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1. Zhong Y, Wang L, Li T, Chen XM: Calcifying epithelial odontogenic tumour showing malignant transformation: a case report and review of the literature. Chin J Dent Res; 2010;13(2):157-62

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Calcifying epithelial odontogenic tumour showing malignant transformation: a case report and review of the literature.
  • Calcifying epithelial odontogenic tumour (CEOT) is a rare and benign odontogenic neoplasm that affects the jaws.
  • [MeSH-major] Cell Transformation, Neoplastic / pathology. Maxillary Neoplasms / pathology
  • [MeSH-minor] Humans. Keratin-19 / analysis. Ki-67 Antigen / analysis. Male. Middle Aged. Odontogenic Tumors / pathology. Odontogenic Tumors / surgery. Skin Neoplasms / pathology. Skin Neoplasms / surgery

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  • (PMID = 21264368.001).
  • [ISSN] 1462-6446
  • [Journal-full-title] The Chinese journal of dental research : the official journal of the Scientific Section of the Chinese Stomatological Association (CSA)
  • [ISO-abbreviation] Chin J Dent Res
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Keratin-19; 0 / Ki-67 Antigen; Calcifying Epithelial Odontogenic Tumor
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2. Wang A, Zhang B, Huang H, Zhang L, Zeng D, Tao Q, Wang J, Pan C: Suppression of local invasion of ameloblastoma by inhibition of matrix metalloproteinase-2 in vitro. BMC Cancer; 2008;8:182
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • BACKGROUND: Ameloblastomas are odontogenic neoplasms characterized by local invasiveness.
  • [MeSH-major] Ameloblastoma / enzymology. Jaw Neoplasms / enzymology. Matrix Metalloproteinase Inhibitors
  • [MeSH-minor] Humans. Matrix Metalloproteinase 2 / genetics. Matrix Metalloproteinase 2 / metabolism. Neoplasm Invasiveness. Plasmids / genetics. RNA, Messenger / genetics. RNA, Small Interfering / genetics. Tissue Inhibitor of Metalloproteinase-2 / biosynthesis. Tissue Inhibitor of Metalloproteinase-2 / genetics. Tissue Inhibitor of Metalloproteinase-2 / metabolism. Transfection. Tumor Cells, Cultured

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  • (PMID = 18588710.001).
  • [ISSN] 1471-2407
  • [Journal-full-title] BMC cancer
  • [ISO-abbreviation] BMC Cancer
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Matrix Metalloproteinase Inhibitors; 0 / RNA, Messenger; 0 / RNA, Small Interfering; 127497-59-0 / Tissue Inhibitor of Metalloproteinase-2; EC 3.4.24.24 / Matrix Metalloproteinase 2
  • [Other-IDs] NLM/ PMC2443806
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3. Deboni MC, Naclério-Homem Mda G, Pinto Junior DS, Traina AA, Cavalcanti MG: Clinical, radiological and histological features of calcifying epithelial odontogenic tumor: case report. Braz Dent J; 2006;17(2):171-4
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Clinical, radiological and histological features of calcifying epithelial odontogenic tumor: case report.
  • The calcifying epithelial odontogenic tumor (CEOT) is a rare benign odontogenic neoplasm that accounts for approximately 1% of all odontogenic tumors.
  • [MeSH-major] Mandibular Neoplasms. Odontogenic Cyst, Calcifying. Tooth, Impacted

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  • (PMID = 16924348.001).
  • [ISSN] 0103-6440
  • [Journal-full-title] Brazilian dental journal
  • [ISO-abbreviation] Braz Dent J
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Brazil
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4. Etit D, Uyaroglu MA, Erdogan N: Mixed odontogenic tumor: ameloblastoma and calcifying epithelial odontogenic tumor. Indian J Pathol Microbiol; 2010 Jan-Mar;53(1):122-4
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Mixed odontogenic tumor: ameloblastoma and calcifying epithelial odontogenic tumor.
  • Odontogenic tumors constitute a group of heterogeneous disease derived from epithelial, mesenchymal and/or ectomesenchymal elements.
  • Ameloblastoma is the best known and the most frequent form of odontogenic tumors.
  • Calcifying epithelial odontogenic tumor (CEOT), known as Pindborg tumor, is locally invasive lesion which has a characteristic amiloid deposition.
  • [MeSH-major] Ameloblastoma / diagnosis. Ameloblastoma / pathology. Odontogenic Tumors / diagnosis. Odontogenic Tumors / pathology

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  • (PMID = 20090240.001).
  • [ISSN] 0974-5130
  • [Journal-full-title] Indian journal of pathology & microbiology
  • [ISO-abbreviation] Indian J Pathol Microbiol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] India
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5. Koutlas IG, Allen CM, Warnock GR, Manivel JC: Sclerosing odontogenic carcinoma: a previously unreported variant of a locally aggressive odontogenic neoplasm without apparent metastatic potential. Am J Surg Pathol; 2008 Nov;32(11):1613-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Sclerosing odontogenic carcinoma: a previously unreported variant of a locally aggressive odontogenic neoplasm without apparent metastatic potential.
  • We describe 3 cases of apparent odontogenic carcinomas that share histologic features not previously reported in the literature.
  • The tumors involved one 72-year-old man and 2 women, 46-year old and 73-year old, respectively.
  • The tumors presented as expansile radiolucencies.
  • Immunohistochemically, all tumors stained for cytokeratins (CK) 5/6, CK19, and epsilon-cadherin, the latter exhibiting a fragmented cytoplasmic membranous staining.
  • All tumors were negative for CK20, carcinoembryonic antigen, and CAM5.2.
  • Microscopic tumor nests and cords were found far beyond of what surgically seemed to be tumor-free margins.
  • All patients are free of disease 5, 12, and 4 years after treatment, respectively.
  • We propose the name sclerosing odontogenic carcinoma.
  • As current classifications of odontogenic tumors do not include such examples, it is important that pathologists become aware of this entity.
  • Finally, this report supports recent literature on positive nuclear p63 in odontogenic cysts and tumors.
  • [MeSH-major] Mandibular Neoplasms / pathology. Maxillary Neoplasms / pathology. Odontogenic Tumors / pathology

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  • [CommentIn] Am J Surg Pathol. 2010 Apr;34(4):595; author reply 595-6, 596 [20154593.001]
  • (PMID = 18753945.001).
  • [ISSN] 1532-0979
  • [Journal-full-title] The American journal of surgical pathology
  • [ISO-abbreviation] Am. J. Surg. Pathol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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6. Garg D, Palaskar S, Shetty VP, Bhushan A: Adenomatoid odontogenic tumor - hamartoma or true neoplasm: a case report. J Oral Sci; 2009 Mar;51(1):155-9

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Adenomatoid odontogenic tumor - hamartoma or true neoplasm: a case report.
  • Adenomatoid odontogenic tumor (AOT) is an uncommon tumor of odontogenic origin with a relative frequency of 2.2 - 7.1%.
  • As the histogenesis of AOT is still uncertain, it is sometimes categorized as a hamartomatous lesion rather than a true neoplasm.
  • The tumor showed some unusual and aggressive features that suggested it was a true neoplasm.
  • [MeSH-major] Hamartoma / diagnosis. Maxillary Diseases / diagnosis. Maxillary Neoplasms / diagnosis. Odontogenic Tumors / diagnosis
  • [MeSH-minor] Cuspid / radiography. Diagnosis, Differential. Facial Asymmetry / diagnosis. Female. Humans. Radiography, Panoramic. Root Resorption / radiography. Tooth, Impacted / radiography. Young Adult

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  • (PMID = 19325215.001).
  • [ISSN] 1343-4934
  • [Journal-full-title] Journal of oral science
  • [ISO-abbreviation] J Oral Sci
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Japan
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7. Ramesh RS, Manjunath S, Ustad TH, Pais S, Shivakumar K: Unicystic ameloblastoma of the mandible--an unusual case report and review of literature. Head Neck Oncol; 2010;2:1

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Ameloblastoma is a true neoplasm of odontogenic epithelial origin.
  • It is the second most common odontogenic neoplasm, and only odontoma outnumbers it in reported frequency of occurrence.
  • Its incidence, combined with its clinical behavior, makes ameloblastoma the most significant odontogenic neoplasm.
  • Unicystic ameloblastoma (UA) refers to those cystic lesions that show clinical, radiographic, or gross features of a mandibular cyst, but on histologic examination show a typical ameloblastomatous epithelium lining part of the cyst cavity, with or without luminal and/or mural tumor growth.
  • [MeSH-major] Ameloblastoma / pathology. Mandibular Neoplasms / pathology

