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1. Dromain C, Baudin E: [Endocrine pancreas]. J Radiol; 2005 Jun;86(6 Pt 2):797-804; quiz 805
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Endocrine pancreas].
  • [Transliterated title] Pancréas endocrine.
  • Pancreatic endocrine tumors (PET) are characterised by their hormone synthesis capability and can be associated with an hereditary syndrome-related cancer.
  • [MeSH-minor] Adenoma, Islet Cell / diagnosis. Carcinoma, Islet Cell / diagnosis. Endosonography. Humans. Magnetic Resonance Imaging. Positron-Emission Tomography. Somatostatin. Tomography, X-Ray Computed

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  • (PMID = 16142073.001).
  • [ISSN] 0221-0363
  • [Journal-full-title] Journal de radiologie
  • [ISO-abbreviation] J Radiol
  • [Language] fre
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] France
  • [Chemical-registry-number] 51110-01-1 / Somatostatin
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2. Hsieh MS, Liu KL, Tien YW, Shun CT: Combined pancreatic endocrine tumor and serous cystadenoma. J Formos Med Assoc; 2009 Sep;108(9):739-45
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  • [Title] Combined pancreatic endocrine tumor and serous cystadenoma.
  • Pancreatic serous cystadenomas account for 1-2% of all exocrine pancreatic tumors, and endocrine tumors account for 1-2% of all pancreatic neoplasms.
  • The combination of pancreatic serous cystadenoma and endocrine tumor is even rarer.
  • Here, we report two cases of combined pancreatic serous adenoma and endocrine tumor.
  • One was a 64-year-old woman with serous cystadenoma and pancreatic endocrine tumor.
  • The other case was a 28-year-old woman with von Hippel-Lindau disease with combined pancreatic serous oligocystic adenoma and well-differentiated malignant endocrine carcinoma.
  • Careful examination of benign serous cystadenoma should be kept in mind during clinical practice, to rule out the possibility of combined malignant endocrine tumor.
  • In addition, von Hippel-Lindau disease should also be suspected when a young adult presents with combination of pancreatic serous cystadenoma and endocrine tumor.

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  • (PMID = 19773214.001).
  • [ISSN] 0929-6646
  • [Journal-full-title] Journal of the Formosan Medical Association = Taiwan yi zhi
  • [ISO-abbreviation] J. Formos. Med. Assoc.
  • [Language] ENG
  • [Publication-type] Case Reports; Journal Article; Review
  • [Publication-country] Singapore
  • [Number-of-references] 15
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3. Hemminki K, Försti A, Ji J: Incidence and familial risks in pituitary adenoma and associated tumors. Endocr Relat Cancer; 2007 Mar;14(1):103-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Incidence and familial risks in pituitary adenoma and associated tumors.
  • The incidence of pituitary adenoma has increased moderately from 1958 to the 1990s and declined thereafter.
  • Parental skin cancer (SIR 1.60) and leukemia (1.90, chronic lymphatic leukemia 2.59) were associated with offspring pituitary adenoma diagnosed at any age up to 70 years.
  • The risk of pituitary adenoma was marginally increased in individuals whose siblings were diagnosed with colorectal cancer.
  • Whether these associations can be explained by the recently identified pituitary adenoma predisposing gene, AIP, remains to be established.
  • [MeSH-major] Adenoma / epidemiology. Genetic Predisposition to Disease. Pituitary Neoplasms / epidemiology

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  • (PMID = 17395979.001).
  • [ISSN] 1351-0088
  • [Journal-full-title] Endocrine-related cancer
  • [ISO-abbreviation] Endocr. Relat. Cancer
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
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4. Rust DW, Bianchi DW: Microchimerism in endocrine pathology. Endocr Pathol; 2009;20(1):11-6
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  • [Title] Microchimerism in endocrine pathology.
  • Many of the diseases associated with microchimerism affect the endocrine system (e.g., autoimmune thyroid disease and diabetes mellitus type 1).
  • Microchimerism is relevant to endocrine pathology because (a) it is associated with pregnancy, a condition of complex endocrine physiology;.
  • (b) materno-fetal and feto-maternal cellular migration must involve the placenta, itself an endocrine organ; and (c) in some species, chimerism results in states of intersexuality, a condition intimately involved with endocrine physiology.
  • Studies of feto-maternal microchimerism in the thyroid have documented the presence of fetal cells in association with Hashimoto thyroiditis, Graves' disease, thyroid adenoma, and papillary thyroid carcinoma.
  • [MeSH-major] Chimerism. Endocrine System Diseases / genetics

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  • (PMID = 19214801.001).
  • [ISSN] 1046-3976
  • [Journal-full-title] Endocrine pathology
  • [ISO-abbreviation] Endocr. Pathol.
  • [Language] eng
  • [Grant] United States / NICHD NIH HHS / HD / R01 HD049469
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 29
  • [Other-IDs] NLM/ NIHMS694640; NLM/ PMC4459518
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5. Liu ZX, Yuan XR, Fang JS, Huang J, Li YB, Luo C, Yang ZQ, Liu YS: [Mononostril-septum-transsphenoidal approach for pituitary adenoma]. Zhong Nan Da Xue Xue Bao Yi Xue Ban; 2006 Apr;31(2):281-3
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Mononostril-septum-transsphenoidal approach for pituitary adenoma].
  • OBJECTIVE: To summarize the mononostril-septum-transsphenoidal approach for pituitary adenoma.
  • METHODS: The clinical features, operative techniques, and outcome of 36 patients with pituitary adenoma were analyzed retrospectively.
  • Endocrine symptom of 31 patients returned to the normal level, the symptom of the other 5 cases were improved.
  • [MeSH-major] Adenoma / surgery. Nasal Septum / surgery. Pituitary Neoplasms / surgery. Sphenoid Sinus / surgery

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  • (PMID = 16706134.001).
  • [ISSN] 1672-7347
  • [Journal-full-title] Zhong nan da xue xue bao. Yi xue ban = Journal of Central South University. Medical sciences
  • [ISO-abbreviation] Zhong Nan Da Xue Xue Bao Yi Xue Ban
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] China
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6. Zhu X, Zhai H, Tang SF, Cheng Y: Intrathyroidal parathyroid adenoma presenting with neuromuscular manifestation. Neurol India; 2009 May-Jun;57(3):340-3

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Intrathyroidal parathyroid adenoma presenting with neuromuscular manifestation.
  • Primary hyperparathyroidism (PHPT) is not an uncommon endocrine disorder.
  • The right lobectomy examination confirmed the diagnosis of an intrathyroidal parathyroid adenoma of the right gland.

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  • (PMID = 19587481.001).
  • [ISSN] 0028-3886
  • [Journal-full-title] Neurology India
  • [ISO-abbreviation] Neurol India
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] India
  • [Chemical-registry-number] 0 / Radiopharmaceuticals; 971Z4W1S09 / Technetium Tc 99m Sestamibi
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7. Pawlikowski M, Gruszka A, Kurnatowska I, Winczyk K, Kunert-Radek J, Radek A: Proliferating cell nuclear antigen (PCNA) expression in pituitary adenomas: relationship to the endocrine phenotype of adenoma. Folia Histochem Cytobiol; 2006;44(1):37-41
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  • [Title] Proliferating cell nuclear antigen (PCNA) expression in pituitary adenomas: relationship to the endocrine phenotype of adenoma.
  • Since proliferation rate is an important factor determining the tumor aggressiveness, the evaluation of PCNA index (the percentage of PCNA-immunopositive nuclei in the investigated tumor sample) is suggested as useful in predicting pituitary adenoma outcome.
  • This value was significantly different in comparison to other adenoma subtypes including corticotropinomas manifesting themselves by Cushing's disease.

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  • (PMID = 16584090.001).
  • [ISSN] 0239-8508
  • [Journal-full-title] Folia histochemica et cytobiologica
  • [ISO-abbreviation] Folia Histochem. Cytobiol.
  • [Language] ENG
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Poland
  • [Chemical-registry-number] 0 / Follicle Stimulating Hormone, Human; 0 / Gonadotropins; 0 / Proliferating Cell Nuclear Antigen; 9002-60-2 / Adrenocorticotropic Hormone; 9002-62-4 / Prolactin; 9002-67-9 / Luteinizing Hormone
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8. Coyne JD, O'Connor B: Mixed adenoma-endocrine tumour of the stomach. Histopathology; 2010 Sep;57(3):492-4
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Mixed adenoma-endocrine tumour of the stomach.
  • [MeSH-major] Adenoma / pathology. Endocrine Gland Neoplasms / pathology. Neoplasms, Complex and Mixed / pathology. Stomach Neoplasms / pathology

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  • (PMID = 20840678.001).
  • [ISSN] 1365-2559
  • [Journal-full-title] Histopathology
  • [ISO-abbreviation] Histopathology
  • [Language] eng
  • [Publication-type] Case Reports; Letter
  • [Publication-country] England
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9. Bakhos D, Lescanne E, Fetissof F, Robier A, Morinière S: Neuro-endocrine adenoma of the middle ear: a case study. Eur Arch Otorhinolaryngol; 2007 Dec;264(12):1525-8

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Neuro-endocrine adenoma of the middle ear: a case study.
  • Microscopic examination and immunohistochemistry revealed an endocrine adenoma of the middle ear.
  • The adenoma is composed of two cellular types: neuroendocrine (which closely resemble carcinoid tumors) and glandular.
  • [MeSH-major] Adenoma / pathology. Adenoma / surgery. Ear Neoplasms / pathology. Ear Neoplasms / surgery. Ear, Middle

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  • [Cites] Laryngoscope. 1983 Aug;93(8):1041-4 [6308369.001]
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  • (PMID = 17639440.001).
  • [ISSN] 0937-4477
  • [Journal-full-title] European archives of oto-rhino-laryngology : official journal of the European Federation of Oto-Rhino-Laryngological Societies (EUFOS) : affiliated with the German Society for Oto-Rhino-Laryngology - Head and Neck Surgery
  • [ISO-abbreviation] Eur Arch Otorhinolaryngol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Germany
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10. Târcoveanu E, Moldovanu R, Georgescu S, Niculescu D, Lupaşcu C, Dimofte G: [Pancreatic endocrine tumors]. Chirurgia (Bucur); 2006 Mar-Apr;101(2):175-81
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Pancreatic endocrine tumors].
  • [Transliterated title] Tumori pancreatice endocrine.
  • Incidence of the endocrine tumors of the pancreas is about 4 to 10/1.000.000 peoples.
  • We present 10 cases of endocrine pancreatic tumors which were operated in the First Surgical Clinic Iaşi in the last 20 years (1984-2003); these cases represent about 2.21% from all the pancreatic tumors (454 cases).
  • It was 4 insulinoma, 2 gastrinoma, 2 gastrinoma associated with other endocrine neoplasia (Wermer syndrome) and 2 non-functioning endocrine pancreatic tumors.
  • Both patients died and diagnosis of pancreatic endocrine tumors was post-mortem.
  • The two patients with Wermer syndrome also had ulcers complicated with haemorrhage and peritonitis and parathyroid adenoma.
  • One case also had ante-hypophyseal and pituitary adenoma and the other had thyroid colloid hypertrophy.
  • The two cases of non-functioning pancreatic endocrine tumors had a non-specific symptoms.
  • [MeSH-minor] Adult. Aged. Female. Humans. Male. Medical Records. Middle Aged. Multiple Endocrine Neoplasia Type 1 / diagnosis. Pancreatectomy. Retrospective Studies. Romania. Splenectomy. Survival Analysis. Zollinger-Ellison Syndrome / diagnosis

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  • (PMID = 16752684.001).
  • [ISSN] 1221-9118
  • [Journal-full-title] Chirurgia (Bucharest, Romania : 1990)
  • [ISO-abbreviation] Chirurgia (Bucur)
  • [Language] rum
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] Romania
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11. Târcoveanu E, Niculescu D, Cotea E, Moldovanu R, Vasilescu A, Crumpei F, Zbranca E, Zugun F, Rusu V, Ferariu D: [Parathyroid glands involvement in multiple endocrine neoplasia]. Rev Med Chir Soc Med Nat Iasi; 2009 Apr-Jun;113(2):482-96
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Parathyroid glands involvement in multiple endocrine neoplasia].
  • [Transliterated title] Interesarea paratiroidelor in neoplaziile endocrine multiple.
  • Multiple endocrine neoplasias are syndromes characterized by the involvement of at least two endocrine glands.
  • Parathyroid gland involvement is usually noted in Multiple Endocrine Neoplasia (MEN) type I and type II.
  • Parathyroid glands tumor associated with endocrine pancreatic tumor, as well as pituitary tumors is the typical pattern of MEN I.
  • In three cases with young ages (28-33 years old) and familial setting, the MEN I syndrome was "complete" (parathyroid adenoma, gastrinoma or insulinoma and pituitary adenoma--prolactinoma or GH-secreting tumors), and, in the other two cases, with 57 and 68 years old respectively, the MEN I syndrome was "incomplete" with parathyroid glands and pituitary gland involvement.
  • Unfortunately one patients died due to severe endocrine disorder.
  • During the necropsy, the pituitary adenoma has been diagnosed.
  • The imagistic and laboratory test diagnosed a tumor situated into the pancreatic body, and an parathyroid adenoma.
  • The resection of pancreatic tumor associated with resection of the parathyroid adenoma, in the same time, were performed.
  • The resection of parathyroid gland adenoma has been performed in both cases, with uneventful postoperative course.
  • There are two kinds of MEN associated with parathyroid gland involvement: the "complete" form, especially in young patients, with diffuse involvement of the parathyroid glands, and the subtotal parathyroidy is the best choice, and the "incomplete" form, especially in elderly, with the involvement of a single parathyroid gland; in this way, the resection of the adenoma associated with biopsy from the other parathyroid gland is the best approach.
  • [MeSH-major] Adenoma / surgery. Gastrinoma / surgery. Insulinoma / surgery. Multiple Endocrine Neoplasia Type 1 / surgery. Pancreatectomy. Pancreatic Neoplasms / surgery. Parathyroid Neoplasms / surgery. Parathyroidectomy
  • [MeSH-minor] Adult. Aged. Fatal Outcome. Female. Humans. Hyperparathyroidism, Secondary / diagnosis. Hyperparathyroidism, Secondary / etiology. Male. Middle Aged. Multiple Endocrine Neoplasia / surgery. Pituitary Neoplasms / diagnosis. Pituitary Neoplasms / surgery. Prolactinoma / diagnosis. Prolactinoma / surgery. Treatment Outcome

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  • (PMID = 21495355.001).
  • [ISSN] 0048-7848
  • [Journal-full-title] Revista medico-chirurgicală̆ a Societă̆ţ̜ii de Medici ş̧i Naturaliş̧ti din Iaş̧i
  • [ISO-abbreviation] Rev Med Chir Soc Med Nat Iasi
  • [Language] rum
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Romania
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12. Amar L, Lepoutre C, Bobrie G, Plouin PF: [Endocrine hypertension]. Rev Med Interne; 2010 Oct;31(10):697-704
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Endocrine hypertension].
  • [Transliterated title] Hypertension artérielle endocrine.
  • Endocrine hypertension represents more than half of the causes of secondary hypertension.
  • The screening of endocrine hypertension should be performed in all the patients presenting with:.
  • (3) the coexistence of hypertension with an adrenal adenoma, clinical or biological abnormalities.
  • However, the diagnosis of endocrine hypertension allows diagnosing surgical correctable form of hypertension, which is not possible in essential hypertension.

