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1. De Ferro SM, Lage P, Suspiro A, Fidalgo P, Fragoso S, Baltazar C, Vitoriano I, Rodrigues P, Albuquerque C, Leitão CN: [MYH associated polyposis: severe phenotype in the homozygosity for the 1103delC mutation]. Acta Med Port; 2007 May-Jun;20(3):243-7
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  • [Title] [MYH associated polyposis: severe phenotype in the homozygosity for the 1103delC mutation].
  • MYH-associated polyposis (MAP) is an autosomal recessive disease associated with multiple colonic adenomas and colorectal cancer.
  • Her sister aged 24 had 20 colonic adenomas and a severe Spigelman's III duodenal adenomatosis.
  • [MeSH-major] Adenomatous Polyposis Coli / genetics. DNA Glycosylases / genetics. Mutation

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  • (PMID = 17868535.001).
  • [ISSN] 1646-0758
  • [Journal-full-title] Acta médica portuguesa
  • [ISO-abbreviation] Acta Med Port
  • [Language] por
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Portugal
  • [Chemical-registry-number] EC 3.2.2.- / DNA Glycosylases; EC 3.2.2.- / mutY adenine glycosylase
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2. Oyama T, Yamada Y, Hata K, Tomita H, Hirata A, Sheng H, Hara A, Aoki H, Kunisada T, Yamashita S, Mori H: Further upregulation of beta-catenin/Tcf transcription is involved in the development of macroscopic tumors in the colon of ApcMin/+ mice. Carcinogenesis; 2008 Mar;29(3):666-72
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Apc(Min/+) mouse, a mouse model for human familial adenomatosis polyposis, contains a truncating mutation in the Apc gene and spontaneously develops intestinal tumors.

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  • (PMID = 18204079.001).
  • [ISSN] 1460-2180
  • [Journal-full-title] Carcinogenesis
  • [ISO-abbreviation] Carcinogenesis
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Dkk1 protein, mouse; 0 / Intercellular Signaling Peptides and Proteins; 0 / T Cell Transcription Factor 1; 0 / Wnt Proteins; 0 / beta Catenin
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3. Kobayashi S, Sakaguchi H, Takatsuka M, Suekane T, Iwai S, Morikawa H, Enomoto M, Tamori A, Kawada N: Two cases of hepatocellular adenomatosis treated with transcatheter arterial embolization. Hepatol Int; 2009 Jun;3(2):416-20
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  • [Title] Two cases of hepatocellular adenomatosis treated with transcatheter arterial embolization.
  • Surgical resection is considered to be the best choice of treatment, when adenomas are increasing in size, while resection is difficult to perform when multiple adenomas develop throughout the liver.
  • Here, we report two cases of multiple hepatocellular adenomatosis.
  • These cases suggest that TAE is an effective treatment of hepatocellular adenomatosis.

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  • [Cites] Liver Transpl Surg. 1998 Sep;4(5):388-98 [9724476.001]
  • [Cites] Ann Surg. 2000 Jan;231(1):74-81 [10636105.001]
  • [Cites] Am Surg. 1996 Oct;62(10):825-9 [8813164.001]
  • [Cites] Ann Intern Med. 1994 Feb 1;120(3):218-26 [8273986.001]
  • [Cites] Gastroenterology. 1985 Nov;89(5):1132-8 [2412930.001]
  • [Cites] Am J Gastroenterol. 1989 Apr;84(4):429-32 [2929567.001]
  • [Cites] Ann Surg. 1988 Nov;208(5):558-64 [3190282.001]
  • [Cites] Gastroenterology. 1983 May;84(5 Pt 1):994-1002 [6299876.001]
  • [Cites] Gastroenterology. 1982 Apr;82(4):775-82 [6277724.001]
  • [Cites] JAMA. 1979 Aug 17;242(7):644-8 [221698.001]
  • [Cites] Arch Pathol Lab Med. 2003 Oct;127(10):e402-5 [14521443.001]
  • [Cites] World J Gastroenterol. 2003 Mar;9(3):627-30 [12632534.001]
  • [Cites] Clin Liver Dis. 2002 May;6(2):513-26, ix [12122868.001]
  • [Cites] J Clin Gastroenterol. 2002 Apr;34(4):463-71 [11907365.001]
  • [Cites] Abdom Imaging. 2001 May-Jun;26(3):264-8 [11429950.001]
  • [Cites] Radiographics. 2001 Jul-Aug;21(4):877-92; discussion 892-4 [11452062.001]
  • [Cites] Radiology. 2000 Aug;216(2):395-402 [10924560.001]
  • [Cites] Radiol Clin North Am. 1998 Mar;36(2):319-31 [9520985.001]
  • (PMID = 19669369.001).
  • [ISSN] 1936-0533
  • [Journal-full-title] Hepatology international
  • [ISO-abbreviation] Hepatol Int
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC2716763
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4. Xue Y, Johnson R, Desmet M, Snyder PW, Fleet JC: Generation of a transgenic mouse for colorectal cancer research with intestinal cre expression limited to the large intestine. Mol Cancer Res; 2010 Aug;8(8):1095-104
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  • Genetically modified mice have been used for colon cancer research, but findings from these models are confounded by expression of cancer in multiple organs.
  • We sought to create a transgenic mouse with Cre recombinase (Cre) expression limited to the epithelial cells of the large intestine and used this model to study colon cancer driven by adenomatosis polyposis coli (APC) gene inactivation.
  • By 10 weeks, 20% of CAC;APC(580S/+) mice developed adenomatous lesions in the distal colon (3.0 +/- 0.4 mm; 1.1 per mouse).


5. Peters WH, te Morsche RH, Roelofs HM, Mathus-Vliegen EM, Berkhout M, Nagengast FM: COX-2 polymorphisms in patients with familial adenomatous polyposis. Oncol Res; 2009;17(8):347-51
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  • [Title] COX-2 polymorphisms in patients with familial adenomatous polyposis.
  • We investigated whether functional genetic polymorphisms in COX-2 may have a risk-modifying effect on duodenal adenomatosis in patients with familial adenomatous polyposis (FAP).
  • The COX-2 genotypes showed no association with the severity of duodenal adenomatosis.
  • [MeSH-major] Adenomatous Polyposis Coli / enzymology. Adenomatous Polyposis Coli / genetics. Cyclooxygenase 2 / genetics. Genetic Predisposition to Disease. Polymorphism, Single Nucleotide

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  • (PMID = 19544971.001).
  • [ISSN] 0965-0407
  • [Journal-full-title] Oncology research
  • [ISO-abbreviation] Oncol. Res.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] EC 1.14.99.1 / Cyclooxygenase 2
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6. Buttrick GJ, Beaumont LM, Leitch J, Yau C, Hughes JR, Wakefield JG: Akt regulates centrosome migration and spindle orientation in the early Drosophila melanogaster embryo. J Cell Biol; 2008 Feb 11;180(3):537-48
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  • We show that Akt is enriched at the embryonic cortex and is required for phosphorylation of the glycogen synthase kinase-3beta homologue Zeste-white 3 kinase (Zw3) and for the cortical localizations of the adenomatosis polyposis coli (APC)-related protein APC2/E-APC and the MT + Tip protein EB1.

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  • [Cites] J Cell Sci. 2003 Feb 15;116(Pt 4):637-46 [12538764.001]
  • [Cites] Development. 1998 Apr;125(7):1295-303 [9477328.001]
  • [Cites] Nature. 2003 Feb 13;421(6924):753-6 [12610628.001]
  • [Cites] Science. 2003 Sep 12;301(5639):1547-50 [12970569.001]
  • [Cites] Mol Cell Biol. 2004 Jun;24(11):4909-19 [15143183.001]
  • [Cites] Neuron. 2004 Jun 24;42(6):897-912 [15207235.001]
  • [Cites] BMC Cell Biol. 2004 Sep 30;5:37 [15458577.001]
  • [Cites] Development. 1989 Sep;107(1):1-22 [2516798.001]
  • [Cites] EMBO J. 1993 Apr;12(4):1657-69 [8467811.001]
  • [Cites] Development. 1993 Aug;118(4):1245-54 [8269851.001]
  • [Cites] Curr Opin Cell Biol. 1995 Feb;7(1):18-22 [7755985.001]
  • [Cites] Cancer Res. 1995 Jul 15;55(14):2972-7 [7606712.001]
  • [Cites] Nature. 1995 Dec 21-28;378(6559):785-9 [8524413.001]
  • [Cites] J Cell Biol. 1996 Jul;134(1):165-79 [8698812.001]
  • [Cites] J Cell Sci. 1996 Nov;109 ( Pt 11):2649-60 [8937983.001]
  • [Cites] FEBS Lett. 1997 Apr 7;406(1-2):211-5 [9109420.001]
  • [Cites] FEBS Lett. 1997 Oct 27;416(3):307-11 [9373175.001]
  • [Cites] J Biol Chem. 1997 Dec 12;272(50):31515-24 [9395488.001]
  • [Cites] Curr Biol. 1998 May 7;8(10):599-602 [9601646.001]
  • [Cites] Biochem J. 1998 Oct 1;335 ( Pt 1):1-13 [9742206.001]
  • [Cites] Cell. 1999 Mar 19;96(6):857-68 [10102273.001]
  • [Cites] EMBO J. 1999 Apr 15;18(8):2184-95 [10205172.001]
  • [Cites] Curr Biol. 1999 Apr 22;9(8):425-8 [10226031.001]
  • [Cites] J Cell Biol. 1999 Aug 9;146(3):597-608 [10444068.001]
  • [Cites] Genetics. 1999 Sep;153(1):135-77 [10471706.001]
  • [Cites] J Cell Biol. 1999 Sep 20;146(6):1303-18 [10491393.001]
  • [Cites] Mol Biol Cell. 2005 Oct;16(10):4609-22 [16030254.001]
  • [Cites] Mol Biol Cell. 2005 Oct;16(10):4967-81 [16079179.001]
  • [Cites] Nat Cell Biol. 2006 Sep;8(9):963-70 [16921364.001]
  • [Cites] Genes Dev. 2007 Mar 1;21(5):483-96 [17344411.001]
  • [Cites] Genes Cells. 2007 Apr;12(4):535-46 [17397400.001]
  • [Cites] Curr Opin Cell Biol. 2003 Oct;15(5):590-7 [14519394.001]
  • [Cites] Nat Cell Biol. 1999 Jul;1(3):144-51 [10559900.001]
  • [Cites] Mol Biol Cell. 2000 Jan;11(1):241-53 [10637305.001]
  • [Cites] EMBO J. 2000 Jan 17;19(2):241-52 [10637228.001]
  • [Cites] Curr Opin Cell Biol. 2000 Feb;12(1):63-71 [10679357.001]
  • [Cites] Science. 2000 Mar 3;287(5458):1606-9 [10733430.001]
  • [Cites] Curr Biol. 2000 Nov 2;10(21):1339-48 [11084333.001]
  • [Cites] Nat Cell Biol. 2001 Jan;3(1):68-75 [11146628.001]
  • [Cites] Nature. 2001 Jan 25;409(6819):522-5 [11206549.001]
  • [Cites] Curr Biol. 2001 Jan 9;11(1):44-9 [11166179.001]
  • [Cites] Nat Cell Biol. 2001 Oct;3(10):933-8 [11584277.001]
  • [Cites] Nat Cell Biol. 2001 Oct;3(10):E226-8 [11584281.001]
  • [Cites] Curr Biol. 2002 Jun 4;12(11):919-24 [12062056.001]
  • [Cites] J Cell Biol. 2002 Sep 2;158(5):873-84 [12213835.001]
  • [Cites] J Biol Chem. 2002 Sep 20;277(38):35364-70 [12167664.001]
  • [Cites] Mol Endocrinol. 1997 Dec;11(13):1881-90 [9415393.001]
  • [Cites] Biophys J. 2003 Feb;84(2 Pt 1):757-69 [12547760.001]
  • (PMID = 18268102.001).
  • [ISSN] 1540-8140
  • [Journal-full-title] The Journal of cell biology
  • [ISO-abbreviation] J. Cell Biol.
  • [Language] eng
  • [Grant] United Kingdom / Biotechnology and Biological Sciences Research Council / / BBS/B/08019
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / APC2 protein, Drosophila; 0 / Armadillo Domain Proteins; 0 / Drosophila Proteins; 0 / Transcription Factors; 0 / Tumor Suppressor Proteins; 0 / armadillo protein, Drosophila; EC 2.7.11.1 / Proto-Oncogene Proteins c-akt; EC 2.7.11.1 / shaggy protein, Drosophila; EC 2.7.11.26 / Glycogen Synthase Kinase 3
  • [Other-IDs] NLM/ PMC2234228
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7. El Idrissi F, Fadli A: [Erosive adenomatosis of the nipple]. J Gynecol Obstet Biol Reprod (Paris); 2005 Dec;34(8):813-4
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  • [Title] [Erosive adenomatosis of the nipple].
  • The histological analysis led to the diagnosis of erosive nipple adenomatosis.
  • Erosive adenomatosis of the nipple is a benign tumor which developing within the lactic nipple.
  • Erosive adenomatosis of the nipple is very rare and should be suspected in patients with nipple erosions or a nipple tumor.

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  • (PMID = 16319774.001).
  • [ISSN] 0368-2315
  • [Journal-full-title] Journal de gynécologie, obstétrique et biologie de la reproduction
  • [ISO-abbreviation] J Gynecol Obstet Biol Reprod (Paris)
  • [Language] fre
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] France
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8. Plevová P, Drobcinská L, Stekrová J, Silhánová E: [Single nucleotide c.645+32c>T substitution in the APC gene is a non-pathogenic polymorphism appearing in about 16% of the Czech population]. Cas Lek Cesk; 2008;147(5):266-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • BACKGROUND: Familial adenomatous polyposis is an autosomal dominant disease characterised by predisposition to colon polyposis and colorectal cancer and caused by germline mutations in the APC gene.
  • The aim of the study was to establish the frequency of c.645+32C>T substitution in intron 5 of the APC gene in patients with multiple colon polyposis and in the general population and to determine if this substitution is a nonpathogenic polymorphism or a pathogenic mutation associated with multiple polyposis coli.
  • METHODS AND RESULTS: The frequency of c.645+32C>T substitution in the APC gene was established in 170 patients with the clinical phenotype of familial adenomatous polyposis or its attenuated form using denaturating gradient gel electrophoresis and direct sequencing.
  • The c.645+32C>T substitution was detected in 27 of 170 patients with multiple colon polyposis (i.e. 15.9%).
  • The difference between patients with polyposis and the control group was not statistically significant (p = 0.979; chí-square test).
  • [MeSH-major] Adenomatous Polyposis Coli / genetics. Genes, APC. Polymorphism, Single Nucleotide

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  • (PMID = 18630182.001).
  • [ISSN] 0008-7335
  • [Journal-full-title] Casopís lékar̆ů c̆eských
  • [ISO-abbreviation] Cas. Lek. Cesk.
  • [Language] cze
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] Czech Republic
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9. Chimenos-Küstner E, Pascual M, Blanco I, Finestres F: Hereditary familial polyposis and Gardner's syndrome: contribution of the odonto-stomatology examination in its diagnosis and a case description. Med Oral Patol Oral Cir Bucal; 2005 Nov-Dec;10(5):402-9
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  • [Title] Hereditary familial polyposis and Gardner's syndrome: contribution of the odonto-stomatology examination in its diagnosis and a case description.
  • Familial Adenomatous Polyposis (FAP) and its phenotype variant, Gardner's syndrome, constitute a rare autosomal dominant inherited disorder.
  • They are characterised by the development, generally during the second and third decades of life, of multiple adenomatous polyps in the colon and rectum.
  • These polyps have a high risk of subsequently becoming malignant, which normally occurs in the third and fourth decades of life.
  • As well as colorectal polyps, these individuals can present with extra-colonic symptoms, among which are particularly: gastro-duodenal polyps, dermoid and epidermoid cysts, desmoid tumours, congenital hypertrophy of the retinal pigment epithelium, disorders of the maxillary and skeletal bones and dental anomalies.
  • [MeSH-major] Adenomatous Polyposis Coli / diagnosis. Jaw Neoplasms / etiology. Osteoma / etiology
  • [MeSH-minor] Adenomatous Polyposis Coli Protein / genetics. Adult. Cementoma / diagnosis. Cementoma / etiology. Codon, Nonsense. Diagnosis, Differential. Early Diagnosis. Epidermal Cyst / diagnosis. Epidermal Cyst / etiology. Female. Fibroma / diagnosis. Fibroma / etiology. Gardner Syndrome / complications. Gardner Syndrome / diagnosis. Gardner Syndrome / genetics. Genes, APC. Humans. Hypertrophy. Pedigree. Pigment Epithelium of Eye / abnormalities. Prognosis

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  • (PMID = 16264375.001).
  • [ISSN] 1698-6946
  • [Journal-full-title] Medicina oral, patología oral y cirugía bucal
  • [ISO-abbreviation] Med Oral Patol Oral Cir Bucal
  • [Language] eng; spa
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Spain
  • [Chemical-registry-number] 0 / Adenomatous Polyposis Coli Protein; 0 / Codon, Nonsense
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10. Luo JR, Liu XH, Yu QY, Zhang SQ, Zhou JH, Ma XE: [A specific PCR assay and a nested PCR assay for the screening of ovine pulmonary adenomatosis]. Bing Du Xue Bao; 2009 Mar;25(2):125-30
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  • [Title] [A specific PCR assay and a nested PCR assay for the screening of ovine pulmonary adenomatosis].
  • Ovine pulmonary adenomatosis (OPA) is a naturally occurring contagious lung tumor of sheep which was caused by an exogenous retrovirus of sheep, jaagsiekte retrovirus (JSRV).

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  • (PMID = 19678567.001).
  • [ISSN] 1000-8721
  • [Journal-full-title] Bing du xue bao = Chinese journal of virology
  • [ISO-abbreviation] Bing Du Xue Bao
  • [Language] CHI
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] China
  • [Chemical-registry-number] 0 / DNA, Viral
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11. Fujita S, Mekeel KL, Fujikawa T, Kim RD, Foley DP, Hemming AW, Howard RJ, Reed AI, Dixon LR: Liver-occupying focal nodular hyperplasia and adenomatosis associated with intrahepatic portal vein agenesis requiring orthotopic liver transplantation. Transplantation; 2006 Feb 15;81(3):490-2
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  • [Title] Liver-occupying focal nodular hyperplasia and adenomatosis associated with intrahepatic portal vein agenesis requiring orthotopic liver transplantation.


