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1. Corrigendum to "Reduced Genotoxicity of Avian Sarcoma Leukosis Virus Vectors in Rhesus Long-term Repopulating Cells Compared to Standard Murine Retrovirus Vectors". Mol Ther; 2008 Oct;16(10):1770

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Corrigendum to "Reduced Genotoxicity of Avian Sarcoma Leukosis Virus Vectors in Rhesus Long-term Repopulating Cells Compared to Standard Murine Retrovirus Vectors".

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  • (PMID = 28189005.001).
  • [ISSN] 1525-0024
  • [Journal-full-title] Molecular therapy : the journal of the American Society of Gene Therapy
  • [ISO-abbreviation] Mol. Ther.
  • [Language] eng
  • [Publication-type] Published Erratum
  • [Publication-country] United States
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2. Kim YJ, Park SI, Park SJ, Kim HH, Jung YW, Kwon JT, Jang BG, Kim HK, Cho KO: Molecular analysis of endogenous avian leukosis/sarcoma virus genomes in Korean chicken embryos. J Vet Med Sci; 2008 Jan;70(1):17-23

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Molecular analysis of endogenous avian leukosis/sarcoma virus genomes in Korean chicken embryos.
  • Since the status of endogenous avian leucosis/sarcoma virus (ALSV) infections in Korean broiler chickens is unclear, this study examined embryonated eggs obtained from broiler farms and Korean native chicken breeds in Korea using PCR with the primer sets specific for endogenous ALSvs. The PCR assays detected the genomes of EAV, ev, ev/J and ART-CH belonging to the endogenous ALSV from all embryos tested.

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  • (PMID = 18250567.001).
  • [ISSN] 0916-7250
  • [Journal-full-title] The Journal of veterinary medical science
  • [ISO-abbreviation] J. Vet. Med. Sci.
  • [Language] ENG
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Japan
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3. Siwko SK, Bu W, Gutierrez C, Lewis B, Jechlinger M, Schaffhausen B, Li Y: Lentivirus-mediated oncogene introduction into mammary cells in vivo induces tumors. Neoplasia; 2008 Jul;10(7):653-62, 1 p following 662
Mouse Genome Informatics (MGI). Mouse Genome Informatics (MGI) .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • We recently reported the introduction of oncogene-expressing avian retroviruses into somatic mammary cells in mice susceptible to infection by transgenic expression of tva, encoding the receptor for subgroup A avian leukosis-sarcoma virus (ALSV).

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  • (PMID = 18592025.001).
  • [ISSN] 1476-5586
  • [Journal-full-title] Neoplasia (New York, N.Y.)
  • [ISO-abbreviation] Neoplasia
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / T32 CA090221; United States / NCI NIH HHS / CA / R01 CA113869; United States / NCI NIH HHS / CA / P01CA50661; United States / NCI NIH HHS / CA / P01 CA030195; United States / NCI NIH HHS / CA / R01CA34722; United States / NCI NIH HHS / CA / R01 CA034722; United States / NCI NIH HHS / CA / P01 CA050661
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antigens, Polyomavirus Transforming; 0 / Keratin-9
  • [Other-IDs] NLM/ PMC2435599
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4. Barnard RJ, Elleder D, Young JA: Avian sarcoma and leukosis virus-receptor interactions: from classical genetics to novel insights into virus-cell membrane fusion. Virology; 2006 Jan 5;344(1):25-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Avian sarcoma and leukosis virus-receptor interactions: from classical genetics to novel insights into virus-cell membrane fusion.
  • For over 40 years, avian sarcoma and leukosis virus (ASLV)-receptor interactions have been employed as a useful model system to study the mechanism of retroviral entry into cells.
  • [MeSH-major] Avian Leukosis Virus / metabolism. Receptors, Virus / metabolism. Sarcoma, Avian / virology. Viral Fusion Proteins / metabolism

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  • (PMID = 16364732.001).
  • [ISSN] 0042-6822
  • [Journal-full-title] Virology
  • [ISO-abbreviation] Virology
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Receptors, Virus; 0 / Viral Fusion Proteins; 0 / Viral Proteins
  • [Number-of-references] 32
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5. Hu J, Renaud G, Gomes TJ, Ferris A, Hendrie PC, Donahue RE, Hughes SH, Wolfsberg TG, Russell DW, Dunbar CE: Reduced genotoxicity of avian sarcoma leukosis virus vectors in rhesus long-term repopulating cells compared to standard murine retrovirus vectors. Mol Ther; 2008 Sep;16(9):1617-23
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Reduced genotoxicity of avian sarcoma leukosis virus vectors in rhesus long-term repopulating cells compared to standard murine retrovirus vectors.
  • In this study, we report for the first time a systematic analysis of 198 avian sarcoma leukosis virus (ASLV) insertion sites identified in rhesus long-term repopulating cells, and a comparison of ASLV insertion patterns to Moloney murine leukemia virus (MLV) (n = 396) and simian immunodeficiency virus (SIV) (n = 289) using the newly released rhesus genome databank.
  • [MeSH-major] Avian Leukosis Virus / genetics. Avian Sarcoma Viruses / genetics. Genetic Vectors / genetics. Hematopoietic Stem Cells / metabolism. Moloney murine leukemia virus / genetics. Transduction, Genetic. Virus Integration

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  • (PMID = 18578011.001).
  • [ISSN] 1525-0024
  • [Journal-full-title] Molecular therapy : the journal of the American Society of Gene Therapy
  • [ISO-abbreviation] Mol. Ther.
  • [Language] eng
  • [Grant] United States / Intramural NIH HHS / / Z01 HL002339-16; United States / Intramural NIH HHS / / Z99 HL999999
  • [Publication-type] Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / MDS1-EVI1 fusion protein, mouse; 0 / Oncogene Proteins, Fusion
  • [Other-IDs] NLM/ NIHMS51855; NLM/ PMC2561952
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6. Holman AG, Coffin JM: Symmetrical base preferences surrounding HIV-1, avian sarcoma/leukosis virus, and murine leukemia virus integration sites. Proc Natl Acad Sci U S A; 2005 Apr 26;102(17):6103-7
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Symmetrical base preferences surrounding HIV-1, avian sarcoma/leukosis virus, and murine leukemia virus integration sites.
  • To investigate retroviral integration targeting on a nucleotide scale, we examined the base frequencies directly surrounding cloned in vivo HIV-1, murine leukemia virus, and avian sarcoma/leukosis virus integrations.
  • Offset symmetry reflecting the topology of the integration reaction was found for HIV-1 and avian sarcoma/leukosis virus but not murine leukemia virus, suggesting fundamental differences in the way different retroviral integration complexes interact with host-cell DNA.

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  • [CommentIn] Proc Natl Acad Sci U S A. 2005 Apr 26;102(17):5903-4 [15840713.001]
  • [ErratumIn] Proc Natl Acad Sci U S A. 2005 Apr 26;102(17):6238
  • (PMID = 15802467.001).
  • [ISSN] 0027-8424
  • [Journal-full-title] Proceedings of the National Academy of Sciences of the United States of America
  • [ISO-abbreviation] Proc. Natl. Acad. Sci. U.S.A.
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / R01 CA092192; United States / NCI NIH HHS / CA / T32 CA065441; United States / NCI NIH HHS / CA / R01-CA-92192; United States / NCI NIH HHS / CA / T32 CA65441
  • [Publication-type] Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC1087937
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7. Hu J, Renaud G, Golmes T, Ferris A, Hendrie PC, Donahue RE, Hughes SH, Wolfsberg TG, Russell DW, Dunbar CE: Reduced Genotoxicity of Avian Sarcoma Leukosis Virus Vectors in Rhesus Long-term Repopulating Cells Compared to Standard Murine Retrovirus Vectors. Mol Ther; 2008 Sep;16(9):1617-1623

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Reduced Genotoxicity of Avian Sarcoma Leukosis Virus Vectors in Rhesus Long-term Repopulating Cells Compared to Standard Murine Retrovirus Vectors.
  • In this study, we report for the first time a systematic analysis of 198 avian sarcoma leukosis virus (ASLV) insertion sites identified in rhesus long-term repopulating cells, and a comparison of ASLV insertion patterns to Moloney murine leukemia virus (MLV) (n = 396) and simian immunodeficiency virus (SIV) (n = 289) using the newly released rhesus genome databank.

