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Items 1 to 14 of about 14
1. Seimiya YM, Takahashi M, Furukawa T, Mizutani K, Kimura K, Haritani M: An aged bull with concurrent thyroid C cell carcinoma, adrenal pheochromocytoma and pituitary chromophobe adenoma. J Vet Med Sci; 2009 Feb;71(2):225-8
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  • [Title] An aged bull with concurrent thyroid C cell carcinoma, adrenal pheochromocytoma and pituitary chromophobe adenoma.
  • Pathological examination disclosed multiple endocrine tumors including thyroid C cell carcinoma with metastases to the cervical lymph nodes and lung, adrenal pheochromocytoma and pituitary chromophobe adenoma in the pars distalis.


2. Brown RL, Muzzafar T, Wollman R, Weiss RE: A pituitary carcinoma secreting TSH and prolactin: a non-secreting adenoma gone awry. Eur J Endocrinol; 2006 May;154(5):639-43
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  • [Title] A pituitary carcinoma secreting TSH and prolactin: a non-secreting adenoma gone awry.
  • To our knowledge, only one case of a TSH-secreting carcinoma has previously been reported.
  • We describe here a second patient with a pituitary carcinoma producing TSH and prolactin (PRL).
  • Pathologic examination revealed a chromophobe adenoma with increased mitotic forms.
  • The patient completed a course of external beam radiation to the pituitary and was prescribed l-thyroxine, bromocriptine, and hydrocortisone.
  • Emergent resection of the larger mass revealed a pituitary cancer with positive staining for PRL, but not for TSH.
  • Nine months later, the patient underwent further debulking of metastatic disease.
  • Although development of a carcinoma from a pituitary adenoma is very rare (<0.5%), macroadenomas that become hormonally active should be suspect for transformation into pituitary cancer.

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  • (PMID = 16645009.001).
  • [ISSN] 0804-4643
  • [Journal-full-title] European journal of endocrinology
  • [ISO-abbreviation] Eur. J. Endocrinol.
  • [Language] ENG
  • [Grant] United States / NIDDK NIH HHS / DK / DK07011; United States / NCRR NIH HHS / RR / RR00055; United States / NCRR NIH HHS / RR / RR18372
  • [Publication-type] Case Reports; Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] England
  • [Chemical-registry-number] 9002-62-4 / Prolactin; 9002-71-5 / Thyrotropin
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3. Minami I, Tateno T, Yoshimoto T, Doi M, Izumiyama H, Akashi T, Hirata Y: Subclinical Cushings disease with amelioration of metabolic comorbidities after removal of pituitary tumor. Intern Med; 2006;45(21):1231-5
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  • [Title] Subclinical Cushings disease with amelioration of metabolic comorbidities after removal of pituitary tumor.
  • A 49-year-old woman with hypertension, obesity and impaired glucose tolerance (IGT) was admitted for evaluation of pituitary incidentaloma.
  • Although she presented no Cushingoid feature, endocrine examination of hypothalamo-pituitary-adrenal (HPA) axis showed elevated basal plasma ACTH and cortisol levels, their lack of circadian rhythm, non-suppressibility to low-dose (1 mg) dexamethasone, and responsiveness to CRH, suggesting autonomous ACTH secretion from a pituitary tumor.
  • She underwent transsphenoidal surgery, and was diagnosed as chromophobe adenoma with positive ACTH immunoreactivity.
  • Thus, her metabolic comorbidities are likely due to subclinical Cushings disease.
  • [MeSH-major] Glucose Intolerance / surgery. Hypertension / surgery. Obesity / surgery. Pituitary ACTH Hypersecretion / surgery. Pituitary Neoplasms / surgery
  • [MeSH-minor] Female. Humans. Middle Aged. Pituitary-Adrenal System / metabolism. Pituitary-Adrenal System / surgery

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  • (PMID = 17139124.001).
  • [ISSN] 1349-7235
  • [Journal-full-title] Internal medicine (Tokyo, Japan)
  • [ISO-abbreviation] Intern. Med.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Japan
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4. Kageyama K, Ikeda H, Nigawara T, Sakihara S, Suda T: Expression of adrenocorticotropic hormone, prolactin and transcriptional factors in clinically nonfunctioning pituitary adenoma. Endocr J; 2007 Dec;54(6):961-8
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  • [Title] Expression of adrenocorticotropic hormone, prolactin and transcriptional factors in clinically nonfunctioning pituitary adenoma.
  • We describe here a case of a clinically nonfunctioning pituitary adenoma, but with expression of ACTH and PRL.
  • A 42-year-old woman was referred to our department for further evaluation of pituitary tumor.
  • Based on these findings we did not clinically suspect ACTH-producing tumor, however immunohistochemistry revealed ACTH immunoreactivity in the pituitary adenoma.
  • Therefore, the tumor was considered a silent corticotroph adenoma.
  • Ptx1, Neuro D1, and T pit were densely expressed and Pit-1 was sparsely expressed in the nuclei of adenoma cells.
  • [MeSH-major] Adenoma, Chromophobe / metabolism. Adrenocorticotropic Hormone / biosynthesis. Pituitary Neoplasms / metabolism. Prolactin / biosynthesis. Transcription Factors / biosynthesis