12. Shemerovskiĭ KA: [Constipation--a risk factor for colorectal cancer]. Klin Med (Mosk); 2005;83(12):60-4
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The morphological signs of large bowel pre-cancer (dysplasia, adenomatosis, polyposis) seem to be secondary to the primary functional disorder of the circadian rhythm.
  • [MeSH-minor] Adenoma / epidemiology. Adenoma / etiology. Circadian Rhythm. Colonic Polyps / epidemiology. Colonic Polyps / etiology. Gastrointestinal Diseases / epidemiology. Humans. Precancerous Conditions / epidemiology. Precancerous Conditions / etiology. Quality of Life. Risk Factors. Russia / epidemiology


13. Lepistö A, Kiviluoto T, Halttunen J, Järvinen HJ: Surveillance and treatment of duodenal adenomatosis in familial adenomatous polyposis. Endoscopy; 2009 Jun;41(6):504-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Surveillance and treatment of duodenal adenomatosis in familial adenomatous polyposis.
  • BACKGROUND AND STUDY AIMS: Patients with familial adenomatous polyposis (FAP) are at increased risk for duodenal cancer whereas colorectal cancer is largely prevented by prophylactic colectomy.
  • We analyzed the results of endoscopic surveillance and different treatment modalities of duodenal adenomatosis in patients with FAP.
  • The cumulative incidences of duodenal adenomatosis and severe dysplasia and cancer were calculated using Kaplan-Meier analysis.
  • RESULTS: By the age of 60 years, the cumulative incidence was 80% for any adenomatosis and 23% for severe dysplasia or cancer.
  • CONCLUSIONS: The high incidence of severe dysplasia and cancer in duodenal polyps suggests that endoscopic surveillance is essential.
  • High-risk patients with Spigelman IV adenomatosis or adenomas with persisting severe dysplasia should undergo surgery with pylorus-preserving pancreaticoduodenectomy before invasive cancer develops.
  • [MeSH-major] Adenomatous Polyposis Coli / complications. Duodenal Neoplasms / diagnosis. Duodenal Neoplasms / epidemiology. Intestinal Polyps / complications


14. Durno CA, Holter S, Sherman PM, Gallinger S: The gastrointestinal phenotype of germline biallelic mismatch repair gene mutations. Am J Gastroenterol; 2010 Nov;105(11):2449-56
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • More than one-third of patients had multiple colorectal adenomas (>10 polyps).
  • This distinct phenotype includes multiple adenomatous polyps and CAL skin lesions.

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  • (PMID = 20531397.001).
  • [ISSN] 1572-0241
  • [Journal-full-title] The American journal of gastroenterology
  • [ISO-abbreviation] Am. J. Gastroenterol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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15. Belingheri M, Ghio L, Sala A, Menni F, Trespidi L, Ferraresso M, Berardinelli L, Rossi G, Edefonti A, Parini R: Combined liver-kidney transplantation in glycogen storage disease Ia: a case beyond the guidelines. Liver Transpl; 2007 May;13(5):762-4
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Liver transplantation is the only radical treatment, for which the main indications are hepatic adenomatosis, hepatocellular carcinoma, or severe hepatic dysfunction.


16. Akatsu T, Aiura K, Takahashi S, Kameyama K, Kitajima M, Kitagawa Y: Recurrent pancreatitis caused by ampullary carcinoma and minor papilla adenoma in familial polyposis: report of a case. Surg Today; 2008;38(5):440-4
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Recurrent pancreatitis caused by ampullary carcinoma and minor papilla adenoma in familial polyposis: report of a case.
  • We report a case of relapsing pancreatitis in familial adenomatous polyposis (FAP) with severe duodenal adenomatosis (Spigelman's stage IV).
  • Upper endoscopy showed multiple duodenal adenomas including carcinoma involving the papilla of Vater.
  • [MeSH-major] Adenocarcinoma / etiology. Adenoma / etiology. Adenomatous Polyposis Coli / complications. Ampulla of Vater. Common Bile Duct Neoplasms / etiology. Pancreatic Neoplasms / etiology. Pancreatitis / etiology


17. Bliznashki I, Minev M, Mikhova A, Velev M: [A rare case of Gardner's syndrome complicated with rectal carcinoma]. Khirurgiia (Sofiia); 2007;(3):60-3
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  • Gardner's syndrome is a rare variant of the Familial Adenomatous Polyposis (FAP) in which affected individuals develop thousands of polyps within the gastrointestinal tract, with a 100 % risk of eventual malignant change.
  • Gardner's syndrome is an autosomal dominant disease, caused by mutations in APC ( adenomatous polyposis coli ) gene, which is located in chromosomal locus 5q21- q22.
  • They have investigated a family of 51 members with polyposis, some of them with multiple epidermoid cysts, fibromas and jaw osteomas.
  • He had multiple adenomatous polyposis of colon, rectal cancer, osteomas of skull bones, subcutaneous fibromas and lipomas.
  • We discovered also by ultrasound examination a polyp of gall bladder.
  • His father has had also multiple polyposis with malignancy and metastatic lesions and he has died at age of 49 years.
  • [MeSH-minor] Adenomatous Polyposis Coli / diagnosis. Adenomatous Polyposis Coli / genetics. Adenomatous Polyposis Coli / surgery. Adult. Cholecystectomy. Epidermal Cyst / diagnosis. Epidermal Cyst / genetics. Epidermal Cyst / surgery. Humans. Male. Osteoma / diagnosis. Osteoma / genetics. Osteoma / surgery. Proctocolectomy, Restorative

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  • (PMID = 18437113.001).
  • [ISSN] 0450-2167
  • [Journal-full-title] Khirurgii︠a︡
  • [ISO-abbreviation] Khirurgiia (Sofiia)
  • [Language] bul
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Bulgaria
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18. Zhang W, Chuang YJ, Jin T, Swanson R, Xiong Y, Leung L, Olson ST: Antiangiogenic antithrombin induces global changes in the gene expression profile of endothelial cells. Cancer Res; 2006 May 15;66(10):5047-55
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  • In contrast, most up-regulated genes (e.g., caspase-3, p21, tissue inhibitor of metalloproteinases 1, 2, and 3, and adenomatosis polyposis coli) are known for their antiangiogenic functions which include the promotion of cell apoptosis and cell cycle arrest and the inhibition of tumor growth and metastasis.

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  • (PMID = 16707426.001).
  • [ISSN] 0008-5472
  • [Journal-full-title] Cancer research
  • [ISO-abbreviation] Cancer Res.
  • [Language] eng
  • [Grant] United States / NHLBI NIH HHS / HL / HL-39888
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Angiogenesis Inhibitors; 0 / Antithrombins; 103107-01-3 / Fibroblast Growth Factor 2
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19. Barreda Costa C, Vila Gutierrez S, Salazar Cabrera F, Barriga Calle E, Velarde Criado H, Barriga Briceño J: [Advanced adenoma in 3700 colonoscopies]. Rev Gastroenterol Peru; 2010 Apr-Jun;30(2):113-20
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  • OBJECTIVES: To determine the prevalence of colon polyps, distribution in the colon, as well as their histological characteristics, with special mention on advanced adenomas, in an adult population at an endoscopy center in Lima.
  • Each polyp was studied separately and its histological findings recorded.
  • RESULTS: 3,701 colonoscopies were done in 3,690 patients; 1,492 (40,4%) had polyps and were included in the study.
  • 997 (27%) had adenomatous polyps and 495 (13.4%) non adenomatous polyps.
  • Adenomatous polyps were found throughout the colon, with a predominance of hyperplasic polyps in the rectum.
  • 78% of the adenomatous polyps and 84% of the advanced adenomas were found in patients older than 50 years old.
  • In this group of patients, significant risk factors were: prior history of adenomas or colorectal cancer (46/203 vs. 63/495; p<0.01), and multiple adenomatous polyps found during colonoscopy (67/203 vs. 121/794; p<0.01).
  • CONCLUSIONS: Adenomatous polyps were the most frequently found polyps in our study; approximately 20% were advanced lesions, especially in patients older than 50 years old.
  • [MeSH-major] Adenocarcinoma / epidemiology. Adenoma / epidemiology. Colonic Neoplasms / epidemiology. Colonic Polyps / epidemiology. Colonoscopy / statistics & numerical data. Rectal Neoplasms / epidemiology
  • [MeSH-minor] Adenomatous Polyps / diagnosis. Adenomatous Polyps / epidemiology. Adenomatous Polyps / pathology. Adult. Age Factors. Aged. Aged, 80 and over. Early Diagnosis. Humans. Hyperplasia. Middle Aged. Motivation. Neoplasms, Multiple Primary / diagnosis. Neoplasms, Multiple Primary / epidemiology. Neoplasms, Multiple Primary / pathology. Organ Specificity. Peru / epidemiology. Prospective Studies. Young Adult

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  • (PMID = 20644602.001).
  • [ISSN] 1609-722X
  • [Journal-full-title] Revista de gastroenterología del Perú : órgano oficial de la Sociedad de Gastroenterología del Perú
  • [ISO-abbreviation] Rev Gastroenterol Peru
  • [Language] spa
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] Peru
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20. Gorbacheva OS, Popova LP, Ivanov SV, Iakovleva MV, Bondarev VP: [Cytological classification of nontoxic nodular goiter]. Klin Lab Diagn; 2008 Jan;(1):37-9
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  • By comparing the cytological and histological data on thyroid nodular lesions in 715 patients, the authors have arrived at the conclusion that with a cytological response of colloidal goiter and colloidal goiter with adenomatosis, the cancer detection rate is not greater than 4%.
  • With a cytological response of adenomatosis with atypia and follicular tumor, a malignancy occurs in 9 to 13%.
  • It is recommended that cytological data should be grouped by the type of a morphological classification of thyroid nodular diseases: colloidal goiter (with regression, proliferation, adenomatosis, cystic cavity); tumors (adenomatosis with atypia, follicular tumor); cancer.

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  • (PMID = 18320618.001).
  • [ISSN] 0869-2084
  • [Journal-full-title] Klinicheskaia laboratornaia diagnostika
  • [ISO-abbreviation] Klin. Lab. Diagn.
  • [Language] rus
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] Russia (Federation)
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21. Astakhova TM, Delone GV, Lyupina YV, Abramova EB, Uryvaeva IV, Sharova NP: Changes in the Proteasome Pool during Malignant Transformation of Mouse Liver Cells. Acta Naturae; 2010 Apr;2(1):102-8
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  • Multiple forms of proteasomes regulate cellular processes by destroying proteins or forming the peptides involved in those processes.
  • Our results indicate that nodular regenerative hyperplasia and adenomatosis may be stages preceding carcinogenesis.

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  • [Cites] Biochem Biophys Res Commun. 2007 Apr 6;355(2):549-54 [17303071.001]
  • [Cites] Breast Cancer Res Treat. 2007 Jul;104(1):21-30 [17004105.001]
  • [Cites] Ther Clin Risk Manag. 2006 Sep;2(3):271-9 [18360602.001]
  • [Cites] J Virol. 2009 Jan;83(2):612-21 [18971267.001]
  • [Cites] Monogr Dev Biol. 1992;23:230-6 [1614438.001]
  • [Cites] Cancer Res. 1982 Oct;42(10):3918-23 [7104991.001]
  • [Cites] J Biol Chem. 1997 May 9;272(19):12328-41 [9139677.001]
  • [Cites] Arch Biochem Biophys. 2002 Sep 15;405(2):147-53 [12220526.001]
  • [Cites] J Biol Chem. 1951 Nov;193(1):265-75 [14907713.001]
  • [Cites] Int J Oncol. 2004 Dec;25(6):1625-9 [15547699.001]
  • [Cites] J Immunol. 2006 Apr 1;176(7):4075-82 [16547243.001]
  • [Cites] Acta Neuropathol. 2007 Aug;114(2):111-9 [17541610.001]
  • [Cites] Mol Med. 2008 Mar-Apr;14(3-4):141-9 [18091979.001]
  • [Cites] Proc Jpn Acad Ser B Phys Biol Sci. 2009;85(1):12-36 [19145068.001]
  • [Cites] J Biol Chem. 1996 Jun 28;271(26):15504-9 [8663134.001]
  • [Cites] Free Radic Biol Med. 1996;20(1):63-73 [8903680.001]
  • [Cites] Proc Natl Acad Sci U S A. 1997 Aug 19;94(17):8970-5 [9256419.001]
  • [Cites] Biull Eksp Biol Med. 1997 Oct;124(10):364-8 [9410159.001]
  • [Cites] J Exp Med. 1998 Jan 5;187(1):97-104 [9419215.001]
  • [Cites] Cell Res. 1998 Sep;8(3):195-207 [9791733.001]
  • [Cites] Annu Rev Immunol. 1999;17:739-79 [10358773.001]
  • [Cites] J Biol Chem. 1999 Jul 30;274(31):21963-72 [10419519.001]
  • [Cites] Cancer Res. 2004 Sep 1;64(17):6319-26 [15342421.001]
  • [Cites] Clin Cancer Res. 2005 Apr 15;11(8):2868-74 [15837734.001]
  • [Cites] Cancer Res. 2005 Jul 1;65(13):5599-606 [15994932.001]
  • [Cites] Free Radic Biol Med. 2006 Mar 15;40(6):1034-44 [16540399.001]
  • [Cites] Ontogenez. 2006 May-Jun;37(3):171-8 [16813218.001]
  • [Cites] Biochim Biophys Acta. 2007 Sep;1773(9):1389-97 [17582523.001]
  • (PMID = 22649635.001).
  • [ISSN] 2075-8251
  • [Journal-full-title] Acta naturae
  • [ISO-abbreviation] Acta Naturae
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Russia (Federation)
  • [Other-IDs] NLM/ PMC3347545
  • [Keywords] NOTNLM ; 19S proteasome activator / Western blot analysis / adenoma / chymotrypsin–like activity of proteasomes / hepatocellular carcinoma / immunoproteasomes / mouse liver / nodular regenerative hyperplasia of the liver
  •  go-up   go-down


22. Upadhyay G, Goessling W, North TE, Xavier R, Zon LI, Yajnik V: Molecular association between beta-catenin degradation complex and Rac guanine exchange factor DOCK4 is essential for Wnt/beta-catenin signaling. Oncogene; 2008 Oct 2;27(44):5845-55
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  • Biochemical studies demonstrate that DOCK4 interacts with the beta-catenin degradation complex, consisting of the proteins adenomatosis polyposis coli, Axin and glycogen synthase kinase 3beta (GSK3beta).


23. Okegawa T, Nutahara K, Higashihara E: Association of circulating tumor cells with tumor-related methylated DNA in patients with hormone-refractory prostate cancer. Int J Urol; 2010 May;17(5):466-75
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  • Hypermethylation at adenomatosis polyposis coli, glutathione-S-transferase-pi, prostaglandin-endoperoxide synthase 2, multidrug resistance 1 and Ras association domain family 1 isoform A was analyzed using a sensitive SYBR green methylation-specific polymerase chain reaction.
  • [MeSH-minor] Adenomatous Polyposis Coli Protein / genetics. Aged. Aged, 80 and over. Antineoplastic Agents, Hormonal / therapeutic use. Biopsy. Cell Count. CpG Islands / physiology. Drug Resistance, Neoplasm. Glutathione S-Transferase pi / genetics. Humans. Kaplan-Meier Estimate. Male. Middle Aged. P-Glycoprotein / genetics. Prognosis. Proportional Hazards Models. Prostaglandin-Endoperoxide Synthases / genetics. Reverse Transcriptase Polymerase Chain Reaction. Tumor Suppressor Proteins / genetics

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  • (PMID = 20337729.001).
  • [ISSN] 1442-2042
  • [Journal-full-title] International journal of urology : official journal of the Japanese Urological Association
  • [ISO-abbreviation] Int. J. Urol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Australia
  • [Chemical-registry-number] 0 / Adenomatous Polyposis Coli Protein; 0 / Antineoplastic Agents, Hormonal; 0 / Biomarkers, Tumor; 0 / DNA, Neoplasm; 0 / P-Glycoprotein; 0 / RASSF1 protein, human; 0 / Tumor Suppressor Proteins; EC 1.14.99.1 / Prostaglandin-Endoperoxide Synthases; EC 2.5.1.18 / Glutathione S-Transferase pi
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24. Kazanjian A, Noah T, Brown D, Burkart J, Shroyer NF: Atonal homolog 1 is required for growth and differentiation effects of notch/gamma-secretase inhibitors on normal and cancerous intestinal epithelial cells. Gastroenterology; 2010 Sep;139(3):918-28, 928.e1-6
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  • METHODS: We studied mice with intestine-specific disruption of Atoh1 (Atoh1(Deltaintestine)), the adenomatosis polyposis coli (APC)(min) mutation, both mutations (Atoh1(Deltaintestine); APC(min)), or littermate controls; mice were given GSI or vehicle.