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  • [Copyright] Copyright © 2010 Société nationale française de médecine interne (SNFMI). Published by Elsevier SAS. All rights reserved.
  • (PMID = 20674105.001).
  • [ISSN] 1768-3122
  • [Journal-full-title] La Revue de medecine interne
  • [ISO-abbreviation] Rev Med Interne
  • [Language] FRE
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] France
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13. Raica M, Coculescu M, Cimpean AM, Ribatti D: Endocrine gland derived-VEGF is down-regulated in human pituitary adenoma. Anticancer Res; 2010 Oct;30(10):3981-6
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Endocrine gland derived-VEGF is down-regulated in human pituitary adenoma.
  • BACKGROUND: Endocrine gland-derived vascular endothelial growth factor (EG-VEGF) is an angiogenic molecule restricted to endocrine glands and, particularly, to steroid-secreting cells.
  • MATERIALS AND METHODS: In this study, we investigated by immunohistochemistry the expression of EG-VEGF in 2 samples of normal adenohypophysis and 43 bioptic samples of pituitary adenoma.
  • RESULTS: The results of this study for the first time demonstrate a down-regulation of EG-VEGF expression in human pituitary adenoma as compared to normal adenohypophysis, suggesting an impaired function of the neoplastic cells in terms of hormone release in the blood stream, as a consequence of impaired tumor angiogenesis in the tumor.
  • CONCLUSION: On the basis of our data showing a marked decrease in the expression of EG-VEGF in pituitary adenoma, with the exception of LH-secreting adenomas, we suggest that LH might be involved in the induction of EG-VEGF secretion.
  • [MeSH-major] Adenoma / metabolism. Pituitary Neoplasms / metabolism. Vascular Endothelial Growth Factor, Endocrine-Gland-Derived / biosynthesis

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  • (PMID = 21036711.001).
  • [ISSN] 1791-7530
  • [Journal-full-title] Anticancer research
  • [ISO-abbreviation] Anticancer Res.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Greece
  • [Chemical-registry-number] 0 / Vascular Endothelial Growth Factor, Endocrine-Gland-Derived; 12629-01-5 / Human Growth Hormone; 9002-60-2 / Adrenocorticotropic Hormone; 9002-62-4 / Prolactin; 9002-67-9 / Luteinizing Hormone; 9002-68-0 / Follicle Stimulating Hormone; 9002-71-5 / Thyrotropin
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14. Hong JW, Lee MK, Kim SH, Lee EJ: Discrimination of prolactinoma from hyperprolactinemic non-functioning adenoma. Endocrine; 2010 Feb;37(1):140-7
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  • [Title] Discrimination of prolactinoma from hyperprolactinemic non-functioning adenoma.
  • (B) prolactinoma requiring surgical treatment (PRS); and (C) non-functioning pituitary adenoma with hyperprolactinemia (NFPAH).
  • In conclusion, old age, extrasellar tumor extension with relatively low prolactin levels, visual defect, and GH deficiency were considered suggestive of non-functioning pituitary adenoma rather than prolactinoma in hyperprolactinemic pituitary macroadenoma.
  • [MeSH-major] Adenoma / blood. Adenoma / diagnosis. Hyperprolactinemia / etiology. Pituitary Neoplasms / blood. Pituitary Neoplasms / diagnosis. Prolactinoma / diagnosis

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  • (PMID = 20963563.001).
  • [ISSN] 1559-0100
  • [Journal-full-title] Endocrine
  • [ISO-abbreviation] Endocrine
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Dopamine Agonists; 9002-62-4 / Prolactin
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15. Schittenhelm J, Ebner FH, Harter P, Bornemann A: Symptomatic intraspinal oncocytic adrenocortical adenoma. Endocr Pathol; 2009;20(1):73-7
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Symptomatic intraspinal oncocytic adrenocortical adenoma.
  • The diagnosis of an oncocytic adrenal cortical adenoma was made.
  • [MeSH-major] Adenoma, Oxyphilic / pathology. Adrenocortical Adenoma / pathology. Spinal Cord Neoplasms / pathology

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  • (PMID = 19039533.001).
  • [ISSN] 1046-3976
  • [Journal-full-title] Endocrine pathology
  • [ISO-abbreviation] Endocr. Pathol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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16. Tinnel BA, Henderson MA, Witt TC, Fakiris AJ, Worth RM, Des Rosiers PM, Edmondson JW, Timmerman RD, Lo SS: Endocrine response after gamma knife-based stereotactic radiosurgery for secretory pituitary adenoma. Stereotact Funct Neurosurg; 2008;86(5):292-6
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  • [Title] Endocrine response after gamma knife-based stereotactic radiosurgery for secretory pituitary adenoma.
  • For adrenocorticotrophic hormone-secreting tumors, 6 of 12 patients (50%) showed normalization of their endocrine levels at a median of 10 months.
  • For growth hormone-secreting tumors, 4 of 9 patients (44%) showed normalization of endocrine levels at a median time of 30 months.
  • Two patients (22%) had >or=50% lower but abnormal endocrine levels.
  • CONCLUSION: GK-based SRS provides a reasonable rate of endocrine normalization of secretory pituitary adenoma.
  • The time to endocrine response is shorter than reported for fractionated external beam radiotherapy.
  • [MeSH-minor] ACTH-Secreting Pituitary Adenoma / secretion. ACTH-Secreting Pituitary Adenoma / surgery. Adenoma / secretion. Adenoma / surgery. Adrenocorticotropic Hormone / secretion. Adult. Aged. Aged, 80 and over. Female. Follow-Up Studies. Growth Hormone-Secreting Pituitary Adenoma / secretion. Growth Hormone-Secreting Pituitary Adenoma / surgery. Human Growth Hormone / secretion. Humans. Hydrocortisone / blood. Insulin-Like Growth Factor I / metabolism. Male. Middle Aged. Retrospective Studies. Treatment Outcome

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  • [Copyright] Copyright 2008 S. Karger AG, Basel.
  • (PMID = 18758206.001).
  • [ISSN] 1423-0372
  • [Journal-full-title] Stereotactic and functional neurosurgery
  • [ISO-abbreviation] Stereotact Funct Neurosurg
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 12629-01-5 / Human Growth Hormone; 67763-96-6 / Insulin-Like Growth Factor I; 9002-60-2 / Adrenocorticotropic Hormone; 9002-62-4 / Prolactin; WI4X0X7BPJ / Hydrocortisone
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17. Al-Brahim N, Asa S: Myelolipoma with adrenocortical adenoma: an unusual combination that can resemble carcinoma. Endocr Pathol; 2007;18(2):103-5
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  • [Title] Myelolipoma with adrenocortical adenoma: an unusual combination that can resemble carcinoma.
  • It has very rarely been reported to occur in association with adrenocortical adenoma.
  • In this paper, we report three cases of adrenal myelolipoma associated with adrenocortical adenoma; in all three patients, the radiological appearance resembled adrenocortical carcinoma.
  • [MeSH-major] Adenoma / pathology. Adrenal Cortex Neoplasms / pathology. Carcinoma / pathology. Myelolipoma / pathology

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  • (PMID = 17917001.001).
  • [ISSN] 1046-3976
  • [Journal-full-title] Endocrine pathology
  • [ISO-abbreviation] Endocr. Pathol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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18. Ballian N, Chrisoulidou A, Nomikos P, Samara C, Kontogeorgos G, Kaltsas GA: Hypophysitis superimposed on a non-functioning pituitary adenoma: diagnostic clinical, endocrine, and radiologic features. J Endocrinol Invest; 2007 Sep;30(8):677-83
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  • [Title] Hypophysitis superimposed on a non-functioning pituitary adenoma: diagnostic clinical, endocrine, and radiologic features.
  • We describe herein a 38-yr-old woman who presented with a pituitary lesion that was considered to be a non-functioning pituitary adenoma.
  • Repeated endocrine investigation revealed partial anterior pituitary insufficiency.
  • The patient underwent trans-sphenoidal resection of the pituitary lesion; histology revealed a null cell pituitary adenoma and lymphocytic hypophysitis (LYH) of the non-neoplastic adenohypophysial gland.
  • This case illustrates the effects of new-onset LYH in a patient with a pre-existing non-functioning pituitary adenoma.
  • [MeSH-major] Adenoma / pathology. Hypopituitarism / pathology. Inflammation. Magnetic Resonance Imaging. Pituitary Neoplasms / pathology

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  • (PMID = 17923800.001).
  • [ISSN] 1720-8386
  • [Journal-full-title] Journal of endocrinological investigation
  • [ISO-abbreviation] J. Endocrinol. Invest.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Italy
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19. Sztal-Mazer S, Topliss DJ, Simpson RW, Hamblin PS, Rosenfeld JV, McLean CA: Gonadotroph adenoma in multiple endocrine neoplasia type 1. Endocr Pract; 2008 Jul-Aug;14(5):592-4
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  • [Title] Gonadotroph adenoma in multiple endocrine neoplasia type 1.
  • OBJECTIVE: To describe a case of an in vivo follicle-stimulating hormone (FSH)-secreting gonadotroph adenoma in a man with multiple endocrine neoplasia type 1 (MEN 1) syndrome.
  • A family history of endocrine neoplasia was obtained of one sibling with a nonfunctioning pituitary adenoma, another sibling who had died of pancreatic carcinoma, and a third sibling, along with her son, who has primary hyperparathyroidism.
  • CONCLUSION: We report an unusual case of clearly high circulating immunoreactive FSH due to a functioning FSH-secreting gonadotroph adenoma in a man with the MEN 1 syndrome.
  • [MeSH-major] Adenoma / blood. Multiple Endocrine Neoplasia Type 1 / blood. Pituitary Neoplasms / blood


20. Scheithauer BW, Silva AI, Atkinson JL, Nippoldt TB, Kaufmann TJ, Kovacs K, Horvath E, Lloyd R: Pituitary adenoma with tumoral granulomatous reaction. Endocr Pathol; 2007;18(2):86-90
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  • [Title] Pituitary adenoma with tumoral granulomatous reaction.
  • Herein, we report a unique case of an adult male with a corticotrophic pituitary adenoma of silent subtype 1 exhibiting conspicuous idiopathic tumoral noncaseating granulomatous inflammation.
  • [MeSH-major] ACTH-Secreting Pituitary Adenoma / pathology. Adenoma / pathology. Granuloma / pathology. Pituitary Neoplasms / pathology

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  • (PMID = 17916998.001).
  • [ISSN] 1046-3976
  • [Journal-full-title] Endocrine pathology
  • [ISO-abbreviation] Endocr. Pathol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Pituitary Hormones; 3XMK78S47O / Testosterone; 9002-71-5 / Thyrotropin; Q51BO43MG4 / Thyroxine
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21. Falchetti A, Marini F, Luzi E, Tonelli F, Brandi ML: Multiple endocrine neoplasms. Best Pract Res Clin Rheumatol; 2008 Mar;22(1):149-63
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Multiple endocrine neoplasms.
  • Multiple endocrine neoplasia type 1 (MEN1) and type 2 (MEN2) are rare autosomal-dominant disorders characterized by primary tumours in at least two different endocrine tissues.
  • Both syndromes present as sporadic (a single case with two of the characteristic endocrine tumours) or familial form (an MEN case plus at least one first-degree relative showing one of the characteristic endocrine tumours).
  • MEN1 is characterized by the occurrence of parathyroid, gastro-entero-pancreatic and anterior pituitary tumours, but it can include various combinations of more than 20 endocrine and non-endocrine tumours.
  • MEN2 is characterized by medullary thyroid carcinoma (MTC), uni- or bi-lateral pheochromocytoma, and other tumours of different endocrine tissues.
  • [MeSH-major] Multiple Endocrine Neoplasia / genetics
  • [MeSH-minor] Adenoma / genetics. Adrenocortical Carcinoma / genetics. Gastrointestinal Neoplasms / genetics. Humans. Pancreatic Neoplasms / genetics. Pheochromocytoma / genetics. Pituitary Neoplasms / genetics. Proto-Oncogene Proteins / genetics. Proto-Oncogene Proteins c-ret / genetics

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  • [ErratumIn] Best Pract Res Clin Rheumatol. 2008 Dec;22(6):III. Brandt, Maria Luisa [corrected to Brandi, Maria Luisa]
  • (PMID = 18328987.001).
  • [ISSN] 1521-6942
  • [Journal-full-title] Best practice & research. Clinical rheumatology
  • [ISO-abbreviation] Best Pract Res Clin Rheumatol
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / MEN1 protein, human; 0 / Proto-Oncogene Proteins; EC 2.7.10.1 / Proto-Oncogene Proteins c-ret
  • [Number-of-references] 68
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22. Kandil E, Alabbas HH, Lum YW, Tufaro AP: Familial isolated primary hyperparathyroidism with double adenoma. South Med J; 2010 Mar;103(3):236-8
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  • [Title] Familial isolated primary hyperparathyroidism with double adenoma.
  • Familial hyperparathyroidism includes the diagnoses of multiple endocrine neoplasia type 1, type 2A, and familial isolated primary hyperparathyroidism.
  • Familial isolated primary hyperparathyroidism is a rare, distinct form of familial primary hyperparathyroidism, mainly due to four-gland hyperplasia or single-gland adenoma.
  • We describe our success in treating a 24-year-old woman with familial isolated primary hyperparathyroidism with resection of double adenoma, using the guide of intraoperative parathyroid hormone (PTH) monitoring.
  • Familial isolated primary hyperparathyroidism usually presents with four-gland hyperplasia or single-gland adenoma.
  • However, double adenoma should be considered in the differential diagnosis.
  • [MeSH-major] Adenoma / genetics. Hyperparathyroidism, Primary / genetics. Parathyroid Neoplasms / genetics. Pedigree


23. Ramachandra C, Manjunath S, Joseph B, Appaji L, Kumari BS, Rao CR, Prabhakaran PS: Mixed exocrine-endocrine pancreatic carcinoma in childhood. Indian J Gastroenterol; 2005 May-Jun;24(3):116
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  • [Title] Mixed exocrine-endocrine pancreatic carcinoma in childhood.
  • A 7-year-old boy with mixed exocrine-endocrine pancreatic cancer is presented.
  • [MeSH-major] Adenoma, Islet Cell / epidemiology. Carcinoma, Acinar Cell / epidemiology. Pancreatic Neoplasms / epidemiology

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  • (PMID = 16041106.001).
  • [ISSN] 0254-8860
  • [Journal-full-title] Indian journal of gastroenterology : official journal of the Indian Society of Gastroenterology
  • [ISO-abbreviation] Indian J Gastroenterol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Review
  • [Publication-country] India
  • [Number-of-references] 5
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24. Zhang W, Tang ZY, Wang WQ, Ning G: [Metabolic syndrome in patients with adrenocortical adenoma]. Zhonghua Yi Xue Za Zhi; 2006 Dec 26;86(48):3397-400
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  • [Title] [Metabolic syndrome in patients with adrenocortical adenoma].
  • OBJECTIVE: To investigate the prevalence and characteristics of metabolic syndrome (MS) in patients of adrenocortical adenoma.
  • METHODS: MS-related indexes, including waist circumference (WC), blood pressure, triglyceride (TG), high-density lipoprotein cholesterol (HDL-C), fasting plasma glucose, plasma and urinary aldosterone, plasma potassium, basic and upright plasma renin activity (PRA), plasma and urinary cortisol, and plasma ACTH, were examined among 61 patients of aldosterone producing adenoma (APA, Group 1), 23 patients of cortisol-secreting adenoma (CSA, Group 2), 24 patients of nonfunctional adrenal adenoma (NAA, Group 3), and 26 healthy controls (Group 4).
  • CONCLUSION: Patients with adrenocortical adenoma have a significantly high prevalence of metabolic syndrome, especially the patients of CSA and NAA.
  • [MeSH-major] Adrenocortical Adenoma / pathology. Metabolic Syndrome X / pathology

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  • (PMID = 17313850.001).
  • [ISSN] 0376-2491
  • [Journal-full-title] Zhonghua yi xue za zhi
  • [ISO-abbreviation] Zhonghua Yi Xue Za Zhi
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] China
  • [Chemical-registry-number] 0 / Blood Glucose; 0 / Lipids; 4964P6T9RB / Aldosterone; 9002-60-2 / Adrenocorticotropic Hormone; WI4X0X7BPJ / Hydrocortisone
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25. Rocque BG, Herold KA, Salamat MS, Shenker Y, Kuo JS: Symptomatic hyperprolactinemia from an ectopic pituitary adenoma located in the clivus. Endocr Pract; 2009 Mar;15(2):143-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Symptomatic hyperprolactinemia from an ectopic pituitary adenoma located in the clivus.
  • OBJECTIVE: To report a case of an ectopic pituitary adenoma in the clivus.
  • After the resected tissue was examined, the patient was diagnosed as having an ectopic prolactin-producing pituitary adenoma.