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  • (PMID = 20157425.001).
  • [ISSN] 1758-3284
  • [Journal-full-title] Head & neck oncology
  • [ISO-abbreviation] Head Neck Oncol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] England
  • [Other-IDs] NLM/ PMC2821325
  • [General-notes] NLM/ Original DateCompleted: 20100629
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8. Angiero F, Borloni R, Macchi M, Stefani M: Ameloblastic carcinoma of the maxillary sinus. Anticancer Res; 2008 Nov-Dec;28(6B):3847-54
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Ameloblastic carcinoma is a very rare malignant odontogenic neoplasm of the mandible and maxilla, accounting for some 66 reported cases.
  • The initial microscopic evaluation of the biopsy and radiographic findings were consistent with benign peripheral ameloblastoma without cellular atypia and extensive fields of acantomatous pattern, but immunohistochemical investigation found strong positivity for Bcl-2, cytokeratins CAM 5 and 6, and for Ki-67/MIB-1, changing our diagnosis.
  • Cellular features of malignancy in the surgical specimen confirmed the diagnosis of ameloblastic carcinoma.
  • This case of an aggressive ameloblastic carcinoma of the maxillary gingiva that presented with an unusual histological pattern illustrates that these tumors can create a diagnostic challenge that may require extensive surgical sampling and/or removal to establish the diagnosis.
  • [MeSH-major] Ameloblastoma / diagnosis. Maxillary Sinus Neoplasms / diagnosis

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  • (PMID = 19192639.001).
  • [ISSN] 0250-7005
  • [Journal-full-title] Anticancer research
  • [ISO-abbreviation] Anticancer Res.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Greece
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9. Gratzinger D, Salama ME, Poh CF, Rouse RV: Ameloblastoma, calcifying epithelial odontogenic tumor, and glandular odontogenic cyst show a distinctive immunophenotype with some myoepithelial antigen expression. J Oral Pathol Med; 2008 Mar;37(3):177-84
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Ameloblastoma, calcifying epithelial odontogenic tumor, and glandular odontogenic cyst show a distinctive immunophenotype with some myoepithelial antigen expression.
  • BACKGROUND: Odontogenic neoplasms have some morphologic overlap with salivary gland neoplasms, many of which show myoepithelial differentiation.
  • In the 1980s, an ultrastructural study identified a population of myoepithelial-like cells in calcifying epithelial odontogenic tumor.
  • Myoepithelial derived tumors have since been shown to have distinct immunohistochemical profiles.
  • METHODS: We examined a series of odontogenic neoplasms, including 11 ameloblastomas, four calcifying epithelial odontogenic tumors, five glandular odontogenic cysts (GOCs), and five keratocystic odontogenic tumors with a panel of myoepithelial-associated immunohistochemical stains.
  • We also assessed representative control examples of oral mucosa, odontogenic rests, and dentigerous cysts.
  • Calponin reactivity was at least focally present in two of four calcifying epithelial odontogenic tumors, three of five GOCs, and 10 of 11 ameloblastomas; the sole completely non-reactive ameloblastoma represents a lung metastasis.
  • One case of calcifying epithelial odontogenic tumor was focally positive for glial fibrillary acidic protein.
  • Two of three calcifying epithelial odontogenic tumors and five of five GOCs were also positive for a low molecular weight cytokeratin (CK7).
  • CONCLUSIONS: Ameloblastomas, GOCs, and calcifying epithelial odontogenic tumors show a distinctive immunophenotype which overlaps with that of myoepithelial-derived salivary gland neoplasms but does not provide definitive support for myoepithelial differentiation.
  • [MeSH-major] Ameloblastoma / pathology. Odontogenic Cysts / pathology. Odontogenic Tumors / pathology. Salivary Gland Neoplasms / pathology
  • [MeSH-minor] Antigens, Neoplasm / biosynthesis. Calcium-Binding Proteins / biosynthesis. Cell Differentiation. Epithelial Cells / pathology. Gene Expression. Humans. Immunophenotyping. Keratins, Type II / biosynthesis. Keratins, Type II / immunology. Membrane Proteins / biosynthesis. Microfilament Proteins / biosynthesis. Muscle Cells / pathology. Muscle Proteins / biosynthesis. Retrospective Studies

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  • (PMID = 18251942.001).
  • [ISSN] 1600-0714
  • [Journal-full-title] Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology
  • [ISO-abbreviation] J. Oral Pathol. Med.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Denmark
  • [Chemical-registry-number] 0 / Antigens, Neoplasm; 0 / CKAP4 protein, human; 0 / Calcium-Binding Proteins; 0 / Keratins, Type II; 0 / Membrane Proteins; 0 / Microfilament Proteins; 0 / Muscle Proteins; 0 / calponin
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10. Jundt G, Reichart PA: [Malignant odontogenic tumors]. Pathologe; 2008 May;29(3):205-13

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Malignant odontogenic tumors].
  • [Transliterated title] Maligne odontogene Tumoren.
  • Malignant odontogenic tumors are extremely rare.
  • As with benign odontogenic tumors, malignant epithelial odontogenic tumors or odontogenic carcinomas are distinguished from the even rarer mesenchymal ones, the odontogenic sarcomas.
  • The existence of odontogenic carcinosarcomas is not yet acknowledged by the World Health Organization.
  • Odontogenic carcinomas comprise ameloblastic carcinoma (AmCa), primary intraosseous carcinoma (PIOC), clear cell odontogenic carcinoma, odontogenic ghost cell carcinoma (OGCC), and the special case of metastasizing ameloblastoma.
  • Odontogenic sarcomas consist of ameloblastic fibrosarcoma and ameloblastic fibrodentinosarcoma and fibroodontosarcoma.
  • Whereas metastasizing ameloblastoma can be diagnosed only after having metastasized, all other malignant odontogenic tumors present with atypia, increased cellularity and mitoses, and invasion.
  • Odontogenic sarcomas are regarded as low-grade tumors that rarely metastasize.
  • Odontogenic carcinomas, however, especially AmCa, OGCC, and PIOC, are more aggressive, with a 5-year survival rate of about 70% for AmCa and OGCC and a 3-year survival rate of about 37% for PIOC.
  • [MeSH-major] Jaw Neoplasms / pathology. Odontogenic Tumors / pathology
  • [MeSH-minor] Ameloblastoma / classification. Ameloblastoma / pathology. Carcinoma / classification. Carcinoma / pathology. Carcinosarcoma / classification. Carcinosarcoma / pathology. Diagnosis, Differential. Humans. Jaw / pathology. Neoplasm Invasiveness. Sarcoma / classification. Sarcoma / pathology

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  • (PMID = 18392827.001).
  • [ISSN] 1432-1963
  • [Journal-full-title] Der Pathologe
  • [ISO-abbreviation] Pathologe
  • [Language] ger
  • [Publication-type] English Abstract; Journal Article; Review
  • [Publication-country] Germany
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11. Jovanović G, Burić N, Mihailović D, Tijanić M: [Marsupialization and enucleation of keratocystic odontogenic tumor with the use of Carnoy's solution]. Vojnosanit Pregl; 2010 May;67(5):431-5
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  • [Title] [Marsupialization and enucleation of keratocystic odontogenic tumor with the use of Carnoy's solution].
  • INTRODUCTION: Keratocystic odontogenic tumors (KCOT) or odontogenic keratocysts are aggressive and expansive odontogenic neoplasms with high recurrence rate (25%-60%).
  • [MeSH-major] Acetic Acid / administration & dosage. Chloroform / administration & dosage. Ethanol / administration & dosage. Mandibular Neoplasms / surgery. Odontogenic Cysts / surgery. Odontogenic Tumors / surgery

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  • (PMID = 20499739.001).
  • [ISSN] 0042-8450
  • [Journal-full-title] Vojnosanitetski pregled
  • [ISO-abbreviation] Vojnosanit Pregl
  • [Language] srp
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Serbia
  • [Chemical-registry-number] 0 / Carnoy's solution; 3K9958V90M / Ethanol; 7V31YC746X / Chloroform; Q40Q9N063P / Acetic Acid
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12. Hermes D, Krüger S, Entenmann A: [Odontogenic tumors in childhood - case report and review of literature]. Klin Padiatr; 2007 Jul-Aug;219(4):234-9

