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1
adenocarcinoma of cardia of stomach 2005:2010[pubdate] *count=100
355 results
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adenocarcinoma of cardia of stomach
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Items 1 to 100 of about 355
1.
Haas M, Dimmler A, Hohenberger W, Grabenbauer GG, Niedobitek G, Distel LV:
Stromal regulatory T-cells are associated with a favourable prognosis in gastric cancer of the cardia.
BMC Gastroenterol
; 2009;9:65
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[Title]
Stromal regulatory T-cells are associated with a favourable prognosis in
gastric
cancer of the
cardia
.
We assessed the prognostic significance of tumour infiltrating lymphocytes (TIL) in intestinal-type
gastric
cardiac cancer.
CONCLUSION: Our results suggest that inflammatory processes within the tumour stroma of
gastric
intestinal-type adenocarcinomas located at
the gastric
cardia
may affect outcome in two ways.
Thus, inhibition of Treg may not be a feasible treatment option in
gastric
adenocarcinoma
.
[MeSH-major]
Adenocarcinoma
/ diagnosis.
Adenocarcinoma
/ pathology.
Cardia
/ pathology.
Stomach
Neoplasms / diagnosis.
Stomach
Neoplasms / pathology. Stromal Cells / pathology. T-Lymphocytes, Regulatory / pathology
MedlinePlus Health Information.
consumer health - Stomach Cancer
.
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[Cites]
Surg Oncol Clin N Am. 2002 Apr;11(2):235-56
[
12424848.001
]
[Cites]
APMIS. 2008 Jul-Aug;116(7-8):685-94
[
18834412.001
]
[Cites]
Anticancer Res. 2003 Sep-Oct;23(5A):4079-83
[
14666722.001
]
[Cites]
BMJ. 2004 May 1;328(7447):1073
[
15117797.001
]
[Cites]
Nat Med. 2004 Sep;10(9):942-9
[
15322536.001
]
[Cites]
Nature. 2004 Sep 23;431(7007):405-6
[
15385993.001
]
[Cites]
Cancer Detect Prev. 1995;19(2):156-64
[
7750103.001
]
[Cites]
J Surg Res. 2005 Mar;124(1):151-7
[
15734494.001
]
[Cites]
J Clin Oncol. 2009 Jan 10;27(2):186-92
[
19064967.001
]
[Cites]
Nat Rev Immunol. 2009 Feb;9(2):83-9
[
19114986.001
]
[Cites]
Annu Rev Immunol. 2000;18:423-49
[
10837065.001
]
[Cites]
Cancer Res. 2005 May 15;65(10):3998-4004
[
15899788.001
]
[Cites]
Nature. 2005 Jun 9;435(7043):752-3
[
15944689.001
]
[Cites]
Proc Natl Acad Sci U S A. 2005 Dec 20;102(51):18538-43
[
16344461.001
]
[Cites]
Cancer Immunol Immunother. 2006 Sep;55(9):1064-71
[
16328385.001
]
[Cites]
Clin Cancer Res. 2006 Jun 1;12(11 Pt 1):3355-60
[
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]
[Cites]
Best Pract Res Clin Gastroenterol. 2006;20(4):687-96
[
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]
[Cites]
Clin Immunol. 2006 Dec;121(3):358-68
[
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]
[Cites]
J Clin Invest. 2007 Jan;117(1):60-9
[
17200707.001
]
[Cites]
Aliment Pharmacol Ther. 2007 Feb 15;25(4):447-53
[
17270000.001
]
[Cites]
Jpn J Clin Oncol. 2007 May;37(5):365-9
[
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]
[Cites]
Am J Gastroenterol. 2007 Aug;102(8):1596-602
[
17459024.001
]
[Cites]
Curr Opin Investig Drugs. 2007 Dec;8(12):1002-8
[
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]
[Cites]
Br J Cancer. 2008 Jan 15;98(1):148-53
[
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]
[Cites]
Haematologica. 2008 Feb;93(2):193-200
[
18223287.001
]
[Cites]
Cell Immunol. 2007 Nov-Dec;250(1-2):1-13
[
18313653.001
]
[Cites]
Integr Cancer Ther. 2008 Jun;7(2):90-5
[
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]
[Cites]
EMBO J. 2008 Jun 18;27(12):1671-81
[
18511911.001
]
[Cites]
N Engl J Med. 2003 Jan 16;348(3):203-13
[
12529460.001
]
(PMID = 19732435.001).
[ISSN]
1471-230X
[Journal-full-title]
BMC gastroenterology
[ISO-abbreviation]
BMC Gastroenterol
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / FOXP3 protein, human; 0 / Forkhead Transcription Factors
[Other-IDs]
NLM/ PMC2749861
2.
Wang GQ, Jiao GG, Song JX, Fang WH, Lü N, Lin DM, Xie YQ, Zhang JH, Wei WQ:
[Diagnosis and long-term results of surgical resection of early cardiac adenocarcinoma].
Zhonghua Wai Ke Za Zhi
; 2008 Jul 15;46(14):1045-7
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[Title]
[Diagnosis and long-term results of surgical resection of early cardiac
adenocarcinoma
].
OBJECTIVE: To summarize therapeutic experience and the long-term results of early cardiac
adenocarcinoma
with surgical resection.
METHODS: Ninety cases were diagnosed with early cardiac
adenocarcinoma
during endoscopic screening in high incidence rate area of esophageal cancer from 1972 to 1997.
Cardiectomy included partial
stomach
and esophagus was performed through left thoracotomy in all patients.
[MeSH-major]
Adenocarcinoma
/ surgery.
Cardia
.
Stomach
Neoplasms / surgery
MedlinePlus Health Information.
consumer health - Stomach Cancer
.
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(PMID = 19094526.001).
[ISSN]
0529-5815
[Journal-full-title]
Zhonghua wai ke za zhi [Chinese journal of surgery]
[ISO-abbreviation]
Zhonghua Wai Ke Za Zhi
[Language]
chi
[Publication-type]
English Abstract; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
China
3.
Jakszyn P, Bingham S, Pera G, Agudo A, Luben R, Welch A, Boeing H, Del Giudice G, Palli D, Saieva C, Krogh V, Sacerdote C, Tumino R, Panico S, Berglund G, Simán H, Hallmans G, Sanchez MJ, Larrañaga N, Barricarte A, Chirlaque MD, Quirós JR, Key TJ, Allen N, Lund E, Carneiro F, Linseisen J, Nagel G, Overvad K, Tjonneland A, Olsen A, Bueno-de-Mesquita HB, Ocké MO, Peeters PH, Numans ME, Clavel-Chapelon F, Trichopoulou A, Fenger C, Stenling R, Ferrari P, Jenab M, Norat T, Riboli E, Gonzalez CA:
Endogenous versus exogenous exposure to N-nitroso compounds and gastric cancer risk in the European Prospective Investigation into Cancer and Nutrition (EPIC-EURGAST) study.
Carcinogenesis
; 2006 Jul;27(7):1497-501
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[Title]
Endogenous versus exogenous exposure to N-nitroso compounds and
gastric
cancer risk in the European Prospective Investigation into Cancer and Nutrition (EPIC-EURGAST) study.
The risk of
gastric
cancer (GC) associated with dietary intake of nitrosodimethylamine (NDMA) and endogenous formation of nitroso compounds (NOCs) was investigated in the European Prospective Investigation into Cancer and Nutrition (EPIC).
ENOC was significantly associated with non-
cardia
cancer risk (HR, 1.42; 95% CI, 1.14-1.78 for an increase of 40 microg/day) but not with
cardia
cancer (HR, 0.96; 95% CI, 0.69-1.33).
ENOC formation may account for our previously reported association between red and processed meat consumption and
gastric
cancer risk.
[MeSH-major]
Adenocarcinoma
/ epidemiology. Diet. Nitrosamines / metabolism. Nitrosamines / pharmacology.
Stomach
Neoplasms / epidemiology
MedlinePlus Health Information.
consumer health - Stomach Cancer
.
COS Scholar Universe.
author profiles
.
Hazardous Substances Data Bank.
Sodium ascorbate
.
Hazardous Substances Data Bank.
L-Ascorbic Acid
.
Hazardous Substances Data Bank.
N-NITROSODIMETHYLAMINE
.
Hazardous Substances Data Bank.
IRON, ELEMENTAL
.
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(PMID = 16571648.001).
[ISSN]
0143-3334
[Journal-full-title]
Carcinogenesis
[ISO-abbreviation]
Carcinogenesis
[Language]
eng
[Grant]
United Kingdom / Wellcome Trust / /
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / Nitrosamines; E1UOL152H7 / Iron; M43H21IO8R / Dimethylnitrosamine; PQ6CK8PD0R / Ascorbic Acid
Advertisement
4.
Lu CC, De-Chuan C, Lee HS, Chu HC:
Pure hepatoid adenocarcinoma of the stomach with spleen and lymph-node metastases.
Am J Surg
; 2010 Apr;199(4):e42-4
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[Title]
Pure hepatoid
adenocarcinoma of
the
stomach
with spleen and lymph-node metastases.
The authors report a rare case of hepatoid
adenocarcinoma of
the
stomach
, presenting initially with spleen and lymph node metastases.
[MeSH-major]
Adenocarcinoma
/ diagnosis.
Cardia
. Liver Neoplasms / diagnosis. Lymph Nodes / pathology. Splenic Neoplasms / diagnosis.
Stomach
Neoplasms / diagnosis
MedlinePlus Health Information.
consumer health - Liver Cancer
.
MedlinePlus Health Information.
consumer health - Stomach Cancer
.
Hazardous Substances Data Bank.
DOXORUBICIN
.
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[Copyright]
Copyright 2010 Elsevier Inc. All rights reserved.
(PMID = 20359564.001).
[ISSN]
1879-1883
[Journal-full-title]
American journal of surgery
[ISO-abbreviation]
Am. J. Surg.
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Biomarkers, Tumor; 0 / alpha-Fetoproteins; 8001-40-9 / Iodized Oil; 80168379AG / Doxorubicin
5.
Kofoed SC, Brandt B, Brenø J, Bardram L, Gustafsen J, Holm J, Jendresen M, Svendsen LB:
[Long-term survival after curative resection for oesophageal and cardia cancer].
Ugeskr Laeger
; 2010 May 24;172(21):1597-602
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[Title]
[Long-term survival after curative resection for oesophageal and
cardia
cancer].
Adenocarcinoma
was found in 93% of the patients and squamous cell carcinoma in 7%.
Cardia
resection was performed in 78%, while 22% underwent gastrectomy.
[MeSH-major]
Adenocarcinoma
/ mortality. Carcinoma, Squamous Cell / mortality.
Cardia
. Esophageal Neoplasms / mortality.
Stomach
Neoplasms / mortality
MedlinePlus Health Information.
consumer health - Esophageal Cancer
.
MedlinePlus Health Information.
consumer health - Stomach Cancer
.
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(PMID = 20525472.001).
[ISSN]
1603-6824
[Journal-full-title]
Ugeskrift for laeger
[ISO-abbreviation]
Ugeskr. Laeg.
[Language]
dan
[Publication-type]
English Abstract; Journal Article
[Publication-country]
Denmark
6.
Moenig SP, Luebke T, Baldus SE, Schroeder W, Bollschweiler E, Schneider PM, Hoelscher AH:
Feasibility of sentinel node concept in gastric carcinoma: clinicopathological analysis of gastric cancer with solitary lymph node metastases.
Anticancer Res
; 2005 Mar-Apr;25(2B):1349-52
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[Title]
Feasibility of sentinel node concept in
gastric
carcinoma: clinicopathological analysis of
gastric
cancer with solitary lymph node metastases.
BACKGROUND: The feasibility and diagnostic reliability of sentinel lymph node biopsy of
gastric
carcinoma are still unclear and controversial.
PATIENTS AND METHODS: To assess the applicability of the sentinel node concept to
gastric
carcinoma, we retrospectively analyzed the location of metastatic lymph nodes in patients with only one or two lymph node metastases.
RESULTS: A total of 135 patients, who underwent gastrectomy with D2 lymphadenectomy for primary
gastric
adenocarcinoma
between 1997 and 2001, were enrolled in this study.
Skip metastases were only seen in one patient with
cardia
carcinoma and lymph node involvement of compartment II (left
gastric
artery).
CONCLUSION: In patients with
gastric
carcinoma, especially in early stage carcinoma, the phenomenon of skip metastasis is infrequent.
Therefore, the sentinel node concept may be feasible in
gastric
cancer.
[MeSH-major]
Sentinel Lymph Node Biopsy.
Stomach
Neoplasms / diagnosis
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(PMID = 15865090.001).
[ISSN]
0250-7005
[Journal-full-title]
Anticancer research
[ISO-abbreviation]
Anticancer Res.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Greece
7.
Gao SG, Wang LD, Fan ZM, Li JL, He X, Guo RF, Xie DL, He XW, Gao SS, Guo HQ, Wang JK, Feng XS, Ma BG:
Histochemical studies on intestinal metaplasia adjacent to gastric cardia adenocarcinoma in subjects at high-incidence area in Henan, north China.
World J Gastroenterol
; 2005 Aug 14;11(30):4634-7
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[Title]
Histochemical studies on intestinal metaplasia adjacent to
gastric
cardia adenocarcinoma
in subjects at high-incidence area in Henan, north China.
AIM: To characterize the histochemical type and pattern of intestinal metaplasia (IM) adjacent to
gastric
cardia adenocarcinoma
(GCA) and distal
gastric
cancer (GC) in Linzhou, Henan Province, China.
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(PMID = 16094701.001).
[ISSN]
1007-9327
[Journal-full-title]
World journal of gastroenterology
[ISO-abbreviation]
World J. Gastroenterol.
[Language]
ENG
[Grant]
United States / NCI NIH HHS / CA / R01 CA065871; United States / NCI NIH HHS / CA / CA65871
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, P.H.S.
[Publication-country]
United States
[Other-IDs]
NLM/ PMC4615402
8.
Chandanos E, Lindblad M, Jia C, Rubio CA, Ye W, Lagergren J:
Tamoxifen exposure and risk of oesophageal and gastric adenocarcinoma: a population-based cohort study of breast cancer patients in Sweden.
Br J Cancer
; 2006 Jul 3;95(1):118-22
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[Title]
Tamoxifen exposure and risk of oesophageal and
gastric
adenocarcinoma
: a population-based cohort study of breast cancer patients in Sweden.
Standardised incidence ratios (SIRs) of oesophageal and
gastric
cancer represented relative risks.
Among 138 885 cohort members contributing with 1 075 724 person-years of follow-up, we found a nonsignificantly increased risk of oesophageal
adenocarcinoma
during the potential tamoxifen exposure period (SIR 1.60, 95% confidence interval (CI) 0.83-3.08), but the risk estimates decreased with increasing latency interval.
No increased risk
of cardia adenocarcinoma
was identified in either period.
The risk of non-
cardia
gastric
adenocarcinoma
was increased in the potential tamoxifen period (SIR 1.27, 1.03-1.57), and almost doubled (SIR 1.86, 95% CI 1.10-3.14) in the period of longest latency (10-14 years).
We concluded that there might be a link between tamoxifen and risk of non-
cardia
gastric
adenocarcinoma
.
[MeSH-major]
Adenocarcinoma
/ chemically induced. Breast Neoplasms / drug therapy. Carcinoma, Squamous Cell / chemically induced. Esophageal Neoplasms / chemically induced. Lung Neoplasms / chemically induced.
Stomach
Neoplasms / chemically induced. Tamoxifen / adverse effects
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[Cites]
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Cancer Causes Control. 2000 Oct;11(9):869-74
[
11075877.001
]
(PMID = 16755290.001).
[ISSN]
0007-0920
[Journal-full-title]
British journal of cancer
[ISO-abbreviation]
Br. J. Cancer
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
094ZI81Y45 / Tamoxifen
[Other-IDs]
NLM/ PMC2360495
9.
Kamangar F, Qiao YL, Schiller JT, Dawsey SM, Fears T, Sun XD, Abnet CC, Zhao P, Taylor PR, Mark SD:
Human papillomavirus serology and the risk of esophageal and gastric cancers: results from a cohort in a high-risk region in China.
Int J Cancer
; 2006 Aug 1;119(3):579-84
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[Title]
Human papillomavirus serology and the risk of esophageal and
gastric
cancers: results from a cohort in a high-risk region in China.
Each year, esophageal and
gastric
cancers cause more than 900,000 deaths worldwide.
We conducted a large prospective study to examine the association between serum antibodies to HPV 16, HPV 18 and HPV 73 and subsequent development of esophageal squamous cell carcinoma (ESCC),
gastric
cardia adenocarcinoma
(GCA), and
gastric
noncardia
adenocarcinoma
(GNCA) in a high-risk population for these cancers in Linxian, China.
The results of this study do not support a major role for HPV 16, HPV 18 and HPV 73 in the etiology of esophageal and
gastric
cancers in Linxian, China.
[MeSH-major]
Antibodies, Viral / blood. Esophageal Neoplasms / etiology. Papillomavirus Infections / complications.
Stomach
Neoplasms / etiology
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[Copyright]
Copyright (c) 2006 Wiley-Liss, Inc.
(PMID = 16496409.001).
[ISSN]
0020-7136
[Journal-full-title]
International journal of cancer
[ISO-abbreviation]
Int. J. Cancer
[Language]
eng
[Grant]
United States / Intramural NIH HHS / /
[Publication-type]
Journal Article; Research Support, N.I.H., Intramural
[Publication-country]
United States
[Chemical-registry-number]
0 / Antibodies, Viral
10.
Jenab M, Riboli E, Ferrari P, Friesen M, Sabate J, Norat T, Slimani N, Tjønneland A, Olsen A, Overvad K, Boutron-Ruault MC, Clavel-Chapelon F, Boeing H, Schulz M, Linseisen J, Nagel G, Trichopoulou A, Naska A, Oikonomou E, Berrino F, Panico S, Palli D, Sacerdote C, Tumino R, Peeters PH, Numans ME, Bueno-de-Mesquita HB, Büchner FL, Lund E, Pera G, Chirlaque MD, Sánchez MJ, Arriola L, Barricarte A, Quirós JR, Johansson I, Johansson A, Berglund G, Bingham S, Khaw KT, Allen N, Key T, Carneiro F, Save V, Del Giudice G, Plebani M, Kaaks R, Gonzalez CA:
Plasma and dietary carotenoid, retinol and tocopherol levels and the risk of gastric adenocarcinomas in the European prospective investigation into cancer and nutrition.
Br J Cancer
; 2006 Aug 07;95(3):406-15
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[Title]
Plasma and dietary carotenoid, retinol and tocopherol levels and the risk of
gastric
adenocarcinomas in the European prospective investigation into cancer and nutrition.
Despite declining incidence rates,
gastric
cancer (GC) is a major cause of death worldwide.
The objective of this study was to determine the association of plasma levels of seven common carotenoids, their total plasma concentration, retinol and alpha- and gamma-tocopherol, with the risk of
gastric
adenocarcinoma
in a case-control study nested within the European Prospective Investigation into Cancer and Nutrition (EPIC), a large cohort involving 10 countries.
A secondary objective was to determine the association of total sum of carotenoids, retinol and alpha-tocopherol on GCs by anatomical subsite (
cardia
/noncardia) and histological subtype (diffuse/intestinal).
[MeSH-major]
Adenocarcinoma
/ epidemiology. Carotenoids / administration & dosage. Diet.
Stomach
Neoplasms / epidemiology. Tocopherols / administration & dosage. Vitamin A / administration & dosage
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[ISSN]
0007-0920
[Journal-full-title]
British journal of cancer
[ISO-abbreviation]
Br. J. Cancer
[Language]
eng
[Grant]
United Kingdom / Medical Research Council / / G0401527; United Kingdom / Wellcome Trust / /
[Publication-type]
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
11103-57-4 / Vitamin A; 1406-66-2 / Tocopherols; 36-88-4 / Carotenoids
[Other-IDs]
NLM/ PMC2360629
11.
Lamb PJ, Griffin SM, Burt AD, Lloyd J, Karat D, Hayes N:
Sentinel node biopsy to evaluate the metastatic dissemination of oesophageal adenocarcinoma.
Br J Surg
; 2005 Jan;92(1):60-7
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[Title]
Sentinel node biopsy to evaluate the metastatic dissemination of oesophageal
adenocarcinoma
.
BACKGROUND: The aim of this study was to determine the feasibility and accuracy of sentinel lymph node (SLN) biopsy for oesophageal
adenocarcinoma
.
METHODS: Fifty-seven patients with
adenocarcinoma of
the lower oesophagus (n = 40) or
gastric
cardia
(n = 17) underwent endoscopic peritumoral injection of (99m)Tc-radiolabelled nanocolloid before en bloc resection with extended lymphadenectomy.
Lower oesophageal tumours had a greater proportion of SLNs (P = 0.018), radioactive uptake (P < 0.001) and malignant nodes (P = 0.004) in the mediastinum than
gastric
cardia
tumours.
CONCLUSION: The sentinel node concept is applicable to oesophageal
adenocarcinoma
and could be used to tailor the extent of lymphadenectomy.
There is a close relationship between patterns of radioactive uptake and lymphatic tumour dissemination, which differ for lower oesophageal and
gastric
cardia
tumours.
[MeSH-major]
Adenocarcinoma
/ secondary. Esophageal Neoplasms / pathology. Sentinel Lymph Node Biopsy / methods.
Stomach
Neoplasms / pathology
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(PMID = 15584066.001).
[ISSN]
0007-1323
[Journal-full-title]
The British journal of surgery
[ISO-abbreviation]
Br J Surg
[Language]
eng
[Publication-type]
Evaluation Studies; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
12.
Hampel H, Abraham NS, El-Serag HB:
Meta-analysis: obesity and the risk for gastroesophageal reflux disease and its complications.
Ann Intern Med
; 2005 Aug 2;143(3):199-211
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BACKGROUND: The association of body mass index and gastroesophageal reflux disease (GERD), including its complications (esophagitis, Barrett esophagus, and esophageal
adenocarcinoma
), is unclear.
