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1. Low PA, Vernino S, Suarez G: Autonomic dysfunction in peripheral nerve disease. Muscle Nerve; 2003 Jun;27(6):646-61
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Autonomic dysfunction in peripheral nerve disease.
  • Autonomic neuropathies are inherited or acquired neuropathies in which autonomic nerve fibers are selectively or disproportionately affected.
  • Acquired cases can be autoimmune; due to diabetes, amyloidosis, drugs, or toxins; or idiopathic.
  • Autoimmune autonomic neuropathy is often subacute, sometimes associated with a neoplasm, and associated with high titers of antibody to ganglionic nicotinic acetylcholine receptor in about half of the severe cases.
  • The molecular basis of inherited autonomic neuropathies is better known, including recent identification of the loci and genes of hereditary sensory and autonomic neuropathies types I, III, and IV.
  • The inherited amyloid neuropathies are due to mutations of three proteins: transthyretin, apolipoprotein A1, and gelsolin.
  • Non-invasive autonomic testing complements clinical and electrophysiological characterization of the autonomic neuropathies.
  • [MeSH-major] Autonomic Nervous System / immunology. Autonomic Nervous System Diseases / immunology. Peripheral Nervous System Diseases / immunology
  • [MeSH-minor] Amyloid Neuropathies / pathology. Amyloid Neuropathies / physiopathology. Autoimmune Diseases of the Nervous System / immunology. Autoimmune Diseases of the Nervous System / pathology. Autoimmune Diseases of the Nervous System / therapy. Diabetic Neuropathies / pathology. Diabetic Neuropathies / physiopathology. Humans. Neurotoxins / adverse effects


2. Masaki T, Takayama M, Matsuoka H, Abe N, Ueki H, Sugiyama M, Tonari A, Kusuda J, Mizumoto S, Atomi Y: Intraoperative radiotherapy for oncological and function-preserving surgery in patients with advanced lower rectal cancer. Langenbecks Arch Surg; 2008 Mar;393(2):173-80
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • BACKGROUND: Pelvic autonomic nerve preservation (PANP) with lateral lymph node dissection (LLND) has been introduced in rectal cancer surgery in Japan; however, its indication has not been standardized yet.
  • In the long term, medication for urination was necessitated in four patients in the control group, whereas in none in the IORT group (p = 0.059).
  • [MeSH-major] Autonomic Nervous System / surgery. Intraoperative Care. Lymph Node Excision. Microsurgery. Pelvis / innervation. Rectal Neoplasms / radiotherapy. Rectal Neoplasms / surgery. Rectum / innervation
  • [MeSH-minor] Aged. Combined Modality Therapy. Disease-Free Survival. Female. Humans. Japan. Kaplan-Meier Estimate. Lymphatic Metastasis / pathology. Male. Middle Aged. Neoplasm Recurrence, Local / etiology. Neoplasm Recurrence, Local / mortality. Neoplasm Staging. Postoperative Complications / etiology. Postoperative Complications / mortality. Prognosis. Radiotherapy, Adjuvant. Risk Factors. Urinary Catheterization. Urination Disorders / etiology

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  • (PMID = 18172677.001).
  • [ISSN] 1435-2451
  • [Journal-full-title] Langenbeck's archives of surgery
  • [ISO-abbreviation] Langenbecks Arch Surg
  • [Language] eng
  • [Publication-type] Journal Article; Randomized Controlled Trial
  • [Publication-country] Germany
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3. Sharifi AM, Mousavi SH, Farhadi M, Larijani B: Study of high glucose-induced apoptosis in PC12 cells: role of bax protein. J Pharmacol Sci; 2007 Jul;104(3):258-62
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Diabetic neuropathies, affecting the autonomic, sensory, and motor peripheral nervous system, are among the most frequent complications of diabetes.
  • [MeSH-major] Apoptosis / drug effects. Glucose / pharmacology. bcl-2-Associated X Protein / metabolism
  • [MeSH-minor] Animals. Blotting, Western. Cell Survival / drug effects. DNA Fragmentation / drug effects. DNA, Neoplasm / drug effects. DNA, Neoplasm / genetics. PC12 Cells. Proto-Oncogene Proteins c-bcl-2 / metabolism. Rats. Tetrazolium Salts. Thiazoles