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  • [Title] [Odontogenic tumors in childhood - case report and review of literature].
  • [Transliterated title] Odontogene Tumoren im Kindesalter - Fallbeschreibung und Literaturübersicht.
  • BACKGROUND: Odontogenic tumors are a heterogenic group of neoplasias of the oral cavity, whose biological behaviour reaches from true hamartomas to highly malignant tumors.
  • PATIENT: We describe the case of a 4 year old caucasian girl presenting with eruption disturbance of a primary tooth and corresponding maxillary tumor.
  • After surgical removal of the mass, histological examination classified the tumor as ameloblastic fibroodontoma.
  • This very rare neoplasm is of odontogenic origin.
  • DISCUSSION: Following our case report, the variety of odontogenic tumors regarding tumor biology, clinical symptoms and therapeutic options in childhood are discussed.
  • [MeSH-major] Maxillary Neoplasms / diagnosis. Odontogenic Tumors / diagnosis. Odontoma / diagnosis. Tooth, Unerupted / etiology
  • [MeSH-minor] Alveolar Process / pathology. Alveolar Process / surgery. Child, Preschool. Diagnosis, Differential. Female. Humans

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  • (PMID = 16865654.001).
  • [ISSN] 0300-8630
  • [Journal-full-title] Klinische Pädiatrie
  • [ISO-abbreviation] Klin Padiatr
  • [Language] ger
  • [Publication-type] Case Reports; English Abstract; Journal Article; Review
  • [Publication-country] Germany
  • [Number-of-references] 39
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13. Carnelio S, Vij H: Expression of tenascin and nucleolar organizer region in ameloblastoma and ameloblastic fibroma. J Oral Pathol Med; 2010 Mar;39(3):223-9

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • BACKGROUND: The aim of this study was to assess the expression, distribution and comparison of tenascin, a glycoprotein of the extracellular matrix in ameloblastoma and ameloblastic fibroma, both odontogenic neoplasms with diverse biological behavior and to understand the proliferative activity by using the morphometric analysis.
  • METHODS: Paraffin embedded tissue from 25 cases of odontogenic tumors i.e., ameloblastoma (n = 15) and ameloblastic fibroma (n = 10) were used.
  • CONCLUSIONS: Expression of tenascin in these neoplasms suggest that it could play a role in epithelial- mesenchymal interaction, while AgNORs reveal that ameloblastomas are more aggressive when compared with ameloblastic fibromas.
  • [MeSH-major] Ameloblastoma / pathology. Nucleolus Organizer Region / ultrastructure. Odontogenic Tumors / pathology. Tenascin / analysis

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  • (PMID = 19943856.001).
  • [ISSN] 1600-0714
  • [Journal-full-title] Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology
  • [ISO-abbreviation] J. Oral Pathol. Med.
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article
  • [Publication-country] Denmark
  • [Chemical-registry-number] 0 / Tenascin
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14. Siar CH, Nagatsuka H, Chuah KS, Rivera RS, Nakano K, Ng KH, Kawakami T: Notch4 overexpression in ameloblastoma correlates with the solid/multicystic phenotype. Oral Surg Oral Med Oral Pathol Oral Radiol Endod; 2010 Aug;110(2):224-33
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  • OBJECTIVE: Notch signaling has been implicated in cell fate decisions during odontogenesis and tumorigenesis of some odontogenic neoplasms; however, its role in solid/multicystic (SA), unicystic (UA), and recurrent (RA) ameloblastoma remains unclear.
  • [MeSH-major] Ameloblastoma / genetics. Intercellular Signaling Peptides and Proteins / biosynthesis. Mandibular Neoplasms / genetics. Maxillary Neoplasms / genetics. Proto-Oncogene Proteins / biosynthesis. Receptors, Notch / biosynthesis

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  • [Copyright] Copyright 2010 Mosby, Inc. All rights reserved.
  • (PMID = 20659700.001).
  • [ISSN] 1528-395X
  • [Journal-full-title] Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics
  • [ISO-abbreviation] Oral Surg Oral Med Oral Pathol Oral Radiol Endod
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Intercellular Signaling Peptides and Proteins; 0 / Ligands; 0 / NOTCH4 protein, human; 0 / Proto-Oncogene Proteins; 0 / Receptors, Notch
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15. Ebert CS Jr, Dubin MG, Hart CF, Chalian AA, Shockley WW: Clear cell odontogenic carcinoma: a comprehensive analysis of treatment strategies. Head Neck; 2005 Jun;27(6):536-42
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Clear cell odontogenic carcinoma: a comprehensive analysis of treatment strategies.
  • BACKGROUND: Odontogenic neoplasms of predominately clear cells are unusual.
  • Our goal is to present two new cases, summarize reported cases of clear cell odontogenic carcinoma (CCOC), assess potential risk factors for recurrence, and propose definitive surgical and therapeutic strategies.
  • The overall rate of recurrent disease was 55%.
  • Age (p = .20), sex (p = .28), and tumor site (p = .50) did not predict risk for recurrence.
  • CONCLUSIONS: CCOC is a potentially aggressive tumor with a tendency for recurrence.
  • Treatment strategies should be directed toward wide surgical resection with confirmation of tumor-free margins.
  • [MeSH-major] Adenocarcinoma, Clear Cell / therapy. Mandibular Neoplasms / therapy. Odontogenic Tumors / therapy

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  • [Copyright] (c) 2005 Wiley Periodicals, Inc.
  • (PMID = 15772956.001).
  • [ISSN] 1043-3074
  • [Journal-full-title] Head & neck
  • [ISO-abbreviation] Head Neck
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 33
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16. Nakano K, Siar CH, Tsujigiwa H, Nagatsuka H, Nagai N, Kawakami T: Notch signaling in benign and malignant ameloblastic neoplasms. Eur J Med Res; 2008 Oct 27;13(10):476-80

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Notch signaling in benign and malignant ameloblastic neoplasms.
  • BACKGROUND: In general, Notch is a representative signal which controls morphosis and differentiation of cells, but its role in human odontogenic neoplasms, especially in ameloblastoma and its malignant counterpart, ameloblastic carcinoma, is not known.
  • METHODS: We examined Notch1 peptide and its gene (mRNA) in an ameloblastoma (case 1: 27-year-old female, right mandibular tumor) and an ameloblastic carcinoma (case 2: 93-year-old female, right mandibular tumor), using immunohistochemistry (IHC) and in situ hybridization (ISH) techniques.
  • RESULTS: Notch1 intracellular domain (NICD) positive products were observed in the cells at the peripheral layer of most proliferating epithelial tumor nests in case 1.
  • CONCLUSIONS: Notch signaling plays some role in cytological differentiation or acquisition of tissue specific characteristics in neoplastic cells of odontogenic neoplasms, including ameloblastoma and ameloblastic carcinoma.
  • [MeSH-major] Ameloblastoma / genetics. Ameloblastoma / metabolism. Mandibular Neoplasms / genetics. Mandibular Neoplasms / metabolism. Receptor, Notch1 / genetics. Receptor, Notch1 / metabolism
  • [MeSH-minor] Adult. Aged. Aged, 80 and over. Female. Gene Expression. Humans. Immunohistochemistry. In Situ Hybridization. Protein Structure, Tertiary. RNA, Messenger / genetics. RNA, Messenger / metabolism. RNA, Neoplasm / genetics. RNA, Neoplasm / metabolism

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  • (PMID = 19008176.001).
  • [ISSN] 0949-2321
  • [Journal-full-title] European journal of medical research
  • [ISO-abbreviation] Eur. J. Med. Res.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / NOTCH1 protein, human; 0 / RNA, Messenger; 0 / RNA, Neoplasm; 0 / Receptor, Notch1
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17. Capodiferro S, Maiorano E, Giardina C, Lacaita MG, Lo Muzio L, Favia G: Osteoblastoma of the mandible: clinicopathologic study of four cases and literature review. Head Neck; 2005 Jul;27(7):616-21

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • BACKGROUND: Osteoblastoma is a benign bone tumor accounting for 1% of all bone tumors; it commonly involves the spine and the sacrum of young individuals, with less than 5% being localized to the posterior mandible.
  • RESULTS: The tumors involved the posterior mandible of young patients (age range, 10-21 years; two male and two female patients) and appeared as painful bone expansions.
  • All patients were disease free after prolonged follow-up.
  • CONCLUSIONS: Osteoblastomas may be distinguished from other bone tumors, fibro-osseous lesions, and odontogenic neoplasms on the basis of integrated clinical, radiologic, and histologic features and usually manifest an indolent clinical course.
  • [MeSH-major] Mandibular Neoplasms / pathology. Osteoblastoma / pathology
  • [MeSH-minor] Adolescent. Adult. Child. Diagnosis, Differential. Female. Humans. Male. Retrospective Studies. Tomography, X-Ray Computed