PURPOSE: To conduct a systematic review and meta-analysis to estimate the magnitude and determinants of an association between obesity and GERD symptoms, erosive esophagitis, Barrett esophagus, and
adenocarcinoma of
the esophagus and of
the gastric
cardia
.
Six of 7 studies found significant associations of BMI with erosive esophagitis, 6 of 7 found significant associations with esophageal
adenocarcinoma
, and 4 of 6 found significant associations with
gastric
cardia adenocarcinoma
.
Similarly, the pooled adjusted odds ratios for esophageal
adenocarcinoma
for BMI of 25 kg/m2 to 30 kg/m2 and BMI greater than 30 kg/m2 were 1.52 (CI, 1.147 to 2.009) and 2.78 (CI, 1.850 to 4.164), respectively.
CONCLUSION: Obesity is associated with a statistically significant increase in the risk for GERD symptoms, erosive esophagitis, and esophageal
adenocarcinoma
.
[MeSH-minor]
Adenocarcinoma
/ etiology. Barrett Esophagus / etiology. Body Mass Index.
Cardia
. Esophageal Neoplasms / etiology. Esophagitis / etiology. Humans. Odds Ratio. Risk Factors
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(PMID = 16061918.001).
[ISSN]
1539-3704
[Journal-full-title]
Annals of internal medicine
[ISO-abbreviation]
Ann. Intern. Med.
[Language]
eng
[Publication-type]
Journal Article; Meta-Analysis; Research Support, U.S. Gov't, Non-P.H.S.
[Publication-country]
United States
13.
Chung JW, Lee GH, Choi KS, Kim DH, Jung KW, Song HJ, Choi KD, Jung HY, Kim JH, Yook JH, Kim BS, Jang SJ:
Unchanging trend of esophagogastric junction adenocarcinoma in Korea: experience at a single institution based on Siewert's classification.
Dis Esophagus
; 2009;22(8):676-81
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[Title]
Unchanging trend of esophagogastric junction
adenocarcinoma
in Korea: experience at a single institution based on Siewert's classification.
The incidence
of adenocarcinoma
of the esophagogastric junction (AEG) has been increasing in Western countries.
We retrospectively reviewed the medical records of 16 811 patients diagnosed with esophageal squamous cell carcinoma (ESC, n= 1450) or
gastric
noncardiac
adenocarcinoma
(GNCA, n= 14 751) between 1992 and 2006.
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(PMID = 19222529.001).
[ISSN]
1442-2050
[Journal-full-title]
Diseases of the esophagus : official journal of the International Society for Diseases of the Esophagus
[ISO-abbreviation]
Dis. Esophagus
[Language]
ENG
[Publication-type]
Journal Article
[Publication-country]
United States
14.
Heinrich H, Bauerfeind P:
Endoscopic mucosal resection for staging and therapy of adenocarcinoma of the esophagus, gastric cardia, and upper gastric third.
Recent Results Cancer Res
; 2010;182:85-91
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The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
[Title]
Endoscopic mucosal resection for staging and therapy
of adenocarcinoma
of the esophagus,
gastric
cardia
, and upper
gastric
third.
Endoscopic submucosal dissection (ESD) is a useful therapeutic option for HGD or early cancer in the squamous epithelium of the esophagus or in the
stomach
when en bloc resection is needed in large lesions.
[MeSH-major]
Adenocarcinoma
/ pathology.
Cardia
. Esophageal Neoplasms / pathology.
Stomach
Neoplasms / pathology
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(PMID = 20676873.001).
[ISSN]
0080-0015
[Journal-full-title]
Recent results in cancer research. Fortschritte der Krebsforschung. Progrès dans les recherches sur le cancer
[ISO-abbreviation]
Recent Results Cancer Res.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
Germany
15.
Man XH, Xu Y, Wang ZN, Lü Z, Xu MD, Jiang L, Luo Y, Xu HM, Zhang X:
[Loss of heterozygosity at chromosome 8p21-p23 in adenocarcinoma of gastric cardia].
Yi Chuan
; 2006 Jun;28(6):641-5
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[Title]
[Loss of heterozygosity at chromosome 8p21-p23 in
adenocarcinoma of
gastric
cardia
].
To investigate the involvement of the gene(s) in the carcinogenesis
of adenocarcinoma
of
gastric
cardia
, loss of heterozygosity (LOH) for microsatellite markers at chromosome 8p21-p23 was examined.
Our data indicated that the tumor suppressor gene at chromosome 8p22 might play an important role in the development
of adenocarcinoma
of
gastric
cardia
.
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(PMID = 16818423.001).
[ISSN]
0253-9772
[Journal-full-title]
Yi chuan = Hereditas
[ISO-abbreviation]
Yi Chuan
[Language]
CHI
[Publication-type]
English Abstract; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
China
[Chemical-registry-number]
0 / DNA, Neoplasm
16.
Zhang XH, Wang QZ:
[Understanding and controversy of the gastroesophageal junction adenocarcinoma].
Zhonghua Zhong Liu Za Zhi
; 2008 Dec;30(12):947-9
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[Title]
[Understanding and controversy of the gastroesophageal junction
adenocarcinoma
].
[MeSH-major]
Adenocarcinoma
.
Cardia
. Esophageal Neoplasms. Esophagogastric Junction.
Stomach
Neoplasms
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(PMID = 19174001.001).
[ISSN]
0253-3766
[Journal-full-title]
Zhonghua zhong liu za zhi [Chinese journal of oncology]
[ISO-abbreviation]
Zhonghua Zhong Liu Za Zhi
[Language]
chi
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
China
[Number-of-references]
23
17.
von Rahden BH, Stein HJ, Feith M, Becker K, Siewert JR:
Lymphatic vessel invasion as a prognostic factor in patients with primary resected adenocarcinomas of the esophagogastric junction.
J Clin Oncol
; 2005 Feb 1;23(4):874-9
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PATIENTS AND METHODS: We prospectively evaluated 459 patients undergoing primary surgical resection for tumors of the esophagogastric junction at our institution between 1992 and 2000 (180 adenocarcinomas of the distal esophagus, AEG I; 140 carcinomas of the
cardia
, AEG II; and 139 subcardial
gastric
cancers, AEG III).
[MeSH-major]
Adenocarcinoma
/ pathology. Esophageal Neoplasms / pathology. Esophagogastric Junction. Lymphatic Vessels / pathology.
Stomach
Neoplasms / pathology
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(PMID = 15681533.001).
[ISSN]
0732-183X
[Journal-full-title]
Journal of clinical oncology : official journal of the American Society of Clinical Oncology
[ISO-abbreviation]
J. Clin. Oncol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
18.
Guo W, Dong Z, Guo Y, Kuang G, Yang Z, Chen Z:
Detection of promoter hypermethylation of the CpG island of E-cadherin in gastric cardiac adenocarcinoma.
Eur J Med Res
; 2009 Sep 28;14(10):453-8
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[Title]
Detection of promoter hypermethylation of the CpG island of E-cadherin in
gastric
cardiac
adenocarcinoma
.
The aim of this study was to investigate the promoter methylation and expression of E-cadherin gene in
gastric
cardiac
adenocarcinoma
(GCA).
CONCLUSIONS: High methylation status of the 5' CpG island of E-cadherin gene may be one of the mechanisms in the development of
gastric
cardiac
adenocarcinoma
.
[MeSH-major]
Adenocarcinoma
/ genetics. Cadherins / genetics.
Cardia
. CpG Islands. DNA Methylation. Promoter Regions, Genetic.
Stomach
Neoplasms / genetics
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[Cites]
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[ISSN]
0949-2321
[Journal-full-title]
European journal of medical research
[ISO-abbreviation]
Eur. J. Med. Res.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Germany
[Chemical-registry-number]
0 / Cadherins; 0 / RNA, Messenger
[Other-IDs]
NLM/ PMC3352230
19.
McColl KE:
Cancer of the gastric cardia.
Best Pract Res Clin Gastroenterol
; 2006;20(4):687-96
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[Title]
Cancer of
the gastric
cardia
.
Current evidence indicates that
cardia
cancers are of at least two distinct and disparate aetiologies.
One type resembles cancer of the more distal
stomach
(Type A), being a consequence of atrophic gastritis due to Helicobacter pylori infection or more rarely autoimmune atrophic gastritis.
Another type (Type B) resembles oesophageal
adenocarcinoma
and is likely to be a consequence of short-segment gastro-oesophageal reflux disease.
The two cancers are themselves indistinguishable but examination of
the gastric
phenotype indicates the aetiology: Type A occurring in patients with evidence of atrophic gastritis whereas Type B occurs in subjects with healthy acid secreting stomachs.
In subjects with healthy acid secreting stomachs the
cardia
has a specific luminal chemistry remaining highly acidic and unbuffered following a meal and having very active nitrosative chemistry due to the acidification of nitrite in saliva.
[MeSH-major]
Adenocarcinoma
/ pathology.
Cardia
/ pathology.
Stomach
Neoplasms / pathology
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(PMID = 16997153.001).
[ISSN]
1521-6918
[Journal-full-title]
Best practice & research. Clinical gastroenterology
[ISO-abbreviation]
Best Pract Res Clin Gastroenterol
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
England
[Number-of-references]
54
20.
Abnet CC, Freedman ND, Kamangar F, Leitzmann MF, Hollenbeck AR, Schatzkin A:
Non-steroidal anti-inflammatory drugs and risk of gastric and oesophageal adenocarcinomas: results from a cohort study and a meta-analysis.
Br J Cancer
; 2009 Feb 10;100(3):551-7
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[Title]
Non-steroidal anti-inflammatory drugs and risk of
gastric
and oesophageal adenocarcinomas: results from a cohort study and a meta-analysis.
Use of aspirin or other non-steroidal anti-inflammatory drugs (NSAIDs) may reduce the risk of
gastric
or oesophageal adenocarcinomas.
We examined the association between self-reported use of aspirin or non-aspirin NSAIDs in the earlier 12 months and
gastric
non-
cardia
(N=182),
gastric
cardia
(N=178), and oesophageal adenocarcinomas (N=228) in a prospective cohort (N=311 115) followed for 7 years.
Use of any aspirin (HR, 95% CI: 0.64, 0.47-0.86) or other NSAIDs (0.68, 0.51-0.92) was associated with a significantly lower risk of
gastric
non-
cardia adenocarcinoma
.
Neither aspirin (0.86, 0.61-1.20) nor other NSAIDs (0.91, 0.67-1.22) had a significant association with
gastric
cardia
cancer.
We found no significant association between using aspirin (1.00, 0.73-1.37) or other NSAIDs (0.90, 69-1.17) and oesophageal
adenocarcinoma
.
We also performed a meta-analysis of the association between the use of NSAIDs and risk of
gastric
and oesophageal
adenocarcinoma
.
In this analysis, aspirin use was inversely associated with both
gastric
and oesophageal adenocarcinomas, with summary odds ratios (95% CI) for non-
cardia
,
cardia
, and oesophageal adenocarcinomas of 0.64 (0.52-0.80), 0.82 (0.65-1.04), and 0.64 (0.52-0.79), respectively.
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[ISSN]
1532-1827
[Journal-full-title]
British journal of cancer
[ISO-abbreviation]
Br. J. Cancer
[Language]
ENG
[Grant]
United States / Intramural NIH HHS / /
[Publication-type]
Journal Article; Meta-Analysis; Research Support, N.I.H., Intramural; Research Support, U.S. Gov't, Non-P.H.S.
[Publication-country]
England
[Chemical-registry-number]
0 / Anti-Inflammatory Agents, Non-Steroidal
[Other-IDs]
NLM/ PMC2658549
21.
Bastos J, Lunet N, Peleteiro B, Lopes C, Barros H:
Dietary patterns and gastric cancer in a Portuguese urban population.
Int J Cancer
; 2010 Jul 15;127(2):433-41
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[Title]
Dietary patterns and
gastric
cancer in a Portuguese urban population.
We aimed to quantify the association between dietary patterns and
gastric
cancer, by location and histological type, according to Helicobacter pylori infection status.
We analyzed 591 incident cases of
gastric
adenocarcinoma
and 1,463 community controls.
Compared to pattern I, the risk of
gastric
cancer was higher for pattern II (OR = 1.68, 95% CI: 1.31-2.14) but not for pattern III (OR = 0.80, 95% CI: 0.57-1.14), with no effect modification by H. pylori infection.
The association was similar for
cardia
and non-
cardia
gastric
cancer, but for tumors of the diffuse Laurén histological type, the association was weaker for pattern II vs. I (OR = 1.32, 95% CI: 0.83-2.08) and a protective effect was observed for pattern III vs. I (OR = 0.43, 95% CI: 0.22-0.87).
[MeSH-major]
Adenocarcinoma
/ epidemiology. Diet.
Stomach
Neoplasms / epidemiology. Urban Population / statistics & numerical data
[MeSH-minor]
Adult. Aged. Aged, 80 and over.
Cardia
. Case-Control Studies. Female. Helicobacter Infections / complications. Helicobacter Infections / microbiology. Helicobacter pylori / isolation & purification. Humans. Male. Middle Aged. Nutrition Surveys. Portugal / epidemiology. Prognosis. Surveys and Questionnaires. Young Adult
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(PMID = 19876925.001).
[ISSN]
1097-0215
[Journal-full-title]
International journal of cancer
[ISO-abbreviation]
Int. J. Cancer
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Multicenter Study; Research Support, Non-U.S. Gov't
[Publication-country]
United States
22.
Cordin J, Lehmann K, Schneider PM:
Clinical staging of adenocarcinoma of the esophagogastric junction.
Recent Results Cancer Res
; 2010;182:73-83
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[Title]
Clinical staging
of adenocarcinoma
of the esophagogastric junction.
Upper endoscopy establishes the tumor diagnosis by multiple biopsies and defines the tumor type (Siewert I-III), based on tumor localization in relation to the endoscopic
cardia
.
[MeSH-major]
Adenocarcinoma
/ pathology. Esophageal Neoplasms / pathology. Esophagogastric Junction.
Stomach
Neoplasms / pathology
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(PMID = 20676872.001).
[ISSN]
0080-0015
[Journal-full-title]
Recent results in cancer research. Fortschritte der Krebsforschung. Progrès dans les recherches sur le cancer
[ISO-abbreviation]
Recent Results Cancer Res.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
Germany
23.
Figueroa JD, Terry MB, Gammon MD, Vaughan TL, Risch HA, Zhang FF, Kleiner DE, Bennett WP, Howe CL, Dubrow R, Mayne ST, Fraumeni JF Jr, Chow WH:
Cigarette smoking, body mass index, gastro-esophageal reflux disease, and non-steroidal anti-inflammatory drug use and risk of subtypes of esophageal and gastric cancers by P53 overexpression.
Cancer Causes Control
; 2009 Apr;20(3):361-8
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[Title]
Cigarette smoking, body mass index, gastro-esophageal reflux disease, and non-steroidal anti-inflammatory drug use and risk of subtypes of esophageal and
gastric
cancers by P53 overexpression.
A number of risk factors for esophageal and
gastric
cancers have emerged, yet little is known whether risk factors map to molecular tumor markers such as overexpression of the tumor suppressor TP53.
Using a US multicenter, population-based case-control study (170 cases of esophageal adenocarcinomas, 147
gastric
cardia
adenocarcinomas, 220 non-
cardia
gastric
adenocarcinomas, and 112 esophageal squamous cell carcinomas), we examined whether the risk associated with cigarette smoking, body mass index (BMI), gastroesophageal reflux disease (GERD), and non-steroidal anti-inflammatory drug (NSAID) use varied by P53 overexpression.
The proportion of cases overexpressing P53 by tumor subtype was 72% for esophageal
adenocarcinoma
, 69% for
gastric
cardia adenocarcinoma
, 52% for non-
cardia
gastric
adenocarcinoma
, and 67% for esophageal squamous cell carcinoma.
For non-
cardia
gastric
cancer however, an association with cigarette smoking was suggested for tumors that do not overexpress P53, whereas larger BMI was related to adenocarcinomas that overexpress P53 versus no overexpression.
Overall, this study did not find a clear relationship between P53 protein overexpression and the known risk factors for subtypes of esophageal and
gastric
cancers.
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[Cites]
J Histochem Cytochem. 1981 Apr;29(4):577-80
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[ISSN]
1573-7225
[Journal-full-title]
Cancer causes & control : CCC
[ISO-abbreviation]
Cancer Causes Control
[Language]
ENG
[Grant]
None / None / / U01 CA057923-03; United States / NCI NIH HHS / CA / U01 CA057949; United States / NCI NIH HHS / CA / U01 CA057983; None / None / / U01 CA057983-03; United States / Intramural NIH HHS / / Z01 CP010136-12; United States / NCI NIH HHS / CA / U01 CA057923; United States / NCI NIH HHS / CA / U01 CA 57923; United States / NCI NIH HHS / CA / U01 CA057949-03; United States / NCI NIH HHS / CA / U01 CA057923-03; United States / NCI NIH HHS / CA / U01 CA057983-03; United States / NCI NIH HHS / CA / U01 CA 57983; United States / NCI NIH HHS / CA / U01 CA 57049; None / None / / U01 CA057949-03
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural
[Publication-country]
Netherlands
[Chemical-registry-number]
0 / Anti-Inflammatory Agents, Non-Steroidal; 0 / Tumor Suppressor Protein p53
[Other-IDs]
NLM/ NIHMS100106; NLM/ PMC2726999
24.
Wang N, Dong XJ, Zhou RM, Guo W, Zhang XJ, Li Y:
An investigation on the polymorphisms of two DNA repair genes and susceptibility to ESCC and GCA of high-incidence region in northern China.
Mol Biol Rep
; 2009 Feb;36(2):357-64
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AIM: To investigate the possible association of three SNPs, XRCC2 C41657T, XRCC2 G4234C and XRCC3 A17893G with susceptibility to esophageal squamous cell carcinoma (ESCC) and
gastric
cardia adenocarcinoma
(GCA) in a population of northern China.
[MeSH-major]
Adenocarcinoma
/ genetics. Carcinoma, Squamous Cell / genetics. DNA Repair / genetics. DNA-Binding Proteins / genetics. Esophageal Neoplasms / genetics. Polymorphism, Single Nucleotide.
Stomach
Neoplasms / genetics
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[ISSN]
1573-4978
[Journal-full-title]
Molecular biology reports
[ISO-abbreviation]
Mol. Biol. Rep.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Netherlands
[Chemical-registry-number]
0 / DNA-Binding Proteins; 0 / X-ray repair cross complementing protein 3; 0 / XRCC2 protein, human
25.
Isgüder AS, Nazli O, Tansug T, Bozdag AD, Onal MA:
Total gastrectomy for gastric carcinoma.
Hepatogastroenterology
; 2005 Jan-Feb;52(61):302-4
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[Title]
Total gastrectomy for
gastric
carcinoma.
BACKGROUND/AIMS:
Gastric
cancer is one of the most common organ cancers all around the world and surgical resection is essential for treatment.
Total gastrectomy is the procedure of choice for treatment of proximal
gastric
cancer.
METHODOLOGY: Thirty-eight
gastric
cancer patients underwent total gastrectomy in the Third Surgical Clinic of Izmir Ataturk Training and Research Hospital between 1996 and 2001.
Sites of the tumors were:
cardia
28.9%,
cardia
and corpus 15.8%, corpus 34.3%, corpus and antrum 18.4%, linitis plastica 2.6%.
Histological types were
adenocarcinoma
(97.4%), and squamous cell carcinoma (2.6%).
Gastric
tubes were removed on the fourth postoperative day.
[MeSH-major]
Adenocarcinoma
/ surgery. Carcinoma, Squamous Cell / surgery. Gastrectomy.
Stomach
Neoplasms / surgery
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(PMID = 15783055.001).
[ISSN]
0172-6390
[Journal-full-title]
Hepato-gastroenterology
[ISO-abbreviation]
Hepatogastroenterology
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Greece
26.
Langman M, Logan R:
Risk of cancer and acid suppressant treatment.
Gut
; 2007 Jul;56(7):1023
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[MeSH-major]
Adenocarcinoma
/ chemically induced. Antacids / adverse effects. Esophageal Neoplasms / chemically induced.
Stomach
Neoplasms / chemically induced
[MeSH-minor]
Anti-Ulcer Agents / adverse effects.
Cardia
. Humans. Research Design
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[Cites]
Gut. 2003 Jul;52(7):942-6
[
12801948.001
]
[Cites]
Br Med J (Clin Res Ed). 1983 May 28;286(6379):1713-6
[
6405946.001
]
[CommentOn]
Gut. 2006 Nov;55(11):1538-44
[
16785284.001
]
(PMID = 17566037.001).
[ISSN]
0017-5749
[Journal-full-title]
Gut
[ISO-abbreviation]
Gut
[Language]
eng
[Publication-type]
Comment; Letter
[Publication-country]
England
[Chemical-registry-number]
0 / Antacids; 0 / Anti-Ulcer Agents
[Other-IDs]
NLM/ PMC1994362
27.
Bor S, Vardar R, Ormeci N, Memik F, Suleymanlar I, Oguz D, Colakoglu S, Yucesoy M, Turkdogan K, Gurel S, Dogan I, Yildirim B, Goral V, Dokmeci G, Okcu N, Duman D, Simsek I, Demir A:
Prevalence patterns of gastric cancers in Turkey: model of a developing country with high occurrence of Helicobacter pylori.
J Gastroenterol Hepatol
; 2007 Dec;22(12):2242-5
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[Title]
Prevalence patterns of
gastric
cancers in Turkey: model of a developing country with high occurrence of Helicobacter pylori.
AIM: In developed countries, there has been a recent increase in the prevalence
of adenocarcinoma
of the esophagus and
cardia
, along with a decrease in distal
gastric
cancers.
The aim of the present study was to evaluate changes in the prevalence of
gastric
adenocarcinomas in Turkey as a function of anatomic location.
Owing to the exclusion criteria, a total of 4065 cases of tumors of the
stomach
and distal esophagus were included.
The ratio of distal/proximal
adenocarcinoma
was 2,1 [corrected] for the western part of Turkey and 3,8 [corrected] for the eastern part of the country (P < 0.0001), and this did not change during the 11 years. H. pylori was detected significantly less in the west compared to the east for distal tumors (65.7 vs 38.7%, respectively, P = 0.02).
