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1. Morikawa Y, Dai YS, Hao J, Bonin C, Hwang S, Cserjesi P: The basic helix-loop-helix factor Hand 2 regulates autonomic nervous system development. Dev Dyn; 2005 Nov;234(3):613-21
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  • [Title] The basic helix-loop-helix factor Hand 2 regulates autonomic nervous system development.
  • Mammalian autonomic nervous system (ANS) development requires the combinatorial action of a number of transcription factors, which include Mash 1, Phox 2b, and GATA 3.
  • Here we show that the bHLH transcription factor, Hand 2 (dHAND), is expressed concurrently with Mash 1 during sympathetic nervous system (SNS) development and that the expression of Hand 2 is not dependent on Mash 1.
  • We propose that Hand 2 works in parallel with other members of the transcriptional network to regulate ANS developmental but can ectopically activate the program by a cross-regulatory mechanism that includes the activation of Mash 1.
  • We show that this function is dependent on its interaction with the histone acetyltransferase p300/CBP, indicating that Hand 2 functions to promote ANS development as part of a larger transcriptional complex.

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  • [Copyright] Developmental Dynamics 234:602-612, 2005. (c) 2005 Wiley-Liss, Inc.
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  • (PMID = 16145670.001).
  • [ISSN] 1058-8388
  • [Journal-full-title] Developmental dynamics : an official publication of the American Association of Anatomists
  • [ISO-abbreviation] Dev. Dyn.
  • [Language] ENG
  • [Grant] United States / NINDS NIH HHS / NS / 2R01 NS015547-22; United States / NINDS NIH HHS / NS / NS015547-27; United States / NINDS NIH HHS / NS / R01 NS015547; United States / NINDS NIH HHS / NS / T32 NS007062; United States / NINDS NIH HHS / NS / R01 NS015547-25; United States / NINDS NIH HHS / NS / 5T32 NS07062; United States / NINDS NIH HHS / NS / NS015547-25; United States / NINDS NIH HHS / NS / R01 NS015547-27
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Ascl1 protein, mouse; 0 / Basic Helix-Loop-Helix Transcription Factors; 0 / Hand2 protein, mouse; EC 2.3.1.48 / p300-CBP Transcription Factors
  • [Other-IDs] NLM/ NIHMS75813; NLM/ PMC2653092
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2. Wang Z, Low PA, Jordan J, Freeman R, Gibbons CH, Schroeder C, Sandroni P, Vernino S: Autoimmune autonomic ganglionopathy: IgG effects on ganglionic acetylcholine receptor current. Neurology; 2007 May 29;68(22):1917-21
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  • [Title] Autoimmune autonomic ganglionopathy: IgG effects on ganglionic acetylcholine receptor current.
  • BACKGROUND: Autoimmune autonomic ganglionopathy (AAG) is an acquired immune-mediated form of diffuse autonomic failure.
  • Many patients have serum antibodies that bind to the ganglionic acetylcholine receptors (AChRs) that mediate fast synaptic transmission in autonomic ganglia.
  • Our observations demonstrate that antibodies in patients with autoimmune autonomic ganglionopathy (AAG) cause physiologic changes in ganglionic AChR function and confirm that AAG is an antibody-mediated disorder.

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  • (PMID = 17536048.001).
  • [ISSN] 1526-632X
  • [Journal-full-title] Neurology
  • [ISO-abbreviation] Neurology
  • [Language] ENG
  • [Grant] United States / NINDS NIH HHS / NS / P50 NS032352; United States / NINDS NIH HHS / NS / NS049899-03; United States / NINDS NIH HHS / NS / NS048077-03; United States / NINDS NIH HHS / NS / F32 NS049899; United States / NINDS NIH HHS / NS / R01 NS048077-03; United States / NIDDK NIH HHS / DK / P01 DK068055; United States / NINDS NIH HHS / NS / F32 NS049899-03; United States / NINDS NIH HHS / NS / N01NS32352; United States / NINDS NIH HHS / NS / NS48077; United States / NINDS NIH HHS / NS / P50 NS032352-110004; United States / NINDS NIH HHS / NS / R01 NS048077-04; United States / NIDDK NIH HHS / DK / DK68055; United States / NINDS NIH HHS / NS / NS048077-04; United States / NINDS NIH HHS / NS / NS32352; United States / NINDS NIH HHS / NS / NS032352-110004; United States / NINDS NIH HHS / NS / R01 NS048077
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Autoantibodies; 0 / Immunoglobulin G; 0 / Receptors, Cholinergic
  • [Other-IDs] NLM/ NIHMS35218; NLM/ PMC2615674
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3. Boettger MK, Weber K, Grossmann D, Gajda M, Bauer R, Bär KJ, Schulz S, Voss A, Geis C, Bräuer R, Schaible HG: Spinal tumor necrosis factor alpha neutralization reduces peripheral inflammation and hyperalgesia and suppresses autonomic responses in experimental arthritis: a role for spinal tumor necrosis factor alpha during induction and maintenance of peripheral inflammation. Arthritis Rheum; 2010 May;62(5):1308-18
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  • [Title] Spinal tumor necrosis factor alpha neutralization reduces peripheral inflammation and hyperalgesia and suppresses autonomic responses in experimental arthritis: a role for spinal tumor necrosis factor alpha during induction and maintenance of peripheral inflammation.
  • This study was undertaken to test the hypothesis that spinal actions of the proinflammatory cytokine tumor necrosis factor alpha (TNFalpha) add significantly to both hyperalgesia and maintenance of peripheral inflammation.
  • Since a role for autonomic control of inflammation was proposed, measures from heart rate time series were obtained in the acute phase.
  • Parameters indicating autonomic modulation showed a shift toward a sympathetically dominated state in vehicle-treated animals, which was prevented by intrathecal etanercept.
  • [MeSH-major] Antirheumatic Agents / pharmacology. Arthritis, Experimental. Hyperalgesia. Immunoglobulin G / pharmacology. Tumor Necrosis Factor-alpha
  • [MeSH-minor] Acute Disease. Animals. Autonomic Nervous System / drug effects. Autonomic Nervous System / immunology. Behavior, Animal / drug effects. Etanercept. Female. Heart Rate / physiology. Infusion Pumps, Implantable. Injections, Spinal. Knee Joint / immunology. Knee Joint / metabolism. Locomotion / drug effects. Nonlinear Dynamics. Rats. Rats, Inbred Lew. Receptors, Tumor Necrosis Factor. Spinal Cord / immunology. Spinal Cord / metabolism

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  • (PMID = 20213802.001).
  • [ISSN] 1529-0131
  • [Journal-full-title] Arthritis and rheumatism
  • [ISO-abbreviation] Arthritis Rheum.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antirheumatic Agents; 0 / Immunoglobulin G; 0 / Receptors, Tumor Necrosis Factor; 0 / Tumor Necrosis Factor-alpha; OP401G7OJC / Etanercept
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4. Bougnères P, Pantalone L, Linglart A, Rothenbühler A, Le Stunff C: Endocrine manifestations of the rapid-onset obesity with hypoventilation, hypothalamic, autonomic dysregulation, and neural tumor syndrome in childhood. J Clin Endocrinol Metab; 2008 Oct;93(10):3971-80
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  • [Title] Endocrine manifestations of the rapid-onset obesity with hypoventilation, hypothalamic, autonomic dysregulation, and neural tumor syndrome in childhood.
  • CONTEXT: Rapid-onset obesity with hypoventilation, hypothalamic, autonomic dysregulation, and neural tumor (ROHHADNET) is a newly described syndrome that can cause cardiorespiratory arrests and death.
  • [MeSH-major] Autonomic Nervous System Diseases / complications. Endocrine System / physiopathology. Hypothalamic Diseases / complications. Hypoventilation / complications. Nervous System Neoplasms / complications. Obesity / complications
  • [MeSH-minor] Age of Onset. Autonomic Nervous System / physiopathology. Child. Child, Preschool. Female. Humans. Hypothalamus / physiopathology. Infant. Male. Pituitary-Adrenal System / physiopathology

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  • (PMID = 18628522.001).
  • [ISSN] 0021-972X
  • [Journal-full-title] The Journal of clinical endocrinology and metabolism
  • [ISO-abbreviation] J. Clin. Endocrinol. Metab.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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5. Kaufman CL, Kaiser DR, Steinberger J, Kelly AS, Dengel DR: Relationships of cardiac autonomic function with metabolic abnormalities in childhood obesity. Obesity (Silver Spring); 2007 May;15(5):1164-71
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  • [Title] Relationships of cardiac autonomic function with metabolic abnormalities in childhood obesity.
  • OBJECTIVE: The objective was to examine cardiovascular autonomic (cANS) function and its potential relationships with leptin resistance, insulin resistance, oxidative stress, and inflammation in a pediatric sample with varying levels of obesity.
  • Standard time-domain and frequency-domain measures [high-frequency normalized units (HFnu; measure of parasympathetic nervous system activity) and low frequency:high-frequency ratio (LF:HF; overall sympathovagal balance)] of HRV were calculated.
  • Fasting blood samples were drawn for measurement of glucose, insulin, lipids, 8-isoprostane, leptin, soluble leptin-receptor (sOB-R), C-reactive protein (CRP), interleukin-6 (IL-6), and tumor necrosis factor-alpha (TNF-alpha).
  • [MeSH-major] Heart / physiology. Heart Conduction System / physiopathology. Heart Rate / physiology. Metabolic Diseases / physiopathology. Obesity / physiopathology
  • [MeSH-minor] Blood Glucose / analysis. C-Reactive Protein / metabolism. Child. Female. Heart Diseases / epidemiology. Humans. Insulin / blood. Interleukin-6 / blood. Leptin / blood. Male. Minnesota / epidemiology. Reference Values. Tumor Necrosis Factor-alpha / blood

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  • (PMID = 17495192.001).
  • [ISSN] 1930-7381
  • [Journal-full-title] Obesity (Silver Spring, Md.)
  • [ISO-abbreviation] Obesity (Silver Spring)
  • [Language] eng
  • [Grant] United States / NIDDK NIH HHS / DK / 1 P30 DK 50456-08; United States / NCRR NIH HHS / RR / M01-RR00400
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Blood Glucose; 0 / Insulin; 0 / Interleukin-6; 0 / Leptin; 0 / Tumor Necrosis Factor-alpha; 9007-41-4 / C-Reactive Protein
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6. Hermann GE, Rogers RC: TNFalpha: a trigger of autonomic dysfunction. Neuroscientist; 2008 Feb;14(1):53-67
MedlinePlus Health Information. consumer health - Autonomic Nervous System Disorders.

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  • [Title] TNFalpha: a trigger of autonomic dysfunction.
  • During disease, infection, or trauma, the cytokine tumor necrosis factor alpha (TNF alpha) causes fever, fatigue, malaise, allodynia, anorexia, gastric stasis associated with nausea, and emesis via interactions with the central nervous system.
  • The constitutive presence of TNF alpha receptors on these afferents and their ability to amplify afferent signals may explain how TNF alpha can completely disrupt autonomic control of the gut.

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  • (PMID = 17911224.001).
  • [ISSN] 1073-8584
  • [Journal-full-title] The Neuroscientist : a review journal bringing neurobiology, neurology and psychiatry
  • [ISO-abbreviation] Neuroscientist
  • [Language] ENG
  • [Grant] United States / NIDDK NIH HHS / DK / R01 DK056373; United States / NICHD NIH HHS / HD / HD47643; United States / NIDDK NIH HHS / DK / DK52142; United States / NICHD NIH HHS / HD / R21 HD047643; United States / NIDDK NIH HHS / DK / R01 DK052142; United States / NIDDK NIH HHS / DK / DK56373
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Receptors, Tumor Necrosis Factor; 0 / Tumor Necrosis Factor-alpha
  • [Number-of-references] 76
  • [Other-IDs] NLM/ NIHMS556569; NLM/ PMC3951357
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7. Leyva-Grado VH, Churchill L, Wu M, Williams TJ, Taishi P, Majde JA, Krueger JM: Influenza virus- and cytokine-immunoreactive cells in the murine olfactory and central autonomic nervous systems before and after illness onset. J Neuroimmunol; 2009 Jun 25;211(1-2):73-83
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  • [Title] Influenza virus- and cytokine-immunoreactive cells in the murine olfactory and central autonomic nervous systems before and after illness onset.
  • Interleukin 1beta- and tumor necrosis factor alpha-immunoreactivity co-localized with neuronal markers in olfactory and central autonomic systems, and the number of cytokine-immunoreactive neurons increased at the time of illness onset [15 h post-inoculation (PI)] but not before (10 h PI).

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  • (PMID = 19410300.001).
  • [ISSN] 1872-8421
  • [Journal-full-title] Journal of neuroimmunology
  • [ISO-abbreviation] J. Neuroimmunol.
  • [Language] ENG
  • [Grant] United States / NINDS NIH HHS / NS / R01 NS025378; United States / NINDS NIH HHS / NS / NS025378-22; United States / NINDS NIH HHS / NS / R01 NS031453-16; United States / NINDS NIH HHS / NS / NS031453-16; United States / NICHD NIH HHS / HD / R01 HD036520; United States / NICHD NIH HHS / HD / R01 HD036520-11; United States / NINDS NIH HHS / NS / NS31453; United States / NINDS NIH HHS / NS / R01 NS031453; United States / NICHD NIH HHS / HD / HD36520; United States / NINDS NIH HHS / NS / R01 NS025378-22; United States / NICHD NIH HHS / HD / HD036520-11; United States / NINDS NIH HHS / NS / NS25378
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Cytokines
  • [Other-IDs] NLM/ NIHMS108077; NLM/ PMC2696569
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8. Basile A, Kettenbach J, Mundo E, Natoli A, Caloggero S, Minciullo M, Garito A: Primitive mesenteric gastrointestinal stromal tumor with autonomic nerve/ganglionic differentiation presenting as a huge mass with small synchronous nodules. Eur Radiol; 2006 Feb;16(2):422-7
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  • [Title] Primitive mesenteric gastrointestinal stromal tumor with autonomic nerve/ganglionic differentiation presenting as a huge mass with small synchronous nodules.
  • We present a case of a primary mesenteric ectopic gastrointestinal stromal tumor.
  • The pathologic characteristics of this neoplasm, autonomic nerve/ganglionic differentiation and presentation as a huge mass with small synchronous nodules, have not been previously described in the literature.
  • [MeSH-major] Autonomic Nervous System / pathology. Cell Transformation, Neoplastic / pathology. Choristoma / radiography. Ganglia, Autonomic / pathology. Gastrointestinal Stromal Tumors / radiography. Image Enhancement. Image Processing, Computer-Assisted. Mesentery / radiography. Neoplasms, Multiple Primary / pathology. Omentum / radiography. Peritoneal Neoplasms / radiography. Tomography, X-Ray Computed
  • [MeSH-minor] Angiography. Biomarkers, Tumor / genetics. Embolization, Therapeutic. Follow-Up Studies. Humans. Male. Middle Aged. Neoplasm Recurrence, Local / blood supply. Neoplasm Recurrence, Local / pathology. Neoplasm Recurrence, Local / radiography. Neoplasm Recurrence, Local / surgery. Reoperation

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  • [ErratumIn] Eur Radiol. 2006 Feb;16(2):519. Kettenback, Joachim [corrected to Kettenbach, Joachim]
  • (PMID = 15726376.001).
  • [ISSN] 0938-7994
  • [Journal-full-title] European radiology
  • [ISO-abbreviation] Eur Radiol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / Biomarkers, Tumor
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9. Nada T, Nomura M, Koshiba K, Kawano T, Mikawa J, Ito S: Clinical study with azelnidipine in patients with essential hypertension. Antiarteriosclerotic and cardiac hypertrophy-inhibitory effects and influence on autonomic nervous activity. Arzneimittelforschung; 2007;57(11):698-704
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  • [Title] Clinical study with azelnidipine in patients with essential hypertension. Antiarteriosclerotic and cardiac hypertrophy-inhibitory effects and influence on autonomic nervous activity.
  • In this study, the antiarteriosclerotic and cardiac hypertrophy-inhibitory effects, and the autonomic nervous activity in essential hypertension of azelnidipine were investigated.
  • 1) Pulse wave velocity (PWV), carotid arterial intima media thickness (IMT), echocardiography, high sensitive C-reactive protein (hs-CRP), interleukin-6 (IL-6), tumor necrosis factor alpha (TNF-alpha), adiponectin, brain natriuretic peptide (BNP), and 8-isoprostane were measured after an initial treatment with azelnidipine.
  • These findings suggest that azelnidipine inhibits the enhancement of sympathetic nervous activity and the progression of arteriosclerosis through its antioxidative effects.
  • [MeSH-minor] 3-Iodobenzylguanidine. Adipokines / metabolism. Aged. Antioxidants / metabolism. Arteriosclerosis / complications. Arteriosclerosis / radionuclide imaging. Autonomic Nervous System / drug effects. Cardiomegaly / etiology. Cardiomegaly / radionuclide imaging. Carotid Arteries / radionuclide imaging. Catecholamines / blood. Cytokines / metabolism. Electrocardiography / drug effects. Female. Heart Rate / drug effects. Humans. Male. Middle Aged. Natriuretic Peptide, Brain / metabolism. Pulse. Radiopharmaceuticals

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  • (PMID = 18193691.001).
  • [ISSN] 0004-4172
  • [Journal-full-title] Arzneimittel-Forschung
  • [ISO-abbreviation] Arzneimittelforschung
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / Adipokines; 0 / Antihypertensive Agents; 0 / Antioxidants; 0 / Calcium Channel Blockers; 0 / Catecholamines; 0 / Cytokines; 0 / Dihydropyridines; 0 / Radiopharmaceuticals; 114471-18-0 / Natriuretic Peptide, Brain; 2517-04-6 / Azetidinecarboxylic Acid; 35MRW7B4AD / 3-Iodobenzylguanidine; PV23P19YUG / azelnidipine
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10. Gershon MD: Developmental determinants of the independence and complexity of the enteric nervous system. Trends Neurosci; 2010 Oct;33(10):446-56
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  • [Title] Developmental determinants of the independence and complexity of the enteric nervous system.
  • Enteric nervous system (ENS) development is relevant to Hirschsprung's disease (HSCR; congenital aganglionosis of the terminal bowel), which is still imperfectly treated.
  • [MeSH-major] Enteric Nervous System / pathology. Hirschsprung Disease / etiology. Hirschsprung Disease / pathology. Neural Crest / pathology. Neurons / metabolism
  • [MeSH-minor] Cell Differentiation / genetics. Cell Movement / genetics. Endothelin-3 / genetics. Extracellular Matrix / metabolism. Ganglia, Autonomic / growth & development. Ganglia, Autonomic / metabolism. Ganglia, Autonomic / pathology. Gastrointestinal Tract / innervation. Glial Cell Line-Derived Neurotrophic Factor / genetics. Glial Cell Line-Derived Neurotrophic Factor Receptors / genetics. Humans. Mutation. Nerve Growth Factors / genetics. Proto-Oncogene Proteins c-ret / genetics. Risk Factors. Tumor Suppressor Proteins / genetics

