[X] Close
You are about to erase all the values you have customized, search history, page format, etc.
Click here to RESET all values       Click here to GO BACK without resetting any value
Items 1 to 100 of about 1270
31. van Bodegom PM, Sorrell BK, Oosthoek A, Bakker C, Aerts R: Separating the effects of partial submergence and soil oxygen demand on plant physiology. Ecology; 2008 Jan;89(1):193-204
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Increased SOD increased root oxygen deprivation, as indicated by either raised porosity or increased alcohol dehydrogenase (ADH) activity in roots of flood-intolerant species (Calamagrostis epigejos and Carex arenaria).
  • In contrast, the most flood-tolerant species (Juncus subnodulosus and Typha latifolia) were unaffected by any treatment and maintained high internal oxygen concentrations at the shoot : root junction and low root ADH activity in all treatments.

  • Hazardous Substances Data Bank. Water .
  • Hazardous Substances Data Bank. OXYGEN .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 18376561.001).
  • [ISSN] 0012-9658
  • [Journal-full-title] Ecology
  • [ISO-abbreviation] Ecology
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Soil; 059QF0KO0R / Water; EC 1.1.1.1 / Alcohol Dehydrogenase; S88TT14065 / Oxygen
  •  go-up   go-down


32. Terry MB, Gammon MD, Zhang FF, Vaughan TL, Chow WH, Risch HA, Schoenberg JB, Mayne ST, Stanford JL, West AB, Rotterdam H, Blot WJ, Fraumeni JF Jr, Santella RM: Alcohol dehydrogenase 3 and risk of esophageal and gastric adenocarcinomas. Cancer Causes Control; 2007 Nov;18(9):1039-46
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • RESULTS: Individuals homozygous for ADH ( 3 ) (1-1) had a higher risk of each tumor type compared to individuals who had ADH ( 3 ) (2-2) or ADH ( 3 ) (1-2) genotype (OR = 1.7, 95% CI = 1.0-2.9 for esophageal and gastric cardia adenocarcinomas; OR = 1.7, 95% CI = 0.7-4.3 for esophageal squamous cell carcinoma; and OR = 2.8, 95% CI = 1.5-5.1 for non-cardia gastric cancer).
  • The elevation in risk from homozygosity of the ADH ( 3 ) (1) allele was seen in drinkers and nondrinkers, although the risk estimate was only significant for drinkers, particularly of liquor.

  • MedlinePlus Health Information. consumer health - Esophageal Cancer.
  • MedlinePlus Health Information. consumer health - Stomach Cancer.
  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17665311.001).
  • [ISSN] 0957-5243
  • [Journal-full-title] Cancer causes & control : CCC
  • [ISO-abbreviation] Cancer Causes Control
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / U01-CA57923; United States / NCI NIH HHS / CA / U01-CA57983; United States / NCI NIH HHS / CN / N01-CN05230; United States / NCI NIH HHS / CA / U01-CA57949; United States / NCI NIH HHS / CP / N02-CP40501; United States / NIEHS NIH HHS / ES / P30-ES09089; United States / NIEHS NIH HHS / ES / P30-ES10126; United States / NIEHS NIH HHS / ES / P30 ES009089
  • [Publication-type] Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  • [Chemical-registry-number] EC 1.1.1.1 / ADH1C protein, human; EC 1.1.1.1 / Alcohol Dehydrogenase
  •  go-up   go-down


33. Macgregor S, Lind PA, Bucholz KK, Hansell NK, Madden PA, Richter MM, Montgomery GW, Martin NG, Heath AC, Whitfield JB: Associations of ADH and ALDH2 gene variation with self report alcohol reactions, consumption and dependence: an integrated analysis. Hum Mol Genet; 2009 Feb 1;18(3):580-93
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Associations of ADH and ALDH2 gene variation with self report alcohol reactions, consumption and dependence: an integrated analysis.
  • Alcohol dependence (AD) is a complex disorder with environmental and genetic origins.
  • This study tested for associations between nine polymorphisms in ALDH2 and 41 in the seven ADH genes, and alcohol-related flushing, alcohol use and dependence symptom scores in 4597 Australian twins.

  • Genetic Alliance. consumer health - Alcohol dependence.
  • MedlinePlus Health Information. consumer health - Alcoholism and Alcohol Abuse.
  • COS Scholar Universe. author profiles.
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • SciCrunch. OMIM: Data: Gene Annotation .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Pharmacogenomics J. 2008 Jun;8(3):220-7 [17923853.001]
  • [Cites] Twin Res Hum Genet. 2008 Jun;11(3):287-305 [18498207.001]
  • [Cites] Alcohol Clin Exp Res. 1997 Jun;21(4):596-601 [9194910.001]
  • [Cites] Psychol Med. 1997 Nov;27(6):1381-96 [9403910.001]
  • [Cites] J Stud Alcohol. 1998 Mar;59(2):133-9 [9500299.001]
  • [Cites] J Abnorm Psychol. 1998 Aug;107(3):363-74 [9715572.001]
  • [Cites] Alcohol Clin Exp Res. 1998 Aug;22(5):1048-52 [9726271.001]
  • [Cites] Alcohol Clin Exp Res. 1998 Oct;22(7):1463-9 [9802529.001]
  • [Cites] Biol Psychiatry. 1999 Mar 1;45(5):518-21 [10088041.001]
  • [Cites] Biol Psychiatry. 1999 Mar 1;45(5):522-32 [10088042.001]
  • [Cites] Psychol Med. 1999 Sep;29(5):1069-81 [10576299.001]
  • [Cites] Addiction. 1999 Sep;94(9):1361-70 [10615721.001]
  • [Cites] Am J Hum Genet. 2000 Jan;66(1):279-92 [10631157.001]
  • [Cites] Hepatology. 2000 Apr;31(4):984-9 [10733556.001]
  • [Cites] Eur J Hum Genet. 2000 Jul;8(7):545-51 [10909856.001]
  • [Cites] Am J Med Genet. 2000 Oct 9;96(5):632-7 [11054770.001]
  • [Cites] J Stud Alcohol. 2000 Nov;61(6):827-35 [11188488.001]
  • [Cites] Behav Genet. 2001 Mar;31(2):231-9 [11545539.001]
  • [Cites] Alcohol Clin Exp Res. 2001 Sep;25(9):1257-63 [11584143.001]
  • [Cites] Alcohol Clin Exp Res. 2001 Sep;25(9):1264-9 [11584144.001]
  • [Cites] Nat Genet. 2002 Jan;30(1):97-101 [11731797.001]
  • [Cites] Alcohol Clin Exp Res. 2001 Dec;25(12):1773-7 [11781511.001]
  • [Cites] Am J Hum Genet. 2002 Jul;71(1):84-99 [12050823.001]
  • [Cites] Am J Hum Genet. 2002 Nov;71(5):1247-50; author reply 1250-1 [12452180.001]
  • [Cites] Alcohol Clin Exp Res. 2004 Jan;28(1):10-4 [14745297.001]
  • [Cites] Science. 1972 Jan 28;175(4020):449-50 [5007912.001]
  • [Cites] Behav Genet. 1985 Jul;15(4):305-47 [4041178.001]
  • [Cites] Hepatology. 1986 May-Jun;6(3):502-10 [3519419.001]
  • [Cites] Eur J Biochem. 1986 Sep 1;159(2):215-8 [3758060.001]
  • [Cites] Hum Genet. 1989 Apr;82(1):14-6 [2714775.001]
  • [Cites] Arch Gen Psychiatry. 1991 Jan;48(1):19-28 [1984758.001]
  • [Cites] Arukoru Kenkyuto Yakubutsu Ison. 1993 Feb;28(1):13-25 [8512495.001]
  • [Cites] Arch Gen Psychiatry. 1993 Sep;50(9):690-8 [8357294.001]
  • [Cites] Hepatology. 1994 Feb;19(2):360-6 [7904979.001]
  • [Cites] J Stud Alcohol. 1994 Mar;55(2):149-58 [8189735.001]
  • [Cites] Alcohol Clin Exp Res. 1994 Jun;18(3):640-3 [7943668.001]
  • [Cites] Am J Psychiatry. 1995 Aug;152(8):1219-21 [7625477.001]
  • [Cites] Alcohol Clin Exp Res. 1995 Jun;19(3):582-92 [7573778.001]
  • [Cites] J Clin Invest. 1995 Nov;96(5):2180-6 [7593603.001]
  • [Cites] Alcohol Clin Exp Res. 1996 Feb;20(1):81-6 [8651467.001]
  • [Cites] Br J Psychiatry. 1996 Jun;168(6):762-7 [8773821.001]
  • [Cites] Hum Genet. 1996 Apr;97(4):409-13 [8834233.001]
  • [Cites] J Clin Invest. 1996 Nov 1;98(9):2027-32 [8903321.001]
  • [Cites] Alcohol Alcohol Suppl. 1994;2:113-9 [8974324.001]
  • [Cites] Biol Psychiatry. 1997 Mar 15;41(6):703-9 [9066994.001]
  • [Cites] Am J Hum Genet. 1999 Apr;64(4):1147-57 [10090900.001]
  • [Cites] Am J Hum Genet. 1999 Sep;65(3):795-807 [10441588.001]
  • [Cites] Bioinformatics. 2005 Jan 15;21(2):263-5 [15297300.001]
  • [Cites] Psychol Med. 2004 Nov;34(8):1519-30 [15724882.001]
  • [Cites] Am J Psychiatry. 2005 May;162(5):1005-7 [15863808.001]
  • [Cites] J Stud Alcohol. 2005 Mar;66(2):196-204 [15957670.001]
  • [Cites] Am J Cardiol. 2005 Jul 15;96(2):227-32 [16018848.001]
  • [Cites] Bioinformatics. 2005 Aug 15;21(16):3445-7 [15947021.001]
  • [Cites] Ther Drug Monit. 2005 Dec;27(6):700-3 [16404797.001]
  • [Cites] Curr Psychiatry Rep. 2006 Apr;8(2):151-7 [16539893.001]
  • [Cites] Twin Res Hum Genet. 2006 Apr;9(2):194-7 [16611487.001]
  • [Cites] Hum Mol Genet. 2006 May 1;15(9):1539-49 [16571603.001]
  • [Cites] Neuropsychopharmacology. 2006 May;31(5):1085-95 [16237392.001]
  • [Cites] Am J Hum Genet. 2006 Jun;78(6):973-87 [16685648.001]
  • [Cites] Cancer Epidemiol Biomarkers Prev. 2006 May;15(5):1009-13 [16702384.001]
  • [Cites] Alcohol Clin Exp Res. 2006 Jul;30(7):1093-100 [16792555.001]
  • [Cites] Alcohol Clin Exp Res. 2006 Sep;30(9):1470-8 [16930209.001]
  • [Cites] Alcohol Clin Exp Res. 2007 Feb;31(2):216-20 [17250612.001]
  • [Cites] Hum Mutat. 2007 May;28(5):506-10 [17285601.001]
  • [Cites] Alcohol Clin Exp Res. 2007 Aug;31(8):1297-301 [17559546.001]
  • [Cites] Alcohol Clin Exp Res. 2008 May;32(5):785-95 [18331377.001]
  • (PMID = 18996923.001).
  • [ISSN] 1460-2083
  • [Journal-full-title] Human molecular genetics
  • [ISO-abbreviation] Hum. Mol. Genet.
  • [Language] ENG
  • [Grant] United States / NIAAA NIH HHS / AA / AA13320; United States / NIAAA NIH HHS / AA / AA13321; United States / NIAAA NIH HHS / AA / AA013326; United States / NIAAA NIH HHS / AA / AA07728; United States / NIAAA NIH HHS / AA / AA11998; United States / NIAAA NIH HHS / AA / AA014041; United States / NIAAA NIH HHS / AA / AA07535
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Twin Study
  • [Publication-country] England
  • [Chemical-registry-number] EC 1.1.1.1 / ADH1B protein, human; EC 1.1.1.1 / Alcohol Dehydrogenase; EC 1.2.1.3 / ALDH2 protein, human; EC 1.2.1.3 / Aldehyde Dehydrogenase
  • [Other-IDs] NLM/ PMC2722191
  •  go-up   go-down


3
Advertisement
4. Haris A, Demeter P, Tóth I, Polner K: [Severe hyponatremia and comatose state during colonoscopy preparation]. Orv Hetil; 2010 May 16;151(20):828-31
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Hyponatremia was likely induced by stress provoked ADH secretion, due to the patients' fear for bowel cleansing and colonoscopy, and by large fluid intake consumed as "misunderstanding" of the instructions.
  • [MeSH-major] Colonoscopy. Coma / etiology. Drinking. Hyponatremia / etiology. Inappropriate ADH Syndrome / etiology

  • MedlinePlus Health Information. consumer health - Colonoscopy.
  • MedlinePlus Health Information. consumer health - Coma.
  • Hazardous Substances Data Bank. POTASSIUM, ELEMENTAL .
  • Hazardous Substances Data Bank. SODIUM .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 20442054.001).
  • [ISSN] 0030-6002
  • [Journal-full-title] Orvosi hetilap
  • [ISO-abbreviation] Orv Hetil
  • [Language] hun
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Hungary
  • [Chemical-registry-number] 0 / Laxatives; 0 / Phosphates; 9NEZ333N27 / Sodium; RWP5GA015D / Potassium; SE337SVY37 / sodium phosphate
  •  go-up   go-down


35. Ghosh S, Bierut LJ, Porjesz B, Edenberg HJ, Dick D, Goate A, Hesselbrock V, Nurnberger J Jr, Foroud T, Kramer J, Rice J, Begleiter H: A novel non-parametric regression reveals linkage on chromosome 4 for the number of externalizing symptoms in sib-pairs. Am J Med Genet B Neuropsychiatr Genet; 2008 Oct 5;147B(7):1301-5
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • We report a statistically significant linkage close to the ADH gene cluster on Chromosome 4.

  • MedlinePlus Health Information. consumer health - Alcoholism and Alcohol Abuse.
  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Addiction. 1999 Sep;94(9):1361-70 [10615721.001]
  • [Cites] Alcohol Clin Exp Res. 1998 Dec;22(9):2035-42 [9884148.001]
  • [Cites] Addiction. 2000 Apr;95(4):553-67 [10829331.001]
  • [Cites] Alcohol Clin Exp Res. 2000 Jul;24(7):933-45 [10923994.001]
  • [Cites] Am J Hum Genet. 1999 Oct;65(4):1148-60 [10486334.001]
  • [Cites] Am J Med Genet. 2000 Oct 9;96(5):632-7 [11054770.001]
  • [Cites] Am J Hum Genet. 2001 Jan;68(1):128-135 [11102287.001]
  • [Cites] Am J Psychiatry. 2001 May;158(5):718-24 [11329392.001]
  • [Cites] Am J Hum Genet. 2002 Feb;70(2):461-71 [11791213.001]
  • [Cites] Proc Natl Acad Sci U S A. 2002 Mar 19;99(6):3729-33 [11891318.001]
  • [Cites] Ann Hum Genet. 2002 Mar;66(Pt 2):169-82 [12174220.001]
  • [Cites] Biol Psychol. 2002 Oct;61(1-2):229-48 [12385677.001]
  • [Cites] Am J Med Genet B Neuropsychiatr Genet. 2003 Apr 1;118B(1):66-71 [12627469.001]
  • [Cites] Arch Gen Psychiatry. 2003 Sep;60(9):929-37 [12963675.001]
  • [Cites] Am J Med Genet A. 2004 Jan 1;124A(1):19-27 [14679582.001]
  • [Cites] Mol Psychiatry. 2004 Jan;9(1):81-6 [14699444.001]
  • [Cites] Int J Psychophysiol. 2004 Jul;53(2):75-90 [15210286.001]
  • [Cites] Arch Gen Psychiatry. 1972 Jan;26(1):57-63 [5009428.001]
  • [Cites] Arch Gen Psychiatry. 1981 Aug;38(8):861-8 [7259422.001]
  • [Cites] J Stud Alcohol. 1986 May;47(3):207-12 [3724155.001]
  • [Cites] Proc Natl Acad Sci U S A. 1987 Apr;84(8):2363-7 [3470801.001]
  • [Cites] Am J Hum Genet. 1991 Jan;48(1):22-5 [1985459.001]
  • [Cites] Arch Gen Psychiatry. 1992 Aug;49(8):599-608 [1637250.001]
  • [Cites] J Stud Alcohol. 1994 Mar;55(2):149-58 [8189735.001]
  • [Cites] Am J Hum Genet. 1995 May;56(5):1224-33 [7726180.001]
  • [Cites] J Stud Alcohol. 1995 Sep;56(5):500-5 [7475029.001]
  • [Cites] Alcohol Clin Exp Res. 1995 Dec;19(6):1528-36 [8749822.001]
  • [Cites] Am J Med Genet. 1998 May 8;81(3):207-15 [9603606.001]
  • [Cites] Am J Med Genet. 1998 May 8;81(3):216-21 [9603607.001]
  • [Cites] Electroencephalogr Clin Neurophysiol. 1998 Apr;108(3):244-50 [9607513.001]
  • [Cites] J Abnorm Psychol. 1998 Aug;107(3):363-74 [9715572.001]
  • [Cites] Alcohol Clin Exp Res. 1998 Sep;22(6):1317-23 [9756048.001]
  • [Cites] Am J Hum Genet. 2000 Mar;66(3):1046-61 [10712217.001]
  • (PMID = 18454434.001).
  • [ISSN] 1552-485X
  • [Journal-full-title] American journal of medical genetics. Part B, Neuropsychiatric genetics : the official publication of the International Society of Psychiatric Genetics
  • [ISO-abbreviation] Am. J. Med. Genet. B Neuropsychiatr. Genet.
  • [Language] ENG
  • [Grant] United States / FIC NIH HHS / TW / R01 TW006604; United States / FIC NIH HHS / TW / R01 TW006604-05; United States / FIC NIH HHS / TW / R01-TW-6604; United States / NIAAA NIH HHS / AA / U10AA08403
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] EC 1.1.1.1 / Alcohol Dehydrogenase
  • [Other-IDs] NLM/ NIHMS203246; NLM/ PMC2879266
  •  go-up   go-down


36. Zografos GC, Zagouri F, Sergentanis TN, Koulocheri D, Nonni A, Oikonomou V, Domeyer P, Kotsani M, Fotiadis C, Bramis J: Is zero underestimation feasible? Extended Vacuum-Assisted Breast Biopsy in solid lesions - a blind study. World J Surg Oncol; 2007;5:53
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Is zero underestimation feasible? Extended Vacuum-Assisted Breast Biopsy in solid lesions - a blind study.
  • BACKGROUND: Vacuum-Assisted Breast Biopsy (VABB) is effective for the preoperative diagnosis of non-palpable mammographic solid lesions.
  • The main disadvantage is underestimation, which might render the management of atypical ductal hyperplasia (ADH), and ductal carcinoma in situ (DCIS) difficult.
  • PATIENTS AND METHODS: A total of 107 women with non-palpable mammographic breast solid tumors BI-RADS 3 and 4 underwent VABB with 11G, on the stereotactic Fischer's table.
  • A preoperative diagnosis was established.
  • Women with a preoperative diagnosis of precursor/preinvasive/invasive lesion underwent open surgery.
  • A second pathologist, blind to the preoperative results and to the protocol made the postoperative diagnosis.
  • In the extended protocol, the postoperative diagnosis was less severe than the preoperative in 55.5% of cases (55.5% vs. 0%, p = 0.029), and preoperative ADH was totally removed.
  • This might possibly point to a modified management of ADH lesions.
  • [MeSH-major] Biopsy, Needle / methods. Breast Neoplasms / pathology. Carcinoma, Intraductal, Noninfiltrating / pathology. Precancerous Conditions / pathology

