acute
kidney
injury (CI-AKI) is one of the leading causes of hospital-acquired acute
kidney
injury.
CI-AKI is highly prevalent in patients with well-known risk factors, including older age, chronic
renal
insufficiency, congestive heart failure, and diabetes.
[MeSH-major]
Angioplasty, Balloon, Coronary. Contrast Media / adverse effects.
Kidney
Diseases / prevention & control. Radiography, Interventional / adverse effects
MedlinePlus Health Information.
consumer health - Kidney Diseases
.
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.
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.
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[Copyright]
(c) 2010 Wiley-Liss, Inc.
(PMID = 20333702.001).
[ISSN]
1522-726X
[Journal-full-title]
Catheterization and cardiovascular interventions : official journal of the Society for Cardiac Angiography & Interventions
[ISO-abbreviation]
Catheter Cardiovasc Interv
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Chemical-registry-number]
0 / Contrast Media; 0 / Protective Agents; 8MDF5V39QO / Sodium Bicarbonate; INU8H2KAWG / Fenoldopam; VTD58H1Z2X / Dopamine; WYQ7N0BPYC / Acetylcysteine
[Number-of-references]
47
32.
Sato S, Mukai Y, Yamate J, Kato J, Kurasaki M, Hatai A, Sagai M:
Effect of polyphenol-containing azuki bean (Vigna angularis) extract on blood pressure elevation and macrophage infiltration in the heart and kidney of spontaneously hypertensive rats.
Clin Exp Pharmacol Physiol
; 2008 Jan;35(1):43-9
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[Title]
Effect of polyphenol-containing azuki bean (Vigna angularis) extract on blood pressure elevation and macrophage infiltration in the heart and
kidney of
spontaneously hypertensive rats.
1. Hypertension is a major risk factor for myocardial infarction and
renal
damage, and it has also been shown to have pro-inflammatory actions that increase the formation of reactive oxygen species.
Therefore, the aim of the present study was to investigate the effect of polyphenol-containing azuki bean extract (ABE) on systolic blood pressure (SBP) and macrophage infiltration in the heart and
kidney of
spontaneously hypertensive rats (SHR).
Tail SBP and macrophage kinetics in the heart and
kidney
were examined.
4. Nicotinamide adenine dinucleotide (NADH) or nicotinamide adenine dinucleotide phosphate (NADPH)-stimulated superoxide (O2-) production was enhanced in the
kidney
and heart in SHR and WKY rats compared with levels in the absence of NADH or NADPH.
The NADPH-stimulated superoxide (O2-) levels in the
kidney
in untreated SHR was significantly higher than that in untreated WKY rats.
5. In immunohistochemical analyses, the number of macrophages in the heart and in the glomeruli and tubulointerstitium of the
kidney
was significantly higher in ABE-untreated SHR than in ABE-untreated WKY rats.
There were significant positive correlations between SBP and the number of ED1-positive macrophages in the heart and tubulointerstitial and glomerular areas of the
kidney
in WKY rats and SHR.
6. In conclusion, the results of the present study suggest that ABE attenuates the elevation of SBP and macrophage infiltration in the heart, as well as in the glomeruli and tubulointerstitium of the
kidney
, in our SHR model.
[MeSH-major]
Antihypertensive Agents / pharmacology. Blood Pressure / drug effects. Fabaceae. Flavonoids / pharmacology. Heart / drug effects. Hypertension / drug therapy.
Kidney
/ drug effects. Macrophages / drug effects. Phenols / pharmacology
MedlinePlus Health Information.
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.
MedlinePlus Health Information.
consumer health - High Blood Pressure
.
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(PMID = 18047626.001).
[ISSN]
1440-1681
[Journal-full-title]
Clinical and experimental pharmacology & physiology
[ISO-abbreviation]
Clin. Exp. Pharmacol. Physiol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Australia
[Chemical-registry-number]
0 / Antihypertensive Agents; 0 / Flavonoids; 0 / Phenols; 0 / Plant Extracts; 0 / Polyphenols; 0 / Thiobarbituric Acid Reactive Substances; 0U46U6E8UK / NAD; 11062-77-4 / Superoxides
33.
Cozzolino M, Malindretos P:
The role of vitamin D receptor activation in chronic kidney disease.
Hippokratia
; 2010 Jan;14(1):7-9
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[Title]
The role of vitamin D receptor activation in chronic
kidney
disease.
Observational data indicate that there is a close inter-relationship between progressive
renal
dysfunction in patients with chronic
kidney
disease cardiovascular disease and mortality.
Continuously evidence indicates that deficiencies in vitamin D receptor activation represents one of key players in adversely affecting cardiovascular health, as well as inducing to secondary hyperparathyroidism in chromic
kidney
disease patients.
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[
15037495.001
]
(PMID = 20411052.001).
[ISSN]
1790-8019
[Journal-full-title]
Hippokratia
[ISO-abbreviation]
Hippokratia
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Greece
[Other-IDs]
NLM/ PMC2843577
[Keywords]
NOTNLM ; cardiovascular disease / haemodialysis / vitamin D
34.
Danovitch GM, Delmonico FL:
The prohibition of kidney sales and organ markets should remain.
Curr Opin Organ Transplant
; 2008 Aug;13(4):386-94
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[Title]
The prohibition
of kidney
sales and organ markets should remain.
RECENT FINDINGS: In 'natural experiments' performed in developing countries the outcome for
kidney
vendors, in terms of both their medical and psychosocial health, has been shown to be poor.
SUMMARY: Commercialization of living
kidney
donation does not serve the interests of the donors, endangers the health of recipients, and undermines the healthy development of the international transplant endeavor.
[MeSH-major]
Financing, Organized / legislation & jurisprudence. Government Regulation. Health Policy / economics.
Kidney
Transplantation / economics. Living Donors / legislation & jurisprudence. Tissue and Organ Procurement / economics
[MeSH-minor]
Altruism. Cost-Benefit Analysis. Crime / prevention & control. Gift Giving. Health Care Costs. Health Services Accessibility / economics. Health Services Accessibility / legislation & jurisprudence. Health Services Needs and Demand / economics. Health Services Needs and Demand / legislation & jurisprudence. Humans. Motivation. Organizational Objectives. Public Opinion.
Renal
Dialysis / economics. Risk Assessment. United States. Waiting Lists
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(PMID = 18685334.001).
[ISSN]
1531-7013
[Journal-full-title]
Current opinion in organ transplantation
[ISO-abbreviation]
Curr Opin Organ Transplant
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Number-of-references]
37
35.
Bang H, Vupputuri S, Shoham DA, Klemmer PJ, Falk RJ, Mazumdar M, Gipson D, Colindres RE, Kshirsagar AV:
SCreening for Occult REnal Disease (SCORED): a simple prediction model for chronic kidney disease.
Arch Intern Med
; 2007 Feb 26;167(4):374-81
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[Title]
SCreening for Occult
REnal
Disease (SCORED): a simple prediction model for chronic
kidney
disease.
BACKGROUND: Despite the wide availability and low cost of serum creatinine measurement, at-risk populations are not routinely tested for chronic
kidney
disease (CKD).
METHODS: We used a cross-sectional analysis of a nationally representative, population-based survey to develop a system, SCORED (SCreening for Occult
REnal
Disease), that uses routinely available demographic and medical information to identify individuals with an increased likelihood of CKD.
Chronic
kidney
disease was defined as a glomerular filtration rate less than 60 mL/min per 1.73 m(2).
CONCLUSION: This scoring system, weighted toward common variables associated with CKD, may be a useful tool to identify individuals with a high likelihood of occult
kidney
disease.
[MeSH-major]
Creatinine / blood.
Kidney
Failure, Chronic /
diagnosis
. Mass Screening / methods. Models, Biological. Population Surveillance
Genetic Alliance.
consumer health - Kidney Disease
.
MedlinePlus Health Information.
consumer health - Creatinine
.
MedlinePlus Health Information.
consumer health - Kidney Failure
.
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(PMID = 17325299.001).
[ISSN]
0003-9926
[Journal-full-title]
Archives of internal medicine
[ISO-abbreviation]
Arch. Intern. Med.
[Language]
eng
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
United States
[Chemical-registry-number]
AYI8EX34EU / Creatinine
36.
Barbari AG, Masri MA, Stephan AG, El Ghoul B, Rizk S, Mourad N, Kamel GS, Kilani HE, Karam AS:
Cyclosporine lymphocyte maximum level monitoring in de novo kidney transplant patients: a prospective study.
Exp Clin Transplant
; 2006 Jun;4(1):400-5
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[Title]
Cyclosporine lymphocyte maximum level monitoring in de novo
kidney
transplant patients: a prospective study.
OBJECTIVES: To determine prospectively the temporal variations of cyclosporine-A lymphocyte maximum level, whole blood maximum concentration, and total lymphocyte count in patients with de novo
kidney
transplantation.
MATERIALS AND METHODS: Lymphocyte maximum level, whole blood maximum concentration, and total lymphocyte count were prospectively measured in 35 patients at 1, 2, and 3 months after
kidney
transplantation.
Cyclosporine-A lymphocyte maximum level seems to offer a more reliable alternative than does whole blood maximum concentration for cyclosporine-A monitoring in patients with
kidney
transplantation.
[MeSH-major]
Cyclosporine / blood. Immunosuppressive Agents / blood.
Kidney
Transplantation. Lymphocytes / metabolism
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.
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(PMID = 16827634.001).
[ISSN]
1304-0855
[Journal-full-title]
Experimental and clinical transplantation : official journal of the Middle East Society for Organ Transplantation
[ISO-abbreviation]
Exp Clin Transplant
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Turkey
[Chemical-registry-number]
0 / Immunosuppressive Agents; 83HN0GTJ6D / Cyclosporine
37.
van der Mei SF, van Sonderen EL, van Son WJ, de Jong PE, Groothoff JW, van den Heuvel WJ:
Social participation after successful kidney transplantation.
Disabil Rehabil
; 2007 Mar 30;29(6):473-83
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[Title]
Social participation after successful
kidney
transplantation.
PURPOSE: To explore and describe the degree of social participation after
kidney
transplantation and to examine associated factors.
METHOD: A cross-sectional study on 239 adult patients 1-7.3 years after
kidney
transplantation was performed via in-home interviews on participation in obligatory activities (i.e., employment, education, household tasks) and leisure activities (volunteer work, assisting others, recreation, sports, clubs/associations, socializing, going out).
RESULTS:
Kidney
transplantation patients had a lower educational level, spent less time on obligatory activities, had part-time jobs more often, and participated less in sports compared to a control group from the general population.
CONCLUSIONS: Although
kidney
transplantation patients participate less in employment and sports, they do participate in household tasks, volunteer work, going out, socializing and other leisure activities.
[MeSH-major]
Activities of Daily Living. Employment. Interpersonal Relations.
Kidney
Transplantation. Leisure Activities
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consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Family Issues
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
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(PMID = 17364802.001).
[ISSN]
0963-8288
[Journal-full-title]
Disability and rehabilitation
[ISO-abbreviation]
Disabil Rehabil
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
38.
Hahn H:
Genetics of kidney development: pathogenesis of renal anomalies.
Korean J Pediatr
; 2010 Jul;53(7):729-34
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[Title]
Genetics
of kidney
development: pathogenesis of
renal
anomalies.
Congenital anomalies of the
kidney
and urinary tract (CAKUT) account for more than 50% of abdominal masses found in neonates and involve about 0.5% of all pregnancies.
CAKUT has a major role in
renal
failure, and increasing evidence suggests that certain abnormalities predispose to the development of hypertension and cardiovascular disease in adulthood.
To understand the pathogenesis of human
renal
anomalies, understanding the development
of kidney
is important.
Diverse anomalies of the
kidney
corresponding to defects at a particular stage of development have been documented recently; however, more research is required to understand the molecular networks underlying
kidney
development, and such an investigation will provide a clue to the therapeutic intervention for CAKUT.
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(PMID = 21189947.001).
[ISSN]
2092-7258
[Journal-full-title]
Korean journal of pediatrics
[ISO-abbreviation]
Korean J Pediatr
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Korea (South)
[Other-IDs]
NLM/ PMC3004483
[Keywords]
NOTNLM ; Congenital anomalies / Development / Kidney / Urinary tract
39.
Negri AL:
Hereditary hypophosphatemias: new genes in the bone-kidney axis.
Nephrology (Carlton)
; 2007 Aug;12(4):317-20
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[Title]
Hereditary hypophosphatemias: new genes in the bone-
kidney
axis.
Hypophosphatemia due to isolated
renal
phosphate wasting is a genetically heterogeneous disease.
Hereditary hypophosphatemic rickets with hypercalciuria is another rare
disorder of
autosomal recessive inheritance.
The disease was mapped to a 1.6 Mbp region on chromosome 9q34, which contains SLC34A3, the gene encoding
the renal
sodium-phosphate cotransporter NaPi-IIc.
Thus, DMP1 and NaPi-IIc add two new members to the bone-
kidney
axis proposed since it was discovered that the first phosphatonin, FGF23, was of osteoblastic/osteocyte origin.
This provides a mechanism for the skeleton to communicate with the
kidney
to coordinate the mineralization of extracelular matrix and
the renal
handling of phosphate.
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(PMID = 17635744.001).
[ISSN]
1320-5358
[Journal-full-title]
Nephrology (Carlton, Vic.)
[ISO-abbreviation]
Nephrology (Carlton)
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
Australia
[Chemical-registry-number]
0 / DMP1 protein, human; 0 / Extracellular Matrix Proteins; 0 / Phosphoproteins; 0 / SLC34A3 protein, human; 0 / Sodium-Phosphate Cotransporter Proteins, Type IIc
[Number-of-references]
22
40.