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  • (PMID = 19289326.001).
  • [ISSN] 1934-2403
  • [Journal-full-title] Endocrine practice : official journal of the American College of Endocrinology and the American Association of Clinical Endocrinologists
  • [ISO-abbreviation] Endocr Pract
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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26. Zogheri A, Di Mambro A, Mannelli M, Serio M, Forti G, Peri A: Hyponatremia and pituitary adenoma: think twice about the etiopathogenesis. J Endocrinol Invest; 2006 Sep;29(8):750-3
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Hyponatremia and pituitary adenoma: think twice about the etiopathogenesis.
  • This case very well demonstrates that, in the presence of hyponatremia due to SIADH, more frequently associated co-morbidities (ie mediastinic diseases) have to be searched, even in the presence of a possible, yet rare, cause of this syndrome (ie pituitary adenoma).

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  • (PMID = 17033267.001).
  • [ISSN] 1720-8386
  • [Journal-full-title] Journal of endocrinological investigation
  • [ISO-abbreviation] J. Endocrinol. Invest.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Italy
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27. Lewis RB, Lattin GE Jr, Paal E: Pancreatic endocrine tumors: radiologic-clinicopathologic correlation. Radiographics; 2010 Oct;30(6):1445-64
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  • [Title] Pancreatic endocrine tumors: radiologic-clinicopathologic correlation.
  • Pancreatic endocrine tumors (PETs) are primarily well-differentiated tumors composed of cells that resemble normal islet cells but that arise from pancreatic ductal cells.
  • Most are sporadic, but some are associated with familial syndromes such as multiple endocrine neoplasia type 1, von Hippel-Lindau syndrome, and neurofibromatosis type 1.
  • [MeSH-minor] Adenoma, Islet Cell / diagnosis. Adenoma, Islet Cell / epidemiology. Adenoma, Islet Cell / pathology. Carcinoma, Islet Cell / diagnosis. Carcinoma, Islet Cell / epidemiology. Carcinoma, Islet Cell / pathology. Diagnosis, Differential. Humans. Multiple Endocrine Neoplasia Type 1 / pathology. Neurofibromatosis 1 / pathology. Prevalence. von Hippel-Lindau Disease / pathology

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  • [Copyright] © RSNA, 2010.
  • (PMID = 21071369.001).
  • [ISSN] 1527-1323
  • [Journal-full-title] Radiographics : a review publication of the Radiological Society of North America, Inc
  • [ISO-abbreviation] Radiographics
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
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28. Frankel WL: Update on pancreatic endocrine tumors. Arch Pathol Lab Med; 2006 Jul;130(7):963-6
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  • [Title] Update on pancreatic endocrine tumors.
  • Endocrine tumors of the pancreas represent 1% to 2% of all pancreatic neoplasms.
  • It is important to be aware that unusual morphologic variants of pancreatic endocrine tumors are common, and immunohistochemical stains can help avoid misdiagnosis.
  • [MeSH-major] Adenoma, Islet Cell / pathology. Carcinoma, Islet Cell / pathology. Pancreatic Neoplasms / pathology

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  • (PMID = 16831051.001).
  • [ISSN] 1543-2165
  • [Journal-full-title] Archives of pathology & laboratory medicine
  • [ISO-abbreviation] Arch. Pathol. Lab. Med.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 26
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29. Daly AF, Tichomirowa MA, Beckers A: Update on familial pituitary tumors: from multiple endocrine neoplasia type 1 to familial isolated pituitary adenoma. Horm Res; 2009 Jan;71 Suppl 1:105-11
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Update on familial pituitary tumors: from multiple endocrine neoplasia type 1 to familial isolated pituitary adenoma.
  • BACKGROUND: Pituitary adenomas occur in a familial setting in about 5% of all cases and over half of these are due to multiple endocrine neoplasia type 1 (MEN1) and Carney complex (CNC).
  • Since the late 1990s, we have described non-MEN1/CNC familial pituitary tumors that include all tumor phenotypes and have named this condition 'familial isolated pituitary adenoma' (FIPA).
  • [MeSH-major] Adenoma / etiology. Multiple Endocrine Neoplasia Type 1 / complications. Pituitary Neoplasms / etiology


30. Bäcklund LM, Grandér D, Brandt L, Hall P, Ekbom A: Parathyroid adenoma and primary CNS tumors. Int J Cancer; 2005 Mar 1;113(6):866-9

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Parathyroid adenoma and primary CNS tumors.
  • Hyperparathyroidism onset at a young age is one feature in multiple endocrine neoplasia (MEN) type 1 and MEN type 2A cancer syndromes.
  • There were 70 observed cases of a CNS tumor diagnosed after a parathyroid adenoma, to be compared to 35 expected (standard incidence ratio [SIR] = 2.0; 95% confidence interval [CI] = 1.5-2.5).
  • [MeSH-major] Adenoma / epidemiology. Central Nervous System Neoplasms / epidemiology. Parathyroid Neoplasms / epidemiology

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  • [Copyright] (c) 2004 Wiley-Liss, Inc.
  • (PMID = 15515018.001).
  • [ISSN] 0020-7136
  • [Journal-full-title] International journal of cancer
  • [ISO-abbreviation] Int. J. Cancer
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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31. Lonser RR, Kindzelski BA, Mehta GU, Jane JA Jr, Oldfield EH: Acromegaly without imaging evidence of pituitary adenoma. J Clin Endocrinol Metab; 2010 Sep;95(9):4192-6
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Acromegaly without imaging evidence of pituitary adenoma.
  • PATIENTS OR OTHER PARTICIPANTS: Consecutive acromegalic patients without imaging evidence of a pituitary adenoma on pre- and postcontrast, spin echo T1-weighted MR imaging and who lacked evidence of an ectopic (nonpituitary) source causing GH excess were included.
  • INTERVENTIONS: Surgical exploration with identification and resection of a pituitary adenoma was performed.
  • RESULTS: Six patients (three males, three females; 3% of all patients) with suspected GH-secreting adenomas did not demonstrate imaging evidence of pituitary adenoma on conventional MR imaging.
  • Three patients underwent a postcontrast, volumetric interpolated breath-hold examination MR-imaging sequence (1.2-mm slice thickness), which revealed a 4-mm pituitary adenoma not seen on the spin echo T1-weighted MR imaging in one patient.
  • A pituitary adenoma was identified and removed in all patients (mean diameter, 5.6 mm; range, 5 to 6.7 mm).
  • Surgical exploration of the pituitary gland in acromegalic patients with endocrine findings consistent with a GH-secreting adenoma but negative MR imaging can lead to identification and removal of an adenoma.

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  • (PMID = 20610592.001).
  • [ISSN] 1945-7197
  • [Journal-full-title] The Journal of clinical endocrinology and metabolism
  • [ISO-abbreviation] J. Clin. Endocrinol. Metab.
  • [Language] ENG
  • [Grant] United States / Intramural NIH HHS / /
  • [Publication-type] Case Reports; Journal Article; Multicenter Study; Research Support, N.I.H., Intramural
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC2936064
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32. Kamoshima Y, Sawamura Y, Iwasaki YK, Fujieda K, Takahashi H: [Case of Carney complex complicated with pituitary adenoma and Rathke cleft cyst]. No Shinkei Geka; 2008 Jun;36(6):535-9
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  • [Title] [Case of Carney complex complicated with pituitary adenoma and Rathke cleft cyst].
  • This syndrome is multiple neoplasia syndrome featuring cardiac, endocrine, cutaneous, and neural tumor, in addition to a variety of pigmented lesions of the skin and mucosa.
  • In addition to the pituitary adenoma, pathological examination revealed the presence of a Rathke cleft cyst.
  • Therefore, the present case seems to be the first case of Carney Complex complicated with pituitary adenoma and Rathke cleft cyst.
  • [MeSH-major] Adenoma / complications. Angiomyoma / complications. Central Nervous System Cysts / complications. Hyperpigmentation / complications. Multiple Endocrine Neoplasia / complications. Neoplasms, Multiple Primary. Pituitary Neoplasms / complications. Skin Neoplasms / complications

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  • (PMID = 18548895.001).
  • [ISSN] 0301-2603
  • [Journal-full-title] No shinkei geka. Neurological surgery
  • [ISO-abbreviation] No Shinkei Geka
  • [Language] jpn
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / Biomarkers, Tumor
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33. Kouniavsky G, Zeiger MA: The role of telomeres and telomerase in endocrine tumors. Discov Med; 2010 Oct;10(53):340-7

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] The role of telomeres and telomerase in endocrine tumors.
  • Herein we discuss the role of telomerase in normal, benign, and cancerous endocrine tissues.
  • [MeSH-major] Adenoma / etiology. Carcinoma / etiology. Endocrine Gland Neoplasms / etiology. Telomerase / physiology. Telomere / physiology

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  • (PMID = 21034675.001).
  • [ISSN] 1944-7930
  • [Journal-full-title] Discovery medicine
  • [ISO-abbreviation] Discov Med
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Chemical-registry-number] EC 2.7.7.49 / Telomerase
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34. Onoda N, Ishikawa T, Nishio K, Tahara H, Inaba M, Wakasa K, Sumi T, Yamazaki T, Shigematsu K, Hirakawa K: Cushing's syndrome by left adrenocortical adenoma synchronously associated with primary aldosteronism by right adrenocortical adenoma: report of a case. Endocr J; 2009;56(3):495-502
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  • [Title] Cushing's syndrome by left adrenocortical adenoma synchronously associated with primary aldosteronism by right adrenocortical adenoma: report of a case.
  • We diagnosed that the present case had 1) cortisol-producing right adrenocortical adenoma, 2) aldosterone producing left adrenocortical adenoma, and 3) cortical minute nodules with aldosterone production in both adrenal glands compatible with idiopathic adrenal hyperplasia.
  • We reviewed the cases reported, and discussed the significance of the minute nodules in the adrenal cortex, often found in association with the adrenocortical adenoma.
  • [MeSH-major] Adrenal Cortex Neoplasms / complications. Adrenocortical Adenoma / complications. Cushing Syndrome / etiology. Hyperaldosteronism / etiology

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  • (PMID = 19270420.001).
  • [ISSN] 1348-4540
  • [Journal-full-title] Endocrine journal
  • [ISO-abbreviation] Endocr. J.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Japan
  • [Chemical-registry-number] EC 1.1.1.145 / 3 beta-hydroxysteroid dehydrogenase type II; EC 1.1.1.145 / Progesterone Reductase; EC 1.14.15.4 / Steroid 11-beta-Hydroxylase
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35. Tateno T, Izumiyama H, Doi M, Akashi T, Ohno K, Hirata Y: Defective expression of prohormone convertase 1/3 in silent corticotroph adenoma. Endocr J; 2007 Dec;54(5):777-82
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Defective expression of prohormone convertase 1/3 in silent corticotroph adenoma.
  • Silent corticotroph adenoma (SCA) is defined as an ACTH-producing pituitary tumor not associated with clinical and endocrine feartures of Cushing's syndrome, but its underlying molecular mechanism(s) remains unknown thus far.
  • Preoperative endocrine and diagnostic image tests did not reveal any differences between SCA and the remaining NFA except for the higher recurrence rate of SCA.
  • [MeSH-major] ACTH-Secreting Pituitary Adenoma / genetics. Adenoma / genetics. Proprotein Convertase 1 / genetics

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  • (PMID = 17917309.001).
  • [ISSN] 1348-4540
  • [Journal-full-title] Endocrine journal
  • [ISO-abbreviation] Endocr. J.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / Protein Isoforms; 66796-54-1 / Pro-Opiomelanocortin; 9002-60-2 / Adrenocorticotropic Hormone; EC 3.4.21.93 / Proprotein Convertase 1
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36. Sugihara A, Nakasho K, Ikuta S, Aihara T, Kawai T, Iida H, Yoshie H, Yasui C, Mitsunobu M, Kishi K, Mori T, Yamada N, Yamanegi K, Ohyama H, Terada N, Ohike N, Morohoshi T, Yamanaka N: Oncocytic non-functioning endocrine tumor of the pancreas. Pathol Int; 2006 Dec;56(12):755-9
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  • [Title] Oncocytic non-functioning endocrine tumor of the pancreas.
  • Herein is presented the case of a malignant non-functioning endocrine tumor of the pancreas with oncocytic features, and a discussion on the high incidence of malignancy in oncocytic endocrine pancreatic tumors.
  • The patient was a 65-year-old woman who showed no paraneoplastic symptoms produced by functioning pancreatic endocrine tumors.
  • Analysis of 18 oncocytic pancreatic endocrine tumors, consisting of those reported previously and that in the present case, suggests that the high incidence of malignancy in oncocytic endocrine tumors is associated with the high incidence of non-functioning endocrine tumors among them, most of which are malignant.
  • [MeSH-major] Adenoma, Oxyphilic / pathology. Pancreatic Neoplasms / pathology

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  • (PMID = 17096734.001).
  • [ISSN] 1320-5463
  • [Journal-full-title] Pathology international
  • [ISO-abbreviation] Pathol. Int.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Australia
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37. Maser C, Toset A, Roman S: Gastrointestinal manifestations of endocrine disease. World J Gastroenterol; 2006 May 28;12(20):3174-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Gastrointestinal manifestations of endocrine disease.
  • The hormonal interactions among the systems throughout the body are not fully understood; many vague clinical symptoms may in fact be manifestations of underlying endocrine diseases.
  • The aim of the following review is to discuss gastrointestinal manifestations of surgically correctable endocrine diseases, focusing on abnormalities of thyroid function, cancer and finally autoimmune diseases.
  • We also review manifestations of pancreatic endocrine tumors, and multiple endocrine neoplasia.
  • [MeSH-major] Endocrine System Diseases / complications. Gastrointestinal Diseases / etiology. Gastrointestinal Tract / physiopathology
  • [MeSH-minor] Adenoma, Islet Cell / complications. Adenoma, Islet Cell / diagnosis. Adenoma, Islet Cell / physiopathology. Adenoma, Islet Cell / surgery. Humans. Neuroendocrine Tumors / complications. Neuroendocrine Tumors / diagnosis. Neuroendocrine Tumors / physiopathology. Neuroendocrine Tumors / surgery. Thyroid Diseases / complications. Thyroid Diseases / diagnosis. Thyroid Diseases / physiopathology. Thyroid Diseases / surgery

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  • (PMID = 16718836.001).
  • [ISSN] 1007-9327
  • [Journal-full-title] World journal of gastroenterology
  • [ISO-abbreviation] World J. Gastroenterol.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] China
  • [Number-of-references] 58
  • [Other-IDs] NLM/ PMC4087959
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38. Borka K, Kaliszky P, Szabó E, Lotz G, Kupcsulik P, Schaff Z, Kiss A: Claudin expression in pancreatic endocrine tumors as compared with ductal adenocarcinomas. Virchows Arch; 2007 May;450(5):549-57
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Claudin expression in pancreatic endocrine tumors as compared with ductal adenocarcinomas.
  • We aimed to analyze protein and messenger RNA (mRNA) expressions of different CLDNs in human pancreatic endocrine tumors (PET) and ductal adenocarcinomas.
  • CLDN 2 expression was found in the half of ductal adenocarcinomas, while the vast majority of endocrine tumors were negative.
  • CLDN 1, -4, and -7 immunohistochemistry was positive in all adenocarcinomas, whereas endocrine tumors were completely negative for CLDNs 1 and -4.
  • CLDN 3 and -7 proteins were detected in all endocrine tumors, while CLDN 3 in ductal adenocarcinomas was negative.
  • The mRNA expression of CLDNs showed differences between endocrine tumors and ductal adenocarcinomas, similar as found for protein expression.
  • High expressions of CLDN 3 in endocrine tumors and CLDN 4 in ductal carcinomas might attract them as targets for adjuvant therapy.
  • [MeSH-major] Adenoma, Islet Cell / metabolism. Carcinoma, Islet Cell / metabolism. Carcinoma, Pancreatic Ductal / metabolism. Membrane Proteins / metabolism. Pancreatic Neoplasms / metabolism

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  • (PMID = 17429687.001).
  • [ISSN] 0945-6317
  • [Journal-full-title] Virchows Archiv : an international journal of pathology
  • [ISO-abbreviation] Virchows Arch.
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / Biomarkers, Tumor; 0 / Membrane Proteins; 0 / RNA, Messenger
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39. Vance ML: Pituitary adenoma: a clinician's perspective. Endocr Pract; 2008 Sep;14(6):757-63
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  • [Title] Pituitary adenoma: a clinician's perspective.
  • RESULTS: A pituitary adenoma is quite common and poses a challenge to the clinician to determine the clinical significance, the appropriate diagnosis, the need for treatment, and the appropriate therapy or therapies.
  • The treatment of a pituitary adenoma depends on the type of tumor; a prolactinoma is treated medically with a dopamine agonist drug, and other lesions are usually treated by transsphenoidal surgical removal.
  • CONCLUSION: The goal is to decrease the mass effect of the adenoma, to restore normal pituitary function, and to suppress hormone hypersecretion.