  • MedlinePlus Health Information. consumer health - Breast Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Breast J. 2002 Jul-Aug;8(4):230-3 [12100116.001]
  • [Cites] AJR Am J Roentgenol. 2002 Sep;179(3):679-83 [12185043.001]
  • [Cites] AJR Am J Roentgenol. 2002 Sep;179(3):691-5 [12185047.001]
  • [Cites] Rofo. 2003 Apr;175(4):489-94 [12677503.001]
  • [Cites] Lancet. 2003 Apr 26;361(9367):1405-10 [12727392.001]
  • [Cites] Eur J Cancer. 2003 Aug;39(12):1676-83 [12888361.001]
  • [Cites] Ann Intern Med. 2003 Aug 19;139(4):274-84 [12965983.001]
  • [Cites] Ann Oncol. 2004 Mar;15(3):450-4 [14998847.001]
  • [Cites] J Radiol. 2004 Apr;85(4 Pt 1):391-401 [15213649.001]
  • [Cites] Breast Cancer Res Treat. 2007 Mar;102(1):103-10 [16838109.001]
  • [Cites] JAMA. 1993 Nov 24;270(20):2444-50 [8230621.001]
  • [Cites] Radiology. 1995 May;195(2):529-32 [7724778.001]
  • [Cites] Radiology. 2004 Sep;232(3):897-903 [15273332.001]
  • [Cites] Cancer. 1998 Feb 15;82(4):679-88 [9477100.001]
  • [Cites] Radiology. 1998 Mar;206(3):711-5 [9494489.001]
  • [Cites] Radiology. 1999 Jun;211(3):845-50 [10352614.001]
  • [Cites] J Am Coll Surg. 1999 Sep;189(3):237-40 [10472922.001]
  • [Cites] Eur Radiol. 2005 May;15(5):1027-36 [15856253.001]
  • [Cites] Invest Radiol. 2005 Jul;40(7):465-71 [15973139.001]
  • [Cites] Eur J Radiol. 2005 Aug;55(2):270-6 [16036159.001]
  • [Cites] G Chir. 2005 May;26(5):187-93 [16184700.001]
  • [Cites] Clin Radiol. 2006 Mar;61(3):276-81 [16488210.001]
  • [Cites] J Am Board Fam Med. 2006 Mar-Apr;19(2):161-4 [16513904.001]
  • [Cites] Nihon Rinsho. 2006 Mar;64(3):469-74 [16529037.001]
  • [Cites] Breast. 2006 Apr;15(2):196-202 [16055333.001]
  • [Cites] Breast. 2006 Apr;15(2):210-8 [16081287.001]
  • [Cites] Cas Lek Cesk. 2006;145(5):399-402; discussion 402-3 [16755779.001]
  • [Cites] AJR Am J Roentgenol. 2007 Mar;188(3):684-90 [17312054.001]
  • [Cites] Radiology. 2001 Feb;218(2):497-502 [11161168.001]
  • [Cites] Cancer. 2001 Jan 1;91(1):173-7 [11148574.001]
  • [Cites] Lancet. 2000 Jan 8;355(9198):129-34 [10675181.001]
  • [Cites] J Radiol. 2002 Apr;83(4 Pt 2):531-4 [12075159.001]
  • [Cites] Rofo. 2001 Aug;173(8):696-701 [11570238.001]
  • [Cites] Radiology. 2001 Jun;219(3):793-6 [11376271.001]
  • [Cites] AJR Am J Roentgenol. 1998 Jan;170(1):97-104 [9423608.001]
  • (PMID = 17501997.001).
  • [ISSN] 1477-7819
  • [Journal-full-title] World journal of surgical oncology
  • [ISO-abbreviation] World J Surg Oncol
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Randomized Controlled Trial
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC1885798
  • [General-notes] NLM/ Original DateCompleted: 20070813
  •  go-up   go-down


37. Rahko E, Kauppila S, Pääkkö P, Blanco G, Apaja-Sarkkinen M, Talvensaari-Mattila A, Turpeenniemi-Hujanen T, Jukkola A: Immunohistochemical study of matrix metalloproteinase 9 and tissue inhibitor of matrix metalloproteinase 1 in benign and malignant breast tissue--strong expression in intraductal carcinomas of the breast. Tumour Biol; 2009;30(5-6):257-64
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Immunohistochemical study of matrix metalloproteinase 9 and tissue inhibitor of matrix metalloproteinase 1 in benign and malignant breast tissue--strong expression in intraductal carcinomas of the breast.
  • Only few data exist on MMP-9 or TIMP-1 expression in tissue samples of different breast histology.
  • METHODS: MMP-9 and TIMP-1 immunoreactivity was examined in a wide range of breast tissue samples differing in histology from usual ductal hyperplasia (UDH) to fully developed ductal breast carcinoma.
  • Immunohistochemical expression of MMP-9 was studied in 178 samples: 31 UDH samples, 29 atypical ductal hyperplasia (ADH) samples, 28 ductal carcinoma in situ (DCIS) samples and 90 ductal invasive carcinoma samples (30 samples of malignancy grades I, II and III, respectively).
  • TIMP-1 expression was also analyzed in 178 breast tissue samples: 41 UDH, 21 ADH and 34 DCIS lesions, and 82 invasive ductal breast carcinomas (25 in grade I, 30 in grade II and 27 in grade III).
  • Negative or weakly positive MMP-9 staining was the most prominent finding in UDH (71%), ADH (69%) as well as in invasive carcinoma samples (64.4%).
  • CONCLUSIONS: These results suggest that MMP-9 and TIMP-1 overexpression are early markers of breast carcinogenesis preceding tumor invasion.
  • [MeSH-major] Breast Neoplasms / enzymology. Breast Neoplasms / pathology. Matrix Metalloproteinase 9 / biosynthesis. Tissue Inhibitor of Metalloproteinase-1 / biosynthesis
  • [MeSH-minor] Biomarkers, Tumor / biosynthesis. Breast / enzymology. Breast / pathology. Carcinoma, Ductal, Breast / enzymology. Carcinoma, Ductal, Breast / pathology. Carcinoma, Intraductal, Noninfiltrating / enzymology. Carcinoma, Intraductal, Noninfiltrating / pathology. Female. Humans. Hyperplasia. Immunohistochemistry

  • MedlinePlus Health Information. consumer health - Breast Cancer.
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • The Lens. Cited by Patents in .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] (c) 2009 S. Karger AG, Basel.
  • (PMID = 19887890.001).
  • [ISSN] 1423-0380
  • [Journal-full-title] Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine
  • [ISO-abbreviation] Tumour Biol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Biomarkers, Tumor; 0 / Tissue Inhibitor of Metalloproteinase-1; EC 3.4.24.35 / Matrix Metalloproteinase 9
  •  go-up   go-down


38. Trivedi AH, Spiess AC, Daussmann T, Büchs J: Study on mesophilic and thermophilic alcohol dehydrogenases in gas-phase reaction. Biotechnol Prog; 2006 Mar-Apr;22(2):454-8
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The initial reaction rate and the thermostability of the mesophilic alcohol dehydrogenase (ADH) from Lactobacillus brevis (LBADH), and the thermophilic ADH from Thermoanaerobacter sp. (ADH T) in gas-phase reaction were compared.
  • An excellent optimal productivity of 1,000 g x L(-1) x d(-1) for LBADH and 600 g x L(-1) x d(-1)for ADH T was found at 60 degrees C, while the highest total turnover numbers with respect to the enzyme were achieved at 30 degrees C and amounted to 4.2 million for LBADH and 1.7 million for ADH T, respectively.
  • Interestingly, the ADH from the mesophilic L. brevisshowed the higher thermostability in the nonconventional medium gas phase.

  • Hazardous Substances Data Bank. Water .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16599562.001).
  • [ISSN] 8756-7938
  • [Journal-full-title] Biotechnology progress
  • [ISO-abbreviation] Biotechnol. Prog.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Enzymes, Immobilized; 059QF0KO0R / Water; EC 1.1.1.1 / Alcohol Dehydrogenase
  •  go-up   go-down


39. García-Otín AL, Cofán M, Junyent M, Recalde D, Cenarro A, Pocoví M, Ros E, Civeira F: Increased intestinal cholesterol absorption in autosomal dominant hypercholesterolemia and no mutations in the low-density lipoprotein receptor or apolipoprotein B genes. J Clin Endocrinol Metab; 2007 Sep;92(9):3667-73
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • CONTEXT: Autosomal dominant hypercholesterolemia (ADH) is frequently caused by functional mutations in the low-density lipoprotein receptor (LDLR) or apolipoprotein B-100 (APOB) genes, but approximately 40% of ADH subjects disclose no such molecular defects, possibly pointing to alternative genetic mechanisms.
  • OBJECTIVE: Our objective was to test the hypothesis that increased intestinal cholesterol absorption might play a role in the lipid abnormalities of subjects with ADH without identified genetic defects.
  • STUDY SUBJECTS: A total of 52 subjects with a clinical diagnosis of ADH were examined for molecular defects in LDLR and APOB.
  • Functional LDLR mutations occurred in 31 (60%) subjects, who received a diagnosis of familial hypercholesterolemia (FH).
  • Those for whom no mutations could be identified were labeled as non-FH ADH.
  • RESULTS: Adjusted campesterol to cholesterol ratios increased in the order non-FH ADH more than FH more than controls more than FCH, with mean values (95% confidence interval) in 10(2) mmol/mol cholesterol of 505 (424-600), 397 (345-458), 335 (294-382), and 284 (247-328), respectively.
  • Thus, cholesterol absorption was lowest in FCH and highest in non-FH ADH.
  • CONCLUSIONS: Increased intestinal cholesterol absorption may partially explain the high cholesterol levels of non-FH ADH subjects.
  • Serum noncholesterol sterols are a useful tool for the differential diagnosis of genetic hypercholesterolemias, especially FCH and ADH unrelated to LDLR or APOB defects.

  • Genetic Alliance. consumer health - Hypercholesterolemia.
  • MedlinePlus Health Information. consumer health - Cholesterol.
  • Hazardous Substances Data Bank. CHOLESTEROL .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17566095.001).
  • [ISSN] 0021-972X
  • [Journal-full-title] The Journal of clinical endocrinology and metabolism
  • [ISO-abbreviation] J. Clin. Endocrinol. Metab.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Apolipoproteins B; 0 / Receptors, LDL; 0 / Sterols; 97C5T2UQ7J / Cholesterol
  •  go-up   go-down


40. Campos FM, Liarte DB, Mortara RA, Romanha AJ, Murta SM: Characterization of a gene encoding alcohol dehydrogenase in benznidazole-susceptible and -resistant populations of Trypanosoma cruzi. Acta Trop; 2009 Jul;111(1):56-63
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Alcohol dehydrogenases (ADH) are a class of oxidoreductases that catalyse the reversible oxidation of ethanol to acetaldehyde.

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 19426664.001).
  • [ISSN] 1873-6254
  • [Journal-full-title] Acta tropica
  • [ISO-abbreviation] Acta Trop.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / DNA, Protozoan; 0 / Nitroimidazoles; 0 / Protozoan Proteins; 0 / RNA, Messenger; 0 / RNA, Protozoan; 0 / Trypanocidal Agents; EC 1.1.1.1 / Alcohol Dehydrogenase; M84I3K7C2O / Nifurtimox; YC42NRJ1ZD / benzonidazole
  •  go-up   go-down


41. Trabold R, Krieg S, Schöller K, Plesnila N: Role of vasopressin V(1a) and V2 receptors for the development of secondary brain damage after traumatic brain injury in mice. J Neurotrauma; 2008 Dec;25(12):1459-65
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The goal of the current study was to investigate the role of arginine vasopressin (AVP), also known as antidiuretic hormone (ADH), an important regulator of tissue water homeostasis, for the formation of post-traumatic brain edema, intracranial pressure (ICP), brain damage, and functional deficits following brain trauma.

  • ClinicalTrials.gov. clinical trials - ClinicalTrials.gov .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 19118456.001).
  • [ISSN] 0897-7151
  • [Journal-full-title] Journal of neurotrauma
  • [ISO-abbreviation] J. Neurotrauma
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antidiuretic Hormone Receptor Antagonists; 0 / Receptors, Vasopressin; 113-79-1 / Arginine Vasopressin
  •  go-up   go-down


42. Zhang W, SLi, Visocchi M, Jiang J, Wang X: Natriuretic peptides, antidiuretic hormone and hyponatraemia after acute craniocerebral injury. J Int Med Res; 2008 Jul-Aug;36(4):648-55
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • We measured blood concentrations of natriuretic peptides, antidiuretic hormone (ADH), and endogenous digitalis-like substance (EDLS), blood and urine sodium concentrations, and the plasma and urine osmolality in 68 patients with ACI and 24 healthy control subjects.
  • Blood concentrations of EDLS and ADH in hyponatraemic ACI patients were significantly higher compared with normonatraemic ACI patients and control subjects.

  • MedlinePlus Health Information. consumer health - Head Injuries.
  • Hazardous Substances Data Bank. VASOPRESSIN .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 18652759.001).
  • [ISSN] 0300-0605
  • [Journal-full-title] The Journal of international medical research
  • [ISO-abbreviation] J. Int. Med. Res.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Cardenolides; 0 / Natriuretic Peptides; 0 / Saponins; 0 / digoxin-like factors; 11000-17-2 / Vasopressins
  •  go-up   go-down


43. Montane-Jaime K, Moore S, Shafe S, Joseph R, Crooks H, Carr L, Ehlers CL: ADH1C*2 allele is associated with alcohol dependence and elevated liver enzymes in Trinidad and Tobago. Alcohol; 2006 Jun;39(2):81-6
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Variants in alcohol dehydrogenase (ADH) genes differ between ethnic groups and have, in some studies, been found to be associated with alcohol dependence and alcoholic liver disease.
  • This study sought to determine whether an association exists between ADH (ADH1C previously ADH3, ADH1B*2 previously ADH2*2) genotypes, alcohol dependence, drinking history, and liver function tests in the two major ethnic groups of Trinidad and Tobago (TT).

  • Genetic Alliance. consumer health - Alcohol dependence.
  • MedlinePlus Health Information. consumer health - Alcoholism and Alcohol Abuse.
  • COS Scholar Universe. author profiles.
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17134660.001).
  • [ISSN] 0741-8329
  • [Journal-full-title] Alcohol (Fayetteville, N.Y.)
  • [ISO-abbreviation] Alcohol
  • [Language] ENG
  • [Grant] United States / NIAAA NIH HHS / AA / R21 AA014370; United States / PHS HHS / / 014370; United States / PHS HHS / / 06420; United States / NIAAA NIH HHS / AA / AA07611
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] EC 1.1.1.1 / ADH1C protein, human; EC 1.1.1.1 / Alcohol Dehydrogenase; EC 1.1.1.27 / L-Lactate Dehydrogenase; EC 2.3.2.2 / gamma-Glutamyltransferase; EC 2.6.1.1 / Aspartate Aminotransferases; EC 2.6.1.2 / Alanine Transaminase; EC 3.1.3.1 / Alkaline Phosphatase
  •  go-up   go-down


44. Alberti C: [Paraneoplastic syndromes associated with urogenital tumours: an up-to-date review]. G Chir; 2008 Oct;29(10):437-48
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • On the basis of an up-to-date review of the literature, this article discusses pathogenetic mechanisms, diagnostic features and essential therapeutic measures concerning the syndromes of inappropriate ACTH/CRH, PTHrp, TRH/TSH, ADH and MSH secretion.

  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 18947470.001).
  • [ISSN] 0391-9005
  • [Journal-full-title] Il Giornale di chirurgia
  • [ISO-abbreviation] G Chir
  • [Language] ita
  • [Publication-type] English Abstract; Journal Article; Review
  • [Publication-country] Italy
  • [Chemical-registry-number] 0 / Cytokines; 0 / Intercellular Signaling Peptides and Proteins; 0 / Peptide Hormones; 333DO1RDJY / Serotonin
  • [Number-of-references] 89
  •  go-up   go-down


45. Theman TA, Collins MT, Dempster DW, Zhou H, Reynolds JC, Brahim JS, Roschger P, Klaushofer K, Winer KK: PTH(1-34) replacement therapy in a child with hypoparathyroidism caused by a sporadic calcium receptor mutation. J Bone Miner Res; 2009 May;24(5):964-73
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Autosomal dominant hypocalcemia (ADH) is an inherited form of hypoparathyroidism caused by activating mutations in the calcium-sensing receptor (CaR).
  • The patient is a 20-yr-old female with ADH treated with PTH continuously since 6 yr and 2 mo of age.
  • Her data were compared with one age-, sex-, and length of hypoparathyroidism-matched control not on PTH and two sex-matched ADH controls before and after 1 yr of PTH.
  • Cancellous bone volume was dramatically elevated in the patient and in ADH controls after 1 yr of PTH.
  • BMD distribution (BMDD) by qBEI of the patient and ADH controls was strikingly shifted toward lower mineralization compared with the non-ADH control.
  • Moreover, the ADH controls exhibited a further reduction in mineralization after 1 yr of PTH.
  • In conclusion, long-term PTH replacement in a child with ADH was not unsafe, increased bone mass without negatively impacting mineralization, and improved serum mineral control but did not prevent nephrocalcinosis.

  • Genetic Alliance. consumer health - Hypoparathyroidism.
  • MedlinePlus Health Information. consumer health - Hormone Replacement Therapy.
  • COS Scholar Universe. author profiles.
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Bone. 2000 Feb;26(2):103-9 [10678403.001]
  • [Cites] Bone. 2008 Mar;42(3):456-66 [18096457.001]
  • [Cites] J Bone Miner Res. 2001 Oct;16(10):1846-53 [11585349.001]
  • [Cites] Toxicol Pathol. 2002 May-Jun;30(3):312-21 [12051548.001]
  • [Cites] J Bone Miner Res. 2002 Jul;17(7):1151-61 [12096828.001]
  • [Cites] J Clin Endocrinol Metab. 2003 Mar;88(3):1150-6 [12629098.001]
  • [Cites] J Bone Miner Res. 2003 Apr;18(4):769-75 [12674338.001]
  • [Cites] J Clin Endocrinol Metab. 2003 Sep;88(9):4214-20 [12970289.001]
  • [Cites] Proc Natl Acad Sci U S A. 2004 Apr 6;101(14):5140-5 [15051872.001]
  • [Cites] Toxicol Pathol. 2004 Jul-Aug;32(4):426-38 [15204966.001]
  • [Cites] Eur J Pediatr. 1986 Apr;145(1-2):27-33 [3015628.001]
  • [Cites] Miner Electrolyte Metab. 1986;12(5-6):342-6 [3027519.001]
  • [Cites] J Bone Miner Res. 1987 Dec;2(6):595-610 [3455637.001]
  • [Cites] J Bone Miner Res. 1986 Aug;1(4):377-81 [3503551.001]
  • [Cites] Physiol Rev. 1991 Apr;71(2):371-411 [2006218.001]
  • [Cites] Eur J Surg. 1992 Aug;158(8):397-402 [1356477.001]
  • [Cites] N Engl J Med. 1994 Dec 15;331(24):1618-23 [7969342.001]
  • [Cites] Mech Ageing Dev. 1995 Nov 24;85(2-3):125-31 [8786659.001]
  • [Cites] Hum Mol Genet. 1996 May;5(5):601-6 [8733126.001]
  • [Cites] JAMA. 1996 Aug 28;276(8):631-6 [8773636.001]
  • [Cites] N Engl J Med. 1996 Oct 10;335(15):1115-22 [8813042.001]
  • [Cites] J Exp Biol. 1997 Jan;200(Pt 2):295-302 [9050237.001]
  • [Cites] Lancet. 1997 Aug 23;350(9077):550-5 [9284777.001]
  • [Cites] Endocrinology. 1997 Oct;138(10):4330-7 [9322947.001]
  • [Cites] Bone. 1998 Oct;23(4):319-26 [9763143.001]
  • [Cites] J Clin Endocrinol Metab. 1998 Oct;83(10):3480-6 [9768650.001]
  • [Cites] J Clin Endocrinol Metab. 1999 May;84(5):1562-6 [10323380.001]
  • [Cites] Yonsei Med J. 1999 Apr;40(2):166-72 [10333721.001]
  • [Cites] J Clin Endocrinol Metab. 1999 Oct;84(10):3757-63 [10523026.001]
  • [Cites] J Bone Miner Res. 2005 Jul;20(7):1244-53 [15940379.001]
  • [Cites] J Musculoskelet Neuronal Interact. 2005 Oct-Dec;5(4):356-7 [16340137.001]
  • [Cites] J Bone Miner Res. 2006 Mar;21(3):354-65 [16491282.001]
  • [Cites] Bone. 2006 Sep;39(3):616-22 [16644299.001]
  • [Cites] Endocr J. 2006 Dec;53(6):797-802 [16983178.001]
  • [Cites] J Bone Miner Res. 2007 Feb;22(2):260-73 [17087625.001]
  • [Cites] J Clin Endocrinol Metab. 2007 Feb;92(2):443-9 [17105843.001]
  • [Cites] J Bone Miner Res. 2007 Apr;22(4):495-502 [17227219.001]
  • [Cites] J Clin Endocrinol Metab. 2007 Jun;92(6):2087-99 [17311856.001]
  • [Cites] Calcif Tissue Int. 2007 Aug;81(2):73-80 [17612779.001]
  • [Cites] N Engl J Med. 2001 May 10;344(19):1434-41 [11346808.001]
  • (PMID = 19063686.001).
  • [ISSN] 1523-4681
  • [Journal-full-title] Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research
  • [ISO-abbreviation] J. Bone Miner. Res.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Parathyroid Hormone; 0 / Receptors, Calcium-Sensing
  • [Other-IDs] NLM/ PMC2672210
  •  go-up   go-down


46. Chen HJ, Tian H, Edenberg HJ: Natural haplotypes in the regulatory sequences affect human alcohol dehydrogenase 1C (ADH1C) gene expression. Hum Mutat; 2005 Feb;25(2):150-5
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Human alcohol dehydrogenases (ADHs) play important roles in metabolizing alcohol, and several lines of evidence suggest that variations in ADH genes affect the risk for alcoholism.
  • Differences in regulatory sequences could affect the expression of ADH genes and thereby modify the risk for alcoholism.

