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1. De Vos M, Hayward BE, Charlton R, Taylor GR, Glaser AW, Picton S, Cole TR, Maher ER, McKeown CM, Mann JR, Yates JR, Baralle D, Rankin J, Bonthron DT, Sheridan E: PMS2 mutations in childhood cancer. J Natl Cancer Inst; 2006 Mar 1;98(5):358-61
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  • [Title] PMS2 mutations in childhood cancer.
  • Such families have instead been ascertained through childhood-onset cancers in homozygotes or through apparently sporadic colorectal cancer in heterozygotes.
  • [MeSH-minor] Adolescent. Arginine. Astrocytoma / genetics. Cafe-au-Lait Spots / genetics. Child. Colonic Polyps / genetics. DNA Repair. Female. Genetic Predisposition to Disease. Glioma / genetics. Great Britain / epidemiology. Humans. Leukemia, T-Cell / genetics. Lymphoma, B-Cell / genetics. Lymphoma, T-Cell / genetics. Male. Neoplasms, Second Primary / genetics. Pakistan / ethnology. Precursor Cell Lymphoblastic Leukemia-Lymphoma / genetics

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  • (PMID = 16507833.001).
  • [ISSN] 1460-2105
  • [Journal-full-title] Journal of the National Cancer Institute
  • [ISO-abbreviation] J. Natl. Cancer Inst.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / DNA-Binding Proteins; 94ZLA3W45F / Arginine; EC 3.6.1.- / Adenosine Triphosphatases; EC 3.6.1.- / PMS2 protein, human; EC 6.5.1.- / DNA Repair Enzymes
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2. Mizushima Y, Taki T, Shimada A, Yui Y, Hiraumi Y, Matsubara H, Watanabe M, Watanabe K, Kamitsuji Y, Hayashi Y, Tsukimoto I, Kobayashi R, Horibe K, Tawa A, Nakahata T, Adachi S: Prognostic significance of the BAALC isoform pattern and CEBPA mutations in pediatric acute myeloid leukemia with normal karyotype: a study by the Japanese Childhood AML Cooperative Study Group. Int J Hematol; 2010 Jun;91(5):831-7
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  • [Title] Prognostic significance of the BAALC isoform pattern and CEBPA mutations in pediatric acute myeloid leukemia with normal karyotype: a study by the Japanese Childhood AML Cooperative Study Group.
  • High BAALC (brain and acute leukemia, cytoplasmic) gene expression may indicate an adverse prognosis for adults who have acute myeloid leukemia (AML) and a normal karyotype, but its prognostic significance for pediatric AML cases is unclear.
  • Newly diagnosed AML patients with normal karyotype who were treated by the Japanese Childhood AML Cooperative Treatment Protocol AML 99 were analyzed in terms of their BAALC expression levels (n = 29), BAALC isoforms (n = 29), and CEBPA mutations (n = 49).
  • Thus, 1-5-6-8 isoform expression may be associated with an adverse prognosis in pediatric AML with normal karyotype.
  • [MeSH-major] Asian Continental Ancestry Group / genetics. CCAAT-Enhancer-Binding Proteins. Gene Expression Regulation, Leukemic. Leukemia, Myeloid, Acute / diagnosis. Mutation. Neoplasm Proteins


3. Russell LJ, Capasso M, Vater I, Akasaka T, Bernard OA, Calasanz MJ, Chandrasekaran T, Chapiro E, Gesk S, Griffiths M, Guttery DS, Haferlach C, Harder L, Heidenreich O, Irving J, Kearney L, Nguyen-Khac F, Machado L, Minto L, Majid A, Moorman AV, Morrison H, Rand V, Strefford JC, Schwab C, Tönnies H, Dyer MJ, Siebert R, Harrison CJ: Deregulated expression of cytokine receptor gene, CRLF2, is involved in lymphoid transformation in B-cell precursor acute lymphoblastic leukemia. Blood; 2009 Sep 24;114(13):2688-98
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  • [Title] Deregulated expression of cytokine receptor gene, CRLF2, is involved in lymphoid transformation in B-cell precursor acute lymphoblastic leukemia.
  • We report 2 novel, cryptic chromosomal abnormalities in precursor B-cell acute lymphoblastic leukemia (BCP-ALL): a translocation, either t(X;14)(p22;q32) or t(Y;14)(p11;q32), in 33 patients and an interstitial deletion, either del(X)(p22.33p22.33) or del(Y)(p11.32p11.32), in 64 patients, involving the pseudoautosomal region (PAR1) of the sex chromosomes.
  • The incidence of these abnormalities was 5% in childhood ALL (0.8% with the translocation, 4.2% with the deletion).
  • In Down syndrome (DS) ALL and 2 non-DS BCP-ALL cell lines, CRLF2 deregulation was associated with mutations of the JAK2 pseudokinase domain, suggesting oncogenic cooperation as well as highlighting a link between non-DS ALL and JAK2 mutations.
  • [MeSH-major] Cell Transformation, Neoplastic / genetics. Lymphocytes / pathology. Precursor B-Cell Lymphoblastic Leukemia-Lymphoma / genetics. Receptors, Cytokine / genetics


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4. Poppe B, De Paepe A, Speleman F: Acquired chromosomal rearrangements targeting selected transcription factors: contribution of molecular cytogenetic and expression analyses to the identification of clinically and biologically relevant subgroups in hematological malignancies. Verh K Acad Geneeskd Belg; 2007;69(1):47-64
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  • We contributed to an extensive analysis of the phenotypical and prognostic features of T-ALL characterized by HOX11L2 expression and identified HOX11L2 overexpression as one of the most frequent genetic defects in childhood T-ALL, associated with intermediate prognosis.
  • [MeSH-major] Chromosome Aberrations. Leukemia, Myeloid / classification. Leukemia, Myeloid / genetics. Translocation, Genetic

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  • (PMID = 17427874.001).
  • [ISSN] 0302-6469
  • [Journal-full-title] Verhandelingen - Koninklijke Academie voor Geneeskunde van België
  • [ISO-abbreviation] Verh. K. Acad. Geneeskd. Belg.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] Belgium
  • [Chemical-registry-number] 0 / Genetic Markers
  • [Number-of-references] 20
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5. Chowdhury S, Bandyopadhyay S, Mandal C, Chandra S, Mandal C: Flow-cytometric monitoring of disease-associated expression of 9-O-acetylated sialoglycoproteins in combination with known CD antigens, as an index for MRD in children with acute lymphoblastic leukaemia: a two-year longitudinal follow-up study. BMC Cancer; 2008;8:40
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  • [Title] Flow-cytometric monitoring of disease-associated expression of 9-O-acetylated sialoglycoproteins in combination with known CD antigens, as an index for MRD in children with acute lymphoblastic leukaemia: a two-year longitudinal follow-up study.
  • BACKGROUND: Over expression of 9-O-acetylated sialoglycoproteins (Neu5,9Ac2-GPs, abbreviated as OAcSGP) has been demonstrated as a disease-associated antigen on the lymphoblasts of childhood acute lymphoblastic leukaemia (ALL).
  • [MeSH-major] Antigens, CD / metabolism. Biomarkers, Tumor / metabolism. Neoplasm Recurrence, Local / diagnosis. Neoplasm Recurrence, Local / metabolism. Precursor Cell Lymphoblastic Leukemia-Lymphoma / metabolism. Sialoglycoproteins / metabolism

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  • (PMID = 18241334.001).
  • [ISSN] 1471-2407
  • [Journal-full-title] BMC cancer
  • [ISO-abbreviation] BMC Cancer
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Antigens, CD; 0 / Biomarkers, Tumor; 0 / Sialoglycoproteins
  • [Other-IDs] NLM/ PMC2268943
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6. Mejstrikova E, Volejnikova J, Fronkova E, Zdrahalova K, Kalina T, Sterba J, Jabali Y, Mihal V, Blazek B, Cerna Z, Prochazkova D, Hak J, Zemanova Z, Jarosova M, Oltova A, Sedlacek P, Schwarz J, Zuna J, Trka J, Stary J, Hrusak O: Prognosis of children with mixed phenotype acute leukemia treated on the basis of consistent immunophenotypic criteria. Haematologica; 2010 Jun;95(6):928-35
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  • [Title] Prognosis of children with mixed phenotype acute leukemia treated on the basis of consistent immunophenotypic criteria.
  • BACKGROUND: Mixed phenotype acute leukemia (MPAL) represents a diagnostic and therapeutic dilemma.
  • DESIGN AND METHODS: Simple immunophenotypic criteria were used to classify all cases of childhood acute leukemia in order to provide therapy directed against acute lymphoblastic leukemia or acute myeloid leukemia.
  • RESULTS: The incidences of MPAL were 28/582 and 4/107 for children treated with acute lymphoblastic leukemia and acute myeloid leukemia regimens, respectively.
  • In immunophenotypic principal component analysis, MPAL treated as T-cell acute lymphoblastic leukemia clustered between cases of non-mixed T-cell acute lymphoblastic leukemia and acute myeloid leukemia, while other MPAL cases were included in the respective non-mixed B-cell progenitor acute lymphoblastic leukemia or acute myeloid leukemia clusters.
  • Analogously, immunoglobulin/T-cell receptor gene rearrangements followed the expected pattern in patients treated as having acute myeloid leukemia (non-rearranged, 4/4) or as having B-cell progenitor acute lymphoblastic leukemia (rearranged, 20/20), but were missing in 3/5 analyzed cases of MPAL treated as having T-cell acute lymphobastic leukemia.
  • In patients who received acute lymphoblastic leukemia treatment, the 5-year event-free survival of the MPAL cases was worse than that of the non-mixed cases (53+/-10% and 76+/-2% at 5 years, respectively, P=0.0075), with a more pronounced difference among B lineage cases.
  • The small numbers of MPAL cases treated as T-cell acute lymphoblastic leukemia or as acute myeloid leukemia hampered separate statistics.
  • In B lineage leukemia, MPAL confers poorer prognosis.
  • However, our data do not justify a preferential use of current acute myeloid leukemia-based therapy in MPAL.
  • [MeSH-major] Immunophenotyping. Leukemia / diagnosis. Leukemia / therapy. Phenotype
  • [MeSH-minor] Adolescent. Child. Child, Preschool. Diagnosis, Differential. Follow-Up Studies. Humans. Infant. Infant, Newborn. Leukemia, Myeloid, Acute / diagnosis. Leukemia, Myeloid, Acute / immunology. Leukemia, Myeloid, Acute / therapy. Precursor Cell Lymphoblastic Leukemia-Lymphoma / diagnosis. Precursor Cell Lymphoblastic Leukemia-Lymphoma / immunology. Precursor Cell Lymphoblastic Leukemia-Lymphoma / therapy. Prognosis. Receptors, Antigen, T-Cell / immunology

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  • (PMID = 20145275.001).
  • [ISSN] 1592-8721
  • [Journal-full-title] Haematologica
  • [ISO-abbreviation] Haematologica
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Multicenter Study; Research Support, Non-U.S. Gov't
  • [Publication-country] Italy
  • [Chemical-registry-number] 0 / Receptors, Antigen, T-Cell
  • [Other-IDs] NLM/ PMC2878790
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7. Constantin T, Kálovics T, Ponyi A, Nagy E, Sallai K, Szabó L, Garami M, Müller J, Gergely P, Dankó K, Fekete G, Kálmánchey R: Prevalence of antiphospholipid and antinuclear antibodies in children with epilepsy. Med Sci Monit; 2009 Apr;15(4):CR164-9
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  • CONCLUSIONS: The clinical relevance of aPL tests in childhood are difficult to explain.
  • The wide spectrum of detected immunological alterations highlight the importance of the participation of pediatric rheumatologists in the management of patients with idiopathic epilepsy or with secondary induced autoimmune disease due to antiepileptic medications.

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  • (PMID = 19333200.001).
  • [ISSN] 1643-3750
  • [Journal-full-title] Medical science monitor : international medical journal of experimental and clinical research
  • [ISO-abbreviation] Med. Sci. Monit.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Poland
  • [Chemical-registry-number] 0 / Antibodies, Antinuclear; 0 / Antibodies, Antiphospholipid
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8. Weir DC, Glickman JN, Roiff T, Valim C, Leichtner AM: Variability of histopathological changes in childhood celiac disease. Am J Gastroenterol; 2010 Jan;105(1):207-12
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  • [Title] Variability of histopathological changes in childhood celiac disease.
  • Few related pediatric data exist.
  • CONCLUSIONS: Duodenal involvement in pediatric CD is frequently patchy and may show variable severity even within a single biopsy fragment.
  • Multiple endoscopic biopsies, including the duodenal bulb, should be obtained in suspected pediatric CD cases to maximize diagnostic yield.

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  • (PMID = 19809405.001).
  • [ISSN] 1572-0241
  • [Journal-full-title] The American journal of gastroenterology
  • [ISO-abbreviation] Am. J. Gastroenterol.
  • [Language] eng
  • [Grant] United States / NIDDK NIH HHS / DK / T32 DK07477
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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9. Stasevich I, Utskevich R, Kustanovich A, Litvinko N, Savitskaya T, Chernyavskaya S, Saharova O, Aleinikova O: Translocation (10;11)(p12;q23) in childhood acute myeloid leukemia: incidence and complex mechanism. Cancer Genet Cytogenet; 2006 Sep;169(2):114-20
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  • [Title] Translocation (10;11)(p12;q23) in childhood acute myeloid leukemia: incidence and complex mechanism.
  • This translocation represented 28% of all cases of childhood AML treated at our center in 2004, and 63% of AML with rearrangements of 11q23.
  • The median event-free survival of patients was 8.1 months, and we conclude that the t(10;11)(p12;q23) is associated with unfavorable prognosis in childhood acute myeloid leukemia.
  • [MeSH-major] Chromosomes, Human, Pair 10. Chromosomes, Human, Pair 11. Leukemia, Myeloid / genetics. Translocation, Genetic
  • [MeSH-minor] Acute Disease. Adolescent. Child. Child, Preschool. Chromosome Banding. Female. Humans. In Situ Hybridization, Fluorescence. Infant. Karyotyping. Male. Reverse Transcriptase Polymerase Chain Reaction

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  • (PMID = 16938568.001).
  • [ISSN] 0165-4608
  • [Journal-full-title] Cancer genetics and cytogenetics
  • [ISO-abbreviation] Cancer Genet. Cytogenet.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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10. Scalapino K, Arkachaisri T, Lucas M, Fertig N, Helfrich DJ, Londino AV Jr, Steen VD, Medsger TA Jr: Childhood onset systemic sclerosis: classification, clinical and serologic features, and survival in comparison with adult onset disease. J Rheumatol; 2006 May;33(5):1004-13
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Childhood onset systemic sclerosis: classification, clinical and serologic features, and survival in comparison with adult onset disease.
  • OBJECTIVE: To describe the differences between patients with systemic sclerosis (SSc) having childhood versus adult onset evaluated at a single medical center.
  • METHODS: Patients were divided into those with childhood onset (first SSc symptom or finding before age 16 yrs) and those with early adult and late adult onset.
  • RESULTS: One hundred eleven childhood onset SSc cases seen between 1960 and 2003 were compared with 2559 adult onset SSc cases (1087 with onset age 16-40 and 1472 with onset after age 40 yrs) first evaluated between 1972 and 2001.
  • Age distribution at onset was unimodal, suggesting that childhood disease is part of the spectrum of adult onset SSc.
  • A significantly greater proportion of childhood onset patients had overlap syndromes, most frequently with polymyositis-dermatomyositis (PM-DM), and skeletal muscle involvement.
  • Renal involvement was uncommon in childhood onset cases, and the frequency increased with age of onset.
  • Serum anti-PM-Scl and anti-U1RNP antibodies were detected significantly more frequently in childhood than in adult onset cases.
  • Survival was significantly better among childhood than all adult onset cases combined, but similar to survival in young adult onset SSc cases.

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  • [CommentIn] J Rheumatol. 2006 May;33(5):840-1 [16652415.001]
  • (PMID = 16583463.001).
  • [ISSN] 0315-162X
  • [Journal-full-title] The Journal of rheumatology
  • [ISO-abbreviation] J. Rheumatol.
  • [Language] eng
  • [Grant] United States / NCRR NIH HHS / RR / 5M01RR00056
  • [Publication-type] Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] Canada
  • [Chemical-registry-number] 0 / Autoantibodies; 0 / Autoantigens; EC 2.7.7.- / RNA Polymerase III; EC 3.1.- / Exoribonucleases; EC 3.1.- / Exosome Multienzyme Ribonuclease Complex; EC 3.1.13.- / EXOSC10 protein, human; EC 5.99.1.2 / DNA Topoisomerases, Type I
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11. Shi L, Lin Y, Wang Y, Ma LJ, Zheng T, Li LP, Li J, Pan Y, Wang TY: [Clinical features and changes of 5-hydroxytryptamine in children with vasovagal syncope]. Zhonghua Er Ke Za Zhi; 2010 Jan;48(1):39-43
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  • OBJECTIVE: To investigate clinical features of childhood vasovagal syncope (VVS) and the possible relationship between changes of plasma and platelet 5-hydroxytryptamine (5-HT) and childhood Vvs.