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  • (PMID = 18996799.001).
  • [ISSN] 1934-2403
  • [Journal-full-title] Endocrine practice : official journal of the American College of Endocrinology and the American Association of Clinical Endocrinologists
  • [ISO-abbreviation] Endocr Pract
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 19
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40. Lee LS, Canter RJ, Fraker DL: Intraoperative jugular venous sampling AIDS detection of an undescended parathyroid adenoma. World J Surg; 2006 Apr;30(4):620-3
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Intraoperative jugular venous sampling AIDS detection of an undescended parathyroid adenoma.
  • INTRODUCTION: A missed parathyroid adenoma is an important cause of persistent or recurrent primary hyperparathyroidism.
  • In all patients, a two- to fourfold iPTH gradient was observed between the affected and unaffected sides, and an undescended adenoma was located near the carotid bifurcation.
  • CONCLUSIONS: Intraoperative jugular venous sampling with iPTH analysis may be a useful technique for successfully detecting an undescended adenoma when other, more routine measures have failed.
  • [MeSH-major] Adenoma / surgery. Hyperparathyroidism / surgery. Monitoring, Intraoperative / methods. Parathyroid Hormone / blood. Parathyroid Neoplasms / surgery. Parathyroidectomy / methods

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  • [Cites] Surgery. 1994 Dec;116(6):982-9; discussion 989-90 [7985106.001]
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  • (PMID = 16555025.001).
  • [ISSN] 0364-2313
  • [Journal-full-title] World journal of surgery
  • [ISO-abbreviation] World J Surg
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Parathyroid Hormone
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41. Khamidullina GA, Kapuller LL, Sereda EN, Izbagambetov NA, Zharkov NV: [Small-cell rectal carcinoma coexisted with tubular-villous adenoma]. Arkh Patol; 2006 Sep-Oct;68(5):37-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Small-cell rectal carcinoma coexisted with tubular-villous adenoma].
  • The paper describes a case of small-cell high-grade endocrine carcinoma of the rectum concurrent with tubular-villous adenoma in a 58 year-old woman.
  • All epithelial cells of adenoma have been ascertained to be "mixed" exoendocrine endocrinocytes (secrete mucus and contain neuroendocrine granules), contain progesterone receptors, and express CK 8, 18, and 19.
  • [MeSH-major] Adenoma, Villous / pathology. Carcinoma, Small Cell / pathology. Rectal Neoplasms / pathology

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  • (PMID = 17144530.001).
  • [ISSN] 0004-1955
  • [Journal-full-title] Arkhiv patologii
  • [ISO-abbreviation] Arkh. Patol.
  • [Language] rus
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Russia (Federation)
  • [Chemical-registry-number] 0 / Keratin-18; 0 / Ki-67 Antigen; 0 / Tumor Suppressor Protein p53
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42. House MG, Schulick RD: Endocrine tumors of the pancreas. Curr Opin Oncol; 2006 Jan;18(1):23-9
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  • [Title] Endocrine tumors of the pancreas.
  • PURPOSE OF REVIEW: Neoplasms of the endocrine pancreas, commonly referenced as pancreatic islet cell tumors, are rare, often well differentiated endocrine neoplasms, whose biology remains poorly characterized.
  • [MeSH-major] Adenoma, Islet Cell / therapy. Carcinoma, Islet Cell / therapy. Pancreatic Neoplasms / therapy

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  • [RetractionIn] Klastersky J. Curr Opin Oncol. 2006 Jul;18(4):414 [16721138.001]
  • (PMID = 16357560.001).
  • [ISSN] 1040-8746
  • [Journal-full-title] Current opinion in oncology
  • [ISO-abbreviation] Curr Opin Oncol
  • [Language] eng
  • [Publication-type] Journal Article; Retracted Publication; Review
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Biomarkers; 0 / ELL protein, human; 0 / Transcriptional Elongation Factors
  • [Number-of-references] 51
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43. Conte-Devolx B, Niccoli P, Groupe d'étude des Tumeurs Endocrines: [Clinical characteristics of multiple endocrine neoplasia]. Bull Acad Natl Med; 2010 Jan;194(1):69-78; discussion 78-9
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  • [Title] [Clinical characteristics of multiple endocrine neoplasia].
  • Multiple endocrine neoplasia type 1 (MEN1) and type 2 (MEN2) are autosomal dominant inherited multiglandular diseases with familial and individual age-related penetrance and variable expression.
  • The most frequent endocrine features of MEN1 are parathyroid involvement (> 95%), duodeno-pancreatic endocrine tissue involvement (80%), pituitary adenoma (30%), and adrenal cortex tumors (25%), with no clear syndromic variants.
  • [MeSH-major] Multiple Endocrine Neoplasia

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  • (PMID = 20669560.001).
  • [ISSN] 0001-4079
  • [Journal-full-title] Bulletin de l'Académie nationale de médecine
  • [ISO-abbreviation] Bull. Acad. Natl. Med.
  • [Language] fre
  • [Publication-type] English Abstract; Journal Article; Review
  • [Publication-country] Netherlands
  • [Number-of-references] 29
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44. Qu QY, Zhang QH, Wang XY: [Expression of E-cadherin catenin complex and invasiveness of pituitary adenoma]. Zhonghua Er Bi Yan Hou Tou Jing Wai Ke Za Zhi; 2006 Jun;41(6):433-6
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  • [Title] [Expression of E-cadherin catenin complex and invasiveness of pituitary adenoma].
  • OBJECTIVE: To explore the relationship between the E-cadherin catenin complex and invasiveness of pituitary adenoma.
  • METHODS: The expression of E-cadherin catenin complex was determined by immunohistochemistry in 78 cases of human pituitary adenomas including invasive adenoma 44 cases, noninvasive adenoma 34 cases and the relativity of their expressions with hormone-producing, pituitary apoplexy and necrosis or cystoid change, tumor diameter were analyzed.
  • Moreover, the expressions of beta-cad and alpha-cat were significantly lower in macro-adenoma group than that in micro-adenoma group (chi-squared = 5.038, P < 0.05).
  • The expression of E-cad was significantly lower in endocrine inactive group than that in endocrine active group (chi-squared = 4.614, P < 0.05).

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  • (PMID = 16927799.001).
  • [ISSN] 1673-0860
  • [Journal-full-title] Zhonghua er bi yan hou tou jing wai ke za zhi = Chinese journal of otorhinolaryngology head and neck surgery
  • [ISO-abbreviation] Zhonghua Er Bi Yan Hou Tou Jing Wai Ke Za Zhi
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] China
  • [Chemical-registry-number] 0 / Cadherins; 0 / alpha Catenin; 0 / beta Catenin
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45. Kijima Y, Matsukita S, Yoshinaka H, Owaki T, Aikou T: Adenoma of the nipple: report of a case. Breast Cancer; 2006;13(1):95-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Adenoma of the nipple: report of a case.
  • We report a case of an adenoma of the nipple in a 33-year-old Japanese woman who presented with a 2-year history of itching, eczema, and discharge from the left nipple.
  • The histological diagnosis was adenoma of the nipple.
  • [MeSH-major] Adenoma / pathology. Breast Neoplasms / pathology. Nipples / pathology

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  • (PMID = 16518068.001).
  • [ISSN] 1340-6868
  • [Journal-full-title] Breast cancer (Tokyo, Japan)
  • [ISO-abbreviation] Breast Cancer
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Japan
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46. Manouras A, Toutouzas KG, Markogiannakis H, Lagoudianakis E, Papadima A, Antonakis PT, Kafiri G, Bramis I: Intracystic hemorrhage in a mediastinal cystic adenoma causing parathyrotoxic crisis. Head Neck; 2008 Jan;30(1):127-31
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  • [Title] Intracystic hemorrhage in a mediastinal cystic adenoma causing parathyrotoxic crisis.
  • BACKGROUND: We report a case of intracystic hemorrhage in a mediastinal cystic parathyroid adenoma causing parathyrotoxic crisis.
  • Histopathologic examination revealed a double parathyroid adenoma and identified the mediastinal lesion as a cystic adenoma with intracystic hemorrhage.
  • CONCLUSIONS: Intracystic hemorrhage in a functional mediastinal cystic parathyroid adenoma is an extremely rare cause of parathyrotoxic crisis.
  • [MeSH-minor] Adenoma / pathology. Adenoma / surgery. Adult. Bone Density Conservation Agents / therapeutic use. Diphosphonates / therapeutic use. Diuretics / therapeutic use. Fluid Therapy. Furosemide / therapeutic use. Humans. Hyperparathyroidism / etiology. Hyperparathyroidism / therapy. Male. Parathyroid Neoplasms / pathology. Parathyroid Neoplasms / surgery. Thyroidectomy

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  • (PMID = 17615565.001).
  • [ISSN] 1043-3074
  • [Journal-full-title] Head & neck
  • [ISO-abbreviation] Head Neck
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Bone Density Conservation Agents; 0 / Diphosphonates; 0 / Diuretics; 7LXU5N7ZO5 / Furosemide; OYY3447OMC / pamidronate
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47. Moura FC, Gonçalves AC, Monteiro ML: Seesaw nystagmus caused by giant pituitary adenoma: case report. Arq Neuropsiquiatr; 2006 Mar;64(1):139-41
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Seesaw nystagmus caused by giant pituitary adenoma: case report.
  • Giant pituitary adenomas are uncommonly large tumors, greater than 4 cm in size that can produces endocrine symptoms, visual loss and cranial nerve palsies.
  • We report the rare occurrence of seesaw nystagmus as the presenting sign of giant pituitary adenoma.
  • Perimetry revealed bitemporal hemianopia and magnetic resonance imaging showed a giant pituitary adenoma.
  • [MeSH-major] Adenoma / complications. Hemianopsia / etiology. Nystagmus, Pathologic / etiology. Pituitary Neoplasms / complications

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  • (PMID = 16622572.001).
  • [ISSN] 0004-282X
  • [Journal-full-title] Arquivos de neuro-psiquiatria
  • [ISO-abbreviation] Arq Neuropsiquiatr
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Brazil
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48. Carney JA: Familial multiple endocrine neoplasia: the first 100 years. Am J Surg Pathol; 2005 Feb;29(2):254-74
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Familial multiple endocrine neoplasia: the first 100 years.
  • In 1903, Erdheim described the case of an acromegalic patient with a pituitary adenoma and three enlarged parathyroid glands.
  • In 1959, Hazard et al described medullary (solid) thyroid carcinoma (MTC), a tumor that later was found to be a component of two endocrine syndromes.
  • The first of these described by Sipple in 1961 comprised pheochromocytoma, MTC, and parathyroid adenoma.
  • In 1968, Steiner et al introduced the term "multiple endocrine neoplasia" (MEN) to describe disorders featuring combinations of endocrine tumors; they designated the Wermer syndrome as MEN 1 and the Sipple syndrome as MEN 2.
  • [MeSH-major] Multiple Endocrine Neoplasia / history

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  • (PMID = 15644784.001).
  • [ISSN] 0147-5185
  • [Journal-full-title] The American journal of surgical pathology
  • [ISO-abbreviation] Am. J. Surg. Pathol.
  • [Language] eng
  • [Publication-type] Historical Article; Journal Article; Portraits; Review
  • [Publication-country] United States
  • [Number-of-references] 92
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49. Akinci B, Demir T, Yener S, Comlekci A, Binicier O, Ozdogan O, Sevinc A, Kocdor MA, Bayraktar F, Canda T, Yesil S: Beneficial effect of endocrinologist-performed ultrasonography on preoperative parathyroid adenoma localization. Endocr Pract; 2009 Jan-Feb;15(1):17-23

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Beneficial effect of endocrinologist-performed ultrasonography on preoperative parathyroid adenoma localization.
  • METHODS: We performed a retrospective analysis of consecutive patients with primary hyperparathyroidism due to a single adenoma who underwent surgery at the Dokuz Eylul University Hospital in Izmir, Turkey, between January 2000 and January 2008.
  • RESULTS: A total of 156 patients with primary hyperparathyroidism due to a single adenoma were included.
  • Endocrinologist-performed ultrasonography localized the adenoma correctly in 19 patients for whom the staff radiologist had reported a negative or unsuccessful localization.
  • CONCLUSION: Our results suggest that endocrinologist-performed ultrasonography improves the preoperative localization of parathyroid adenoma.
  • [MeSH-major] Adenoma / ultrasonography. Clinical Competence. Parathyroid Neoplasms / ultrasonography. Ultrasonography, Doppler, Color / standards

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  • [ErratumIn] Endocr Pract. 2009 Nov-Dec;15(7):768. Secil, Mustafa [removed]
  • (PMID = 19211392.001).
  • [ISSN] 1934-2403
  • [Journal-full-title] Endocrine practice : official journal of the American College of Endocrinology and the American Association of Clinical Endocrinologists
  • [ISO-abbreviation] Endocr Pract
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 971Z4W1S09 / Technetium Tc 99m Sestamibi
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50. Kageyama K, Ikeda H, Nigawara T, Sakihara S, Suda T: Expression of adrenocorticotropic hormone, prolactin and transcriptional factors in clinically nonfunctioning pituitary adenoma. Endocr J; 2007 Dec;54(6):961-8
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  • [Title] Expression of adrenocorticotropic hormone, prolactin and transcriptional factors in clinically nonfunctioning pituitary adenoma.
  • We describe here a case of a clinically nonfunctioning pituitary adenoma, but with expression of ACTH and PRL.
  • Based on these findings we did not clinically suspect ACTH-producing tumor, however immunohistochemistry revealed ACTH immunoreactivity in the pituitary adenoma.
  • Therefore, the tumor was considered a silent corticotroph adenoma.
  • Ptx1, Neuro D1, and T pit were densely expressed and Pit-1 was sparsely expressed in the nuclei of adenoma cells.
  • [MeSH-major] Adenoma, Chromophobe / metabolism. Adrenocorticotropic Hormone / biosynthesis. Pituitary Neoplasms / metabolism. Prolactin / biosynthesis. Transcription Factors / biosynthesis

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  • (PMID = 18079591.001).
  • [ISSN] 1348-4540
  • [Journal-full-title] Endocrine journal
  • [ISO-abbreviation] Endocr. J.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / Basic Helix-Loop-Helix Transcription Factors; 0 / ERGIC2 protein, human; 0 / Homeodomain Proteins; 0 / NEUROD1 protein, human; 0 / T-Box Domain Proteins; 0 / TBX19 protein, human; 0 / Transcription Factor Pit-1; 0 / Transcription Factors; 0 / Vesicular Transport Proteins; 12629-01-5 / Human Growth Hormone; 9002-60-2 / Adrenocorticotropic Hormone; 9002-62-4 / Prolactin; 9002-71-5 / Thyrotropin
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51. Nakamura N, Erickson LA, Jin L, Kajita S, Zhang H, Qian X, Rumilla K, Lloyd RV: Immunohistochemical separation of follicular variant of papillary thyroid carcinoma from follicular adenoma. Endocr Pathol; 2006;17(3):213-23
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  • [Title] Immunohistochemical separation of follicular variant of papillary thyroid carcinoma from follicular adenoma.
  • A combination of markers consisting of a panel of HBME-1, galectin-3, and CK19 or a panel of HBME-1, CITED1, and galectin-3 was usually most effective in distinguishing follicular adenoma from follicular variant of papillary thyroid carcinoma.
  • These results indicate that some individual antibodies or a panel of antibodies combined with histopathological analysis can be useful in separating follicular adenoma (FA) from follicular variant of papillary thyroid carcinoma (FVPTC).
  • [MeSH-major] Adenoma / diagnosis. Biomarkers, Tumor / analysis. Carcinoma, Papillary, Follicular / diagnosis. Thyroid Neoplasms / diagnosis

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  • (PMID = 17308358.001).
  • [ISSN] 1046-3976
  • [Journal-full-title] Endocrine pathology
  • [ISO-abbreviation] Endocr. Pathol.
  • [Language] eng
  • [Publication-type] Historical Article; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Biomarkers, Tumor
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52. Giacchetti G, Ronconi V, Rilli S, Guerrieri M, Turchi F, Boscaro M: Small tumor size as favorable prognostic factor after adrenalectomy in Conn's adenoma. Eur J Endocrinol; 2009 Apr;160(4):639-46

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Small tumor size as favorable prognostic factor after adrenalectomy in Conn's adenoma.
  • OBJECTIVE: Primary aldosteronism (PA) due to aldosterone-producing adenoma (APA) is the most common curable form of secondary hypertension.
  • Drug history and adenoma size at morphological evaluation were also collected.
  • RESULTS: Multiple regression analysis showed that, before surgery, patients with a small adenoma (diameter <20 mm) displayed higher postsaline aldosterone values (P=0.0001), and lower serum potassium levels (P=0.020), than patients with adenoma >20 mm.
  • Recovered patients had a shorter duration of hypertension (P=0.038), and a smaller adenoma size (P=0.035).