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  • [Copyright] Copyright © 2008 The American Society of Gene Therapy. Published by Elsevier Inc. All rights reserved.
  • (PMID = 28189014.001).
  • [ISSN] 1525-0024
  • [Journal-full-title] Molecular therapy : the journal of the American Society of Gene Therapy
  • [ISO-abbreviation] Mol. Ther.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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8. Hatai H, Ochiai K, Nagakura K, Imanishi S, Ochi A, Kozakura R, Ono M, Goryo M, Ohashi K, Umemura T: A recombinant avian leukosis virus associated with fowl glioma in layer chickens in Japan. Avian Pathol; 2008 Apr;37(2):127-37

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] A recombinant avian leukosis virus associated with fowl glioma in layer chickens in Japan.
  • Fowl glioma is characterized by multiple nodular growth of astrocytes, and fowl glioma-inducing virus belonging to avian leukosis virus has been isolated from Japanese bantam as a causal agent.
  • The genome of one isolate mainly consisted of ev loci and contained several parts of other avian leukosis/sarcoma viruses.
  • These results show that the causal avian leukosis virus of fowl glioma is not just fowl glioma-inducing virus and that different avian leukosis virus strains having oncogenicity in the central nervous system by recombination are spread in layers in Japan.
  • [MeSH-major] Avian Leukosis / virology. Avian Leukosis Virus / classification. Chickens / virology. Glioma / veterinary. Reassortant Viruses

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  • (PMID = 18393089.001).
  • [ISSN] 1465-3338
  • [Journal-full-title] Avian pathology : journal of the W.V.P.A
  • [ISO-abbreviation] Avian Pathol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
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9. Hu J, Ferris A, Larochelle A, Krouse AE, Metzger ME, Donahue RE, Hughes SH, Dunbar CE: Transduction of rhesus macaque hematopoietic stem and progenitor cells with avian sarcoma and leukosis virus vectors. Hum Gene Ther; 2007 Aug;18(8):691-700
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Transduction of rhesus macaque hematopoietic stem and progenitor cells with avian sarcoma and leukosis virus vectors.
  • Avian sarcoma and leukosis viruses (ASLVs) were previously reported to have a weak preference for gene-coding regions in a cell line study as compared with human immunodeficiency virus and MoMLV; however, thus far these vectors have not been studied for their potential efficacy in transduction of hematopoietic progenitor and stem cells.
  • In this study we investigated for the first time the ability of ASLV-derived RCAS (replication-competent ALV LTR [avian leukosis virus long terminal repeat] with a splice acceptor) vectors to transduce rhesus macaque hematopoietic progenitors and long-term repopulating cells, in an autologous transplantation model.
  • [MeSH-major] Avian Leukosis Virus. Avian Sarcoma Viruses. Genetic Vectors. Hematopoietic Stem Cells / metabolism. Transduction, Genetic

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  • (PMID = 17655493.001).
  • [ISSN] 1043-0342
  • [Journal-full-title] Human gene therapy
  • [ISO-abbreviation] Hum. Gene Ther.
  • [Language] eng
  • [Grant] United States / Intramural NIH HHS / /
  • [Publication-type] Journal Article; Research Support, N.I.H., Intramural
  • [Publication-country] United States
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10. Torne-Celer C, Moreau K, Faure C, Verdier G, Ronfort C: An improved self-deleting retroviral vector derived from avian leukemia and sarcoma virus. Arch Virol; 2008;153(12):2233-43

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] An improved self-deleting retroviral vector derived from avian leukemia and sarcoma virus.
  • We have previously developed a self-deleting avian leukosis and sarcoma virus (ALSV)- based retroviral vector carrying an additional attachment (att) sequence.

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  • (PMID = 19018452.001).
  • [ISSN] 1432-8798
  • [Journal-full-title] Archives of virology
  • [ISO-abbreviation] Arch. Virol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Austria
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11. Beniaminov AD, Samokhin AB, Ul'ianov NB, Miniat EE: [Characteristic of the two-step RNA dimerization in avian sarcoma and leukosis viruses]. Mol Biol (Mosk); 2005 Jan-Feb;39(1):147-54

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Characteristic of the two-step RNA dimerization in avian sarcoma and leukosis viruses].
  • Similar model of two-step dimerization mechanism can be considered for avian sarcoma and leukosis virus group (ASLV) in spite of the absence of homology between dimer initiation site (DIS) of ASLV and that of HIV-1.
  • In this paper, short RNA fragments of two viruses: avian sarcoma virus CT-10 and avian leukosis virus HPRS-103 have been chosen in order to investigate the structural requirements of dimerization process and compare them to that of HIV-1.
  • Although both types of dimers were observed for both avian retroviruses chosen, fragments of CT-10 requires much higher RNA concentration to form loose dimer.
  • In spite of identical sequence of the loops (5'-A-CUGCAG-3') avian sarcoma virus CT-10 RNA fragments dimerization was greatly impaired.
  • The differences can be explained by deletion of adenine 271 in avian sarcoma virus CT-10 in the stem and by resulting shortening of the self-complementary loop.
  • [MeSH-major] Avian Leukosis Virus / genetics. Avian Sarcoma Viruses / genetics. Genome, Viral. RNA, Viral / genetics

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  • (PMID = 15773559.001).
  • [ISSN] 0026-8984
  • [Journal-full-title] Molekuliarnaia biologiia
  • [ISO-abbreviation] Mol. Biol. (Mosk.)
  • [Language] rus
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] Russia
  • [Chemical-registry-number] 0 / RNA, Viral
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12. Barr SD, Leipzig J, Shinn P, Ecker JR, Bushman FD: Integration targeting by avian sarcoma-leukosis virus and human immunodeficiency virus in the chicken genome. J Virol; 2005 Sep;79(18):12035-44
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Integration targeting by avian sarcoma-leukosis virus and human immunodeficiency virus in the chicken genome.
  • We have analyzed the placement of sites of integration of avian sarcoma-leukosis virus (ASLV) and human immunodeficiency virus (HIV) DNA in the draft chicken genome sequence, with the goals of assessing species-specific effects on integration and allowing comparison to the distribution of chicken endogenous retroviruses (ERVs).

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  • (PMID = 16140779.001).
  • [ISSN] 0022-538X
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Databank-accession-numbers] GENBANK/ CZ905239/ CZ905240/ CZ905241/ CZ905242/ CZ905243/ CZ905244/ CZ905245/ CZ905246/ CZ905247/ CZ905248/ CZ905249/ CZ905250/ CZ905251/ CZ905252/ CZ905253/ CZ905254/ CZ905255/ CZ905256/ CZ905257/ CZ905258/ CZ905259/ CZ905260/ CZ905261/ CZ905262/ CZ905263/ CZ905264/ CZ905265/ CZ905266/ CZ905267/ CZ905268/ CZ905269/ CZ905270/ CZ905271/ CZ905272/ CZ905273/ CZ905274/ CZ905275/ CZ905276/ CZ905277/ CZ905278/ CZ905279/ CZ905280/ CZ905281/ CZ905282/ CZ905283/ CZ905284/ CZ905285/ CZ905286/ CZ905287/ CZ905288/ CZ905289/ CZ905290/ CZ905291/ CZ905292/ CZ905293/ CZ905294/ CZ905295/ CZ905296/ CZ905297/ CZ905298/ CZ905299/ CZ905300/ CZ905301/ CZ905302/ CZ905303/ CZ905304/ CZ905305/ CZ905306/ CZ905307/ CZ905308/ CZ905309/ CZ905310/ CZ905311/ CZ905312/ CZ905313/ CZ905314/ CZ905315/ CZ905316/ CZ905317/ CZ905318/ CZ905319/ CZ905320/ CZ905321/ CZ905322/ CZ905323/ CZ905324/ CZ905325/ CZ905326/ CZ905327/ CZ905328/ CZ905329/ CZ905330/ CZ905331/ CZ905332/ CZ905333/ 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  • [Grant] United States / NIAID NIH HHS / AI / R01 AI034786; United States / NIAID NIH HHS / AI / R01 AI052845; United States / NIAID NIH HHS / AI / AI34786; United States / NIAID NIH HHS / AI / AI52845
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 9007-49-2 / DNA
  • [Other-IDs] NLM/ PMC1212630
  •  go-up   go-down