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  • (PMID = 20441702.001).
  • [ISSN] 0578-1310
  • [Journal-full-title] Zhonghua er ke za zhi = Chinese journal of pediatrics
  • [ISO-abbreviation] Zhonghua Er Ke Za Zhi
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] China
  • [Chemical-registry-number] 333DO1RDJY / Serotonin
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12. Kaspers GJ, Wijnands JJ, Hartmann R, Huismans L, Loonen AH, Stackelberg A, Henze G, Pieters R, Hählen K, Van Wering ER, Veerman AJ: Immunophenotypic cell lineage and in vitro cellular drug resistance in childhood relapsed acute lymphoblastic leukaemia. Eur J Cancer; 2005 Jun;41(9):1300-3
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  • [Title] Immunophenotypic cell lineage and in vitro cellular drug resistance in childhood relapsed acute lymphoblastic leukaemia.
  • At relapse, T-cell acute lymphoblastic leukaemia (ALL) has a worse patient outcome than B-cell precursor (BCP-) ALL.
  • [MeSH-major] Antineoplastic Agents / therapeutic use. Burkitt Lymphoma / drug therapy. Drug Resistance, Neoplasm / immunology. Leukemia-Lymphoma, Adult T-Cell / drug therapy. Precursor Cell Lymphoblastic Leukemia-Lymphoma / drug therapy

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  • (PMID = 15869873.001).
  • [ISSN] 0959-8049
  • [Journal-full-title] European journal of cancer (Oxford, England : 1990)
  • [ISO-abbreviation] Eur. J. Cancer
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Antineoplastic Agents
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13. Yourstone J, Lindholm T, Kristiansson M: Women who kill: a comparison of the psychosocial background of female and male perpetrators. Int J Law Psychiatry; 2008 Aug-Sep;31(4):374-83
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  • Areas of interest were psychosocial variables during childhood and at the time of the current crime.
  • Homicidal women had more severe childhood circumstances, but less aggressive childhood behaviour than did their male counterparts.

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  • (PMID = 18678408.001).
  • [ISSN] 0160-2527
  • [Journal-full-title] International journal of law and psychiatry
  • [ISO-abbreviation] Int J Law Psychiatry
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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14. Su XY, Della-Valle V, Andre-Schmutz I, Lemercier C, Radford-Weiss I, Ballerini P, Lessard M, Lafage-Pochitaloff M, Mugneret F, Berger R, Romana SP, Bernard OA, Penard-Lacronique V: HOX11L2/TLX3 is transcriptionally activated through T-cell regulatory elements downstream of BCL11B as a result of the t(5;14)(q35;q32). Blood; 2006 Dec 15;108(13):4198-201
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  • The t(5;14)(q35;q32) chromosomal translocation is specifically observed in up to 20% of childhood T-cell acute lymphoblastic leukemia (T-ALL).
  • [MeSH-major] Chromosomes, Human, Pair 14 / genetics. Chromosomes, Human, Pair 5 / genetics. DNA-Binding Proteins / genetics. Homeodomain Proteins / genetics. Leukemia-Lymphoma, Adult T-Cell / genetics. Oncogene Proteins / genetics. Oncogene Proteins, Fusion / genetics. Repressor Proteins / genetics. Translocation, Genetic. Tumor Suppressor Proteins / genetics

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  • (PMID = 16926283.001).
  • [ISSN] 0006-4971
  • [Journal-full-title] Blood
  • [ISO-abbreviation] Blood
  • [Language] eng
  • [Publication-type] Clinical Trial; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / BCL11B protein, human; 0 / DNA-Binding Proteins; 0 / Homeodomain Proteins; 0 / Oncogene Proteins; 0 / Oncogene Proteins, Fusion; 0 / Repressor Proteins; 0 / TLX3 protein, human; 0 / Tumor Suppressor Proteins
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15. Ozen S, Ruperto N, Dillon MJ, Bagga A, Barron K, Davin JC, Kawasaki T, Lindsley C, Petty RE, Prieur AM, Ravelli A, Woo P: EULAR/PReS endorsed consensus criteria for the classification of childhood vasculitides. Ann Rheum Dis; 2006 Jul;65(7):936-41
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  • [Title] EULAR/PReS endorsed consensus criteria for the classification of childhood vasculitides.
  • OBJECTIVE: To develop a widely accepted general classification for the vasculitides observed in children and specific and realistic classification criteria for common childhood vasculitides (Henoch-Schönlein purpura (HSP), Kawasaki disease (KD), childhood polyarteritis nodosa (PAN), Wegener's granulomatosis (WG), and Takayasu arteritis (TA)).
  • RESULTS: Consensus was reached to base the general working classification for childhood vasculitides on vessel size.
  • The small vessel disease was further subcategorised into "granulomatous" and "non-granulomatous."
  • Final criteria were developed to classify a child as HSP, KD, childhood PAN, WG, or TA, with changes introduced based on paediatric experience.

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  • (PMID = 16322081.001).
  • [ISSN] 0003-4967
  • [Journal-full-title] Annals of the rheumatic diseases
  • [ISO-abbreviation] Ann. Rheum. Dis.
  • [Language] eng
  • [Publication-type] Consensus Development Conference; Journal Article
  • [Publication-country] England
  • [Number-of-references] 23
  • [Other-IDs] NLM/ PMC1798210
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16. Wilzén A, Nilsson S, Sjöberg RM, Kogner P, Martinsson T, Abel F: The Phox2 pathway is differentially expressed in neuroblastoma tumors, but no mutations were found in the candidate tumor suppressor gene PHOX2A. Int J Oncol; 2009 Mar;34(3):697-705
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  • Neuroblastoma (NB), a tumor of the sympathetic nervous system, is the most common solid tumor in childhood.

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  • (PMID = 19212675.001).
  • [ISSN] 1019-6439
  • [Journal-full-title] International journal of oncology
  • [ISO-abbreviation] Int. J. Oncol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Greece
  • [Chemical-registry-number] 0 / Homeodomain Proteins; 0 / PHOX2A protein, human
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17. Taylor GM, Hussain A, Verhage V, Thompson PD, Fergusson WD, Watkins G, Lightfoot T, Harrison CJ, Birch JM, UKCCS Investigators: Strong association of the HLA-DP6 supertype with childhood leukaemia is due to a single allele, DPB1*0601. Leukemia; 2009 May;23(5):863-9
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  • [Title] Strong association of the HLA-DP6 supertype with childhood leukaemia is due to a single allele, DPB1*0601.
  • We previously reported that susceptibility to childhood B cell precursor ALL (BCP ALL) is associated with HLA-DPB1 alleles having glutamic acid (E) rather than lysine (K) in the P4 antigenic peptide-binding pocket.
  • Clustering approximately 90% of DPB1 alleles into DPB69E (DP2, 6, 8) and DPB69K (DP1, 3, 4) supertypes revealed that DP2 and DP8 are associated with BCP ALL, but DP6 is also associated with non-BCP leukaemia.
  • Here, we report that only one of seven alleles with the DP6 supertype (DPB1(*)0601) is associated with childhood leukaemia (leukaemia vs controls: odds ratio, 95% confidence interval [OR, CI]: 4.6, 2.0-10.4; corrected P=0.019), but not with childhood solid tumours or lymphomas.
  • DPB1(*)0601 is also significantly associated with leukaemia subtypes, including BCP ALL, Pro-B ALL, T-ALL and AML.
  • Sequencing the coding region of DPB1(*)0601 revealed an exon 1-4 haplotype [T-DEAV-KIL-RVI] shared with DPB1(*)0301 and 0901, but no evidence of germline mutations in childhood leukaemia.
  • These results suggest that the DPbeta0601 molecule may be functionally involved in childhood leukaemia.
  • Analysis of peptide binding and T-cell activation by DPbeta0601-peptide complexes should help determine its role in childhood leukaemia causation.
  • [MeSH-major] HLA-DP Antigens / genetics. Haplotypes / genetics. Leukemia, Myeloid, Acute / genetics. Precursor Cell Lymphoblastic Leukemia-Lymphoma / genetics

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  • (PMID = 19148140.001).
  • [ISSN] 1476-5551
  • [Journal-full-title] Leukemia
  • [ISO-abbreviation] Leukemia
  • [Language] eng
  • [Grant] United Kingdom / Cancer Research UK / /
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / HLA-DP Antigens; 0 / HLA-DP beta-Chains; 0 / HLA-DPB1 antigen
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18. Carlet M, Janjetovic K, Rainer J, Schmidt S, Panzer-Grümayer R, Mann G, Prelog M, Meister B, Ploner C, Kofler R: Expression, regulation and function of phosphofructo-kinase/fructose-biphosphatases (PFKFBs) in glucocorticoid-induced apoptosis of acute lymphoblastic leukemia cells. BMC Cancer; 2010 Nov 23;10:638
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  • [Title] Expression, regulation and function of phosphofructo-kinase/fructose-biphosphatases (PFKFBs) in glucocorticoid-induced apoptosis of acute lymphoblastic leukemia cells.
  • BACKGROUND: Glucocorticoids (GCs) cause apoptosis and cell cycle arrest in lymphoid cells and constitute a central component in the therapy of lymphoid malignancies, most notably childhood acute lymphoblastic leukemia (ALL).
  • RESULTS: Expression analyses in additional ALL children, non-leukemic individuals and leukemic cell lines confirmed frequent PFKFB2 induction by GC in most systems sensitive to GC-induced apoptosis, particularly T-ALL cells.
  • [MeSH-major] Antineoplastic Agents / therapeutic use. Apoptosis / drug effects. Dexamethasone / therapeutic use. Glucocorticoids / therapeutic use. Lymphocytes / drug effects. Phosphofructokinase-2 / metabolism. Precursor Cell Lymphoblastic Leukemia-Lymphoma / drug therapy

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  • (PMID = 21092265.001).
  • [ISSN] 1471-2407
  • [Journal-full-title] BMC cancer
  • [ISO-abbreviation] BMC Cancer
  • [Language] eng
  • [Grant] Austria / Austrian Science Fund FWF / / W 1101
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Antineoplastic Agents; 0 / Glucocorticoids; 0 / PFKFB4 protein, human; 7S5I7G3JQL / Dexamethasone; EC 2.7.1.105 / PFKFB2 protein, human; EC 2.7.1.105 / PFKFB3 protein, human; EC 2.7.1.105 / Phosphofructokinase-2; N12000U13O / Doxycycline
  • [Other-IDs] NLM/ PMC3002928
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19. Dickson NP, van Roode T, Herbison P, Paul C: Circumcision and risk of sexually transmitted infections in a birth cohort. J Pediatr; 2008 Mar;152(3):383-7
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  • OBJECTIVE: To determine the impact of early childhood circumcision on sexually transmitted infection (STI) acquisition to age 32 years.
  • CONCLUSIONS: These findings are consistent with recent population-based cross-sectional studies in developed countries, which found that early childhood circumcision does not markedly reduce the risk of the common STIs in the general population in such countries.

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  • [CommentIn] J Pediatr. 2008 Mar;152(3):A3 [18280826.001]
  • (PMID = 18280846.001).
  • [ISSN] 1097-6833
  • [Journal-full-title] The Journal of pediatrics
  • [ISO-abbreviation] J. Pediatr.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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20. Marques-Salles Tde J, Mkrtchyan H, Leite EP, Soares-Ventura EM, Muniz MT, Silva EF, Liehr T, Silva ML, Santos N: Complex karyotype defined by molecular cytogenetic FISH and M-FISH in an infant with acute megakaryoblastic leukemia and neurofibromatosis. Cancer Genet Cytogenet; 2010 Jul 15;200(2):167-9
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  • [Title] Complex karyotype defined by molecular cytogenetic FISH and M-FISH in an infant with acute megakaryoblastic leukemia and neurofibromatosis.
  • Acute myeloid leukemia in childhood is a heterogeneous group of diseases, and different epidemiologic factors are involved in the etiopathogenesis.
  • Genetic syndromes are one of the predisposing factors of acute myeloid leukemia (AML), including Down syndrome, Bloom syndrome, and neurofibromatosis.
  • Acute megakaryoblastic leukemia (AMKL) is the main subtype in Down syndrome infants, and acquired chromosomal anomalies are closely related to the physiopathology of the illness.
  • [MeSH-major] In Situ Hybridization, Fluorescence / methods. Leukemia, Megakaryoblastic, Acute / genetics. Neurofibromatoses / genetics
  • [MeSH-minor] Female. Genes, p53. Histone-Lysine N-Methyltransferase. Humans. Infant. Karyotyping. Myeloid-Lymphoid Leukemia Protein / genetics

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  • [Copyright] Copyright (c) 2010 Elsevier Inc. All rights reserved.
  • (PMID = 20620601.001).
  • [ISSN] 1873-4456
  • [Journal-full-title] Cancer genetics and cytogenetics
  • [ISO-abbreviation] Cancer Genet. Cytogenet.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / MLL protein, human; 149025-06-9 / Myeloid-Lymphoid Leukemia Protein; EC 2.1.1.43 / Histone-Lysine N-Methyltransferase
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21. Fowler A, Stödberg T, Eriksson M, Wickström R: Long-term outcomes of acute encephalitis in childhood. Pediatrics; 2010 Oct;126(4):e828-35
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  • [Title] Long-term outcomes of acute encephalitis in childhood.
  • OBJECTIVES: The aims of this study were to investigate the long-term outcomes of childhood encephalitis and to examine possible prognostic factors.
  • METHODS: Of 93 children who were treated for acute encephalitis in 2000-2004, 71 were eligible for follow-up evaluations.
  • CONCLUSION: Persisting symptoms after childhood encephalitis were present for a substantial number of children.
  • Children who made a full recovery did so within 6 to 12 months, which suggests that all children with encephalitis should be monitored for 1 year after the acute illness.
  • [MeSH-minor] Acute Disease. Adolescent. Child. Child, Preschool. Cognition Disorders / etiology. Electroencephalography. Epilepsy / etiology. Female. Humans. Infant. Male. Personality. Prognosis. Surveys and Questionnaires


22. Orihuela CJ, Mahdavi J, Thornton J, Mann B, Wooldridge KG, Abouseada N, Oldfield NJ, Self T, Ala'Aldeen DA, Tuomanen EI: Laminin receptor initiates bacterial contact with the blood brain barrier in experimental meningitis models. J Clin Invest; 2009 Jun;119(6):1638-46
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  • Bacterial meningitis in childhood is almost exclusively caused by the respiratory tract pathogens Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae, but the mechanism by which they initiate contact with the vascular endothelium of the blood brain barrier (BBB) is unknown.

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  • (PMID = 19436113.001).
  • [ISSN] 1558-8238
  • [Journal-full-title] The Journal of clinical investigation
  • [ISO-abbreviation] J. Clin. Invest.
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / P30 CA021765; United States / NIAID NIH HHS / AI / R01 AI027913; United States / NCI NIH HHS / CA / CA21765; United States / NIAID NIH HHS / AI / R01 AI27913
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Ligands; 0 / Receptors, Laminin
  • [Other-IDs] NLM/ PMC2689107
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23. Pascual CY, Reche M, Fiandor A, Valbuena T, Cuevas T, Esteban MM: Fish allergy in childhood. Pediatr Allergy Immunol; 2008 Nov;19(7):573-9
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  • [Title] Fish allergy in childhood.
  • An adverse reaction to fish may be of non-allergic origin, due to food contamination or newly formed toxic products, but the most frequent type of adverse reactions to fish are immunologic-mediated reactions (allergic reactions).
  • Such allergic reactions may be both IgE-mediated and non-IgE-mediated.
  • Some children develop non-IgE-mediated type allergies such as food protein induced enterocolitis syndrome.
  • The clinical symptoms related to IgE-mediated fish allergy are most frequently acute urticaria and angioedema as well as mild oral symptoms, worsening of atopic dermatitis, respiratory symptoms such as rhinitis or asthma, and gastrointestinal symptoms such as nausea and vomiting.