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  • [Copyright] Copyright © 2010 AGA Institute. Published by Elsevier Inc. All rights reserved.
  • [Cites] Nat Genet. 2000 Jan;24(1):36-44 [10615124.001]
  • [Cites] Curr Alzheimer Res. 2010 May;7(3):207-9 [20088802.001]
  • [Cites] J Biol Chem. 2003 Nov 14;278(46):46107-16 [12949072.001]
  • [Cites] J Biol Chem. 2004 Mar 26;279(13):12876-82 [14709552.001]
  • [Cites] Cancer Res. 2004 Sep 1;64(17):6050-7 [15342386.001]
  • [Cites] Toxicol Sci. 2004 Nov;82(1):341-58 [15319485.001]
  • [Cites] Genomics. 1994 Mar 1;20(1):56-61 [8020957.001]
  • [Cites] Hum Mol Genet. 1996 Sep;5(9):1207-16 [8872459.001]
  • [Cites] Curr Biol. 1996 Oct 1;6(10):1290-301 [8939576.001]
  • [Cites] J Med Chem. 1998 Jan 1;41(1):6-9 [9438016.001]
  • [Cites] Nature. 1999 Apr 8;398(6727):518-22 [10206645.001]
  • [Cites] Development. 2005 Mar;132(5):1093-104 [15689380.001]
  • [Cites] Nature. 2005 Jun 16;435(7044):959-63 [15959515.001]
  • [Cites] Nature. 2005 Jun 16;435(7044):964-8 [15959516.001]
  • [Cites] Am J Physiol Gastrointest Liver Physiol. 2005 Sep;289(3):G381-7 [16093419.001]
  • [Cites] Proc Natl Acad Sci U S A. 2005 Aug 30;102(35):12443-8 [16107537.001]
  • [Cites] Int J Hematol. 2005 Nov;82(4):295-301 [16298817.001]
  • [Cites] Development. 2007 Feb;134(3):535-44 [17215306.001]
  • [Cites] Gastroenterology. 2007 Jan;132(1):208-20 [17241872.001]
  • [Cites] Gastroenterology. 2007 Jun;132(7):2478-88 [17570220.001]
  • [Cites] Gastroenterology. 2008 Jan;134(1):131-44 [18166351.001]
  • [Cites] EMBO Rep. 2008 Apr;9(4):377-83 [18274550.001]
  • [Cites] Mol Cancer Res. 2008 Dec;6(12):1920-7 [19074836.001]
  • [Cites] Nat Med. 2009 Jan;15(1):50-8 [19098907.001]
  • [Cites] PLoS Biol. 2009 Feb 24;7(2):e39 [19243219.001]
  • [Cites] Proc Natl Acad Sci U S A. 2009 Apr 14;106(15):6315-20 [19325125.001]
  • [Cites] Science. 2001 Dec 7;294(5549):2155-8 [11739954.001]
  • (PMID = 20621629.001).
  • [ISSN] 1528-0012
  • [Journal-full-title] Gastroenterology
  • [ISO-abbreviation] Gastroenterology
  • [Language] eng
  • [Grant] United States / NIDDK NIH HHS / DK / K01 DK071686; United States / NIDDK NIH HHS / DK / K01 DK071686; United States / NIDDK NIH HHS / DK / P30 DK078392; United States / NCI NIH HHS / CA / R01 CA142826; United States / NCI NIH HHS / CA / R01 CA142826; United States / NCI NIH HHS / CA / R01 CA142826-01; United States / NIDDK NIH HHS / DK / R03 DK084167; United States / NIDDK NIH HHS / DK / R03 DK084167-01; United States / NIDDK NIH HHS / DK / T35 DK060444
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / ATOH1 protein, human; 0 / Antineoplastic Agents; 0 / Atoh1 protein, mouse; 0 / Basic Helix-Loop-Helix Transcription Factors; 0 / RNA, Messenger; 0 / Receptors, Notch; EC 3.4.- / Amyloid Precursor Protein Secretases
  • [Other-IDs] NLM/ NIHMS215773; NLM/ PMC3197859
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25. Chan OT, Haghighi P: Hamartomatous polyps of the colon: ganglioneuromatous, stromal, and lipomatous. Arch Pathol Lab Med; 2006 Oct;130(10):1561-6
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  • [Title] Hamartomatous polyps of the colon: ganglioneuromatous, stromal, and lipomatous.
  • Intestinal ganglioneuromas comprise benign, hamartomatous polyps characterized by an overgrowth of nerve ganglion cells, nerve fibers, and supporting cells in the gastrointestinal tract.
  • This polyposis has been divided into 3 subgroups, each with a different degree of ganglioneuroma formation: polypoid ganglioneuroma, ganglioneuromatous polyposis, and diffuse ganglioneuromatosis.
  • The ganglioneuromatous polyposis subgroup is not known to coexist with systemic disorders that often have an associated intestinal polyposis, such as multiple endocrine neoplasia type IIb, neurofibromatosis type I, and Cowden syndrome.
  • We report a case of ganglioneuromatous polyposis plus cutaneous lipomatosis in a 41-year-old man with no established systemic disease.
  • This case report and brief review of the literature provide an overview of intestinal ganglioneuromatosis in relation to the hereditary polyposis syndromes and describe the individual ganglioneuromatosis subgroups.
  • [MeSH-major] Colonic Neoplasms / pathology. Ganglioneuroma / pathology. Hamartoma / pathology. Intestinal Polyposis / pathology. Lipomatosis / complications. Skin Neoplasms / complications. Stromal Cells / pathology

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  • (PMID = 17090203.001).
  • [ISSN] 1543-2165
  • [Journal-full-title] Archives of pathology & laboratory medicine
  • [ISO-abbreviation] Arch. Pathol. Lab. Med.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 47
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26. Sa Cunha A, Blanc JF, Lazaro E, Mellottee L, Le Bail B, Zucman-Rossi J, Balabaud C, Bioulac-Sage P: Inflammatory syndrome with liver adenomatosis: the beneficial effects of surgical management. Gut; 2007 Feb;56(2):307-9
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  • [Title] Inflammatory syndrome with liver adenomatosis: the beneficial effects of surgical management.

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  • [Cites] J La State Med Soc. 1999 Sep;151(9):474-8 [11284149.001]
  • [Cites] Nat Genet. 2002 Oct;32(2):312-5 [12355088.001]
  • [Cites] Gastroenterology. 2005 May;128(5):1211-8 [15887105.001]
  • [Cites] Hepatology. 2006 Mar;43(3):515-24 [16496320.001]
  • [Cites] Nephron. 1997;75(3):350-3 [9069459.001]
  • [Cites] Gut. 1990 Mar;31(3):361-3 [2157638.001]
  • [Cites] Nephron. 1993;64(1):157-8 [8502327.001]
  • [Cites] J Chir (Paris). 1993 Jun-Jul;130(6-7):297-9 [8408330.001]
  • [Cites] Liver. 1995 Jun;15(3):164-7 [7674844.001]
  • [Cites] J Hepatol. 1986;2(3):450-7 [3722793.001]
  • (PMID = 17303609.001).
  • [ISSN] 0017-5749
  • [Journal-full-title] Gut
  • [ISO-abbreviation] Gut
  • [Language] eng
  • [Publication-type] Case Reports; Letter
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC1856757
  •  go-up   go-down


27. Esteban JM, Gutiérrez del Olmo A, Baki W, Fernández S, Soria MT, Díaz Mediavilla J, Ramírez Armengol JA: Colonic mucosa-associated lymphoid tissue lymphoma presenting as multiple polyposis. Gastrointest Endosc; 2005 Jun;61(7):928-30
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  • [Title] Colonic mucosa-associated lymphoid tissue lymphoma presenting as multiple polyposis.
  • [MeSH-major] Colonic Neoplasms / pathology. Colonic Polyps / pathology. Intestinal Polyposis / pathology. Lymphoma, B-Cell, Marginal Zone / pathology

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  • (PMID = 15933708.001).
  • [ISSN] 0016-5107
  • [Journal-full-title] Gastrointestinal endoscopy
  • [ISO-abbreviation] Gastrointest. Endosc.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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28. Bober A: Passing it on: should health care professionals be permitted to disclose patients' genetic information to their reproductive partners? J Law Med; 2006 Nov;14(2):262-81
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  • The issue is addressed with reference to four genetic disorders (Huntington Disease, Familial Adenomatous Polyposis, Multiple Endocrine Neoplasia Type 2A and Cystic Fibrosis) which illustrate differences in inheritance traits and availability of effective treatments.
  • [MeSH-minor] Adenomatous Polyposis Coli / genetics. Australia. Cystic Fibrosis / genetics. Humans. Huntington Disease / genetics. Multiple Endocrine Neoplasia Type 2a / genetics

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  • (PMID = 17153530.001).
  • [ISSN] 1320-159X
  • [Journal-full-title] Journal of law and medicine
  • [ISO-abbreviation] J Law Med
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Australia
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29. Dawson S, Else RW, Rhind SM, Collie DD: Diagnostic value of cytology of bronchoalveolar fluid for lung diseases of sheep. Vet Rec; 2005 Oct 8;157(15):433-6
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  • The diffuse lung diseases, maedi and adenomatosis, were both characterised by an increase in overall cellularity and by increases in the percentages of lymphocytes and neutrophils, respectively.

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  • (PMID = 16215243.001).
  • [ISSN] 0042-4900
  • [Journal-full-title] The Veterinary record
  • [ISO-abbreviation] Vet. Rec.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
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30. Lejeune S, Guillemot F, Triboulet JP, Cattan S, Mouton C, PAFNORD Group, Porchet N, Manouvrier S, Buisine MP: Low frequency of AXIN2 mutations and high frequency of MUTYH mutations in patients with multiple polyposis. Hum Mutat; 2006 Oct;27(10):1064
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  • [Title] Low frequency of AXIN2 mutations and high frequency of MUTYH mutations in patients with multiple polyposis.
  • Familial adenomatous polyposis has been linked to germline mutations in the APC tumor suppressor gene.
  • However, a number of patients with familial adenomatous polyposis (with either classical or attenuated phenotype) have no APC mutation.
  • Moreover, biallelic mutations in the base excision repair gene MUTYH have been associated with polyposis and early-onset colorectal cancer.
  • AXIN2 and MUTYH genes were screened for germline mutations by PCR and direct sequencing in 39 unrelated patients with multiple adenomas or colorectal cancer without evidence of APC mutation nor mismatch repair defect.
  • Two novel AXIN2 variants were detected in one patient with multiple adenomas, but no clearly pathogenic mutation.
  • Biallelic MUTYH mutations were only found in patients with multiple adenomatous polyposis (7 out of 22 (32%)).
  • Moreover, one patient with biallelic MUTYH mutations presented with multiple adenomas and severe tooth agenesis.
  • Therefore, germline mutations are rare in AXIN2 but frequent in MUTYH in patients with multiple adenomas.
  • [MeSH-major] Adenomatous Polyposis Coli / genetics. Cytoskeletal Proteins / genetics. DNA Glycosylases / genetics. Mutation / genetics

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  • (PMID = 16941501.001).
  • [ISSN] 1098-1004
  • [Journal-full-title] Human mutation
  • [ISO-abbreviation] Hum. Mutat.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / AXIN2 protein, human; 0 / Axin Protein; 0 / Cytoskeletal Proteins; EC 3.2.2.- / DNA Glycosylases; EC 3.2.2.- / mutY adenine glycosylase
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31. Radford IR, Lobachevsky PN: Clustered DNA lesion sites as a source of mutations during human colorectal tumourigenesis. Mutat Res; 2008 Nov 10;646(1-2):60-8
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  • We used statistical analyses to search for sets of dinucleotide sequences (designated target sequences) that are present at and in close proximity to mutation sites in four genes associated with human colorectal tumourigenesis (adenomatosis polyposis coli (APC), v-Ki-ras2 Kirsten rat sarcoma viral oncogene homolog (KRAS), phosphoinositide-3-kinase, catalytic, alpha polypeptide (PIK3CA), and tumour protein p53 (TP53)).
  • Our results strongly suggest a statistical association between the presence of multiple, clustered target sequences and mutational events.

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  • (PMID = 18824008.001).
  • [ISSN] 0027-5107
  • [Journal-full-title] Mutation research
  • [ISO-abbreviation] Mutat. Res.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Tumor Suppressor Protein p53; 9007-49-2 / DNA; EC 2.7.1.- / Phosphatidylinositol 3-Kinases; EC 2.7.1.137 / PIK3CA protein, human
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32. Kwon JE, Park YN: [Hepatic adenomatosis in glycogen storage disease]. Korean J Hepatol; 2008 Mar;14(1):108-12
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  • [Title] [Hepatic adenomatosis in glycogen storage disease].

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  • (PMID = 18367864.001).
  • [ISSN] 1738-222X
  • [Journal-full-title] The Korean journal of hepatology
  • [ISO-abbreviation] Korean J Hepatol
  • [Language] kor
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Korea (South)
  • [Chemical-registry-number] 0 / Biomarkers, Tumor
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33. Zlobec I, Terracciano L, Jass JR, Lugli A: Value of staining intensity in the interpretation of immunohistochemistry for tumor markers in colorectal cancer. Virchows Arch; 2007 Oct;451(4):763-9
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  • A tissue microarray of 1,197 colorectal cancers was immunostained for p53, Her2/neu, epidermal growth factor receptor (EGFR), adenomatosis polyposis coli (APC), and beta-catenin.
  • [MeSH-minor] Adenomatous Polyposis Coli Protein / metabolism. Humans. Multivariate Analysis. Neoplasm Staging. Receptor, Epidermal Growth Factor / metabolism. Receptor, ErbB-2 / metabolism. Tumor Suppressor Protein p53 / metabolism. beta Catenin / metabolism


34. Svrcek M, Jeannot E, Arrivé L, Poupon R, Fromont G, Fléjou JF, Zucman-Rossi J, Bouchard P, Wendum D: Regressive liver adenomatosis following androgenic progestin therapy withdrawal: a case report with a 10-year follow-up and a molecular analysis. Eur J Endocrinol; 2007 Jun;156(6):617-21
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  • [Title] Regressive liver adenomatosis following androgenic progestin therapy withdrawal: a case report with a 10-year follow-up and a molecular analysis.
  • On the contrary, their contribution to liver adenomatosis (LA) development is still a debatable issue.

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  • (PMID = 17535860.001).
  • [ISSN] 0804-4643
  • [Journal-full-title] European journal of endocrinology
  • [ISO-abbreviation] Eur. J. Endocrinol.
  • [Language] ENG
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Contraceptives, Oral, Synthetic; 0 / Hepatocyte Nuclear Factor 1-alpha; 0 / Receptors, Androgen; 0 / Receptors, Estrogen; 0 / Receptors, Progesterone; N2Z8ALG4U5 / Lynestrenol
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35. Ek ET, Ek EW, Moulton CA, Mackay S: Incidental discovery of massive hepatic adenomatosis. ANZ J Surg; 2005 Jan-Feb;75(1-2):90-1
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  • [Title] Incidental discovery of massive hepatic adenomatosis.

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  • (PMID = 15740530.001).
  • [ISSN] 1445-1433
  • [Journal-full-title] ANZ journal of surgery
  • [ISO-abbreviation] ANZ J Surg
  • [Language] eng
  • [Publication-type] Case Reports; Letter
  • [Publication-country] Australia
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36. Kim JC, Cho YK, Roh SA, Yu CS, Gong G, Jang SJ, Kim SY, Kim YS: Individual tumorigenesis pathways of sporadic colorectal adenocarcinomas are associated with the biological behavior of tumors. Cancer Sci; 2008 Jul;99(7):1348-54
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  • Individual steps in the tumorigenesis pathway, that is, adenomatosis polyposis coli (APC), Wnt-activated, base excision repair mutations, mismatch repair defects, RAF-mediated, transforming growth factor (TGF)-beta-suppressed, bone morphogenic protein (BMP)-suppressed, and p53 alterations, were examined in terms of genetic and epigenetic changes, as well as protein expression.

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  • [ErratumIn] Cancer Sci. 2008 Jul;99(7):1499
  • (PMID = 18422752.001).
  • [ISSN] 1349-7006
  • [Journal-full-title] Cancer science
  • [ISO-abbreviation] Cancer Sci.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] EC 2.7.11.1 / raf Kinases
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37. Kim YT, Park JY, Jeon YK, Park SJ, Song JY, Kang CH, Sung SW, Kim JH: Aberrant promoter CpG island hypermethylation of the adenomatosis polyposis coli gene can serve as a good prognostic factor by affecting lymph node metastasis in squamous cell carcinoma of the esophagus. Dis Esophagus; 2009;22(2):143-50
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  • [Title] Aberrant promoter CpG island hypermethylation of the adenomatosis polyposis coli gene can serve as a good prognostic factor by affecting lymph node metastasis in squamous cell carcinoma of the esophagus.
  • CpG island hypermethylations were observed in 10% for p16, 34% for RARbetaP2, 46% for adenomatosis polyposis coli (APC), 14% for RASSF1A, 84% for FHIT, and 8% for hMLH1.

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  • (PMID = 18847451.001).
  • [ISSN] 1442-2050
  • [Journal-full-title] Diseases of the esophagus : official journal of the International Society for Diseases of the Esophagus
  • [ISO-abbreviation] Dis. Esophagus
  • [Language] ENG
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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38. Poomthavorn P, Wong SH, Higgins S, Werther GA, Russo VC: Activation of a prometastatic gene expression program in hypoxic neuroblastoma cells. Endocr Relat Cancer; 2009 Sep;16(3):991-1004
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  • In hypoxic SH-SY5Y NB cells, real-time PCR analysis showed that the genes involved in maintenance of cell-cell and cell-matrix interactions (i.e. adenomatosis polyposis coli, E-cadherin, catenin, EphB2, fibronectin-1, HTATIP2, tissue inhibitor of metalloprotease-4) were down-regulated by up to 90%, while genes involved in enhancement of metastatic behavior (integrin a7b1, hepatocyte growth factor receptor, transforming growth factor-beta1, VEGF, kisspeptin, interleukin-1beta) were dramatically up-regulated above 200%.

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  • (PMID = 19423615.001).
  • [ISSN] 1479-6821
  • [Journal-full-title] Endocrine-related cancer
  • [ISO-abbreviation] Endocr. Relat. Cancer
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Antimutagenic Agents; 0 / HIF1A protein, human; 0 / Hypoxia-Inducible Factor 1, alpha Subunit; 0 / ID1 protein, human; 0 / ID2 protein, human; 0 / Inhibitor of Differentiation Protein 1; 0 / Inhibitor of Differentiation Protein 2; 0 / VEGFA protein, human; 0 / Vascular Endothelial Growth Factor A; 3G0H8C9362 / Cobalt; EVS87XF13W / cobaltous chloride
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39. Flemming P, Lehmann U, Steinemann D, Kreipe H, Wilkens L: [Hepatocellular adenoma. Malignancy potential and differentiation from hepatocellular carcinoma]. Pathologe; 2006 Jul;27(4):238-43
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  • A limitation of particular HCA in genetic and metabolic diseases, children, adult males, adenomatosis, and HCA-like tumors with known risk factors of HCC would seem pragmatically meaningful.