Moody EM, Clemens KK, Storsley L, Waterman A, Parikh CR, Garg AX, Donor Nephrectomy Outcomes Research (Donor) Network:
Improving on-line information for potential living kidney donors.
Kidney Int
; 2007 May;71(10):1062-70
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[Title]
Improving on-line information for potential living
kidney
donors.
Individuals who consider becoming living
kidney
donors often search the internet for reliable information before contacting the transplant center.
Eighty-six unique websites on living
kidney
donation were found.
[MeSH-major]
Information Dissemination. Internet.
Kidney
Transplantation. Living Donors
MedlinePlus Health Information.
consumer health - Evaluating Health Information
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Organ Donation
.
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(PMID = 17361119.001).
[ISSN]
0085-2538
[Journal-full-title]
Kidney international
[ISO-abbreviation]
Kidney Int.
[Language]
eng
[Grant]
United States / NIDDK NIH HHS / DK / K23-DK064689
[Publication-type]
Evaluation Studies; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
United States
41.
Colović R, Colović N, Grubor N, Radak V, Micev M, Stojković M:
[Bilateral angiomyolipoma of the kidney in patient with tuberous sclerosis].
Srp Arh Celok Lek
; 2005 Sep-Oct;133(9-10):433-7
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[Title]
[Bilateral angiomyolipoma of the
kidney
in patient with tuberous sclerosis].
Angiomyolipomas are relatively frequent tumours of the
kidney
.
It is believed that about 10 million people worldwide have such a
tumour
.
Due to the
benign
nature of angiomyolipomas, surgical treatment and embolisation of the tumours are generally not recommended, unless
renal
function is endangered, the symptoms are severe, or the
kidney
in question becomes completely dysfunctional.
We present a 24-year-old woman with tuberous sclerosis in whom bilateral
kidney
tumours were diagnosed 7 years earlier and in whom we carried out a left nephrectomy of a 5300 gram angiomyolipoma, which caused pain and complete loss of function.
Although tumourous, the right
kidney
was functional, so it was left untouched.
After an uneventful recovery, a close follow-up was recommended, as well as HLA typing, as it is highly probable that the right
kidney
will gradually become inadequate or completely dysfunctional, so that haemodialysis and/or
kidney
transplantation along with nephrectomy will become necessary.
[MeSH-major]
Angiomyolipoma / complications.
Kidney
Neoplasms
/ complications. Tuberous Sclerosis / complications
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.
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(PMID = 16640189.001).
[ISSN]
0370-8179
[Journal-full-title]
Srpski arhiv za celokupno lekarstvo
[ISO-abbreviation]
Srp Arh Celok Lek
[Language]
srp
[Publication-type]
Case Reports; English Abstract; Journal Article
[Publication-country]
Serbia and Montenegro
42.
Al-Taan OS, Featherstone JM, Rees AM, Young WT, Stephenson TP:
Renal cell carcinoma in a horseshoe kidney presenting as an acute, left sided varicocele.
Int Urol Nephrol
; 2007;39(2):369-71
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[Title]
Renal
cell carcinoma in a horseshoe
kidney
presenting as an acute, left sided varicocele.
We present a rare case of
renal
cell carcinoma (RCC) in a horseshoe
kidney
presenting as an acute left sided varicocele.
A left sided varicocele is a well-described presentation of RCC, usually caused by
tumour
thrombus extending along
the renal
vein with resultant testicular vein occlusion.
However, in our case a
tumour
in the lower pole of a horseshoe
kidney
caused an acute varicocele by direct involvement and occlusion of the testicular vein.
[MeSH-major]
Carcinoma,
Renal
Cell / complications.
Kidney
/ abnormalities.
Kidney
Neoplasms
/ complications. Varicocele / etiology
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[Cites]
Eur Urol. 1998;33(2):175-9
[
9519360.001
]
[Cites]
Urology. 1976 Aug;8(2):146-8
[
960345.001
]
(PMID = 16835726.001).
[ISSN]
0301-1623
[Journal-full-title]
International urology and nephrology
[ISO-abbreviation]
Int Urol Nephrol
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Netherlands
43.
Shah SV, Rajapurkar MM:
The role of labile iron in kidney disease and treatment with chelation.
Hemoglobin
; 2009;33(5):378-85
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[Title]
The role of labile iron in
kidney
disease and treatment with chelation.
There are two major forms
of kidney
disease: acute
renal
failure [also referred to as acute
kidney
injury (AKI)] and chronic
kidney
disease (CKD).
Acute
renal
failure is an abrupt loss
of kidney
function within 48 h, whereas CKD is a loss
of kidney
function greater than 3 months.
There is a large amount of experimental evidence for an increase of labile iron in a wide variety of models
of kidney
disease.
These observations suggest that iron chelators may provide a new modality of prevention and treatment
of kidney
disease.
[MeSH-major]
Acute
Kidney
Injury / drug therapy. Acute
Kidney
Injury / metabolism. Iron / metabolism. Iron Chelating Agents / therapeutic use.
Kidney
Diseases / drug therapy.
Kidney
Diseases / metabolism
Genetic Alliance.
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.
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(PMID = 19821781.001).
[ISSN]
1532-432X
[Journal-full-title]
Hemoglobin
[ISO-abbreviation]
Hemoglobin
[Language]
eng
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
[Publication-country]
England
[Chemical-registry-number]
0 / Iron Chelating Agents; E1UOL152H7 / Iron
44.
Palmer BF:
Hypertension management in patients with chronic kidney disease.
Curr Hypertens Rep
; 2008 Oct;10(5):367-73
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[Title]
Hypertension management in patients with chronic
kidney
disease.
Hypertension is one of the major risk factors for the development and progression of chronic
kidney
disease.
The loss of
renal
function leads to impaired
renal
autoregulation and renders the
kidney
vulnerable to the damaging effects of uncontrolled hypertension.
Mounting evidence indicates that angiotensin-converting enzyme inhibitors and angiotensin receptor blockers slow the progression of chronic
kidney
disease through effects beyond lowering blood pressure.
Studies are needed to determine whether high doses of the single agent or combination therapy is most effective in providing
renal
protection.
Urinary protein excretion is a useful tool for monitoring and titrating therapy to maximize
renal
protection.
Changes in the serum creatinine concentration and hyperkalemia are complications of antihypertensive therapy in patients with chronic
kidney
disease that can be successfully managed to allow continued use of renin-angiotensin blockade.
[MeSH-major]
Hypertension / prevention & control.
Renal
Insufficiency, Chronic / etiology.
Renal
Insufficiency, Chronic / prevention & control
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.
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.
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Am J Med Sci. 2001 Jun;321(6):388-400
[
11417753.001
]
(PMID = 18775113.001).
[ISSN]
1534-3111
[Journal-full-title]
Current hypertension reports
[ISO-abbreviation]
Curr. Hypertens. Rep.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Chemical-registry-number]
0 / Antihypertensive Agents; EC 3.4.23.15 / Renin
[Number-of-references]
40
45.
López Gómez JM:
[Management of anemia in chronic kidney disease].
Nefrologia
; 2008;28 Suppl 3:63-6
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[Title]
[Management of anemia in chronic
kidney
disease].
[Transliterated title]
Manejo de la anemia en la enfermedad
renal
cronica.
1. All patients with anemia secondary to CKD should be treated and evaluated for possible treatment, irrespective of underlying disease, associated comorbidity or possibility
of kidney
replacement therapy.
Normalization of Hb levels in CKD is associated with an improvement in health-related quality of life, but without differences in mortality or the rate of loss
of kidney
function (Strength of Recommendation A).
[MeSH-major]
Anemia / drug therapy. Anemia / etiology.
Kidney
Diseases / complications
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.
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.
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.
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(PMID = 19018741.001).
[ISSN]
0211-6995
[Journal-full-title]
Nefrología : publicación oficial de la Sociedad Española Nefrologia
[ISO-abbreviation]
Nefrologia
[Language]
spa
[Publication-type]
English Abstract; Journal Article; Practice Guideline
[Publication-country]
Spain
46.
Wołyniec W, Zdrojewski Z, Rutkowski B:
[Metabolic acidosis after kidney transplantation].
Przegl Lek
; 2005;62(1):68-71
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[Title]
[Metabolic acidosis after
kidney
transplantation].
Metabolic acidosis is a common complication in patients after
kidney
transplantation.
It is caused by inability of transplanted
kidney
to regenerate bicarbonate buffer.
Although every type of acidosis may be present in these patients, the most frequent are
renal
tubular acidosis and uremic acidosis.
Ketoacidosis due to a post-transplant diabetes mellitus and bicarbonate loss in patients after simultaneous pancreas/
kidney
transplantation are rarely observed.
The proper
diagnosis
and appropriate treatment with oral supplements are essential.
[MeSH-major]
Acidosis,
Renal
Tubular / metabolism.
Kidney
Transplantation / adverse effects
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(PMID = 16053226.001).
[ISSN]
0033-2240
[Journal-full-title]
Przegla̧d lekarski
[ISO-abbreviation]
Prz. Lek.
[Language]
pol
[Publication-type]
English Abstract; Journal Article; Review
[Publication-country]
Poland
[Chemical-registry-number]
0 / Bicarbonates
[Number-of-references]
28
47.
el-Assmy A, el-Nahas AR, Hekal IA, Badran M, Youssef RF, Sheir KZ:
Long-term effects of extracorporeal shock wave lithotripsy on renal function: our experience with 156 patients with solitary kidney.
J Urol
; 2008 Jun;179(6):2229-32
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[Title]
Long-term effects of extracorporeal shock wave lithotripsy on
renal
function: our experience with 156 patients with solitary
kidney
.
PURPOSE: We studied the long-term impact of shock wave lithotripsy on
renal
function, stone recurrence and hypertension in patients with a solitary
kidney
.
Patients with a solitary
kidney
provide a unique opportunity to evaluate any clinically significant change in
renal
function.
MATERIALS AND METHODS: We retrospectively reviewed the records of 156 patients with stones in a solitary
kidney
treated with shock wave lithotripsy monotherapy.
Serum creatinine, systolic and diastolic blood pressure, new onset hypertension, calculated glomerular filtration rate, and
kidney
morphology were determined before and after treatment, and compared by chi-square, paired and unpaired t tests.
Renal
obstruction caused by steinstrasse after shock wave lithotripsy occurred in 14 (8.9%) patients.
CONCLUSIONS: The demonstrated effectiveness, small number of complications at short-term followup, insignificant effect on
renal
function, blood pressure and relatively small number of recurrences at the long-term followup confirm that shock wave lithotripsy is not only effective but is also safe in the long run.
[MeSH-major]
Kidney
/ abnormalities.
Kidney
/ physiopathology.
Kidney
Calculi / physiopathology.
Kidney
Calculi / therapy.
Kidney
Failure, Chronic
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(PMID = 18423733.001).
[ISSN]
1527-3792
[Journal-full-title]
The Journal of urology
[ISO-abbreviation]
J. Urol.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
48.
Ekiel AM, Pietrzak B, Kamiński P, Dolezych H, Jóźwiak J, Martirosian G:
Prevalence of urogenital mycoplasmas and ureaplasmas in women after kidney transplantation.
Transplantation
; 2009 Mar 27;87(6):848-51
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[Title]
Prevalence of urogenital mycoplasmas and ureaplasmas in women after
kidney
transplantation.
BACKGROUND: The prevalence of urogenital mycoplasmas and ureaplasmas in
kidney
transplant and hemodialyzed patients was studied.
CONCLUSION: Our observation showed the necessity of careful examination of possible atypical pathogens in diagnostic materials from hemodialyzed and
kidney
transplant patients.
[MeSH-major]
Female Urogenital Diseases / epidemiology.
Kidney
Transplantation / adverse effects. Mycoplasma Infections / epidemiology. Postoperative Complications / microbiology. Ureaplasma Infections / epidemiology
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.
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(PMID = 19300187.001).
[ISSN]
1534-6080
[Journal-full-title]
Transplantation
[ISO-abbreviation]
Transplantation
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
49.
Peters J:
Secretory and cytosolic (pro)renin in kidney, heart, and adrenal gland.
J Mol Med (Berl)
; 2008 Jun;86(6):711-4
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[Title]
Secretory and cytosolic (pro)renin in
kidney
, heart, and adrenal gland.
Renin is commonly known as a secretory glycoprotein, which is expressed, stored, and secreted in a regulated manner by the
kidney
.
The rat
kidney
exclusively expresses secretory renin.
[MeSH-major]
Adrenal Glands / metabolism. Cytosol / metabolism.
Kidney
/ metabolism. Myocardium / metabolism. Renin / metabolism
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(PMID = 18368380.001).
[ISSN]
0946-2716
[Journal-full-title]
Journal of molecular medicine (Berlin, Germany)
[ISO-abbreviation]
J. Mol. Med.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
Germany
[Chemical-registry-number]
EC 3.4.23.15 / Renin
[Number-of-references]
30
50.
Wesson JA, Ward MD:
Role of crystal surface adhesion in kidney stone disease.
Curr Opin Nephrol Hypertens
; 2006 Jul;15(4):386-93
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[Title]
Role of crystal surface adhesion in
kidney
stone disease.
These studies have produced new insight into the specific chemical interactions that regulate
kidney
stone formation.
SUMMARY: The low adhesion force for calcium oxalate dihydrate predicts a decreased ability of these crystals to aggregate or attach to cells, and correlates with the relative absence of calcium oxalate dihydrate in
kidney
stones.