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  • (PMID = 15880408.001).
  • [ISSN] 1043-3074
  • [Journal-full-title] Head & neck
  • [ISO-abbreviation] Head Neck
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 20
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18. Crincoli V, Scivetti M, Di Bisceglie MB, Lucchese A, Favia G: Complex odontoma: confocal laser scanning microscopy analysis of a case. Minerva Stomatol; 2006 May;55(5):315-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • This study shows the confocal laser scanning microscopy versatility in the analysis of odontogenic neoplasms with production of mineralized tissues.
  • [MeSH-major] Maxillary Neoplasms / ultrastructure. Microscopy, Confocal. Odontoma / ultrastructure

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  • [ErratumIn] Minerva Stomatol. 2006 Jun;55(6):XVII. Di Risceglie, M B [corrected to Di Bisceglie, M B]
  • (PMID = 16688108.001).
  • [ISSN] 0026-4970
  • [Journal-full-title] Minerva stomatologica
  • [ISO-abbreviation] Minerva Stomatol
  • [Language] eng; ita
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Italy
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19. Heikinheimo K, Jee KJ, Morgan PR, Nagy B, Knuutila S, Leivo I: Genetic changes in sporadic keratocystic odontogenic tumors (odontogenic keratocysts). J Dent Res; 2007 Jun;86(6):544-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Genetic changes in sporadic keratocystic odontogenic tumors (odontogenic keratocysts).
  • Little is known about the genetic background of keratocystic odontogenic tumors (KCOT, odontogenic keratocysts).
  • Over-expressed and amplified genes in 12q13, also reported in several other tumors and cell lines, may contribute to the persistent growth characteristics of KCOT.
  • [MeSH-major] Odontogenic Cysts / genetics

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  • (PMID = 17525355.001).
  • [ISSN] 0022-0345
  • [Journal-full-title] Journal of dental research
  • [ISO-abbreviation] J. Dent. Res.
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Activated-Leukocyte Cell Adhesion Molecule; 0 / CDH18 protein, human; 0 / Cadherins; 0 / GLI1 protein, human; 0 / Keratin-6; 0 / Transcription Factors; 0 / Zinc Finger Protein GLI1; 9007-49-2 / DNA
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20. Mendes RA, Carvalho JF, Waal Iv: An overview on the expression of cyclooxygenase-2 in tumors of the head and neck. Oral Oncol; 2009 Oct;45(10):e124-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] An overview on the expression of cyclooxygenase-2 in tumors of the head and neck.
  • Cyclooxygenase-2 (COX-2) levels are increased in various tumors, particularly those involving the esophagus, stomach, breast, pancreas, lung, colon, skin, urinary bladder, prostate and head and neck.
  • Nevertheless, the tumorigenic mechanisms of COX-2 overexpression still remain poorly understood and may include mechanisms that may act at different stages of the disease.
  • Thus, the literature shows increasing evidence that overexpression of the COX-2 plays an important role in tumor growth and spread of tumors by interfering with different biological processes such as cell proliferation, cellular adhesion, immune surveillance, apoptosis, and angiogenesis.
  • Furthermore, the expression of COX-2 might shed some light over the physiopathology and clinical behavior of tumors of the head and neck, including benign odontogenic neoplasms of the jaws with an aggressive behavior, such as keratocystic odontogenic tumors (KCOT).
  • Ultimately, the research of molecular markers associated with the biological behavior of tumors will help to understand the underlying molecular mechanisms and to predict the clinical outcome, leading to the development of new therapeutic applications, such as molecular-targeted treatment and patient tailored therapy.
  • [MeSH-major] Cyclooxygenase 2 / metabolism. Head and Neck Neoplasms / metabolism. Neoplasm Proteins / metabolism

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  • (PMID = 19457709.001).
  • [ISSN] 1879-0593
  • [Journal-full-title] Oral oncology
  • [ISO-abbreviation] Oral Oncol.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Neoplasm Proteins; 0 / Prostaglandins; EC 1.14.99.1 / Cyclooxygenase 2
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21. Peña-Torres LM, Monterrubio-Guerrero A, Díaz de León-Sandoval LA: [A case of the calcifying epithelial odontogenic tumor (Pindborg's tumor). Reported and literature review]. Rev Med Inst Mex Seguro Soc; 2010 Mar-Apr;48(2):205-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [A case of the calcifying epithelial odontogenic tumor (Pindborg's tumor). Reported and literature review].
  • [Transliterated title] Tumor odontogénico epitelial calcificante (tumor de Pindborg). Informe de un caso y revisión de la literatura.
  • The calcifying epithelial odontogenic tumor known as Pindborg's tumor, is a rare odontogenic neoplasm of the jaws.
  • This article reviews the literature and describes a case of patient who presented calcifying epithelial odontogenic tumor in the jaw undergoing surgical excision treatment with an evolution without complications.
  • [MeSH-major] Mandibular Neoplasms. Odontogenic Tumors
  • [MeSH-minor] Adult. Calcinosis / complications. Calcinosis / diagnosis. Calcinosis / surgery. Female. Humans. Mandibular Diseases / complications. Mandibular Diseases / diagnosis. Mandibular Diseases / surgery

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  • (PMID = 20929626.001).
  • [ISSN] 0443-5117
  • [Journal-full-title] Revista médica del Instituto Mexicano del Seguro Social
  • [ISO-abbreviation] Rev Med Inst Mex Seguro Soc
  • [Language] spa
  • [Publication-type] Case Reports; English Abstract; Journal Article; Review
  • [Publication-country] Mexico
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22. Mendes RA, Carvalho JF, van der Waal I: Characterization and management of the keratocystic odontogenic tumor in relation to its histopathological and biological features. Oral Oncol; 2010 Apr;46(4):219-25

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Characterization and management of the keratocystic odontogenic tumor in relation to its histopathological and biological features.
  • Keratocystic odontogenic tumor (KCOT), formerly referred to as odontogenic keratocyst, is a benign neoplasm of odontogenic origin which may present an aggressive and infiltrative behavior leading to high recurrence rates.
  • Although prognostic factors based on clinico-pathologic and immunohistochemical findings for determining the potential for recurrence of KCOT still remains unclear, its use for determining the potential for recurrence of KCOT after surgical treatment may become important to successfully manage this neoplasm's aggressive behavior.
  • The key element for future management of KCOTs will probably be based on thorough knowledge of the biological basis of this tumor, thereby enabling a more tailored treatment approach.
  • [MeSH-major] Jaw Neoplasms. Neoplasm Recurrence, Local. Odontogenic Cysts. Odontogenic Tumors
  • [MeSH-minor] Diagnosis, Differential. Female. Humans. Male. Prognosis

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  • [Copyright] Copyright 2010 Elsevier Ltd. All rights reserved.
  • (PMID = 20189443.001).
  • [ISSN] 1879-0593
  • [Journal-full-title] Oral oncology
  • [ISO-abbreviation] Oral Oncol.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
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23. Reichart PA, Jundt G: [Benign epithelial odontogenic tumors]. Pathologe; 2008 May;29(3):175-6, 178-88

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Benign epithelial odontogenic tumors].
  • [Transliterated title] Benigne epitheliale odontogene Tumoren.
  • The group of benign epithelial odontogenic tumors consists of the four member types of the ameloblastoma family (solid/multicystic, extraosseous/peripheral, desmoplastic, unicystic), squamous odontogenic tumors, calcifying odontogenic tumors, adenomatoid odontogenic tumors, and keratocystic odontogenic tumors, the former "keratocysts" that were recently reclassified by the World Health Organization and are now regarded as tumors.
  • The latter are by far the most frequent tumors in this group, followed by solid/multicystic ameloblastoma.
  • Although the etiology of these lesions is still unknown, a close relationship to normal tooth development is obvious, which is partially imitated by some tumors.
  • The diagnosis is essentially based on localization (intraosseous vs. extraosseous/peripheral) and histology, whereupon the correlation of histological findings with radiographic morphology may be of additional diagnostic value.
  • [MeSH-major] Jaw Neoplasms / pathology. Odontogenic Cyst, Calcifying / pathology. Odontogenic Tumor, Squamous / pathology. Odontoma / pathology
  • [MeSH-minor] Ameloblastoma / classification. Ameloblastoma / pathology. Ameloblastoma / surgery. Cementoma / classification. Cementoma / pathology. Cementoma / surgery. Diagnosis, Differential. Humans. Jaw / pathology. Jaw / surgery. Prognosis. Tomography, X-Ray Computed