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  • [Cites] Arch Pathol Lab Med. 1981 Jun;105(6):296-9 [6263214.001]
  • [Cites] World J Gastroenterol. 2003 Oct;9(10):2379-81 [14562419.001]
  • [Cites] Hepatology. 2002 Oct;36(4 Pt 1):927-35 [12297840.001]
  • [Cites] Cancer Res. 1976 Jul;36(7 PT 2):2584-8 [179705.001]
  • [Cites] Gastroenterology. 2005 May;128(5):1211-8 [15887105.001]
  • [Cites] Mod Pathol. 2002 Mar;15(3):189-96 [11904335.001]
  • [Cites] Clin Cancer Res. 2005 May 15;11(10):3654-60 [15897561.001]
  • [Cites] Zentralbl Chir. 1998;123(2):140-4 [9556886.001]
  • [Cites] Ann Surg. 1988 Nov;208(5):558-64 [3190282.001]
  • [Cites] Eur J Contracept Reprod Health Care. 1998 Dec;3(4):194-200 [10036602.001]
  • [Cites] Genes Chromosomes Cancer. 2002 Oct;35(2):138-43 [12203777.001]
  • [Cites] J Hepatol. 2004 Jun;40(6):1036-9 [15158349.001]
  • [Cites] Cancer. 1991 Jul 15;68(2):341-7 [1712664.001]
  • [Cites] Mod Pathol. 2002 Apr;15(4):470-5 [11950923.001]
  • [Cites] Gastroenterology. 1997 Mar;112(3):919-22 [9041254.001]
  • [Cites] Hepatology. 1999 Jun;29(6):1858-62 [10347130.001]
  • [Cites] Radiology. 2000 Aug;216(2):395-402 [10924560.001]
  • [Cites] Pathologica. 2003 Apr;95(2):71-82 [12768875.001]
  • [Cites] Pathologe. 2001 May;22(3):184-90 [11402848.001]
  • [Cites] J Pathol. 2001 Apr;193(4):476-82 [11276006.001]
  • [Cites] J Mol Diagn. 2001 May;3(2):68-73 [11333302.001]
  • [Cites] Am J Surg Pathol. 1993 May;17(5):525-9 [8385884.001]
  • [Cites] Histopathology. 2006 Jun;48(7):876-8 [16722942.001]
  • [Cites] Am J Surg Pathol. 1993 Nov;17(11):1113-23 [8214256.001]
  • [Cites] Eur J Pediatr. 1993;152 Suppl 1:S63-70 [8391447.001]
  • [Cites] Hum Pathol. 2002 Aug;33(8):852-5 [12203220.001]
  • [Cites] Gut. 2002 Aug;51(2):253-8 [12117890.001]
  • [Cites] Rev Hosp Clin Fac Med Sao Paulo. 1994 Jan-Feb;49(1):30-3 [8029613.001]
  • [Cites] Expert Rev Mol Diagn. 2002 Mar;2(2):120-8 [11962332.001]
  • [Cites] Hepatology. 1993 Apr;17 (4):583-5 [7682980.001]
  • [Cites] Cancer. 1983 Oct 15;52(8):1510-5 [6311396.001]
  • [Cites] Ann Intern Med. 1986 Oct;105(4):547-9 [3019201.001]
  • [Cites] Arch Surg. 1994 Jul;129(7):712-7 [7517661.001]
  • [Cites] Arch Pathol Lab Med. 2004 Feb;128(2):222-6 [14736278.001]
  • [Cites] Arch Surg. 2001 Sep;136(9):1033-8 [11529826.001]
  • [Cites] Proc Natl Acad Sci U S A. 2004 Feb 3;101(5):1309-14 [14745031.001]
  • [Cites] J Hepatol. 1996 Dec;25(6):976-9 [9007729.001]
  • [Cites] Histopathology. 1996 May;28(5):472-4 [8735726.001]
  • [Cites] Ann Intern Med. 1989 Mar 15;110(6):489-90 [2537593.001]
  • (PMID = 16736176.001).
  • [ISSN] 0172-8113
  • [Journal-full-title] Der Pathologe
  • [ISO-abbreviation] Pathologe
  • [Language] ger
  • [Publication-type] Comparative Study; English Abstract; Journal Article; Review
  • [Publication-country] Germany
  • [Number-of-references] 39
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40. Watkins J, Balabaud C, Bioulac-Sage P, Sharma D, Dhillon A: Hepatocellular adenoma in advanced-stage fatty liver disease. Eur J Gastroenterol Hepatol; 2009 Aug;21(8):932-6
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  • Hepatocellular adenomas are defined as benign hepatocellular neoplasms occurring in otherwise near normal liver and adenomatosis as 10 or more such lesions.
  • We present a case of a 45-year-old man with radiological adenomatosis, histologically confirmed with resection of one lesion, arising within advanced-stage nonalcoholic fatty liver disease (nearly cirrhotic).
  • Hepatocellular adenomas have recently been divided into four categories and the lesion in the present case had morphologic and immunohistochemical features of an inflammatory hepatocellular adenoma.

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  • (PMID = 19404203.001).
  • [ISSN] 1473-5687
  • [Journal-full-title] European journal of gastroenterology & hepatology
  • [ISO-abbreviation] Eur J Gastroenterol Hepatol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] England
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41. Bisceglia M, Gatta A, Tomezzoli A, Donataccio M: Multiple spontaneous liver cell adenomas of different types (liver adenomatosis). Adv Anat Pathol; 2009 May;16(3):172-9
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  • [Title] Multiple spontaneous liver cell adenomas of different types (liver adenomatosis).
  • Notwithstanding the nosologic advancement, and outside the "archetypal" types, the differential morphologic diagnosis of spontaneous HCA versus FNH may remain problematic as ductular proliferation in some cases of FNH may be scanty and hard to find.
  • In some histologic overlapping cases of FNH and HCA the morphologic diagnosis may be very difficult or even impossible (especially in small lesions) and molecular biology may be of critical assistance.
  • A prototypical case of surgically resected multiple spontaneous liver cell adenomas of various types, including HCA of T-FNH type and steatotic type, previously interpreted in different ways, affecting a young girl, is presented herein.
  • [MeSH-major] Adenoma, Liver Cell / pathology. Liver Neoplasms / pathology. Neoplasms, Multiple Primary / pathology

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  • (PMID = 19395881.001).
  • [ISSN] 1533-4031
  • [Journal-full-title] Advances in anatomic pathology
  • [ISO-abbreviation] Adv Anat Pathol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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42. Jo WS, Bandipalliam P, Shannon KM, Niendorf KB, Chan-Smutko G, Hur C, Syngal S, Chung DC: Correlation of polyp number and family history of colon cancer with germline MYH mutations. Clin Gastroenterol Hepatol; 2005 Oct;3(10):1022-8
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  • [Title] Correlation of polyp number and family history of colon cancer with germline MYH mutations.
  • BACKGROUND & AIMS: Affected individuals with biallelic MYH mutations are believed to display multiple adenomatous polyps without evidence of vertical transmission.
  • Our goal was to determine the detection rate of germline MYH mutations in a high-risk gastrointestinal cancer clinic population by using polyp number as a selection criterion.
  • RESULTS: Among 45 patients with more than 15 adenomatous polyps not diagnosed with familial adenomatous polyposis, 7 (15.6%) had biallelic MYH mutations.
  • Both had young-onset colorectal cancer (age, <50 y) with fewer than 15 polyps.
  • CONCLUSIONS: Most individuals with MYH mutations exhibit multiple adenomatous polyps.

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  • (PMID = 16234049.001).
  • [ISSN] 1542-3565
  • [Journal-full-title] Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association
  • [ISO-abbreviation] Clin. Gastroenterol. Hepatol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] EC 3.2.2.- / DNA Glycosylases; EC 3.2.2.- / mutY adenine glycosylase
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43. Virsaladze DK, Tkeshelashvili BD, Dzhavashvili LV, Kadzhaia NR, Khutsishvili MP: [Peculiarities of metabolic characteristics in females of reproductive age with hyperplastic processes of cervix and mammary glands]. Georgian Med News; 2006 Jul;(136):58-61
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  • 72 patients with hyperplastic processes of endometrium and endocervix (hyperplasia, polyposis), myoma of uterus, fibroadenoms and adenomatosis of mammary glands were selected in main group.

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  • (PMID = 16905847.001).
  • [ISSN] 1512-0112
  • [Journal-full-title] Georgian medical news
  • [ISO-abbreviation] Georgian Med News
  • [Language] rus
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] Georgia (Republic)
  • [Chemical-registry-number] 0 / Cholesterol, HDL; 0 / Triglycerides
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44. Peranteau WH, Bathaii SM, Pawel B, Hardy O, Alavi A, Stanley CA, Adzick NS: Multiple ectopic lesions of focal islet adenomatosis identified by positron emission tomography scan in an infant with congenital hyperinsulinism. J Pediatr Surg; 2007 Jan;42(1):188-92
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  • [Title] Multiple ectopic lesions of focal islet adenomatosis identified by positron emission tomography scan in an infant with congenital hyperinsulinism.


45. Stephen JK, Chen KM, Raitanen M, Grénman S, Worsham MJ: DNA hypermethylation profiles in squamous cell carcinoma of the vulva. Int J Gynecol Pathol; 2009 Jan;28(1):63-75
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  • Of the 22 tumor suppressor genes examined, aberrant methylation was observed for 9 genes: tumor protein p73 (TP73), fragile histidine triad (FHIT), von Hippel-Lindau (VHL), adenomatosis polyposis coli (APC), estrogen receptor 1 (ESR1), cyclin-dependent kinase inhibitor 2B (CDKN2B), death-associated protein kinase 1 (DAPK1), glutathione S-transferase pi (GSTP1), and immunoglobin superfamily, member 4 (IGSF4).
  • Frequent genetic alterations of loss and gain of gene copy number included gain of GSTP1 and multiple endocrine neoplasia type 1 (MEN1), and loss of malignant fibrous histiocytoma amplified sequence 1 (MFHAS1) and IGSF4 in over 50% of the squamous cell carcinoma of the vulva cell lines.


46. Moya Herráiz A, Torres-Quevedo R, Mir Pallardó J: [Liver transplantation in patients with benign hepatic lesions]. Cir Esp; 2008 Aug;84(2):60-6
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  • We present a review of the subject, commenting on the aspects that have been subsidiary to liver transplantation, of which are highlighted: adenomatosis, polycystosis and hepatic epithelioid haemangioendothelioma (although this process may be a low to intermediate malignant grade).


47. Komaba H, Takeda Y, Shin J, Tanaka R, Kakuta T, Tominaga Y, Fukagawa M: Reversed whole PTH/intact PTH ratio as an indicator of marked parathyroid enlargement: five case studies and a literature review. NDT Plus; 2008 Aug;1(Suppl 3):iii54-iii58
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  • In the case of a single enlarged gland, the pathological pattern and heterogeneous expression of parathyroid adenomatosis 1/cyclin D1 suggested it to be a single nodule of uraemic hyperparathyroidism rather than sporadic primary adenoma.

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  • (PMID = 25983975.001).
  • [ISSN] 1753-0784
  • [Journal-full-title] NDT plus
  • [ISO-abbreviation] NDT Plus
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC4421125
  • [Keywords] NOTNLM ; N-PTH / intact PTH / secondary hyperparathyroidism / single nodule / whole PTH
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48. Wellen JR, Anderson CD, Doyle M, Shenoy S, Nadler M, Turmelle Y, Shepherd R, Chapman WC, Lowell JA: The role of liver transplantation for hepatic adenomatosis in the pediatric population: case report and review of the literature. Pediatr Transplant; 2010 May;14(3):E16-9
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  • [Title] The role of liver transplantation for hepatic adenomatosis in the pediatric population: case report and review of the literature.
  • These tumors are often solitary but can also be multiple in which case this is referred to as hepatic adenomatosis (HA).


49. Sugita A, Koganei K, Kimura H, Yamada K, Futatuki R, Kitoh F, Fukushima T: [Reconstruction of proctocolectomy: which is the best surgical procedure?]. Nihon Geka Gakkai Zasshi; 2008 Sep;109(5):269-73
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  • Total proctocolectomy is commonly performed in patients with ulcerative colitis or familial adenomatosis coli.
  • Postoperative cancer surveillance colonoscopy is recommended in patients with stapled IPAA, and patients with IPAA have also at risk for cancer in the anastomotic site, although at very low incidence.

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  • (PMID = 18939461.001).
  • [ISSN] 0301-4894
  • [Journal-full-title] Nihon Geka Gakkai zasshi
  • [ISO-abbreviation] Nihon Geka Gakkai Zasshi
  • [Language] jpn
  • [Publication-type] Comparative Study; English Abstract; Journal Article
  • [Publication-country] Japan
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50. Lorenzo Garrido H, Rotaetxe Vacas O, García Barcina MJ, Muñoz García G, Martín Bejarano E: [Hepatic adenomatosis]. An Pediatr (Barc); 2008 May;68(5):526-7
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  • [Title] [Hepatic adenomatosis].
  • [Transliterated title] Adenomatosis hepática.

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  • (PMID = 18448003.001).
  • [ISSN] 1695-4033
  • [Journal-full-title] Anales de pediatría (Barcelona, Spain : 2003)
  • [ISO-abbreviation] An Pediatr (Barc)
  • [Language] spa
  • [Publication-type] Case Reports; Letter
  • [Publication-country] Spain
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51. Rosoff PM, Larrier N, Rice HE: Intra-abdominal desmoid tumor after successful treatment for Hodgkin disease. Pediatr Blood Cancer; 2005 Oct 15;45(5):728-31
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  • The tumor did not occur at either a surgical site or within a radiation field.
  • She did not carry a mutation for familial adenomatosis polypoli.

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  • (PMID = 16035093.001).
  • [ISSN] 1545-5009
  • [Journal-full-title] Pediatric blood & cancer
  • [ISO-abbreviation] Pediatr Blood Cancer
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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52. Kámory E, Olasz J, Csuka O: Somatic APC inactivation mechanisms in sporadic colorectal cancer cases in Hungary. Pathol Oncol Res; 2008 Mar;14(1):51-6
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  • The role of germline inactivation of the adenomatosis polyposis coli (APC) gene in hereditary colorectal cancer is well known, being the most important cause of familial adenomatosus polyposis (FAP) syndrome.

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  • [Cites] Proc Natl Acad Sci U S A. 1992 May 15;89(10):4452-6 [1316610.001]
  • [Cites] J Biol Chem. 2006 Jun 30;281(26):17751-7 [16798748.001]
  • [Cites] Oncogene. 2006 Dec 4;25(57):7531-7 [17143297.001]
  • [Cites] Scand J Gastroenterol. 2002 Sep;37(9):1048-53 [12374230.001]
  • [Cites] Cancer Genet Cytogenet. 2007 Sep;177(2):95-7 [17854661.001]
  • [Cites] Nat Genet. 1997 Sep;17(1):79-83 [9288102.001]
  • [Cites] Cell. 1991 Aug 9;66(3):589-600 [1651174.001]
  • [Cites] Am J Hum Genet. 1993 Feb;52(2):280-5 [8381581.001]
  • [Cites] Hum Mol Genet. 2001 Apr;10(7):721-33 [11257105.001]
  • [Cites] Histopathology. 2007 Feb;50(3):318-30 [17257127.001]
  • [Cites] Genes Chromosomes Cancer. 2002 Sep;35(1):1-10 [12203784.001]
  • [Cites] BMC Cancer. 2005 Apr 15;5:40 [15833136.001]
  • [Cites] Proteins. 2004 Dec 1;57(4):811-9 [15390262.001]
  • [Cites] Proc Natl Acad Sci U S A. 1992 Mar 1;89(5):1827-31 [1542678.001]
  • [Cites] Cancer Res. 2000 Aug 15;60(16):4366-71 [10969779.001]
  • [Cites] Mol Cancer Res. 2007 Feb;5(2):165-70 [17293392.001]
  • [Cites] Cell. 1990 Jun 1;61(5):759-67 [2188735.001]
  • [Cites] Cell Oncol. 2006;28(5-6):247-57 [17167178.001]
  • [Cites] Cancer Res. 2001 Apr 15;61(8):3410-8 [11309301.001]
  • [Cites] Nat Genet. 1994 Jun;7(2 Spec No):246-339 [7545953.001]
  • [Cites] Neoplasia. 2005 Feb;7(2):99-108 [15802015.001]
  • [Cites] Nat Med. 1999 Sep;5(9):1071-5 [10470088.001]
  • [Cites] Hum Mutat. 1993;2(6):425-34 [8111410.001]
  • [Cites] J Pathol. 2001 Sep;195(1):111-34 [11568897.001]
  • [Cites] Hum Genet. 1993 Feb;90(6):650-2 [8383094.001]
  • [Cites] Cancer Biol Ther. 2004 Oct;3(10):960-4 [15326380.001]
  • [Cites] Cancer Res. 1994 Jun 1;54(11):2991-5 [8187087.001]
  • [Cites] Clin Genet. 2006 May;69(5):404-9 [16650078.001]
  • [Cites] Genes Chromosomes Cancer. 2002 Aug;34(4):349-53 [12112523.001]
  • [Cites] Gastroenterology. 2007 Jan;132(1):127-38 [17087942.001]
  • [Cites] Hum Mol Genet. 1993 Jul;2(7):925-31 [8395941.001]
  • [Cites] Hum Mol Genet. 2001 Mar 15;10(6):591-7 [11230178.001]
  • [Cites] J Med Genet. 1999 Jan;36(1):14-20 [9950360.001]
  • [Cites] Hum Mutat. 2003 Apr;21(4):446-7 [12655564.001]
  • [Cites] Nucleic Acids Res. 2003 Jul 1;31(13):3812-4 [12824425.001]
  • [Cites] Med Mol Morphol. 2007 Jun;40(2):68-81 [17572842.001]
  • [Cites] Cancer Epidemiol Biomarkers Prev. 2001 Jul;10(7):799-803 [11440966.001]
  • [Cites] Hum Mutat. 2002 Jun;19(6):664 [12007223.001]
  • (PMID = 18369740.001).
  • [ISSN] 1219-4956
  • [Journal-full-title] Pathology oncology research : POR
  • [ISO-abbreviation] Pathol. Oncol. Res.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Netherlands
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53. Campos FG, Habr-Gama A, Kiss DR, Atuí FC, Rawet V, Goldstein PJ, Gama-Rodrigues J: Cowden syndrome: report of two cases and review of clinical presentation and management of a rare colorectal polyposis. Curr Surg; 2006 Jan-Feb;63(1):15-9
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  • [Title] Cowden syndrome: report of two cases and review of clinical presentation and management of a rare colorectal polyposis.
  • REVIEW: Although it exhibits variable clinical expressivity, the diagnosis is based on characteristic mucocutaneous alterations such as multiple facial trichilemmomas, oral mucosal papillomatosis, and acral and palmoplantar keratoses.
  • Extracutaneous lesions include fibrocystic disease of the breast, thyroid goiters or adenomas, multiple polyposis of the gastrointestinal tract, and ovarian cysts.
  • Gastrointestinal polyps are usually asymptomatic, and the risk of gastrointestinal cancer is not greatly increased.
  • [MeSH-major] Hamartoma Syndrome, Multiple / diagnosis. Hamartoma Syndrome, Multiple / therapy

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  • (PMID = 16373153.001).
  • [ISSN] 0149-7944
  • [Journal-full-title] Current surgery
  • [ISO-abbreviation] Curr Surg
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] EC 3.1.3.48 / PTEN protein, human; EC 3.1.3.67 / PTEN Phosphohydrolase
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54. Widimský J Jr, Zelinka T, Petrák O, Strauch B, Safarík L, Kasalický M, Vranková A, Holaj R: [Diagnostic and therapeutic procedures in pheochromocytoma: current trends]. Vnitr Lek; 2007 Apr;53(4):428-33
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  • 5 gene mutations have so far been identified that may be responsible for the familial form of pheochromacytoma: mutation of the von Hippel-Lindau (VHL) gene, leading to the onset of VHL syndrome, mutation of the RET-proto-oncogene in multiple endocrine adenomatosis type 2, mutation of the type 1 gene for neurofibromatosis, which is associated with von Recklinghausen's disease and finally mutation of the genes encoding the B and D subunits of succinated hydrogenase (SDHB, SDHD), which are associated with familial paragangliomas and pheochromacytoma.
  • The diagnosis of extraadrenal or multiple forms can use not only CT/MR but also imaging using the radiopharmaceutical 123I-Metaiodobenzylguanidine (MIBG) or 18F-fluorodopamine PET (only available in the USA).
  • [MeSH-minor] Humans. Hypertension / etiology. Multiple Endocrine Neoplasia Type 2a / diagnosis. Neurofibromatosis 1 / diagnosis. Paraganglioma / diagnosis. von Hippel-Lindau Disease / diagnosis. von Hippel-Lindau Disease / etiology

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  • (PMID = 17578179.001).
  • [ISSN] 0042-773X
  • [Journal-full-title] Vnitr̆ní lékar̆ství
  • [ISO-abbreviation] Vnitr Lek
  • [Language] cze
  • [Publication-type] English Abstract; Journal Article; Review
  • [Publication-country] Czech Republic
  • [Number-of-references] 25
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55. Kemp ZE, Carvajal-Carmona LG, Barclay E, Gorman M, Martin L, Wood W, Rowan A, Donohue C, Spain S, Jaeger E, Evans DG, Maher ER, Bishop T, Thomas H, Houlston R, Tomlinson I, Colorectal Tumour Gene Identification Study Consortium: Evidence of linkage to chromosome 9q22.33 in colorectal cancer kindreds from the United Kingdom. Cancer Res; 2006 May 15;66(10):5003-6
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  • Exclusion from the analysis of kindreds with a phenotype of multiple polyposis also found evidence of linkage in the same region (NPL = 2.47 at close to D9S277, P = 0.009).