13. Rai T, Caffrey M, Rong L: Identification of two residues within the LDL-A module of Tva that dictate the altered receptor specificity of mutant subgroup A avian sarcoma and leukosis viruses. J Virol; 2005 Dec;79(23):14962-6
SciCrunch. OMIA - Online Mendelian Inheritance in Animals: Data: Gene Expression .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Identification of two residues within the LDL-A module of Tva that dictate the altered receptor specificity of mutant subgroup A avian sarcoma and leukosis viruses.
  • Avian sarcoma and leukosis virus subgroup A (ASLV-A) entry is mediated by interactions between the viral glycoprotein EnvA and its cognate receptor Tva.

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  • (PMID = 16282495.001).
  • [ISSN] 0022-538X
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / R01 CA092459; United States / NCI NIH HHS / CA / CA 092459
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Amino Acids; 0 / Avian Proteins; 0 / Receptors, Virus; 0 / Tva receptor
  • [Other-IDs] NLM/ PMC1287562
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14. Delos SE, Brecher MB, Chen Z, Melder DC, Federspiel MJ, White JM: Cysteines flanking the internal fusion peptide are required for the avian sarcoma/leukosis virus glycoprotein to mediate the lipid mixing stage of fusion with high efficiency. J Virol; 2008 Mar;82(6):3131-4
Hazardous Substances Data Bank. CYSTEINE .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Cysteines flanking the internal fusion peptide are required for the avian sarcoma/leukosis virus glycoprotein to mediate the lipid mixing stage of fusion with high efficiency.
  • We previously showed that the cysteines flanking the internal fusion peptide of the avian sarcoma/leukosis virus subtype A (ASLV-A) Env (EnvA) are important for infectivity and cell-cell fusion.

  • COS Scholar Universe. author profiles.
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  • (PMID = 18184714.001).
  • [ISSN] 1098-5514
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NIAID NIH HHS / AI / AI22470; United States / NIAID NIH HHS / AI / R01 AI048682; United States / NIAID NIH HHS / AI / R01 AI022470; United States / NIAID NIH HHS / AI / AI48682; United States / NIAID NIH HHS / AI / T32 AI07076-27; United States / NIAID NIH HHS / AI / T32 AI055432
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Glycoproteins; 0 / Viral Proteins; K848JZ4886 / Cysteine
  • [Other-IDs] NLM/ PMC2259008
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15. Zhang XY, Kutner RH, Bialkowska A, Marino MP, Klimstra WB, Reiser J: Cell-specific targeting of lentiviral vectors mediated by fusion proteins derived from Sindbis virus, vesicular stomatitis virus, or avian sarcoma/leukosis virus. Retrovirology; 2010 Jan 25;7:3
The Lens. Cited by Patents in .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Cell-specific targeting of lentiviral vectors mediated by fusion proteins derived from Sindbis virus, vesicular stomatitis virus, or avian sarcoma/leukosis virus.
  • An alternative approach involved soluble avian sarcoma/leukosis virus receptors fused to cell-specific ligands including stem cell factor and erythropoietin for targeting lentiviral vectors pseudotyped with avian sarcoma/leukosis virus envelope proteins to cells that express the corresponding receptors.
  • Lentiviral vectors pseudotyped with avian sarcoma/leukosis virus subgroup A or B envelope proteins and bearing bi-functional bridge proteins encoding erythropoietin or stem cell factor fused to the soluble extracellular domains of the avian sarcoma/leukosis virus subgroup A or B receptors resulted in efficient transduction of erythropoietin receptor or c-kit-expressing cells.
  • CONCLUSIONS: Our results indicate that the avian sarcoma/leukosis virus bridge strategy provides a reliable approach for cell-specific lentiviral vector targeting.

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  • (PMID = 20100344.001).
  • [ISSN] 1742-4690
  • [Journal-full-title] Retrovirology
  • [ISO-abbreviation] Retrovirology
  • [Language] ENG
  • [Grant] United States / NINDS NIH HHS / NS / NS044832
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Viral Fusion Proteins
  • [Other-IDs] NLM/ PMC2823649
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16. Klucking S, Collins AS, Young JA: Avian sarcoma and leukosis virus cytopathic effect in the absence of TVB death domain signaling. J Virol; 2005 Jul;79(13):8243-8
NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Avian sarcoma and leukosis virus cytopathic effect in the absence of TVB death domain signaling.
  • The cytopathic effect (CPE) seen with some subgroups of avian sarcoma and leukosis virus (ASLV) is associated with viral Env activation of the death-promoting activity of TVB (a tumor necrosis factor receptor-related receptor that is most closely related to mammalian TNF-related apoptosis-inducing ligand [TRAIL] receptors) and with viral superinfection leading to unintegrated viral DNA (UVD) accumulation, which is presumed to activate a cellular DNA damage response.

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  • (PMID = 15956570.001).
  • [ISSN] 0022-538X
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / R01 CA062000; United States / NCI NIH HHS / CA / CA62000
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / DNA Primers; 0 / Gene Products, env; 0 / Receptors, Virus
  • [Other-IDs] NLM/ PMC1143719
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17. Elleder D, Stepanets V, Melder DC, Senigl F, Geryk J, Pajer P, Plachý J, Hejnar J, Svoboda J, Federspiel MJ: The receptor for the subgroup C avian sarcoma and leukosis viruses, Tvc, is related to mammalian butyrophilins, members of the immunoglobulin superfamily. J Virol; 2005 Aug;79(16):10408-19
SciCrunch. OMIA - Online Mendelian Inheritance in Animals: Data: Gene Expression .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The receptor for the subgroup C avian sarcoma and leukosis viruses, Tvc, is related to mammalian butyrophilins, members of the immunoglobulin superfamily.
  • The five highly related envelope subgroups of the avian sarcoma and leukosis viruses (ASLVs), subgroup A [ASLV(A)] to ASLV(E), are thought to have evolved from an ancestral envelope glycoprotein yet utilize different cellular proteins as receptors.

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  • (PMID = 16051833.001).
  • [ISSN] 0022-538X
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NIAID NIH HHS / AI / R01 AI048682; United States / NIAID NIH HHS / AI / AI48682
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Codon, Terminator; 0 / Membrane Glycoproteins; 0 / Receptors, Virus; 0 / butyrophilin
  • [Other-IDs] NLM/ PMC1182627
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18. Babel AR, Bruce J, Young JA: The hr1 and fusion peptide regions of the subgroup B avian sarcoma and leukosis virus envelope glycoprotein influence low pH-dependent membrane fusion. PLoS One; 2007 Jan 24;2(1):e171

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The hr1 and fusion peptide regions of the subgroup B avian sarcoma and leukosis virus envelope glycoprotein influence low pH-dependent membrane fusion.
  • The avian sarcoma and leukosis virus (ASLV) envelope glycoprotein (Env) is activated to trigger fusion by a two-step mechanism involving receptor-priming and low pH fusion activation.