CONCLUSION: In Turkey, a developing country with a high H. pylori prevalence, contrary to the state of developed countries, the ratio of distal versus proximal
gastric
adenocarcinomas has not changed.
Geographical distribution should be taken into the account in projecting the changing patterns of
gastric
cancers.
[MeSH-major]
Developing Countries. Helicobacter pylori.
Stomach
Neoplasms / epidemiology.
Stomach
Neoplasms / microbiology
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[ErratumIn]
J Gastroenterol Hepatol. 2008 Aug;23(8 Pt 1):1309
(PMID = 18031388.001).
[ISSN]
0815-9319
[Journal-full-title]
Journal of gastroenterology and hepatology
[ISO-abbreviation]
J. Gastroenterol. Hepatol.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Australia
28.
Zhang CH, Zhan WH, He YL, Chen CQ, Huang MJ, Cai SR:
Spleen preservation in radical surgery for gastric cardia cancer.
Ann Surg Oncol
; 2007 Apr;14(4):1312-9
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[Title]
Spleen preservation in radical surgery for
gastric
cardia
cancer.
BACKGROUND: In
gastric
cardia
cancer (GCC), the spleen is usually removed when the tumor is resected.
The purpose of this study was to investigate the effect of spleen preservation on survival following radical resection for
gastric
cardia
cancer.
CONCLUSIONS: Splenectomy does not improve survival of patients who undergo curative resection for
gastric
cardia
cancer.
[MeSH-major]
Adenocarcinoma
/ surgery.
Cardia
. Spleen / physiology.
Stomach
Neoplasms / surgery
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(PMID = 17265118.001).
[ISSN]
1068-9265
[Journal-full-title]
Annals of surgical oncology
[ISO-abbreviation]
Ann. Surg. Oncol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
29.
Huerta JM, Navarro C, Chirlaque MD, Tormo MJ, Steindorf K, Buckland G, Carneiro F, Johnsen NF, Overvad K, Stegger J, Tjønneland A, Boutron-Ruault MC, Clavel-Chapelon F, Morois S, Boeing H, Kaaks R, Rohrmann S, Vigl M, Lagiou P, Trichopoulos D, Trichopoulou A, Bas Bueno-de-Mesquita H, Monninkhof EM, Numans ME, Peeters PH, Mattiello A, Pala V, Palli D, Tumino R, Vineis P, Agudo A, Ardanaz E, Arriola L, Molina-Montes E, Rodríguez L, Lindkvist B, Manjer J, Stenling R, Lund E, Crowe FL, Key TJ, Khaw KT, Wareham NJ, Jenab M, Norat T, Romaguera D, Riboli E, González CA:
Prospective study of physical activity and risk of primary adenocarcinomas of the oesophagus and stomach in the EPIC (European Prospective Investigation into Cancer and nutrition) cohort.
Cancer Causes Control
; 2010 May;21(5):657-69
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[Title]
Prospective study of physical activity and risk of primary adenocarcinomas of the oesophagus and
stomach
in the EPIC (European Prospective Investigation into Cancer and nutrition) cohort.
OBJECTIVE: To analyse the association between types of physical activity (occupational, recreational and household, vigorous and overall) and risk of primary oesophageal (OAC) or
gastric
adenocarcinoma
(GAC).
Analyses were repeated by tumour site (
cardia
/non-
cardia
) and histological type (intestinal/diffuse).
A lower risk of overall and non-
cardia
GAC was found for increasing levels of a PA index which combined occupational PA with weekly time spent in sports and cycling.
The hazard ratio (HR) of GAC was 0.69, 95% CI: 0.50-0.94, for the comparison between active and inactive participants according to the PA index (HR = 0.44, 95% CI:0.26-0.74, for non-
cardia
GAC).
No effect was found for
cardia
tumours or histological subtypes of GAC.
CONCLUSIONS: Overall and distal (non-
cardia
)
gastric
tumours were inversely associated with time spent on cycling and sports and a total PA index.
No association was found for any type of PA and risk
of cardia
cancers of the
stomach
.
[MeSH-major]
Adenocarcinoma
/ epidemiology. Esophageal Neoplasms / epidemiology. Exercise. Health Behavior. Nutrition Surveys.
Stomach
Neoplasms / epidemiology
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(PMID = 20052611.001).
[ISSN]
1573-7225
[Journal-full-title]
Cancer causes & control : CCC
[ISO-abbreviation]
Cancer Causes Control
[Language]
eng
[Grant]
United Kingdom / Medical Research Council / / G0401527; United Kingdom / Medical Research Council / / MC/ U106179471; United Kingdom / Medical Research Council / / ; United Kingdom / Cancer Research UK / /
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Netherlands
30.
Trouillet N, Robert B, Charfi S, Bartoli E, Joly JP, Chatelain D:
Gastric metastases. An endoscopic series of ten cases.
Gastroenterol Clin Biol
; 2010 Apr-May;34(4-5):305-9
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[Title]
Gastric
metastases. An endoscopic series of ten cases.
We report a series of ten cases of the clinical, endoscopic and pathological features of
gastric
metastases.
Patients were six women and four men between 54 and 88 years old, with
gastric
metastases from breast carcinoma (4), lung carcinoma (4) and melanoma (2).
Metastases were located in the
cardia
(2), fundus (5) and antrum (3).
The microscopic features of
the gastric
metastases resembled a primary
gastric
cancer in eight cases.
Thanks to clinical data, the pathologist confirmed the diagnosis of
gastric
metastases on immunohistochemistry.
Gastric
metastases are rare, occur at a late stage of the neoplastic disease, and have a poor prognosis.
Diagnosis of
gastric
metastases is difficult because they simulate primary
gastric
cancer on endoscopy and on microscopic examination.
[MeSH-major]
Endoscopy, Gastrointestinal.
Stomach
Neoplasms / pathology.
Stomach
Neoplasms / secondary
[MeSH-minor]
Adenocarcinoma
/ pathology.
Adenocarcinoma
/ secondary. Aged. Aged, 80 and over. Carcinoma, Lobular / pathology. Carcinoma, Lobular / secondary. Female. Humans. Male. Middle Aged. Tomography, X-Ray Computed
Genetic Alliance.
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(PMID = 20627637.001).
[ISSN]
0399-8320
[Journal-full-title]
Gastroentérologie clinique et biologique
[ISO-abbreviation]
Gastroenterol. Clin. Biol.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
France
31.
DiMusto PD, Orringer MB:
Transhiatal esophagectomy for distal and cardia cancers: implications of a positive gastric margin.
Ann Thorac Surg
; 2007 Jun;83(6):1993-8; discussion 1998-9
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[Title]
Transhiatal esophagectomy for distal and
cardia
cancers: implications of a positive
gastric
margin.
BACKGROUND: A common operation for cancer of the esophagus and
cardia
consists of transhiatal esophagectomy, proximal gastrectomy, and a cervical esophagogastric anastomosis.
The oncologic adequacy of dividing the
stomach
4 to 6 cm distal to palpable tumor is not well documented, and when a positive
gastric
margin is present on the final pathologic analysis, the appropriate management is not established.
This study was undertaken to determine the incidence of a positive
gastric
margin in these patients and the impact of adjuvant treatment.
METHODS: A retrospective review was performed of 1044 patients undergoing transhiatal esophagectomy for
adenocarcinoma of
the distal esophagus or
cardia
.
Twenty (1.9%) had a positive
gastric
margin on final the pathologic evaluation and met inclusion criteria for this study.
CONCLUSIONS: A transhiatal esophagectomy and proximal gastrectomy for carcinoma of the distal esophagus and
cardia
, dividing the
stomach
4 to 6 cm from palpable tumor, provides a negative
gastric
margin in 98% of patients.
In the few patients who have a positive
gastric
margin, 80% die with distant metastases, which would not be influenced by more extensive
gastric
resection, and in about 20%, local tumor recurrence develops in the intrathoracic
stomach
, seldom causing dysphagia.
Adjuvant therapy for a positive
gastric
margin neither improves survival nor reduces local tumor recurrence.
[MeSH-major]
Adenocarcinoma
/ surgery. Esophageal Neoplasms / surgery. Esophagectomy / methods.
Stomach
Neoplasms / surgery
[MeSH-minor]
Anastomosis, Surgical.
Cardia
/ surgery. Combined Modality Therapy. Deglutition Disorders / prevention & control. Esophagus / surgery. Gastrectomy / methods. Humans. Neoplasm Recurrence, Local / prevention & control. Retrospective Studies.
Stomach
/ surgery. Survival Analysis
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(PMID = 17532385.001).
[ISSN]
1552-6259
[Journal-full-title]
The Annals of thoracic surgery
[ISO-abbreviation]
Ann. Thorac. Surg.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Netherlands
32.
Sadighi S, Raafat J, Mohagheghi M, Meemary F:
Gastric carcinoma: 5 year experience of a single institute.
Asian Pac J Cancer Prev
; 2005 Apr-Jun;6(2):195-6
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[Title]
Gastric
carcinoma: 5 year experience of a single institute.
PURPOSE:
Gastric
cancer (GC) is the most common cause of cancer death registered in cancer institute.
Most common site of involvement was
cardia
(43%).
[MeSH-major]
Adenocarcinoma
/ therapy.
Stomach
Neoplasms / therapy
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(PMID = 16101332.001).
[ISSN]
1513-7368
[Journal-full-title]
Asian Pacific journal of cancer prevention : APJCP
[ISO-abbreviation]
Asian Pac. J. Cancer Prev.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Thailand
33.
Guo W, Cui YJ, Fang SM, Li Y, Wang N, Zhang JH:
[Association of polymorphisms of p21cip1 and p27kip1 genes with susceptibilities of esophageal squamous cell carcinoma and gastric cardiac adenocarcinoma].
Ai Zheng
; 2006 Feb;25(2):194-9
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[Title]
[Association of polymorphisms of p21cip1 and p27kip1 genes with susceptibilities of esophageal squamous cell carcinoma and
gastric
cardiac
adenocarcinoma
].
This study was to investigate the possible association of functional polymorphisms of p21(cip1) and p27(kip1) genes with susceptibilities of esophageal squamous cell carcinoma (ESCC) and
gastric
cardiac
adenocarcinoma
(GCA) in a population from a high incidence region in north China.
[MeSH-major]
Cyclin-Dependent Kinase Inhibitor p21 / genetics. Cyclin-Dependent Kinase Inhibitor p27 / genetics. Esophageal Neoplasms / genetics. Polymorphism, Single Nucleotide / genetics.
Stomach
Neoplasms / genetics
[MeSH-minor]
Adenocarcinoma
/ genetics. Carcinoma, Squamous Cell / genetics.
Cardia
. Female. Gene Frequency. Genetic Predisposition to Disease. Genotype. Humans. Male. Middle Aged. Smoking
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(PMID = 16480585.001).
[Journal-full-title]
Ai zheng = Aizheng = Chinese journal of cancer
[ISO-abbreviation]
Ai Zheng
[Language]
chi
[Publication-type]
English Abstract; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
China
[Chemical-registry-number]
0 / CDKN1A protein, human; 0 / Cyclin-Dependent Kinase Inhibitor p21; 147604-94-2 / Cyclin-Dependent Kinase Inhibitor p27
34.
Hida Y, Kubo Y, Nishio Y, Murakami S, Fukumoto D, Sayama K, Hashimoto K, Arase S:
Malignant acanthosis nigricans with enhanced expression of fibroblast growth factor receptor 3.
Acta Derm Venereol
; 2009;89(4):435-7
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[MeSH-minor]
Adenocarcinoma
/ complications. Aged.
Cardia
. Fatal Outcome. Humans. Hypertrophy. Immunohistochemistry. Male. Skin / pathology.
Stomach
Neoplasms / metabolism
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(PMID = 19688170.001).
[ISSN]
1651-2057
[Journal-full-title]
Acta dermato-venereologica
[ISO-abbreviation]
Acta Derm. Venereol.
[Language]
eng
[Publication-type]
Case Reports; Letter
[Publication-country]
Sweden
[Chemical-registry-number]
EC 2.7.10.1 / Receptor, Fibroblast Growth Factor, Type 3
35.
Sampliner RE, Camargo E, Prasad AR:
Association of ablation of Barrett's esophagus with high grade dysplasia and adenocarcinoma of the gastric cardia.
Dis Esophagus
; 2006;19(4):277-9
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[Title]
Association of ablation of Barrett's esophagus with high grade dysplasia and
adenocarcinoma of
the gastric
cardia
.
Three cases are reported in which the patient developed
adenocarcinoma of
the gastric
cardia
after thermal ablation of HGD.
Three cases are reported with long-segment BE and a nodule or mass in the endoscopic
cardia
post-thermal ablation.
Biopsies documented
adenocarcinoma of
the gastric
cardia
.
The development
of adenocarcinoma
of the
cardia
is unexpected.
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(PMID = 16866860.001).
[ISSN]
1120-8694
[Journal-full-title]
Diseases of the esophagus : official journal of the International Society for Diseases of the Esophagus
[ISO-abbreviation]
Dis. Esophagus
[Language]
ENG
[Publication-type]
Case Reports; Journal Article
[Publication-country]
United States
36.
Capellá G, Pera G, Sala N, Agudo A, Rico F, Del Giudicce G, Plebani M, Palli D, Boeing H, Bueno-de-Mesquita HB, Carneiro F, Berrino F, Vineis P, Tumino R, Panico S, Berglund G, Simán H, Nyrén O, Hallmans G, Martinez C, Dorronsoro M, Barricarte A, Navarro C, Quirós JR, Allen N, Key T, Bingham S, Caldas C, Linseisen J, Nagel G, Overvad K, Tjonneland A, Boshuizen HC, Peeters PH, Numans ME, Clavel-Chapelon F, Trichopoulou A, Lund E, Jenab M, Kaaks R, Riboli E, González CA:
DNA repair polymorphisms and the risk of stomach adenocarcinoma and severe chronic gastritis in the EPIC-EURGAST study.
Int J Epidemiol
; 2008 Dec;37(6):1316-25
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[Title]
DNA repair polymorphisms and the risk
of stomach adenocarcinoma
and severe chronic gastritis in the EPIC-EURGAST study.
BACKGROUND: The contribution of genetic variation in DNA repair genes to
gastric
cancer (GC) risk remains essentially unknown.
Method A nested case control study within the EPIC cohort was performed including 246
gastric
adenocarcinomas and 1175 matched controls.
RESULTS: No association was observed for any of these polymorphisms with
stomach
cancer risk.
This is the first prospective study suggesting that individual variation in DNA repair may be relevant for
gastric
carcinogenesis, a finding that will require further confirmation validation in larger independent studies.
[MeSH-major]
Adenocarcinoma
/ genetics. DNA Repair / genetics. Gastritis, Atrophic / genetics. Polymorphism, Genetic.
Stomach
Neoplasms / genetics
[MeSH-minor]
Adult. Aged. Antibodies, Bacterial / blood. Biomarkers / blood.
Cardia
/ pathology. Case-Control Studies. Chronic Disease. Europe. Female. Follow-Up Studies. Gene Frequency. Genes, p53. Genetic Predisposition to Disease. Helicobacter Infections / complications. Helicobacter Infections / immunology. Helicobacter pylori / immunology. Humans. Male. Middle Aged. Odds Ratio. Pepsinogen A / blood. Prospective Studies. Risk. Xeroderma Pigmentosum Group D Protein / genetics
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(PMID = 18641418.001).
[ISSN]
1464-3685
[Journal-full-title]
International journal of epidemiology
[ISO-abbreviation]
Int J Epidemiol
[Language]
eng
[Grant]
United Kingdom / British Heart Foundation / / ; United Kingdom / Cancer Research UK / / ; United Kingdom / Medical Research Council / / ; United Kingdom / Wellcome Trust / /
[Publication-type]
Journal Article; Multicenter Study; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / Antibodies, Bacterial; 0 / Biomarkers; 9001-10-9 / Pepsinogen A; EC 3.6.4.12 / Xeroderma Pigmentosum Group D Protein; EC 5.99.- / ERCC2 protein, human
37.
Wen D, Shan B, Wang S, Zhang L, Wei L, Zhou W, Peng Q:
A positive family history of esophageal/gastric cardia cancer with gastric cardia adenocarcinoma is associated with a younger age at onset and more likely with another synchronous esophageal/gastric cardia cancer in a Chinese high-risk area.
Eur J Med Genet
; 2010 Sep-Oct;53(5):250-5
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[Title]
A positive family history of esophageal/
gastric
cardia
cancer with
gastric
cardia adenocarcinoma
is associated with a younger age at onset and more likely with another synchronous esophageal/
gastric
cardia
cancer in a Chinese high-risk area.
BACKGROUND: To find a genetic component in
gastric
cardia
adenocarcinomas (GCA).
[MeSH-major]
Adenocarcinoma
/ genetics. Asian Continental Ancestry Group.
Cardia
/ pathology. Esophageal Neoplasms / genetics. Neoplasms, Multiple Primary / pathology.
Stomach
Neoplasms / genetics
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[Copyright]
Copyright © 2010 Elsevier Masson SAS. All rights reserved.
(PMID = 20603233.001).
[ISSN]
1878-0849
[Journal-full-title]
European journal of medical genetics
[ISO-abbreviation]
Eur J Med Genet
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Netherlands
38.
Mottershead M, Karteris E, Barclay JY, Suortamo S, Newbold M, Randeva H, Nwokolo CU:
Immunohistochemical and quantitative mRNA assessment of ghrelin expression in gastric and oesophageal adenocarcinoma.
J Clin Pathol
; 2007 Apr;60(4):405-9
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[Title]
Immunohistochemical and quantitative mRNA assessment of ghrelin expression in
gastric
and oesophageal
adenocarcinoma
.
BACKGROUND: Ghrelin is an orexigenic gut peptide produced predominantly by the
stomach
.
Gastric
mucosal ghrelin production could be compromised by an infiltrating
adenocarcinoma
.
METHODS: 10
gastric
and 22 oesophageal
adenocarcinoma
archival samples were randomly selected from a database.
Quantitative reverse transcriptase polymerase chain reaction (PCR) for ghrelin mRNA was also performed on 24
gastric
and 8 oesophageal
adenocarcinoma
specimens and adjacent non-neoplastic mucosa.
RESULTS: Immunohistochemistry and reverse transcriptase PCR confirm a negligible expression of ghrelin in
adenocarcinoma
specimens.
By contrast, non-neoplastic
gastric
mucosa was rich in ghrelin-positive cells and ghrelin mRNA.
The number (median and range) of ghrelin-positive cells per 2 mm section of non-neoplastic mucosa was 73 (45-215) in the corpus; this was significantly higher than in
cardia
mucosa (9 (0-64), p<0.001) and antral mucosa (5 (0-14), p<0.001).
CONCLUSIONS:
Gastric
and oesophageal adenocarcinomas have no ghrelin-producing cells.
The highest level of ghrelin expression was noted in the non-neoplastic mucosa of
the gastric
corpus.
Disruption of
the gastric
ghrelin-producing mechanism may occur during oesophagogastric malignancy.
[MeSH-major]
Adenocarcinoma
/ metabolism. Esophageal Neoplasms / metabolism. Peptide Hormones / biosynthesis.
Stomach
Neoplasms / metabolism
[MeSH-minor]
Gastric
Mucosa / metabolism. Ghrelin. Humans. Immunoenzyme Techniques. Neurosecretory Systems / metabolism. RNA, Messenger / genetics. RNA, Neoplasm / genetics. Reverse Transcriptase Polymerase Chain Reaction / methods
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[ISSN]
0021-9746
[Journal-full-title]
Journal of clinical pathology
[ISO-abbreviation]
J. Clin. Pathol.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
England
[Chemical-registry-number]
0 / Ghrelin; 0 / Peptide Hormones; 0 / RNA, Messenger; 0 / RNA, Neoplasm
[Other-IDs]
NLM/ PMC2001112
39.
Cao YY, Ge H, Chen LQ, Chen ZF, Wen DG, Li Y, Zhang JH:
[Correlation of 53BP1 and p53 polymorphisms to susceptibility to esophageal squamous cell carcinoma and gastric cardiac adenocarcinoma].
Ai Zheng
; 2007 Oct;26(10):1052-7
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[Title]
[Correlation of 53BP1 and p53 polymorphisms to susceptibility to esophageal squamous cell carcinoma and
gastric
cardiac
adenocarcinoma
].
This study was to investigate the correlation of 53BP1 and p53 SNPs to susceptibility to esophageal squamous cell carcinoma (ESCC) and
gastric
cardiac
adenocarcinoma
(GCA) in a high incidence area of Hebei Province in China.
[MeSH-major]
Cardia
. Esophageal Neoplasms / genetics. Intracellular Signaling Peptides and Proteins / genetics. Polymorphism, Single Nucleotide.
Stomach
Neoplasms / genetics. Tumor Suppressor Protein p53 / genetics
[MeSH-minor]
Adenocarcinoma
/ epidemiology.
Adenocarcinoma
/ genetics.
Adenocarcinoma
/ metabolism. Adult. Aged. Aged, 80 and over. Alleles. Carcinoma, Squamous Cell / epidemiology. Carcinoma, Squamous Cell / genetics. Carcinoma, Squamous Cell / metabolism. China / epidemiology. Female. Gene Frequency. Genetic Predisposition to Disease. Genotype. Humans. Male. Middle Aged
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.
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(PMID = 17927872.001).
[Journal-full-title]
Ai zheng = Aizheng = Chinese journal of cancer
[ISO-abbreviation]
Ai Zheng
[Language]
chi
[Publication-type]
English Abstract; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
China
[Chemical-registry-number]
0 / Intracellular Signaling Peptides and Proteins; 0 / TP53BP1 protein, human; 0 / Tumor Suppressor Protein p53
40.
Shen JG, Cheong JH, Hyung WJ, Kim J, Choi SH, Noh SH:
Influence of a microscopic positive proximal margin in the treatment of gastric adenocarcinoma of the cardia.
World J Gastroenterol
; 2006 Jun 28;12(24):3883-6
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[Title]
Influence of a microscopic positive proximal margin in the treatment of
gastric
adenocarcinoma of
the
cardia
.