[MeSH-major]
Calcium Oxalate / chemistry.
Kidney
Calculi / chemistry
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(PMID = 16775453.001).
[ISSN]
1062-4821
[Journal-full-title]
Current opinion in nephrology and hypertension
[ISO-abbreviation]
Curr. Opin. Nephrol. Hypertens.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
England
[Chemical-registry-number]
2612HC57YE / Calcium Oxalate
[Number-of-references]
51
51.
Brown CT, Kooiman G, Sharma DM, Poulsen J, Grange P:
Scarless single-port laparoscopic pelvic kidney nephrectomy.
J Laparoendosc Adv Surg Tech A
; 2010 Nov;20(9):743-6
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[Title]
Scarless single-port laparoscopic pelvic
kidney
nephrectomy.
INTRODUCTION: We report the first pelvic
kidney
removal through the umbilicus using a scarless pure single-port technique in a young woman.
PATIENTS AND METHODS: A 27-year-old woman presented with uro-sepsis and acute
renal
failure secondary to a dilated, chronically infected, nonfunctioning left-sided pelvic
kidney
with ureteropelvic obstruction causing an obstruction to the right
kidney
.
Definitive treatment involved removal of the diseased pelvic
kidney
through the umbilicus via a single-port access device (TriPor™; Olympus).
[MeSH-major]
Acute
Kidney
Injury / surgery.
Kidney
Pelvis / surgery. Laparoscopy / methods. Nephrectomy / methods. Ureteral Obstruction / surgery
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(PMID = 20874248.001).
[ISSN]
1557-9034
[Journal-full-title]
Journal of laparoendoscopic & advanced surgical techniques. Part A
[ISO-abbreviation]
J Laparoendosc Adv Surg Tech A
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
United States
52.
Cassuto JR, Reese PP, Sonnad S, Bloom RD, Levine MH, Olthoff KM, Shaked A, Naji A, Abt P:
Wait list death and survival benefit of kidney transplantation among nonrenal transplant recipients.
Am J Transplant
; 2010 Nov;10(11):2502-11
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[Title]
Wait list death and survival benefit
of kidney
transplantation among nonrenal transplant recipients.
The disparity between the number of patients waiting for
kidney
transplantation and the limited supply
of kidney
allografts has renewed interest in the benefit from
kidney
transplantation experienced by different groups.
This study evaluated
kidney
transplant survival benefit in prior nonrenal transplant recipients (
kidney
after liver, KALi; lung, KALu; heart, KAH) compared to primary isolated (KA1) or repeat isolated
kidney
(KA2) transplant.
Following
kidney
transplant, patient survival was greatest for KA1, similar among KA2, KALi, KAH, and inferior for KALu.
Compared to the entire wait list,
renal
transplantation was associated with a survival benefit among all groups except KALu (p = 0.017; HR = 1.61; CI = 1.09-2.38), where posttransplant survival was inferior to the wait list population.
Recipients of KA1
kidney
transplantation have the greatest posttransplant survival and compared to the overall
kidney
wait list, the greatest survival benefit.
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[Copyright]
©2010 The Authors Journal compilation©2010 The American Society of Transplantation and the American Society of Transplant Surgeons.
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(PMID = 20977641.001).
[ISSN]
1600-6143
[Journal-full-title]
American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons
[ISO-abbreviation]
Am. J. Transplant.
[Language]
ENG
[Grant]
United States / NIDDK NIH HHS / DK / K23 DK078688; United States / NIDDK NIH HHS / DK / K23 DK078688-01
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Other-IDs]
NLM/ NIHMS235283; NLM/ PMC2966021
53.
Jung JY, Song JH, Li C, Yang CW, Kang TC, Won MH, Jeong YG, Han KH, Choi KB, Lee SH, Kim J:
Expression of epidermal growth factor in the developing rat kidney.
Am J Physiol Renal Physiol
; 2005 Jan;288(1):F227-35
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[Title]
Expression of epidermal growth factor in the developing rat
kidney
.
Epidermal growth factor (EGF) is important in mammalian
renal
development.
In our study, we investigated the detailed distribution and the time of the first appearance of EGF in developing rat
kidney
.
Kidneys
from embryonic 18 (E18)- and 20-day-old (E20) fetuses, postnatal 1 (P1)-, 3 (P3)-, 7 (P7)-, 14 (P14)-, and 21-day-old (P21) pups, and adults were processed for immunohistochemistry and electronmicroscopy.
In adult rat
kidney
, EGF immunoreactivity was found in distal tubule including the thick ascending limb (TAL) and portion 1 of distal convoluted tubule (DCT1), whereas no EGF immunoreactivity was seen in portion 2 of distal convoluted tubule (DCT2) and connecting tubule.
In developing
kidney
, EGF-positive cells first appeared at P3 and were localized in the middle portion of the differentiating TAL of the corticomedullary junction.
However, EGF-positive and EGF-negative cells were in the TAL in developing rat
kidney
.
Our results suggest that the expression of EGF in developing
kidney
plays an important role in the regulation of growth and differentiation of the loop of Henle during
kidney
development and that this may act in the paracrine mode.
[MeSH-major]
Epidermal Growth Factor / biosynthesis. Gene Expression Regulation, Developmental / physiology.
Kidney
/ growth & development
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BROMODEOXYURIDINE
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(PMID = 15353402.001).
[ISSN]
1931-857X
[Journal-full-title]
American journal of physiology. Renal physiology
[ISO-abbreviation]
Am. J. Physiol. Renal Physiol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
62229-50-9 / Epidermal Growth Factor; G34N38R2N1 / Bromodeoxyuridine
54.
Grgic I, Wulff H, Eichler I, Flothmann C, Köhler R, Hoyer J:
Blockade of T-lymphocyte KCa3.1 and Kv1.3 channels as novel immunosuppression strategy to prevent kidney allograft rejection.
Transplant Proc
; 2009 Jul-Aug;41(6):2601-6
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[Title]
Blockade of T-lymphocyte KCa3.1 and Kv1.3 channels as novel immunosuppression strategy to prevent
kidney
allograft rejection.
Currently, there is an unmet clinical need for novel immunosuppressive agents for long-term prevention
of kidney
transplant rejection as alternatives to the nephrotoxic calcineurin inhibitor cyclosporine (CsA).
We investigated whether combined blockade of the T-cell K(+) channels K(Ca)3.1 and K(v)1.3, both of which regulate calcium signaling during lymphocyte activation, is effective in prevention of rejection
of kidney
allografts from Fisher rats to Lewis rats.
Kidney
sections were stained with periodic acid-Schiff or hematoxylin-eosin and histochemically for markers of macrophages (CD68), T-lymphocytes (CD43), or cytotoxic T-cells (CD8).
Thus, selective blockade of T-lymphocyte K(Ca)3.1 and K(v)1.3 channels may represent a novel alternative therapy for prevention
of kidney
allograft rejection.
[MeSH-major]
Graft Rejection / prevention & control. Immunosuppression / methods. Intermediate-Conductance Calcium-Activated Potassium Channels / immunology.
Kidney
Transplantation / immunology. Kv1.1 Potassium Channel / immunology. T-Lymphocytes / immunology
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[Cites]
Proc Natl Acad Sci U S A. 2000 Jul 5;97(14):8151-6
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10884437.001
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J Biol Chem. 2000 Nov 24;275(47):37137-49
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10961988.001
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J Immunol. 2007 Oct 1;179(7):4563-70
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J Bone Miner Res. 2004 Jan;19(1):155-64
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Curr Opin Drug Discov Devel. 2003 Sep;6(5):640-7
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J Clin Invest. 2003 Jun;111(11):1703-13
[
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[Cites]
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[
11276202.001
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[Cites]
Annu Rev Immunol. 2001;19:497-521
[
11244045.001
]
(PMID = 19715983.001).
[ISSN]
1873-2623
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Grant]
United States / NIGMS NIH HHS / GM / R01 GM076063; United States / NIGMS NIH HHS / GM / R01 GM076063; United States / NIGMS NIH HHS / GM / R01 GM076063-04
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Cnidarian Venoms; 0 / Intermediate-Conductance Calcium-Activated Potassium Channels; 0 / Kcnn4 protein, rat; 0 / Pyrazoles; 0 / ShK neurotoxin; 0 / TRAM 34; 147173-20-4 / Kv1.1 Potassium Channel
[Other-IDs]
NLM/ NIHMS370561; NLM/ PMC3343637
56.
Gerrits JH, van de Wetering J, Drabbels JJ, IJzermans JN, Claas FH, Weimar W, van Besouw NM:
Non-HLA T-cell reactivity during the first year after HLA-identical living-related kidney transplantation.
Clin Transplant
; 2009 Sep-Oct;23(5):740-7
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[Title]
Non-HLA T-cell reactivity during the first year after HLA-identical living-related
kidney
transplantation.
We wondered whether donor-reactive T-cell responses directed to minor histocompatibility antigens (mHAgs) or other non-HLA antigens also decrease after HLA-identical living-related (LR)
kidney
transplantation.
METHODS: We studied donor-reactive T-cell responses by IFN-gamma and granzyme B (GrB) Elispot assays in 15 HLA-identical LR
kidney
transplant recipients before, six months and one yr after transplantation.
CONCLUSIONS: Donor-reactivity could be detected before and after HLA-identical LR
kidney
transplantation, but was not related with the number of mHAg mismatches, and did not decrease after transplantation.
[MeSH-major]
Graft Rejection / immunology. HLA Antigens / immunology.
Kidney
Transplantation / immunology. Minor Histocompatibility Antigens / immunology. T-Lymphocytes / immunology
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(PMID = 19563488.001).
[ISSN]
1399-0012
[Journal-full-title]
Clinical transplantation
[ISO-abbreviation]
Clin Transplant
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Denmark
[Chemical-registry-number]
0 / HLA Antigens; 0 / Minor Histocompatibility Antigens; 82115-62-6 / Interferon-gamma; EC 3.4.21.- / GZMB protein, human; EC 3.4.21.- / Granzymes
57.
Pascual J, Galeano C, Royuela A, Zamora J:
A systematic review on steroid withdrawal between 3 and 6 months after kidney transplantation.
Transplantation
; 2010 Aug 27;90(4):343-9
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[Title]
A systematic review on steroid withdrawal between 3 and 6 months after
kidney
transplantation.
BACKGROUND: Steroid withdrawal (SW) after the first posttransplant months in patients receiving a
kidney
transplant has been recently discouraged in clinical guidelines.
METHODS: A systematic review and meta-analysis of randomized controlled trials assessing SW (beyond the second week after
kidney
transplantation) was performed.
CONCLUSIONS: SW after 3 to 6 months
of kidney
transplantation is associated with increased rates of acute rejection only if CsA is used but not with tacrolimus.
[MeSH-major]
Adrenal Cortex Hormones / administration & dosage.
Kidney
Transplantation / immunology
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.
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.
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consumer health - Steroids
.
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CYCLOSPORIN A
.
NCI CPTAC Assay Portal.
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.
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[CommentIn]
Transplantation. 2010 Aug 27;90(4):350-2
[
20548264.001
]
[CommentIn]
Transplantation. 2011 Mar 15;91(5):e27-8
[
21336084.001
]
[CommentIn]
Transplantation. 2011 Mar 15;91(5):e25; author reply e26-7
[
21336081.001
]
(PMID = 20574419.001).
[ISSN]
1534-6080
[Journal-full-title]
Transplantation
[ISO-abbreviation]
Transplantation
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Chemical-registry-number]
0 / Adrenal Cortex Hormones; 0 / Immunosuppressive Agents; 83HN0GTJ6D / Cyclosporine
[Number-of-references]
23
58.
Saha SA, Tuttle KR:
Influence of glycemic control on the development of diabetic cardiovascular and kidney disease.
Cardiol Clin
; 2010 Aug;28(3):497-516
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[Title]
Influence of glycemic control on the development of diabetic cardiovascular and
kidney
disease.
Among the microvascular complications of diabetes, diabetic
kidney
disease is the most common.
This article discusses the various drug classes used to treat diabetes mellitus, and reviews the current clinical evidence linking glycemic control using these drug classes on diabetic
kidney
and cardiovascular disease.
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.
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consumer health - Diabetes
.
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.
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consumer health - Diabetic Kidney Problems
.
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Acarbose
.
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[Copyright]
2010 Elsevier Inc. All rights reserved.
(PMID = 20621253.001).
[ISSN]
1558-2264
[Journal-full-title]
Cardiology clinics
[ISO-abbreviation]
Cardiol Clin
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
Netherlands
[Chemical-registry-number]
0 / Biguanides; 0 / Hypoglycemic Agents; 0 / Insulin; 0 / Sulfonylurea Compounds; 0 / Thiazolidinediones; T58MSI464G / Acarbose
59.
Stratta RJ, Sundberg AK, Farney AC, Rohr MS, Hartmann EL, Adams PL:
Successful simultaneous kidney-pancreas transplantation from extreme donors.
Transplant Proc
; 2005 Oct;37(8):3535-7
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[Title]
Successful simultaneous
kidney
-pancreas transplantation from extreme donors.
RESULTS: From January 2002 through January 2005, we performed 40 simultaneous
kidney
-pancreas transplants (SKPT) with Thymoglobulin induction, including 9 (22.5%) from EX and 31 from CONV DDs.
With a mean follow-up of 16.8 months in the EX DD group, patient and
kidney
graft survival rates are both 100%, and the pancreas graft survival rate is 89%.
With a mean follow-up of 21.7 months in the CONV DD group, patient and
kidney
graft survival rates are both 93.5% and the pancreas graft survival rate is 77.4%.