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  • [Copyright] Copyright © 2010 Elsevier Ltd. All rights reserved.
  • (PMID = 20633936.001).
  • [ISSN] 1878-108X
  • [Journal-full-title] Trends in neurosciences
  • [ISO-abbreviation] Trends Neurosci.
  • [Language] eng
  • [Grant] United States / NINDS NIH HHS / NS / NS12969; United States / NINDS NIH HHS / NS / NS15547
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Endothelin-3; 0 / GFRA1 protein, human; 0 / Glial Cell Line-Derived Neurotrophic Factor; 0 / Glial Cell Line-Derived Neurotrophic Factor Receptors; 0 / Nerve Growth Factors; 0 / Tumor Suppressor Proteins; 158651-98-0 / netrin-1; EC 2.7.10.1 / Proto-Oncogene Proteins c-ret; EC 2.7.10.1 / RET protein, human
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11. Wang SJ, Hung CW, Fuh JL, Lirng JF, Hwu CM: Cranial autonomic symptoms in patients with pituitary adenoma presenting with headaches. Acta Neurol Taiwan; 2009 Jun;18(2):104-12
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  • [Title] Cranial autonomic symptoms in patients with pituitary adenoma presenting with headaches.
  • Different types of symptomatic trigeminal autonomic cephalalgias (TACs) have been reported in patients with pituitary adenoma.
  • We investigated the significance of the presence of cranial autonomic symptoms (CAS) in patients with pituitary adenoma presenting with headaches.
  • CAS were ascertained if one or more autonomic symptoms defined for the diagnosis of TACs in the International Classification of Headache Disorders, 2nd edition (ICHD-2) was identified.
  • In the group with CAS (CAS+), the sides of the tumor were significantly concordant with the sides of headaches (kappa=0.58, p<0.001) and those of CAS (kappa=0.67, p<0.001).
  • [MeSH-major] Adenoma / complications. Autonomic Nervous System Diseases / complications. Cranial Nerve Diseases / complications. Headache / etiology. Pituitary Neoplasms / complications
  • [MeSH-minor] Acromegaly / etiology. Adult. Aged. Female. Humans. Magnetic Resonance Imaging. Male. Middle Aged. Trigeminal Autonomic Cephalalgias / etiology

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  • (PMID = 19673362.001).
  • [ISSN] 1028-768X
  • [Journal-full-title] Acta neurologica Taiwanica
  • [ISO-abbreviation] Acta Neurol Taiwan
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] China (Republic : 1949- )
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12. Barone L, Colicchio G, Policicchio D, Di Clemente F, Di Monaco A, Meglio M, Lanza GA, Crea F: Effect of vagal nerve stimulation on systemic inflammation and cardiac autonomic function in patients with refractory epilepsy. Neuroimmunomodulation; 2007;14(6):331-6
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  • [Title] Effect of vagal nerve stimulation on systemic inflammation and cardiac autonomic function in patients with refractory epilepsy.
  • Tumor necrosis factor-alpha, interleukin-6 and C-reactive protein serum levels were measured, as markers of inflammation, at the same times.
  • CONCLUSIONS: Our data in epileptic patients without cardiovascular disease failed to show a significant effect of left VNS on cardiac autonomic function and on systemic inflammation at short-term follow-up.
  • [MeSH-major] Autonomic Nervous System / physiology. Electric Stimulation Therapy. Epilepsy / therapy. Heart / physiology. Inflammation / blood. Vagus Nerve / physiology
  • [MeSH-minor] Adolescent. Adult. Aged. C-Reactive Protein / analysis. Child. Electrocardiography, Ambulatory. Female. Heart Rate / physiology. Humans. Interleukin-6 / blood. Male. Neuroimmunomodulation / physiology. Tumor Necrosis Factor-alpha / blood

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  • [Copyright] (c) 2008 S. Karger AG, Basel.
  • (PMID = 18418007.001).
  • [ISSN] 1423-0216
  • [Journal-full-title] Neuroimmunomodulation
  • [ISO-abbreviation] Neuroimmunomodulation
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 0 / Interleukin-6; 0 / Tumor Necrosis Factor-alpha; 9007-41-4 / C-Reactive Protein
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13. Hermann GE, Van Meter MJ, Rogers RC: CXCR4 receptors in the dorsal medulla: implications for autonomic dysfunction. Eur J Neurosci; 2008 Feb;27(4):855-64
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  • [Title] CXCR4 receptors in the dorsal medulla: implications for autonomic dysfunction.
  • Anorexia, nausea and failed autonomic regulation of gastrointestinal (GI) function cause morbidity and contribute to the mortality associated with these disease states.
  • Our previous work on the peripheral cytokine, tumor necrosis factor-alpha, demonstrated that similar morbidity factors involving GI dysfunction are attributable to agonist action on neural circuit elements of the dorsal vagal complex (DVC) of the hindbrain.

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  • (PMID = 18333961.001).
  • [ISSN] 1460-9568
  • [Journal-full-title] The European journal of neuroscience
  • [ISO-abbreviation] Eur. J. Neurosci.
  • [Language] ENG
  • [Grant] United States / NIDDK NIH HHS / DK / R01 DK056373; United States / NIDDK NIH HHS / DK / DK52142; United States / NICHD NIH HHS / HD / HD047643; United States / NICHD NIH HHS / HD / R21 HD047643; United States / NIDDK NIH HHS / DK / R01 DK052142; United States / NIDDK NIH HHS / DK / DK56373
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] France
  • [Chemical-registry-number] 0 / Chemokine CXCL12; 0 / Cxcr4 protein, rat; 0 / Proto-Oncogene Proteins c-fos; 0 / Receptors, CXCR4
  • [Other-IDs] NLM/ NIHMS556562; NLM/ PMC3951345
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14. Pomara C, Neri M, Riezzo I, Turillazzi E, Fineschi V: Autonomic nervous system instability, tetanic necrosis of the heart and myocardial TNFalpha expression in a tetanus fatal case. Int J Cardiol; 2009 Aug 21;136(3):e54-7
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  • [Title] Autonomic nervous system instability, tetanic necrosis of the heart and myocardial TNFalpha expression in a tetanus fatal case.
  • [MeSH-major] Autonomic Nervous System Diseases / etiology. Heart Diseases / etiology. Heart Diseases / pathology. Tetanus / complications. Tumor Necrosis Factor-alpha / metabolism

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  • (PMID = 18649956.001).
  • [ISSN] 1874-1754
  • [Journal-full-title] International journal of cardiology
  • [ISO-abbreviation] Int. J. Cardiol.
  • [Language] eng
  • [Publication-type] Case Reports; Letter
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Tumor Necrosis Factor-alpha
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15. Irwin MR, Olmos L, Wang M, Valladares EM, Motivala SJ, Fong T, Newton T, Butch A, Olmstead R, Cole SW: Cocaine dependence and acute cocaine induce decreases of monocyte proinflammatory cytokine expression across the diurnal period: autonomic mechanisms. J Pharmacol Exp Ther; 2007 Feb;320(2):507-15
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Cocaine dependence and acute cocaine induce decreases of monocyte proinflammatory cytokine expression across the diurnal period: autonomic mechanisms.
  • The innate immune system triggers effector pathways to combat microbial pathogens through expression of tumor necrosis factor-alpha (TNF-alpha) and interleukin-6 (IL-6).
  • Cocaine alters autonomic activity and induces protracted decreases in innate immune mechanisms.
  • [MeSH-major] Autonomic Nervous System / drug effects. Cocaine / pharmacology. Cocaine-Related Disorders / immunology. Cytokines / biosynthesis. Immunity, Innate / drug effects. Monocytes / immunology
  • [MeSH-minor] Adult. HIV Infections / immunology. Humans. Interleukin-6 / biosynthesis. Lipopolysaccharides / pharmacology. Middle Aged. Receptors, Tumor Necrosis Factor, Type II / blood. Time Factors. Tumor Necrosis Factor-alpha / biosynthesis

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  • (PMID = 17068203.001).
  • [ISSN] 0022-3565
  • [Journal-full-title] The Journal of pharmacology and experimental therapeutics
  • [ISO-abbreviation] J. Pharmacol. Exp. Ther.
  • [Language] eng
  • [Grant] United States / NIAAA NIH HHS / AA / AA13239; United States / NIDA NIH HHS / DA / DA16541; United States / NCRR NIH HHS / RR / M01 RR00827; United States / NIMH NIH HHS / MH / T32-MH19925
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Cytokines; 0 / Interleukin-6; 0 / Lipopolysaccharides; 0 / Receptors, Tumor Necrosis Factor, Type II; 0 / Tumor Necrosis Factor-alpha; I5Y540LHVR / Cocaine
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16. Liang JT, Lai HS, Lee PH, Chang KJ: Laparoscopic pelvic autonomic nerve-preserving surgery for sigmoid colon cancer. Ann Surg Oncol; 2008 Jun;15(6):1609-16

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Laparoscopic pelvic autonomic nerve-preserving surgery for sigmoid colon cancer.
  • BACKGROUND: To test the feasibility of laparoscopic approach in performing the simultaneous pelvic autonomic nerve preservation during standard anterior resection of sigmoid colon cancer.
  • RESULTS: A total of 112 patients (tumor, node, metastasis system stage I, n = 8; stage II, n = 54; stage III, n = 50; male, n = 58; female, n = 54; age [mean +/- standard deviation], 55.8 +/- 6.4 years) with good baseline genitourinary function were operated on with the intent of total preservation of pelvic autonomic nerves and curative resection of sigmoid colon cancer.
  • CONCLUSIONS: Under laparoscopy, we can clearly identify and preserve the pelvic autonomic nerves to retain genitourinary function in most patients undergoing oncologic resection of sigmoid colon cancer.
  • [MeSH-major] Adenocarcinoma / surgery. Autonomic Pathways / surgery. Colectomy. Sigmoid Neoplasms / surgery. Trauma, Nervous System / prevention & control

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  • (PMID = 18365285.001).
  • [ISSN] 1534-4681
  • [Journal-full-title] Annals of surgical oncology
  • [ISO-abbreviation] Ann. Surg. Oncol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC2373867
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17. Kessler W, Traeger T, Westerholt A, Neher F, Mikulcak M, Müller A, Maier S, Heidecke CD: The vagal nerve as a link between the nervous and immune system in the instance of polymicrobial sepsis. Langenbecks Arch Surg; 2006 Apr;391(2):83-7
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  • [Title] The vagal nerve as a link between the nervous and immune system in the instance of polymicrobial sepsis.
  • BACKGROUND: The role of the vagal nerve in the autonomic nervous system is widely well known.
  • Recently, an additional function was revealed serving as a connector between the nervous and immune system.
  • Through stimulation of the acetylcholine receptors located upon the macrophages, the "unspecific" immune system can be directly influenced.
  • CONCLUSION: The vagal nerve is therefore an important modulator of the immune system.

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  • (PMID = 16568325.001).
  • [ISSN] 1435-2443
  • [Journal-full-title] Langenbeck's archives of surgery
  • [ISO-abbreviation] Langenbecks Arch Surg
  • [Language] ENG
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / CCL2 protein, human; 0 / Chemokine CCL2; 0 / Interleukin-6; 0 / Tumor Necrosis Factor-alpha; 130068-27-8 / Interleukin-10
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18. Yun AJ, Bazar KA, Lee PY, Gerber A, Daniel SM: The smoking gun: many conditions associated with tobacco exposure may be attributable to paradoxical compensatory autonomic responses to nicotine. Med Hypotheses; 2005;64(6):1073-9
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  • [Title] The smoking gun: many conditions associated with tobacco exposure may be attributable to paradoxical compensatory autonomic responses to nicotine.
  • We hypothesize that the illnesses associated with smoking may be partly attributable to autonomic dysfunction, sympathetic bias, and T helper (Th)2 inflammation induced by a paradoxical compensatory response to intermittent nicotinic exposure.
  • Other conditions associated with tobacco exposure may also operate through similar autonomic and immune dysfunctions.
  • [MeSH-major] Autonomic Nervous System / physiopathology. Models, Biological. Nicotine / adverse effects. Smoking / adverse effects. Substance Withdrawal Syndrome / physiopathology. Tobacco, Smokeless / adverse effects
  • [MeSH-minor] Anxiety / chemically induced. Anxiety / physiopathology. Humans. Hypertension / chemically induced. Hypertension / physiopathology. Inflammation / chemically induced. Inflammation / physiopathology. Neoplasms / etiology. Receptors, Nicotinic / drug effects. Receptors, Nicotinic / physiology. Sleep Initiation and Maintenance Disorders / chemically induced. Sleep Initiation and Maintenance Disorders / physiopathology. Smoking Cessation. Sympathetic Nervous System / drug effects. Sympathetic Nervous System / physiopathology. Tachycardia / chemically induced. Tachycardia / physiopathology. Th2 Cells / immunology. Tumor Escape. Vagus Nerve / physiopathology

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  • (PMID = 15823687.001).
  • [ISSN] 0306-9877
  • [Journal-full-title] Medical hypotheses
  • [ISO-abbreviation] Med. Hypotheses
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Scotland
  • [Chemical-registry-number] 0 / Receptors, Nicotinic; 6M3C89ZY6R / Nicotine
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19. Liang JT, Lai HS, Lee PH: Laparoscopic pelvic autonomic nerve-preserving surgery for patients with lower rectal cancer after chemoradiation therapy. Ann Surg Oncol; 2007 Apr;14(4):1285-7
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Laparoscopic pelvic autonomic nerve-preserving surgery for patients with lower rectal cancer after chemoradiation therapy.
  • OBJECTIVE: This is a phase II study, the aim of which is to determine if a laparoscopic approach can be used in pelvic autonomic nerve-preserving surgery for patients with lower rectal cancer following chemoradiation therapy.
  • METHODS: Patients with T3 lower rectal cancer treated by preoperative chemoradiation were recruited and subjected to laparoscopic pelvic autonomic nerve-preserving surgery with total mesorectal excision and a sphincter-saving procedure.
  • Postoperatively, only patients successfully operated on by total pelvic autonomic nerve-preserving surgery were included in the statistical analysis of surgical outcomes.
  • The reason for these four parameters is because the influence of pelvic autonomic nerve damage on female sexual function has been ambiguous but would most likely result in impairment of vaginal lubrication and congestion of the genitals.
  • With respect to scrutiny of surgical specimens, the distal safety margin was adequate (mean: 2.4 cm; range: 1.2-5.6 cm), and the circumferential resection margins were free of tumor invasion (mean: 8.6 mm; range: 2-18 mm).
  • CONCLUSIONS: By the laparoscopic approach, total preservation of pelvic autonomic nerves without compromise of the radical extirpation of tumor is technically feasible in the vast majority of patients with lower rectal cancer who have undergone concurrent chemoradiation therapy, thus facilitating the retention of genitourinary function in a significant proportion of such patients.
  • [MeSH-major] Autonomic Nervous System / physiology. Laparoscopy. Neurosurgical Procedures. Pelvis / innervation. Postoperative Complications / prevention & control. Rectal Neoplasms / surgery
  • [MeSH-minor] Combined Modality Therapy. Female. Humans. Male. Neoplasm Staging. Treatment Outcome

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  • (PMID = 17235719.001).
  • [ISSN] 1068-9265
  • [Journal-full-title] Annals of surgical oncology
  • [ISO-abbreviation] Ann. Surg. Oncol.
  • [Language] eng
  • [Publication-type] Clinical Trial, Phase II; Journal Article
  • [Publication-country] United States
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20. Franzius C, Hermann K, Weckesser M, Kopka K, Juergens KU, Vormoor J, Schober O: Whole-body PET/CT with 11C-meta-hydroxyephedrine in tumors of the sympathetic nervous system: feasibility study and comparison with 123I-MIBG SPECT/CT. J Nucl Med; 2006 Oct;47(10):1635-42
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Whole-body PET/CT with 11C-meta-hydroxyephedrine in tumors of the sympathetic nervous system: feasibility study and comparison with 123I-MIBG SPECT/CT.
  • The 11C-labeled tracer meta-hydroxyephedrine (11C-HED) is a noradrenaline analog that was developed to visualize the sympathetic nervous system with PET.
  • Initial clinical studies show a rapid uptake of 11C-HED in localized tumors of this system.
  • The aim of this study was to evaluate the feasibility of whole-body 11C-HED PET/CT for examination of tumors of the sympathetic nervous system and to directly compare the results with 123I-labeled meta-iodobenzylguanidine (123I-MIBG) scintigraphy, including SPECT/CT.
  • In 5 patients, each having 1 pair of examinations, clinical follow-up and/or histologic examination did not reveal any tumor deriving from the sympathetic nervous system.
  • 11C-HED PET/CT detected 80 of 81 totally depicted tumor lesions (sensitivity, 0.99; soft tissue, 61; bone, 19).
  • The tumor-to-background contrast of 11C-HED uptake was higher in comparison with 123I-MIBG uptake in 26 lesions (0.32; soft tissue, 18; bone, 8), equal in 39 lesions (0.48; soft tissue, 30; bone, 9), and lower than 123I-MIBG uptake in 16 lesions (0.20; soft tissue, 14; bone, 2).
  • CONCLUSION: Whole-body imaging using 11C-HED PET/CT is feasible in the clinical setting of patients with tumors of the sympathetic nervous system.
  • 11C-HED PET/CT detected more tumor lesions than 123I-MIBG SPECT/CT.
  • However, tumor-to-background contrast of 11C-HED in lesions can be higher, equal, or lower compared with 123I-MIBG.
  • [MeSH-major] 3-Iodobenzylguanidine. Autonomic Nervous System Diseases / radionuclide imaging. Ephedrine / analogs & derivatives. Peripheral Nervous System Neoplasms / radionuclide imaging

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  • (PMID = 17015899.001).
  • [ISSN] 0161-5505
  • [Journal-full-title] Journal of nuclear medicine : official publication, Society of Nuclear Medicine
  • [ISO-abbreviation] J. Nucl. Med.
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Carbon Radioisotopes; 0 / Iodine Radioisotopes; 0 / Radiopharmaceuticals; 3002-40-2 / 3-hydroxyephedrine; 35MRW7B4AD / 3-Iodobenzylguanidine; GN83C131XS / Ephedrine
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21. Dorosevich AE, Bekhtereva IA, Sudilovskaia VV: [The morphological features of the nervous and vascular components of communication systems in the cervix uteri]. Arkh Patol; 2009 Sep-Oct;71(5):43-6
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  • [Title] [The morphological features of the nervous and vascular components of communication systems in the cervix uteri].
  • The investigation has indicated the presence of adrenergic and cholinergic autonomic nerve terminals (ANT) in the tissues of squamous cell carcinomas of the cervix uteri in a tumor growth area and contralaterally.
  • Heterogeneity of the local neuromediator background in the tumor growth area and contralaterally may be explained, by studying the specific features of the cell microenvironment of ANT.
  • [MeSH-major] Carcinoma, Squamous Cell / blood supply. Cervix Uteri / blood supply. Cervix Uteri / innervation. Uterine Cervical Neoplasms / blood supply
  • [MeSH-minor] Adolescent. Adult. Autonomic Nervous System / pathology. Female. Humans. Middle Aged

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  • (PMID = 19938703.001).
  • [ISSN] 0004-1955
  • [Journal-full-title] Arkhiv patologii
  • [ISO-abbreviation] Arkh. Patol.
  • [Language] rus
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] Russia (Federation)
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22. Trochet D, O'Brien LM, Gozal D, Trang H, Nordenskjöld A, Laudier B, Svensson PJ, Uhrig S, Cole T, Niemann S, Munnich A, Gaultier C, Lyonnet S, Amiel J: PHOX2B genotype allows for prediction of tumor risk in congenital central hypoventilation syndrome. Am J Hum Genet; 2005 Mar;76(3):421-6
SciCrunch. OMIM: Data: Gene Annotation .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] PHOX2B genotype allows for prediction of tumor risk in congenital central hypoventilation syndrome.
  • The Phox2b gene is necessary for autonomic nervous-system development.
  • Phox2b-/- mice die in utero with absent autonomic nervous system circuits, since autonomic nervous system neurons either fail to form or degenerate.
  • We have subsequently shown that heterozygous mutations of PHOX2B may account for several combined or isolated disorders of autonomic nervous-system development--namely, tumors of the sympathetic nervous system (TSNS), such as neuroblastoma and late-onset central hypoventilation syndrome.
  • Such findings suggest PHOX2B mutation screening as a simple and reliable tool for the diagnosis of CCHS, independent of the clinically variable phenotype.
  • These data further highlight the link between congenital malformations and tumor predisposition when a master gene in development is mutated.
  • [MeSH-major] Autonomic Nervous System Diseases / genetics. Homeodomain Proteins / genetics. Nervous System Neoplasms / genetics. Sleep Apnea, Central / congenital. Sleep Apnea, Central / genetics. Transcription Factors / genetics

