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1. Bennett LM, McAllister KA, Ward T, Malphurs J, Collins NK, Seely JC, Davis BJ, Wiseman RW: Mammary tumor induction and premature ovarian failure in ApcMin mice are not enhanced by Brca2 deficiency. Toxicol Pathol; 2001 Jan-Feb;29(1):117-25
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Female and male offspring treated with a single IP injection of 50 mg/kg N-ethyl-N-nitrosourea (ENU) at 35 days of age developed mammary adenoacanthomas by 100 days of age.
  • The female Apc-mutant and Brca2/Apc double-mutant progeny had mean mammary tumor multiplicities of 6.7+/-2.8 and 7.2+/-2.7, respectively, compared to wild-type and Brca2-mutant females, which had mean mammary tumor multiplicities of 0.1+/-0.4 and 0.3+/-0.5, respectively.
  • [MeSH-major] Adenocarcinoma / chemically induced. Adenocarcinoma / genetics. Genes, APC / genetics. Mammary Neoplasms, Experimental / chemically induced. Mammary Neoplasms, Experimental / genetics. Metaplasia / chemically induced. Metaplasia / genetics. Neoplasm Proteins / deficiency. Neoplasm Proteins / genetics. Ovarian Diseases / chemically induced. Ovarian Diseases / genetics. Transcription Factors / deficiency. Transcription Factors / genetics
  • [MeSH-minor] Animals. BRCA2 Protein. Body Weight / drug effects. Carcinogens / toxicity. Ethylnitrosourea / toxicity. Female. Heterozygote. Male. Mice. Mice, Inbred C57BL. Mice, Knockout


2. Ghourab S: Synchronous endometrioid carcinoma of the ovary and endometrium associated with ovulation induction. Saudi Med J; 2001 Oct;22(10):914-6
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Between the first reported case in 1982 and the end of year 2000, there have been 44 cases of ovarian carcinoma reported to occur in women previously treated with ovulation induction drugs.
  • Most of these tumors were of the serous type with low malignant potential.
  • Intraoperative and histological examinations showed stage 1A endometrioid ovarian cancer and well-differentiated endometrial adenoacanthoma with minimal myometrial invasion.
  • [MeSH-major] Adenocarcinoma / etiology. Endometrial Neoplasms / etiology. Neoplasms, Multiple Primary / etiology. Ovarian Neoplasms / etiology. Ovulation Induction / adverse effects
  • [MeSH-minor] Adult. Female. Humans. Metaplasia

  • MedlinePlus Health Information. consumer health - Ovarian Cancer.
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  • (PMID = 11744954.001).
  • [ISSN] 0379-5284
  • [Journal-full-title] Saudi medical journal
  • [ISO-abbreviation] Saudi Med J
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Saudi Arabia
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3. Franchimont D, Covas A, Brasseur C, Laethem JL, El-Nakadi I, Devière J: Newly developed Barrett's esophagus after subtotal esophagectomy. Endoscopy; 2003 Oct;35(10):850-3
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • BACKGROUND AND STUDY AIMS: More detailed information regarding the early mucosal events that lead to intestinal metaplasia would be very beneficial for understanding the pathogenesis of Barrett's esophagus (BE).
  • RESULTS: Based on the Savary-Miller classification, 47 patients developed either type I (n = 2), II (n = 8), III (n = 11) or IV (n = 26) esophagitis after surgery.
  • Newly developed BE was observed in nine patients (13.5 %) after subtotal esophagectomy (median time to diagnosis: 489 days, range 43 - 1172).
  • CONCLUSIONS: Newly developed BE after subtotal esophagectomy may provide further insights into the early mucosal events that lead to intestinal metaplasia and into the roles of gastroesophageal and duodenoesophageal reflux in the pathogenesis of BE.
  • [MeSH-minor] Adenocarcinoma / surgery. Aged. Carcinoma, Squamous Cell / drug therapy. Carcinoma, Squamous Cell / surgery. Chemotherapy, Adjuvant. Esophageal Neoplasms / drug therapy. Esophageal Neoplasms / surgery. Female. Humans. Male. Middle Aged. Proton Pump Inhibitors. Retrospective Studies

  • Genetic Alliance. consumer health - Barrett's Esophagus.
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  • (PMID = 14551864.001).
  • [ISSN] 0013-726X
  • [Journal-full-title] Endoscopy
  • [ISO-abbreviation] Endoscopy
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / Proton Pump Inhibitors
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4. Gadour M, Ayoola EA: Barrett's esophagus: a review. Trop Gastroenterol; 2002 Oct-Dec;23(4):157-61
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Barrett's esophagus [BE] is usually an acquired, condition in which specialized metaplastic intestinal columnar epithelium with goblet cells replaces the normal stratified squamous epithelium anywhere in the esophagus.
  • The importance of BE comes from its potential risk of progression to adenocarcinoma.
  • The development of dysplastic changes in Barrett's metaplasia increases this risk markedly.
  • However, the true incidence and the likelihood of adenocarcinoma developing in such individuals with dysplasia over a lifetime are not well defined.
  • Histopathology of esophageal biopsy is mandatory for the diagnosis, because clinical, radiological and endoscopic findings in such cases can only suspect the disease.
  • The exact prevalence of BE in the general population is difficult to estimate, however worldwide distribution of the disease vary considerably.
  • To date, the optimal form of treatment whether by drugs and/or endoscopic ablation or by surgery remains undetermined.
  • [MeSH-minor] Adenocarcinoma / etiology. Deglutition Disorders / etiology. Endoscopy, Gastrointestinal. Esophageal Neoplasms / etiology. Humans. Population Surveillance. Prevalence

  • Genetic Alliance. consumer health - Barrett's Esophagus.
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  • (PMID = 12833699.001).
  • [ISSN] 0250-636X
  • [Journal-full-title] Tropical gastroenterology : official journal of the Digestive Diseases Foundation
  • [ISO-abbreviation] Trop Gastroenterol
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] India
  • [Number-of-references] 59
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