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  • (PMID = 18079591.001).
  • [ISSN] 1348-4540
  • [Journal-full-title] Endocrine journal
  • [ISO-abbreviation] Endocr. J.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Japan
  • [Chemical-registry-number] 0 / Basic Helix-Loop-Helix Transcription Factors; 0 / ERGIC2 protein, human; 0 / Homeodomain Proteins; 0 / NEUROD1 protein, human; 0 / T-Box Domain Proteins; 0 / TBX19 protein, human; 0 / Transcription Factor Pit-1; 0 / Transcription Factors; 0 / Vesicular Transport Proteins; 12629-01-5 / Human Growth Hormone; 9002-60-2 / Adrenocorticotropic Hormone; 9002-62-4 / Prolactin; 9002-71-5 / Thyrotropin
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5. Chung TT, Evanson J, Walker D, Akker SA, Besser GM, Monson JP, Grossman AB, Drake WM: Safety of GH replacement in hypopituitary patients with nonirradiated pituitary and peripituitary tumours. Clin Endocrinol (Oxf); 2008 Jun;68(6):965-9
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  • [Title] Safety of GH replacement in hypopituitary patients with nonirradiated pituitary and peripituitary tumours.
  • BACKGROUND: Published data suggest that growth hormone replacement (GHR) may be given safely to patients with hypopituitarism consequent upon a pituitary/peripituitary tumour.
  • However, a preponderance of patients treated with external pituitary irradiation were included.
  • OBJECTIVE: To assess the safety of GHR in nonirradiated pituitary/peripituitary tumour.
  • PATIENTS: We imaged prospectively the pituitary glands of 48 patients (18 males; mean age 51.6 years range 21-77) who had adult onset growth hormone deficiency (AO-GHD) after appropriate treatment for a pituitary/peripituitary tumour but who did not receive external pituitary irradiation.
  • Pituitary surveillance imaging was performed prior to the commencement of GHR, at 6-12 months and then yearly.
  • Three patients were judged to have an apparent increase in tumour volume and/or marker, although only one was thought to be possibly GH related--a patient with a cystic chromophobe adenoma who demonstrated a marginal increase in residual tumour volume 4 years after commencement of GHR.
  • CONCLUSION: These data add to the growing body of evidence for the safety of GHR in hypopituitary patients consequent upon pituitary/peripituitary mass lesions and represents the first reported series in a heterogeneous group of nonirradiated patients.
  • [MeSH-major] Human Growth Hormone / adverse effects. Human Growth Hormone / therapeutic use. Hypopituitarism / drug therapy. Pituitary Neoplasms / drug therapy

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  • (PMID = 18031317.001).
  • [ISSN] 1365-2265
  • [Journal-full-title] Clinical endocrinology
  • [ISO-abbreviation] Clin. Endocrinol. (Oxf)
  • [Language] eng
  • [Publication-type] Clinical Trial; Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 12629-01-5 / Human Growth Hormone
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6. Díaz-Rubio JL, González-Carrillo CP, Hernández-Abrego MP, Quiroz-Mercado H: [Congenital hypertrophy of the retinal pigment epithelium in a patient with a pituitary tumour]. Rev Neurol; 2007 Nov 1-15;45(9):571-2
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  • [Title] [Congenital hypertrophy of the retinal pigment epithelium in a patient with a pituitary tumour].
  • [MeSH-major] Adenoma, Chromophobe / complications. Diagnostic Errors. Pigment Epithelium of Eye / abnormalities. Pituitary Neoplasms / complications

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  • (PMID = 17979088.001).
  • [ISSN] 0210-0010
  • [Journal-full-title] Revista de neurologia
  • [ISO-abbreviation] Rev Neurol
  • [Language] spa
  • [Publication-type] Case Reports; Letter
  • [Publication-country] Spain
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7. Rockwell J, Monroe WE, Tromblee TC: Spontaneous hypoadrenocorticism in a dog after a diagnosis of hyperadrenocorticism. J Vet Intern Med; 2005 May-Jun;19(3):299-300
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  • [Title] Spontaneous hypoadrenocorticism in a dog after a diagnosis of hyperadrenocorticism.