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  • (PMID = 17245447.001).
  • [ISSN] 1932-6203
  • [Journal-full-title] PloS one
  • [ISO-abbreviation] PLoS ONE
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / P01-CA22443; United States / NIAID NIH HHS / AI / R01 AI053668; United States / NCI NIH HHS / CA / CA70810; United States / NIAID NIH HHS / AI / AI053668; United States / NCI NIH HHS / CA / P01 CA022443; United States / NCI NIH HHS / CA / T32 CA009135; United States / NCI NIH HHS / CA / R01 CA070810
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Gene Products, env; 0 / Peptides; 0 / Viral Fusion Proteins
  • [Other-IDs] NLM/ PMC1764858
  • [General-notes] NLM/ Original DateCompleted: 20070723
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19. Melder DC, Yin X, Delos SE, Federspiel MJ: A charged second-site mutation in the fusion peptide rescues replication of a mutant avian sarcoma and leukosis virus lacking critical cysteine residues flanking the internal fusion domain. J Virol; 2009 Sep;83(17):8575-86
NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .

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  • [Title] A charged second-site mutation in the fusion peptide rescues replication of a mutant avian sarcoma and leukosis virus lacking critical cysteine residues flanking the internal fusion domain.
  • The entry process of the avian sarcoma and leukosis virus (ASLV) family of retroviruses requires first a specific interaction between the viral surface (SU) glycoproteins and a receptor on the cell surface at a neutral pH, triggering conformational changes in the viral SU and transmembrane (TM) glycoproteins, followed by exposure to low pH to complete fusion.

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  • (PMID = 19515762.001).
  • [ISSN] 1098-5514
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NIAID NIH HHS / AI / R01 AI022470; United States / NIAID NIH HHS / AI / R01 AI048682; United States / NIAID NIH HHS / AI / AI22470; United States / NIAID NIH HHS / AI / AI48682
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Avian Proteins; 0 / Receptors, Virus; 0 / Tva receptor; 0 / Viral Fusion Proteins; K848JZ4886 / Cysteine
  • [Other-IDs] NLM/ PMC2738199
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20. Delos SE, La B, Gilmartin A, White JM: Studies of the "chain reversal regions" of the avian sarcoma/leukosis virus (ASLV) and ebolavirus fusion proteins: analogous residues are important, and a His residue unique to EnvA affects the pH dependence of ASLV entry. J Virol; 2010 Jun;84(11):5687-94
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Studies of the "chain reversal regions" of the avian sarcoma/leukosis virus (ASLV) and ebolavirus fusion proteins: analogous residues are important, and a His residue unique to EnvA affects the pH dependence of ASLV entry.
  • Here, we used mutagenesis and pseudovirus entry assays to test this model for the avian retrovirus avian sarcoma/leukosis virus (ASLV) and the filovirus ebolavirus Zaire.

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  • (PMID = 20335266.001).
  • [ISSN] 1098-5514
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NIAID NIH HHS / AI / R01 AI022470; United States / NIAID NIH HHS / AI / U54 AI057168; United States / NIAID NIH HHS / AI / R01 AI22470; United States / NIAID NIH HHS / AI / U54 AI57168
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Viral Envelope Proteins; 0 / Viral Fusion Proteins; 4QD397987E / Histidine
  • [Other-IDs] NLM/ PMC2876614
  •  go-up   go-down


21. Munguia A, Federspiel MJ: Efficient subgroup C avian sarcoma and leukosis virus receptor activity requires the IgV domain of the Tvc receptor and proper display on the cell membrane. J Virol; 2008 Nov;82(22):11419-28
SciCrunch. OMIA - Online Mendelian Inheritance in Animals: Data: Gene Expression .

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  • [Title] Efficient subgroup C avian sarcoma and leukosis virus receptor activity requires the IgV domain of the Tvc receptor and proper display on the cell membrane.
  • We recently identified and cloned the receptor for subgroup C avian sarcoma and leukosis viruses [ASLV(C)], i.e., Tvc, a protein most closely related to mammalian butyrophilins, which are members of the immunoglobulin protein family.

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  • (PMID = 18768966.001).
  • [ISSN] 1098-5514
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NIAID NIH HHS / AI / R01 AI048682; United States / NIAID NIH HHS / AI / AI48682
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Receptors, Virus; 0 / Viral Envelope Proteins
  • [Other-IDs] NLM/ PMC2573256
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22. Delos SE, Godby JA, White JM: Receptor-induced conformational changes in the SU subunit of the avian sarcoma/leukosis virus A envelope protein: implications for fusion activation. J Virol; 2005 Mar;79(6):3488-99
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Receptor-induced conformational changes in the SU subunit of the avian sarcoma/leukosis virus A envelope protein: implications for fusion activation.
  • The avian sarcoma/leukosis virus (ASLV) is activated for fusion by a two-step mechanism.

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  • (PMID = 15731243.001).
  • [ISSN] 0022-538X
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NIAID NIH HHS / AI / R01 AI022470; United States / NIAID NIH HHS / AI / AI22470
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Avian Proteins; 0 / Protein Subunits; 0 / Receptors, Virus; 0 / Recombinant Proteins; 0 / Tva receptor; 0 / Viral Envelope Proteins; 0 / glycoprotein 85, avian leukosis virus
  • [Other-IDs] NLM/ PMC1075726
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23. Reinisová M, Senigl F, Yin X, Plachy J, Geryk J, Elleder D, Svoboda J, Federspiel MJ, Hejnar J: A single-amino-acid substitution in the TvbS1 receptor results in decreased susceptibility to infection by avian sarcoma and leukosis virus subgroups B and D and resistance to infection by subgroup E in vitro and in vivo. J Virol; 2008 Mar;82(5):2097-105
SciCrunch. OMIA - Online Mendelian Inheritance in Animals: Data: Gene Expression .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] A single-amino-acid substitution in the TvbS1 receptor results in decreased susceptibility to infection by avian sarcoma and leukosis virus subgroups B and D and resistance to infection by subgroup E in vitro and in vivo.
  • The avian sarcoma and leukosis virus (ASLV) family of retroviruses contains five highly related envelope subgroups (A to E) thought to have evolved from a common viral ancestor in the chicken population.
  • Here, we show that the C125S substitution in Tvb(S1) significantly reduces the susceptibility of line M cells to infection by ASLV(B) and ASLV(D) and virtually eliminates susceptibility to ASLV(E) infection both in cultured cells and in the incidence and growth of avian sarcoma virus-induced sarcomas in chickens.

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  • (PMID = 18094190.001).
  • [ISSN] 1098-5514
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NIAID NIH HHS / AI / R01 AI048682; United States / NIAID NIH HHS / AI / AI48682
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / DNA Primers; 0 / Receptors, Virus
  • [Other-IDs] NLM/ PMC2258910
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24. Senigl F, Plachý J, Hejnar J: The core element of a CpG island protects avian sarcoma and leukosis virus-derived vectors from transcriptional silencing. J Virol; 2008 Aug;82(16):7818-27
The Lens. Cited by Patents in .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The core element of a CpG island protects avian sarcoma and leukosis virus-derived vectors from transcriptional silencing.
  • We report here the insertion of this CpG island core element, IE, into the long terminal repeat of a retroviral vector derived from Rous sarcoma virus, which normally suffers from progressive transcriptional silencing in mammalian cells.
  • [MeSH-major] Avian Leukosis Virus / metabolism. CpG Islands. Gene Silencing. Sarcoma, Avian / genetics. Sarcoma, Avian / virology. Transcription, Genetic
  • [MeSH-minor] Animals. Binding Sites. Birds. Flow Cytometry. Genes, Reporter. Humans. Models, Biological. Mutation. Rous sarcoma virus / metabolism. Sp1 Transcription Factor / metabolism. Terminal Repeat Sequences

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  • (PMID = 18550662.001).
  • [ISSN] 1098-5514
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Sp1 Transcription Factor
  • [Other-IDs] NLM/ PMC2519551
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25. Cheng Z, Liu J, Cui Z, Zhang L: Tumors associated with avian leukosis virus subgroup J in layer hens during 2007 to 2009 in China. J Vet Med Sci; 2010 Aug;72(8):1027-33

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Tumors associated with avian leukosis virus subgroup J in layer hens during 2007 to 2009 in China.
  • In the 3 years leading up to November 2009, 6 different types of naturally occurring neoplasms associated with avian leukosis virus subgroup J (ALV-J) were diagnosed by histopathology, polymerase chain reaction (PCR) and immunohistochemistry (IHC) in 140 layer hens out of approximately 100,000.
  • The most prevalent tumor type was hemangioma (50%) in commercial layer flocks; the second most prevalent neoplasm type was myelocytoma (38.6%); a small number of ALV-J positive lymphomas (4.3%) that were not associated with Marek's disease (MD) or lymphoid leukosis (LL) was observed.

