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1
adenocarcinoma bronchiolo alveolar disease finding 2005:2010[pubdate] *count=100
3710 results
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Items 1 to 100 of about 3710
1.
Mensah-Osman E, Labut E, Zavros Y, El-Zaatari M, Law DJ, Merchant JL:
Regulated expression of the human gastrin gene in mice.
Regul Pept
; 2008 Nov 29;151(1-3):115-22
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Thus, we describe here the expression of the human gastrin gene using
a bacterial
artificial chromosome (
BAC
) in the antral and duodenal cells of gastrin null mice.
All 5 founder lines expressed the 253 kb human gastrin
BAC
. hGasBAC transgenic mice were bred onto a gastrin null background so that the levels of human gastrin peptide could be analyzed by immunohistochemistry and radioimmunoassay without detecting endogenous mouse gastrin.
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[Cites]
J Gastroenterol. 1999;34 Suppl 11:10-7
[
10616759.001
]
[Cites]
Curr Protoc Mol Biol. 2005 Aug;Chapter 23:Unit 23.11
[
18265362.001
]
[Cites]
Proc Natl Acad Sci U S A. 2001 Jan 30;98(3):1118-23
[
11158604.001
]
[Cites]
Transgenic Res. 2001 Aug;10(4):329-41
[
11592712.001
]
[Cites]
Am J Physiol Gastrointest Liver Physiol. 2002 Jan;282(1):G175-83
[
11751171.001
]
[Cites]
Proc Natl Acad Sci U S A. 2003 Oct 28;100(22):12944-9
[
14555768.001
]
[Cites]
Dev Biol. 2004 Jun 15;270(2):443-54
[
15183725.001
]
[Cites]
Life Sci. 1979 Nov 12;25(20):1749-53
[
529985.001
]
[Cites]
Am J Physiol. 1988 Jan;254(1 Pt 1):G20-4
[
2892424.001
]
[Cites]
J Biol Chem. 1988 Apr 15;263(11):5341-7
[
3356689.001
]
[Cites]
J Clin Invest. 1988 Sep;82(3):1059-66
[
2901431.001
]
[Cites]
Gastroenterology. 1991 Dec;101(6):1552-8
[
1683325.001
]
[Cites]
Gastroenterology. 1993 Jun;104(6):1655-60
[
8500723.001
]
[Cites]
Proc Natl Acad Sci U S A. 1993 Jul 15;90(14):6696-700
[
8393573.001
]
[Cites]
Yale J Biol Med. 1992 Nov-Dec;65(6):553-60; discussion 621-3
[
1364124.001
]
[Cites]
Am J Gastroenterol. 1994 Sep;89(9):1515-9
[
7915874.001
]
[Cites]
Am J Physiol. 1995 Jun;268(6 Pt 1):G1025-36
[
7611402.001
]
[Cites]
FEBS Lett. 1995 Aug 7;369(2-3):225-8
[
7649261.001
]
[Cites]
Biochem Biophys Res Commun. 1995 Nov 2;216(1):34-41
[
7488110.001
]
[Cites]
Eur J Gastroenterol Hepatol. 1997 Apr;9(4):361-5
[
9160198.001
]
[Cites]
Am J Physiol. 1998 Mar;274(3 Pt 1):G561-8
[
9530158.001
]
[Cites]
Korean J Intern Med. 1999 Jan;14(1):15-20
[
10063309.001
]
[Cites]
Genomics. 1999 Jun 15;58(3):250-3
[
10373322.001
]
[Cites]
Gastroenterology. 2005 May;128(5):1187-98
[
15887103.001
]
[Cites]
Annu Rev Genet. 2006;40:107-38
[
16953792.001
]
[Cites]
Scand J Clin Lab Invest. 2006;66(7):607-21
[
17101553.001
]
[Cites]
Dig Dis Sci. 2007 Oct;52(10):2482-9
[
17415644.001
]
[Cites]
Cancer Lett. 2007 Nov 8;257(1):1-15
[
17698287.001
]
[Cites]
Curr Opin Gastroenterol. 2007 Nov;23(6):595-601
[
17906434.001
]
[Cites]
Curr Top Dev Biol. 2008;80:337-60
[
17950379.001
]
[Cites]
Gastroenterol Clin North Am. 2007 Dec;36(4):851-65, vi
[
17996794.001
]
[Cites]
Nucleic Acids Res. 2008 Jan;36(Database issue):D83-7
[
17981843.001
]
[Cites]
Dig Dis Sci. 2000 Jul;45(7):1308-14
[
10961708.001
]
(PMID = 18456349.001).
[ISSN]
0167-0115
[Journal-full-title]
Regulatory peptides
[ISO-abbreviation]
Regul. Pept.
[Language]
ENG
[Grant]
United States / NIDDK NIH HHS / DK / P30 DK034933-219001; United States / NIDDK NIH HHS / DK / R01 DK045729; United States / NIDDK NIH HHS / DK / DK034933-219001; United States / NIDDK NIH HHS / DK / P30 DK034933; United States / NIDDK NIH HHS / DK / DK045729-14A1; United States / NIDDK NIH HHS / DK / R37 DK045729-14A1; United States / NIDDK NIH HHS / DK / R01-DK45729; United States / NIDDK NIH HHS / DK / R37 DK045729
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
Netherlands
[Chemical-registry-number]
0 / DNA Primers; 0 / Gastrins; 51110-01-1 / Somatostatin
[Other-IDs]
NLM/ NIHMS82617; NLM/ PMC2617792
2.
Gorman-Lewis D, Elias PE, Fein JB:
Adsorption of aqueous uranyl complexes onto Bacillus subtilis cells.
Environ Sci Technol
; 2005 Jul 1;39(13):4906-12
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[Title]
Adsorption of aqueous uranyl
complexes
onto Bacillus subtilis cells.
In oxygenated, CO2-rich systems, negatively charged uranyl
complexes
dominate the aqueous uranium speciation, and it is commonly assumed that these
complexes
exhibit negligible adsorption onto negatively charged surfaces such as bacteria.
We measured the adsorption of 4.2 x 10(-6) M aqueous uranium onto Bacillus subtilis from pH 1.5 to 9 and with wet weight
bacterial
concentrations from 0.125 to 0.5 g/L.
We observed extensive uranium adsorption onto
the bacterial
surface under all conditions.
Thermodynamic modeling of the data suggests that uranylhydroxide, uranyl-carbonate, and calcium-uranylcarbonate species each can form stable surface
complexes
on
the bacterial cell
wall.
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Hazardous Substances Data Bank.
Carbon dioxide
.
Hazardous Substances Data Bank.
URANIUM, ELEMENTAL
.
Hazardous Substances Data Bank.
CALCIUM, ELEMENTAL
.
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(PMID = 16053091.001).
[ISSN]
0013-936X
[Journal-full-title]
Environmental science & technology
[ISO-abbreviation]
Environ. Sci. Technol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, U.S. Gov't, Non-P.H.S.
[Publication-country]
United States
[Chemical-registry-number]
142M471B3J / Carbon Dioxide; 4OC371KSTK / Uranium; SY7Q814VUP / Calcium
3.
Norimatsu Y, Moriya T, Kobayashi TK, Sakurai T, Shimizu K, Tsukayama C, Ohno E:
Immunohistochemical expression of PTEN and beta-catenin for endometrial intraepithelial neoplasia in Japanese women.
Ann Diagn Pathol
; 2007 Apr;11(2):103-8
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PTEN and beta-catenin are the most common genes for which genetic abnormalities are found in endometrioid
adenocarcinoma
(type I) and even in their precursors.
Currently, the World Health Organization (WHO) classifies endometrial hyperplasia as a premalignant
disease
.
A total of 117 cases that were initially diagnosed as endometrial hyperplasia according to WHO classification were reevaluated histopathologically by the EIN
diagnosis
category.
They were classified into 38 EIN and 32 benign architectural changes of unopposed estrogen (
BAC
), and 47 cases were excluded.
Glandular epithelium was positive for PTEN in all the cases of NPE (20/20), whereas 12.5% (4/32)
of BAC
and 34.2% (13/38) of EIN were PTEN-null (negative).
Instead, none of the
BAC
or NPE showed positive nuclear staining.
This combination was statistically significantly more frequently seen than
BAC
(4/32, 12.5%) (P < .001) and NPE (0/20, 0%) (P < .0001).
[MeSH-major]
Carcinoma
in Situ / metabolism. Endometrial Neoplasms / metabolism. PTEN Phosphohydrolase / metabolism. beta Catenin / metabolism
[MeSH-minor]
Adult. Aged. Biomarkers, Tumor / metabolism.
Cell
Nucleus / metabolism.
Cell
Nucleus / pathology. Endometrium / metabolism. Female. Humans. Immunoenzyme Techniques. Japan. Middle Aged. Premenopause
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
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(PMID = 17349568.001).
[ISSN]
1092-9134
[Journal-full-title]
Annals of diagnostic pathology
[ISO-abbreviation]
Ann Diagn Pathol
[Language]
eng
[Publication-type]
Comparative Study; Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Biomarkers, Tumor; 0 / beta Catenin; EC 3.1.3.48 / PTEN protein, human; EC 3.1.3.67 / PTEN Phosphohydrolase
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4.
Rios JJ, Perelygin AA, Long MT, Lear TL, Zharkikh AA, Brinton MA, Adelson DL:
Characterization of the equine 2'-5' oligoadenylate synthetase 1 (OAS1) and ribonuclease L (RNASEL) innate immunity genes.
BMC Genomics
; 2007 Sep 07;8:313
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Mutations in either the OAS1 or RNASEL genes may also modulate the outcome of WNV-induced
disease
or other viral infections in horses.
Polymorphisms in the human OAS gene cluster have been previously utilized for case-control analysis of virus-induced
disease
in humans.
RESULTS: Genomic sequence for equine OAS1 was obtained from a contig assembly generated from a shotgun subclone library of CHORI-241
BAC
100I10.
The chromosomal location of the RNASEL gene was assigned by FISH to ECA5p17-p16 using two selected CHORI-241
BAC
clones.
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[Cites]
Nucleic Acids Res. 2002 Sep 1;30(17):3894-900
[
12202775.001
]
[Cites]
Physiol Genomics. 2007 Sep 19;31(1):96-103
[
17550993.001
]
[Cites]
Gene. 2004 Apr 14;330:75-84
[
15087126.001
]
[Cites]
Nucleic Acids Res. 2004 Jul 1;32(Web Server issue):W273-9
[
15215394.001
]
[Cites]
EMBO J. 2004 Oct 13;23(20):3929-38
[
15385955.001
]
[Cites]
Nature. 1976 Dec 2;264(5585):477-80
[
1004583.001
]
[Cites]
Nature. 1977 Aug 11;268(5620):537-40
[
560630.001
]
[Cites]
Nature. 1977 Aug 11;268(5620):540-2
[
196217.001
]
[Cites]
Proc Natl Acad Sci U S A. 1978 Jan;75(1):256-60
[
272640.001
]
[Cites]
Cell. 1978 Mar;13(3):565-72
[
657268.001
]
[Cites]
Nature. 1978 Jun 22;273(5664):684-7
[
208001.001
]
[Cites]
Biochem Biophys Res Commun. 1982 Oct 15;108(3):1243-50
[
6185117.001
]
[Cites]
Proc Natl Acad Sci U S A. 1983 Aug;80(16):4904-8
[
6348777.001
]
[Cites]
Nucleic Acids Res. 1985 Feb 25;13(4):1267-81
[
2860635.001
]
[Cites]
J Biol Chem. 1987 Mar 15;262(8):3852-7
[
2434505.001
]
[Cites]
EMBO J. 1987 May;6(5):1273-80
[
2440675.001
]
[Cites]
J Mol Biol. 1990 Oct 5;215(3):403-10
[
2231712.001
]
[Cites]
Cell. 1993 Mar 12;72(5):753-65
[
7680958.001
]
[Cites]
Genomics. 1993 Jun;16(3):768-70
[
7686884.001
]
[Cites]
Genomics. 1994 Jan 1;19(1):174-5
[
7514564.001
]
[Cites]
J Biol Chem. 1995 Feb 24;270(8):4133-7
[
7876164.001
]
[Cites]
Genomics. 1995 Jun 10;27(3):489-96
[
7558031.001
]
[Cites]
J Biol Chem. 1996 Feb 23;271(8):3979-81
[
8626728.001
]
[Cites]
Chromosome Res. 1996 Apr;4(3):218-25
[
8793207.001
]
[Cites]
Nucleic Acids Res. 1997 Sep 1;25(17):3389-402
[
9254694.001
]
[Cites]
J Biol Chem. 1997 Aug 29;272(35):22236-42
[
9268370.001
]
[Cites]
Virology. 1997 Sep 29;236(2):354-63
[
9325243.001
]
[Cites]
EMBO J. 1997 Nov 3;16(21):6355-63
[
9351818.001
]
[Cites]
Chromosome Res. 1997 Nov;5(7):433-43
[
9421259.001
]
[Cites]
Genome Res. 1998 Mar;8(3):175-85
[
9521921.001
]
[Cites]
Genome Res. 1998 Mar;8(3):186-94
[
9521922.001
]
[Cites]
Genome Res. 1998 Mar;8(3):195-202
[
9521923.001
]
[Cites]
Genomics. 1998 Sep 15;52(3):267-77
[
9790745.001
]
[Cites]
Cell Death Differ. 1998 Apr;5(4):313-20
[
10200477.001
]
[Cites]
Am J Hum Genet. 2005 Mar;76(3):449-62
[
15700229.001
]
[Cites]
Am J Hum Genet. 2005 Apr;76(4):623-33
[
15732009.001
]
[Cites]
Biochem Biophys Res Commun. 2005 Apr 22;329(4):1234-9
[
15766558.001
]
[Cites]
Mol Pharmacol. 2005 Jun;67(6):2126-36
[
15774772.001
]
[Cites]
Proteins. 2005 Jul 1;60(1):131-8
[
15849753.001
]
[Cites]
Genomics. 1999 Oct 15;61(2):145-55
[
10534400.001
]
[Cites]
Genomics. 1999 Dec 1;62(2):189-202
[
10610712.001
]
[Cites]
Trends Genet. 2000 May;16(5):198-200
[
10782110.001
]
[Cites]
Bioinformatics. 2000 Nov;16(11):1046-7
[
11159318.001
]
[Cites]
Am J Hum Genet. 2001 Apr;68(4):978-89
[
11254454.001
]
[Cites]
Hum Mol Genet. 2001 Mar 15;10(6):591-7
[
11230178.001
]
[Cites]
Genomics. 2001 Jun 15;74(3):287-98
[
11414756.001
]
[Cites]
J Biol Chem. 2002 Jul 5;277(27):24321-30
[
11986302.001
]
[Cites]
Proc Natl Acad Sci U S A. 2002 Jul 9;99(14):9322-7
[
12080145.001
]
[Cites]
Proc Natl Acad Sci U S A. 2002 Aug 20;99(17):11311-6
[
12186974.001
]
[Cites]
Cytogenet Genome Res. 2005;111(1):51-6
[
16093721.001
]
[Cites]
Oncogene. 2005 Aug 18;24(35):5492-501
[
15940267.001
]
[Cites]
J Infect Dis. 2005 Nov 15;192(10):1741-8
[
16235172.001
]
[Cites]
Nature. 2005 Dec 8;438(7069):803-19
[
16341006.001
]
[Cites]
Apoptosis. 2006 May;11(5):725-38
[
16532271.001
]
[Cites]
BMC Infect Dis. 2006;6:106
[
16824203.001
]
[Cites]
Genet Sel Evol. 2006 Sep-Oct;38(5):551-63
[
16954046.001
]
[Cites]
J Pharmacol Exp Ther. 2006 Oct;319(1):317-22
[
16809478.001
]
[Cites]
J Mol Evol. 2006 Oct;63(4):562-76
[
17024523.001
]
[Cites]
Hum Mutat. 2007 Jan;28(1):98
[
17154280.001
]
[Cites]
Genes Immun. 2003 Sep;4(6):411-9
[
12944978.001
]
(PMID = 17822564.001).
[ISSN]
1471-2164
[Journal-full-title]
BMC genomics
[ISO-abbreviation]
BMC Genomics
[Language]
ENG
[Grant]
United States / NCPDCID CDC HHS / CI / R01 CI000216; United States / NCPDCID CDC HHS / CI / CI000216
[Publication-type]
Journal Article; Research Support, U.S. Gov't, P.H.S.
[Publication-country]
England
[Chemical-registry-number]
0 / Codon, Terminator; EC 2.7.7.- / 2',5'-Oligoadenylate Synthetase; EC 3.1.- / Endoribonucleases
[Other-IDs]
NLM/ PMC2048516
5.
Vlamakis H, Aguilar C, Losick R, Kolter R:
Control of cell fate by the formation of an architecturally complex bacterial community.
Genes Dev
; 2008 Apr 1;22(7):945-53
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[Title]
Control
of cell
fate by the formation of an architecturally
complex bacterial
community.
Bacteria form architecturally
complex
communities known as biofilms in which cells are held together by an extracellular matrix.
Biofilms harbor multiple
cell
types, and it has been proposed that within biofilms individual cells follow different developmental pathways, resulting in heterogeneous populations.
We show that motile, matrix-producing, and sporulating cells localize to distinct regions within the biofilm, and that the localization and percentage of each
cell
type is dynamic throughout development of the community.
We propose that sporulation is a culminating feature of biofilm formation, and that spore formation is coupled to the formation of an architecturally
complex
community of cells.
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[Cites]
J Bacteriol. 2005 Feb;187(4):1357-68
[
15687200.001
]
[Cites]
Mol Microbiol. 2005 Feb;55(3):739-49
[
15661000.001
]
[Cites]
Curr Opin Microbiol. 2005 Apr;8(2):222-7
[
15802256.001
]
[Cites]
Genes Dev. 2005 Sep 15;19(18):2236-44
[
16166384.001
]
[Cites]
Science. 2005 Sep 23;309(5743):2010-3
[
16179466.001
]
[Cites]
Mol Microbiol. 2006 Feb;59(4):1216-28
[
16430695.001
]
[Cites]
Mol Microbiol. 2006 Feb;59(4):1229-38
[
16430696.001
]
[Cites]
Nat Rev Microbiol. 2006 Apr;4(4):259-71
[
16541134.001
]
[Cites]
J Bacteriol. 2006 Apr;188(8):3099-109
[
16585769.001
]
[Cites]
Nature. 2006 May 18;441(7091):300-2
[
16710410.001
]
[Cites]
Mol Microbiol. 2006 Aug;61(3):564-72
[
16879639.001
]
[Cites]
J Appl Microbiol. 2006 Sep;101(3):531-41
[
16907804.001
]
[Cites]
J Bacteriol. 2007 Jun;189(11):4223-33
[
17337582.001
]
[Cites]
Curr Opin Microbiol. 2007 Jun;10(3):292-6
[
17573234.001
]
[Cites]
Mol Microbiol. 2008 Jan;67(2):254-63
[
18047568.001
]
[Cites]
J Bacteriol. 2000 May;182(10):2675-9
[
10781532.001
]
[Cites]
Microbiol Mol Biol Rev. 2000 Jun;64(2):316-53
[
10839819.001
]
[Cites]
Annu Rev Microbiol. 2000;54:49-79
[
11018124.001
]
[Cites]
J Bacteriol. 2001 Apr;183(8):2696-9
[
11274134.001
]
[Cites]
Proc Natl Acad Sci U S A. 2001 Sep 25;98(20):11621-6
[
11572999.001
]
[Cites]
Curr Opin Microbiol. 2001 Dec;4(6):667-73
[
11731318.001
]
[Cites]
J Bacteriol. 2002 Jan;184(2):564-71
[
11751836.001
]
[Cites]
Annu Rev Microbiol. 2002;56:187-209
[
12142477.001
]
[Cites]
Mol Microbiol. 2002 Oct;46(2):297-304
[
12406209.001
]
[Cites]
Mol Microbiol. 2003 Apr;48(1):1-8
[
12657040.001
]
[Cites]
Science. 2003 Jul 25;301(5632):510-3
[
12817086.001
]
[Cites]
Appl Environ Microbiol. 2004 Oct;70(10):6188-96
[
15466566.001
]
[Cites]
J Virol. 1974 Dec;14(6):1343-8
[
4214946.001
]
[Cites]
Annu Rev Microbiol. 1988;42:319-38
[
3059997.001
]
[Cites]
J Bacteriol. 1989 Jun;171(6):3095-101
[
2498284.001
]
[Cites]
Cell. 1991 Feb 8;64(3):545-52
[
1846779.001
]
[Cites]
J Bacteriol. 1994 Apr;176(7):1977-84
[
8144465.001
]
[Cites]
Gene. 1996 Nov 21;180(1-2):57-61
[
8973347.001
]
[Cites]
J Bacteriol. 1999 Mar;181(5):1664-72
[
10049401.001
]
[Cites]
Mol Microbiol. 2005 Mar;55(6):1767-81
[
15752199.001
]
(PMID = 18381896.001).
[ISSN]
0890-9369
[Journal-full-title]
Genes & development
[ISO-abbreviation]
Genes Dev.
[Language]
ENG
[Grant]
United States / NIGMS NIH HHS / GM / R01 GM018568; United States / NIGMS NIH HHS / GM / GM18568; United States / NIGMS NIH HHS / GM / R37 GM018568; United States / NIGMS NIH HHS / GM / GM58213; United States / NIGMS NIH HHS / GM / R01 GM058213
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
United States
[Other-IDs]
NLM/ NIHMS49745; NLM/ PMC2279205
6.
Kurosu K, Takiguchi Y, Okada O, Yumoto N, Sakao S, Tada Y, Kasahara Y, Tanabe N, Tatsumi K, Weiden M, Rom WN, Kuriyama T:
Identification of annexin 1 as a novel autoantigen in acute exacerbation of idiopathic pulmonary fibrosis.
J Immunol
; 2008 Jul 1;181(1):756-67
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Consistent with the hypothesis that pulmonary epithelial apoptosis is the key to the acute exacerbation of idiopathic pulmonary fibrosis (IPF), we conducted serological identification of Ags by recombinant expression cloning (SEREX) analysis using type II
alveolar
cell carcinoma
(A549)
cell
lines to identify
disease
-related Abs.
Abs to annexin 1 were detected in 47 and 53% of the sera and
bronchoalveolar
lavage materials from patients with acute exacerbation of IPF.
Some identical TCR Vbeta genes were identified in sequential materials obtained at 1-3 mo in all 10 acute exacerbation IPF cases, suggesting that some infiltrating CD4-positive T cells sharing limited epitopes expand by Ag-driven stimulation during
disease
extension.
The CDR3 region of these identical TCR Vbeta genes showed high homology with the N-
terminal
portion of annexin 1, including in the HLA-DR ligand epitopes predicted by TEPITOPE analysis.
By Western blotting analysis and observation of the CD4-positive T
cell
responses in
bronchoalveolar
lavage samples, the N-
terminal
portion of annexin 1 was cleaved and found to induce marked proliferative responses of CD4-positive T cells in three patients.
Our study demonstrates that annexin 1 is an autoantigen that raises both Ab production and T
cell
response in patients with acute exacerbation of IPF, and that the N-
terminal
portion of annexin 1 plays some role in the pathogenesis of acute exacerbation in IPF patients.
[MeSH-minor]
Acute
Disease
. Aged. Antibodies / immunology. Base Sequence.
Bronchoalveolar
Lavage Fluid / immunology. DNA, Complementary / genetics. Female. Gene Expression Regulation. Humans. Male. Middle Aged. Molecular Sequence Data. Pancreatic Elastase / metabolism. Receptors,
Antigen
, T-
Cell
, alpha-beta / genetics. Recombinant Proteins / immunology
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consumer health - Pulmonary Fibrosis
.
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author profiles
.
Immune Epitope Database and Analysis Resource.
gene/protein/disease-specific - Related Immune Epitope Information
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
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(PMID = 18566442.001).
[ISSN]
0022-1767
[Journal-full-title]
Journal of immunology (Baltimore, Md. : 1950)
[ISO-abbreviation]
J. Immunol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Annexins; 0 / Antibodies; 0 / Autoantigens; 0 / DNA, Complementary; 0 / Receptors, Antigen, T-Cell, alpha-beta; 0 / Recombinant Proteins; EC 3.4.21.36 / Pancreatic Elastase
7.
Hayes DN, Monti S, Parmigiani G, Gilks CB, Naoki K, Bhattacharjee A, Socinski MA, Perou C, Meyerson M:
Gene expression profiling reveals reproducible human lung adenocarcinoma subtypes in multiple independent patient cohorts.
J Clin Oncol
; 2006 Nov 1;24(31):5079-90
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[Title]
Gene expression profiling reveals reproducible human
lung
adenocarcinoma
subtypes in multiple independent patient cohorts.
PURPOSE: Published reports suggest that DNA microarrays identify clinically meaningful subtypes
of lung adenocarcinomas
not recognizable by other routine tests.
METHODS: Three independent cohorts of patients with
lung cancer
were evaluated using a variety of DNA microarray assays.
RESULTS: Cohorts of 31, 72, and 128
adenocarcinomas
were generated for a total of 231 microarrays, each with 2,553 reliable genes.
Three
adenocarcinoma
subtypes were identified in each cohort.
These were named bronchioid, squamoid, and magnoid according to their respective correlations with gene expression patterns from histologically defined
bronchioalveolar carcinoma
, squamous
cell carcinoma
, and large-
cell carcinoma
.
Most notably, bronchioid tumors were correlated with improved survival in early-stage
disease
, whereas squamoid tumors were associated with better survival in advanced
disease
.
CONCLUSION: DNA microarray analysis
of lung adenocarcinomas
identified reproducible tumor subtypes which differ significantly in clinically important behaviors such as stage-specific survival.
[MeSH-major]
Adenocarcinoma
/ classification. Gene Expression Profiling.
Lung
Neoplasms / classification
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(PMID = 17075127.001).
[ISSN]
1527-7755
[Journal-full-title]
Journal of clinical oncology : official journal of the American Society of Clinical Oncology
[ISO-abbreviation]
J. Clin. Oncol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
United States
[Number-of-references]
58
8.
Lichtenzveig J, Scheuring C, Dodge J, Abbo S, Zhang HB:
Construction of BAC and BIBAC libraries and their applications for generation of SSR markers for genome analysis of chickpea, Cicer arietinum L.
Theor Appl Genet
; 2005 Feb;110(3):492-510
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[Title]
Construction
of BAC
and BIBAC libraries and their applications for generation of SSR markers for genome analysis of chickpea, Cicer arietinum L.
Large-insert
bacterial
artificial chromosome (
BAC
) libraries, plant-transformation-competent binary
BAC
(BIBAC) libraries, and simple sequence repeat (SSR) markers are essential for many aspects of genomics research.
We constructed
a BAC
library and a BIBAC library from the nuclear DNA of chickpea, Cicer arietinum L., cv.
The BAC
library has 14,976 clones, with an average insert size of 121 kb, and the BIBAC library consists of 23,040 clones, with an average insert size of 145 kb.
We screened
the BAC
library with eight synthetic SSR oligos, (GA)10, (GAA)7, (AT)10, (TAA)7, (TGA)7, (CA)10, (CAA)7, and (CCA)7.
Positive
BACs
were selected, subcloned, and sequenced for SSR marker development.
These results have demonstrated that
BACs
are an excellent source for SSR marker development in chickpea.
The BAC
and BIBAC libraries and new SSR markers will provide valuable resources for chickpea genomics research and breeding (the libraries and their filters are available to the public at http://hbz.tamu.edu).
[MeSH-minor]
Base Sequence. Chromosomes, Artificial,
Bacterial
. Cloning, Molecular. DNA Primers. Minisatellite Repeats / genetics. Molecular Sequence Data. Oligonucleotides. Sequence Analysis, DNA
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[Cites]
Theor Appl Genet. 2004 Sep;109(5):1041-50
[
15164176.001
]
[Cites]
Proc Natl Acad Sci U S A. 1999 May 25;96(11):6535-40
[
10339623.001
]
[Cites]
Plant Cell Rep. 1996 Nov;16(1-2):32-7
[
24178649.001
]
[Cites]
Genetics. 2001 Aug;158(4):1711-24
[
11514457.001
]
[Cites]
Gene. 2002 Jan 9;282(1-2):247-55
[
11814697.001
]
[Cites]
Theor Appl Genet. 1992 Mar;83(5):620-7
[
24202680.001
]
[Cites]
Theor Appl Genet. 2002 Sep;105(4):604-607
[
12582510.001
]
[Cites]
Mol Gen Genet. 1999 Mar;261(2):354-63
[
10102371.001
]
[Cites]
Genome Res. 2004 Feb;14(2):319-26
[
14718376.001
]
[Cites]
Mol Gen Genet. 1999 Aug;262(1):90-101
[
10503540.001
]
[Cites]
Genome Res. 2003 Dec;13(12):2754-8
[
14656976.001
]
[Cites]
Nucleic Acids Res. 1996 Apr 15;24(8):1574-5
[
8628694.001
]
[Cites]
Biotechniques. 1996 May;20(5):758-60
[
8723911.001
]
[Cites]
Theor Appl Genet. 2003 May;106(8):1447-56
[
12750788.001
]
[Cites]
Theor Appl Genet. 1995 Jan;90(1):90-6
[
24173788.001
]
[Cites]
Theor Appl Genet. 2002 Nov;105(6-7):847-854
[
12582909.001
]
[Cites]
Genetics. 2001 Nov;159(3):1231-42
[
11729165.001
]
[Cites]
Nucleic Acids Res. 1984 May 25;12(10):4127-38
[
6328411.001
]
[Cites]
Genomics. 2004 Dec;84(6):941-51
[
15533711.001
]
[Cites]
Nucleic Acids Res. 1998 Nov 1;26(21):4901-9
[
9776751.001
]
[Cites]
Theor Appl Genet. 2004 Feb;108(4):663-9
[
14564396.001
]
[Cites]
Genetics. 1998 Aug;149(4):2007-23
[
9691054.001
]
[Cites]
Theor Appl Genet. 2003 May;106(7):1196-202
[
12748770.001
]
[Cites]
Proc Natl Acad Sci U S A. 1996 Sep 3;93(18):9975-9
[
8790442.001
]
[Cites]
Genome. 1999 Apr;42(2):210-7
[
10231957.001
]
[Cites]
Gene. 2003 Dec 4;321:113-21
[
14636998.001
]
[Cites]
Genome. 2000 Dec;43(6):988-1002
[
11195353.001
]
[Cites]
Plant Cell. 2002 Mar;14(3):537-45
[
11910002.001
]
[Cites]
Theor Appl Genet. 1993 May;86(4):417-26
[
24193588.001
]
(PMID = 15712010.001).
[ISSN]
0040-5752
[Journal-full-title]
TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik
[ISO-abbreviation]
Theor. Appl. Genet.
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Germany
[Chemical-registry-number]
0 / DNA Primers; 0 / Genetic Markers; 0 / Oligonucleotides
9.
Künzle F, Gerber V, Van Der Haegen A, Wampfler B, Straub R, Marti E:
IgE-bearing cells in bronchoalveolar lavage fluid and allergen-specific IgE levels in sera from RAO-affected horses.
J Vet Med A Physiol Pathol Clin Med
; 2007 Feb;54(1):40-7
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[Title]
IgE-bearing cells in
bronchoalveolar
lavage fluid and allergen-specific IgE levels in sera from RAO-affected horses.
We hypothesized that the number of cells with receptor-bound IgE in
bronchoalveolar
lavage fluid (BALF) and IgE levels in serum would be higher in RAO-affected than in healthy horses living in the same environment.
Age had no significant effect on BALF
cell
ratios or on specific serum IgE levels.
[MeSH-major]
Bronchoalveolar
Lavage Fluid / immunology. Horse Diseases / immunology. Immunoglobulin E / analysis.
Lung
Diseases, Obstructive / veterinary. Mitosporic Fungi / immunology
[MeSH-minor]
Allergens / immunology. Animals. Female. Horses.
Lung
Diseases, Fungal / immunology.
Lung
Diseases, Fungal / veterinary. Male. Mast Cells / immunology
SciCrunch.