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  • [CommentOn] J Vet Intern Med. 2005 Mar-Apr;19(2):255-8 [15822572.001]
  • (PMID = 15954541.001).
  • [ISSN] 0891-6640
  • [Journal-full-title] Journal of veterinary internal medicine
  • [ISO-abbreviation] J. Vet. Intern. Med.
  • [Language] ENG
  • [Publication-type] Case Reports; Comment; Letter
  • [Publication-country] United States
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8. D'Amore M, Minenna G, D'Amore S, Scagliusi P, Caprio S: [The strange case of a patient affected by acromegaly with osteoporomalacia without hypogonadism]. Reumatismo; 2005 Dec;57(4):291-4
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  • [Title] [The strange case of a patient affected by acromegaly with osteoporomalacia without hypogonadism].
  • Acromegaly is a rare disease that, in the majority of cases, is due to the presence of a benign growth hormone (GH)-producing tumor of the pituitary.
  • [MeSH-major] Acromegaly / diagnosis. Adenoma, Chromophobe / diagnosis. Osteomalacia / diagnosis. Osteoporosis / diagnosis. Pituitary Neoplasms / diagnosis

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  • (PMID = 16380758.001).
  • [ISSN] 0048-7449
  • [Journal-full-title] Reumatismo
  • [ISO-abbreviation] Reumatismo
  • [Language] ita
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Italy
  • [Chemical-registry-number] 0 / Androgens; 12629-01-5 / Human Growth Hormone; 3XMK78S47O / Testosterone
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9. Delides A, Velegrakis G, Kontogeorgos G, Karagianni E, Nakas D, Helidonis E: Familial bilateral acinic cell carcinoma of the parotid synchronous with pituitary adenoma: case report. Head Neck; 2005 Sep;27(9):825-8
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  • [Title] Familial bilateral acinic cell carcinoma of the parotid synchronous with pituitary adenoma: case report.
  • Only one case of a familial recurrence of such a neoplasm and 16 cases of bilateral tumors have been reported.
  • METHODS: History files and histologic reports of a patient with bilateral multifocal acinic cell carcinoma of the parotid and a synchronous pituitary adenoma, and of the patient's sister and his father, also treated for parotid tumours, were retrieved.
  • A pituitary tumor was a chromophobe gonotrophic adenoma.
  • CONCLUSIONS: This is the 17th case of bilateral acinic cell carcinoma of the parotid gland and the second reported case with a familial recurrence.
  • It is the first with a synchronous pituitary adenoma.
  • [MeSH-major] Adenoma / diagnosis. Carcinoma, Acinar Cell / diagnosis. Neoplasms, Multiple Primary / diagnosis. Parotid Neoplasms / diagnosis. Pituitary Neoplasms / diagnosis
  • [MeSH-minor] Genetic Predisposition to Disease. Humans. Male. Middle Aged

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  • [Copyright] (c) 2005 Wiley Periodicals, Inc.
  • (PMID = 15920750.001).
  • [ISSN] 1043-3074
  • [Journal-full-title] Head & neck
  • [ISO-abbreviation] Head Neck
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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10. Ruggeri RM, Santarpia L, Curtò L, Torre ML, Galatioto M, Galatioto S, Trimarchi F, Cannavò S: Non-functioning pituitary adenomas infrequently harbor G-protein gene mutations. J Endocrinol Invest; 2008 Nov;31(11):946-9
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  • [Title] Non-functioning pituitary adenomas infrequently harbor G-protein gene mutations.
  • BACKGROUND: Mutations of the genes encoding the alpha subunit of the stimulatory G protein (Gs) and of the inhibiting Gi2 protein (GNAS1 and GNAI2 genes, respectively) have been described in various endocrine neoplasias, including pituitary tumors.
  • AIM: To search for mutations of GNAS1 and GNAI2 in a continuous series of non-functioning pituitary adenoma (NFPA) patients neurosurgically treated.
  • CONCLUSIONS: This finding suggests and confirms that G-protein mutations are rare and not crucial in NFPA development.
  • [MeSH-major] Adenoma, Acidophil / genetics. Adenoma, Chromophobe / genetics. GTP-Binding Protein alpha Subunits, Gs / genetics. Intracellular Signaling Peptides and Proteins / genetics. Pituitary Neoplasms / genetics