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  • (PMID = 19131503.001).
  • [ISSN] 1479-683X
  • [Journal-full-title] European journal of endocrinology
  • [ISO-abbreviation] Eur. J. Endocrinol.
  • [Language] ENG
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 4964P6T9RB / Aldosterone
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53. Imelda F, Bandar IN, Setiyohadi B, Suwondo P, Nasar IM, Darwito: Postural shortening due to primary hyperparathyroidism caused by parathyroid adenoma. Acta Med Indones; 2006 Apr-Jun;38(2):89-91
Genetic Alliance. consumer health - Hyperparathyroidism, primary.

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Postural shortening due to primary hyperparathyroidism caused by parathyroid adenoma.
  • Secondary osteoporosis is the result of an underlying disease such as an endocrine abnormality, and an example of such is primary hyperparathyroidism.
  • The most common cause of primary hyperparathyroidism is parathyroid gland adenoma.
  • The patient was diagnosed with primary hyperparathyroidism caused by parathyroid gland adenoma.
  • [MeSH-major] Adenoma / diagnosis. Body Height / physiology. Hyperparathyroidism, Primary / physiopathology. Parathyroid Neoplasms / diagnosis

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  • (PMID = 16799210.001).
  • [ISSN] 0125-9326
  • [Journal-full-title] Acta medica Indonesiana
  • [ISO-abbreviation] Acta Med Indones
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Indonesia
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54. Tagami T, Usui T, Shimatsu A, Naruse M: Toxic thyroid adenoma presenting as hypokalemic periodic paralysis. Endocr J; 2007 Dec;54(5):797-803
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  • [Title] Toxic thyroid adenoma presenting as hypokalemic periodic paralysis.
  • Toxic thyroid adenoma presenting as hypokalemic periodic paralysis is extraordinarily rare.
  • No somatic mutation of TSH receptor was found in his thyroid adenoma and no known genetic mutations of ionic channel genes, such as calcium (CACN1S), sodium (SCN4A) and potassium (KCNE3), were found.
  • [MeSH-major] Adenoma / diagnosis. Hypokalemic Periodic Paralysis / diagnosis. Thyroid Neoplasms / diagnosis. Thyrotoxicosis / diagnosis


55. Tarakji B, Nassani MZ, Sloan P: Immunohistochemical expression of estrogens and progesterone receptors in carcinoma ex pleomorphic adenoma-undifferentiated and adenocarcinoma types. Med Oral Patol Oral Cir Bucal; 2010 May;15(3):e432-6
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  • [Title] Immunohistochemical expression of estrogens and progesterone receptors in carcinoma ex pleomorphic adenoma-undifferentiated and adenocarcinoma types.
  • OBJECTIVE: Our study aimed to characterize alteration in the immunohistochemical expression of oestrogens receptor and progesterone receptor in the tumour cells of carcinoma arising in pleomorphic adenoma.
  • STUDY DESIGN: 27 cases of carcinoma arising in pleomorphic adenoma (undifferentiated and adenocarcinoma types) were examined.
  • CONCLUSION: Our data suggest that carcinomas arising in pleomorphic adenoma were not dependent on endocrine function.
  • [MeSH-major] Adenocarcinoma / metabolism. Adenoma, Pleomorphic / metabolism. Neoplasms, Multiple Primary / metabolism. Receptors, Estrogen / biosynthesis. Receptors, Progesterone / biosynthesis. Salivary Gland Neoplasms / metabolism

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  • (PMID = 20038908.001).
  • [ISSN] 1698-6946
  • [Journal-full-title] Medicina oral, patología oral y cirugía bucal
  • [ISO-abbreviation] Med Oral Patol Oral Cir Bucal
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Spain
  • [Chemical-registry-number] 0 / Receptors, Estrogen; 0 / Receptors, Progesterone
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56. Zhan X, Desiderio DM: Signaling pathway networks mined from human pituitary adenoma proteomics data. BMC Med Genomics; 2010 Apr 28;3:13
NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .

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  • [Title] Signaling pathway networks mined from human pituitary adenoma proteomics data.
  • BACKGROUND: We obtained a series of pituitary adenoma proteomic expression data, including protein-mapping data (111 proteins), comparative proteomic data (56 differentially expressed proteins), and nitroproteomic data (17 nitroproteins).
  • There is a pressing need to clarify the significant signaling pathway networks that derive from those proteins in order to clarify and to better understand the molecular basis of pituitary adenoma pathogenesis and to discover biomarkers.
  • Here, we describe the significant signaling pathway networks that were mined from human pituitary adenoma proteomic data with the Ingenuity pathway analysis system.
  • METHODS: The Ingenuity pathway analysis system was used to analyze signal pathway networks and canonical pathways from protein-mapping data, comparative proteomic data, adenoma nitroproteomic data, and control nitroproteomic data.
  • Statistical significant results were rationalized within the pituitary adenoma biological system with literature-based bioinformatics analyses.
  • For the comparative proteomic data, top pathway networks were related to cancer, endocrine system development and function, and lipid metabolism; the top canonical toxicity pathways included mitochondrial dysfunction, oxidative phosphorylation, oxidative-stress response, and ERK/MAPK signaling.
  • The nitroproteomic data from a pituitary adenoma were related to cancer, cell death, lipid metabolism, and reproductive system disease, and the top canonical toxicity pathways mainly related to p38 MAPK signaling and cell-cycle G2/M transition regulation.
  • CONCLUSIONS: This pathway network analysis demonstrated that mitochondrial dysfunction, oxidative stress, cell-cycle dysregulation, and the MAPK-signaling abnormality are significantly associated with a pituitary adenoma.
  • These pathway-network data provide new insights into the molecular mechanisms of human pituitary adenoma pathogenesis, and new clues for an in-depth investigation of pituitary adenoma and biomarker discovery.

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  • (PMID = 20426862.001).
  • [ISSN] 1755-8794
  • [Journal-full-title] BMC medical genomics
  • [ISO-abbreviation] BMC Med Genomics
  • [Language] ENG
  • [Grant] United States / NCRR NIH HHS / RR / RR016679
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Biomarkers; 0 / Cell Cycle Proteins; EC 2.7.11.24 / p38 Mitogen-Activated Protein Kinases
  • [Other-IDs] NLM/ PMC2884164
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57. Ogawa T, Tsuji E, Kanauchi H, Yamada K, Mimura Y, Kaminishi M: Excision of postesophageal parathyroid adenoma in posterior mediastinum with intraoperative 99mTechnetium sestamibi scanning. Ann Thorac Surg; 2007 Nov;84(5):1754-6

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Excision of postesophageal parathyroid adenoma in posterior mediastinum with intraoperative 99mTechnetium sestamibi scanning.
  • We present a case of retroesophageal mediastinal parathyroid adenoma that developed in the left superior parathyroid gland.
  • [MeSH-major] Adenoma / surgery. Choristoma / surgery. Mediastinal Neoplasms / surgery. Parathyroid Neoplasms / surgery. Technetium Tc 99m Sestamibi

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  • (PMID = 17954108.001).
  • [ISSN] 1552-6259
  • [Journal-full-title] The Annals of thoracic surgery
  • [ISO-abbreviation] Ann. Thorac. Surg.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 971Z4W1S09 / Technetium Tc 99m Sestamibi
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58. Blandamura S, Parenti A, Famengo B, Canesso A, Moschino P, Pasquali C, Pizzi S, Guzzardo V, Ninfo V: Three cases of pancreatic serous cystadenoma and endocrine tumour. J Clin Pathol; 2007 Mar;60(3):278-82
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Three cases of pancreatic serous cystadenoma and endocrine tumour.
  • AIMS: To report three cases of serous cystadenoma and endocrine tumour in the same pancreas, to review the literature and to evaluate the clinicopathological features of the tumours.
  • RESULTS: Histological examination of the pancreas showed one serous oligocystic adenoma associated with a benign, well-differentiated endocrine tumour, one serous oligocystic adenoma associated with an endocrine microadenoma, and a von Hippel-Lindau-related cystic neoplasm with a well-differentiated endocrine carcinoma.
  • CONCLUSIONS: Serous cystadenoma associated with endocrine tumour shows some clinicopathological differences with respect to the two tumours considered separately, and with respect to von Hippel-Lindau-related cases, although there is no convincing evidence at present to justify considering this association as a separate entity.
  • [MeSH-major] Cystadenoma, Serous / pathology. Endocrine Gland Neoplasms / pathology. Neoplasms, Multiple Primary / pathology. Pancreatic Neoplasms / pathology

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  • (PMID = 16644876.001).
  • [ISSN] 0021-9746
  • [Journal-full-title] Journal of clinical pathology
  • [ISO-abbreviation] J. Clin. Pathol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Neoplasm Proteins
  • [Number-of-references] 32
  • [Other-IDs] NLM/ PMC1860571
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59. Kapran Y, Bauersfeld J, Anlauf M, Sipos B, Klöppel G: Multihormonality and entrapment of islets in pancreatic endocrine tumors. Virchows Arch; 2006 Apr;448(4):394-8
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  • [Title] Multihormonality and entrapment of islets in pancreatic endocrine tumors.
  • We analyzed pancreatic endocrine tumors (PETs) from 200 patients for the incidence of multihormonality and entrapped islets and correlated the results with clinicopathological features.
  • [MeSH-major] Adenoma, Islet Cell / pathology. Carcinoma, Islet Cell / pathology. Islets of Langerhans / pathology. Pancreatic Hormones / metabolism. Pancreatic Neoplasms / pathology

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  • (PMID = 16418841.001).
  • [ISSN] 0945-6317
  • [Journal-full-title] Virchows Archiv : an international journal of pathology
  • [ISO-abbreviation] Virchows Arch.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / Biomarkers, Tumor; 0 / Pancreatic Hormones
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60. Berker D, Aydin Y, Tutuncu YA, Isik S, Delibasi T, Berker M, Guler S, Kamel N: Somatotropin adenoma and resistance to thyroid hormone. J Endocrinol Invest; 2009 Mar;32(3):284-6
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Somatotropin adenoma and resistance to thyroid hormone.
  • Up to date, in the literature, there are more than 600 RTH cases, but co-incidental hypophyseal adenoma was reported in only 1 case.
  • The patient was followed with the diagnosis of RTH and incidental hypophyseal adenoma.
  • [MeSH-major] Growth Hormone-Secreting Pituitary Adenoma / complications. Pituitary Neoplasms / complications. Thyroid Hormone Resistance Syndrome / complications
  • [MeSH-minor] Adult. Diagnostic Techniques, Endocrine. Humans. Incidental Findings. Male

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  • (PMID = 19542750.001).
  • [ISSN] 1720-8386
  • [Journal-full-title] Journal of endocrinological investigation
  • [ISO-abbreviation] J. Endocrinol. Invest.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Italy
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61. Kimata-Hayashi N, Takano K, Yasufuku-Takano J, Teramoto A, Fujita T: Mechanism of adrenomedullin-induced prolactin release from human prolactin-releasing adenoma cells. Endocr J; 2005 Dec;52(6):769-73
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  • [Title] Mechanism of adrenomedullin-induced prolactin release from human prolactin-releasing adenoma cells.
  • The mechanism of adrenomedullin-induced prolactin release was investigated in prolactin-secreting human pituitary adenoma cells by intracellular calcium measurement and static incubation study.

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  • (PMID = 16410671.001).
  • [ISSN] 0918-8959
  • [Journal-full-title] Endocrine journal
  • [ISO-abbreviation] Endocr. J.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / Calcium Channels; 0 / Intracellular Signaling Peptides and Proteins; 0 / Peptides; 0 / protein kinase modulator; 148498-78-6 / Adrenomedullin; 9002-62-4 / Prolactin; 9NEZ333N27 / Sodium; SY7Q814VUP / Calcium; TSN3DL106G / Fura-2
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62. Mete O, Asa SL: Aldosterone-producing adrenal cortical adenoma with oncocytic change and cytoplasmic eosinophilic globular inclusions. Endocr Pathol; 2009;20(3):182-5
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  • [Title] Aldosterone-producing adrenal cortical adenoma with oncocytic change and cytoplasmic eosinophilic globular inclusions.
  • The laparoscopic left adrenalectomy specimen revealed an adrenal cortical adenoma composed of varying proportions of oncocytic and clear cells, predominantly showing central oncocytic change.
  • [MeSH-major] Adrenal Cortex Neoplasms / pathology. Adrenocortical Adenoma / pathology. Aldosterone / secretion. Inclusion Bodies / pathology

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  • (PMID = 19462261.001).
  • [ISSN] 1559-0097
  • [Journal-full-title] Endocrine pathology
  • [ISO-abbreviation] Endocr. Pathol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antihypertensive Agents; 4964P6T9RB / Aldosterone
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63. Toledo RA, Lourenço DM Jr, Toledo SP: Familial isolated pituitary adenoma: evidence for genetic heterogeneity. Front Horm Res; 2010;38:77-86
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  • [Title] Familial isolated pituitary adenoma: evidence for genetic heterogeneity.
  • The identification of mutations in the Aryl hydrocarbon receptor interacting protein (AIP) gene in a subset of familial isolated pituitary adenoma (FIPA) cases has recently expanded our understanding of the pathophysiology of inherited pituitary adenoma disorders.
  • [MeSH-major] Adenoma / genetics. Genetic Heterogeneity. Pituitary Neoplasms / genetics

