[X] Close
You are about to erase all the values you have customized, search history, page format, etc.
Click here to RESET all values       Click here to GO BACK without resetting any value
Items 1 to 5 of about 5
1. Nistal M, Castillo MC, Regadera J, GarcĂ­a-Cabezas MA: Adenomatous hyperplasia of the rete testis. A review and report of new cases. Histol Histopathol; 2003 07;18(3):741-52
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Adenomatous hyperplasia of the rete testis (AHRT) is an uncommon benign lesion that preferentially involves the septal rete testis and mediastinal rete testis.
  • Some authors suggest a role for hormonal changes, tumour invasion and action of chemical agents.
  • The cases associated with different kidney and spermatic duct diseases, most cases associated with cryptorchidic testis and some cases associated with testicular germ cell tumour should be included in the congenital group.
  • The remaining cases associated with chemical agents, some hormonal changes (i.e. androgen blockade) and most of the germ cell tumour cases can be considered as acquired AHRT.
  • [MeSH-minor] Adenocarcinoma / pathology. Adolescent. Adult. Aged. Child. Child, Preschool. Cryptorchidism / pathology. Diagnosis, Differential. Humans. Immunohistochemistry. Infant. Kidney / pathology. Male. Middle Aged. Neoplasm Metastasis. Prostatic Neoplasms / diagnosis. Spermatozoa / pathology. Testicular Neoplasms / pathology. Testis / pathology

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 12792886.001).
  • [ISSN] 0213-3911
  • [Journal-full-title] Histology and histopathology
  • [ISO-abbreviation] Histol. Histopathol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] Spain
  • [Number-of-references] 50
  •  go-up   go-down


2. Biesheuvel CJ, Vergouwe Y, Steyerberg EW, Grobbee DE, Moons KG: Polytomous logistic regression analysis could be applied more often in diagnostic research. J Clin Epidemiol; 2008 Feb;61(2):125-34
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Polytomous logistic regression analysis could be applied more often in diagnostic research.
  • We discuss and (empirically) illustrate the value of this method for diagnostic research.
  • STUDY DESIGN AND SETTING: We used data from a study on the diagnosis of residual retroperitoneal mass histology in patients presenting with nonseminomatous testicular germ cell tumor.
  • The differential diagnoses include benign tissue, mature teratoma, and viable cancer.
  • For both modeling methods, we show the calibration plots and receiver operating characteristics curve (ROC) areas comparing each diagnostic outcome category with the other two.
  • The ROC areas for benign tissue, mature teratoma, and viable cancer were similar for both modeling methods, 0.83 (95% confidence interval [CI]=0.80-0.85) vs. 0.83 (95% CI=0.80-0.85), 0.78 (95% CI=0.75-0.81) vs. 0.78 (95% CI=0.75-0.81), and 0.66 (95% CI=0.61-0.71) vs. 0.64 (95% CI=0.59-0.69), for polytomous and dichotomous regression models, respectively.
  • CONCLUSION: Polytomous logistic regression is a useful technique to simultaneously model predicted probabilities of multiple diagnostic outcome categories.
  • Because the simultaneous consideration of the presence of multiple (differential) conditions serves clinical practice better than consideration of the presence of only one target condition, polytomous logistic regression could be applied more often in diagnostic research.
  • [MeSH-minor] Antineoplastic Agents / therapeutic use. Data Interpretation, Statistical. Humans. Male. Neoplasm, Residual. Retroperitoneal Neoplasms / diagnosis. Retroperitoneal Neoplasms / secondary. Teratoma / diagnosis. Teratoma / drug therapy. Teratoma / secondary. Testicular Neoplasms / drug therapy

  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 18177785.001).
  • [ISSN] 0895-4356
  • [Journal-full-title] Journal of clinical epidemiology
  • [ISO-abbreviation] J Clin Epidemiol
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Antineoplastic Agents
  •  go-up   go-down