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  • (PMID = 20467208.001).
  • [ISSN] 0916-7250
  • [Journal-full-title] The Journal of veterinary medical science
  • [ISO-abbreviation] J. Vet. Med. Sci.
  • [Language] ENG
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / DNA Primers
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26. Du Z, Podsypanina K, Huang S, McGrath A, Toneff MJ, Bogoslovskaia E, Zhang X, Moraes RC, Fluck M, Allred DC, Lewis MT, Varmus HE, Li Y: Introduction of oncogenes into mammary glands in vivo with an avian retroviral vector initiates and promotes carcinogenesis in mouse models. Proc Natl Acad Sci U S A; 2006 Nov 14;103(46):17396-401
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Introduction of oncogenes into mammary glands in vivo with an avian retroviral vector initiates and promotes carcinogenesis in mouse models.
  • We have adapted the avian leukosis virus RCAS (replication-competent avian sarcoma-leukosis virus LTR splice acceptor)-mediated somatic gene transfer technique to introduce oncogenes into mammary cells in mice transgenic for the avian subgroup A receptor gene, tva, under control of the mouse mammary tumor virus (MMTV) promoter.

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  • (PMID = 17090666.001).
  • [ISSN] 0027-8424
  • [Journal-full-title] Proceedings of the National Academy of Sciences of the United States of America
  • [ISO-abbreviation] Proc. Natl. Acad. Sci. U.S.A.
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / U01 CA084243-07; United States / NCI NIH HHS / CA / R01 CA113869; United States / NCI NIH HHS / CA / P01 CA94060-02; United States / NCI NIH HHS / CA / U01 CA084243; United States / NCI NIH HHS / CA / P50 CA058183; United States / NCI NIH HHS / CA / P01 CA094060
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antigens, Polyomavirus Transforming; 0 / Myc protein, mouse; 0 / Proto-Oncogene Proteins c-myc; 136601-57-5 / Cyclin D1
  • [Other-IDs] NLM/ PMC1635021
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27. Pandiri AR, Gimeno IM, Reed WM, Fitzgerald SD, Fadly AM: Subgroup J avian leukosis virus-induced histiocytic sarcomatosis occurs only in persistently viremic but not immunotolerized meat-type chickens. Vet Pathol; 2009 Mar;46(2):282-7
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Subgroup J avian leukosis virus-induced histiocytic sarcomatosis occurs only in persistently viremic but not immunotolerized meat-type chickens.
  • The role of subgroup J avian leukosis virus (ALV J) infection profile in the development of histiocytic sarcomatosis (HS) in chickens was evaluated using retrospective analysis of 2 experiments involving in ovo and at-hatch inoculation of commercial meat-type and ADOL line 0 chickens with 100 or 10,000 TCID(50) of various strains ALV J.
  • [MeSH-major] Avian Leukosis Virus / classification. Chickens. Histiocytic Sarcoma / veterinary. Poultry Diseases / pathology. Viremia

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  • (PMID = 19261641.001).
  • [ISSN] 0300-9858
  • [Journal-full-title] Veterinary pathology
  • [ISO-abbreviation] Vet. Pathol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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28. Wu X, Qin A, Qian K, Jin W, Shao H, Zhu Y, Shen H: [Full-length cDNA sequence analysis of avian leukosis viruses subgroup J isolated from chickens with clinical hemangioma]. Wei Sheng Wu Xue Bao; 2010 Sep;50(9):1264-72
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Full-length cDNA sequence analysis of avian leukosis viruses subgroup J isolated from chickens with clinical hemangioma].
  • OBJECTIVE: To understand the molecular characteristics of subgroup J Avian Leukosis Viruses (ALV-J) isolated from chickens with clinical hemangioma, as well as to get more information for controlling the spread of ALV-J in layer chickens flocks.
  • METHOD: We amplified the full-length viral cDNA sequences of three layers isolates associated with simple hemangioma or coexisting of hemangioma and myeloid leukosis (ML) by PCR.
  • We noted a special 19bp insertion mutation in Primer Binding Site (PBS)-Leader sequences in both JS09GY3 and JS09GY6 isolates from layer chickens with hemangioma and ML, which sequence was same to Rous Associated Virus type 1 (RAV-1), Rous Associated Virus type 2 (RAV-2) and Rous sarcoma virus (strain Schmidt-Ruppin B) (RSV-SRB).
  • [MeSH-major] Avian Leukosis Virus / genetics. Avian Leukosis Virus / isolation & purification. Hemangioma / virology

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  • (PMID = 21090269.001).
  • [ISSN] 0001-6209
  • [Journal-full-title] Wei sheng wu xue bao = Acta microbiologica Sinica
  • [ISO-abbreviation] Wei Sheng Wu Xue Bao
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] China
  • [Chemical-registry-number] 0 / DNA, Complementary; 0 / DNA, Viral
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29. Wu X, Qian K, Qin A, Shen H, Wang P, Jin W, Eltahir YM: Recombinant avian leukosis viruses of subgroup J isolated from field infected commercial layer chickens with hemangioma and myeloid leukosis possess an insertion in the E element. Vet Res Commun; 2010 Oct;34(7):619-32
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Recombinant avian leukosis viruses of subgroup J isolated from field infected commercial layer chickens with hemangioma and myeloid leukosis possess an insertion in the E element.
  • BACKGROUND: Five isolates (JS09GY2, JS09GY3, JS09GY4, JS09GY5, and JS09GY6) of avian leukosis virus subgroup J (ALV-J) were isolated from six infected commercial layer flocks displaying both hemangioma and myeloid leukosis (ML), which shared the same parental line, in China in 2009.
  • By full genome sequences analysis, a 19-nucleotide repeat sequence was identified in the primer binding site (PBS)-leader region of isolates JS09GY3 and JS09GY6, located between sites 249 and 250 according to the sequence of reference strain HPRS103, and also present in Rous sarcoma virus strain Schmidt-Ruppin B (RSV-SRB), Rous associated virus type 1 (RAV-1), and Rous associated virus type 2 (RAV-2).
  • [MeSH-major] Avian Leukosis / virology. Avian Leukosis Virus / classification. Avian Leukosis Virus / genetics. Chickens. Hemangioma / veterinary

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  • (PMID = 20676760.001).
  • [ISSN] 1573-7446
  • [Journal-full-title] Veterinary research communications
  • [ISO-abbreviation] Vet. Res. Commun.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / DNA Transposable Elements
  • [Other-IDs] NLM/ PMC2931761
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30. Zhang HM, Bacon LD, Cheng HH, Hunt HD: Development and validation of a PCR-RFLP assay to evaluate TVB haplotypes coding receptors for subgroup B and subgroup E avian leukosis viruses in White Leghorns. Avian Pathol; 2005 Aug;34(4):324-31
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Development and validation of a PCR-RFLP assay to evaluate TVB haplotypes coding receptors for subgroup B and subgroup E avian leukosis viruses in White Leghorns.
  • The cellular receptor of subgroup B avian leukosis virus (ALVB) is encoded by a gene at the tumour virus B (TVB) locus.
  • TVB alleles encode specific receptors permitting infection by exogenous ALVB or avian leukosis virus subgroup D (ALVD) as well as endogenous avian leukosis virus subgroup E (ALVE), and thus susceptibility is dominant to resistance.
  • Chickens from parents heterozygous for different TVB alleles were challenged with Rous sarcoma viruses of subgroup ALVB and ALVE to induce wing-web tumours.
  • [MeSH-major] Avian Leukosis Virus. Avian Proteins / genetics. Chickens / genetics. Polymerase Chain Reaction / methods. Receptors, Virus / genetics
  • [MeSH-minor] Alleles. Animals. Avian Sarcoma Viruses. Haplotypes. Polymorphism, Restriction Fragment Length. Sarcoma, Avian / virology