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  • [Copyright] Copyright (c) 2010 S. Karger AG, Basel.
  • (PMID = 20616498.001).
  • [ISSN] 0301-3073
  • [Journal-full-title] Frontiers of hormone research
  • [ISO-abbreviation] Front Horm Res
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] Switzerland
  • [Chemical-registry-number] 0 / CDKN1B protein, human; 0 / Intracellular Signaling Peptides and Proteins; 0 / aryl hydrocarbon receptor-interacting protein; 147604-94-2 / Cyclin-Dependent Kinase Inhibitor p27
  • [Number-of-references] 41
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64. Nielsen EH, Lindholm J, Bjerre P, Christiansen JS, Hagen C, Juul S, Jørgensen J, Kruse A, Laurberg P: Frequent occurrence of pituitary apoplexy in patients with non-functioning pituitary adenoma. Clin Endocrinol (Oxf); 2006 Mar;64(3):319-22
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  • [Title] Frequent occurrence of pituitary apoplexy in patients with non-functioning pituitary adenoma.
  • As our experience differs from this view, we analysed the incidence in patients operated on for a nonfunctioning pituitary adenoma.
  • PATIENTS AND DESIGN: One hundred ninety-two consecutive patients with a suprasellar, clinically inactive adenoma operated on during the period 1985-1996 were retrospectively reviewed.
  • The outcome as regards survival and endocrine function was not different from that in patients with a nonfunctioning adenoma who did not suffer pituitary apoplexy.
  • [MeSH-major] Adenoma / complications. Pituitary Apoplexy / etiology. Pituitary Neoplasms / complications

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  • (PMID = 16487443.001).
  • [ISSN] 0300-0664
  • [Journal-full-title] Clinical endocrinology
  • [ISO-abbreviation] Clin. Endocrinol. (Oxf)
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] England
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65. Yoshida A, Sen C, Asa SL, Rosenblum MK: Composite pituitary adenoma and craniopharyngioma?: an unusual sellar neoplasm with divergent differentiation. Am J Surg Pathol; 2008 Nov;32(11):1736-41
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  • [Title] Composite pituitary adenoma and craniopharyngioma?: an unusual sellar neoplasm with divergent differentiation.
  • At transsphenoidal surgery, a tumor consisting of a pituitary adenoma and adamantinomatous craniopharyngiomalike components was resected.
  • Immunohistochemically, the adenoma was not only positive for beta-thyroid stimulating hormone, alpha subunit, and pituitary transcription factor 1, but also stained for beta-follicle stimulating hormone, steroidogenic factor-1, adrenocorticotropic hormone, and pituitary-restricted transcription factor (Tpit), exhibiting an unusual plurihormonal profile.
  • Alternatively, it may be viewed as a pituitary adenoma showing metaplastic change analogous to the development of squamous cell nests of the pars tuberalis from adenohypophyseal endocrine cells.
  • [MeSH-major] Adenoma / pathology. Craniopharyngioma / pathology. Neoplasms, Multiple Primary / pathology. Pituitary Neoplasms / pathology

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  • (PMID = 18769335.001).
  • [ISSN] 1532-0979
  • [Journal-full-title] The American journal of surgical pathology
  • [ISO-abbreviation] Am. J. Surg. Pathol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 9002-71-5 / Thyrotropin
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66. Wolf A, Willenberg HS, Cupisti K, Schott M, Geddert H, Raffel A, Bornstein SR, Scherbaum WA, Knoefel WT: Adrenal pheochromocytoma with contralateral cortisol-producing adrenal adenoma: diagnostic and therapeutic management. Horm Metab Res; 2005 Jun;37(6):391-5
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  • [Title] Adrenal pheochromocytoma with contralateral cortisol-producing adrenal adenoma: diagnostic and therapeutic management.
  • To contribute to the studies of systemic interactions between these tissues, we studied a 52-year-old female patient with a pheochromocytoma and a contralateral cortisol-producing adenoma.
  • Due to excess catecholamine secretion, bilateral pheochromocytomas based on multiple endocrine neoplasia syndrome were suspected.
  • Laboratory work-up, selective adrenal venous sampling and magnetic resonance imaging studies established the diagnosis of a pheochromocytoma in the right-hand adrenal gland and a cortisol-producing adenoma on the left.
  • Although presence of bilateral adrenal adenomas or bilateral adrenal pheochromocytomas in certain inherited disorders are possible, this rare case of an adrenal pheochromocytoma combined with a contralateral cortisol-producing adrenal adenoma may further underline the wide range of complex interactions between the two endocrine systems.
  • [MeSH-major] Adrenal Cortex Neoplasms / radiography. Adrenocortical Adenoma / radiography. Pheochromocytoma / radiography

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  • (PMID = 16001333.001).
  • [ISSN] 0018-5043
  • [Journal-full-title] Hormone and metabolic research = Hormon- und Stoffwechselforschung = Hormones et métabolisme
  • [ISO-abbreviation] Horm. Metab. Res.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Germany
  • [Chemical-registry-number] WI4X0X7BPJ / Hydrocortisone
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67. Calender A, Groupe d'étude des Tumeurs Endocrines: [Multiple endocrine neoplasia: genetic aspects]. Bull Acad Natl Med; 2010 Jan;194(1):81-95; discussion 95-6
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  • [Title] [Multiple endocrine neoplasia: genetic aspects].
  • Multiple endocrine neoplasia type 1 (MEN1) and type 2 (MEN2) are major genetic disorders carrying a high risk of endocrine tumor development.
  • We also briefly describe the genetic basis of three other inherited states predisposing individuals to endocrine tumors, namely Carney's syndrome, hyperparathyroidism type 2 (HRPT2) and familial isolated pituitary adenoma (FIPA), which are related to inactivating mutations in the PRKAR1-alpha, HRPT2 and AIP genes, respectively.
  • [MeSH-major] Genes, Tumor Suppressor. Multiple Endocrine Neoplasia / genetics. Proto-Oncogene Proteins / genetics. Proto-Oncogene Proteins c-ret / genetics

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  • (PMID = 20669561.001).
  • [ISSN] 0001-4079
  • [Journal-full-title] Bulletin de l'Académie nationale de médecine
  • [ISO-abbreviation] Bull. Acad. Natl. Med.
  • [Language] fre
  • [Publication-type] English Abstract; Journal Article; Review
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Glial Cell Line-Derived Neurotrophic Factor Receptors; 0 / MEN1 protein, human; 0 / Proto-Oncogene Proteins; EC 2.7.10.1 / Proto-Oncogene Proteins c-ret; EC 2.7.10.1 / RET protein, human
  • [Number-of-references] 30
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68. Fatemi N, Dusick JR, Mattozo C, McArthur DL, Cohan P, Boscardin J, Wang C, Swerdloff RS, Kelly DF: Pituitary hormonal loss and recovery after transsphenoidal adenoma removal. Neurosurgery; 2008 Oct;63(4):709-18; discussion 718-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Pituitary hormonal loss and recovery after transsphenoidal adenoma removal.
  • METHODS: All consecutive patients who underwent endonasal transsphenoidal adenoma removal over an 8-year period were analyzed.
  • Of 223 patients with an endocrine-inactive adenoma, new hypopituitarism was seen in 0, 7.2, and 13.6% of patients with tumor diameters of <20, 20 to 29, and >or=30 mm, respectively (P = 0.005).
  • Multivariate analysis revealed that resolution of hypopituitarism was related to younger age (39 versus 52 years, P < 0.0001), absence of an intraoperative cerebrospinal fluid leak and, in patients with an endocrine-inactive adenoma, absence of systemic hypertension (24% versus 6%, P = 0.009).

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  • [CommentIn] Neurosurgery. 2010 Jul;67(1):221; author reply 221 [20559079.001]
  • (PMID = 18981881.001).
  • [ISSN] 1524-4040
  • [Journal-full-title] Neurosurgery
  • [ISO-abbreviation] Neurosurgery
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Pituitary Hormones
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69. Hamidi S, Hedayat A, Esfahanian F, Kamalian N: Distribution of solitary parathyroid adenoma over the parathyroid glands and its surgical management. J Coll Physicians Surg Pak; 2007 Oct;17(10):619-21

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  • [Title] Distribution of solitary parathyroid adenoma over the parathyroid glands and its surgical management.
  • OBJECTIVE: To determine the distribution of solitary parathyroid adenoma over the parathyroid glands in a group of patients with primary hyperparathyroidism Design: A case-series.
  • All of the patients had solitary parathyroid adenoma and the anatomical location of each adenoma was clearly defined during operation.
  • RESULTS: The distribution of adenomas over the superior and inferior parathyroid glands showed a significant higher incidence of adenoma in the lower parathyroids (p < 0.001).

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  • (PMID = 17999854.001).
  • [ISSN] 1022-386X
  • [Journal-full-title] Journal of the College of Physicians and Surgeons--Pakistan : JCPSP
  • [ISO-abbreviation] J Coll Physicians Surg Pak
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Pakistan
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70. Alzahrani AS, Al-Khaldi N, Shi Y, Al-Rijjal RA, Zou M, Baitei EY, Amin T: Diagnosis by serendipity: Cushing syndrome attributable to cortisol-producing adrenal adenoma as the initial manifestation of multiple endocrine neoplasia type 1 due to a rare splicing site MEN1 gene mutation. Endocr Pract; 2008 Jul-Aug;14(5):595-602
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  • [Title] Diagnosis by serendipity: Cushing syndrome attributable to cortisol-producing adrenal adenoma as the initial manifestation of multiple endocrine neoplasia type 1 due to a rare splicing site MEN1 gene mutation.
  • OBJECTIVE: To report a case that highlights the potential for Cushing syndrome to be the first manifestation of multiple endocrine neoplasia type 1 (MEN 1) syndrome and to describe the rare underlying genetic mutation and the heterogeneous manifestations of the syndrome within the same family.
  • RESULTS: A 16-year-old girl who was not known to have any medical illness and had no known family history of MEN 1 syndrome presented with Cushing syndrome attributable to a cortisol-producing adrenal adenoma.
  • [MeSH-major] Adrenocortical Adenoma / pathology. Cushing Syndrome / diagnosis. Hydrocortisone / secretion. Multiple Endocrine Neoplasia Type 1 / pathology


71. Lantz M, Abraham-Nordling M, Svensson J, Wallin G, Hallengren B: Immigration and the incidence of Graves' thyrotoxicosis, thyrotoxic multinodular goiter and solitary toxic adenoma. Eur J Endocrinol; 2009 Feb;160(2):201-6
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  • [Title] Immigration and the incidence of Graves' thyrotoxicosis, thyrotoxic multinodular goiter and solitary toxic adenoma.
  • The aim was to investigate whether the incidence of Graves' thyrotoxicosis (GD), toxic multinodular goiter (TMNG), and solitary toxic adenoma (STA) has changed in Malmö since 1990 and to study the influence of geographic origin.

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  • (PMID = 19004985.001).
  • [ISSN] 1479-683X
  • [Journal-full-title] European journal of endocrinology
  • [ISO-abbreviation] Eur. J. Endocrinol.
  • [Language] ENG
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
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72. Kimura N, Komuro K, Uchino S, Yagihashi S, Ishidate T, Ishizaka M: Multiple endocrine neoplasia type 1-associated cystic pancreatic endocrine neoplasia and multifocal cholesterol granulomas. Pathol Int; 2010 Apr;60(4):321-5
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  • [Title] Multiple endocrine neoplasia type 1-associated cystic pancreatic endocrine neoplasia and multifocal cholesterol granulomas.
  • A novel combination of tumors was found in a 68 year-old female with Multiple Endocrine Neoplasia type-1 (MEN 1) that included a cystic pancreatic endocrine neoplasm (CPEN), a pituitary adenoma, and multifocal cholesterol granulomas (MCGs) in the breast, pleura, and the extremities.
  • The tumor cells of the pancreas were immunohistochemically positive for both endocrine and pancreatic acinar markers including chromogranin A, synaptophysin, glucagon, lipase, and reg protein.
  • Analysis of the DNA extracted from the tissues revealed that there is a MEN1 germline mutation in exon 10 codon 527, and somatic mutation in exon 2 codon 32 in the pancreatic tumor, and one base pair deletion in exon 2 codon 79 in the pituitary adenoma.
  • [MeSH-major] Adenoma / pathology. Breast Neoplasms / pathology. Cholesterol. Granuloma, Foreign-Body / pathology. Multiple Endocrine Neoplasia Type 1 / pathology. Pancreas / pathology. Pancreatic Neoplasms / pathology. Pituitary Neoplasms / pathology


73. Revill K, Dudley KJ, Clayton RN, McNicol AM, Farrell WE: Loss of neuronatin expression is associated with promoter hypermethylation in pituitary adenoma. Endocr Relat Cancer; 2009 Jun;16(2):537-48
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  • [Title] Loss of neuronatin expression is associated with promoter hypermethylation in pituitary adenoma.
  • In this report, we determined the expression pattern of NNAT in individual cell types of the normal gland and within each of the different pituitary adenoma subtypes.

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  • (PMID = 19218280.001).
  • [ISSN] 1351-0088
  • [Journal-full-title] Endocrine-related cancer
  • [ISO-abbreviation] Endocr. Relat. Cancer
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Membrane Proteins; 0 / NNAT protein, human; 0 / Nerve Tissue Proteins; 0 / RNA, Messenger
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74. Kahara T, Seto C, Uchiyama A, Usuda D, Akahori H, Tajika E, Miwa A, Usuda R, Suzuki T, Sasano H: Preclinical Cushing's syndrome resulting from adrenal black adenoma diagnosed with diabetic ketoacidosis. Endocr J; 2007 Aug;54(4):543-51
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Preclinical Cushing's syndrome resulting from adrenal black adenoma diagnosed with diabetic ketoacidosis.
  • Histological examination of the adrenalectomy specimen demonstrated adrenal black adenoma.
  • [MeSH-major] Adenoma / complications. Adrenal Gland Neoplasms / complications. Cushing Syndrome / etiology. Diabetes Mellitus, Type 2 / etiology. Diabetic Ketoacidosis / etiology


75. Souza ER, Scrignoli JA, Bezerra FC, Ribeiro SL, Passos LF: Devastating skeletal effects of delayed diagnosis of complicated primary hyperparathyroidism because of ectopic adenoma. J Clin Rheumatol; 2008 Oct;14(5):281-4
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  • [Title] Devastating skeletal effects of delayed diagnosis of complicated primary hyperparathyroidism because of ectopic adenoma.
  • Primary hyperparathyroidism is a disease caused by exaggerated secretion of the parathyroid gland hormone, produced by an adenoma in 80% of cases.
  • Herein, the authors report a 72-year-old woman with a delayed diagnosis of primary hyperparathyroidism, produced by an intrathoracic adenoma, with a longstanding course, presenting with severe osteoporosis, multiple fractures, bone deformities, and neurologic impairments.
  • After surgical removal, the histopathological examination confirmed an ectopic adenoma of the parathyroid gland and the patient achieved some improvement in her clinical picture.
  • [MeSH-major] Adenoma / complications. Bone Diseases, Endocrine / etiology. Hyperparathyroidism, Primary / complications. Hyperparathyroidism, Primary / diagnosis. Parathyroid Neoplasms / complications

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  • (PMID = 18824925.001).
  • [ISSN] 1536-7355
  • [Journal-full-title] Journal of clinical rheumatology : practical reports on rheumatic & musculoskeletal diseases
  • [ISO-abbreviation] J Clin Rheumatol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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76. Georgitsi M, Raitila A, Karhu A, van der Luijt RB, Aalfs CM, Sane T, Vierimaa O, Mäkinen MJ, Tuppurainen K, Paschke R, Gimm O, Koch CA, Gündogdu S, Lucassen A, Tischkowitz M, Izatt L, Aylwin S, Bano G, Hodgson S, De Menis E, Launonen V, Vahteristo P, Aaltonen LA: Germline CDKN1B/p27Kip1 mutation in multiple endocrine neoplasia. J Clin Endocrinol Metab; 2007 Aug;92(8):3321-5
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  • [Title] Germline CDKN1B/p27Kip1 mutation in multiple endocrine neoplasia.
  • CONTEXT: Germline mutations in the MEN1 gene predispose to multiple endocrine neoplasia type 1 (MEN1) syndrome, but in up to 20-25% of clinical MEN1 cases, no MEN1 mutations can be found.
  • OBJECTIVE: Our objective was to evaluate the role of CDKN1B/p27(Kip1) in human tumor predisposition in patients clinically suspected of MEN1 but testing negative for MEN1 germline mutation as well as in familial and sporadic acromegaly/pituitary adenoma patients.
  • In addition, 19 familial and 50 sporadic acromegaly/pituitary adenoma patients from Europe and the United States were included in the study.
  • MAIN OUTCOME MEASURES: We analyzed germline CDKN1B/p27(Kip1) mutations in individuals with pituitary adenoma and MEN1-like features.
  • RESULTS: A heterozygous 19-bp duplication (c.59_77dup19) leading to a truncated protein product was identified in one Dutch patient with suspected MEN1 phenotype, pituitary adenoma, carcinoid tumor, and hyperparathyroidism (one of 36, 2.8%).
  • No mutations were detected in either familial or sporadic acromegaly/pituitary adenoma patients.
  • However, such mutations appear uncommon in suspected MEN1 cases and rare or nonexistent in familial or sporadic acromegaly/pituitary adenoma patients.
  • [MeSH-major] DNA / genetics. Intracellular Signaling Peptides and Proteins / genetics. Multiple Endocrine Neoplasia Type 1 / genetics