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  • (PMID = 18389236.001).
  • [ISSN] 1432-1963
  • [Journal-full-title] Der Pathologe
  • [ISO-abbreviation] Pathologe
  • [Language] ger
  • [Publication-type] English Abstract; Journal Article; Review
  • [Publication-country] Germany
  • [Chemical-registry-number] Adenoameloblastoma
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24. Zhang L, Zeng D, Huang H, Wang J, Tao Q, Pan C, Xu J, Zhang B, Wang A: Tissue inhibitor of metalloproteinase-2 inhibits ameloblastoma growth in a new mouse xenograft disease model. J Oral Pathol Med; 2010 Jan;39(1):94-102
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Tissue inhibitor of metalloproteinase-2 inhibits ameloblastoma growth in a new mouse xenograft disease model.
  • BACKGROUND: Ameloblastomas are odontogenic neoplasms characterized by local invasiveness.
  • Newly established tumors on each mouse were injected with: (i) transfection reagent;.
  • Tumors were monitored for 5 weeks and excised for histopathology, RNA, and protein analyses.
  • In addition, we demonstrated the successful introduction of the TIMP-2 gene in tumor xenograft cells in vivo, resulting in xenograft growth inhibition.
  • [MeSH-major] Ameloblastoma / drug therapy. Neoplasm Transplantation. Soft Tissue Neoplasms / drug therapy. Tissue Inhibitor of Metalloproteinase-2 / therapeutic use. Transplantation, Heterologous
  • [MeSH-minor] Adult. Animals. Blotting, Western. Disease Models, Animal. Female. Genetic Vectors / therapeutic use. Humans. Matrix Metalloproteinase 2. Matrix Metalloproteinase Inhibitors. Mice. Mice, Inbred BALB C. Mice, Nude. Neoplasm Invasiveness. Plasmids / therapeutic use. Reverse Transcriptase Polymerase Chain Reaction. Subcutaneous Tissue / surgery. Transfection. Tumor Cells, Cultured

  • Mouse Genome Informatics (MGI). Mouse Genome Informatics (MGI) .
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  • (PMID = 19895658.001).
  • [ISSN] 1600-0714
  • [Journal-full-title] Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology
  • [ISO-abbreviation] J. Oral Pathol. Med.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Denmark
  • [Chemical-registry-number] 0 / Matrix Metalloproteinase Inhibitors; 127497-59-0 / Tissue Inhibitor of Metalloproteinase-2; EC 3.4.24.24 / Matrix Metalloproteinase 2; EC 3.4.24.24 / Mmp2 protein, mouse
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25. Reichart PA, Jundt G: [Benign "mixed" odontogenic tumors]. Pathologe; 2008 May;29(3):189-98

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Benign "mixed" odontogenic tumors].
  • [Transliterated title] Benigne „gemischte“ odontogene Tumoren.
  • Benign "mixed"odontogenic tumors consist of an epithelial and ectomesenchymal tumor component, distinguishing them from pure epithelial and pure ectomesenchymal odontogenic tumors.
  • Therefore, they can sometimes already be differentiated radiologically from epithelial odontogenic tumors.
  • Some of the mixed odontogenic lesions are regarded as true tumors (ameloblastic fibroma, odontoameloblastoma, dentinogenic ghost cell tumor), while others are assumed to represent hamartomatous lesions (complex and compound odontoma, probably also ameloblastic fibrodentinoma and ameloblastic fibroodontoma).
  • Preceded by keratocystic odontogenic tumor, complex and compound odontomas are the second most common odontogenic tumors, while other members of the "mixed" odontogenic tumor group are far less frequently diagnosed.
  • Odontoameloblastoma and dentinogenic ghost cell tumors are locally aggressive lesions that require total resection.
  • [MeSH-major] Jaw Neoplasms / pathology. Mixed Tumor, Malignant / pathology. Odontogenic Tumors / pathology

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  • (PMID = 18369623.001).
  • [ISSN] 1432-1963
  • [Journal-full-title] Der Pathologe
  • [ISO-abbreviation] Pathologe
  • [Language] ger
  • [Publication-type] English Abstract; Journal Article; Review
  • [Publication-country] Germany
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26. Kuroyanagi N, Sakuma H, Miyabe S, Machida J, Kaetsu A, Yokoi M, Maeda H, Warnakulasuriya S, Nagao T, Shimozato K: Prognostic factors for keratocystic odontogenic tumor (odontogenic keratocyst): analysis of clinico-pathologic and immunohistochemical findings in cysts treated by enucleation. J Oral Pathol Med; 2009 Apr;38(4):386-92
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Prognostic factors for keratocystic odontogenic tumor (odontogenic keratocyst): analysis of clinico-pathologic and immunohistochemical findings in cysts treated by enucleation.
  • BACKGROUND: The purpose of this study was to determine prognostic factors for the recurrence of keratocystic odontogenic tumors (KCOTs) following simple enucleation by examining clinico-pathologic and immunohistochemical findings.
  • METHODS: Following enucleation, the frequency of recurrence among 32 subjects diagnosed with KCOT was analyzed for tumor site, radiographic and histologic features, and immunopositivity for Ki-67 and p53.
  • RESULTS: Keratocystic odontogenic tumors in four out of 32 subjects (12.5%) recurred during the follow-up period (median: 33 months, range: 7-114 months).
  • CONCLUSION: The results suggested that the evaluation of Ki-67 expression in KCOT at the time of pathological diagnosis might be helpful for consideration of appropriate adjunctive surgical procedures to avoid a recurrence and may serve as a prognostic marker.
  • [MeSH-major] Odontogenic Cysts / metabolism. Odontogenic Cysts / pathology
  • [MeSH-minor] Adolescent. Adult. Aged. Female. Humans. Immunohistochemistry. Keratins. Ki-67 Antigen / biosynthesis. Male. Middle Aged. Prognosis. Recurrence. Tumor Suppressor Protein p53 / biosynthesis. Young Adult

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  • (PMID = 19141056.001).
  • [ISSN] 1600-0714
  • [Journal-full-title] Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology
  • [ISO-abbreviation] J. Oral Pathol. Med.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Denmark
  • [Chemical-registry-number] 0 / Ki-67 Antigen; 0 / Tumor Suppressor Protein p53; 68238-35-7 / Keratins
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27. Hyun HK, Hong SD, Kim JW: Recurrent keratocystic odontogenic tumor in the mandible: a case report and literature review. Oral Surg Oral Med Oral Pathol Oral Radiol Endod; 2009 Aug;108(2):e7-10

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Recurrent keratocystic odontogenic tumor in the mandible: a case report and literature review.
  • Keratocystic odontogenic tumor is characterized by benign intraosseous neoplasm of odontogenic origin with high recurrence rates.
  • Conservative approaches have some advantages in that they avoid adverse effects on the development of involved teeth and the jaw when the patient is young, although aggressive methods, including surgical resection, can be recommended considering the nature of the neoplasm.
  • Here we report a case of a 7-year-old patient with a recurrent keratocystic odontogenic tumor treated by marsupialization for normal development of the involved teeth.
  • [MeSH-major] Mandibular Neoplasms / surgery. Neoplasm Recurrence, Local / pathology. Odontogenic Cysts / surgery. Odontogenic Tumors / surgery

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  • (PMID = 19615649.001).
  • [ISSN] 1528-395X
  • [Journal-full-title] Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics
  • [ISO-abbreviation] Oral Surg Oral Med Oral Pathol Oral Radiol Endod
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Research Support, Non-U.S. Gov't; Review
  • [Publication-country] United States
  • [Number-of-references] 18
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28. Oliveira-Neto HH, Spíndula-Filho JV, Dallara MC, Silva CM, Mendonça EF, Batista AC: Unicystic ameloblastoma in a child: a differential diagnosis from the dentigerous cyst and the inflammatory follicular cyst. J Dent Child (Chic); 2007 Sep-Dec;74(3):245-9

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Unicystic ameloblastoma in a child: a differential diagnosis from the dentigerous cyst and the inflammatory follicular cyst.
  • Unicystic ameloblastoma (UA) is a benign epithelial odontogenic tumor of the jaws with an aggressive potential that commonly occurs in children.
  • This cystic odontogenic neoplasm is generally asymptomatic and found during routine radiographs.
  • The purposes of this report were to describe a case of UA involving the crown of the unerupted right mandibular second premolar in an 11-year-old girl under orthodontic treatment, and discuss its diagnosis and radiographic and microscopic findings, emphasizing its distinction from the dentigerous cyst and the inflammatory follicular cyst.
  • [MeSH-major] Ameloblastoma / pathology. Jaw Cysts / pathology. Mandibular Neoplasms / pathology
  • [MeSH-minor] Bicuspid / physiopathology. Child. Dentigerous Cyst / diagnosis. Diagnosis, Differential. Female. Follicular Cyst / diagnosis. Humans. Tooth, Unerupted / etiology