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  • (PMID = 16147569.001).
  • [ISSN] 0307-9457
  • [Journal-full-title] Avian pathology : journal of the W.V.P.A
  • [ISO-abbreviation] Avian Pathol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Avian Proteins; 0 / Receptors, Virus
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31. Smith CA, Roeszler KN, Sinclair AH: Robust and ubiquitous GFP expression in a single generation of chicken embryos using the avian retroviral vector, RCASBP. Differentiation; 2009 Jun;77(5):473-82

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Robust and ubiquitous GFP expression in a single generation of chicken embryos using the avian retroviral vector, RCASBP.
  • Functional genomics in avian models has lagged behind that of mammals, and the production of transgenic birds has proven to be challenging and time-consuming.
  • Here, we report a rapid method for the ubiquitous expression of GFP in chicken embryos in a single generation (G-0), using the avian retroviral vector, Replication-Competent Avian sarcoma-leukosis virus, with a Splice acceptor, Bryan RSV Pol (RCASBP).
  • The method can be used to generate avian cells with a stable genetic marker, or to induce global expression of a gene of choice.
  • This transgenic chicken model is of value in developmental studies, and for the isolation and study of avian primordial germ cells.

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  • (PMID = 19395148.001).
  • [ISSN] 1432-0436
  • [Journal-full-title] Differentiation; research in biological diversity
  • [ISO-abbreviation] Differentiation
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 147336-22-9 / Green Fluorescent Proteins
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32. Seidler B, Schmidt A, Mayr U, Nakhai H, Schmid RM, Schneider G, Saur D: A Cre-loxP-based mouse model for conditional somatic gene expression and knockdown in vivo by using avian retroviral vectors. Proc Natl Acad Sci U S A; 2008 Jul 22;105(29):10137-42
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] A Cre-loxP-based mouse model for conditional somatic gene expression and knockdown in vivo by using avian retroviral vectors.
  • Site- and time-specific somatic gene transfer by using the avian sarcoma-leukosis retrovirus RCAS (replication-competent avian sarcoma-leukosis virus long terminal repeat with splice acceptor) has been shown to be a powerful tool to analyze gene function in vivo.
  • RCAS retroviruses that express the avian subgroup A envelope transduce only mammalian cells genetically engineered to express the avian retroviral receptor, tumor virus A (TVA).
  • [MeSH-major] Alpharetrovirus / genetics. Avian Proteins / genetics. Carcinoma, Pancreatic Ductal / genetics. Genetic Vectors. Pancreatic Neoplasms / genetics. Receptors, Virus / genetics

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  • (PMID = 18621715.001).
  • [ISSN] 1091-6490
  • [Journal-full-title] Proceedings of the National Academy of Sciences of the United States of America
  • [ISO-abbreviation] Proc. Natl. Acad. Sci. U.S.A.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Avian Proteins; 0 / RNA, Small Interfering; 0 / Receptors, Virus; 0 / Tva receptor
  • [Other-IDs] NLM/ PMC2481330
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33. Chang KW, Barsov EV, Ferris AL, Hughes SH: Mutations of a residue within the polyproline-rich region of Env alter the replication rate and level of cytopathic effects in chimeric avian retroviral vectors. J Virol; 2005 Aug;79(16):10258-67
NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Mutations of a residue within the polyproline-rich region of Env alter the replication rate and level of cytopathic effects in chimeric avian retroviral vectors.
  • Previous attempts to extend the host range of the avian sarcoma/leukosis virus (ASLV)-based RCASBP vectors produced two viral vectors, RCASBP M2C (4070A) and RCASBP M2C (797-8), which replicate using the amphotropic murine leukemia virus 4070A Env protein (2).
  • Both viruses were adapted to replicate efficiently in the avian cell line DF-1, but RCASBP M2C (4070A) caused extensive cytopathic effects (CPE) in DF-1 cells whereas RCASBP M2C (797-8) induced low levels of CPE.
  • [MeSH-major] Cytopathogenic Effect, Viral. Gene Products, env / chemistry. Orthoreovirus, Avian / physiology. Virus Replication

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  • (PMID = 16051819.001).
  • [ISSN] 0022-538X
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / DNA, Viral; 0 / Gene Products, env; 0 / Peptides; 25191-13-3 / polyproline; F64QU97QCR / Dantrolene; SY7Q814VUP / Calcium
  • [Other-IDs] NLM/ PMC1182669
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34. Amberg SM, Netter RC, Simmons G, Bates P: Expanded tropism and altered activation of a retroviral glycoprotein resistant to an entry inhibitor peptide. J Virol; 2006 Jan;80(1):353-9

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Resistance to an HR2-based peptide inhibitor of a model retrovirus, subgroup A of the Avian Sarcoma and Leukosis Virus genus (ASLV-A), was studied by examining mutants derived by viral passage in the presence of inhibitor.

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  • (PMID = 16352560.001).
  • [ISSN] 0022-538X
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / R01-CA76256; United States / NIAID NIH HHS / AI / F32-AI050341; United States / NIAID NIH HHS / AI / R01 AI043455; United States / NIAID NIH HHS / AI / F32 AI050341; United States / NIGMS NIH HHS / GM / T32 GM007229; United States / NIAID NIH HHS / AI / R01-AI43455; United States / NCI NIH HHS / CA / R01 CA076256; United States / NIGMS NIH HHS / GM / T32-GM07229
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Peptides; 0 / Viral Envelope Proteins; 0 / Viral Fusion Proteins
  • [Other-IDs] NLM/ PMC1317511
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35. Yang X, Lipchina I, Lifton M, Wang L, Sodroski J: Antibody binding in proximity to the receptor/glycoprotein complex leads to a basal level of virus neutralization. J Virol; 2007 Aug;81(16):8809-13
COS Scholar Universe. author profiles.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Here, we demonstrate that retroviral infection mediated by the avian sarcoma-leukosis virus (ASLV-A) envelope glycoproteins can be neutralized by an antibody directed against a functionally unimportant component of a chimeric receptor protein.

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  • (PMID = 17537847.001).
  • [ISSN] 0022-538X
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NIAID NIH HHS / AI / R37 AI024755; United States / NIAID NIH HHS / AI / U01 AI067854; United States / NIAID NIH HHS / AI / AI 073133; United States / NIAID NIH HHS / AI / R21 AI064009; United States / NIAID NIH HHS / AI / R01 AI039420; United States / NIAID NIH HHS / AI / AI 064009; United States / NIAID NIH HHS / AI / AI 39420; United States / NIAID NIH HHS / AI / P30 AI042848; United States / NIAID NIH HHS / AI / AI 42848; United States / NIAID NIH HHS / AI / U19 AI067854; United States / NIAID NIH HHS / AI / R01 AI040895; United States / NIAID NIH HHS / AI / AI 40895; United States / NIAID NIH HHS / AI / AI 24755; United States / NIAID NIH HHS / AI / R01 AI073133; United States / NIAID NIH HHS / AI / AI 67854
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antibodies, Monoclonal; 0 / Antibodies, Viral; 0 / CCR5 Receptor Antagonists; 0 / Glycoproteins; 0 / Ligands; 0 / Receptors, Virus; 0 / Viral Envelope Proteins
  • [Other-IDs] NLM/ PMC1951382
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36. Pincetic A, Kuang Z, Seo EJ, Leis J: The interferon-induced gene ISG15 blocks retrovirus release from cells late in the budding process. J Virol; 2010 May;84(9):4725-36
NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The budding of avian sarcoma leukosis virus and HIV-1 Gag virus-like particles containing L-domain mutations can be rescued by fusion to ESCRT proteins, which cause entry into the budding pathway beyond these early steps.
  • We now demonstrate that the Vps4 protein does not associate with the avian sarcoma leukosis virus or the HIV-1 budding complexes when ISG15 is expressed.