OMIA - Online Mendelian Inheritance in Animals: Data: Gene Expression
.
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(PMID = 17359454.001).
[ISSN]
0931-184X
[Journal-full-title]
Journal of veterinary medicine. A, Physiology, pathology, clinical medicine
[ISO-abbreviation]
J Vet Med A Physiol Pathol Clin Med
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Germany
[Chemical-registry-number]
0 / Allergens; 37341-29-0 / Immunoglobulin E
10.
Halder S, Murakami M, Verma SC, Kumar P, Yi F, Robertson ES:
Early events associated with infection of Epstein-Barr virus infection of primary B-cells.
PLoS One
; 2009 Sep 28;4(9):e7214
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Epstein Barr virus (EBV) is closely associated with the development
of a
vast number of human cancers.
To develop a system for monitoring early cellular and viral events associated with EBV infection a self-recombining
BAC
containing 172-kb of the Epstein Barr virus genome
BAC
-EBV designated as MD1
BAC
(Chen et al., 2005, J.Virology) was used to introduce an expression cassette of green fluorescent protein (GFP) by homologous recombination, and the resultant
BAC
clone,
BAC
-GFP-EBV was transfected into the HEK 293T epithelial
cell
line.
The resulting recombinant GFP EBV was induced to produce progeny virus by chemical inducer from the stable HEK 293T
BAC
GFP EBV
cell
line and the virus was used to immortalize human primary B-
cell
as monitored by green fluorescence and outgrowth of the primary B cells.
The infection, B-
cell
activation and
cell
proliferation due to GFP EBV was monitored by the expression of the B-
cell
surface antigens CD5, CD10, CD19, CD23, CD39, CD40 , CD44 and the intercellular proliferation marker Ki-67 using Flow cytometry.
This up-regulation of lytic gene expression prior to latent genes during early infection strongly suggests that EBV infects primary B-
cell
with an initial burst of lytic gene expression and the resulting progeny virus is competent for infecting new primary B-cells.
The newly infected primary B-cells can be further analyzed for investigating B
cell
activation due to EBV infection.
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[Cites]
J Virol. 2004 Jul;78(13):7004-15
[
15194777.001
]
[Cites]
Science. 1967 Sep 1;157(3792):1064-5
[
6036237.001
]
[Cites]
Int J Cancer. 1968 Nov 15;3(6):857-66
[
4894385.001
]
[Cites]
J Reticuloendothel Soc. 1972 Apr;11(4):383-93
[
4112527.001
]
[Cites]
Int J Cancer. 1974 Dec 15;14(6):704-15
[
4377001.001
]
[Cites]
Int J Cancer. 1975 Dec 15;16(6):1008-14
[
172457.001
]
[Cites]
Int J Cancer. 1977 Jun 15;19(6):775-82
[
194845.001
]
[Cites]
Nature. 1978 Apr 13;272(5654):583-5
[
205792.001
]
[Cites]
J Virol. 1978 Oct;28(1):344-51
[
81320.001
]
[Cites]
J Virol. 1980 May;34(2):560-8
[
6246281.001
]
[Cites]
Nature. 1980 May 29;285(5763):333-5
[
6246453.001
]
[Cites]
J Virol. 1993 Jun;67(6):3182-90
[
8388496.001
]
[Cites]
Intervirology. 1993;35(1-4):26-39
[
8407248.001
]
[Cites]
Proc Natl Acad Sci U S A. 1993 Oct 1;90(19):9150-4
[
8415670.001
]
[Cites]
Science. 1994 Feb 11;263(5148):802-5
[
8303295.001
]
[Cites]
J Gen Virol. 1994 Jan;75 ( Pt 1):1-13
[
8113717.001
]
[Cites]
J Gen Virol. 1994 Apr;75 ( Pt 4):769-78
[
7512118.001
]
[Cites]
Proc Natl Acad Sci U S A. 1995 Jun 20;92(13):5875-9
[
7597045.001
]
[Cites]
Photochem Photobiol. 1995 Oct;62(4):651-6
[
7480149.001
]
[Cites]
Immunology. 1995 Sep;86(1):41-8
[
7590880.001
]
[Cites]
J Virol. 1996 Mar;70(3):2049-54
[
8627735.001
]
[Cites]
Science. 1996 Dec 13;274(5294):1906-9
[
8943203.001
]
[Cites]
Curr Opin Immunol. 1997 Jun;9(3):330-7
[
9203418.001
]
[Cites]
EMBO J. 1998 Mar 16;17(6):1700-9
[
9501091.001
]
[Cites]
Proc Natl Acad Sci U S A. 1998 Jul 7;95(14):8245-50
[
9653172.001
]
[Cites]
Immunol Lett. 1999 May 3;68(1):147-54
[
10397170.001
]
[Cites]
Arch Virol. 1999;144(6):1123-37
[
10446648.001
]
[Cites]
J Pathol. 1999 Sep;189(1):34-9
[
10451485.001
]
[Cites]
J Virol. 2005 Apr;79(7):4506-9
[
15767450.001
]
[Cites]
J Gen Virol. 1987 Mar;68 ( Pt 3):805-11
[
3029307.001
]
[Cites]
Int J Cancer. 1987 Apr 15;39(4):472-6
[
3030940.001
]
[Cites]
Cell. 1987 Jul 17;50(2):203-13
[
3036369.001
]
[Cites]
Proc Natl Acad Sci U S A. 1987 Nov;84(22):8060-4
[
2825176.001
]
[Cites]
Blood. 1988 Jan;71(1):13-29
[
3257143.001
]
[Cites]
N Engl J Med. 1988 Mar 24;318(12):733-41
[
2831453.001
]
[Cites]
J Virol. 1988 Nov;62(11):4173-84
[
2845129.001
]
[Cites]
J Virol. 1989 Apr;63(4):1729-36
[
2538653.001
]
[Cites]
EMBO J. 1989 Jan;8(1):127-32
[
2540954.001
]
[Cites]
Cell. 1980 Nov;22(1 Pt 1):243-55
[
6253078.001
]
[Cites]
Proc Natl Acad Sci U S A. 1980 Sep;77(9):5163-6
[
6254061.001
]
[Cites]
Nature. 1981 Dec 3;294(5840):458-60
[
6273741.001
]
[Cites]
Int J Cancer. 1983 Jan 15;31(1):13-20
[
6339421.001
]
[Cites]
J Immunol. 1983 Jul;131(1):244-50
[
6408173.001
]
[Cites]
J Immunol. 1984 Dec;133(6):3408-14
[
6436381.001
]
[Cites]
Hematol Oncol. 1984 Oct-Dec;2(4):365-71
[
6396192.001
]
[Cites]
J Immunol. 1985 May;134(5):3007-12
[
2984280.001
]
[Cites]
J Virol. 1985 Aug;55(2):347-51
[
2410629.001
]
[Cites]
J Virol. 1985 Sep;55(3):710-20
[
2991591.001
]
[Cites]
J Virol. 1987 Feb;61(2):499-508
[
3027378.001
]
[Cites]
J Virol. 1990 Mar;64(3):1227-32
[
2154606.001
]
[Cites]
J Virol. 1990 May;64(5):2309-18
[
2157887.001
]
[Cites]
J Virol. 1990 Sep;64(9):4549-52
[
2166830.001
]
[Cites]
Virology. 1991 Apr;181(2):595-608
[
1849678.001
]
[Cites]
J Virol. 1991 May;65(5):2728-31
[
1850046.001
]
[Cites]
Annu Rev Immunol. 1991;9:97-127
[
1910693.001
]
[Cites]
Int J Cancer. 1993 Jan 2;53(1):153-60
[
8416201.001
]
[Cites]
J Virol. 1993 Apr;67(4):2014-25
[
8445720.001
]
[Cites]
J Virol. 2006 Mar;80(5):2548-65
[
16474161.001
]
[Cites]
Cell Host Microbe. 2007 Aug 16;2(2):106-18
[
18005725.001
]
[Cites]
Curr Protoc Microbiol. 2007 Aug;Chapter 14:Unit 14E.2
[
18770612.001
]
[Cites]
Microbes Infect. 2009 Mar;11(3):429-33
[
19397878.001
]
[Cites]
Cell. 1981 Jul;25(1):227-32
[
6268303.001
]
[Cites]
Mol Pathol. 1999 Dec;52(6):307-22
[
10748864.001
]
[Cites]
Proc Natl Acad Sci U S A. 2000 Apr 25;97(9):4873-8
[
10781094.001
]
[Cites]
Trends Genet. 2000 Jun;16(6):254-9
[
10827452.001
]
[Cites]
Immunology. 2000 Oct;101(2):201-9
[
11012773.001
]
[Cites]
Curr Top Microbiol Immunol. 2001;258:141-51
[
11443859.001
]
[Cites]
Proc Natl Acad Sci U S A. 2001 Nov 20;98(24):13850-3
[
11707593.001
]
[Cites]
Methods. 2001 Dec;25(4):402-8
[
11846609.001
]
[Cites]
Proc Natl Acad Sci U S A. 2002 Nov 12;99(23):15036-41
[
12409611.001
]
[Cites]
Proc Natl Acad Sci U S A. 2003 Jun 24;100(13):7836-40
[
12805559.001
]
[Cites]
Oncogene. 2003 Aug 11;22(33):5108-21
[
12910248.001
]
[Cites]
Nat Rev Immunol. 2003 Oct;3(10):801-12
[
14523386.001
]
[Cites]
Proc Natl Acad Sci U S A. 2004 Jan 6;101(1):239-44
[
14688409.001
]
[Cites]
Clin Cancer Res. 2004 Feb 1;10(3):803-21
[
14871955.001
]
(PMID = 19784370.001).
[ISSN]
1932-6203
[Journal-full-title]
PloS one
[ISO-abbreviation]
PLoS ONE
[Language]
ENG
[Grant]
United States / NCI NIH HHS / CA / 1R01CA137894-01; United States / NCI NIH HHS / CA / K99 CA126182-02; United States / NCI NIH HHS / CA / K99 CA126182; United States / NCI NIH HHS / CA / 5R01CA108461-05; United States / NCI NIH HHS / CA / CA126182-02; United States / NCI NIH HHS / CA / R01 CA108461; United States / NCI NIH HHS / CA / R01 CA137894
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
United States
[Chemical-registry-number]
0 / Antigens, Surface; 0 / DNA Primers; 147336-22-9 / Green Fluorescent Proteins
[Other-IDs]
NLM/ PMC2746279
11.
Greulich-Bode KM, Wang M, Rhein AP, Weier JF, Weier HU:
Validation of DNA probes for molecular cytogenetics by mapping onto immobilized circular DNA.
Mol Cytogenet
; 2008 Dec 23;1:28
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Large insert recombinant DNA clones such as
bacterial
artificial chromosome (
BAC
) or P1/PAC clones have established themselves in recent years as preferred starting material for probe preparations due to their low rates of chimerism and ease of use.
The method takes advantage of the fact that P1/PAC/
BAC
's can be isolated as circular DNA molecules, stretched out on glass slides and fine-mapped by multicolor hybridization with smaller probe molecules.
Two examples demonstrate the application of this technique: mapping
of a
gene-specific ~6 kb plasmid onto an unusually small, ~55 kb circular P1 molecule and the determination of the extent of overlap between P1 molecules homologous to the human NF-kappaB2 locus.
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[Cites]
Genome. 2006 Sep;49(9):1057-68
[
17110986.001
]
[Cites]
Nucleic Acids Res. 2000 Apr 15;28(8):E30
[
10734207.001
]
[Cites]
Genes Dev. 2006 Jan 15;20(2):159-73
[
16384935.001
]
[Cites]
Cytogenet Genome Res. 2005;109(1-3):79-82
[
15753562.001
]
[Cites]
Cytometry A. 2005 Feb;63(2):114-7
[
15651009.001
]
[Cites]
Genomics. 1997 Nov 1;45(3):479-86
[
9367672.001
]
[Cites]
Cytogenet Cell Genet. 1997;79(1-2):64-70
[
9533015.001
]
[Cites]
Chromosome Res. 1998 Aug;6(5):415-8
[
9872672.001
]
[Cites]
Nat Genet. 1997 May;16(1):28-36
[
9140392.001
]
[Cites]
Genome Res. 1996 May;6(5):414-30
[
8743991.001
]
[Cites]
Hum Mol Genet. 1995 Oct;4(10):1903-10
[
8595414.001
]
[Cites]
Hum Mol Genet. 1995 May;4(5):831-6
[
7633442.001
]
[Cites]
Genomics. 1995 Mar 20;26(2):390-3
[
7601468.001
]
[Cites]
Hum Genet. 1994 Mar;93(3):229-35
[
8125473.001
]
[Cites]
Science. 1994 Sep 30;265(5181):2096-8
[
7522347.001
]
[Cites]
Methods Mol Biol. 1994;33:109-22
[
7894573.001
]
[Cites]
Biotechniques. 1994 Nov;17(5):928-9, 932-3
[
7840975.001
]
[Cites]
Proc Natl Acad Sci U S A. 1994 Jun 7;91(12):5513-7
[
8202519.001
]
[Cites]
Proc Natl Acad Sci U S A. 1994 Mar 29;91(7):2629-33
[
8146166.001
]
[Cites]
Nat Genet. 1993 Sep;5(1):17-21
[
8106079.001
]
[Cites]
DNA Cell Biol. 1992 Sep;11(7):523-37
[
1388725.001
]
[Cites]
Mol Cell Biol. 1992 Feb;12(2):685-95
[
1531086.001
]
[Cites]
Cell. 1991 Dec 20;67(6):1075-87
[
1760839.001
]
[Cites]
Proc Natl Acad Sci U S A. 1992 Oct 15;89(20):9509-13
[
1384055.001
]
[Cites]
Nat Genet. 1992 Nov;2(3):171-2
[
1345163.001
]
[Cites]
Hum Mol Genet. 1992 Nov;1(8):587-91
[
1301167.001
]
[Cites]
Genomics. 1992 Oct;14(2):536-41
[
1427876.001
]
[Cites]
Genomics. 1992 May;13(1):122-8
[
1577477.001
]
[Cites]
Proc Natl Acad Sci U S A. 1992 Mar 15;89(6):2056-60
[
1549564.001
]
[Cites]
Am J Hum Genet. 1984 Nov;36(6):1159-71
[
6097109.001
]
[Cites]
Nucleic Acids Res. 1979 Nov 24;7(6):1513-23
[
388356.001
]
[Cites]
Am J Hum Genet. 2003 Dec;73(6):1261-70
[
14628292.001
]
[Cites]
J Histochem Cytochem. 2003 Oct;51(10):1249-53
[
14500692.001
]
[Cites]
J Histochem Cytochem. 2001 Aug;49(8):939-48
[
11457922.001
]
[Cites]
Methods Mol Biol. 2006;338:31-57
[
16888349.001
]
(PMID = 19108707.001).
[ISSN]
1755-8166
[Journal-full-title]
Molecular cytogenetics
[ISO-abbreviation]
Mol Cytogenet
[Language]
ENG
[Grant]
United States / NCI NIH HHS / CA / CA123370-01A1; United States / NICHD NIH HHS / HD / R44 HD044313-01; United States / NICHD NIH HHS / HD / HD044313-01; United States / NCI NIH HHS / CA / R33 CA088258; United States / NCI NIH HHS / CA / R21 CA123370; United States / NCI NIH HHS / CA / CA088258-03; United States / NCI NIH HHS / CA / R33 CA088258-03; United States / NCI NIH HHS / CA / R21 CA123370-01A1; United States / NICHD NIH HHS / HD / R44 HD044313
[Publication-type]
Journal Article
[Publication-country]
England
[Other-IDs]
NLM/ PMC2630919
12.
Lang M, Miyake T, Braasch I, Tinnemore D, Siegel N, Salzburger W, Amemiya CT, Meyer A:
A BAC library of the East African haplochromine cichlid fish Astatotilapia burtoni.
J Exp Zool B Mol Dev Evol
; 2006 Jan 15;306(1):35-44
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[Title]
A BAC
library of the East African haplochromine cichlid fish Astatotilapia burtoni.
A BAC
library was constructed from Astatotilapia burtoni, a haplochromine cichlid that is found in Lake Tanganyika, East Africa, and its surrounding rivers.
The BAC
library described here is expected to be useful to the scientific community interested in cichlid genomics as an important resource to gain new insights into the rapid evolution of the great species diversity of haplochromine cichlid fishes.
[MeSH-major]
Chromosomes, Artificial,
Bacterial
. Cichlids / genetics. Gene Library. Phylogeny
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[Copyright]
(c) 2005 Wiley-Liss, Inc.
(PMID = 16254984.001).
[ISSN]
1552-5007
[Journal-full-title]
Journal of experimental zoology. Part B, Molecular and developmental evolution
[ISO-abbreviation]
J. Exp. Zool. B Mol. Dev. Evol.
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
[Publication-country]
United States
[Chemical-registry-number]
0 / DNA Primers
13.
Yang TJ, Kim JS, Lim KB, Kwon SJ, Kim JA, Jin M, Park JY, Lim MH, Kim HI, Kim SH, Lim YP, Park BS:
The Korea brassica genome project: a glimpse of the brassica genome based on comparative genome analysis with Arabidopsis.
Comp Funct Genomics
; 2005;6(3):138-46
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We have selected 48 seed
BACs
on chromosome 1 using EST genetic markers and FISH analyses.
Among them, 30
BAC
clones have been sequenced and 18 are on the way.
Comparative genome analyses of the EST sequences and sequenced
BAC
clones from Brassica chromosome 1 revealed their homeologous partner regions on the Arabidopsis genome and a syntenic comparative map between Brassica chromosome 1 and Arabidopsis chromosomes.
In silico chromosome walking and clone validation have been successfully applied to extending sequence contigs based on the comparative map and
BAC
end sequences.
In-depth sequence analyses of five homeologous
BAC
clones and an Arabidopsis chromosomal region reveal overall co-linearity, with 82% sequence similarity.
In 2005 we intend to construct an integrated physical map, including sequence information from 500
BAC
clones and integration of fingerprinting data and end sequence data of more than 100,000
BAC
clones.
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[Cites]
Genome Res. 1998 Mar;8(3):175-85
[
9521921.001
]
[Cites]
Genome Res. 1998 Mar;8(3):186-94
[
9521922.001
]
[Cites]
Genetics. 1994 Oct;138(2):499-510
[
7828831.001
]
[Cites]
Genetics. 2003 May;164(1):359-72
[
12750346.001
]
[Cites]
Nature. 2003 Mar 27;422(6930):433-8
[
12660784.001
]
[Cites]
Genome. 2001 Oct;44(5):911-8
[
11681616.001
]
[Cites]
Genome Biol. 2001;2(3):REVIEWS1011
[
11276431.001
]
[Cites]
Plant Cell. 2000 Sep;12(9):1523-40
[
11006329.001
]
[Cites]
Nature. 2000 Dec 14;408(6814):796-815
[
11130711.001
]
[Cites]
Plant J. 2000 Jul;23(2):233-43
[
10929117.001
]
[Cites]
Genome Res. 2000 Apr;10(4):577-86
[
10779500.001
]
[Cites]
Comp Funct Genomics. 2004;5(3):276-80
[
18629157.001
]
[Cites]
Plant J. 2004 Dec;40(5):725-33
[
15546355.001
]
[Cites]
Theor Appl Genet. 2004 Nov;109(7):1346-52
[
15365626.001
]
[Cites]
Ann Bot. 2005 Jan;95(1):229-35
[
15596470.001
]
[Cites]
Genome Res. 1998 Mar;8(3):195-202
[
9521923.001
]
[Cites]
Plant J. 1996 Jan;9(1):13-20
[
8580970.001
]
(PMID = 18629219.001).
[ISSN]
1531-6912
[Journal-full-title]
Comparative and functional genomics
[ISO-abbreviation]
Comp. Funct. Genomics
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Egypt
[Other-IDs]
NLM/ PMC2447515
14.
Mack DG, Lanham AM, Palmer BE, Maier LA, Fontenot AP:
CD27 expression on CD4+ T cells differentiates effector from regulatory T cell subsets in the lung.
J Immunol
; 2009 Jun 1;182(11):7317-24
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[Title]
CD27 expression on CD4+ T cells differentiates effector from regulatory T
cell
subsets in
the lung
.
Beryllium exposure in the workplace can result in chronic beryllium
disease
, a granulomatous
lung disorder
characterized by CD4(+) T
cell
alveolitis and progressive
lung
fibrosis.
A large number of the CD4(+) T cells recruited to
the lung
in chronic beryllium
disease
recognize beryllium in an Ag-specific manner and express Th1-type cytokines following T
cell
activation.
Beryllium-responsive CD4(+) T cells in
the bronchoalveolar
lavage (BAL) express an effector memory T
cell
phenotype and recognize beryllium in a CD28-independent manner.
In addition, loss of CD27 on BAL CD4(+) T cells inversely correlates with markers
of lung
inflammation.
A small population of BAL CD4(+) T cells retains CD27 expression, and these CD4(+)CD27(+) T cells contain the FoxP3-expressing, naturally occurring regulatory T (T(reg))
cell
subset.
Taken together, these findings suggest that CD27 is differentially expressed between effector T cells from the inflamed
lung
and can be used in conjunction with CD25 to isolate T(reg) cells and assess their functional capacity in an ongoing adaptive immune response in a target organ.
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BERYLLIUM, ELEMENTAL
.
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[Cites]
J Immunol. 1993 Sep 1;151(5):2426-35
[
7689607.001
]
[Cites]
J Allergy Clin Immunol. 1991 Jul;88(1):54-60
[
2071785.001
]
[Cites]
J Immunol. 1995 Mar 15;154(6):2612-23
[
7876536.001
]
[Cites]
J Immunol. 1995 Aug 1;155(3):1151-64
[
7636184.001
]
[Cites]
Environ Health Perspect. 1996 Oct;104 Suppl 5:953-6
[
8933041.001
]
[Cites]
J Immunol. 1997 Jan 1;158(1):518-26
[
8977230.001
]
[Cites]
J Exp Med. 1997 Nov 3;186(9):1407-18
[
9348298.001
]
[Cites]
Am J Respir Cell Mol Biol. 1998 Apr;18(4):581-9
[
9533947.001
]
[Cites]
J Exp Med. 1998 Jul 20;188(2):287-96
[
9670041.001
]
[Cites]
J Immunol. 1999 Jul 15;163(2):1019-26
[
10395700.001
]
[Cites]
J Immunol. 1999 Aug 1;163(3):1647-53
[
10415070.001
]
[Cites]
Annu Rev Immunol. 2005;23:23-68
[
15771565.001
]
[Cites]
Nat Immunol. 2005 Apr;6(4):345-52
[
15785760.001
]
[Cites]
J Exp Med. 2005 Apr 4;201(7):1061-7
[
15809351.001
]
[Cites]
J Exp Med. 2005 Jun 6;201(11):1793-803
[
15939793.001
]
[Cites]
J Clin Invest. 2005 Oct;115(10):2886-93
[
16151531.001
]
[Cites]
J Immunol. 2005 Nov 15;175(10):6489-97
[
16272303.001
]
[Cites]
J Exp Med. 2005 Nov 21;202(10):1433-42
[
16301748.001
]
[Cites]
J Exp Med. 2006 Jul 10;203(7):1701-11
[
16818678.001
]
[Cites]
Immunology. 2007 May;121(1):129-39
[
17425604.001
]
[Cites]
J Immunol. 2008 Feb 15;180(4):2704-12
[
18250483.001
]
[Cites]
J Immunol. 2008 Sep 15;181(6):4381-8
[
18768897.001
]
[Cites]
Annu Rev Immunol. 2000;18:423-49
[
10837065.001
]
[Cites]
Cell. 2000 May 26;101(5):455-8
[
10850488.001
]
[Cites]
Nat Genet. 2001 Jan;27(1):68-73
[
11138001.001
]
[Cites]
J Immunol. 2001 Jan 15;166(2):877-84
[
11145663.001
]
[Cites]
Nat Immunol. 2000 Nov;1(5):433-40
[
11062504.001
]
[Cites]
J Immunol. 2001 Aug 1;167(3):1245-53
[
11466340.001
]
[Cites]
Am J Respir Crit Care Med. 2002 Mar 15;165(6):788-94
[
11897645.001
]
[Cites]
J Clin Invest. 2002 Nov;110(10):1473-82
[
12438445.001
]
[Cites]
Nat Immunol. 2003 Apr;4(4):330-6
[
12612578.001
]
[Cites]
J Clin Invest. 2003 Sep;112(5):776-84
[
12952926.001
]
[Cites]
J Exp Med. 2003 Nov 3;198(9):1369-80
[
14581610.001
]
[Cites]
J Immunol. 2003 Dec 15;171(12):6910-8
[
14662898.001
]
[Cites]
J Exp Med. 1985 Jan 1;161(1):72-87
[
3871469.001
]
[Cites]
J Immunol. 1987 Sep 1;139(5):1589-96
[
2442250.001
]
[Cites]
Ann Intern Med. 1988 May;108(5):687-93
[
3282464.001
]
[Cites]
Am Rev Respir Dis. 1989 Jun;139(6):1479-86
[
2729754.001
]
[Cites]
Science. 1993 Oct 8;262(5131):242-4
[
8105536.001
]
(PMID = 19454729.001).
[ISSN]
1550-6606
[Journal-full-title]
Journal of immunology (Baltimore, Md. : 1950)
[ISO-abbreviation]
J. Immunol.
[Language]
ENG
[Grant]
United States / NIEHS NIH HHS / ES / P01 ES011810; United States / NIEHS NIH HHS / ES / ES011810; United States / NHLBI NIH HHS / HL / HL62410; United States / NHLBI NIH HHS / HL / K24 HL102245; United States / NIAID NIH HHS / AI / AI050864; United States / NIAID NIH HHS / AI / U19 AI050864; United States / NHLBI NIH HHS / HL / R01 HL062410; United States / NCRR NIH HHS / RR / M01 RR000051; United States / NCRR NIH HHS / RR / M01-RR00051; United States / NCRR NIH HHS / RR / M01 RR000051-46
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
United States
[Chemical-registry-number]
0 / Antigens, CD27; OW5102UV6N / Beryllium
[Other-IDs]
NLM/ NIHMS273500; NLM/ PMC3061566
15.
Dekio I, Sakamoto M, Hayashi H, Amagai M, Suematsu M, Benno Y:
Characterization of skin microbiota in patients with atopic dermatitis and in normal subjects using 16S rRNA gene-based comprehensive analysis.
J Med Microbiol
; 2007 Dec;56(Pt 12):1675-83
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A previous study using
bacterial
16S rRNA gene-based clone libraries revealed that the microbiota in healthy human skin included uncultured micro-organisms, although the micro-organisms in skin exposed to
disease
conditions remain to be examined.
To compare the profiles of skin microbiota in 13 patients with atopic dermatitis (
AD
) and 10 healthy controls,
terminal
RFLP analysis
of bacterial
16S rRNA genes was applied to 23 swab-scrubbed samples from facial skin.
This culture-independent analysis successfully revealed
the complex bacterial
members of the microbiota as peak patterns following capillary electrophoresis
of terminal
restriction fragments (T-RFs).
Each T-RF peak reflected a micro-organism, and the micro-organism to which each peak was assigned could be identified by computer simulation of T-RF length using the nucleotide sequence data
of bacterial
species residing in the skin.
Among 18 species detected in the study, Stenotrophomonas maltophilia was detected significantly more commonly in
AD
patients (5/13 for
AD
patients vs 0/10 for controls), whilst Dietzia maris was detected significantly more commonly in normal controls (8/10 for controls vs 2/13 for
AD
patients).
Although further studies should be undertaken to investigate the roles of these micro-organisms in
AD
, the microbiota were presumed to include hitherto uninvestigated
bacterial
species in the major population of patients with
AD
and of healthy controls.
[MeSH-minor]
Adult. DNA,
Bacterial
/ genetics. DNA,
Bacterial
/ isolation & purification. Female. Humans. Male. Polymorphism, Restriction Fragment Length. RNA,
Bacterial
/ analysis. RNA,
Bacterial
/ genetics
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(PMID = 18033838.001).
[ISSN]
0022-2615
[Journal-full-title]
Journal of medical microbiology
[ISO-abbreviation]
J. Med. Microbiol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / DNA, Bacterial; 0 / RNA, Bacterial; 0 / RNA, Ribosomal, 16S
16.
Chen B, Tong Z, Ye Q, Nakamura S, Costabel U, Guzman J:
Expression of tumour necrosis factor receptors by bronchoalveolar cells in hypersensitivity pneumonitis.
Eur Respir J
; 2005 Jun;25(6):1039-43
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[Title]
Expression of tumour necrosis factor receptors by
bronchoalveolar
cells in hypersensitivity pneumonitis.
Tumour necrosis factor receptors (TNFR) and the Fas receptor (FasR) have been implicated in the pathogenesis of interstitial
lung
diseases.
The current authors examined the expression of TNFR-1, TNFR-2 and FasR by
bronchoalveolar
cells in hypersensitivity pneumonitis (HP).
Cell
surface receptor expression on
bronchoalveolar
lavage cells was analysed by immunocytochemistry in 11 HP patients, 11 idiopathic pulmonary fibrosis (IPF) patients and 10 controls.
TNFR-1, TNFR-2 and FasR were expressed on a higher percentage of
alveolar
macrophages (AM) in HP compared with controls and IPF patients.
[MeSH-minor]
Aged. Antigens, CD95 / metabolism.
Bronchoalveolar
Lavage Fluid / cytology. Female. Humans. Male. Middle Aged. Pulmonary Fibrosis / metabolism. Pulmonary Fibrosis / pathology. Receptors, Tumor Necrosis Factor, Type I / metabolism. Receptors, Tumor Necrosis Factor, Type II / metabolism
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(PMID = 15929959.001).
[ISSN]
0903-1936
[Journal-full-title]
The European respiratory journal
[ISO-abbreviation]
Eur. Respir. J.
[Language]
eng
[Publication-type]
Clinical Trial; Comparative Study; Controlled Clinical Trial; Journal Article
[Publication-country]
Denmark
[Chemical-registry-number]
0 / Antigens, CD95; 0 / Receptors, Tumor Necrosis Factor; 0 / Receptors, Tumor Necrosis Factor, Type I; 0 / Receptors, Tumor Necrosis Factor, Type II
17.
Yu J, Müller H, Hehn S, Koschmieder S, Schönig K, Berdel WE, Serve H, Müller-Tidow C:
Construction and application of an inducible system for homogenous expression levels in bulk cell lines.
PLoS One
; 2009;4(7):e6445
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[Title]
Construction and application of an inducible system for homogenous expression levels in bulk
cell
lines.
Here, we describe a system fusing Tet (tetracycline)-inducible elements,
BAC
(
bacterial
artificial chromosome) and Gateway technology together to allow tight control of gene expression in
BAC
-transfected eukaryotic bulk
cell
cultures.
Recombinase cloning into the shuttle vector and
the BAC
facilitates vector construction.
An EGFP (enhanced green fluorescent protein) allows FACS (fluorescence activated
cell
sorting) and
the BAC
technology ensures tight control of gene expression that is independent of the integrating site.
Tested by luciferase assay and western blotting, in HTB56
lung cancer
cells the final
BAC
E11-IGR-beta-catenin-ERalpha vector demonstrated sensitive inducibility by Tet or Dox (doxycycline) in a dose-dependent manner with low background, and the EGFP was an effective selection marker by FACS in bulk culture HTB56 and myeloblastic 32D cells.
This is a highly efficient tool for the rapid generation of stringently controlled Tet-inducible systems in
cell
lines.
[MeSH-minor]
Blotting, Western.