  • COS Scholar Universe. author profiles.
  • Coriell Cell Repositories. culture/stock collections - Coriell Cell Repositories .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] (c) 2005 Wiley-Liss, Inc.
  • (PMID = 15643610.001).
  • [ISSN] 1098-1004
  • [Journal-full-title] Human mutation
  • [ISO-abbreviation] Hum. Mutat.
  • [Language] eng
  • [Databank-accession-numbers] OMIM/ 103730
  • [Grant] United States / NIAAA NIH HHS / AA / R37-AA06460
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] EC 1.1.1.1 / ADH1C protein, human; EC 1.1.1.1 / Alcohol Dehydrogenase
  •  go-up   go-down


47. Yu HS, Oyama T, Isse T, Kitagawa K, Pham TT, Tanaka M, Kawamoto T: Formation of acetaldehyde-derived DNA adducts due to alcohol exposure. Chem Biol Interact; 2010 Dec 5;188(3):367-75
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Ingested ethanol is mainly oxidized by the enzymes alcohol dehydrogenase (ADH), cytochrome P-450 2E1 (CYP2E1), and catalase to form acetaldehyde, which is subsequently oxidized by aldehyde dehydrogenase 2 (ALDH2) to produce acetate.
  • ADH1B*2 allele (ADH1B, one of the enzyme in ADH family) is commonly observed in Asian population, has much higher enzymatic activity than ADH1B*1 allele.

  • Hazardous Substances Data Bank. ACETALDEHYDE .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] Copyright © 2010 Elsevier Ireland Ltd. All rights reserved.
  • (PMID = 20813101.001).
  • [ISSN] 1872-7786
  • [Journal-full-title] Chemico-biological interactions
  • [ISO-abbreviation] Chem. Biol. Interact.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] Ireland
  • [Chemical-registry-number] 0 / Alcohols; 0 / DNA Adducts; EC 1.1.1.1 / Alcohol Dehydrogenase; EC 1.2.1.3 / Aldehyde Dehydrogenase; GO1N1ZPR3B / Acetaldehyde
  •  go-up   go-down


48. Belokon' MM, Belokon' IuS, Politov DV, Altukhov IuP: [Allozyme polymorphism of Swiss stone pine Pinus cembra L. in mountainous populations of the Alps and the Eastern Carpathians]. Genetika; 2005 Nov;41(11):1538-51
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The allele frequencies of 30 isozyme loci, coding for enzymes ADH, FDH, FEST, GDH, GOT, IDH, LAP, MNR, MDH, PEPCA, 6PGD, PGI, PGM, SDH, SKDH, SOD, were analyzed using cluster analysis and methods of principal components.
  • The main contribution to these differences were made by loci Adh-1, Adh-2, Fest-2, Lap-3, Mdh-4, and Sod-4.

  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16358721.001).
  • [ISSN] 0016-6758
  • [Journal-full-title] Genetika
  • [ISO-abbreviation] Genetika
  • [Language] rus
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] Russia (Federation)
  • [Chemical-registry-number] 0 / Enzymes; 0 / Plant Proteins
  •  go-up   go-down


49. Charlot A, Heyraud A, Guenot P, Rinaudo M, Auzély-Velty R: Controlled synthesis and inclusion ability of a hyaluronic acid derivative bearing beta-cyclodextrin molecules. Biomacromolecules; 2006 Mar;7(3):907-13
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • This was based on the preparation of a HA derivative selectively modified with adipic dihydrazide (HA-ADH) and a beta-cyclodextrin derivative possessing an aldehyde function on the primary face, followed by their coupling by a reductive amination-type reaction.

  • Hazardous Substances Data Bank. IBUPROFEN .
  • Hazardous Substances Data Bank. HYALURONIC ACID .
  • The Lens. Cited by Patents in .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16529430.001).
  • [ISSN] 1525-7797
  • [Journal-full-title] Biomacromolecules
  • [ISO-abbreviation] Biomacromolecules
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Acetates; 0 / Polymers; 0 / beta-Cyclodextrins; 9004-61-9 / Hyaluronic Acid; JV039JZZ3A / betadex; PJY633525U / Adamantane; WK2XYI10QM / Ibuprofen
  •  go-up   go-down


50. Defilippi BG, Dandekar AM, Kader AA: Relationship of ethylene biosynthesis to volatile production, related enzymes, and precursor availability in apple peel and flesh tissues. J Agric Food Chem; 2005 Apr 20;53(8):3133-41
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The study was focused at the level of the aroma volatile-related enzymes, including alcohol acyltransferase (AAT), alcohol dehydrogenase (ADH), and lipoxygenase (LOX) and at the level of amino acids and fatty acids as aroma volatile precursors in peel and flesh tissues.
  • Contrarily, ADH and LOX seem to be independent of ethylene modulation.

  • Hazardous Substances Data Bank. L-Isoleucine .
  • Hazardous Substances Data Bank. ETHYLENE .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 15826070.001).
  • [ISSN] 0021-8561
  • [Journal-full-title] Journal of agricultural and food chemistry
  • [ISO-abbreviation] J. Agric. Food Chem.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Amino Acids; 0 / Ethylenes; 0 / Fatty Acids; 04Y7590D77 / Isoleucine; 91GW059KN7 / ethylene; EC 1.1.1.1 / Alcohol Dehydrogenase; EC 1.13.11.12 / Lipoxygenase; EC 2.3.- / Acyltransferases
  •  go-up   go-down


56. Yalcin-Cakmakli G, Karli Oguz K, Shorbagi A, Funda Bas D, Ergan-Arsava B, Kunt M, Topcuoglu MA: Hyponatremic encephalopathy after excessive water ingestion prior to pelvic ultrasound: neuroimaging findings. Intern Med; 2010;49(16):1807-11
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • In addition to the first illustration of multimodality MR imaging features of water-intoxication, these two cases also highlight the need for standardized practice on the quantity of water intake recommended to distend the bladder for pelvic ultrasound, especially in patients at risk for serum inappropriate ADH syndrome-related hyponatremia.
  • [MeSH-major] Brain Edema / diagnosis. Hyponatremia / diagnosis. Pelvis / ultrasonography. Water Intoxication / diagnosis

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 20720364.001).
  • [ISSN] 1349-7235
  • [Journal-full-title] Internal medicine (Tokyo, Japan)
  • [ISO-abbreviation] Intern. Med.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Japan
  •  go-up   go-down


57. Lee SM, Alam R, Ho CJ, Kim JH, Kang CW, Park JH, Lee MS: Involvement of p42/44 MAPK in the effects of ethanol on secretion of insulin-like growth factor (IGF)-I and insulin-like growth factor binding protein (IGFBP)-1 in primary cultured rat hepatocytes. Int J Neurosci; 2007 Feb;117(2):187-201
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • In addition, inhibition of p42/44 MAPK activity by ethanol occurs via the activity of ADH.

  • Hazardous Substances Data Bank. ETHANOL .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17365107.001).
  • [ISSN] 0020-7454
  • [Journal-full-title] The International journal of neuroscience
  • [ISO-abbreviation] Int. J. Neurosci.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Central Nervous System Depressants; 0 / Enzyme Inhibitors; 0 / Insulin-Like Growth Factor Binding Protein 1; 3K9958V90M / Ethanol; 67763-96-6 / Insulin-Like Growth Factor I; EC 1.1.1.1 / Alcohol Dehydrogenase; EC 2.7.11.24 / Extracellular Signal-Regulated MAP Kinases; EC 2.7.11.24 / Mitogen-Activated Protein Kinase 1; EC 2.7.11.24 / Mitogen-Activated Protein Kinase 3
  •  go-up   go-down


58. Chiang CP, Wu CW, Lee SP, Chung CC, Wang CW, Lee SL, Nieh S, Yin SJ: Expression pattern, ethanol-metabolizing activities, and cellular localization of alcohol and aldehyde dehydrogenases in human pancreas: implications for pathogenesis of alcohol-induced pancreatic injury. Alcohol Clin Exp Res; 2009 Jun;33(6):1059-68
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • BACKGROUND: Alcohol dehydrogenase (ADH) and aldehyde dehydrogenase (ALDH) are major enzymes responsible for metabolism of ethanol.
  • The goal of this study was to determine the functional expressions and cellular localization of ADH and ALDH families in human pancreas.
  • The isozyme expression patterns of ADH/ALDH were identified by isoelectric focusing, and the activities were assayed spectrophotometrically.
  • The protein contents of ADH/ALDH isozymes were determined by immunoblotting, and the cellular localizations were detected by immunohistochemistry and histochemistry.

  • MedlinePlus Health Information. consumer health - Pancreatic Cancer.
  • MedlinePlus Health Information. consumer health - Pancreatitis.
  • Hazardous Substances Data Bank. ETHANOL .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 19382905.001).
  • [ISSN] 1530-0277
  • [Journal-full-title] Alcoholism, clinical and experimental research
  • [ISO-abbreviation] Alcohol. Clin. Exp. Res.
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Central Nervous System Depressants; 0 / Isoenzymes; 3K9958V90M / Ethanol; EC 1.1.1.1 / ADH1B protein, human; EC 1.1.1.1 / ADH1C protein, human; EC 1.1.1.1 / Alcohol Dehydrogenase; EC 1.2.1.3 / ALDH2 protein, human; EC 1.2.1.3 / Aldehyde Dehydrogenase
  •  go-up   go-down


59. Yeom J, Bhang SH, Kim BS, Seo MS, Hwang EJ, Cho IH, Park JK, Hahn SK: Effect of cross-linking reagents for hyaluronic acid hydrogel dermal fillers on tissue augmentation and regeneration. Bioconjug Chem; 2010 Feb 17;21(2):240-7
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Five kinds of samples, a normal control, a negative control, a positive control of Restylane, adipic acid dihydrazide grafted HA (HA-ADH) hydrogels, and HA-HMDA hydrogels, were subcutaneously injected to wrinkled model mice.

  • Hazardous Substances Data Bank. HEXAMETHYLENE DIAMINE .
  • Hazardous Substances Data Bank. HYALURONIC ACID .
  • The Lens. Cited by Patents in .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 20078098.001).
  • [ISSN] 1520-4812
  • [Journal-full-title] Bioconjugate chemistry
  • [ISO-abbreviation] Bioconjug. Chem.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Biocompatible Materials; 0 / Cross-Linking Reagents; 0 / Diamines; 0 / Hydrogels; 9004-61-9 / Hyaluronic Acid; 9007-34-5 / Collagen; ZRA5J5B2QW / 1,6-diaminohexane
  •  go-up   go-down


60. Dendorfer U, Mann J: [Drug-related disorders of water and electrolyte metabolism]. Internist (Berl); 2006 Nov;47(11):1121-2, 1124-6, 8
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Drug-induced disorders of Na+ concentration may usually be attributed to altered antidiuretic hormone (ADH) effects, either as diabetes insipidus or as the syndrome of inappropriate ADH secretion.

  • MedlinePlus Health Information. consumer health - Blood Pressure Medicines.
  • MedlinePlus Health Information. consumer health - Fluid and Electrolyte Balance.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Internist (Berl). 2004 May;45(5):598-605 [15133614.001]
  • [Cites] Ann Pharmacother. 1993 Jul-Aug;27(7-8):832-40 [8364259.001]
  • [Cites] Eur J Intern Med. 2003 Aug;14(5):302-309 [13678755.001]
  • [Cites] Pharmacotherapy. 2002 Jul;22(7):915-23 [12126224.001]
  • [Cites] N Engl J Med. 1998 Aug 13;339(7):451-8 [9700180.001]
  • [Cites] N Engl J Med. 2004 Aug 5;351(6):543-51 [15295047.001]
  • [Cites] Best Pract Res Clin Endocrinol Metab. 2003 Dec;17(4):471-503 [14687585.001]
  • [Cites] QJM. 2005 Jul;98(7):529-40 [15955797.001]
  • [Cites] Best Pract Res Clin Endocrinol Metab. 2003 Dec;17(4):623-51 [14687593.001]
  • [Cites] Internist (Berl). 2006 Jun;47(6):623-4, 626-8 [16767477.001]
  • [Cites] Am J Nephrol. 1999;19(4):447-52 [10460932.001]
  • [Cites] BMJ. 2006 Mar 25;332(7543):702-5 [16565125.001]
  • [Cites] Am J Med Sci. 2000 Jan;319(1):10-24 [10653441.001]
  • [Cites] Ann Pharmacother. 2002 Sep;36(9):1331-6 [12196047.001]
  • [Cites] N Engl J Med. 2004 Aug 5;351(6):585-92 [15295051.001]
  • [Cites] N Engl J Med. 2000 May 25;342(21):1581-9 [10824078.001]
  • [Cites] JAMA. 1999 Mar 17;281(11):1022-9 [10086438.001]
  • [Cites] N Engl J Med. 2000 May 18;342(20):1493-9 [10816188.001]
  • [Cites] J Am Board Fam Med. 2006 Mar-Apr;19(2):148-60 [16513903.001]
  • (PMID = 16988802.001).
  • [ISSN] 0020-9554
  • [Journal-full-title] Der Internist
  • [ISO-abbreviation] Internist (Berl)
  • [Language] ger
  • [Publication-type] English Abstract; Journal Article; Review
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / Anti-Inflammatory Agents, Non-Steroidal; 0 / Antidiuretic Agents; 0 / Antihypertensive Agents; 0 / Gonadal Steroid Hormones
  • [Number-of-references] 24
  •  go-up   go-down


61. Chen YY, DeVries S, Anderson J, Lessing J, Swain R, Chin K, Shim V, Esserman LJ, Waldman FM, Hwang ES: Pathologic and biologic response to preoperative endocrine therapy in patients with ER-positive ductal carcinoma in situ. BMC Cancer; 2009 Aug 18;9:285
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Pathologic and biologic response to preoperative endocrine therapy in patients with ER-positive ductal carcinoma in situ.
  • BACKGROUND: Endocrine therapy is commonly recommended in the adjuvant setting for patients as treatment for ductal carcinoma in situ (DCIS).
  • Following treatment, predominant morphologic changes included increased multinucleated histiocytes and degenerated cells, decreased duct extension, and prominent periductal fibrosis.
  • Two postmenopausal patients had ADH only with no residual DCIS at excision.
  • [MeSH-major] Breast Neoplasms / drug therapy. Breast Neoplasms / pathology. Estrogen Antagonists / therapeutic use. Hormone Replacement Therapy. Receptors, Estrogen / metabolism. Tamoxifen / therapeutic use
  • [MeSH-minor] Adult. Aged. Carcinoma, Ductal. Cohort Studies. Female. Gene Expression Regulation, Neoplastic. Humans. Middle Aged. Nitriles / therapeutic use. Triazoles / therapeutic use

  • MedlinePlus Health Information. consumer health - Breast Cancer.
  • MedlinePlus Health Information. consumer health - Hormone Replacement Therapy.
  • COS Scholar Universe. author profiles.
  • ClinicalTrials.gov. clinical trials - ClinicalTrials.gov .
  • Hazardous Substances Data Bank. TAMOXIFEN .
  • Hazardous Substances Data Bank. LETROZOLE .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Clin Cancer Res. 2000 Feb;6(2):616-21 [10690547.001]
  • [Cites] Lancet Oncol. 2008 Jan;9(1):45-53 [18083636.001]
  • [Cites] Cancer Res. 2000 Aug 1;60(15):4284-8 [10945643.001]
  • [Cites] Radiology. 2000 Nov;217(2):466-70 [11058647.001]
  • [Cites] Cancer Res. 2000 Nov 1;60(21):5995-6000 [11085519.001]
  • [Cites] Radiology. 2001 Feb;218(2):497-502 [11161168.001]
  • [Cites] Cancer Epidemiol Biomarkers Prev. 2001 Sep;10(9):961-6 [11535548.001]
  • [Cites] Cancer Res. 2001 Sep 15;61(18):6739-46 [11559545.001]
  • [Cites] Ann Oncol. 2001 Nov;12(11):1527-32 [11822750.001]
  • [Cites] J Clin Oncol. 2002 Feb 15;20(4):1026-35 [11844826.001]
  • [Cites] J Steroid Biochem Mol Biol. 2001 Dec;79(1-5):103-7 [11850213.001]
  • [Cites] J Biol Chem. 2002 Nov 22;277(47):45695-703 [12244117.001]
  • [Cites] Br J Cancer. 2003 Jul 21;89(2):277-83 [12865917.001]
  • [Cites] N Engl J Med. 2004 Apr 1;350(14):1430-41 [15070793.001]
  • [Cites] Cancer. 1984 Aug 15;54(4):612-5 [6744199.001]
  • [Cites] Br J Cancer. 1985 Feb;51(2):271-8 [3966983.001]
  • [Cites] Arch Pathol Lab Med. 1985 Aug;109(8):716-21 [3893381.001]
  • [Cites] Br J Cancer. 1987 Dec;56(6):814-9 [2829956.001]
  • [Cites] N Engl J Med. 1989 Feb 23;320(8):479-84 [2644532.001]
  • [Cites] Hum Pathol. 1992 Oct;23(10):1095-7 [1328030.001]
  • [Cites] Br J Cancer. 1993 Mar;67(3):606-11 [8439511.001]
  • [Cites] Histopathology. 1993 Sep;23(3):233-8 [8225241.001]
  • [Cites] Semin Diagn Pathol. 1994 Aug;11(3):223-35 [7831534.001]
  • [Cites] JAMA. 1996 Mar 27;275(12):913-8 [8598618.001]
  • [Cites] Cancer. 1995 Oct 1;76(7):1197-200 [8630897.001]
  • [Cites] Breast Cancer Res Treat. 1996;41(1):31-41 [8932874.001]
  • [Cites] Int J Cancer. 1997 Aug 7;72(4):608-13 [9259399.001]
  • [Cites] Int J Oncol. 1998 Apr;12(4):853-8 [9499446.001]
  • [Cites] Lancet. 1999 Jun 12;353(9169):1993-2000 [10376613.001]
  • [Cites] Clin Cancer Res. 2005 Jan 15;11(2 Pt 2):951s-8s [15701892.001]
  • [Cites] J Clin Oncol. 2005 Apr 10;23(11):2477-92 [15767642.001]
  • [Cites] Cancer. 2005 May 1;103(9):1778-84 [15770688.001]
  • [Cites] J Clin Oncol. 2005 May 1;23(13):2980-7 [15860853.001]
  • [Cites] Cancer. 2005 Jun 15;103(12):2481-4 [15884091.001]
  • [Cites] J Clin Oncol. 2005 Aug 1;23(22):5108-16 [15998903.001]
  • [Cites] N Engl J Med. 2005 Dec 29;353(26):2747-57 [16382061.001]
  • [Cites] Cancer. 2006 May 15;106(10):2095-103 [16598749.001]
  • [Cites] Breast Cancer Res Treat. 2006 May;97(2):135-44 [16319971.001]
  • [Cites] J Steroid Biochem Mol Biol. 2007 Aug-Sep;106(1-5):173-9 [17604618.001]
  • [Cites] Arch Intern Med. 2000 Apr 10;160(7):953-8 [10761960.001]
  • (PMID = 19689789.001).
  • [ISSN] 1471-2407
  • [Journal-full-title] BMC cancer
  • [ISO-abbreviation] BMC Cancer
  • [Language] eng
  • [Databank-accession-numbers] ClinicalTrials.gov/ NCT00290745
  • [Grant] United States / NCI NIH HHS / CA / K23CA097181; United States / NCI NIH HHS / CA / P50 CA58207
  • [Publication-type] Clinical Trial; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Estrogen Antagonists; 0 / Nitriles; 0 / Receptors, Estrogen; 0 / Triazoles; 094ZI81Y45 / Tamoxifen; 7LKK855W8I / letrozole
  • [Other-IDs] NLM/ PMC2744704
  •  go-up   go-down