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  • (PMID = 16707420.001).
  • [ISSN] 0008-5472
  • [Journal-full-title] Cancer research
  • [ISO-abbreviation] Cancer Res.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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56. Arvind N, Duraimurugan D, Rajkumar JS: Hepatic adenomatosis--a rare double complication of multiple adenoma rupture and malignant transformation. Indian J Gastroenterol; 2006 Jul-Aug;25(4):209-10
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  • [Title] Hepatic adenomatosis--a rare double complication of multiple adenoma rupture and malignant transformation.
  • Hepatic adenomatosis (HA) is a rare disorder that is susceptible to hemorrhagic complications and, rarely, to malignant transformation.

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  • (PMID = 16974041.001).
  • [ISSN] 0254-8860
  • [Journal-full-title] Indian journal of gastroenterology : official journal of the Indian Society of Gastroenterology
  • [ISO-abbreviation] Indian J Gastroenterol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] India
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57. Katsinelos P, Chatzimavroudis G, Kotakidou R, Zavos C, Paroutoglou G, Kountouras J: Diffuse gastric carcinoid polyposis in multiple endocrine neoplasia 1 syndrome without hypergastrinemia. Gastrointest Endosc; 2007 Aug;66(2):411-2
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  • [Title] Diffuse gastric carcinoid polyposis in multiple endocrine neoplasia 1 syndrome without hypergastrinemia.
  • [MeSH-major] Carcinoid Tumor / pathology. Gastrins / blood. Multiple Endocrine Neoplasia Type 1 / pathology. Stomach Neoplasms / pathology


58. Amemori H, Hyakutake M, Maeda Y, Tomita Y, Mitsumizo S, Oishi H, Koizumi S, Nakashima M, Araki K: Survival case of Vibrio vulnificus infection with multiple endocrine neoplasia type I. Fukuoka Igaku Zasshi; 2007 Sep;98(9):346-52
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  • [Title] Survival case of Vibrio vulnificus infection with multiple endocrine neoplasia type I.
  • A 35-year-old female with malabsorption syndrome who underwent a pancreatoduodenectomy for multiple endocrine adenomatosis 13 years prior was admitted to our hospital with diarrhea, general fatigue, high fever, and eruption in the lower legs.
  • [MeSH-major] Immunocompromised Host. Multiple Endocrine Neoplasia Type 1 / complications. Vibrio Infections / etiology. Vibrio Infections / therapy. Vibrio vulnificus

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  • (PMID = 17974078.001).
  • [ISSN] 0016-254X
  • [Journal-full-title] Fukuoka igaku zasshi = Hukuoka acta medica
  • [ISO-abbreviation] Fukuoka Igaku Zasshi
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / Anti-Bacterial Agents; 0 / Catecholamines; 9M416Z9QNR / Ceftazidime; FYY3R43WGO / Minocycline; IY9XDZ35W2 / Glucose
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59. Lipton L, Tomlinson I: The genetics of FAP and FAP-like syndromes. Fam Cancer; 2006;5(3):221-6
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  • The presence of multiple adenomatous polyps in the large bowel confers a high lifetime risk of colorectal cancer.
  • Although many cases of classical familial adenomatous polyposis (> 100 polyps) can be accounted for by mutations in the adenomatous polyposis coli (APC) gene, a large group of patients remains with multiple (5-100) adenomas and in whom there is no detectable APC mutation.
  • Recently two new genetic variants have been found to be associated with multiple colorectal adenomas and cancer, MYH/MUTYH on chromosome 1p and the HMPS/CRAC1 locus on chromosome 15q13-q14.
  • New information also continues to emerge regarding the less common hamartomatous polyposis conditions, Peutz-Jeghers syndrome and Juvenile Polyposis syndrome.
  • In this review we draw together some of the most recent information pertinent to the molecular pathogenesis of colorectal polyposis.
  • [MeSH-major] Adenomatous Polyposis Coli / genetics. Genes, APC / physiology. Neoplastic Syndromes, Hereditary / genetics

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  • [Cites] Proc Natl Acad Sci U S A. 2002 Mar 5;99(5):2954-8 [11867715.001]
  • [Cites] Am J Hum Genet. 2005 Jul;77(1):112-9 [15931596.001]
  • [Cites] Cancer Res. 2003 Nov 15;63(22):7595-9 [14633673.001]
  • [Cites] Nat Genet. 2002 Feb;30(2):227-32 [11818965.001]
  • [Cites] Lancet. 2003 Jul 5;362(9377):39-41 [12853198.001]
  • [Cites] Gastroenterology. 1999 Apr;116(4):789-95 [10092300.001]
  • [Cites] Genes Chromosomes Cancer. 1999 Jun;25(2):75-81 [10337989.001]
  • [Cites] J Natl Cancer Inst. 2004 Nov 3;96(21):1631-4 [15523092.001]
  • [Cites] Am J Hum Genet. 1998 Jun;62(6):1290-301 [9585611.001]
  • [Cites] Am J Hum Genet. 1996 Apr;58(4):770-6 [8644741.001]
  • [Cites] Am J Hum Genet. 2003 May;72(5):1261-7 [12696020.001]
  • [Cites] Int J Cancer. 2004 Apr 20;109 (4):554-8 [14991577.001]
  • [Cites] Cell. 1991 Aug 9;66(3):589-600 [1651174.001]
  • [Cites] Cell Biochem Biophys. 2001;35(2):141-70 [11892789.001]
  • [Cites] Science. 1987 Dec 4;238(4832):1411-3 [3479843.001]
  • [Cites] Am J Hum Genet. 1993 Feb;52(2):263-72 [8381579.001]
  • [Cites] Int J Cancer. 1995 Nov 3;63(3):435-41 [7591245.001]
  • [Cites] Hum Genet. 1996 Dec;98(6):727-34 [8931709.001]
  • [Cites] Am J Pathol. 2003 Sep;163(3):827-32 [12937124.001]
  • [Cites] Cell. 1993 Dec 3;75(5):951-7 [8252630.001]
  • [Cites] Science. 1998 May 15;280(5366):1086-8 [9582123.001]
  • [Cites] Am J Pathol. 2003 May;162(5):1545-8 [12707038.001]
  • [Cites] Nature. 1991 Jan 31;349(6308):431-4 [1992344.001]
  • [Cites] J Med Genet. 1999 May;36(5):365-8 [10353780.001]
  • [Cites] Hum Mol Genet. 2002 Nov 1;11(23):2961-7 [12393807.001]
  • [Cites] Curr Biol. 2002 Feb 19;12(4):335-9 [11864576.001]
  • [Cites] Am J Hum Genet. 2001 Oct;69(4):704-11 [11536076.001]
  • [Cites] J Bacteriol. 1999 Oct;181(19):6210-3 [10498741.001]
  • [Cites] N Engl J Med. 1995 Aug 24;333(8):524 [7662026.001]
  • [Cites] J Bacteriol. 1996 Jul;178(13):3885-92 [8682794.001]
  • [Cites] Gastroenterology. 1997 Feb;112(2):327-34 [9024286.001]
  • [Cites] Nature. 1998 Jan 8;391(6663):184-7 [9428765.001]
  • [Cites] Genes Chromosomes Cancer. 2004 May;40(1):1-9 [15034862.001]
  • [Cites] N Engl J Med. 2003 Feb 27;348(9):791-9 [12606733.001]
  • [Cites] Int J Cancer. 2004 May 1;109(5):680-4 [14999774.001]
  • [Cites] Hum Mol Genet. 2000 Sep 22;9(15):2215-21 [11001924.001]
  • (PMID = 16998667.001).
  • [ISSN] 1389-9600
  • [Journal-full-title] Familial cancer
  • [ISO-abbreviation] Fam. Cancer
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Netherlands
  • [Number-of-references] 36
  •  go-up   go-down


60. Hagiwara S, Takagi H, Kanda D, Sohara N, Kakizaki S, Katakai K, Yoshinaga T, Higuchi T, Nomoto K, Kuwano H, Mori M: Hepatic adenomatosis associated with hormone replacement therapy and hemosiderosis: a case report. World J Gastroenterol; 2006 Jan 28;12(4):652-5
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  • [Title] Hepatic adenomatosis associated with hormone replacement therapy and hemosiderosis: a case report.
  • We have reported a case of hepatic adenomatosis associated with hormone replacement therapy (estrogen and progesterone) and hemosiderosis caused by excessive blood transfusion for the treatment of chronic myeloid leukemia.
  • Histopathological examination of the surgical specimen showed hepatic adenomatosis with hemosiderosis.
  • Both hormone replacement therapy and iron overload could be the cause of hepatic adenomatosis.

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  • [Cites] Postgrad Med J. 2000 Feb;76(892):100-2 [10644389.001]
  • [Cites] Radiology. 2000 Aug;216(2):395-402 [10924560.001]
  • [Cites] Curr Med Chem. 2001 Jun;8(7):839-49 [11375754.001]
  • [Cites] Abdom Imaging. 2001 May-Jun;26(3):264-8 [11429950.001]
  • [Cites] Lancet. 1973 Oct 27;2(7835):926-9 [4126557.001]
  • [Cites] Gastroenterology. 1980 Feb;78(2):374-81 [6985597.001]
  • [Cites] Arch Biochem Biophys. 1997 Oct 15;346(2):180-6 [9343364.001]
  • [Cites] Dig Dis Sci. 1981 Nov;26(11):1045-50 [6271517.001]
  • [Cites] Gastroenterology. 1982 Apr;82(4):775-82 [6277724.001]
  • [Cites] Gastroenterology. 1985 Nov;89(5):1132-8 [2412930.001]
  • [Cites] Am J Gastroenterol. 1989 Apr;84(4):429-32 [2929567.001]
  • [Cites] J Med Assoc Thai. 1994 Feb;77(2):108-12 [7798835.001]
  • [Cites] Gastroenterology. 1981 Aug;81(2):352-5 [6263745.001]
  • (PMID = 16489686.001).
  • [ISSN] 1007-9327
  • [Journal-full-title] World journal of gastroenterology
  • [ISO-abbreviation] World J. Gastroenterol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] China
  • [Other-IDs] NLM/ PMC4066105
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61. Pechenikova VA: [Cancer transformation of adenomyosis]. Arkh Patol; 2005 Jul-Aug;67(4):51-3
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  • Various forms of transformation (stages of AMCT morphogensis) in and outside of denomyosis (AM) foci are described (adenomatosis, invasive cancer, metastases).

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  • (PMID = 16209300.001).
  • [ISSN] 0004-1955
  • [Journal-full-title] Arkhiv patologii
  • [ISO-abbreviation] Arkh. Patol.
  • [Language] rus
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Russia (Federation)
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62. Beaton MD, Taylor B, Driman D, Ainsworth P, Adams PC: Colonic interposition in a woman with attenuated familial adenomatosis polyposis: does the location of the colon affect polyp formation? Can J Gastroenterol; 2008 Jul;22(7):634-6
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  • [Title] Colonic interposition in a woman with attenuated familial adenomatosis polyposis: does the location of the colon affect polyp formation?
  • Attenuated familial adenomatous polyposis (AFAP) is a rare but well-established cause of colorectal carcinoma and multiple polyps.
  • While she had developed adenomatous polyps in her native cecum, there was no evidence of polyps or cancer in the segment of large intestine interposed between her upper esophagus and stomach.
  • [MeSH-major] Adenomatous Polyposis Coli / pathology. Colonic Polyps / pathology

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  • [Cites] Gut. 1999 Dec;45(6):829-33 [10562580.001]
  • [Cites] Transl Res. 2007 Sep;150(3):147-9 [17761365.001]
  • [Cites] Surg Today. 2001;31(11):1020-3 [11766074.001]
  • [Cites] Dis Colon Rectum. 2002 Jan;45(1):127-34; discussion 134-6 [11786778.001]
  • [Cites] Gut. 2002 May;50(5):636-41 [11950808.001]
  • [Cites] Am J Pathol. 2002 Jun;160(6):2253-7 [12057927.001]
  • [Cites] Am J Gastroenterol. 2002 Jul;97(7):1822-7 [12135043.001]
  • [Cites] J Postgrad Med. 1986 Oct;32(4):206-9 [3585794.001]
  • [Cites] Cancer. 1988 Apr 15;61(8):1563-70 [3349420.001]
  • [Cites] IARC Monogr Eval Carcinog Risks Hum. 1994;61:1-241 [7715068.001]
  • [Cites] Cancer Res. 1997 Mar 1;57(5):812-4 [9041176.001]
  • [Cites] Hum Genet. 1997 Oct;100(5-6):601-5 [9341879.001]
  • [Cites] Am J Hum Genet. 1998 Jun;62(6):1290-301 [9585611.001]
  • [Cites] Nutr Cancer. 1998;31(2):111-8 [9770722.001]
  • [Cites] N Engl J Med. 1999 Mar 18;340(11):825-31 [10080844.001]
  • [Cites] Cancer. 1999 Apr 15;85(8):1664-9 [10223558.001]
  • [Cites] World J Gastroenterol. 2006 Nov 14;12(42):6741-6 [17106919.001]
  • [Cites] Tech Coloproctol. 2006 Dec;10(4):287-96 [17115321.001]
  • [Cites] Crit Rev Oncol Hematol. 2007 Feb;61(2):153-61 [17064931.001]
  • [Cites] Cancer Res. 2001 Mar 15;61(6):2395-8 [11289103.001]
  • (PMID = 18629394.001).
  • [ISSN] 0835-7900
  • [Journal-full-title] Canadian journal of gastroenterology = Journal canadien de gastroenterologie
  • [ISO-abbreviation] Can. J. Gastroenterol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Canada
  • [Other-IDs] NLM/ PMC2661270
  •  go-up   go-down


63. Hsiao HP, Kirschner LS, Bourdeau I, Keil MF, Boikos SA, Verma S, Robinson-White AJ, Nesterova M, Lacroix A, Stratakis CA: Clinical and genetic heterogeneity, overlap with other tumor syndromes, and atypical glucocorticoid hormone secretion in adrenocorticotropin-independent macronodular adrenal hyperplasia compared with other adrenocortical tumors. J Clin Endocrinol Metab; 2009 Aug;94(8):2930-7
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  • For three patients with AIMAH, there was family history of CS; germline mutations were identified in three other patients in the genes for menin (one), fumarate hydratase (one), and adenomatosis polyposis coli (APC) (one); a PDE11A gene variant was found in another.

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  • [Cites] J Clin Endocrinol Metab. 2004 Jul;89(7):3173-82 [15240590.001]
  • [Cites] Clin Endocrinol (Oxf). 2004 Feb;60(2):192-200 [14725680.001]
  • [Cites] J Clin Endocrinol Metab. 1968 Mar;28(3):343-8 [4296070.001]
  • [Cites] J Pediatr. 1991 Feb;118(2):256-8 [1993957.001]
  • [Cites] N Engl J Med. 1992 Oct 1;327(14):974-80 [1325608.001]
  • [Cites] N Engl J Med. 1992 Oct 1;327(14):981-6 [1325609.001]
  • [Cites] Eur J Endocrinol. 1994 Jul;131(1):67-73 [8038906.001]
  • [Cites] J Clin Endocrinol Metab. 1995 Aug;80(8):2336-41 [7629226.001]
  • [Cites] N Engl J Med. 1997 Nov 13;337(20):1429-34 [9358140.001]
  • [Cites] Endocrinol Metab Clin North Am. 1997 Dec;26(4):973-91 [9429868.001]
  • [Cites] J Clin Endocrinol Metab. 1998 Apr;83(4):1163-7 [9543134.001]
  • [Cites] J Clin Endocrinol Metab. 1998 Jun;83(6):2029-35 [9626135.001]
  • [Cites] N Engl J Med. 1998 Jul 2;339(1):27-31 [9647876.001]
  • [Cites] Horm Metab Res. 1998 Jun-Jul;30(6-7):456-63 [9694579.001]
  • [Cites] Ann Intern Med. 1999 Oct 19;131(8):585-91 [10523219.001]
  • [Cites] J Clin Endocrinol Metab. 1999 Oct;84(10):3817-22 [10523036.001]
  • [Cites] J Clin Endocrinol Metab. 1960 Dec;20:1539-60 [13761950.001]
  • [Cites] Endocr Res. 2004 Nov;30(4):575-83 [15666794.001]
  • [Cites] J Clin Endocrinol Metab. 2005 Mar;90(3):1302-10 [15585558.001]
  • [Cites] J Clin Endocrinol Metab. 2005 Jun;90(6):3773-9 [15741255.001]
  • [Cites] Horm Res. 2005;64(3):119-31 [16215323.001]
  • [Cites] Clin Endocrinol (Oxf). 2005 Dec;63(6):625-30 [16343095.001]
  • [Cites] Clin Endocrinol (Oxf). 2006 Jan;64(1):37-45 [16402926.001]
  • [Cites] J Clin Endocrinol Metab. 2006 Apr;91(4):1566-72 [16449345.001]
  • [Cites] Nat Genet. 2006 Jul;38(7):794-800 [16767104.001]
  • [Cites] J Clin Endocrinol Metab. 2006 Sep;91(9):3626-32 [16772351.001]
  • [Cites] Cancer Res. 2006 Dec 15;66(24):11571-5 [17178847.001]
  • [Cites] Eur J Endocrinol. 2007 Jan;156(1):21-31 [17218722.001]
  • [Cites] Pediatrics. 2007 Sep;120(3):e575-86 [17698579.001]
  • [Cites] Hum Mutat. 2008 Jan;29(1):22-32 [17879353.001]
  • [Cites] Surgery. 2008 Jun;143(6):750-8 [18549891.001]
  • [Cites] Mol Cell Endocrinol. 2009 Mar 5;300(1-2):152-7 [19063937.001]
  • [Cites] N Engl J Med. 1999 Nov 18;341(21):1577-81 [10564687.001]
  • [Cites] Radiology. 2000 Sep;216(3):797-802 [10966714.001]
  • [Cites] J Clin Endocrinol Metab. 2000 Oct;85(10):3531-6 [11061496.001]
  • [Cites] J Clin Endocrinol Metab. 2001 Sep;86(9):4041-6 [11549623.001]
  • [Cites] J Clin Endocrinol Metab. 2001 Nov;86(11):5534-40 [11701732.001]
  • [Cites] Ann N Y Acad Sci. 2002 Jun;968:240-55 [12119280.001]
  • [Cites] Am J Hum Genet. 2002 Dec;71(6):1433-42 [12424709.001]
  • [Cites] Endocrine. 2002 Dec;19(3):319-26 [12624433.001]
  • [Cites] J Clin Endocrinol Metab. 2003 May;88(5):2147-51 [12727968.001]
  • [Cites] J Clin Endocrinol Metab. 2003 Aug;88(8):3931-7 [12915689.001]
  • [Cites] Cancer Res. 2003 Sep 1;63(17):5308-19 [14500362.001]
  • [Cites] Trends Endocrinol Metab. 2004 Oct;15(8):375-82 [15380809.001]
  • (PMID = 19509103.001).
  • [ISSN] 1945-7197
  • [Journal-full-title] The Journal of clinical endocrinology and metabolism
  • [ISO-abbreviation] J. Clin. Endocrinol. Metab.
  • [Language] ENG
  • [Grant] United States / NICHD NIH HHS / HD / Z01 HD000642; United States / NICHD NIH HHS / HD / Z01-HD-000642-04
  • [Publication-type] Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Glucocorticoids; 7S5I7G3JQL / Dexamethasone; 9002-60-2 / Adrenocorticotropic Hormone; 9015-71-8 / Corticotropin-Releasing Hormone; EC 3.6.1.- / GNAS protein, human; EC 3.6.5.1 / GTP-Binding Protein alpha Subunits, Gs
  • [Other-IDs] NLM/ PMC2730864
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64. de Ferro SM, Suspiro A, Fidalgo P, Lage P, Rodrigues P, Fragoso S, Vitoriano I, Baltazar C, Albuquerque C, Bettencourt A, Leitão CN: Aggressive phenotype of MYH-associated polyposis with jejunal cancer and intra-abdominal desmoid tumor: report of a case. Dis Colon Rectum; 2009 Apr;52(4):742-5
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  • [Title] Aggressive phenotype of MYH-associated polyposis with jejunal cancer and intra-abdominal desmoid tumor: report of a case.
  • MYH-associated polyposis is an inherited autosomal recessive disease, linked to biallelic germline MYH mutations, which predisposes to the development of multiple colorectal adenomas and cancer.
  • We report the case of a young male patient with an aggressive MYH-associated polyposis phenotype.
  • He presented at aged 30 years with more than 100 colonic polyps and 4 colonic adenocarcinomas.
  • At aged 35 years, Spigelman Stage IV duodenal adenomatosis was detected.
  • Based on this report, we believe that screening of the entire small bowel should be recommended in MYH-associated polyposis patients, especially in those with duodenal adenomas.
  • Similar to patients with familial adenomatous polyposis, desmoid tumors also may be part of the clinical spectrum of MYH-associated polyposis and may prove to be a significant clinical problem in patients submitted to prophylactic colectomy.
  • [MeSH-major] Adenomatous Polyposis Coli / genetics. Colorectal Neoplasms / genetics. Fibromatosis, Aggressive / genetics. Jejunal Neoplasms / genetics. Neoplasms, Multiple Primary / genetics. Peritoneal Neoplasms / genetics