Cell
Line. Chromosomes, Artificial,
Bacterial
. Flow Cytometry. Green Fluorescent Proteins / genetics
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[Cites]
Proc Natl Acad Sci U S A. 2000 Jul 5;97(14):7963-8
[
10859354.001
]
[Cites]
J Virol. 2005 Jun;79(11):6772-80
[
15890916.001
]
[Cites]
Mol Ther. 2000 Dec;2(6):579-87
[
11124058.001
]
[Cites]
Anal Biochem. 2001 Apr 1;291(1):142-8
[
11262167.001
]
[Cites]
Transgenic Res. 2001 Apr;10(2):83-103
[
11305364.001
]
[Cites]
Nat Biotechnol. 2001 Jun;19(6):582-5
[
11385466.001
]
[Cites]
Hum Gene Ther. 2002 Jan 20;13(2):335-42
[
11812288.001
]
[Cites]
Gene Ther. 2002 Nov;9(21):1415-21
[
12378403.001
]
[Cites]
Gene Ther. 2003 Mar;10(6):459-66
[
12621450.001
]
[Cites]
Hum Gene Ther. 2003 May 20;14(8):749-61
[
12804138.001
]
[Cites]
Hum Gene Ther. 2003 Sep 1;14(13):1265-77
[
12952598.001
]
[Cites]
Gastroenterology. 2004 Jan;126(1):278-89
[
14699506.001
]
[Cites]
Mol Ther. 2004 Mar;9(3):410-8
[
15006608.001
]
[Cites]
Gene Ther. 2004 Jul;11(13):1057-67
[
15152187.001
]
[Cites]
Annu Rev Biochem. 1989;58:913-49
[
2528323.001
]
[Cites]
Science. 1995 Jun 23;268(5218):1766-9
[
7792603.001
]
[Cites]
J Virol. 1996 Sep;70(9):6054-9
[
8709228.001
]
[Cites]
Proc Natl Acad Sci U S A. 1996 Oct 1;93(20):10933-8
[
8855286.001
]
[Cites]
Nat Genet. 1999 Aug;22(4):319-20
[
10431229.001
]
[Cites]
Hum Gene Ther. 2004 Oct;15(10):934-44
[
15585109.001
]
[Cites]
Exp Neurol. 2005 Feb;191 Suppl 1:S80-94
[
15629764.001
]
[Cites]
Proc Natl Acad Sci U S A. 2005 Feb 15;102(7):2396-401
[
15695330.001
]
[Cites]
Gene Ther. 2005 Mar;12(6):504-11
[
15660114.001
]
[Cites]
Blood. 2005 May 1;105(9):3699-706
[
15650056.001
]
[Cites]
J Gene Med. 2005 May;7(5):576-83
[
15580589.001
]
[Cites]
Blood. 2000 Dec 1;96(12):3907-14
[
11090077.001
]
(PMID = 19649290.001).
[ISSN]
1932-6203
[Journal-full-title]
PloS one
[ISO-abbreviation]
PLoS ONE
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / enhanced green fluorescent protein; 147336-22-9 / Green Fluorescent Proteins
[Other-IDs]
NLM/ PMC2714175
18.
Tsutsui S, Ashizawa K, Minami K, Tagawa T, Nagayasu T, Hayashi T, Uetani M:
Multiple focal pure ground-glass opacities on high-resolution CT images: Clinical significance in patients with lung cancer.
AJR Am J Roentgenol
; 2010 Aug;195(2):W131-8
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[Title]
Multiple focal pure ground-glass opacities on high-resolution CT images: Clinical significance in patients with
lung cancer
.
OBJECTIVE: The purpose of this study was to evaluate the clinical significance of multiple focal pure ground-glass opacities (GGOs) on high-resolution CT images of patients with
lung cancer
.
MATERIALS AND METHODS: The cases of 23 patients with proven
lung cancer
and associated multiple focal pure GGOs on high-resolution CT images were retrospectively reviewed.
The number, size, distribution, and
morphologic
characteristics of focal pure GGOs were evaluated.
Lung cancer
and focal pure GGOs were seen in the same lobe and/or in the other lobes.
Histologic findings were obtained for 15 lesions representing 74 focal pure GGOs that were surgically resected: 11 atypical adenomatous hyperplasia lesions, three
bronchioloalveolar carcinomas
, and one lesion of focal fibrosis.
CONCLUSION: The size of most focal pure GGOs associated with
lung cancer
did not change during the follow-up period.
Most of the small number of lesions histologically diagnosed were atypical adenomatous hyperplasia or
bronchioloalveolar carcinoma
.
[MeSH-major]
Algorithms.
Lung
Neoplasms / radiography. Radiographic Image Enhancement / methods. Radiographic Image Interpretation, Computer-Assisted / methods. Tomography, X-Ray Computed / methods
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.
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consumer health - Lung Cancer
.
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(PMID = 20651172.001).
[ISSN]
1546-3141
[Journal-full-title]
AJR. American journal of roentgenology
[ISO-abbreviation]
AJR Am J Roentgenol
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
19.
Le Saux A, Houdebine LM, Jolivet G:
Chromosome integration of BAC (bacterial artificial chromosome): evidence of multiple rearrangements.
Transgenic Res
; 2010 Oct;19(5):923-31
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[Title]
Chromosome integration
of BAC
(
bacterial
artificial chromosome): evidence of multiple rearrangements.
The possibility of such
complex
rearrangements should be carefully considered when transgenic animals are produced with large genomic DNA fragments.
[MeSH-major]
Chromosomes / ultrastructure. Chromosomes, Artificial,
Bacterial
/ genetics. Mice, Transgenic / genetics. Recombination, Genetic
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[Cites]
Gene. 2007 Oct 15;401(1-2):97-107
[
17692477.001
]
[Cites]
Transgenic Res. 2003 Dec;12(6):751-5
[
14713206.001
]
[Cites]
Comp Immunol Microbiol Infect Dis. 2009 Mar;32(2):81-90
[
18328563.001
]
[Cites]
Transgenic Res. 1995 Jan;4(1):52-9
[
7881462.001
]
[Cites]
Plant J. 1995 Sep;8(3):457-63
[
7550382.001
]
[Cites]
Genomics. 1995 Feb 10;25(3):674-81
[
7759102.001
]
[Cites]
Mamm Genome. 2007 Oct;18(10):693-708
[
17882484.001
]
[Cites]
Genetics. 1988 Nov;120(3):621-3
[
2852134.001
]
[Cites]
Plant J. 2008 Apr;54(2):335-47
[
18208518.001
]
[Cites]
Biotechniques. 2007 Nov;43(5):649-50, 652, 654 passim
[
18072594.001
]
[Cites]
Methods Mol Biol. 2007;360:163-202
[
17172731.001
]
[Cites]
Gene Ther. 2003 Oct;10(21):1791-9
[
12960968.001
]
[Cites]
Methods Mol Biol. 2010;597:55-69
[
20013225.001
]
[Cites]
Gene. 1995 Nov 20;165(2):173-81
[
8522172.001
]
[Cites]
Reprod Nutr Dev. 1996;36(6):607-18
[
9021872.001
]
[Cites]
J Biol Chem. 1999 Dec 17;274(51):36585-91
[
10593959.001
]
[Cites]
Transgenic Res. 2009 Oct;18(5):769-85
[
19396621.001
]
[Cites]
Nucleic Acids Res. 2006;34(21):6205-14
[
17090599.001
]
[Cites]
Transgenic Res. 2008 Oct;17(5):979-83
[
18612840.001
]
[Cites]
Genesis. 2003 Jul;36(3):134-41
[
12872244.001
]
[Cites]
Gene. 2009 Jan 1;428(1-2):20-4
[
18976699.001
]
(PMID = 20107893.001).
[ISSN]
1573-9368
[Journal-full-title]
Transgenic research
[ISO-abbreviation]
Transgenic Res.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Netherlands
[Chemical-registry-number]
0 / DNA, Recombinant; 0 / Milk Proteins; 0 / whey acidic proteins
20.
Emad A, Emad Y:
Increased in CD8 T lymphocytes in the BAL fluid of patients with sulfur mustard gas-induced pulmonary fibrosis.
Respir Med
; 2007 Apr;101(4):786-92
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OBJECTIVE: In an attempt to understand better the potential role of the T
cell
in the pathogenesis of pulmonary fibrosis (PF) due to sulfur mustard gas inhalation, this study was designed to analyze
bronchoalveolar
lavage (BAL) lymphocyte subsets and to determine the ratio of CD4 to CD8 lymphocytes in BAL fluid.
INTERVENTION: Chest roentgenograms, pulmonary function tests (PFTs), tests for carbon monoxide diffusing capacity of the
lung
(DLCO), high-resolution CT scans of the chest, BAL via fiberoptic bronchoscopy, analyses of BAL fluids for cellular and Flow-cytometric analysis of the phenotype
of bronchoalveolar
cells were performed in all cases.
A transbronchial
lung
biopsy was done in all patients following BAL.
Neutrophils (P<0.0001) and eosinophils (P=0.0006) were the predominant
cell
types in the BAL fluid of patients with PF.
[MeSH-major]
Bronchoalveolar
Lavage Fluid / immunology. CD8-Positive T-Lymphocytes / immunology. Chemical Warfare Agents / adverse effects. Mustard Gas / adverse effects. Pulmonary Fibrosis / chemically induced
[MeSH-minor]
Adult. CD4-CD8 Ratio / methods. CD4-Positive T-Lymphocytes / immunology. Carbon Monoxide / physiology. Humans. Inhalation Exposure / adverse effects.
Lung
/ physiopathology.
Lung
/ radiography. Lymphocyte Count / methods. Tomography, X-Ray Computed / methods
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.
MedlinePlus Health Information.
consumer health - Pulmonary Fibrosis
.
Hazardous Substances Data Bank.
BIS(2-CHLOROETHYL)SULFIDE
.
Hazardous Substances Data Bank.
Carbon monoxide
.
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(PMID = 16982181.001).
[ISSN]
0954-6111
[Journal-full-title]
Respiratory medicine
[ISO-abbreviation]
Respir Med
[Language]
eng
[Publication-type]
Comparative Study; Journal Article
[Publication-country]
England
[Chemical-registry-number]
0 / Chemical Warfare Agents; 7U1EE4V452 / Carbon Monoxide; T8KEC9FH9P / Mustard Gas
21.
Meng N, Zhang SQ, Zhou NL, Shen J:
Biopolymer-modified graphite oxide nanocomposite films based on benzalkonium chloride-heparin intercalated in graphite oxide.
Nanotechnology
; 2010 May 7;21(18):185101
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The heparin-benzalkonium chloride (
BAC
-HEP) was intercalated into graphite oxide (GO) layers to form GO-
BAC
-HEP (modified graphite oxide).
Cell
culture assay indicated that PDMS/GO-BCA-HEP nanocomposite films showed enhanced
cell
adhesion.
[MeSH-minor]
Biopolymers / chemistry. Blood Coagulation / drug effects.
Cell
Proliferation / drug effects. Hemolysis. Humans. Leukocytes, Mononuclear / drug effects. Microscopy, Electron, Scanning. Microscopy, Electron, Transmission. P-Selectin / metabolism. Platelet Adhesiveness / drug effects. Spectroscopy, Fourier Transform Infrared. Thermogravimetry. X-Ray Diffraction
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.
Hazardous Substances Data Bank.
GRAPHITE
.
Hazardous Substances Data Bank.
HEPARIN
.
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(PMID = 20378948.001).
[ISSN]
1361-6528
[Journal-full-title]
Nanotechnology
[ISO-abbreviation]
Nanotechnology
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / Anticoagulants; 0 / Benzalkonium Compounds; 0 / Biopolymers; 0 / Dimethylpolysiloxanes; 0 / Nylons; 0 / Oxides; 0 / P-Selectin; 0 / poly(dimethylsiloxane)-polyamide copolymer; 7782-42-5 / Graphite; 9005-49-6 / Heparin
22.
Lavi-Moshayoff V, Silver J, Naveh-Many T:
Human PTH gene regulation in vivo using transgenic mice.
Am J Physiol Renal Physiol
; 2009 Sep;297(3):F713-9
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To study the regulation of the human PTH (hPTH) gene in vivo, we generated transgenic mice with the hPTH gene expressed in the mouse parathyroid using
a bacterial
artificial chromosome (
BAC
) containing the hPTH gene within its 144-kb chromosomal region.
The BAC
construct maintains the native hPTH gene surrounding sequences and isolates it from positional effects.
[MeSH-minor]
Age Factors. Aniline Compounds / pharmacology. Animals. Calcitriol / blood. Calcium / agonists. Calcium / blood. Chromosomes, Artificial,
Bacterial
. Fibroblast Growth Factors / metabolism. Glucuronidase / metabolism. Humans. Mice. Mice, Transgenic. RNA, Messenger / blood. Receptor, Fibroblast Growth Factor, Type 1 / metabolism. Receptor, Parathyroid Hormone, Type 1 / metabolism. Receptors, Calcium-Sensing / metabolism. Recombinant Proteins / metabolism. Tachyphylaxis
Hazardous Substances Data Bank.
1,25-DIHYDROXYCHOLECALCIFEROL
.
Hazardous Substances Data Bank.
CALCIUM, ELEMENTAL
.
Mouse Genome Informatics (MGI).
Mouse Genome Informatics (MGI)
.
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(PMID = 19570881.001).
[ISSN]
1522-1466
[Journal-full-title]
American journal of physiology. Renal physiology
[ISO-abbreviation]
Am. J. Physiol. Renal Physiol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Aniline Compounds; 0 / N-(2-chlorophenylpropyl)-1-(3-methoxyphenyl)ethylamine; 0 / Parathyroid Hormone; 0 / RNA, Messenger; 0 / Receptor, Parathyroid Hormone, Type 1; 0 / Receptors, Calcium-Sensing; 0 / Recombinant Proteins; 0 / fibroblast growth factor 23; 62031-54-3 / Fibroblast Growth Factors; EC 2.7.10.1 / Fgfr1 protein, mouse; EC 2.7.10.1 / Receptor, Fibroblast Growth Factor, Type 1; EC 3.2.1.31 / Glucuronidase; EC 3.2.1.31 / klotho protein; FXC9231JVH / Calcitriol; SY7Q814VUP / Calcium
23.
Noaín D, Avale ME, Wedemeyer C, Calvo D, Peper M, Rubinstein M:
Identification of brain neurons expressing the dopamine D4 receptor gene using BAC transgenic mice.
Eur J Neurosci
; 2006 Nov;24(9):2429-38
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[Title]
Identification of brain neurons expressing the dopamine D4 receptor gene using
BAC
transgenic mice.
The dopamine D4 receptor (D4R) has received considerable interest because of its higher affinity for atypical antipsychotics, the extremely polymorphic nature of the human gene and the genetic association with attention deficit and hyperactivity
disorder
(ADHD).
Here, we have explored an alternative genetic approach by studying
bacterial
artificial chromosome (
BAC
) transgenic mice that express enhanced green fluorescent protein (EGFP) under the transcriptional control of the mouse dopamine D4 receptor gene (Drd4).
Given the fine specificity of EGFP expression in
BAC
transgenic mice and the high sensitivity of the EGFP antibody used in this study, our results indicate that Drd4 expression in the adult mouse brain is limited to a more restricted number of areas than previously reported.
Its leading expression in the prefrontal cortex supports the importance of the D4R in
complex
behaviours depending on cortical dopamine (DA) transmission and its possible role in the etiopathophysiology of ADHD.
[MeSH-major]
Brain / metabolism. Chromosomes, Artificial,
Bacterial
. Neurons / metabolism. Receptors, Dopamine D4 / metabolism
KOMP Repository.
gene/protein/disease-specific - KOMP Repository
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(PMID = 17100831.001).
[ISSN]
0953-816X
[Journal-full-title]
The European journal of neuroscience
[ISO-abbreviation]
Eur. J. Neurosci.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
France
[Chemical-registry-number]
137750-34-6 / Receptors, Dopamine D4; 147336-22-9 / Green Fluorescent Proteins
24.
Ge Y, He G, Wang Z, Guo D, Qin R, Li R:
GISH and BAC-FISH study of apomictic Beta M14.
Sci China C Life Sci
; 2007 Apr;50(2):242-50
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[Title]
GISH and
BAC
-FISH study of apomictic Beta M14.
We analyzed
BAC
microarrays of B. corolliflora chromosome 9 by using fluorescence-specific mRNA of B. vulgaris and Beta M14.
BAC
clones 16-M11 and 26-L15 were detected as fluorescence-specifics
of BAC
DNA of Beta M14.
Then both
BAC
clones 16-M11 and 26-L15 were in situ hybridized to M14 chromosomes.
The two hybridized
BAC
clones were located close to the telomere region of the long arm
of a
single chromosome 9, and showed hemizygosity.
The results
of BAC
microarrays showed that these developments of embryo and endosperm have conservative expression patterns, indicating that sexual reproduction and apomixis have an interrelated pathway with common regulatory components and that the induction
of a
modified sexual reproduction program may enable the manifestation of apomixis in Beta species.
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(PMID = 17447032.001).
[ISSN]
1006-9305
[Journal-full-title]
Science in China. Series C, Life sciences
[ISO-abbreviation]
Sci. China, C, Life Sci.
[Language]
ENG
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
China
25.
Webster L, Russell M, Walsham P, Phillips LA, Packer G, Hussy I, Scurfield JA, Dalgarno EJ, Moffat CF:
An assessment of persistent organic pollutants (POPs) in wild and rope grown blue mussels (Mytilius edulis) from Scottish coastal waters.
J Environ Monit
; 2009 Jun;11(6):1169-84
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Concentrations were compared to Background Assessment Concentrations (
BAC
; blue/green transition) and Environmental Assessment Concentrations (EACs; green/red transition).
The pristine sites were also below
BACs
for some PAHs and therefore would be classed as 'blue' for these PAHs.
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(PMID = 19513448.001).
[ISSN]
1464-0333
[Journal-full-title]
Journal of environmental monitoring : JEM
[ISO-abbreviation]
J Environ Monit
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
England
[Chemical-registry-number]
0 / Organic Chemicals; 0 / Polycyclic Hydrocarbons, Aromatic; 0 / Water Pollutants, Chemical; DFC2HB4I0K / Polychlorinated Biphenyls
26.
Wislez M, Antoine M, Baudrin L, Poulot V, Neuville A, Pradere M, Longchampt E, Isaac-Sibille S, Lebitasy MP, Cadranel J:
Non-mucinous and mucinous subtypes of adenocarcinoma with bronchioloalveolar carcinoma features differ by biomarker expression and in the response to gefitinib.
Lung Cancer
; 2010 May;68(2):185-91
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[Title]
Non-mucinous and mucinous subtypes of
adenocarcinoma
with
bronchioloalveolar carcinoma
features differ by biomarker expression and in the response to gefitinib.
There is no optimal established therapy for treating advanced or recurrent
adenocarcinoma
with
bronchioloalveolar carcinoma
features (ADC-
BAC
), and it remains unclear whether chemotherapy achieves therapeutic results comparable to those seen in the more common non-small
lung carcinoma
subtypes.
In order to improve the decisions made during the treatment of advanced ADC-
BAC
, we attempted to better characterize the mucinous and non-mucinous ADC-
BAC
subtypes.
Fifty pathological samples were obtained from 62 patients included in a multicenter prospective phase II trial (IFCT0401) conducted to evaluate gefitinib as a first-line therapy for non-resectable ADC-
BAC
.
We demonstrated that demographic data, clinical characteristics and stage at presentation (extrathoracic versus
lung
metastasis, as well as TNM staging) did not distinguish between the two subtypes.
In contrast, three biological markers (PAS staining, TTF-1 expression and EGFR genomic gain combined with mutation analysis) enabled us to independently segregate all but 2 of the 50 patients into the mucinous and non-mucinous ADC-
BAC
subtypes.
[MeSH-major]
Adenocarcinoma
,
Bronchiolo
-
Alveolar
/
diagnosis
.
Adenocarcinoma
, Mucinous /
diagnosis
. DNA-Binding Proteins / metabolism.
Lung
Neoplasms /
diagnosis
. Receptor, Epidermal Growth Factor / genetics
[MeSH-minor]
Adult. Aged. Aged, 80 and over. Biomarkers, Tumor / analysis. Biomarkers, Tumor / genetics. Biomarkers, Tumor / metabolism. DNA Mutational Analysis.
Disease
Progression. Drug Resistance, Neoplasm. Female. Humans. Male. Middle Aged. Quinazolines / therapeutic use
MedlinePlus Health Information.
consumer health - Lung Cancer
.
ClinicalTrials.gov.
clinical trials - ClinicalTrials.gov
.
NCI CPTAC Assay Portal.
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.
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[Copyright]
Copyright 2009 Elsevier Ireland Ltd. All rights reserved.
(PMID = 19581016.001).
[ISSN]
1872-8332
[Journal-full-title]
Lung cancer (Amsterdam, Netherlands)
[ISO-abbreviation]
Lung Cancer
[Language]
eng
[Publication-type]
Clinical Trial, Phase II; Comparative Study; Journal Article; Multicenter Study; Research Support, Non-U.S. Gov't
[Publication-country]
Ireland
[Chemical-registry-number]
0 / Biomarkers, Tumor; 0 / DNA-Binding Proteins; 0 / Quinazolines; 0 / TTF1 protein, human; EC 2.7.10.1 / Receptor, Epidermal Growth Factor; S65743JHBS / gefitinib
27.
Murisier F, Guichard S, Beermann F:
Distinct distal regulatory elements control tyrosinase expression in melanocytes and the retinal pigment epithelium.
Dev Biol
; 2007 Mar 15;303(2):838-47
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The tyrosinase promoter does not confer strong expression in pigment cells in vivo, while inclusion
of a
distal regulatory element at position -15 kb is necessary and sufficient to provide strong expression in melanocytes.
In this report, we show that a 186 kb
BAC
containing the tyrosinase gene provides transgene expression in both RPE and melanocytes indicating the presence of regulatory sequences required for expression in the RPE.
A deletion analysis of the
BAC
was performed demonstrating that a RPE-regulatory element resides between -17 and -75 kb.
In addition, deletion of this regulatory element within a tyrosinase::lacZ
BAC
provided evidence that this sequence is not only sufficient but also required for correct spatial and temporal expression in the RPE.
[MeSH-minor]
Albinism, Oculocutaneous / enzymology. Albinism, Oculocutaneous / genetics. Animals. Base Sequence. Chromosomes, Artificial,
Bacterial
/ genetics. DNA Primers / genetics. Enhancer Elements, Genetic. Gene Expression Regulation, Enzymologic. Genes, Regulator. Mice. Mice, Mutant Strains. Mice, Transgenic. Mutation. Phenotype
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(PMID = 17196956.001).
[ISSN]
0012-1606
[Journal-full-title]
Developmental biology
[ISO-abbreviation]
Dev. Biol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / DNA Primers; EC 1.14.18.1 / Monophenol Monooxygenase
28.
Stanton RJ, Baluchova K, Dargan DJ, Cunningham C, Sheehy O, Seirafian S, McSharry BP, Neale ML, Davies JA, Tomasec P, Davison AJ, Wilkinson GW:
Reconstruction of the complete human cytomegalovirus genome in a BAC reveals RL13 to be a potent inhibitor of replication.
J Clin Invest
; 2010 Sep;120(9):3191-208
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[Title]
Reconstruction of the complete human cytomegalovirus genome in
a BAC
reveals RL13 to be a potent inhibitor of replication.
To generate a genetically intact source of HCMV, we cloned strain Merlin into a self-excising
BAC
.
The Merlin
BAC
clone had mutations in the RL13 gene and UL128 locus that were acquired during limited replication in vitro prior to cloning.
Characterization of viruses generated from repaired
BACs
revealed that RL13 efficiently repressed HCMV replication in multiple
cell
types; moreover, RL13 mutants rapidly and reproducibly emerged in transfectants.
[MeSH-major]
Chromosomes, Artificial,
Bacterial
. Cytomegalovirus / genetics. Cytomegalovirus / metabolism. Genes, Viral. Virus Replication
[MeSH-minor]
Cell
Line. Fibroblasts / metabolism. Fibroblasts / virology. Genome, Viral. Mutation. Tropism / genetics. Virion / genetics. Virion / metabolism
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[Cites]
J Immunol. 2005 Dec 1;175(11):7457-65
[
16301653.001
]
[Cites]
J Virol. 2006 Jan;80(2):710-22
[
16378974.001
]
[Cites]
Virol J. 2006;3:4
[
16409621.001
]
[Cites]
Intervirology. 2006;49(4):215-23
[
16491016.001
]
[Cites]
J Gen Virol. 2006 Sep;87(Pt 9):2451-60
[
16894182.001
]
[Cites]
Virol J. 2006;3:63
[
16945126.001
]
[Cites]
Nat Protoc. 2007;2(4):953-71
[
17446895.001
]
[Cites]
J Virol. 2007 Aug;81(15):7860-72
[
17522202.001
]
[Cites]
Blood. 2007 Aug 1;110(3):937-45
[
17440050.001
]
[Cites]
J Virol. 2007 Oct;81(19):10424-36
[
17652378.001
]
[Cites]
J Gen Virol. 2003 Mar;84(Pt 3):657-63
[
12604818.001
]
[Cites]
Virology. 2003 Mar 1;307(1):164-77
[
12667824.001
]
[Cites]
J Gen Virol. 2003 May;84(Pt 5):1117-22
[
12692276.001
]
[Cites]
Exp Hematol. 2003 Nov;31(11):1007-14
[
14585362.001
]
[Cites]
Proc Natl Acad Sci U S A. 2003 Dec 9;100(25):14976-81
[
14657367.001
]
[Cites]
Virology. 2004 Jan 20;318(2):582-97
[
14972526.001
]
[Cites]
J Gen Virol. 2004 May;85(Pt 5):1301-12
[
15105547.001
]
[Cites]
J Virol. 2004 Sep;78(18):10023-33
[
15331735.001
]
[Cites]
J Med Virol. 2004 Dec;74(4):573-9
[
15484281.001
]
[Cites]
J Mol Biol. 1967 Jun 14;26(2):365-9
[
4291934.001
]
[Cites]
Lancet. 1974 Jan 5;1(7845):1-5
[
4128996.001
]
[Cites]
Virology. 1983 Oct 15;130(1):118-33
[
6314643.001
]
[Cites]
Ann Intern Med. 1984 Oct;101(4):478-83
[
6089634.001
]
[Cites]
J Virol. 2007 Oct;81(20):11479-88
[
17686875.001
]
[Cites]
J Virol. 2008 Jan;82(1):60-70
[
17942555.001
]
[Cites]
J Gen Virol. 2008 Feb;89(Pt 2):359-68
[
18198366.001
]
[Cites]
Neuro Oncol. 2008 Feb;10(1):10-8
[
17951512.001
]
[Cites]
J Med Virol. 2008 Sep;80(9):1615-23
[
18649324.001
]
[Cites]
Proc Natl Acad Sci U S A. 2008 Sep 16;105(37):14118-23
[
18768787.001
]
[Cites]
Proc Natl Acad Sci U S A. 2008 Dec 16;105(50):19950-5
[
19064925.001
]
[Cites]
Biotechniques. 2008 Dec;45(6):659-62, 664-8
[
19238796.001
]
[Cites]
J Virol. 2009 Jun;83(11):5615-29
[
19297488.001
]
[Cites]
J Gen Virol. 2009 Oct;90(Pt 10):2375-80
[
19553388.001
]
[Cites]
J Gen Virol. 2010 Mar;91(Pt 3):605-15
[
19906940.001
]
[Cites]
J Gen Virol. 2010 Jun;91(Pt 6):1535-46
[
20479471.001
]
[Cites]
Proc Natl Acad Sci U S A. 1992 Sep 15;89(18):8794-7
[
1528894.001
]
[Cites]
J Infect Dis. 1995 Jun;171(6):1599-603
[
7769298.001
]
[Cites]
J Virol. 1996 Jan;70(1):78-83
[
8523595.001
]
[Cites]
J Infect Dis. 1996 Jan;173(1):240-5
[
8537667.001
]
[Cites]
Exp Eye Res. 1996 Feb;62(2):155-69
[
8698076.001
]
[Cites]
Hum Gene Ther. 1996 Aug 1;7(12):1405-13
[
8844199.001
]
[Cites]
J Virol Methods. 1997 Jan;63(1-2):103-12
[
9015280.001
]
[Cites]
J Gen Virol. 1995 Apr;76 ( Pt 4):741-50
[
9049319.001
]
[Cites]
J Virol. 1997 Dec;71(12):9833-6
[
9371656.001
]
[Cites]
Virology. 1997 Dec 8;239(1):169-75
[
9426456.001
]
[Cites]
Hum Gene Ther. 1998 Sep 1;9(13):1939-50
[
9741432.001
]
[Cites]
J Gen Virol. 1999 Nov;80 ( Pt 11):2867-77
[
10580048.001
]
[Cites]
J Gen Virol. 2000 Feb;81(Pt 2):393-9
[
10644837.001
]
[Cites]
J Immunol. 2000 May 1;164(9):4775-82
[
10779784.001
]
[Cites]
Proc Natl Acad Sci U S A. 2000 Apr 25;97(9):4873-8
[
10781094.001
]
[Cites]
J Virol. 2000 Aug;74(16):7628-35
[
10906217.001
]
[Cites]
J Virol. 2000 Sep;74(17):7720-9
[
10933677.001
]
[Cites]
J Virol. 2001 Feb;75(4):1870-8
[
11160686.001
]
[Cites]
J Gen Virol. 2001 Jun;82(Pt 6):1429-38
[
11369888.001
]
[Cites]
Rev Med Virol. 2001 May-Jun;11(3):191-200
[
11376481.001
]
[Cites]
Proc Natl Acad Sci U S A. 2001 Jul 3;98(14):7829-34
[
11427719.001
]
[Cites]
Nat Rev Genet. 2001 Oct;2(10):769-79
[
11584293.001
]
[Cites]
J Virol. 2002 Mar;76(5):2316-28
[
11836410.001
]
[Cites]
J Virol. 2002 Mar;76(6):2973-89
[
11861863.001
]
[Cites]
J Med Virol. 2002 Jun;67(2):200-6
[
11992580.001
]
[Cites]
Cancer Res. 2002 Jun 15;62(12):3347-50
[
12067971.001
]
[Cites]
J Virol. 2002 Nov;76(21):10841-8
[
12368327.001
]
[Cites]
Annu Rev Genet. 2002;36:361-88
[
12429697.001
]
[Cites]
J Biol Chem. 2002 Nov 22;277(47):45122-8
[
12244063.001
]
[Cites]
J Gen Virol. 2003 Jan;84(Pt 1):17-28
[
12533697.001
]
[Cites]
J Infect Dis. 2003 Mar 1;187(5):809-19
[
12599055.001
]
[Cites]
J Infect Dis. 1984 Dec;150(6):952-3
[
6094678.001
]
[Cites]
J Infect Dis. 1984 Dec;150(6):953-6
[
6094679.001
]
[Cites]
J Infect Dis. 1987 Apr;155(4):655-60
[
3029241.001
]
[Cites]
J Infect Dis. 1989 Jan;159(1):123-6
[
2535864.001
]
[Cites]
J Infect Dis. 1989 May;159(5):860-5
[
2540247.001
]
[Cites]
J Infect Dis. 1989 Jul;160(1):11-5
[
2543706.001
]
[Cites]
Arch Virol. 1991;117(3-4):143-64
[
1850227.001
]
[Cites]
Arch Virol. 1998;143(9):1701-9
[
9787655.001
]
[Cites]
J Med Virol. 1999 Mar;57(3):298-307
[
10022803.001
]
[Cites]
J Immunol. 1999 Jun 15;162(12):6967-70
[
10358135.001
]
[Cites]
Proc Natl Acad Sci U S A. 1999 Aug 17;96(17):9839-44
[
10449781.001
]
[Cites]
J Virol. 1999 Oct;73(10):8320-9
[
10482582.001
]
[Cites]
J Med Virol. 2005 Jan;75(1):42-6
[
15543586.001
]
[Cites]
Glycobiology. 2005 Feb;15(2):153-64
[
15385431.001
]
[Cites]
Nat Immunol. 2005 Feb;6(2):181-8
[
15640804.001
]
[Cites]
Nucleic Acids Res. 2005;33(4):e36
[
15731329.001
]
[Cites]
Genetics. 2005 Jun;170(2):969-70
[
15802515.001
]
[Cites]
J Virol. 2005 Aug;79(16):10330-8
[
16051825.001
]
[Cites]
J Virol. 2005 Sep;79(18):12095-9
[
16140786.001
]
[Cites]
J Exp Med. 2005 Sep 5;202(5):673-85
[
16147978.001
]
[Cites]
J Gen Virol. 2005 Nov;86(Pt 11):2999-3008
[
16227221.001
]
(PMID = 20679731.001).