All patients with surviving grafts exhibited good initial (1 case of delayed
kidney
graft function in a CONV DD) and stable long-term
kidney
and pancreas graft function.
[MeSH-major]
Antilymphocyte Serum / therapeutic use.
Kidney
Transplantation / physiology. Pancreas Transplantation / physiology. Tissue Donors / statistics & numerical data
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.
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(PMID = 16298652.001).
[ISSN]
0041-1345
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Comparative Study; Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Antilymphocyte Serum; 0 / Immunosuppressive Agents
60.
Dhaun N, Macintyre IM, Melville V, Lilitkarntakul P, Johnston NR, Goddard J, Webb DJ:
Blood pressure-independent reduction in proteinuria and arterial stiffness after acute endothelin-a receptor antagonism in chronic kidney disease.
Hypertension
; 2009 Jul;54(1):113-9
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[Title]
Blood pressure-independent reduction in proteinuria and arterial stiffness after acute endothelin-a receptor antagonism in chronic
kidney
disease.
Endothelin 1 is implicated in the development and progression of chronic
kidney
disease and associated cardiovascular disease.
We, therefore, studied the effects of selective endothelin-A receptor antagonism with BQ-123 on key independent surrogate markers of cardiovascular risk (blood pressure, proteinuria and
renal
hemodynamics, arterial stiffness, and endothelial function) in patients with nondiabetic chronic
kidney
disease.
In a double-blind, randomized crossover study, 22 subjects with proteinuric chronic
kidney
disease received, on 2 separate occasions, placebo or BQ-123.
Blood pressure, pulse wave velocity, flow-mediated dilation,
renal
blood flow, and glomerular filtration rate were monitored after drug dosing.
BQ-123 reduced blood pressure (mean arterial pressure: -7+/-1%; P<0.001 versus placebo) and increased
renal
blood flow (17+/-4%; P<0.01 versus placebo).
Nifedipine matched the blood pressure and
renal
blood flow changes seen with BQ-123.
Selective endothelin-A receptor antagonism reduced blood pressure, proteinuria, and arterial stiffness on top of standard treatment in
renal
patients.
If maintained longer term, selective endothelin-A receptor antagonism may confer cardiovascular and
renal
benefits in patients with chronic
kidney
disease.
[MeSH-major]
Arteries / drug effects. Blood Pressure / drug effects.
Kidney
Failure, Chronic / drug therapy. Peptides, Cyclic / therapeutic use. Proteinuria / prevention & control
[MeSH-minor]
Adult. Aged. Antihypertensive Agents / administration & dosage. Antihypertensive Agents / therapeutic use. Calcium Channel Blockers / administration & dosage. Calcium Channel Blockers / therapeutic use. Cross-Over Studies. Double-Blind Method. Endothelin Receptor Antagonists. Endothelin-1 / blood. Hemodynamics / drug effects. Hemodynamics / physiology. Humans. Middle Aged. Nifedipine / administration & dosage. Nifedipine / therapeutic use.
Renal
Circulation / drug effects. Sodium / urine. Treatment Outcome
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.
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.
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commentaries/discussion - See the articles recommended by F1000Prime's Faculty of more than 8,000 leading experts in Biology and Medicine.
(subscription/membership/fee required).
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SODIUM
.
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[CommentIn]
Hypertension. 2009 Jul;54(1):29-31
[
19506097.001
]
(PMID = 19506099.001).
[ISSN]
1524-4563
[Journal-full-title]
Hypertension (Dallas, Tex. : 1979)
[ISO-abbreviation]
Hypertension
[Language]
eng
[Grant]
United Kingdom / British Heart Foundation / / PG/05/91
[Publication-type]
Journal Article; Randomized Controlled Trial; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Antihypertensive Agents; 0 / Calcium Channel Blockers; 0 / Endothelin Receptor Antagonists; 0 / Endothelin-1; 0 / Peptides, Cyclic; 136553-81-6 / cyclo(Trp-Asp-Pro-Val-Leu); 9NEZ333N27 / Sodium; I9ZF7L6G2L / Nifedipine
61.
Ix JH, De Boer IH, Peralta CA, Adeney KL, Duprez DA, Jenny NS, Siscovick DS, Kestenbaum BR:
Serum phosphorus concentrations and arterial stiffness among individuals with normal kidney function to moderate kidney disease in MESA.
Clin J Am Soc Nephrol
; 2009 Mar;4(3):609-15
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[Title]
Serum phosphorus concentrations and arterial stiffness among individuals with normal
kidney
function to moderate
kidney
disease in MESA.
DESIGN, SETTING, PARTICIPANTS, & MEASUREMENTS: This study examined the cross-sectional associations of serum phosphorus with ankle brachial index (ABI), pulse pressure, and large and small artery elasticity by radial artery waveform analysis among 1370 individuals (440 with moderate chronic
kidney
disease) who did not have clinical CVD and participated in the Multi-Ethnic Study of Atherosclerosis.
Participants with phosphorus levels >4 mg/dl had greater than four-fold risk for high ABI compared with participants with phosphate levels <3 mg/dl (relative risk 4.6; 95% confidence interval 1.6 to 13.2; P = 0.01) after adjustment for demographics, traditional CVD risk factors, and
kidney
function.
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12937218.001
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J Am Coll Cardiol. 2004 Aug 4;44(3):618-23
[
15358030.001
]
(PMID = 19211667.001).
[ISSN]
1555-905X
[Journal-full-title]
Clinical journal of the American Society of Nephrology : CJASN
[ISO-abbreviation]
Clin J Am Soc Nephrol
[Language]
ENG
[Grant]
United States / NHLBI NIH HHS / HL / N01HC95169; United States / NHLBI NIH HHS / HC / N01-HC-95165; United States / NHLBI NIH HHS / HC / N01-HC-95169; United States / NHLBI NIH HHS / HL / N01HC95165; United States / NHLBI NIH HHS / HL / N01HC95159
[Publication-type]
Journal Article; Multicenter Study; Research Support, N.I.H., Extramural
[Publication-country]
United States
[Chemical-registry-number]
27YLU75U4W / Phosphorus
[Other-IDs]
NLM/ PMC2653665
62.
Berger A, Preiser W, Kachel HG, Stürmer M, Doerr HW:
HBV reactivation after kidney transplantation.
J Clin Virol
; 2005 Feb;32(2):162-5
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[Title]
HBV reactivation after
kidney
transplantation.
In this study, we reviewed the virologic laboratory records from
kidney
recipients.
Out of 1512 patients, 228 had been diagnosed with resolved HBV infection (anti-HBc positive, HBsAg negative) but normal liver enzyme levels prior to
kidney
transplantation.
Reappearance of HBsAg after
kidney
transplantation was observed in two (0.9%) of those patients, which may be attributed to reactivation of a latent infection or to a new HBV infection.
Periodic follow-up of HBV serology for early
diagnosis
of reactivation is highly recommended in transplant recipients.
[MeSH-major]
Hepatitis B / virology. Hepatitis B Surface Antigens / blood. Hepatitis B virus / physiology.
Kidney
Transplantation / adverse effects. Virus Activation
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(PMID = 15653420.001).
[ISSN]
1386-6532
[Journal-full-title]
Journal of clinical virology : the official publication of the Pan American Society for Clinical Virology
[ISO-abbreviation]
J. Clin. Virol.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Netherlands
[Chemical-registry-number]
0 / Hepatitis B Surface Antigens
63.
Genberg H, Hansson A, Wernerson A, Wennberg L, Tydén G:
Pharmacodynamics of rituximab in kidney allotransplantation.
Am J Transplant
; 2006 Oct;6(10):2418-28
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[Title]
Pharmacodynamics of rituximab in
kidney
allotransplantation.
The anti-CD20 antibody rituximab has recently gained interest as a B-cell depleting agent in
renal
transplantation.
However, little is known about the pharmacodynamics of rituximab in
renal
transplant recipients.
We have therefore studied the effect of single-dose rituximab in combination with conventional triple immunosuppressive therapy on
the B
-cell population in peripheral blood as well as in tissues.
A total of 49
renal
transplant recipients received single-dose rituximab, as induction therapy (n = 36) or as anti-rejection therapy (n = 13).
We counted B cells in peripheral blood and performed immunohistochemical staining on lymph nodes and
kidney
transplant tissue samples to assess the prevalence of B cells.
The immunohistochemical staining showed a complete elimination of B cells in
kidney
tissue and a reduction of B cells in lymph nodes.
In conclusion, single-dose rituximab in
kidney
transplant recipients evokes a long-term elimination of B cells in peripheral blood as well as within the
kidney
transplant.
[MeSH-major]
Antibodies, Monoclonal / pharmacokinetics. Graft Rejection / drug therapy. Immunologic Factors / pharmacokinetics.
Kidney
Transplantation / pathology
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(PMID = 16925569.001).
[ISSN]
1600-6135
[Journal-full-title]
American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons
[ISO-abbreviation]
Am. J. Transplant.
[Language]
eng
[Databank-accession-numbers]
ClinicalTrials.gov/ NCT00255593
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Denmark
[Chemical-registry-number]
0 / Antibodies, Monoclonal; 0 / Antibodies, Monoclonal, Murine-Derived; 0 / Antigens, CD19; 0 / Antigens, CD20; 0 / Immunologic Factors; 4F4X42SYQ6 / Rituximab
64.
Montagner J, Michelon T, Fontanelle B, Oliveira A, Silveira J, Schroeder R, Neumann J, Keitel E, Alexandre CO:
BKV-infection in kidney graft dysfunction.
Braz J Infect Dis
; 2010 Mar-Apr;14(2):170-4
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[Title]
BKV-infection in
kidney
graft dysfunction.
INTRODUCTION: BKV nephropathy (BKN) causes
kidney
graft loss, whose specific
diagnosis
is invasive and might be predicted by the early detection of active viral infection.
OBJECTIVE: Determine the BKV-infection prevalence in late
kidney
graft dysfunction by urinary decoy cell (DC) and viral DNA detection in urine (viruria) and blood (viremia; active infection).
METHODS:
Kidney
recipients with >1 month follow-up and creatinine >1.5 mg/dL and/or recent increasing >20% (n = 120) had their urine and blood tested for BKV by semi-nested PCR, DC searching, and graft biopsy.
Diagnosis
efficacy of DC and viruria were compared to viremia.
Intense viruria was the single predictive test for active infection (3+; LR = 2.8).1,6-4,9 CONCLUSION: DC, BKV-viruria and -viremia are commun findings under late
kidney
graft dysfunction.
[MeSH-major]
BK Virus / isolation & purification.
Kidney
Transplantation / adverse effects. Polyomavirus Infections /
diagnosis
. Primary Graft Dysfunction / virology.
Tumor
Virus Infections /
diagnosis
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(PMID = 20563444.001).
[ISSN]
1678-4391
[Journal-full-title]
The Brazilian journal of infectious diseases : an official publication of the Brazilian Society of Infectious Diseases
[ISO-abbreviation]
Braz J Infect Dis
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Brazil
[Chemical-registry-number]
0 / DNA, Viral
65.
Szeto CC, Kwan BC, Lai KB, Lai FM, Chow KM, Wang G, Luk CC, Li PK:
Urinary expression of kidney injury markers in renal transplant recipients.
Clin J Am Soc Nephrol
; 2010 Dec;5(12):2329-37
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[Title]
Urinary expression
of kidney
injury markers in
renal
transplant recipients.
BACKGROUND AND OBJECTIVES: The outcome of
renal
transplantation after an episode of acute rejection is difficult to predict, even with an allograft biopsy.
We examined whether urinary expression of specific biomarker mRNA could be used as a noninvasive prognostic marker in
kidney
transplant recipients.
DESIGN, SETTING, PARTICIPANTS, & MEASUREMENTS: We studied 63
kidney
transplant recipients who require graft biopsy because of progressive worsening
of kidney
function.
The mRNA of neutrophil gelatinase-associated lipocalin,
kidney
injury molecule-1 (KIM-1), IL-18, surfactant protein-C, and S100 calcium-binding proteins A8 and A9 in urinary sediment were quantified.
After followed for an average of 39.7 ± 21.1 months, the rate of
renal
function decline significantly correlated with urinary KIM-1 expression (r = -0.434, P = 0.0004) but not other target genes.
CONCLUSIONS: In
kidney
allograft recipients, urinary KIM-1 expression provides prognostic information in relation to the rate of
renal
function decline, irrespective of the
kidney
pathology.
[MeSH-major]
Acute
Kidney
Injury / urine. Biomarkers / urine.
Kidney
Transplantation
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[Cites]
Kidney Int. 1999 Nov;56(5):1920-7
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[Cites]
Transplantation. 1999 Nov 27;68(10):1578-82
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[Cites]
N Engl J Med. 2000 Mar 2;342(9):605-12
[
10699159.001
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[Cites]
Kidney Int. 2000 Nov;58(5):2206-14
[
11044243.001
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[Cites]
N Engl J Med. 2001 Mar 29;344(13):947-54
[
11274620.001
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[Cites]
Transplantation. 2001 Sep 15;72(5):948-53
[
11571464.001
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[Cites]
Kidney Int. 2002 Feb;61(2):686-96
[
11849412.001
]
[Cites]
Transplantation. 2002 Feb 27;73(4):573-9
[
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]
[Cites]
J Am Soc Nephrol. 2002 Jul;13(7):1961-72
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Kidney Int. 2002 Oct;62(4):1125-35
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Nephrol Dial Transplant. 2002 Dec;17(12):2099-107
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[Cites]
Am J Transplant. 2003 Jan;3(1):17-22
[
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[Cites]
Transplantation. 2003 Apr 27;75(8):1323-30
[
12717224.001
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N Engl J Med. 2003 Jul 10;349(2):125-38
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[CommentIn]
Clin J Am Soc Nephrol. 2010 Dec;5(12):2141-3
[
21115630.001
]
(PMID = 20671224.001).