AIM: To investigate the influence of a positive proximal margin in total gastrectomy patients with
gastric
adenocarcinoma of
the
cardia
.
METHODS: Medical records of 191 patients with total gastrectomies for
adenocarcinoma of
the
cardia
between 1995 and 2000 were reviewed.
CONCLUSION: A positive margin is more of an indication of advanced disease in patients with
gastric
adenocarcinoma of
the
cardia
rather than an independent prognostic factor for survival.
[MeSH-major]
Adenocarcinoma
/ pathology.
Adenocarcinoma
/ surgery.
Cardia
/ pathology. Gastrectomy / methods.
Stomach
Neoplasms / pathology.
Stomach
Neoplasms / surgery
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[ISSN]
1007-9327
[Journal-full-title]
World journal of gastroenterology
[ISO-abbreviation]
World J. Gastroenterol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
China
[Other-IDs]
NLM/ PMC4087938
41.
Vissers KJ, Dinjens WN, Riegman PH, Tilanus HW, van Dekken H:
Allelic imbalance on distal 7q (7q36.1-q36.3) in gastric cardia and oesophageal (Barrett's) adenocarcinoma.
Anticancer Res
; 2005 Mar-Apr;25(2A):913-6
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[Title]
Allelic imbalance on distal 7q (7q36.1-q36.3) in
gastric
cardia
and oesophageal (Barrett's)
adenocarcinoma
.
BACKGROUND: Oesophageal (Barrett's) and
gastric
cardia
adenocarcinomas are cancers arising at and around the gastro-oesophageal junction.
In addition, 40
gastric
cardia
cancers were investigated to compare the pattern of imbalance at these loci.
RESULTS: Overall, the number of allelic loss was higher in Barrett's cancers than in
gastric
cardia
carcinomas (p=0.04).
In
gastric
cardia
cancers, loss ranged from 12% to 27% (of informative cases), being most frequent at marker D7S3037.
The difference between oesophageal and
gastric
adenocarcinomas was highest for polymorphic marker D7S483 (p=0.05).
CONCLUSION: Marker D7S483 can aid in discriminating oesophageal (Barrett's) and
gastric
cardia
carcinomas.
[MeSH-major]
Adenocarcinoma
/ genetics. Allelic Imbalance. Barrett Esophagus / genetics.
Cardia
/ pathology. Chromosomes, Human, Pair 7 / genetics. Esophageal Neoplasms / genetics.
Stomach
Neoplasms / genetics
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(PMID = 15868927.001).
[ISSN]
0250-7005
[Journal-full-title]
Anticancer research
[ISO-abbreviation]
Anticancer Res.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Greece
42.
Freedman ND, Abnet CC, Leitzmann MF, Mouw T, Subar AF, Hollenbeck AR, Schatzkin A:
A prospective study of tobacco, alcohol, and the risk of esophageal and gastric cancer subtypes.
Am J Epidemiol
; 2007 Jun 15;165(12):1424-33
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[Title]
A prospective study of tobacco, alcohol, and the risk of esophageal and
gastric
cancer subtypes.
Rates of esophageal
adenocarcinoma
and
gastric
cardia adenocarcinoma
have increased, while rates of esophageal squamous cell carcinoma (ESCC) and
gastric
noncardia
adenocarcinoma
have decreased, suggesting distinct etiologies.
Between 1995/1996 and 2000, 97 incident cases of ESCC, 205 of esophageal
adenocarcinoma
, 188 of
gastric
cardia
, and 187 of
gastric
noncardia cancer occurred.
Compared with nonsmokers, current smokers were at increased risk for ESCC (hazard ratio (HR) = 9.27, 95% confidence interval (CI): 4.04, 21.29), esophageal
adenocarcinoma
(HR = 3.70, 95% CI: 2.20, 6.22),
gastric
cardia
(HR = 2.86, 95% CI: 1.73, 4.70), and
gastric
noncardia (HR = 2.04, 95% CI: 1.32, 3.16).
Assuming causality, ever smoking had population attributable risks of 77% (95% CI: 0.55, 0.89) for ESCC, 58% (95% CI: 0.38, 0.72) for esophageal
adenocarcinoma
, 47% (95% CI: 0.27, 0.63) for
gastric
cardia
, and 19% (95% CI: 0.00, 0.37) for
gastric
noncardia.
For drinkers of more than three alcoholic beverages per day, compared with those whose intake was up to one drink per day, the authors found significant associations between alcohol intake and ESCC risk (HR = 4.93, 95% CI: 2.69, 9.03) but not risk for esophageal,
gastric
cardia
, or
gastric
noncardia
adenocarcinoma
.
[MeSH-major]
Adenocarcinoma
/ etiology. Alcohol Drinking / adverse effects. Carcinoma, Squamous Cell / etiology. Esophageal Neoplasms / etiology. Smoking / adverse effects.
Stomach
Neoplasms / etiology
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(PMID = 17420181.001).
[ISSN]
0002-9262
[Journal-full-title]
American journal of epidemiology
[ISO-abbreviation]
Am. J. Epidemiol.
[Language]
eng
[Grant]
United States / Intramural NIH HHS / /
[Publication-type]
Journal Article; Research Support, N.I.H., Intramural
[Publication-country]
United States
43.
Steevens J, Botterweck AA, Dirx MJ, van den Brandt PA, Schouten LJ:
Trends in incidence of oesophageal and stomach cancer subtypes in Europe.
Eur J Gastroenterol Hepatol
; 2010 Jun;22(6):669-78
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[Title]
Trends in incidence of oesophageal and
stomach
cancer subtypes in Europe.
OBJECTIVE: Time trend studies in the USA have shown that the incidences of adenocarcinomas of the oesophagus and
gastric
cardia
have risen strongly since the 1970s, whereas the incidence of squamous cell carcinomas of the oesophagus has declined.
The main goal of this study was to investigate more recent trends in the incidence of oesophageal and
stomach
cancer subtypes in the European countries.
METHODS: Eurocim cancer incidence data of 23 cancer registries from 13 European countries were used to investigate the incidence trends in oesophageal and
stomach
cancer subtypes during the 1983-1997 period.
RESULTS: The incidence of adenocarcinomas of the oesophagus and
gastric
cardia
rose in most, but not all, registration areas (EAPCs were usually 1-7%), the strongest in the UK and Ireland.
CONCLUSION: Our results are partly in line with earlier findings on adenocarcinomas of the oesophagus and
gastric
cardia
.
[MeSH-major]
Adenocarcinoma
/ epidemiology. Esophageal Neoplasms / epidemiology. Neoplasms, Squamous Cell / epidemiology.
Stomach
Neoplasms / epidemiology
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(PMID = 19474750.001).
[ISSN]
1473-5687
[Journal-full-title]
European journal of gastroenterology & hepatology
[ISO-abbreviation]
Eur J Gastroenterol Hepatol
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
44.
Geddert H, Kiel S, Zotz RB, Zhang J, Willers R, Gabbert HE, Sarbia M:
Polymorphism of p16 INK4A and cyclin D1 in adenocarcinomas of the upper gastrointestinal tract.
J Cancer Res Clin Oncol
; 2005 Dec;131(12):803-8
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METHODS: Using PCR based restriction fragment length polymorphism and single strand conformational polymorphism, we determined single nucleotide exchanges in the p16 and cyclin D1 genes among 56 esophageal adenocarcinomas (ADC) arising in Barrett's esophagus, 95 cardiac
gastric
ADC, and in 191 distal
gastric
ADC.
RESULTS: The C/G genotype of p16 was identified in 10.4% of esophageal carcinomas, 13.3% of cardiac carcinomas, and in 14.1% of
gastric
carcinomas, compared to 17.4% in the healthy control group.
In distal
gastric
carcinoma, both homozygous genotypes (G/G and A/A) had a frequency of 15.2% each, while the heterozygous A/G genotype occurred in 69.6% of patients.
CONCLUSIONS: Our results show that frequencies of p16 or cyclin D1 polymorphisms in
gastric
and esophageal ADC do not differ significantly from the healthy control group.
[MeSH-major]
Adenocarcinoma
/ genetics. Cyclin D1 / genetics. Cyclin-Dependent Kinase Inhibitor p16 / genetics. Esophageal Neoplasms / genetics. Polymorphism, Genetic.
Stomach
Neoplasms / genetics
[MeSH-minor]
Adult. Aged. Aged, 80 and over. Alanine. Barrett Esophagus / complications.
Cardia
. Case-Control Studies. Cysteine. DNA, Neoplasm / analysis. Female. Gene Frequency. Glycine. Homozygote. Humans. Male. Middle Aged. Polymerase Chain Reaction. Polymorphism, Restriction Fragment Length. Polymorphism, Single Nucleotide. Polymorphism, Single-Stranded Conformational
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[Cites]
Carcinogenesis. 2001 Aug;22(8):1195-9
[
11470749.001
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Oncology. 2001;61(1):1-9
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Cancer Epidemiol Biomarkers Prev. 2003 Feb;12(2):176
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Hum Pathol. 1994 Oct;25(10):982-93
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Genes Chromosomes Cancer. 2000 Aug;28(4):404-14
[
10862049.001
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Cancer Epidemiol Biomarkers Prev. 2002 Jul;11(7):640-5
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12101111.001
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Oncogene. 1995 Sep 7;11(5):1005-11
[
7675441.001
]
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Semin Oncol. 2004 Aug;31(4):476-86
[
15297940.001
]
[Cites]
Gastroenterology. 2002 May;122(6):1569-91
[
12016424.001
]
[Cites]
Carcinogenesis. 2003 Sep;24(9):1499-503
[
12807740.001
]
[Cites]
Int J Cancer. 2003 Mar 10;104(1):98-103
[
12532425.001
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Int J Cancer. 2004 Mar;109(1):138-43
[
14735480.001
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[
9462707.001
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[
9823902.001
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Urology. 2004 Jul;64(1):74-8
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Hepatogastroenterology. 2001 Jul-Aug;48(40):1007-10
[
11490786.001
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Biochim Biophys Acta. 1998 Oct 14;1378(2):F115-77
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9823374.001
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Carcinogenesis. 2002 Aug;23(8):1405
[
12151361.001
]
[Cites]
JAMA. 2003 Dec 3;290(21):2843-8
[
14657069.001
]
(PMID = 16163549.001).
[ISSN]
0171-5216
[Journal-full-title]
Journal of cancer research and clinical oncology
[ISO-abbreviation]
J. Cancer Res. Clin. Oncol.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Germany
[Chemical-registry-number]
0 / Cyclin-Dependent Kinase Inhibitor p16; 0 / DNA, Neoplasm; 136601-57-5 / Cyclin D1; K848JZ4886 / Cysteine; OF5P57N2ZX / Alanine; TE7660XO1C / Glycine
45.
Nomura AM, Kolonel LN, Miki K, Stemmermann GN, Wilkens LR, Goodman MT, Perez-Perez GI, Blaser MJ:
Helicobacter pylori, pepsinogen, and gastric adenocarcinoma in Hawaii.
J Infect Dis
; 2005 Jun 15;191(12):2075-81
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[Title]
Helicobacter pylori, pepsinogen, and
gastric
adenocarcinoma
in Hawaii.
BACKGROUND: The objective was to investigate the association of Helicobacter pylori and serum pepsinogen (PG) levels with
gastric
adenocarcinoma
.
RESULTS: Subjects with low PG I levels or low PG I/II ratios were at increased risk for
cardia
and noncardia
gastric
cancer, whereas those with H. pylori or CagA seropositivity had an elevated risk for noncardia cancer only.
CONCLUSIONS: The results suggest that persons with both H. pylori or CagA seropositivity and a low PG I level or PG I/II ratio are highly susceptible to development of noncardia
gastric
cancer.
[MeSH-major]
Adenocarcinoma
/ etiology. Helicobacter Infections / complications. Helicobacter pylori / pathogenicity. Pepsinogen A / blood.
Stomach
Neoplasms / etiology
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(PMID = 15897993.001).
[ISSN]
0022-1899
[Journal-full-title]
The Journal of infectious diseases
[ISO-abbreviation]
J. Infect. Dis.
[Language]
eng
[Grant]
United States / NCI NIH HHS / CA / P01 CA 33619
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, P.H.S.
[Publication-country]
United States
[Chemical-registry-number]
0 / Antibodies, Bacterial; 0 / Antigens, Bacterial; 0 / Bacterial Proteins; 0 / cagA protein, Helicobacter pylori; 9001-10-9 / Pepsinogen A
46.
Alvarez Herrero L, Pouw RE, van Vilsteren FG, ten Kate FJ, Visser M, van Berge Henegouwen MI, Weusten BL, Bergman JJ:
Risk of lymph node metastasis associated with deeper invasion by early adenocarcinoma of the esophagus and cardia: study based on endoscopic resection specimens.
Endoscopy
; 2010 Dec;42(12):1030-6
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[Title]
Risk of lymph node metastasis associated with deeper invasion by early
adenocarcinoma of
the esophagus and
cardia
: study based on endoscopic resection specimens.
BACKGROUND: Most risk estimations for lymph node metastasis in
adenocarcinoma of
the esophagus and
cardia
(AEC) with invasion into the muscularis mucosae (m3) or submucosa are based on surgical series.
[MeSH-major]
Adenocarcinoma
/ secondary.
Cardia
/ pathology. Esophageal Neoplasms / pathology. Mucous Membrane / pathology.
Stomach
Neoplasms / pathology
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[Copyright]
© Georg Thieme Verlag KG Stuttgart · New York.
(PMID = 20960392.001).
[ISSN]
1438-8812
[Journal-full-title]
Endoscopy
[ISO-abbreviation]
Endoscopy
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Germany
47.
Zhang XH:
[Adenocarcinoma arising in gastroesophageal junction: a reappraisal].
Zhonghua Bing Li Xue Za Zhi
; 2007 Jun;36(6):363-5
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[Title]
[
Adenocarcinoma
arising in gastroesophageal junction: a reappraisal].
[MeSH-major]
Adenocarcinoma
/ pathology.
Cardia
. Esophageal Neoplasms / pathology. Esophagogastric Junction / pathology.
Stomach
Neoplasms / pathology
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(PMID = 17822618.001).
[ISSN]
0529-5807
[Journal-full-title]
Zhonghua bing li xue za zhi = Chinese journal of pathology
[ISO-abbreviation]
Zhonghua Bing Li Xue Za Zhi
[Language]
chi
[Publication-type]
Journal Article; Review
[Publication-country]
China
[Number-of-references]
31
48.
Ryan AM, Rowley SP, Fitzgerald AP, Ravi N, Reynolds JV:
Adenocarcinoma of the oesophagus and gastric cardia: male preponderance in association with obesity.
Eur J Cancer
; 2006 May;42(8):1151-8
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[Title]
Adenocarcinoma of
the oesophagus and
gastric
cardia
: male preponderance in association with obesity.
Recent evidence links obesity with the rising incidence of oesophageal
adenocarcinoma
.
In Ireland between 1995 and 2004 the incidence of oesophageal
adenocarcinoma
increased by 38%, and this coincided with a 67% increase in the prevalence of obesity.
In this study, a prospective case-control study was undertaken in 760 patients presenting to a tertiary centre between 1994 and 2004 diagnosed with cancer of the oesophagus,
gastric
cardia
or
stomach
.
Based on pre-illness BMI, 82% of patients who developed
adenocarcinoma of
the oesophagus were either overweight or obese compared with 59% of the healthy control population (P<0.001).
A dose-dependent relationship existed between BMI and oesophageal
adenocarcinoma
in males.
Using common cut-off points for BMI, the OR
of adenocarcinoma
of the lower oesophagus was 11.3 times higher (95% CI: 3.5-36.4) for individuals with a BMI >30 kg/m2 versus individuals with a BMI <22 kg/m2 (P<0.001 for trend).
For
adenocarcinoma of
the gastric
cardia
, males in the top quartile of BMI had an OR of 3.5 (95% CI: 1.3-9.4) compared with the lowest quartile (P=0.03 for trend).
The odds ratio for
adenocarcinoma of
the oesophagus, the oesophago-
gastric
junction and
gastric
cardia
rose significantly with increasing BMI.
[MeSH-major]
Adenocarcinoma
/ etiology. Carcinoma, Squamous Cell / etiology.
Cardia
. Esophageal Neoplasms / etiology. Obesity / complications.
Stomach
Neoplasms / etiology
Genetic Alliance.
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(PMID = 16630714.001).
[ISSN]
0959-8049
[Journal-full-title]
European journal of cancer (Oxford, England : 1990)
[ISO-abbreviation]
Eur. J. Cancer
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
England
49.
Fock KM, Talley N, Moayyedi P, Hunt R, Azuma T, Sugano K, Xiao SD, Lam SK, Goh KL, Chiba T, Uemura N, Kim JG, Kim N, Ang TL, Mahachai V, Mitchell H, Rani AA, Liou JM, Vilaichone RK, Sollano J, Asia-Pacific Gastric Cancer Consensus Conference:
Asia-Pacific consensus guidelines on gastric cancer prevention.
J Gastroenterol Hepatol
; 2008 Mar;23(3):351-65
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[Title]
Asia-Pacific consensus guidelines on
gastric
cancer prevention.
BACKGROUND AND AIM:
Gastric
cancer is a major health burden in the Asia-Pacific region but consensus on prevention strategies has been lacking.
We aimed to critically evaluate strategies for preventing
gastric
cancer.
RESULTS: Helicobacter pylori infection is a necessary but not sufficient causal factor for non-
cardia
gastric
adenocarcinoma
.
A high intake of salt is strongly associated with
gastric
cancer.
Fresh fruits and vegetables are protective but the use of vitamins and other dietary supplements does not prevent
gastric
cancer.
Host-bacterial interaction in H. pylori infection results in different patterns of gastritis and differences in
gastric
acid secretion which determine disease outcome.
A positive family history of
gastric
cancer is an important risk factor.
Low serum pepsinogens reflect
gastric
atrophy and may be useful as a marker to identify populations at high risk for
gastric
cancer. H. pylori screening and treatment is a recommended
gastric
cancer risk reduction strategy in high-risk populations. H. pylori screening and treatment is most effective before atrophic gastritis has developed.
It does not exclude the existing practice of
gastric
cancer surveillance in high-risk populations.
In populations at low risk for
gastric
cancer, H. pylori screening is not recommended.
CONCLUSION: A strategy of H. pylori screening and eradication in high-risk populations will probably reduce
gastric
cancer incidence, and based on current evidence is recommended by consensus.
[MeSH-major]
Adenocarcinoma
/ prevention & control. Anticarcinogenic Agents / therapeutic use. Biomarkers, Tumor / analysis. Helicobacter Infections / drug therapy. Helicobacter pylori. Mass Screening.
Stomach
Neoplasms / prevention & control
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(PMID = 18318820.001).
[ISSN]
1440-1746
[Journal-full-title]
Journal of gastroenterology and hepatology
[ISO-abbreviation]
J. Gastroenterol. Hepatol.
[Language]
eng
[Publication-type]
Consensus Development Conference; Journal Article; Practice Guideline
[Publication-country]
Australia
[Chemical-registry-number]
0 / Anti-Bacterial Agents; 0 / Anticarcinogenic Agents; 0 / Biomarkers, Tumor; 0 / Pepsinogens; 0 / Sodium Chloride, Dietary; 0 / Vitamins; PQ6CK8PD0R / Ascorbic Acid
[Number-of-references]
140
50.
Ghotli ZA, Serra S, Chetty R:
Clear cell (glycogen rich) gastric adenocarcinoma: a distinct tubulo-papillary variant with a predilection for the cardia/gastro-oesophageal region.
Pathology
; 2007 Oct;39(5):466-9
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[Title]
Clear cell (glycogen rich)
gastric
adenocarcinoma
: a distinct tubulo-papillary variant with a predilection for the
cardia
/gastro-oesophageal region.
AIMS: To explore the clinicopathological and immunohistochemical profile of clear cell
gastric
cancers with a tubulo-papillary pattern.
METHODS: Twelve cases of clear cell
gastric
cancer (containing a minimum of 10% of clear cells) were studied.
Ten cases were located in
the gastric
cardia
with extension into the gastro-oesophageal junction and two were in the pylorus/pre-pyloric area.
CONCLUSION: Clear cell
gastric
cancers have a predilection for the gastro-oesophageal junction, are polypoid, have a tubulopapillary pattern, and show over-expression of cyclin D1 but normal E-cadherin.
[MeSH-major]
Adenocarcinoma
, Clear Cell / pathology.
Cardia
/ pathology. Esophagogastric Junction / pathology.
Stomach
Neoplasms / pathology
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[CommentIn]
Pathology. 2008 Apr;40(3):333
[
18428065.001
]
(PMID = 17886094.001).
[ISSN]
0031-3025
[Journal-full-title]
Pathology
[ISO-abbreviation]
Pathology
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
England
[Chemical-registry-number]
0 / Biomarkers, Tumor
51.
Li Y, Sun DL, Duan YN, Zhang XJ, Wang N, Zhou RM, Chen ZF, Wang SJ:
Association of functional polymorphisms in MMPs genes with gastric cardia adenocarcinoma and esophageal squamous cell carcinoma in high incidence region of North China.
Mol Biol Rep
; 2010 Jan;37(1):197-205
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[Title]
Association of functional polymorphisms in MMPs genes with
gastric
cardia adenocarcinoma
and esophageal squamous cell carcinoma in high incidence region of North China.
The aim of the present study was to investigate the association of single nucleotide polymorphisms (SNPs) in matrix metalloproteinase (MMPs) with the risk of
gastric
cardia adenocarcinoma
(GCA) and esophageal squamous cell carcinoma (ESCC).
[MeSH-major]
Esophageal Neoplasms / epidemiology. Esophageal Neoplasms / genetics. Genetic Predisposition to Disease. Matrix Metalloproteinases / genetics. Polymorphism, Single Nucleotide / genetics.
Stomach
Neoplasms / epidemiology.
Stomach
Neoplasms / genetics
[MeSH-minor]
Adenocarcinoma
/ enzymology.
Adenocarcinoma
/ epidemiology.