[ISSN]
1558-8238
[Journal-full-title]
The Journal of clinical investigation
[ISO-abbreviation]
J. Clin. Invest.
[Language]
eng
[Grant]
United Kingdom / Medical Research Council / / G1000236; United Kingdom / Medical Research Council / / G0700142; United Kingdom / Medical Research Council / / G0801822; United Kingdom / Medical Research Council / / ; United Kingdom / Biotechnology and Biological Sciences Research Council / / ; United Kingdom / Medical Research Council / / G0900740; United Kingdom / Medical Research Council / / MC/ U130184136; United Kingdom / Wellcome Trust / /
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Other-IDs]
NLM/ PMC2929729
29.
Li Y, Uhm T, Ren C, Wu C, Santos TS, Lee MK, Yan B, Santos F, Zhang A, Scheuring C, Sanchez A, Millena AC, Nguyen HT, Kou H, Liu D, Zhang HB:
A plant-transformation-competent BIBAC/BAC-based map of rice for functional analysis and genetic engineering of its genomic sequence.
Genome
; 2007 Mar;50(3):278-88
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[Title]
A plant-transformation-competent BIBAC/
BAC
-based map of rice for functional analysis and genetic engineering of its genomic sequence.
Here, we report a plant-transformation-competent, binary
bacterial
artificial chromosome (BIBAC) and
bacterial
artificial chromosome (
BAC
) based map of rice to facilitate these studies.
The map was constructed from 20 835 BIBAC and
BAC
clones, and consisted of 579 overlapping BIBAC/
BAC
contigs.
Comparative analysis between the genetic and physical maps of chromosome 8 showed that there are 3 "hot" and 2 "cold" spots of genetic recombination along the chromosomal arms in addition to the "cold spot" in the centromeric region, suggesting that the sequence component contents
of a
chromosome may affect its local genetic recombination frequencies.
Because of its plant transformability, the BIBAC/
BAC
map could provide a platform for functional analysis of the rice genome sequence and effective use of the sequencing results for gene and QTL cloning and molecular breeding.
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(PMID = 17502901.001).
[ISSN]
0831-2796
[Journal-full-title]
Genome
[ISO-abbreviation]
Genome
[Language]
ENG
[Publication-type]
Journal Article
[Publication-country]
Canada
[Chemical-registry-number]
0 / DNA, Plant
30.
Jin C, Jin Y, Gisselsson D, Wennerberg J, Wah TS, Strömbäck B, Kwong YL, Mertens F:
Molecular cytogenetic characterization of the 11q13 amplicon in head and neck squamous cell carcinoma.
Cytogenet Genome Res
; 2006;115(2):99-106
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[Title]
Molecular cytogenetic characterization of the 11q13 amplicon in head and neck squamous
cell carcinoma
.
Amplification of 11q13 DNA sequences and overexpression of CCND1 are common findings in head and neck squamous
cell carcinoma
(HNSCC), identified in about 30% of the cases.
In order to study the structure of the amplicon in more detail and to learn more about the mechanisms involved in its initiation, prometaphase, metaphase, and anaphase fluorescence in situ hybridization (FISH) with 40
BAC
clones spanning a 16-Mb region in chromosome bands 11q12.2 to 11q13.5 was performed in nine HNSCC
cell
lines with homogeneously staining regions.
FISH analysis showed that the size of the amplicon varied among the nine
cell
lines, the smallest being 2.12 Mb and the largest 8.97 Mb.
The smallest overlapping region of amplification was approximately 1.61 Mb, covering the region from
BAC
729E14 to
BAC
102B19.
The cell
lines were also used to study the internal structure of the amplicon.
Various patterns of amplified DNA sequences within the amplicon were found among the nine
cell
lines.
Even within the same
cell
line, different amplicon structures could be found in different
cell
populations, indicating that the mechanisms involved in the development of the amplicons in HNSCC were more
complex
than previously assumed.
The frequent
finding
of inverted repeats within the amplicons, however, suggests that breakage-fusion-bridge cycles are important in the initiation, but the fact that such repeats constituted only small parts of the amplicons indicate that they are further rearranged during tumor progression.
[MeSH-major]
Carcinoma
, Squamous
Cell
/ genetics. Chromosomes, Human, Pair 11 / genetics. DNA, Neoplasm / genetics. Gene Amplification. Head and Neck Neoplasms / genetics. In Situ Hybridization, Fluorescence
[MeSH-minor]
Anaphase.
Cell
Line, Tumor / ultrastructure. Chromosome Banding. Chromosome Breakage. Chromosomes, Artificial,
Bacterial
. DNA Repair.
Disease
Progression. Female. Gene Expression Regulation, Neoplastic. Humans. Male. Metaphase. Repetitive Sequences, Nucleic Acid
Genetic Alliance.
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.
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.
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.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
The Lens.
Cited by Patents in
.
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[Copyright]
Copyright 2006 S. Karger AG, Basel.
(PMID = 17065789.001).
[ISSN]
1424-859X
[Journal-full-title]
Cytogenetic and genome research
[ISO-abbreviation]
Cytogenet. Genome Res.
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Switzerland
[Chemical-registry-number]
0 / DNA, Neoplasm
31.
Roca Vanaclocha Y, Narváez JA, Pozuelo C, Monés L:
[Alveolar microlithiasis: an uncommon cause of the crazy paving pattern].
Radiologia
; 2008 Jan-Feb;50(1):75-8
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[Title]
[
Alveolar
microlithiasis: an uncommon cause of the crazy paving pattern].
[Transliterated title]
Microlitiasis
alveolar
: presentación infrecuente del "patrón en empedrado"
Alveolar
microlithiasis is an uncommon
disease
of unknown etiology characterized by the presence of multiple, predominantly subpleural, intra-
alveolar
microcalcifications.
This pattern is not specific for
alveolar
microlithiasis; it has also been reported in other entities, including
alveolar
proteinosis, lipoid pneumonia, and bronchial
alveolar
carcinoma
.
[MeSH-major]
Lithiasis / radiography.
Lung
Diseases / radiography. Pulmonary Alveoli / radiography
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(PMID = 18275793.001).
[ISSN]
0033-8338
[Journal-full-title]
Radiología
[ISO-abbreviation]
Radiologia
[Language]
spa
[Publication-type]
Case Reports; English Abstract; Journal Article
[Publication-country]
Spain
32.
Schneider D, Pohl T, Walter J, Dörner K, Kohlstädt M, Berger A, Spehr V, Friedrich T:
Assembly of the Escherichia coli NADH:ubiquinone oxidoreductase (complex I).
Biochim Biophys Acta
; 2008 Jul-Aug;1777(7-8):735-9
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[Title]
Assembly of the Escherichia coli NADH:ubiquinone oxidoreductase (
complex
I).
The proton-pumping NADH:ubiquinone oxidoreductase is the first of the respiratory chain
complexes
in many bacteria and the mitochondria of most eukaryotes.
In general,
the bacterial complex
consists of 14 different subunits.
In addition to the homologues of these subunits, the mitochondrial
complex
contains approximately 31 additional proteins.
While it was shown that the mitochondrial
complex
is assembled from distinct intermediates, nothing is known about the assembly of the
bacterial complex
.
We used Escherichia coli mutants, in which the nuo-genes coding the subunits
of complex
I were individually disrupted by an insertion
of a
resistance cartridge to determine whether they are required for the assembly
of a
functional
complex
I.
No
complex
I-mediated enzyme activity was detectable in the mutant membranes and it was not possible to extract a structurally intact
complex
I from the mutant membranes.
However, the subunits and the cofactors of the soluble NADH dehydrogenase fragment of the
complex
were detected in the cytoplasm of some of the nuo-mutants.
[MeSH-major]
Electron Transport
Complex
I / genetics. Escherichia coli / enzymology
[MeSH-minor]
Centrifugation, Density Gradient. Cytoplasm / enzymology. Electron Spin Resonance Spectroscopy. Escherichia coli Proteins / chemistry. Escherichia coli Proteins / genetics. Escherichia coli Proteins / isolation & purification. Escherichia coli Proteins / metabolism. Genes,
Bacterial
. Kinetics. Mutation
BioCyc.
gene/protein/disease-specific - nuoJ
.
BioCyc.
gene/protein/disease-specific - nuoK
.
BioCyc.
gene/protein/disease-specific - nuoN
.
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(PMID = 18394423.001).
[ISSN]
0006-3002
[Journal-full-title]
Biochimica et biophysica acta
[ISO-abbreviation]
Biochim. Biophys. Acta
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
Netherlands
[Chemical-registry-number]
0 / Escherichia coli Proteins; EC 1.6.5.3 / Electron Transport Complex I
[Number-of-references]
44
33.
Pusztaszeri M, Orcurto MV, Schmidt S, Krueger T:
Solitary nodular pure bronchioloalveolar carcinoma.
Respiration
; 2010;79(3):243-4
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[Title]
Solitary nodular pure
bronchioloalveolar carcinoma
.
[MeSH-major]
Adenocarcinoma
,
Bronchiolo
-
Alveolar
/ pathology.
Lung
/ pathology.
Lung
Neoplasms / pathology
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(PMID = 19147987.001).
[ISSN]
1423-0356
[Journal-full-title]
Respiration; international review of thoracic diseases
[ISO-abbreviation]
Respiration
[Language]
eng
[Publication-type]
Case Reports; Journal Article
[Publication-country]
Switzerland
34.
Shan X, Ray DA, Bunge JA, Peterson DG:
A bacterial artificial chromosome library for the Australian saltwater crocodile (Crocodylus porosus) and its utilization in gene isolation and genome characterization.
BMC Genomics
; 2009;10 Suppl 2:S9
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[Title]
A bacterial
artificial chromosome library for the Australian saltwater crocodile (Crocodylus porosus) and its utilization in gene isolation and genome characterization.
To facilitate comparative genomics within Crocodylia and between crocodilians and other archosaurs, we have constructed
a bacterial
artificial chromosome (
BAC
) library for the Australian saltwater crocodile, Crocodylus porosus.
This is the first
BAC
library for a crocodile and only the second
BAC
resource for a crocodilian.
RESULTS:
The C
. porosus
BAC
library consists of 101,760 individually archived clones stored in 384-well microtiter plates.
To demonstrate the utility of the library in gene isolation, we probed
the C
. porosus macroarrays with an overgo designed from
a C
-mos (oocyte maturation factor) partial cDNA.
A BAC
containing C-mos was identified and
the C
-mos locus was sequenced.
CONCLUSION: We have demonstrated the utility of the Crocodylus porosus
BAC
library as a tool in genomics research.
The BAC
library should expedite complete genome sequencing
of C
. porosus and facilitate detailed analysis of genome evolution within Crocodylia and between crocodilians and diverse amniote lineages including birds, mammals, and other non-avian reptiles.
[MeSH-minor]
Animals. Chromosomes, Artificial,
Bacterial
/ genetics. Genes, mos. Male. Retroelements. Sequence Analysis, DNA
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[Cites]
Nature. 2001 Feb 15;409(6822):934-41
[
11237014.001
]
[Cites]
Mol Phylogenet Evol. 2007 Nov;45(2):663-73
[
17719245.001
]
[Cites]
J Exp Zool. 2002 Dec 15;294(4):302-11
[
12461810.001
]
[Cites]
Basic Appl Histochem. 1987;31(2):119-26
[
3115252.001
]
[Cites]
Proc Natl Acad Sci U S A. 1991 Jul 1;88(13):5814-8
[
1829530.001
]
[Cites]
Mol Biol Evol. 1994 Nov;11(6):886-98
[
7815928.001
]
[Cites]
Genetics. 1997 Oct;147(2):533-44
[
9335591.001
]
[Cites]
Mol Biol Evol. 1997 Dec;14(12):1266-72
[
9402737.001
]
[Cites]
Genome Res. 1998 Mar;8(3):175-85
[
9521921.001
]
[Cites]
Nature. 1998 Apr 30;392(6679):917-20
[
9582070.001
]
[Cites]
Mol Phylogenet Evol. 1998 Oct;10(2):259-63
[
9878236.001
]
[Cites]
Comp Biochem Physiol C Toxicol Pharmacol. 2004 Jul;138(3):233-44
[
15533781.001
]
[Cites]
Nature. 2004 Dec 9;432(7018):695-716
[
15592404.001
]
[Cites]
Mol Biol Evol. 2005 Apr;22(4):810-3
[
15625185.001
]
[Cites]
Mol Biol Evol. 2007 Jan;24(1):26-53
[
17047029.001
]
[Cites]
Mutat Res. 2007 Mar 1;616(1-2):24-33
[
17161440.001
]
[Cites]
BMC Genomics. 2007;8:52
[
17300727.001
]
[Cites]
Syst Biol. 2006 Dec;55(6):902-11
[
17345672.001
]
[Cites]
Syst Biol. 2006 Dec;55(6):928-35
[
17345674.001
]
[Cites]
Proc Natl Acad Sci U S A. 2007 Feb 20;104(8):2767-72
[
17307883.001
]
[Cites]
Mol Phylogenet Evol. 2007 Jun;43(3):787-94
[
17433721.001
]
[Cites]
PLoS One. 2007;2(5):e484
[
17534434.001
]
[Cites]
Genome Res. 2002 May;12(5):795-807
[
11997346.001
]
(PMID = 19607660.001).
[ISSN]
1471-2164
[Journal-full-title]
BMC genomics
[ISO-abbreviation]
BMC Genomics
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S.
[Publication-country]
England
[Chemical-registry-number]
0 / Retroelements
[Other-IDs]
NLM/ PMC2966330
35.
Idbaih A, Marie Y, Lucchesi C, Pierron G, Manié E, Raynal V, Mosseri V, Hoang-Xuan K, Kujas M, Brito I, Mokhtari K, Sanson M, Barillot E, Aurias A, Delattre JY, Delattre O:
BAC array CGH distinguishes mutually exclusive alterations that define clinicogenetic subtypes of gliomas.
Int J Cancer
; 2008 Apr 15;122(8):1778-86
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[Title]
BAC
array CGH distinguishes mutually exclusive alterations that define clinicogenetic subtypes of gliomas.
To assess the relationship between genetic abnormalities and clinicopathological characteristics, we have performed a genetic and clinical analysis
of a
series of gliomas.
A total of 112 gliomas were analyzed by comparative genomic hybridization on
a BAC
array with a 1 megabase resolution.
Finally, our results highlight the potential
of a
whole-genome analysis as an additional diagnostic in cases of unclear conventional genetic findings.
[MeSH-minor]
Adult. Aged. Astrocytoma / genetics. Astrocytoma / pathology. Chromosomes, Artificial,
Bacterial
. Chromosomes, Human, Pair 1. Chromosomes, Human, Pair 10. Chromosomes, Human, Pair 19. Chromosomes, Human, Pair 7. Chromosomes, Human, Pair 9.
Disease
-Free Survival. Female. Humans. Loss of Heterozygosity. Male. Middle Aged. Multivariate Analysis. Nucleic Acid Hybridization. Oligodendroglioma / genetics. Oligodendroglioma / pathology. Oligonucleotides. Predictive Value of Tests. Prognosis. Proportional Hazards Models. Survival Analysis. World Health Organization
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(PMID = 18076069.001).
[ISSN]
1097-0215
[Journal-full-title]
International journal of cancer
[ISO-abbreviation]
Int. J. Cancer
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Oligonucleotides; EC 2.7.10.1 / Receptor, Epidermal Growth Factor
36.
Zgheib MH, Buchbinder SS, Abi Rafeh N, Elya M, Raia C, Ahern K, Smith MC, Costantino T, Flory MJ, Lafferty JC, Castellanos MR:
Breast arterial calcifications on mammograms do not predict coronary heart disease at coronary angiography.
Radiology
; 2010 Feb;254(2):367-73
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[Title]
Breast arterial calcifications on mammograms do not predict coronary heart
disease
at coronary angiography.
PURPOSE: To examine, in women who underwent cardiac catheterization, whether breast arterial calcifications (
BACs
) seen at screening mammography correlate with coronary heart
disease
(CHD) seen at coronary angiography.
Presence
of BAC
was noted and correlated with presence of CHD and presence of cardiac risk factors.
Thirty-seven (36%) women in the CHD+ group versus 20 (29%) in the CHD-group (P = .40) had
BAC
.
The mean age of the patients with
BAC
, 72 years +/- 9.8, was significantly older than the mean age of the patients without
BAC
, 60.4 years +/- 11.1 (P < .001).
No correlation existed, despite the fact that
BAC
was associated with some cardiac risk factors.
CONCLUSION: The authors did not observe a correlation between
BAC
and coronary angiography-detected CHD, even when CHD severity was considered.
On the basis of these results, caution should be exercised when using screening mammography-detected
BAC
to identify patients with CHD.
[MeSH-major]
Breast / blood supply. Coronary Angiography. Coronary
Disease
/ radiography. Mammography
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[Copyright]
(c) RSNA, 2010.
(PMID = 20093509.001).
[ISSN]
1527-1315
[Journal-full-title]
Radiology
[ISO-abbreviation]
Radiology
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
37.
Sabia C, de Niederhäusern S, Guerrieri E, Bondi M, Anacarso I, Iseppi R, Messi P:
Interference of Lactobacillus plantarum strains in the in vitro conjugative transfer of R-plasmids.
Curr Microbiol
; 2009 Feb;58(2):101-5
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[Title]
Interference of Lactobacillus plantarum strains in the in vitro conjugative transfer
of R
-plasmids.
For this purpose different matings were performed adding to the donor and recipient cells L. plantarum 35d
bac
+ and L. plantarum 396/1
bac
- as agents of interference.
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[Cites]
Trends Biotechnol. 1997 Jul;15(7):270-4
[
9237406.001
]
[Cites]
J Dairy Sci. 2000 Apr;83(4):894-907
[
10791807.001
]
[Cites]
Zh Mikrobiol Epidemiol Immunobiol. 1977 Aug;(8):69-73
[
335743.001
]
[Cites]
Lett Appl Microbiol. 2004;39(6):483-9
[
15548299.001
]
[Cites]
Antonie Van Leeuwenhoek. 1998 Jan;73(1):95-102
[
9602283.001
]
[Cites]
Antimicrob Agents Chemother. 1998 Jan;42(1):53-8
[
9449260.001
]
[Cites]
Br J Nutr. 2002 Feb;87(2):121-9
[
11895164.001
]
[Cites]
Antimicrob Agents Chemother. 1987 May;31(5):723-7
[
3496848.001
]
[Cites]
Water Res. 2003 Apr;37(8):1685-90
[
12697213.001
]
[Cites]
Scand J Infect Dis Suppl. 1988;53:50-8
[
3166543.001
]
[Cites]
Plasmid. 1978 Jun;1(3):417-20
[
372973.001
]
[Cites]
J Bacteriol. 1974 Feb;117(2):360-72
[
4204433.001
]
[Cites]
Annu Rev Microbiol. 1989;43:69-87
[
2679366.001
]
[Cites]
Dig Liver Dis. 2002 Sep;34 Suppl 2:S2-7
[
12408431.001
]
[Cites]
Int J Antimicrob Agents. 2000 Dec;16(4):531-6
[
11118874.001
]
[Cites]
Trends Microbiol. 2001 Sep;9(9):424-8
[
11553454.001
]
[Cites]
Acta Astronaut. 2005 May-Jun;56(9-12):839-50
[
15835023.001
]
[Cites]
Int J Food Microbiol. 2006 Mar 15;107(2):218-22
[
16280183.001
]
[Cites]
FEMS Microbiol Ecol. 2000 Mar 1;31(3):241-248
[
10719205.001
]
[Cites]
Epidemiol Infect. 1994 Oct;113(2):247-58
[
7925663.001
]
[Cites]
Int J Food Microbiol. 2001 Feb 28;64(1-2):193-8
[
11252503.001
]
[Cites]
Antonie Van Leeuwenhoek. 2001 Jun;79(2):199-207
[
11520006.001
]
(PMID = 18810532.001).
[ISSN]
1432-0991
[Journal-full-title]
Current microbiology
[ISO-abbreviation]
Curr. Microbiol.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
38.
Miller AD, Bergholz U, Ziegler M, Stocking C:
Identification of the myelin protein plasmolipin as the cell entry receptor for Mus caroli endogenous retrovirus.
J Virol
; 2008 Jul;82(14):6862-8
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[Title]
Identification of the myelin protein plasmolipin as
the cell
entry receptor for Mus caroli endogenous retrovirus.
McERV could infect some
cell
types from humans, dogs, and rats, but not all, and did not infect any mouse
cell
line tested.
We determined the chromosomal location of the receptor gene in the human genome by phenotypic screening of the G3 human-hamster radiation hybrid
cell
line panel and confirmed the localization by assaying for receptor activity conferred by
bacterial
artificial chromosome (
BAC
) clones spanning the region.
We next localized the gene more precisely in one positive
BAC
by assaying for receptor activity following
BAC
digestion with several restriction enzymes that cleaved different sets of genes, and we confirmed that the final candidate gene, plasmolipin (PLLP; TM4SF11), is the novel receptor by showing that the expression of the human PLLP cDNA renders hamster and mouse cells susceptible to McERV infection.
PLLP functions as a voltage-dependent potassium ion channel and is expressed primarily in kidney and brain, helping to explain the limited range
of cell
types that McERV can infect.
Interestingly, mouse PLLP also functioned well as a receptor for McERV but was simply not expressed in the mouse
cell
types that we originally tested.
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[Cites]
Proc Natl Acad Sci U S A. 2000 Jun 20;97(13):7388-92
[
10852967.001
]
[Cites]
J Virol. 1997 Jun;71(6):4663-70
[
9151860.001
]
[Cites]
Bioinformatics. 2001 Sep;17(9):849-50
[
11590105.001
]
[Cites]
Mamm Genome. 2001 Dec;12(12):933-7
[
11707781.001
]
[Cites]
FASEB J. 2002 Jun;16(8):869-71
[
11967234.001
]
[Cites]
Proc Natl Acad Sci U S A. 2002 Sep 17;99(19):12386-90
[
12218182.001
]
[Cites]
J Exp Med. 2002 Nov 4;196(9):1227-40
[
12417632.001
]
[Cites]
J Virol. 2003 May;77(10):5926-32
[
12719585.001
]
[Cites]
J Neurochem. 2003 Jul;86(2):508-18
[
12871592.001
]
[Cites]
J Virol. 2004 Jun;78(11):5784-98
[
15140976.001
]
[Cites]
Cancer. 1969 Sep;24(3):520-6
[
4241949.001
]
[Cites]
Nat New Biol. 1971 Nov 3;234(44):20-4
[
5286856.001
]
[Cites]
Cancer. 1974 Apr;33(4):1027-33
[
4132053.001
]
[Cites]
Virology. 1974 Jul;60(1):282-7
[
4366800.001
]
[Cites]
J Gen Virol. 1974 Oct;25(1):21-9
[
4372315.001
]
[Cites]
Proc Natl Acad Sci U S A. 1975 Jun;72(6):2315-9
[
49058.001
]
[Cites]
J Cell Physiol. 1978 Mar;94(3):335-42
[
304450.001
]
[Cites]
Biochim Biophys Acta. 1978 Oct 27;516(2):193-230
[
365240.001
]
[Cites]
Proc Soc Exp Biol Med. 1958 Jul;98(3):574-6
[
13567776.001
]
[Cites]
Virology. 1962 Feb;16:147-51
[
14468055.001
]
[Cites]
J Virol. 2006 Mar;80(6):3104-7
[
16501122.001
]
[Cites]
Nature. 2006 Jul 6;442(7098):79-81
[
16823453.001
]
[Cites]
Cell Microbiol. 2008 Apr;10(4):821-7
[
18081727.001
]
[Cites]
J Exp Med. 1953 May;97(5):695-710
[
13052828.001
]
[Cites]
J Virol. 2000 May;74(9):4264-72
[
10756041.001
]
[Cites]
Cell. 1981 Jan;23(1):175-82
[
6260373.001
]
[Cites]
Virology. 1981 Aug;113(1):408-11
[
7269249.001
]
[Cites]
J Membr Biol. 1981;63(1-2):77-84
[
6273572.001
]
[Cites]
J Virol. 1984 Nov;52(2):695-8
[
6092693.001
]
[Cites]
Proc Natl Acad Sci U S A. 1987 Dec;84(23):8458-62
[
3317408.001
]
[Cites]
J Cell Physiol. 1989 Aug;140(2):323-34
[
2663885.001
]
[Cites]
J Neurochem. 1990 Aug;55(2):602-10
[
1695242.001
]
[Cites]
Proc Natl Acad Sci U S A. 1992 Jan 15;89(2):693-7
[
1731342.001
]
[Cites]
J Virol. 1993 Aug;67(8):4704-11
[
8392609.001
]
[Cites]
J Virol. 1993 Sep;67(9):5346-52
[
8394452.001
]
[Cites]
Proc Natl Acad Sci U S A. 1994 Jan 4;91(1):78-82
[
8278411.001
]
[Cites]
J Biol Chem. 1994 Oct 7;269(40):24912-9
[
7929173.001
]
[Cites]
J Virol. 1994 Dec;68(12):8270-6
[
7966619.001
]
[Cites]
Neurochem Res. 1994 Aug;19(8):959-66
[
7800123.001
]
[Cites]
J Virol. 1996 Mar;70(3):1804-9
[
8627704.001
]
[Cites]
Hum Gene Ther. 1996 Oct 1;7(15):1871-81
[
8894679.001
]
[Cites]
Genome Res. 1997 May;7(5):422-33
[
9149939.001
]
[Cites]
J Virol. 1997 Jun;71(6):4531-5
[
9151846.001
]
[Cites]
Proc Natl Acad Sci U S A. 2001 Apr 10;98(8):4443-8
[
11296287.001
]
(PMID = 18463156.001).
[ISSN]
1098-5514
[Journal-full-title]
Journal of virology
[ISO-abbreviation]
J. Virol.
[Language]
ENG
[Grant]
United States / NCI NIH HHS / CA / P30 CA015704; United States / NIDDK NIH HHS / DK / P30 DK047754; United States / NCI NIH HHS / CA / CA15704; United States / NIDDK NIH HHS / DK / DK47754
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Membrane Proteins; 0 / Myelin and Lymphocyte-Associated Proteolipid Proteins; 0 / Nerve Tissue Proteins; 0 / PLLP protein, human; 0 / Pllp protein, mouse; 0 / Proteolipids; 0 / Receptors, Virus; 147336-22-9 / Green Fluorescent Proteins
[Other-IDs]
NLM/ PMC2446966
39.
Okada K, Tonaka N, Moriya Y, Norioka N, Sawamura Y, Matsumoto T, Nakanishi T, Takasaki-Yasuda T:
Deletion of a 236 kb region around S 4-RNase in a stylar-part mutant S 4sm-haplotype of Japanese pear.
Plant Mol Biol
; 2008 Mar;66(4):389-400
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[Title]
Deletion
of a
236 kb region around S 4-RNase in a stylar-part mutant S 4sm-haplotype of Japanese pear.
To delineate the deletion breakpoint in the S(4)sm-haplotype, we constructed
a bacterial
artificial chromosome (
BAC
) library from an S (4)-homozygote, and assembled
a BAC
contig of 570 kb around the S (4)-RNase.
Genomic PCR of DNA from S (4)- and S(4)sm-homozygotes and the DNA sequence of the
BAC
contig allowed the identification
of a
deletion of 236 kb spanning from 48 kb upstream to 188 kb downstream of S (4)-RNase.
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[Cites]
Nature. 2004 May 20;429(6989):302-5
[
15152253.001
]
[Cites]
Genes Cells. 2003 Mar;8(3):203-13
[
12622718.001
]
[Cites]
Plant Cell. 2005 Jan;17(1):37-51
[
15598801.001
]
[Cites]
Plant Mol Biol. 1998 Aug;37(6):931-41
[
9700066.001
]
[Cites]
Curr Opin Plant Biol. 2006 Dec;9(6):631-8
[
17005440.001
]
[Cites]
Plant Mol Biol. 2006 Oct;62(3):371-83
[
16915517.001
]
[Cites]
Nucleic Acids Res. 1994 Jun 11;22(11):2168-9
[
8029028.001
]
[Cites]
Plant J. 2004 Aug;39(4):573-86
[
15272875.001
]
[Cites]
J Mol Biol. 1997 Apr 25;268(1):78-94
[
9149143.001
]
[Cites]
J Biochem. 1996 Aug;120(2):326-34
[
8889818.001
]
[Cites]
Plant Mol Biol. 2002 Sep;50(1):29-42
[
12139007.001
]
[Cites]
Plant Cell. 2003 Mar;15(3):771-81
[
12615948.001
]
[Cites]
Plant Mol Biol. 1992 Feb;18(4):725-37
[
1558946.001
]
[Cites]
Genetics. 2007 Apr;175(4):1869-81
[
17237509.001
]
[Cites]
Plant Mol Biol. 1995 Aug;28(5):847-58
[
7640357.001
]
[Cites]
J Biochem. 1996 Aug;120(2):335-45
[
8889819.001
]
[Cites]
Plant Mol Biol. 2004 Mar;54(5):727-42
[
15356391.001
]
[Cites]
Nucleic Acids Res. 1997 Sep 1;25(17):3389-402
[
9254694.001
]
[Cites]
Genetics. 2001 May;158(1):379-86
[
11333246.001
]
(PMID = 18175198.001).
[ISSN]
0167-4412
[Journal-full-title]
Plant molecular biology
[ISO-abbreviation]
Plant Mol. Biol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Netherlands
[Chemical-registry-number]
0 / Plant Proteins; EC 3.1.- / Endoribonucleases; EC 3.1.26.- / 2-5A-dependent ribonuclease
40.
Bearz A, Talamini R, Vaccher E, Spina M, Simonelli C, Steffan A, Berretta M, Chimienti E, Tirelli U:
MUC-1 (CA 15-3 antigen) as a highly reliable predictor of response to EGFR inhibitors in patients with bronchioloalveolar carcinoma: an experience on 26 patients.
Int J Biol Markers
; 2007 Oct-Dec;22(4):307-11
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[Title]
MUC-1 (CA 15-3
antigen
) as a highly reliable predictor of response to EGFR inhibitors in patients with
bronchioloalveolar carcinoma
: an experience on 26 patients.
BACKGROUND:
Bronchioloalveolar carcinoma
(
BAC
) is a histological subtype of non-small
cell lung cancer
(NSCLC), particularly of
adenocarcinoma
.
Given its multifocality and the poor activity of chemotherapy, there is no established treatment for
BAC
, although promising results have been achieved with inhibitors of the epidermal growth factor receptor (EGFR).
No tumor marker has been validated in
the diagnosis
and follow-up
of lung cancer
, in particular to predict the outcome of treatment with EGFR inhibitors.
PURPOSE: As CA 15-3
antigen
serum levels are reported to be pathologically abnormal in
adenocarcinoma
of the
lung
, we chose this tumor marker to monitor treatment with EGFR inhibitors of patients affected by
adenocarcinoma
with
BAC
features or pure
BAC
.
PATIENTS AND METHODS: We collected data from 26 consecutive Caucasian patients with
BAC
, mostly women and never smokers, who received EGFR inhibitors.
CONCLUSION: Our data suggest that CA 15-3 levels might be a predictive factor for the response to EGFR inhibitors in patients with
BAC
.
[MeSH-major]
Adenocarcinoma
,
Bronchiolo
-
Alveolar
/ drug therapy.
Adenocarcinoma
,
Bronchiolo
-
Alveolar
/ metabolism. Biomarkers, Tumor.
Carcinoma
, Non-Small-
Cell Lung
/ metabolism. Gene Expression Regulation, Neoplastic.
Lung
Neoplasms / metabolism. Mucin-1 / biosynthesis. Mucin-1 / physiology. Receptor, Epidermal Growth Factor / antagonists & inhibitors
[MeSH-minor]
Adult. Aged. Aged, 80 and over. Female. Genetic Predisposition to
Disease
. Humans. Male. Middle Aged
MedlinePlus Health Information.
consumer health - Lung Cancer
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(PMID = 18161663.001).