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  • (PMID = 15657873.001).
  • [ISSN] 0002-9297
  • [Journal-full-title] American journal of human genetics
  • [ISO-abbreviation] Am. J. Hum. Genet.
  • [Language] eng
  • [Databank-accession-numbers] OMIM/ 142623/ 209880/ 603851
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Homeodomain Proteins; 0 / NBPhox protein; 0 / Transcription Factors
  • [Other-IDs] NLM/ PMC1196394
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23. Kilinçer C, Oztürk L, Hamamcioglu MK, Altunrende E, Cobanoglu S: An upper thoracic spinal cord tumor presenting as hemifacial hyperhidrosis. Surg Neurol; 2007 Oct;68(4):461-3; discussion 463
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  • [Title] An upper thoracic spinal cord tumor presenting as hemifacial hyperhidrosis.
  • BACKGROUND: Hyperhidrosis as the sole presenting symptom of an upper thoracic intramedullary tumor has never been reported in the English literature.
  • The MRI revealed a large spinal cord tumor at the T1-T2 levels.
  • The patient underwent total excision of the intramedullary tumor via a posterior myelotomy.
  • The histopathological diagnosis was low-grade astrocytoma.
  • Its unusual presentation and lack of motor and sensory deficits resulted in delayed diagnosis of this potentially disabling lesion.
  • When autonomic dysfunction of the face and neck is encountered, in addition to the cranial and cervical regions, the upper thoracic levels should be investigated using MRI.
  • [MeSH-major] Astrocytoma / diagnosis. Hyperhidrosis / etiology. Spinal Cord Neoplasms / diagnosis
  • [MeSH-minor] Adolescent. Autonomic Nervous System Diseases / etiology. Autonomic Nervous System Diseases / physiopathology. Flushing. Functional Laterality / physiology. Humans. Magnetic Resonance Imaging. Male

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  • (PMID = 17905075.001).
  • [ISSN] 0090-3019
  • [Journal-full-title] Surgical neurology
  • [ISO-abbreviation] Surg Neurol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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24. Vassilopoulos D, Mantzoukis D: Dialogue between the brain and the immune system in inflammatory arthritis. Ann N Y Acad Sci; 2006 Nov;1088:132-8
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  • [Title] Dialogue between the brain and the immune system in inflammatory arthritis.
  • The crosstalk between the brain and the immune system in inflammatory arthritis is exerted mainly through the activation or downregulation of the hypothalamic-pituitary-adrenal (HPA), the hypothalamic-pituitary-gonadal (HPG), and the hypothalamic-autonomic nervous system (HANS) axes.
  • Furthermore, the effect of the most recently available biologic therapies like anti-tumor necrosis factor (TNF-a) on the neuroendocrine function in patients with RA will be reviewed.
  • [MeSH-major] Arthritis, Rheumatoid / immunology. Brain / immunology. Immune System / immunology. Neuroimmunomodulation / immunology. Neurosecretory Systems / immunology

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  • (PMID = 17192561.001).
  • [ISSN] 0077-8923
  • [Journal-full-title] Annals of the New York Academy of Sciences
  • [ISO-abbreviation] Ann. N. Y. Acad. Sci.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 16
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25. Guinjoan SM, Vigo DE, Castro MN, Tateosian N, Chuluyan E, Costanzo E, Fahrer R, Grancelli H, Leiguarda R, Cardinali DP: Mood, Th-1/Th-2 cytokine profile, and autonomic activity in older adults with acute/decompensated heart failure: preliminary observations. World J Biol Psychiatry; 2009;10(4 Pt 3):913-8
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  • [Title] Mood, Th-1/Th-2 cytokine profile, and autonomic activity in older adults with acute/decompensated heart failure: preliminary observations.
  • In order to assess the relationships among mood, peripheral autonomic output and circulating immunoinflammatory mediators in older individuals with decompensated heart failure (CHF), 20 consecutive patients (78+/-7 years, 35% women) admitted to the coronary care unit with a clinical diagnosis of acute/decompensated CHF of coronary origin were examined.
  • The results add to the concept that mood and autonomic unbalance are associated with increased systemic inflammation in old patients with decompensated CHF, a potential mechanism for mood-related worsened prognosis of heart failure at an advanced age.
  • [MeSH-major] Autonomic Nervous System Diseases / immunology. Autonomic Nervous System Diseases / physiopathology. Heart Failure. Interferon-gamma / immunology. Interleukin-10 / immunology. Interleukin-2 / immunology. Interleukin-4 / immunology. Interleukin-6 / immunology. Mood Disorders / epidemiology. Mood Disorders / psychology. Peptide Fragments / immunology. Th1 Cells / immunology. Th2 Cells / immunology. Tumor Necrosis Factor-alpha / immunology. Vagus Nerve / immunology. Vagus Nerve / physiopathology
  • [MeSH-minor] Acute Disease. Aged, 80 and over. Aging / physiology. Dyspnea / diagnosis. Dyspnea / epidemiology. Female. Heart Rate / physiology. Humans. Male. Prevalence. Respiratory Sounds. Systemic Inflammatory Response Syndrome / epidemiology. Systemic Inflammatory Response Syndrome / immunology. Systemic Inflammatory Response Syndrome / physiopathology

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  • (PMID = 18937152.001).
  • [ISSN] 1814-1412
  • [Journal-full-title] The world journal of biological psychiatry : the official journal of the World Federation of Societies of Biological Psychiatry
  • [ISO-abbreviation] World J. Biol. Psychiatry
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Interleukin-2; 0 / Interleukin-6; 0 / Peptide Fragments; 0 / Tumor Necrosis Factor-alpha; 0 / interferon gamma (1-39); 130068-27-8 / Interleukin-10; 207137-56-2 / Interleukin-4; 82115-62-6 / Interferon-gamma
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26. Holman AJ, Ng E: Heart rate variability predicts anti-tumor necrosis factor therapy response for inflammatory arthritis. Auton Neurosci; 2008 Dec 5;143(1-2):58-67
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  • [Title] Heart rate variability predicts anti-tumor necrosis factor therapy response for inflammatory arthritis.
  • To consider autonomic status as a predictor of anti-tumor necrosis factor (TNF) treatment response for inflammatory arthritis, we conducted an exploratory, double-blind, 52-week study with 33 patients with rheumatoid (25) or psoriatic (8) arthritis using heart rate variability (HRV).
  • All were assessed for parasympathetic, sympathetic, total power and tension index measures of autonomic reactivity at initiation of anti-TNF therapy with etanercept (15) or adalimumab (18).
  • In conclusion, this unique, exploratory study suggests that HRV may be a novel, useful predictor of response to anti-TNF therapy in patients with inflammatory arthritis, and emphasizes the importance of autonomic influence of autoimmune disease expression.
  • [MeSH-major] Antirheumatic Agents / therapeutic use. Arthritis, Psoriatic / drug therapy. Arthritis, Rheumatoid / drug therapy. Heart Rate / drug effects. Tumor Necrosis Factors / antagonists & inhibitors
  • [MeSH-minor] Adalimumab. Adult. Antibodies, Monoclonal / therapeutic use. Antibodies, Monoclonal, Humanized. Autonomic Nervous System / drug effects. Autonomic Nervous System / physiopathology. Double-Blind Method. Etanercept. Female. Humans. Immunoglobulin G / therapeutic use. Male. Middle Aged. Predictive Value of Tests. Receptors, Tumor Necrosis Factor / therapeutic use. Severity of Illness Index. Treatment Outcome

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  • (PMID = 18632310.001).
  • [ISSN] 1872-7484
  • [Journal-full-title] Autonomic neuroscience : basic & clinical
  • [ISO-abbreviation] Auton Neurosci
  • [Language] eng
  • [Publication-type] Clinical Trial; Journal Article
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Antibodies, Monoclonal; 0 / Antibodies, Monoclonal, Humanized; 0 / Antirheumatic Agents; 0 / Immunoglobulin G; 0 / Receptors, Tumor Necrosis Factor; 0 / Tumor Necrosis Factors; FYS6T7F842 / Adalimumab; OP401G7OJC / Etanercept
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27. Kimura N, Ishidate T, Kogawa T, Miura Y, Ishizaka M, Ogita M: A retroperitoneal sympathetic paraganglioma invading the duodenum and mimicking a submucosal tumor. Endocr Pathol; 2008;19(2):128-32
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  • [Title] A retroperitoneal sympathetic paraganglioma invading the duodenum and mimicking a submucosal tumor.
  • We report a case of an autopsy of unusual retroperitoneal sympathetic paraganglioma (SPG) that directly invaded the duodenum and showed expansive growth mimicking a submucosal tumor.
  • The tumor was clinically suspected to be a gastrointestinal stromal tumor (GIST) of the duodenum because of its location and extension to the retroperitoneum without catecholamine-associated symptoms.
  • However, a small biopsy specimen of the tumor showed diffuse proliferation of large basophilic cells that were negative for C-kit and CD34, ruling out GIST and indicating an epithelial malignancy.
  • An autopsy revealed that the tumor was mainly in the retroperitoneum, measuring 7.5 x 9.5 cm, weighing 600 g and extending into the duodenum, adjacent to the pancreas but free of the adrenal glands.
  • On cut section, the tumor involved the entire wall of the duodenum.
  • For differential diagnosis, endocrine tumors of the duodenum or pancreas and extra-adrenal SPG were considered.
  • The tumor cells were immunohistochemically strongly positive for chromogranin A and were surrounded by cells positive for S100 protein.
  • The four catecholamine-synthesizing enzymes were detected in the tumor cells.
  • We report this case of SPG with emphasis on differential diagnosis and the significance of its local invasion.
  • [MeSH-major] Autonomic Nervous System Diseases / pathology. Duodenal Neoplasms / secondary. Nervous System Neoplasms / pathology. Paraganglioma / secondary
  • [MeSH-minor] Aged, 80 and over. Autopsy. Catecholamines / metabolism. Cell Proliferation. Diagnosis, Differential. Dopamine beta-Hydroxylase / metabolism. Fatal Outcome. Humans. Immunohistochemistry. Ki-67 Antigen / metabolism. Male. Paraffin Embedding. Phenylethanolamine N-Methyltransferase / metabolism. Pneumonia / etiology. Pneumonia / pathology. Retroperitoneal Neoplasms / metabolism. Retroperitoneal Neoplasms / pathology. Retroperitoneal Neoplasms / secondary. Tyrosine 3-Monooxygenase / metabolism

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  • (PMID = 18438726.001).
  • [ISSN] 1046-3976
  • [Journal-full-title] Endocrine pathology
  • [ISO-abbreviation] Endocr. Pathol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Catecholamines; 0 / Ki-67 Antigen; EC 1.14.16.2 / Tyrosine 3-Monooxygenase; EC 1.14.17.1 / Dopamine beta-Hydroxylase; EC 2.1.1.28 / Phenylethanolamine N-Methyltransferase
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28. Hermann GE, Rogers RC: TNF activates astrocytes and catecholaminergic neurons in the solitary nucleus: implications for autonomic control. Brain Res; 2009 Jun 1;1273:72-82
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  • [Title] TNF activates astrocytes and catecholaminergic neurons in the solitary nucleus: implications for autonomic control.
  • Tumor necrosis factor [TNF] produces a profound anorexia associated with gastrointestinal stasis.

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  • (PMID = 19348788.001).
  • [ISSN] 1872-6240
  • [Journal-full-title] Brain research
  • [ISO-abbreviation] Brain Res.
  • [Language] ENG
  • [Grant] United States / NIDDK NIH HHS / DK / R01 DK056373-11; United States / NIDDK NIH HHS / DK / R01 DK056373; United States / NIDDK NIH HHS / DK / R01 DK052142-09; United States / NICHD NIH HHS / HD / HD47643; United States / NIDDK NIH HHS / DK / DK 52142; United States / NIDDK NIH HHS / DK / DK 56373; United States / NICHD NIH HHS / HD / R21 HD047643-02; United States / NICHD NIH HHS / HD / R21 HD047643; United States / NIDDK NIH HHS / DK / R01 DK052142; United States / NICHD NIH HHS / HD / HD047643-02
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Proto-Oncogene Proteins c-fos; 0 / Tumor Necrosis Factor-alpha; EC 1.14.16.2 / Tyrosine 3-Monooxygenase; X4W3ENH1CV / Norepinephrine
  • [Other-IDs] NLM/ NIHMS107940; NLM/ PMC2693276
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29. Scottish Bone Tumor Registry, Sharma H, Mehdi SA, MacDuff E, Reece AT, Jane MJ, Reid R: Paget sarcoma of the spine: Scottish Bone Tumor Registry experience. Spine (Phila Pa 1976); 2006 May 20;31(12):1344-50
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  • [Title] Paget sarcoma of the spine: Scottish Bone Tumor Registry experience.
  • STUDY DESIGN: Retrospective case study of 13 cases of Paget sarcoma of the spine accrued from a prospectively collected Tumor Registry database.
  • METHODS: Between January 1944 and December 2003, 89 patients were registered with a diagnosis of Paget sarcoma in the Scottish Bone Tumor Registry.
  • The mode of presentation was progressively increasing low back pain (in all 13), unilateral sciatica (six; left-sided, five; right-sided, one), bilateral sciatica (two), lower limb weakness (eight), and autonomic dysfunction (four).
  • [MeSH-major] Lumbar Vertebrae. Osteitis Deformans. Sacrum. Sarcoma. Spinal Neoplasms
  • [MeSH-minor] Adult. Aged. Aged, 80 and over. Female. Humans. Male. Middle Aged. Nerve Compression Syndromes / complications. Nerve Compression Syndromes / etiology. Nervous System Diseases / etiology. Registries. Retrospective Studies. Spinal Cord Compression / complications. Spinal Cord Compression / etiology. Spinal Nerve Roots. Survival Analysis. Tomography, X-Ray Computed

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  • (PMID = 16721297.001).
  • [ISSN] 1528-1159
  • [Journal-full-title] Spine
  • [ISO-abbreviation] Spine
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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30. von Düring M, Fricke B: Organization of peripheral nerves in skin, musculoskeletal system and viscera. Prog Exp Tumor Res; 2007;39:30-44

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  • [Title] Organization of peripheral nerves in skin, musculoskeletal system and viscera.
  • Skin, musculoskeletal system and all organs of the body are supplied by nerve fibers of the somatic and autonomic nervous system, each of the systems with its specific nerve fiber types, fiber composition, fiber density and targets.
  • Experimental data support the hypothesis that tumor tissue might interact with nerve fibers.
  • The peripheral nervous system possesses an extraordinary cellular equipment to protect the axons against pathological stimuli.
  • Only restricted areas lacking a cellular barrier are weak points within the nervous network.
  • Therefore, this article focuses on the functional morphology of the peripheral nervous system and its regional differences.
  • [MeSH-major] Musculoskeletal System / innervation. Peripheral Nerves / anatomy & histology. Skin / innervation. Viscera / innervation

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  • (PMID = 17314499.001).
  • [ISSN] 0079-6263
  • [Journal-full-title] Progress in experimental tumor research
  • [ISO-abbreviation] Prog Exp Tumor Res
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Switzerland
  • [Number-of-references] 67
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31. Shen J, Horii Y, Fujisaki Y, Nagai K: Effects of L-arginine and L-lysine mixtures on splenic sympathetic nerve activity and tumor proliferation. Auton Neurosci; 2009 May 11;147(1-2):86-90
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  • [Title] Effects of L-arginine and L-lysine mixtures on splenic sympathetic nerve activity and tumor proliferation.
  • Oral supplementations of L-arginine and L-lysine show tumor inhibition abilities.
  • An increase in splenic-SNA and tumor volume (2 mM), no effect (10 mM), and a decrease in both values (50 mM) were seen.
  • Bivariate correlation analysis revealed a positive correlation between changes in splenic-SNA and tumor volume, indicating the tumor suppressing ability of weakened splenic-SNA.
  • [MeSH-major] Arginine / pharmacology. Immune Tolerance / drug effects. Lysine / pharmacology. Neoplasms / drug therapy. Neuroimmunomodulation / drug effects. Sympathetic Nervous System / drug effects
  • [MeSH-minor] Animals. Antineoplastic Agents / pharmacology. Antineoplastic Agents / therapeutic use. Antineoplastic Combined Chemotherapy Protocols / pharmacology. Antineoplastic Combined Chemotherapy Protocols / therapeutic use. Carcinoma / drug therapy. Carcinoma / immunology. Carcinoma / physiopathology. Cell Proliferation / drug effects. Colonic Neoplasms / drug therapy. Colonic Neoplasms / immunology. Colonic Neoplasms / physiopathology. Disease Models, Animal. Female. Humans. Immunologic Factors / pharmacology. Immunologic Factors / therapeutic use. Killer Cells, Natural / immunology. Male. Mice. Mice, Inbred BALB C. Rats. Rats, Wistar. Spleen / cytology. Spleen / immunology. Spleen / innervation. Treatment Outcome

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  • (PMID = 19237319.001).
  • [ISSN] 1872-7484
  • [Journal-full-title] Autonomic neuroscience : basic & clinical
  • [ISO-abbreviation] Auton Neurosci
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Antineoplastic Agents; 0 / Immunologic Factors; 94ZLA3W45F / Arginine; K3Z4F929H6 / Lysine
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32. Kellogg AP, Converso K, Wiggin T, Stevens M, Pop-Busui R: Effects of cyclooxygenase-2 gene inactivation on cardiac autonomic and left ventricular function in experimental diabetes. Am J Physiol Heart Circ Physiol; 2009 Feb;296(2):H453-61
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  • [Title] Effects of cyclooxygenase-2 gene inactivation on cardiac autonomic and left ventricular function in experimental diabetes.

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  • (PMID = 19060127.001).
  • [ISSN] 0363-6135
  • [Journal-full-title] American journal of physiology. Heart and circulatory physiology
  • [ISO-abbreviation] Am. J. Physiol. Heart Circ. Physiol.
  • [Language] ENG
  • [Grant] United States / NIDDK NIH HHS / DK / NIH5P60 DK20572; United States / NINDS NIH HHS / NS / NS047653-03; United States / NCCIH NIH HHS / AT / R1AT002146
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Blood Glucose; 0 / Inflammation Mediators; 0 / Prostaglandins; 0 / Tumor Necrosis Factor-alpha; 4Y8F71G49Q / Malondialdehyde; 9007-34-5 / Collagen; EC 1.14.99.- / Ptgs2 protein, mouse; EC 1.14.99.1 / Cyclooxygenase 2; GAN16C9B8O / Glutathione
  • [Other-IDs] NLM/ PMC2643890
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33. Lorton D, Lubahn CL, Estus C, Millar BA, Carter JL, Wood CA, Bellinger DL: Bidirectional communication between the brain and the immune system: implications for physiological sleep and disorders with disrupted sleep. Neuroimmunomodulation; 2006;13(5-6):357-74
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Bidirectional communication between the brain and the immune system: implications for physiological sleep and disorders with disrupted sleep.
  • The central nervous system (CNS) modulates immune function by signaling target cells of the immune system through autonomic and neuroendocrine pathways.
  • Cytokines produced by cells of the immune and nervous systems regulate sleep.
  • Cytokines released by immune cells, particularly interleukin-1beta and tumor necrosis factor-alpha, signal neuroendocrine, autonomic, limbic and cortical areas of the CNS to affect neural activity and modify behaviors (including sleep), hormone release and autonomic function.
  • The immune system's ability to affect behavior has important implications for understanding normal and pathological sleep.
  • [MeSH-major] Brain / physiology. Immune System / physiology. Neuroimmunomodulation / physiology. Sleep / physiology. Sleep Wake Disorders / physiopathology
  • [MeSH-minor] Animals. Cytokines / metabolism. Humans. Hypothalamo-Hypophyseal System / physiology. Pituitary-Adrenal System / physiology

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  • [Copyright] Copyright (c) 2006 S. Karger AG, Basel.
  • (PMID = 17709958.001).
  • [ISSN] 1021-7401
  • [Journal-full-title] Neuroimmunomodulation
  • [ISO-abbreviation] Neuroimmunomodulation
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 0 / Cytokines
  • [Number-of-references] 189
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34. Krychun II: [Effect of a complex treatment, including glutargin and erbisol on the blood plasma content of protein P53, apoptotic markers of type II, activity of caspases and the level of sCD 117 in patients with autonomic-vascular dystonia]. Lik Sprava; 2008 Apr-Jun;(3-4):49-56
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Effect of a complex treatment, including glutargin and erbisol on the blood plasma content of protein P53, apoptotic markers of type II, activity of caspases and the level of sCD 117 in patients with autonomic-vascular dystonia].