Adenocarcinoma
/ genetics. Adult. Aged. Aged, 80 and over. Carcinoma, Squamous Cell / enzymology. Carcinoma, Squamous Cell / epidemiology. Carcinoma, Squamous Cell / genetics. Case-Control Studies. China / epidemiology. Female. Gene Frequency / genetics. Haplotypes / genetics. Humans. Incidence. Male. Middle Aged
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[Cites]
Int J Epidemiol. 2000 Aug;29(4):645-54
[
10922340.001
]
[Cites]
Carcinogenesis. 2005 Jun;26(6):1117-21
[
15731163.001
]
[Cites]
J Biol Chem. 2001 Mar 9;276(10):7549-58
[
11114309.001
]
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(PMID = 19562509.001).
[ISSN]
1573-4978
[Journal-full-title]
Molecular biology reports
[ISO-abbreviation]
Mol. Biol. Rep.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Netherlands
[Chemical-registry-number]
EC 3.4.24.- / Matrix Metalloproteinases
52.
Tsavaris N, Kosmas C, Kopterides P, Tsikalakis D, Skopelitis H, Sakelaridi F, Papadoniou N, Tzivras M, Balatsos V, Koufos C, Archimandritis A:
Retinol-binding protein, acute phase reactants and Helicobacter pylori infection in patients with gastric adenocarcinoma.
World J Gastroenterol
; 2005 Dec 7;11(45):7174-8
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[Title]
Retinol-binding protein, acute phase reactants and Helicobacter pylori infection in patients with
gastric
adenocarcinoma
.
AIM: To determine the serum levels of c-reactive protein (CRP), transferrin (TRF), a2-macroglobulin (A2M), ceruloplasmin (CER), a1-acid glycoprotein (AAG), pre-albumin (P-ALB) and retinol-binding protein (RBP) in
gastric
carcinoma patients and to explore their possible correlation with underlying Helicobacter pylori (H pylori) infection.
METHODS: We measured the serum levels of CRP, TRF, A2M, CER, AAG, P-ALB, and RBP in 153 preoperative patients (93 males; mean age: 63.1+/-11.3 years) with non-
cardia
gastric
adenocarcinoma
and 19 healthy subjects.
Retinol-binding protein seems to discriminate between infected and non-infected patients with
gastric
carcinoma.
[MeSH-major]
Adenocarcinoma
/ complications. Helicobacter Infections / complications. Helicobacter pylori.
Stomach
Neoplasms / complications
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(PMID = 16437667.001).
[ISSN]
1007-9327
[Journal-full-title]
World journal of gastroenterology
[ISO-abbreviation]
World J. Gastroenterol.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
China
[Chemical-registry-number]
0 / Acute-Phase Proteins; 0 / Biomarkers, Tumor; 0 / Retinol-Binding Proteins
[Other-IDs]
NLM/ PMC4725071
53.
Mandong BM, Manasseh AN, Tanko MN, Echejoh GO, Madaki AJ:
Epidemiology of gastric cancer in Jos University Teaching Hospital Jos a 20 year review of cases.
Niger J Med
; 2010 Oct-Dec;19(4):451-4
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[Title]
Epidemiology of
gastric
cancer in Jos University Teaching Hospital Jos a 20 year review of cases.
BACKGROUND:
Gastric
cancer believed to be rare in the past in Africa, is now one of the leading cancer morbidity and mortality.
It is now known
gastric
cancer is 2-3 times higher in males than females living in the same environment.
We aim to describe the comprehensive histological characteristics of
gastric
cancer with age and sex distribution.
The study materials were obtained from all stained specimens of
gastric
cancer recorded in the histopathology laboratory of the teaching hospital between 1985 to 2004.
These were divided into the following groups:
Cardia
, body and an thrum/pyloric regions respectively.
RESULTS: There were a total of 205
gastric
cancer histological confirmed, out of 5705 malignant tumours recorded in the same period.
The highest frequencies of
gastric
cancers were located in the anthral and
cardia
regions which accounted for 79% of all the tumours.
Well differentiated
adenocarcinoma
(intestinal type) was the most frequent histological subtypes 51.2%), this was followed by poorly and diffusely infiltrating carcinoma.
The study has also demonstrated H pylori at the background of intestinal type
adenocarcinoma
which was seen in the body and an thrum.
CONCLUSION: The study has shown that
gastric
cancer is not only common but it occur more males than females.
Therefore eradication of H pylori might reduce the prevalence of
gastric
carcinoma.
[MeSH-major]
Carcinoma / epidemiology. Lymphoma, Non-Hodgkin / epidemiology.
Stomach
Neoplasms / epidemiology
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(PMID = 21526638.001).
[ISSN]
1115-2613
[Journal-full-title]
Nigerian journal of medicine : journal of the National Association of Resident Doctors of Nigeria
[ISO-abbreviation]
Niger J Med
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Nigeria
54.
Shen H, Newmann AS, Hu Z, Zhang Z, Xu Y, Wang L, Hu X, Guo J, Wang X, Wei Q:
Methylenetetrahydrofolate reductase polymorphisms/haplotypes and risk of gastric cancer: a case-control analysis in China.
Oncol Rep
; 2005 Feb;13(2):355-60
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[Title]
Methylenetetrahydrofolate reductase polymorphisms/haplotypes and risk of
gastric
cancer: a case-control analysis in China.
Studies have suggested that low dietary folate intake is associated with an increased risk of
gastric
cancer.
C677T, A1298C and G1793A) and their haplotypes are associated with the risk of
gastric
cancer.
To test this hypothesis, we genotyped these polymorphisms in a population-based case-control study of 320 incident
gastric
adenocarcinoma
cases and 313 cancer-free controls in a Chinese population.
Consistent with our previous observations, the 677TT genotype was associated with a significantly increased risk for
gastric
cancer (adjusted OR =1.79, 95% CI =1.02-3.15) compared with the 677CC genotype; the association was more evident for
gastric
cardia adenocarcinoma
(adjusted OR =2.60, 95% CI =1.30-5.21).
When we used the haplotype analyses and assumed MTHFR 677T, 1298C and 1793A as risk alleles, individuals with 6 variant alleles had a significantly (4.64-fold) increased risk for
gastric
cardia adenocarcinoma
(OR =4.64, 95% CI =1.34-16.01) compared with those having 0-2 variants.
These findings suggest that the MTHFR common variants and their haplotypes may play a role in the etiology of
gastric
cancer, particularly
gastric
cardia adenocarcinoma
.
[MeSH-major]
Adenocarcinoma
/ genetics. Methylenetetrahydrofolate Reductase (NADPH2) / genetics. Polymorphism, Genetic.
Stomach
Neoplasms / genetics
[MeSH-minor]
Aged. Asian Continental Ancestry Group / genetics.
Cardia
. Case-Control Studies. China / epidemiology. Female. Gene Frequency. Genetic Predisposition to Disease. Haplotypes. Humans. Male. Middle Aged. Risk Factors
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(PMID = 15643524.001).
[ISSN]
1021-335X
[Journal-full-title]
Oncology reports
[ISO-abbreviation]
Oncol. Rep.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Greece
[Chemical-registry-number]
EC 1.5.1.20 / Methylenetetrahydrofolate Reductase (NADPH2)
55.
Gao Y, Hu N, Han X, Giffen C, Ding T, Goldstein A, Taylor P:
Family history of cancer and risk for esophageal and gastric cancer in Shanxi, China.
BMC Cancer
; 2009 Aug 05;9:269
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[Title]
Family history of cancer and risk for esophageal and
gastric
cancer in Shanxi, China.
METHODS: 600 esophageal squamous cell carcinoma (ESCC) cases, 598
gastric
cardia adenocarcinoma
cases, and 316
gastric
non-
cardia adenocarcinoma
cases, and 1514 age-, gender-, and neighborhood-matched controls were asked for FH in first degree relatives and non-blood relatives.
FH of
gastric
cardia
cancer was associated with an increased risk of all three cancers.
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[ISSN]
1471-2407
[Journal-full-title]
BMC cancer
[ISO-abbreviation]
BMC Cancer
[Language]
ENG
[Grant]
United States / Intramural NIH HHS / /
[Publication-type]
Journal Article; Research Support, N.I.H., Intramural
[Publication-country]
England
[Other-IDs]
NLM/ PMC2729777
56.
Shi H, Lu D, Shu Y, Shi W, Lu S, Wang K:
Expression of multidrug resistance-related proteins p-glycoprotein, glutathione-s-transferases, topoisomerase-II and lung resistance protein in primary gastric cardiac adenocarcinoma.
Hepatogastroenterology
; 2008 Sep-Oct;55(86-87):1530-6
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[Title]
Expression of multidrug resistance-related proteins p-glycoprotein, glutathione-s-transferases, topoisomerase-II and lung resistance protein in primary
gastric
cardiac
adenocarcinoma
.
However, the clinical significance of the expression of MDR-related proteins p-glycoprotein (PGP), glutathione-s-transferases (GST-pi), topoisomerase-II (Topo-II) and lung resistance protein (LRP) in primary
gastric
cardiac
adenocarcinoma
(PGCA) remains unclear.
[MeSH-major]
Adenocarcinoma
/ chemistry.
Cardia
/ chemistry. DNA Topoisomerases, Type II / analysis. Glutathione S-Transferase pi / analysis. P-Glycoprotein / analysis.
Stomach
Neoplasms / chemistry. Vault Ribonucleoprotein Particles / analysis
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(PMID = 19102336.001).
[ISSN]
0172-6390
[Journal-full-title]
Hepato-gastroenterology
[ISO-abbreviation]
Hepatogastroenterology
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Greece
[Chemical-registry-number]
0 / P-Glycoprotein; 0 / Vault Ribonucleoprotein Particles; 0 / major vault protein; EC 2.5.1.18 / Glutathione S-Transferase pi; EC 5.99.1.3 / DNA Topoisomerases, Type II
57.
van Blankenstein M, Looman CW, Siersema PD, Kuipers EJ, Coebergh JW:
Trends in the incidence of adenocarcinoma of the oesophagus and cardia in the Netherlands 1989-2003.
Br J Cancer
; 2007 Jun 4;96(11):1767-71
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[Title]
Trends in the incidence
of adenocarcinoma
of the oesophagus and
cardia
in the Netherlands 1989-2003.
Over the 15-year period 1989-2003, the incidence of oesophagus-
cardia adenocarcinoma
in the Netherlands rose annually by 2.6% for males and 1.2% for females.
This was the net outcome of annual increases in the incidence
of adenocarcinoma
of the oesophagus (ACO) of 7.2% for males and 3.5% for females and annual declines in the incidence
of adenocarcinoma
of
the gastric
cardia
(AGC) of more than 1% for both genders.
[MeSH-major]
Adenocarcinoma
/ epidemiology.
Cardia
. Esophageal Neoplasms / epidemiology.
Stomach
Neoplasms / epidemiology
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Scand J Gastroenterol. 1999 Apr;34(4):353-60
[
10365894.001
]
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Int J Epidemiol. 1999 Jun;28(3):391-5
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]
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14954081.001
]
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Gut. 2005 Jan;54(1):1-3
[
15591495.001
]
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[
15784017.001
]
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Gut. 2005 Aug;54(8):1062-6
[
15857935.001
]
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[
16061918.001
]
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[
16181362.001
]
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]
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Gut. 2002 Mar;50(3):368-72
[
11839716.001
]
(PMID = 17505507.001).
[ISSN]
0007-0920
[Journal-full-title]
British journal of cancer
[ISO-abbreviation]
Br. J. Cancer
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
England
[Other-IDs]
NLM/ PMC2359916
58.
Trivers KF, De Roos AJ, Gammon MD, Vaughan TL, Risch HA, Olshan AF, Schoenberg JB, Mayne ST, Dubrow R, Stanford JL, Abrahamson P, Rotterdam H, West AB, Fraumeni JF, Chow WH:
Demographic and lifestyle predictors of survival in patients with esophageal or gastric cancers.
Clin Gastroenterol Hepatol
; 2005 Mar;3(3):225-30
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[Title]
Demographic and lifestyle predictors of survival in patients with esophageal or
gastric
cancers.
BACKGROUND AND AIMS: Risk factors for subtypes of esophageal and
gastric
cancer recently have been identified, but their effect on survival is unknown.
Cox regression models were used to estimate adjusted hazard ratios (HRs) and 95% confidence intervals (CIs) for esophageal and
gastric
cancer in relation to prediagnostic factors.
RESULTS: Relative to distant stage, esophageal
adenocarcinoma
(EA) patients with localized disease had a decreased risk for death (HR, .22; 95% CI, .15-.31), followed by those with regional spread (HR, .32; 95% CI, .23-.45).
Except for other (non-
cardia
)
gastric
adenocarcinomas (OGA), higher household income (> or =15,000 US dollars/y vs. <15,000 US dollars/y) was associated with a 33%-38% decrease in risk for death.
CONCLUSIONS: Predictors of lengthened esophageal and
gastric
cancer survival included higher income (except in OGA), overweight (among EA and OGA patients), and female sex (among ES and OGA patients).
[MeSH-major]
Adenocarcinoma
/ mortality. Carcinoma, Squamous Cell / mortality. Esophageal Neoplasms / mortality.
Stomach
Neoplasms / mortality
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(PMID = 15765441.001).
[ISSN]
1542-3565
[Journal-full-title]
Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association
[ISO-abbreviation]
Clin. Gastroenterol. Hepatol.
[Language]
eng
[Grant]
United States / NCI NIH HHS / CN / N01-CN05230; United States / NCI NIH HHS / CP / N02-CP40501; United States / NIEHS NIH HHS / ES / P30ES10126; United States / NCI NIH HHS / CA / T32-CA09330; United States / NCI NIH HHS / CA / U01-CA57923; United States / NCI NIH HHS / CA / U01-CA57949; United States / NCI NIH HHS / CA / U01-CA57983
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, P.H.S.
[Publication-country]
United States
59.
Wang DC, Wang LD, Zheng S, Fan ZM, Li JL, Feng CW, Zhang YR, Liu B, Gao SS, He X:
[The application of surface-enhanced laser desorption/ionization-time of flight-mass spectrometry in diagnosing dysplasia and chronic atrophic gastric-carditis in population with high risk of gastric-cardia adenocarcinoma].
Zhonghua Nei Ke Za Zhi
; 2005 Aug;44(8):573-6
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[Title]
[The application of surface-enhanced laser desorption/ionization-time of flight-mass spectrometry in diagnosing dysplasia and chronic atrophic
gastric
-carditis in population with high risk of
gastric
-
cardia adenocarcinoma
].
OBJECTIVES: To evaluate the serum biomarkers for diagnosis of
gastric
cardia
dysplasia (DYS) and chronic atrophic
gastric
-carditis (CAG) and to provide a novel screening method for high risk population of
gastric
-
cardia adenocarcinoma
(GCA).
A set of spectra derived from analysis of serum from 143 symptom-free subjects at high-risk area for GCA, including 63 cases with histologically normal
gastric
cardia
epithelia, 57 of CAG and 23 of DYS, were analyzed by bioinformatics like decision tree classification algorithm.
CONCLUSIONS:
The gastric
cardia
lesions of DYS and CAG could be identified by SELDI-TOF-MS technique specifically in symptom-free subjects at high incidence area for GCA.
[MeSH-major]
Biomarkers, Tumor / blood.
Cardia
. Gastritis, Atrophic / diagnosis. Spectrometry, Mass, Matrix-Assisted Laser Desorption-Ionization / methods.
Stomach
Neoplasms / diagnosis
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(PMID = 16194406.001).
[ISSN]
0578-1426
[Journal-full-title]
Zhonghua nei ke za zhi
[ISO-abbreviation]
Zhonghua Nei Ke Za Zhi
[Language]
chi
[Publication-type]
English Abstract; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
China
[Chemical-registry-number]
0 / Biomarkers, Tumor
60.
Ychou M, Gory-Delabaere G, Blanc P, Bosquet L, Duffour J, Giovannini M, Guillemin F, Lemanski C, Marchal F, Masson B, Merrouche Y, Monges G, Adenis A, Bosset JF, Bouché O, Conroy T, Pezet D, Triboulet JP, Fédération nationale des centres de lutte contre le cancer (FNCLCC), Fédération hospitalière de France (FHF), Fédération nationale de Cancérologie des CHRU (FNCHRU), Fédération française de cancérologie des CHG (FFCCHG), Centres régionaux de lutte contre le cancer (CRLCC):
[Clinical Practice Guidelines 2004. Standards, Options and Recommendations for the management of patient with adenocarcinoma of the stomach: radiotherapy (therapeutic evaluation)].
Bull Cancer
; 2005 Apr;92(4):381-409
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[Title]
[Clinical Practice Guidelines 2004. Standards, Options and Recommendations for the management of patient with
adenocarcinoma of
the
stomach
: radiotherapy (therapeutic evaluation)].
[Transliterated title]
Recommandations pour la pratique clinique. Standards, Options et Recommandations 2004 pour la prise en charge des patients atteints d'adénocarcinomes de l'estomac, cancers du
cardia
, autres types histologiques exclus (évaluation des thérapeutiques).
OBJECTIVES: To elaborate clinical practice guidelines for patients with
stomach adenocarcinoma
.
[MeSH-major]
Adenocarcinoma
/ therapy.
Stomach
Neoplasms / therapy
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(PMID = 15888395.001).
[ISSN]
1769-6917
[Journal-full-title]
Bulletin du cancer
[ISO-abbreviation]
Bull Cancer
[Language]
fre
[Publication-type]
English Abstract; Guideline; Journal Article; Practice Guideline; Research Support, Non-U.S. Gov't; Review
[Publication-country]
France
[Chemical-registry-number]
0 / Antineoplastic Agents
[Number-of-references]
192
61.
Kountouras J, Zavos C, Chatzopoulos D, Katsinelos P:
New aspects of Helicobacter pylori infection involvement in gastric oncogenesis.
J Surg Res
; 2008 May 1;146(1):149-58
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[Title]
New aspects of Helicobacter pylori infection involvement in
gastric
oncogenesis.
Gastric
adenocarcinoma
not located in the
cardia
still remains second only to lung cancer as the leading cause of cancer-related mortality worldwide, whereas
adenocarcinoma of
the
cardia
and gastroesophageal junction has been rapidly rising over the past two decades.
Gastric
malignancy can be subdivided into diffuse and intestinal pathologic entities that have different epidemiological and prognostic features.
Oncogene overexpression, tumor suppressor loss, and defective DNA mismatch repair is associated with
gastric
cancer.
The latter leads to
gastric
carcinogenesis through changes related to E-cadherin-catenin complex, which plays a critical role in normal tissue architecture maintenance.
Putative trophic factors have also been involved in
gastric
oncogenesis.
This review focuses mainly on Helicobacter pylori infection involved in
gastric
carcinogenesis through various mechanisms, including repopulation of the
stomach
with bone marrow-derived stem cells that may facilitate
gastric
cancer progression, thereby necessitating eradication of this bacterium.
[MeSH-major]
Adenocarcinoma
/ microbiology. Helicobacter Infections / complications.
Stomach
Neoplasms / microbiology
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.
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.
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(PMID = 17720195.001).
[ISSN]
0022-4804
[Journal-full-title]
The Journal of surgical research
[ISO-abbreviation]
J. Surg. Res.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Number-of-references]
53
62.
Schuhmacher C, Gretschel S, Lordick F, Reichardt P, Hohenberger W, Eisenberger CF, Haag C, Mauer ME, Hasan B, Welch J, Ott K, Hoelscher A, Schneider PM, Bechstein W, Wilke H, Lutz MP, Nordlinger B, Van Cutsem E, Siewert JR, Schlag PM:
Neoadjuvant chemotherapy compared with surgery alone for locally advanced cancer of the stomach and cardia: European Organisation for Research and Treatment of Cancer randomized trial 40954.
J Clin Oncol
; 2010 Dec 10;28(35):5210-8
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[Title]
Neoadjuvant chemotherapy compared with surgery alone for locally advanced cancer of the
stomach
and
cardia
: European Organisation for Research and Treatment of Cancer randomized trial 40954.
PURPOSE: Patients with locally advanced
gastric
cancer benefit from combined pre- and postoperative chemotherapy, although fewer than 50% could receive postoperative chemotherapy.
PATIENTS AND METHODS: Patients with locally advanced
adenocarcinoma of
the
stomach
or esophagogastric junction (AEG II and III) were randomly assigned to preoperative chemotherapy followed by surgery or to surgery alone.
RESULTS: This trial was stopped for poor accrual after 144 patients were randomly assigned (72:72); 52.8% patients had tumors located in the proximal third of the
stomach
, including AEG type II and III.
Possible explanations are low statistical power, a high rate of proximal
gastric
cancer including AEG and/or a better outcome than expected after radical surgery alone due to the high quality of surgery with resections of regional lymph nodes outside the perigastic area (celiac trunc, hepatic ligament, lymph node at a. lienalis; D2).
[MeSH-major]
Adenocarcinoma
/ drug therapy.
Adenocarcinoma
/ surgery. Neoadjuvant Therapy / methods.
Stomach
Neoplasms / drug therapy.
Stomach
Neoplasms / surgery
[MeSH-minor]
Adult. Aged. Antineoplastic Agents / therapeutic use.
Cardia
/ pathology.
Cardia
/ surgery. Combined Modality Therapy. Digestive System Surgical Procedures. Disease-Free Survival. Esophagogastric Junction / drug effects. Esophagogastric Junction / pathology. Esophagogastric Junction / surgery. Female. Humans. Kaplan-Meier Estimate. Male. Middle Aged. Neoplasm Staging
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[Cites]
CA Cancer J Clin. 2000 Jan-Feb;50(1):7-33
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10735013.001
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JAMA. 2010 May 5;303(17):1729-37
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Ann Surg Oncol. 2000 Mar;7(2):139-44
[
10761793.001
]
(PMID = 21060024.001).
[ISSN]
1527-7755
[Journal-full-title]
Journal of clinical oncology : official journal of the American Society of Clinical Oncology
[ISO-abbreviation]
J. Clin. Oncol.
[Language]
eng
[Databank-accession-numbers]
ClinicalTrials.gov/ NCT00004099
[Grant]
United States / NCI NIH HHS / CA / U10 CA011488; United States / NCI NIH HHS / CA / 5U10 CA11488-38; United States / NCI NIH HHS / CA / 5U10-CA11488-29
[Publication-type]
Clinical Trial, Phase III; Journal Article; Multicenter Study; Randomized Controlled Trial; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Antineoplastic Agents
[Other-IDs]
NLM/ PMC3020693
63.