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  • (PMID = 20164219.001).
  • [ISSN] 1098-5514
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NIAID NIH HHS / AI / R21 AI079025; United States / NIAID NIH HHS / AI / T32 AI060523; United States / NIAID NIH HHS / AI / AI079025
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / CHMP5 protein, human; 0 / Cytokines; 0 / Endosomal Sorting Complexes Required for Transport; 0 / Ubiquitins; 0 / VTA1 protein, human; 60267-61-0 / ISG15 protein, human; 9008-11-1 / Interferons; EC 3.6.1.- / Adenosine Triphosphatases; EC 3.6.1.- / VPS4A protein, human; EC 3.6.1.- / Vacuolar Proton-Translocating ATPases
  • [Other-IDs] NLM/ PMC2863725
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37. Côté M, Zheng YM, Liu SL: Receptor binding and low pH coactivate oncogenic retrovirus envelope-mediated fusion. J Virol; 2009 Nov;83(22):11447-55
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  • Fusion of enveloped viruses with host cells is triggered by either receptor binding or low pH but rarely requires both except for avian sarcoma leukosis virus (ASLV).

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  • (PMID = 19726505.001).
  • [ISSN] 1098-5514
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] Canada / Canadian Institutes of Health Research / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Receptors, Virus; 0 / Viral Fusion Proteins
  • [Other-IDs] NLM/ PMC2772678
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38. Melikyan GB, Barnard RJ, Abrahamyan LG, Mothes W, Young JA: Imaging individual retroviral fusion events: from hemifusion to pore formation and growth. Proc Natl Acad Sci U S A; 2005 Jun 14;102(24):8728-33
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  • To elucidate the mechanism of virus entry, we have imaged fusion between single virions bearing avian sarcoma and leukosis virus (ASLV) envelope glycoprotein (Env) and the cell membrane.

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  • (PMID = 15937118.001).
  • [ISSN] 0027-8424
  • [Journal-full-title] Proceedings of the National Academy of Sciences of the United States of America
  • [ISO-abbreviation] Proc. Natl. Acad. Sci. U.S.A.
  • [Language] ENG
  • [Grant] United States / NIAID NIH HHS / AI / R01 AI053668; United States / NCI NIH HHS / CA / R01 CA098727; United States / NIAID NIH HHS / AI / AI053668; United States / NCI NIH HHS / CA / CA098727
  • [Publication-type] Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Fluorescent Dyes; 0 / Viral Envelope Proteins; 0 / Viral Fusion Proteins
  • [Other-IDs] NLM/ PMC1150829
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39. Wu X, Li Y, Crise B, Burgess SM, Munroe DJ: Weak palindromic consensus sequences are a common feature found at the integration target sites of many retroviruses. J Virol; 2005 Apr;79(8):5211-4
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  • We carefully examined a large number of integration target site sequences from several retroviruses, including human immunodeficiency virus type 1, simian immunodeficiency virus, murine leukemia virus, and avian sarcoma-leukosis virus, and found that a statistical palindromic consensus, centered on the virus-specific duplicated target site sequence, was a common feature at integration target sites for these retroviruses.

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  • (PMID = 15795304.001).
  • [ISSN] 0022-538X
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / N01CO12400; United States / NCI NIH HHS / CO / N01-CO-12400
  • [Publication-type] Journal Article; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / DNA, Viral
  • [Other-IDs] NLM/ PMC1069554
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40. Yang X, Kurteva S, Ren X, Lee S, Sodroski J: Stoichiometry of envelope glycoprotein trimers in the entry of human immunodeficiency virus type 1. J Virol; 2005 Oct;79(19):12132-47
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  • A similar approach was applied to investigate the entry stoichiometry of envelope glycoproteins from amphotropic murine leukemia virus (A-MLV), avian sarcoma/leukosis virus type A (ASLV-A), and influenza A virus.

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  • (PMID = 16160141.001).
  • [ISSN] 0022-538X
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NIAID NIH HHS / AI / R37 AI024755; United States / NIAID NIH HHS / AI / AI31783; United States / NIAID NIH HHS / AI / R01 AI031783; United States / NIAID NIH HHS / AI / U01 AI046725; United States / NIAID NIH HHS / AI / AI46725; United States / NIAID NIH HHS / AI / AI24755
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Viral Envelope Proteins
  • [Other-IDs] NLM/ PMC1211524
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41. Contreras-Alcantara S, Godby JA, Delos SE: The single ligand-binding repeat of Tva, a low density lipoprotein receptor-related protein, contains two ligand-binding surfaces. J Biol Chem; 2006 Aug 11;281(32):22827-38
SciCrunch. OMIA - Online Mendelian Inheritance in Animals: Data: Gene Expression .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The receptor for avian sarcoma/leukosis virus subtype A (ASLV-A), Tva, is the simplest member of the low density lipoprotein receptor family containing a single ligand-binding repeat (LBR).
  • [MeSH-major] Avian Proteins / chemistry. Avian Proteins / physiology. Receptors, Virus / chemistry. Receptors, Virus / physiology

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  • (PMID = 16769730.001).
  • [ISSN] 0021-9258
  • [Journal-full-title] The Journal of biological chemistry
  • [ISO-abbreviation] J. Biol. Chem.
  • [Language] eng
  • [Grant] United States / NIAID NIH HHS / AI / AI22470
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Avian Proteins; 0 / Ligands; 0 / Receptors, Virus; 0 / Tva receptor; EC 3.2.1.- / Glycoside Hydrolases; SY7Q814VUP / Calcium
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42. Kang Y, Moressi CJ, Scheetz TE, Xie L, Tran DT, Casavant TL, Ak P, Benham CJ, Davidson BL, McCray PB Jr: Integration site choice of a feline immunodeficiency virus vector. J Virol; 2006 Sep;80(17):8820-3
The Lens. Cited by Patents in .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • FIV integration preferences are more similar to those of primate lentiviruses and distinct from those of Moloney murine leukemia virus, avian sarcoma leukosis virus, and foamy virus.

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  • (PMID = 16912328.001).
  • [ISSN] 0022-538X
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NHLBI NIH HHS / HL / R01 HL079023; United States / NHLBI NIH HHS / HL / HL-51670; United States / NHLBI NIH HHS / HL / P01 HL051670; United States / NHLBI NIH HHS / HL / R01 HL075363; United States / NIGMS NIH HHS / GM / R01 GM068903; United States / NHLBI NIH HHS / HL / HL-75363; United States / NHLBI NIH HHS / HL / HL-79023
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Proteins
  • [Other-IDs] NLM/ PMC1563849
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43. McNally MM, Wahlin KJ, Canto-Soler MV: Endogenous expression of ASLV viral proteins in specific pathogen free chicken embryos: relevance for the developmental biology research field. BMC Dev Biol; 2010 Oct 18;10:106
SciCrunch. The Antibody Registry: Reagent: Antibodies .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • BACKGROUND: The use of Specific Pathogen Free (SPF) eggs in combination with RCAS retrovirus, a member of the Avian Sarcoma-Leukosis Virus (ASLV) family, is of standard practice to study gene function and development.

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  • (PMID = 20955591.001).
  • [ISSN] 1471-213X
  • [Journal-full-title] BMC developmental biology
  • [ISO-abbreviation] BMC Dev. Biol.
  • [Language] ENG
  • [Grant] United States / NEI NIH HHS / EY / R01 EY004859; United States / NEI NIH HHS / EY / EY1765; United States / PHS HHS / / EYO4859
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Viral Proteins
  • [Other-IDs] NLM/ PMC2973938
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44. Torne-Celer C, Moreau K, Faure C, Chebloune Y, Verdier G, Ronfort C: A novel self-deleting retroviral vector carrying an additional sequence recognized by the viral integrase (IN). Virus Res; 2008 Jul;135(1):72-82

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • We have developed a self-deleting Avian Leukosis and Sarcoma Viruses (ALSVs)-based retroviral vector carrying an additional copy of the att sequence, between neo and puro genes.