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  • (PMID = 19145821.001).
  • [ISSN] 1019-5297
  • [Journal-full-title] Likars'ka sprava
  • [ISO-abbreviation] Lik. Sprava
  • [Language] UKR
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] Ukraine
  • [Chemical-registry-number] 0 / Biological Factors; 0 / Biomarkers; 0 / Dipeptides; 0 / TNF-Related Apoptosis-Inducing Ligand; 0 / TNFSF10 protein, human; 0 / Tumor Necrosis Factor-alpha; 0 / Tumor Suppressor Protein p53; 0 / erbisol; EC 2.7.10.1 / Proto-Oncogene Proteins c-kit; EC 3.4.22.- / Caspases; TU1X77K34Q / arginine glutamate
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35. Horenstein MG, Hitchcock TA, Tucker JA: Dual CD117 expression in gastrointestinal stromal tumor (GIST) and paraganglioma of Carney triad: a case report. Int J Surg Pathol; 2005 Jan;13(1):87-92
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  • [Title] Dual CD117 expression in gastrointestinal stromal tumor (GIST) and paraganglioma of Carney triad: a case report.
  • We report a 36-year-old white woman with complete Carney triad, including metastatic gastric stromal tumor (GIST), pulmonary chondroma, and nonfunctioning extra-adrenal paraganglioma.
  • To our knowledge, this is the 21st complete Carney triad case reported and the first report of dual expression CD117 in both GIST and paraganglioma, a finding with intriguing pathogenetic implications related to the organization of the autonomic nervous system.
  • [MeSH-major] Chondroma / pathology. Gastrointestinal Stromal Tumors / pathology. Leiomyosarcoma / secondary. Lung Neoplasms / pathology. Paraganglioma, Extra-Adrenal / pathology. Proto-Oncogene Proteins c-kit / analysis
  • [MeSH-minor] Adult. Antigens, CD34 / analysis. Antineoplastic Agents / therapeutic use. Biomarkers, Tumor / analysis. Chromogranins / analysis. Drug Therapy. Female. Humans. Palliative Care. Syndrome

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  • (PMID = 15735861.001).
  • [ISSN] 1066-8969
  • [Journal-full-title] International journal of surgical pathology
  • [ISO-abbreviation] Int. J. Surg. Pathol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antigens, CD34; 0 / Antineoplastic Agents; 0 / Biomarkers, Tumor; 0 / Chromogranins; EC 2.7.10.1 / Proto-Oncogene Proteins c-kit
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36. Kohr D, Tschernatsch M, Schmitz K, Singh P, Kaps M, Schäfer KH, Diener M, Mathies J, Matz O, Kummer W, Maihöfner C, Fritz T, Birklein F, Blaes F: Autoantibodies in complex regional pain syndrome bind to a differentiation-dependent neuronal surface autoantigen. Pain; 2009 Jun;143(3):246-51
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  • There is an increasing evidence of an involvement of the immune system in CRPS, and recently we showed that CRPS patients have autoantibodies against nervous system structures.
  • Therefore we tested the sera of CRPS patients, neuropathy patients and healthy volunteers for surface-binding autoantibodies to primary cultures of autonomic neurons and differentiated neuroblastoma cell lines using flow cytometry.
  • Thirteen of 30 CRPS patients, but none of 30 healthy controls and only one of the 20 neuropathy sera had specific surface binding to autonomic neurons (p<0.001).
  • Our data show that about 30-40% of CRPS patients have surface-binding autoantibodies against an inducible autonomic nervous system autoantigen.
  • [MeSH-major] Autoantibodies / metabolism. Autoantigens / immunology. Autoimmune Diseases of the Nervous System / immunology. Autonomic Nervous System / immunology. Complex Regional Pain Syndromes / immunology. Neurons / immunology
  • [MeSH-minor] Adult. Antigens, Surface / immunology. Cell Differentiation / immunology. Cell Line, Tumor. Cells, Cultured. Female. Flow Cytometry. Ganglia, Sympathetic / immunology. Ganglia, Sympathetic / physiopathology. Humans. Immune System / physiopathology. Male. Middle Aged. Myenteric Plexus / immunology. Myenteric Plexus / physiopathology. Neurogenesis / immunology. Protein Binding / immunology


37. Mravec B, Gidron Y, Kukanova B, Bizik J, Kiss A, Hulin I: Neural-endocrine-immune complex in the central modulation of tumorigenesis: facts, assumptions, and hypotheses. J Neuroimmunol; 2006 Nov;180(1-2):104-16

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • For the precise coordination of systemic functions, the nervous system uses a variety of peripherally and centrally localized receptors, which transmit information from internal and external environments to the central nervous system.
  • Tight interconnections between the immune, nervous, and endocrine systems provide a base for monitoring and consequent modulation of immune system functions by the brain and vice versa.
  • The immune system plays an important role in tumorigenesis.
  • On the basis of rich interconnections between the immune, nervous and endocrine systems, the possibility that the brain may be informed about tumorigenesis is discussed in this review article.
  • Moreover, the eventual modulation of tumorigenesis by central nervous system is also considered.
  • Prospective consequences of the interactions between tumor and brain for diagnosis and therapy of cancer are emphasized.
  • [MeSH-major] Cell Transformation, Neoplastic / immunology. Neoplasms / immunology. Nervous System / immunology. Neural Pathways / immunology. Neuroimmunomodulation / immunology. Neurosecretory Systems / immunology
  • [MeSH-minor] Animals. Autonomic Pathways / immunology. Autonomic Pathways / physiopathology. Brain / anatomy & histology. Brain / immunology. Humans. Psychophysiologic Disorders / immunology. Psychophysiologic Disorders / physiopathology. Synaptic Transmission / immunology

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  • (PMID = 16945428.001).
  • [ISSN] 0165-5728
  • [Journal-full-title] Journal of neuroimmunology
  • [ISO-abbreviation] J. Neuroimmunol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] Netherlands
  • [Number-of-references] 158
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38. Hashimoto Y, Myojin R, Katoh N, Ohtsu M, Tashiro F, Onoda F, Murakami Y: The bHLH transcription factor Hand2 regulates the expression of nanog in ANS differentiation. Biochem Biophys Res Commun; 2009 Dec 11;390(2):223-9

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The bHLH transcription factor Hand2 regulates the expression of nanog in ANS differentiation.
  • The basic helix-loop-helix transcription factor Hand2 is induced by bone morphogenetic proteins (BMPs) in neural crest-derived precursor cells during the early stage of development of the autonomic nervous system (ANS).
  • Previous studies showed that Hand2 was essential for the ANS differentiation.
  • However, regulatory mechanism of pluripotent genes has not been elucidated in ANS differentiation.
  • Here, we show that Hand2 regulated nanog expression in ANS differentiation.
  • Our studies demonstrated that the forced expression of Hand2 promoted the ANS differentiation program in P19 embryonal carcinoma (EC) cells without aggregation.
  • The rapid downregulation of nanog mRNA during ANS differentiation correlated with the Hand2 transcriptional activity and nanog promoter methylation.
  • These findings are evidence for a presence of the novel regulatory mechanism of nanog in ANS differentiation.
  • [MeSH-major] Autonomic Nervous System / growth & development. Basic Helix-Loop-Helix Transcription Factors / metabolism. Gene Expression Regulation, Developmental. Homeodomain Proteins / genetics. Neurogenesis / genetics
  • [MeSH-minor] Animals. Cell Line, Tumor. Embryonic Stem Cells / metabolism. Mice

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  • (PMID = 19785992.001).
  • [ISSN] 1090-2104
  • [Journal-full-title] Biochemical and biophysical research communications
  • [ISO-abbreviation] Biochem. Biophys. Res. Commun.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Basic Helix-Loop-Helix Transcription Factors; 0 / Hand2 protein, mouse; 0 / Homeodomain Proteins; 0 / Nanog protein, mouse
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39. Palao S, Corral I, Vera R, Alonso de Leciñana M: [Progressive dysautonomia as initial manifestation of anti-Hu antibody-related syndrome]. Neurologia; 2007 Dec;22(10):899-902
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  • INTRODUCTION: The anti-Hu antibody are associated with central nervous system paraneoplastic syndromes such as sensory neuropathy, cerebellar ataxia or limbic encephalitis.
  • The isolated autonomic dysfunction as the presenting manifestation of anti-Hu antibody-related paraneoplastic syndrome is extremely infrequent.
  • Anti-Hu antibodies were positive in serum and cerebrospinal fluid, although tumor was not found.
  • [MeSH-major] Antibodies / immunology. Autonomic Nervous System Diseases / immunology. ELAV Proteins / immunology. Paraneoplastic Syndromes / immunology

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  • (PMID = 18040905.001).
  • [ISSN] 0213-4853
  • [Journal-full-title] Neurología (Barcelona, Spain)
  • [ISO-abbreviation] Neurologia
  • [Language] spa
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Spain
  • [Chemical-registry-number] 0 / Antibodies; 0 / ELAV Proteins
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40. Schuster SR, Tabba M, Sahota P: Relationship between the cardiometabolic syndrome and obstructive sleep apnea. J Cardiometab Syndr; 2006;1(3):204-8
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  • Hypoxemia and sleep fragmentation from OSA appear to produce autonomic activation, alterations in neuroendocrine function, and increased amounts of inflammatory cytokines (interleukin 6 and tumor necrosis factor alpha).
  • [MeSH-minor] Autonomic Nervous System / physiopathology. Continuous Positive Airway Pressure. Glucose / metabolism. Humans. Interleukin-6 / metabolism. Risk Factors. Tumor Necrosis Factor-alpha / metabolism

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  • (PMID = 17679802.001).
  • [ISSN] 1559-4564
  • [Journal-full-title] Journal of the cardiometabolic syndrome
  • [ISO-abbreviation] J Cardiometab Syndr
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / IL6 protein, human; 0 / Interleukin-6; 0 / Tumor Necrosis Factor-alpha; IY9XDZ35W2 / Glucose
  • [Number-of-references] 43
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41. Rosas-Ballina M, Ochani M, Parrish WR, Ochani K, Harris YT, Huston JM, Chavan S, Tracey KJ: Splenic nerve is required for cholinergic antiinflammatory pathway control of TNF in endotoxemia. Proc Natl Acad Sci U S A; 2008 Aug 5;105(31):11008-13
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  • The autonomic nervous system maintains homeostasis through its sympathetic and parasympathetic divisions.
  • During infection, cells of the immune system release cytokines and other mediators that cause fever, hypotension, and tissue injury.
  • Although the effect of cytokines on the nervous system has been known for decades, only recently has it become evident that the autonomic nervous system, in turn, regulates cytokine production through neural pathways.

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  • (PMID = 18669662.001).
  • [ISSN] 1091-6490
  • [Journal-full-title] Proceedings of the National Academy of Sciences of the United States of America
  • [ISO-abbreviation] Proc. Natl. Acad. Sci. U.S.A.
  • [Language] ENG
  • [Grant] United States / NIGMS NIH HHS / GM / R01 GM057226; United States / NIGMS NIH HHS / GM / R01 GM062508; United States / NIGMS NIH HHS / GM / R01 GM57226; United States / NIGMS NIH HHS / GM / R01 GM62508
  • [Publication-type] Comparative Study; Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Chrna7 protein, mouse; 0 / Chrna7 protein, rat; 0 / Receptors, Nicotinic; 0 / Tumor Necrosis Factors; 0 / alpha7 Nicotinic Acetylcholine Receptor; 6M3C89ZY6R / Nicotine
  • [Other-IDs] NLM/ PMC2504833
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42. Efthimiou P, Blanco M: Pathogenesis of neuropsychiatric systemic lupus erythematosus and potential biomarkers. Mod Rheumatol; 2009;19(5):457-68
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  • Systemic lupus erythematosus is a chronic, multisystemic, autoimmune disease that may involve the central, peripheral, and autonomic nervous systems and can present with a wide variety of neurological and psychiatric manifestations.
  • Cytokines that may be involved in the pathology of NPSLE include interleukin (IL)-2, IL-6, IL-8, IL-10, tumor necrosis factor (TNF)-alpha, and interferons (IFN)-alpha and -gamma.

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  • (PMID = 19618249.001).
  • [ISSN] 1439-7609
  • [Journal-full-title] Modern rheumatology
  • [ISO-abbreviation] Mod Rheumatol
  • [Language] ENG
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Autoantibodies; 0 / Biomarkers; 0 / Chemokines; 0 / Cytokines
  • [Number-of-references] 105
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43. Sayk F, Vietheer A, Schaaf B, Wellhoener P, Weitz G, Lehnert H, Dodt C: Endotoxemia causes central downregulation of sympathetic vasomotor tone in healthy humans. Am J Physiol Regul Integr Comp Physiol; 2008 Sep;295(3):R891-8
NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .

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  • Experimental endotoxemia as a model of the initial septic response affects the autonomic nervous system with profound cardiovascular sequelae.
  • [MeSH-major] Endotoxemia / physiopathology. Inflammation / physiopathology. Lipopolysaccharides / toxicity. Sympathetic Nervous System / physiology. Vasomotor System / physiology
  • [MeSH-minor] Adult. Blood Pressure / physiology. Down-Regulation / drug effects. Down-Regulation / physiology. Heart Rate / physiology. Humans. Male. Muscle, Skeletal / blood supply. Muscle, Skeletal / innervation. Placebos. Pressoreceptors / physiology. Tumor Necrosis Factor-alpha / blood

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  • (PMID = 18635446.001).
  • [ISSN] 0363-6119
  • [Journal-full-title] American journal of physiology. Regulatory, integrative and comparative physiology
  • [ISO-abbreviation] Am. J. Physiol. Regul. Integr. Comp. Physiol.
  • [Language] eng
  • [Publication-type] Journal Article; Randomized Controlled Trial; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Lipopolysaccharides; 0 / Placebos; 0 / Tumor Necrosis Factor-alpha
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44. Vernino S, Lindstrom J, Hopkins S, Wang Z, Low PA, Muscle Study Group: Characterization of ganglionic acetylcholine receptor autoantibodies. J Neuroimmunol; 2008 Jun 15;197(1):63-9
The Lens. Cited by Patents in .

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  • Autoimmune autonomic ganglionopathy (AAG) is an antibody-mediated neurological disorder caused by antibodies against neuronal AChRs in autonomic ganglia.

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  • [Cites] J Neurosci. 2004 Aug 11;24(32):7037-42 [15306637.001]
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  • (PMID = 18485491.001).
  • [ISSN] 0165-5728
  • [Journal-full-title] Journal of neuroimmunology
  • [ISO-abbreviation] J. Neuroimmunol.
  • [Language] ENG
  • [Grant] United States / NINDS NIH HHS / NS / P50 NS032352; United States / NINDS NIH HHS / NS / R01 NS011323; United States / NINDS NIH HHS / NS / R01 NS048077-04; United States / NINDS NIH HHS / NS / R01 NS052463; United States / NINDS NIH HHS / NS / NS048077-04; United States / NINDS NIH HHS / NS / NS011323; None / None / / P50 NS032352-150004; United States / NINDS NIH HHS / NS / P50 NS032352-12; United States / NINDS NIH HHS / NS / P50 NS032352-150004; United States / NINDS NIH HHS / NS / P50NS32352; United States / NINDS NIH HHS / NS / P50 NS032352-11; United States / NINDS NIH HHS / NS / NS052463; United States / NINDS NIH HHS / NS / R01 NS048077
  • [Publication-type] Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Autoantibodies; 0 / Chrna7 protein, human; 0 / Chrna7 protein, mouse; 0 / Receptors, Muscarinic; 0 / Receptors, Nicotinic; 0 / alpha7 Nicotinic Acetylcholine Receptor; 0 / nicotinic receptor alpha3beta2; 0 / nicotinic receptor alpha3beta4; 0 / nicotinic receptor alpha4beta2
  • [Other-IDs] NLM/ NIHMS56604; NLM/ PMC2536520
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45. Rudnicki SA, Dalmau J: Paraneoplastic syndromes of the peripheral nerves. Curr Opin Neurol; 2005 Oct;18(5):598-603

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • There is often motor, autonomic, or central nervous system involvement, and electrophysiological studies may demonstrate not only sensory changes, but also motor abnormalities.
  • Whole body fluorodeoxyglucose positron emission tomography scanning helps uncover the associated tumor, and recently proposed criteria may assist in the diagnosis.
  • In many instances, prompt treatment of the tumor and immunotherapy result in symptom stabilization or neurologic improvement.
  • [MeSH-minor] Guillain-Barre Syndrome. Humans. Motor Neuron Disease. Neurons / pathology. Neurons / physiology. Peripheral Nervous System Diseases

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  • (PMID = 16155447.001).
  • [ISSN] 1350-7540
  • [Journal-full-title] Current opinion in neurology
  • [ISO-abbreviation] Curr. Opin. Neurol.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Number-of-references] 69
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46. Carney RM, Freedland KE, Stein PK, Miller GE, Steinmeyer B, Rich MW, Duntley SP: Heart rate variability and markers of inflammation and coagulation in depressed patients with coronary heart disease. J Psychosom Res; 2007 Apr;62(4):463-7
NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .

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  • Cardiac autonomic nervous system (ANS) dysregulation, proinflammatory processes, and procoagulant processes have been suggested as possible explanations.
  • METHODS: Heart rate variability (HRV), an indicator of cardiac autonomic regulation, and markers of inflammation [C-reactive protein (CRP), interleukin-6 (IL-6), tumor necrosis factor-alpha (TNF-alpha)] and coagulation (fibrinogen) were assessed in 44 depressed patients with CHD.
  • The relationship between ANS function and inflammatory and coagulant processes should be investigated in larger mechanistic studies of depression and cardiac morbidity and mortality.

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  • (PMID = 17383498.001).
  • [ISSN] 0022-3999
  • [Journal-full-title] Journal of psychosomatic research
  • [ISO-abbreviation] J Psychosom Res
  • [Language] ENG
  • [Grant] United States / NHLBI NIH HHS / HL / R01 HL065356; United States / NHLBI NIH HHS / HL / R01 HL65356
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Inflammation Mediators; 0 / Interleukin-6; 0 / Tumor Necrosis Factor-alpha; 9001-32-5 / Fibrinogen; 9007-41-4 / C-Reactive Protein
  • [Other-IDs] NLM/ NIHMS21148; NLM/ PMC1924882
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47. Daramola OO, Shinners MJ, Levine SC: Secreting jugulotympanic paraganglioma with venous involvement into the thorax. Laryngoscope; 2008 Jul;118(7):1233-5
Hazardous Substances Data Bank. Norepinephrine .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Paragangliomas are highly vascular tumors that arise from chief cells in extra-adrenal paraganglia of the autonomic nervous system.
  • Multiple imaging options, including magnetic resonance venography, are useful and should include the neck for definitive characterization of tumor anatomy.
  • [MeSH-major] Glomus Jugulare Tumor / diagnosis. Glomus Jugulare Tumor / secretion. Jugular Veins / pathology. Magnetic Resonance Angiography. Norepinephrine / blood. Phlebography
  • [MeSH-minor] Diagnosis, Differential. Ear, Middle / pathology. Ear, Middle / surgery. Embolization, Therapeutic. Female. Hearing Loss, Conductive / etiology. Humans. Mastoid / pathology. Mastoid / surgery. Middle Aged. Neck Dissection. Preoperative Care. Tinnitus / etiology. Tomography, X-Ray Computed

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  • (PMID = 18425050.001).
  • [ISSN] 1531-4995
  • [Journal-full-title] The Laryngoscope
  • [ISO-abbreviation] Laryngoscope
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] X4W3ENH1CV / Norepinephrine
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48. Brauner JS, Clausell N: Neurohumoral, immunoinflammatory and cardiovascular profile of patients with severe tetanus: a prospective study. J Negat Results Biomed; 2006;5:2
MedlinePlus Health Information. consumer health - Tetanus.