Rubenstein JH, Davis J, Marrero JA, Inadomi JM:
Relationship between diabetes mellitus and adenocarcinoma of the oesophagus and gastric cardia.
Aliment Pharmacol Ther
; 2005 Aug 1;22(3):267-71
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[Title]
Relationship between diabetes mellitus and
adenocarcinoma of
the oesophagus and
gastric
cardia
.
BACKGROUND: Obesity is a risk factor for adenocarcinomas of the oesophagus and
gastric
cardia
.
AIM: To estimate the risk of diabetes mellitus on the development
of adenocarcinoma
of distal oesophagus and
gastric
cardia
beyond that of gastro-oesophageal reflux disease.
CONCLUSIONS: Within the limitations of this case-control study, there is no evidence of an association between diabetes and
adenocarcinoma of
the oesophagus or
gastric
cardia
among US veterans with gastro-oesophageal reflux disease.
[MeSH-major]
Adenocarcinoma
/ etiology.
Cardia
. Diabetes Mellitus. Esophageal Neoplasms / etiology.
Stomach
Neoplasms / etiology
Genetic Alliance.
consumer health - Diabetes
.
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.
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(PMID = 16091065.001).
[ISSN]
0269-2813
[Journal-full-title]
Alimentary pharmacology & therapeutics
[ISO-abbreviation]
Aliment. Pharmacol. Ther.
[Language]
eng
[Grant]
United States / NCI NIH HHS / CA / CA864000; United States / NIDDK NIH HHS / DK / DK064909
[Publication-type]
Journal Article; Multicenter Study; Research Support, N.I.H., Extramural; Research Support, U.S. Gov't, P.H.S.
[Publication-country]
England
64.
Lundell LR:
Etiology and risk factors for esophageal carcinoma.
Dig Dis
; 2010;28(4-5):641-4
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Trends toward increasing incidence rates were observed for esophageal and
gastric
cardia adenocarcinoma
in Western countries and were associated with trends toward stabilizing or declining incidence rates for esophageal squamous cell carcinoma, suggesting that these tumors might be associated with distinct risk factors.
Overweight and obesity have been consistently related to esophageal
adenocarcinoma
, but not to squamous cell carcinoma.
The influence of obesity on esophageal
adenocarcinoma
and
gastric
cardia adenocarcinoma
may be related to higher incidence of gastroesophageal reflux in obese persons since the risk of gastroesophageal reflux is strongly related to the risk for Barrett's esophagus.
Tobacco smoking is a strong risk factor for esophageal squamous cell carcinoma, but is only a weak risk factor for esophageal
adenocarcinoma
.
Alcohol consumption is a strong risk factor for esophageal squamous cell carcinoma, but is not consistently related to esophageal
adenocarcinoma
.
It has been suggested that infection with H. pylori is protective to
adenocarcinoma
, but might be a risk factor for squamous cell carcinoma, although the role of H. pylori in the etiology of these cancers remains somewhat unclear.
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[Copyright]
Copyright © 2010 S. Karger AG, Basel.
(PMID = 21088416.001).
[ISSN]
1421-9875
[Journal-full-title]
Digestive diseases (Basel, Switzerland)
[ISO-abbreviation]
Dig Dis
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Switzerland
65.
Lindblad M, García Rodríguez LA, Chandanos E, Lagergren J:
Hormone replacement therapy and risks of oesophageal and gastric adenocarcinomas.
Br J Cancer
; 2006 Jan 16;94(1):136-41
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[Title]
Hormone replacement therapy and risks of oesophageal and
gastric
adenocarcinomas.
Oesophageal and
gastric
adenocarcinoma
share an unexplained male predominance, which would be explained by the hypothesis that oestrogens are protective in this respect.
We carried out a nested case-control study of hormone replacement therapy (HRT) among 299 women with oesophageal cancer, 313 with
gastric
cancer, and 3191 randomly selected control women, frequency matched by age and calendar year in the General Practitioners Research Database in the United Kingdom.
Among 1 619 563 person-years of follow-up, more than 50% reduced risk of
gastric
adenocarcinoma
was found among users of HRT compared to nonusers (odds ratio (OR), 0.48, 95% confidence interval (CI) 0.29-0.79).
This inverse association appeared to be stronger for
gastric
noncardia (OR 0.34, 95% CI 0.14-0.78) and weaker for
gastric
cardia
tumours (OR 0.68, 95% CI 0.23-2.01).
There was no association between HRT and oesophageal
adenocarcinoma
(OR 1.17, 95% CI 0.41-3.32).
[MeSH-major]
Adenocarcinoma
/ etiology. Esophageal Neoplasms / etiology. Hormone Replacement Therapy.
Stomach
Neoplasms / etiology
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Int J Cancer. 2002 Feb 20;97(6):833-8
[
11857364.001
]
[Cites]
Lancet Oncol. 2001 Sep;2(9):533-43
[
11905707.001
]
[Cites]
J Cancer Res Clin Oncol. 2002 Jun;128(6):319-24
[
12073050.001
]
[Cites]
Anticancer Res. 2002 May-Jun;22(3):1459-61
[
12168823.001
]
[Cites]
JAMA. 2002 Aug 21;288(7):872-81
[
12186605.001
]
[Cites]
Gastric Cancer. 2002;5(4):213-9
[
12491079.001
]
[Cites]
J Clin Epidemiol. 2003 Jan;56(1):1-9
[
12589864.001
]
[Cites]
Int J Cancer. 2003 Jun 20;105(3):408-12
[
12704678.001
]
[Cites]
Pharmacotherapy. 2003 May;23(5):686-9
[
12741446.001
]
(PMID = 16404367.001).
[ISSN]
0007-0920
[Journal-full-title]
British journal of cancer
[ISO-abbreviation]
Br. J. Cancer
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Other-IDs]
NLM/ PMC2361087
66.
Fortuny J, Johnson CC, Bohlke K, Chow WH, Hart G, Kucera G, Mujumdar U, Ownby D, Wells K, Yood MU, Engel LS:
Use of anti-inflammatory drugs and lower esophageal sphincter-relaxing drugs and risk of esophageal and gastric cancers.
Clin Gastroenterol Hepatol
; 2007 Oct;5(10):1154-1159.e3
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[Title]
Use of anti-inflammatory drugs and lower esophageal sphincter-relaxing drugs and risk of esophageal and
gastric
cancers.
BACKGROUND & AIMS: The incidence of esophageal and
gastric
cardia adenocarcinoma
has increased in Western countries in recent decades for largely unknown reasons.
We investigated whether use of LES-relaxing drugs was related to an increased risk of esophageal and
gastric
cardia adenocarcinoma
, and whether use of NSAIDs was related to a reduced risk of esophageal and
gastric
cancers.
Cases were incident esophageal adenocarcinomas (n = 163) and squamous cell carcinomas (n = 114) and
gastric
cardia
(n = 176) and non-
cardia
adenocarcinomas (n = 320), diagnosed between 1980-2002 in one health system and between 1993-2002 in the other.
RESULTS: Prescription of corticosteroids was associated with a decreased risk of esophageal
adenocarcinoma
(odds ratio [OR], 0.6; 95% confidence interval [CI], 0.4-0.9), esophageal squamous cell carcinoma (OR, 0.4; 95% CI, 0.2-0.6), and
gastric
non-
cardia
carcinoma (OR, 0.4, 95% CI, 0.3-0.6).
As a group, LES-relaxing drugs showed little evidence of association with increased risk of any esophageal or
gastric
cancer.
CONCLUSIONS: Corticosteroid and aspirin use were associated with significantly decreased risks of esophageal and
gastric
cancer.
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[Cites]
Cancer Epidemiol Biomarkers Prev. 2005 Feb;14(2):444-50
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[
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Cancer Epidemiol Biomarkers Prev. 1998 Feb;7(2):97-102
[
9488582.001
]
(PMID = 17644046.001).
[ISSN]
1542-7714
[Journal-full-title]
Clinical gastroenterology and hepatology : the official clinical practice journal of the American Gastroenterological Association
[ISO-abbreviation]
Clin. Gastroenterol. Hepatol.
[Language]
ENG
[Grant]
United States / NCI NIH HHS / CA / R01 CA140754; United States / NCI NIH HHS / CA / U19 CA079689; United States / Intramural NIH HHS / / Z99 CA999999; United States / NCI NIH HHS / CA / U19 CA79689
[Publication-type]
Journal Article; Multicenter Study; Research Support, N.I.H., Extramural; Research Support, N.I.H., Intramural
[Publication-country]
United States
[Chemical-registry-number]
0 / Anti-Inflammatory Agents, Non-Steroidal; 0 / Antidepressive Agents; 0 / Histamine H1 Antagonists
[Other-IDs]
NLM/ NIHMS32453; NLM/ PMC2140196
67.
Xu DK, Zhao P, Wang CF, Shao YF, Lin HW, Tian YT:
[Clinicopathological characteristics and prognosis of remnant stomach cancer--report of 45 cases].
Zhonghua Zhong Liu Za Zhi
; 2006 Nov;28(11):852-4
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[Title]
[Clinicopathological characteristics and prognosis of remnant
stomach
cancer--report of 45 cases].
OBJECTIVE: To investigate the clinicopathological characteristics and prognostic factors of remnant
stomach
cancer.
METHODS: The clinicopathological and prognosis data of 45 patients with remnant
stomach
cancer were retrospectively analyzed.
RESULTS: The remnant
stomach
cancer are likely to develop in males with a ratio of male to female: 44:1.
The interval from the initial operation to the diagnosis of remnant
stomach
cancer was 5 to 42 years with an average of 23 years.
Of these 45 patients, 28 had lesion at anastomotic site, 9 in
the gastric
cardia
and 8 in other locations; 19 had radical resection, 16 palliative resection and 10 exploration alone except one who had an anastomosis of remnant
stomach
with the jejunum.
The histology types included: 1 un-differentiated
adenocarcinoma
, 36 poorly-differentiated
adenocarcinoma
, 7 moderately-differentiated
adenocarcinoma
and 1 well-differentiated
adenocarcinoma
.
CONCLUSION: Remnant
stomach
cancer prevalently occurs in the male usually 10 years after Birroth II gastrectomy.
Poorly-differentiated
adenocarcinoma
is found to be the prevalent histological type of advanced remnant
stomach
cancer.
The prognosis of remnant
stomach
cancer is correlated with pTNM stage and whether having been treated with complete resection or not.
Patients with early remnant
stomach
cancer may survive for a long time if radical resection can be done.
[MeSH-major]
Adenocarcinoma
/ pathology. Gastrectomy / methods.
Gastric
Stump / pathology.
Stomach
Neoplasms / pathology
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(PMID = 17416009.001).
[ISSN]
0253-3766
[Journal-full-title]
Zhonghua zhong liu za zhi [Chinese journal of oncology]
[ISO-abbreviation]
Zhonghua Zhong Liu Za Zhi
[Language]
chi
[Publication-type]
English Abstract; Journal Article
[Publication-country]
China
68.
Bhurgri Y, Pervez S, Kayani N, Haider S, Ahmed R, Usman A, Bashir I, Bhurgri A, Hasan SH, Zaidi SM:
Rising incidence of gastric malignancies in Karachi, 1995- 2002.
Asian Pac J Cancer Prev
; 2009 Jan-Mar;10(1):41-4
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[Title]
Rising incidence of
gastric
malignancies in Karachi, 1995- 2002.
INTRODUCTION: South Asia is an enigma for
gastric
cancer, a low risk region with a contradictory high prevalence for Helicobacter pylori.
PATIENTS AND METHODS: To examine the demographics, pathology and trends of
gastric
cancer in Pakistan, epidemiological data of 335
gastric
malignancies, registered at Karachi Cancer Registry (KCR) for Karachi South (KS), during 1st January 1995 to 31st December 2002 were reviewed.
RESULTS: Ninety six cases of
gastric
cancers were registered in the 1995-7 period, 61 in males and 35 in females.
In the 1998-02 period 239 cases of
gastric
cancer were registered, 156 cases in males and 83 in females.
The majority of the cases presented as poorly or moderately differentiated distal (non-
cardia
) cancers with a regional spread.
CONCLUSION:
Gastric
cancers in Karachi fall into the prototype of a low risk developing country pattern.
Larger pathology-based studies are required to comment on the precise morphological sub-types of
gastric
adenocarcinoma
.
Etiological studies focused on different strains of H. pylori are required to address
the gastric
cancer enigma, whilst examining possible protective environmental or genetic factors.
[MeSH-major]
Stomach
Neoplasms / epidemiology
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(PMID = 19469622.001).
[ISSN]
2476-762X
[Journal-full-title]
Asian Pacific journal of cancer prevention : APJCP
[ISO-abbreviation]
Asian Pac. J. Cancer Prev.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Thailand
69.
Hold GL, Rabkin CS, Chow WH, Smith MG, Gammon MD, Risch HA, Vaughan TL, McColl KE, Lissowska J, Zatonski W, Schoenberg JB, Blot WJ, Mowat NA, Fraumeni JF Jr, El-Omar EM:
A functional polymorphism of toll-like receptor 4 gene increases risk of gastric carcinoma and its precursors.
Gastroenterology
; 2007 Mar;132(3):905-12
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[Title]
A functional polymorphism of toll-like receptor 4 gene increases risk of
gastric
carcinoma and its precursors.
The TLR4+896A>G polymorphism linked with impaired reactivity to bacterial lipopolysaccharide may play a role in
gastric
carcinogenesis.
METHODS: We assessed associations with premalignant
gastric
changes in 149 relatives of
gastric
cancer patients, including 45 with hypochlorhydria and
gastric
atrophy.
We also genotyped 2 independent Caucasian population-based case-control studies of upper gastrointestinal tract cancer, initially in 312 noncardia
gastric
carcinoma cases and 419 controls and then in 184 noncardia
gastric
carcinomas, 123
cardia
carcinomas, 159 esophageal cancers, and 211 frequency-matched controls.
RESULTS: TLR4+896G carriers had an 11-fold (95% confidence interval [CI], 2.5-48) increased odds ratio (OR) for hypochlorhydria; the polymorphism was unassociated with
gastric
acid output in the absence of H pylori infection.
Carriers also had significantly more severe
gastric
atrophy and inflammation.
Seventeen percent of
gastric
carcinoma patients in the initial study and 15% of the noncardia
gastric
carcinoma patients in the replication study had 1 or 2 TLR4 variant alleles vs 8% of both control populations (combined OR = 2.3; 95% CI = 1.6-3.4).
In contrast, prevalence of TLR4+896G was not significantly increased in esophageal squamous cell (2%, OR = 0.2) or
adenocarcinoma
(9%, OR = 1.4) or
gastric
cardia
carcinoma (11%, OR = 1.4).
CONCLUSIONS: Our data suggest that the TLR4+896A>G polymorphism is a risk factor for noncardia
gastric
carcinoma and its precursors.
[MeSH-major]
Carcinoma / genetics. Helicobacter Infections / microbiology. Helicobacter pylori. Polymorphism, Genetic. Precancerous Conditions / genetics.
Stomach
Neoplasms / genetics. Toll-Like Receptor 4 / genetics
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(PMID = 17324405.001).
[ISSN]
0016-5085
[Journal-full-title]
Gastroenterology
[ISO-abbreviation]
Gastroenterology
[Language]
eng
[Grant]
United Kingdom / Chief Scientist Office / / CZB/4/485; United States / Intramural NIH HHS / /
[Publication-type]
Journal Article; Multicenter Study; Research Support, N.I.H., Intramural; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / TLR4 protein, human; 0 / Toll-Like Receptor 4
70.
Schuhmacher C, Novotny A, Ott K, Feith M, Siewert JR:
[Lymphadenectomy with tumors of the upper gastrointestinal tract].
Chirurg
; 2007 Mar;78(3):203-6, 208-12, 214-6
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Locally advanced Barrett carcinoma is also an indication for classic two-field lymphadenectomy together with abdominothoracic oesophagectomy and creation of a
stomach
tube with intrathoracic anastomosis.
Adenocarcinoma of
the
cardia
and subcardial
gastric
cancer including the
cardia
both require lymphadenectomy analogous to that performed in
gastric
cancer, with special attention paid to the retroperitoneal lymphatic drainage towards the left kidney pedicle.
For therapy of
gastric
cancer, a systematic D2 lymphadenectomy should always be performed.
[MeSH-major]
Adenocarcinoma
/ surgery. Barrett Esophagus / surgery. Carcinoma, Squamous Cell / surgery. Esophageal Neoplasms / surgery. Lymph Node Excision / methods. Precancerous Conditions / surgery.
Stomach
Neoplasms / surgery
[MeSH-minor]
Cardia
/ pathology.
Cardia
/ surgery. Esophagectomy / methods. Humans. Lymph Nodes / pathology. Lymphatic Metastasis / pathology. Neoplasm Staging. Prognosis. Thoracic Cavity / surgery
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[ISSN]
0009-4722
[Journal-full-title]
Der Chirurg; Zeitschrift fur alle Gebiete der operativen Medizen
[ISO-abbreviation]
Chirurg
[Language]
ger
[Publication-type]
English Abstract; Journal Article
[Publication-country]
Germany
71.
Lagergren J, Ye W, Lagergren P, Lu Y:
The risk of esophageal adenocarcinoma after antireflux surgery.
Gastroenterology
; 2010 Apr;138(4):1297-301
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[Title]
The risk of esophageal
adenocarcinoma
after antireflux surgery.
BACKGROUND & AIMS: The question of a possible preventive effect of antireflux surgery on the development of esophageal or
cardia adenocarcinoma
remains unsettled.
We aimed to clarify whether antireflux surgery prevents later development of esophageal
adenocarcinoma
.
Overall risk of esophageal
adenocarcinoma
(n = 39) was increased 12-fold (SIR, 12.3; 95% confidence interval [CI], 8.7-16.8).
For the corresponding overall risk
of cardia adenocarcinoma
(n = 21) the SIR was 4.4 (95% CI, 2.7-6.7), without any major decrease in risk with time (P = .20); the SIR was 3.1 (95% CI, 0.6-9.1) after at least 15 years of follow-up evaluation.
No association between antireflux surgery and
gastric
adenocarcinoma
or esophageal squamous cell carcinoma was identified.
CONCLUSIONS: Antireflux surgery cannot be considered to prevent the development of esophageal or
cardia adenocarcinoma
among persons with reflux.
[MeSH-major]
Adenocarcinoma
/ prevention & control. Esophageal Neoplasms / prevention & control. Gastroesophageal Reflux / surgery
[MeSH-minor]
Cardia
. Cohort Studies. Female. Humans. Male. Middle Aged. Risk.
Stomach
Neoplasms / prevention & control
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[Copyright]
2010 AGA Institute. Published by Elsevier Inc. All rights reserved.
[CommentIn]
Chirurg. 2011 Jan;82(1):78-9
[
21085917.001
]
(PMID = 20080091.001).
[ISSN]
1528-0012
[Journal-full-title]
Gastroenterology
[ISO-abbreviation]
Gastroenterology
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
72.
Lindblad M, Ye W, Lindgren A, Lagergren J:
Disparities in the classification of esophageal and cardia adenocarcinomas and their influence on reported incidence rates.
Ann Surg
; 2006 Apr;243(4):479-85
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[Title]
Disparities in the classification of esophageal and
cardia
adenocarcinomas and their influence on reported incidence rates.
OBJECTIVE: To evaluate the diagnostic accuracy of esophageal and
cardia adenocarcinoma
in the Swedish Cancer Register.
SUMMARY BACKGROUND DATA: Based on cancer registers, a rising incidence of esophageal and
cardia adenocarcinoma
has been reported in several populations, but possible influence of differences in tumor classification has not been evaluated.
METHODS: In a nationwide study in 1995 through 1997, all Swedish patients, born in Sweden and younger than 80 years with esophageal or
cardia adenocarcinoma
and half of all patients with esophageal squamous cell carcinoma, were prospectively, uniformly, and thoroughly classified.
RESULTS: The overall completeness of the Cancer Register was high (98.3%), whereas the site-specific completeness of the Register was 63% for esophageal
adenocarcinoma
, 74% for
cardia adenocarcinoma
, and 91% for esophageal squamous cell carcinoma.
The incidence of esophageal adenocarcinomas was 16% higher in the study classification compared with that of the Register during the study period, whereas the incidence
of cardia adenocarcinoma
was 2% lower in the study classification.
CONCLUSIONS: There is a diagnostic mismatch between esophageal and
cardia adenocarcinoma
in the clinical setting and, therefore, also in Cancer Registers.
The increasing incidence rate of esophageal
adenocarcinoma
in Sweden is unlikely to be explained by such differences in tumor classification, however.
[MeSH-major]
Adenocarcinoma
/ epidemiology. Esophageal Neoplasms / classification. Esophageal Neoplasms / epidemiology.
Stomach
Neoplasms / classification.
Stomach
Neoplasms / epidemiology
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11775726.001
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Cancer Causes Control. 2000 Mar;11(3):231-8
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[
11821356.001
]
(PMID = 16552198.001).
[ISSN]
0003-4932
[Journal-full-title]
Annals of surgery
[ISO-abbreviation]
Ann. Surg.
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Other-IDs]
NLM/ PMC1448962
73.
Schurr PG, Yekebas EF, Kaifi JT, Lasch S, Strate T, Kutup A, Cataldegirmen G, Bubenheim M, Pantel K, Izbicki JR:
Lymphatic spread and microinvolvement in adenocarcinoma of the esophago-gastric junction.
J Surg Oncol
; 2006 Sep 15;94(4):307-15
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[Title]
Lymphatic spread and microinvolvement in
adenocarcinoma of
the esophago-
gastric
junction.
BACKGROUND:
Adenocarcinoma of
the esophago-
gastric
junction (EGJ) potentially spreads to abdominal and mediastinal lymph nodes.
[MeSH-major]
Adenocarcinoma
/ pathology.