65. Lerario AM, Brito LP, Mariani BM, Fragoso MC, Machado MA, Teixeira R: A missense TCF1 mutation in a patient with mody-3 and liver adenomatosis. Clinics (Sao Paulo); 2010;65(10):1059-60
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  • [Title] A missense TCF1 mutation in a patient with mody-3 and liver adenomatosis.

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  • [Cites] J Clin Endocrinol Metab. 2004 Mar;89(3):1476-80 [15001650.001]
  • [Cites] N Engl J Med. 1978 Aug 3;299(5):239-41 [207987.001]
  • [Cites] Gastroenterology. 1985 Nov;89(5):1132-8 [2412930.001]
  • [Cites] Hum Mutat. 2006 Sep;27(9):854-69 [16917892.001]
  • [Cites] Gastroenterology. 2003 Nov;125(5):1470-5 [14598263.001]
  • [Cites] Radiology. 2000 Aug;216(2):395-402 [10924560.001]
  • [Cites] N Engl J Med. 2001 Sep 27;345(13):971-80 [11575290.001]
  • [Cites] Nat Genet. 2002 Oct;32(2):312-5 [12355088.001]
  • [Cites] Diabetologia. 2003 May;46(5):721-7 [12712243.001]
  • [Cites] Ann Surg. 2000 Jan;231(1):74-81 [10636105.001]
  • (PMID = 21120312.001).
  • [ISSN] 1980-5322
  • [Journal-full-title] Clinics (São Paulo, Brazil)
  • [ISO-abbreviation] Clinics (Sao Paulo)
  • [Language] eng
  • [Publication-type] Case Reports; Letter
  • [Publication-country] Brazil
  • [Chemical-registry-number] 0 / HNF1A protein, human; 0 / Hepatocyte Nuclear Factor 1-alpha
  • [Other-IDs] NLM/ PMC2972616
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66. Beach R, Chan AO, Wu TT, White JA, Morris JS, Lunagomez S, Broaddus RR, Issa JP, Hamilton SR, Rashid A: BRAF mutations in aberrant crypt foci and hyperplastic polyposis. Am J Pathol; 2005 Apr;166(4):1069-75
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  • [Title] BRAF mutations in aberrant crypt foci and hyperplastic polyposis.
  • Patients with hyperplastic polyposis have multiple hyperplastic polyps (HPs) and increased risk of colorectal carcinomas.
  • We evaluated BRAF mutations by DNA sequencing in 53 ACF from patients with sporadic colorectal carcinomas and familial adenomatous polyposis, in 18 sporadic HPs from patients with resected colorectal cancer, and in 70 HPs, 4 serrated adenomas, 3 admixed hyperplastic-adenomatous polyps, 10 tubular adenomas, and 6 carcinomas from 17 patients with multiple/large HPs and/or hyperplastic polyposis.
  • In contrast, BRAF mutation was present in 43% of HPs (P = 0.01 versus sporadic HPs), 75% of serrated adenomas, 33% of admixed hyperplastic-adenomatous polyps, 30% of tubular adenomas, and 33% of carcinomas from patients with multiple/large HPs and/or hyperplastic polyposis.
  • BRAF mutation status in patients with multiple/large HPs and/or hyperplastic polyposis correlated with HPs from the same patient (odds ratio, 5.8; P = 0.0002) but associated with younger age (odds ratio, 0.83; P = 0.006 compared to older age), with a large HP (odds ratio, 22.5; P = 0.01 compared with patients with multiple HPs), with location of HPs in the right colon (odds ratio, 3.0; P = 0.03), and with methylation of the p16 gene and the MINT31 locus [odds ratio, 12.2 (P = 0.0001) and 4.4 (P = 0.02), respectively].
  • Our study shows that BRAF mutation status is heterogeneous among patients with multiple/large HPs and/or hyperplastic polyposis, suggesting differences in pathogenesis of HPs that indicate subsets within this phenotype.
  • [MeSH-major] Colorectal Neoplasms / genetics. Intestinal Polyposis / genetics. Mutation. Proto-Oncogene Proteins B-raf / genetics
  • [MeSH-minor] Adenocarcinoma / genetics. Adenoma / genetics. Aged. Aged, 80 and over. Base Sequence. Female. Humans. Intestinal Mucosa. Intestinal Polyps / genetics. Male. Middle Aged. Molecular Sequence Data

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  • [Cites] Nature. 2002 Jun 27;417(6892):949-54 [12068308.001]
  • [Cites] Histopathology. 1988 Dec;13(6):700-2 [2466756.001]
  • [Cites] Cancer Res. 2002 Nov 15;62(22):6451-5 [12438234.001]
  • [Cites] Am J Surg Pathol. 2003 Jan;27(1):65-81 [12502929.001]
  • [Cites] Cancer Res. 2003 Aug 15;63(16):4878-81 [12941809.001]
  • [Cites] Cancer Res. 2003 Sep 1;63(17):5209-12 [14500346.001]
  • [Cites] Oncogene. 2003 Dec 11;22(57):9192-6 [14668801.001]
  • [Cites] Genes Chromosomes Cancer. 2004 Feb;39(2):138-42 [14695993.001]
  • [Cites] Gut. 2004 Apr;53(4):573-80 [15016754.001]
  • [Cites] Carcinogenesis. 2004 Apr;25(4):527-33 [14688025.001]
  • [Cites] Cell. 2004 Mar 19;116(6):855-67 [15035987.001]
  • [Cites] Gut. 2004 Aug;53(8):1137-44 [15247181.001]
  • [Cites] Am J Surg Pathol. 2004 Nov;28(11):1452-9 [15489648.001]
  • [Cites] S Afr Med J. 1978 Mar 25;53(12):454-7 [675387.001]
  • [Cites] Gastrointest Radiol. 1981;6(4):333-5 [7308714.001]
  • [Cites] Am J Surg Pathol. 1984 Jul;8(7):551-6 [6742315.001]
  • [Cites] Am J Surg Pathol. 1990 Jun;14(6):524-37 [2186644.001]
  • [Cites] Cancer. 1993 Jan 15;71(2):306-14 [8422622.001]
  • [Cites] J Natl Cancer Inst. 1993 Dec 15;85(24):2004-7 [8246286.001]
  • [Cites] Cancer Res. 1993 Dec 15;53(24):5849-52 [8261392.001]
  • [Cites] Cancer Res. 1994 Nov 1;54(21):5523-6 [7923189.001]
  • [Cites] Gastroenterology. 1995 Feb;108(2):434-40 [7835585.001]
  • [Cites] Neth J Med. 1995 Apr;46(4):185-8 [7760968.001]
  • [Cites] Genes Chromosomes Cancer. 1995 Nov;14(3):182-8 [8589034.001]
  • [Cites] Gastroenterology. 1996 Mar;110(3):748-55 [8608884.001]
  • [Cites] Cell. 1996 Oct 18;87(2):159-70 [8861899.001]
  • [Cites] Cancer Res. 1996 Dec 1;56(23):5339-41 [8968080.001]
  • [Cites] Pathology. 1997 Feb;29(1):28-33 [9094174.001]
  • [Cites] Gut. 1997 May;40(5):660-3 [9203947.001]
  • [Cites] Gut. 1997 May;40(5):691-2 [9203954.001]
  • [Cites] Hum Pathol. 1997 Dec;28(12):1396-407 [9416697.001]
  • [Cites] J Natl Cancer Inst. 2001 Sep 5;93(17):1307-13 [11535705.001]
  • [Cites] Am J Pathol. 2002 Feb;160(2):529-36 [11839573.001]
  • [Cites] Am J Pathol. 2002 May;160(5):1823-30 [12000733.001]
  • [Cites] Cancer. 1998 Sep 1;83(5):896-900 [9731892.001]
  • [Cites] Curr Biol. 1999 Jan 28;9(2):R62-5 [10021355.001]
  • [Cites] J Clin Pathol. 1999 Jan;52(1):5-9 [10343605.001]
  • [Cites] Pathology. 1989 Apr;21(2):138-42 [2812873.001]
  • [Cites] Gut. 2000 Jul;47(1):43-9 [10861263.001]
  • [Cites] Gastroenterology. 2000 Aug;119(2):323-32 [10930367.001]
  • [Cites] Gastroenterology. 2000 Sep;119(3):854-65 [10982779.001]
  • [Cites] J Pathol. 2001 Mar;193(3):283-5 [11241405.001]
  • [Cites] Gastroenterology. 2001 Sep;121(3):599-611 [11522744.001]
  • [Cites] Am J Surg Pathol. 1987 Apr;11(4):323-7 [3565675.001]
  • [Cites] Gastroenterology. 2002 Sep;123(3):862-76 [12198712.001]
  • (PMID = 15793287.001).
  • [ISSN] 0002-9440
  • [Journal-full-title] The American journal of pathology
  • [ISO-abbreviation] Am. J. Pathol.
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] EC 2.7.11.1 / BRAF protein, human; EC 2.7.11.1 / Proto-Oncogene Proteins B-raf
  • [Other-IDs] NLM/ PMC1602378
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67. Müller MW, Dahmen R, Köninger J, Michalski CW, Hinz U, Hartel M, Kadmon M, Kleeff J, Büchler MW, Friess H: Is there an advantage in performing a pancreas-preserving total duodenectomy in duodenal adenomatosis? Am J Surg; 2008 Jun;195(6):741-8
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  • [Title] Is there an advantage in performing a pancreas-preserving total duodenectomy in duodenal adenomatosis?
  • BACKGROUND: Duodenal adenomatosis is a premalignant condition often not treatable by local resection or endoscopy.
  • Since the introduction of pancreas-preserving total duodenectomy (PPTD), the question has arisen whether a pp-Whipple resection is still needed to treat duodenal adenomatosis.
  • PATIENTS AND METHODS: In a 5-year period 23 PPTDs were performed for duodenal adenomatosis.
  • CONCLUSIONS: PPTD is a safe surgical procedure for duodenal adenomatosis that avoids pancreatic head resection, provides high quality of life, and shows advantages over the pp-Whipple procedure.


68. Méric JB, Rottey S, Olaussen K, Soria JC, Khayat D, Rixe O, Spano JP: Cyclooxygenase-2 as a target for anticancer drug development. Crit Rev Oncol Hematol; 2006 Jul;59(1):51-64
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  • Moreover, clinical trials in patients with familial adenomatosis polyposis syndrome have shown too the efficacy of non-selective COX inhibitors and recently also of selective COX-2 inhibitors in the reduction of the number and the size of colorectal polyps.

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  • (PMID = 16531064.001).
  • [ISSN] 1040-8428
  • [Journal-full-title] Critical reviews in oncology/hematology
  • [ISO-abbreviation] Crit. Rev. Oncol. Hematol.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Ireland
  • [Chemical-registry-number] 0 / Antineoplastic Agents; 0 / Cyclooxygenase Inhibitors; EC 1.14.99.1 / Cyclooxygenase 2
  • [Number-of-references] 130
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69. Ruys AT, Alderlieste YA, Gouma DJ, Dekker E, Mathus-Vliegen EM: Jejunal cancer in patients with familial adenomatous polyposis. Clin Gastroenterol Hepatol; 2010 Aug;8(8):731-3
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Jejunal cancer in patients with familial adenomatous polyposis.
  • BACKGROUND & AIMS: Familial adenomatous polyposis (FAP) is an inherited disease affecting approximately 1:10,000 newborns, characterized by the formation of numerous adenomas in the digestive tract.
  • RESULTS: Three patients with FAP aged 71, 57, and 59 years developed advanced duodenal adenomatosis and a jejunal carcinoma, which was associated with poor prognosis in 2 patients.
  • [MeSH-major] Adenocarcinoma / diagnosis. Adenoma / diagnosis. Adenomatous Polyposis Coli / complications. Jejunal Neoplasms / diagnosis


70. Cajaiba MM, Sarita-Reyes C, Zambrano E, Reyes-Múgica M: Mesenchymal hamartoma of the liver associated with features of Beckwith-Wiedemann syndrome and high serum alpha-fetoprotein levels. Pediatr Dev Pathol; 2007 May-Jun;10(3):233-8
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  • In addition, we analyze the unusual pancreatic lesion (focal endocrine adenomatosis) leading to severe hyperinsulinemic hypoglycemia in a patient with possible BWS.

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  • [CommentIn] Pediatr Dev Pathol. 2008 Jul-Aug;11(4):264-5 [17990933.001]
  • (PMID = 17535089.001).
  • [ISSN] 1093-5266
  • [Journal-full-title] Pediatric and developmental pathology : the official journal of the Society for Pediatric Pathology and the Paediatric Pathology Society
  • [ISO-abbreviation] Pediatr. Dev. Pathol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / alpha-Fetoproteins
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71. Hardy OT, Hernandez-Pampaloni M, Saffer JR, Suchi M, Ruchelli E, Zhuang H, Ganguly A, Freifelder R, Adzick NS, Alavi A, Stanley CA: Diagnosis and localization of focal congenital hyperinsulinism by 18F-fluorodopa PET scan. J Pediatr; 2007 Feb;150(2):140-5
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  • Pathology results confirmed that all 11 had focal adenomatosis, and the locations of these lesions matched the areas of increased [18F]-DOPA uptake on the PET scans in all of the cases.

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  • [CommentIn] J Pediatr. 2007 Feb;150(2):122-4 [17236885.001]
  • (PMID = 17236890.001).
  • [ISSN] 1097-6833
  • [Journal-full-title] The Journal of pediatrics
  • [ISO-abbreviation] J. Pediatr.
  • [Language] eng
  • [Grant] United States / NCRR NIH HHS / RR / M01-RR-00240; United States / NIDDK NIH HHS / DK / R01-DK-56268; United States / NIDDK NIH HHS / DK / T32-DK63688
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 2C598205QX / fluorodopa F 18; 63-84-3 / Dihydroxyphenylalanine
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72. Giovanoli O, Heim M, Terracciano L, Bongartz G, Ledermann HP: MRI of hepatic adenomatosis: initial observations with gadoxetic acid contrast agent in three patients. AJR Am J Roentgenol; 2008 May;190(5):W290-3
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  • [Title] MRI of hepatic adenomatosis: initial observations with gadoxetic acid contrast agent in three patients.
  • OBJECTIVE: The purpose of our study was to describe the MR signal characteristics of histologically proven liver adenomatosis in three patients using gadoxetic acid, a newly developed liver-specific MR contrast agent.

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  • (PMID = 18430814.001).
  • [ISSN] 1546-3141
  • [Journal-full-title] AJR. American journal of roentgenology
  • [ISO-abbreviation] AJR Am J Roentgenol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Contrast Media; 0 / gadolinium ethoxybenzyl DTPA; K2I13DR72L / Gadolinium DTPA
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73. Veteläinen R, Erdogan D, de Graaf W, ten Kate F, Jansen PL, Gouma DJ, van Gulik TM: Liver adenomatosis: re-evaluation of aetiology and management. Liver Int; 2008 Apr;28(4):499-508
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  • [Title] Liver adenomatosis: re-evaluation of aetiology and management.
  • BACKGROUND: Liver adenomatosis (LA) is a rare benign parenchymal liver disease with unknown aetiology.
  • CONCLUSION: Liver adenomatosis is a progressive, benign parenchymal disease mainly occurring in females.

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  • [CommentIn] Liver Int. 2009 Jan;29(1):142 [18828785.001]
  • (PMID = 18339077.001).
  • [ISSN] 1478-3231
  • [Journal-full-title] Liver international : official journal of the International Association for the Study of the Liver
  • [ISO-abbreviation] Liver Int.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Number-of-references] 57
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74. Konstantinova D, Kadiyska T, Sokolova V, Kaneva R, Mirchev M, Savov A, Aleksandrova A, Nedin D, Kostadinov E, Damyanov D, Kremensky I, Mitev V: CHEK2 I157T and colorectal cancer in Bulgaria. J BUON; 2010 Apr-Jun;15(2):314-7
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  • Higher frequencies were found among patients with multiple polyposis (2/40, 5%, p=0.28) and the rarer mucinous histology (1/11, 9.09%, p= 0.26).