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  • INTRODUCTION: Autonomic disturbances in tetanus are traditionally associated with adrenergic variations and/or cardiac dysfunction, based on case report data.
  • Catecholamines (dopamine, norepinephrine, epinephrine and total catecholamines), tumor necrosis factor (TNF)-alpha and LVEF were assessed during the first week of autonomic instability and following tetanus recovery.
  • Troponin T was measured during autonomic instability only.
  • No increase in total catecholamines or in TNF-alpha levels was observed during autonomic instability or in the recovery period.
  • Mean LVEF was similar during autonomic instability and after tetanus recovery, 67 +/- 7% and 65 +/- 7%, respectively.
  • Troponin T levels correlated with pressoric instability during autonomic instability.
  • CONCLUSION: Our study demonstrated that in patients with severe tetanus no significant increased levels of catecholamines or TNF-alpha or evidence of cardiac systolic dysfunction was observed either during autonomic instability or in the recovery period.
  • Elevated values of troponin T detected during autonomic instability were not associated with left ventricular dysfunction.
  • Our data do not support the hypothesis that autonomic disturbances in tetanus are associated with adrenergic variations or cardiac dysfunction.
  • [MeSH-major] Autonomic Nervous System / physiopathology. Cardiovascular System / metabolism. Tetanus / pathology. Tetanus / physiopathology
  • [MeSH-minor] Adult. Aged. Aged, 80 and over. Blood Specimen Collection. Catecholamines / metabolism. Echocardiography. Electrocardiography. Female. Humans. Inflammation / immunology. Inflammation / pathology. Male. Middle Aged. Prospective Studies. Troponin T / metabolism. Tumor Necrosis Factor-alpha / metabolism

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  • (PMID = 16503969.001).
  • [ISSN] 1477-5751
  • [Journal-full-title] Journal of negative results in biomedicine
  • [ISO-abbreviation] J Negat Results Biomed
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Catecholamines; 0 / Troponin T; 0 / Tumor Necrosis Factor-alpha
  • [Other-IDs] NLM/ PMC1434771
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49. Blaes F, Tschernatsch M: Paraneoplastic neurological disorders. Expert Rev Neurother; 2010 Oct;10(10):1559-68
COS Scholar Universe. author profiles.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The article provides an overview on the diagnosis and pathogenesis of paraneoplastic neurological disorders (PNDs), and subsequently the current therapeutic strategies in these patients.
  • PNDs are nervous system dysfunctions in cancer patients, which are not due to a local effect of the tumor or its metastases.
  • Whereas the most central nervous PNDs are more T-cell-mediated, limbic encephalitis can be caused by pathogenic receptor autoantibodies.
  • The PND of the neuromuscular junction and paraneoplastic autonomic neuropathy are mainly associated with receptor or ion channel autoantibodies.
  • By contrast, most CNS PNDs associated with defined antineuronal antibodies directed against intracellular antigens only stabilize after tumor treatment.
  • [MeSH-major] Autoantibodies / immunology. Autoantibodies / physiology. Paraneoplastic Syndromes, Nervous System / diagnosis. Paraneoplastic Syndromes, Nervous System / physiopathology
  • [MeSH-minor] Central Nervous System Diseases / complications. Central Nervous System Diseases / immunology. Female. Humans. Lung Neoplasms / complications. Lung Neoplasms / immunology. Male. Nerve Tissue Proteins / immunology. Nervous System Diseases / complications. Nervous System Diseases / immunology. Paraneoplastic Polyneuropathy / complications. Paraneoplastic Polyneuropathy / immunology

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  • (PMID = 20925471.001).
  • [ISSN] 1744-8360
  • [Journal-full-title] Expert review of neurotherapeutics
  • [ISO-abbreviation] Expert Rev Neurother
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Autoantibodies; 0 / Nerve Tissue Proteins
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50. Mogler C, Kohlhof P, Penzel R, Grenacher L, Haag GM, Schirmacher P, Mueller W: A primary malignant ependymoma of the abdominal cavity: a case report and review of the literature. Virchows Arch; 2009 Apr;454(4):475-8
The Weizmann Institute of Science GeneCards and MalaCards databases. gene/protein/disease-specific - MalaCards for malignant ependymoma .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Ependymomas generally arise in the central nervous system (CNS).
  • Careful reevaluation together with extensive review of the literature and comparison of related cases established the diagnosis after treatment failure and tumor progression.
  • The tumor was large and firm with some small cysts and showed pseudorosettes with strong glial fibrillary acidic protein (GFAP) expression.
  • In conclusion, primary extraneural ependymomas have to be included into the differential diagnosis of abdominal tumors with pseudorosette-formation, even in unusual sites, and GFAP-immunohistochemistry (IHC) supports the diagnosis.
  • [MeSH-major] Abdominal Cavity / pathology. Diagnostic Errors. Ependymoma / diagnosis
  • [MeSH-minor] Adult. Antineoplastic Agents / therapeutic use. Autonomic Pathways / pathology. Combined Modality Therapy. Digestive System Surgical Procedures. Female. Gastrointestinal Neoplasms / pathology. Glial Fibrillary Acidic Protein / biosynthesis. Humans. Immunohistochemistry. Peripheral Nervous System Neoplasms / pathology

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  • (PMID = 19238432.001).
  • [ISSN] 1432-2307
  • [Journal-full-title] Virchows Archiv : an international journal of pathology
  • [ISO-abbreviation] Virchows Arch.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Review
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / Antineoplastic Agents; 0 / Glial Fibrillary Acidic Protein
  • [Number-of-references] 15
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51. Briani C, Doria A, Ruggero S, Toffanin E, Luca M, Albergoni MP, D'Odorico A, Grassivaro F, Lucchetta M, De Lazzari F, Balzani I, Battistin L, Vernino S: Antibodies to muscle and ganglionic acetylcholine receptors (AchR) in celiac disease. Autoimmunity; 2008 Feb;41(1):100-4
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  • Muscle AChR antibodies are found in patients with myasthenia gravis, and ganglionic AChR antibodies in patients with autoimmune autonomic neuropathy.
  • RESULTS: None of our CD patients complained of autonomic symptoms or fatigable weakness.
  • Three of the 4 patients underwent cardiovascular autonomic function tests, which showed no abnormalities.
  • None of our CD patients had autonomic failure or significant levels of ganglionic AChR antibodies.
  • The presence of antibodies in CD and in SLE patients may reflect a non-specific autoimmune response in these patients or may indicate subclinical autoimmune autonomic and neuromuscular involvement.
  • [MeSH-major] Autoantibodies / blood. Autoimmune Diseases of the Nervous System / physiopathology. Celiac Disease / physiopathology. Ganglia, Autonomic / immunology. Muscles / immunology. Receptors, Cholinergic / immunology
  • [MeSH-minor] Adult. Animals. Autonomic Nervous System Diseases / immunology. Autonomic Nervous System Diseases / physiopathology. Cell Line, Tumor. Female. Humans. Male. Mice. Mice, Nude. Middle Aged

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  • (PMID = 18176871.001).
  • [ISSN] 1607-842X
  • [Journal-full-title] Autoimmunity
  • [ISO-abbreviation] Autoimmunity
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Autoantibodies; 0 / Receptors, Cholinergic
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52. Weiser T, Just S: Hyoscine butylbromide potently blocks human nicotinic acetylcholine receptors in SH-SY5Y cells. Neurosci Lett; 2009 Feb 6;450(3):258-61

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • If one hypothesizes that HBB might also affect nicotinic receptors in autonomic neurons in vivo (e. g. in the enteric nervous system), this effect could contribute to its spasmolytic activity.
  • [MeSH-minor] Acetylcholine / pharmacology. Cell Line, Tumor. Dose-Response Relationship, Drug. Enteric Nervous System / drug effects. Enteric Nervous System / metabolism. Ganglionic Blockers / pharmacology. Hexamethonium / pharmacology. Humans. Membrane Potentials / drug effects. Membrane Potentials / physiology. Muscarinic Antagonists / pharmacology. Muscle, Smooth / innervation. Muscle, Smooth / physiopathology. Nicotinic Agonists / pharmacology. Nicotinic Antagonists / pharmacology. Parasympatholytics / pharmacology

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  • (PMID = 19070647.001).
  • [ISSN] 0304-3940
  • [Journal-full-title] Neuroscience letters
  • [ISO-abbreviation] Neurosci. Lett.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Ireland
  • [Chemical-registry-number] 0 / Ganglionic Blockers; 0 / Muscarinic Antagonists; 0 / Nicotinic Agonists; 0 / Nicotinic Antagonists; 0 / Parasympatholytics; 0 / Receptors, Nicotinic; 2Z3E1OF81V / Butylscopolammonium Bromide; 3C9PSP36Z2 / Hexamethonium; N9YNS0M02X / Acetylcholine
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53. Lee L, Niu H, Rueger R, Igawa Y, Deutsch J, Ishii N, Mu S, Sakamoto Y, Busse-Reid R, Gimmi C, Goelzer P, De Schepper S, Yoshimura Y, Barrett J, Ishikawa Y, Weissgerber G, Peck R: The safety, tolerability, pharmacokinetics, and pharmacodynamics of single oral doses of CH4987655 in healthy volunteers: target suppression using a biomarker. Clin Cancer Res; 2009 Dec 1;15(23):7368-74
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  • It potently inhibits mitogen-activated protein kinase signaling pathway activation and tumor cell growth, with an in vitro IC(50) of 5.2 nmol/L for inhibition of MEK1/2.
  • Single-agent oral administration of CH4987655 resulted in complete tumor regressions in xenograft models.
  • Moderate adverse events were experienced by one subject at 1 mg (autonomic nervous system imbalance) and three subjects at 4 mg (diarrhea, abdominal pain, autonomic nervous system and acne).
  • [MeSH-minor] Adolescent. Adult. Antineoplastic Agents. Area Under Curve. Biomarkers, Tumor / metabolism. Dose-Response Relationship, Drug. Double-Blind Method. Female. Humans. Inhibitory Concentration 50. MAP Kinase Signaling System. Male. Middle Aged. Neoplasm Transplantation. Placebos

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  • (PMID = 19934286.001).
  • [ISSN] 1078-0432
  • [Journal-full-title] Clinical cancer research : an official journal of the American Association for Cancer Research
  • [ISO-abbreviation] Clin. Cancer Res.
  • [Language] eng
  • [Publication-type] Journal Article; Randomized Controlled Trial
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antineoplastic Agents; 0 / Biomarkers; 0 / Biomarkers, Tumor; 0 / Enzyme Inhibitors; 0 / Placebos
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54. Peretz A, Kaufman JD, Trenga CA, Allen J, Carlsten C, Aulet MR, Adar SD, Sullivan JH: Effects of diesel exhaust inhalation on heart rate variability in human volunteers. Environ Res; 2008 Jun;107(2):178-84
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  • In this experimental study we assessed whether short-term exposure to DE would result in alterations in heart rate variability (HRV), a non-invasive measure of autonomic control of the heart.
  • CONCLUSIONS: We did not observe a consistent DE effect on the autonomic control of the heart in a controlled-exposure experiment in young participants.

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  • (PMID = 18329013.001).
  • [ISSN] 1096-0953
  • [Journal-full-title] Environmental research
  • [ISO-abbreviation] Environ. Res.
  • [Language] ENG
  • [Grant] United States / NIEHS NIH HHS / ES / ES007033-119016; United States / NIEHS NIH HHS / ES / P30 ES007033-129016; United States / NIEHS NIH HHS / ES / K23ES011139; United States / NCCIH NIH HHS / AT / F32 AT003366; United States / NIEHS NIH HHS / ES / P30 ES007033-139016; United States / NIEHS NIH HHS / ES / ES007033-149016; United States / NIEHS NIH HHS / ES / K23 ES011139; United States / NCRR NIH HHS / RR / M01RR-00037; United States / NIEHS NIH HHS / ES / ES011139-04; United States / NIEHS NIH HHS / ES / K24 ES013195; United States / NCCIH NIH HHS / AT / F32 AT003366-03; United States / NIEHS NIH HHS / ES / K24ES013195; United States / NCRR NIH HHS / RR / RR000037-460950; United States / NIEHS NIH HHS / ES / P30ES07033; United States / NCRR NIH HHS / RR / M01 RR000037; United States / NIEHS NIH HHS / ES / ES013195; United States / NCRR NIH HHS / RR / RR000037-450972; United States / NCRR NIH HHS / RR / RR000037-450950; United States / NCRR NIH HHS / RR / RR000037-460972; United States / NIEHS NIH HHS / ES / P50 ES015915; United States / NCRR NIH HHS / RR / RR000037-440886; United States / NCRR NIH HHS / RR / M01 RR000037-450950; United States / NIEHS NIH HHS / ES / P30 ES007033; United States / NCRR NIH HHS / RR / M01 RR000037-450972; None / None / / P50 ES015915-01; United States / NIEHS NIH HHS / ES / ES015915-016704; United States / NIEHS NIH HHS / ES / K23 ES011139-04; United States / NCRR NIH HHS / RR / M01 RR000037-440886; United States / NCRR NIH HHS / RR / M01 RR000037-475376; United States / NIEHS NIH HHS / ES / K23 ES011139-05; United States / NIEHS NIH HHS / ES / ES007033-129016; United States / NCCIH NIH HHS / AT / AT003366-03; United States / NIEHS NIH HHS / ES / K24 ES013195-03; United States / NIEHS NIH HHS / ES / K24 ES013195-01A1; United States / NCRR NIH HHS / RR / M01 RR000037-460950; United States / NIEHS NIH HHS / ES / P30 ES007033-149016; United States / NCRR NIH HHS / RR / M01 RR000037-460972; United States / NIEHS NIH HHS / ES / P50 ES015915-016704; United States / NCRR NIH HHS / RR / M01 RR000037-450886; United States / NIEHS NIH HHS / ES / K24 ES013195-02; United States / NCRR NIH HHS / RR / RR000037-450886; United States / NIEHS NIH HHS / ES / P50 ES015915-01; United States / NIEHS NIH HHS / ES / P30 ES007033-119016; United States / NCRR NIH HHS / RR / RR000037-475376; United States / NIEHS NIH HHS / ES / ES007033-139016; United States / NIEHS NIH HHS / ES / ES011139-05
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Particulate Matter; 0 / Vehicle Emissions
  • [Other-IDs] NLM/ NIHMS52690; NLM/ PMC2518070
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55. Unal M, Polat A, Pata YS, Vayisoğlu Y, Yildiz A, Ismi O: Paraganglioma of the skull base: a case report. Auris Nasus Larynx; 2007 Sep;34(3):427-30

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Paragangliomas are rare benign neuroendocrine tumors derived from the extra-adrenal paraganglia of the autonomic nervous system.
  • [MeSH-major] Paraganglioma / diagnosis. Skull Base Neoplasms / diagnosis
  • [MeSH-minor] Aged. Biomarkers, Tumor / analysis. Biopsy. Chromogranin A / analysis. Endoscopy. Female. Humans. Magnetic Resonance Imaging. Respiratory Insufficiency / etiology. S100 Proteins / analysis. Skull Base / pathology. Synaptophysin / analysis. Tomography, X-Ray Computed. Vocal Cord Paralysis / etiology

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  • (PMID = 17331688.001).
  • [ISSN] 0385-8146
  • [Journal-full-title] Auris, nasus, larynx
  • [ISO-abbreviation] Auris Nasus Larynx
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Biomarkers, Tumor; 0 / Chromogranin A; 0 / S100 Proteins; 0 / Synaptophysin
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56. Mazur KA: Neuroblastoma: What the nurse practitioner should know. J Am Acad Nurse Pract; 2010 May;22(5):236-45
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  • PURPOSE: To provide a comprehensive review of the background, diagnosis, and primary care management of neuroblastoma (NBL) and to describe the pathophysiology, signs and symptoms, and diagnostic tests for the patient with NBL.
  • CONCLUSIONS: NBL is one of the most common tumors of childhood and clinical presentation depends on the site of the primary tumor as well as the presence and location of any metastasis.
  • The clinical presentation of NBL, the vital facts needed to ensure that this diagnosis will not be overlooked, and follow-up in a primary care setting will be reviewed.
  • [MeSH-minor] Anthracyclines / therapeutic use. Antineoplastic Agents / therapeutic use. Autonomic Nervous System Diseases / diagnosis. Autonomic Nervous System Diseases / nursing. Autonomic Nervous System Diseases / pathology. Immunotherapy. Neural Crest / cytology. Neural Crest / pathology. Prognosis. Radiotherapy

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  • (PMID = 20500737.001).
  • [ISSN] 1745-7599
  • [Journal-full-title] Journal of the American Academy of Nurse Practitioners
  • [ISO-abbreviation] J Am Acad Nurse Pract
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Anthracyclines; 0 / Antineoplastic Agents
  • [Number-of-references] 26
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57. Kakuta Y, Kawamura N, Fukuhara S, Imazu T, Hara T, Yamaguchi S, Adachi S: [Case of retroperitoneal paraganglioma]. Hinyokika Kiyo; 2007 Nov;53(11):801-4

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Paragangliomas are rare neuroendocrine tumors that arise from hyperplastic paraganglionic cells and occur in or near the ganglia of the autonomic nervous system.
  • There was no pedicle and no connection of the tumor to the abdominal or retroperitoneal organs.
  • Complete excision of the tumor was performed smoothly.
  • Nine months after surgery, there were no signs of tumor recurrence.
  • [MeSH-major] Paraganglioma / surgery. Retroperitoneal Neoplasms / surgery
  • [MeSH-minor] Adult. Biomarkers, Tumor / analysis. Catecholamines / analysis. Humans. Magnetic Resonance Imaging. Male. Tomography, X-Ray Computed. Treatment Outcome

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  • (PMID = 18051805.001).
  • [ISSN] 0018-1994
  • [Journal-full-title] Hinyokika kiyo. Acta urologica Japonica
  • [ISO-abbreviation] Hinyokika Kiyo
  • [Language] jpn
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / Biomarkers, Tumor; 0 / Catecholamines
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58. Sevilla MA, Hermsen MA, Weiss MM, Grimbergen A, Balbín M, Llorente JL, Rodrigo JP, Suárez C: Chromosomal changes in sporadic and familial head and neck paragangliomas. Otolaryngol Head Neck Surg; 2009 May;140(5):724-9
MedlinePlus Health Information. consumer health - Head and Neck Cancer.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • OBJECTIVE: Paragangliomas (PGLs) of the head and neck are benign neoplasms derived from the autonomic nervous system.
  • [MeSH-major] Head and Neck Neoplasms / genetics. Paraganglioma / genetics. Succinate Dehydrogenase / genetics
  • [MeSH-minor] Adolescent. Adult. Aged. Chi-Square Distribution. Comparative Genomic Hybridization. DNA Mutational Analysis. Female. Germ-Line Mutation. Humans. Male. Middle Aged. Proto-Oncogene Proteins c-ret / genetics. Von Hippel-Lindau Tumor Suppressor Protein / genetics

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  • (PMID = 19393419.001).
  • [ISSN] 0194-5998
  • [Journal-full-title] Otolaryngology--head and neck surgery : official journal of American Academy of Otolaryngology-Head and Neck Surgery
  • [ISO-abbreviation] Otolaryngol Head Neck Surg
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] EC 1.3.99.1 / Succinate Dehydrogenase; EC 2.7.10.1 / Proto-Oncogene Proteins c-ret; EC 2.7.10.1 / RET protein, human; EC 6.3.2.19 / VHL protein, human; EC 6.3.2.19 / Von Hippel-Lindau Tumor Suppressor Protein
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59. Muriová K, Maláska J, Otevrel F, Slezák M, Kratochvíl M, Sevík P: [Myocardial dysfunction in sepsis--definition and pathogenetic mechanisms]. Vnitr Lek; 2010 Mar;56(3):220-5
Hazardous Substances Data Bank. NITRIC OXIDE .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [MeSH-minor] Animals. Autonomic Nervous System / physiopathology. Coronary Circulation. Cytokines / physiology. Endotoxins / physiology. Humans. Mitochondria, Heart / physiology. Myocardium / metabolism. Myofibrils / physiology. Nitric Oxide / physiology. Tumor Necrosis Factor-alpha / physiology

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  • (PMID = 20394208.001).
  • [ISSN] 0042-773X
  • [Journal-full-title] Vnitr̆ní lékar̆ství
  • [ISO-abbreviation] Vnitr Lek
  • [Language] cze
  • [Publication-type] English Abstract; Journal Article; Review
  • [Publication-country] Czech Republic
  • [Chemical-registry-number] 0 / Cytokines; 0 / Endotoxins; 0 / Tumor Necrosis Factor-alpha; 31C4KY9ESH / Nitric Oxide
  • [Number-of-references] 55
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60. Yu Y, Zhang ZH, Wei SG, Chu Y, Weiss RM, Heistad DD, Felder RB: Central gene transfer of interleukin-10 reduces hypothalamic inflammation and evidence of heart failure in rats after myocardial infarction. Circ Res; 2007 Aug 3;101(3):304-12
Hazardous Substances Data Bank. Norepinephrine .