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  • (PMID = 18950323.001).
  • [ISSN] 1399-3038
  • [Journal-full-title] Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology
  • [ISO-abbreviation] Pediatr Allergy Immunol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Allergens; 37341-29-0 / Immunoglobulin E
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24. Halton J, Gaboury I, Grant R, Alos N, Cummings EA, Matzinger M, Shenouda N, Lentle B, Abish S, Atkinson S, Cairney E, Dix D, Israels S, Stephure D, Wilson B, Hay J, Moher D, Rauch F, Siminoski K, Ward LM, Canadian STOPP Consortium: Advanced vertebral fracture among newly diagnosed children with acute lymphoblastic leukemia: results of the Canadian Steroid-Associated Osteoporosis in the Pediatric Population (STOPP) research program. J Bone Miner Res; 2009 Jul;24(7):1326-34
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  • [Title] Advanced vertebral fracture among newly diagnosed children with acute lymphoblastic leukemia: results of the Canadian Steroid-Associated Osteoporosis in the Pediatric Population (STOPP) research program.
  • Vertebral compression is a serious complication of childhood acute lymphoblastic leukemia (ALL).
  • Whether the fractures will resolve through bone growth during or after leukemia chemotherapy remains to be determined.

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  • (PMID = 19210218.001).
  • [ISSN] 1523-4681
  • [Journal-full-title] Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research
  • [ISO-abbreviation] J. Bone Miner. Res.
  • [Language] ENG
  • [Grant] None / None / / 64285-1; Canada / Canadian Institutes of Health Research / / 64285-1
  • [Publication-type] Clinical Trial; Journal Article; Multicenter Study; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Glucocorticoids
  • [Other-IDs] NLM/ CAMS3788; NLM/ PMC3890351
  • [Investigator] Sykes E; Scharke M; Konji V; Riddell C; Ward LM; Feber J; Gaboury I; Halton J; Matzinger M; Moher D; Roth J; Shenouda N; Stephure D; Kloiber R; Lewis V; Midgley J; Miettunen P; Cabral D; Dix DB; Nadel HR; White C; Lentle BC; Hay J; Clarson C; Cairney E; Filler G; Grimmer J; Sparrow K; Cummings E; Fernandez C; Huber AM; Lang B; O'Brien K; Ross A; Atkinson S; Arora S; Barr R; Coblentz C; Dent PB; Webber C; Rodd C; Abish S; Bell L; Scuccimarri R; Rauch F; Glorieux F; Alos N; Dubois J; Laverdière C; Phan V; Saint-Cyr C; Couch R; Ellsworth J; LeBlanc C; Pinsk M; Siminoski K; Wilson B; Grant R; Charron M; Hebert D; Taback S; Blydt-Hansen T; Israels S; Oen K; Reed M
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25. Schmidt S, Rainer J, Riml S, Ploner C, Jesacher S, Achmüller C, Presul E, Skvortsov S, Crazzolara R, Fiegl M, Raivio T, Jänne OA, Geley S, Meister B, Kofler R: Identification of glucocorticoid-response genes in children with acute lymphoblastic leukemia. Blood; 2006 Mar 1;107(5):2061-9
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  • [Title] Identification of glucocorticoid-response genes in children with acute lymphoblastic leukemia.
  • The ability of glucocorticoids (GCs) to kill lymphoid cells led to their inclusion in essentially all chemotherapy protocols for lymphoid malignancies, particularly childhood acute lymphoblastic leukemia (ALL).
  • [MeSH-major] Apoptosis / drug effects. Drug Resistance, Neoplasm / genetics. Gene Expression Regulation, Leukemic / drug effects. Glucocorticoids / pharmacology. Neoplasm Proteins / genetics. Precursor Cell Lymphoblastic Leukemia-Lymphoma / genetics

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  • [ErratumIn] Blood. 2007 Apr 15;109(8):3234
  • (PMID = 16293608.001).
  • [ISSN] 0006-4971
  • [Journal-full-title] Blood
  • [ISO-abbreviation] Blood
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Glucocorticoids; 0 / Neoplasm Proteins
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26. George RE, Sanda T, Hanna M, Fröhling S, Luther W 2nd, Zhang J, Ahn Y, Zhou W, London WB, McGrady P, Xue L, Zozulya S, Gregor VE, Webb TR, Gray NS, Gilliland DG, Diller L, Greulich H, Morris SW, Meyerson M, Look AT: Activating mutations in ALK provide a therapeutic target in neuroblastoma. Nature; 2008 Oct 16;455(7215):975-8
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  • Neuroblastoma, an embryonal tumour of the peripheral sympathetic nervous system, accounts for approximately 15% of all deaths due to childhood cancer.
  • Five non-synonymous sequence variations were identified in the kinase domain of ALK, of which three were somatic and two were germ line.


27. Roman-Gomez J, Jimenez-Velasco A, Barrios M, Prosper F, Heiniger A, Torres A, Agirre X: Poor prognosis in acute lymphoblastic leukemia may relate to promoter hypermethylation of cancer-related genes. Leuk Lymphoma; 2007 Jul;48(7):1269-82
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  • [Title] Poor prognosis in acute lymphoblastic leukemia may relate to promoter hypermethylation of cancer-related genes.
  • The hallmark of acute lymphoblastic leukemia (ALL) is a progressive appearance of malignant cell behavior that is triggered by the evolution of altered gene function.
  • The presence in individual tumors of multiple genes simultaneously methylated is an independent factor of poor prognosis in both childhood and adult ALL in terms of disease-free survival and overall survival.
  • [MeSH-major] DNA Methylation. Precursor Cell Lymphoblastic Leukemia-Lymphoma / genetics

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  • (PMID = 17613754.001).
  • [ISSN] 1042-8194
  • [Journal-full-title] Leukemia & lymphoma
  • [ISO-abbreviation] Leuk. Lymphoma
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] England
  • [Number-of-references] 100
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28. Stams WA, den Boer ML, Beverloo HB, Kazemier KM, van Wering ER, Janka-Schaub GE, Pieters R: Effect of the histone deacetylase inhibitor depsipeptide on B-cell differentiation in both TEL-AML1-positive and negative childhood acute lymphoblastic leukemia. Haematologica; 2005 Dec;90(12):1697-9
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  • [Title] Effect of the histone deacetylase inhibitor depsipeptide on B-cell differentiation in both TEL-AML1-positive and negative childhood acute lymphoblastic leukemia.
  • The fusion protein TEL-AML1 in t(12;21)+ acute lymphoblastic leukemia (ALL) recruits co-repressors and histone deacetylases (HDAC), which transrepress AML1 target genes.
  • [MeSH-major] Antibiotics, Antineoplastic / pharmacology. B-Lymphocytes / drug effects. Core Binding Factor Alpha 2 Subunit / analysis. Depsipeptides / pharmacology. Histone Deacetylase Inhibitors. Oncogene Proteins, Fusion / analysis. Precursor B-Cell Lymphoblastic Leukemia-Lymphoma / pathology. Precursor Cell Lymphoblastic Leukemia-Lymphoma / pathology

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  • (PMID = 16330447.001).
  • [ISSN] 1592-8721
  • [Journal-full-title] Haematologica
  • [ISO-abbreviation] Haematologica
  • [Language] eng
  • [Publication-type] Comparative Study; Letter; Research Support, Non-U.S. Gov't
  • [Publication-country] Italy
  • [Chemical-registry-number] 0 / Antibiotics, Antineoplastic; 0 / Antigens, Differentiation, B-Lymphocyte; 0 / Antineoplastic Agents; 0 / Butyrates; 0 / Core Binding Factor Alpha 2 Subunit; 0 / Depsipeptides; 0 / Histone Deacetylase Inhibitors; 0 / Oncogene Proteins, Fusion; 0 / TEL-AML1 fusion protein; CX3T89XQBK / romidepsin; EC 3.5.1.1 / Asparaginase
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29. Scott RH, Homfray T, Huxter NL, Mitton SG, Nash R, Potter MN, Lancaster D, Rahman N: Familial T-cell non-Hodgkin lymphoma caused by biallelic MSH2 mutations. J Med Genet; 2007 Jul;44(7):e83
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  • [Title] Familial T-cell non-Hodgkin lymphoma caused by biallelic MSH2 mutations.
  • Familial non-Hodgkin lymphoma (NHL) is rare and in most cases, no underlying cause is identifiable.
  • Q76X) in three siblings who each developed T-cell NHL in early childhood.
  • Constitutional biallelic MSH2 mutations have previously been reported in five individuals, all of whom developed malignancy in childhood.


30. Bryce J, Gouws E, Adam T, Black RE, Schellenberg JA, Manzi F, Victora CG, Habicht JP: Improving quality and efficiency of facility-based child health care through Integrated Management of Childhood Illness in Tanzania. Health Policy Plan; 2005 Dec;20 Suppl 1:i69-i76
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  • [Title] Improving quality and efficiency of facility-based child health care through Integrated Management of Childhood Illness in Tanzania.
  • OBJECTIVES: To assess the effect of Integrated Management of Childhood Illness (IMCI) relative to routine care on the quality and efficiency of providing care for sick children in first-level health facilities in Tanzania, and to disseminate the results for use in health sector decision-making.
  • DESIGN: Non-randomized controlled trial to compare child health care quality and economic costs in two intervention (>90% of health care workers trained in IMCI) and two comparison districts in rural Tanzania.

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  • (PMID = 16306072.001).
  • [ISSN] 0268-1080
  • [Journal-full-title] Health policy and planning
  • [ISO-abbreviation] Health Policy Plan
  • [Language] eng
  • [Publication-type] Comparative Study; Controlled Clinical Trial; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
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31. Johansson LE, Danielsson P, Norgren S, Marcus C, Ridderstråle M: Interaction between PPARG Pro12Ala and ADIPOQ G276T concerning cholesterol levels in childhood obesity. Int J Pediatr Obes; 2009;4(2):119-25
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  • [Title] Interaction between PPARG Pro12Ala and ADIPOQ G276T concerning cholesterol levels in childhood obesity.

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  • (PMID = 18645733.001).
  • [ISSN] 1747-7174
  • [Journal-full-title] International journal of pediatric obesity : IJPO : an official journal of the International Association for the Study of Obesity
  • [ISO-abbreviation] Int J Pediatr Obes
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Adiponectin; 0 / Lipids; 0 / PPAR gamma; 97C5T2UQ7J / Cholesterol; 9DLQ4CIU6V / Proline; OF5P57N2ZX / Alanine
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32. Moe K, Hummelman EG, Oo WM, Lwin T, Htwe TT: Hospital-based surveillance for rotavirus diarrhea in children in Yangon, Myanmar. J Infect Dis; 2005 Sep 1;192 Suppl 1:S111-3
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  • Diarrhea is a common childhood illness in Myanmar, and rotavirus is the single most important etiological agent of diarrhea.
  • Surveillance for rotavirus diarrhea in children <5 years of age was conducted in a tertiary pediatric hospital in Yangon, Myanmar, from January 2002 through December 2003.
  • Stool specimens obtained from children admitted to the hospital for acute diarrhea were tested for the presence of rotavirus by use of an enzyme-linked immunosorbent assay.
  • The present study confirms the importance of the etiological role that rotavirus plays in childhood diarrhea.

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  • (PMID = 16088793.001).
  • [ISSN] 0022-1899
  • [Journal-full-title] The Journal of infectious diseases
  • [ISO-abbreviation] J. Infect. Dis.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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33. Eiholzer U, Meinhardt U, Petrò R, Witassek F, Gutzwiller F, Gasser T: High-intensity training increases spontaneous physical activity in children: a randomized controlled study. J Pediatr; 2010 Feb;156(2):242-6
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  • If this was confirmed in unselected children, resistance training might be a new strategy for childhood obesity prevention programs.


34. Rodríguez-López R, González-Carpio M, Serrano MV, Torres G, García de Cáceres MT, Herrera T, Román A, Rubio M, Méndez P, Hernández-Sáez R, Núñez M, Luengo LM: [Association of FTO gene polymorphisms and morbid obesity in the population of Extremadura (Spain)]. Endocrinol Nutr; 2010 May;57(5):203-9
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  • RESULTS: The A allele of rs9939609 was associated with severe obesity starting in childhood among the Spanish population.

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  • [Copyright] Copyright (c) 2009 SEEN. Published by Elsevier Espana. All rights reserved.
  • (PMID = 20418190.001).
  • [ISSN] 1575-0922
  • [Journal-full-title] Endocrinología y nutrición : órgano de la Sociedad Española de Endocrinología y Nutrición
  • [ISO-abbreviation] Endocrinol Nutr
  • [Language] spa
  • [Publication-type] English Abstract; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Spain
  • [Chemical-registry-number] 0 / FTO protein, human; 0 / Proteins
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35. Westby Wold SH, Sommerfelt K, Reigstad H, Rønnestad A, Medbø S, Farstad T, Kaaresen PI, Støen R, Leversen KT, Irgens LM, Markestad T: Neonatal mortality and morbidity in extremely preterm small for gestational age infants: a population based study. Arch Dis Child Fetal Neonatal Ed; 2009 Sep;94(5):F363-7
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  • [Title] Neonatal mortality and morbidity in extremely preterm small for gestational age infants: a population based study.
  • AIM: To assess if growth restricted (small for gestational age, SGA) extremely preterm infants have excess neonatal mortality and morbidity.
  • METHODS: This was a cohort study of all infants born alive at 22-27 weeks' post menstrual age in Norway during 1999-2000.
  • Outcomes were compared between those who were SGA, defined as a birth weight less than the fifth percentile for post menstrual age, and those who had weights at or above the fifth percentile.
  • RESULTS: Of 365 infants with a post menstrual age of <28 weeks, 31 (8%) were SGA.
  • Among infants with a post menstrual age of <28 weeks, only chronic lung disease was associated with SGA status (OR 2.7, 95% CI 1.0 to 7.2).
  • SGA infants with a post menstrual age of 26-27 weeks had excess neonatal mortality (OR 3.8, 95% CI 1.3 to 11), chronic lung disease and a significantly higher mean number of days (age) before tolerating full enteral nutrition.
  • SGA infants with a post menstrual age of 22-25 weeks had an excess risk of necrotising enterocolitis.
  • CONCLUSION: Extremely preterm SGA infants had excess neonatal mortality and morbidity in terms of necrotising enterocolitis and chronic lung disease.
  • [MeSH-major] Infant, Premature, Diseases / epidemiology. Infant, Small for Gestational Age. Intensive Care, Neonatal / standards. Lung Diseases / epidemiology
  • [MeSH-minor] Female. Humans. Infant, Newborn. Infant, Premature. Male. Neonatal Screening. Norway / epidemiology. Prenatal Diagnosis. Risk Factors

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  • (PMID = 19439434.001).
  • [ISSN] 1468-2052
  • [Journal-full-title] Archives of disease in childhood. Fetal and neonatal edition
  • [ISO-abbreviation] Arch. Dis. Child. Fetal Neonatal Ed.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
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36. Baldo M, Bhogal B, Groves RW, Powell J, Wojnarowska F: Childhood vulval lichen sclerosus: autoimmunity to the basement membrane zone protein BP180 and its relationship to autoimmunity. Clin Exp Dermatol; 2010 Jul;35(5):543-5
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  • [Title] Childhood vulval lichen sclerosus: autoimmunity to the basement membrane zone protein BP180 and its relationship to autoimmunity.
  • [MeSH-major] Autoantibodies / immunology. Autoantigens / immunology. Basement Membrane / immunology. Non-Fibrillar Collagens / immunology. Vulvar Lichen Sclerosus / immunology

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  • (PMID = 20456392.001).
  • [ISSN] 1365-2230
  • [Journal-full-title] Clinical and experimental dermatology
  • [ISO-abbreviation] Clin. Exp. Dermatol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Autoantibodies; 0 / Autoantigens; 0 / Immunoglobulin A; 0 / Immunoglobulin G; 0 / Non-Fibrillar Collagens; 0 / collagen type XVII
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37. Nurmio M, Keros V, Lähteenmäki P, Salmi T, Kallajoki M, Jahnukainen K: Effect of childhood acute lymphoblastic leukemia therapy on spermatogonia populations and future fertility. J Clin Endocrinol Metab; 2009 Jun;94(6):2119-22
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  • [Title] Effect of childhood acute lymphoblastic leukemia therapy on spermatogonia populations and future fertility.
  • CONTEXT: Isolation of spermatogonial stem cells before potentially sterilizing cancer therapy, followed by transplantation of these cells into the testis after such treatment, may be an effective approach to prevent infertility among prepubertal boys suffering from acute lymphoblastic leukemia (ALL).
  • A key clinical consideration in this context is the timing of biopsy, if collection of spermatogonia could be delayed from diagnosis to the later phase of leukemia treatment, better patient selection could be offered.
  • OBJECTIVE: The objective of the study was to examine the routine testicular biopsy material collected to detect testicular leukemia to evaluate if treatment for leukemia affects numbers and maturation of the spermatogonia during the prepubertal period.
  • No significant alteration in spermatogonial numbers was associated with testicular leukemia.
  • CONCLUSION: Treatment for childhood leukemia without high-dose cyclophosphamide seldom depletes the spermatogonial stem cell pool totally.
  • [MeSH-major] Cyclophosphamide / adverse effects. Fertility / drug effects. Precursor Cell Lymphoblastic Leukemia-Lymphoma / drug therapy. Sperm Count. Spermatogonia / pathology

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  • (PMID = 19318447.001).
  • [ISSN] 1945-7197
  • [Journal-full-title] The Journal of clinical endocrinology and metabolism
  • [ISO-abbreviation] J. Clin. Endocrinol. Metab.
  • [Language] eng
  • [Publication-type] Evaluation Studies; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antineoplastic Agents, Alkylating; 8N3DW7272P / Cyclophosphamide
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38. Hassapidou M, Papadopoulou SK, Frossinis A, Kaklamanos I, Tzotzas T: Sociodemographic, ethnic and dietary factors associated with childhood obesity in Thessaloniki, Northern Greece. Hormones (Athens); 2009 Jan-Mar;8(1):53-9
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  • [Title] Sociodemographic, ethnic and dietary factors associated with childhood obesity in Thessaloniki, Northern Greece.
  • OBJECTIVE: To investigate sociodemographic, ethnic and dietary factors associated with the development of childhood obesity.
  • Obese children compared to their non-obese counterparts a) had parents who were obese at a higher percentage (p=0.001), b) reported that food preparation was carried out by their grandmother (p=0.006) and c) had less pocket money (p=0.004).
  • CONCLUSIONS: In planning interventions for childhood obesity, sociodemographic factors in addition to food intake and physical activity patterns have to be considered.