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  • (PMID = 17652910.001).
  • [ISSN] 1347-8613
  • [Journal-full-title] Journal of pharmacological sciences
  • [ISO-abbreviation] J. Pharmacol. Sci.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / DNA, Neoplasm; 0 / Proto-Oncogene Proteins c-bcl-2; 0 / Tetrazolium Salts; 0 / Thiazoles; 0 / bcl-2-Associated X Protein; 298-93-1 / thiazolyl blue; IY9XDZ35W2 / Glucose
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4. Dorobisz AT, Zaleska P: [Head and neck paraganglioma--neoplasm of nervous origin]. Pol Merkur Lekarski; 2004 Sep;17(99):292-6
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  • [Title] [Head and neck paraganglioma--neoplasm of nervous origin].
  • Paragangliomas are relatively rarely occurring tumors, stemming from the autonomic nervous system.
  • Not large size of the tumor contributes to the late diagnosis which is most regrettable due to its potential malignancy (10%).
  • Diagnostic and therapeutic problems cause it's medication reasonable only in specialist centers.
  • During the previous years many centers carried a preoperative embolization of a tumor during angiographies.
  • An alternative to the large tumor vessels obliteration may be a radium therapy, although several reports confirm its low radiosensitivity.
  • [MeSH-major] Head and Neck Neoplasms / pathology. Nervous System Neoplasms / pathology. Paraganglioma / pathology

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  • (PMID = 15628063.001).
  • [ISSN] 1426-9686
  • [Journal-full-title] Polski merkuriusz lekarski : organ Polskiego Towarzystwa Lekarskiego
  • [ISO-abbreviation] Pol. Merkur. Lekarski
  • [Language] pol
  • [Publication-type] English Abstract; Journal Article; Review
  • [Publication-country] Poland
  • [Number-of-references] 23
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5. Oh SJ, Hong YK, Song EK: Paroxysmal autonomic dysregulation with fever that was controlled by propranolol in a brain neoplasm patient. Korean J Intern Med; 2007 Mar;22(1):51-4
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  • [Title] Paroxysmal autonomic dysregulation with fever that was controlled by propranolol in a brain neoplasm patient.
  • Paroxysmal persistent hyperthermia after sudden mental change or neurologic deficit can develop via autonomic dysregulation without infection or any other causes of fever.
  • Paroxysmal hyperthermic autonomic dysregulation is a rare disease entity.
  • It manifests as a form of paroxysmal hypertension, fever, tachycardia, tachypnea, pupillary dilation, agitation and extensor posturing after traumatic brain injury, hydrocephalus, brain hemorrhage or brain neoplasm.
  • We recently experienced a case of paroxysmal hyperthermia following intracerebral hemorrhage along with brain neoplasm.
  • Empirical treatments with antibiotics, antipyretics, morphine, steroid and antiepileptic agents were also ineffective.
  • Propranolol is one of the most effective drugs for treating paroxysmal hyperthermia that is due to autonomic dysregulation.
  • [MeSH-major] Adrenergic beta-Antagonists / therapeutic use. Autonomic Nervous System Diseases / drug therapy. Brain Neoplasms / complications. Cerebral Hemorrhage / complications. Fever / drug therapy. Propranolol / therapeutic use