[ISSN]
1555-905X
[Journal-full-title]
Clinical journal of the American Society of Nephrology : CJASN
[ISO-abbreviation]
Clin J Am Soc Nephrol
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Acute-Phase Proteins; 0 / Biomarkers; 0 / Calgranulin A; 0 / HAVCR1 protein, human; 0 / Interleukin-18; 0 / LCN2 protein, human; 0 / Lipocalins; 0 / Membrane Glycoproteins; 0 / Proto-Oncogene Proteins; 0 / Pulmonary Surfactant-Associated Protein C; 0 / RNA, Messenger; 0 / Receptors, Virus; 0 / SFTPC protein, human
[Other-IDs]
NLM/ PMC2994096
66.
Moudouni SM, Lakmichi A, Tligui M, Rafii A, Tchala K, Haab F, Gattegno B, Thibault P, Doublet JD:
Renal cell carcinoma of native kidney in renal transplant recipients.
BJU Int
; 2006 Aug;98(2):298-302
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[Title]
Renal
cell carcinoma of native
kidney
in
renal
transplant recipients.
OBJECTIVE: To evaluate the prevalence, prognosis and possible risk factors of
renal
cell carcinoma (RCC) of the native
kidney
in
renal
transplant recipients.
PATIENTS AND METHODS: We retrospectively re-examined the follow-up data of 373 consecutive
renal
transplant recipients at our institution between August 1993 and September 2004.
We collected the data of all de novo RCC of the native
kidney
in the current analysis.
RESULTS: Of the 373 patients examined, 12 tumours of the native
kidney
were diagnosed in 10 individuals.
The mean ages at transplantation and
diagnosis
were 33 and 45.8 years, respectively.
Among
the renal
ultrasonograms there were two false-negative results.
The mean
tumour
size was 21 mm.
Among the 12
kidney
malignancies, there were five conventional RCCs and seven papillary RCCs.
One of the 10 patients died, from progression of metastases 6 years after
diagnosis
.
One patient had a local recurrence 2 years after
diagnosis
.
CONCLUSIONS: There appears to be a greater risk of RCC of the native
kidney
in patients with end-stage
renal
disease.
The present results suggest that an annual examination of the native
kidney
before and after
renal
transplantation is essential.
[MeSH-major]
Carcinoma,
Renal
Cell / etiology.
Kidney
Failure, Chronic / surgery.
Kidney
Neoplasms
/ etiology.
Kidney
Transplantation. Postoperative Complications / etiology
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(PMID = 16879668.001).
[ISSN]
1464-4096
[Journal-full-title]
BJU international
[ISO-abbreviation]
BJU Int.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
England
67.
Chang MY, Ong AC:
Autosomal dominant polycystic kidney disease: recent advances in pathogenesis and treatment.
Nephron Physiol
; 2008;108(1):p1-7
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[Title]
Autosomal dominant polycystic
kidney
disease: recent advances in pathogenesis and treatment.
Autosomal dominant polycystic
kidney
disease (ADPKD) is a common genetic
disorder
affecting 1 in 1,000 people in the general population and accounts for up to 10% of all patients on
renal
replacement therapy.
Numerous fluid-filled epithelial cysts arise from different nephron segments as spherical dilatations or small out-pouchings, enlarge progressively and eventually become disconnected from the rest of
the renal
tubule.
The development of cysts is accompanied by destruction of
the renal
parenchyma, interstitial fibrosis, cellular infiltration and loss of functional nephrons.
ADPKD is not only a
kidney
disease but also a systemic
disorder
associated with intracranial arterial aneurysms, cardiac valvular defects, colonic diverticulosis and cyst formation in other organs such as the liver, spleen and pancreas.
The identification of PKD1 and PKD2 together with the drive to elucidate the functions of their encoded proteins, polycystin-1 (PC1) and polycystin-2 (PC2), has led to an explosion of clinical and scientific interest in this common
disorder
.
[MeSH-major]
Polycystic
Kidney
, Autosomal Dominant / etiology. Polycystic
Kidney
, Autosomal Dominant / therapy
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[Copyright]
(c) 2007 S. Karger AG, Basel
(PMID = 18075279.001).
[ISSN]
1660-2137
[Journal-full-title]
Nephron. Physiology
[ISO-abbreviation]
Nephron Physiol
[Language]
eng
[Grant]
United Kingdom / Wellcome Trust / /
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
Switzerland
[Number-of-references]
70
68.
Jasuja D, Mor MK, Hartwig KC, Palevsky PM, Fine MJ, Weisbord SD:
Provider knowledge of contrast-induced acute kidney injury.
Am J Med Sci
; 2009 Oct;338(4):280-6
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[Title]
Provider knowledge of contrast-induced acute
kidney
injury.
BACKGROUND: Although past research has elucidated the principal risk factors and efficacy of preventive interventions for contrast-induced acute
kidney
injury (CIAKI), provider awareness of this empiric evidence base is largely unknown.
RESULTS: Of the 87 participating providers, nearly all (n = 85; 98%) recognized chronic
kidney
disease and intravascular volume depletion as risk factors.
[MeSH-major]
Contrast Media / adverse effects. Health Personnel / psychology.
Kidney
/ drug effects
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(PMID = 19641454.001).
[ISSN]
1538-2990
[Journal-full-title]
The American journal of the medical sciences
[ISO-abbreviation]
Am. J. Med. Sci.
[Language]
eng
[Grant]
United States / NIAID NIH HHS / AI / K24 AI001769
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
[Publication-country]
United States
[Chemical-registry-number]
0 / Contrast Media
69.
Stasević Z, Gorgieva GS, Vasić S, Ristić S, Djukanović L, Lezaić V:
High prevalence of kidney disease in two rural communities in Kosovo and Metohia.
Ren Fail
; 2010 Jun;32(5):541-6
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[Title]
High prevalence
of kidney
disease in two rural communities in Kosovo and Metohia.
A systematic survey was carried out in an enclave in Kosovo and Metohia, with the aim of assessing the prevalence
of kidney
diseases.
Persons with any detected
disorder
indicating
kidney
disease were invited for additional examination
of kidney
function.
Glomerular filtration rate (Modification of Diet in
Renal
Disease (MDRD) formula) below 60 mL/min/1.73 m(2) was found in 5.2% of subjects.
Kidney
ultrasound examination detected reduced length of right and left
kidneys
in 38 and 24 persons, respectively.
Cysts were also a frequent finding, but polycystic
kidney
, hydronephrosis, and
kidney
stones were found in about 2% each.
The analysis of data obtained by the present examination and available medical documentation revealed
kidney
and urinary tract diseases in 98 persons: 52 patients with already known disease and 46 patients detected in the survey.
Out of them in 22 patients
diagnosis
of kidney
disease could not be established during the survey but laboratory analyses indicated that they might suffer from tubulointerstitial disease: 14 had tubular dysfunctions, 8 of them low-grade proteinuria, and 12 had a positive family history for
kidney
disease.
In the enclave of Velika Hoca and Orahovac the prevalence
of kidney
disease was 7.0% indicating that these communities might be placed among those with a high prevalence
of kidney
disease in Serbia.
[MeSH-major]
Kidney
Diseases / epidemiology
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(PMID = 20486835.001).
[ISSN]
1525-6049
[Journal-full-title]
Renal failure
[ISO-abbreviation]
Ren Fail
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
70.
Sulejmanagić H, Sulejmanagić N, Prohić S, Secić S, Miseljić S:
Dental treatment of patients with kidney diseases-review.
Bosn J Basic Med Sci
; 2005 Feb;5(1):52-6
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[Title]
Dental treatment of patients with
kidney
diseases-review.
In their practice every dentist is brought into a situation to treat patients with grossly impaired
kidney
function.
Kidney
diseases, whether acute or acquired, imply a number of body dysfunctions such as prolonged bleeding, high blood pressure, infection tendency etc. which, in turn, pose a threat involving serious complications in cases of dental interventions in these patients.
The aim of this article is to provide a review of current dental practice in patients with
kidney
disease.
This implies dental intervention and preparations of patients with chronic
renal
disease, nephritic syndrome, patients on dialysis, and patients with
kidney
transplants.
Certainly, cooperation between the dentist and nephrologist is an imperative for the appropriate dental treatment of patients with grossly impaired
renal
function.
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(PMID = 15771603.001).
[ISSN]
1512-8601
[Journal-full-title]
Bosnian journal of basic medical sciences
[ISO-abbreviation]
Bosn J Basic Med Sci
[Language]
ENG
[Publication-type]
Journal Article; Review
[Publication-country]
Bosnia and Herzegovina
[Number-of-references]
12
71.
Perkins BA, Ficociello LH, Roshan B, Warram JH, Krolewski AS:
In patients with type 1 diabetes and new-onset microalbuminuria the development of advanced chronic kidney disease may not require progression to proteinuria.
Kidney Int
; 2010 Jan;77(1):57-64
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[Title]
In patients with type 1 diabetes and new-onset microalbuminuria the development of advanced chronic
kidney
disease may not require progression to proteinuria.
We sought to study new-onset microalbuminuria, its progression, and the decline of
renal
function in patients with type 1 diabetes.
Using a cohort of 109 patients who developed new-onset microalbuminuria in the first 4 years following enrollment in the 1st Joslin
Kidney
Study, we simultaneously tracked the change in their
renal
function and urinary albumin excretion.
Of these, 79 patients were followed for an average of 12 years after microalbuminuria onset, wherein their glomerular filtration rate was estimated by the Modification of Diet in
Renal
Disease Study formula and compared with their microalbuminuria and proteinuria.
Only 12 of the 23 patients who progressed to advanced (stage 3-5) chronic
kidney
disease developed proteinuria, which, in general, did not precede but accompanied the progression to advanced chronic
kidney
disease.
Thus, we found that one-third of patients with type 1 diabetes developed advanced chronic
kidney
disease relatively soon after the onset of microalbuminuria and this was not conditional on the presence of proteinuria.
Contrary to the existing concept of early nephropathy in type 1 diabetes, less emphasis should be placed on the mechanisms of progression to proteinuria and more placed on mechanisms initiating and promoting the early decline of
renal
function that eventually progresses to advanced chronic
kidney
disease.
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[CommentIn]
Kidney Int. 2010 Nov;78(10):1049; author reply 1049
[
21030977.001
]
(PMID = 19847154.001).
[ISSN]
1523-1755
[Journal-full-title]
Kidney international
[ISO-abbreviation]
Kidney Int.
[Language]
ENG
[Grant]
United States / NIDDK NIH HHS / DK / R01 DK041526; United States / NIDDK NIH HHS / DK / R01 DK067638; United States / NIDDK NIH HHS / DK / DK041526; United States / NIDDK NIH HHS / DK / DK067638
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Other-IDs]
NLM/ NIHMS493137; NLM/ PMC3725722
72.
Neuhofer W, Pittrow D:
Endothelin receptor selectivity in chronic kidney disease: rationale and review of recent evidence.
Eur J Clin Invest
; 2009 Jun;39 Suppl 2:50-67
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[Title]
Endothelin receptor selectivity in chronic
kidney
disease: rationale and review of recent evidence.
In addition to its substantial contribution to normal
renal
function, a large body of evidence suggests that derangement of
the renal
ET system is involved in the initiation and progression of chronic
kidney
disease (CKD) in diabetes, hypertension and glomerulonephritis.
Recent literature on the role of
the renal
ET system in the healthy
kidney
was reviewed.
ET-1 acts primarily as an autocrine or paracrine factor in the
kidney
.
In normal physiology, ET-1 promotes diuresis and natriuresis by local production and action through ET(B) receptors in
the renal
medulla.
[MeSH-major]
Endothelin Receptor Antagonists. Endothelin-1 / physiology.
Kidney
Failure, Chronic / physiopathology
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consumer health - Kidney Disease
.
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consumer health - Kidney Failure
.
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[ErratumIn]
Eur J Clin Invest. 2009 Jul;39(7):630
(PMID = 19335747.001).
[ISSN]
1365-2362
[Journal-full-title]
European journal of clinical investigation
[ISO-abbreviation]
Eur. J. Clin. Invest.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
England
[Chemical-registry-number]
0 / Antihypertensive Agents; 0 / Endothelin A Receptor Antagonists; 0 / Endothelin B Receptor Antagonists; 0 / Endothelin Receptor Antagonists; 0 / Endothelin-1; 0 / Receptors, Endothelin
[Number-of-references]
125
[General-notes]
NLM/ Original DateCompleted: 20090407
73.
Kim YW, Park BS, Ryu CH, Park SJ, Kang SW, Kim YH, Song IS, Shon JH:
Allopurinol-induced aplastic anemia in a patient with chronic kidney disease.
Clin Nephrol
; 2009 Feb;71(2):203-6
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[Title]
Allopurinol-induced aplastic anemia in a patient with chronic
kidney
disease.
We report an unusual case of aplastic anemia associated with allopurinol therapy for hyperuricemia in a patient with chronic
kidney
disease.
A 37-year-old female patient diagnosed with Stage III chronic
kidney
disease was admitted with pancytopenia.
These results suggested a
diagnosis
of aplastic anemia.
[MeSH-major]
Allopurinol / adverse effects. Anemia, Aplastic / chemically induced. Enzyme Inhibitors / adverse effects.