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  • (PMID = 19169048.001).
  • [ISSN] 1720-8386
  • [Journal-full-title] Journal of endocrinological investigation
  • [ISO-abbreviation] J. Endocrinol. Invest.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Italy
  • [Chemical-registry-number] 0 / Codon; 0 / Intracellular Signaling Peptides and Proteins; 0 / URI1 protein, human; EC 3.6.1.- / GNAS protein, human; EC 3.6.5.1 / GTP-Binding Protein alpha Subunits, Gs
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11. Gołkowski F, Buziak-Bereza M, Stefańska A, Trofimiuk M, Pantofliński J, Huszno B, Czepko R, Adamek D: [A case of GH and TSH secreting pituitary macroadenoma]. Przegl Lek; 2006;63(2):106-8
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  • [Title] [A case of GH and TSH secreting pituitary macroadenoma].
  • A case of GH and TSH secreting pituitary macroadenoma is reported.
  • NMR pituitary imaging revealed intra and extrasellar tumor.
  • Histological and immunohistochemical examination of the tissue obtained at transsphenoidal surgery showed GH and TSH secreting adenoma.
  • The very low plasma levels of TSH and free thyroid hormones after surgery and immunohistochemical examination suggest central hyperthyroidism due to TSH secreting pituitary tumor (thyrotropinoma).
  • [MeSH-major] Adenoma, Chromophobe / secretion. Adenoma, Chromophobe / surgery. Growth Hormone / secretion. Pituitary Neoplasms / secretion. Pituitary Neoplasms / surgery. Thyrotropin / secretion
  • [MeSH-minor] Acromegaly / diagnosis. Acromegaly / etiology. Acromegaly / surgery. Female. Humans. Hyperthyroidism / blood. Hyperthyroidism / etiology. Middle Aged. Pituitary Gland / pathology. Pituitary Gland / surgery

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  • (PMID = 16967720.001).
  • [ISSN] 0033-2240
  • [Journal-full-title] Przegla̧d lekarski
  • [ISO-abbreviation] Prz. Lek.
  • [Language] pol
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Poland
  • [Chemical-registry-number] 9002-71-5 / Thyrotropin; 9002-72-6 / Growth Hormone
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12. Brunotte I, Haubitz B, Winter R, Meyer MW: [Differential diagnosis in visual field defects of glaucoma patients]. Klin Monbl Augenheilkd; 2008 Feb;225(2):169-72
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  • We recommend the performance of a radiological examination in patients with visual field defects if the intraocular pressure is normal and thus glaucoma may not be the cause of the defects.
  • [MeSH-major] Adenoma, Chromophobe / diagnosis. Glaucoma / diagnosis. Meningeal Neoplasms / diagnosis. Meningioma / diagnosis. Optic Nerve Diseases / diagnosis. Pituitary Neoplasms / diagnosis. Vision Disorders / diagnosis. Visual Fields


13. Zhang J, Lefkowitz RA, Ishill NM, Wang L, Moskowitz CS, Russo P, Eisenberg H, Hricak H: Solid renal cortical tumors: differentiation with CT. Radiology; 2007 Aug;244(2):494-504
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  • PURPOSE: To retrospectively determine if solid renal cortical tumors can be differentiated on computed tomographic (CT) images on the basis of their morphologic features and enhancement patterns.
  • RESULTS: Of the 198 renal tumors (median size, 3.4 cm; range, 1.1-20.0 cm) included in this study, 108 (55%) were clear cell renal cell carcinomas (RCCs); 30 (15%), papillary lesions; 24 (12%), chromophobe adenomas; 14 (7%), oncocytomas; six (3%), lipid-poor angiomyolipomas; and 16 (8%), other or unclassified renal tumors.
  • This pattern was highly predictive of clear cell RCC (odds ratio of 22 and 54 for readers 1 and 2, respectively, for comparison with homogeneous pattern), whereas the homogeneous and peripheral enhancing patterns were more predictive of less aggressive papillary and chromophobe lesions.
  • Clear cell RCCs and oncocytomas tended to be hypervascular, chromophobe lesions and angiomyolipomas tended to enhance moderately, and papillary lesions were mostly hypovascular.

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  • (PMID = 17641370.001).
  • [ISSN] 0033-8419
  • [Journal-full-title] Radiology
  • [ISO-abbreviation] Radiology
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Contrast Media; 4419T9MX03 / Iohexol
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14. Provenzale JM: Morphology predicts oncology: a commentary on Vezina and Sutton's article, "Prolactin-secreting pituitary microadenomas: roentgenologic diagnosis". AJR Am J Roentgenol; 2007 Jan;188(1):9-10
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  • [Title] Morphology predicts oncology: a commentary on Vezina and Sutton's article, "Prolactin-secreting pituitary microadenomas: roentgenologic diagnosis".
  • [MeSH-major] Adenoma, Chromophobe / radiography. Craniopharyngioma / radiography. Craniopharyngioma / secretion. Lactation Disorders / etiology. Pituitary Neoplasms / complications. Pituitary Neoplasms / secretion. Prolactin / secretion

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  • [CommentOn] Am J Roentgenol Radium Ther Nucl Med. 1974 Jan;120(1):46-54 [4855697.001]
  • (PMID = 17179340.001).
  • [ISSN] 1546-3141
  • [Journal-full-title] AJR. American journal of roentgenology
  • [ISO-abbreviation] AJR Am J Roentgenol
  • [Language] eng
  • [Publication-type] Comment; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Hormones, Ectopic; 9002-62-4 / Prolactin
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