62. Zagouri F, Sergentanis TN, Nonni A, Koulocheri D, Fotou M, Panopoulou E, Panou M, Fotiadis C, Bramis J, Zografos GC: Vacuum-assisted breast biopsy: the value and limitations of cores with microcalcifications. Pathol Res Pract; 2007;203(8):563-6
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Vacuum-assisted breast biopsy: the value and limitations of cores with microcalcifications.
  • The aim of this study was to assess cores with microcalcifications (CM) and without microcalcifications (CWM) obtained from vacuum-assisted breast biopsy (VABB).
  • The study included 12 atypical ductal hyperplasias (ADH), 37 ductal carcinomas in situ (DCIS), and seven invasive ductal carcinomas (IDC) diagnosed by VABB (11G) on the Fischer's table.
  • CWM failed to make the diagnosis in 8.3% and 35.1% of ADH and DCIS, respectively.
  • In 28.6% of IDC, diagnosis was made through CWM.
  • The underestimation rate was 8.3% in ADH, and 10.8% in DCIS.
  • CMs are superior in DCIS/ADH diagnosis.
  • However, CWM may be valuable for the diagnosis of the invasive component.
  • [MeSH-major] Biopsy / methods. Breast Neoplasms / pathology. Breast Neoplasms / surgery. Calcinosis / pathology. Calcinosis / surgery

  • MedlinePlus Health Information. consumer health - Biopsy.
  • MedlinePlus Health Information. consumer health - Breast Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17611039.001).
  • [ISSN] 1618-0631
  • [Journal-full-title] Pathology, research and practice
  • [ISO-abbreviation] Pathol. Res. Pract.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Germany
  •  go-up   go-down


63. Scaperrotta G, Ferranti C, Costa C, Mariani L, Marchesini M, Suman L, Folini C, Bergonzi S: Role of sonoelastography in non-palpable breast lesions. Eur Radiol; 2008 Nov;18(11):2381-9
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Role of sonoelastography in non-palpable breast lesions.
  • The purpose of this study was to evaluate the diagnostic utility of sonoelastography in differentiating benign from malignant non-palpable breast lesions.
  • A total of 293 BI-RADS 3-5 (Breast Imaging Reporting And Data System) impalpable breast lesions in 278 women was evaluated with B-mode ultrasound (US) and subsequently with sonoelastography (SE) before performing US-guided biopsy.
  • Lesions that were malignant or showed atypical ductal hyperplasia were referred for surgical excision, as well as 32 benign lesions showing discordance between US/SE results and histology.
  • SE is a simple, fast and non-invasive diagnostic method that may be a useful aid to US for less experienced radiologists in the assessment of solid non-palpable breast lesions, especially BI-RADS 3, where specificity was higher (88.7%).
  • [MeSH-major] Breast Neoplasms / diagnostic imaging. Elasticity Imaging Techniques / methods. Ultrasonography, Mammary / methods

  • MedlinePlus Health Information. consumer health - Breast Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Radiology. 1983 Sep;148(3):839-43 [6878708.001]
  • [Cites] Eur J Radiol. 2002 Apr;42(1):2-9 [12039015.001]
  • [Cites] Radiol Clin North Am. 1995 Nov;33(6):1067-80 [7480656.001]
  • [Cites] Radiol Med. 2005 Jul-Aug;110(1-2):69-76 [16163141.001]
  • [Cites] Ultrason Imaging. 1991 Apr;13(2):111-34 [1858217.001]
  • [Cites] Lancet. 1987 Dec 5;2(8571):1316-9 [2890912.001]
  • [Cites] Acad Radiol. 2006 Dec;13(12 ):1496-504 [17138118.001]
  • [Cites] Radiology. 1982 Apr;143(1):29-36 [7063747.001]
  • [Cites] Radiology. 1997 Jan;202(1):79-86 [8988195.001]
  • [Cites] Ultrasound Med Biol. 2001 Nov;27(11):1461-9 [11750744.001]
  • [Cites] Eur Radiol. 2003 Jan;13(1):68-79 [12541112.001]
  • [Cites] Ultrasonics. 2004 Apr;42(1-9):331-6 [15047307.001]
  • [Cites] Arch Gynecol Obstet. 1999;262(3-4):159-71 [10326635.001]
  • [Cites] Eur Radiol. 2000;10 (10 ):1562-8 [11044925.001]
  • [Cites] Radiology. 2006 May;239(2):341-50 [16484352.001]
  • [Cites] Radiology. 1995 Jul;196 (1):123-34 [7784555.001]
  • (PMID = 18523780.001).
  • [ISSN] 1432-1084
  • [Journal-full-title] European radiology
  • [ISO-abbreviation] Eur Radiol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Germany
  •  go-up   go-down


64. Calaf GM, Echiburú-Chau C, Zhao YL, Hei TK: BigH3 protein expression as a marker for breast cancer. Int J Mol Med; 2008 May;21(5):561-8
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] BigH3 protein expression as a marker for breast cancer.
  • For breast cancer development, this multistep process may manifest itself as a sequence of pathologically defined stages.
  • It is widely held that breast cancer originates at the premalignant stage of atypical ductal hyperplasia, progresses to the preinvasive stage of ductal carcinoma in situ, and culminates in the potentially lethal stage of invasive ductal carcinoma.
  • Tumor grade has been a highly valuable prognostic factor for breast cancer, and high-grade ductal carcinoma in situ lesions are associated with poor clinical outcome.
  • The aim of this work was to investigate the BigH3 protein expression changes associated with various stages of breast cancer progression in comparison to benign specimens using tissue microarray technology.
  • Pathological characteristics of breast tissues ranged from benign lesions to breast cancers either of lobular or ductal carcinomas in origin, and included in situ ductal carcinomas, lobular carcinomas, infiltrating ductal carcinomas, carcinomas, scirrhous carcinomas, adenocarcinomas and infiltrating colloid carcinomas.
  • BigH3 protein expression was analyzed by immunohistochemistry in 192 cases of breast tumors.
  • Results indicated a decrease in BigH3 protein expression from benign tissues to in situ ductal carcinoma, lobular carcinoma, infiltrating ductal carcinomas, carcinomas, scirrhous carcinoma, adenocarcinomas to infiltrating colloid carcinomas.
  • In summary, these studies confirmed the suppressor effect of the BigH3 gene expressed as protein expression in those processes related to the progression of breast tumorigenesis.
  • We conclude that this protein can be used as a marker for breast cancer progression.
  • [MeSH-major] Biomarkers, Tumor / metabolism. Breast Neoplasms / metabolism. Extracellular Matrix Proteins / metabolism. Transforming Growth Factor beta / metabolism
  • [MeSH-minor] Aged. Aged, 80 and over. Female. Humans. Mammary Glands, Human / cytology. Mammary Glands, Human / metabolism. Mammary Glands, Human / pathology. Microarray Analysis. Middle Aged

  • Genetic Alliance. consumer health - Breast Cancer.
  • MedlinePlus Health Information. consumer health - Breast Cancer.
  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 18425347.001).
  • [ISSN] 1107-3756
  • [Journal-full-title] International journal of molecular medicine
  • [ISO-abbreviation] Int. J. Mol. Med.
  • [Language] eng
  • [Grant] United States / PHS HHS / / NAG2 1637; United States / NIEHS NIH HHS / ES / P30 ES 09089
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] Greece
  • [Chemical-registry-number] 0 / Biomarkers, Tumor; 0 / Extracellular Matrix Proteins; 0 / Transforming Growth Factor beta; 148710-76-3 / betaIG-H3 protein
  •  go-up   go-down


65. Duchesne N, Parker SH, Lechner MC, Gittleman MA, Kusnick CA, Elvecrog EE, Kaske TI, Gizienski TA: Multicenter evaluation of a new ultrasound-guided biopsy device: Improved ergonomics, sampling and rebiopsy rates. Breast J; 2007 Jan-Feb;13(1):36-43
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The purpose of this study was to evaluate performance, ergonomics, and immediate rebiopsy rate of a new vacuum-assisted biopsy (VAB) device for ultrasound-guided breast biopsies.
  • Patient and procedural data included breast composition, lesion characteristics, number of samples, procedure time, and complications.
  • One hundred thirteen patients aged 20-83 years (mean 52) were successfully biopsied with dense/fibrous breast tissue in 60% and dense/fibrous lesions in 49%.
  • Operators rated safety and comfort comparable with existing devices and rated sample quality, breast/lesion penetration, and positioning ease/accuracy superior (p < 0.01).
  • Diagnoses included 37 cancers, 70 benign, and six high-risk lesions with one upgrade from atypical ductal hyperplasia to ductal carcinoma in situ at surgery.
  • Compared with other devices, it is more ergonomic to target and position for sampling, particularly in dense breast tissue or lesions.
  • [MeSH-major] Biopsy / instrumentation. Breast Neoplasms / pathology. Ultrasonography, Interventional / instrumentation

  • MedlinePlus Health Information. consumer health - Biopsy.
  • MedlinePlus Health Information. consumer health - Breast Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17214791.001).
  • [ISSN] 1075-122X
  • [Journal-full-title] The breast journal
  • [ISO-abbreviation] Breast J
  • [Language] eng
  • [Publication-type] Evaluation Studies; Journal Article; Multicenter Study; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  •  go-up   go-down


66. Kristoffersen L, Skuterud B, Larssen BR, Skurtveit S, Smith-Kielland A: Fast quantification of ethanol in whole blood specimens by the enzymatic alcohol dehydrogenase method. Optimization by experimental design. J Anal Toxicol; 2005 Jan-Feb;29(1):66-70
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Alcohol dehydrogenase (ADH) oxidizes ethanol to acetaldehyde using the coenzyme nicotinamide adenine dinucleotide (NAD), which is concurrently reduced to form NADH.
  • Method development was performed with the aid of factorial design, varying pH, and concentrations of NAD+ and ADH.

  • Hazardous Substances Data Bank. ETHANOL .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 15808017.001).
  • [ISSN] 0146-4760
  • [Journal-full-title] Journal of analytical toxicology
  • [ISO-abbreviation] J Anal Toxicol
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 3K9958V90M / Ethanol; EC 1.1.1.1 / Alcohol Dehydrogenase
  •  go-up   go-down


67. Tang F, Gu DH, Wang H, Zhu TF, Zhu HG, Xu ZD, Hu XQ: [Expression and significance of hTERT mRNA in breast carcinoma and its relation to p53]. Zhonghua Zhong Liu Za Zhi; 2006 Mar;28(3):192-5
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Expression and significance of hTERT mRNA in breast carcinoma and its relation to p53].
  • OBJECTIVE: This study was designed to investigate the significance of hTERT mRNA in breast carcinogenesis and to explore the diagnostic efficacy, and to study the effect of tumor suppressor gene p53 on the expression of hTERT mRNA.
  • METHODS: The expression of hTERT mRNA was examined by in situ hybridization in 12 cases of normal breast tissue nearby cancer, 7 of simple ductal hyperplasia, 20 of atypical hyperplasia, 18 of ductal carcinoma in situ and 25 with invasive ductal carcinoma.
  • RESULTS: hTERT was not detected in normal breast tissue nearby cancer and simple ductal hyperplasia.
  • The positive rate of hTERT mRNA in atypical hyperplasia, ductal carcinoma in situ and invasive ductal carcinoma were 25.0%, 83.3% and 88.0%, respectively.
  • The prevalence and intensity of hTERT mRNA expression were much greater in carcinoma than those in simple or atypical hyperplasia and normal breast tissue nearby cancer (P < 0.05).
  • The positive correlation between hTERT mRNA and p53 was found in breast carcinoma (r = 0.5540, P < 0.01).
  • CONCLUSION: hTERT mRNA expression is closely related to the malignant transformation of breast tissue.
  • Semi-quantitative detection of hTERT mRNA expression in situ is helpful in differentiated diagnosis of carcinoma in situ and atypical hyperplasia.
  • Inactivation of p53 may play a role in the transcriptive activation of hTERT gene in breast carcinoma.
  • [MeSH-major] Breast Neoplasms / metabolism. Carcinoma, Ductal, Breast / metabolism. Telomerase / biosynthesis
  • [MeSH-minor] Adult. Breast / metabolism. Breast / pathology. Carcinoma, Intraductal, Noninfiltrating / metabolism. Carcinoma, Intraductal, Noninfiltrating / pathology. Diagnosis, Differential. Disease Progression. Humans. Hyperplasia. Lymphatic Metastasis. Middle Aged. RNA, Messenger / biosynthesis. RNA, Messenger / genetics. Tumor Suppressor Protein p53

  • MedlinePlus Health Information. consumer health - Breast Cancer.
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16875603.001).
  • [ISSN] 0253-3766
  • [Journal-full-title] Zhonghua zhong liu za zhi [Chinese journal of oncology]
  • [ISO-abbreviation] Zhonghua Zhong Liu Za Zhi
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] China
  • [Chemical-registry-number] 0 / RNA, Messenger; 0 / Tumor Suppressor Protein p53; EC 2.7.7.49 / TERT protein, human; EC 2.7.7.49 / Telomerase
  •  go-up   go-down


68. Guth AA, Shanker BA, Roses DF, Axelrod D, Singh B, Toth H, Shapiro RL, Hiotis K, Diflo T, Cangiarella JF: A decade of change: an institutional experience with breast surgery in 1995 and 2005. Breast Cancer (Auckl); 2008;1:51-5
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] A decade of change: an institutional experience with breast surgery in 1995 and 2005.
  • INTRODUCTION: With the adoption of routine screening mammography, breast cancers are being diagnosed at earlier stages, with DCIS now accouting for 22.5% of all newly diagnosed breast cancers.
  • This has been attributed to both increased breast cancer awareness and improvements in breast imaging techniques.
  • How have these changes, including the increased use of image-guided sampling techniques, influenced the clinical practice of breast surgery?
  • METHODS: The institutional pathology database was queried for all breast surgeries, including breast reconstruction, performed in 1995 and 2005.
  • ADH, and to a lesser extent, ALH, became indications for surgical excision.
  • Fewer surgical biopsies were performed for indeterminate abnormalities on breast imaging, due to the introduction of stereotactic large core biopsy.
  • While the rate of benign breast biopsies remained constant, there was a higher percentage of precancerous and DCIS cases in 2005.
  • CONCLUSIONS: While the rate of benign breast biopsies has remained constant over a recent 10-year period, fewer diagnostic surgical image-guided biopsies were performed in 2005.
  • A greater percentage of patients with breast cancer or preinvasive disease have these diagnoses determined before surgery.

  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Am J Surg. 2002 Oct;184(4):353-5 [12383901.001]
  • [Cites] Am J Surg. 2003 Aug;186(2):112-6 [12885600.001]
  • [Cites] N Engl J Med. 2005 Oct 27;353(17):1773-83 [16169887.001]
  • [Cites] Breast. 2005 Dec;14(6):509-19 [16246564.001]
  • [Cites] CA Cancer J Clin. 2006 May-Jun;56(3):168-83 [16737949.001]
  • [Cites] Eur J Surg Oncol. 2006 Nov;32(9):901-10 [16920327.001]
  • [Cites] MMWR Morb Mortal Wkly Rep. 2007 Jan 26;56(3):49-51 [17251897.001]
  • [Cites] N Engl J Med. 2007 Mar 29;356(13):1295-303 [17392300.001]
  • [Cites] Breast J. 2006 Sep-Oct;12(5 Suppl 2):S174-80 [16958998.001]
  • [Cites] Cancer. 2006 Jan 1;106(1):4-16 [16329134.001]
  • [Cites] J Clin Oncol. 2006 Oct 1;24(28):4603-10 [17008702.001]
  • (PMID = 21655372.001).
  • [Journal-full-title] Breast cancer : basic and clinical research
  • [ISO-abbreviation] Breast Cancer (Auckl)
  • [Language] ENG
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC3091402
  • [Keywords] NOTNLM ; breast cancer / breast cancer trends / breast radiology / breast surgery / sentinel nodes
  •  go-up   go-down


69. Balasundaram B, Harrison ST: Disruption of Brewers' yeast by hydrodynamic cavitation: Process variables and their influence on selective release. Biotechnol Bioeng; 2006 Jun 5;94(2):303-11
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The release profile of the enzymes studied include alpha-glucosidase (periplasmic), invertase (cell wall bound), alcohol dehydrogenase (ADH; cytoplasmic) and glucose-6-phosphate dehydrogenase (G6PDH; cytoplasmic).

  • Saccharomyces Genome Database. Saccharomyces Genome Database .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] Copyright 2006 Wiley Periodicals, Inc.
  • (PMID = 16570316.001).
  • [ISSN] 0006-3592
  • [Journal-full-title] Biotechnology and bioengineering
  • [ISO-abbreviation] Biotechnol. Bioeng.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] EC 1.1.1.1 / Alcohol Dehydrogenase; EC 3.2.1.- / Glycoside Hydrolases
  •  go-up   go-down


70. Atsumi S, Wu TY, Eckl EM, Hawkins SD, Buelter T, Liao JC: Engineering the isobutanol biosynthetic pathway in Escherichia coli by comparison of three aldehyde reductase/alcohol dehydrogenase genes. Appl Microbiol Biotechnol; 2010 Jan;85(3):651-7
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Here, we have compared the effect of various alcohol dehydrogenases (ADH) for the last step of the isobutanol production. E. coli has the yqhD gene which encodes a broad-range ADH.
  • The adh genes of two bacteria and one yeast were also compared in E. coli harboring the isobutanol synthesis pathway.