[ISSN]
0393-6155
[Journal-full-title]
The International journal of biological markers
[ISO-abbreviation]
Int. J. Biol. Markers
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Biomarkers, Tumor; 0 / Mucin-1; EC 2.7.10.1 / Receptor, Epidermal Growth Factor
41.
Yi CD, Gong ZY, Liang GH, Wang FH, Tang SZ, Gu MH:
[Isolation and characterization of the centromeric BAC clones from different genomes in genus Oryza].
Yi Chuan
; 2007 Jul;29(7):851-8
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[Title]
[Isolation and characterization of the centromeric
BAC
clones from different genomes in genus Oryza].
In this research, we constructed five
BAC
libraries for diploid wild rice with different genomes.
Together with the technique of colony blot hybridization and fluorescence in situ hybridization (FISH), centromere-related
BAC
clones were screened and characterized from different genomes.
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(PMID = 17646152.001).
[ISSN]
0253-9772
[Journal-full-title]
Yi chuan = Hereditas
[ISO-abbreviation]
Yi Chuan
[Language]
CHI
[Publication-type]
English Abstract; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
China
42.
Zhang Z, Huang Y, Zhu H:
A highly efficient protocol of generating and analyzing VZV ORF deletion mutants based on a newly developed luciferase VZV BAC system.
J Virol Methods
; 2008 Mar;148(1-2):197-204
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[Title]
A highly efficient protocol of generating and analyzing VZV ORF deletion mutants based on a newly developed luciferase VZV
BAC
system.
Recently, the full-length VZV (P-Oka strain) genome was cloned as a VZV bacteria artificial chromosome (
BAC
) and additionally a firefly luciferase cassette was inserted to generate a novel luciferase VZV
BAC
.
In this study, a highly efficient protocol has been developed exploiting the new luciferase VZV
BAC
system to rapidly isolate and characterize VZV ORF deletion mutants by growth curve analysis in
cell
culture.
[MeSH-minor]
Cell
Line. Chromosomes, Artificial,
Bacterial
. Genes, Reporter. Humans. Luciferases, Firefly / biosynthesis. Luciferases, Firefly / genetics. Viral Plaque Assay
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[Cites]
Proc Natl Acad Sci U S A. 2000 May 23;97(11):5978-83
[
10811905.001
]
[Cites]
J Virol. 2003 May;77(10):6062-5
[
12719598.001
]
[Cites]
J Virol. 2003 Nov;77(22):12369-72
[
14581575.001
]
[Cites]
Vaccine. 2004 Sep 28;22(29-30):4069-74
[
15364458.001
]
[Cites]
J Virol. 2007 Sep;81(17):9024-33
[
17581997.001
]
[Cites]
J Virol. 2005 Jun;79(11):6969-75
[
15890936.001
]
[Cites]
J Virol. 2005 Apr;79(8):5035-46
[
15795289.001
]
[Cites]
Immunol Rev. 1999 Apr;168:143-56
[
10399071.001
]
[Cites]
J Virol Methods. 2004 Nov;121(2):137-43
[
15381350.001
]
[Cites]
Proc Natl Acad Sci U S A. 1993 Aug 1;90(15):7376-80
[
8394020.001
]
(PMID = 18215429.001).
[ISSN]
0166-0934
[Journal-full-title]
Journal of virological methods
[ISO-abbreviation]
J. Virol. Methods
[Language]
eng
[Grant]
United States / NIAID NIH HHS / AI / R01 AI055381-04; United States / NIAID NIH HHS / AI / R01 AI055381; United States / NIAID NIH HHS / AI / AI050709-01; United States / NIAID NIH HHS / AI / R01 AI050709-05; United States / NIAID NIH HHS / AI / R01 AI050709
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
Netherlands
[Chemical-registry-number]
EC 1.13.12.7 / Luciferases, Firefly
[Other-IDs]
NLM/ NIHMS42696; NLM/ PMC2291443
43.
Warming S, Costantino N, Court DL, Jenkins NA, Copeland NG:
Simple and highly efficient BAC recombineering using galK selection.
Nucleic Acids Res
; 2005;33(4):e36
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[Title]
Simple and highly efficient
BAC
recombineering using galK selection.
Here, we describe the construction of three new recombineering strains (SW102, SW105 and SW106) that allow
bacterial
artificial chromosomes (
BACs
) to be modified using galK positive/negative selection.
We also show how galK selection can be used to rapidly introduce point mutations, deletions and loxP sites into
BAC
DNA and thus facilitate functional studies of SNP and/or
disease
-causing point mutations, the identification of long-range regulatory elements and the construction of conditional targeting vectors.
[MeSH-major]
Chromosomes, Artificial,
Bacterial
. Escherichia coli / genetics. Escherichia coli Proteins / genetics. Galactokinase / genetics. Genetic Engineering. Recombination, Genetic
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[Cites]
Proc Natl Acad Sci U S A. 2000 May 23;97(11):5978-83
[
10811905.001
]
[Cites]
Nucleic Acids Res. 1999 Mar 15;27(6):1555-7
[
10037821.001
]
[Cites]
Genomics. 2001 Apr 1;73(1):56-65
[
11352566.001
]
[Cites]
Nat Rev Genet. 2001 Oct;2(10):769-79
[
11584293.001
]
[Cites]
Annu Rev Genet. 2002;36:361-88
[
12429697.001
]
[Cites]
Genome Res. 2002 Dec;12(12):1992-8
[
12466304.001
]
[Cites]
Genome Res. 2003 Mar;13(3):476-84
[
12618378.001
]
[Cites]
Proc Natl Acad Sci U S A. 2003 Jun 10;100(12):7207-12
[
12771385.001
]
[Cites]
Genomics. 2003 Jul;82(1):68-77
[
12809677.001
]
[Cites]
Genome Res. 2003 Sep;13(9):2190-4
[
12915491.001
]
[Cites]
Virology. 2004 Feb 20;319(2):185-9
[
14980479.001
]
[Cites]
J Bacteriol. 1975 Jan;121(1):259-66
[
1090571.001
]
[Cites]
Nucleic Acids Res. 1986 Mar 11;14(5):2287-300
[
3457367.001
]
[Cites]
Proc Natl Acad Sci U S A. 1990 Jun;87(12):4645-9
[
2162051.001
]
[Cites]
Proc Natl Acad Sci U S A. 1992 Sep 15;89(18):8794-7
[
1528894.001
]
[Cites]
Cell. 1997 May 16;89(4):655-67
[
9160756.001
]
[Cites]
Nat Biotechnol. 1997 Sep;15(9):859-65
[
9306400.001
]
[Cites]
Nat Genet. 1998 Oct;20(2):123-8
[
9771703.001
]
[Cites]
Genesis. 2001 Jan;29(1):14-21
[
11135458.001
]
(PMID = 15731329.001).
[ISSN]
1362-4962
[Journal-full-title]
Nucleic acids research
[ISO-abbreviation]
Nucleic Acids Res.
[Language]
eng
[Publication-type]
Journal Article; Research Support, U.S. Gov't, P.H.S.
[Publication-country]
England
[Chemical-registry-number]
0 / Escherichia coli Proteins; EC 2.7.1.6 / Galactokinase; X2RN3Q8DNE / Galactose
[Other-IDs]
NLM/ PMC549575
44.
Lou JC, Chang TW, Huang CE:
Effective removal of disinfection by-products and assimilable organic carbon: an advanced water treatment system.
J Hazard Mater
; 2009 Dec 30;172(2-3):1365-71
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In this investigation, the AOC was reduced effectively by ozonation and biological activated carbon (
BAC
) processes.
Hazardous Substances Data Bank.
CARBON
.
Hazardous Substances Data Bank.
CHLOROACETIC ACID
.
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(PMID = 19744776.001).
[ISSN]
1873-3336
[Journal-full-title]
Journal of hazardous materials
[ISO-abbreviation]
J. Hazard. Mater.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Netherlands
[Chemical-registry-number]
0 / Acetates; 0 / Disinfectants; 0 / Trihalomethanes; 0 / Water Pollutants, Chemical; 5GD84Y125G / chloroacetic acid; 68-10-0 / bromoacetate; 7440-44-0 / Carbon
45.
Wang T, Huang W, Chen F:
Baclofen, a GABAB receptor agonist, inhibits human hepatocellular carcinoma cell growth in vitro and in vivo.
Life Sci
; 2008 Feb 27;82(9-10):536-41
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[Title]
Baclofen, a GABAB receptor agonist, inhibits human hepatocellular
carcinoma cell
growth in vitro and in vivo.
Gamma aminobutyric acid (GABA) has been reported to affect
cancer
development, but the activation of its type B receptor (GABABR) has shown contradictory effects on the progress of human
carcinoma
.
In this study, we investigated the antitumor effect of the GABABR agonist baclofen (
Bac
) on growth of human hepatocellular
carcinoma
(HCC) in vitro and in vivo.
We found
Bac
induced G(0)/G(1) phase arrest which was associated with down-regulation of intracellular cAMP level, and up-regulation of p21(WAF1) protein expression as well as its phosphorylation level.
Moreover, systemic administration
of Bac
significantly suppressed Bel-7402 xenograft tumor growth.
Our data support the inhibitory effect of GABABR activation on HCC development, which would raise the possibility to develop
Bac
as a therapeutic drug for the treatment of HCC.
[MeSH-major]
Baclofen / pharmacology.
Carcinoma
, Hepatocellular / prevention & control.
Cell
Proliferation / drug effects. GABA-B Receptor Agonists. Liver Neoplasms, Experimental / prevention & control
[MeSH-minor]
Animals.
Cell
Cycle / drug effects.
Cell
Line, Tumor. Cyclic AMP / metabolism. Cyclin-Dependent Kinase Inhibitor p21 / metabolism. GABA Agonists / pharmacology. Humans. Male. Mice. Mice, Inbred BALB C. Mice, Nude. RNA, Messenger / genetics. RNA, Messenger / metabolism. Radioimmunoassay. Receptors, GABA-B / genetics. Receptors, GABA-B / metabolism. Reverse Transcriptase Polymerase Chain Reaction. Time Factors. Xenograft Model Antitumor Assays
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(PMID = 18222491.001).
[ISSN]
0024-3205
[Journal-full-title]
Life sciences
[ISO-abbreviation]
Life Sci.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / CDKN1A protein, human; 0 / Cyclin-Dependent Kinase Inhibitor p21; 0 / GABA Agonists; 0 / GABA-B Receptor Agonists; 0 / RNA, Messenger; 0 / Receptors, GABA-B; E0399OZS9N / Cyclic AMP; H789N3FKE8 / Baclofen
46.
Groheux D, Hindié E, Trédaniel J, Giraudet AL, Vaylet F, Berenger N, Moretti JL:
[PET-CT for evaluation of the solitary pulmonary nodule: an update].
Rev Mal Respir
; 2009 Dec;26(10):1041-55
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False negative results are mainly due to certain histological types with low metabolic activity (such as
bronchiolo
-
alveolar
carcinoma
and typical carcinoid), or small size (nodules less than 8 mm).
[MeSH-major]
Positron-Emission Tomography. Solitary Pulmonary Nodule /
diagnosis
. Tomography, X-Ray Computed
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consumer health - CT Scans
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(PMID = 20032840.001).
[ISSN]
1776-2588
[Journal-full-title]
Revue des maladies respiratoires
[ISO-abbreviation]
Rev Mal Respir
[Language]
fre
[Publication-type]
English Abstract; Journal Article; Review
[Publication-country]
France
[Number-of-references]
69
47.
Värnik A, Kõlves K, Väli M, Tooding LM, Wasserman D:
Do alcohol restrictions reduce suicide mortality?
Addiction
; 2007 Feb;102(2):251-6
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AIM: Blood alcohol concentration (
BAC
) at the time of suicide was examined in relation to the marked falls in suicide rates and per capita alcohol consumption in Estonia during the major Soviet anti-alcohol campaign from 1 June 1985.
Cases were divided by gender and
BAC
level (0.5-1.49, 1.5-2.49 and > 2.5 per thousand).
During the intervention,
BAC
-positive, i.e. alcohol-positive, suicides decreased by 39.2% for males and 41.4% for females, with the largest fall occurring at
the BAC
2.5 per thousand + level for both sexes.
Changes in
BAC
-negative suicides were modest.
CONCLUSIONS: Investigation on an individual level showed that alcohol consumption was a common precursor to suicide and that rigorous alcohol restrictions were accompanied particularly by a decrease in
BAC
-positive suicide mortality among both sexes.
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.
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consumer health - Suicide
.
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(PMID = 17222279.001).
[ISSN]
0965-2140
[Journal-full-title]
Addiction (Abingdon, England)
[ISO-abbreviation]
Addiction
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
48.
Miyauchi T, Mori M, Ito K:
Quantitative determination of benzalkonium chloride in treated wood by solid-phase extraction followed by liquid chromatography with ultraviolet detection.
J Chromatogr A
; 2005 Nov 18;1095(1-2):74-80
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Ammoniacal copper quat (ACQ) compound wood preservative is comprised of copper and quaternary ammonium compounds with benzalkonium chloride (
BAC
) as the active ingredient.
Solid-phase extraction (SPE) followed by liquid chromatography with ultraviolet detection (LC-UV) was developed for quantitative determination
of BAC
in treated wood.
BAC
used in the present study was composed of 66% C12, 33% C14 and less than 1% C16.
BAC
was added to each wood species (500 mg) then extracted with HCl-ethanol (20 ml) and quantitatively determined with LC-UV (262 nm).
Wood extractives from the heartwood of each species, except western hemlock, interfered with quantitative determination
of BAC
, but SPE with an Oasis MCX cartridge was effective in preventing this.
Using the present methods,
BAC
homologue peaks were clearly confirmed without interference.
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(PMID = 16275285.001).
[ISSN]
0021-9673
[Journal-full-title]
Journal of chromatography. A
[ISO-abbreviation]
J Chromatogr A
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Netherlands
[Chemical-registry-number]
0 / Benzalkonium Compounds
49.
Hong CP, Kwon SJ, Kim JS, Yang TJ, Park BS, Lim YP:
Progress in understanding and sequencing the genome of Brassica rapa.
Int J Plant Genomics
; 2008;2008:582837
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Triplicated genomic segments of B. rapa are collinear to those
of A
. thaliana with InDels.
The genome triplication has led to an approximately 1.7-fold increase in the B. rapa gene number compared to that
of A
. thaliana.
Repetitive DNA of B. rapa has also been extensively amplified and has diverged from that
of A
. thaliana.
Ten chromosomes of B. rapa are being allocated to BrGSP consortium participants, and each chromosome will be sequenced by
a BAC
-by-
BAC
approach.
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[Cites]
Plant J. 2004 Dec;40(5):725-33
[
15546355.001
]
[Cites]
Genetics. 2006 Sep;174(1):29-39
[
16988107.001
]
[Cites]
Mol Genet Genomics. 2005 Dec;274(6):579-88
[
16283385.001
]
[Cites]
Genetics. 2005 Oct;171(2):765-81
[
16020789.001
]
[Cites]
Trends Genet. 2005 Nov;21(11):602-7
[
16140417.001
]
[Cites]
Trends Genet. 2005 Oct;21(10):539-43
[
16098633.001
]
[Cites]
Mol Cells. 2005 Jun 30;19(3):436-44
[
15995362.001
]
[Cites]
BMC Biol. 2005;3:7
[
15784138.001
]
[Cites]
Genome Res. 2005 Apr;15(4):487-95
[
15805490.001
]
[Cites]
Genome Res. 2005 Apr;15(4):516-25
[
15781573.001
]
[Cites]
Curr Opin Plant Biol. 2005 Apr;8(2):122-8
[
15752990.001
]
[Cites]
Chromosoma. 2004 Dec;113(6):276-83
[
15480726.001
]
[Cites]
Ann Bot. 2005 Jan;95(1):229-35
[
15596470.001
]
[Cites]
Theor Appl Genet. 2004 Nov;109(7):1346-52
[
15365626.001
]
[Cites]
Proc Natl Acad Sci U S A. 1999 Aug 17;96(17):9739-44
[
10449764.001
]
[Cites]
Genetics. 1998 Nov;150(3):1217-28
[
9799273.001
]
[Cites]
Proc Natl Acad Sci U S A. 1998 Oct 27;95(22):13073-8
[
9789043.001
]
[Cites]
Genome Res. 1997 Nov;7(11):1045-53
[
9371740.001
]
[Cites]
Nature. 1996 May 30;381(6581):364-6
[
8632789.001
]
[Cites]
Genetics. 1994 Oct;138(2):499-510
[
7828831.001
]
[Cites]
Plant Mol Biol. 1994 Nov;26(3):817-32
[
7999997.001
]
[Cites]
Plant Mol Biol. 1993 Jan;21(2):213-24
[
8425054.001
]
[Cites]
Proc Natl Acad Sci U S A. 2004 Oct 5;101(40):14349-54
[
15388850.001
]
[Cites]
Mol Genet Genomics. 2004 Jul;271(6):709-16
[
15197578.001
]
[Cites]
Nat Genet. 2004 Jun;36(6):577-9
[
15122255.001
]
[Cites]
Nucleic Acids Res. 2004;32(6):2023-30
[
15064362.001
]
[Cites]
Proc Natl Acad Sci U S A. 2004 Apr 13;101(15):5589-94
[
15064405.001
]
[Cites]
Trends Plant Sci. 2003 Dec;8(12):570-5
[
14659705.001
]
[Cites]
Mol Phylogenet Evol. 2003 Dec;29(3):396-409
[
14615182.001
]
[Cites]
Genomics. 2003 Sep;82(3):378-89
[
12906862.001
]
[Cites]
Genetics. 2003 Mar;163(3):1221-5
[
12663558.001
]
[Cites]
Nature. 2003 Mar 27;422(6930):433-8
[
12660784.001
]
[Cites]
Mol Genet Genomics. 2003 Feb;268(5):656-65
[
12589440.001
]
[Cites]
Plant Cell. 2002 Aug;14(8):1691-704
[
12172016.001
]
[Cites]
Plant J. 2006 Jul;47(1):63-74
[
16824180.001
]
[Cites]
Plant Cell. 2006 Jun;18(6):1348-59
[
16632643.001
]
[Cites]
Genetics. 2006 May;173(1):309-19
[
16723420.001
]
[Cites]
Plant Physiol. 2006 Jan;140(1):336-48
[
16377753.001
]
[Cites]
Plant J. 2001 Jul;27(1):49-58
[
11489182.001
]
[Cites]
Genome Res. 2001 Apr;11(4):595-9
[
11282974.001
]
[Cites]
Genetics. 2001 Mar;157(3):1321-30
[
11238417.001
]
[Cites]
Nature. 2001 Feb 15;409(6822):934-41
[
11237014.001
]
[Cites]
Nature. 2000 Dec 14;408(6814):796-815
[
11130711.001
]
[Cites]
Science. 2000 Nov 10;290(5494):1151-5
[
11073452.001
]
[Cites]
Genetics. 2000 Oct;156(2):833-8
[
11014828.001
]
[Cites]
Plant J. 2000 Jul;23(2):233-43
[
10929117.001
]
[Cites]
Genome Res. 2000 Jun;10(6):776-88
[
10854410.001
]
[Cites]
Comp Funct Genomics. 2005;6(3):138-46
[
18629219.001
]
[Cites]
Genome. 1995 Dec;38(6):1122-31
[
18470236.001
]
[Cites]
Genome. 1995 Apr;38(2):313-9
[
18470170.001
]
[Cites]
Theor Appl Genet. 2007 Oct;115(6):777-92
[
17646962.001
]
[Cites]
Mol Cells. 2007 Jun 30;23(3):349-56
[
17646709.001
]
[Cites]
Mol Cells. 2007 Apr 30;23(2):145-53
[
17464190.001
]
[Cites]
Plant J. 2007 Jan;49(2):173-83
[
17156411.001
]
[Cites]
Mol Cells. 2006 Dec 31;22(3):300-7
[
17202858.001
]
[Cites]
Plant Cell. 2006 Jun;18(6):1339-47
[
16632644.001
]
[Cites]
Genome Res. 2005 Dec;15(12):1632-42
[
16339360.001
]
(PMID = 18288250.001).
[ISSN]
1687-5370
[Journal-full-title]
International journal of plant genomics
[ISO-abbreviation]
Int J Plant Genomics
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Egypt
[Other-IDs]
NLM/ PMC2233773
50.
Amarillo FI, Bass HW:
A transgenomic cytogenetic sorghum (Sorghum propinquum) bacterial artificial chromosome fluorescence in situ hybridization map of maize (Zea mays L.) pachytene chromosome 9, evidence for regions of genome hyperexpansion.
Genetics
; 2007 Nov;177(3):1509-26
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[Source]
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[Title]
A transgenomic cytogenetic sorghum (Sorghum propinquum)
bacterial
artificial chromosome fluorescence in situ hybridization map of maize (Zea mays L.) pachytene chromosome 9, evidence for regions of genome hyperexpansion.
A cytogenetic FISH map of maize pachytene-stage chromosome 9 was produced with 32 maize marker-selected sorghum
BACs
as probes.
Each locus was mapped by means of multicolor direct FISH with a fluorescently labeled probe mix containing a whole-chromosome paint, a single sorghum
BAC
clone, and the centromeric sequence, CentC.
The locations of the sorghum
BAC
-FISH signals were determined, and each new cytogenetic locus was assigned a centiMcClintock position on the short (9S) or long (9L) arm.
[MeSH-minor]
Centromere / genetics. Chromosome Mapping. Chromosomes, Artificial,
Bacterial
/ genetics. Chromosomes, Plant / genetics. Cytogenetics. Genetic Markers. Genome, Plant. In Situ Hybridization, Fluorescence. Meiosis / genetics. Plants, Genetically Modified. Polymorphism, Restriction Fragment Length
COS Scholar Universe.
author profiles
.
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[Cites]
Genome Res. 2004 Oct;14(10A):1916-23
[
15466289.001
]
[Cites]
Proc Natl Acad Sci U S A. 2004 Oct 5;101(40):14349-54
[
15388850.001
]
[Cites]
Genetics. 1988 Mar;118(3):519-26
[
3366363.001
]
[Cites]
Proc Natl Acad Sci U S A. 1990 Jun;87(11):4251-5
[
1971947.001
]
[Cites]
Plant Physiol. 2005 May;138(1):116-26
[
15888684.001
]
[Cites]
Genome Res. 2005 Jun;15(6):885-92
[
15930498.001
]
[Cites]
Proc Natl Acad Sci U S A. 2005 Aug 16;102(33):11793-8
[
16040802.001
]
[Cites]
Proc Natl Acad Sci U S A. 2005 Sep 13;102(37):13206-11
[
16141333.001
]
[Cites]
Plant Physiol. 2005 Sep;139(1):27-38
[
16166258.001
]
[Cites]
Cell Mol Biol Lett. 2005;10(3):425-37
[
16217554.001
]
[Cites]
Genome Res. 2005 Dec;15(12):1643-50
[
16339361.001
]
[Cites]
Plant Physiol. 2005 Dec;139(4):1612-24
[
16339807.001
]
[Cites]
Genetics. 2005 Dec;171(4):1963-76
[
16143604.001
]
[Cites]
Curr Opin Plant Biol. 2006 Apr;9(2):172-6
[
16459128.001
]
[Cites]
Curr Opin Plant Biol. 2006 Apr;9(2):157-63
[
16459130.001
]
[Cites]
Plant Cell. 2006 Mar;18(3):529-44
[
16461583.001
]
[Cites]
Genetics. 2006 Mar;172(3):1893-900
[
16361231.001
]
[Cites]
Genetics. 2006 Mar;172(3):2007-9
[
16387866.001
]
[Cites]
Genetics. 2006 Jun;173(2):1007-21
[
16582446.001
]
[Cites]
BMC Genomics. 2006;7:199
[
16895597.001
]
[Cites]
Genome Res. 2006 Oct;16(10):1241-51
[
16902087.001
]
[Cites]
Genetics. 2006 Oct;174(2):1057-61
[
16951073.001
]
[Cites]
Genetics. 2006 Nov;174(3):1671-83
[
16951074.001
]
[Cites]
Genome Res. 2006 Dec;16(12):1557-65
[
16983148.001
]
[Cites]
Genome Res. 2006 Dec;16(12):1441-4
[
17053088.001
]
[Cites]
Genetics. 2006 Dec;174(4):1755-65
[
17057247.001
]
[Cites]
Curr Opin Plant Biol. 2005 Apr;8(2):155-62
[
15752995.001
]
[Cites]
Annu Rev Genet. 1999;33:479-532
[
10690416.001
]
[Cites]
Genome. 2000 Feb;43(1):199-204
[
10701131.001
]
[Cites]
Proc Natl Acad Sci U S A. 2000 Jun 20;97(13):7008-15
[
10860964.001
]
[Cites]
Genetics. 2000 Oct;156(2):833-8
[
11014828.001
]
[Cites]
Genome. 2000 Dec;43(6):1081-3
[
11195341.001
]
[Cites]
Genome Res. 2001 Jan;11(1):55-66
[
11156615.001
]
[Cites]
Nature. 2001 Feb 15;409(6822):953-8
[
11237021.001
]
[Cites]
Plant Physiol. 2001 Mar;125(3):1216-27
[
11244103.001
]
[Cites]
Plant Physiol. 2001 Mar;125(3):1325-41
[
11244113.001
]
[Cites]
Plant Mol Biol. 2001 Jan;45(1):113-22
[
11247602.001
]
[Cites]
Genetics. 2001 Apr;157(4):1749-57
[
11290728.001
]
[Cites]
Plant J. 2001 Jul;27(1):49-58
[
11489182.001
]
[Cites]
Genome Res. 2001 Oct;11(10):1660-76
[
11591643.001
]
[Cites]
Genome Res. 2001 Dec;11(12):2133-41
[
11731505.001
]
[Cites]
Plant Physiol. 2001 Dec;127(4):1572-8
[
11743101.001
]
[Cites]
Funct Integr Genomics. 2000 Sep;1(2):89-98
[
11793225.001
]
[Cites]
Plant J. 2001 Dec;28(6):689-97
[
11851915.001
]
[Cites]
Plant Mol Biol. 2002 Mar-Apr;48(5-6):453-61
[
11999829.001
]
[Cites]
Plant Mol Biol. 2002 Mar-Apr;48(5-6):463-81
[
12004892.001
]
[Cites]
Genetics. 2002 May;161(1):345-53
[
12019248.001
]
[Cites]
Proc Natl Acad Sci U S A. 2002 Jul 9;99(14):9573-8
[
12060715.001
]
[Cites]
Genetics. 2002 Jul;161(3):1225-34
[
12136025.001
]
[Cites]
Chromosome Res. 2002;10(5):379-87
[
12296520.001
]
[Cites]
Nat Rev Genet. 2002 Oct;3(10):769-78
[
12360235.001
]
[Cites]
Plant Physiol. 2002 Dec;130(4):1594-7
[
12481042.001
]
[Cites]
Plant J. 2003 Apr;34(2):249-55
[
12694599.001
]
[Cites]
Plant J. 2003 Sep;35(5):647-59
[
12940957.001
]
[Cites]
Genetics. 2003 Sep;165(1):367-86
[
14504243.001
]
[Cites]
BMC Genomics. 2007;8:47
[
17291341.001
]
[Cites]
Genetics. 2007 Mar;175(3):1047-58
[
17237520.001
]
[Cites]
Proc Natl Acad Sci U S A. 2007 Jul 10;104(28):11844-9
[
17615239.001
]
[Cites]
Genetics. 2000 Jan;154(1):397-412
[
10628998.001
]
[Cites]
Genome. 1993 Oct;36(5):884-9
[
7903654.001
]
[Cites]
Chromosome Res. 1994 Jan;2(1):65-71
[
8162323.001
]
[Cites]
Nucleic Acids Res. 1994 Nov 25;22(23):4922-31
[
7800481.001
]
[Cites]
Proc Natl Acad Sci U S A. 1995 May 9;92(10):4487-91
[
7753830.001
]
[Cites]
Genome. 1995 Aug;38(4):646-51
[
7672600.001
]
[Cites]
Genetics. 1995 Oct;141(2):683-708
[
8647403.001
]
[Cites]
Chromosoma. 1996;104(5):315-20
[
8575242.001
]
[Cites]
Chromosome Res. 1996 Jan;4(1):24-8
[
8653264.001
]
[Cites]
Genetics. 1996 Jun;143(2):1001-12
[
8725245.001
]
[Cites]
Plant J. 1996 Mar;9(3):421-30
[
8919917.001
]
[Cites]
Genome. 1996 Apr;39(2):277-87
[
8984002.001
]
[Cites]
Genetics. 1998 Jan;148(1):479-94
[
9475757.001
]
[Cites]
Genetics. 1998 Apr;148(4):1983-92
[
9560411.001
]
[Cites]
Trends Genet. 1998 Aug;14(8):327-32
[
9724966.001
]
[Cites]
Proc Natl Acad Sci U S A. 1998 Oct 27;95(22):13073-8
[
9789043.001
]
[Cites]
Plant Mol Biol. 1998 Dec;38(6):1043-52
[
9869410.001
]
[Cites]
Genetics. 1999 May;152(1):427-39
[
10224272.001
]
[Cites]
Genetics. 1999 Jul;152(3):1137-72
[
10388831.001
]
[Cites]
Genome Res. 1999 Oct;9(10):994-1001
[
10523528.001
]
[Cites]
J Hered. 1950 Mar;41(3):59-67
[
15422115.001
]
[Cites]
Genetics. 2005 Feb;169(2):955-65
[
15489513.001
]
[Cites]
Curr Opin Plant Biol. 2005 Apr;8(2):148-54
[
15752994.001
]
[Cites]
Genetics. 2003 Oct;165(2):849-65
[
14573493.001
]
[Cites]
Genetics. 2003 Dec;165(4):2117-28
[
14704191.001
]
[Cites]
Nat Genet. 2004 Feb;36(2):138-45
[
14716315.001
]
[Cites]
Curr Opin Plant Biol. 2004 Apr;7(2):102-7
[
15003207.001
]
[Cites]
Plant Cell. 2004 Mar;16(3):571-81
[
14973167.001
]
[Cites]
Plant Physiol. 2004 Apr;134(4):1317-26
[
15020742.001
]
[Cites]
Genetics. 2004 Apr;166(4):1923-33
[
15126409.001
]
[Cites]
J Histochem Cytochem. 2004 Aug;52(8):1113-6
[
15258188.001
]
[Cites]
Proc Natl Acad Sci U S A. 2004 Sep 14;101(37):13554-9
[
15342909.001
]
(PMID = 17947405.001).
[ISSN]
0016-6731
[Journal-full-title]
Genetics
[ISO-abbreviation]
Genetics
[Language]
eng
[Publication-type]
Journal Article; Research Support, U.S. Gov't, Non-P.H.S.
[Publication-country]
United States
[Chemical-registry-number]
0 / Genetic Markers
[Other-IDs]
NLM/ PMC2147981
51.
Bazzini AA, Asís R, González V, Bassi S, Conte M, Soria M, Fernie AR, Asurmendi S, Carrari F:
miSolRNA: A tomato micro RNA relational database.
BMC Plant Biol
; 2010;10:240
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It permits i) to map miRNAs and their predicted target sites both on expressed (SGN-UNIGENES) and newly annotated sequences (
BAC
sequences released), ii) to co-locate any predicted miRNA-target interaction with metabolic QTL found in tomato fruits, iii) to retrieve expression data of target genes in tomato fruit along their developmental period and iv) to design further experiments for unresolved questions in
complex
trait biology based on the use of genetic materials that have been proven to be a useful tools for map-based cloning experiments in Solanaceae plant species.