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  • Hypothalamic tumor necrosis factor-alpha and circulating tumor necrosis factor-alpha and IL-1beta levels, also increased in MI+betaGal, were not affected by AdIL-10 treatment.
  • The results demonstrate that cardiovascular and autonomic mechanisms leading to heart failure after MI can be modulated by manipulating the balance between proinflammatory and antiinflammatory cytokines in the brain.
  • [MeSH-major] Genetic Vectors / therapeutic use. Heart Failure / prevention & control. Interleukin-10 / physiology. Myocardial Infarction / complications. Paraventricular Hypothalamic Nucleus / physiopathology. Sympathetic Nervous System / physiopathology
  • [MeSH-minor] Adenoviridae / genetics. Animals. Brain. Cyclooxygenase 2 / biosynthesis. Cyclooxygenase 2 / genetics. Cytokines / biosynthesis. Cytokines / genetics. Dinoprostone / biosynthesis. Dinoprostone / cerebrospinal fluid. Disease Progression. Gene Transfer Techniques. Heart Ventricles / pathology. Injections. Interleukin-1beta / biosynthesis. Interleukin-1beta / genetics. Ligation. Lung / pathology. Male. NF-kappa B / metabolism. Norepinephrine / blood. Organ Size. Rats. Rats, Sprague-Dawley. Recombinant Fusion Proteins / genetics. Recombinant Fusion Proteins / physiology. Tumor Necrosis Factor-alpha / biosynthesis. Tumor Necrosis Factor-alpha / genetics. Ventricular Remodeling

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  • [CommentIn] Circ Res. 2007 Aug 3;101(3):221-3 [17673679.001]
  • (PMID = 17569888.001).
  • [ISSN] 1524-4571
  • [Journal-full-title] Circulation research
  • [ISO-abbreviation] Circ. Res.
  • [Language] eng
  • [Grant] United States / NHLBI NIH HHS / HL / R01 HL-073986
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, Non-P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Cytokines; 0 / IL10 protein, human; 0 / Interleukin-1beta; 0 / NF-kappa B; 0 / Recombinant Fusion Proteins; 0 / Tumor Necrosis Factor-alpha; 130068-27-8 / Interleukin-10; EC 1.14.99.1 / Cyclooxygenase 2; EC 1.14.99.1 / Ptgs2 protein, rat; K7Q1JQR04M / Dinoprostone; X4W3ENH1CV / Norepinephrine
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61. Rich T, Innominato PF, Boerner J, Mormont MC, Iacobelli S, Baron B, Jasmin C, Lévi F: Elevated serum cytokines correlated with altered behavior, serum cortisol rhythm, and dampened 24-hour rest-activity patterns in patients with metastatic colorectal cancer. Clin Cancer Res; 2005 Mar 1;11(5):1757-64
The Lens. Cited by Patents in .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Actigraphy patterns were correlated with QOL indices, serum cortisol obtained at 8:00 a.m. and 4:00 p.m. and with serum levels of transforming growth factor-alpha, tumor necrosis factor-alpha, and interleukin 6 (IL-6) obtained at 8:00 a.m. and analyzed in duplicate by ELISA.
  • CONCLUSIONS: Significant correlations were found between serum levels of TGF-alpha and IL-6, circadian patterns in wrist activity and serum cortisol and tumor-related symptoms in patients with metastatic colorectal cancer.
  • These data support the hypothesis that some cancer patient's symptoms of fatigue, poor QOL, and treatment outcome are related to tumor or host generated cytokines and could reflect cytokine effects on the circadian timing system.
  • This interplay between cytokine signaling pathways, the hypothalamic-pituitary-adrenal axis, the autonomic nervous system, and efferent pathways of the suprachiasmatic nucleus that control circadian physiology, opens the way to new rational interventions for symptom management in cancer patients.
  • [MeSH-major] Circadian Rhythm. Colorectal Neoplasms / immunology. Colorectal Neoplasms / psychology. Interleukin-6 / blood. Neoplasm Metastasis. Transforming Growth Factor alpha / blood. Tumor Necrosis Factor-alpha / analysis

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  • (PMID = 15755997.001).
  • [ISSN] 1078-0432
  • [Journal-full-title] Clinical cancer research : an official journal of the American Association for Cancer Research
  • [ISO-abbreviation] Clin. Cancer Res.
  • [Language] eng
  • [Grant] United States / NCRR NIH HHS / RR / M01 RR 00847
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Interleukin-6; 0 / Transforming Growth Factor alpha; 0 / Tumor Necrosis Factor-alpha; WI4X0X7BPJ / Hydrocortisone
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62. Shimoyama M, Tatsuoka H, Ohtori S, Tanaka K, Shimoyama N: Change of dorsal horn neurochemistry in a mouse model of neuropathic cancer pain. Pain; 2005 Mar;114(1-2):221-30
SciCrunch. The Antibody Registry: Reagent: Antibodies .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The model was produced by inoculation of Meth-A sarcoma cells to the vicinity of the sciatic nerve, which resulted in growth of a tumor mass embedding the nerve.
  • [MeSH-major] Neoplasms / metabolism. Pain / metabolism. Peripheral Nervous System Diseases / physiopathology. Posterior Horn Cells / chemistry. Posterior Horn Cells / metabolism

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  • (PMID = 15733648.001).
  • [ISSN] 0304-3959
  • [Journal-full-title] Pain
  • [ISO-abbreviation] Pain
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 33507-63-0 / Substance P; 83652-28-2 / Calcitonin Gene-Related Peptide
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63. Loram LC, Harrison JA, Chao L, Taylor FR, Reddy A, Travis CL, Giffard R, Al-Abed Y, Tracey K, Maier SF, Watkins LR: Intrathecal injection of an alpha seven nicotinic acetylcholine receptor agonist attenuates gp120-induced mechanical allodynia and spinal pro-inflammatory cytokine profiles in rats. Brain Behav Immun; 2010 Aug;24(6):959-67
The Lens. Cited by Patents in .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Nicotinic acetylcholine receptors (nAchRs) are not only key receptors in the autonomic nervous system, but also are present on immune cells.

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  • [Copyright] Published by Elsevier Inc.
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  • (PMID = 20353818.001).
  • [ISSN] 1090-2139
  • [Journal-full-title] Brain, behavior, and immunity
  • [ISO-abbreviation] Brain Behav. Immun.
  • [Language] ENG
  • [Grant] United States / NIDA NIH HHS / DA / DA024044; United States / NIDA NIH HHS / DA / R01 DA017670-02; United States / NIDA NIH HHS / DA / K05 DA024044-01A1; United States / NIGMS NIH HHS / GM / R01 GM057226; United States / NIDCR NIH HHS / DE / DE020247; United States / NIGMS NIH HHS / GM / R01 GM057226-10; United States / NIDA NIH HHS / DA / DA017670-02; United States / NIDA NIH HHS / DA / K05 DA024044-02; United States / NIDA NIH HHS / DA / DA017670-01A1; United States / CCR NIH HHS / RC / NS067807-01; United States / NIDA NIH HHS / DA / R01 DA017670-01A1; United States / NIGMS NIH HHS / GM / GM057226-10; United States / NIDA NIH HHS / DA / DA017670; United States / NIDCR NIH HHS / DE / RC1 DE020247; United States / NINDS NIH HHS / NS / NS067807; United States / NIDA NIH HHS / DA / K05 DA024044; United States / NINDS NIH HHS / NS / RC1 NS067807-01; United States / NIDA NIH HHS / DA / DA017670-03; United States / CCR NIH HHS / RC / DE020247-01; United States / NIDCR NIH HHS / DE / RC1 DE020247-01; United States / NIDA NIH HHS / DA / R01 DA017670; United States / NIDA NIH HHS / DA / R01 DA017670-03; United States / NINDS NIH HHS / NS / RC1 NS067807; United States / NIDA NIH HHS / DA / DA024044-02
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Benzylidene Compounds; 0 / Chrna7 protein, rat; 0 / Cytokines; 0 / DNA Primers; 0 / DNA, Complementary; 0 / HIV Envelope Protein gp120; 0 / Interleukin-1beta; 0 / Nerve Tissue Proteins; 0 / Nicotinic Agonists; 0 / Pyridines; 0 / RNA, Messenger; 0 / Receptors, Nicotinic; 0 / Tumor Necrosis Factor-alpha; 0 / alpha7 Nicotinic Acetylcholine Receptor; 156223-05-1 / 3-(2,4-dimethoxybenzylidene)anabaseine
  • [Other-IDs] NLM/ NIHMS197760; NLM/ PMC2902784
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64. An MM, Zou Z, Shen H, Liu P, Chen ML, Cao YB, Jiang YY: Incidence and risk of significantly raised blood pressure in cancer patients treated with bevacizumab: an updated meta-analysis. Eur J Clin Pharmacol; 2010 Aug;66(8):813-21
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The risk may be dose-dependent and vary with tumor type.
  • [MeSH-major] Antibodies, Monoclonal / adverse effects. Autonomic Nervous System Diseases / epidemiology. Cardiovascular Diseases / epidemiology. Hypertension / chemically induced. Meta-Analysis as Topic. Neoplasms / drug therapy
  • [MeSH-minor] Antibodies, Monoclonal, Humanized. Bevacizumab. Breast Neoplasms / chemically induced. Breast Neoplasms / drug therapy. Carcinoma, Renal Cell / chemically induced. Carcinoma, Renal Cell / drug therapy. Female. Humans. Incidence. Randomized Controlled Trials as Topic. Risk Factors. Vascular Endothelial Growth Factor A / therapeutic use

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  • (PMID = 20401474.001).
  • [ISSN] 1432-1041
  • [Journal-full-title] European journal of clinical pharmacology
  • [ISO-abbreviation] Eur. J. Clin. Pharmacol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / Antibodies, Monoclonal; 0 / Antibodies, Monoclonal, Humanized; 0 / Vascular Endothelial Growth Factor A; 2S9ZZM9Q9V / Bevacizumab
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65. Kenemans P, Kubista E, Foidart JM, Yip CH, von Schoultz B, Sismondi P, Vassilopoulou-Sellin R, Beckmann MW, Bundred NJ, Egberts J, van Os S, Planellas J: Safety of tibolone in the treatment of vasomotor symptoms in breast cancer patients--design and baseline data 'LIBERATE' trial. Breast; 2007 Dec;16 Suppl 2:S182-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • For the primary tumor, Stage IIA or higher was reported for >70% of the patients.
  • [MeSH-major] Antineoplastic Agents, Hormonal / pharmacology. Autonomic Nervous System Diseases / drug therapy. Breast Neoplasms / drug therapy. Norpregnenes / pharmacology. Vasomotor System / drug effects
  • [MeSH-minor] Aromatase Inhibitors / adverse effects. Aromatase Inhibitors / pharmacology. Aromatase Inhibitors / therapeutic use. Bone Density. Double-Blind Method. Female. Humans. Middle Aged. Neoplasm Recurrence, Local. Neoplasm Staging. Quality of Life. Survival Analysis. Tamoxifen / adverse effects. Tamoxifen / pharmacology. Tamoxifen / therapeutic use. Treatment Outcome

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  • [ErratumIn] Breast. 2008 Apr;17(2):214-5. Egberts, J [added]; van Os, S [added]; Planellas, J [added]
  • (PMID = 17983942.001).
  • [ISSN] 0960-9776
  • [Journal-full-title] Breast (Edinburgh, Scotland)
  • [ISO-abbreviation] Breast
  • [Language] eng
  • [Publication-type] Journal Article; Multicenter Study; Randomized Controlled Trial; Research Support, Non-U.S. Gov't
  • [Publication-country] Scotland
  • [Chemical-registry-number] 0 / Antineoplastic Agents, Hormonal; 0 / Aromatase Inhibitors; 0 / Norpregnenes; 094ZI81Y45 / Tamoxifen; FF9X0205V2 / tibolone
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66. Kunitz A, Pahl S, Podrabsky P, Wardelmann E, Sturm I: Large paraganglioma of the abdominal cavity: a case report and review of the literature. Onkologie; 2010;33(7):377-80
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  • BACKGROUND: Paragangliomas are rare tumors that derive from cells of the autonomic nervous system.
  • CASE REPORT: We report a case of an oligosymptomatic 50-year-old man presenting with a large intraabdominal tumor mass measuring 24 x 22 x 12 cm.
  • Core needle biopsy revealed a tumor of mesenchymal origin with no clear-line differentiation, so the highly vascularized tumor was resected after embolization of the tumor vessels.
  • Differential diagnosis included histiocytic sarcoma and c-kit-negative gastrointestinal stromal tumor; the final diagnosis was paraganglioma.
  • CONCLUSIONS: Paraganglioma is a rare disease and should be considered in the differential diagnosis of abdominal masses.
  • [MeSH-major] Abdominal Neoplasms / diagnosis. Paraganglioma / diagnosis
  • [MeSH-minor] Angiography. Biomarkers, Tumor / analysis. Diagnosis, Differential. Embolization, Therapeutic. Follow-Up Studies. Humans. Infant, Newborn. Male. Middle Aged. Neoadjuvant Therapy. Neoplasm Invasiveness. Tomography, X-Ray Computed

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  • [Copyright] Copyright 2010 S. Karger AG, Basel.
  • (PMID = 20631484.001).
  • [ISSN] 1423-0240
  • [Journal-full-title] Onkologie
  • [ISO-abbreviation] Onkologie
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Review
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 0 / Biomarkers, Tumor
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67. Rosas-Ballina M, Tracey KJ: Cholinergic control of inflammation. J Intern Med; 2009 Jun;265(6):663-79
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  • The central nervous system interacts dynamically with the immune system to modulate inflammation through humoral and neural pathways.
  • We summarize autonomic regulation of local and systemic inflammation through the 'cholinergic anti-inflammatory pathway', a mechanism consisting of the vagus nerve and its major neurotransmitter, acetylcholine, a process dependent on the nicotinic acetylcholine receptor alpha7 subunit.

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  • (PMID = 19493060.001).
  • [ISSN] 1365-2796
  • [Journal-full-title] Journal of internal medicine
  • [ISO-abbreviation] J. Intern. Med.
  • [Language] ENG
  • [Grant] United States / NIGMS NIH HHS / GM / GM057226-10; United States / NIGMS NIH HHS / GM / R01 GM057226; United States / NIGMS NIH HHS / GM / R01 GM057226-10
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Cholinergic Agents; 0 / Receptors, Cholinergic; 0 / Receptors, Nicotinic; 0 / Tumor Necrosis Factor-alpha; N9YNS0M02X / Acetylcholine
  • [Number-of-references] 124
  • [Other-IDs] NLM/ NIHMS228567; NLM/ PMC4540232
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68. Szutowicz A, Bielarczyk H, Gul S, Ronowska A, Pawełczyk T, Jankowska-Kulawy A: Phenotype-dependent susceptibility of cholinergic neuroblastoma cells to neurotoxic inputs. Metab Brain Dis; 2006 Sep;21(2-3):149-61
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [MeSH-major] Autonomic Nervous System Diseases / pathology. Brain Neoplasms / pathology. Neuroblastoma / pathology. Neurotoxins / toxicity
  • [MeSH-minor] Acetyl Coenzyme A / metabolism. Acetylcholine / biosynthesis. Aluminum / toxicity. Amyloid beta-Peptides / toxicity. Animals. Calcium / metabolism. Cell Differentiation / physiology. Cell Line, Tumor. Choline O-Acetyltransferase / genetics. Cytochromes c / metabolism. DNA, Complementary / genetics. Energy Metabolism / physiology. Immunohistochemistry. Mice. Nitroprusside / toxicity. Peptide Fragments / toxicity. Phenotype. Rats. Receptor, trkA / genetics. Trypan Blue

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  • (PMID = 16724269.001).
  • [ISSN] 0885-7490
  • [Journal-full-title] Metabolic brain disease
  • [ISO-abbreviation] Metab Brain Dis
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Amyloid beta-Peptides; 0 / DNA, Complementary; 0 / Neurotoxins; 0 / Peptide Fragments; 0 / amyloid beta-protein (25-35); 169D1260KM / Nitroprusside; 72-89-9 / Acetyl Coenzyme A; 9007-43-6 / Cytochromes c; CPD4NFA903 / Aluminum; EC 2.3.1.6 / Choline O-Acetyltransferase; EC 2.7.10.1 / Receptor, trkA; I2ZWO3LS3M / Trypan Blue; N9YNS0M02X / Acetylcholine; SY7Q814VUP / Calcium
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69. Batchelor T, Loeffler JS: Primary CNS lymphoma. J Clin Oncol; 2006 Mar 10;24(8):1281-8
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  • Characteristic imaging features should be suggestive of the diagnosis, avoidance of corticosteroids, if possible, and early neurosurgical consultation for stereotactic biopsy.
  • Whole-brain radiation therapy (WBRT) alone is insufficient for durable tumor control and is associated with a high risk of neurotoxicity in patients older than age 60.
  • Neurotoxicity typically is associated with significant cognitive, motor, and autonomic dysfunction, and has a negative impact on quality of life.
  • Chemotherapy and WBRT together improve tumor response rates and survival compared with WBRT alone.
  • Methotrexate-based multiagent chemotherapy without WBRT is associated with similar tumor response rates and survival compared with regimens that include WBRT, although controlled trials have not been performed.
  • [MeSH-major] Central Nervous System Neoplasms. Lymphoma, Non-Hodgkin