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  • (PMID = 17427648.001).
  • [ISSN] 1226-3303
  • [Journal-full-title] The Korean journal of internal medicine
  • [ISO-abbreviation] Korean J. Intern. Med.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Korea (South)
  • [Chemical-registry-number] 0 / Adrenergic beta-Antagonists; 9Y8NXQ24VQ / Propranolol
  • [Other-IDs] NLM/ PMC2687607
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6. Gouider-Khouja N, Mekaouar A, Larnaout A, Miladi N, Ben Khelifa F, Hentati F: Progressive encephalomyelitis with rigidity presenting as a stiff-person syndrome. Parkinsonism Relat Disord; 2002 Mar;8(4):285-8
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  • Diagnosis criteria of stiff-person syndrome (SPS) include progressive, fluctuating muscular rigidity and spasms with normal neurological examination.
  • The presence of unusual features such as prominent limb rigidity with segmental signs and contracture, evidence of brainstem dysfunction, profound autonomic disturbances, CSF pleiocytosis or MRI abnormalities in patients with SPS presentation allows to classify these patients as progressive encephalomyelitis with rigidity (PER).
  • Search for profound neoplasm was negative.
  • The initial clinical presentation mimicking a SPS with subsequent diffuse involvement of the central nervous system and a striking localisation of a severe rigidity to one arm allowed to suspect the diagnosis of PER.
  • [MeSH-major] Encephalomyelitis / complications. Encephalomyelitis / diagnosis. Muscle Rigidity / diagnosis. Muscle Rigidity / etiology. Stiff-Person Syndrome / diagnosis
  • [MeSH-minor] Anti-Inflammatory Agents / therapeutic use. Baclofen / therapeutic use. Diagnosis, Differential. Diazepam / therapeutic use. Drug Therapy, Combination. Electromyography. Evoked Potentials, Somatosensory. Female. Humans. Methylprednisolone / therapeutic use. Middle Aged. Muscle Relaxants, Central / therapeutic use

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  • (PMID = 12039424.001).
  • [ISSN] 1353-8020
  • [Journal-full-title] Parkinsonism & related disorders
  • [ISO-abbreviation] Parkinsonism Relat. Disord.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Anti-Inflammatory Agents; 0 / Muscle Relaxants, Central; H789N3FKE8 / Baclofen; Q3JTX2Q7TU / Diazepam; X4W7ZR7023 / Methylprednisolone
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7. Pasha SF, Lunsford TN, Lennon VA: Autoimmune gastrointestinal dysmotility treated successfully with pyridostigmine. Gastroenterology; 2006 Nov;131(5):1592-6
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • BACKGROUND & AIMS: Autoimmune gastrointestinal dysmotility (AGID) is a limited form of autoimmune autonomic neuropathy occurring idiopathically or in a paraneoplastic context.
  • We describe the diagnosis and management of an instructive case.
  • Paraneoplastic autoantibody evaluation aided the diagnosis of AGID.
  • RESULTS: Serologic testing revealed both ganglionic neuronal acetylcholine receptor and N-type voltage-gated calcium channel autoantibodies.
  • An underlying neoplasm was excluded by appropriate investigations.
  • CONCLUSIONS: Autoimmune serology is a valuable adjunct to the diagnosis and guide to management of patients with AGID.