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[Cites]
Nucleic Acids Res. 2008 Jul 1;36(Web Server issue):W70-4
[
18424795.001
]
[Cites]
Plant J. 2008 Jun;54(5):876-87
[
18298674.001
]
[Cites]
Trends Plant Sci. 2008 Jul;13(7):359-67
[
18501664.001
]
[Cites]
Plant Physiol. 2008 Aug;147(4):1788-99
[
18539779.001
]
[Cites]
Genome Res. 2008 Oct;18(10):1602-9
[
18653800.001
]
[Cites]
Biochim Biophys Acta. 2008 Nov;1779(11):743-8
[
18457682.001
]
[Cites]
Plant J. 2009 Jan;57(2):313-21
[
18801012.001
]
[Cites]
Nucleic Acids Res. 2009 Jul;37(12):4010-21
[
19417064.001
]
[Cites]
Genome Biol. 2003;5(1):R1
[
14709173.001
]
[Cites]
Plant J. 2004 Mar;37(6):914-39
[
14996223.001
]
[Cites]
Mol Cell. 2004 Jun 18;14(6):787-99
[
15200956.001
]
[Cites]
Nucleic Acids Res. 2004 Jul 1;32(Web Server issue):W309-12
[
15215400.001
]
[Cites]
Plant Cell. 2004 Aug;16(8):2001-19
[
15258262.001
]
[Cites]
Genetics. 1995 Nov;141(3):1147-62
[
8582620.001
]
[Cites]
J Exp Bot. 2001 Jul;52(360):1519-26
[
11457912.001
]
[Cites]
Genes Dev. 2002 Jul 1;16(13):1616-26
[
12101121.001
]
[Cites]
Plant Physiol. 2003 Jun;132(2):709-17
[
12805599.001
]
[Cites]
Nucleic Acids Res. 2004 Jan 1;32(Database issue):D109-11
[
14681370.001
]
[Cites]
J Mol Biol. 1999 May 21;288(5):911-40
[
10329189.001
]
[Cites]
Genome Res. 2005 Jan;15(1):78-91
[
15632092.001
]
[Cites]
Cell. 2005 Apr 22;121(2):207-21
[
15851028.001
]
[Cites]
Plant Physiol. 2005 May;138(1):1-3
[
15888672.001
]
[Cites]
Plant Cell. 2005 Jun;17(6):1658-73
[
15849273.001
]
[Cites]
Nucleic Acids Res. 2005 Jul 1;33(Web Server issue):W701-4
[
15980567.001
]
[Cites]
Science. 2005 Sep 2;309(5740):1567-9
[
16141074.001
]
[Cites]
Nat Biotechnol. 2006 Apr;24(4):447-54
[
16531992.001
]
[Cites]
Plant Mol Biol. 2006 Mar;60(5):773-92
[
16649112.001
]
[Cites]
Bioinformatics. 2006 Dec 1;22(23):2958-9
[
17046975.001
]
[Cites]
Plant Physiol. 2006 Dec;142(4):1380-96
[
17071647.001
]
[Cites]
Nat Rev Genet. 2007 Feb;8(2):93-103
[
17230196.001
]
[Cites]
Plant Cell Environ. 2007 Mar;30(3):323-32
[
17263777.001
]
[Cites]
Nat Genet. 2007 Jun;39(6):787-91
[
17486095.001
]
[Cites]
Bioinformatics. 2007 Jun 1;23(11):1418-23
[
17344243.001
]
[Cites]
Curr Opin Genet Dev. 2007 Dec;17(6):545-52
[
17723293.001
]
[Cites]
Nucleic Acids Res. 2008 Jan;36(Database issue):D947-53
[
17984077.001
]
[Cites]
Biochim Biophys Acta. 2008 Feb;1779(2):99-107
[
18078843.001
]
[Cites]
Bioinformatics. 2008 Feb 1;24(3):422-3
[
18202028.001
]
[Cites]
Plant Physiol. 2008 Apr;146(4):1738-58
[
18281415.001
]
[Cites]
Plant Cell. 2008 Mar;20(3):509-23
[
18364465.001
]
[Cites]
J Exp Bot. 2008;59(10):2875-90
[
18552354.001
]
(PMID = 21059227.001).
[ISSN]
1471-2229
[Journal-full-title]
BMC plant biology
[ISO-abbreviation]
BMC Plant Biol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / MicroRNAs
[Other-IDs]
NLM/ PMC3095322
52.
Kang WH, Hoang NH, Yang HB, Kwon JK, Jo SH, Seo JK, Kim KH, Choi D, Kang BC:
Molecular mapping and characterization of a single dominant gene controlling CMV resistance in peppers (Capsicum annuum L.).
Theor Appl Genet
; 2010 May;120(8):1587-96
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[Title]
Molecular mapping and characterization
of a
single dominant gene controlling CMV resistance in peppers (Capsicum annuum L.).
Analysis of the cellular localization of CMV using a CMV green fluorescent protein construct showed that in 'Bukang,' systemic movement of the virus from the epidermal
cell
layer to mesophyll cells is inhibited.
Three SNP markers were developed by comparative genetic mapping: one intron-based marker using a pepper homolog of Tm-1, and two SNP markers using tomato and pepper
BAC
sequences mapped near Cmr1.
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[Cites]
Virology. 1958 Oct;6(2):303-16
[
13593173.001
]
[Cites]
Theor Appl Genet. 2006 Sep;113(5):895-905
[
16874489.001
]
[Cites]
Theor Appl Genet. 2003 Aug;107(3):540-3
[
12748763.001
]
[Cites]
Theor Appl Genet. 2006 Dec;114(1):113-30
[
17047912.001
]
[Cites]
Plant Cell Rep. 2009 Feb;28(2):223-32
[
19018536.001
]
[Cites]
Genetics. 1992 Dec;132(4):1141-60
[
1360934.001
]
[Cites]
Proc Natl Acad Sci U S A. 2007 Aug 21;104(34):13833-8
[
17699618.001
]
[Cites]
Annu Rev Phytopathol. 2005;43:581-621
[
16078896.001
]
[Cites]
Plant Cell Physiol. 2001 Mar;42(3):340-7
[
11266586.001
]
[Cites]
Theor Appl Genet. 2002 Mar;104(4):586-591
[
12582662.001
]
[Cites]
Cytogenet Genome Res. 2008;120(3-4):194-201
[
18504347.001
]
[Cites]
Theor Appl Genet. 2009 May;118(7):1279-93
[
19229514.001
]
[Cites]
Genetics. 2000 Jun;155(2):873-87
[
10835406.001
]
[Cites]
Mol Plant Microbe Interact. 2002 Jul;15(7):647-53
[
12118880.001
]
[Cites]
Mol Plant Pathol. 2008 Jan;9(1):73-83
[
18705886.001
]
[Cites]
Genetics. 1999 Jul;152(3):1183-202
[
10388833.001
]
[Cites]
J Virol. 2004 Jun;78(12):6102-11
[
15163703.001
]
[Cites]
Plant J. 2002 Dec;32(5):655-67
[
12472683.001
]
[Cites]
Mol Cells. 2009 Feb 28;27(2):205-9
[
19277503.001
]
[Cites]
Plant J. 2005 May;42(3):392-405
[
15842624.001
]
[Cites]
Adv Virus Res. 1992;41:281-348
[
1575085.001
]
[Cites]
Plant J. 2005 Apr;42(2):251-61
[
15807786.001
]
[Cites]
Mol Cells. 2009 Mar 31;27(3):329-36
[
19326080.001
]
[Cites]
Micron. 2009 Dec;40(8):851-9
[
19646883.001
]
[Cites]
J Virol. 2001 Oct;75(19):9114-20
[
11533175.001
]
[Cites]
Adv Virus Res. 2003;62:241-323
[
14719367.001
]
(PMID = 20180096.001).
[ISSN]
1432-2242
[Journal-full-title]
TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik
[ISO-abbreviation]
Theor. Appl. Genet.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Germany
[Chemical-registry-number]
0 / DNA, Plant
53.
Chochi Y, Kawauchi S, Nakao M, Furuya T, Hashimoto K, Oga A, Oka M, Sasaki K:
A copy number gain of the 6p arm is linked with advanced hepatocellular carcinoma: an array-based comparative genomic hybridization study.
J Pathol
; 2009 Apr;217(5):677-84
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[Title]
A copy number gain of the 6p arm is linked with advanced hepatocellular
carcinoma
: an array-based comparative genomic hybridization study.
In accordance with
cancer
progression, genomic aberrations accumulate in
cancer
cells in a stepwise fashion.
The purpose of this study is to elucidate the relationship between genomic alterations and clinical stages in hepatocellular
carcinoma
(HCC).
A technology of array-based CGH using DNA chips spotted with 1440
BAC
clones was applied to 42 surgically removed HCCs to examine the DNA copy number aberrations.
[MeSH-major]
Carcinoma
, Hepatocellular / genetics. Chromosomes, Human, Pair 6 / genetics. Gene Dosage / genetics. Liver Neoplasms / genetics
[MeSH-minor]
Adult. Aged. Algorithms. Chromosome Aberrations. Comparative Genomic Hybridization / methods. DNA, Neoplasm / genetics. Female. Genetic Predisposition to
Disease
. Humans. Male. Middle Aged. Neoplasm Staging
MedlinePlus Health Information.
consumer health - Liver Cancer
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(PMID = 19097070.001).
[ISSN]
1096-9896
[Journal-full-title]
The Journal of pathology
[ISO-abbreviation]
J. Pathol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / DNA, Neoplasm
54.
Howell EC, Kearsey MJ, Jones GH, King GJ, Armstrong SJ:
A and C genome distinction and chromosome identification in brassica napus by sequential fluorescence in situ hybridization and genomic in situ hybridization.
Genetics
; 2008 Dec;180(4):1849-57
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Fluorescence in situ hybridization (FISH)-with 45S rDNA and
a BAC
that hybridizes to the pericentromeric heterochromatin of several chromosomes-followed by GISH allowed identification of six chromosomes and also three chromosome groups.
Using B. oleracea chromosome-specific
BACs
as FISH probes followed by GISH, the chromosomes involved were confirmed to be A7 and C6.
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[Cites]
Plant Mol Biol. 2000 Jan;42(1):225-49
[
10688139.001
]
[Cites]
Genetics. 2007 Feb;175(2):487-503
[
17151256.001
]
[Cites]
Genetics. 2002 Jul;161(3):1225-34
[
12136025.001
]
[Cites]
Genetics. 2003 Jun;164(2):645-53
[
12807785.001
]
[Cites]
Genetics. 2003 Nov;165(3):1569-77
[
14668403.001
]
[Cites]
Proc Natl Acad Sci U S A. 2004 Apr 13;101(15):5589-94
[
15064405.001
]
[Cites]
Nucleic Acids Res. 1979 Dec 11;7(7):1869-85
[
537913.001
]
[Cites]
J Mol Evol. 1996 Nov;43(5):460-8
[
8875860.001
]
[Cites]
Proc Natl Acad Sci U S A. 1997 Apr 1;94(7):3442-7
[
9096413.001
]
[Cites]
Plant Mol Biol. 1998 Dec;38(6):1081-7
[
9869414.001
]
[Cites]
Plant Mol Biol. 2004 Apr;54(6):895-909
[
15612105.001
]
[Cites]
Genetics. 2005 Feb;169(2):967-79
[
15520255.001
]
[Cites]
Genetics. 2005 Feb;169(2):931-44
[
15654116.001
]
[Cites]
Cytogenet Genome Res. 2005;109(1-3):310-4
[
15753591.001
]
[Cites]
Cytogenet Genome Res. 2005;109(1-3):373-7
[
15753599.001
]
[Cites]
Mol Phylogenet Evol. 1999 Dec;13(3):455-62
[
10620403.001
]
[Cites]
Chromosome Res. 2007;15(7):849-61
[
17899408.001
]
[Cites]
Cytogenet Genome Res. 2007;119(1-2):147-53
[
18160795.001
]
[Cites]
Plant J. 2008 Dec;56(6):1030-44
[
18764926.001
]
[Cites]
Genome Res. 2005 Apr;15(4):516-25
[
15781573.001
]
[Cites]
Mol Cells. 2005 Jun 30;19(3):436-44
[
15995362.001
]
[Cites]
Theor Appl Genet. 2005 Jul;111(2):196-205
[
15756535.001
]
[Cites]
Genome. 2005 Dec;48(6):1093-103
[
16391678.001
]
[Cites]
Ann Bot. 2006 Feb;97(2):205-16
[
16357054.001
]
[Cites]
Theor Appl Genet. 2006 Nov;113(8):1467-80
[
16983552.001
]
[Cites]
Genetics. 2006 Nov;174(3):1583-96
[
16951054.001
]
[Cites]
Plant J. 2007 Jan;49(2):173-83
[
17156411.001
]
[Cites]
Methods Cell Sci. 2001;23(1-3):83-104
[
11741146.001
]
(PMID = 18845839.001).
[ISSN]
0016-6731
[Journal-full-title]
Genetics
[ISO-abbreviation]
Genetics
[Language]
ENG
[Grant]
United Kingdom / Biotechnology and Biological Sciences Research Council / / G18621
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / DNA, Plant; 0 / DNA, Ribosomal
[Other-IDs]
NLM/ PMC2600926
55.
López CE, Quesada-Ocampo LM, Bohórquez A, Duque MC, Vargas J, Tohme J, Verdier V:
Mapping EST-derived SSRs and ESTs involved in resistance to bacterial blight in Manihot esculenta.
Genome
; 2007 Dec;50(12):1078-88
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[Title]
Mapping EST-derived SSRs and ESTs involved in resistance to
bacterial
blight in Manihot esculenta.
Cassava
bacterial
blight, caused by Xanthomonas axonopodis pv. manihotis (Xam), is an important
disease
in Latin America and Africa resulting in significant losses.
In total, 10 defense-related genes and 2
bacterial
artificial chromosomes (
BACs
) containing resistance gene candidates (RGCs) were mapped in 11 linkage groups.
The QTL in linkage group U explained 61.6% of the phenotypic variance and was associated with an RGC-containing
BAC
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
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(PMID = 18059536.001).
[ISSN]
0831-2796
[Journal-full-title]
Genome
[ISO-abbreviation]
Genome
[Language]
ENG
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Canada
[Chemical-registry-number]
0 / Biomarkers
56.
Kaplan DH, Li MO, Jenison MC, Shlomchik WD, Flavell RA, Shlomchik MJ:
Autocrine/paracrine TGFbeta1 is required for the development of epidermal Langerhans cells.
J Exp Med
; 2007 Oct 29;204(11):2545-52
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To address these issues, we created mice transgenic for
a bacterial
artificial chromosome (
BAC
) containing the gene for human Langerin into which Cre recombinase had been inserted by homologous recombination (Langerin-Cre).
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(subscription/membership/fee required).
Mouse Genome Informatics (MGI).
Mouse Genome Informatics (MGI)
.
SciCrunch.
Marmoset Gene list: Data: Gene Annotation
.
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[Cites]
J Invest Dermatol. 2001 Dec;117(6):1574-80
[
11886525.001
]
[Cites]
Genesis. 2002 Feb;32(2):73-5
[
11857781.001
]
[Cites]
Nat Immunol. 2002 Dec;3(12):1135-41
[
12415265.001
]
[Cites]
Nat Immunol. 2003 Apr;4(4):380-6
[
12598895.001
]
[Cites]
APMIS. 2003 Jul-Aug;111(7-8):725-40
[
12974775.001
]
[Cites]
Blood. 2003 Nov 1;102(9):3129-35
[
12842983.001
]
[Cites]
J Invest Dermatol. 2004 Mar;122(3):670-2
[
15086552.001
]
[Cites]
Nat Med. 2004 May;10(5):510-7
[
15098028.001
]
[Cites]
J Invest Dermatol. 2004 May;122(5):1165-74
[
15140219.001
]
[Cites]
J Cell Biol. 1989 Feb;108(2):661-9
[
2645303.001
]
[Cites]
J Immunol. 1990 Nov 1;145(9):2833-8
[
2212665.001
]
[Cites]
Int Immunol. 1993 Oct;5(10):1329-41
[
8268138.001
]
[Cites]
J Immunol. 1996 Aug 15;157(4):1499-507
[
8759731.001
]
[Cites]
J Exp Med. 1996 Dec 1;184(6):2417-22
[
8976197.001
]
[Cites]
J Clin Invest. 1997 Aug 1;100(3):575-81
[
9239404.001
]
[Cites]
J Exp Med. 1998 Mar 16;187(6):961-6
[
9500798.001
]
[Cites]
Immunity. 2005 May;22(5):643-54
[
15894281.001
]
[Cites]
Eur J Cell Biol. 2005 Aug;84(8):733-41
[
16180311.001
]
[Cites]
J Invest Dermatol. 2005 Nov;125(5):983-94
[
16297200.001
]
[Cites]
Immunity. 2005 Dec;23(6):611-20
[
16356859.001
]
[Cites]
Nat Immunol. 2006 Mar;7(3):265-73
[
16444257.001
]
[Cites]
Annu Rev Immunol. 2006;24:99-146
[
16551245.001
]
[Cites]
Immunity. 2007 May;26(5):579-91
[
17481928.001
]
[Cites]
Immunity. 2000 Jan;12(1):71-81
[
10661407.001
]
[Cites]
Blood. 2001 Jan 1;97(1):324-6
[
11133778.001
]
[Cites]
Int Immunol. 2001 May;13(5):695-704
[
11312257.001
]
[Cites]
J Immunol Methods. 2001 Nov 1;257(1-2):99-105
[
11687243.001
]
[Cites]
Nat Immunol. 2001 Dec;2(12):1151-8
[
11702065.001
]
[Cites]
Genesis. 2002 Jan;32(1):19-26
[
11835670.001
]
[Cites]
Int Immunol. 2002 May;14(5):433-44
[
11978773.001
]
(PMID = 17938236.001).
[ISSN]
1540-9538
[Journal-full-title]
The Journal of experimental medicine
[ISO-abbreviation]
J. Exp. Med.
[Language]
ENG
[Grant]
United States / NHLBI NIH HHS / HL / R01 HL066279; United States / NIAMS NIH HHS / AR / R01 AR044077; United States / NIAMS NIH HHS / AR / R01 AR44077; United States / NIAMS NIH HHS / AR / K08 AR651092; United States / NHLBI NIH HHS / HL / R01 HL66279
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
United States
[Chemical-registry-number]
0 / Antigens, CD; 0 / CD207 protein, human; 0 / Lectins, C-Type; 0 / Mannose-Binding Lectins; 0 / TGFB1 protein, human; 0 / Transforming Growth Factor beta1; EC 2.7.7.- / Cre recombinase; EC 2.7.7.- / Integrases
[Other-IDs]
NLM/ PMC2118472
57.
Borg K, Stankiewicz P, Bocian E, Kruczek A, Obersztyn E, Lupski JR, Mazurczak T:
Molecular analysis of a constitutional complex genome rearrangement with 11 breakpoints involving chromosomes 3, 11, 12, and 21 and a approximately 0.5-Mb submicroscopic deletion in a patient with mild mental retardation.
Hum Genet
; 2005 Nov;118(2):267-75
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[Title]
Molecular analysis
of a
constitutional
complex
genome rearrangement with 11 breakpoints involving chromosomes 3, 11, 12, and 21 and a approximately 0.5-Mb submicroscopic deletion in a patient with mild mental retardation.
Complex
chromosome rearrangements (CCRs) are extremely rare but often associated with mental retardation, congenital anomalies, or recurrent spontaneous abortions.
We report
a de
novo apparently balanced CCR involving chromosomes 3 and 12 and a two-way translocation between chromosomes 11 and 21 in a woman with mild intellectual disability, obesity, coarse facies, and apparent synophrys without other distinctive dysmorphia or congenital anomalies.
Molecular analysis of breakpoints using fluorescence in situ hybridization (FISH) with region-specific
BAC
clones revealed a more
complex
character for the CCR.
The rearrangement is a result of nine breaks and involves reciprocal translocation
of terminal
chromosome fragments 3p24.1-->pter and 12q23.1-->qter, insertion of four fragments of the long arm of chromosome 12: q14.1-->q21?
The deletion involves the chromosome region that has been previously associated with Cornelia
de
Lange syndrome (CdLS) in which a novel gene NAALADL2 has been mapped recently.
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[ErratumIn]
Hum Genet. 2006 Jan;118(5):668
[Cites]
Nat Genet. 2002 May;31(1):79-83
[
11941370.001
]
[Cites]
Nat Genet. 2004 Jun;36(6):636-41
[
15146185.001
]
[Cites]
Am J Hum Genet. 1991 Nov;49(5):995-1013
[
1928105.001
]
[Cites]
Eur J Hum Genet. 2004 Aug;12(8):647-53
[
15162125.001
]
[Cites]
J Med Genet. 2002 Jun;39(6):391-9
[
12070244.001
]
[Cites]
Am J Med Genet A. 2003 Mar 15;117A(3):207-11
[
12599183.001
]
[Cites]
Am J Hum Genet. 2005 Jan;76(1):8-32
[
15549674.001
]
[Cites]
Eur J Biochem. 1986 Jan 2;154(1):193-6
[
3080313.001
]
[Cites]
Hum Genet. 2004 Jul;115(2):139-48
[
15168106.001
]
[Cites]
Cell. 1994 Dec 16;79(6):1111-20
[
8001137.001
]
[Cites]
Am J Med Genet A. 2004 Jan 1;124A(1):10-8
[
14679581.001
]
[Cites]
Nat Genet. 1992 Apr;1(1):3-6
[
1301996.001
]
[Cites]
Am J Med Genet. 1998 Jun 16;78(1):44-51
[
9637422.001
]
[Cites]
J Med Genet. 2001 Nov;38(11):740-4
[
11694545.001
]
[Cites]
Am J Med Genet. 2001 Jun 15;101(2):120-9
[
11391654.001
]
[Cites]
Clin Genet. 2004 Jun;65(6):496-500
[
15151510.001
]
[Cites]
Proc Natl Acad Sci U S A. 2002 Sep 3;99(18):11754-9
[
12195014.001
]
[Cites]
J Med Genet. 1991 Sep;28(9):639-40
[
1956066.001
]
[Cites]
J Med Genet. 1993 Feb;30(2):167-70
[
8445625.001
]
[Cites]
J Biol Chem. 1987 Oct 25;262(30):14498-506
[
3667587.001
]
[Cites]
Genet Med. 2004 Mar-Apr;6(2):81-9
[
15017330.001
]
[Cites]
Cytogenet Genome Res. 2003;103(1-2):14-6
[
15004457.001
]
[Cites]
J Med Genet. 1999 Apr;36(4):271-8
[
10227392.001
]
[Cites]
Learn Mem. 2004 Jul-Aug;11(4):365-72
[
15254214.001
]
[Cites]
Genomics. 2004 Apr;83(4):727-34
[
15028294.001
]
[Cites]
J Med Genet. 1993 Sep;30(9):713-27
[
8411066.001
]
[Cites]
Proc Natl Acad Sci U S A. 1998 May 26;95(11):6413-8
[
9600980.001
]
[Cites]
Hum Mol Genet. 1995 Mar;4(3):415-22
[
7795596.001
]
[Cites]
Nat Genet. 2004 Jun;36(6):631-5
[
15146186.001
]
[Cites]
J Med Genet. 1990 Jul;27(7):426-32
[
2395160.001
]
[Cites]
Pharmacol Ther. 1999 Mar;81(3):163-221
[
10334661.001
]
[Cites]
Clin Genet. 2002 Oct;62(4):315-20
[
12372060.001
]
[Cites]
Genomics. 2003 May;81(5):489-503
[
12706107.001
]
[Cites]
J Med Genet. 2000 Nov;37(11):858-65
[
11073540.001
]
[Cites]
J Med Genet. 2005 Jan;42(1):8-16
[
15635069.001
]
[Cites]
Int Rev Neurobiol. 2004;59:111-74
[
15006487.001
]
[Cites]
Eur J Hum Genet. 1999 May-Jun;7(4):487-95
[
10352939.001
]
[Cites]
Hum Genet. 1998 Aug;103(2):173-8
[
9760201.001
]
[Cites]
Eur J Hum Genet. 2004 May;12(5):419-21
[
14997185.001
]
[Cites]
J Med Genet. 2004 Dec;41(12):e128
[
15591270.001
]
[Cites]
Am J Med Genet. 2001 Feb 1;98(4):317-9
[
11170074.001
]
[Cites]
Nat Genet. 1996 Nov;14(3):353-6
[
8896571.001
]
[Cites]
Am J Med Genet. 1988 Oct;31(2):415-20
[
3068990.001
]
[Cites]
Am J Med Genet A. 2004 Jan 15;124A(2):179-91
[
14699618.001
]
[Cites]
J Genet Hum. 1982 Oct;30(3):199-214
[
6759619.001
]
[Cites]
Am J Med Genet A. 2003 Apr 30;118A(3):235-40
[
12673653.001
]
(PMID = 16160854.001).
[ISSN]
0340-6717
[Journal-full-title]
Human genetics
[ISO-abbreviation]
Hum. Genet.
[Language]
eng
[Grant]
United States / NICHD NIH HHS / HD / HD24064; United States / NICHD NIH HHS / HD / P01 HD39420
[Publication-type]
Case Reports; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
Germany
58.
Dohm JC, Lottaz C, Borodina T, Himmelbauer H:
SHARCGS, a fast and highly accurate short-read assembly algorithm for de novo genomic sequencing.
Genome Res
; 2007 Nov;17(11):1697-706
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[Title]
SHARCGS, a fast and highly accurate short-read assembly algorithm for
de
novo genomic sequencing.
In order to extend the fields of application to
de
novo sequencing, we developed the SHARCGS algorithm to assemble short-read (25-40-mer) data with high accuracy and speed.
The efficiency of SHARCGS was tested on
BAC
inserts from three eukaryotic species, on two yeast chromosomes, and on two
bacterial
genomes (Haemophilus influenzae, Escherichia coli).
We show that 30-mer-based
BAC
assemblies have N50 sizes >20 kbp for Drosophila and Arabidopsis and >4 kbp for human in simulations taking missing reads and wrong base calls into account.
Thus, SHARCGS is a suitable tool for fully exploiting novel sequencing technologies by assembling sequence contigs
de
novo with high confidence and by outperforming existing assembly algorithms in terms of speed and accuracy.
[MeSH-minor]
Chromosomes, Artificial,
Bacterial
/ chemistry. Contig Mapping. Humans. Sequence Analysis, DNA / methods
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[Cites]
Science. 2000 Mar 24;287(5461):2271-4
[
10731150.001
]
[Cites]
Science. 2000 Mar 24;287(5461):2196-204
[
10731133.001
]
[Cites]
Proc Natl Acad Sci U S A. 2001 Aug 14;98(17):9748-53
[
11504945.001
]
[Cites]
Genome Res. 2002 Jan;12(1):177-89
[
11779843.001
]
[Cites]
Science. 2002 Aug 23;297(5585):1301-10
[
12142439.001
]
[Cites]
Nature. 2002 Dec 5;420(6915):520-62
[
12466850.001
]
[Cites]
Genome Res. 2003 Jan;13(1):81-90
[
12529309.001
]
[Cites]
Genome Res. 2003 Jan;13(1):91-6
[
12529310.001
]
[Cites]
Proc Natl Acad Sci U S A. 1977 Dec;74(12):5463-7
[
271968.001
]
[Cites]
Genome Res. 1998 Mar;8(3):186-94
[
9521922.001
]
[Cites]
Genome Res. 1999 Sep;9(9):868-77
[
10508846.001
]
[Cites]
Nature. 2005 Sep 15;437(7057):376-80
[
16056220.001
]
[Cites]
PLoS Genet. 2006 Jul;2(7):e120
[
16789826.001
]
[Cites]
Curr Opin Genet Dev. 2006 Dec;16(6):545-52
[
17055251.001
]
[Cites]
Bioinformatics. 2007 Feb 15;23(4):500-1
[
17158514.001
]
[Cites]
Science. 2000 Mar 24;287(5461):2185-95
[
10731132.001
]
[Cites]
Genome Res. 2001 Mar;11(3):483-96
[
11230172.001
]
(PMID = 17908823.001).
[ISSN]
1088-9051
[Journal-full-title]
Genome research
[ISO-abbreviation]
Genome Res.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
[Other-IDs]
NLM/ PMC2045152
59.
Atallah E, Abrams J, Ayash L, Bentley G, Abidi M, Ratanatharathorn V, Uberti J:
Long term follow-up of allogeneic stem cell transplantation in patients with myelodysplastic syndromes using busulfan, cytosine arabinoside, and cyclophosphamide.
Am J Hematol
; 2010 Aug;85(8):579-83
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[Title]
Long term follow-up of allogeneic stem
cell
transplantation in patients with myelodysplastic syndromes using busulfan, cytosine arabinoside, and cyclophosphamide.
We report here the 10-year follow-up of 86 patients who underwent allogeneic stem
cell
transplantation (ASCT) for myelodysplastic syndrome (MDS).
All patients received the busulfan, cytosine arabinoside, and cyclophosphamide (
BAC
) preparative regimen which consisted of busulfan 16 mg/kg, cytosine arabinoside 8 g/m(2) IV, and cyclophosphamide 120 mg/kg IV.
Fifty-nine patients (69%) had
de
novo MDS; 26 (30%) had secondary MDS (treatment related), and one had a preceding aplastic anemia which progressed to MDS before transplant.
Younger age (P = 0.05), human leukocyte
antigen
(HLA) match (P = 0.002), good risk cytogenetics (P = 0.008), and having a related donor (P = 0.03) significantly improved overall and RFS in the multivariable analysis.
The long-term follow-up of patients receiving
the BAC
regimen with ASCT in this study indicated durable relapse-free and OS with acceptable toxicity in this group of patients with high-risk features.
[MeSH-major]
Busulfan / therapeutic use. Cyclophosphamide / therapeutic use. Cytarabine / therapeutic use. Myeloablative Agonists / therapeutic use. Myelodysplastic Syndromes / surgery. Stem
Cell
Transplantation. Transplantation Conditioning
[MeSH-minor]
Adolescent. Adult. Aged.
Disease
-Free Survival. Female. Follow-Up Studies. Graft vs Host
Disease
/ mortality. Graft vs Host
Disease
/ prevention & control. Humans. Immunosuppressive Agents / therapeutic use. Infection / mortality. Kaplan-Meier Estimate. Male. Middle Aged. Postoperative Complications / mortality. Recurrence. Retrospective Studies. Transplantation, Homologous. Treatment Outcome. Young Adult
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consumer health - Myelodysplastic Syndromes
.
Hazardous Substances Data Bank.
CYTARABINE
.
Hazardous Substances Data Bank.
CYCLOPHOSPHAMIDE
.
Hazardous Substances Data Bank.
BUSULFAN
.
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[Copyright]
(c) 2010 Wiley-Liss, Inc.
[Cites]
Blood Rev. 2000 Jun;14(2):63-77
[
10913969.001
]
[Cites]
Blood. 2006 Aug 1;108(3):836-46
[
16597592.001
]
[Cites]
Blood. 2002 Mar 15;99(6):1943-51
[
11877264.001
]
[Cites]
Biol Blood Marrow Transplant. 2002;8(3):161-9
[
11939606.001
]
[Cites]
Blood. 2002 Aug 15;100(4):1201-7
[
12149198.001
]
[Cites]
Blood. 2002 Sep 15;100(6):1997-2004
[
12200358.001
]
[Cites]
Bone Marrow Transplant. 1992 Jan;9(1):41-7
[
1543948.001
]
[Cites]
Bone Marrow Transplant. 1992 Jan;9(1):49-55
[
1543949.001
]
[Cites]
Blood. 1993 Apr 15;81(8):2194-9
[
8471779.001
]
[Cites]
Blood. 1993 Jul 15;82(2):677-81
[
8329721.001
]
[Cites]
J Clin Oncol. 1996 Jan;14(1):220-6
[
8558201.001
]
[Cites]
Blood. 1996 Jul 1;88(1):358-65
[
8704196.001
]
[Cites]
Lancet Oncol. 2009 Mar;10(3):223-32
[
19230772.001
]
[Cites]
Blood. 1997 Mar 15;89(6):2079-88
[
9058730.001
]
[Cites]
Blood. 1998 Sep 15;92(6):1910-7
[
9731047.001
]
[Cites]
Stat Med. 1999 Mar 30;18(6):695-706
[
10204198.001
]
[Cites]
N Engl J Med. 2005 Feb 10;352(6):549-57
[
15703420.001
]
[Cites]
Blood. 2005 May 1;105(9):3420-7
[
15572587.001
]
[Cites]
Cancer. 2006 Apr 15;106(8):1794-803
[
16532500.001
]
[Cites]
Am J Hematol. 2001 Aug;67(4):227-33
[
11443634.001
]
(PMID = 20578198.001).