  • COS Scholar Universe. author profiles.
  • BioCyc. gene/protein/disease-specific - yqhD .
  • Hazardous Substances Data Bank. ISOBUTYL ALCOHOL .
  • Saccharomyces Genome Database. Saccharomyces Genome Database .
  • The Lens. Cited by Patents in .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Nature. 2008 Jan 3;451(7174):86-9 [18172501.001]
  • [Cites] Mol Syst Biol. 2006;2:2006.0008 [16738554.001]
  • [Cites] J Biol Chem. 2008 Mar 21;283(12):7346-53 [18211903.001]
  • [Cites] Appl Environ Microbiol. 2008 Sep;74(18):5769-75 [18676713.001]
  • [Cites] Appl Microbiol Biotechnol. 2008 Nov;81(1):89-98 [18758769.001]
  • [Cites] Metab Eng. 2008 Nov;10(6):305-11 [17942358.001]
  • [Cites] Metab Eng. 2008 Nov;10(6):312-20 [18775501.001]
  • [Cites] Proc Natl Acad Sci U S A. 2000 Jun 6;97(12):6640-5 [10829079.001]
  • [Cites] Genome Res. 2001 May;11(5):731-53 [11337471.001]
  • [Cites] Proteins. 2004 Jan 1;54(1):174-7 [14705036.001]
  • [Cites] J Mol Biol. 2004 Aug 20;341(4):1049-62 [15289102.001]
  • [Cites] J Mol Biol. 2004 Sep 10;342(2):489-502 [15327949.001]
  • [Cites] FEMS Microbiol Lett. 2004 Sep 15;238(2):367-74 [15358422.001]
  • [Cites] Eur J Biochem. 1987 Sep 1;167(2):195-201 [3622514.001]
  • [Cites] Nature. 1990 Jan 4;343(6253):38-43 [2296288.001]
  • [Cites] Nucleic Acids Res. 1997 Mar 15;25(6):1203-10 [9092630.001]
  • [Cites] Nat Biotechnol. 2004 Nov;22(11):1399-408 [15529165.001]
  • [Cites] Proc Natl Acad Sci U S A. 2005 Jul 19;102(29):10105-10 [16006528.001]
  • [Cites] Protein Sci. 2006 Mar;15(3):449-58 [16452624.001]
  • [Cites] Appl Microbiol Biotechnol. 2008 Jan;77(6):1305-16 [18060402.001]
  • (PMID = 19609521.001).
  • [ISSN] 1432-0614
  • [Journal-full-title] Applied microbiology and biotechnology
  • [ISO-abbreviation] Appl. Microbiol. Biotechnol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / Butanols; 0 / Escherichia coli Proteins; 0 / Fungal Proteins; 0 / Recombinant Proteins; 56F9Z98TEM / isobutyl alcohol; EC 1.1.1.1 / Alcohol Dehydrogenase; EC 1.1.1.21 / Aldehyde Reductase
  • [Other-IDs] NLM/ PMC2802489
  •  go-up   go-down


71. Yang Z, Lu CD: Functional genomics enables identification of genes of the arginine transaminase pathway in Pseudomonas aeruginosa. J Bacteriol; 2007 Jun;189(11):3945-53
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • While most of the genes involved in arginine uptake, regulation, and metabolism have been identified as members of the ArgR (arginine-responsive regulatory protein) regulon in our previous study, they did not include any genes of the arginine dehydrogenase (ADH) pathway.
  • In this study, 18 putative transcriptional units of 38 genes, including the two known genes of the ADH pathway, kauB and gbuA, were found to be inducible by exogenous L-arginine in the absence of ArgR.
  • To identify the missing genes that encode enzymes for the initial steps of the ADH pathway, the potential physiological functions of those candidate genes in arginine utilization were studied by growth phenotype analysis of knockout mutants.

  • Gene Ontology. gene/protein/disease-specific - Gene Ontology annotations from this paper .
  • Hazardous Substances Data Bank. (L)-ARGININE .
  • The Lens. Cited by Patents in .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Nature. 2000 Aug 31;406(6799):959-64 [10984043.001]
  • [Cites] J Bacteriol. 2007 Jun;189(11):3954-9 [17416668.001]
  • [Cites] FEMS Microbiol Lett. 2001 Feb 20;195(2):191-6 [11179651.001]
  • [Cites] J Bacteriol. 2001 Nov;183(22):6517-24 [11673419.001]
  • [Cites] J Bacteriol. 2002 Jun;184(12):3377-84 [12029055.001]
  • [Cites] J Bacteriol. 2002 Jul;184(14):3765-73 [12081945.001]
  • [Cites] Microbiology. 2003 Mar;149(Pt 3):707-14 [12634339.001]
  • [Cites] J Bacteriol. 2004 Jun;186(12):3848-54 [15175298.001]
  • [Cites] J Bacteriol. 2004 Jun;186(12):3855-61 [15175299.001]
  • [Cites] J Biol Chem. 1971 May 10;246(9):2758-64 [5554291.001]
  • [Cites] J Biol Chem. 1971 Aug 25;246(16):5053-8 [5570437.001]
  • [Cites] J Biol Chem. 1972 Jul 25;247(14):4486-90 [5043851.001]
  • [Cites] J Biol Chem. 1975 Jul 25;250(14):5322-9 [237915.001]
  • [Cites] Anal Biochem. 1976 May 7;72:248-54 [942051.001]
  • [Cites] Mol Gen Genet. 1977 Jul 7;154(1):7-22 [408599.001]
  • [Cites] J Gen Microbiol. 1980 Feb;116(2):381-9 [6768836.001]
  • [Cites] Mol Gen Genet. 1984;193(3):437-44 [6423933.001]
  • [Cites] J Bacteriol. 1984 Dec;160(3):928-34 [6438064.001]
  • [Cites] Nucleic Acids Res. 1985 Jun 11;13(11):3979-93 [2989781.001]
  • [Cites] Biochem J. 1986 Mar 15;234(3):593-604 [3521591.001]
  • [Cites] Gene. 1986;50(1-3):133-40 [2884169.001]
  • [Cites] J Gen Microbiol. 1988 Apr;134(4):1043-53 [3141581.001]
  • [Cites] J Bacteriol. 1990 Jun;172(6):3496-9 [2111810.001]
  • [Cites] Gene. 1991 Jan 2;97(1):109-21 [1899844.001]
  • [Cites] J Gen Microbiol. 1990 Dec;136(12):2351-7 [1964171.001]
  • [Cites] J Bacteriol. 1991 May;173(9):3000-9 [1902216.001]
  • [Cites] J Bacteriol. 1991 Aug;173(15):4742-50 [1906871.001]
  • [Cites] J Gen Microbiol. 1991 Dec;137(12):2911-8 [1791443.001]
  • [Cites] Microbiology. 1994 Nov;140 ( Pt 11):2907-29 [7812433.001]
  • [Cites] J Bacteriol. 1997 Sep;179(17):5300-8 [9286980.001]
  • [Cites] J Bacteriol. 1997 Sep;179(17):5309-17 [9286981.001]
  • [Cites] J Bacteriol. 1997 Dec;179(23):7280-90 [9393691.001]
  • [Cites] Microbiol Mol Biol Rev. 1997 Dec;61(4):393-410 [9409145.001]
  • [Cites] J Bacteriol. 1998 Nov;180(21):5559-66 [9791103.001]
  • [Cites] J Bacteriol. 1999 Mar;181(6):1934-8 [10074092.001]
  • [Cites] J Bacteriol. 2001 Jan;183(2):490-9 [11133942.001]
  • (PMID = 17416670.001).
  • [ISSN] 0021-9193
  • [Journal-full-title] Journal of bacteriology
  • [ISO-abbreviation] J. Bacteriol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Bacterial Proteins; 0 / Recombinant Fusion Proteins; 94ZLA3W45F / Arginine; EC 2.3.- / Acyltransferases; EC 2.3.1.- / arginine succinyltransferase; EC 2.6.1.- / Transaminases; EC 4.1.1.- / Carboxy-Lyases
  • [Other-IDs] NLM/ PMC1913404
  •  go-up   go-down


72. Jelski W, Chrostek L, Laszewicz W, Szmitkowski M: Alcohol dehydrogenase (ADH) isoenzyme activity in the sera of patients with Helicobacter pylori infection. Dig Dis Sci; 2007 Jun;52(6):1513-6
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Alcohol dehydrogenase (ADH) isoenzyme activity in the sera of patients with Helicobacter pylori infection.
  • Human gastric mucosa contains three classes of alcohol dehydrogenase (ADH) isoenzymes: I, III, and IV.
  • Various factors have been found to influence gastric ADH activity.
  • One of them is Helicobacter pylori infection, which is associated with gastric mucosal injury and leads to a decrease in gastric ADH activity.
  • The aim of the study was to assess the effect of H. pylori infection on the serum activity of ADH isoenzymes.
  • The activities of class III and IV ADH isoenzymes were measured by the photometric method with formaldehyde and with m-nitrobenzaldehyde as substrate, respectively.
  • Total activity of ADH was measured by a photometric method with p-nitrosodimethylaniline.
  • The total activities of ADH and class IV isoenzyme were significantly higher in sera of patients with H. pylori infection compared to healthy subjects.
  • The serum activity of other tested isoenzymes of ADH did not differ significantly between infected and noninfected groups.
  • We conclude that H. pylori infection of gastric mucosa is reflected in the serum by a significant increase in class IV and total ADH activity.

  • MedlinePlus Health Information. consumer health - Helicobacter Pylori Infections.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Alcohol Clin Exp Res. 1993 Dec;17(6):1337-44 [8116851.001]
  • [Cites] Gut. 1993 Oct;34(10):1433-7 [8244116.001]
  • [Cites] FEBS Lett. 1989 Oct 23;257(1):105-9 [2806555.001]
  • [Cites] Alcohol Clin Exp Res. 1990 Dec;14(6):946-50 [1982399.001]
  • [Cites] Anal Biochem. 1989 Apr;178(1):57-62 [2729580.001]
  • [Cites] N Engl J Med. 1990 Jan 11;322(2):95-9 [2248624.001]
  • [Cites] Anal Biochem. 1979 Oct 15;99(1):65-71 [231394.001]
  • [Cites] Life Sci. 1991;49(25):1929-34 [1745108.001]
  • [Cites] Digestion. 2002;66(1):14-8 [12379810.001]
  • [Cites] Alcohol Alcohol. 1994 Nov;29(6):663-71 [7695781.001]
  • [Cites] J Clin Lab Anal. 2003;17(3):93-6 [12696080.001]
  • [Cites] Alcohol Clin Exp Res. 1994 Dec;18(6):1294-9 [7695020.001]
  • [Cites] Clin Sci (Lond). 2006 Mar;110(3):305-14 [16464172.001]
  • [Cites] Digestion. 1998 Jul-Aug;59(4):314-20 [9693201.001]
  • [Cites] Alcohol Clin Exp Res. 2001 Apr;25(4):508-12 [11329489.001]
  • [Cites] Alcohol Clin Exp Res. 2001 Jun;25(6 Suppl):29S-34S [11410738.001]
  • [Cites] Alcohol Alcohol. 1997 Sep-Oct;32(5):543-9 [9373695.001]
  • [Cites] Alcohol Clin Exp Res. 1994 Aug;18(4):795-8 [7978087.001]
  • [Cites] Alcohol Clin Exp Res. 1996 Dec;20(9):1569-76 [8986205.001]
  • [Cites] J Gastroenterol Hepatol. 1991 May-Jun;6(3):209-22 [1912431.001]
  • [Cites] Am J Physiol Gastrointest Liver Physiol. 2005 Sep;289(3):G429-33 [15860641.001]
  • [Cites] Dig Dis Sci. 2002 Jul;47(7):1554-7 [12141816.001]
  • [Cites] J Biol Chem. 1991 Jan 15;266(2):1128-33 [1985938.001]
  • (PMID = 17404875.001).
  • [ISSN] 0163-2116
  • [Journal-full-title] Digestive diseases and sciences
  • [ISO-abbreviation] Dig. Dis. Sci.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Isoenzymes; EC 1.1.1.1 / Alcohol Dehydrogenase
  •  go-up   go-down


73. Peña-Fronteras JT, Villalobos MC, Baltazar AM, Merca FE, Ismail AM, Johnson DE: Adaptation to flooding in upland and lowland ecotypes of Cyperus rotundus, a troublesome sedge weed of rice: tuber morphology and carbohydrate metabolism. Ann Bot; 2009 Jan;103(2):295-302
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • METHODS: Upland and lowland ecotypes of C. rotundus were compared in terms of growth habit, carbohydrate reserves and metabolism, and activities of enzymes involved in alcoholic fermentation - alcohol dehydrogenase (ADH) and pyruvate decarboxylase (PDC).
  • At 24 h after germination in hypoxic conditions, PDC and ADH activities in the lowland plants increased, before decreasing at 48 h following germination.
  • In contrast, ADH and PDC activities in the upland plants increased from 24 to 48 h after germination.
  • This is coupled with the modulation of ADH and PDC activities during germination, possibly to control the use of carbohydrate reserves and sustain substrate supply in order to avoid starvation and death of seedlings with prolonged flooding.

  • MedlinePlus Health Information. consumer health - Floods.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Ann Bot. 2003 Jan;91 Spec No:143-8 [12509335.001]
  • [Cites] J Exp Bot. 2003 Oct;54(391):2363-73 [14504303.001]
  • [Cites] J Biol Chem. 1951 Nov;193(1):113-24 [14907695.001]
  • [Cites] Plant Physiol. 1993 Oct;103(2):441-8 [8029333.001]
  • [Cites] Anal Biochem. 1976 May 7;72:248-54 [942051.001]
  • (PMID = 18515404.001).
  • [ISSN] 1095-8290
  • [Journal-full-title] Annals of botany
  • [ISO-abbreviation] Ann. Bot.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 9005-25-8 / Starch; EC 1.1.1.1 / Alcohol Dehydrogenase; EC 3.2.1.- / Amylases; EC 4.1.1.1 / Pyruvate Decarboxylase
  • [Other-IDs] NLM/ PMC2707299
  •  go-up   go-down


74. Hanai T, Atsumi S, Liao JC: Engineered synthetic pathway for isopropanol production in Escherichia coli. Appl Environ Microbiol; 2007 Dec;73(24):7814-8
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The strain with the combination of C. acetobutylicum thl (acetyl-coenzyme A [CoA] acetyltransferase), E. coli atoAD (acetoacetyl-CoA transferase), C. acetobutylicum adc (acetoacetate decarboxylase), and C. beijerinckii adh (secondary alcohol dehydrogenase) achieved the highest titer.

  • COS Scholar Universe. author profiles.
  • Hazardous Substances Data Bank. ISOPROPANOL .
  • The Lens. Cited by Patents in .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Nucleic Acids Res. 1997 Mar 15;25(6):1203-10 [9092630.001]
  • [Cites] FEMS Microbiol Rev. 1995 Oct;17(3):263-73 [7576768.001]
  • [Cites] Biotechnol Bioeng. 1999 Jan 20;62(2):235-41 [10099534.001]
  • [Cites] Biotechnol Prog. 1999 Sep-Oct;15(5):855-66 [10514255.001]
  • [Cites] Biotechnol Bioeng. 2005 Jan 20;89(2):148-56 [15543598.001]
  • [Cites] Nat Biotechnol. 2000 May;18(5):533-7 [10802621.001]
  • [Cites] Nat Biotechnol. 2003 Jul;21(7):796-802 [12778056.001]
  • [Cites] Proc Natl Acad Sci U S A. 2004 Feb 24;101(8):2235-40 [14982993.001]
  • [Cites] Arch Biochem Biophys. 1977 May;181(1):178-84 [18103.001]
  • [Cites] Biochem J. 1981 Apr 1;195(1):183-90 [7030321.001]
  • [Cites] J Bacteriol. 1987 Jan;169(1):42-52 [3025185.001]
  • [Cites] Appl Environ Microbiol. 1989 Feb;55(2):323-9 [2719476.001]
  • [Cites] Appl Environ Microbiol. 1990 Apr;56(4):1004-11 [2187400.001]
  • [Cites] Appl Environ Microbiol. 1990 Nov;56(11):3491-8 [2268159.001]
  • [Cites] J Chromatogr. 1991 Feb 15;563(2):231-42 [2055989.001]
  • [Cites] J Bacteriol. 1993 Aug;175(16):5097-105 [8349550.001]
  • [Cites] Appl Environ Microbiol. 1998 Mar;64(3):1079-85 [9501448.001]
  • (PMID = 17933911.001).
  • [ISSN] 1098-5336
  • [Journal-full-title] Applied and environmental microbiology
  • [ISO-abbreviation] Appl. Environ. Microbiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Bacterial Proteins; EC 1.1.- / Alcohol Oxidoreductases; EC 1.1.1.80 / isopropanol dehydrogenase (NADP); EC 2.3.1.9 / Acetyl-CoA C-Acetyltransferase; EC 4.1.1.- / Carboxy-Lyases; EC 4.1.1.4 / acetoacetate decarboxylase; ND2M416302 / 2-Propanol
  • [Other-IDs] NLM/ PMC2168132
  •  go-up   go-down


75. Campbell CT: Transition metal oxides: extra thermodynamic stability as thin films. Phys Rev Lett; 2006 Feb 17;96(6):066106
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The adhesion energy at this interface (E(adh,ox/m)) provides extra stabilization, which lowers the O2 pressure required for oxide stability as a thin film below that required for bulk-oxide stability by the factor exp[(2gamma(g/ox) - E(adh,ox/m))/(tN(ox)RT)], where gamma(g/ox) is the surface energy of the oxide, t is the oxide film thickness, and N(ox) is the oxygen concentration in the bulk oxide (moles O2 per volume).

  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16606017.001).
  • [ISSN] 0031-9007
  • [Journal-full-title] Physical review letters
  • [ISO-abbreviation] Phys. Rev. Lett.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  •  go-up   go-down


76. Fadare O, Clement NF, Ghofrani M: High and intermediate grade ductal carcinoma in-situ of the breast: a comparison of pathologic features in core biopsies and excisions and an evaluation of core biopsy features that may predict a close or positive margin in the excision. Diagn Pathol; 2009;4:26
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] High and intermediate grade ductal carcinoma in-situ of the breast: a comparison of pathologic features in core biopsies and excisions and an evaluation of core biopsy features that may predict a close or positive margin in the excision.
  • Low and high-grade ductal carcinoma in-situ (DCIS) are known to be highly disparate by a multitude of parameters, including progression potential, immunophenotype, gene expression profile and DNA ploidy.
  • In this study, we analyzed a group of intermediate and high-grade DCIS cases to determine how well the core biopsy predicts the maximal pathology in the associated excisions, and to determine if there are any core biopsy morphologic features that may predict a close (< or = 0.2 cm) or positive margin in the subsequent excision.
  • Forty-nine consecutive paired specimens [core biopsies with a maximal diagnosis of DCIS, and their corresponding excisions, which included 20 and 29 specimens from mastectomies and breast conserving surgeries respectively] were evaluated in detail.
  • In another 4 cases, the changes were diagnostic only of atypical ductal hyperplasia.
  • Among the 40 cases with at least residual DCIS in the excision, there were 5 cases in which comedo-pattern DCIS was present in the excision but not in the core biopsy, attributed to the lower maximal nuclear grade in the biopsy proliferation in 4 cases and the absence of central necrosis in the 5th.
  • In 6(15%) of the 40 cases, the maximum nuclear grade of the excision (grade 3) was higher than that seen in the core biopsy (grade 2).
  • Overall, however, the maximum nuclear grade in the excision was significantly predicted by maximum nuclear grade in the core biopsy (p = 0.028), with a Phi of 0.347, indicating a moderately strong association.
  • Finally, among a variety of core biopsy features that were evaluated, including maximum nuclear grade, necrosis, cancerization of lobules, number of tissue cores with DCIS, number of DCIS ducts per tissue core, total DCIS ducts, or comedo-pattern, only necrosis was significantly associated with a positive or close (< or = 0.2 cm) margin on multivariate analysis (Phi of 0.350).
  • It is concluded that a significant change [to invasive disease (14%) or to no residual disease (10%)] is seen in approximately 24% of excisions that follow a core biopsy diagnosis of intermediate or high-grade DCIS.