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  • (PMID = 18482524.001).
  • [ISSN] 1935-5068
  • [Journal-full-title] Journal of dentistry for children (Chicago, Ill.)
  • [ISO-abbreviation] J Dent Child (Chic)
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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29. Boffano P, Ruga E, Gallesio C: Keratocystic odontogenic tumor (odontogenic keratocyst): preliminary retrospective review of epidemiologic, clinical, and radiologic features of 261 lesions from University of Turin. J Oral Maxillofac Surg; 2010 Dec;68(12):2994-9

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Keratocystic odontogenic tumor (odontogenic keratocyst): preliminary retrospective review of epidemiologic, clinical, and radiologic features of 261 lesions from University of Turin.
  • PURPOSE: To analyze and discuss the demographic, clinical, and surgical aspects of 261 surgically treated keratocystic odontogenic tumors.
  • PATIENTS AND METHODS: A retrospective review was performed of all cases of treated keratocystic odontogenic tumors.
  • RESULTS: Keratocystic odontogenic tumors most commonly occurred in patients in the third and fourth decades of life.
  • [MeSH-major] Mandibular Neoplasms / pathology. Maxillary Neoplasms / pathology. Odontogenic Cysts / pathology. Odontogenic Tumors / pathology

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  • [Copyright] Copyright © 2010 American Association of Oral and Maxillofacial Surgeons. Published by Elsevier Inc. All rights reserved.
  • [CommentIn] J Oral Maxillofac Surg. 2011 May;69(5):1267-8; author reply 1268-9 [21501776.001]
  • (PMID = 20970907.001).
  • [ISSN] 1531-5053
  • [Journal-full-title] Journal of oral and maxillofacial surgery : official journal of the American Association of Oral and Maxillofacial Surgeons
  • [ISO-abbreviation] J. Oral Maxillofac. Surg.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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30. Mendes RA, Carvalho JF, van der Waal I: Biological pathways involved in the aggressive behavior of the keratocystic odontogenic tumor and possible implications for molecular oriented treatment - an overview. Oral Oncol; 2010 Jan;46(1):19-24
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Biological pathways involved in the aggressive behavior of the keratocystic odontogenic tumor and possible implications for molecular oriented treatment - an overview.
  • In the classification of Head and Neck Tumors, published in 2005 by the World Health Organization Classification, the odontogenic keratocyst has been reclassified as a benign intraosseous neoplasm, calling it "keratocystic odontogenic tumor" (KCOT).
  • Significant differences on the molecular level between KCOT and other odontogenic cystic lesions suggest a different biological origin.
  • Genetic and molecular research regarding odontogenic tumors, and KCOTs in particular, has led to an increasing amount of knowledge and understanding of their physiopathological pathways.
  • A review of the biological behavior of this recognized aggressive pathological entity of the jaws and a contemporary outline of the molecular (growth factors, p53, PCNA and Ki-67, bcl-2) and genetic (PTCH, SHH) alterations associated with this odontogenic neoplasm provides a better understanding of the mechanisms involved in its development and strengthen the current concept that the KCOT should, indeed, be regarded as a neoplasm.
  • Furthermore, markers known to be rapidly induced in response to growth factors, tumor promoters, cytokines, bacterial endotoxins, oncogenes, hormones and shear stress, such as COX-2, may also shed new light on the biological mechanisms involved in the development of these benign but sometimes aggressive neoplasms of the jaws.
  • [MeSH-major] Jaw Neoplasms. Odontogenic Cysts. Odontogenic Tumors

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  • (PMID = 20004133.001).
  • [ISSN] 1879-0593
  • [Journal-full-title] Oral oncology
  • [ISO-abbreviation] Oral Oncol.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] England
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31. Sedghizadeh PP, Wong D, Shuler CF, Linz V, Kalmar JR, Allen CM: Multifocal calcifying epithelial odontogenic tumor. Oral Surg Oral Med Oral Pathol Oral Radiol Endod; 2007 Aug;104(2):e30-4
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Multifocal calcifying epithelial odontogenic tumor.
  • The calcifying epithelial odontogenic tumor (CEOT), or Pindborg tumor, is a rare and benign odontogenic neoplasm that affects the jaw.
  • Multifocal odontogenic lesions are not typical but have been observed in conditions associated with known genetic mutations.
  • For example, multiple odontogenic keratocysts are the most common feature of the inherited condition known as nevoid basal cell carcinoma syndrome.
  • [MeSH-major] Jaw Neoplasms / pathology. Odontogenic Tumors / pathology
  • [MeSH-minor] Humans. Male. Middle Aged. Neoplasm Recurrence, Local. Rare Diseases

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  • (PMID = 17630096.001).
  • [ISSN] 1528-395X
  • [Journal-full-title] Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics
  • [ISO-abbreviation] Oral Surg Oral Med Oral Pathol Oral Radiol Endod
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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32. Sakai H, Mori T, Iida T, Tokuma Y, Maruo K, Masegi T: Immunohistochemical features of proliferative marker and basement membrane components of two feline inductive odontogenic tumours. J Feline Med Surg; 2008 Jul;10(3):296-9
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  • [Title] Immunohistochemical features of proliferative marker and basement membrane components of two feline inductive odontogenic tumours.
  • Feline inductive odontogenic tumour (FIOT) is a rare and interesting odontogenic neoplasm in which the odontogenic epithelium has inductive potential to form aggregated foci of dental pulp-like mesenchymal cells.
  • Histopathologically, the masses consisted of non-encapsulated invasive neoplasms exhibiting proliferation of epithelial and mesenchymal components with local infiltration into the maxillary bone in both cases.
  • [MeSH-major] Cat Diseases / diagnosis. Cat Diseases / immunology. Mandibular Neoplasms / veterinary. Odontogenic Tumors / veterinary

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  • (PMID = 17766158.001).
  • [ISSN] 1098-612X
  • [Journal-full-title] Journal of feline medicine and surgery
  • [ISO-abbreviation] J. Feline Med. Surg.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Biomarkers; 0 / Collagen Type IV; 0 / Ki-67 Antigen
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33. Szporek BJ, Cieślik T, Jedrzejewski PW, Lipiarz LZ: [Calcifying epithelial odontogenic tumor (Pindborg tumor)]. Wiad Lek; 2005;58(7-8):458-61
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Calcifying epithelial odontogenic tumor (Pindborg tumor)].
  • [Transliterated title] Nabłonkowy wapniejacy guz zebopochodny (guz Pindborga).
  • The calcifying epithelial odontogenic tumor (CEOT) is a rare benign odontogenic neoplasm which was first described by Pindborg in 1955 representing only 1% of all odontogenic tumors.
  • The tumor has an ectodermal odontogenic origin.
  • This tumor are considered benign but can be locally aggressive in nature with recurrence rates of 10-15% reported.
  • Since 1973 three cases only of the Pinborg tumor have been presented in the Polish literature.
  • We described the case of a 44-year-old man with Pindborg tumor in the right maxilla.
  • Standard x-ray examinations and CT scan were performed in order to obtain information about tumor's localization.
  • Autors discuss the radiologic features of calcifying epithelial odontogenic tumor and treatment method and the relevant literature.
  • [MeSH-major] Calcinosis / diagnosis. Maxillary Neoplasms / diagnosis. Odontogenic Tumors / diagnosis
  • [MeSH-minor] Adult. Humans. Male. Neoplasm Invasiveness

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  • (PMID = 16425804.001).
  • [ISSN] 0043-5147
  • [Journal-full-title] Wiadomości lekarskie (Warsaw, Poland : 1960)
  • [ISO-abbreviation] Wiad. Lek.
  • [Language] pol
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Poland
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34. Souza Andrade ES, da Costa Miguel MC, Pinto LP, de Souza LB: Ameloblastoma and adenomatoid odontogenic tumor: the role of alpha2beta1, alpha3beta1, and alpha5beta1 integrins in local invasiveness and architectural characteristics. Ann Diagn Pathol; 2007 Jun;11(3):199-205

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Ameloblastoma and adenomatoid odontogenic tumor: the role of alpha2beta1, alpha3beta1, and alpha5beta1 integrins in local invasiveness and architectural characteristics.
  • Ameloblastoma is an odontogenic neoplasm characterized by local invasiveness and a tendency toward recurrence, whereas adenomatoid odontogenic tumor (AOT) is an indolent neoplasm.
  • The objective of the present study was to immunohistochemically analyze the role of alpha2beta1, alpha3beta1, and alpha5beta1 integrins in the cellular events and cell-matrix interactions that occur in these tumors and their consequent repercussions on the architectural arrangement and biologic behavior of these lesions.
  • Paraffin-embedded specimens from 30 ameloblastomas (20 solid and 10 unicystic tumors) and 12 AOTs were submitted to immunohistochemistry using the catalyzed signal amplification system.
  • [MeSH-major] Ameloblastoma / pathology. Integrin alpha2beta1 / physiology. Integrin alpha3beta1 / physiology. Integrin alpha5beta1 / physiology. Jaw Neoplasms / pathology. Odontogenic Tumors / pathology
  • [MeSH-minor] Cell Communication. Gene Expression Regulation, Neoplastic. Humans. Neoplasm Invasiveness. Neoplasm Recurrence, Local / pathology