[ISSN]
1096-8652
[Journal-full-title]
American journal of hematology
[ISO-abbreviation]
Am. J. Hematol.
[Language]
eng
[Grant]
United States / NCI NIH HHS / CA / P30 CA022453
[Publication-type]
Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Immunosuppressive Agents; 0 / Myeloablative Agonists; 04079A1RDZ / Cytarabine; 8N3DW7272P / Cyclophosphamide; G1LN9045DK / Busulfan
[Other-IDs]
NLM/ NIHMS513633; NLM/ PMC3800118
60.
Bossolini E, Krattinger SG, Keller B:
Development of simple sequence repeat markers specific for the Lr34 resistance region of wheat using sequence information from rice and Aegilops tauschii.
Theor Appl Genet
; 2006 Oct;113(6):1049-62
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To derive new polymorphic markers,
bacterial
artificial chromosome (
BAC
) clones representing a total physical size of approximately 1 Mb and belonging to four contigs were isolated from Ae. tauschii by hybridization screening with wheat ESTs.
Several
BAC
clones were low-pass sequenced, resulting in a total of approximately 560 kb of sequence.
[MeSH-minor]
Alleles. Chromosome Mapping. Chromosomes, Artificial,
Bacterial
. Expressed Sequence Tags. Genetic Markers. Genome, Plant. Immunity, Innate / genetics. Microsatellite Repeats. Nucleic Acid Hybridization. Oryza / genetics. Polymorphism, Genetic. Polyploidy. Sequence Analysis, DNA. Triticum / genetics
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[Cites]
Genome. 2000 Aug;43(4):689-97
[
10984182.001
]
[Cites]
Phytopathology. 2004 Oct;94(10):1036-41
[
18943790.001
]
[Cites]
Theor Appl Genet. 2004 Feb;108(3):477-84
[
14523520.001
]
[Cites]
Phytopathology. 2003 Jul;93(7):881-90
[
18943170.001
]
[Cites]
Theor Appl Genet. 2002 Oct;105(5):699-707
[
12582483.001
]
[Cites]
Proc Natl Acad Sci U S A. 1998 Jan 6;95(1):370-5
[
9419382.001
]
[Cites]
Curr Opin Plant Biol. 2003 Apr;6(2):128-33
[
12667868.001
]
[Cites]
Nucleic Acids Res. 2002 Jan 1;30(1):98-102
[
11752265.001
]
[Cites]
Funct Integr Genomics. 2004 Mar;4(1):47-58
[
14767678.001
]
[Cites]
Theor Appl Genet. 2005 Aug;111(4):731-5
[
15965649.001
]
[Cites]
Genomics. 1987 Oct;1(2):174-81
[
3692487.001
]
[Cites]
Plant Cell. 2003 May;15(5):1186-97
[
12724543.001
]
[Cites]
Genome Res. 2005 Apr;15(4):526-36
[
15805493.001
]
[Cites]
Theor Appl Genet. 2005 Feb;110(3):550-60
[
15655666.001
]
[Cites]
Theor Appl Genet. 2005 Nov;111(8):1489-94
[
16187119.001
]
[Cites]
Theor Appl Genet. 2003 Nov;107(7):1235-42
[
12898031.001
]
[Cites]
Theor Appl Genet. 2002 Oct;105(5):736-744
[
12582487.001
]
[Cites]
Genome. 2003 Oct;46(5):870-8
[
14608404.001
]
[Cites]
Genet Res. 2005 Apr;85(2):93-100
[
16174327.001
]
[Cites]
Genome Res. 2003 Aug;13(8):1818-27
[
12902377.001
]
[Cites]
Genetics. 1998 Aug;149(4):2007-23
[
9691054.001
]
[Cites]
Genetics. 2003 Jun;164(2):673-83
[
12807788.001
]
[Cites]
Plant Mol Biol. 2002 Mar-Apr;48(5-6):821-7
[
11999852.001
]
[Cites]
Theor Appl Genet. 2004 Oct;109(6):1105-14
[
15490101.001
]
[Cites]
Plant Physiol. 2001 Mar;125(3):1342-53
[
11244114.001
]
[Cites]
Ann Bot. 2002 Jan;89(1):3-10
[
12096816.001
]
[Cites]
Genomics. 2003 Sep;82(3):378-89
[
12906862.001
]
[Cites]
Theor Appl Genet. 2002 Aug;105(2-3):413-422
[
12582546.001
]
[Cites]
Proc Natl Acad Sci U S A. 2000 Nov 21;97(24):13436-41
[
11078510.001
]
[Cites]
Plant Cell. 2002 Sep;14(9):2095-106
[
12215508.001
]
[Cites]
J Hered. 2002 Jan-Feb;93(1):77-8
[
12011185.001
]
[Cites]
J Hered. 1946 Mar;37:81 107
[
20985728.001
]
[Cites]
Genome Res. 2003 May;13(5):753-63
[
12695326.001
]
[Cites]
Nucleic Acids Res. 1997 Sep 1;25(17):3389-402
[
9254694.001
]
(PMID = 16896711.001).
[ISSN]
0040-5752
[Journal-full-title]
TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik
[ISO-abbreviation]
Theor. Appl. Genet.
[Language]
eng
[Publication-type]
Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Germany
[Chemical-registry-number]
0 / Genetic Markers
61.
Athanasiou A, Vanel D, El Mesbahi O, Theodore C, Fizazi K:
Non-germ cell tumours arising in germ cell tumours (teratoma with malignant transformation) in men: CT and MR findings.
Eur J Radiol
; 2009 Feb;69(2):230-5
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[Title]
Non-germ
cell
tumours arising in germ
cell
tumours (teratoma with
malignant
transformation) in men: CT and MR findings.
PURPOSE: To describe the imaging findings of germ
cell
tumours (GCT) containing non-germ
cell malignant
components (also designated teratoma with
malignant
transformation or TMT).
PATIENTS AND METHODS: The records of 14 male patients with GCT and a non-germ
cell
histological component TMT were retrospectively reviewed.
Sarcoma was identified in 10 out of 14 patients, with rhabdomyosarcoma ranking first (n=4), followed by osteosarcoma (n=2), fusiform
cell
sarcoma (n=1), undifferentiated sarcoma (n=1), neurosarcoma (n=1) and myxoid sarcoma (n=1).
Other histological types
of malignant
transformation included
adenocarcinoma
(n=3) and
bronchoalveolar carcinoma
(n=1).
RESULTS: Non-GCT
malignant
transformation was identified in the retroperitoneum (5), testis (3), mediastinum (3), peritoneum (2) and lungs (1).
The CT and MR imaging findings before treatment and after relapse were evaluated with emphasis on imaging features that could possibly imply the presence
of malignant
transformation (heterogeneously enhancing soft-tissue masses, ossified masses with calcified lymph nodes, diffuse epiploic thickening associated with ascites and peritoneal nodules, pulmonary
alveolar
infiltration with septal thickening).
[MeSH-major]
Cell
Transformation, Neoplastic / pathology. Magnetic Resonance Imaging. Teratoma /
diagnosis
. Tomography, X-Ray Computed
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(PMID = 19056194.001).
[ISSN]
1872-7727
[Journal-full-title]
European journal of radiology
[ISO-abbreviation]
Eur J Radiol
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Ireland
62.
Hein I, Williamson S, Russell J, Powell W:
Isolation of high molecular weight DNA suitable for BAC library construction from woody perennial soft-fruit species.
Biotechniques
; 2005 Jan;38(1):69-71
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[Title]
Isolation of high molecular weight DNA suitable for
BAC
library construction from woody perennial soft-fruit species.
Extracted DNA was readily digested by restriction enzymes and, as shown for raspberry, suitable for
bacterial
artificial chromosome (
BAC
) library construction.
[MeSH-major]
Chromosomes, Artificial,
Bacterial
/ genetics. DNA, Plant / chemistry. DNA, Plant / isolation & purification. Gene Library. Plant Leaves / genetics. Rosales / genetics. Ultrafiltration / methods
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(PMID = 15679088.001).
[ISSN]
0736-6205
[Journal-full-title]
BioTechniques
[ISO-abbreviation]
BioTechniques
[Language]
eng
[Publication-type]
Evaluation Studies; Journal Article; Technical Report
[Publication-country]
United States
[Chemical-registry-number]
0 / DNA, Plant
63.
Rhoden CR, Ghelfi E, González-Flecha B:
Pulmonary inflammation by ambient air particles is mediated by superoxide anion.
Inhal Toxicol
; 2008 Jan;20(1):11-5
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Lung
inflammation is a key response to increased levels of particulate air pollution (PM); however, the cellular mechanisms leading to this response remain poorly understood.
Here we tested the possible role
of a
specific oxidant, superoxide anion, by using the membrane-permeable analog of superoxide dismutase, Mn(III) tetrakis(4-benzoic acid)porphyrin chloride (MnTBAP).
Recruitment of inflammatory cells into
bronchoalveolar
lavage was evaluated 4 h after instillation.
Rats exposed to UAP showed significant increases in the total
cell
number (8.9 +/- 0.6 x 10(6); sham: 5.1 +/- 0.6 x 10(6), p < .02), the numbers of polymorphonuclear leukocytes (26 +/- 4%; sham: 6 +/- 1%, p < .0001), protein levels (1.2 +/- 0.5 mg/ml, sham: 0.4 +/- 0.1 mg/ml, p < .001), and a trend of increase in myeloperoxidase levels (5 +/- 1; sham: 2 +/- 1 mU/ml) in
bronchoalveolar
lavage (BAL).
These data indicate that superoxide anion is a critical mediator of the inflammatory response elicited by PM deposition in
the lung
.
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(PMID = 18236216.001).
[ISSN]
1091-7691
[Journal-full-title]
Inhalation toxicology
[ISO-abbreviation]
Inhal Toxicol
[Language]
eng
[Grant]
United States / NIEHS NIH HHS / ES / R01 HL/ES68073
[Publication-type]
Comparative Study; Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
United States
[Chemical-registry-number]
0 / Free Radicals; 0 / Particulate Matter; 11062-77-4 / Superoxides
64.
Bruckner L, Gigliotti F, Wright T, Harmsen A, Notter RH, Chess P, Wang Z, Finkelstein J:
Pneumocystis carinii infection sensitizes lung to radiation-induced injury after syngeneic marrow transplantation: role of CD4+ T cells.
Am J Physiol Lung Cell Mol Physiol
; 2006 Jun;290(6):L1087-96
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[Title]
Pneumocystis carinii infection sensitizes
lung
to radiation-induced injury after syngeneic marrow transplantation: role of CD4+ T cells.
A murine model of bone marrow transplant (BMT)-related
lung
injury was developed to study how infection sensitizes
lung
to the damaging effects of total body irradiation (TBI) at infectious and TBI doses that individually do not cause injury.
Mice infected with Pneumocystis carinii exhibited an asymptomatic, rapid, and transient influx of eosinophils and T cells in
bronchoalveolar
lavage fluid (BALF).
BALF from P. carinii/TBI mice contained a disproportionate initial influx of CD4(+) T cells (CD4(+):CD8(+) ratio of 2.7) that correlated with progressive
lung
injury (from 8 to 17 days post-BMT).
In vivo depletion of CD4(+) T cells in P. carinii/TBI mice abrogated pulmonary dysfunction and reduced TNF-alpha levels in BALF, whereas depletion of CD8(+) T cells did not affect
lung
compliance or TNF-alpha.
Lung
injury was not attributable to direct P. carinii damage, since CD4-depleted P. carinii/TBI mice that exhibited no injury had higher average
lung P
. carinii burdens than either mice given P. carinii alone or undepleted P. carinii/TBI mice.
Together, these results indicate that P. carinii infection can sensitize
the lung
to subsequent TBI-mediated
lung
injury via a process dependent on non-alloreactive CD4(+) T cells.
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(PMID = 16399785.001).
[ISSN]
1040-0605
[Journal-full-title]
American journal of physiology. Lung cellular and molecular physiology
[ISO-abbreviation]
Am. J. Physiol. Lung Cell Mol. Physiol.
[Language]
eng
[Grant]
United States / NHLBI NIH HHS / HL / HL 71659; United States / NHLBI NIH HHS / HL / R01 HL 57176
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
United States
65.
Huang XQ, Cloutier S:
Molecular characterization and genomic organization of low molecular weight glutenin subunit genes at the Glu-3 loci in hexaploid wheat (Triticum aestivum L.).
Theor Appl Genet
; 2008 May;116(7):953-66
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Eighty-two positive
BAC
clones were identified to contain LMW-GS genes from the hexaploid wheat 'Glenlea'
BAC
library via filter hybridization and PCR validation.
Twelve unique LMW glutenin genes and seven pseudogenes were isolated from these positive
BAC
clones by primer-template mismatch PCR and subsequent primer walking using hemi-nested touchdown PCR.
These genes were sequenced and each consisted
of a
single-open reading frame (ORF) and untranslated 5' and 3' flanking regions.
On the basis of their N-
terminal
sequences, they were classified into nine groups.
Fingerprinting of the 82
BAC
clones indicated 30
BAC
clones assembled into eight contigs, while the remaining clones were singletons.
BAC
end sequencing of the 82 clones revealed that long
terminal
repeat (LTR) retrotransposons were abundant in the Glu-3 regions.
Alignments of sequences indicated that the same type (starting with the same N-
terminal
sequence) LMW-GS genes were highly conserved in the homologous genomes between hexaploid wheat and its donors such as durum wheat and T. tauschii.
[MeSH-minor]
Amino Acid Sequence. Chromosomes, Artificial,
Bacterial
. Gene Library. Molecular Sequence Data. Molecular Weight. Phylogeny. Polymerase Chain Reaction. Retroelements. Sequence Homology, Amino Acid
Hazardous Substances Data Bank.
GLUTEN
.
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[Cites]
Theor Appl Genet. 2004 May;108(7):1409-19
[
14727031.001
]
[Cites]
BMC Genet. 2007 May 04;8:18
[
17480230.001
]
[Cites]
Genome. 2005 Jun;48(3):401-10
[
16121237.001
]
[Cites]
Plant Mol Biol. 2004 Jan;54(1):55-69
[
15159634.001
]
[Cites]
Theor Appl Genet. 2004 Sep;109 (5):1028-40
[
15164175.001
]
[Cites]
Theor Appl Genet. 2006 Jan;112(2):327-34
[
16283233.001
]
[Cites]
Theor Appl Genet. 2006 Aug;113(3):383-95
[
16775696.001
]
[Cites]
Theor Appl Genet. 2005 Nov;111(7):1251-9
[
16187122.001
]
[Cites]
Genetics. 2006 Nov;174(3):1493-504
[
17028342.001
]
[Cites]
Plant Mol Biol. 2007 Sep;65(1-2):189-203
[
17629796.001
]
[Cites]
Plant Cell. 2003 May;15(5):1186-97
[
12724543.001
]
[Cites]
Plant Physiol. 1998 Dec;118(4):1147-58
[
9847089.001
]
[Cites]
Funct Integr Genomics. 2002 May;2(1-2):70-80
[
12021852.001
]
[Cites]
Genome. 2003 Oct;46(5):870-8
[
14608404.001
]
[Cites]
Bioinformatics. 2003 Dec 12;19(18):2496-7
[
14668244.001
]
[Cites]
Theor Appl Genet. 1994 Apr;88(1):81-8
[
24185886.001
]
[Cites]
Methods Mol Biol. 2000;132:365-86
[
10547847.001
]
[Cites]
Plant Physiol. 2004 May;135(1):459-70
[
15122014.001
]
[Cites]
Theor Appl Genet. 2007 Feb;114(3):451-60
[
17106734.001
]
[Cites]
Theor Appl Genet. 2004 Aug;109(3):648-57
[
15103408.001
]
[Cites]
Theor Appl Genet. 1989 Jan;77(1):57-64
[
24232474.001
]
[Cites]
Theor Appl Genet. 1990 Jul;80(1):65-74
[
24220812.001
]
[Cites]
Genomics. 2003 Sep;82(3):378-89
[
12906862.001
]
[Cites]
Funct Integr Genomics. 2003 Mar;3(1-2):56-68
[
12590343.001
]
[Cites]
Brief Bioinform. 2004 Jun;5(2):150-63
[
15260895.001
]
[Cites]
Nucleic Acids Res. 1994 Nov 11;22(22):4673-80
[
7984417.001
]
[Cites]
Theor Appl Genet. 2002 Mar;104(4):680-687
[
12582674.001
]
[Cites]
Genome Res. 1999 Sep;9(9):868-77
[
10508846.001
]
[Cites]
Genome Res. 1998 Mar;8(3):175-85
[
9521921.001
]
[Cites]
Theor Appl Genet. 2006 Nov;113(7):1247-59
[
16941095.001
]
[Cites]
Mol Gen Genet. 1989 Mar;216(1):81-90
[
2733691.001
]
(PMID = 18305921.001).
[ISSN]
0040-5752
[Journal-full-title]
TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik
[ISO-abbreviation]
Theor. Appl. Genet.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Germany
[Chemical-registry-number]
0 / Retroelements; 8002-80-0 / Glutens; FX065C7O71 / glutenin
66.
Mackay A, Tamber N, Fenwick K, Iravani M, Grigoriadis A, Dexter T, Lord CJ, Reis-Filho JS, Ashworth A:
A high-resolution integrated analysis of genetic and expression profiles of breast cancer cell lines.
Breast Cancer Res Treat
; 2009 Dec;118(3):481-98
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[Title]
A high-resolution integrated analysis of genetic and expression profiles of breast
cancer cell
lines.
Tumour
cell
lines derived from breast
cancer
patients constitute one of the cornerstones of breast
cancer
research.
To characterise breast
cancer cell
lines at the genetic level, we have developed a full tiling path
bacterial
artificial chromosome (
BAC
) array collection for comparative genomic hybridisation (aCGH).
This aCGH
BAC
collection covers 98% of the entire human genome at a resolution of 40-60 kbp.
We have used this platform alongside an in-house produced 17 K cDNA microarray set to characterise the genetic and transcriptomic profiles of 24 breast
cancer cell
lines, as well as
cell
types derived from non-diseased breast.
We demonstrate that breast
cancer cell
lines have genomic and transcriptomic features that recapitulate those of primary breast cancers and can be reliably subclassified into basal-like and luminal subgroups.
By overlaying aCGH and transcriptomic data, we have identified 753 genes whose expression correlate with copy number; this list comprised numerous oncogenes recurrently amplified and overexpressed in breast
cancer
(e.g., HER2, MYC, CCND1 and AURKA).
Finally, we demonstrate that although breast
cancer cell
lines have genomic features usually found in grade III breast cancers (i.e., gains of 1q, 8q and 20q), basal-like and luminal
cell
lines are characterised by distinct genomic aberrations.
[MeSH-major]
Breast Neoplasms / genetics.
Cell
Line, Tumor / physiology. Gene Expression Profiling
[MeSH-minor]
Chromosomes, Artificial,
Bacterial
. Cluster Analysis. Comparative Genomic Hybridization. Female. Gene Expression. Humans. Oligonucleotide Array Sequence Analysis
Genetic Alliance.
consumer health - Breast Cancer
.
MedlinePlus Health Information.
consumer health - Breast Cancer
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTAC Assay Portal.
NCI CPTAC Assay Portal
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
NCI CPTC Antibody Characterization Program.
NCI CPTC Antibody Characterization Program
.
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.
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(PMID = 19169812.001).
[ISSN]
1573-7217
[Journal-full-title]
Breast cancer research and treatment
[ISO-abbreviation]
Breast Cancer Res. Treat.
[Language]
eng
[Grant]
United Kingdom / Cancer Research UK / /
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Netherlands
67.
Satoh N, Kawashima T, Shoguchi E, Satou Y:
Urochordate genomes.
Genome Dyn
; 2006;2:198-212
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The C
. intestinalis genome is composed of approximately 160 Mbp, similar to other invertebrate genomes, and contains approximately 16,000 protein-coding genes that represent the basic set of chordate genes without the extensive gene duplications seen in vertebrates.
The C
. intestinalis gene models are intensively annotated and supported by corresponding cDNAs.
With the aid of two-color fluorescent in situ hybridization
of BAC
clones, approximately 65% of the assembled genome information has been mapped onto the 14 pairs
of C
. intestinalis chromosomes.
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(PMID = 18753780.001).
[ISSN]
1660-9263
[Journal-full-title]
Genome dynamics
[ISO-abbreviation]
Genome Dyn
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't; Review
[Publication-country]
Switzerland
[Number-of-references]
30
68.
Kim DW, Chae SH, Kang BR, Choi SH, Kim A, Woo S, Park HS:
Comparative genomic analysis of the whale (Pseudorca crassidens) PRNP locus.
Genome
; 2008 Jun;51(6):452-64
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In this paper, we sequenced three novel
BAC
clones corresponding to a total length of 308 kb and spanning the PRNP, PRND, and RASSF2 loci, and conducted comparative genomic analysis to examine the genomic structure of the false killer whale PRNP locus.
Our results will provide novel insights into the genomic changes that have occurred during evolution of mammalian PRNP loci, and may also have implications for research into prion
disease
.
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[CommentIn]
Genome. 2008 Dec;51(12):1062
[
19088820.001
]
(PMID = 18521124.001).
[ISSN]
0831-2796
[Journal-full-title]
Genome
[ISO-abbreviation]
Genome
[Language]
ENG
[Publication-type]
Comparative Study; Journal Article
[Publication-country]
Canada
[Chemical-registry-number]
0 / Prions
69.
Zhang ZF, Ruivenkamp C, Staaf J, Zhu H, Barbaro M, Petillo D, Khoo SK, Borg A, Fan YS, Schoumans J:
Detection of submicroscopic constitutional chromosome aberrations in clinical diagnostics: a validation of the practical performance of different array platforms.
Eur J Hum Genet
; 2008 Jul;16(7):786-92
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For several decades etiological
diagnosis of
patients with idiopathic mental retardation (MR) and multiple congenital anomalies (MCA) has relied on chromosome analysis by karyotyping.
Here, we investigated four high-density array platforms with a theoretical resolution < or =100 kb: 33K tiling path
BAC
array, 500K Affymetrix SNP array, 385K NimbleGen oligonucleotide array and 244K Agilent oligonucleotide array for their robustness and implementation in our diagnostic setting.
[MeSH-minor]
Chromosomes, Artificial,
Bacterial
/ genetics. Female. Genome, Human / genetics. Humans. Male. Reproducibility of Results
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(PMID = 18285835.001).
[ISSN]
1018-4813
[Journal-full-title]
European journal of human genetics : EJHG
[ISO-abbreviation]
Eur. J. Hum. Genet.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
England
70.
Somboonthum P, Yoshii H, Okamoto S, Koike M, Gomi Y, Uchiyama Y, Takahashi M, Yamanishi K, Mori Y:
Generation of a recombinant Oka varicella vaccine expressing mumps virus hemagglutinin-neuraminidase protein as a polyvalent live vaccine.
Vaccine
; 2007 Dec 17;25(52):8741-55
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[Title]
Generation
of a
recombinant Oka varicella vaccine expressing mumps virus hemagglutinin-neuraminidase protein as a polyvalent live vaccine.
We constructed a recombinant varicella-zoster virus (VZV) Oka vaccine strain (vOka) that contained the mumps virus (MuV) hemagglutinin-neuraminidase (HN) gene, inserted into the site of the ORF 13 gene by using
the bacterial
artificial chromosome (
BAC
) system in Escherichia coli.
Interestingly, the induced antibodies had a strong neutralizing activity against virus-
cell
infections of both MuV and VZV.
[MeSH-minor]
Animals. Antibodies, Viral / blood.
Cell
Line.
Cell
Membrane / chemistry. Chromosomes, Artificial,
Bacterial
. Escherichia coli / genetics. Guinea Pigs. Humans. Male. Microscopy, Electron, Transmission. Microscopy, Immunoelectron. Neuraminidase / genetics. Neuraminidase / immunology. Neutralization Tests. Vaccines, Attenuated / genetics. Vaccines, Attenuated / immunology. Vaccines, Synthetic / genetics. Vaccines, Synthetic / immunology. Virion / chemistry
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(PMID = 18053621.001).
[ISSN]
0264-410X
[Journal-full-title]
Vaccine
[ISO-abbreviation]
Vaccine
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
Netherlands
[Chemical-registry-number]
0 / Antibodies, Viral; 0 / Chickenpox Vaccine; 0 / Hemagglutinins, Viral; 0 / Vaccines, Attenuated; 0 / Vaccines, Synthetic; EC 3.2.1.18 / Neuraminidase
71.
Tallini YN, Brekke JF, Shui B, Doran R, Hwang SM, Nakai J, Salama G, Segal SS, Kotlikoff MI:
Propagated endothelial Ca2+ waves and arteriolar dilation in vivo: measurements in Cx40BAC GCaMP2 transgenic mice.
Circ Res
; 2007 Dec 7;101(12):1300-9
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To study endothelial
cell
(EC)- specific Ca(2+) signaling in vivo we engineered transgenic mice in which the Ca(2+) sensor GCaMP2 is placed under control of endogenous connexin40 (Cx40) transcription regulatory elements within
a bacterial
artificial chromosome (
BAC
), resulting in high sensor expression in arterial ECs, atrial myocytes, and cardiac Purkinje fibers.
High signal/noise Ca(2+) signals were obtained in Cx40(
BAC
)-GCaMP2 mice within the ventricular Purkinje
cell
network in vitro and in ECs of cremaster muscle arterioles in vivo.
At intermediate distances (300 to 600 microm), rapidly-conducted vasodilation occurred without changing EC Ca(2+), and additional dilation occurred after arrival
of a
Ca(2+) wave.
[MeSH-minor]
Animals. Chromosomes, Artificial,
Bacterial
/ genetics. Chromosomes, Artificial,
Bacterial
/ physiology. Mice. Mice, Transgenic
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(subscription/membership/fee required).
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gene/protein/disease-specific - KOMP Repository
(subscription/membership/fee required).
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.
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SciCrunch.
Marmoset Gene list: Data: Gene Annotation
.
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(PMID = 17932328.001).
[ISSN]
1524-4571
[Journal-full-title]
Circulation research
[ISO-abbreviation]
Circ. Res.
[Language]
eng
[Grant]
United States / NIDDK NIH HHS / DK / DK65992; United States / NHLBI NIH HHS / HL / F2HL76999; United States / NHLBI NIH HHS / HL / HL057929; United States / NHLBI NIH HHS / HL / HL41026; United States / NHLBI NIH HHS / HL / HL45239; United States / NHLBI NIH HHS / HL / HL56786; United States / NHLBI NIH HHS / HL / HL69097; United States / NHLBI NIH HHS / HL / HL70722
[Publication-type]
Comparative Study; Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Connexins; 0 / Intracellular Calcium-Sensing Proteins; 0 / connexin 40; EC 2.7.11.17 / Calcium-Calmodulin-Dependent Protein Kinase Type 2
72.
Righini ChA, Soriano E, Morel N, Hitter A, Bolla M, Reyt E:
[Combined induction chemotherapy and radiotherapy in case of undifferentiated carcinoma of nasopharynx tumours (UCNT)].
Rev Laryngol Otol Rhinol (Bord)
; 2006;127(4):223-8
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[Title]
[Combined induction chemotherapy and radiotherapy in case of undifferentiated
carcinoma of
nasopharynx tumours (UCNT)].
[Transliterated title]
Traitement par chimiothérapie d'induction et radiothérapie
des
cancers du cavum
de
type UCNT.
OBJECTIVE: The objectives of our study were to consider the morbidity and the effectiveness of combined induction chemotherapy and radiotherapy in the treatment of Undifferentiated
Carcinoma of
Nasopharynx Tumor (UCNT).
Two types of chemotherapy were administered:
The BAC
regime (Bleomycin, Adriamycin, Cisplatinum) and the FUCIFOL regime (Fluorouracil, Cispaltinum, Elvorin).
The BAC
regime was the most effective.
The overall
disease
free survival rates at 3 years were respectively 78% and 69%.
[MeSH-major]
Antineoplastic Agents / therapeutic use.
Carcinoma
/ drug therapy.
Carcinoma
/ radiotherapy. Nasopharyngeal Neoplasms / drug therapy. Nasopharyngeal Neoplasms / radiotherapy
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(PMID = 17315786.001).
[ISSN]
0035-1334
[Journal-full-title]
Revue de laryngologie - otologie - rhinologie
[ISO-abbreviation]
Rev Laryngol Otol Rhinol (Bord)
[Language]
fre
[Publication-type]
English Abstract; Journal Article
[Publication-country]
France
[Chemical-registry-number]
0 / Antineoplastic Agents
73.
Fillmore MT, Marczinski CA, Bowman AM:
Acute tolerance to alcohol effects on inhibitory and activational mechanisms of behavioral control.
J Stud Alcohol
; 2005 Sep;66(5):663-72
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OBJECTIVE: Acute alcohol tolerance refers to the observation of reduced impairment at a given blood alcohol concentration (
BAC
) on the descending versus ascending limb of the blood alcohol curve.
This study examined the expression of acute alcohol tolerance to impaired behavioral control in terms of changes in a drinker's ability to activate and inhibit behavioral responses as
BAC
ascended and declined following a dose.
The development of acute tolerance was measured by testing behavioral control twice: once during the ascending phase and again at comparable
BACs
during the descending phase of the blood alcohol curve.
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.
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ETHANOL
.
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(PMID = 16331852.001).
[ISSN]
0096-882X
[Journal-full-title]
Journal of studies on alcohol
[ISO-abbreviation]
J. Stud. Alcohol
[Language]
eng
[Grant]
United States / NIAAA NIH HHS / AA / R01 AA12895
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural
[Publication-country]
United States
[Chemical-registry-number]
3K9958V90M / Ethanol
74.
Varet J, Douglas SK, Gilmartin L, Medford AR, Bates DO, Harper SJ, Millar AB:
VEGF in the lung: a role for novel isoforms.
Am J Physiol Lung Cell Mol Physiol
; 2010 Jun;298(6):L768-74
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[Title]
VEGF in
the lung
: a role for novel isoforms.
Vascular endothelial
cell
growth factor (VEGF) is a potent mitogen and permogen that increases in the plasma and decreases in the
alveolar
space in respiratory diseases such as acute respiratory distress syndrome (ARDS).
This observation has led to controversy over the role of this potent molecule in
lung
physiology and
disease
.
We hypothesized that some of the VEGF previously detected in normal
lung
may be of the anti-angiogenic family (VEGF(xxx)b) with significant potential effects on VEGF bioactivity.
Expression of VEGF(xxx)b was also detected by immunoblotting in normal
lung
tissue, primary human
alveolar
type II (ATII) cells, and
bronchoalveolar
lavage (BAL) fluid in normal subjects and by ELISA in normal, "at risk," and ARDS subjects.
The effect of VEGF(165) and VEGF(165)b on both human primary endothelial cells and
alveolar
epithelial
cell
proliferation was assessed by [(3)H]thymidine uptake.
We found that VEGF(165)b was widely expressed in normal healthy
lung
tissue but is reduced in ARDS
lung
.
VEGF(121)b and VEGF(165)b were present in whole
lung
, BAL, and ATII lysate.
The proliferative effect of VEGF(165) on both human primary endothelial cells and human
alveolar
epithelial cells was significantly inhibited by VEGF(165)b (P < 0.01).
These data demonstrate that the novel VEGF(xxx)b family members are expressed in normal
lung
and are reduced in ARDS.
A specific functional effect on primary human endothelial and
alveolar
epithelial cells has also been shown.
These data suggest that the VEGF(xxx)b family may have a role in repair after
lung
injury.