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  • (PMID = 20658728.001).
  • [ISSN] 1107-0625
  • [Journal-full-title] Journal of B.U.ON. : official journal of the Balkan Union of Oncology
  • [ISO-abbreviation] J BUON
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Greece
  • [Chemical-registry-number] 0 / DNA Primers; EC 2.7.1.11 / Checkpoint Kinase 2; EC 2.7.11.1 / CHEK2 protein, human; EC 2.7.11.1 / Protein-Serine-Threonine Kinases
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75. McCart A, Latchford A, Volikos E, Rowan A, Tomlinson I, Silver A: A novel exon duplication event leading to a truncating germ-line mutation of the APC gene in a familial adenomatous polyposis family. Fam Cancer; 2006;5(2):205-8
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  • [Title] A novel exon duplication event leading to a truncating germ-line mutation of the APC gene in a familial adenomatous polyposis family.
  • Familial Adenomatous Polyposis (FAP) is an autosomal dominant condition predisposing to multiple adenomatous polyps of the colon.
  • [MeSH-major] Adenomatous Polyposis Coli / genetics. Exons. Gene Duplication. Genes, APC. Germ-Line Mutation

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  • [Cites] Proc Natl Acad Sci U S A. 1992 May 15;89(10):4452-6 [1316610.001]
  • [Cites] Proc Natl Acad Sci U S A. 2002 Mar 5;99(5):2954-8 [11867715.001]
  • [Cites] Hum Mutat. 1997;9(1):7-16 [8990002.001]
  • [Cites] Cell. 1993 Dec 3;75(5):959-68 [8252631.001]
  • [Cites] Br J Cancer. 2004 Sep 13;91(6):1155-9 [15475941.001]
  • [Cites] Hum Mutat. 2005 Feb;25(2):125-34 [15643602.001]
  • [Cites] Nucleic Acids Res. 2002 Jun 15;30(12 ):e57 [12060695.001]
  • [Cites] Gut. 2005 Feb;54(2):264-7 [15647192.001]
  • [Cites] Hum Mol Genet. 1994 Jan;3(1):181-4 [8162022.001]
  • [Cites] J Clin Pathol. 1994 Aug;47(8):709-10 [7962621.001]
  • [Cites] Hum Mol Genet. 1994 Sep;3(9):1703-4 [7833936.001]
  • [Cites] Gut. 2001 Apr;48(4):515-21 [11247896.001]
  • [Cites] Am J Hum Genet. 2003 Oct;73(4):823-34 [14505274.001]
  • [Cites] Hum Mutat. 2003 Dec;22(6):428-33 [14635101.001]
  • [Cites] J Med Genet. 1999 Jan;36(1):14-20 [9950360.001]
  • (PMID = 16736293.001).
  • [ISSN] 1389-9600
  • [Journal-full-title] Familial cancer
  • [ISO-abbreviation] Fam. Cancer
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / APC2 protein, human; 0 / Cytoskeletal Proteins
  •  go-up   go-down


76. Kaufmann C, Brechbühl M, Oevermann A, Müller S, Zimmer K, Schönmann M, Leroux C, Weibel D, Bertoni G: [Enzootic nasal adenocarcinoma in a dairy sheep flock]. Schweiz Arch Tierheilkd; 2008 Jun;150(6):297-302
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  • The significance of the disease in Switzerland is discussed, also in the context of lung adenomatosis.
  • [MeSH-major] Adenocarcinoma / veterinary. Jaagsiekte sheep retrovirus / isolation & purification. Nose Neoplasms / veterinary. Pulmonary Adenomatosis, Ovine / diagnosis. Sheep Diseases / diagnosis

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  • (PMID = 18605021.001).
  • [ISSN] 0036-7281
  • [Journal-full-title] Schweizer Archiv für Tierheilkunde
  • [ISO-abbreviation] Schweiz. Arch. Tierheilkd.
  • [Language] ger
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] Switzerland
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77. Kadzhaia NR, Virsaladze DK, Tkeshelashvili BD, Dzhavashvili LV, Dzhugeli MK: [Endocrine-metabolic peculiarities in women of reproductive age with hyperplastic processes of cervix and mammary glands]. Georgian Med News; 2006 May;(134):60-3
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  • 72 patients with hyperplastic processes in endomyometrium, uterine cervix (hyperplasia, polyposis, myoma) and mammary glands (fibroadenomatosis, adenomatosis) were selected in main group.


78. Berkhout M, Roelofs HM, te Morsche RH, Dekker E, van Krieken JH, Nagengast FM, Peters WH: Detoxification enzyme polymorphisms are not involved in duodenal adenomatosis in familial adenomatous polyposis. Br J Surg; 2008 Apr;95(4):499-505
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  • [Title] Detoxification enzyme polymorphisms are not involved in duodenal adenomatosis in familial adenomatous polyposis.
  • BACKGROUND: Patients with familial adenomatous polyposis (FAP) are at high risk of developing duodenal adenomas and carcinomas.
  • Besides germline mutations in the adenomatous polyposis coli (APC) gene, additional factors may influence the age of onset and number of duodenal adenomas.
  • The polymorphisms investigated had no predictive value for the severity of duodenal adenomatosis in patients with FAP.
  • CONCLUSION: Although the variant genotypes of UGT1A3 were less common in patients with FAP than in those without, this did not modulate the severity of duodenal adenomatosis.
  • [MeSH-major] Adenomatous Polyposis Coli / enzymology. Duodenal Neoplasms / enzymology. Genes, APC. Glucuronosyltransferase / genetics. Glutathione Transferase / genetics. Polymorphism, Genetic / genetics


79. Ku BS, Kwon OE, Kim DC, Song KH, Lee CW, Kim KH: A case of erosive adenomatosis of nipple treated with total excision using purse-string suture. Dermatol Surg; 2006 Aug;32(8):1093-6
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  • [Title] A case of erosive adenomatosis of nipple treated with total excision using purse-string suture.

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  • [CommentIn] Dermatol Surg. 2007 Aug;33(8):1001 [17661951.001]
  • (PMID = 16918575.001).
  • [ISSN] 1076-0512
  • [Journal-full-title] Dermatologic surgery : official publication for American Society for Dermatologic Surgery [et al.]
  • [ISO-abbreviation] Dermatol Surg
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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80. Marín Hernández C, Robles Campos R, López Conesa A, Parrilla P: [Multiple liver adenomatosis treated with surgical resection and radiofrequency]. Cir Esp; 2010 Nov;88(5):337-8
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  • [Title] [Multiple liver adenomatosis treated with surgical resection and radiofrequency].
  • [Transliterated title] Adenomatosis hepática múltiple tratada con resección quirúrgica y radiofrecuencia.

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  • (PMID = 20206344.001).
  • [ISSN] 1578-147X
  • [Journal-full-title] Cirugía española
  • [ISO-abbreviation] Cir Esp
  • [Language] spa
  • [Publication-type] Case Reports; Letter
  • [Publication-country] Spain
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81. Raphe R, Felício HC, Rocha MF, Duca WJ, Arroyo PC Jr, D'Santi Neto D, da Silva RC, da Silva RF: Histopathologic characteristics of incidental hepatocellular carcinoma after liver transplantation. Transplant Proc; 2010 Mar;42(2):505-6
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  • One patient presented with adenomatosis and focus of HCC.

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  • [Copyright] Copyright (c) 2010 Elsevier Inc. All rights reserved.
  • (PMID = 20304178.001).
  • [ISSN] 1873-2623
  • [Journal-full-title] Transplantation proceedings
  • [ISO-abbreviation] Transplant. Proc.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / alpha-Fetoproteins
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82. Matsumoto T, Esaki M, Yanaru-Fujisawa R, Moriyama T, Yada S, Nakamura S, Yao T, Iida M: Small-intestinal involvement in familial adenomatous polyposis: evaluation by double-balloon endoscopy and intraoperative enteroscopy. Gastrointest Endosc; 2008 Nov;68(5):911-9
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  • [Title] Small-intestinal involvement in familial adenomatous polyposis: evaluation by double-balloon endoscopy and intraoperative enteroscopy.
  • BACKGROUND: Small-intestinal adenoma occurs in patients with familial adenomatous polyposis (FAP).
  • Phenotypes of FAP and genotypes of adenomatous polyposis coli (APC) were then compared between patients with small-intestinal adenomas and those without.
  • The adenomas occurred predominantly in the jejunum, with a configuration of diminutive polyps in 22 patients.
  • Patients with small-intestinal adenoma had more severe duodenal adenomatosis than those patients without small-intestinal adenoma (P < .001).
  • [MeSH-major] Adenomatous Polyposis Coli / pathology. Duodenal Neoplasms / pathology. Endoscopy, Gastrointestinal. Jejunal Neoplasms / pathology


83. Vermeil V, D'Amore L, Ceci F, Dassatti MR, Negro A, Gossetti F, Negro P: [Familial polyposis coli associated with carcinoma of the uterine cervix]. Chir Ital; 2008 May-Jun;60(3):355-9
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  • [Title] [Familial polyposis coli associated with carcinoma of the uterine cervix].
  • Familial polyposis coli is a heterogeneous disease with a broad spectrum of clinical manifestations including not only multiple polyposis of the small bowel, but also multiple primary tumours, such as carcinoma of the ampulla of Vater, subcutaneous tumours, bone tumours, central nervous system tumous and gynaecological malignancies.
  • This report is of two brothers with familial polyposis, each showing peculiar distinctive features.
  • In one case, polyposis was diagnosed during emergency surgery for ileo-colic intussusception.
  • Polyposis coli was identified late in the second patient who showed an evolution towards colonic adenocarcinoma with multiple hepatic metastases.
  • The possible association of familial polyposis and extracolonic malignancies has already been emphasized in the literature.
  • [MeSH-major] Adenomatous Polyposis Coli. Carcinoma in Situ. Neoplasms, Multiple Primary. Uterine Cervical Neoplasms


84. Woldemeskel M, Tibbo M: Pulmonary adenomatosis and maedi-visna in Ethiopian central highland sheep: a microscopic study. Trop Anim Health Prod; 2010 Jun;42(5):995-9
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  • [Title] Pulmonary adenomatosis and maedi-visna in Ethiopian central highland sheep: a microscopic study.
  • Lesions compatible with sheep pulmonary adenomatosis (SPA; 8/35, 22.8%), and maedi-visna (MV; 9/35, 25.7%) were recorded only in sheep from the central highlands.
  • [MeSH-major] Pulmonary Adenomatosis, Ovine / epidemiology. Sheep Diseases / epidemiology. Visna / epidemiology. Visna-maedi virus / isolation & purification

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  • [Cites] Trop Anim Health Prod. 2009 Jun;41(5):803-6 [18989743.001]
  • [Cites] Vet J. 2003 May;165(3):276-87 [12672374.001]
  • [Cites] Dtsch Tierarztl Wochenschr. 2002 Nov;109(11):486-8 [12494555.001]
  • [Cites] Vet Rec. 1991 Jul 20;129(3):51-4 [1656581.001]
  • [Cites] Res Vet Sci. 1993 Mar;54(2):140-6 [8384727.001]
  • [Cites] Vet Parasitol. 2006 Dec 20;142(3-4):330-5 [16919877.001]
  • [Cites] Small Rumin Res. 2000 Jul 1;37(1-2):1-12 [10818297.001]
  • (PMID = 20024674.001).
  • [ISSN] 1573-7438
  • [Journal-full-title] Tropical animal health and production
  • [ISO-abbreviation] Trop Anim Health Prod
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  •  go-up   go-down


85. Miller MJ, Reuter BK, Wallace JL, Sharkey KA: A unique therapeutic approach to emesis and itch with a proanthocyanidin-rich genonutrient. J Transl Med; 2008;6:3
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  • In the ApcMinmouse model of precancerous adenomatosis polyposis, mice received Zangrado (100 mug/ml in drinking water) from the age of 6 - 16 weeks for effects on polyp number.

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  • [Cites] Curr Opin Pharmacol. 2004 Dec;4(6):572-9 [15525546.001]
  • [Cites] J Neurosci. 1997 Oct 15;17(20):8003-8 [9315918.001]
  • [Cites] J Clin Invest. 2007 Mar;117(3):636-47 [17304351.001]
  • [Cites] BMC Complement Altern Med. 2007;7:22 [17578576.001]
  • [Cites] JAMA. 2007 Sep 12;298(10):1196-207 [17848654.001]
  • [Cites] CNS Drugs. 2007;21(10):813-33 [17850171.001]
  • [Cites] J Clin Anesth. 2007 Aug;19(5):356-9 [17869986.001]
  • [Cites] Am J Physiol Gastrointest Liver Physiol. 2000 Jul;279(1):G192-200 [10898763.001]
  • [Cites] Skin Pharmacol Appl Skin Physiol. 2001 Mar-Apr;14(2):87-96 [11316967.001]
  • [Cites] Anesthesiology. 2001 May;94(5):882-7 [11388542.001]
  • [Cites] J Invest Dermatol. 2001 Sep;117(3):725-30 [11564183.001]
  • [Cites] Gastroenterology. 2001 Oct;121(4):767-74 [11606489.001]
  • [Cites] Acta Derm Venereol. 2001 Aug-Sep;81(4):250-4 [11720170.001]
  • [Cites] J Neurophysiol. 2002 Mar;87(3):1280-9 [11877502.001]
  • [Cites] J Ethnopharmacol. 2002 May;80(2-3):121-9 [12007701.001]
  • [Cites] Expert Opin Investig Drugs. 2002 Jun;11(6):801-5 [12036423.001]
  • [Cites] Phytomedicine. 2002 May;9(4):325-37 [12120814.001]
  • [Cites] BMC Cancer. 2002 Aug 9;2:19 [12171603.001]
  • [Cites] Oncology (Williston Park). 2002 Oct;16(10):1401-7; discussion 1407-8, 1412 [12435208.001]
  • [Cites] Carcinogenesis. 2003 Mar;24(3):605-11 [12663524.001]
  • [Cites] J Neurophysiol. 2003 May;89(5):2441-8 [12611975.001]
  • [Cites] Am J Physiol Gastrointest Liver Physiol. 2003 Sep;285(3):G566-76 [12791597.001]
  • [Cites] J Neurosci. 2003 Nov 26;23(34):10784-90 [14645470.001]
  • [Cites] Am Fam Physician. 2004 Mar 1;69(5):1169-74 [15023018.001]
  • [Cites] Toxicon. 2004 Sep 15;44(4):461-7 [15302527.001]
  • [Cites] N Engl J Med. 1995 Sep 7;333(10):609-14 [7637720.001]
  • [Cites] Pharmacol Biochem Behav. 2005 Jan;80(1):35-44 [15652378.001]
  • (PMID = 18205911.001).
  • [ISSN] 1479-5876
  • [Journal-full-title] Journal of translational medicine
  • [ISO-abbreviation] J Transl Med
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / AM 251; 0 / Antiemetics; 0 / Antipruritics; 0 / Morphine Derivatives; 0 / Piperidines; 0 / Plant Extracts; 0 / Proanthocyanidins; 0 / Pyrazoles; 0 / Receptor, Cannabinoid, CB1; 18206-61-6 / proanthocyanidin; 20290-10-2 / morphine-6-glucuronide
  • [Other-IDs] NLM/ PMC2254379
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86. Di Sandro S, Slim AO, Lauterio A, Giacomoni A, Mangoni I, Aseni P, Pirotta V, Aldumour A, Mihaylov P, De Carlis L: Liver adenomatosis: a rare indication for living donor liver transplantation. Transplant Proc; 2009 May;41(4):1375-7
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  • [Title] Liver adenomatosis: a rare indication for living donor liver transplantation.
  • Liver adenomatosis (LA) is a rare benign disease of the liver with unclear pathogenesis, which is characterized by multiple hepatic adenomas.
  • A 48-year-old woman developed multiple liver adenomas.
  • Thirty-four months later, she developed recurrence of multiple hepatic adenomas and 2 nodules were highly suspect for hepatocellular carcinoma.


87. Cadigan KM, Liu YI: Wnt signaling: complexity at the surface. J Cell Sci; 2006 Feb 1;119(Pt 3):395-402
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  • Activation of Fz-LRP promotes the stability and nuclear localization of beta-catenin by compromising the ability of a multiprotein complex containing axin, adenomatosis polyposis coli (APC) and glycogen synthase kinase 3 (GSK3) to target it for degradation and block its nuclear import.
  • The Fz-LRP receptor complex probably accomplishes this by generating multiple signals in the cytoplasm.

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  • (PMID = 16443747.001).
  • [ISSN] 0021-9533
  • [Journal-full-title] Journal of cell science
  • [ISO-abbreviation] J. Cell. Sci.
  • [Language] eng
  • [Grant] United States / NCI NIH HHS / CA / CA95869; United States / NIGMS NIH HHS / GM / R01 GM59846
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Low Density Lipoprotein Receptor-Related Protein-1; 0 / Multiprotein Complexes; 0 / Receptors, Neurotransmitter; 0 / Wnt Proteins
  • [Number-of-references] 131
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88. Beytut E, Sözmen M, Ergínsoy S: Immunohistochemical detection of pulmonary surfactant proteins and retroviral antigens in the lungs of sheep with pulmonary adenomatosis. J Comp Pathol; 2009 Jan;140(1):43-53
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  • [Title] Immunohistochemical detection of pulmonary surfactant proteins and retroviral antigens in the lungs of sheep with pulmonary adenomatosis.
  • The lungs and mediastinal and bronchial lymph nodes from 26 sheep with ovine pulmonary adenomatosis (OPA) were examined.
  • [MeSH-major] Antigens, Viral / metabolism. Lung / metabolism. Pulmonary Adenomatosis, Ovine / metabolism. Pulmonary Surfactant-Associated Protein A / metabolism. Pulmonary Surfactant-Associated Protein B / metabolism. Pulmonary Surfactant-Associated Protein C / metabolism. Retroviridae / immunology

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  • (PMID = 19081577.001).
  • [ISSN] 0021-9975
  • [Journal-full-title] Journal of comparative pathology
  • [ISO-abbreviation] J. Comp. Pathol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Antigens, CD3; 0 / Antigens, Viral; 0 / Proliferating Cell Nuclear Antigen; 0 / Pulmonary Surfactant-Associated Protein A; 0 / Pulmonary Surfactant-Associated Protein B; 0 / Pulmonary Surfactant-Associated Protein C; 9060-09-7 / Uteroglobin
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89. Greaves WO, Bhattacharya B: Hepatic adenomatosis. Arch Pathol Lab Med; 2008 Dec;132(12):1951-5
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  • [Title] Hepatic adenomatosis.
  • Hepatic adenomatosis was first described in 1985 by Flejou et al as multiple adenomas, arbitrarily more than 10, in an otherwise normal liver parenchyma.