Adenocarcinoma
/ surgery. Esophagogastric Junction. Lymph Nodes / pathology
[MeSH-minor]
Aged. Aged, 80 and over. Barrett Esophagus / pathology.
Cardia
. Esophageal Neoplasms / mortality. Esophageal Neoplasms / pathology. Esophageal Neoplasms / surgery. Esophagectomy. Female. Gastrectomy. Humans. Lymph Node Excision. Lymphatic Metastasis. Male. Mediastinum. Middle Aged. Risk.
Stomach
Neoplasms / mortality.
Stomach
Neoplasms / pathology.
Stomach
Neoplasms / surgery. Survival Rate
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[Copyright]
(c) 2006 Wiley-Liss, Inc.
(PMID = 16917878.001).
[ISSN]
0022-4790
[Journal-full-title]
Journal of surgical oncology
[ISO-abbreviation]
J Surg Oncol
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
74.
Deveci MS, Deveci G:
Prognostic value of p53 protein and MK-1 (a tumor-associated antigen) expression in gastric carcinoma.
Gastric Cancer
; 2007;10(2):112-6
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[Title]
Prognostic value of p53 protein and MK-1 (a tumor-associated antigen) expression in
gastric
carcinoma.
BACKGROUND: MK-1, the target molecule of FU-MK-1, is encoded by the GA733-2 gene, which is currently being used as a target in clinical trials for
gastric
, intestinal and biliary cancer treatment with monoclonal antibodies.
METHODS: The expression of p53 protein and MK-1 antigen was investigated in specimens from 42 patients with
gastric
carcinoma.
MK-1 expression was more frequent in
cardia
tumors (71%), in large (>3 cm) tumors (60%-64%), and in specimens from patients with more than five metastatic lymph nodes (69%).
Of these 20 patients, 15 (52%) had tubular
adenocarcinoma
(TA) and 5 (38%) had signet ring cell carcinoma. p53 expression was more frequent in the tumors of male patients (55% vs 27%); in poorly differentiated TAs (60% vs 47% in well-to-moderately differentiated TAs); in smaller tumors (< or = 3 cm, 72% vs 43%-50% in larger tumors); in patients with a prominent inflammatory response (61% vs 21%; P < 0.02); and in patients with lymphatic vessel invasion (77% vs 34%; P < 0.02).
Most patients with p53- and MK-1-positive
gastric
carcinomas and those more than five metastatic lymph nodes had a poor prognosis.
CONCLUSION: The study found that the expression of both p53 and MK-1 was frequent in aggressive
gastric
carcinomas; however, extensive lymph node involvement (more than five nodes) was the only significant factor related to overall survival.
[MeSH-major]
Adenocarcinoma
/ metabolism. Antigens, Neoplasm / metabolism. Biomarkers, Tumor / metabolism. Cell Adhesion Molecules / metabolism.
Stomach
Neoplasms / metabolism. Tumor Suppressor Protein p53 / metabolism
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[Cites]
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(PMID = 17577621.001).
[ISSN]
1436-3291
[Journal-full-title]
Gastric cancer : official journal of the International Gastric Cancer Association and the Japanese Gastric Cancer Association
[ISO-abbreviation]
Gastric Cancer
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Japan
[Chemical-registry-number]
0 / Antigens, Neoplasm; 0 / Biomarkers, Tumor; 0 / Cell Adhesion Molecules; 0 / EPCAM protein, human; 0 / Epithelial Cell Adhesion Molecule; 0 / Tumor Suppressor Protein p53
75.
Palli D, Masala G, Del Giudice G, Plebani M, Basso D, Berti D, Numans ME, Ceroti M, Peeters PH, Bueno de Mesquita HB, Buchner FL, Clavel-Chapelon F, Boutron-Ruault MC, Krogh V, Saieva C, Vineis P, Panico S, Tumino R, Nyrén O, Simán H, Berglund G, Hallmans G, Sanchez MJ, Larrãnaga N, Barricarte A, Navarro C, Quiros JR, Key T, Allen N, Bingham S, Khaw KT, Boeing H, Weikert C, Linseisen J, Nagel G, Overvad K, Thomsen RW, Tjonneland A, Olsen A, Trichoupoulou A, Trichopoulos D, Arvaniti A, Pera G, Kaaks R, Jenab M, Ferrari P, Nesi G, Carneiro F, Riboli E, Gonzalez CA:
CagA+ Helicobacter pylori infection and gastric cancer risk in the EPIC-EURGAST study.
Int J Cancer
; 2007 Feb 15;120(4):859-67
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[Title]
CagA+ Helicobacter pylori infection and
gastric
cancer risk in the EPIC-EURGAST study.
Helicobacter pylori (H. pylori), atrophic gastritis, dietary and life-style factors have been associated with
gastric
cancer (GC).
According to site, the risk of noncardia GC associated with CagA seropositivity showed a further increase (OR 6.5; 95% CI 3.3-12.6); on the other hand, a ten-fold increased risk
of cardia
GC was associated with SCAG (OR 11.0; 95% CI 3.0-40.9).
This association was limited to distal GC, while serologically defined SCAG was strongly associated with
cardia
GC, thus suggesting a divergent risk pattern for these 2 sites.
[MeSH-major]
Adenocarcinoma
/ microbiology. Antibodies, Bacterial / blood. Antigens, Bacterial / immunology. Bacterial Proteins / immunology. Helicobacter Infections / microbiology. Helicobacter pylori / isolation & purification.
Stomach
Neoplasms / microbiology
[MeSH-minor]
Cardia
/ microbiology.
Cardia
/ pathology. Case-Control Studies. Cohort Studies. Humans. International Agencies. Male. Middle Aged. Prevalence. Prospective Studies. Risk Assessment. Risk Factors. Seroepidemiologic Studies. Time Factors
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[ErratumIn]
Int J Cancer. 2007 Aug 15;121(4):928. E Numans, Mattijs [corrected to Numans, Mattijs E]
(PMID = 17131317.001).
[ISSN]
0020-7136
[Journal-full-title]
International journal of cancer
[ISO-abbreviation]
Int. J. Cancer
[Language]
eng
[Grant]
United Kingdom / Medical Research Council / / G0401527; United Kingdom / Wellcome Trust / /
[Publication-type]
Comparative Study; Journal Article; Multicenter Study; Randomized Controlled Trial; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Antibodies, Bacterial; 0 / Antigens, Bacterial; 0 / Bacterial Proteins; 0 / cagA protein, Helicobacter pylori
76.
Hwang SW, Lee DH, Lee SH, Park YS, Hwang JH, Kim JW, Jung SH, Kim NY, Kim YH, Lee KH, Kim HH, Park DJ, Lee HS, Jung HC, Song IS:
Preoperative staging of gastric cancer by endoscopic ultrasonography and multidetector-row computed tomography.
J Gastroenterol Hepatol
; 2010 Mar;25(3):512-8
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[Title]
Preoperative staging of
gastric
cancer by endoscopic ultrasonography and multidetector-row computed tomography.
BACKGROUND AND AIM: The aim of this study was to determine the accuracy of endoscopic ultrasonography (EUS) and multidetector-row computed tomography (MDCT) for the locoregional staging of
gastric
cancer.
EUS and computed tomography (CT) are valuable tools for the preoperative evaluation of
gastric
cancer.
The performance of EUS and MDCT for large lesions and lesions at the
cardia
and angle had significantly lower accuracy than that of other groups.
For EUS, the early
gastric
cancer lesions with ulcerative changes had significantly lower accuracy than those without ulcerative changes.
CONCLUSIONS: For the preoperative assessment of individual T and N staging in patients with
gastric
cancer, the accuracy of MDCT was close to that of EUS.
Both EUS and MDCT are useful complementary modalities for the locoregional staging of
gastric
cancer.
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(PMID = 20370729.001).
[ISSN]
1440-1746
[Journal-full-title]
Journal of gastroenterology and hepatology
[ISO-abbreviation]
J. Gastroenterol. Hepatol.
[Language]
ENG
[Publication-type]
Journal Article
[Publication-country]
Australia
77.
Lagergren J, Jansson C, Viklund P:
Chewing gum and risk of oesophageal adenocarcinoma: a new hypothesis tested in a population-based study.
Eur J Cancer
; 2006 Sep;42(14):2359-62
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[Title]
Chewing gum and risk of oesophageal
adenocarcinoma
: a new hypothesis tested in a population-based study.
The aim of this study was to test the hypothesis that chewing gum is associated with risk of oesophageal and
cardia adenocarcinoma
.
In all, 189 and 262 patients with oesophageal and
cardia adenocarcinoma
, respectively, and 820 population-based control subjects were interviewed.
Regular users of chewing gum (P3 times/week for P6 months) were not at increased risk of oesophageal
adenocarcinoma
(OR 1.0, 95% CI 0.6-2.2), and no duration-response relation was observed (P = 0.38).
No association between regular gum chewing and
cardia adenocarcinoma
was found (OR 1.0, 95% CI 0.6-1.7), irrespective of duration of use (P = 0.56).
In conclusion, with regard to risk of oesophageal or
cardia adenocarcinoma
, gum chewing seems harmless.
[MeSH-major]
Adenocarcinoma
/ etiology.
Cardia
. Chewing Gum / adverse effects. Esophageal Neoplasms / etiology.
Stomach
Neoplasms / etiology
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(PMID = 16890425.001).
[ISSN]
0959-8049
[Journal-full-title]
European journal of cancer (Oxford, England : 1990)
[ISO-abbreviation]
Eur. J. Cancer
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / Chewing Gum
78.
Derakhshan MH, Liptrot S, Paul J, Brown IL, Morrison D, McColl KE:
Oesophageal and gastric intestinal-type adenocarcinomas show the same male predominance due to a 17 year delayed development in females.
Gut
; 2009 Jan;58(1):16-23
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[Title]
Oesophageal and
gastric
intestinal-type adenocarcinomas show the same male predominance due to a 17 year delayed development in females.
BACKGROUND AND AIMS: Upper gastrointestinal adenocarcinomas show an unexplained male predominance that is more apparent in oesophagus than
stomach
and in intestinal than diffuse histological subtype.
METHOD AND MATERIALS: Of 3270
gastric
and oesophageal cancers recorded in the West of Scotland Cancer Registry, 1998-2002, 812 were randomly selected for detailed analysis.
The Lauren histological subtype
of adenocarcinoma
was determined by reviewing 1204 original reports and 3241 biopsies.
RESULTS: Analysis included 405 non-
cardia
cancers, 173
cardia
cancers and 209 oesophageal adenocarcinomas.
The M/F ratios for oesophageal,
cardia
and non-
cardia
gastric
cancer were 3.5, 2.0 and 1.6, respectively.
CONCLUSION: Male predominance of upper gastrointestinal
adenocarcinoma
is related to the intestinal histological subtype rather than tumour location and is due to marked delayed development of this subtype in females prior to 50-60 years of age.
[MeSH-major]
Adenocarcinoma
/ epidemiology. Esophageal Neoplasms / epidemiology.
Stomach
Neoplasms / epidemiology
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(PMID = 18838486.001).
[ISSN]
1468-3288
[Journal-full-title]
Gut
[ISO-abbreviation]
Gut
[Language]
eng
[Grant]
United Kingdom / Chief Scientist Office / / CZB/4/485
[Publication-type]
Journal Article
[Publication-country]
England
79.
Siewert JR, Feith M, Stein HJ:
Biologic and clinical variations of adenocarcinoma at the esophago-gastric junction: relevance of a topographic-anatomic subclassification.
J Surg Oncol
; 2005 Jun 1;90(3):139-46; discussion 146
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[Title]
Biologic and clinical variations
of adenocarcinoma
at the esophago-
gastric
junction: relevance of a topographic-anatomic subclassification.
A topographic-anatomic subclassification of adenocarcinomas of the esophago-
gastric
junction (AEG) in distal esophageal
adenocarcinoma
(AEG Type I), true carcinoma of the
cardia
(AEG Type II), and subcardial
gastric
cancer (AEG Type III) was introduced in 1987 and is now increasingly accepted and used worldwide.
[MeSH-major]
Adenocarcinoma
/ classification.
Cardia
. Esophageal Neoplasms / classification. Esophagogastric Junction.
Stomach
Neoplasms / classification
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[Copyright]
Copyright 2005 Wiley-Liss, Inc
(PMID = 15895452.001).
[ISSN]
0022-4790
[Journal-full-title]
Journal of surgical oncology
[ISO-abbreviation]
J Surg Oncol
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
United States
[Number-of-references]
35
80.
Lee WA:
Gastric extremely well differentiated adenocarcinoma of gastric phenotype: as a gastric counterpart of adenoma malignum of the uterine cervix.
World J Surg Oncol
; 2005 May 23;3:28
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[Title]
Gastric
extremely well differentiated
adenocarcinoma of
gastric
phenotype: as a
gastric
counterpart of adenoma malignum of the uterine cervix.
BACKGROUND: Most of
gastric
adenocarcinoma
can be simply diagnosed by microscopic examination of biopsy specimen.
CASE PRESENTATION: A 67-year-old male presented with a
gastric
mass incidentally found on the abdominal computed tomography (CT) for routine medical examination.
Gastric
endoscopic examination revealed a huge fungating mass at the
cardia
and mucosal biopsy was performed.
The resected
stomach
revealed a huge fungating tumor at the
cardia
.
Microscopically the tumor was sharply demarcated from surrounding mucosa and composed of very well formed glandular structures without significant cellular atypia, which invaded into the whole layer of
the gastric
wall.
CONCLUSION: The clinicopathologic profiles of
gastric
extremely well differentiated
adenocarcinoma of
gastric
phenotype include cardiac location, fungating gross type, very similar histology to foveolar epithelial hyperplasia, foveolar mucin phenotype, lack of p53 overexpressoin and high proliferative index.
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[Cites]
Hum Pathol. 1999 Jul;30(7):826-32
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10414502.001
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Hum Pathol. 2000 Sep;31(9):1031-5
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[
12428787.001
]
(PMID = 15907218.001).
[ISSN]
1477-7819
[Journal-full-title]
World journal of surgical oncology
[ISO-abbreviation]
World J Surg Oncol
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
England
[Other-IDs]
NLM/ PMC1180859
81.
Chandrasoma P, Wickramasinghe K, Ma Y, DeMeester T:
Is intestinal metaplasia a necessary precursor lesion for adenocarcinomas of the distal esophagus, gastroesophageal junction and gastric cardia?
Dis Esophagus
; 2007;20(1):36-41
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[Title]
Is intestinal metaplasia a necessary precursor lesion for adenocarcinomas of the distal esophagus, gastroesophageal junction and
gastric
cardia
?
Adenocarcinoma of
the distal esophagus and gastroesophageal junction are believed to arise in Barrett's esophagus with intestinal metaplasia.
Whether
adenocarcinoma
can arise in columnar lined esophagus without intestinal metaplasia is in doubt.
Whether
adenocarcinoma of
the gastric
cardia
arises in intestinal metaplasia of
the gastric
cardia
is also in doubt.
We aim to evaluate the relationship of size and stage
of adenocarcinoma
of the distal esophagus, gastroesophageal junction and
gastric
cardia
to intestinal metaplasia and other types of columnar epithelium.
Residual intestinal metaplasia was present in 48 (65%) tumors, including 33/38 (87%) distal esophageal, 10/25 (45%) junctional and 5/11 (45%)
gastric
cardia
tumors.
These data strongly support the contention that adenocarcinomas of this region, including those in
the gastric
cardia
, arise in intestinal metaplastic epithelium.
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(PMID = 17227308.001).
[ISSN]
1120-8694
[Journal-full-title]
Diseases of the esophagus : official journal of the International Society for Diseases of the Esophagus
[ISO-abbreviation]
Dis. Esophagus
[Language]
ENG
[Publication-type]
Journal Article
[Publication-country]
United States
82.
Wen S, Moss SF:
Helicobacter pylori virulence factors in gastric carcinogenesis.
Cancer Lett
; 2009 Sep 8;282(1):1-8
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[Title]
Helicobacter pylori virulence factors in
gastric
carcinogenesis.
Helicobacter pylori infection is the most important risk factor in the development of non-
cardia
gastric
adenocarcinoma
; host genetic variability and dietary co-factors also modulate risk.
Improved understanding of the pathogenesis of H. pylori-associated
gastric
cancer may improve risk stratification for prevention and therapy.
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[Cites]
Oncogene. 2007 May 24;26(24):3462-72
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17160020.001
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Mol Microbiol. 2001 Dec;42(5):1337-48
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]
(PMID = 19111390.001).
[ISSN]
1872-7980
[Journal-full-title]
Cancer letters
[ISO-abbreviation]
Cancer Lett.
[Language]
ENG
[Grant]
United States / NCI NIH HHS / CA / CA125126-01A1; United States / NCI NIH HHS / CA / R21 CA125126-01A1; United States / NCI NIH HHS / CA / R01 CA111533-03; United States / NCI NIH HHS / CA / R21 CA125126; United States / NCI NIH HHS / CA / CA111533-03; United States / NCI NIH HHS / CA / R01 CA111533
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Review
[Publication-country]
Ireland
[Chemical-registry-number]
0 / Antigens, Bacterial; 0 / Bacterial Proteins; 0 / VacA protein, Helicobacter pylori; 0 / Virulence Factors; 0 / cagA protein, Helicobacter pylori
[Number-of-references]
96
[Other-IDs]
NLM/ NIHMS133225; NLM/ PMC2746929
83.
Carboni F, Lorusso R, Santoro R, Lepiane P, Mancini P, Sperduti I, Santoro E:
Adenocarcinoma of the esophagogastric junction: the role of abdominal-transhiatal resection.
Ann Surg Oncol
; 2009 Feb;16(2):304-10
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[Title]
Adenocarcinoma of
the esophagogastric junction: the role of abdominal-transhiatal resection.
The surgical strategy for
adenocarcinoma of
the esophagogastric junction is still controversial.
The aim of this study was to evaluate surgical results of the abdominal-transhiatal approach for 100 consecutively operated type II and III
cardia adenocarcinoma
, to clarify clinicopathological differences between these tumors, and to define prognostic factors.
A prospectively maintained database identified 100 consecutively operated patients with Siewert type II and III
cardia adenocarcinoma
.
True carcinoma of the
cardia
may be a distinct clinical entity with a more aggressive natural history than subcardial
gastric
carcinoma.
[MeSH-major]
Adenocarcinoma
/ surgery. Digestive System Surgical Procedures. Esophagogastric Junction / surgery.
Stomach
Neoplasms / surgery
[MeSH-minor]
Adult. Aged. Aged, 80 and over.
Cardia
/ pathology.
Cardia
/ surgery. Female. Humans. Lymph Nodes / pathology. Lymph Nodes / surgery. Lymphatic Metastasis. Male. Middle Aged. Neoplasm Invasiveness. Neoplasm Staging. Prognosis. Prospective Studies. Survival Rate. Treatment Outcome
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[CommentIn]
Ann Surg Oncol. 2009 Jul;16(7):2074-5; author reply 2076
[
19365623.001
]
(PMID = 19050964.001).
[ISSN]
1534-4681
[Journal-full-title]
Annals of surgical oncology
[ISO-abbreviation]
Ann. Surg. Oncol.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
84.
Hanazono K, Natsugoe S, Stein HJ, Aikou T, Hoefler H, Siewert JR:
Distribution of p53 mutations in esophageal and gastric carcinomas and the relationship with p53 expression.
Oncol Rep
; 2006 Apr;15(4):821-4
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[Title]
Distribution of p53 mutations in esophageal and
gastric
carcinomas and the relationship with p53 expression.
This study investigated the distribution of p53 mutations within both esophageal and
gastric
adenocarcinomas.
The patients included 8 cases
of adenocarcinoma
from the
cardia
(esophagogastric junction) and 9 cases of
gastric
carcinoma.
DHPLC demonstrated that 37.5% (3/8) of esophageal carcinomas and 44.4% (4/9) of
gastric
carcinomas have p53 mutations.
[MeSH-major]
Mutation.
Stomach
Neoplasms / pathology. Tumor Suppressor Protein p53 / genetics
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.
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.
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(PMID = 16525665.001).
[ISSN]
1021-335X
[Journal-full-title]
Oncology reports
[ISO-abbreviation]
Oncol. Rep.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Greece
[Chemical-registry-number]
0 / DNA, Neoplasm; 0 / Tumor Suppressor Protein p53
85.
Ding GC, Ren JL, Chang FB, Li JL, Yuan L, Song X, Zhou SL, Guo T, Fan ZM, Zeng Y, Wang LD:
Human papillomavirus DNA and P16(INK4A) expression in concurrent esophageal and gastric cardia cancers.
World J Gastroenterol
; 2010 Dec 14;16(46):5901-6
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[Title]
Human papillomavirus DNA and P16(INK4A) expression in concurrent esophageal and
gastric
cardia
cancers.
AIM: To investigate the relationship between human papillomavirus (HPV) infection and concurrent esophagus and
gastric
cardia
cancer from the same patient (CC) and examine the significance of P16(INK4A) protein expression.
RESULTS: Among the CC specimens, HPV16-DNA was found in eight cases of esophageal squamous cell carcinoma (ESCC) and five cases of
gastric
cardia adenocarcinoma
(GCA), respectively (47% vs 29%), and two of both ESCC and GCA.
P16(INK4A) may be a screening index in the HPV-associated carcinoma of
gastric
cardia
.
[MeSH-major]
Cardia
/ pathology. Cyclin-Dependent Kinase Inhibitor p16 / metabolism. DNA, Viral / genetics. Esophageal Neoplasms / virology. Human papillomavirus 16 / genetics.
Stomach
Neoplasms / virology
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.
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consumer health - Stomach Cancer
.
The Lens.
Cited by Patents in
.
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[Cites]
Int J Cancer. 2000 Jun 15;86(6):874-8
[
10842204.001
]
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]
[Cites]
J Cancer Res Clin Oncol. 2000 Nov;126(11):655-60
[
11079730.001
]
(PMID = 21155014.001).
[ISSN]
2219-2840
[Journal-full-title]
World journal of gastroenterology
[ISO-abbreviation]
World J. Gastroenterol.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
China
[Chemical-registry-number]
0 / Cyclin-Dependent Kinase Inhibitor p16; 0 / DNA, Viral
[Other-IDs]
NLM/ PMC3001984
86.