Kidney
Failure, Chronic / complications
Genetic Alliance.
consumer health - Kidney Disease
.
Genetic Alliance.
consumer health - Anemia
.
Genetic Alliance.
consumer health - Aplastic Anemia
.
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consumer health - Aplastic Anemia
.
MedlinePlus Health Information.
consumer health - Kidney Failure
.
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Allopurinol
.
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(PMID = 19203517.001).
[ISSN]
0301-0430
[Journal-full-title]
Clinical nephrology
[ISO-abbreviation]
Clin. Nephrol.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Germany
[Chemical-registry-number]
0 / Enzyme Inhibitors; 63CZ7GJN5I / Allopurinol
74.
Peces R, Peces C, Pérez-Dueñas V, Cuesta-López E, Azorín S, Selgas R:
Rapamycin reduces kidney volume and delays the loss of renal function in a patient with autosomal-dominant polycystic kidney disease.
NDT Plus
; 2009 Apr;2(2):133-5
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[Title]
Rapamycin reduces
kidney
volume and delays the loss of
renal
function in a patient with autosomal-dominant polycystic
kidney
disease.
This is the first report of a case of a reduction in
kidney
volume and preservation of
renal
function in a patient with autosomal-dominant polycystic
kidney
disease (ADPKD) receiving rapamycin.
A 42-year-old man with ADPKD and a severe persistent bleeding from his solitary left
kidney
was successfully treated with tranexamic acid (TXA).
He also received low-dose rapamycin for 8 months, and this was associated with a 23.5% reduction in
kidney
volume, improvement and stabilization of
renal
function, and normalization of haemoglobin levels.
When treatment with rapamycin was interrupted,
renal
function deteriorated within an 8-month period and haemodialysis (HD) became necessary.
Kidney
volume increased at once, and life-threatening bleeding prompted a nephrectomy 4 months after the onset of HD.
These data suggest that the reduction in
kidney
volume and preservation of
renal
function with rapamycin could be the result of the antiangiogenic, antiproliferative effects of rapamycin.
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(PMID = 25949309.001).
[ISSN]
1753-0784
[Journal-full-title]
NDT plus
[ISO-abbreviation]
NDT Plus
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
England
[Other-IDs]
NLM/ PMC4421355
[Keywords]
NOTNLM ; ADPKD / mTOR / rapamycin / renal function / volume change
75.
Lee M, Partridge NC:
Parathyroid hormone signaling in bone and kidney.
Curr Opin Nephrol Hypertens
; 2009 Jul;18(4):298-302
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[Title]
Parathyroid hormone signaling in bone and
kidney
.
PURPOSE OF REVIEW: Parathyroid hormone (PTH) maintains a physiological balance of calcium and phosphate concentrations by binding to its receptor on the plasma membrane of cells in bone and
kidney
.
Here, we will review the recent advancements regarding PTH signaling in bone and
kidney
.
In
kidney
, sodium-hydrogen exchanger regulatory factor 1, originally known for its role in the retention of NaPi-IIa at the apical membrane, was shown to have multiple roles in PTH signaling, both as a mediator and regulator.
SUMMARY: PTH activates a number of different signaling pathways by binding to a single receptor in bone and
kidney
.
[MeSH-major]
Bone and Bones / metabolism.
Kidney
/ metabolism. Parathyroid Hormone / physiology. Signal Transduction / physiology
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CALCIUM, ELEMENTAL
.
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(PMID = 19395963.001).
[ISSN]
1473-6543
[Journal-full-title]
Current opinion in nephrology and hypertension
[ISO-abbreviation]
Curr. Opin. Nephrol. Hypertens.
[Language]
eng
[Grant]
United States / NIDDK NIH HHS / DK / DK47420
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Review
[Publication-country]
England
[Chemical-registry-number]
0 / Parathyroid Hormone; 0 / Phosphoproteins; 0 / Receptor, Parathyroid Hormone, Type 1; 0 / Sodium-Hydrogen Antiporter; 0 / sodium-hydrogen exchanger regulatory factor; EC 2.7.11.11 / Cyclic AMP-Dependent Protein Kinases; EC 2.7.11.13 / Protein Kinase C; SY7Q814VUP / Calcium
[Number-of-references]
58
76.
Gerosa C, Fanni D, Nemolato S, Locci A, Marinelli V, Cabras T, Messana I, Castagnola M, Monga G, Fanos V, Faa G:
Thymosin beta-10 expression in developing human kidney.
J Matern Fetal Neonatal Med
; 2010 Oct;23 Suppl 3:125-8
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[Title]
Thymosin beta-10 expression in developing human
kidney
.
The report that the Tβ10 gene is expressed at high levels in embryonic human brain as well in human
kidney
induced us to study Tβ10 reactivity in the preterm
kidney
in order to verify, at tissue level, the expression of this peptide during
renal
embryogenesis.
To this end, we analyzed, using immunocytochemistry, the expression of Tβ10 in samples of human
kidney
obtained, at autopsy, from 8 fetuses, 12 preterm infants, ranging from 25 to 36 weeks of gestation and 3 at term newborns.
Tβ10 immunoreactivity was detected in 20 out of 22
kidneys
examined, and was mainly localized in proximal and distal tubular structures, in the cytoplasm and occasionally in the nuclei of ductal cells.
In 13
kidneys
, we also observed immunostaining for Tβ10 inside the "comma-shaped bodies" and the "S-shaped bodies" during active glomerulogenesis.
Our data show, for the first time, the expression of Tβ10 in the human
kidney
during the initial phases of its physiological development, mainly restricted in the proximal and the distal tubuli.
Further studies are needed in order to better characterize the role of Tβ10 in
kidney
embryogenesis.
[MeSH-major]
Kidney
/ embryology.
Kidney
/ metabolism. Thymosin / metabolism
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(PMID = 20836742.001).
[ISSN]
1476-4954
[Journal-full-title]
The journal of maternal-fetal & neonatal medicine : the official journal of the European Association of Perinatal Medicine, the Federation of Asia and Oceania Perinatal Societies, the International Society of Perinatal Obstetricians
[ISO-abbreviation]
J. Matern. Fetal. Neonatal. Med.
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
61512-21-8 / Thymosin; 87397-91-9 / thymosin beta(10)
77.
Zukowski M, Bohatyrewicz R, Biernawska J, Kotfis K, Zegan M, Knap R, Janeczek M, Zietek Z:
Risk factors for septic complications in kidney transplant recipients.
Transplant Proc
; 2009 Oct;41(8):3043-5
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[Title]
Risk factors for septic complications in
kidney
transplant recipients.
INTRODUCTION: Septic complications following
kidney
transplantation are a leading cause of therapeutic failure.
An early
diagnosis
may protect the recipient from the severe consequences of sepsis.
We sought to determine the risk factors influencing the occurrence of septic complications among
kidney
transplant recipients.
Recipient data included age, gender, period of prior hemodialysis, panel reactive antibodies, cold ischemia time, and cause of
renal
insufficiency.
The 232
kidney
recipients were examined for occurrence of septic complications including septicemia, pneumonia, peritonitis, or graft infection.
RESULTS:
Kidney
transplants from donors with MAP < 70 mm Hg and SVRI < 1200 dyne x s/cm(5) x m(2) showed a significantly higher occurrence of septic complications in recipients (P < .05) where mortality rate was also significantly greater (P < .01).
CONCLUSIONS: MAP < 70 mm Hg and SVRI < 1200 dyne x s/cm(5) x m(2) among organ donors predicted greater occurrence of septic complications and increased mortality among
kidney
transplant recipients.
[MeSH-major]
Kidney
Transplantation / physiology. Sepsis / epidemiology
[MeSH-minor]
Adolescent. Adult. Blood Pressure / physiology. Brain Death. Cadaver. Cause of Death. Central Venous Pressure / physiology. Child. Female. Heart Rate. Humans. Male. Middle Aged.
Renal
Dialysis. Risk Factors. Shock, Septic / epidemiology. Tissue Donors. Vascular Resistance. Young Adult
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Sepsis
.
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(PMID = 19857672.001).
[ISSN]
1873-2623
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
78.
Li L, Okusa MD:
Macrophages, dendritic cells, and kidney ischemia-reperfusion injury.
Semin Nephrol
; 2010 May;30(3):268-77
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[Title]
Macrophages, dendritic cells, and
kidney
ischemia-reperfusion injury.
Dendritic cells and macrophages are critical early initiators of innate immunity in the
kidney
and orchestrate inflammation subsequent to ischemia-reperfusion injury.
They are the most abundant leukocytes present in the
kidney
, and they represent a heterogeneous population of cells that are capable of inducing sterile inflammation after reperfusion directly through the production of proinflammatory cytokines and other soluble inflammatory mediators or indirectly through activation of effector T lymphocytes and natural killer T cells.
In addition, recent studies have indicated that
kidney
and immune cell micro-RNAs control gene expression and have the ability to regulate the initial inflammatory response to injury.
Although dendritic cells and macrophages contribute to both innate and adaptive immunity and to injury and repair, this review focuses on the initial innate response to
kidney
ischemia-reperfusion injury.
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[ISSN]
1558-4488
[Journal-full-title]
Seminars in nephrology
[ISO-abbreviation]
Semin. Nephrol.
[Language]
ENG
[Grant]
United States / NIDDK NIH HHS / DK / DK062324-08; United States / NIDDK NIH HHS / DK / DK062324-07; United States / NIDDK NIH HHS / DK / R01 DK056223-08; United States / NIDDK NIH HHS / DK / R01 DK085259; United States / NIDDK NIH HHS / DK / R01DK56223; United States / NIDDK NIH HHS / DK / R01 DK062324; United States / NIDDK NIH HHS / DK / R01 DK056223; United States / NIDDK NIH HHS / DK / R01DK62324; United States / NIDDK NIH HHS / DK / DK056223-08; United States / NIDDK NIH HHS / DK / R01 DK062324-07; United States / NIDDK NIH HHS / DK / R01 DK062324-08
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review
[Publication-country]
United States
[Other-IDs]
NLM/ NIHMS188223; NLM/ PMC2904394
79.
Sanchez-Niño MD, Benito-Martin A, Gonçalves S, Sanz AB, Ucero AC, Izquierdo MC, Ramos AM, Berzal S, Selgas R, Ruiz-Ortega M, Egido J, Ortiz A:
TNF superfamily: a growing saga of kidney injury modulators.
Mediators Inflamm
; 2010;2010
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[Title]
TNF superfamily: a growing saga
of kidney
injury modulators.
Members of the TNF superfamily participate in
kidney
disease.
Tumor
necrosis factor (TNF) and Fas ligand regulate
renal
cell survival and inflammation, and therapeutic targeting improves the outcome of experimental
renal
injury.
In vivo TWEAK promotes postnephrectomy compensatory
renal
cell proliferation in a noninflammatory milieu.
However, in the inflammatory milieu of acute
kidney
injury, TWEAK promotes tubular cell death and inflammation.
[MeSH-major]
Kidney
/ injuries.
Tumor
Necrosis Factors / metabolism
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(PMID = 20953353.001).
[ISSN]
1466-1861
[Journal-full-title]
Mediators of inflammation
[ISO-abbreviation]
Mediators Inflamm.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
United States
[Chemical-registry-number]
0 / Cytokines; 0 / Fas Ligand Protein; 0 / TNF-Related Apoptosis-Inducing Ligand; 0 / Tumor Necrosis Factors
[Other-IDs]
NLM/ PMC2952810
80.
Bansal N, Hsu CY:
Long-term outcomes of patients with chronic kidney disease.
Nat Clin Pract Nephrol
; 2008 Oct;4(10):532-3
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[Title]
Long-term outcomes of patients with chronic
kidney
disease.
This Practice Point commentary discusses a recent paper by Menon et al. that described the natural history of a cohort of nondiabetic patients with stage 2-4 chronic
kidney
disease (CKD) who had been recruited from nephrology practices and screened for entry into the Modification of Diet in
Renal
Disease (MDRD) trial.
Kidney
failure was the most common outcome during long-term follow-up among these patients and there was a low competing risk of death, findings in contrast to observations in other cohorts of patients with CKD.
Patients with lower glomerular filtration rate and greater proteinuria at baseline were at increased risk for both
kidney
failure and death, but
kidney
failure remained more likely than death in all glomerular filtration rate subgroups.
Nephrologists should not rely on CKD staging alone to direct management of or risk-stratify patients with CKD, but should also consider the etiology and rate of progression
of kidney
disease, patient age and cardiovascular disease risk factors.
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[CommentOn]
Kidney Int. 2008 Jun;73(11):1310-5
[
18337713.001
]
(PMID = 18695707.001).
[ISSN]
1745-8331
[Journal-full-title]
Nature clinical practice. Nephrology
[ISO-abbreviation]
Nat Clin Pract Nephrol
[Language]
eng
[Grant]
United States / NIDDK NIH HHS / DK / K23 DK088865
[Publication-type]
Comment; Journal Article
[Publication-country]
England
81.
Pecoits-Filho R:
Dietary protein intake and kidney disease in Western diet.
Contrib Nephrol
; 2007;155:102-12
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[Source]
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[Title]
Dietary protein intake and
kidney
disease in Western diet.
Components of the diet related to changes in eating habits that characterize the modern Western world are important factors in the increasingly high prevalence of chronic disease, including obesity, diabetes, hypertension and as a consequence, chronic
kidney
disease.
Unfortunately, very few clinical studies focused on diet-based strategies of prevention
of kidney
disorders.
Furthermore, this review will propose that the concept that protein restricted diets decrease the risk of developing
kidney
disease in the general population is not supported by the scientific literature.