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  • (PMID = 19269921.001).
  • [ISSN] 1109-3099
  • [Journal-full-title] Hormones (Athens, Greece)
  • [ISO-abbreviation] Hormones (Athens)
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Greece
  • [Chemical-registry-number] 0 / Dietary Carbohydrates
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39. Jin Y, Mertens F, Kullendorff CM, Panagopoulos I: Fusion of the tumor-suppressor gene CHEK2 and the gene for the regulatory subunit B of protein phosphatase 2 PPP2R2A in childhood teratoma. Neoplasia; 2006 May;8(5):413-8
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  • [Title] Fusion of the tumor-suppressor gene CHEK2 and the gene for the regulatory subunit B of protein phosphatase 2 PPP2R2A in childhood teratoma.

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  • (PMID = 16790090.001).
  • [ISSN] 1476-5586
  • [Journal-full-title] Neoplasia (New York, N.Y.)
  • [ISO-abbreviation] Neoplasia
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Canada
  • [Chemical-registry-number] 0 / Oncogene Proteins, Fusion; 0 / PPP2R2A protein, human; EC 2.7.1.11 / Checkpoint Kinase 2; EC 2.7.11.1 / CHEK2 protein, human; EC 2.7.11.1 / Protein-Serine-Threonine Kinases; EC 3.1.3.16 / Phosphoprotein Phosphatases; EC 3.1.3.16 / Protein Phosphatase 2
  • [Other-IDs] NLM/ PMC1592448
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40. Horsley SW, Colman S, McKinley M, Bateman CM, Jenney M, Chaplin T, Young BD, Greaves M, Kearney L: Genetic lesions in a preleukemic aplasia phase in a child with acute lymphoblastic leukemia. Genes Chromosomes Cancer; 2008 Apr;47(4):333-40
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  • [Title] Genetic lesions in a preleukemic aplasia phase in a child with acute lymphoblastic leukemia.
  • In a small fraction ( approximately 2%) of cases of childhood acute lymphoblastic leukemia (ALL) clinical presentation of leukemia is preceded, some 2-9 months earlier, by a transient, remitting phase of nonclassical aplastic anemia, usually in connection with infection.
  • [MeSH-major] Anemia, Aplastic / complications. Anemia, Aplastic / genetics. Core Binding Factor Alpha 2 Subunit / genetics. Oncogene Proteins, Fusion / genetics. Precursor Cell Lymphoblastic Leukemia-Lymphoma / genetics. Preleukemia / genetics

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  • [Copyright] (c) 2008 Wiley-Liss, Inc.
  • (PMID = 18181181.001).
  • [ISSN] 1098-2264
  • [Journal-full-title] Genes, chromosomes & cancer
  • [ISO-abbreviation] Genes Chromosomes Cancer
  • [Language] eng
  • [Grant] United Kingdom / Cancer Research UK / / A6438; United Kingdom / Cancer Research UK / / A6789
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Core Binding Factor Alpha 2 Subunit; 0 / Oncogene Proteins, Fusion; 0 / TEL-AML1 fusion protein
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41. Spence W, Mulholland C, Lynch G, McHugh S, Dempster M, Shannon C: Rates of childhood trauma in a sample of patients with schizophrenia as compared with a sample of patients with non-psychotic psychiatric diagnoses. J Trauma Dissociation; 2006;7(3):7-22
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  • [Title] Rates of childhood trauma in a sample of patients with schizophrenia as compared with a sample of patients with non-psychotic psychiatric diagnoses.
  • Despite strong evidence of high rates of childhood and adult trauma in schizophrenia, the area remains under-researched.
  • Our objectives in the study were first, to examine the rates of exposure to childhood, adult and lifetime (child plus adult) trauma in a population with schizophrenia and a population with non-psychotic psychiatric diagnoses and second, to examine the effect of trauma on the symptoms of schizophrenia.
  • Two groups, those with schizophrenia (n = 40), and those with a non-psychotic diagnosis (n = 30), were recruited.
  • Childhood exposure to trauma was significantly more common in the schizophrenia group (t = 5.196, df = 68, p < 0.001, Eta squared = 0.28), with the strongest relationship being childhood physical assault.
  • Evidence that childhood exposure to trauma is more common in a population with schizophrenia is consistent with other studies and raises the possibility that such trauma is of etiological importance.
  • Further research is required to replicate those findings, to elucidate possible pathways by which the experience of trauma may contribute to the development of schizophrenia, and to explore the relationship between a history of childhood trauma and the experience of depressive symptoms in schizophrenia.

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  • (PMID = 16873227.001).
  • [ISSN] 1529-9732
  • [Journal-full-title] Journal of trauma & dissociation : the official journal of the International Society for the Study of Dissociation (ISSD)
  • [ISO-abbreviation] J Trauma Dissociation
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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42. Hunninghake GM, Soto-Quirós ME, Avila L, Kim HP, Lasky-Su J, Rafaels N, Ruczinski I, Beaty TH, Mathias RA, Barnes KC, Wilk JB, O'Connor GT, Gauderman WJ, Vora H, Baurley JW, Gilliland F, Liang C, Sylvia JS, Klanderman BJ, Sharma SS, Himes BE, Bossley CJ, Israel E, Raby BA, Bush A, Choi AM, Weiss ST, Celedón JC: TSLP polymorphisms are associated with asthma in a sex-specific fashion. Allergy; 2010 Dec;65(12):1566-75
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  • Significant findings were replicated in whites and African-American participants in the Childhood Asthma Management Program, in African-Americans in the Genomic Research on Asthma in the African Diaspora study, in whites and Hispanics in the Children's Health Study, and in whites in the Framingham Heart Study (FHS).

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  • [Copyright] © 2010 John Wiley & Sons A/S.
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  • (PMID = 20560908.001).
  • [ISSN] 1398-9995
  • [Journal-full-title] Allergy
  • [ISO-abbreviation] Allergy
  • [Language] ENG
  • [Grant] United States / NHLBI NIH HHS / HL / P01 HL083069-02; United States / NIEHS NIH HHS / ES / ES011627-010003; United States / NIEHS NIH HHS / ES / P30 ES007048; United States / NHLBI NIH HHS / HL / P01 HL083069; United States / NHLBI NIH HHS / HL / R00 HL096840; United States / NHLBI NIH HHS / HL / K01 HL004370; United States / NHLBI NIH HHS / HL / U01 HL65899; United States / NHLBI NIH HHS / HL / HL083069-02; United States / NHLBI NIH HHS / HL / K08HL092222; United States / NLM NIH HHS / LM / T15 LM007092; United States / NHLBI NIH HHS / HC / N01 HC025195; United States / NHLBI NIH HHS / HL / U01 HL065899; United States / NHLBI NIH HHS / HL / R01 HL066289-01; United States / NHLBI NIH HHS / HL / R37 HL066289; United States / NHLBI NIH HHS / HL / R01 HL086601-01; United States / NHLBI NIH HHS / HL / R01 HL087699-01; United States / NIEHS NIH HHS / ES / P01 ES011627; United States / NHLBI NIH HHS / HL / R01 HL087699; United States / NHLBI NIH HHS / HL / R01 HL087680; United States / NHLBI NIH HHS / HL / K08 HL092222; United States / NHLBI NIH HHS / HL / K01 HL004370-02; United States / NHLBI NIH HHS / HL / U01 HL075419; United States / NIEHS NIH HHS / ES / P01 ES011627-010003; United States / NHLBI NIH HHS / HL / T32 HL007427-19; United States / NHLBI NIH HHS / HL / HL004370-02; United States / NHLBI NIH HHS / HL / HL092222-03; United States / NHLBI NIH HHS / HL / K08 HL096833; United States / NHLBI NIH HHS / HL / HL04370; United States / NHLBI NIH HHS / HL / HL66289; United States / NHLBI NIH HHS / HL / R01 HL066289; United States / NHLBI NIH HHS / HL / N01HC25195; United States / NHLBI NIH HHS / HL / K12 HL089990; United States / NHLBI NIH HHS / HL / U01 HL065899-01; United States / NIEHS NIH HHS / ES / P30 ES007048-04S19001; United States / NHLBI NIH HHS / HL / HL087680-01; United States / NHLBI NIH HHS / HL / R01 HL086601; United States / NHLBI NIH HHS / HC / N01-HC25195; United States / NLM NIH HHS / LM / 2T15LM007092-16; United States / NIEHS NIH HHS / ES / ES007048-04S19001; United States / NHLBI NIH HHS / HL / U01 HL075419-01; None / None / / R01 HL066289-01; United States / NHLBI NIH HHS / HL / K08 HL092222-03; United States / NHLBI NIH HHS / HL / T32 HL07427; United States / NHLBI NIH HHS / HL / T32 HL007427; United States / NHLBI NIH HHS / HL / R01 HL087680-01; United States / NHLBI NIH HHS / HL / HL087699-01
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] Denmark
  • [Chemical-registry-number] 0 / Cytokines; 0 / thymic stromal lymphopoietin
  • [Other-IDs] NLM/ NIHMS233299; NLM/ PMC2970693
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43. Unger K, Zurnadzhy L, Walch A, Mall M, Bogdanova T, Braselmann H, Hieber L, Tronko N, Hutzler P, Jeremiah S, Thomas G, Zitzelsberger H: RET rearrangements in post-Chernobyl papillary thyroid carcinomas with a short latency analysed by interphase FISH. Br J Cancer; 2006 May 22;94(10):1472-7
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  • Tissue samples from 13 post-Chernobyl childhood thyroid tumours that occurred within a short period of time (4-8 years) after the Chernobyl accident have been investigated by interphase FISH analysis for rearrangements of RET.

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  • (PMID = 16641909.001).
  • [ISSN] 0007-0920
  • [Journal-full-title] British journal of cancer
  • [ISO-abbreviation] Br. J. Cancer
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] EC 2.7.10.1 / Proto-Oncogene Proteins c-ret; EC 2.7.10.1 / RET protein, human
  • [Other-IDs] NLM/ PMC2365029
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44. Buschmann A, Jooss B, Rupp A, Feldhusen F, Pietz J, Philippi H: Parent based language intervention for 2-year-old children with specific expressive language delay: a randomised controlled trial. Arch Dis Child; 2009 Feb;94(2):110-6
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  • Standardised instruments were used to assess the language and non-verbal cognitive abilities of these children and of 36 other children with normal language development (reference group; mean age 24.6 months, SD 0.8).

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  • (PMID = 18703544.001).
  • [ISSN] 1468-2044
  • [Journal-full-title] Archives of disease in childhood
  • [ISO-abbreviation] Arch. Dis. Child.
  • [Language] eng
  • [Publication-type] Journal Article; Multicenter Study; Randomized Controlled Trial; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC2614563
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45. Hosking FJ, Papaemmanuil E, Sheridan E, Kinsey SE, Lightfoot T, Roman E, Irving JA, Allan JM, Taylor M, Tomlinson IP, Greaves M, Houlston RS: Genome-wide homozygosity signatures and childhood acute lymphoblastic leukemia risk. Blood; 2010 Jun 3;115(22):4472-7
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  • [Title] Genome-wide homozygosity signatures and childhood acute lymphoblastic leukemia risk.
  • To examine whether homozygosity is associated with an increased risk of developing childhood B-cell precursor acute lymphoblastic leukemia (BCP-ALL), we analyzed 824 ALL cases and 2398 controls genotyped for 292 200 tagging SNPs.

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  • (PMID = 20231427.001).
  • [ISSN] 1528-0020
  • [Journal-full-title] Blood
  • [ISO-abbreviation] Blood
  • [Language] ENG
  • [Grant] United Kingdom / Cancer Research UK / / 10417; United Kingdom / Cancer Research UK / / C1298/A8362
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Receptors, Erythropoietin
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46. Taylor M, Harrison C, Eden T, Birch J, Greaves M, Lightfoot T, Hussain A, UKCCS Investigators: HLA-DPB1 supertype-associated protection from childhood leukaemia: relationship to leukaemia karyotype and implications for prevention. Cancer Immunol Immunother; 2008 Jan;57(1):53-61
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  • [Title] HLA-DPB1 supertype-associated protection from childhood leukaemia: relationship to leukaemia karyotype and implications for prevention.
  • Most childhood B cell precursor (BCP) acute lymphoblastic leukaemia (ALL) cases carry the reciprocal translocation t(12;21)(p13;q22) ( approximately 25%), or a high hyperdiploid (HeH) karyotype (30%).
  • Based on our previous analysis of HLA-DP in childhood ALL, and evidence from in vitro studies that TEL-AML1 can activate HLA-DP-restricted T cell responses, we hypothesised that the development of TEL-AML1+ ALL might be influenced by the child's DPB1 genotype.
  • To test this, we analysed the frequency of six HLA-DPB1 supertypes in a population-based series of childhood leukaemias (n = 776) classified by their karyotype (TEL-AML1+, HeH and others), in comparison with newborn controls (n = 864).
  • [MeSH-major] Core Binding Factor Alpha 2 Subunit / genetics. Genetic Predisposition to Disease. HLA-DP Antigens / genetics. Oncogene Proteins, Fusion / genetics. Precursor Cell Lymphoblastic Leukemia-Lymphoma / genetics. Precursor Cell Lymphoblastic Leukemia-Lymphoma / prevention & control

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  • (PMID = 17622527.001).
  • [ISSN] 0340-7004
  • [Journal-full-title] Cancer immunology, immunotherapy : CII
  • [ISO-abbreviation] Cancer Immunol. Immunother.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / Core Binding Factor Alpha 2 Subunit; 0 / HLA-DP Antigens; 0 / HLA-DP beta-Chains; 0 / HLA-DPB1 antigen; 0 / Oncogene Proteins, Fusion; 0 / TEL-AML1 fusion protein
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47. Sanjeevi CB, Sedimbi SK, Landin-Olsson M, Kockum I, Lernmark A, Swedish Childhood Diabetes and the Diabetes Incidence in Sweden Study Groups: Risk conferred by HLA-DR and DQ for type 1 diabetes in 0-35-year age group in Sweden. Ann N Y Acad Sci; 2008 Dec;1150:106-11
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  • [Title] Risk conferred by HLA-DR and DQ for type 1 diabetes in 0-35-year age group in Sweden.
  • HLA DR4-DQ8 and DR3-DQ2 haplotypes account for 89% of newly diagnosed cases of type 1 diabetes (T1D) in Sweden.
  • The presence of a single copy of DQ6 confers protection.
  • The aim of the present study is to evaluate whether the risk conferred by high risk HLA DR and DQ to T1D is similar in all regions of Sweden and see whether there are any significant regional differences.
  • The subjects comprised 799 consecutively diagnosed T1D patients and 585 age-, sex-, and geography-matched healthy controls in the age group 0-35 years.
  • HLA typing for high-risk haplotypes was previously performed using PCR-SSOP and RFLP.
  • The results showed that HLA DR3-DR4 gave an odds ratio of 8.14 for the whole of Sweden.
  • However, when the study group was divided into six geographical regions, subjects from Stockholm had the highest OR, followed by those from Lund, Linköping, Gothenburg, Umeå, and Uppsala.
  • Absolute protection was conferred by the presence of DQ6 in subjects from the Linköping region, but varied in the other regions.
  • The frequency of DR3 and DQ2, DR4 and DQ8, DR15, and DQ6 in patients showed high linkage for each region, but were different between regions.
  • IN CONCLUSION: The risk conferred by high-risk HLA varies in different regions for a homogenous population in Sweden.
  • The results highlight the important role played by the various environmental factors in the precipitation of T1D.
  • [MeSH-major] Diabetes Mellitus, Type 1 / etiology. Diabetes Mellitus, Type 1 / genetics. HLA-DQ Antigens / genetics. HLA-DR Antigens / genetics
  • [MeSH-minor] Adolescent. Adult. Age Distribution. Case-Control Studies. Child. Child, Preschool. Environment. Female. Gene Frequency. Genetic Predisposition to Disease. Geography. Histocompatibility Testing. Humans. Infant. Infant, Newborn. Male. Risk Factors. Sweden / epidemiology. Young Adult