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  • (PMID = 17519308.001).
  • [ISSN] 0021-972X
  • [Journal-full-title] The Journal of clinical endocrinology and metabolism
  • [ISO-abbreviation] J. Clin. Endocrinol. Metab.
  • [Language] eng
  • [Databank-accession-numbers] GENBANK/ EF474465
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / CDKN1B protein, human; 0 / Intracellular Signaling Peptides and Proteins; 147604-94-2 / Cyclin-Dependent Kinase Inhibitor p27; 9007-49-2 / DNA
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77. Moshkin O, Scheithauer BW, Syro LV, Velasquez A, Horvath E, Kovacs K: Collision tumors of the sella: craniopharyngioma and silent pituitary adenoma subtype 3: case report. Endocr Pathol; 2009;20(1):50-5
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  • [Title] Collision tumors of the sella: craniopharyngioma and silent pituitary adenoma subtype 3: case report.
  • Collision tumors of the sella turcica are rare and consist mainly of more than one type of pituitary adenoma, usually a corticotropin- and a prolactin-producing adenoma.
  • The association of a craniopharyngioma and a pituitary adenoma is rare.
  • Herein, we report the first case of an association between craniopharyngioma and silent pituitary adenoma subtype 3.
  • Histology revealed an adamantinomatous craniopharyngioma; however, by electron microscopy, there was conclusive evidence of adenoma cells showing the ultrastructural features of silent pituitary adenoma subtype 3.
  • Endocrine and neuroimaging as well as detailed immunohistochemical and ultrastructural studies were undertaken.
  • [MeSH-major] Adenoma / ultrastructure. Craniopharyngioma / ultrastructure. Neoplasms, Multiple Primary / pathology. Pituitary Neoplasms / ultrastructure

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  • (PMID = 19238590.001).
  • [ISSN] 1046-3976
  • [Journal-full-title] Endocrine pathology
  • [ISO-abbreviation] Endocr. Pathol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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78. Davis JR, McNeilly JR, Norris AJ, Pope C, Wilding M, McDowell G, Holland JP, McNeilly AS: Fetal gonadotrope cell origin of FSH-secreting pituitary adenoma - insight into human pituitary tumour pathogenesis. Clin Endocrinol (Oxf); 2006 Nov;65(5):648-54
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  • [Title] Fetal gonadotrope cell origin of FSH-secreting pituitary adenoma - insight into human pituitary tumour pathogenesis.
  • OBJECTIVE: The pathogenesis of human pituitary adenomas remains unclear, but we report a case of FSH-secreting pituitary adenoma whose monohormonal phenotype suggests it was of fetal origin.
  • MEASUREMENTS: Endocrine studies were performed before and after curative surgery, with assessment of tumour hormone secretion in vitro, and immunostaining of tumour tissue for a series of gonadotrope proteins.
  • CONCLUSIONS: We propose that this pituitary adenoma represents an indolent tumour of monohormonal fetal gonadotrope cells that originated early in gestation.
  • Pituitary tumours may therefore arise from abnormal persistence of fetal cell types, with extremely slow growth over many years until reaching a size threshold to generate an endocrine syndrome.
  • [MeSH-major] Adenoma / embryology. Gonadotrophs / secretion. Pituitary Neoplasms / embryology

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  • (PMID = 17054468.001).
  • [ISSN] 0300-0664
  • [Journal-full-title] Clinical endocrinology
  • [ISO-abbreviation] Clin. Endocrinol. (Oxf)
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 4TI98Z838E / Estradiol; 9002-67-9 / Luteinizing Hormone; 9002-68-0 / Follicle Stimulating Hormone
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79. Zinnamosca L, Petramala L, Cotesta D, Marinelli C, Sciomer S, Cavallaro G, Ciardi A, Massa R, De Toma G, Filetti S, Letizia C: Primary aldosteronism due to adrenocortical adenoma with concurrent ileum carcinoid tumor: case report. Endocrine; 2010 Dec;38(3):313-9
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  • [Title] Primary aldosteronism due to adrenocortical adenoma with concurrent ileum carcinoid tumor: case report.
  • In this article, we describe a case of PA due to adrenocortical adenoma ("aldosteronoma") and concurrent malignant carcinoid tumor of ileum.
  • We report a case of a nonclassical form of multiple endocrine neoplasia type 1 (MEN 1) syndrome.
  • [MeSH-major] Adrenal Cortex Neoplasms / complications. Adrenocortical Adenoma / complications. Carcinoid Tumor / complications. Hyperaldosteronism / complications. Hyperaldosteronism / etiology. Ileal Neoplasms / complications

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  • (PMID = 20972725.001).
  • [ISSN] 1559-0100
  • [Journal-full-title] Endocrine
  • [ISO-abbreviation] Endocrine
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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80. Mohammed S, Syro LV, Scheithauer BW, Abad A, Uribe H, Rotondo F, Horvath E, Cusimano M, Kovacs K: Pituitary adenoma with peliosis: a report of two cases. Endocr Pathol; 2009;20(1):41-5
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  • [Title] Pituitary adenoma with peliosis: a report of two cases.
  • The mechanism responsible for the development of peliosis remains unknown. (1) A 69 year-old man with a 6-year history of acromegaly underwent transsphenoidal surgery for pituitary adenoma.
  • Morphologic findings demonstrated a plurimorphous plurihormonal pituitary adenoma consisting of somatotrophs, lactotrophs and mammosomatotrophs.
  • The tumor contained several blood-filled cavities characteristic of peliosis. (2) A 61-year-old man with a prolactin-producing pituitary adenoma who underwent transsphenoidal surgery.
  • Peliosis in a pituitary adenoma is an intriguing finding.
  • [MeSH-major] Adenoma / pathology. Cysts / pathology. Pituitary Neoplasms / pathology

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  • (PMID = 19089655.001).
  • [ISSN] 1046-3976
  • [Journal-full-title] Endocrine pathology
  • [ISO-abbreviation] Endocr. Pathol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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81. Almeida MQ, Stratakis CA: Solid tumors associated with multiple endocrine neoplasias. Cancer Genet Cytogenet; 2010 Nov;203(1):30-6
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  • [Title] Solid tumors associated with multiple endocrine neoplasias.
  • We present an update on molecular and clinical genetics of solid tumors associated with the various multiple endocrine neoplasias (MEN) syndromes.
  • In the Carney triad, patients can manifest gastrointestinal stromal tumors, lung chondroma, paraganglioma, adrenal adenoma and pheochromocytoma, esophageal leiomyoma, and other conditions.
  • The Carney complex is yet another form of MEN that is characterized by skin tumors and pigmented lesions, myxomas, schwannomas, and various endocrine neoplasias.
  • [MeSH-major] Multiple Endocrine Neoplasia / complications. Neoplasms / genetics
  • [MeSH-minor] Adrenal Gland Neoplasms / genetics. Germ-Line Mutation. Humans. Multiple Endocrine Neoplasia Type 1 / complications. Multiple Endocrine Neoplasia Type 2a / complications. Pheochromocytoma / genetics. Thyroid Neoplasms / etiology

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  • [Copyright] Copyright © 2010 Elsevier Inc. All rights reserved.
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  • (PMID = 20951316.001).
  • [ISSN] 1873-4456
  • [Journal-full-title] Cancer genetics and cytogenetics
  • [ISO-abbreviation] Cancer Genet. Cytogenet.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Other-IDs] NLM/ NIHMS241024; NLM/ PMC2957471
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82. Morita S, Otsuki M, Izumi M, Asanuma N, Izumoto S, Saitoh Y, Yoshimine T, Kasayama S: Reduced epinephrine reserve in response to insulin-induced hypoglycemia in patients with pituitary adenoma. Eur J Endocrinol; 2007 Sep;157(3):265-70
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  • [Title] Reduced epinephrine reserve in response to insulin-induced hypoglycemia in patients with pituitary adenoma.
  • The aim of this study was to determine whether the response of catecholamine secretion to hypoglycemia is disrupted in patients with pituitary adenoma.
  • METHODS: The study population comprised 23 patients with pituitary adenoma (non-functioning adenoma or prolactinoma).
  • CONCLUSIONS: Impaired epinephrine secretion in response to insulin-induced hypoglycemia was frequently observed in patients with pituitary adenoma.

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  • (PMID = 17766707.001).
  • [ISSN] 0804-4643
  • [Journal-full-title] European journal of endocrinology
  • [ISO-abbreviation] Eur. J. Endocrinol.
  • [Language] ENG
  • [Publication-type] Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Biomarkers; 0 / Hypoglycemic Agents; 0 / Insulin; X4W3ENH1CV / Norepinephrine; YKH834O4BH / Epinephrine
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83. Balasanthiran A, Sandler B, Amonoo-Kuofi K, Swamy R, Kaniyur S, Kaplan F: Sarcoid granulomas in the parathyroid gland - a case of dual pathology: hypercalcaemia due to a parathyroid adenoma and coexistent sarcoidosis with granulomas located within the parathyroid adenoma and thyroid gland. Endocr J; 2010;57(7):603-7
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  • [Title] Sarcoid granulomas in the parathyroid gland - a case of dual pathology: hypercalcaemia due to a parathyroid adenoma and coexistent sarcoidosis with granulomas located within the parathyroid adenoma and thyroid gland.
  • Parathyroid imaging was then requested and an adenoma was identified.
  • Surprisingly, surgery revealed the coexistence of a parathyroid adenoma with the unexpected finding of sarcoid granulomas within the parathyroid and thyroid glands.
  • [MeSH-major] Adenoma / complications. Granuloma / complications. Hypercalcemia / etiology. Parathyroid Diseases / complications. Parathyroid Neoplasms / complications. Sarcoidosis / complications. Thyroid Neoplasms / complications


84. Salgado LR, Machado MC, Cukiert A, Liberman B, Kanamura CT, Alves VA: Cushing's disease arising from a clinically nonfunctioning pituitary adenoma. Endocr Pathol; 2006;17(2):191-9
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  • [Title] Cushing's disease arising from a clinically nonfunctioning pituitary adenoma.
  • A 49-yr-old woman with a large pituitary tumor leading to visual loss and galactorrhea- amenorrhea was submitted to transcranial pituitary surgery, when a clinically nonfunctioning pituitary adenoma was partially removed.
  • Histopathology and immunohistochemistry confirmed the diagnosis of "non-secreting atypical adenoma."
  • [MeSH-major] Adenoma / complications. Adenoma / pathology. Pituitary ACTH Hypersecretion / etiology. Pituitary Neoplasms / complications. Pituitary Neoplasms / pathology

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  • (PMID = 17159252.001).
  • [ISSN] 1046-3976
  • [Journal-full-title] Endocrine pathology
  • [ISO-abbreviation] Endocr. Pathol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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85. Oertli D: [Current concepts in minimal invasive endocrine surgery]. Ther Umsch; 2005 Feb;62(2):90-5
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  • [Title] [Current concepts in minimal invasive endocrine surgery].
  • Prerequisits for all of these approaches are a positive preoperative localisation of the adenoma, an intraoperative parathormone testing, and the respective surgical experience in the minimal invasive technique.
  • The experience with the laparoscopic approach to endocrine pancreatic tumours is still limited.
  • [MeSH-minor] Adenoma / diagnosis. Adenoma / surgery. Aged. Female. Humans. Insulinoma / surgery. Length of Stay. Male. Pancreatic Neoplasms / surgery. Parathyroid Neoplasms / diagnosis. Parathyroid Neoplasms / surgery. Patient Selection. Postoperative Complications. Safety. Time Factors

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  • (PMID = 15756917.001).
  • [ISSN] 0040-5930
  • [Journal-full-title] Therapeutische Umschau. Revue thérapeutique
  • [ISO-abbreviation] Ther Umsch
  • [Language] ger
  • [Publication-type] Comparative Study; English Abstract; Journal Article; Review
  • [Publication-country] Switzerland
  • [Number-of-references] 52
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86. Cuthbertson DJ, Ritchie D, Crooks D, Main G, Smith C, Vora J, Eljamel MS, Leese GP: Lymphocytic hypophysitis occurring simultaneously with a functioning pituitary adenoma. Endocr J; 2008 Aug;55(4):729-35
MedlinePlus Health Information. consumer health - Pituitary Tumors.

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  • [Title] Lymphocytic hypophysitis occurring simultaneously with a functioning pituitary adenoma.
  • It presents clinically with symptoms and signs related to either a pituitary mass or hypopituitarism, frequently mimicking a pituitary adenoma; the diagnosis of LH can only be made histologically with the presence of a dense lymphocytic infiltration usually confined to the anterior pituitary.
  • We present two case histories of patients who presented with symptoms suggestive of a functioning pituitary adenoma who also had concomitant LH confirmed histologically.
  • In both cases a joint diagnosis of a functioning pituitary adenoma with LH was made.
  • [MeSH-major] Adenoma / complications. Pituitary Diseases / complications. Pituitary Neoplasms / complications

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  • (PMID = 18497455.001).
  • [ISSN] 1348-4540
  • [Journal-full-title] Endocrine journal
  • [ISO-abbreviation] Endocr. J.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Japan
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87. Rao S, Rajkumar A, Kuruvilla S: Sellar lesion: not always a pituitary adenoma. Indian J Pathol Microbiol; 2008 Apr-Jun;51(2):269-70
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  • [Title] Sellar lesion: not always a pituitary adenoma.
  • Based on preliminary investigations, a clinical diagnosis of pituitary adenoma was made and the pituitary gland was surgically excised.
  • [MeSH-major] Adenoma / diagnosis. Pituitary Diseases / diagnosis. Pituitary Neoplasms / diagnosis. Tuberculosis, Endocrine / diagnosis

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  • (PMID = 18603706.001).
  • [ISSN] 0377-4929
  • [Journal-full-title] Indian journal of pathology & microbiology
  • [ISO-abbreviation] Indian J Pathol Microbiol
  • [Language] eng
  • [Publication-type] Case Reports; Comparative Study; Journal Article
  • [Publication-country] India
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88. Sangeetha SR, Singh N, Vender JR, Dhandapani KM: Suberoylanilide hydroxamic acid (SAHA) induces growth arrest and apoptosis in pituitary adenoma cells. Endocrine; 2009 Jun;35(3):389-96
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  • [Title] Suberoylanilide hydroxamic acid (SAHA) induces growth arrest and apoptosis in pituitary adenoma cells.
  • This study tested the efficacy of the histone deacetylase inhibitor (HDACi), suberoylanilide hydroxamic acid (SAHA) in GH3, and MMQ pituitary adenoma cells.
  • Clinically achievable concentrations of SAHA (500 nM-4 microM) induced growth arrest and increased cell death in GH3 pituitary adenoma cells.
  • Together, these findings support a possible utility for SAHA alone or in combination with radiation for the treatment of pituitary adenoma.
  • [MeSH-major] Adenoma / pathology. Apoptosis / drug effects. Cell Proliferation / drug effects. Hydroxamic Acids / pharmacology. Pituitary Neoplasms / pathology

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  • (PMID = 19291426.001).
  • [ISSN] 1355-008X
  • [Journal-full-title] Endocrine
  • [ISO-abbreviation] Endocrine
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Antineoplastic Agents; 0 / Enzyme Inhibitors; 0 / Histone Deacetylase Inhibitors; 0 / Hydroxamic Acids; 58IFB293JI / vorinostat; EC 2.4.2.30 / Poly(ADP-ribose) Polymerases; EC 3.4.22.- / Caspases
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89. Imachi H, Murao K, Kontani K, Yokomise H, Miyai Y, Yamamoto Y, Kushida Y, Haba R, Ishida T: Ectopic mediastinal parathyroid adenoma: a cause of acute pancreatitis. Endocrine; 2009 Oct;36(2):194-7
MedlinePlus Health Information. consumer health - Pancreatitis.