3. Staal FJ, van der Luijt RB, Baert MR, van Drunen J, van Bakel H, Peters E, de Valk I, van Amstel HK, Taphoorn MJ, Jansen GH, van Veelen CW, Burgering B, Staal GE: A novel germline mutation of PTEN associated with brain tumours of multiple lineages. Br J Cancer; 2002 May 20;86(10):1586-91
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The biological consequences of this germline mutation were investigated by means of transfection studies of the mutant PTEN molecule compared to wild-type PTEN.
  • In contrast to the wild-type molecule, the mutant PTEN protein is not capable of inducing apoptosis, induces increased cell proliferation and leads to high constitutive PKB/Akt activation, which cannot be increased anymore by stimulation with insulin.
  • The reported patient, in addition to glioma, had suffered from benign meningioma in the past but did not show any clinical signs of Cowden disease or other hereditary diseases typically associated with PTEN germline mutations.
  • [MeSH-major] Amino Acid Substitution. Brain Neoplasms / genetics. Frontal Lobe. Germ-Line Mutation. Meningeal Neoplasms / genetics. Meningioma / genetics. Mutation, Missense. Neoplasm Proteins / genetics. Neoplasms, Multiple Primary / genetics. Oligodendroglioma / genetics. Phosphoric Monoester Hydrolases / genetics. Point Mutation. Protein-Serine-Threonine Kinases. Tumor Suppressor Proteins / genetics
  • [MeSH-minor] Adult. Apoptosis / genetics. Cell Division. Cell Lineage. DNA Mutational Analysis. DNA, Neoplasm / genetics. Enzyme Activation / drug effects. Genetic Predisposition to Disease. Humans. Insulin / pharmacology. Loss of Heterozygosity. Male. Models, Molecular. PTEN Phosphohydrolase. Protein Conformation. Proto-Oncogene Proteins / metabolism. Proto-Oncogene Proteins c-akt. Transfection. U937 Cells / drug effects. U937 Cells / enzymology

  • MedlinePlus Health Information. consumer health - Brain Tumors.
  • COS Scholar Universe. author profiles.
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • SciCrunch. OMIM: Data: Gene Annotation .
  • SciCrunch. Clinical Genomic Database: Data: Gene Annotation .
  • The Lens. Cited by Patents in .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] comCopyright 2002 Cancer Research UK
  • [Cites] Nat Genet. 1998 Aug;19(4):348-55 [9697695.001]
  • [Cites] Cancer Res. 1998 Jun 1;58(11):2331-4 [9622068.001]
  • [Cites] Curr Biol. 1998 Oct 22;8(21):1169-78 [9799734.001]
  • [Cites] Cancer Res. 1998 Nov 15;58(22):5002-8 [9823298.001]
  • [Cites] Proc Natl Acad Sci U S A. 1998 Dec 22;95(26):15406-11 [9860981.001]
  • [Cites] Proc Natl Acad Sci U S A. 1999 Feb 16;96(4):1563-8 [9990064.001]
  • [Cites] Science. 1999 Sep 24;285(5436):2122-5 [10497129.001]
  • [Cites] Cancer Res. 1999 Nov 1;59(21):5479-82 [10554022.001]
  • [Cites] Cell. 1999 Oct 29;99(3):323-34 [10555148.001]
  • [Cites] Annu Rev Biochem. 1999;68:965-1014 [10872470.001]
  • [Cites] Hum Mutat. 2000;16(2):109-22 [10923032.001]
  • [Cites] Science. 1997 Mar 28;275(5308):1943-7 [9072974.001]
  • [Cites] Nat Genet. 1997 Apr;15(4):356-62 [9090379.001]
  • [Cites] Cancer Res. 1997 Jun 1;57(11):2124-9 [9187108.001]
  • [Cites] Hum Mol Genet. 1997 Aug;6(8):1383-7 [9259288.001]
  • [Cites] Cytometry. 1997 Dec 1;29(4):286-91 [9415410.001]
  • [Cites] Am J Hum Genet. 1997 Dec;61(6):1234-8 [9399917.001]
  • [Cites] J Biol Chem. 1998 May 29;273(22):13375-8 [9593664.001]
  • [Cites] Cell. 1998 Oct 2;95(1):29-39 [9778245.001]
  • (PMID = 12085208.001).
  • [ISSN] 0007-0920
  • [Journal-full-title] British journal of cancer
  • [ISO-abbreviation] Br. J. Cancer
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Scotland
  • [Chemical-registry-number] 0 / DNA, Neoplasm; 0 / Insulin; 0 / Neoplasm Proteins; 0 / Proto-Oncogene Proteins; 0 / Tumor Suppressor Proteins; EC 2.7.11.1 / AKT1 protein, human; EC 2.7.11.1 / Protein-Serine-Threonine Kinases; EC 2.7.11.1 / Proto-Oncogene Proteins c-akt; EC 3.1.3.- / Phosphoric Monoester Hydrolases; EC 3.1.3.48 / PTEN protein, human; EC 3.1.3.67 / PTEN Phosphohydrolase
  • [Other-IDs] NLM/ PMC2746590
  •  go-up   go-down