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  • (PMID = 19120278.001).
  • [ISSN] 1749-6632
  • [Journal-full-title] Annals of the New York Academy of Sciences
  • [ISO-abbreviation] Ann. N. Y. Acad. Sci.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / HLA-DQ Antigens; 0 / HLA-DR Antigens
  • [Investigator] Aili M; Bååth LE; Carlsson E; Edenwall H; Forsander G; Granstöm BW; Gustavsson I; Hanås R; Hellenberg L; Hellgren H; Holmberg E; Hörnell H; Ivarsson SA; Johansson C; Jonsell G; Kockum K; Lindblad B; Lindh A; Ludvigsson J; Myrdal U; Neiderud J; Segnestam K; Sjöblad S; Skogsberg L; Strömberg L; Ståhle U; Thalme B; Tullus K; Tuvemo T; Wallensteen M
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48. Baldassano RN, Bradfield JP, Monos DS, Kim CE, Glessner JT, Casalunovo T, Frackelton EC, Otieno FG, Kanterakis S, Shaner JL, Smith RM, Eckert AW, Robinson LJ, Onyiah CC, Abrams DJ, Chiavacci RM, Skraban R, Devoto M, Grant SF, Hakonarson H: Association of variants of the interleukin-23 receptor gene with susceptibility to pediatric Crohn's disease. Clin Gastroenterol Hepatol; 2007 Aug;5(8):972-6
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  • [Title] Association of variants of the interleukin-23 receptor gene with susceptibility to pediatric Crohn's disease.
  • METHODS: By using data from our ongoing genome-wide association study in our cohort of 142 pediatric CD patients and 281 matched controls, we investigated the association of the previously reported SNPs at the IL23R locus with the childhood form of this disease.
  • CONCLUSIONS: The OR of the IL23R variant in our pediatric study is highly comparable with that reported previously in a non-Jewish adult inflammatory bowel disease case-control cohort (OR, 0.26).

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  • (PMID = 17618837.001).
  • [ISSN] 1542-7714
  • [Journal-full-title] Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association
  • [ISO-abbreviation] Clin. Gastroenterol. Hepatol.
  • [Language] ENG
  • [Grant] United States / NCRR NIH HHS / RR / M01 RR000040; United States / NCRR NIH HHS / RR / M01 RR000040-46; United States / NCRR NIH HHS / RR / M01 RR000240; United States / NCRR NIH HHS / RR / 5-M01-RR-000240
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / DNA Probes; 0 / IL23R protein, human; 0 / Receptors, Interleukin; 9007-49-2 / DNA
  • [Other-IDs] NLM/ NIHMS29859; NLM/ PMC4287202
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49. Cambuli VM, Incani M, Pilia S, Congiu T, Cavallo MG, Cossu E, Sentinelli F, Mariotti S, Loche S, Baroni MG: Oral glucose tolerance test in Italian overweight/obese children and adolescents results in a very high prevalence of impaired fasting glycaemia, but not of diabetes. Diabetes Metab Res Rev; 2009 Sep;25(6):528-34
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  • BACKGROUND: Very few studies on glucose abnormalities in European overweight/obese children and adolescents are available, and scientific evidence on the value of standard oral glucose tolerance test (OGTT) in childhood is lacking.
  • IFG appears not useful to detect IGT in childhood.


50. Attarbaschi A, Mann G, König M, Steiner M, Strehl S, Schreiberhuber A, Schneider B, Meyer C, Marschalek R, Borkhardt A, Pickl WF, Lion T, Gadner H, Haas OA, Dworzak MN: Mixed lineage leukemia-rearranged childhood pro-B and CD10-negative pre-B acute lymphoblastic leukemia constitute a distinct clinical entity. Clin Cancer Res; 2006 May 15;12(10):2988-94
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  • [Title] Mixed lineage leukemia-rearranged childhood pro-B and CD10-negative pre-B acute lymphoblastic leukemia constitute a distinct clinical entity.
  • PURPOSE: Mixed lineage leukemia (MLL) abnormalities occur in approximately 50% of childhood pro-B acute lymphoblastic leukemia (ALL).
  • However, the incidence and type of MLL rearrangements have not been determined in common ALL (cALL) and CD10+ or CD10- pre-B ALL.
  • EXPERIMENTAL DESIGN: To address this question, we analyzed 29 patients with pro-B ALL, 11 patients with CD10- pre-B ALL, 23 pre-B, and 26 cALL patients with CD10 on 20% to 80%, as well as 136 pre-B and 143 cALL patients with CD10 > or = 80% of blasts.
  • RESULTS: We found that 15 of 29 pro-B ALL, 7 of 11 CD10- pre-B ALL, and 1 of 2 French-American-British classification L1 mature B-cell leukemia cases had a MLL rearrangement.
  • MLL-rearranged pro-B and CD10- pre-B ALL cases had similar clinical and immunophenotypic (coexpression of CDw65 and CD15) features at initial diagnosis.
  • CONCLUSIONS: The striking similarities between the two CD10- ALL subsets imply that CD10- pre-B ALL variants may represent pro-B ALL cases that maintained the propensity to rearrange and express their immunoglobulin heavy chain rather than actual pre-B ALL forms transformed at this later stage of B-cell differentiation.
  • [MeSH-major] Chromosome Aberrations. Myeloid-Lymphoid Leukemia Protein / genetics. Precursor B-Cell Lymphoblastic Leukemia-Lymphoma / genetics. Precursor Cell Lymphoblastic Leukemia-Lymphoma / genetics

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  • (PMID = 16707593.001).
  • [ISSN] 1078-0432
  • [Journal-full-title] Clinical cancer research : an official journal of the American Association for Cancer Research
  • [ISO-abbreviation] Clin. Cancer Res.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Immunoglobulin Heavy Chains; 149025-06-9 / Myeloid-Lymphoid Leukemia Protein; EC 3.4.24.11 / Neprilysin
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51. Borup MT, Trusina A, Andersson AM: Aging mechanism as the "down side" of adaptation: a network approach. J Theor Biol; 2008 Jan 7;250(1):66-74
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  • (1) general plasticity (childhood), (2) optimization/adaptation to given conditions (youth and adolescence) and (3) steady state associated with high rigidity (aging).

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  • (PMID = 17961600.001).
  • [ISSN] 0022-5193
  • [Journal-full-title] Journal of theoretical biology
  • [ISO-abbreviation] J. Theor. Biol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
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52. Yang MH, Jia WG, Cao LZ, He YL, Liao N, Chen GL, Luo JM, Xu WQ, Yang J: [Efficacy and prognosis analysis in 188 children with acute lymphoblastic leukemia of China]. Zhonghua Er Ke Za Zhi; 2008 Jul;46(7):498-501
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  • [Title] [Efficacy and prognosis analysis in 188 children with acute lymphoblastic leukemia of China].
  • OBJECTIVE: To analyze the therapeutic effect and the influencing factors of event-free survival (EFS) of childhood acute lymphoblastic leukemia (ALL) in Xiangya Hospital of Central South University and the First Affiliated Hospital of Guangxi Medical University.
  • CONCLUSIONS: The total EFS of childhood ALL patients treated with Rongcheng ALL-98 Protocol in two hospitals was close to 70%.
  • [MeSH-major] Antineoplastic Combined Chemotherapy Protocols / therapeutic use. Precursor Cell Lymphoblastic Leukemia-Lymphoma / drug therapy. Precursor Cell Lymphoblastic Leukemia-Lymphoma / mortality

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  • (PMID = 19099804.001).
  • [ISSN] 0578-1310
  • [Journal-full-title] Zhonghua er ke za zhi = Chinese journal of pediatrics
  • [ISO-abbreviation] Zhonghua Er Ke Za Zhi
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] China
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53. Humiston SG, Lerner EB, Hepworth E, Blythe T, Goepp JG: Parent opinions about universal influenza vaccination for infants and toddlers. Arch Pediatr Adolesc Med; 2005 Feb;159(2):108-12
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  • OBJECTIVE: To assess among English-speaking caregivers of children aged 6 to 23 months opinions about childhood influenza vaccination and potential knowledge, attitudinal, and demographic factors that might influence such opinions.
  • METHODS: A structured, interviewer-administered survey of knowledge, attitudes, and beliefs about the influenza vaccine among parents and caregivers of children at the ambulatory pediatric clinic or the pediatric emergency department of a large tertiary care teaching hospital.

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  • (PMID = 15699302.001).
  • [ISSN] 1072-4710
  • [Journal-full-title] Archives of pediatrics & adolescent medicine
  • [ISO-abbreviation] Arch Pediatr Adolesc Med
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Influenza Vaccines
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54. Sadarangani M, Seaton C, Scott JA, Ogutu B, Edwards T, Prins A, Gatakaa H, Idro R, Berkley JA, Peshu N, Neville BG, Newton CR: Incidence and outcome of convulsive status epilepticus in Kenyan children: a cohort study. Lancet Neurol; 2008 Feb;7(2):145-50
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  • BACKGROUND: Convulsive status epilepticus (CSE) is the most common neurological emergency in childhood and is often associated with fever.
  • We aimed to provide data on the incidence, causes, and outcomes of childhood CSE in this region.

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  • (PMID = 18248771.001).
  • [ISSN] 1474-4422
  • [Journal-full-title] The Lancet. Neurology
  • [ISO-abbreviation] Lancet Neurol
  • [Language] eng
  • [Grant] United Kingdom / Wellcome Trust / / 061089; United Kingdom / Wellcome Trust / / 081835; United Kingdom / Medical Research Council / / G0700837; United Kingdom / Wellcome Trust / / 070114
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Anticonvulsants; 6158TKW0C5 / Phenytoin; YQE403BP4D / Phenobarbital
  • [Other-IDs] NLM/ PMC2258310
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55. Fernald LC, Gertler PJ, Neufeld LM: 10-year effect of Oportunidades, Mexico's conditional cash transfer programme, on child growth, cognition, language, and behaviour: a longitudinal follow-up study. Lancet; 2009 Dec 12;374(9706):1997-2005
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  • The money itself also has significant effects on most outcomes, adding to existing evidence for interventions in early childhood.


56. Mullighan CG, Zhang J, Harvey RC, Collins-Underwood JR, Schulman BA, Phillips LA, Tasian SK, Loh ML, Su X, Liu W, Devidas M, Atlas SR, Chen IM, Clifford RJ, Gerhard DS, Carroll WL, Reaman GH, Smith M, Downing JR, Hunger SP, Willman CL: JAK mutations in high-risk childhood acute lymphoblastic leukemia. Proc Natl Acad Sci U S A; 2009 Jun 9;106(23):9414-8
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  • [Title] JAK mutations in high-risk childhood acute lymphoblastic leukemia.
  • Pediatric acute lymphoblastic leukemia (ALL) is a heterogeneous disease consisting of distinct clinical and biological subtypes that are characterized by specific chromosomal abnormalities or gene mutations.
  • We recently identified a poor prognostic subgroup of pediatric BCR-ABL1-negative ALL patients characterized by deletion of IKZF1 (encoding the lymphoid transcription factor IKAROS) and a gene expression signature similar to BCR-ABL1-positive ALL, raising the possibility of activated tyrosine kinase signaling within this leukemia subtype.
  • Here, we report activating mutations in the Janus kinases JAK1 (n = 3), JAK2 (n = 16), and JAK3 (n = 1) in 20 (10.7%) of 187 BCR-ABL1-negative, high-risk pediatric ALL cases.
  • The JAK-mutated cases had a gene expression signature similar to BCR-ABL1 pediatric ALL, and they had a poor outcome.

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  • (PMID = 19470474.001).
  • [ISSN] 1091-6490
  • [Journal-full-title] Proceedings of the National Academy of Sciences of the United States of America
  • [ISO-abbreviation] Proc. Natl. Acad. Sci. U.S.A.
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / P30 CA118100; United States / NCI NIH HHS / CA / U10 CA98413; United States / NCI NIH HHS / CA / L40 CA142226; United States / NCI NIH HHS / CA / U01 CA114762; United States / NCI NIH HHS / CA / CA114762; United States / NCI NIH HHS / CA / U10 CA098413; United States / PHS HHS / / N01-C0-12400; United States / NCI NIH HHS / CA / CA098543; United States / Howard Hughes Medical Institute / / ; United States / NICHD NIH HHS / HD / T32 HD044331; United States / NCI NIH HHS / CA / U10 CA098543; United States / NCI NIH HHS / CA / T32 CA128583; United States / NCI NIH HHS / CA / U10 CA98543
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / IKZF1 protein, human; 148971-36-2 / Ikaros Transcription Factor; EC 2.7.10.2 / Janus Kinase 1; EC 2.7.10.2 / Janus Kinase 3; EC 2.7.10.2 / Janus Kinases
  • [Other-IDs] NLM/ PMC2695045
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57. Matz K, Junghöfer M, Elbert T, Weber K, Wienbruch C, Rockstroh B: Adverse experiences in childhood influence brain responses to emotional stimuli in adult psychiatric patients. Int J Psychophysiol; 2010 Mar;75(3):277-86
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  • [Title] Adverse experiences in childhood influence brain responses to emotional stimuli in adult psychiatric patients.

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  • [Copyright] Copyright 2009 Elsevier B.V. All rights reserved.
  • (PMID = 20045438.001).
  • [ISSN] 1872-7697
  • [Journal-full-title] International journal of psychophysiology : official journal of the International Organization of Psychophysiology
  • [ISO-abbreviation] Int J Psychophysiol
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Netherlands
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58. Sameith K, Antczak P, Marston E, Turan N, Maier D, Stankovic T, Falciani F: Functional modules integrating essential cellular functions are predictive of the response of leukaemia cells to DNA damage. Bioinformatics; 2008 Nov 15;24(22):2602-7
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  • MOTIVATION: Childhood B-precursor lymphoblastic leukaemia (ALL) is the most common paediatric malignancy.

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  • (PMID = 18801750.001).
  • [ISSN] 1367-4811
  • [Journal-full-title] Bioinformatics (Oxford, England)
  • [ISO-abbreviation] Bioinformatics
  • [Language] eng
  • [Grant] United Kingdom / Biotechnology and Biological Sciences Research Council / / BB/D524624/1; United Kingdom / Biotechnology and Biological Sciences Research Council / / S20214
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
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59. Lo Curto M, D'Angelo P, Cecchetto G, Klersy C, Dall'Igna P, Federico A, Siracusa F, Alaggio R, Bernini G, Conte M, De Laurentis T, Di Cataldo A, Inserra A, Santoro N, Tamaro P, Indolfi P: Mature and immature teratomas: results of the first paediatric Italian study. Pediatr Surg Int; 2007 Apr;23(4):315-22
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  • Teratoma is the most common germ cell tumour in childhood; mature (MT) and immature teratomas (IT) are benign tumours, but if they recur, they can be in some cases malignant.

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  • (PMID = 17333214.001).
  • [ISSN] 0179-0358
  • [Journal-full-title] Pediatric surgery international
  • [ISO-abbreviation] Pediatr. Surg. Int.
  • [Language] eng
  • [Publication-type] Journal Article; Multicenter Study; Research Support, Non-U.S. Gov't
  • [Publication-country] Germany
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60. Harrison AN, Regelmann WE, Zirbes JM, Milla CE: Longitudinal assessment of lung function from infancy to childhood in patients with cystic fibrosis. Pediatr Pulmonol; 2009 Apr;44(4):330-9
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  • [Title] Longitudinal assessment of lung function from infancy to childhood in patients with cystic fibrosis.
  • We hypothesized that measures of airflow obstruction by IPFT would correlate strongly with lung function by conventional spirometry later in childhood.