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  • (PMID = 16525183.001).
  • [ISSN] 1527-7755
  • [Journal-full-title] Journal of clinical oncology : official journal of the American Society of Clinical Oncology
  • [ISO-abbreviation] J. Clin. Oncol.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 51
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70. Gerstner E, Batchelor T: Primary CNS lymphoma. Expert Rev Anticancer Ther; 2007 May;7(5):689-700
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  • Characteristic imaging features should lead to suspicion of the diagnosis, avoidance of corticosteroids (if possible) and early neurosurgical consultation for stereotactic biopsy.
  • Whole-brain radiation therapy alone is insufficient for durable tumor control and is associated with a high risk of neurotoxicity in patients over 60 years of age.
  • Neurotoxicity is typically associated with significant cognitive, motor and autonomic dysfunction and has a negative impact on quality of life.
  • Chemotherapy and whole-brain radiation therapy together improve tumor response rates and survival compared with whole-brain radiation therapy alone.
  • Methotrexate-based multiagent chemotherapy without whole-brain radiation therapy is associated with similar tumor response rates and survival compared with regimens that include whole-brain radiation therapy, although controlled trials have not been performed.
  • [MeSH-major] Antineoplastic Combined Chemotherapy Protocols / therapeutic use. Central Nervous System Neoplasms / drug therapy. Central Nervous System Neoplasms / radiotherapy. Lymphoma, Non-Hodgkin / drug therapy. Lymphoma, Non-Hodgkin / radiotherapy

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  • (PMID = 17492932.001).
  • [ISSN] 1744-8328
  • [Journal-full-title] Expert review of anticancer therapy
  • [ISO-abbreviation] Expert Rev Anticancer Ther
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Chemical-registry-number] YL5FZ2Y5U1 / Methotrexate
  • [Number-of-references] 71
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71. Lazzerini PE, Acampa M, Hammoud M, Maffei S, Capecchi PL, Selvi E, Bisogno S, Guideri F, Galeazzi M, Pasini FL: Arrhythmic risk during acute infusion of infliximab: a prospective, single-blind, placebo-controlled, crossover study in patients with chronic arthritis. J Rheumatol; 2008 Oct;35(10):1958-65
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  • The acute effects of IFX on autonomic balance may substantiate the role of the complex interaction between autonomic nervous system and inflammation during chronic arthritis.
  • [MeSH-major] Antibodies, Monoclonal / adverse effects. Antirheumatic Agents / adverse effects. Arthritis, Rheumatoid / drug therapy. Tachycardia / chemically induced. Tumor Necrosis Factor-alpha / antagonists & inhibitors

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  • (PMID = 18709695.001).
  • [ISSN] 0315-162X
  • [Journal-full-title] The Journal of rheumatology
  • [ISO-abbreviation] J. Rheumatol.
  • [Language] eng
  • [Publication-type] Clinical Trial; Journal Article
  • [Publication-country] Canada
  • [Chemical-registry-number] 0 / Antibodies, Monoclonal; 0 / Antirheumatic Agents; 0 / Placebos; 0 / Tumor Necrosis Factor-alpha; B72HH48FLU / Infliximab
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72. Casserly P, Kiely P, Fenton JE: Cervical sympathetic chain schwannoma masquerading as a carotid body tumour with a postoperative complication of first-bite syndrome. Eur Arch Otorhinolaryngol; 2009 Oct;266(10):1659-62
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  • The patient had a postoperative complication of first-bite syndrome (FBS).Although rare, CSCS should be considered in the differential diagnosis for tumours at the carotid bifurcation.
  • [MeSH-major] Autonomic Nervous System Diseases / diagnosis. Carotid Body Tumor / diagnosis. Head and Neck Neoplasms / diagnosis. Horner Syndrome / etiology. Neurilemmoma / diagnosis. Peripheral Nervous System Neoplasms / diagnosis. Postoperative Complications / etiology
  • [MeSH-minor] Carotid Artery, External / pathology. Carotid Artery, Internal / pathology. Diagnosis, Differential. Female. Humans. Jugular Veins / pathology. Magnetic Resonance Angiography. Magnetic Resonance Imaging. Middle Aged. Tomography, X-Ray Computed

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  • (PMID = 19130071.001).
  • [ISSN] 1434-4726
  • [Journal-full-title] European archives of oto-rhino-laryngology : official journal of the European Federation of Oto-Rhino-Laryngological Societies (EUFOS) : affiliated with the German Society for Oto-Rhino-Laryngology - Head and Neck Surgery
  • [ISO-abbreviation] Eur Arch Otorhinolaryngol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Germany
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73. Pawlu C, Bausch B, Reisch N, Neumann HP: Genetic testing for pheochromocytoma-associated syndromes. Ann Endocrinol (Paris); 2005 Jun;66(3):178-85
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Pheochromocytoma and paraganglioma are tumors of the autonomic nervous system.
  • Mutational analysis of the susceptibility genes of these syndromes in patients presenting with pheochromocytoma or paraganglioma may help to judge the risks of multifocality of the tumor as well as development of malignant pheochromocytoma or of other malignant tumors.
  • Based on tumor characteristics and prevalence data we give recommendations for an efficient genetic testing procedure in patients presenting with pheochromocytomas and paragangliomas.
  • [MeSH-major] Adrenal Gland Neoplasms / genetics. Multiple Endocrine Neoplasia Type 2a / genetics. Mutation. Pheochromocytoma / genetics
  • [MeSH-minor] Genes, Neurofibromatosis 1. Humans. Neurofibromatoses / genetics. Oncogene Proteins / genetics. Proto-Oncogene Proteins c-ret. Receptor Protein-Tyrosine Kinases / genetics. Thyroid Neoplasms / genetics. Tumor Suppressor Proteins / genetics. Ubiquitin-Protein Ligases / genetics. Von Hippel-Lindau Tumor Suppressor Protein

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  • (PMID = 15988378.001).
  • [ISSN] 0003-4266
  • [Journal-full-title] Annales d'endocrinologie
  • [ISO-abbreviation] Ann. Endocrinol. (Paris)
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] France
  • [Chemical-registry-number] 0 / Oncogene Proteins; 0 / Tumor Suppressor Proteins; EC 2.7.10.1 / Proto-Oncogene Proteins c-ret; EC 2.7.10.1 / RET protein, human; EC 2.7.10.1 / Receptor Protein-Tyrosine Kinases; EC 6.3.2.19 / Ubiquitin-Protein Ligases; EC 6.3.2.19 / VHL protein, human; EC 6.3.2.19 / Von Hippel-Lindau Tumor Suppressor Protein
  • [Number-of-references] 43
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74. Marsland AL, Gianaros PJ, Prather AA, Jennings JR, Neumann SA, Manuck SB: Stimulated production of proinflammatory cytokines covaries inversely with heart rate variability. Psychosom Med; 2007 Nov;69(8):709-16
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  • Convergent evidence from the animal literature shows that the autonomic nervous system plays a key role in regulating the magnitude of immune responses to inflammatory stimuli.
  • Signaling by the parasympathetic system inhibits the production of proinflammatory cytokines by activated monocytes/macrophages and thus decreases local and systemic inflammation.
  • METHODS: We examined the relationship of variations in heart rate, recorded during paced respiration, to lipopolysaccharide-induced production of the inflammatory cytokines interleukin (IL)-1beta, IL-6, tumor necrosis factor (TNF)-alpha, and IL-10 among a community sample of 183 healthy adults (mean age = 45 years; 59% male; 92% White, 7% African-American).

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  • (PMID = 17942840.001).
  • [ISSN] 1534-7796
  • [Journal-full-title] Psychosomatic medicine
  • [ISO-abbreviation] Psychosom Med
  • [Language] eng
  • [Grant] United States / NHLBI NIH HHS / HL / HL007560; United States / NINR NIH HHS / NR / NR008237; United States / NHLBI NIH HHS / HL / P01HL40962
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Cytokines
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75. Suzuki S: [Lambert-Eaton myasthenic syndrome (LEMS)]. Brain Nerve; 2010 Apr;62(4):419-26
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The clinical features of LEMS include proximal weakness, areflexia, ptosis, cerebellar ataxia and autonomic dysfunction.
  • Tumor removal is the primary treatment of LEMS.
  • [MeSH-major] Autoantibodies. Lambert-Eaton Myasthenic Syndrome. Paraneoplastic Syndromes, Nervous System

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  • (PMID = 20420183.001).
  • [ISSN] 1881-6096
  • [Journal-full-title] Brain and nerve = Shinkei kenkyū no shinpo
  • [ISO-abbreviation] Brain Nerve
  • [Language] jpn
  • [Publication-type] English Abstract; Journal Article; Review
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / Autoantibodies; 0 / Calcium Channels, P-Type; 0 / Calcium Channels, Q-Type; 0 / HLA Antigens; 0 / SOX1 protein, human; 0 / SOXB1 Transcription Factors; 134193-27-4 / Synaptotagmins; BH3B64OKL9 / 4-Aminopyridine; RU4S6E2G0J / 3,4-diaminopyridine
  • [Number-of-references] 53
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76. Benfante R, Flora A, Di Lascio S, Cargnin F, Longhi R, Colombo S, Clementi F, Fornasari D: Transcription factor PHOX2A regulates the human alpha3 nicotinic receptor subunit gene promoter. J Biol Chem; 2007 May 4;282(18):13290-302
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • PHOX2A is a paired-like homeodomain transcription factor that participates in specifying the autonomic nervous system.
  • As the alpha3 subunit is expressed in every terminally differentiated ganglionic cell, this is the first example of a "pan-autonomic" gene whose expression is regulated by PHOX2 proteins.
  • [MeSH-minor] Cell Differentiation / physiology. Cell Line, Tumor. Chromatin Immunoprecipitation. Humans. Protein Binding / genetics. Sp1 Transcription Factor / genetics. Sp1 Transcription Factor / metabolism

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  • (PMID = 17344216.001).
  • [ISSN] 0021-9258
  • [Journal-full-title] The Journal of biological chemistry
  • [ISO-abbreviation] J. Biol. Chem.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Homeodomain Proteins; 0 / PHOX2A protein, human; 0 / Receptors, Nicotinic; 0 / Sp1 Transcription Factor; 0 / nicotinic receptor subunit alpha3
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77. Kirby S, Purdy RA: Headache and brain tumors. Curr Neurol Neurosci Rep; 2007 Mar;7(2):110-6
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  • The epidemiology of this clinically relevant area is highlighted along with general clinical features of headache disorders seen in brain tumor patients.
  • Some rarer clinical presentations are noted, particularly in relationship to the newly described trigeminal autonomic cephalalgias, as well as the relationships of headache to pituitary tumors and paroxysmal and positional headaches.
  • Headaches as a result of brain tumor therapy are noted, as is the important area of treatment of headaches in patients with central nervous system neoplasms of a primary or secondary nature.
  • [MeSH-major] Antineoplastic Agents / adverse effects. Brain Neoplasms / complications. Brain Neoplasms / drug therapy. Headache / chemically induced. Headache / etiology

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  • (PMID = 17324360.001).
  • [ISSN] 1528-4042
  • [Journal-full-title] Current neurology and neuroscience reports
  • [ISO-abbreviation] Curr Neurol Neurosci Rep
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antineoplastic Agents
  • [Number-of-references] 62
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78. Ideguchi M, Kajiwara K, Yoshikawa K, Kato S, Ishihara H, Fujii M, Fujisawa H, Suzuki M: Continuous hypertension and tachycardia after resection of a hemangioblastoma behind the dorsal medulla oblongata: relationship to sympathetic overactivity at the neurogenic vasomotor center. J Neurosurg; 2010 Aug;113(2):369-73
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  • Radiological examination revealed a tumor located behind the dorsal medulla oblongata and compressing it substantially.
  • The tumor was completely resected, but after the surgery the patient experienced prolonged hypertension and tachycardia.
  • [MeSH-major] Autonomic Nervous System Diseases / etiology. Brain Stem Neoplasms / surgery. Hemangioblastoma / surgery. Hypertension / etiology. Postoperative Complications / etiology. Tachycardia / etiology
  • [MeSH-minor] Adrenergic beta-Antagonists / administration & dosage. Baroreflex. Calcium Channel Blockers / administration & dosage. Diltiazem / administration & dosage. Humans. Magnetic Resonance Imaging. Male. Medulla Oblongata / pathology. Morpholines / administration & dosage. Solitary Nucleus / pathology. Urea / administration & dosage. Urea / analogs & derivatives. Vasomotor System / pathology. Young Adult


79. Saadat P, Cesnorek S, Ram R, Kelly L, Vadmal M: Primary cutaneous paraganglioma of the scalp. J Am Acad Dermatol; 2006 May;54(5 Suppl):S220-3
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  • Paragangliomas are neuroendocrine tumors that arise in relation to the sympathetic or parasympathetic ganglia of the autonomic nervous system.
  • The tumor was studied histologically, immunohistochemically, and ultrastructurally.
  • [MeSH-major] Paraganglioma / pathology. Scalp. Skin Neoplasms / pathology

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  • (PMID = 16631945.001).
  • [ISSN] 1097-6787
  • [Journal-full-title] Journal of the American Academy of Dermatology
  • [ISO-abbreviation] J. Am. Acad. Dermatol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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80. Shusterman V, McTiernan CF, Goldberg A, Saba S, Salama G, London B: Adrenergic stimulation promotes T-wave alternans and arrhythmia inducibility in a TNF-alpha genetic mouse model of congestive heart failure. Am J Physiol Heart Circ Physiol; 2010 Feb;298(2):H440-50
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  • [Title] Adrenergic stimulation promotes T-wave alternans and arrhythmia inducibility in a TNF-alpha genetic mouse model of congestive heart failure.
  • T-wave alternans (TWA) is a proarrhythmic repolarization instability that is common in congestive heart failure (CHF).
  • Although transgenic mice are commonly used to study the mechanisms of arrhythmogenesis in CHF, little is known about the dynamics of TWA in these species.
  • We hypothesized that TWA is present in a TNF-alpha model of CHF and can be further promoted by adrenergic stimulation.
  • We studied 16 TNF-alpha mice and 12 FVB controls using 1) in vivo intracardiac electrophysiological testing and 2) ambulatory telemetry during 30 min before and after an intraperitoneal injection of isoproterenol.
  • TWA was examined using both linear and nonlinear filtering applied in the time domain.
  • In addition, changes in the mean amplitude of the T wave and area under the T wave were computed.
  • During intracardiac electrophysiological testing, none of the animals had TWA or inducible arrhythmias before the injection of isoproterenol.
  • After the injection, sustained TWA and inducible ventricular tachyarrhythmias were observed in TNF-alpha mice but not in FVB mice.
  • In ambulatory telemetry, before the isoproterenol injection, the cardiac cycle length (CL) was longer in TNF-alpha mice than in FVB mice (98 +/- 9 and 88 +/- 3 ms, P = 0.04).
  • After the injection of isoproterenol, the CL became 8% and 6% shorter in TNF-alpha and FVB mice (P < 10(-4)); however, the 2% difference between the groups in the magnitude of CL changes was not significant.
  • In TNF-alpha mice, the magnitude of TWA was 1.5-2 times greater than in FVB mice both before and after the isoproterenol injection.
  • The magnitude of TWA increased significantly after the isoproterenol injection compared with the baseline in TNF-alpha mice (P = 0.003) but not in FVB mice.
  • The mean amplitude of the T wave and area under the T wave increased 60% and 80% in FVB mice (P = 0.006 and 0.009) but not in TNF-alpha mice.
  • In conclusion, TWA is present in a TNF-alpha model of CHF and can be further promoted by adrenergic stimulation, along with the enhanced susceptibility for ventricular arrhythmias.

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  • (PMID = 19940073.001).
  • [ISSN] 1522-1539
  • [Journal-full-title] American journal of physiology. Heart and circulatory physiology
  • [ISO-abbreviation] Am. J. Physiol. Heart Circ. Physiol.
  • [Language] ENG
  • [Grant] United States / NHLBI NIH HHS / HL / R01 HL062300; United States / NHLBI NIH HHS / HL / R44 HL077116; United States / NHLBI NIH HHS / HL / R01-HL-62300; United States / NHLBI NIH HHS / HL / R44-HL-077116
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Adrenergic beta-Agonists; 0 / Receptors, Adrenergic; 0 / Tumor Necrosis Factor-alpha; L628TT009W / Isoproterenol
  • [Other-IDs] NLM/ PMC2822584
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81. Gjuric M, Gleeson M: Consensus statement and guidelines on the management of paragangliomas of the head and neck. Skull Base; 2009 Jan;19(1):109-16

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Hereditary PGL is an autosomal-dominant tumor predisposition syndrome in which an affected individual has greatly increased risk of developing PGL at any or several sites in the autonomic nervous system.

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  • (PMID = 19568348.001).
  • [ISSN] 1531-5010
  • [Journal-full-title] Skull base : official journal of North American Skull Base Society ... [et al.]
  • [ISO-abbreviation] Skull Base
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC2637566
  • [Keywords] NOTNLM ; Head and neck paraganglioma / genetics / management / molecular biology / surgery
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82. Hermann GE, Holmes GM, Rogers RC: TNF(alpha) modulation of visceral and spinal sensory processing. Curr Pharm Des; 2005;11(11):1391-409

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The cytokine tumor necrosis factor(alpha) (TNF(alpha)) is associated with a constellation of physiological and behavioral characteristics that follow in response to infection such as fever, fatigue, listlessness, loss of appetite, malaise, and tactile hypersensitivity.
  • These responses are examples of central nervous system (CNS) functions modified by the activated immune system.
  • [MeSH-major] Neurons, Afferent / physiology. Receptors, Tumor Necrosis Factor / metabolism. Spinal Cord / drug effects. Tumor Necrosis Factor-alpha / pharmacology. Viscera / innervation

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  • (PMID = 15853670.001).
  • [ISSN] 1381-6128
  • [Journal-full-title] Current pharmaceutical design
  • [ISO-abbreviation] Curr. Pharm. Des.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Receptors, Tumor Necrosis Factor; 0 / Tumor Necrosis Factor-alpha
  • [Number-of-references] 122
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83. Osaka K, Yokonishi T, Ito Y, Komiya A, Kobayashi K, Sakai N, Noguchi S, Kishi H, Tsuura Y: [A case of giant retroperitoneal paraganglioma]. Hinyokika Kiyo; 2010 Jul;56(7):377-80
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Paraganglioma is a rare neuroendocrine tumor which arises from extra adrenal paraganglionic cells of the autonomic nervous system.
  • Angiography showed the left renal, splenic, middle suprarenal and left inferior diaphragmatic artery feeding the tumor.
  • The tumor, left kidney, adrenal gland and spleen were surgically resected.
  • Histological examination revealed extra-adrenal paraganglioma, and there was no invasion of the tumor to the left kidney, adrenal gland and spleen.
  • The patient has now survived more than 10 months following the surgery without tumor recurrence.
  • [MeSH-major] Paraganglioma / pathology. Retroperitoneal Neoplasms / pathology

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  • (PMID = 20724811.001).
  • [ISSN] 0018-1994
  • [Journal-full-title] Hinyokika kiyo. Acta urologica Japonica
  • [ISO-abbreviation] Hinyokika Kiyo
  • [Language] jpn
  • [Publication-type] Case Reports; English Abstract; Journal Article; Review
  • [Publication-country] Japan
  • [Number-of-references] 13
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84. Evans JM, Collins M: Clinically diagnosed glomus vagale tumour treated with external beam radiotherapy: a review of the published reports. J Med Imaging Radiat Oncol; 2008 Dec;52(6):617-21

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The details of presentation, diagnosis, treatment and follow up of the patient are discussed.
  • A review of the published work was carry out using MEDLINE database with respect to aetiology, clinical presentation, diagnosis, treatment and expected outcomes.
  • Glomus vagale tumours are a subtype of paragangliomas of the head and neck derived from extra-adrenal paraganglia of the autonomic nervous system.
  • [MeSH-major] Aortic Bodies / radiation effects. Carotid Body Tumor / diagnosis. Carotid Body Tumor / radiotherapy. Radiotherapy, Conformal / methods