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  • [Title] Ectopic mediastinal parathyroid adenoma: a cause of acute pancreatitis.
  • Microscopic examination revealed the presence of a parathyroid adenoma (1.3 x 0.4 cm(2)) adjacent to the atrophic parathyroid gland in right thymus gland.
  • We report the case of a patient diagnosed with primary hyperparathyroidism due to an ectopic mediastinal parathyroid adenoma.
  • An ectopic mediastinal parathyroid adenoma may manifest as an episode of acute pancreatitis.
  • Preoperative investigation to determine the exact location of an adenoma should include two types of imaging studies, preferably (99m)Tc sestamibi scintigraphy and CT of the neck and chest.
  • [MeSH-major] Adenoma / complications. Choristoma. Mediastinal Diseases / complications. Pancreatitis / etiology. Parathyroid Glands. Parathyroid Neoplasms / complications

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  • (PMID = 19598003.001).
  • [ISSN] 1559-0100
  • [Journal-full-title] Endocrine
  • [ISO-abbreviation] Endocrine
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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90. Unger N, Petersenn S, Mann K: [Diagnosis and therapy of endocrine hypertension]. Med Klin (Munich); 2006 Mar 22;101 Suppl 1:170-2
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  • [Title] [Diagnosis and therapy of endocrine hypertension].
  • A unilateral adrenal adenoma has to be separated from bilateral adrenal hyperplasia.
  • Endocrine hypertension due to adrenal tumors may be treated by minimally invasive surgery or specific antihypertensive treatment.
  • [MeSH-major] Adrenal Gland Neoplasms / diagnosis. Adrenocortical Adenoma / diagnosis. Hyperaldosteronism / diagnosis. Hypertension / etiology. Pheochromocytoma / diagnosis. Pituitary ACTH Hypersecretion / diagnosis

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  • (PMID = 16802547.001).
  • [ISSN] 0723-5003
  • [Journal-full-title] Medizinische Klinik (Munich, Germany : 1983)
  • [ISO-abbreviation] Med. Klin. (Munich)
  • [Language] ger
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] Germany
  • [Chemical-registry-number] WI4X0X7BPJ / Hydrocortisone; X4W3ENH1CV / Norepinephrine; YKH834O4BH / Epinephrine
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91. Moskowitz SI, Hamrahian A, Prayson RA, Pineyro M, Lorenz RR, Weil RJ: Concurrent lymphocytic hypophysitis and pituitary adenoma. Case report and review of the literature. J Neurosurg; 2006 Aug;105(2):309-14
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  • [Title] Concurrent lymphocytic hypophysitis and pituitary adenoma. Case report and review of the literature.
  • Evidence suggests that the cause is autoimmune, and the symptoms are usually related to either a mass effect or endocrine dysfunction.
  • Microscopic and immunohistopathological examinations revealed a nonsecreting pituitary adenoma with concurrent lymphocytic adenohypophysitis.
  • This is the first documented case of LyH in the setting of a null-cell pituitary adenoma.
  • The authors review the related literature and outline potential mechanisms for the concurrent development of LyH and a pituitary adenoma.
  • [MeSH-major] Adenoma / complications. Lymphocytosis / complications. Pituitary Diseases / complications. Pituitary Neoplasms / complications


92. Dorward NL: Endocrine outcomes in endoscopic pituitary surgery: a literature review. Acta Neurochir (Wien); 2010 Aug;152(8):1275-9
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  • [Title] Endocrine outcomes in endoscopic pituitary surgery: a literature review.
  • Whilst some advantages of minimal access have been recognised, the information on endocrine outcomes has been slow to accumulate.
  • METHOD: This literature review examined all the endoscopic papers for endocrine results and complication rates.
  • FINDINGS: The review revealed that the fully endoscopic method is the equal of the microscopic technique for endocrine remission in functioning pituitary tumours.
  • CONCLUSIONS: Endoscopic pituitary surgery offers significant advantages over the microscopic technique and should become the standard method of pituitary adenoma surgery.
  • [MeSH-major] Adenoma / surgery. Endocrine System Diseases / surgery. Endoscopy / adverse effects. Neurosurgical Procedures / adverse effects. Pituitary Neoplasms / surgery. Postoperative Complications / epidemiology


93. Takenaka Y, Tsukamoto T, Mizoshita T, Ogasawara N, Hirano N, Otsuka T, Ban H, Nakamura T, Yamamura Y, Kaminishi M, Tatematsu M: Gastric and intestinal phenotypic correlation between exocrine and endocrine components in human stomach tumors. Histol Histopathol; 2007 03;22(3):273-84
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  • [Title] Gastric and intestinal phenotypic correlation between exocrine and endocrine components in human stomach tumors.
  • To clarify whether our hypothesis is correct or not, we analyzed the expression of Exo-cell and endocrine cell (End-cell) markers in a series of lesions for comparison.
  • We evaluated chromogranin A (CgA) expression in 37 early and 73 advanced stomach cancers, in 30 stomach adenomas, in 8 carcinoid tumors, and in 4 endocrine cell carcinomas (ECCs) with assessment of gastric and/or intestinal (G/I) phenotypes in both Exo-cell and End-cell by immunohistochemistry.
  • All of the adenoma cases had the intestinal Exo-cell phenotypic expression, with the positive link between Exo-cell and End-cell G/I phenotypes.
  • [MeSH-major] Adenocarcinoma / secondary. Adenoma / pathology. Biomarkers, Tumor / metabolism. Carcinoid Tumor / pathology. Neoplasm Proteins / metabolism. Stomach Neoplasms / pathology
  • [MeSH-minor] Adult. Aged. Cell Count. Chromogranin A / metabolism. Endocrine Glands / metabolism. Endocrine Glands / pathology. Exocrine Glands / metabolism. Exocrine Glands / pathology. Female. Gastric Mucosa / metabolism. Gastric Mucosa / pathology. Humans. Intestinal Mucosa / metabolism. Intestinal Mucosa / pathology. Lymphatic Metastasis / pathology. Male. Middle Aged. Phenotype

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  • (PMID = 17163401.001).
  • [ISSN] 1699-5848
  • [Journal-full-title] Histology and histopathology
  • [ISO-abbreviation] Histol. Histopathol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Spain
  • [Chemical-registry-number] 0 / Biomarkers, Tumor; 0 / Chromogranin A; 0 / Neoplasm Proteins
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94. Chaychi L, Chaidarun S, Golding A, Kinlaw WB, Memoli V, Siegel A: Unusual recurrence of hypercalcemia due to concurrent parathyroid adenoma and parathyroid sarcoidosis with lymph node involvement. Endocr Pract; 2010 May-Jun;16(3):463-7
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Unusual recurrence of hypercalcemia due to concurrent parathyroid adenoma and parathyroid sarcoidosis with lymph node involvement.
  • OBJECTIVE: To describe a patient presenting with the rare findings of synchronous parathyroid adenoma and parathyroid sarcoidosis.
  • Pathologic examination of the resected gland confirmed the diagnosis of parathyroid adenoma, and subsequent review disclosed the presence of noncaseating granulomas within the adenoma.
  • Recurrent hypercalcemia after successful resection of a parathyroid adenoma may require consideration of potential causes other than the initial diagnosis.

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  • (PMID = 20061287.001).
  • [ISSN] 1934-2403
  • [Journal-full-title] Endocrine practice : official journal of the American College of Endocrinology and the American Association of Clinical Endocrinologists
  • [ISO-abbreviation] Endocr Pract
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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95. Choe JH, Lee KS, Jeun SS, Cho JH, Hong YK: Endocrine outcome of endoscopic endonasal transsphenoidal surgery in functioning pituitary adenomas. J Korean Neurosurg Soc; 2008 Sep;44(3):151-5
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Endocrine outcome of endoscopic endonasal transsphenoidal surgery in functioning pituitary adenomas.
  • OBJECTIVE: Microscopic and endoscopic transsphenoidal approach (TSA) are major surgical techniques in the treatment of pituitary adenoma.
  • Endoscopic endonasal transsphenoidal approach (EETSA) has been increasingly used for pituitary adenomas, however, its surgical outcome particularly in functioning pituitary adenoma has been debated.
  • Here, we investigated the endocrine outcome of the patients with growth hormone (GH) and adrenocorticotropic hormone (ACTH) secreting pituitary adenoma treated by EETSA.
  • METHODS: We treated 80 patients with pituitary adenoma by EETSA since 2004, of which 12 patients were affected by functioning pituitary adenomas (9 GH, 3 ACTH, 0 PRL; 9 macro, 3 micro).
  • Surgical outcome of those patients treated by EETSA was compared with that of the 11 functioning pituitary adenoma patients (8 GH, 3 ACTH; 8 macro, 3 micro) who underwent sublabial microscopic TSA between 1997 and 2003.

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  • (PMID = 19096666.001).
  • [ISSN] 2005-3711
  • [Journal-full-title] Journal of Korean Neurosurgical Society
  • [ISO-abbreviation] J Korean Neurosurg Soc
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Korea (South)
  • [Other-IDs] NLM/ PMC2588303
  • [Keywords] NOTNLM ; ACTH-secreting pituitary adenoma / Endoscopy / GH-secreting pituitary adenoma / Transsphenoidal approach
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96. Sakamoto N, Tojo K, Saito T, Fujimoto K, Isaka T, Tajima N, Ikeda K, Yamada H, Furuta N, Sasano H: Coexistence of aldosterone-producing adrenocortical adenoma and pheochromocytoma in an ipsilateral adrenal gland. Endocr J; 2009;56(2):213-9
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  • [Title] Coexistence of aldosterone-producing adrenocortical adenoma and pheochromocytoma in an ipsilateral adrenal gland.
  • Furthermore, selective adrenal venous sampling with intravenous ACTH infusion indicated aldosterone-producing adrenocortical adenoma (APA) in left adrenal gland.
  • [MeSH-major] Adrenal Gland Neoplasms / pathology. Adrenocortical Adenoma / pathology. Pheochromocytoma / pathology

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  • (PMID = 19023159.001).
  • [ISSN] 1348-4540
  • [Journal-full-title] Endocrine journal
  • [ISO-abbreviation] Endocr. J.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Japan
  • [Chemical-registry-number] 4964P6T9RB / Aldosterone
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97. Atabek ME, Pirgon O, Sert A, Esen HH: Extensive brown tumors caused by parathyroid adenoma in an adolescent patient. Eur J Pediatr; 2008 Jan;167(1):117-9

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Extensive brown tumors caused by parathyroid adenoma in an adolescent patient.
  • Primary hyperparathyroidism is a rare endocrine disease in children and young adults.
  • Moreover, the skeletal involvement in primary hyperparathyroidism secondary to parathyroid adenoma is extremely rare.
  • We report on an adolescent girl with multiple brown tumors and a history of recurrent fractures as the manifestation of primary hyperparathyroidism associated with a parathyroid adenoma.
  • [MeSH-major] Adenoma / complications. Hyperparathyroidism, Primary / etiology. Osteitis Fibrosa Cystica / etiology. Parathyroid Neoplasms / complications

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  • (PMID = 17273830.001).
  • [ISSN] 1432-1076
  • [Journal-full-title] European journal of pediatrics
  • [ISO-abbreviation] Eur. J. Pediatr.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / Parathyroid Hormone
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98. Leenstra JL, Tanaka S, Kline RW, Brown PD, Link MJ, Nippoldt TB, Young WF Jr, Pollock BE: Factors associated with endocrine deficits after stereotactic radiosurgery of pituitary adenomas. Neurosurgery; 2010 Jul;67(1):27-32; discussion 32-3
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Factors associated with endocrine deficits after stereotactic radiosurgery of pituitary adenomas.
  • OBJECTIVE: To analyze the factors associated with anterior pituitary deficits after pituitary adenoma stereotactic radiosurgery (SRS).
  • CONCLUSION: New endocrine deficits after pituitary adenoma radiosurgery were correlated with increasing radiation dose to the pituitary gland.
  • [MeSH-major] Adenoma / surgery. Hypopituitarism / etiology. Pituitary Gland, Anterior / radiation effects. Pituitary Gland, Anterior / surgery. Pituitary Neoplasms / surgery. Postoperative Complications / etiology. Radiosurgery / adverse effects

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  • (PMID = 20559089.001).
  • [ISSN] 1524-4040
  • [Journal-full-title] Neurosurgery
  • [ISO-abbreviation] Neurosurgery
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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99. Mazza A, Zamboni S, Armigliato M, Zennaro R, Cuppini S, Rempelou P, Rubello D, Pessina AC: Endocrine arterial hypertension: diagnostic approach in clinical practice. Minerva Endocrinol; 2008 Jun;33(2):127-46
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Endocrine arterial hypertension: diagnostic approach in clinical practice.
  • Endocrine arterial hypertension (EAH) a condition in which hormone excess results in clinically significant hypertension is a rare cause of hypertension.
  • Differential diagnosis of PA requires adrenal gland imaging by computed tomography (CT) or magnetic resonance imaging (MRI), biochemical testing of the aldosterone response to posture, and selective adrenal venous sampling to differentiate unilateral aldosterone-producing adenoma from bilateral hyperplasia.
  • [MeSH-minor] Adenoma / complications. Adrenal Gland Neoplasms / complications. Aldosterone / blood. Algorithms. Catecholamines / blood. Cushing Syndrome / complications. Diagnosis, Differential. Glucocorticoids / blood. Humans. Hydrocortisone / urine. Hyperaldosteronism / complications. Hyperaldosteronism / diagnosis. Hyperaldosteronism / etiology. Mass Screening. Renin / blood. Tomography, X-Ray Computed

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  • (PMID = 18272953.001).
  • [ISSN] 0391-1977
  • [Journal-full-title] Minerva endocrinologica
  • [ISO-abbreviation] Minerva Endocrinol.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Italy
  • [Chemical-registry-number] 0 / Catecholamines; 0 / Glucocorticoids; 4964P6T9RB / Aldosterone; EC 3.4.23.15 / Renin; WI4X0X7BPJ / Hydrocortisone
  • [Number-of-references] 100
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100. Yan QZ, Li HZ, Pan DL: [Causes of delaying diagnosis of primary hyperaldosteronism with adrenal adenoma and strategy therefore]. Zhonghua Yi Xue Za Zhi; 2006 Nov 14;86(42):3002-4
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Causes of delaying diagnosis of primary hyperaldosteronism with adrenal adenoma and strategy therefore].
  • OBJECTIVE: To analyze the causes of delaying diagnosis of primary hyperaldosteronism with adrenal adenoma and discuss corrective strategies.
  • METHODS: The clinical data of 267 patients of primary hyperaldosteronism with adrenal adenoma confirmed by operation 1995 - 2005 were analyzed.
  • 95.9% of the patients were diagnosed as with primary hypertension at the first visit without receiving relevant imaging examination of adrenal and endocrine examination.
  • Adrenal adenoma was discovered by computed tomography with thin coat screening in 267 patients and by ultrasonography in 151 patients.
  • [MeSH-major] Adrenal Cortex Neoplasms / diagnosis. Adrenocortical Adenoma / diagnosis. Hyperaldosteronism / diagnosis

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  • (PMID = 17288816.001).
  • [ISSN] 0376-2491
  • [Journal-full-title] Zhonghua yi xue za zhi
  • [ISO-abbreviation] Zhonghua Yi Xue Za Zhi
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] China
  • [Chemical-registry-number] 0 / Biomarkers, Tumor; 4964P6T9RB / Aldosterone; EC 3.4.23.4 / Chymosin
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