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  • (PMID = 17498594.001).
  • [ISSN] 1092-9134
  • [Journal-full-title] Annals of diagnostic pathology
  • [ISO-abbreviation] Ann Diagn Pathol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Integrin alpha2beta1; 0 / Integrin alpha3beta1; 0 / Integrin alpha5beta1
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35. Greer RO: Pathology of malignant and premalignant oral epithelial lesions. Otolaryngol Clin North Am; 2006 Apr;39(2):249-75, v
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  • Malignant neoplasia of bone, cartilage, salivary glands, and connective tissue and those of lymphoproliferative derivatives are far less common occurrences in the oral cavity.
  • Malignant neoplasms can and do arise from the tooth germ apparatus, but neoplasms of odontogenic elements are rare and are not included in this discussion.
  • [MeSH-major] Carcinoma, Squamous Cell / pathology. Mouth Mucosa / pathology. Mouth Neoplasms / pathology. Precancerous Conditions / pathology

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  • (PMID = 16580910.001).
  • [ISSN] 0030-6665
  • [Journal-full-title] Otolaryngologic clinics of North America
  • [ISO-abbreviation] Otolaryngol. Clin. North Am.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 54
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36. Kim K, Velez I, Kaltman SI, Lopez E, Stern D: Odontogenic carcinoma differentiation into rhabdomyosarcoma: report of a rare case. Quintessence Int; 2009 Nov-Dec;40(10):837-42

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Odontogenic carcinoma differentiation into rhabdomyosarcoma: report of a rare case.
  • To the authors' knowledge, this is the first reported case of an odontogenic carcinoma with documented skeletal muscle differentiation (rhabdomyosarcoma).
  • The histology and clinical features of this aggressive odontogenic neoplasm are described.
  • Within the English-language literature, only 2 cases are reported of an odontogenic tumor with muscle differentiation: a benign odontogenic tumor (ameloblastoma) with differentiation into a rhabdomyosarcoma and an odontogenic sarcoma with smooth-muscle differentiation.
  • The general practitioner should be aware that odontogenic lesions may be malignant, even though this is extremely rare, and all tissue removed from the oral cavity should be submitted for biopsy.
  • [MeSH-major] Gingival Neoplasms / pathology. Neoplasms, Multiple Primary / pathology. Odontogenic Tumors / pathology. Rhabdomyosarcoma / pathology
  • [MeSH-minor] Aged. Biopsy. Diagnosis, Differential. Follow-Up Studies. Gingival Diseases / diagnosis. Granuloma, Giant Cell / diagnosis. Granuloma, Pyogenic / diagnosis. Humans. Male. Maxillary Neoplasms / pathology

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  • (PMID = 19898715.001).
  • [ISSN] 1936-7163
  • [Journal-full-title] Quintessence international (Berlin, Germany : 1985)
  • [ISO-abbreviation] Quintessence Int
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Germany
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37. Bray D, Michael A, Falconer DT, Kaddour HS: Ameloblastoma: a rare nasal polyp. J Laryngol Otol; 2007 Jan;121(1):72-5

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Ameloblastoma is an odontogenic neoplasm of enamel organ type tissue which does not undergo transformation to the point of enamel formation.
  • The unusual presenting symptoms, as well as the highly destructive nature of these lesions when arising in the maxilla, make them worthy of consideration in the differential diagnosis of nasal and maxillary masses.
  • [MeSH-major] Ameloblastoma / pathology. Maxillary Neoplasms / pathology. Nasal Polyps / pathology

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  • (PMID = 17076931.001).
  • [ISSN] 1748-5460
  • [Journal-full-title] The Journal of laryngology and otology
  • [ISO-abbreviation] J Laryngol Otol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Review
  • [Publication-country] England
  • [Number-of-references] 20
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38. Siqueira AS, Carvalho MR, Monteiro AC, Freitas VM, Jaeger RG, Pinheiro JJ: Matrix metalloproteinases, TIMPs and growth factors regulating ameloblastoma behaviour. Histopathology; 2010 Jul;57(1):128-37
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  • AIMS: Ameloblastoma is an odontogenic neoplasm with local invasiveness and recurrence.
  • As a control, calcifying cystic odontogenic tumour (CCOT), a non-invasive odontogenic neoplasm with ameloblastomatous epithelium was also studied.
  • This neoplasm showed greater expression of MMPs, TIMPs and growth factors compared with CCOT.
  • [MeSH-major] Ameloblastoma / metabolism. Ameloblastoma / pathology. Intercellular Signaling Peptides and Proteins / metabolism. Jaw Neoplasms / metabolism. Jaw Neoplasms / pathology. Matrix Metalloproteinases / metabolism. Tissue Inhibitor of Metalloproteinases / metabolism
  • [MeSH-minor] Cell Proliferation. Epidermal Growth Factor / metabolism. Humans. Immunohistochemistry. MAP Kinase Signaling System. Matrix Metalloproteinase 1 / metabolism. Matrix Metalloproteinase 9 / metabolism. Odontogenic Cyst, Calcifying / metabolism. Odontogenic Cyst, Calcifying / pathology. Receptor, Epidermal Growth Factor / metabolism. Stromal Cells / metabolism. Stromal Cells / pathology. Tissue Inhibitor of Metalloproteinase-2 / metabolism. Transforming Growth Factor alpha / metabolism

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  • (PMID = 20653784.001).
  • [ISSN] 1365-2559
  • [Journal-full-title] Histopathology
  • [ISO-abbreviation] Histopathology
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Intercellular Signaling Peptides and Proteins; 0 / Tissue Inhibitor of Metalloproteinases; 0 / Transforming Growth Factor alpha; 127497-59-0 / Tissue Inhibitor of Metalloproteinase-2; 62229-50-9 / Epidermal Growth Factor; EC 2.7.10.1 / Receptor, Epidermal Growth Factor; EC 3.4.24.- / Matrix Metalloproteinases; EC 3.4.24.35 / Matrix Metalloproteinase 9; EC 3.4.24.7 / MMP1 protein, human; EC 3.4.24.7 / Matrix Metalloproteinase 1
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39. Nishioka H, Shibuya M, Izawa H, Ikeda Y: Primary suprasellar malignant tumor with odontogenic features: case report. Neurosurgery; 2009 Aug;65(2):E380-2; discussion E382
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  • [Title] Primary suprasellar malignant tumor with odontogenic features: case report.
  • OBJECTIVE: Primary malignant odontogenic tumors in the suprasellar region have rarely been reported.
  • CLINICAL PRESENTATION: A partially cystic tumor that originated from the stalk and showed rapid enlargement was observed in a 56-year-old woman.
  • INTERVENTION: Gross total removal of the tumor was performed with sacrifice of the stalk.
  • The tumor was a malignant epithelial type with odontogenic features, whereas findings consistent with craniopharyngioma were absent.
  • The tumor recurred 14 months after surgery.
  • CONCLUSION: The present unusual tumor may represent a novel member of malignant tumors of the suprasellar region.
  • Given the common embryological origin of craniopharyngiomas to odontogenic lesions, a possible correlation with craniopharyngioma was suggested.
  • [MeSH-major] Brain Neoplasms / pathology. Craniopharyngioma / pathology. Odontogenic Tumors / pathology. Pituitary Gland / pathology. Pituitary Neoplasms / pathology
  • [MeSH-minor] Biomarkers, Tumor / analysis. Biomarkers, Tumor / metabolism. Female. Headache / etiology. Humans. Ki-67 Antigen / analysis. Ki-67 Antigen / metabolism. Magnetic Resonance Imaging. Middle Aged. Mitotic Index. Neoplasm Invasiveness / pathology. Neoplasm Invasiveness / physiopathology. Neoplasm Invasiveness / prevention & control. Neoplasm Recurrence, Local / physiopathology. Neoplasm Recurrence, Local / prevention & control. Neoplasm Recurrence, Local / surgery. Neurosurgical Procedures. Sella Turcica / pathology. Sella Turcica / radiography. Sella Turcica / surgery. Tomography, X-Ray Computed. Tooth / cytology. Tooth / embryology. Treatment Outcome

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  • (PMID = 19625894.001).
  • [ISSN] 1524-4040
  • [Journal-full-title] Neurosurgery
  • [ISO-abbreviation] Neurosurgery
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Biomarkers, Tumor; 0 / Ki-67 Antigen
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40. Bozza F, Marcelli VA, Pistilli R, Govoni FA, Marsico C: Maxillary ameloblastoma. Minerva Stomatol; 2006 Apr;55(4):215-22