Indeed, preliminary studies showing a positive effect of relatively high protein diets on risk factors for chronic
kidney
disease (particularly on obesity, hypertension and diabetes) point to the need for future studies addressing diets that could prevent the increasingly high prevalence
of kidney
disease in the Western world.
On the other hand, there is a potential role for protein restriction in patients with established
kidney
disease, particularly in patients with significant decrease in glomerular filtration rate.
The exact protective action of protein restriction in patients with established
renal
disease needs further analysis, taking into account the more broad effects of protein restriction (lower phosphate, acidosis, uric acid) and a more current definition of malnutrition.
[MeSH-major]
Diet / ethnology. Diet, Protein-Restricted. Dietary Proteins.
Kidney
Diseases / prevention & control
Genetic Alliance.
consumer health - Kidney Disease
.
MedlinePlus Health Information.
consumer health - Dietary Proteins
.
MedlinePlus Health Information.
consumer health - Kidney Diseases
.
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(PMID = 17369718.001).
[ISSN]
0302-5144
[Journal-full-title]
Contributions to nephrology
[ISO-abbreviation]
Contrib Nephrol
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
Switzerland
[Chemical-registry-number]
0 / Dietary Proteins
[Number-of-references]
41
82.
Stratta RJ, Sundberg AK, Rohr MS, Farney AC, Hartmann EL, Roskopf JA, Iskandar SS, Hairston G, Kiger DF, Gautreaux MD, Anderson TK, Adams PL:
Optimal use of older donors and recipients in kidney transplantation.
Surgery
; 2006 Mar;139(3):324-33
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[Source]
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[Title]
Optimal use of older donors and recipients in
kidney
transplantation.
BACKGROUND: The aging donor and recipient population have led to new challenges in
kidney
transplantation.
The purpose of this study was to review retrospectively our single center experience in deceased-donor
kidney
transplantation, with respect to donor and recipient age.
METHODS: From October 1, 2001, through February 20, 2004, we performed 144 deceased-donor
kidney
transplantations, which included 37 procedures (26%) in recipients > or =60 years old and 107 procedures (74%) in recipients 19 to 59 years old.
ECD
kidneys
were used by matching estimated
renal
functional mass to recipient size (body mass index, <25 kg/m(2)), which included the use of dual
kidney
transplantations (n = 9).
ECD
kidney
recipients were further selected on the basis of age >40 years and low immunologic risk.
In recipients > or =60 years old, 23 recipients (62%) received
kidney
transplants from ECDs compared with 34
kidney
transplants from ECDs (32%; P < .001) in recipients who were <60 years old.
Kidney
graft survival rates were 84% in both recipient groups.
CONCLUSION: By the matching of nephron mass with recipient size and avoiding the use of ECD
kidneys
in recipients with a high immunologic risk, short-term outcomes that are comparable with standard criteria donor
kidneys
in younger patients can be achieved with either older donors or recipients, regardless of age.
[MeSH-major]
Kidney
Transplantation. Patient Selection. Tissue Donors
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Organ Donation
.
COS Scholar Universe.
author profiles
.
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(PMID = 16546496.001).
[ISSN]
0039-6060
[Journal-full-title]
Surgery
[ISO-abbreviation]
Surgery
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Immunosuppressive Agents
83.
Lora CM, Daviglus ML, Kusek JW, Porter A, Ricardo AC, Go AS, Lash JP:
Chronic kidney disease in United States Hispanics: a growing public health problem.
Ethn Dis
; 2009;19(4):466-72
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[Source]
The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Chronic
kidney
disease in United States Hispanics: a growing public health problem.
The incidence of end-stage
renal
disease (ESRD) in Hispanics is higher than non-Hispanic Whites and Hispanics with chronic
kidney
disease (CKD) are at increased risk for
kidney
failure.
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(PMID = 20073150.001).
[ISSN]
1049-510X
[Journal-full-title]
Ethnicity & disease
[ISO-abbreviation]
Ethn Dis
[Language]
ENG
[Grant]
United States / NIDDK NIH HHS / DK / DK072231-05S2; United States / NIDDK NIH HHS / DK / 5U01DK60980; United States / NIDDK NIH HHS / DK / R01 DK072231; United States / NIDDK NIH HHS / DK / DK072231-05; United States / NIDDK NIH HHS / DK / R01 DK072231-03S1; United States / NIDDK NIH HHS / DK / R01 DK072231-04; United States / NIDDK NIH HHS / DK / U01 DK060980; United States / NIDDK NIH HHS / DK / DK072231-03; United States / NIDDK NIH HHS / DK / R01 DK072231-07; United States / NIDDK NIH HHS / DK / DK072231-02; United States / NIDDK NIH HHS / DK / DK072231-03S1; United States / NIDDK NIH HHS / DK / R01 DK072231-03; United States / NIDDK NIH HHS / DK / DK072231-04S1; United States / NIDDK NIH HHS / DK / R01 DK072231-04S1; United States / NIDDK NIH HHS / DK / DK072231-06; United States / NIDDK NIH HHS / DK / R56 DK072231; United States / NIDDK NIH HHS / DK / R01 DK72231; United States / NCRR NIH HHS / RR / M01 RR013987; United States / NIDDK NIH HHS / DK / R01 DK072231-02; United States / NIDDK NIH HHS / DK / R01 DK072231-05; United States / NIDDK NIH HHS / DK / DK072231-04; United States / NIDDK NIH HHS / DK / R01 DK072231-05S1; United States / NIDDK NIH HHS / DK / DK072231-07; United States / NIDDK NIH HHS / DK / R01 DK072231-06A1; United States / NIDDK NIH HHS / DK / R56 DK072231-06; United States / NIDDK NIH HHS / DK / R01 DK072231-01; United States / NIDDK NIH HHS / DK / DK072231-05S1; United States / NIDDK NIH HHS / DK / DK072231-06A1; United States / NIDDK NIH HHS / DK / R01 DK072231-05S2
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review
[Publication-country]
United States
[Number-of-references]
76
[Other-IDs]
NLM/ NIHMS316436; NLM/ PMC3587111
84.
Okamoto M, Suzuki T, Fujiki M, Nobori S, Ushigome H, Sakamoto S, Yoshimura N:
The consequences for live kidney donors with preexisting glucose intolerance without diabetic complication: analysis at a single Japanese center.
Transplantation
; 2010 Jun 15;89(11):1391-5
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[Title]
The consequences for live
kidney
donors with preexisting glucose intolerance without diabetic complication: analysis at a single Japanese center.
BACKGROUND: Because of lack of deceased donors in Japan, there has been a need to expand the eligibility criteria for live
kidney
donation.
To assess the indications for live
kidney
donation in glucose intolerance (GI), we analyzed perioperative complications associated with donor nephrectomies performed at our institution and followed up the long-term consequences.
METHODS: The 444 live
kidney
donors were divided into two groups based on the results of the 75-g oral glucose tolerance test: a GI group (n=71) who showed a diabetic (n=27) or impaired glucose tolerance (n=44) pattern, and a non-GI group (n=373) who showed a normal oral glucose tolerance test pattern.
Perioperative complications, longterm survival rate, and frequencies of hypertension, diabetes, hyperlipidemia, and
renal
dysfunction in long term were compared in each group.
None of the patients in the diabetes mellitus group had developed severe diabetic complications or end-stage
renal
disease at a mean follow-up point of 88+/-71 (range, 14-225) months.
CONCLUSIONS: Our results suggest that individuals who have GI without diabetic complication may be able to donate their
kidney
safely with little surgical complication and little major morbidity if strict evaluation is performed before transplant.
[MeSH-major]
Glucose Intolerance / epidemiology.
Kidney
Transplantation / physiology. Living Donors. Nephrectomy. Tissue and Organ Procurement / statistics & numerical data
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(PMID = 20535851.001).
[ISSN]
1534-6080
[Journal-full-title]
Transplantation
[ISO-abbreviation]
Transplantation
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
85.
Kestenbaum B, Belozeroff V:
Mineral metabolism disturbances in patients with chronic kidney disease.
Eur J Clin Invest
; 2007 Aug;37(8):607-22
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[Title]
Mineral metabolism disturbances in patients with chronic
kidney
disease.
BACKGROUND:
Kidney
disease, especially chronic
kidney
disease (CKD), is a worldwide public health problem with serious adverse health consequences for affected individuals.
Secondary hyperparathyroidism, a
disorder
characterized by elevated serum parathyroid hormone levels, and alteration of calcium and phosphorus homeostasis are common metabolic complications of CKD that may impact cardiovascular health.
RESULTS: Mineral metabolism disturbances begin early during the course of chronic
kidney
disease, and are associated with cardiovascular disease and mortality in observational studies.
Vascular calcification is one plausible mechanism connecting
renal
-related mineral metabolism with cardiovascular risk.
CONCLUSIONS: There exists a potential to improve outcomes for patients with CKD through increased awareness of the Bone Metabolism and Disease guidelines set forth by the National
Kidney
Foundation-
Kidney
Disease Outcomes Quality Initiative.
[MeSH-major]
Calcium / metabolism. Cardiovascular Diseases / etiology. Hyperparathyroidism, Secondary / etiology.
Kidney
Failure, Chronic / complications. Parathyroid Hormone / secretion. Phosphorus / metabolism
[MeSH-minor]
Calcinosis / etiology. Female. Humans. Male.
Renal
Dialysis. Risk Factors. Vitamin D / analogs & derivatives. Vitamin D / blood. Vitamin D / therapeutic use
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(PMID = 17635571.001).
[ISSN]
0014-2972
[Journal-full-title]
European journal of clinical investigation
[ISO-abbreviation]
Eur. J. Clin. Invest.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
England
[Chemical-registry-number]
0 / Parathyroid Hormone; 1406-16-2 / Vitamin D; 27YLU75U4W / Phosphorus; SY7Q814VUP / Calcium
[Number-of-references]
137
86.
Hanratty R, Chonchol M, Miriam Dickinson L, Beaty BL, Estacio RO, Mackenzie TD, Hurley LP, Linas SL, Steiner JF, Havranek EP:
Incident chronic kidney disease and the rate of kidney function decline in individuals with hypertension.
Nephrol Dial Transplant
; 2010 Mar;25(3):801-7
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[Title]
Incident chronic
kidney
disease and the rate
of kidney
function decline in individuals with hypertension.
BACKGROUND: Little is known about the decline
of kidney
function in patients with normal
kidney
function at baseline.
Our objectives were to (i) identify predictors of incident chronic
kidney
disease (CKD) and (ii) to estimate rate of decline in
kidney
function.
Rates of incident CKD or in decline
of kidney
function did not differ by race or ethnicity in this cohort.
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(PMID = 19889870.001).
[ISSN]
1460-2385
[Journal-full-title]
Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association
[ISO-abbreviation]
Nephrol. Dial. Transplant.
[Language]
ENG
[Grant]
United States / NHLBI NIH HHS / HL / U01 HL079160; United States / NHLBI NIH HHS / HL / U01 HL079208; United States / NHLBI NIH HHS / HL / 1U01 HL079160; United States / NHLBI NIH HHS / HL / 1U01 HL079208
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
England
[Other-IDs]
NLM/ PMC2828608
87.
Cunningham J, Cass A, Anderson K, Snelling P, Devitt J, Preece C, Eris J:
Australian nephrologists' attitudes towards living kidney donation.
Nephrol Dial Transplant
; 2006 May;21(5):1178-83
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[Title]
Australian nephrologists' attitudes towards living
kidney
donation.
BACKGROUND: The demand for deceased donor
kidneys
far outweighs the supply.
The rate of living
kidney
donation (LKD) has been steadily increasing world-wide and is associated with excellent outcomes for the recipient.
The vast majority (95%) of respondents indicated that they would recommend it to a suitable donor or would themselves (97%) donate a
kidney
to an immediate family member.
However, fewer than half (43%) would recommend LKD to a potential donor, where their relative's end-stage
kidney
disease (ESKD) had been attributed to diabetes and where there was a strong family history of diabetes.
Meeting the growing demand for
kidney
transplantation will require an increased supply of both live and deceased donor
kidneys
.
[MeSH-major]
Attitude of Health Personnel.
Kidney
Transplantation / psychology. Living Donors. Tissue and Organ Procurement
[MeSH-minor]
Adult. Australia. Diabetic Nephropathies /
diagnosis
. Diabetic Nephropathies / surgery. Female. Health Care Surveys. Humans. Logistic Models. Male. Middle Aged. Multivariate Analysis. Nephrectomy / methods. Nephrology / standards. Nephrology / trends. Practice Patterns, Physicians'. Risk Assessment. Surveys and Questionnaires
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(PMID = 16490747.001).
[ISSN]
0931-0509
[Journal-full-title]
Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association
[ISO-abbreviation]
Nephrol. Dial. Transplant.
[Language]
eng
[Publication-type]
Comparative Study; Editorial; Research Support, Non-U.S. Gov't
[Publication-country]
England
88.
Thomas MC, Atkins RC:
Blood pressure lowering for the prevention and treatment of diabetic kidney disease.
Drugs
; 2006;66(17):2213-34
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[Title]
Blood pressure lowering for the prevention and treatment of diabetic
kidney
disease.
The current pandemic of diabetes mellitus will inevitably be followed by an epidemic of chronic
kidney
disease.
It is anticipated that 25-40% of patients with type 1 diabetes and 5-40% of patients with type 2 diabetes will ultimately develop diabetic
kidney
disease.
Diabetic nephropathy may lead to hypertension through direct actions on
renal
sodium handling, vascular compliance and vasomotor function.