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[Cites]
Gene Ther. 2004 Mar;11(6):512-21
[
14999223.001
]
[Cites]
Trends Biochem Sci. 2003 Sep;28(9):488-94
[
13678960.001
]
[Cites]
Mol Med. 2004 Jan-Jun;10(1-6):12-8
[
15502878.001
]
[Cites]
J Biol Chem. 1991 Jun 25;266(18):11947-54
[
1711045.001
]
[Cites]
J Biol Chem. 1996 Mar 8;271(10):5761-7
[
8621443.001
]
[Cites]
Am J Physiol. 1996 Dec;271(6 Pt 2):H2520-8
[
8997313.001
]
[Cites]
Cancer Res. 2004 Nov 1;64(21):7822-35
[
15520188.001
]
[Cites]
Eur Respir J. 2005 Jan;25(1):139-46
[
15640335.001
]
[Cites]
Thorax. 2005 Feb;60(2):153-8
[
15681505.001
]
[Cites]
Thorax. 2006 Jul;61(7):621-6
[
16807391.001
]
[Cites]
Cell Mol Life Sci. 2006 Sep;63(17):2067-77
[
16909199.001
]
[Cites]
Br J Cancer. 2008 Apr 22;98(8):1366-79
[
18349829.001
]
[Cites]
Cancer Res. 2008 Jun 15;68(12):4683-92
[
18559514.001
]
[Cites]
FASEB J. 2008 Aug;22(8):3078-86
[
18467594.001
]
[Cites]
Nat Rev Cancer. 2008 Nov;8(11):880-7
[
18923433.001
]
[Cites]
Nephron Physiol. 2008;110(4):p57-67
[
19039247.001
]
[Cites]
Respir Res. 2009;10:27
[
19358726.001
]
[Cites]
J Crit Care. 2009 Jun;24(2):236-42
[
19327291.001
]
[Cites]
Am J Respir Crit Care Med. 2009 Sep 1;180(5):396-406
[
19520907.001
]
[Cites]
Mol Med. 2001 Apr;7(4):240-6
[
11471568.001
]
[Cites]
Am J Respir Cell Mol Biol. 2000 Jun;22(6):657-64
[
10837361.001
]
[Cites]
J Cell Sci. 2001 Mar;114(Pt 5):853-65
[
11181169.001
]
[Cites]
J Biol Chem. 2001 Jun 1;276(22):18688-94
[
11278319.001
]
[Cites]
Eur Respir J. 2001 Jul;18(1):100-6
[
11510779.001
]
[Cites]
Am J Respir Crit Care Med. 2001 Nov 1;164(9):1601-5
[
11719296.001
]
[Cites]
Cancer Res. 2002 Jul 15;62(14):4123-31
[
12124351.001
]
[Cites]
Am J Respir Crit Care Med. 2002 Aug 1;166(3):382-5
[
12153975.001
]
[Cites]
Am J Respir Crit Care Med. 2002 Nov 15;166(10):1332-7
[
12421742.001
]
[Cites]
J Appl Physiol (1985). 2003 May;94(5):1836-40
[
12524373.001
]
[Cites]
Am J Physiol Renal Physiol. 2003 Jun;284(6):F1263-73
[
12620928.001
]
[Cites]
Nat Med. 2003 Jun;9(6):669-76
[
12778165.001
]
[Cites]
Am J Respir Cell Mol Biol. 2004 Aug;31(2):241-5
[
15044215.001
]
(PMID = 20228180.001).
[ISSN]
1522-1504
[Journal-full-title]
American journal of physiology. Lung cellular and molecular physiology
[ISO-abbreviation]
Am. J. Physiol. Lung Cell Mol. Physiol.
[Language]
ENG
[Grant]
United Kingdom / Wellcome Trust / / 074702; United Kingdom / British Heart Foundation / / BS/006/005
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Protein Isoforms; 0 / VEGFA protein, human; 0 / Vascular Endothelial Growth Factor A; 0 / Vascular Endothelial Growth Factors
[Other-IDs]
NLM/ PMC2886605
75.
Wu Y, Zhou H, Xu Y, Li S:
Enhanced expression of urocortin in lung tissues of rats with allergic asthma.
Biochem Biophys Res Commun
; 2006 Mar 10;341(2):532-40
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[Title]
Enhanced expression of urocortin in
lung
tissues of rats with allergic asthma.
Bronchial asthma is defined as a chronic airway inflammatory
disease
characterized by sustained activation of many inflammatory cells including mast cells.
On the other hand, UCN can induce mast
cell
degranulation and generation of many proinflammatory factors.
The purpose of this study was to examine the expression profile of UCN in rat
lung
with allergic asthma.
In contrast, treatment with dexamethasone resulted in markedly ameliorated airway inflammation and alleviated airway inflammatory
cell
infiltration, coupled with a significantly decreased urocortin expression.
Regression analysis revealed a positive correlation between urocortin expression and the number of inflammatory cells in
bronchoalveolar
lavage fluid (P<0.01).
Glucocorticoid treatment markedly reduced the production of UCN in asthmatic
lung
tissues.
Peripherally produced UCN in
lung
may act as a possible local autocrine and paracrine immune-inflammatory mediator in inflammatory airway of allergic asthma rats.
[MeSH-major]
Asthma / metabolism. Corticotropin-Releasing Hormone / biosynthesis. Corticotropin-Releasing Hormone / chemistry. Gene Expression Regulation. Hypersensitivity / metabolism.
Lung
/ metabolism
[MeSH-minor]
Actins / metabolism. Animals. Blotting, Western. Bronchi / pathology.
Bronchoalveolar
Lavage. Dexamethasone / pharmacology.
Disease
Models, Animal. Epithelium / metabolism. Glucocorticoids / metabolism. Immunohistochemistry. Inflammation. Male. Mast Cells / metabolism. Models, Statistical. Ovalbumin / metabolism. Peptides / chemistry. RNA, Messenger / metabolism. Rats. Rats, Sprague-Dawley. Reverse Transcriptase Polymerase Chain Reaction. Time Factors. Up-Regulation. Urocortins
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DEXAMETHASONE
.
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(PMID = 16427607.001).
[ISSN]
0006-291X
[Journal-full-title]
Biochemical and biophysical research communications
[ISO-abbreviation]
Biochem. Biophys. Res. Commun.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / Actins; 0 / Glucocorticoids; 0 / Peptides; 0 / RNA, Messenger; 0 / Urocortins; 7S5I7G3JQL / Dexamethasone; 9006-59-1 / Ovalbumin; 9015-71-8 / Corticotropin-Releasing Hormone
76.
Politi K, Zakowski MF, Fan PD, Schonfeld EA, Pao W, Varmus HE:
Lung adenocarcinomas induced in mice by mutant EGF receptors found in human lung cancers respond to a tyrosine kinase inhibitor or to down-regulation of the receptors.
Genes Dev
; 2006 Jun 1;20(11):1496-510
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[Title]
Lung adenocarcinomas
induced in mice by mutant EGF receptors found in human
lung
cancers respond to a tyrosine kinase inhibitor or to down-regulation of the receptors.
Somatic mutations in exons encoding the tyrosine kinase domain of the epidermal growth factor receptor (EGFR) gene are found in human
lung adenocarcinomas
and are associated with sensitivity to the tyrosine kinase inhibitors gefitinib and erlotinib.
To study further the role of these mutations in the initiation and maintenance
of lung cancer
, we have developed transgenic mice that express an exon 19 deletion mutant (EGFR(DeltaL747-S752)) or the L858R mutant (EGFR(L858R)) in type II pneumocytes under the control of doxycycline.
Expression of either EGFR mutant leads to the development
of lung adenocarcinomas
.
Two weeks after induction with doxycycline, mice that express the EGFR(L858R) allele show diffuse
lung cancer
highly reminiscent of human
bronchioloalveolar carcinoma
and later develop interspersed multifocal
adenocarcinomas
.
In contrast, mice expressing EGFR(DeltaL747-S752) develop multifocal tumors embedded in normal
lung
parenchyma with a longer latency.
These models may be useful for developing improved therapies for patients with
lung
cancers bearing EGFR mutations.
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[Cites]
Proc Natl Acad Sci U S A. 2005 May 24;102(21):7665-70
[
15897464.001
]
[Cites]
Proc Natl Acad Sci U S A. 2005 Mar 8;102(10):3788-93
[
15731348.001
]
[Cites]
PLoS Med. 2005 Jan;2(1):e17
[
15696205.001
]
[Cites]
PLoS Med. 2005 Mar;2(3):e73
[
15737014.001
]
[Cites]
N Engl J Med. 2005 Jul 14;353(2):133-44
[
16014883.001
]
[Cites]
Nature. 2005 Aug 4;436(7051):642
[
16079833.001
]
[Cites]
J Clin Oncol. 2005 Sep 1;23(25):5900-9
[
16043828.001
]
[Cites]
Cancer Cell. 2005 Sep;8(3):197-209
[
16169465.001
]
[Cites]
Cancer Res. 2005 Oct 1;65(19):8968-74
[
16204070.001
]
[Cites]
Cancer Res. 2005 Dec 15;65(24):11478-85
[
16357156.001
]
[Cites]
Clin Cancer Res. 2006 Feb 1;12(3 Pt 1):839-44
[
16467097.001
]
[Cites]
PLoS Med. 2005 Nov;2(11):e313
[
16187797.001
]
[Cites]
Oncogene. 2006 Mar 30;25(14):2105-12
[
16288213.001
]
[Cites]
Cancer Cell. 2006 Jun;9(6):485-95
[
16730237.001
]
[Cites]
Cold Spring Harb Symp Quant Biol. 2005;70:1-9
[
16869733.001
]
[Cites]
Proc Natl Acad Sci U S A. 2000 Mar 28;97(7):3444-9
[
10716706.001
]
[Cites]
J Biol Chem. 2000 Apr 21;275(16):11858-64
[
10766812.001
]
[Cites]
Nature. 2001 Apr 26;410(6832):1111-6
[
11323676.001
]
[Cites]
Genes Dev. 2001 Dec 15;15(24):3249-62
[
11751631.001
]
[Cites]
Ann Thorac Surg. 2002 Nov;74(5):1640-6; discussion 1646-7
[
12440623.001
]
[Cites]
J Biol Chem. 2002 Nov 29;277(48):46265-72
[
12196540.001
]
[Cites]
Genes Dev. 2003 Feb 15;17(4):488-501
[
12600942.001
]
[Cites]
J Clin Oncol. 2003 Oct 15;21(20):3798-807
[
12953099.001
]
[Cites]
Oncogene. 2003 Dec 8;22(56):8999-9006
[
14663478.001
]
[Cites]
J Clin Oncol. 2004 Mar 15;22(6):1103-9
[
15020612.001
]
[Cites]
Curr Opin Genet Dev. 2004 Feb;14(1):37-42
[
15108803.001
]
[Cites]
N Engl J Med. 2004 May 20;350(21):2129-39
[
15118073.001
]
[Cites]
Science. 2004 Jun 4;304(5676):1497-500
[
15118125.001
]
[Cites]
Science. 2004 Aug 20;305(5687):1163-7
[
15284455.001
]
[Cites]
Proc Natl Acad Sci U S A. 2004 Sep 7;101(36):13306-11
[
15329413.001
]
[Cites]
Cancer Res. 2004 Oct 15;64(20):7241-4
[
15492241.001
]
[Cites]
Proc Natl Acad Sci U S A. 1987 Oct;84(19):6899-903
[
3477813.001
]
[Cites]
Proc Natl Acad Sci U S A. 1992 May 15;89(10):4309-13
[
1584765.001
]
[Cites]
Curr Biol. 1994 Jan 1;4(1):1-7
[
7922305.001
]
[Cites]
J Clin Oncol. 2005 Apr 10;23(11):2493-501
[
15710947.001
]
[Cites]
J Clin Oncol. 2005 Apr 10;23(11):2513-20
[
15738541.001
]
[Cites]
J Natl Cancer Inst. 2005 May 4;97(9):643-55
[
15870435.001
]
[Cites]
Clin Cancer Res. 2005 May 15;11(10):3750-7
[
15897572.001
]
[Cites]
J Clin Oncol. 1996 Aug;14(8):2377-86
[
8708731.001
]
[Cites]
J Pharmacol Exp Ther. 1999 Nov;291(2):739-48
[
10525095.001
]
[Cites]
Cancer Cell. 2004 Dec;6(6):577-86
[
15607962.001
]
[Cites]
Clin Cancer Res. 2004 Dec 15;10(24):8195-203
[
15623594.001
]
[Cites]
Cancer Res. 2005 Jan 1;65(1):226-35
[
15665299.001
]
[Cites]
J Clin Oncol. 2005 Feb 1;23(4):857-65
[
15681531.001
]
[Cites]
Clin Cancer Res. 2005 Feb 1;11(3):1167-73
[
15709185.001
]
[Cites]
N Engl J Med. 2005 Feb 24;352(8):786-92
[
15728811.001
]
[Cites]
J Natl Cancer Inst. 2005 Mar 2;97(5):339-46
[
15741570.001
]
[Cites]
Cell. 2005 Jun 17;121(6):823-35
[
15960971.001
]
(PMID = 16705038.001).
[ISSN]
0890-9369
[Journal-full-title]
Genes & development
[ISO-abbreviation]
Genes Dev.
[Language]
ENG
[Grant]
United States / NCI NIH HHS / CA / K08 CA097980; United States / NCI NIH HHS / CA / R24 CA083084; United States / NCI NIH HHS / CA / K08-CA097980
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
United States
[Chemical-registry-number]
0 / DNA Primers; EC 2.7.10.1 / Protein-Tyrosine Kinases; EC 2.7.10.1 / Receptor, Epidermal Growth Factor
[Other-IDs]
NLM/ PMC1475762
77.
Wang C, Yang R, Yue D, Zhang Z:
Expression of FAK and PTEN in bronchioloalveolar carcinoma and lung adenocarcinoma.
Lung
; 2009 Mar-Apr;187(2):104-9
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[Title]
Expression of FAK and PTEN in
bronchioloalveolar carcinoma
and
lung
adenocarcinoma
.
Bronchioloalveolar carcinoma
(
BAC
) is classified as a subset
of lung
adenocarcinoma
but has a distinct clinical presentation, tumor biology, response to therapy, and prognosis compared with other subtypes
of lung
adenocarcinoma
.
This study was designed to investigate the clinicopathological differences between
BAC
and
adenocarcinoma
and the expression of focal adhesion kinase (FAK) and phosphatase and tensin homologue (PTEN) and their clinical significance in
BAC
and
adenocarcinoma
.
A retrospective analysis was performed on 77 patients with
BAC
and 172 patients with pure
adenocarcinoma
seen during the period from January 1998 to December 2000.
Clinicopathological characteristics and survival outcome were reviewed and compared between patients with
BAC
and
adenocarcinoma
.
Lymph node status, clinical symptoms, CT appearance and expression of FAK were different between
BAC
and
adenocarcinoma
.
The overall survival
of BAC
was better than that of
adenocarcinoma
.
In patients with FAK(-), the overall survival was not different between
BAC
and
adenocarcinoma
.
In patients with
adenocarcinoma
, the overall survival was better for FAK(-) compared with FAK(+).
Expression of PTEN had a prognostic significance in patients with
BAC
and
adenocarcinoma
.
BAC
and
adenocarcinoma
have different clinicopathological presentations.
Expression of FAK has some effect on such differences and affects survival
of lung
adenocarcinoma
.
Expression of PTEN can predict outcome of resected
lung
adenocarcinoma
and
BAC
.
[MeSH-major]
Adenocarcinoma
/ enzymology.
Adenocarcinoma
,
Bronchiolo
-
Alveolar
/ enzymology. Biomarkers, Tumor / analysis. Focal Adhesion Kinase 1 / analysis.
Lung
Neoplasms / enzymology. PTEN Phosphohydrolase / analysis
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[CommentIn]
Lung. 2009 May-Jun;187(3):207-8
[
19408043.001
]
[Cites]
Ann Oncol. 2005 Jul;16(7):1076-80
[
15860488.001
]
[Cites]
J Clin Oncol. 2004 May 15;22(10):1878-85
[
15143080.001
]
[Cites]
Eur J Cardiothorac Surg. 2003 May;23(5):818-23
[
12754039.001
]
[Cites]
Clin Cancer Res. 2006 Jun 1;12(11 Pt 1):3233-7
[
16740741.001
]
[Cites]
Front Biosci. 2003 May 01;8:s705-14
[
12700131.001
]
[Cites]
J Biol Chem. 1999 Jul 16;274(29):20693-703
[
10400703.001
]
[Cites]
Cancer Metastasis Rev. 2003 Dec;22(4):359-74
[
12884911.001
]
[Cites]
J Clin Oncol. 2004 Jul 15;22(14 ):2954-63
[
15254063.001
]
[Cites]
Science. 1997 Mar 28;275(5308):1943-7
[
9072974.001
]
[Cites]
Adv Intern Med. 1960;10:329-58
[
13762021.001
]
[Cites]
Cancer. 1994 Feb 15;73(4):1163-70
[
8313318.001
]
[Cites]
Cancer. 1991 Nov 1;68(9):1973-7
[
1655232.001
]
[Cites]
Cancer Res. 2006 Sep 1;66(17 ):8389-96
[
16951148.001
]
[Cites]
Chest. 1997 Jun;111(6):1718-23
[
9187199.001
]
[Cites]
Anticancer Res. 2004 Nov-Dec;24(6):3899-905
[
15736429.001
]
[Cites]
Clin Lung Cancer. 2006 Mar;7(5):313-22
[
16640802.001
]
[Cites]
Cancer Epidemiol Biomarkers Prev. 2006 May;15(5):1021-5
[
16702386.001
]
(PMID = 19242756.001).
[ISSN]
1432-1750
[Journal-full-title]
Lung
[ISO-abbreviation]
Lung
[Language]
eng
[Publication-type]
Comparative Study; Journal Article
[Publication-country]
United States
[Chemical-registry-number]
0 / Biomarkers, Tumor; EC 2.7.10.2 / Focal Adhesion Kinase 1; EC 2.7.10.2 / PTK2 protein, human; EC 3.1.3.67 / PTEN Phosphohydrolase; EC 3.1.3.67 / PTEN protein, human
78.
Cui H, Wang Y, Shi X, Tong G, Lan D, He L, Qiu H, Liu C, Wang M:
[Construction of Marek's disease virus serotype 814 strain as an infectious bacterial artificial chromosome].
Sheng Wu Gong Cheng Xue Bao
; 2008 Apr;24(4):569-75
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[Title]
[Construction of Marek's
disease
virus serotype 814 strain as an infectious
bacterial
artificial chromosome].
The aim of this study was to construct the complete genome of Marek's
disease
virus serotype 814 strain as an infectious
bacterial
artificial chromosome (
BAC
).
Using self-designed selection marker Eco-gpt (1.3 kb) and
BAC
vector pBeloBAC11 (7.5 kb), we constructed the transfer plasmid pUAB-gpt-BAC11.
Recombinant viral genomes were extracted and electroporated into E. coli,
BAC
clones were identified by restriction enzyme digestion and PCR analysis.
Finally, we obtained 38
BAC
clones, DNA from various MDV-1
BACs
was transfected into CEFs, and recombinant virus was reconstituted by transfection of MDV-BAC2 DNA.
We successfully cloned the complete genome of MDV-1814 strain as an infectious
bacterial
artificial chromosome.
In addition, it opens the possibility to generate novel MDV-1 vaccines based on
the BACs
.
[MeSH-major]
Chickens / virology. Chromosomes, Artificial,
Bacterial
/ genetics. Genetic Engineering / methods. Mardivirus / genetics. Viral Proteins / physiology
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(PMID = 18616164.001).
[ISSN]
1000-3061
[Journal-full-title]
Sheng wu gong cheng xue bao = Chinese journal of biotechnology
[ISO-abbreviation]
Sheng Wu Gong Cheng Xue Bao
[Language]
chi
[Publication-type]
English Abstract; Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
China
[Chemical-registry-number]
0 / DNA, Recombinant; 0 / DNA, Viral; 0 / Viral Proteins
79.
Stuart LM, Boulais J, Charriere GM, Hennessy EJ, Brunet S, Jutras I, Goyette G, Rondeau C, Letarte S, Huang H, Ye P, Morales F, Kocks C, Bader JS, Desjardins M, Ezekowitz RA:
A systems biology analysis of the Drosophila phagosome.
Nature
; 2007 Jan 4;445(7123):95-101
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During phagocytosis, particles are internalized into 'phagosomes', organelles from which immune processes such as microbial destruction and
antigen
presentation are initiated.
Here we present a systems biology analysis of phagosomes isolated from cells derived from the genetically tractable model organism Drosophila melanogaster and address
the complex
dynamic interactions between proteins within this organelle and their involvement in particle engulfment.
The contribution of each protein and
complex
to
bacterial
internalization was tested by RNA-mediated interference and identified known components of the phagocytic machinery.
In addition, the prediction and validation of regulators of phagocytosis such as the 'exocyst', a macromolecular
complex
required for exocytosis but not previously implicated in phagocytosis, validates this strategy.
In generating this 'systems-based model', we show the power of applying this approach to the study
of complex
cellular processes and organelles and expect that this detailed model of the phagosome will provide a new framework for studying host-pathogen interactions and innate immunity.
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(PMID = 17151602.001).
[ISSN]
1476-4687
[Journal-full-title]
Nature
[ISO-abbreviation]
Nature
[Language]
eng
[Grant]
United Kingdom / Wellcome Trust / /
[Publication-type]
Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
[Publication-country]
England
[Chemical-registry-number]
0 / Drosophila Proteins
80.
Rozema JJ, Coeckelbergh T, Van den Berg TJ, Trau R, Duchateau NC, Lemmens S, Tassignon MJ:
Straylight before and after LASEK in myopia: changes in retinal straylight.
Invest Ophthalmol Vis Sci
; 2010 May;51(5):2800-4
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The difference with the average straylight increase with age, called base- and age-corrected (
BAC
) straylight, was also studied.
RESULTS: Before surgery,
BAC
straylight was found to be strongly elevated, with a value of 0.15 +/- 0.14 log units.
The reduction was significant (paired t-test, P << 0.01) and correlated with preoperative
BAC
straylight levels (r(2) = 0.332; P << 0.01).
Preoperative wear of soft contact lenses increased
the BAC
straylight by approximately 0.06 log units, with respect to the spectacles groups (P < 0.05, unpaired t-test), but after surgery, this difference was no longer found (P > 0.05).
CONCLUSIONS: Higher than normal preoperative
BAC
straylight was found to normalize after LASEK refractive surgery.
Wearing soft contact lenses causes an additional increase in preoperative
BAC
straylight that is eliminated after LASEK.
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(PMID = 20007829.001).
[ISSN]
1552-5783
[Journal-full-title]
Investigative ophthalmology & visual science
[ISO-abbreviation]
Invest. Ophthalmol. Vis. Sci.
[Language]
eng
[Publication-type]
Journal Article
[Publication-country]
United States
81.
Tomita R, Murai J, Miura Y, Ishihara H, Liu S, Kubotera Y, Honda A, Hatta R, Kuroda T, Hamada H, Sakamoto M, Munemura I, Nunomura O, Ishikawa K, Genda Y, Kawasaki S, Suzuki K, Meksem K, Kobayashi K:
Fine mapping and DNA fiber FISH analysis locates the tobamovirus resistance gene L3 of Capsicum chinense in a 400-kb region of R-like genes cluster embedded in highly repetitive sequences.
Theor Appl Genet
; 2008 Nov;117(7):1107-18
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[Title]
Fine mapping and DNA fiber FISH analysis locates the tobamovirus resistance gene L3 of Capsicum chinense in a 400-kb region
of R
-like genes cluster embedded in highly repetitive sequences.
The tobamovirus resistance gene L(3) of Capsicum chinense was mapped using an intra-specific F2 population (2,016 individuals) of Capsicum annuum cultivars, into one of which had been introduced
the C
. chinense L(3) gene, and an inter-specific F2 population (3,391 individuals) between C. chinense and Capsicum frutescence.
Analysis
of a BAC
library with an AFLP marker closely linked to L(3)-resistance revealed the presence of homologs of the tomato
disease
resistance gene I2.
The L(3) gene was mapped between I2 homolog marker IH1-04 and
BAC
-end marker 189D23M, and located within a region encompassing two different
BAC
contigs consisting of four and one clones, respectively.
DNA fiber FISH results and Southern blotting of the
BAC
clones suggested that the L(3) locus-containing region is rich in highly repetitive sequences.
Southern blot analysis indicated that the two
BAC
contigs contain more than ten copies of the I2 homologs.
In contrast to the inter-specific F2 population, no recombinant progeny were identified to have a crossover point within two
BAC
contigs consisting of seven and two clones in the intra-specific F2 population.
[MeSH-minor]
Amplified Fragment Length Polymorphism Analysis. Blotting, Southern. Chromosome Walking. Chromosomes, Artificial,
Bacterial
. Cloning, Molecular. Contig Mapping. Genetic Markers. Immunity, Innate / genetics. In Situ Hybridization, Fluorescence. Linkage Disequilibrium
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[Cites]
Curr Opin Plant Biol. 2007 Apr;10(2):116-22
[
17291819.001
]
[Cites]
Proc Natl Acad Sci U S A. 2006 Dec 5;103(49):18828-33
[
17021014.001
]
[Cites]
Proc Natl Acad Sci U S A. 2007 Aug 21;104(34):13833-8
[
17699618.001
]
[Cites]
Arch Virol. 2008;153(4):645-50
[
18236125.001
]
[Cites]
Genome. 1999 Dec;42(6):1100-10
[
10659776.001
]
[Cites]
Genetics. 2000 Jun;155(2):873-87
[
10835406.001
]
[Cites]
Bioinformatics. 2001 Sep;17(9):838-9
[
11590100.001
]
[Cites]
Mol Genet Genomics. 2001 Oct;266(2):157-66
[
11683256.001
]
[Cites]
Genome. 2002 Oct;45(5):839-54
[
12416616.001
]
[Cites]
Trends Biotechnol. 2003 Apr;21(4):178-83
[
12679066.001
]
[Cites]
Theor Appl Genet. 2003 Aug;107(3):540-3
[
12748763.001
]
[Cites]
Theor Appl Genet. 2004 Jan;108(2):190-6
[
14504748.001
]
[Cites]
Plant J. 2004 Jun;38(6):898-909
[
15165183.001
]
[Cites]
J Gen Virol. 2004 Jul;85(Pt 7):2077-85
[
15218193.001
]
[Cites]
Nucleic Acids Res. 1989 Oct 25;17(20):8093-9
[
2573037.001
]
[Cites]
Arch Virol. 1993;131(1-2):75-88
[
8328918.001
]
[Cites]
Virology. 1995 Jun 1;209(2):498-505
[
7778282.001
]
[Cites]
Nucleic Acids Res. 1995 Nov 11;23(21):4407-14
[
7501463.001
]
[Cites]
Plant J. 1996 Mar;9(3):421-30
[
8919917.001
]
[Cites]
Mol Plant Microbe Interact. 1997 Jan;10(1):107-13
[
9002274.001
]
[Cites]
Plant Cell. 1997 Apr;9(4):521-32
[
9144960.001
]
[Cites]
Mol Plant Microbe Interact. 1998 Apr;11(4):327-31
[
9530869.001
]
[Cites]
Plant Cell. 1998 Jun;10(6):1055-68
[
9634592.001
]
[Cites]
Genome Res. 1998 Nov;8(11):1113-30
[
9847076.001
]
[Cites]
Genetics. 1999 Jul;152(3):1183-202
[
10388833.001
]
[Cites]
Cytometry A. 2005 Feb;63(2):114-7
[
15651009.001
]
[Cites]
Plant J. 2005 Apr;42(2):251-61
[
15807786.001
]
[Cites]
Methods Enzymol. 2005;395:443-60
[
15865979.001
]
[Cites]
Virus Genes. 2007 Apr;34(2):205-14
[
17160553.001
]
(PMID = 18663424.001).
[ISSN]
0040-5752
[Journal-full-title]
TAG. Theoretical and applied genetics. Theoretische und angewandte Genetik
[ISO-abbreviation]
Theor. Appl. Genet.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Germany
[Chemical-registry-number]
0 / DNA, Plant; 0 / Genetic Markers
[Other-IDs]
NLM/ PMC2755798
82.
Kifayathullah LA, Arunachalam JP, Bodda C, Agbemenyah HY, Laccone FA, Mannan AU:
MeCP2 mutant protein is expressed in astrocytes as well as in neurons and localizes in the nucleus.
Cytogenet Genome Res
; 2010;129(4):290-7
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RTT is a progressive neurodevelopmental
disorder
, which affects primarily girls during early childhood and it is one of the most common causes of mental retardation in females.
To evaluate the functional role of the R270X mutation, we generated a transgenic mouse model expressing MeCP2(270_EGFP) (human mutation equivalent) by
BAC
recombineering.
[MeSH-major]
Astrocytes / metabolism.
Cell
Nucleus / metabolism. Methyl-CpG-Binding Protein 2 / metabolism. Mutation. Neurons / metabolism
[MeSH-minor]
Active Transport,
Cell
Nucleus. Animals. Cells, Cultured. Mice. Mice, Transgenic
figshare.
supplemental materials - Supporting Data and Materials for the article
.
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[Copyright]
Copyright (c) 2010 S. Karger AG, Basel.
(PMID = 20625242.001).
[ISSN]
1424-859X
[Journal-full-title]
Cytogenetic and genome research
[ISO-abbreviation]
Cytogenet. Genome Res.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Switzerland
[Chemical-registry-number]
0 / Mecp2 protein, mouse; 0 / Methyl-CpG-Binding Protein 2
83.
Richter M, Schudel L, Tobler K, Matheis F, Vögtlin A, Vanderplasschen A, Costes B, Spiess B, Ackermann M:
Clinical, virological, and immunological parameters associated with superinfection of latently with FeHV-1 infected cats.
Vet Microbiol
; 2009 Sep 18;138(3-4):205-16
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Infections with feline herpesvirus type 1 (FeHV-1) are frequently associated with recurrent ocular
disease
, which may occur even in vaccinated cats.
To begin addressing this
complex
question, we reconstituted a marker-tagged mutant FeHV-1 from
a bacterial
artificial chromosome (
BAC
) harboring the FeHV-1 genome.
Reactivation was accompanied by recrudescence of ocular
disease
signs.
[MeSH-minor]
Animals. Antibodies, Viral / blood. Cats.
Cell
Line. Cyclophosphamide / pharmacology. Dexamethasone / pharmacology. Female. Immunosuppressive Agents / pharmacology. Male. Specific Pathogen-Free Organisms. Viral Proteins
Hazardous Substances Data Bank.
DEXAMETHASONE
.
Hazardous Substances Data Bank.
CYCLOPHOSPHAMIDE
.
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(PMID = 19359108.001).
[ISSN]
1873-2542
[Journal-full-title]
Veterinary microbiology
[ISO-abbreviation]
Vet. Microbiol.
[Language]
eng
[Publication-type]
Journal Article; Research Support, Non-U.S. Gov't
[Publication-country]
Netherlands
[Chemical-registry-number]
0 / Antibodies, Viral; 0 / Immunosuppressive Agents; 0 / Viral Proteins; 7S5I7G3JQL / Dexamethasone; 8N3DW7272P / Cyclophosphamide
84.
Prazeres da Costa O, González J, Ruiz A:
Cloning and sequencing of the breakpoint regions of inversion 5g fixed in Drosophila buzzatii.
Chromosoma
; 2009 Jun;118(3):349-60
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First, D. buzzatii
BAC
clones encompassing the breakpoints were identified and their ends sequenced.
FlyBase.
FlyBase
.
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[Cites]
Science. 1999 Jul 16;285(5426):415-8
[
10411506.001
]
[Cites]
Genetics. 2000 Apr;154(4):1681-91
[
10747062.001
]
[Cites]
Annu Rev Ecol Evol Syst. 2008 Dec 1;39:21-42
[
20419035.001
]
[Cites]
Mol Biol Evol. 2004 Jan;21(1):36-44
[
12949132.001
]
[Cites]
Annu Rev Genet. 2007;41:169-92
[
17608616.001
]
[Cites]
Genetica. 2002 May;115(1):81-91
[