Gulmann C, Lantuejoul S, Grace A, Leader M, Patchett S, Kay E:
Telomerase activity in proximal and distal gastric neoplastic and preneoplastic lesions using immunohistochemical detection of hTERT.
Dig Liver Dis
; 2005 Jun;37(6):439-45
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[Title]
Telomerase activity in proximal and distal
gastric
neoplastic and preneoplastic lesions using immunohistochemical detection of hTERT.
BACKGROUND: The incidence of distal (corpus and antrum)
gastric
adenocarcinoma
is decreasing with a simultaneous increase in incidence of proximal (
cardia
)
adenocarcinoma
.
Intestinal metaplasia may have a lower malignant potential in the proximal
stomach
but regardless of the locations, its specificity as a predictor of carcinoma is low.
AIMS: The aim of this study was to establish whether human telomerase reverse transcriptase expression differs at various points in proximal versus distal
gastric
carcinogenesis and to test the utility of human telomerase reverse transcriptase expression as a marker of cancer risk in intestinal metaplasia.
MATERIAL AND METHODS: Wax-embedded tissue from proximal and distal
stomach
including normal mucosa (n=86), intestinal metaplasia (n=83) and carcinoma (n=101) were used and slides were immunostained for human telomerase reverse transcriptase and pRb and scored semi-quantitatively.
RESULTS: The results showed that in both proximal and distal
stomach
, human telomerase reverse transcriptase expression rates increased from normal mucosa to cancer.
CONCLUSIONS: In conclusion, telomerase activity appears to be an early event in both proximal and distal
gastric
carcinogenesis and human telomerase reverse transcriptase is expressed in intestinal metaplasia.
[MeSH-major]
Carcinoma / metabolism. DNA-Binding Proteins / metabolism. Precancerous Conditions / metabolism.
Stomach
Neoplasms / metabolism. Telomerase / metabolism
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NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
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(PMID = 15893283.001).
[ISSN]
1590-8658
[Journal-full-title]
Digestive and liver disease : official journal of the Italian Society of Gastroenterology and the Italian Association for the Study of the Liver
[ISO-abbreviation]
Dig Liver Dis
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Netherlands
[Chemical-registry-number]
0 / Biomarkers, Tumor; 0 / DNA-Binding Proteins; 0 / Retinoblastoma Protein; EC 2.7.7.49 / Telomerase
87.
Zheng B, Chen YB, Hu Y, Wang JY, Zhou ZW, Fu JH:
[Trend analysis for clinical characteristics and prognosis of adenocarcinoma of cardia].
Chin J Cancer
; 2010 Jan;29(1):94-7
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[Title]
[Trend analysis for clinical characteristics and prognosis
of adenocarcinoma of cardia
].
BACKGROUND AND OBJECTIVE: The incidence
of adenocarcinoma
of the
cardia
has recently increased.
This study compared the clinicopathology and prognosis of patients with
gastric
cardia adenocarcinoma
in different periods between 1984 and 2003.
METHODS: A total of 589 patients with pathologically confirmed
gastric
cardia adenocarcinoma
hospitalized in Sun Yat-sen University Cancer Center between 1984 and 2003 were divided into 5-year groups.
CONCLUSIONS: During the past 20 years, associated with the upward-trending incidence of
gastric
cardia adenocarcinoma
, the admission rate at our hospital of patients with the tumor increased.
[MeSH-major]
Adenocarcinoma
.
Cardia
/ pathology.
Stomach
Neoplasms
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(PMID = 20038318.001).
[ISSN]
1000-467X
[Journal-full-title]
Chinese journal of cancer
[ISO-abbreviation]
Chin J Cancer
[Language]
chi
[Publication-type]
English Abstract; Journal Article
[Publication-country]
China
88.
Buchs NC, Bucher P, Pugin F, Hagen ME, Morel P:
Robot-assisted oncologic resection for large gastric gastrointestinal stromal tumor: a preliminary case series.
J Laparoendosc Adv Surg Tech A
; 2010 Jun;20(5):411-5
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[Title]
Robot-assisted oncologic resection for large
gastric
gastrointestinal stromal tumor: a preliminary case series.
BACKGROUND: Laparoscopic resection of
gastric
gastrointestinal stromal tumor (GIST) has been shown as feasible and safe in terms of oncologic results.
The robotic approach may, by its characteristics, enable the surgeon to perform atypical gastrectomies in an unfavorable location (i.e., close to pylorus or
cardia
).
Its use in oncologic
gastric
surgery has been poorly defined and has never been reported for GIST.
MATERIALS AND METHODS: All patients who underwent robotic-assisted
gastric
resection for GIST at a single institution from 2006 to 2009 were prospectively followed-up.
One patient had a conversion to open surgery because of a suspicion of diffuse
adenocarcinoma
on fresh frozen section and necessitated a total gastrectomy with a radical lymph node dissection.
CONCLUSIONS: The da Vinci robot (Intuitive Surgical, Inc., Sunnyvale, CA) is a valuable instrument for oncologically safe resection with esogastric or duodenogastric junction preservation for an unfavorably located
gastric
GIST.
Moreover, the three-dimensional, high-definition vision, instrument mobility, and ease of performing a difficult suturing enable a safe, large atypical gastrectomy, close to the pylorus or
cardia
.
[MeSH-major]
Gastrectomy / instrumentation. Gastrointestinal Stromal Tumors / surgery.
Stomach
Neoplasms / surgery
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(PMID = 20459328.001).
[ISSN]
1557-9034
[Journal-full-title]
Journal of laparoendoscopic & advanced surgical techniques. Part A
[ISO-abbreviation]
J Laparoendosc Adv Surg Tech A
[Language]
eng
[Publication-type]
Clinical Trial; Journal Article
[Publication-country]
United States
89.
Schiesser M, Schneider PM:
Surgical strategies for adenocarcinoma of the esophagogastric junction.
Recent Results Cancer Res
; 2010;182:93-106
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[Title]
Surgical strategies for
adenocarcinoma of
the esophagogastric junction.
This chapter summarizes the surgical strategies for adenocarcinomas of the distal esophagus,
gastric
cardia
, and subcardial
gastric
cancer invading the
cardia
+/-distal esophagus known as adenocarcinomas of the esophagogastric junction (AEG).
[MeSH-major]
Adenocarcinoma
/ surgery. Esophageal Neoplasms / surgery. Esophagogastric Junction.
Stomach
Neoplasms / surgery
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(PMID = 20676874.001).
[ISSN]
0080-0015
[Journal-full-title]
Recent results in cancer research. Fortschritte der Krebsforschung. Progrès dans les recherches sur le cancer
[ISO-abbreviation]
Recent Results Cancer Res.
[Language]
eng
[Publication-type]
Journal Article; Review
[Publication-country]
Germany
90.
Stahl M, Walz MK, Stuschke M, Lehmann N, Meyer HJ, Riera-Knorrenschild J, Langer P, Engenhart-Cabillic R, Bitzer M, Königsrainer A, Budach W, Wilke H:
Phase III comparison of preoperative chemotherapy compared with chemoradiotherapy in patients with locally advanced adenocarcinoma of the esophagogastric junction.
J Clin Oncol
; 2009 Feb 20;27(6):851-6
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[Title]
Phase III comparison of preoperative chemotherapy compared with chemoradiotherapy in patients with locally advanced
adenocarcinoma of
the esophagogastric junction.
PURPOSE: Preoperative chemotherapy is an accepted standard in the treatment of localized esophagogastric
adenocarcinoma
.
PATIENTS AND METHODS: Patients with locally advanced (uT3-4NXM0)
adenocarcinoma of
the lower esophagus or
gastric
cardia
were randomly allocated to one of two treatment groups: induction chemotherapy (15 weeks) followed by surgery (arm A); or chemotherapy (12 weeks) followed by chemoradiotherapy (3 weeks) followed by surgery (arm B).
[MeSH-major]
Adenocarcinoma
/ therapy. Antineoplastic Agents / administration & dosage. Esophageal Neoplasms / therapy. Esophagogastric Junction.
Stomach
Neoplasms / therapy
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[CommentIn]
J Clin Oncol. 2009 Feb 20;27(6):836-7
[
19139425.001
]
(PMID = 19139439.001).
[ISSN]
1527-7755
[Journal-full-title]
Journal of clinical oncology : official journal of the American Society of Clinical Oncology
[ISO-abbreviation]
J. Clin. Oncol.
[Language]
eng
[Publication-type]
Clinical Trial, Phase III; Comparative Study; Journal Article; Randomized Controlled Trial; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Antineoplastic Agents
91.
Rutegård M, Shore R, Lu Y, Lagergren P, Lindblad M:
Sex differences in the incidence of gastrointestinal adenocarcinoma in Sweden 1970-2006.
Eur J Cancer
; 2010 Apr;46(6):1093-100
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[Title]
Sex differences in the incidence of gastrointestinal
adenocarcinoma
in Sweden 1970-2006.
BACKGROUND: Oesophageal and
gastric
adenocarcinoma
share a male predominance not seen for other adenocarcinomas of the gastrointestinal tract.
METHODS: The Swedish Cancer Register was used to collect primary oesophageal,
gastric
cardia
, non-
cardia
gastric
, colonic and pancreatic
adenocarcinoma
cases aged 25-84, during the study period of 1970-2006.
RESULTS: The sex ratio for oesophageal
adenocarcinoma
ranged from approximately 10:1 to 4:1, presenting a seemingly consistent decline with age.
The sex ratio for non-
cardia
gastric
adenocarcinoma
, however, increased with age to reach 2:1 at a point one to two decades after menopause, where the ratio levelled off and eventually declined.
There was no discernible time period effect concerning any type
of adenocarcinoma
.
The ratios for
gastric
cardia
, colonic and pancreatic
adenocarcinoma
were stable with age.
CONCLUSION: This study indicates separate patterns of age-dependency of the sex difference in oesophageal and non-
cardia
gastric
adenocarcinoma
incidence.
The non-
cardia
gastric
adenocarcinoma
pattern might be due to a protective effect during premenopausal years for the female population, while the seemingly steady decline in sex ratio in oesophageal
adenocarcinoma
indicates a mechanism independent of menopause.
[MeSH-major]
Adenocarcinoma
/ epidemiology. Colonic Neoplasms / epidemiology. Esophageal Neoplasms / epidemiology. Pancreatic Neoplasms / epidemiology.
Stomach
Neoplasms / epidemiology
[MeSH-minor]
Adult. Age Distribution. Aged. Aged, 80 and over.
Cardia
. Female. Humans. Incidence. Male. Middle Aged. Sex Distribution. Sweden / epidemiology. Time Factors
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[Copyright]
Copyright (c) 2010 Elsevier Ltd. All rights reserved.
(PMID = 20188539.001).
[ISSN]
1879-0852
[Journal-full-title]
European journal of cancer (Oxford, England : 1990)
[ISO-abbreviation]
Eur. J. Cancer
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
92.
Cheng Y, Zhang J, Li Y, Wang Y, Gong J:
Proteome analysis of human gastric cardia adenocarcinoma by laser capture microdissection.
BMC Cancer
; 2007;7:191
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[Title]
Proteome analysis of human
gastric
cardia adenocarcinoma
by laser capture microdissection.
BACKGROUND: The incidence of
gastric
cardiac
adenocarcinoma
(GCA) has been increasing in the past two decades in China, but the molecular changes relating to carcinogenesis have not been well characterised.
METHODS: In this study, we used a comparative proteomic approach to analyse the malignant and nonmalignant
gastric
cardia
epithelial cells isolated by navigated laser capture microdissection (LCM) from paired surgical specimens of human GCA.
[MeSH-major]
Adenocarcinoma
/ metabolism.
Cardia
/ metabolism. Proteome / analysis.
Stomach
Neoplasms / metabolism
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[ISSN]
1471-2407
[Journal-full-title]
BMC cancer
[ISO-abbreviation]
BMC Cancer
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / Proteome
[Other-IDs]
NLM/ PMC2151079
93.
Lee JH, Kim SH, Han SH, An JS, Lee ES, Kim YS:
Clinicopathological and molecular characteristics of Epstein-Barr virus-associated gastric carcinoma: a meta-analysis.
J Gastroenterol Hepatol
; 2009 Mar;24(3):354-65
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[Title]
Clinicopathological and molecular characteristics of Epstein-Barr virus-associated
gastric
carcinoma: a meta-analysis.
There is conflicting data regarding the clinicopathological significance of the risk factors associated with Epstein-Barr virus (EBV)-associated
gastric
carcinoma (EBVaGC).
EBVaGC developed most often in the
cardia
and body, and it generally showed the diffuse histological type.
The clinicopathological and molecular characteristics of EBVaGC are quite different from those of conventional
gastric
adenocarcinoma
.
[MeSH-major]
Carcinoma / virology. Epstein-Barr Virus Infections / complications.
Stomach
Neoplasms / virology
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(PMID = 19335785.001).
[ISSN]
1440-1746
[Journal-full-title]
Journal of gastroenterology and hepatology
[ISO-abbreviation]
J. Gastroenterol. Hepatol.
[Language]
eng
[Publication-type]
Journal Article; Meta-Analysis; Review
[Publication-country]
Australia
[Chemical-registry-number]
0 / TP53 protein, human; 0 / Tumor Suppressor Protein p53
[Number-of-references]
75
94.
Maeda H, Okabayashi T, Nishimori I, Sugimoto T, Namikawa T, Dabanaka K, Tsujii S, Onishi S, Kobayashi M, Hanazaki K:
Clinicopathologic features of adenocarcinoma at the gastric cardia: is it different from distal cancer of the stomach?
J Am Coll Surg
; 2008 Feb;206(2):306-10
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[Title]
Clinicopathologic features
of adenocarcinoma
at
the gastric
cardia
: is it different from distal cancer of the
stomach
?
BACKGROUND: Although the incidence of
gastric
cardia
cancer is considerably less than more distal
gastric
cancer, the rate of occurrence is now increasing.
The objective of this study was to evaluate and compare the clinicopathologic findings of
gastric
cardia
and more distal
stomach adenocarcinoma
.
STUDY DESIGN: Patients included in our study were those who underwent operations for
gastric
adenocarcinoma
in our institute from 1981 to 2006, and who had undergone complete medical history, including history of daily alcohol consumption; smoking; body mass index; and pathologic examinations.
A total of 843 patients were included in our study, and were divided into
cardia
and noncardia cancer groups.
RESULTS: Among the 843 patients, 23 (2.8%) had
gastric
cardia
cancer.
Mean size
of cardia
tumors was larger than noncardia tumors.
Although noncardia cancer was often detected at an early stage,
gastric
cardia
cancer was most often diagnosed at an advanced stage.
Pathologically,
cardia
cancer was more invasive and had more lymphatic permeation and lymph node metastasis than noncardia cancer.
CONCLUSIONS:
Gastric
cardia
cancer occurs at a low incidence of only 2.8% of resected
gastric
cancers.
Unlike cases of
gastric
cardia
cancer in Western populations, body mass index is not associated with occurrence of
gastric
cardia
cancer in our study.
Because
gastric
cardia
cancer appears more aggressive than noncardia
gastric
cancer, early diagnosis and intervention are important.
[MeSH-major]
Adenocarcinoma
/ pathology.
Cardia
/ pathology.
Stomach
Neoplasms / pathology
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(PMID = 18222384.001).
[ISSN]
1879-1190
[Journal-full-title]
Journal of the American College of Surgeons
[ISO-abbreviation]
J. Am. Coll. Surg.
[Language]
eng
[Publication-type]
Comparative Study; Journal Article
[Publication-country]
United States
95.
Früh M, Ruhstaller T, Neuweiler J, Cerny T:
Resection of skin metastases from gastric carcinoma with long-term follow-up: an unusual clinical presentation.
Onkologie
; 2005 Jan;28(1):38-40
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[Title]
Resection of skin metastases from
gastric
carcinoma with long-term follow-up: an unusual clinical presentation.
BACKGROUND: Skin metastases from
gastric
cancer are rare and generally occur at a very late stage in the course of the disease.
CASE REPORT: A 60-year-old patient with localized
adenocarcinoma of
the
cardia
(stage II) was primarily treated with extended total gastrectomy with transhiatal resection of the distal esophagus.
CONCLUSION: We report a long-term disease-free survival of a patient with isolated cutaneous metastases of a
gastric
cancer.
[MeSH-major]
Adenocarcinoma
/ secondary.
Adenocarcinoma
/ surgery. Skin Neoplasms / secondary. Skin Neoplasms / surgery.
Stomach
Neoplasms / surgery
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(PMID = 15604627.001).
[ISSN]
0378-584X
[Journal-full-title]
Onkologie
[ISO-abbreviation]
Onkologie
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Switzerland
96.
Wu IC, Wu DC, Yu FJ, Wang JY, Kuo CH, Yang SF, Wang CL, Wu MT:
Association between Helicobacter pylori seropositivity and digestive tract cancers.
World J Gastroenterol
; 2009 Nov 21;15(43):5465-71
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METHODS: In total, 199 oral squamous-cell carcinoma (SCC), 317 esophageal SCC, 196
gastric
cardia
and non-
cardia adenocarcinoma
and 240 colon
adenocarcinoma
patients were recruited for serum tests of H pylori infection.
RESULTS: Presence of H pylori infection was significantly inversely associated with esophageal SCC [adjusted odds ratio (AOR): 0.315-0.472, all P-value < 0.05] but positively associated with
gastric
adenocarcinoma
(both
cardia
and non-
cardia
) (AOR: 1.636-3.060, all P-value < 0.05) in comparison to the three control groups.
CONCLUSION: Our findings support the finding that H pylori seropositivity is inversely associated with esophageal SCC risk, but increases the risk of
gastric
cardia adenocarcinoma
.
[MeSH-major]
Adenocarcinoma
/ microbiology. Carcinoma, Squamous Cell / microbiology.
Cardia
/ microbiology. Esophageal Neoplasms / microbiology. Helicobacter Infections / blood. Helicobacter Infections / complications. Helicobacter pylori / immunology.
Stomach
Neoplasms / microbiology
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.
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Am J Gastroenterol. 2000 Feb;95(2):387-94
[
10685740.001
]
(PMID = 19916178.001).
[ISSN]
2219-2840
[Journal-full-title]
World journal of gastroenterology
[ISO-abbreviation]
World J. Gastroenterol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
China
[Chemical-registry-number]
0 / Antigens, Bacterial
[Other-IDs]
NLM/ PMC2778104
97.
Whitson BA, Groth SS, Li Z, Kratzke RA, Maddaus MA:
Survival of patients with distal esophageal and gastric cardia tumors: a population-based analysis of gastroesophageal junction carcinomas.
J Thorac Cardiovasc Surg
; 2010 Jan;139(1):43-8
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[Title]
Survival of patients with distal esophageal and
gastric
cardia
tumors: a population-based analysis of gastroesophageal junction carcinomas.
OBJECTIVE: Distal esophageal tumors and
gastric
cardia
tumors, although only physically separated by centimeters, have different staging systems and are usually treated differently.
We hypothesized that gastroesophageal junction adenocarcinomas (eg,
gastric
cardia
and distal esophageal tumors) were not distinct entities and had similar survival.
METHODS: Using the Surveillance, Epidemiology, and End Results database (1988-2005), we identified patients with adenocarcinomas of the distal esophagus (n = 1474) and
gastric
cardia
(n = 192).
RESULTS: Even after adjusting for potential confounding covariates (location, stage, race, cancer-directed surgery, and radiation therapy), we found no significant difference between distal esophageal and
gastric
cardia
tumors with regard to overall (hazard ratio, 1.18; 95% confidence interval, 0.99-1.41) and cancer-specific (hazard ratio, 1.09; 95% confidence interval, 0.90-1.31) survival.
CONCLUSION: Through a large, population-based analysis of
gastric
cardia
and distal esophageal adenocarcinomas, we found that patients with gastroesophageal junction adenocarcinomas have similar survival rates.
Adenocarcinomas of the gastroesophageal junction are not distinct entities delineated by anatomic boundaries and as such should be managed by one skilled in both esophageal and
gastric
resections.
[MeSH-major]
Cardia
. Esophageal Neoplasms / mortality. Esophagogastric Junction.
Stomach
Neoplasms / mortality
[MeSH-minor]
Adenocarcinoma
/ mortality.
Adenocarcinoma
/ therapy. Aged. Cohort Studies. Female. Humans. Male. Middle Aged. Retrospective Studies. Survival Rate
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consumer health - Esophageal Cancer
.
MedlinePlus Health Information.
consumer health - Stomach Cancer
.
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[Copyright]
Copyright 2010 The American Association for Thoracic Surgery. Published by Mosby, Inc. All rights reserved.
(PMID = 19660401.001).
[ISSN]
1097-685X
[Journal-full-title]
The Journal of thoracic and cardiovascular surgery
[ISO-abbreviation]
J. Thorac. Cardiovasc. Surg.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
98.
Ohdaira H, Noro T, Terada H, Kameyama J, Ohara T, Yoshino K, Kitajima M, Suzuki Y:
New double-stapling technique for esophagojejunostomy and esophagogastrostomy in gastric cancer surgery, using a peroral intraluminal approach with a digital stapling system.
Gastric Cancer
; 2009;12(2):101-5
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[Source]
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[Title]
New double-stapling technique for esophagojejunostomy and esophagogastrostomy in
gastric
cancer surgery, using a peroral intraluminal approach with a digital stapling system.
In the abdominal-transhiatal approach for resection
of adenocarcinoma
of the
cardia
or subcardia, and in laparoscopy-assisted total gastrectomy (LATG), the use of a circular stapling device has potential problems with the placement of the purse-string suture and insertion of the anvil of the instrument.
[MeSH-major]
Digestive System Surgical Procedures / instrumentation. Digestive System Surgical Procedures / methods.
Stomach
Neoplasms / surgery. Surgical Stapling / instrumentation. Surgical Stapling / methods
[MeSH-minor]
Anastomosis, Surgical / instrumentation. Anastomosis, Surgical / methods. Esophagus / surgery. Humans.
Stomach
/ surgery. Surgical Staplers. Sutures
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Gastric Cancer
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