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  • (PMID = 19061298.001).
  • [ISSN] 1543-2165
  • [Journal-full-title] Archives of pathology & laboratory medicine
  • [ISO-abbreviation] Arch. Pathol. Lab. Med.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Hepatocyte Nuclear Factor 1-alpha
  • [Number-of-references] 30
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90. Jeannot E, Poussin K, Chiche L, Bacq Y, Sturm N, Scoazec JY, Buffet C, Van Nhieu JT, Bellanné-Chantelot C, de Toma C, Laurent-Puig P, Bioulac-Sage P, Zucman-Rossi J: Association of CYP1B1 germ line mutations with hepatocyte nuclear factor 1alpha-mutated hepatocellular adenoma. Cancer Res; 2007 Mar 15;67(6):2611-6
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  • In rare cases, HNF1alpha germ line mutations could also predispose to familial adenomatosis.
  • In addition, an E229K CYP1B1 mutation was found in a woman with a germ line HNF1alpha mutation in a familial adenomatosis context.
  • In this large family, all three patients with adenomatosis bore both HNF1 and CYP1B1 germ line mutations.
  • [MeSH-minor] Adolescent. Adult. Alleles. Aryl Hydrocarbon Hydroxylases. Child. Cytochrome P-450 CYP1B1. Female. Genetic Predisposition to Disease. Genotype. Humans. Middle Aged. Mutagenesis, Site-Directed. Pedigree

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  • (PMID = 17363580.001).
  • [ISSN] 0008-5472
  • [Journal-full-title] Cancer research
  • [ISO-abbreviation] Cancer Res.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Hepatocyte Nuclear Factor 1-alpha; 9035-51-2 / Cytochrome P-450 Enzyme System; EC 1.14.14.1 / Aryl Hydrocarbon Hydroxylases; EC 1.14.14.1 / CYP1B1 protein, human; EC 1.14.14.1 / Cytochrome P-450 CYP1B1
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91. Ajith Kumar VK, Gold JA, Mallon E, Thomas S, Hodgson SV: Sebaceous adenomas in an MYH associated polyposis patient of Indian (Gujarati) origin. Fam Cancer; 2008;7(2):187-9
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  • [Title] Sebaceous adenomas in an MYH associated polyposis patient of Indian (Gujarati) origin.
  • MYH associated polyposis is an autosomal recessive polyposis syndrome with a high risk of large bowel cancer, caused by mutations in the DNA repair gene MYH.
  • Muir Torre Syndrome is the association of internal malignancies with sebaceous gland tumours; Lynch Syndrome/Hereditary Non Polyposis Cancer is the best known cause.
  • There has been a previous report of sebaceous gland tumours in an Italian patient with MYH associated polyposis.
  • We describe a man of Indian (Gujarati) descent who has MYH associated polyposis and multiple sebaceous adenomas of the skin.
  • [MeSH-major] Adenoma / genetics. Adenomatous Polyposis Coli / genetics. DNA Glycosylases / genetics. Sebaceous Gland Neoplasms / genetics

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  • [Cites] Nat Genet. 2002 Feb;30(2):227-32 [11818965.001]
  • [Cites] Am J Med Genet A. 2005 Apr 15;134A(2):212-4 [15690400.001]
  • [Cites] Lancet. 2003 Jul 5;362(9377):39-41 [12853198.001]
  • [Cites] Int J Cancer. 2004 Apr 20;109 (4):554-8 [14991577.001]
  • [Cites] Clin Genet. 2005 Nov;68(5):442-7 [16207212.001]
  • [Cites] Int J Cancer. 2004 May 1;109(5):680-4 [14999774.001]
  • (PMID = 17874208.001).
  • [ISSN] 1389-9600
  • [Journal-full-title] Familial cancer
  • [ISO-abbreviation] Fam. Cancer
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Netherlands
  • [Chemical-registry-number] EC 3.2.2.- / DNA Glycosylases; EC 3.2.2.- / mutY adenine glycosylase
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92. Lewin M, Handra-Luca A, Arrivé L, Wendum D, Paradis V, Bridel E, Fléjou JF, Belghiti J, Tubiana JM, Vilgrain V: Liver adenomatosis: classification of MR imaging features and comparison with pathologic findings. Radiology; 2006 Nov;241(2):433-40
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  • [Title] Liver adenomatosis: classification of MR imaging features and comparison with pathologic findings.
  • PURPOSE: To retrospectively compare the clinical manifestation and magnetic resonance (MR) imaging features of liver adenomatosis with pathologic findings.
  • Twenty patients were classified on the basis of pathologic findings into three groups: those with a steatotic, those with a peliotic, and those with a mixed (steatotic and peliotic) form of liver adenomatosis.
  • CONCLUSION: There are three patterns of MR imaging features of liver adenomatosis that are associated with three pathologic forms (steatotic, peliotic, and mixed).

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  • (PMID = 16966481.001).
  • [ISSN] 0033-8419
  • [Journal-full-title] Radiology
  • [ISO-abbreviation] Radiology
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Contrast Media; 0 / Organometallic Compounds; 0 / gadoterate meglumine; 6HG8UB2MUY / Meglumine
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93. Ghosh N, Chaki R, Mandal V, Mandal SC: COX-2 as a target for cancer chemotherapy. Pharmacol Rep; 2010 Mar-Apr;62(2):233-44
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  • Moreover, clinical trials in patients with familial adenomatosis polyposis syndrome have shown the efficacy of selective COX-2 inhibitors in the reduction of the number and size of colorectal polyps.

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  • (PMID = 20508278.001).
  • [ISSN] 1734-1140
  • [Journal-full-title] Pharmacological reports : PR
  • [ISO-abbreviation] Pharmacol Rep
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Poland
  • [Chemical-registry-number] 0 / Anti-Inflammatory Agents, Non-Steroidal; 0 / Cyclooxygenase 2 Inhibitors; 0 / Pyrazoles; 0 / Sulfonamides; EC 1.14.99.1 / Cyclooxygenase 2; EC 2.7.10.1 / Receptor, Epidermal Growth Factor; EC 2.7.11.1 / Protein-Serine-Threonine Kinases; EC 2.7.11.2 / pyruvate dehydrogenase (acetyl-transferring) kinase; EC 3.6.3.8 / Sarcoplasmic Reticulum Calcium-Transporting ATPases; JCX84Q7J1L / Celecoxib
  • [Number-of-references] 72
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94. von Meyenfeldt EM, Mantel SF, Gouma DJ, van Gulik TM: [Tumors in the gallbladder: a possible differentiation between malignant and benign tumours]. Ned Tijdschr Geneeskd; 2007 May 12;151(19):1049-54
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  • In a 59-year-old woman with pain in the right upper abdomen, echography and CT-scan revealed adenomatosis of the gallbladder.
  • Her symptoms did not disappear after cholecystectomy, even though echography showed no further abnormality.
  • In a 46-year-old man with fever and weight loss, echography revealed two polyps in the gallbladder.
  • Following cholecystectomy, histopathology revealed cholesterol polyps and an infection with Entamoeba coli.
  • [MeSH-minor] Aged. Diagnosis, Differential. Female. Humans. Magnetic Resonance Imaging / methods. Male. Middle Aged. Polyps / diagnosis. Polyps / surgery. Tomography, X-Ray Computed / methods. Ultrasonography / methods

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  • [CommentIn] Ned Tijdschr Geneeskd. 2007 Jul 21;151(29):1653; author reply 1653 [17727189.001]
  • [CommentIn] Ned Tijdschr Geneeskd. 2007 Jul 21;151(29):1652-3; author reply 1653 [17729454.001]
  • [CommentOn] Ned Tijdschr Geneeskd. 2007 May 12;151(19):1083-6 [17552418.001]
  • (PMID = 17552411.001).
  • [ISSN] 0028-2162
  • [Journal-full-title] Nederlands tijdschrift voor geneeskunde
  • [ISO-abbreviation] Ned Tijdschr Geneeskd
  • [Language] dut
  • [Publication-type] Case Reports; Comment; English Abstract; Journal Article
  • [Publication-country] Netherlands
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95. Entz-Werlé N, Choquet P, Neuville A, Kuchler-Bopp S, Clauss F, Danse JM, Simo-Noumbissie P, Guérin E, Gaub MP, Freund JN, Boehm N, Constantinesco A, Lutz P, Guenot D, Perrin-Schmitt F: Targeted apc;twist double-mutant mice: a new model of spontaneous osteosarcoma that mimics the human disease. Transl Oncol; 2010;3(6):344-53
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  • TWIST and adenomatosis polyposis coli (APC) are critical signaling factors in normal bone development.

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  • (PMID = 21151473.001).
  • [ISSN] 1936-5233
  • [Journal-full-title] Translational oncology
  • [ISO-abbreviation] Transl Oncol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC3000459
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96. Grazioli L, Bondioni MP, Faccioli N, Gambarini S, Tinti R, Schneider G, Kirchin M: Solid focal liver lesions: dynamic and late enhancement patterns with the dual phase contrast agent gadobenate dimeglumine. J Gastrointest Cancer; 2010 Dec;41(4):221-32
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  • IMAGING FINDINGS: Unenhanced T2- and T1-weighted, dynamic T1-weighted (arterial, portal-venous, and equilibrium) and late phase (1-3 h) Gd-BOPTA-enhanced MR imaging of different focal liver lesions (nodular regenerative hyperplasia, hepatic adenoma, liver adenomatosis, hepatocellular carcinoma, peripheral cholangiocarcinoma, hypervascular metastases, and hypovascular metastases) are shown.

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  • [Cites] Acad Radiol. 1998 Apr;5 Suppl 1:S86-9; discussion S93-4 [9561052.001]
  • [Cites] AJR Am J Roentgenol. 2001 Jun;176(6):1499-507 [11373220.001]
  • [Cites] Prog Liver Dis. 1996;14:265-78 [9055582.001]
  • [Cites] AJR Am J Roentgenol. 2004 May;182(5):1217-23 [15100122.001]
  • [Cites] Invest Radiol. 2001 Nov;36(11):632-41 [11606840.001]
  • [Cites] Hepatology. 1990 May;11(5):787-97 [2189821.001]
  • [Cites] J Comput Assist Tomogr. 1999 Sep-Oct;23 (5):670-7 [10524843.001]
  • [Cites] Radiology. 2000 Jun;215(3):727-36 [10831691.001]
  • [Cites] Eur Radiol. 2004 Jan;14(1):5-13 [14600778.001]
  • [Cites] Ann Surg. 1988 Nov;208(5):558-64 [3190282.001]
  • [Cites] Radiology. 2005 Jul;236(1):166-77 [15955857.001]
  • [Cites] Dig Dis Sci. 1992 Jul;37(7):1105-9 [1618060.001]
  • [Cites] Radiology. 1994 Aug;192(2):393-9 [8029403.001]
  • [Cites] J Comput Assist Tomogr. 1999 Nov;23 Suppl 1:S161-8 [10608412.001]
  • [Cites] Acad Radiol. 1999 May;6(5):282-91 [10228617.001]
  • [Cites] AJR Am J Roentgenol. 2000 Oct;175(4):1111-20 [11000175.001]
  • [Cites] AJR Am J Roentgenol. 2002 Apr;178(4):877-83 [11906867.001]
  • [Cites] J Magn Reson Imaging. 1995 Mar-Apr;5(2):165-70 [7766977.001]
  • [Cites] Hepatogastroenterology. 2001 Sep-Oct;48(41):1425-9 [11677979.001]
  • [Cites] Radiology. 1994 Nov;193(2):507-12 [7972769.001]
  • [Cites] Invest Radiol. 2006 Mar;41(3):213-21 [16481903.001]
  • [Cites] J Hepatol. 2001 Sep;35(3):421-30 [11592607.001]
  • [Cites] J Pediatr Gastroenterol Nutr. 1997 Mar;24(3):276-9 [9138172.001]
  • [Cites] J Magn Reson Imaging. 2003 May;17 (5):593-602 [12720270.001]
  • [Cites] Eur Radiol. 2001;11(8):1374-88 [11519546.001]
  • [Cites] Invest Radiol. 2006 Mar;41(3):222-8 [16481904.001]
  • [Cites] Invest Radiol. 1997 Dec;32(12):780-96 [9406019.001]
  • [Cites] Magn Reson Imaging Clin N Am. 1997 May;5(2):319-30 [9113677.001]
  • [Cites] Hepatology. 1995 Dec;22(6):1674-81 [7489973.001]
  • [Cites] Invest Radiol. 1998 Nov;33(11):798-809 [9818314.001]
  • [Cites] Semin Liver Dis. 1987 Aug;7(3):257-68 [3317862.001]
  • [Cites] Cancer. 1986 Jul 15;58(2):321-8 [3013389.001]
  • [Cites] J Comput Assist Tomogr. 1997 Jan-Feb;21(1):59-65 [9022771.001]
  • [Cites] Radiol Clin North Am. 1994 Jan;32(1):39-54 [8284360.001]
  • [Cites] J Magn Reson Imaging. 1999 May;9(5):704-10 [10331767.001]
  • [Cites] Invest Radiol. 2000 Jan;35(1):25-34 [10639033.001]
  • [Cites] Invest Radiol. 2003 Feb;38(2):85-94 [12544071.001]
  • [Cites] Radiology. 2001 Dec;221(3):731-9 [11719669.001]
  • [Cites] AJR Am J Roentgenol. 1993 Jun;160(6):1177-87 [8388618.001]
  • [Cites] Hum Pathol. 1991 Feb;22(2):172-8 [1848205.001]
  • [Cites] Radiology. 1996 May;199(2):513-20 [8668804.001]
  • [Cites] Hepatology. 1985 Nov-Dec;5(6):1194-200 [4065824.001]
  • [Cites] Eur Radiol. 2005 Feb;15(2):220-8 [15624108.001]
  • (PMID = 20405242.001).
  • [ISSN] 1941-6636
  • [Journal-full-title] Journal of gastrointestinal cancer
  • [ISO-abbreviation] J Gastrointest Cancer
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Contrast Media; 0 / Organometallic Compounds; 15G12L5X8K / gadobenic acid; 6HG8UB2MUY / Meglumine
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97. Ivanov SV, Zhabin SN, Gorbacheva OS, Barsukov VS, Denisova EIu: [Multifactor analysis of nodular goiter by the method of artificial neural network]. Klin Med (Mosk); 2008;86(4):49-50
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  • Study of 297 patients with preparative methods of nontoxic nodular goiter and cytological diagnose "adenomatosis" with the use of artificial neural network (ANN) were analyzed.
  • Painless elastic nodules with sonographically iso- o hyperechogenic structure, with clear outline and cytologically determined adenomatosis in combination with colloid can be interpreted as benign lesion of thyroid gland.

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  • (PMID = 18494287.001).
  • [ISSN] 0023-2149
  • [Journal-full-title] Klinicheskaia meditsina
  • [ISO-abbreviation] Klin Med (Mosk)
  • [Language] rus
  • [Publication-type] Comparative Study; English Abstract; Journal Article
  • [Publication-country] Russia (Federation)
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98. Scurry J, van der Putte SC, Pyman J, Chetty N, Szabo R: Mammary-like gland adenoma of the vulva: review of 46 cases. Pathology; 2009;41(4):372-8
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  • Unusual architectural patterns resembled breast lesions such as erosive adenomatosis, sclerosing adenosis and ductal adenoma.
  • The ductal cell and a site compatible with and/or the presence of adjacent normal MLG are the most characteristic features.
  • Unusual vulvar tumours, which have been previously reported as erosive adenomatosis, sclerosing adenosis, papillary adenofibroma, syringocystadenoma papilliferans, etc., are variants of MLG adenomas.

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  • (PMID = 19404851.001).
  • [ISSN] 1465-3931
  • [Journal-full-title] Pathology
  • [ISO-abbreviation] Pathology
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Number-of-references] 46
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99. Bülow S, Bülow C, Vasen H, Järvinen H, Björk J, Christensen IJ: Colectomy and ileorectal anastomosis is still an option for selected patients with familial adenomatous polyposis. Dis Colon Rectum; 2008 Sep;51(9):1318-23
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  • [Title] Colectomy and ileorectal anastomosis is still an option for selected patients with familial adenomatous polyposis.
  • PURPOSE: The risk of rectal cancer after colectomy and ileorectal anastomosis may be reduced in the last decades, as patients with severe polyposis now have an ileoanal pouch.
  • This indicates that ileorectal anastomosis may still be justified in selected patients with mild adenomatosis, especially in young females.
  • [MeSH-major] Adenomatous Polyposis Coli / surgery. Colectomy. Ileum / surgery. Rectal Neoplasms / epidemiology. Rectum / surgery


100. Alese OB, Irabor DO: Adenomatous polyposis coli in an elderly female nigerian. Ghana Med J; 2009 Sep;43(3):139-41
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  • [Title] Adenomatous polyposis coli in an elderly female nigerian.
  • Colorectal cancer is reported to occur in lower rates in Africans and some of the reasons adduced include a rarity of polyposis coli syndromes.
  • Only two cases of polyposis coli have been documented in Nigeria in the last 15 years.
  • The present case is an elderly lady who had radiologic and colonoscopic evidence of multiple colonic polyps.
  • She had a colonic resection and the histology was reported as multiple polyposis coli, follicular hyperplasia and submucosal fibrosis in the appendix, and reactive hyperplasia in the mesenteric lymph nodes.
  • This is the first case report of polyposis coli in a living elderly patient in Nigeria.

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  • [Cites] Int J Cancer. 2006 Aug 15;119(4):807-14 [16557584.001]
  • [Cites] Cent Afr J Med. 1997 Nov;43(11):339 [9631103.001]
  • [Cites] Ann Surg. 1998 Jan;227(1):57-62 [9445111.001]
  • [Cites] Dis Colon Rectum. 1996 May;39(5):536-40 [8620804.001]
  • [Cites] Cent Afr J Med. 1992 Jan;38(1):44-8 [1321689.001]
  • [Cites] Gut. 2001 Apr;48(4):515-21 [11247896.001]
  • [Cites] Dis Colon Rectum. 1978 Oct;21(7):474-9 [710237.001]
  • [Cites] S Afr Med J. 1972 Sep 2;46(35):1241-6 [4638220.001]
  • [Cites] Br J Cancer. 1975 Apr;31(4):485-91 [1156529.001]
  • [Cites] Fam Cancer. 2003;2(1):43-55 [14574166.001]
  • [Cites] Clin Genet. 2002 Mar;61(3):214-7 [12000364.001]
  • [Cites] Gut. 1981 Aug;22(8):653-7 [7286782.001]
  • (PMID = 20126328.001).
  • [ISSN] 0016-9560
  • [Journal-full-title] Ghana medical journal
  • [ISO-abbreviation] Ghana Med J
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Ghana
  • [Other-IDs] NLM/ PMC2810243
  • [Keywords] NOTNLM ; Nigeria / Polyposis coli / familial adenomatous polyposis / malignancy
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