  • COS Scholar Universe. author profiles.
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Breast Cancer Res Treat. 2009 May;115(1):181-3 [18516674.001]
  • [Cites] J Clin Pathol. 2009 Jun;62(6):534-8 [19190009.001]
  • [Cites] J Surg Oncol. 2009 Feb 1;99(2):99-103 [19065638.001]
  • [Cites] Int J Clin Pract. 2008 Nov;62(11):1730-5 [19143859.001]
  • [Cites] J Clin Oncol. 2009 Jan 10;27(2):279-88 [19064970.001]
  • [Cites] Arch Pathol Lab Med. 2009 Jan;133(1):26-30 [19123732.001]
  • [Cites] Clin Radiol. 2009 Feb;64(2):178-83 [19103348.001]
  • [Cites] J Surg Oncol. 2008 Dec 15;98(8):585-7 [19072849.001]
  • [Cites] Eur J Surg Oncol. 2009 Jan;35(1):43-7 [18723312.001]
  • [Cites] Int J Radiat Oncol Biol Phys. 2008 Nov 15;72(4):1016-20 [18954711.001]
  • [Cites] Clin Breast Cancer. 2008 Jun;8(3):269-74 [18650158.001]
  • [Cites] Ann Ital Chir. 2008 May-Jun;79(3):151-5 [18958961.001]
  • [Cites] Br J Surg. 2008 May;95(5):547-54 [18386775.001]
  • [Cites] Acta Oncol. 2008;47(2):225-31 [17851868.001]
  • [Cites] Expert Rev Anticancer Ther. 2008 Mar;8(3):433-41 [18366290.001]
  • [Cites] Breast J. 2008 Mar-Apr;14(2):135-40 [18315691.001]
  • [Cites] Ann Surg Oncol. 2008 Mar;15(3):833-42 [18163174.001]
  • [Cites] Ann Surg Oncol. 2008 Jan;15(1):235-43 [17987342.001]
  • [Cites] Mod Pathol. 2008 Jan;21(1):39-45 [17948023.001]
  • [Cites] Ann Surg Oncol. 2007 Oct;14(10):2953-60 [17674109.001]
  • [Cites] Acta Oncol. 2007;46(6):798-802 [17653903.001]
  • [Cites] Ann Surg Oncol. 2007 May;14(5):1638-43 [17245612.001]
  • [Cites] Ann Surg Oncol. 2007 May;14(5):1618-28 [17443388.001]
  • [Cites] Breast J. 2007 May-Jun;13(3):251-7 [17461899.001]
  • [Cites] Eur J Cancer. 2007 Apr;43(6):993-1001 [17300928.001]
  • [Cites] Ann Surg Oncol. 2007 Apr;14(4):1458-71 [17260108.001]
  • [Cites] Breast Cancer Res Treat. 2007 Mar;101(3):335-47 [16838110.001]
  • [Cites] ANZ J Surg. 2006 Dec;76(12):1064-7 [17199691.001]
  • [Cites] J Exp Clin Cancer Res. 2006 Jun;25(2):223-7 [16918134.001]
  • [Cites] Breast Cancer Res Treat. 2006 Aug;98(3):311-4 [16552627.001]
  • [Cites] J Clin Pathol. 2006 Jul;59(7):740-3 [16803949.001]
  • [Cites] Asian Pac J Cancer Prev. 2006 Jan-Mar;7(1):69-74 [16629519.001]
  • [Cites] Breast J. 2006 Jan-Feb;12(1):20-7 [16409583.001]
  • [Cites] Pathol Res Pract. 2005;201(11):713-25 [16325514.001]
  • [Cites] Am J Surg Pathol. 2005 Dec;29(12):1625-32 [16327435.001]
  • [Cites] J Surg Oncol. 2005 May 1;90(2):71-6 [15844190.001]
  • [Cites] J Am Coll Surg. 2005 Apr;200(4):516-26 [15804465.001]
  • [Cites] Eur J Radiol. 2005 Apr;54(1):55-61 [15797293.001]
  • [Cites] J Pathol. 2005 Jan;205(2):248-54 [15641021.001]
  • [Cites] Can Assoc Radiol J. 1999 Aug;50(4):235-40 [10459309.001]
  • [Cites] J Pathol. 1999 Mar;187(4):383-4 [10398095.001]
  • [Cites] J Natl Cancer Inst. 1997 Sep 17;89(18):1356-60 [9308705.001]
  • [Cites] Radiology. 1997 Apr;203(1):151-7 [9122384.001]
  • [Cites] JAMA. 1996 Mar 27;275(12):913-8 [8598618.001]
  • [Cites] Semin Diagn Pathol. 1994 Aug;11(3):181-92 [7831529.001]
  • [Cites] Histopathology. 1991 Nov;19(5):403-10 [1757079.001]
  • [Cites] Lancet. 1990 Mar 3;335(8688):519-22 [1968538.001]
  • [Cites] Acad Radiol. 2004 Mar;11(3):293-308 [15035520.001]
  • [Cites] Cancer. 2004 Jan 15;100(2):245-51 [14716757.001]
  • [Cites] Breast Cancer Res. 2003;5(6):313-9 [14580249.001]
  • [Cites] Am J Surg. 2003 Oct;186(4):371-7 [14553853.001]
  • [Cites] ANZ J Surg. 2003 Jun;73(6):404-6 [12801338.001]
  • [Cites] J Natl Cancer Inst. 2002 Oct 16;94(20):1546-54 [12381707.001]
  • [Cites] Cancer. 2002 Apr 1;94(7):1917-24 [11932892.001]
  • [Cites] Clin Radiol. 2001 Oct;56(10):828-32 [11895299.001]
  • [Cites] J Surg Oncol. 2001 Apr;76(4):245-54 [11320515.001]
  • [Cites] Am J Clin Pathol. 2001 May;115(5):709-18 [11345835.001]
  • [Cites] Br J Radiol. 2000 Sep;73(873):938-44 [11064645.001]
  • [Cites] Breast Cancer Res Treat. 2000 May;61(2):151-9 [10942101.001]
  • [Cites] Eur J Surg Oncol. 2000 Nov;26(7):646-51 [11078610.001]
  • [Cites] Am J Surg Pathol. 2000 Aug;24(8):1058-67 [10935646.001]
  • [Cites] Am J Surg Pathol. 2000 Jan;24(1):123-8 [10632496.001]
  • [Cites] Am J Surg Pathol. 1999 Nov;23(11):1340-8 [10555002.001]
  • [Cites] Eur J Cancer. 2009 May;45(7):1162-7 [19121932.001]
  • (PMID = 19691836.001).
  • [ISSN] 1746-1596
  • [Journal-full-title] Diagnostic pathology
  • [ISO-abbreviation] Diagn Pathol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC2740842
  •  go-up   go-down


77. Lührs AK, Guhr S, Schilke R, Borchers L, Geurtsen W, Günay H: Shear bond strength of self-etch adhesives to enamel with additional phosphoric acid etching. Oper Dent; 2008 Mar-Apr;33(2):155-62
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • In half of each test group, ground enamel surfaces were coated with the self-etch adhesives AdheSe (ADH), Xeno III (XE) or Futurabond NR (FNR).

  • COS Scholar Universe. author profiles.
  • Hazardous Substances Data Bank. PHOSPHORIC ACID .
  • Hazardous Substances Data Bank. Water .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 18435189.001).
  • [ISSN] 0361-7734
  • [Journal-full-title] Operative dentistry
  • [ISO-abbreviation] Oper Dent
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Randomized Controlled Trial
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Acrylic Resins; 0 / AdheSE cement; 0 / CeramX; 0 / Composite Resins; 0 / Dentin-Bonding Agents; 0 / Methacrylates; 0 / Phosphoric Acids; 0 / Resin Cements; 0 / Syntac dentine adhesive; 0 / Tetric EvoCeram; 0 / Xeno III; 0 / futurabond NR; 059QF0KO0R / Water; E4GA8884NN / phosphoric acid
  •  go-up   go-down


78. Moulis S, Sgroi DC: Re-evaluating early breast neoplasia. Breast Cancer Res; 2008;10(1):302
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Re-evaluating early breast neoplasia.
  • Historically, histomorphological and epidemiological data suggested that atypical ductal hyperplasia and ductal carcinoma in situ are the earliest recognizable neoplastic stages of breast cancer progression.
  • Over the past several years, detailed high-throughput molecular genetic, gene expression and epigenetic analyses have enhanced our understanding of these early neoplastic lesions and have re-shaped our view of human breast cancer progression to include multiple distinct pathways of evolution.

  • MedlinePlus Health Information. consumer health - Breast Cancer.
  • COS Scholar Universe. author profiles.
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Proc Natl Acad Sci U S A. 2001 Sep 11;98(19):10869-74 [11553815.001]
  • [Cites] J Clin Oncol. 2007 Feb 20;25(6):662-8 [17308270.001]
  • [Cites] Proc Natl Acad Sci U S A. 2003 May 13;100(10):5974-9 [12714683.001]
  • [Cites] Breast Cancer Res. 2003;5(5):254-7 [12927035.001]
  • [Cites] Proc Natl Acad Sci U S A. 2003 Sep 2;100(18):10393-8 [12917485.001]
  • [Cites] Cancer Cell. 2004 Jun;5(6):607-16 [15193263.001]
  • [Cites] N Engl J Med. 1985 Jan 17;312(3):146-51 [3965932.001]
  • [Cites] Cancer. 1990 Feb 1;65(3):518-29 [2297643.001]
  • [Cites] Am J Pathol. 1997 Jan;150(1):297-303 [9006344.001]
  • [Cites] J Natl Cancer Inst. 1998 May 6;90(9):697-703 [9586667.001]
  • [Cites] J Pathol. 1999 Mar;187(4):396-402 [10398097.001]
  • [Cites] J Pathol. 1999 Feb;187(3):272-8 [10398078.001]
  • [Cites] J Natl Cancer Inst. 2006 Feb 15;98(4):262-72 [16478745.001]
  • [Cites] Clin Cancer Res. 2006 Apr 1;12(7 Pt 1):2080-7 [16609019.001]
  • [Cites] Cancer Res. 2006 Apr 15;66(8):4065-78 [16618726.001]
  • [Cites] J Pathol. 2006 Jul;209(3):307-16 [16604511.001]
  • [Cites] Cancer Res. 2006 Nov 1;66(21):10292-301 [17079448.001]
  • [Cites] Cell Cycle. 2006 Oct;5(19):2198-202 [16969100.001]
  • [Cites] Cancer Res. 2006 Dec 15;66(24):11649-58 [17178859.001]
  • [Cites] Mol Cancer Res. 2003 Mar;1(5):362-75 [12651909.001]
  • (PMID = 18279539.001).
  • [ISSN] 1465-542X
  • [Journal-full-title] Breast cancer research : BCR
  • [ISO-abbreviation] Breast Cancer Res.
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / R01 CA112021; United States / NCI NIH HHS / CA / R01-1CA112021-01
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC2374963
  •  go-up   go-down


79. Rao R, Zhang MZ, Zhao M, Cai H, Harris RC, Breyer MD, Hao CM: Lithium treatment inhibits renal GSK-3 activity and promotes cyclooxygenase 2-dependent polyuria. Am J Physiol Renal Physiol; 2005 Apr;288(4):F642-9
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Lithium also induced renal medullary COX2 protein expression in congenitally polyuric antidiuretic hormone (AHD)-deficient rats, demonstrating that lithium-induced COX2 protein expression is not secondary to altered ADH levels or polyuria.

  • COS Scholar Universe. author profiles.
  • Hazardous Substances Data Bank. LITHIUM CHLORIDE .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 15585669.001).
  • [ISSN] 1931-857X
  • [Journal-full-title] American journal of physiology. Renal physiology
  • [ISO-abbreviation] Am. J. Physiol. Renal Physiol.
  • [Language] eng
  • [Grant] United States / NIDDK NIH HHS / DK / 2P50-DK-39261; United States / NIDDK NIH HHS / DK / DK-065024
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Adjuvants, Immunologic; 0 / Membrane Proteins; EC 1.14.99.1 / Cyclooxygenase 1; EC 1.14.99.1 / Cyclooxygenase 2; EC 1.14.99.1 / Prostaglandin-Endoperoxide Synthases; EC 1.14.99.1 / Ptgs1 protein, mouse; EC 1.14.99.1 / Ptgs1 protein, rat; EC 2.7.11.1 / glycogen synthase kinase 3 beta; EC 2.7.11.26 / Glycogen Synthase Kinase 3; G4962QA067 / Lithium Chloride; K7Q1JQR04M / Dinoprostone
  •  go-up   go-down


80. Crichton PG, Affourtit C, Moore AL: Identification of a mitochondrial alcohol dehydrogenase in Schizosaccharomyces pombe: new insights into energy metabolism. Biochem J; 2007 Jan 15;401(2):459-64
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • In the present study we have shown that mitochondria isolated from Schizosaccharomyces pombe exhibit antimycin A-sensitive oxygen uptake activity that is exclusively dependent on ethanol and is inhibited by trifluoroethanol, a potent inhibitor of ADH (alcohol dehydrogenase).
  • Ethanol-dependent respiratory activity has, to our knowledge, not been reported in S. pombe mitochondria to date, which is surprising as it has been concluded previously that only one ADH gene, encoding a cytosolic enzyme, occurs in this yeast.
  • Spectrophotometric enzyme assays reveal that ADH activity in isolated mitochondria is increased approximately 16-fold by Triton X-100, which demonstrates that the enzyme is located in the matrix.
  • Using genetic knockouts, we show conclusively that the novel mitochondrial ADH is encoded by adh4 and, as such, is unrelated to ADH isoenzymes found in mitochondria of other yeasts.

  • COS Scholar Universe. author profiles.
  • Saccharomyces Genome Database. Saccharomyces Genome Database .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Gene. 2000 Mar 21;245(2):299-309 [10717481.001]
  • [Cites] FEBS Lett. 2005 Jan 17;579(2):331-6 [15642340.001]
  • [Cites] J Mol Biol. 2000 Jul 21;300(4):1005-16 [10891285.001]
  • [Cites] J Bacteriol. 2000 Sep;182(17):4730-7 [10940011.001]
  • [Cites] FEMS Microbiol Rev. 2001 Jan;25(1):15-37 [11152939.001]
  • [Cites] Biochim Biophys Acta. 2001 Mar 1;1504(1):58-69 [11239485.001]
  • [Cites] Eur J Biochem. 2001 Oct;268(19):5057-65 [11589696.001]
  • [Cites] J Biol Chem. 2002 Jan 11;277(2):1190-4 [11698414.001]
  • [Cites] Bioinformatics. 2002 Feb;18(2):298-305 [11847077.001]
  • [Cites] Nature. 2002 Feb 21;415(6874):871-80 [11859360.001]
  • [Cites] Trends Plant Sci. 2002 Nov;7(11):478-81 [12417142.001]
  • [Cites] DNA Repair (Amst). 2002 Dec 5;1(12):983-94 [12531008.001]
  • [Cites] DNA Seq. 2002 Oct;13(5):301-6 [12592711.001]
  • [Cites] FEMS Yeast Res. 2004 Mar;4(6):649-54 [15040954.001]
  • [Cites] Eur J Biochem. 1970 Feb;12(3):583-92 [4314881.001]
  • [Cites] Biochem J. 1978 Jun 1;171(3):613-27 [208509.001]
  • [Cites] J Biol Chem. 1983 Jan 10;258(1):143-9 [6294096.001]
  • [Cites] Anal Biochem. 1985 Oct;150(1):76-85 [3843705.001]
  • [Cites] J Bacteriol. 1987 Jun;169(6):2591-7 [3584063.001]
  • [Cites] Mol Gen Genet. 1987 Sep;209(2):374-81 [2823079.001]
  • [Cites] Nucleic Acids Res. 1991 Jul 11;19(13):3525-31 [1852603.001]
  • [Cites] Mol Gen Genet. 1992 Apr;232(3):479-88 [1588917.001]
  • [Cites] Yeast. 1992 Nov;8(11):923-33 [1481570.001]
  • [Cites] Can J Microbiol. 1992 Dec;38(12):1313-9 [1288848.001]
  • [Cites] Crit Rev Microbiol. 1994;20(1):13-56 [8185833.001]
  • [Cites] Eur J Biochem. 1994 Dec 15;226(3):1071-8 [7813462.001]
  • [Cites] J Bioenerg Biomembr. 1994 Aug;26(4):447-56 [7844119.001]
  • [Cites] J Bioenerg Biomembr. 1995 Aug;27(4):387-96 [8595974.001]
  • [Cites] J Biol Chem. 1996 Jul 19;271(29):17062-6 [8663588.001]
  • [Cites] Eur J Biochem. 1996 Nov 1;241(3):779-86 [8944766.001]
  • [Cites] J Biol Chem. 1998 Sep 18;273(38):24529-34 [9733747.001]
  • [Cites] J Biol Chem. 1998 Nov 13;273(46):30301-5 [9804791.001]
  • [Cites] J Biol Chem. 1999 Mar 5;274(10):6212-8 [10037707.001]
  • [Cites] J Bacteriol. 2000 May;182(10):2823-30 [10781551.001]
  • (PMID = 16999687.001).
  • [ISSN] 1470-8728
  • [Journal-full-title] The Biochemical journal
  • [ISO-abbreviation] Biochem. J.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Isoenzymes; EC 1.1.1.1 / Alcohol Dehydrogenase
  • [Other-IDs] NLM/ PMC1820810
  •  go-up   go-down


81. Powers RH, Dean DE: Evaluation of potential lactate/lactate dehydrogenase interference with an enzymatic alcohol analysis. J Anal Toxicol; 2009 Oct;33(8):561-3
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The Connecticut Department of Public Safety laboratory recently addressed a legal challenge to a hospital alcohol dehydrogenase (ADH)-based serum ethanol determination based on the suggestion of interference by lactate dehydrogenase (LDH)-catalyzed oxidation of lactate.
  • Both ADH- and LDH-oxidations require NAD(+) (present in excess in the assay).
  • Calculation of the amount of LDH and ADH present in the ethanol assay suggest an ADH/LDH ratio of 200:1.

  • International Agency for Research on Cancer - Screening Group. diagnostics - Planning and Implementing Cervical Cancer Prevention and Control Programs: A Manual for Managers .
  • Hazardous Substances Data Bank. LACTIC ACID .
  • Hazardous Substances Data Bank. ETHANOL .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 19874670.001).
  • [ISSN] 1945-2403
  • [Journal-full-title] Journal of analytical toxicology
  • [ISO-abbreviation] J Anal Toxicol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Reagent Kits, Diagnostic; 33X04XA5AT / Lactic Acid; 3K9958V90M / Ethanol; EC 1.1.- / Lactate Dehydrogenases; EC 2.6.1.1 / Aspartate Aminotransferases; EC 2.6.1.2 / Alanine Transaminase
  •  go-up   go-down


82. Montjoy CA, Rahman A, Teba L: Ethylene glycol and methanol poisonings: case series and review. W V Med J; 2010 Sep-Oct;106(6):17-23
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • MATERIALS AND METHODS: A retrospective chart review of all adult patients admitted to our institution with a diagnosis of EG or MTH intoxication during a 15-year period was done.
  • Age, sex, EG and MTH levels, degree of acidosis, initial anion and osmolar gaps, renal dysfunction, length of stay in the hospital, need for dialysis, and ADH (alcohol dehydrogenase) blockade treatment were recorded.
  • One must rely on laboratory data for clues in making a diagnosis of intoxication.
  • [MeSH-minor] Acidosis / diagnosis. Adult. Female. Humans. Male. Middle Aged. Poisoning / diagnosis. Poisoning / physiopathology. Retrospective Studies

  • Hazardous Substances Data Bank. ETHYLENE GLYCOL .
  • Hazardous Substances Data Bank. METHANOL .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 21928557.001).
  • [ISSN] 0043-3284
  • [Journal-full-title] The West Virginia medical journal
  • [ISO-abbreviation] W V Med J
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Solvents; FC72KVT52F / Ethylene Glycol; Y4S76JWI15 / Methanol
  •  go-up   go-down


83. Larriba MJ, Valle N, Pálmer HG, Ordóñez-Morán P, Alvarez-Díaz S, Becker KF, Gamallo C, de Herreros AG, González-Sancho JM, Muñoz A: The inhibition of Wnt/beta-catenin signalling by 1alpha,25-dihydroxyvitamin D3 is abrogated by Snail1 in human colon cancer cells. Endocr Relat Cancer; 2007 Mar;14(1):141-51
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The transcription factor Snail1 represses VDR expression and we demonstrate here that Snail1 also abolishes the nuclear export of beta-catenin induced by 1,25(OH)(2)D(3) in SW480-ADH cells.