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  • (PMID = 17101331.001).
  • [ISSN] 0016-5085
  • [Journal-full-title] Gastroenterology
  • [ISO-abbreviation] Gastroenterology
  • [Language] ENG
  • [Grant] United States / NIDDK NIH HHS / DK / P01 DK068055; United States / NIDDK NIH HHS / DK / P01-DK68055
  • [Publication-type] Case Reports; Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Autoantibodies; 0 / Cholinesterase Inhibitors; KVI301NA53 / Pyridostigmine Bromide
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8. Kaestner F, Mostert C, Behnken A, Boeckermann I, Ternes F, Diedrich M, Zavorotnyy M, Arolt V, Weckesser M, Rothermundt M: Therapeutic strategies for catatonia in paraneoplastic encephalitis. World J Biol Psychiatry; 2008;9(3):236-40
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  • This report is about a 40-year-old man suffering from fluctuating catatonia as main symptom of long-lasting paraneoplastic encephalitis caused by a testicular neoplasm.
  • With recurrence of a neoplasm initially diagnosed as seminoma after a 7-year symptom-free interval the patient suddenly developed various neurological and psychopathological symptoms including seizures, autonomic dysregulation, continuous anterograde short-term amnesia and predominantly a long-lasting complex catatonic syndrome with on-off phenomena.
  • Haloperidol and lorazepam effectively relieved catatonic symptoms in contrast to various atypical antipsychotic drugs and diazepam.
  • Further neoplasm recurrences, however, reinforced catatonia until the tumour was successfully treated and the patient fully recovered.
  • [MeSH-major] Catatonia / complications. Catatonia / therapy. Electroconvulsive Therapy / methods. Paraneoplastic Syndromes, Nervous System / complications. Paraneoplastic Syndromes, Nervous System / pathology
  • [MeSH-minor] Adult. Electroencephalography. Humans. Magnetic Resonance Imaging. Male. Plasmapheresis. Testicular Neoplasms / pathology

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  • (PMID = 17853266.001).
  • [ISSN] 1562-2975
  • [Journal-full-title] The world journal of biological psychiatry : the official journal of the World Federation of Societies of Biological Psychiatry
  • [ISO-abbreviation] World J. Biol. Psychiatry
  • [Language] eng
  • [Publication-type] Case Reports; Letter
  • [Publication-country] Scotland
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9. Renton KW, Nicholson TE: Hepatic and central nervous system cytochrome P450 are down-regulated during lipopolysaccharide-evoked localized inflammation in brain. J Pharmacol Exp Ther; 2000 Aug;294(2):524-30
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Hepatic and central nervous system cytochrome P450 are down-regulated during lipopolysaccharide-evoked localized inflammation in brain.
  • The effect of central nervous system inflammation on the levels and activity of hepatic and brain cytochrome P450 were examined in the rat.
  • Similar i.p. doses of LPS had no effect on hepatic cytochrome P450, indicating that the hepatic effect was not caused by LPS leakage from the central nervous system.
  • Tumor necrosis factor-alpha given i.c.v. depressed EROD activity in the brain but this was not accompanied by a concomitant loss in the liver.
  • Hepatic EROD did respond to the i.p. injection of tumor necrosis factor-alpha.
  • This study demonstrates that localized inflammatory responses in the brain cause a concomitant down-regulation of cytochrome P450 and drug-metabolizing activity in the liver and the brain.
  • [MeSH-major] Brain / enzymology. Cytochrome P-450 Enzyme System / metabolism. Encephalitis / enzymology. Heat-Shock Proteins. Lipopolysaccharides / toxicity. Liver / enzymology
  • [MeSH-minor] Adrenergic beta-Antagonists / pharmacology. Animals. Autonomic Nerve Block. Cytochrome P-450 CYP1A1 / metabolism. Cytochrome P-450 Enzyme Inhibitors. Down-Regulation. HSP27 Heat-Shock Proteins. Immunohistochemistry. Male. Membrane Proteins / metabolism. Membranes / enzymology. Membranes / metabolism. Neoplasm Proteins / metabolism. Nerve Tissue Proteins / metabolism. Propranolol / pharmacology. Rats. Rats, Sprague-Dawley