Advertisement
4. Huang R, Jaffer S: Imprint cytology of metastatic sialoblastoma. A case report. Acta Cytol; 2003 Nov-Dec;47(6):1123-6
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • BACKGROUND: Sialoblastoma is a rare, aggressive and potentially malignant perinatal/congenital tumor that recapitulates the developing salivary gland.
  • Imprint smears and frozen section of the mass were diagnostic of metastatic sialoblastoma.
  • The clusters contained admixed benign ductal cells and dense, metachromatic, magenta hyaline globular material with smooth, rounded outlines.
  • The differential diagnoses include neoplasms composed of either basaloid cells and/or admixed hyaline matrix material and included pleomorphic adenoma, basal cell adenoma and adenoid cystic carcinoma.
  • [MeSH-major] Carcinoma, Adenoid Cystic / secondary. Lung Neoplasms / secondary. Neoplasms, Germ Cell and Embryonal / secondary. Neoplasms, Glandular and Epithelial / secondary. Parotid Neoplasms / pathology
  • [MeSH-minor] Antineoplastic Combined Chemotherapy Protocols / therapeutic use. Child, Preschool. Diagnosis, Differential. Disease Progression. Epithelial Cells / pathology. Female. Humans. Neoplasm Metastasis / pathology. Neoplasm Recurrence, Local / drug therapy. Neoplasm Recurrence, Local / pathology. Neoplasm Recurrence, Local / radiotherapy. Parotid Gland / pathology. Parotid Gland / surgery. Treatment Outcome

  • MedlinePlus Health Information. consumer health - Lung Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 14674095.001).
  • [ISSN] 0001-5547
  • [Journal-full-title] Acta cytologica
  • [ISO-abbreviation] Acta Cytol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  •  go-up   go-down


5. Lewis CM, Herbert BS, Bu D, Halloway S, Beck A, Shadeo A, Zhang C, Ashfaq R, Shay JW, Euhus DM: Telomerase immortalization of human mammary epithelial cells derived from a BRCA2 mutation carrier. Breast Cancer Res Treat; 2006 Sep;99(1):103-15
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • A novel human mammary epithelial cell line, HME348, was established from benign breast tissue from a 44-year-old germ-line BRCA2 mutation carrier with a history of stage 1 breast cancer.
  • Three clones derived from this culture were also p63(+)/ESA(+)/EMA(+/-) on glass but formed similar acinar structures with both luminal and myoepithelial cell differentiation in Matrigel confirming the mammary progenitor nature of these cells.
  • As this is the first report establishing and characterizing a benign human mammary epithelial cell line derived from a BRCA2 patient without the use of viral oncogenes, these cells may be useful for the study of BRCA2 function in breast morphogenesis and carcinogenesis.
  • [MeSH-minor] Animals. Cell Culture Techniques / methods. Collagen / pharmacology. Comet Assay. Drug Combinations. Humans. Laminin / pharmacology. Mice. Mice, Nude. Neoplasm Transplantation. Proteoglycans / pharmacology. Telomere / ultrastructure. Tumor Cells, Cultured

  • COS Scholar Universe. author profiles.
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16541310.001).
  • [ISSN] 0167-6806
  • [Journal-full-title] Breast cancer research and treatment
  • [ISO-abbreviation] Breast Cancer Res. Treat.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Drug Combinations; 0 / Laminin; 0 / Proteoglycans; 119978-18-6 / matrigel; 9007-34-5 / Collagen; EC 2.7.7.49 / Telomerase
  •  go-up   go-down






Advertisement