  • MedlinePlus Health Information. consumer health - Vitamin D.
  • Hazardous Substances Data Bank. 1,25-DIHYDROXYCHOLECALCIFEROL .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17395983.001).
  • [ISSN] 1351-0088
  • [Journal-full-title] Endocrine-related cancer
  • [ISO-abbreviation] Endocr. Relat. Cancer
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Microfilament Proteins; 0 / Neuropeptides; 0 / Nuclear Proteins; 0 / Receptors, Calcitriol; 0 / Transcription Factors; 0 / Wnt Proteins; 0 / beta Catenin; 0 / dihydroxy-vitamin D3; 0 / ectodermal-neural cortex 1 protein; 0 / snail family transcription factors; 1406-16-2 / Vitamin D; FXC9231JVH / Calcitriol; Q0OZ0D9223 / seocalcitol
  •  go-up   go-down


84. Wu YC, Chen DR, Kuo SJ: Personal experience of ultrasound-guided 14-gauge core biopsy of breast tumor. Eur J Surg Oncol; 2006 Sep;32(7):715-8
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Personal experience of ultrasound-guided 14-gauge core biopsy of breast tumor.
  • AIM: This study was aimed to examine the efficacy of ultrasound-guided core needle biopsy of breast tumor and compared with the results of previous publications.
  • METHODS: From January 2001 to September 2003, 546 lesions in 513 consecutive patients with the identification of a tumor on ultrasound examination categorized belong and above C3 according to BIRADS (Breast Image Reporting and Data Systems).
  • There were 341 lesions with benign findings, 202 lesions with malignancy and 3 lesions with atypical ductal hyperplasia (ADH).
  • Two patients were "ADH-DCIS (ductal carcinoma in situ) underestimate", and three patients were "DCIS-IDC (invasive ductal carcinoma) underestimate".
  • The ADH underestimation rate was 40% (2/5) and DCIS underestimation rate was 43% (3/7).
  • CONCLUSIONS: Both palpable and impalpable breast lesions should be examined under image guidance and automated core biopsy is the technique of first choice.
  • Fourteen-gauge core biopsy can provide a definitive diagnosis in 99% of solid tumors in this series.
  • [MeSH-major] Biopsy, Fine-Needle. Breast Neoplasms / pathology. Ultrasonography, Interventional
  • [MeSH-minor] Adolescent. Adult. Aged. Aged, 80 and over. Breast / pathology. False Negative Reactions. Female. Humans. Middle Aged. Sensitivity and Specificity

  • MedlinePlus Health Information. consumer health - Breast Cancer.
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16769196.001).
  • [ISSN] 0748-7983
  • [Journal-full-title] European journal of surgical oncology : the journal of the European Society of Surgical Oncology and the British Association of Surgical Oncology
  • [ISO-abbreviation] Eur J Surg Oncol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  •  go-up   go-down


85. Toyama H, Chen ZW, Fukumoto M, Adachi O, Matsushita K, Mathews FS: Molecular cloning and structural analysis of quinohemoprotein alcohol dehydrogenase ADH-IIG from Pseudomonas putida HK5. J Mol Biol; 2005 Sep 9;352(1):91-104
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Molecular cloning and structural analysis of quinohemoprotein alcohol dehydrogenase ADH-IIG from Pseudomonas putida HK5.
  • Depending on the alcohols used as growth substrates, Pseudomonas putida HK5 produces two distinct quinohemoprotein alcohol dehydrogenases, ADH-IIB and ADH-IIG, both of which contain pyrroloquinoline quinone (PQQ) and heme c as the prosthetic groups but show different substrate specificities, especially for diol substrates.
  • Molecular cloning of the gene of ADH-IIB and its crystal structure are already reported.
  • Here, molecular cloning of the gene, qgdA, and solution of the three-dimensional structure of ADH-IIG are reported.
  • The crystal structure of ADH-IIG, determined at 2.2A resolution, shows that the overall structure and the amino acid residues involved in PQQ binding are quite similar to ADH-IIB and to another quinohemoprotein ADH, qhEDH from Comamonas testosteroni.
  • Apart from ADH-IIB and qhEDH, ADH-IIG has an extra 12-residue helix within loop 3 in the PQQ domain and an extra 3(10) helix in the C terminus of the cytochrome domain, and both helices appear parallel and linked by a hydrogen bond.
  • In the crystal structure of ADH-IIG with 1,2-propanediol, one of the hydroxyl groups of the substrate forms a hydrogen bond with O5 of PQQ and OD1 of Asp300, and the other interacts with a water molecule and with NE2 of Trp386, the corresponding residue of which is not found in ADH-IIB and qhEDH, and might be the residue responsible for making ADH-IIG prefer diol substrates.

  • COS Scholar Universe. author profiles.
  • Gene Ontology. gene/protein/disease-specific - Gene Ontology annotations from this paper .
  • The Lens. Cited by Patents in .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16061256.001).
  • [ISSN] 0022-2836
  • [Journal-full-title] Journal of molecular biology
  • [ISO-abbreviation] J. Mol. Biol.
  • [Language] eng
  • [Databank-accession-numbers] GENBANK/ AB204833
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Bacterial Proteins; 0 / Isoenzymes; 9007-43-6 / Cytochromes c; EC 1.1.- / Alcohol Oxidoreductases; EC 1.1.2.8 / alcohol dehydrogenase (acceptor)
  •  go-up   go-down


86. Edenberg HJ, Xuei X, Chen HJ, Tian H, Wetherill LF, Dick DM, Almasy L, Bierut L, Bucholz KK, Goate A, Hesselbrock V, Kuperman S, Nurnberger J, Porjesz B, Rice J, Schuckit M, Tischfield J, Begleiter H, Foroud T: Association of alcohol dehydrogenase genes with alcohol dependence: a comprehensive analysis. Hum Mol Genet; 2006 May 1;15(9):1539-49
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Linkage evidence indicated that gene(s) located on chromosome 4q, in the region of the alcohol dehydrogenase (ADH) genes, affected risk for alcoholism.
  • We genotyped 110 single nucleotide polymorphisms (SNPs) across the seven ADH genes and analyzed their association with alcoholism in a set of families with multiple alcoholic members, using the pedigree disequilibrium test.

  • Genetic Alliance. consumer health - Alcohol dependence.
  • MedlinePlus Health Information. consumer health - Alcoholism and Alcohol Abuse.
  • COS Scholar Universe. author profiles.
  • SciCrunch. OMIM: Data: Gene Annotation .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16571603.001).
  • [ISSN] 0964-6906
  • [Journal-full-title] Human molecular genetics
  • [ISO-abbreviation] Hum. Mol. Genet.
  • [Language] eng
  • [Grant] United States / NIAAA NIH HHS / AA / U10AA008401
  • [Publication-type] Journal Article; Multicenter Study; Research Support, N.I.H., Extramural
  • [Publication-country] England
  • [Chemical-registry-number] EC 1.1.1.1 / Alcohol Dehydrogenase
  •  go-up   go-down


87. Mossé IB, Mikhaĭlova ME, Glushkova IV, Kasinskaia SI, Anoshenko IP, Aksiutik TV, Tikhanovich NI, Kamysh NA: [Genetic monitoring of natural Drosophila populations in radiation contaminated regions of Belarus]. Radiats Biol Radioecol; 2006 May-Jun;46(3):287-95
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The dominant and recessive lethal mutation levels and genetic structure of the populations were analyzed for frequencies of F- and S-alleles of Adh (alcohol dehydrogenase) of Gpdh (glycerinophosphate dehydrogenase) and Sod (superoxide dismutase) loci.

  • FlyBase. FlyBase .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16869160.001).
  • [ISSN] 0869-8031
  • [Journal-full-title] Radiatsionnaia biologiia, radioecologiia
  • [ISO-abbreviation] Radiats Biol Radioecol
  • [Language] RUS
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] Russia (Federation)
  • [Chemical-registry-number] 0 / Drosophila Proteins; 0 / Radioactive Pollutants; EC 1.1.- / Alcohol Oxidoreductases; EC 1.1.- / Glycerolphosphate Dehydrogenase; EC 1.1.1.1 / ADH protein, Drosophila; EC 1.1.1.1 / Alcohol Dehydrogenase; EC 1.15.1.1 / Superoxide Dismutase
  •  go-up   go-down


88. Lutz P, Maring D, Tschampa HJ, Sauerbruch T: [A 25-year-old patient with colonic pseudo-obstruction, hyponatremia, hypertension, and diffuse pain]. Med Klin (Munich); 2010 Apr;105(4):267-72
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Shortly after admission, she developed generalized seizures while hyponatremia increased compatible with SIADH (syndrome of inadequate ADH secretion).
  • Since porphobilinogen deaminase activity was clearly decreased, diagnosis of acute intermittent porphyria could be confirmed.
  • CONCLUSION: This case shows how definite diagnosis of this illness is often delayed because of its rarity and the variety of its possible symptoms and signs.
  • [MeSH-major] Colonic Pseudo-Obstruction / etiology. Hypertension / etiology. Hyponatremia / etiology. Pain / etiology. Porphyria, Acute Intermittent / diagnosis
  • [MeSH-minor] Adult. Delayed Diagnosis. Diagnosis, Differential. Epilepsy, Generalized / etiology. Female. Humans. Hydroxymethylbilane Synthase. Inappropriate ADH Syndrome / diagnosis

  • MedlinePlus Health Information. consumer health - High Blood Pressure.
  • MedlinePlus Health Information. consumer health - Pain.
  • COS Scholar Universe. author profiles.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Ann Neurol. 2005 Apr;57(4):581-4 [15786449.001]
  • [Cites] Medicine (Baltimore). 1967 Mar;46(2):217-23 [6027463.001]
  • [Cites] Clin Sci (Lond). 1982 Feb;62(2):183-91 [7053917.001]
  • [Cites] CMAJ. 2002 Apr 16;166(8):1056-62 [12002984.001]
  • [Cites] Am J Med Genet. 1996 Nov 11;65(4):269-73 [8923933.001]
  • [Cites] Clin Chem. 1989 Dec;35(12):2311-2 [2591048.001]
  • [Cites] Lancet. 2004 Feb 28;363(9410):705-6 [15001330.001]
  • [Cites] N Engl J Med. 1998 Jun 18;338(25):1853-4 [9634374.001]
  • [Cites] Nephrol Dial Transplant. 1992;7(10):986-90 [1331893.001]
  • [Cites] Proc Natl Acad Sci U S A. 1991 Dec 1;88(23):10912-5 [1961762.001]
  • [Cites] Lancet. 1997 May 31;349(9065):1613-7 [9174571.001]
  • [Cites] N Engl J Med. 1971 Jan 7;284(1):17-20 [5538584.001]
  • [Cites] Physiol Res. 2006;55 Suppl 2:S119-36 [17298216.001]
  • [Cites] J Intern Med. 1996 Oct;240(4):195-201 [8918510.001]
  • [Cites] Epilepsia. 2004 May;45(5):559-60 [15101839.001]
  • [Cites] Am J Med. 1966 Jul;41(1):149-62 [5939541.001]
  • [Cites] Ann Clin Biochem. 1998 Nov;35 ( Pt 6):726-32 [9838985.001]
  • [Cites] Medicine (Baltimore). 2005 Jan;84(1):48-60 [15643299.001]
  • [Cites] Epilepsia. 2008 May;49(5):934-6 [18454784.001]
  • [Cites] Ann Intern Med. 2005 Mar 15;142(6):439-50 [15767622.001]
  • [Cites] Am J Psychiatry. 1985 Dec;142(12):1430-6 [4073306.001]
  • [Cites] Cell Mol Biol (Noisy-le-grand). 2009 Jul 01;55(2):147-51 [19656463.001]
  • [Cites] Clin Neuropharmacol. 2005 Sep-Oct;28(5):243-4 [16239766.001]
  • [Cites] Arch Intern Med. 1993 Sep 13;153(17):2004-8 [8357285.001]
  • [Cites] Metabolism. 1964 May;13:396-406 [14169218.001]
  • (PMID = 20455047.001).
  • [ISSN] 1615-6722
  • [Journal-full-title] Medizinische Klinik (Munich, Germany : 1983)
  • [ISO-abbreviation] Med. Klin. (Munich)
  • [Language] ger
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Germany
  • [Chemical-registry-number] EC 2.5.1.61 / Hydroxymethylbilane Synthase
  •  go-up   go-down


89. Szabo-Moskal J, Lasek W, Kozłowska R, Sir J, Serafin Z: [The role of preoperative hooked-wire localization of occult lesions for early detection of breast cancer]. Ginekol Pol; 2005 Jan;76(1):15-9
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [The role of preoperative hooked-wire localization of occult lesions for early detection of breast cancer].
  • OBJECTIVES: Nonpalpable small lesions, usually few millimeters in diameter, consist an important problem in the early diagnostics of breast cancer.
  • DESIGN: An aim of this paper was an evaluation of usefulness of preoperative hooked-wire localization of such lesions for the detection of breast cancer.
  • RESULTS: Histologic examination of the localized lesions revealed benign changes in 71.2%, atypical ductal hyperplasia in 4.6% and cancer in 24.2%.
  • [MeSH-major] Biopsy, Needle / methods. Breast Neoplasms / pathology. Carcinoma in Situ / pathology. Carcinoma, Ductal, Breast / pathology

  • Genetic Alliance. consumer health - Breast Cancer.
  • MedlinePlus Health Information. consumer health - Breast Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 15844561.001).
  • [ISSN] 0017-0011
  • [Journal-full-title] Ginekologia polska
  • [ISO-abbreviation] Ginekol. Pol.
  • [Language] pol
  • [Publication-type] Journal Article
  • [Publication-country] Poland
  •  go-up   go-down


90. Guo Y, Wu XQ, Zhang C, Liao ZX, Wu Y, Xia ZY, Wang H: Effect of indole-3-carbinol on ethanol-induced liver injury and acetaldehyde-stimulated hepatic stellate cells activation using precision-cut rat liver slices. Clin Exp Pharmacol Physiol; 2010 Dec;37(12):1107-13
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • In I3C groups, cytochrome P450 (CYP) 2E1 activities were inhibited by 40.9-51.8%, whereas alcohol dehydrogenase (ADH) activity was enhanced 1.6-fold compared with the ethanol-treated group.

  • Gene Ontology. gene/protein/disease-specific - Gene Ontology annotations from this paper .
  • Hazardous Substances Data Bank. ETHANOL .
  • Hazardous Substances Data Bank. ACETALDEHYDE .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • The Lens. Cited by Patents in .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] © 2010 The Authors. Clinical and Experimental Pharmacology and Physiology © 2010 Blackwell Publishing Asia Pty Ltd.
  • [CommentIn] Clin Exp Pharmacol Physiol. 2012 Jul;39(7):642 [22738366.001]
  • (PMID = 20880187.001).
  • [ISSN] 1440-1681
  • [Journal-full-title] Clinical and experimental pharmacology & physiology
  • [ISO-abbreviation] Clin. Exp. Pharmacol. Physiol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Australia
  • [Chemical-registry-number] 0 / Actins; 0 / Indoles; 0 / Tissue Inhibitor of Metalloproteinase-1; 0 / Transforming Growth Factor beta1; 0 / smooth muscle actin, rat; 3K9958V90M / Ethanol; 9007-34-5 / Collagen; C11E72455F / indole-3-carbinol; EC 3.4.24.7 / Matrix Metalloproteinase 1; GO1N1ZPR3B / Acetaldehyde
  •  go-up   go-down


91. Lysak MA, Koch MA, Beaulieu JM, Meister A, Leitch IJ: The dynamic ups and downs of genome size evolution in Brassicaceae. Mol Biol Evol; 2009 Jan;26(1):85-98
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Evolution of GS was analyzed within a phylogenetic framework based on gene trees built from five data sets (matK, chs, adh, trnLF, and ITS).

  • COS Scholar Universe. author profiles.
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 18842687.001).
  • [ISSN] 1537-1719
  • [Journal-full-title] Molecular biology and evolution
  • [ISO-abbreviation] Mol. Biol. Evol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  •  go-up   go-down


92. Robinson D: Nocturia in women. Int J Clin Pract Suppl; 2007 Sep;(155):23-31
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • When evaluating women complaining of nocturia a urinary diary and clinical examination is integral to making the diagnosis prior to commencing treatment.
  • Should this conservative approach fail then treatment with antimuscarinic agents or desmopressin, a nona-peptide analogue of anti-diuretic hormone (ADH) is often helpful.

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 17727576.001).
  • [ISSN] 1368-504X
  • [Journal-full-title] International journal of clinical practice. Supplement
  • [ISO-abbreviation] Int J Clin Pract Suppl
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Number-of-references] 58
  •  go-up   go-down


93. Thompson CE, Fernandes CL, de Souza ON, de Freitas LB, Salzano FM: Evaluation of the impact of functional diversification on Poaceae, Brassicaceae, Fabaceae, and Pinaceae alcohol dehydrogenase enzymes. J Mol Model; 2010 May;16(5):919-28
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Several studies point to functional diversification of the ADH, with evidence of its importance, in different organisms, in the ethanol, norepinephrine, dopamine, serotonin, and bile acid metabolism.
  • The main objective of this article is to establish a correlation between the functional diversification occurring in the plant alcohol dehydrogenase family and the three-dimensional structures predicted for 17 ADH belonging to Poaceae, Brassicaceae, Fabaceae, and Pinaceae botanical families.

  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Proteomics. 2007 Aug;7(16):2947-62 [17654459.001]
  • [Cites] Mol Biol Evol. 1999 Aug;16(8):1086-97 [10474904.001]
  • [Cites] Proc Natl Acad Sci U S A. 1986 Mar;83(5):1408-12 [2937058.001]
  • [Cites] Proc Natl Acad Sci U S A. 2009 Jan 20;106(3):779-84 [19131516.001]
  • [Cites] Biochemistry. 2003 Mar 18;42(10):2907-15 [12627956.001]
  • [Cites] Proc Natl Acad Sci U S A. 1994 May 24;91(11):4980-4 [8197167.001]
  • [Cites] Plant Physiol. 1997 Feb;113(2):397-402 [12223614.001]
  • [Cites] Curr Opin Struct Biol. 1995 Dec;5(6):775-83 [8749365.001]
  • [Cites] Genetics. 2000 Aug;155(4):1913-26 [10924485.001]
  • [Cites] Proc Natl Acad Sci U S A. 1996 Oct 15;93(21):11735-9 [8876206.001]
  • [Cites] J Mol Biol. 2006 Jul 21;360(4):893-906 [16784753.001]
  • [Cites] Mol Biol Evol. 1998 May;15(5):552-9 [9580984.001]
  • [Cites] Eur J Biochem. 2004 May;271(9):1660-70 [15096205.001]
  • [Cites] Eur J Biochem. 1993 Aug 15;216(1):49-56 [8365416.001]
  • [Cites] Proc Natl Acad Sci U S A. 1992 Nov 15;89(22):10915-9 [1438297.001]
  • [Cites] Plant Mol Biol. 2005 May;58(2):283-94 [16027979.001]
  • [Cites] Appl Bioinformatics. 2002;1(2):107-8 [15130850.001]
  • [Cites] Nature. 1992 Mar 5;356(6364):83-5 [1538787.001]
  • [Cites] Annu Rev Biophys Bioeng. 1977;6:151-76 [326146.001]
  • [Cites] Evol Bioinform Online. 2008 Jun 13;4:203-16 [19204818.001]
  • [Cites] Mol Biol Evol. 2000 Oct;17(10):1483-98 [11018155.001]
  • [Cites] Proc Natl Acad Sci U S A. 2001 Jan 16;98(2):531-6 [11149938.001]
  • [Cites] Annu Rev Genet. 1985;19:297-323 [3002240.001]
  • [Cites] Plant Mol Biol. 1995 Jul;28(4):739-50 [7647304.001]
  • [Cites] Proc Natl Acad Sci U S A. 1993 Jun 1;90(11):5095-9 [8506356.001]
  • [Cites] Annu Rev Biophys Biomol Struct. 2000;29:291-325 [10940251.001]
  • [Cites] J Biol Chem. 1979 May 10;254(9):3458-61 [218969.001]
  • [Cites] J Mol Biol. 1974 Jan 5;82(1):1-14 [4818482.001]
  • [Cites] Bioinformatics. 2002 Mar;18(3):500-1 [11934757.001]
  • [Cites] Science. 1969 Oct 31;166(3905):626-7 [17778204.001]
  • [Cites] J Mol Biol. 1990 Oct 5;215(3):403-10 [2231712.001]
  • [Cites] Arch Biochem Biophys. 1993 Nov 15;307(1):85-90 [8239669.001]
  • [Cites] Proc Natl Acad Sci U S A. 1996 Nov 12;93(23):13020-3 [8917537.001]
  • [Cites] J Biomed Sci. 2001 Jan-Feb;8(1):71-6 [11173978.001]
  • [Cites] Electrophoresis. 1997 Dec;18(15):2714-23 [9504803.001]
  • [Cites] Mol Biol Evol. 1998 Nov;15(11):1420-9 [12572606.001]
  • [Cites] Trends Biochem Sci. 1998 Oct;23(10):403-5 [9810230.001]
  • [Cites] Plant Physiol. 1994 Aug;105(4):1075-87 [7972489.001]
  • [Cites] Ann Ist Super Sanita. 2006;42(1):8-16 [16801720.001]
  • [Cites] Nucleic Acids Res. 1994 Nov 11;22(22):4673-80 [7984417.001]
  • [Cites] J Comput Biol. 2001;8(3):221-34 [11535174.001]
  • [Cites] Nucleic Acids Res. 1997 Sep 1;25(17):3389-402 [9254694.001]
  • [Cites] J Mol Biol. 1993 Dec 5;234(3):779-815 [8254673.001]
  • [Cites] Gene. 2007 Jul 1;396(1):108-15 [17433574.001]
  • (PMID = 19834749.001).
  • [ISSN] 0948-5023
  • [Journal-full-title] Journal of molecular modeling
  • [ISO-abbreviation] J Mol Model
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Germany
  • [Chemical-registry-number] EC 1.1.1.1 / Alcohol Dehydrogenase
  •  go-up   go-down


94. Emerson RO, Sage EH, Ghosh JG, Clark JI: Chaperone-like activity revealed in the matricellular protein SPARC. J Cell Biochem; 2006 Jul 1;98(4):701-5
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Unfolding and aggregation of the chaperone target protein, alcohol dehydrogenase (ADH), were initiated at 50 degrees C.
  • SPARC inhibited the thermal aggregation of ADH in a concentration-dependent manner, with maximal inhibition at a 1:4 molar ratio of SPARC:ADH.