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  • (PMID = 10900228.001).
  • [ISSN] 0022-3565
  • [Journal-full-title] The Journal of pharmacology and experimental therapeutics
  • [ISO-abbreviation] J. Pharmacol. Exp. Ther.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] UNITED STATES
  • [Chemical-registry-number] 0 / Adrenergic beta-Antagonists; 0 / Cytochrome P-450 Enzyme Inhibitors; 0 / HSP27 Heat-Shock Proteins; 0 / Heat-Shock Proteins; 0 / Hspb1 protein, rat; 0 / Lipopolysaccharides; 0 / Membrane Proteins; 0 / Neoplasm Proteins; 0 / Nerve Tissue Proteins; 9035-51-2 / Cytochrome P-450 Enzyme System; 9Y8NXQ24VQ / Propranolol; EC 1.14.14.1 / Cytochrome P-450 CYP1A1
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10. Lee L, Niu H, Rueger R, Igawa Y, Deutsch J, Ishii N, Mu S, Sakamoto Y, Busse-Reid R, Gimmi C, Goelzer P, De Schepper S, Yoshimura Y, Barrett J, Ishikawa Y, Weissgerber G, Peck R: The safety, tolerability, pharmacokinetics, and pharmacodynamics of single oral doses of CH4987655 in healthy volunteers: target suppression using a biomarker. Clin Cancer Res; 2009 Dec 1;15(23):7368-74
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  • It potently inhibits mitogen-activated protein kinase signaling pathway activation and tumor cell growth, with an in vitro IC(50) of 5.2 nmol/L for inhibition of MEK1/2.
  • Single-agent oral administration of CH4987655 resulted in complete tumor regressions in xenograft models.
  • Moderate adverse events were experienced by one subject at 1 mg (autonomic nervous system imbalance) and three subjects at 4 mg (diarrhea, abdominal pain, autonomic nervous system and acne).
  • [MeSH-minor] Adolescent. Adult. Antineoplastic Agents. Area Under Curve. Biomarkers, Tumor / metabolism. Dose-Response Relationship, Drug. Double-Blind Method. Female. Humans. Inhibitory Concentration 50. MAP Kinase Signaling System. Male. Middle Aged. Neoplasm Transplantation. Placebos

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  • (PMID = 19934286.001).
  • [ISSN] 1078-0432
  • [Journal-full-title] Clinical cancer research : an official journal of the American Association for Cancer Research
  • [ISO-abbreviation] Clin. Cancer Res.
  • [Language] eng
  • [Publication-type] Journal Article; Randomized Controlled Trial
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antineoplastic Agents; 0 / Biomarkers; 0 / Biomarkers, Tumor; 0 / Enzyme Inhibitors; 0 / Placebos
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11. Marzocchi N, Cainazzo MM, Catellani D, Pini LA: A case of a GH-producing pituitary adenoma associated with a unilateral headache with autonomic signs. J Headache Pain; 2005 Jun;6(3):152-5
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  • [Title] A case of a GH-producing pituitary adenoma associated with a unilateral headache with autonomic signs.
  • A 66-year-old man suffered from a drug-resistant, left-sided headache with autonomic signs, triggered by the supine position.
  • [MeSH-major] Autonomic Nervous System Diseases / etiology. Growth Hormone-Secreting Pituitary Adenoma / complications. Headache / complications
  • [MeSH-minor] Bromhexine. Diagnosis, Differential. Disease Progression. Fatal Outcome. Growth Hormone / secretion. Humans. Hypophysectomy. Insulin-Like Growth Factor I / metabolism. Magnetic Resonance Imaging. Male. Middle Aged. Myocardial Infarction. Neoplasm Recurrence, Local / complications. Neoplasm Recurrence, Local / pathology. Neoplasm Recurrence, Local / secretion. Pituitary Gland, Anterior / pathology. Pituitary Gland, Anterior / radiography. Pituitary Gland, Anterior / secretion. Sella Turcica / pathology. Sella Turcica / physiopathology. Treatment Outcome

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  • (PMID = 16355297.001).
  • [ISSN] 1129-2369
  • [Journal-full-title] The journal of headache and pain
  • [ISO-abbreviation] J Headache Pain
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Italy
  • [Chemical-registry-number] 67763-96-6 / Insulin-Like Growth Factor I; 9002-72-6 / Growth Hormone; Q1J152VB1P / Bromhexine
  • [Other-IDs] NLM/ PMC3451631
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