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  • (PMID = 18420298.001).
  • [ISSN] 0168-1702
  • [Journal-full-title] Virus research
  • [ISO-abbreviation] Virus Res.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / RNA, Viral; 0 / Viral Proteins; EC 2.7.7.- / Integrases
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45. Klopocki E, Hennig BP, Dathe K, Koll R, de Ravel T, Baten E, Blom E, Gillerot Y, Weigel JF, Krüger G, Hiort O, Seemann P, Mundlos S: Deletion and point mutations of PTHLH cause brachydactyly type E. Am J Hum Genet; 2010 Mar 12;86(3):434-9
SciCrunch. Clinical Genomic Database: Data: Gene Annotation .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The missense mutation L60P was tested in chicken micromass culture with the replication-competent avian sarcoma leukosis virus retroviral expression system and was shown to result in a loss of function.

  • Genetic Alliance. consumer health - Brachydactyly Type E.
  • Genetic Alliance. consumer health - Brachydactyly.
  • KOMP Repository. gene/protein/disease-specific - KOMP Repository (subscription/membership/fee required).
  • Mouse Genome Informatics (MGI). Mouse Genome Informatics (MGI) .
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
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  • [Copyright] Copyright 2010 The American Society of Human Genetics. Published by Elsevier Inc. All rights reserved.
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  • (PMID = 20170896.001).
  • [ISSN] 1537-6605
  • [Journal-full-title] American journal of human genetics
  • [ISO-abbreviation] Am. J. Hum. Genet.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Codon, Nonsense; 0 / PTHLH protein, human; 0 / Parathyroid Hormone-Related Protein; 0 / Recombinant Proteins
  • [Other-IDs] NLM/ PMC2833367
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46. Bruce JW, Hierl M, Young JA, Ahlquist P: Cellular transcription factor ZASC1 regulates murine leukemia virus transcription. J Virol; 2010 Aug;84(15):7473-83
Gene Ontology. gene/protein/disease-specific - Gene Ontology annotations from this paper .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The defect in this cell line was specific for the MLV long terminal repeat (LTR) promoter, as normal levels of reporter gene expression were obtained from both an internal cytomegalovirus (CMV) promoter contained within an LTR-defective MLV vector and LTR expression from an avian sarcoma and leukosis virus (ASLV) vector.

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  • (PMID = 20484494.001).
  • [ISSN] 1098-5514
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NIAID NIH HHS / AI / AI072645-05; United States / NIAID NIH HHS / AI / AI72645; United States / NIAID NIH HHS / AI / R01 AI072645; United States / NIAID NIH HHS / AI / R01 AI072645-05; United States / Howard Hughes Medical Institute / / ; United States / NCI NIH HHS / CA / P01 CA022443; United States / NCI NIH HHS / CA / CA22443
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Transcription Factors
  • [Other-IDs] NLM/ PMC2897642
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47. Markosyan RM, Kielian M, Cohen FS: Fusion induced by a class II viral fusion protein, semliki forest virus E1, is dependent on the voltage of the target cell. J Virol; 2007 Oct;81(20):11218-25

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  • In contrast, fusion induced by the class I fusion proteins of human immunodeficiency virus, avian sarcoma leukosis virus, and influenza virus was independent of the voltage polarity across the target cell.

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  • (PMID = 17686870.001).
  • [ISSN] 0022-538X
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NIGMS NIH HHS / GM / R01 GM027367; United States / NIGMS NIH HHS / GM / R01 GM057454; United States / NIGMS NIH HHS / GM / GM057454; United States / NIGMS NIH HHS / GM / R01 GM27367
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Viral Envelope Proteins; 0 / Viral Fusion Proteins
  • [Other-IDs] NLM/ PMC2045574
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48. Faschinger A, Rouault F, Sollner J, Lukas A, Salmons B, Günzburg WH, Indik S: Mouse mammary tumor virus integration site selection in human and mouse genomes. J Virol; 2008 Feb;82(3):1360-7
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  • Avian sarcoma-leukosis virus (ASLV) and human T-cell leukemia virus (HTLV), forming the third group, show little preference for any genomic feature.

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  • [ISSN] 1098-5514
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] eng
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  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC2224419
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49. Derse D, Crise B, Li Y, Princler G, Lum N, Stewart C, McGrath CF, Hughes SH, Munroe DJ, Wu X: Human T-cell leukemia virus type 1 integration target sites in the human genome: comparison with those of other retroviruses. J Virol; 2007 Jun;81(12):6731-41
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  • On the other hand, integration of avian sarcoma-leukosis virus (ASLV) shows little preference either for genes, transcription start sites, or CpG islands.

  • Genetic Alliance. consumer health - Human T-cell leukemia virus type 1.
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  • (PMID = 17409138.001).
  • [ISSN] 0022-538X
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / N01CO12400; United States / NCI NIH HHS / CO / N01-CO-12400
  • [Publication-type] Comparative Study; Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC1900082
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50. Van Hoeven NS, Miller AD: Use of different but overlapping determinants in a retrovirus receptor accounts for non-reciprocal interference between xenotropic and polytropic murine leukemia viruses. Retrovirology; 2005 Dec 15;2:76
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • In the case of avian sarcoma and leukosis virus types B, D, and E, the two receptors are distinct protein products of a single gene.

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  • (PMID = 16354307.001).
  • [ISSN] 1742-4690
  • [Journal-full-title] Retrovirology
  • [ISO-abbreviation] Retrovirology
  • [Language] ENG
  • [Grant] United States / NIDDK NIH HHS / DK / P30 DK047754; United States / NHLBI NIH HHS / HL / HL36444; United States / NHLBI NIH HHS / HL / P01 HL036444; United States / NHLBI NIH HHS / HL / HL54881; United States / NIDDK NIH HHS / DK / DK47754; United States / NHLBI NIH HHS / HL / P50 HL054881
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Gene Products, env; 0 / Receptors, G-Protein-Coupled; 0 / Receptors, Virus; 0 / Recombinant Fusion Proteins; 0 / xenotropic and polytropic retrovirus receptor
  • [Other-IDs] NLM/ PMC1325250
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51. Bruce JW, Bradley KA, Ahlquist P, Young JA: Isolation of cell lines that show novel, murine leukemia virus-specific blocks to early steps of retroviral replication. J Virol; 2005 Oct;79(20):12969-78
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  • When engineered to express the TVA receptor for subgroup A avian sarcoma and leukosis virus (ASLV-A), the five cell lines were resistant to infection with a MLV vector pseudotyped with the ASLV-A envelope protein but were fully susceptible to infection with an ASLV-A vector.

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  • (PMID = 16188999.001).
  • [ISSN] 0022-538X
  • [Journal-full-title] Journal of virology
  • [ISO-abbreviation] J. Virol.
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / CA70810; United States / NCI NIH HHS / CA / T32 CA009075; United States / NCI NIH HHS / CA / P01 CA022443; United States / NCI NIH HHS / CA / R01 CA070810; United States / NCI NIH HHS / CA / CA22443
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC1235846
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52. Zavala G, Lucio-Martinez B, Cheng S, Barbosa T: Sarcomas and myelocytomas induced by a retrovirus related to myeloblastosis-associated virus type 1 in White Leghorn egg layer chickens. Avian Dis; 2006 Jun;50(2):201-8
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  • There was no microscopic evidence of Marek's disease or lymphoid leukosis.
  • Avian myeloblastosis-associated virus type 1 (MAV-1) was isolated from tumors, plasma, and serum.
  • Upon initial virus neutralization, the viruses isolated seemed at least partially related antigenically to avian leukosis virus (ALV) subgroups A and B but not to subgroup J (ALV-J).
  • [MeSH-major] Chickens / virology. Retroviridae / classification. Retroviridae / isolation & purification. Retroviridae Infections / veterinary. Sarcoma / veterinary. Tumor Virus Infections / veterinary

  • MedlinePlus Health Information. consumer health - Soft Tissue Sarcoma.
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  • (PMID = 16863068.001).
  • [ISSN] 0005-2086
  • [Journal-full-title] Avian diseases
  • [ISO-abbreviation] Avian Dis.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antibodies, Viral
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