  • COS Scholar Universe. author profiles.
  • NCI CPTAC Assay Portal. NCI CPTAC Assay Portal .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Copyright] 2006 Wiley-Liss, Inc.
  • (PMID = 16598771.001).
  • [ISSN] 0730-2312
  • [Journal-full-title] Journal of cellular biochemistry
  • [ISO-abbreviation] J. Cell. Biochem.
  • [Language] ENG
  • [Grant] United States / NEI NIH HHS / EY / R01 EY004542; United States / NEI NIH HHS / EY / EY13180; United States / NIGMS NIH HHS / GM / GM40711
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Molecular Chaperones; 0 / Osteonectin; 0 / alpha-Crystallins; 0 / beta-Crystallins; EC 1.1.1.1 / Alcohol Dehydrogenase
  •  go-up   go-down


95. Birley AJ, Whitfield JB, Neale MC, Duffy DL, Heath AC, Boomsma DI, Martin NG: Genetic time-series analysis identifies a major QTL for in vivo alcohol metabolism not predicted by in vitro studies of structural protein polymorphism at the ADH1B or ADH1C loci. Behav Genet; 2005 Sep;35(5):509-24
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Nearly all participants were subsequently genotyped on two polymorphic SNPs in the ADH1B and ADH1C loci known to affect in vitro ADH activity.
  • In the DZ pairs, 14 microsatellite markers covering a 20.5 cM region on chromosome 4 that includes the ADH gene family were assessed, Variation in the timed series of autocorrelated blood and breath alcohol readings was studied using a bivariate simplex design.
  • The contribution of a quantitative trait locus (QTL) or QTL's linked to the ADH region was estimated via a mixture of likelihoods weighted by identity-by-descent probabilities.
  • This implies that there is a major determinant of variation for in vivo alcohol metabolism in the ADH region that is not accounted for by these polymorphisms.

  • MedlinePlus Health Information. consumer health - Alcoholism and Alcohol Abuse.
  • COS Scholar Universe. author profiles.
  • Hazardous Substances Data Bank. ETHANOL .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16184481.001).
  • [ISSN] 0001-8244
  • [Journal-full-title] Behavior genetics
  • [ISO-abbreviation] Behav. Genet.
  • [Language] eng
  • [Grant] United States / NIAAA NIH HHS / AA / AA013321; United States / NIAAA NIH HHS / AA / AA013326; United States / NIAAA NIH HHS / AA / AA014041; United States / NIAAA NIH HHS / AA / AA077281; United States / NIMH NIH HHS / MH / MH-65322
  • [Publication-type] Clinical Trial; Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Twin Study
  • [Publication-country] United States
  • [Chemical-registry-number] 3K9958V90M / Ethanol; EC 1.1.1.1 / ADH1B protein, human; EC 1.1.1.1 / ADH1C protein, human; EC 1.1.1.1 / Alcohol Dehydrogenase
  •  go-up   go-down


96. Weber WP, Engelberger S, Viehl CT, Zanetti-Dallenbach R, Kuster S, Dirnhofer S, Wruk D, Oertli D, Marti WR: Accuracy of frozen section analysis versus specimen radiography during breast-conserving surgery for nonpalpable lesions. World J Surg; 2008 Dec;32(12):2599-606
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Accuracy of frozen section analysis versus specimen radiography during breast-conserving surgery for nonpalpable lesions.
  • BACKGROUND: Whereas specimen radiography (SR) is an established strategy for intraoperative resection margin analysis during breast-conserving surgery for nonpalpable lesions, the use of frozen section analysis (FSA) is still a matter of debate.
  • METHODS: A retrospective review was conducted of 115 consecutive operations in which the two objectives sought were the excision of nonpalpable malignant lesions and breast conservation.
  • Breast surgery was performed in the Gynecology and the Surgery Departments at the Basel University Hospital Breast Center.
  • Whereas one department preferably uses SR for intraoperative margin assessments of lesions involving ductal carcinoma in situ (DCIS) or atypical ductal hyperplasia, the other uses FSA to increase the rate of complete removal of these lesions with a single procedure.
  • RESULTS: Intraoperative resection margin assessments were performed with FSA in 80 and SR in 35 of a total of 115 operations performed on 111 patients with pTis, pT1, or pT2 nonpalpable breast cancer.
  • CONCLUSIONS: The present results suggest that FSA may be more accurate than SR for analyzing intraoperative resection margins during breast-conserving surgery for nonpalpable lesions.
  • [MeSH-major] Breast Neoplasms / diagnostic imaging. Breast Neoplasms / pathology. Carcinoma / diagnostic imaging. Carcinoma / pathology. Frozen Sections. Mammography

  • MedlinePlus Health Information. consumer health - Breast Cancer.
  • MedlinePlus Health Information. consumer health - Mammography.
  • NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Surg Oncol. 2003 Dec;12(4):221-40 [14998563.001]
  • [Cites] J Am Coll Surg. 2005 Dec;201(6):855-61 [16310688.001]
  • [Cites] N Engl J Med. 1995 Apr 6;332(14):907-11 [7877647.001]
  • [Cites] JAMA. 1996 Mar 27;275(12):913-8 [8598618.001]
  • [Cites] N Engl J Med. 1989 Mar 30;320(13):822-8 [2927449.001]
  • [Cites] Ann Surg Oncol. 2007 Apr;14(4):1458-71 [17260108.001]
  • [Cites] Eur J Cancer. 1990;26(6):668-70 [2144152.001]
  • [Cites] Cancer. 1980 Aug 15;46(4 Suppl):980-2 [7397677.001]
  • [Cites] J Clin Oncol. 1996 May;14(5):1558-64 [8622072.001]
  • [Cites] Semin Surg Oncol. 1991 Sep-Oct;7(5):257-60 [1775809.001]
  • [Cites] N Engl J Med. 2002 Oct 17;347(16):1233-41 [12393820.001]
  • [Cites] J Am Coll Surg. 2005 Aug;201(2):194-8 [16038815.001]
  • [Cites] Surg Oncol. 1999 Aug;8(2):77-84 [10732959.001]
  • [Cites] Clin Radiol. 2006 Sep;61(9):789-96 [16905388.001]
  • [Cites] Cancer. 1980 Aug 15;46(4 Suppl):950-6 [7397674.001]
  • [Cites] J Natl Cancer Inst. 2002 Oct 16;94(20):1546-54 [12381707.001]
  • [Cites] World J Surg. 2005 Apr;29(4):495-9 [15770379.001]
  • [Cites] AJR Am J Roentgenol. 1994 Jan;162(1):33-6 [8273685.001]
  • [Cites] Cancer J Sci Am. 1997 Sep-Oct;3(5):273-7 [9327150.001]
  • (PMID = 18836763.001).
  • [ISSN] 0364-2313
  • [Journal-full-title] World journal of surgery
  • [ISO-abbreviation] World J Surg
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  •  go-up   go-down


97. Zagouri F, Sergentanis TN, Nonni A, Koulocheri D, Domeyer P, Dardamanis D, Michalopoulos NV, Pararas N, Gounaris A, Zografos GC: Secondary breast lymphoma diagnosed by vacuum-assisted breast biopsy: a case report. J Med Case Rep; 2007;1:113
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Secondary breast lymphoma diagnosed by vacuum-assisted breast biopsy: a case report.
  • INTRODUCTION: Breast lymphoma, either as a manifestation of primary extranodal disease or as secondary involvement, is a rare malignancy, and its diagnosis, prognosis, and treatment have not been clearly defined.
  • On the other hand, Vacuum-assisted breast biopsy (VABB) is a minimally invasive technique with ever-growing use for the diagnosis of mammographically detected, non-palpable breast lesions.
  • She had a positive family history for breast cancer and a history of atypical ductal hyperplasia in the ipsilateral breast four years ago.
  • With the suspicion of breast cancer, mammographically guided VABB with 11-gauge probe (on the stereotactic Fisher's table) was performed.
  • VABB made the diagnosis of a non-Hodgkin, grade II, B-cell germinal-center lymphoma.
  • CONCLUSION: This is the first case in the literature demonstrating the successful diagnosis of breast lymphoma by VABB, irrespectively of the level of clinical suspicion.
  • In general, lymphoma should never be omitted in the differential diagnosis, since no pathognomonic radiologic findings exist for its diagnosis.

  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] Cancer. 2002 Jan 1;94(1):6-13 [11815954.001]
  • [Cites] Int J Cancer. 2002 Jun 20;99(6):853-9 [12115488.001]
  • [Cites] Cancer. 2003 Apr 15;97(8):1824-31 [12673707.001]
  • [Cites] Eur J Cancer. 2003 Aug;39(12):1676-83 [12888361.001]
  • [Cites] Acta Cytol. 2003 Sep-Oct;47(5):739-43 [14526671.001]
  • [Cites] Diagn Cytopathol. 2004 May;30(5):332-40 [15108231.001]
  • [Cites] Clin Lymphoma. 2005 Jun;6(1):37-42 [15989705.001]
  • [Cites] Acta Radiol. 2005 Nov;46(7):690-5 [16372687.001]
  • [Cites] Acta Cytol. 2005 Nov-Dec;49(6):661-5 [16450909.001]
  • [Cites] Ann Diagn Pathol. 2006 Jun;10(3):144-8 [16730308.001]
  • [Cites] World J Surg Oncol. 2007;5:53 [17501997.001]
  • [Cites] Radiology. 1994 Jul;192(1):157-60 [8208929.001]
  • [Cites] Cancer. 1998 Feb 15;82(4):679-88 [9477100.001]
  • [Cites] Cancer. 1993 Feb 15;71(4):1258-65 [8435803.001]
  • (PMID = 17956632.001).
  • [ISSN] 1752-1947
  • [Journal-full-title] Journal of medical case reports
  • [ISO-abbreviation] J Med Case Rep
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC2169247
  •  go-up   go-down


98. Fukuhara K, Omura T, Katsuragi K, Osugi H, Lee S, Kishida S: [A case of severe electrolytic disorder after adjuvant chemotherapy for esophageal cancer]. Gan To Kagaku Ryoho; 2009 Aug;36(8):1359-61
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [A case of severe electrolytic disorder after adjuvant chemotherapy for esophageal cancer].
  • An electrolytic disorder was found on day 2, and her consciousness became unclear on 5 day (respective Na, K and Cl values of 113, 2.2 and 67 mEq/L in blood).
  • As the b2-microglobulin level was high in urine and the ADH level in blood was normal, cisplatinum nephrotoxicity was thought to make this accident.
  • Electrolytes should be checked carefully for disorder during chemotherapy.


99. Zhang L, Xu Z, Sun X, Dong S: A novel alcohol dehydrogenase biosensor based on solid-state electrogenerated chemiluminescence by assembling dehydrogenase to Ru(bpy)(3)2+-Au nanoparticles aggregates. Biosens Bioelectron; 2007 Jan 15;22(6):1097-100
PDF icon [Fulltext service] Get downloadable fulltext PDFs of articles closely matching to this article, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Based on electrogenerated chemiluminescence (ECL), a novel method for fabrication of alcohol dehydrogenase (ADH) biosensor by self-assembling ADH to Ru(bpy)(3)(2+)-AuNPs aggregates (Ru-AuNPs) on indium tin oxide (ITO) electrode surface has been developed.

  • Hazardous Substances Data Bank. ETHANOL .
  • Hazardous Substances Data Bank. GOLD, ELEMENTAL .
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • (PMID = 16815003.001).
  • [ISSN] 0956-5663
  • [Journal-full-title] Biosensors & bioelectronics
  • [ISO-abbreviation] Biosens Bioelectron
  • [Language] eng
  • [Publication-type] Evaluation Studies; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Enzymes, Immobilized; 3K9958V90M / Ethanol; 7440-57-5 / Gold; EC 1.1.1.1 / Alcohol Dehydrogenase; MLT4718TJW / Rubidium
  •  go-up   go-down


100. Wells CA, El-Ayat GA: Non-operative breast pathology: apocrine lesions. J Clin Pathol; 2007 Dec;60(12):1313-20
PDF icon [Fulltext service] Download fulltext PDF of this article and others, as many as you want.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Non-operative breast pathology: apocrine lesions.
  • Apocrine metaplasia is a very common finding in the female breast after the age of 25.
  • It is so common that many people regard it as a normal component of the breast.
  • The apocrine cell does, however, contribute to a number of different breast lesions, some of which are very taxing diagnostically; apocrine variants of both in-situ and invasive cancer are encountered.
  • This review considers the common apocrine metaplastic lesions seen in fibrocystic change as well as apocrine adenoma, apocrine change within sclerosing adenosis, atypical apocrine lesions and apocrine malignancies.
  • [MeSH-major] Apocrine Glands / pathology. Breast Neoplasms / pathology. Precancerous Conditions / pathology
  • [MeSH-minor] Adenoma / pathology. Biopsy. Breast / pathology. Carcinoma, Ductal, Breast / pathology. Female. Fibrocystic Breast Disease / pathology. Humans. Hyperplasia / pathology. Metaplasia / pathology. Neoplasm Invasiveness

  • MedlinePlus Health Information. consumer health - Breast Cancer.
  • [Email] Email this result item
    Email the results to the following email address:   [X] Close
  • [Cites] J Clin Pathol. 1999 Nov;52(11):838-41 [10690175.001]
  • [Cites] APMIS. 2006 May;114(5):352-8 [16725011.001]
  • [Cites] Am J Pathol. 2001 Jan;158(1):207-14 [11141494.001]
  • [Cites] Hum Pathol. 2001 May;32(5):487-93 [11381366.001]
  • [Cites] Histopathology. 2001 Aug;39(2):198-202 [11493337.001]
  • [Cites] J Clin Pathol. 2002 Jan;55(1):14-6 [11825917.001]
  • [Cites] Virchows Arch. 2002 Nov;441(5):449-55 [12447674.001]
  • [Cites] Breast J. 2003 Jul-Aug;9(4):335-6 [12846876.001]
  • [Cites] Cancer. 2003 Aug 15;98(4):703-11 [12910513.001]
  • [Cites] Adv Anat Pathol. 2004 Jan;11(1):1-9 [14676636.001]
  • [Cites] Cancer. 1968 Apr;21(4):756-63 [4296681.001]
  • [Cites] Cancer. 1976 Jun;37(6):2891-905 [949710.001]
  • [Cites] Cancer. 1980 Dec 1;46(11):2463-71 [6254632.001]
  • [Cites] Surg Gynecol Obstet. 1981 Apr;152(4):437-40 [7209771.001]
  • [Cites] Am J Pathol. 1983 Feb;110(2):105-12 [6130702.001]
  • [Cites] Am J Surg Pathol. 1983 Jul;7(5):451-61 [6351647.001]
  • [Cites] N Engl J Med. 1985 Jan 17;312(3):146-51 [3965932.001]
  • [Cites] Eur J Cancer Clin Oncol. 1985 Sep;21(9):1047-50 [4065177.001]
  • [Cites] Am J Pathol. 1986 Jun;123(3):532-41 [3717305.001]
  • [Cites] Ann N Y Acad Sci. 1986;464:262-74 [3524352.001]
  • [Cites] Hum Pathol. 1987 Apr;18(4):381-6 [3557440.001]
  • [Cites] Histopathology. 1987 Jun;11(6):603-10 [3305282.001]
  • [Cites] Histopathology. 1987 Mar;11(3):305-15 [2828217.001]
  • [Cites] Br J Cancer. 1987 Dec;56(6):814-9 [2829956.001]
  • [Cites] Breast Cancer Res Treat. 1988 Sep;12(1):37-44 [2848603.001]
  • [Cites] Hum Pathol. 1989 Mar;20(3):281-7 [2542151.001]
  • [Cites] Ann N Y Acad Sci. 1990;586:161-73 [2192632.001]
  • [Cites] Ann N Y Acad Sci. 1990;586:188-97 [2357000.001]
  • [Cites] Am J Clin Pathol. 1990 Oct;94(4):371-7 [2171320.001]
  • [Cites] Am J Surg Pathol. 1991 Jul;15(7):687-94 [2058763.001]
  • [Cites] Br J Cancer. 1991 Nov;64(5):953-5 [1931623.001]
  • [Cites] Hum Pathol. 1992 Jun;23(6):655-62 [1592388.001]
  • [Cites] Cancer Detect Prev. 1992;16(2):99-106 [1600527.001]
  • [Cites] CMAJ. 1992 Jul 1;147(1):45-9 [1393887.001]
  • [Cites] N Engl J Med. 1994 Jul 7;331(1):10-5 [8202095.001]
  • [Cites] Cytopathology. 1994 Apr;5(2):123-8 [8038424.001]
  • [Cites] Mod Pathol. 1994 Oct;7(8):813-8 [7838835.001]
  • [Cites] Cancer. 1996 Jun 15;77(12):2529-37 [8640702.001]
  • [Cites] Cancer Epidemiol Biomarkers Prev. 1996 Jan;5(1):29-32 [8770463.001]
  • [Cites] BMJ. 1997 Mar 29;314(7085):925-8 [9099114.001]
  • [Cites] Hum Pathol. 1997 Aug;28(8):967-73 [9269834.001]
  • [Cites] Cancer. 1997 Nov 1;80(9):1798-802 [9351550.001]
  • [Cites] Pathol Res Pract. 1997;193(11-12):753-8 [9521507.001]
  • [Cites] Virchows Arch. 1998 Dec;433(6):505-9 [9870682.001]
  • [Cites] Arch Pathol Lab Med. 1998 Dec;122(12):1053-5 [9870852.001]
  • [Cites] Lancet. 1999 May 22;353(9166):1742-5 [10347986.001]
  • [Cites] Breast. 2005 Feb;14(1):3-10 [15695074.001]
  • [Cites] Breast. 2005 Feb;14(1):37-41 [15695079.001]
  • [Cites] Breast Cancer Res. 2005;7(2):R296-305 [15743508.001]
  • [Cites] Oncogene. 2005 Jul 7;24(29):4660-71 [15897907.001]
  • [Cites] FEBS Lett. 2006 May 22;580(12):2935-44 [16631754.001]
  • [Cites] J Pathol. 2000 Jun;191(2):138-42 [10861572.001]
  • (PMID = 18042688.001).
  • [ISSN] 1472-4146
  • [Journal-full-title] Journal of clinical pathology
  • [ISO-abbreviation] J. Clin. Pathol.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Number-of-references] 62
  • [Other-IDs] NLM/ PMC2095572
  •  go-up   go-down






Advertisement