61. Velázquez-Cruz R, Orozco L, Espinosa-Rosales F, Carreño-Manjarrez R, Solís-Vallejo E, López-Lara ND, Ruiz-López IK, Rodríguez-Lozano AL, Estrada-Gil JK, Jiménez-Sánchez G, Baca V: Association of PDCD1 polymorphisms with childhood-onset systemic lupus erythematosus. Eur J Hum Genet; 2007 Mar;15(3):336-41
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  • [Title] Association of PDCD1 polymorphisms with childhood-onset systemic lupus erythematosus.
  • However, association to childhood-onset SLE has not been analyzed.
  • The aim of this study was to investigate the association of PDCD1 polymorphisms and haplotypes with susceptibility to childhood-onset SLE in Mexican population.
  • Three PDCD1 SNPs, PD1.3G/A, PD1.5C/T, PD1.6G/A, were analyzed in 250 childhood-onset SLE Mexican patients and 355 healthy controls in a case-control association study.
  • The PD1.3A allele was significantly associated to childhood-onset SLE (P=0.0019, odds ratio (OR) 2.73, 95% confidence interval (95% CI) 1.35-5.56).
  • Our results support association of the PD1.3A SNP to susceptibility of childhood-onset SLE in Mexican population and does not show association to lupus nephritis in this age group.

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  • (PMID = 17228327.001).
  • [ISSN] 1018-4813
  • [Journal-full-title] European journal of human genetics : EJHG
  • [ISO-abbreviation] Eur. J. Hum. Genet.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Antigens, CD; 0 / Apoptosis Regulatory Proteins; 0 / PDCD1 protein, human; 0 / Programmed Cell Death 1 Receptor
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62. Rudnik-Schöneborn S, Berg C, Zerres K, Betzler C, Grimm T, Eggermann T, Eggermann K, Wirth R, Wirth B, Heller R: Genotype-phenotype studies in infantile spinal muscular atrophy (SMA) type I in Germany: implications for clinical trials and genetic counselling. Clin Genet; 2009 Aug;76(2):168-78
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  • [MeSH-major] Clinical Trials as Topic. Genetic Counseling. Spinal Muscular Atrophies of Childhood / genetics. Spinal Muscular Atrophies of Childhood / pathology


63. Harjutsalo V, Podar T, Tuomilehto J: Cumulative incidence of type 1 diabetes in 10,168 siblings of Finnish young-onset type 1 diabetic patients. Diabetes; 2005 Feb;54(2):563-9
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  • The aims of our analysis were to obtain the empirical risk estimates for type 1 diabetes in the siblings of a Finnish population-based cohort of childhood-onset diabetic patients and search for demographic and other factors predicting the risk of type 1 diabetes in siblings.
  • This large prospective family study of type 1 diabetes in siblings of childhood-onset diabetic patients provides reliable empirical estimates for the sibling recurrence risk.

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  • (PMID = 15677516.001).
  • [ISSN] 0012-1797
  • [Journal-full-title] Diabetes
  • [ISO-abbreviation] Diabetes
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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64. Bordon V, Gennery AR, Slatter MA, Vandecruys E, Laureys G, Veys P, Qasim W, Friedrich W, Wulfraat NM, Scherer F, Cant AJ, Fischer A, Cavazzana-Calvo M, Bredius RG, Notarangelo LD, Mazzolari E, Neven B, Güngör T, Inborn Error Working Party of the European Bone Marrow Transplantation (EBMT) group: Clinical and immunologic outcome of patients with cartilage hair hypoplasia after hematopoietic stem cell transplantation. Blood; 2010 Jul 8;116(1):27-35
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  • Thirteen patients were transplanted in early childhood ( approximately 2.5 years).

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  • [ErratumIn] Blood. 2010 Sep 30;116(13):2402. Waseem, Qasim [corrected to Qasim, Waseem];Tayfun, Güngör [corrected to Güngör, Tayfun]
  • [ErratumIn] Blood. 2011 Feb 10;117(6):2077. Cavazanna-Calvo, Marina [corrected to Cavazzana-Calvo, Marina]
  • (PMID = 20375313.001).
  • [ISSN] 1528-0020
  • [Journal-full-title] Blood
  • [ISO-abbreviation] Blood
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Immunoglobulin A; 0 / Immunoglobulin G; 0 / Immunoglobulin M; 0 / RMRP non-coding RNA, human; 0 / RNA, Long Noncoding; 0 / RNA, Untranslated
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65. Schwander M, Sczaniecka A, Grillet N, Bailey JS, Avenarius M, Najmabadi H, Steffy BM, Federe GC, Lagler EA, Banan R, Hice R, Grabowski-Boase L, Keithley EM, Ryan AF, Housley GD, Wiltshire T, Smith RJ, Tarantino LM, Müller U: A forward genetics screen in mice identifies recessive deafness traits and reveals that pejvakin is essential for outer hair cell function. J Neurosci; 2007 Feb 28;27(9):2163-75
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  • [Title] A forward genetics screen in mice identifies recessive deafness traits and reveals that pejvakin is essential for outer hair cell function.
  • Deafness is the most common form of sensory impairment in the human population and is frequently caused by recessive mutations.
  • To obtain animal models for recessive forms of deafness and to identify genes that control the development and function of the auditory sense organs, we performed a forward genetics screen in mice.
  • We identified 13 mouse lines with defects in auditory function and six lines with auditory and vestibular defects.
  • We mapped several of the affected genetic loci and identified point mutations in four genes.
  • Interestingly, all identified genes are expressed in mechanosensory hair cells and required for their function.
  • One mutation maps to the pejvakin gene, which encodes a new member of the gasdermin protein family.
  • Previous studies have described two missense mutations in the human pejvakin gene that cause nonsyndromic recessive deafness (DFNB59) by affecting the function of auditory neurons.
  • In contrast, the pejvakin allele described here introduces a premature stop codon, causes outer hair cell defects, and leads to progressive hearing loss.
  • We also identified a novel allele of the human pejvakin gene in an Iranian pedigree that is afflicted with progressive hearing loss.
  • Our findings suggest that the mechanisms of pathogenesis associated with pejvakin mutations are more diverse than previously appreciated.
  • More generally, our findings demonstrate that recessive screens in mice are powerful tools for identifying genes that control the development and function of mechanosensory hair cells and cause deafness in humans, as well as generating animal models for disease.


66. Yang JJ, Bhojwani D, Yang W, Cai X, Stocco G, Crews K, Wang J, Morrison D, Devidas M, Hunger SP, Willman CL, Raetz EA, Pui CH, Evans WE, Relling MV, Carroll WL: Genome-wide copy number profiling reveals molecular evolution from diagnosis to relapse in childhood acute lymphoblastic leukemia. Blood; 2008 Nov 15;112(10):4178-83
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  • [Title] Genome-wide copy number profiling reveals molecular evolution from diagnosis to relapse in childhood acute lymphoblastic leukemia.
  • The underlying pathways that lead to relapse in childhood acute lymphoblastic leukemia (ALL) are unknown.
  • To comprehensively characterize the molecular evolution of relapsed childhood B-precursor ALL, we used human 500K single-nucleotide polymorphism arrays to identify somatic copy number alterations (CNAs) in 20 diagnosis/relapse pairs relative to germ line.

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  • (PMID = 18768390.001).
  • [ISSN] 1528-0020
  • [Journal-full-title] Blood
  • [ISO-abbreviation] Blood
  • [Language] ENG
  • [Grant] United States / NIGMS NIH HHS / GM / U01 GM061393; United States / NCI NIH HHS / CA / U01 CA114762; United States / NCI NIH HHS / CA / NCI CA 51 001; United States / NIGMS NIH HHS / GM / U01GM61374; United States / NCI NIH HHS / CA / CA21765; United States / NIGMS NIH HHS / GM / U01 GM61393; United States / NCI NIH HHS / CA / P30 CA021765; United States / NIGMS NIH HHS / GM / U01 GM061374; United States / NCI NIH HHS / CA / CA 78 224; United States / NCI NIH HHS / CA / R01 CA093552; United States / NCI NIH HHS / CA / CA093552-02
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antimetabolites, Antineoplastic; 0 / Antineoplastic Agents, Hormonal; 0 / DNA-Binding Proteins; 0 / EBF1 protein, human; 0 / G-T mismatch-binding protein; 0 / IKZF1 protein, human; 0 / Neoplasm Proteins; 0 / Trans-Activators; 148971-36-2 / Ikaros Transcription Factor; 9PHQ9Y1OLM / Prednisolone; E7WED276I5 / 6-Mercaptopurine
  • [Other-IDs] NLM/ PMC2581992
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67. White T, Su S, Schmidt M, Kao CY, Sapiro G: The development of gyrification in childhood and adolescence. Brain Cogn; 2010 Feb;72(1):36-45
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  • [Title] The development of gyrification in childhood and adolescence.
  • Little is known about changes in gyrification during childhood and adolescence, although considering the changes in gray matter volume and thickness during this time period, it is conceivable that alterations in the brain surface morphology could also occur during this period of development.
  • We also present recent findings involving alterations in gyrification during childhood and adolescence.

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  • [Copyright] 2009 Elsevier Inc. All rights reserved.
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  • (PMID = 19942335.001).
  • [ISSN] 1090-2147
  • [Journal-full-title] Brain and cognition
  • [ISO-abbreviation] Brain Cogn
  • [Language] ENG
  • [Grant] United States / NIMH NIH HHS / MH / K08 MH068540; United States / NCRR NIH HHS / RR / P41 RR00807; United States / NIMH NIH HHS / MH / K08 MH068540-05; United States / PHS HHS / / CON000000004051-3014; United States / NINDS NIH HHS / NS / P30 NS057091; United States / NCRR NIH HHS / RR / RR000400-360552; None / None / / K08 MH068540-05; United States / NIMH NIH HHS / MH / MH068540; United States / NCRR NIH HHS / RR / M01 RR000400-360552
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.; Review
  • [Publication-country] United States
  • [Number-of-references] 98
  • [Other-IDs] NLM/ NIHMS162818; NLM/ PMC2815169
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68. Halpern JH, Sherwood AR, Passie T, Blackwell KC, Ruttenber AJ: Evidence of health and safety in American members of a religion who use a hallucinogenic sacrament. Med Sci Monit; 2008 Aug;14(8):SR15-22
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • MATERIAL/METHODS: 32 (out of 40) American members of one branch of the Santo Daime Church were interviewed providing demographic information, physical exam, drug use timeline, a variety of psychological measures, and data about childhood conduct disorder.

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  • (PMID = 18668010.001).
  • [ISSN] 1643-3750
  • [Journal-full-title] Medical science monitor : international medical journal of experimental and clinical research
  • [ISO-abbreviation] Med. Sci. Monit.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Poland
  • [Chemical-registry-number] 0 / Hallucinogens
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69. Apfelbacher CJ, Cairns J, Bruckner T, Möhrenschlager M, Behrendt H, Ring J, Krämer U: Prevalence of overweight and obesity in East and West German children in the decade after reunification: population-based series of cross-sectional studies. J Epidemiol Community Health; 2008 Feb;62(2):125-30
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  • CONCLUSIONS: Unlike in other countries in transition, prevalences of childhood overweight and obesity were increasing in samples of East German children after reunification in 1990, possibly as a result of the rapid adoption of a western lifestyle in the East.

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  • [CommentIn] J Epidemiol Community Health. 2011 Jan;65(1):86 [20961878.001]
  • [ErratumIn] J Epidemiol Community Health. 2011 Jan;65(1):86 [20961878.001]
  • (PMID = 18192600.001).
  • [ISSN] 1470-2738
  • [Journal-full-title] Journal of epidemiology and community health
  • [ISO-abbreviation] J Epidemiol Community Health
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
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70. Fergusson P, Chinkhumba J, Grijalva-Eternod C, Banda T, Mkangama C, Tomkins A: Nutritional recovery in HIV-infected and HIV-uninfected children with severe acute malnutrition. Arch Dis Child; 2009 Jul;94(7):512-6
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  • [Title] Nutritional recovery in HIV-infected and HIV-uninfected children with severe acute malnutrition.
  • RESULTS: In our sample of 454 children with severe acute malnutrition (SAM), 17.4% (n = 79) of children were HIV infected.


71. Sourander A, Jensen P, Rönning JA, Elonheimo H, Niemelä S, Helenius H, Kumpulainen K, Piha J, Tamminen T, Moilanen I, Almqvist F: Childhood bullies and victims and their risk of criminality in late adolescence: the Finnish From a Boy to a Man study. Arch Pediatr Adolesc Med; 2007 Jun;161(6):546-52
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  • [Title] Childhood bullies and victims and their risk of criminality in late adolescence: the Finnish From a Boy to a Man study.
  • OBJECTIVE: To study correlations of childhood bullying and victimization with juvenile criminality.

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  • (PMID = 17548758.001).
  • [ISSN] 1072-4710
  • [Journal-full-title] Archives of pediatrics & adolescent medicine
  • [ISO-abbreviation] Arch Pediatr Adolesc Med
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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72. Lilljebjörn H, Soneson C, Andersson A, Heldrup J, Behrendtz M, Kawamata N, Ogawa S, Koeffler HP, Mitelman F, Johansson B, Fontes M, Fioretos T: The correlation pattern of acquired copy number changes in 164 ETV6/RUNX1-positive childhood acute lymphoblastic leukemias. Hum Mol Genet; 2010 Aug 15;19(16):3150-8
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  • [Title] The correlation pattern of acquired copy number changes in 164 ETV6/RUNX1-positive childhood acute lymphoblastic leukemias.
  • The ETV6/RUNX1 fusion gene, present in 25% of B-lineage childhood acute lymphoblastic leukemia (ALL), is thought to represent an initiating event, which requires additional genetic changes for leukemia development.

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  • (PMID = 20513752.001).
  • [ISSN] 1460-2083
  • [Journal-full-title] Human molecular genetics
  • [ISO-abbreviation] Hum. Mol. Genet.
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / R01 CA026038-31; United States / NCI NIH HHS / CA / R01 CA026038-32; United States / NCI NIH HHS / CA / CA026038-30A2; United States / NCI NIH HHS / CA / R01 CA026038; United States / NCI NIH HHS / CA / R01 CA026038-30A2
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Core Binding Factor Alpha 2 Subunit; 0 / Oncogene Proteins, Fusion; 0 / TEL-AML1 fusion protein
  • [Other-IDs] NLM/ PMC3146010
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73. Dimitrova MM: A study of pregnant women's knowledge of children's feeding practice as a risk factor for early childhood caries. Folia Med (Plovdiv); 2009 Oct-Dec;51(4):40-5
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  • [Title] A study of pregnant women's knowledge of children's feeding practice as a risk factor for early childhood caries.
  • AIM: The aim of the present study was to assess the knowledge pregnant women have of infant and baby's feeding as a risk factor for early childhood caries.
  • The results were analysed using alternative analysis, non-parametric (chi2) test, Student t-test and graphic analysis.
  • RESULTS: The results show a low level of the knowledge pregnant women have of feeding as a potential risk factor for early childhood caries.