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  • (PMID = 19178639.001).
  • [ISSN] 1754-9485
  • [Journal-full-title] Journal of medical imaging and radiation oncology
  • [ISO-abbreviation] J Med Imaging Radiat Oncol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Review
  • [Publication-country] Australia
  • [Number-of-references] 36
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85. Schuller HM: Neurotransmitter receptor-mediated signaling pathways as modulators of carcinogenesis. Prog Exp Tumor Res; 2007;39:45-63
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The autonomic nervous system with its two antagonistic branches, the sympathicus and the parasympathicus, regulates the activities of all body functions that are not under voluntary control.
  • While the autonomic regulation of organ functions has been extensively studied, little attention has been given to the potential role of neurohumoral transmission at the cellular level in the development of cancer.
  • [MeSH-minor] Animals. Cell Proliferation. Cell Transformation, Neoplastic. Humans. Neoplasms / metabolism

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  • (PMID = 17314500.001).
  • [ISSN] 0079-6263
  • [Journal-full-title] Progress in experimental tumor research
  • [ISO-abbreviation] Prog Exp Tumor Res
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 0 / Carcinogens; 0 / Receptors, Neurotransmitter
  • [Number-of-references] 81
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86. Eisenhofer G, Sharabi Y, Pacak K: Unexplained symptomatic paroxysmal hypertension in pseudopheochromocytoma: a stress response disorder? Ann N Y Acad Sci; 2008 Dec;1148:469-78
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Among overall numbers of patients tested for pheochromocytoma, less than 2% harbor the tumor.
  • Similar to patients with the real tumor, patients with pseudopheochromocytoma can be misdiagnosed with panic disorder.
  • Because the clinical manifestations of pseudopheochromocytoma are similar, if not identical, to those due to excess circulating catecholamines in patients with the tumor, the most attractive explanation for the disorder is that it involves altered function of the autonomic nervous system.
  • [MeSH-minor] Adrenal Gland Neoplasms / physiopathology. Adrenal Gland Neoplasms / therapy. Diagnosis, Differential. Epinephrine / metabolism. Humans. Panic Disorder / physiopathology


87. Levy MT, Braun JT, Pennant M, Thompson LD: Primary paraganglioma of the parathyroid: a case report and clinicopathologic review. Head Neck Pathol; 2010 Mar;4(1):37-43
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Paragangliomas are relatively uncommon neoplasms that arise in adrenal and extra-adrenal paraganglia of the autonomic nervous system.
  • [MeSH-major] Paraganglioma, Extra-Adrenal / pathology. Parathyroid Neoplasms / pathology
  • [MeSH-minor] Aged. Antigens, CD56 / metabolism. Biomarkers, Tumor / metabolism. Cell Nucleus / pathology. Chromogranins / metabolism. Cyclin D1 / metabolism. Disease-Free Survival. Female. Humans. Synaptophysin / metabolism. Thyroidectomy

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  • (PMID = 20237987.001).
  • [ISSN] 1936-0568
  • [Journal-full-title] Head and neck pathology
  • [ISO-abbreviation] Head Neck Pathol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antigens, CD56; 0 / Biomarkers, Tumor; 0 / CCND1 protein, human; 0 / Chromogranins; 0 / Synaptophysin; 136601-57-5 / Cyclin D1
  • [Number-of-references] 11
  • [Other-IDs] NLM/ PMC2825535
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88. Fu D, Li CR, He YX, Zu Y, Cao DZ, Wang GB, Deng JK, Wang HP: [Changes of immune function in patients with enterovirus 71 infection]. Zhonghua Er Ke Za Zhi; 2009 Nov;47(11):829-34
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The patients were divided into four groups according to critical degree of enterovirus 71 infection: hand-foot-and-mouth disease (HFMD); central nervous system disease (CNSD); autonomic nervous system dysregulation (ANSD) and pulmonary edema (PE).
  • [MeSH-minor] Adolescent. CD4-Positive T-Lymphocytes / immunology. Case-Control Studies. Child. Child, Preschool. Enterovirus A, Human. Female. HLA-DR Antigens / immunology. Humans. Inflammation. Interleukin-10 / metabolism. Lymphocyte Count. Male. Tumor Necrosis Factor-alpha / metabolism

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  • (PMID = 20078993.001).
  • [ISSN] 0578-1310
  • [Journal-full-title] Zhonghua er ke za zhi = Chinese journal of pediatrics
  • [ISO-abbreviation] Zhonghua Er Ke Za Zhi
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] China
  • [Chemical-registry-number] 0 / HLA-DR Antigens; 0 / Tumor Necrosis Factor-alpha; 130068-27-8 / Interleukin-10
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89. Reusch J, Haag C, Raue F, Badenhoop K: Relapsing pheochromocytoma in a Chinese women caused by a novel mutation in exon 6 of the SDHB gene: a case report. Exp Clin Endocrinol Diabetes; 2007 Oct;115(9):616-8
MedlinePlus Health Information. consumer health - Pheochromocytoma.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Pheochromocytomas are rare catecholamine-secreting, chromaffin tumors of the autonomic nervous system.
  • The transabdominal ultrasonography revealed a tumor with reduced echogenicity in the left suprarenal region, which was suspected to be a recurrent pheochromocytoma.
  • [MeSH-major] Adrenal Gland Neoplasms / genetics. Iron-Sulfur Proteins / genetics. Neoplasm Recurrence, Local / genetics. Pheochromocytoma / genetics. Point Mutation. Succinate Dehydrogenase / genetics
  • [MeSH-minor] Adult. DNA, Neoplasm / chemistry. DNA, Neoplasm / genetics. Female. Humans. Male. Pedigree. Sequence Analysis, DNA

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  • (PMID = 17943698.001).
  • [ISSN] 0947-7349
  • [Journal-full-title] Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association
  • [ISO-abbreviation] Exp. Clin. Endocrinol. Diabetes
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / DNA, Neoplasm; 0 / Iron-Sulfur Proteins; EC 1.3.5.1 / SDHB protein, human; EC 1.3.99.1 / Succinate Dehydrogenase
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90. Borghini S, Vargiolu M, Di Duca M, Ravazzolo R, Ceccherini I: Nuclear factor Y drives basal transcription of the human TLX3, a gene overexpressed in T-cell acute lymphocytic leukemia. Mol Cancer Res; 2006 Sep;4(9):635-43

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Based on a knocked-out mouse model and a few expression studies, TLX3 is regarded as a homeobox gene crucial for the development of the autonomic nervous system.
  • [MeSH-minor] Animals. Base Sequence. Cell Line, Tumor. Humans. Mice. Molecular Sequence Data. Promoter Regions, Genetic. Transcription Initiation Site. Transcription, Genetic

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  • (PMID = 16966433.001).
  • [ISSN] 1541-7786
  • [Journal-full-title] Molecular cancer research : MCR
  • [ISO-abbreviation] Mol. Cancer Res.
  • [Language] eng
  • [Grant] Italy / Telethon / / GGP04257
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / CCAAT-Binding Factor; 0 / Homeodomain Proteins; 0 / Oncogene Proteins; 0 / TLX3 protein, human; 0 / Tlx3 protein, mouse; 0 / Transcription Factors; 0 / nuclear factor Y
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91. Hensen EF, Goeman JJ, Oosting J, Van der Mey AG, Hogendoorn PC, Cremers CW, Devilee P, Cornelisse CJ: Similar gene expression profiles of sporadic, PGL2-, and SDHD-linked paragangliomas suggest a common pathway to tumorigenesis. BMC Med Genomics; 2009;2:25
SciCrunch. ArrayExpress: Data: Microarray .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • BACKGROUND: Paragangliomas of the head and neck are highly vascular and usually clinically benign tumors arising in the paraganglia of the autonomic nervous system.
  • The supervised analysis on pathways implicated in PGL tumor formation also did not reveal significant differences in gene expression between these paraganglioma subgroups.
  • CONCLUSION: The similarity in gene-expression profiles suggests that PGL2, like SDHD, is involved in the functionality of the SDH complex, and that tumor formation in these subgroups involves the same pathways as in SDH linked paragangliomas.

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  • (PMID = 19432956.001).
  • [ISSN] 1755-8794
  • [Journal-full-title] BMC medical genomics
  • [ISO-abbreviation] BMC Med Genomics
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC2694828
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92. Misawa T, Mizusawa H: [Anti-VGKC antibody-associated limbic encephalitis/Morvan syndrome]. Brain Nerve; 2010 Apr;62(4):339-45
MedlinePlus Health Information. consumer health - Syringomyelia.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Anti-voltage-gated potassium channel antibodies (anti-VGKC-Ab) cause hyperexcitability of the peripheral nerve and central nervous system.
  • Morvan syndrome is characterized by neuromyotonia with autonomic and CNS involvement.
  • Morvan syndrome is characterized by widespread neurological symptoms involving the peripheral nervous system (neuromyotonia), autonomic system (hyperhidrosis, severe constipation, urinary incontinence, and cardiac arrhythmia) and the CNS (severe insomnia, hallucinations, impairment of short-term memory and epilepsy).
  • Many patients have an underlying tumor, for example thymoma, lung cancer, testicular cancer and lymphoma; this indicates the paraneoplastic nature of the disease.
  • Plasma exchange is an effective treatment approach, and tumor resection also improves symptoms.
  • [MeSH-minor] Adrenal Cortex Hormones / administration & dosage. Female. Humans. Immunoglobulins, Intravenous / administration & dosage. Middle Aged. Neoplasms / complications. Plasma Exchange

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  • (PMID = 20420173.001).
  • [ISSN] 1881-6096
  • [Journal-full-title] Brain and nerve = Shinkei kenkyū no shinpo
  • [ISO-abbreviation] Brain Nerve
  • [Language] jpn
  • [Publication-type] Case Reports; English Abstract; Journal Article; Review
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / Adrenal Cortex Hormones; 0 / Autoantibodies; 0 / Immunoglobulins, Intravenous; 0 / Potassium Channels, Voltage-Gated
  • [Number-of-references] 36
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93. Likasitwattanakul S: Serotonin syndrome: a case report. J Med Assoc Thai; 2005 Jul;88(7):993-6
Hazardous Substances Data Bank. SERTRALINE .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Serotonin syndrome is a rare but potentially fatal complication of drugs that have effects on central nervous system serotonin.
  • It is characterized by sudden onset of altered mental status, increased neuromuscular activity and autonomic instability.
  • The author reports a child with suprasellar region tumor who presented with depression and obsessive-compulsive disorder and received a combination of sertaline (selective serotonin reuptake inhibitor) and clomipramine (tricyclic antidepressant).
  • [MeSH-major] Depressive Disorder / drug therapy. Obsessive-Compulsive Disorder / drug therapy. Serotonin Syndrome / diagnosis. Serotonin Uptake Inhibitors / adverse effects. Sertraline / adverse effects

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  • (PMID = 16241032.001).
  • [ISSN] 0125-2208
  • [Journal-full-title] Journal of the Medical Association of Thailand = Chotmaihet thangphaet
  • [ISO-abbreviation] J Med Assoc Thai
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Thailand
  • [Chemical-registry-number] 0 / Serotonin Uptake Inhibitors; QUC7NX6WMB / Sertraline
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94. Schulte TL, Adolphs B, Oberdiek D, Osada N, Liljenqvist U, Filler TJ, Marziniak M, Bullmann V: Approach-related lesions of the sympathetic chain in anterior correction and instrumentation of idiopathic scoliosis. Eur Spine J; 2010 Sep;19(9):1558-68
MedlinePlus Health Information. consumer health - Scoliosis.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • During anterior scoliosis instrumentation with a dual-rod system, the vertebrae are dissected anterolaterally.
  • In conclusion, anterior scoliosis instrumentation with a dual-rod system including vertebrae down to L3 regularly leads to lesions in the sympathetic trunk.
  • [MeSH-major] Autonomic Nervous System Diseases / etiology. Internal Fixators / adverse effects. Scoliosis / surgery. Spinal Fusion / adverse effects. Spinal Nerve Roots / injuries

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  • (PMID = 20502925.001).
  • [ISSN] 1432-0932
  • [Journal-full-title] European spine journal : official publication of the European Spine Society, the European Spinal Deformity Society, and the European Section of the Cervical Spine Research Society
  • [ISO-abbreviation] Eur Spine J
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Germany
  • [Other-IDs] NLM/ PMC2989285
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95. Chishima F, Tamura M, Nakazawa T, Sugitani M, Hirano D, Yoshikawa T, Fuchinoue F, Nemoto N, Yamamoto T: Paraaortic paraganglioma arising in an ovarian carcinoma patient mimicking lymph node metastasis. J Obstet Gynaecol Res; 2010 Feb;36(1):204-8
MedlinePlus Health Information. consumer health - Ovarian Cancer.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Paragangliomas are rare tumors arising from the chromaffin cells in the autonomic nervous system.
  • Image analysis showed a 21 x 18 x 10 cm ovarian mass, and a 38 mm tumor at the paraaortic lesion.
  • The paraaortic tumor was completely eliminated in spite of fluctuating blood pressure intraoperatively.
  • Microscopic examination revealed that the paraaortic tumor was paraganglioma.
  • [MeSH-major] Cystadenocarcinoma / diagnosis. Endocrine Gland Neoplasms / diagnosis. Neoplasms, Multiple Primary / diagnosis. Ovarian Neoplasms / diagnosis. Para-Aortic Bodies. Paraganglioma / diagnosis
  • [MeSH-minor] Diagnosis, Differential. Female. Humans. Lymphatic Metastasis / diagnosis. Middle Aged

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  • (PMID = 20178553.001).
  • [ISSN] 1447-0756
  • [Journal-full-title] The journal of obstetrics and gynaecology research
  • [ISO-abbreviation] J. Obstet. Gynaecol. Res.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Australia
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96. Ferrari TC, Moreira PR, Cunha AS: Clinical characterization of neuroschistosomiasis due to Schistosoma mansoni and its treatment. Acta Trop; 2008 Nov-Dec;108(2-3):89-97

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The involvement of the central nervous system (CNS) by Schistosoma mansoni may or may not cause clinical manifestations.
  • Lower limbs pain, weakness and sensory disturbance, and autonomic dysfunctions, particularly bladder dysfunction, are often present.
  • Cerebral NSM presents as a slow-expanding intracranial tumor-like lesion.
  • The diagnosis of spinal cord NSM is based on clinical evidence whereas the cerebral disease is usually diagnosed by biopsy of the nervous tissue.

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  • (PMID = 18499080.001).
  • [ISSN] 1873-6254
  • [Journal-full-title] Acta tropica
  • [ISO-abbreviation] Acta Trop.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] Netherlands
  • [Number-of-references] 55
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97. Ponce-Camacho MA, Diaz de Leon-Medina R, Miranda-Maldonado I, Garza-Guajardo R, Hernandez-Salazar J, Barboza-Quintana O: A 5-year-old girl with a congenital ganglioneuroma diagnosed by fine needle aspiration biopsy: a case report. Cytojournal; 2008;5:5

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • INTRODUCTION: Ganglioneuroma is a rare, benign, neuroblastic tumor arising mainly from the central or peripheral autonomic nervous system, especially the sympathetic system.
  • A diagnosis of ganglioneuroma was suggested.
  • Core biopsy and surgical resection confirmed this diagnosis.
  • CONCLUSION: Congenital ganglioneuroma of the cervical region is an uncommon soft tissue benign neoplasm of neuroblastic origin, and it should be considered in the differential diagnosis of head and neck pediatric soft tissue tumors.

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  • (PMID = 18373875.001).
  • [ISSN] 1742-6413
  • [Journal-full-title] CytoJournal
  • [ISO-abbreviation] Cytojournal
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC2359765
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98. Aragno M, Mastrocola R, Medana C, Catalano MG, Vercellinatto I, Danni O, Boccuzzi G: Oxidative stress-dependent impairment of cardiac-specific transcription factors in experimental diabetes. Endocrinology; 2006 Dec;147(12):5967-74
The Lens. Cited by Patents in .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • DHEA treatment, by preventing the activation of the oxidative pathways induced by hyperglycemia, counteracted the enhanced AGE receptor activation in the heart of STZ-diabetic rats and ZDF rats and normalized downstream signaling, thus avoiding impairment of the cardiac myogenic factors, heart autonomic nervous system and neural crest derivatives (HAND) and myogenic enhancer factor-2, and the switch in MHC gene expression, which are the early events in diabetic cardiomyopathy.
  • [MeSH-minor] Advanced Glycosylation End Product-Specific Receptor. Animals. Dehydroepiandrosterone / pharmacology. Glycosylation End Products, Advanced / blood. Heart / drug effects. Heart Ventricles / chemistry. Heart Ventricles / drug effects. Heart Ventricles / metabolism. Male. Myosin Heavy Chains / metabolism. Organ Specificity. Rats. Rats, Wistar. Rats, Zucker. Receptors, Immunologic / analysis. Receptors, Tumor Necrosis Factor, Type I / metabolism. Tumor Necrosis Factor-alpha / metabolism

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  • (PMID = 16935841.001).
  • [ISSN] 0013-7227
  • [Journal-full-title] Endocrinology
  • [ISO-abbreviation] Endocrinology
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Advanced Glycosylation End Product-Specific Receptor; 0 / Bmyo protein, rat; 0 / Glycosylation End Products, Advanced; 0 / Receptors, Immunologic; 0 / Receptors, Tumor Necrosis Factor, Type I; 0 / Transcription Factors; 0 / Tumor Necrosis Factor-alpha; 459AG36T1B / Dehydroepiandrosterone; EC 3.6.4.1 / Myosin Heavy Chains
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99. Straburzyńska-Migaj E, Ochotny R, Wachowiak-Baszyńska A, Straburzyńska-Lupa A, Leśniewska K, Wiktorowicz K, Cieśliński A: Cytokines and heart rate variability in patients with chronic heart failure. Kardiol Pol; 2005 Nov;63(5):478-85; discussion 486-7
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • INTRODUCTION: Heart rate variability (HRV) analysis is a non-invasive method of assessment of the autonomic nervous system's effects on heart function.
  • Because these molecules are believed to influence the nervous system at both the central and peripheral levels, their potential role in HRV reduction in the course of CHF has been proposed.
  • [MeSH-minor] Adult. Female. Humans. Interleukin-6 / blood. Male. Middle Aged. Receptors, Tumor Necrosis Factor, Type I / blood. Receptors, Tumor Necrosis Factor, Type II / blood. Statistics, Nonparametric. Tumor Necrosis Factor-alpha / metabolism

  • MedlinePlus Health Information. consumer health - Heart Failure.
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  • (PMID = 16362851.001).
  • [ISSN] 0022-9032
  • [Journal-full-title] Kardiologia polska
  • [ISO-abbreviation] Kardiol Pol
  • [Language] eng
  • [Publication-type] Clinical Trial; Journal Article
  • [Publication-country] Poland
  • [Chemical-registry-number] 0 / Cytokines; 0 / Interleukin-6; 0 / Receptors, Tumor Necrosis Factor, Type I; 0 / Receptors, Tumor Necrosis Factor, Type II; 0 / Tumor Necrosis Factor-alpha
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100. Luyer MD, Greve JW, Hadfoune M, Jacobs JA, Dejong CH, Buurman WA: Nutritional stimulation of cholecystokinin receptors inhibits inflammation via the vagus nerve. J Exp Med; 2005 Oct 17;202(8):1023-9
The Lens. Cited by Patents in .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The immune system in vertebrates senses exogenous and endogenous danger signals by way of complex cellular and humoral processes, and responds with an inflammatory reaction to combat putative attacks.
  • The autonomic nervous system plays an important role in sensing luminal contents in the gut by way of hard-wired connections and chemical messengers, such as cholecystokinin (CCK).
  • Vagotomy and administration of antagonists for CCK and nicotinic receptors significantly blunted the inhibitory effect of high-fat enteral nutrition on hemorrhagic shock-induced tumor necrosis factor-alpha and interleukin-6 release (P < 0.05).
  • [MeSH-minor] Animals. Cytokines / blood. Enzyme-Linked Immunosorbent Assay. Interleukin-6 / metabolism. Male. Models, Biological. Rats. Rats, Sprague-Dawley. Receptors, Nicotinic / metabolism. Tumor Necrosis Factor-alpha / metabolism






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