Recent clinical trials also support the utility of blood pressure reduction in the prevention of diabetic
kidney
disease.
In patients with overt nephropathy, blood pressure reduction is associated with reduced urinary albumin excretion and, subsequently, a reduced risk of
renal
impairment or end stage
renal
disease.
Although there is a clear physiological rationale for blockade of the renin angiotensin system, which is strongly supported by clinical studies, to achieve the optimal lowering of blood pressure, particularly in the setting of established diabetic
renal
disease, a number of different antihypertensive agents will always be needed.
Ultimately, no matter how it is achieved, so long as it is achieved,
renal
risk can be reduced by agents that lower blood pressure and albuminuria.
[MeSH-major]
Antihypertensive Agents / therapeutic use. Blood Pressure / drug effects. Diabetes Complications /
diagnosis
. Diabetes Mellitus / therapy. Diabetic Nephropathies / prevention & control. Hypertension / drug therapy
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[ISSN]
0012-6667
[Journal-full-title]
Drugs
[ISO-abbreviation]
Drugs
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
New Zealand
[Chemical-registry-number]
0 / Antihypertensive Agents
[Number-of-references]
141
89.
Ibrahim HN, Foley R, Tan L, Rogers T, Bailey RF, Guo H, Gross CR, Matas AJ:
Long-term consequences of kidney donation.
N Engl J Med
; 2009 Jan 29;360(5):459-69
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[Title]
Long-term consequences
of kidney
donation.
BACKGROUND: The long-term
renal
consequences
of kidney
donation by a living donor are attracting increased appropriate interest.
The overall evidence suggests that living
kidney
donors have survival similar to that of nondonors and that their risk of end-stage
renal
disease (ESRD) is not increased.
METHODS: We ascertained the vital status and lifetime risk of ESRD in 3698
kidney
donors who donated
kidneys
during the period from 1963 through 2007; from 2003 through 2007, we also measured the glomerular filtration rate (GFR) and urinary albumin excretion and assessed the prevalence of hypertension, general health status, and quality of life in 255 donors.
RESULTS: The survival
of kidney
donors was similar to that of controls who were matched for age, sex, and race or ethnic group.
CONCLUSIONS: Survival and the risk of ESRD in carefully screened
kidney
donors appear to be similar to those in the general population.
[MeSH-major]
Health Status.
Kidney
Failure, Chronic / epidemiology.
Kidney
Transplantation. Living Donors. Quality of Life
MedlinePlus Health Information.
consumer health - Kidney Failure
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
MedlinePlus Health Information.
consumer health - Organ Donation
.
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[Copyright]
2009 Massachusetts Medical Society
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[CommentIn]
N Engl J Med. 2010 Aug 19;363(8):797-8
[
20818884.001
]
[CommentIn]
N Engl J Med. 2009 Jan 29;360(5):522-3
[
19179321.001
]
[CommentIn]
N Engl J Med. 2009 May 28;360(22):2371-2; author reply 2372
[
19484820.001
]
[CommentIn]
N Engl J Med. 2009 May 28;360(22):2371; author reply 2372
[
19484821.001
]
[CommentIn]
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[
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]
[CommentIn]
N Engl J Med. 2009 May 28;360(22):2370; author reply 2372
[
19474439.001
]
(PMID = 19179315.001).
[ISSN]
1533-4406
[Journal-full-title]
The New England journal of medicine
[ISO-abbreviation]
N. Engl. J. Med.
[Language]
eng
[Grant]
United States / NCRR NIH HHS / RR / M01 RR000400; United States / NIDDK NIH HHS / DK / P01 DK013083; United States / NCRR NIH HHS / RR / M01-RR00400; United States / NIDDK NIH HHS / DK / P01DK13083
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
AYI8EX34EU / Creatinine
[Other-IDs]
NLM/ NIHMS434270; NLM/ PMC3559132
90.
Hou CP, Chiang YJ, Chu SH, Ng KF, Wang HH:
Mixed epithelial and stromal tumor of the kidney--a case report.
Chang Gung Med J
; 2010 Nov-Dec;33(6):693-8
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[Title]
Mixed epithelial and stromal
tumor
of the
kidney
--a case report.
She visited our hospital and a
renal
echo showed a heterogenous mass on the left
kidney
.
Abdominal computed tomography showed a multicystic
tumor
, about 7 cm, on the left
renal
pelvis and the proximal ureter.
The tumor
was enhanced after contrast injection.
Ureteroscopy showed an intraluminal polypoid
tumor
.
Cystic
renal
cell carcinoma or urothelial carcinoma was suspected preoperatively.
The pathology report demonstrated that
the tumor
was composed of an admixture of stroma and flattened to cuboidal urothelium.
The tumor
stromal cells expressed both estrogen and progesterone receptors, and no malignant cells were found.
There has been no recurrence or deterioration of the patient's
renal
function since surgery.
We suggest keeping in mind
the diagnosis
of mixed epithelial and stromal
tumor
of the
kidney
when encountering perimenopausal women with
renal
cystic
tumors
.
[MeSH-major]
Kidney
Neoplasms
/ pathology.
Neoplasms
, Complex and Mixed / pathology
MedlinePlus Health Information.
consumer health - Kidney Cancer
.
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(PMID = 21199615.001).
[ISSN]
2309-835X
[Journal-full-title]
Chang Gung medical journal
[ISO-abbreviation]
Chang Gung Med J
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
China (Republic : 1949- )
91.
Vassalotti JA, Stevens LA, Levey AS:
Testing for chronic kidney disease: a position statement from the National Kidney Foundation.
Am J Kidney Dis
; 2007 Aug;50(2):169-80
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[Title]
Testing for chronic
kidney
disease: a position statement from the National
Kidney
Foundation.
Chronic
kidney
disease (CKD) is common in the United States.
Major outcomes of CKD include progression to
kidney
failure, development of complications of impaired
kidney
function, and increased risk for cardiovascular disease.
CKD is usually silent until its late stages, thus many patients with CKD are detected only shortly before the onset of symptomatic
kidney
failure, when there are few opportunities to prevent adverse outcomes.
[MeSH-major]
Foundations / standards.
Kidney
Diseases / blood.
Kidney
Diseases / urine. Practice Guidelines as Topic
Genetic Alliance.
consumer health - Kidney Disease
.
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consumer health - Kidney Diseases
.
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[CommentIn]
Am J Kidney Dis. 2008 Jan;51(1):161-2; author reply 162-3
[
18155547.001
]
[CommentIn]
Am J Kidney Dis. 2008 Jan;51(1):162; author reply 162-3
[
18155548.001
]
(PMID = 17660017.001).
[ISSN]
1523-6838
[Journal-full-title]
American journal of kidney diseases : the official journal of the National Kidney Foundation
[ISO-abbreviation]
Am. J. Kidney Dis.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Number-of-references]
42
92.
Jiang L, Liang Y, Qiu B, Wang F, Duan X, Yang X, Huang W, Wang N:
Prevalence of chronic kidney disease in a rural Chinese adult population: the Handan Eye Study.
Nephron Clin Pract
; 2010;114(4):c295-302
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[Title]
Prevalence of chronic
kidney
disease in a rural Chinese adult population: the Handan Eye Study.
BACKGROUND: Chronic
kidney
disease (CKD) is a worldwide public health problem.
Glomerular filtration rate was estimated with the modified Modification of Diet in
Renal
Disease equation.
CKD was defined by the guidelines proposed by the
Kidney
Disease Outcomes Quality Initiative.
The associations between age, gender, diabetes, hypertension, and
kidney
damage were examined.
RESULTS: Albuminuria and reduced
renal
function were detected in 16.8 and 0.4% of subjects, respectively.
The age-standardized prevalence of albuminuria, reduced
renal
function and CKD was 14.9% (95% confidence interval (CI) 13.9-15.9), 0.38% (95% CI 0.21-0.55) and 15.2% (95% CI 14.2-16.2), respectively.
Older age was independently associated with reduced
renal
function.
[MeSH-major]
Asian Continental Ancestry Group / ethnology. Eye Diseases / ethnology.
Kidney
Failure, Chronic /
diagnosis
.
Kidney
Failure, Chronic / ethnology. Rural Population
Genetic Alliance.
consumer health - Kidney Disease
.
MedlinePlus Health Information.
consumer health - Eye Diseases
.
MedlinePlus Health Information.
consumer health - Kidney Failure
.
MedlinePlus Health Information.
consumer health - Rural Health Concerns
.
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[Copyright]
Copyright 2010 S. Karger AG, Basel.
(PMID = 20090372.001).
[ISSN]
1660-2110
[Journal-full-title]
Nephron. Clinical practice
[ISO-abbreviation]
Nephron Clin Pract
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Switzerland
93.
Hwang YH, Ro H, Choi I, Kim H, Oh KH, Hwang JI, Park MH, Kim S, Yang J, Ahn C:
Impact of polymorphisms of TLR4/CD14 and TLR3 on acute rejection in kidney transplantation.
Transplantation
; 2009 Sep 15;88(5):699-705
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[Title]
Impact of polymorphisms of TLR4/CD14 and TLR3 on acute rejection in
kidney
transplantation.
We evaluated the possible association between the TLR4/CD14 and TLR3 polymorphisms of donor-recipient pairs, and acute rejection after living donor
kidney
transplantation.
METHODS: TLR4 -1607T/C (rs10759932), -2026A/G (rs1927914); CD14 -159C/T (rs2569190); and TLR3 rs3775290, rs3775291, and rs3775296 were genotyped using DNA samples from 216 donor-recipient pairs of adult living donor
kidney
transplantation between January 1996 and July 2006.
CONCLUSION: These findings suggest the importance of TLR4 in the pathogenesis of acute rejection in
kidney
transplantation.
[MeSH-major]
Antigens, CD14 / genetics.
Kidney
Transplantation / methods. Polymorphism, Genetic. Toll-Like Receptor 3 / genetics. Toll-Like Receptor 4 / genetics
Genetic Alliance.
consumer health - Transplantation
.
MedlinePlus Health Information.
consumer health - Kidney Transplantation
.
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(PMID = 19741468.001).
[ISSN]
1534-6080
[Journal-full-title]
Transplantation
[ISO-abbreviation]
Transplantation
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Antigens, CD14; 0 / TLR3 protein, human; 0 / TLR4 protein, human; 0 / Toll-Like Receptor 3; 0 / Toll-Like Receptor 4
94.
Tombul ST, Aki FT, Gunay M, Inci K, Hazirolan T, Karcaaltincaba M, Erkan I, Bakkaloglu A, Yasavul U, Bakkaloglu M:
Preoperative evaluation of hilar vessel anatomy with 3-D computerized tomography in living kidney donors.
Transplant Proc
; 2008 Jan-Feb;40(1):47-9
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[Title]
Preoperative evaluation of hilar vessel anatomy with 3-D computerized tomography in living
kidney
donors.
OBJECTIVES: Digital subtract angiography is the gold standard for anatomic assessment of
renal
vasculature for living
renal
donors.
However, multidetector-row computerized tomography (MDCT) is less invasive than digital subtract angiography and provides information
of kidney
stones and other intra-abdominal organs.
In this study, preoperative MDCT angiography results were compared with the peroperative findings to evaluate the accuracy of MDCT for the evaluation of
renal
anatomy.
METHODS: From December 2002 to May 2007, all 60 consecutive living
kidney
donors were evaluated with MDCT angiography preoperatively.
We reported the number and origin of
renal
arteries, presence of early branching arteries, and any intrinsic
renal
artery disease.
Renal
venous anatomy was evaluated for the presence of accessory, retroaortic, and circumaortic veins using venous phase axial images.
RESULTS: A total of 67
renal
arteries were seen peroperatively in 60
renal
units.
CONCLUSION: MDCT angiography offers a less invasive, rapid, and accurate preoperative investigation modality for vascular anatomy in living
kidney
donors.
[MeSH-major]
Kidney
. Living Donors.
Renal
Artery / anatomy & histology.
Renal
Circulation. Tomography, X-Ray Computed
MedlinePlus Health Information.
consumer health - CT Scans
.
MedlinePlus Health Information.
consumer health - Organ Donation
.
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(PMID = 18261544.001).
[ISSN]
0041-1345
[Journal-full-title]
Transplantation proceedings
[ISO-abbreviation]
Transplant. Proc.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
95.
Vora N, Perrone R, Bianchi DW:
Reproductive issues for adults with autosomal dominant polycystic kidney disease.
Am J Kidney Dis
; 2008 Feb;51(2):307-18
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[Title]
Reproductive issues for adults with autosomal dominant polycystic
kidney
disease.
Autosomal dominant polycystic
kidney
disease (ADPKD) is a common
disorder
.
Affected women with ADPKD and normal
renal
function have a high rate of successful uncomplicated pregnancies.
Pregnant women with ADPKD with compromised
kidney
function should be monitored carefully for the development of hypertension and preeclampsia.
The diagnosis
of ADPKD should always be considered when prenatal sonographic findings of hyperechogenic enlarged
kidneys
are found.
In this setting, a family history and
renal
sonogram of both parents is indicated.
Sequencing of the PKD1 and PKD2 genes is available and can be used for both prenatal and preimplantation genetic
diagnosis
.
[MeSH-major]
Infertility, Male / physiopathology. Polycystic
Kidney
, Autosomal Dominant / physiopathology. Pregnancy Complications / physiopathology. Reproduction
[MeSH-minor]
Adult. Age of Onset. Counseling. Cysts / genetics. Female. Fetal Growth Retardation / prevention & control. Genital Diseases, Male / genetics. Humans. Hypertension, Pregnancy-Induced / prevention & control.
Kidne