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  • (PMID = 20232657.001).
  • [ISSN] 0204-8043
  • [Journal-full-title] Folia medica
  • [ISO-abbreviation] Folia Med (Plovdiv)
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article
  • [Publication-country] Bulgaria
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74. Palle J, Frost BM, Forestier E, Gustafsson G, Nygren P, Hellebostad M, Jonsson OG, Kanerva J, Schmiegelow K, Larsson R, Lönnerholm G, Nordic Society for Paediatric Haematology and Oncology: Cellular drug sensitivity in MLL-rearranged childhood acute leukaemia is correlated to partner genes and cell lineage. Br J Haematol; 2005 Apr;129(2):189-98
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Cellular drug sensitivity in MLL-rearranged childhood acute leukaemia is correlated to partner genes and cell lineage.
  • Rearrangements in the 11q23 region, the site of the mixed lineage leukaemia (MLL) gene, are found in both childhood acute myeloid (AML) and lymphoblastic (ALL) leukaemia.
  • In AML, children with t(9;11) (n = 10) were significantly more sensitive to cytarabine (P < 0.001) and doxorubicin (P = 0.005) than non-11q23 rearranged patients (n = 108).
  • Children with other 11q23 rearrangements (n = 14) differed less from non-rearranged children.
  • [MeSH-major] DNA-Binding Proteins / genetics. Drug Resistance, Neoplasm / genetics. Gene Rearrangement. Leukemia, Myeloid / genetics. Precursor Cell Lymphoblastic Leukemia-Lymphoma / genetics. Proto-Oncogenes / genetics. Transcription Factors / genetics
  • [MeSH-minor] Acute Disease. Adolescent. Antineoplastic Agents / pharmacology. Cell Lineage. Child. Child, Preschool. Chromosomes, Human, Pair 11. Chromosomes, Human, Pair 9. Cytarabine / pharmacology. Cytotoxicity Tests, Immunologic. Doxorubicin / pharmacology. Female. Fluorometry. Glucocorticoids / pharmacology. Histone-Lysine N-Methyltransferase. Humans. Infant. Infant, Newborn. Male. Myeloid-Lymphoid Leukemia Protein. Prospective Studies. Statistics, Nonparametric. Translocation, Genetic

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  • (PMID = 15813846.001).
  • [ISSN] 0007-1048
  • [Journal-full-title] British journal of haematology
  • [ISO-abbreviation] Br. J. Haematol.
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Antineoplastic Agents; 0 / DNA-Binding Proteins; 0 / Glucocorticoids; 0 / MLL protein, human; 0 / Transcription Factors; 04079A1RDZ / Cytarabine; 149025-06-9 / Myeloid-Lymphoid Leukemia Protein; 80168379AG / Doxorubicin; EC 2.1.1.43 / Histone-Lysine N-Methyltransferase
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75. Kalina T, Vaskova M, Mejstrikova E, Madzo J, Trka J, Stary J, Hrusak O: Myeloid antigens in childhood lymphoblastic leukemia: clinical data point to regulation of CD66c distinct from other myeloid antigens. BMC Cancer; 2005;5:38
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  • [Title] Myeloid antigens in childhood lymphoblastic leukemia: clinical data point to regulation of CD66c distinct from other myeloid antigens.
  • BACKGROUND: Aberrant expression of myeloid antigens (MyAgs) on acute lymphoblastic leukemia (ALL) cells is a well-documented phenomenon, although its regulating mechanisms are unclear.
  • CONCLUSION: In contrast to general notion we show that different MyAgs in lymphoblastic leukemia represent different biological circumstances.
  • [MeSH-major] Antigens, CD / biosynthesis. Cell Adhesion Molecules / biosynthesis. Gene Expression Regulation, Neoplastic. Precursor Cell Lymphoblastic Leukemia-Lymphoma / metabolism

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  • (PMID = 15826304.001).
  • [ISSN] 1471-2407
  • [Journal-full-title] BMC cancer
  • [ISO-abbreviation] BMC Cancer
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Antigens, CD; 0 / Antigens, CD15; 0 / Antigens, Differentiation, Myelomonocytic; 0 / CD33 protein, human; 0 / CD65s antigen, human; 0 / CEACAM6 protein, human; 0 / Cell Adhesion Molecules; 0 / GPI-Linked Proteins; 0 / Sialic Acid Binding Ig-like Lectin 3; 63231-63-0 / RNA; EC 3.4.11.2 / Antigens, CD13
  • [Other-IDs] NLM/ PMC1112585
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76. Holmqvist AS, Wiebe T, Hjorth L, Lindgren A, Øra I, Moëll C: Young age at diagnosis is a risk factor for negative late socio-economic effects after acute lymphoblastic leukemia in childhood. Pediatr Blood Cancer; 2010 Oct;55(4):698-707
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Young age at diagnosis is a risk factor for negative late socio-economic effects after acute lymphoblastic leukemia in childhood.
  • BACKGROUND: The increasing number of survivors after childhood cancer requires characterization of the late complications of these diseases and their treatment.
  • We examined a large number of possible socio-economic late effects following treatment for acute lymphoblastic leukemia (ALL) in order to identify factors leading to a poor outcome.
  • CONCLUSIONS: Young age at diagnosis, as well as treatment with cranial irradiation, is a risk factor for socio-economic late effects after treatment for ALL in childhood.
  • [MeSH-major] Precursor Cell Lymphoblastic Leukemia-Lymphoma / therapy


77. Moorman AV, Raimondi SC, Pui CH, Baruchel A, Biondi A, Carroll AJ, Forestier E, Gaynon PS, Harbott J, Harms DO, Heerema N, Pieters R, Schrappe M, Silverman LB, Vilmer E, Harrison CJ, Ponte di Legno Working Group: No prognostic effect of additional chromosomal abnormalities in children with acute lymphoblastic leukemia and 11q23 abnormalities. Leukemia; 2005 Apr;19(4):557-63
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  • [Title] No prognostic effect of additional chromosomal abnormalities in children with acute lymphoblastic leukemia and 11q23 abnormalities.
  • This study characterized the additional chromosomal abnormalities (ACA) associated with 11q23 rearrangements in 450 infants and children with acute lymphoblastic leukemia (ALL) and examined the impact of these ACA on survival.
  • This study has conclusively demonstrated that there is no prognostic effect of secondary chromosomal changes in association with 11q23 abnormalities in childhood ALL.
  • [MeSH-major] Chromosome Aberrations. Chromosomes, Human, Pair 11. Precursor Cell Lymphoblastic Leukemia-Lymphoma / genetics. Precursor Cell Lymphoblastic Leukemia-Lymphoma / mortality

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  • (PMID = 15744345.001).
  • [ISSN] 0887-6924
  • [Journal-full-title] Leukemia
  • [ISO-abbreviation] Leukemia
  • [Language] eng
  • [Grant] United States / NCI NIH HHS / CA / CA21765; United States / NCI NIH HHS / CA / CA36401; United States / NCI NIH HHS / CA / CA51001; United States / NCI NIH HHS / CA / CA60419; United States / NCI NIH HHS / CA / CA78224; United States / NIGMS NIH HHS / GM / GM61393
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] England
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78. Turpeinen M, Pelkonen AS, Nikander K, Sorva R, Selroos O, Juntunen-Backman K, Haahtela T: Bone mineral density in children treated with daily or periodical inhaled budesonide: the Helsinki Early Intervention Childhood Asthma study. Pediatr Res; 2010 Aug;68(2):169-73
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  • [Title] Bone mineral density in children treated with daily or periodical inhaled budesonide: the Helsinki Early Intervention Childhood Asthma study.

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  • (PMID = 20485203.001).
  • [ISSN] 1530-0447
  • [Journal-full-title] Pediatric research
  • [ISO-abbreviation] Pediatr. Res.
  • [Language] eng
  • [Publication-type] Journal Article; Randomized Controlled Trial; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Anti-Asthmatic Agents; 0 / Bronchodilator Agents; 51333-22-3 / Budesonide; Q2WXR1I0PK / Cromolyn Sodium
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79. Warnatz K, Bossaller L, Salzer U, Skrabl-Baumgartner A, Schwinger W, van der Burg M, van Dongen JJ, Orlowska-Volk M, Knoth R, Durandy A, Draeger R, Schlesier M, Peter HH, Grimbacher B: Human ICOS deficiency abrogates the germinal center reaction and provides a monogenic model for common variable immunodeficiency. Blood; 2006 Apr 15;107(8):3045-52
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  • The identification of a total of 9 ICOS-deficient patients revealed that this monogenic disease comprises the full clinical phenotype described for common variable immunodeficiency (CVID), including recurrent bacterial infections, adult as well as childhood onset, splenomegaly, autoimmune phenomena (autoimmune neutropenia), intestinal lymphoid hyperplasia, and malignancy (carcinoma of the vulva).


80. Parslow RC, Tasker RC, Draper ES, Parry GJ, Jones S, Chater T, Thiru K, McKinney PA, Paediatric Intensive Care Audit Network: Epidemiology of critically ill children in England and Wales: incidence, mortality, deprivation and ethnicity. Arch Dis Child; 2009 Mar;94(3):210-5
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  • MAIN OUTCOME MEASURES: Incidence rates for admission and odds ratios (OR) for risk-adjusted mortality by an area based measure of deprivation (Townsend score) and ethnic group (south Asian vs non-south Asian determined using two-name analysis algorithms).
  • RESULTS: The incidence for south Asian children was higher than that of non-south Asian children (138 vs 95/100,000, incidence rate ratio 1.36, 95% CI 1.32 to 1.40).

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  • (PMID = 19106117.001).
  • [ISSN] 1468-2044
  • [Journal-full-title] Archives of disease in childhood
  • [ISO-abbreviation] Arch. Dis. Child.
  • [Language] eng
  • [Publication-type] Journal Article; Multicenter Study; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Investigator] Barnes P; Black N; Booth W; Child BB; Chapple J; Chaudhry B; Darowski M; Durkin N; Kerr S; Langfield I; Marsh M; McFadzean J; McFaul R; Nicholl J; Pearson G; Ralph T; Reekie L; Rowan K; Rowe S; Sammut D; Smith J; Stack C; Tanner S; Tasker R; Baines P; Sister CB; Durward A; Gymer G; Fraser J; Klonin H; Mackerness C; McLaughlin V; O'Donnell R; Oldham G; Pryor D; Wardhaugh A; White D
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81. Kestilä L, Koskinen S, Martelin T, Rahkonen O, Pensola T, Pirkola S, Patja K, Aromaa A: Influence of parental education, childhood adversities, and current living conditions on daily smoking in early adulthood. Eur J Public Health; 2006 Dec;16(6):617-26
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  • [Title] Influence of parental education, childhood adversities, and current living conditions on daily smoking in early adulthood.
  • AIMS: To assess the association of parental education, childhood living conditions and adversities with daily smoking in early adulthood and to analyse the effect of the respondent's own education, main economic activity, and current family structure on these associations.
  • CONCLUSIONS: Childhood living conditions are strong determinants of daily smoking.
  • Much of their influence seems to be mediated through current living conditions, which are also determined by childhood conditions.
  • Parental involvement in fostering non-smoking would be important.

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  • (PMID = 16641156.001).
  • [ISSN] 1101-1262
  • [Journal-full-title] European journal of public health
  • [ISO-abbreviation] Eur J Public Health
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
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82. Remes H, Martikainen P, Valkonen T: Mortality inequalities by parental education among children and young adults in Finland 1990-2004. J Epidemiol Community Health; 2010 Feb;64(2):136-41
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  • This study examines the presence and strength of the association between parental education and mortality during different periods of childhood and young adulthood, and changes in the association over time.
  • The convergence of differences in late childhood, and re-emergence in early adulthood, particularly among men, was, however, related to changes in the cause composition of deaths.

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  • (PMID = 19666635.001).
  • [ISSN] 1470-2738
  • [Journal-full-title] Journal of epidemiology and community health
  • [ISO-abbreviation] J Epidemiol Community Health
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
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83. Hemmingsson T, Melin B, Allebeck P, Lundberg I: The association between cognitive ability measured at ages 18-20 and mortality during 30 years of follow-up--a prospective observational study among Swedish males born 1949-51. Int J Epidemiol; 2006 Jun;35(3):665-70
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  • OBJECTIVES: An association between childhood cognitive ability measured with IQ tests and mortality has been reported recently.


84. Vitko I, Bidaud I, Arias JM, Mezghrani A, Lory P, Perez-Reyes E: The I-II loop controls plasma membrane expression and gating of Ca(v)3.2 T-type Ca2+ channels: a paradigm for childhood absence epilepsy mutations. J Neurosci; 2007 Jan 10;27(2):322-30
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  • [Title] The I-II loop controls plasma membrane expression and gating of Ca(v)3.2 T-type Ca2+ channels: a paradigm for childhood absence epilepsy mutations.
  • Single nucleotide polymorphisms in one of the T-channel genes (CACNA1H, which encodes Ca(v)3.2) are associated with childhood absence epilepsy in a Chinese population.

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  • (PMID = 17215393.001).
  • [ISSN] 1529-2401
  • [Journal-full-title] The Journal of neuroscience : the official journal of the Society for Neuroscience
  • [ISO-abbreviation] J. Neurosci.
  • [Language] eng
  • [Grant] United States / NINDS NIH HHS / NS / NS038691
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / CACNA1H protein, human; 0 / Calcium Channels, T-Type; 0 / Membrane Proteins
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85. Ntalla I, Dedoussis G, Yannakoulia M, Smart MC, Louizou E, Sakka SD, Papoutsakis C, Talmud PJ: ADIPOQ gene polymorphism rs1501299 interacts with fibre intake to affect adiponectin concentration in children: the GENe-Diet Attica Investigation on childhood obesity. Eur J Nutr; 2009 Dec;48(8):493-7
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  • [Title] ADIPOQ gene polymorphism rs1501299 interacts with fibre intake to affect adiponectin concentration in children: the GENe-Diet Attica Investigation on childhood obesity.

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  • [ISSN] 1436-6215
  • [Journal-full-title] European journal of nutrition
  • [ISO-abbreviation] Eur J Nutr
  • [Language] eng
  • [Grant] United Kingdom / British Heart Foundation / / PG2005/014; United Kingdom / Biotechnology and Biological Sciences Research Council / /
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Germany
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86. Zhang Q, Zulfiqar F, Xiao X, Riazuddin SA, Ahmad Z, Caruso R, MacDonald I, Sieving P, Riazuddin S, Hejtmancik JF: Severe retinitis pigmentosa mapped to 4p15 and associated with a novel mutation in the PROM1 gene. Hum Genet; 2007 Nov;122(3-4):293-9
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  • The visual acuity of all affected patients in the family was severely compromised beginning in early childhood.

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  • (PMID = 17605048.001).
  • [ISSN] 0340-6717
  • [Journal-full-title] Human genetics
  • [ISO-abbreviation] Hum. Genet.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
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87. Cheng S, Völgyi E, Tylavsky FA, Lyytikäinen A, Törmäkangas T, Xu L, Cheng SM, Kröger H, Alèn M, Kujala UM: Trait-specific tracking and determinants of body composition: a 7-year follow-up study of pubertal growth in girls. BMC Med; 2009;7:5
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  • BACKGROUND: Understanding how bone (BM), lean (LM) and fat mass (FM) develop through childhood, puberty and adolescence is vital since it holds key information regarding current and future health.

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  • (PMID = 19171028.001).
  • [ISSN] 1741-7015
  • [Journal-full-title] BMC medicine
  • [ISO-abbreviation] BMC Med
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC2639618
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88. Fideleff HL, Boquete HR, Stalldecker G, Giaccio AV, Sobrado PG: Comparative results of a 4-year study on cardiovascular parameters, lipid metabolism, body composition and bone mass between untreated and treated adult growth hormone deficient patients. Growth Horm IGF Res; 2008 Aug;18(4):318-24
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  • In the TG, 22 were childhood-onset (CO) GH-deficient patients, 18-44 years (12 males) and 26 were adult-onset (AO) GH-deficient patients, 27-66 years (10 males).

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  • (PMID = 18289903.001).
  • [ISSN] 1096-6374
  • [Journal-full-title] Growth hormone & IGF research : official journal of the Growth Hormone Research Society and the International IGF Research Society
  • [ISO-abbreviation] Growth Horm. IGF Res.
  • [Language] eng
  • [Publication-type] Controlled Clinical Trial; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Scotland
  • [Chemical-registry-number] 12629-01-5 / Human Growth Hormone; 67763-96-6 / Insulin-Like Growth Factor I
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89. Rudin C, Wolbers M, Nadal D, Rickenbach M, Bucher HC, Pediatric Infectious Disease Group of Switzerland (PIGS), Swiss Mother and Child HIV Cohort Study (MoCHiV): Long-term safety and effectiveness of lopinavir/ritonavir in antiretroviral-experienced HIV-1-infected children. Arch Dis Child; 2010 Jun;95(6):478-81
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  • [Title] Long-term safety and effectiveness of lopinavir/ritonavir in antiretroviral-experienced HIV-1-infected children.
  • AIM: To evaluate the long-term safety and effectiveness of lopinavir/ritonavir (LPV/r) in a population-based cohort of HIV-1-infected children.
  • METHODS: All children enrolled in the Swiss Mother and Child HIV Cohort Study, treated with LPV/r-based combination antiretroviral treatment (cART) between November 2000 and October 2008, were included.
  • RESULTS: 88 children (25 (28%) protease inhibitor (PI)-naive, 16 (18%) ART-naive) were analysed (251 patient-years on LPV/r).
  • After 48 weeks on LPV/r, 70 children had a median (interquartile range (IQR)) decrease in HIV-1 viral load of 4.25 log (5.45-3.17; PI-naive, n=17) and 2.53 (3.68-1.38; PI-experienced, n=53).
  • Median (IQR) increase in CD4 count was 429 (203-593; PI-naive) and 177 (21-331; PI-experienced) cells/microl.
  • These effects remained stable throughout 192 weeks for 25 children.
  • Treatment was stopped for viral rebound in seven and suspected toxicity in 12 children.
  • CONCLUSION: Long-term treatment with LPV/r-based cART is safe and effective in HIV-1-infected children.
  • [MeSH-major] HIV Infections / drug therapy. HIV Protease Inhibitors / therapeutic use. HIV-1 / isolation & purification. Pyrimidinones / therapeutic use. Ritonavir / therapeutic use
  • [MeSH-minor] Adolescent. CD4 Lymphocyte Count. Child. Child, Preschool. Drug Therapy, Combination. Female. Follow-Up Studies. Humans. Lopinavir. Male. Prospective Studies. Treatment Outcome. Viral Load

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  • (PMID = 20501542.001).
  • [ISSN] 1468-2044
  • [Journal-full-title] Archives of disease in childhood
  • [ISO-abbreviation] Arch. Dis. Child.
  • [Language] eng
  • [Publication-type] Clinical Trial; Journal Article; Multicenter Study; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / HIV Protease Inhibitors; 0 / Pyrimidinones; 2494G1JF75 / Lopinavir; O3J8G9O825 / Ritonavir
  • [Investigator] Aebi C; Bär W; Berger Ch; Besson F; Bühlmann U; Cheseaux JJ; Desgrandchamps D; Diana A; Duppenthaler A; Gervaix A; Gnehm HP; Heininger U; Hunziker UA; Kahlert C; Kind C; Kuchler H; Loher A; Masserey-Spicher V; Myers C; Nadal D; Posfay-Barbe C; Rudin C; Schaad UB; Siegrist CA; Stähelin J; Vaudaux B; Wyler-Lazarevic CA; Zingg W; Aebi C; Battegay M; Bernasconi E; Böni J; Brazzola P; Bucher HC; Bürgisser P; Calmy A; Cattacin S; Cavassini M; Cheseaux JJ; Drack G; Dubs R; Egger M; Elzi L; Fischer M; Flepp M; Fontana A; Francioli P; Furrer HJ; Fux C; Gayet-Ageron A; Gerber S; Gorgievski M; Günthard H; Gyr T; Hirsch H; Hirschel B; Hösli I; Hüsler M; Kaiser L; Kahlert Ch; Karrer U; Kind C; Klimkait T; Ledergerber B; Martinetti G; Martinez B; Müller N; Nadal D; Paccaud F; Pantaleo G; Raio L; Rauch A; Regenass S; Rickenbach M; Rudin C; Schmid P; Schultze D; Schüpbach J; Speck R; Taffé P; Telenti A; Trkola A; Vernazza P; Weber R; Wyler CA; Yerly S
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90. Zuna J, Cavé H, Eckert C, Szczepanski T, Meyer C, Mejstrikova E, Fronkova E, Muzikova K, Clappier E, Mendelova D, Boutard P, Schrauder A, Sterba J, Marschalek R, van Dongen JJ, Hrusak O, Stary J, Trka J: Childhood secondary ALL after ALL treatment. Leukemia; 2007 Jul;21(7):1431-5
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  • [Title] Childhood secondary ALL after ALL treatment.
  • Data on secondary acute lymphoblastic leukaemia (sALL) following ALL treatment are very rare.
  • We focused on the recurrences of childhood ALL to discover the real frequency of the sALL after ALL treatment.
  • [MeSH-major] Molecular Diagnostic Techniques / methods. Neoplasms, Second Primary / diagnosis. Precursor Cell Lymphoblastic Leukemia-Lymphoma / diagnosis

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  • (PMID = 17460701.001).
  • [ISSN] 0887-6924
  • [Journal-full-title] Leukemia
  • [ISO-abbreviation] Leukemia
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Antineoplastic Agents
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91. James-Todd T, Tehranifar P, Rich-Edwards J, Titievsky L, Terry MB: The impact of socioeconomic status across early life on age at menarche among a racially diverse population of girls. Ann Epidemiol; 2010 Nov;20(11):836-42
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  • PURPOSE: We sought to evaluate the association between childhood socioeconomic status (SES) at two time points and age at menarche in a multiracial sample of U.S. girls.
  • We used multivariable linear regression to examine the association between SES and age at menarche after adjusting for childhood body mass index (BMI) and other covariates associated with age at menarche.
  • CONCLUSIONS: Our results suggest that lower SES at 7 years and reductions in SES in early childhood are both associated with an earlier age at menarche.

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  • [Copyright] Copyright © 2010 Elsevier Inc. All rights reserved.
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  • (PMID = 20933190.001).
  • [ISSN] 1873-2585
  • [Journal-full-title] Annals of epidemiology
  • [ISO-abbreviation] Ann Epidemiol
  • [Language] ENG
  • [Grant] United States / NCI NIH HHS / CA / K07 CA090685; United States / NCI NIH HHS / CA / K07CA90685
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Other-IDs] NLM/ NIHMS232732; NLM/ PMC3018742
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92. Raimondi SC, Zhou Y, Shurtleff SA, Rubnitz JE, Pui CH, Behm FG: Near-triploidy and near-tetraploidy in childhood acute lymphoblastic leukemia: association with B-lineage blast cells carrying the ETV6-RUNX1 fusion, T-lineage immunophenotype, and favorable outcome. Cancer Genet Cytogenet; 2006 Aug;169(1):50-7
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  • [Title] Near-triploidy and near-tetraploidy in childhood acute lymphoblastic leukemia: association with B-lineage blast cells carrying the ETV6-RUNX1 fusion, T-lineage immunophenotype, and favorable outcome.
  • The prognostic significance of near-triploidy (68-80 chromosomes) and near-tetraploidy (>80 chromosomes) in childhood acute lymphoblastic leukemia (ALL) is unclear.
  • [MeSH-major] B-Lymphocytes / immunology. Core Binding Factor Alpha 2 Subunit / genetics. Oncogene Proteins, Fusion / genetics. Polyploidy. Precursor Cell Lymphoblastic Leukemia-Lymphoma / genetics. T-Lymphocytes / immunology


93. Wu QW, Cai PC, Wang L, Li YR, Kong LL, Hu LH: Family-based association study of Tim-1 and Tim-3 gene polymorphisms with childhood asthma in Chinese trios. Int Arch Allergy Immunol; 2009;150(3):252-60
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  • [Title] Family-based association study of Tim-1 and Tim-3 gene polymorphisms with childhood asthma in Chinese trios.

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  • [Copyright] Copyright 2009 S. Karger AG, Basel.
  • (PMID = 19494522.001).
  • [ISSN] 1423-0097
  • [Journal-full-title] International archives of allergy and immunology
  • [ISO-abbreviation] Int. Arch. Allergy Immunol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 0 / HAVCR1 protein, human; 0 / HAVCR2 protein, human; 0 / Membrane Glycoproteins; 0 / Membrane Proteins; 0 / Receptors, Virus
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94. Staley KG, Kuehni CE, Strippoli MP, McNally T, Silverman M, Stover C: Properdin in childhood and its association with wheezing and atopy. Pediatr Allergy Immunol; 2010 Jun;21(4 Pt 2):e787-91
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  • [Title] Properdin in childhood and its association with wheezing and atopy.
  • We aimed to determine properdin levels in a community-based paediatric sample, and to assess whether levels of properdin were associated with childhood wheeze phenotypes and atopy.

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  • (PMID = 20337960.001).
  • [ISSN] 1399-3038
  • [Journal-full-title] Pediatric allergy and immunology : official publication of the European Society of Pediatric Allergy and Immunology
  • [ISO-abbreviation] Pediatr Allergy Immunol
  • [Language] eng
  • [Grant] United Kingdom / Medical Research Council / / G0400300
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 11016-39-0 / Properdin
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95. Zibat A, Uhmann A, Nitzki F, Wijgerde M, Frommhold A, Heller T, Armstrong V, Wojnowski L, Quintanilla-Martinez L, Reifenberger J, Schulz-Schaeffer W, Hahn H: Time-point and dosage of gene inactivation determine the tumor spectrum in conditional Ptch knockouts. Carcinogenesis; 2009 Jun;30(6):918-26
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  • Furthermore, they point to key differences in the tumorigenic mechanisms underlying adult and childhood tumors.


96. Yeates KO, Armstrong K, Janusz J, Taylor HG, Wade S, Stancin T, Drotar D: Long-term attention problems in children with traumatic brain injury. J Am Acad Child Adolesc Psychiatry; 2005 Jun;44(6):574-84
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  • OBJECTIVE: To examine long-term attention problems and their cognitive correlates after childhood traumatic brain injury (TBI).
  • CONCLUSIONS: Childhood TBI exacerbates premorbid attention problems.
  • Long-term behavioral symptoms of attention problems are related to the cognitive deficits in attention and executive functions that often occur in association with childhood TBI.

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  • (PMID = 15908840.001).
  • [ISSN] 0890-8567
  • [Journal-full-title] Journal of the American Academy of Child and Adolescent Psychiatry
  • [ISO-abbreviation] J Am Acad Child Adolesc Psychiatry
  • [Language] eng
  • [Grant] United States / NINDS NIH HHS / NS / 5R01 NS36335; United States / PHS HHS / / MCJ 390611
  • [Publication-type] Journal Article; Multicenter Study; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
  • [Publication-country] United States
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97. Carlsson G, van't Hooft I, Melin M, Entesarian M, Laurencikas E, Nennesmo I, Trebińska A, Grzybowska E, Palmblad J, Dahl N, Nordenskjöld M, Fadeel B, Henter JI: Central nervous system involvement in severe congenital neutropenia: neurological and neuropsychological abnormalities associated with specific HAX1 mutations. J Intern Med; 2008 Oct;264(4):388-400
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  • [Title] Central nervous system involvement in severe congenital neutropenia: neurological and neuropsychological abnormalities associated with specific HAX1 mutations.
  • OBJECTIVES: Homozygous mutations in the HAX1 gene were recently identified in severe congenital neutropenia patients belonging to the original Kostmann family in northern Sweden.
  • Our observations suggested that these patients also develop neurological and neuropsychological symptoms.
  • METHODS: Detailed clinical studies and mutation analyses were performed in the surviving patients belonging to the Kostmann kindred and in two patients not related to this family, along with studies of HAX1 splice variant expression in normal human tissues.
  • RESULTS: Five of six Kostmann family patients and one other patient from northern Sweden harboured homozygous HAX1 mutations (568C-->T, Q190X) and one carried a heterozygous ELA2 gene mutation.
  • One Swedish patient of Kurdish extraction carried alternative homozygous HAX1 mutations (131G-->A, W44X).
  • All the three patients with Q190X mutations who were alive and available for evaluation developed neurological disease with decreased cognitive function, and three of four patients who reached 10 years developed epilepsy.
  • In contrast, the patients with the ELA2 and W44X HAX1 mutations, respectively, showed no obvious neurological abnormalities.
  • Moreover, two alternative HAX1 splice variants were identified in normal human tissues, including the brain.
  • Both transcripts contained exon 5, harbouring the Q190X mutation, whereas the 5' end of exon 2 containing the W44X mutation was spliced out from the second transcript.
  • CONCLUSIONS: We describe neurological and neuropsychological abnormalities for the first time in Kostmann disease patients.
  • These central nervous system symptoms appear to be associated with specific HAX1 mutations.
  • [MeSH-major] Central Nervous System Diseases / diagnosis. Neutropenia / congenital. Proteins / genetics
  • [MeSH-minor] Adaptor Proteins, Signal Transducing. Adolescent. Adult. DNA Mutational Analysis. Female. Homozygote. Humans. Magnetic Resonance Imaging. Male. Neuropsychological Tests. Pedigree. Point Mutation. Protein Isoforms. Reverse Transcriptase Polymerase Chain Reaction. Sweden

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  • (PMID = 18513342.001).
  • [ISSN] 1365-2796
  • [Journal-full-title] Journal of internal medicine
  • [ISO-abbreviation] J. Intern. Med.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Adaptor Proteins, Signal Transducing; 0 / HAX1 protein, human; 0 / Protein Isoforms; 0 / Proteins
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98. van Delft FW, Bellotti T, Luo Z, Jones LK, Patel N, Yiannikouris O, Hill AS, Hubank M, Kempski H, Fletcher D, Chaplin T, Foot N, Young BD, Hann IM, Gammerman A, Saha V: Prospective gene expression analysis accurately subtypes acute leukaemia in children and establishes a commonality between hyperdiploidy and t(12;21) in acute lymphoblastic leukaemia. Br J Haematol; 2005 Jul;130(1):26-35
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  • [Title] Prospective gene expression analysis accurately subtypes acute leukaemia in children and establishes a commonality between hyperdiploidy and t(12;21) in acute lymphoblastic leukaemia.
  • We have prospectively analysed and correlated the gene expression profiles of children presenting with acute leukaemia to the Royal London and Great Ormond Street Hospitals with morphological diagnosis, immunophenotype and karyotype.
  • Total RNA extracted from freshly sorted blast cells was obtained from 84 lymphoblastic [acute lymphoblastic leukaemia (ALL)], 20 myeloid [acute myeloid leukaemia (AML)] and three unclassified acute leukaemias and hybridised to the high density Affymetrix U133A oligonucleotide array.
  • A novel 50-gene set accurately identified all patients with ALL and AML and predicted for a diagnosis of AML in three patients with unclassified acute leukaemia.
  • A unique gene set was derived for each of eight subtypes of acute leukaemia within our data set.
  • Our analyses demonstrate that not only is microarray analysis the single most effective tool for the diagnosis of acute leukaemias of childhood but it has the ability to identify unique biological pathways.
  • To further evaluate its prognostic value it needs to be incorporated into the routine diagnostic analysis for large-scale clinical trials in childhood acute leukaemias.
  • [MeSH-major] Chromosome Aberrations. Chromosomes, Human, Pair 12. Chromosomes, Human, Pair 21. Gene Expression Profiling. Oligonucleotide Array Sequence Analysis. Precursor Cell Lymphoblastic Leukemia-Lymphoma / genetics
  • [MeSH-minor] Acute Disease. Analysis of Variance. Child. Chromosome Banding. Core Binding Factor Alpha 2 Subunit. DNA-Binding Proteins / genetics. Diagnosis, Differential. Gene Rearrangement. Humans. Immunophenotyping. In Situ Hybridization, Fluorescence. Karyotyping. Leukemia / genetics. Leukemia, Myeloid / diagnosis. Leukemia, Myeloid / genetics. Nuclear Proteins / genetics. Ploidies. Principal Component Analysis. Prospective Studies. Proto-Oncogene Proteins / genetics. Proto-Oncogene Proteins c-ets. Repressor Proteins / genetics. Reproducibility of Results. Reverse Transcriptase Polymerase Chain Reaction. Telomeric Repeat Binding Protein 2 / genetics. Transcription Factors / genetics. Translocation, Genetic

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  • (PMID = 15982341.001).
  • [ISSN] 0007-1048
  • [Journal-full-title] British journal of haematology
  • [ISO-abbreviation] Br. J. Haematol.
  • [Language] eng
  • [Grant] United Kingdom / Cancer Research UK / / 14840
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Core Binding Factor Alpha 2 Subunit; 0 / DNA-Binding Proteins; 0 / ETS translocation variant 6 protein; 0 / Nuclear Proteins; 0 / Proto-Oncogene Proteins; 0 / Proto-Oncogene Proteins c-ets; 0 / RUNX1 protein, human; 0 / Repressor Proteins; 0 / Telomeric Repeat Binding Protein 2; 0 / Transcription Factors
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99. Gast A, Bermejo JL, Flohr T, Stanulla M, Burwinkel B, Schrappe M, Bartram CR, Hemminki K, Kumar R: Folate metabolic gene polymorphisms and childhood acute lymphoblastic leukemia: a case-control study. Leukemia; 2007 Feb;21(2):320-5
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  • [Title] Folate metabolic gene polymorphisms and childhood acute lymphoblastic leukemia: a case-control study.
  • We genotyped six folate metabolic pathway genes for 11 polymorphisms in 460 cases of childhood acute lymphoblastic leukemia (ALL) and 552 ethnically matched controls.
  • Our results suggest that, besides a weak association of childhood ALL with the 66A>G polymorphism, haplotypes within the MTRR gene may, in part, account for population-based differences in risk.
  • [MeSH-major] Folic Acid / genetics. Folic Acid / metabolism. Polymorphism, Genetic. Precursor Cell Lymphoblastic Leukemia-Lymphoma / genetics


100. Goshen Y, Stark B, Kornreich L, Michowiz S, Feinmesser M, Yaniv I: High incidence of meningioma in cranial irradiated survivors of childhood acute lymphoblastic leukemia. Pediatr Blood Cancer; 2007 Sep;49(3):294-7
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  • [Title] High incidence of meningioma in cranial irradiated survivors of childhood acute lymphoblastic leukemia.
  • BACKGROUND: Most survivors of childhood acute lymphoblastic leukemia (ALL) and T-cell lymphoma (T-NHL) treated before 1990 received cranial radiation.
  • This study assessed the occurrence of second tumors in irradiated and non-irradiated survivors.
  • Imaging (MRI, CT) was performed every 3-6 years in 76/88 irradiated and 74/122 non-irradiated patients for the last 20 years.
  • Only one of the 74 non-irradiated patients (median follow-up 14 years) developed meningioma.
  • CONCLUSIONS: Survivors of childhood ALL treated with cranial radiation require prolonged surveillance because of a high incidence of late meningiomas.
  • [MeSH-major] Cranial Irradiation / adverse effects. Meningeal Neoplasms / epidemiology. Meningioma / epidemiology. Neoplasms, Radiation-Induced / epidemiology. Neoplasms, Second Primary / epidemiology. Precursor Cell Lymphoblastic Leukemia-Lymphoma / radiotherapy






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