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Maxillary ameloblastoma is a rare odontogenic neoplasm that is histologically benign and originates from epithelial cells present in bone tissue.
  • If excised through conservative surgery, this tumour has a high relapse rate and is locally aggressive.
  • [MeSH-major] Ameloblastoma / surgery. Maxillary Neoplasms / surgery

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  • (PMID = 16618996.001).
  • [ISSN] 0026-4970
  • [Journal-full-title] Minerva stomatologica
  • [ISO-abbreviation] Minerva Stomatol
  • [Language] eng; ita
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Italy
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41. Sulzbacher I, Wick N, Pichlhofer B, Mazal PR: Expression of platelet-derived growth factor-AA and platelet-derived growth factor-alpha receptor in ameloblastomas. J Oral Pathol Med; 2008 Apr;37(4):235-40

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • We investigated the expression of both proteins in ameloblastomas, to contribute the understanding of the potential role of the PDGF/PDGFR system in this odontogenic neoplasm.
  • RESULTS: PDGF-AA and PDGFRA expression were detectable in all cases with the exception of one tumor.
  • Unicystic ameloblastomas did not differ from solid tumors with regard to PDGF-AA, PDGFRA, and MIB-1 expression.
  • One tumor revealed a somatic mutation of exon 12 of the PDGFRA gene.
  • [MeSH-major] Ameloblastoma / metabolism. Jaw Neoplasms / metabolism. Platelet-Derived Growth Factor / biosynthesis. Receptor, Platelet-Derived Growth Factor alpha / biosynthesis

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  • (PMID = 18284546.001).
  • [ISSN] 1600-0714
  • [Journal-full-title] Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology
  • [ISO-abbreviation] J. Oral Pathol. Med.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Denmark
  • [Chemical-registry-number] 0 / Antibodies, Antinuclear; 0 / Antibodies, Monoclonal; 0 / MIB-1 antibody; 0 / Platelet-Derived Growth Factor; 0 / platelet-derived growth factor A; EC 2.7.10.1 / Receptor, Platelet-Derived Growth Factor alpha
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42. Kreutzer R, Wohlsein P, Staszyk C, Nowak M, Sill V, Baumgärtner W: Dental benign cementomas in three horses. Vet Pathol; 2007 Jul;44(4):533-6

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Cementoma is a very rare odontogenic neoplasm of mesenchymal origin.
  • Histopathologically, the tumors consisted of excessive deposition of cementum-like tissue.
  • Cells, resembling cementoblasts, lined irregularly shaped lacunae, which were present in the tumor tissue, and showed minimal cellular pleomorphism.
  • However, due to their high degree of differentiation, hypercementosis must be considered as a differential diagnosis.

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  • (PMID = 17606518.001).
  • [ISSN] 0300-9858
  • [Journal-full-title] Veterinary pathology
  • [ISO-abbreviation] Vet. Pathol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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43. Peron JM, Hardy H: [Mixed odontogenic tumours]. Rev Stomatol Chir Maxillofac; 2009 Sep;110(4):217-20

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Mixed odontogenic tumours].
  • [Transliterated title] Tumeurs odontogéniques mixtes.
  • Mixed odontogenic tumors include all tumors of the teeth both epithelial and mesenchymatous.
  • Most of these tumors are rare (ameloblastic fibroma, ameloblastic fibro-odontoma and fibrodentinoma, odontoameloblastoma, calcified odontogenic cyst).
  • Other tumors such as odontoma are frequent.
  • The diagnosis may be confirmed only by an anatomopathological examination, along with clinical and radiological observations.
  • [MeSH-major] Jaw Neoplasms / diagnosis. Odontogenic Tumors / diagnosis
  • [MeSH-minor] Ameloblastoma / diagnosis. Diagnosis, Differential. Humans. Odontogenic Cyst, Calcifying / diagnosis. Odontoma / diagnosis. Young Adult

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  • (PMID = 19660774.001).
  • [ISSN] 1776-257X
  • [Journal-full-title] Revue de stomatologie et de chirurgie maxillo-faciale
  • [ISO-abbreviation] Rev Stomatol Chir Maxillofac
  • [Language] fre
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] France
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44. Dinkar AD, Sahai S, Sharma M: Primary hyperparathyroidism presenting as an exophytic mandibular mass. Dentomaxillofac Radiol; 2007 Sep;36(6):360-3
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  • The initial clinical and radiological evaluation indicated an aggressive odontogenic neoplasm or a metastasis from an unknown primary; the suspicion of a systemic metabolic or endocrine disorder lay low on the list of differential diagnoses.
  • The total serum parathyroid hormone levels and ultrasound-guided fine needle aspiration cytology (FNAC) of the mandibular and parathyroid lesions provided the diagnosis of primary hyperparathyroidism presenting as a mandibular brown tumour.
  • [MeSH-major] Hyperparathyroidism, Primary / diagnosis. Mandibular Diseases / diagnosis
  • [MeSH-minor] Adenoma / diagnosis. Adult. Biopsy, Fine-Needle. Diagnosis, Differential. Female. Granuloma, Giant Cell / diagnosis. Humans. Osteitis Fibrosa Cystica / diagnosis. Parathyroid Hormone / blood. Parathyroid Neoplasms / diagnosis. Ultrasonography, Interventional

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  • (PMID = 17699707.001).
  • [ISSN] 0250-832X
  • [Journal-full-title] Dento maxillo facial radiology
  • [ISO-abbreviation] Dentomaxillofac Radiol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Parathyroid Hormone
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45. Ritwik P, Brannon RB: Peripheral odontogenic fibroma: a clinicopathologic study of 151 cases and review of the literature with special emphasis on recurrence. Oral Surg Oral Med Oral Pathol Oral Radiol Endod; 2010 Sep;110(3):357-63
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Peripheral odontogenic fibroma: a clinicopathologic study of 151 cases and review of the literature with special emphasis on recurrence.
  • OBJECTIVE: The peripheral odontogenic fibroma (POdF) is a rare benign neoplasm of odontogenic origin with limited data on recurrence.
  • RESULTS: POdF should be considered a mixed odontogenic tumor because it is composed of active odontogenic epithelial and ectomesenchymal components.
  • Budding of the surface epithelium and calcification in apposition to odontogenic epithelial rests are histologic predictors of recurrence.
  • [MeSH-major] Fibroma / pathology. Jaw Neoplasms / pathology. Neoplasm Recurrence, Local / pathology. Odontogenic Tumors / pathology
  • [MeSH-minor] Adolescent. Adult. Age Distribution. Aged. Aged, 80 and over. Child. Child, Preschool. Follow-Up Studies. Humans. Infant. Infant, Newborn. Logistic Models. Male. Middle Aged. Mixed Tumor, Malignant / pathology. Sex Distribution. Time Factors. Young Adult

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  • [Copyright] Copyright (c) 2010 Mosby, Inc. All rights reserved.
  • (PMID = 20674403.001).
  • [ISSN] 1528-395X
  • [Journal-full-title] Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics
  • [ISO-abbreviation] Oral Surg Oral Med Oral Pathol Oral Radiol Endod
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
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46. Mendenhall WM, Werning JW, Fernandes R, Malyapa RS, Mendenhall NP: Ameloblastoma. Am J Clin Oncol; 2007 Dec;30(6):645-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Ameloblastoma is an uncommon benign, locally aggressive odontogenic neoplasm that usually occurs in the vicinity of the mandibular molars or ramus.
  • Limited experience with radiotherapy indicates that it may reduce the risk of progression and result in long-term local control in the occasional patient with incompletely resectable disease.
  • Radiotherapy may improve the likelihood of local control in the occasional patient with incompletely resectable tumor.
  • [MeSH-major] Ameloblastoma / radiotherapy. Ameloblastoma / surgery. Jaw Neoplasms / radiotherapy. Jaw Neoplasms / surgery

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  • (PMID = 18091060.001).
  • [ISSN] 1537-453X
  • [Journal-full-title] American journal of clinical oncology
  • [ISO-abbreviation] Am. J. Clin. Oncol.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 27
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47. Kallel R, Ayadi L, Gouiaa N, Chaabouni S, Karray F, Daoud E, Chaari C, Khabir A, Makni S, Boudawara T: [Extrafollicular adenomatoid odontogenic tumour: report of two cases]. Rev Med Brux; 2009 Sep-Oct;30(5):511-4

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Extrafollicular adenomatoid odontogenic tumour: report of two cases].
  • [Transliterated title] Tumeur odontogène adénomatoïde extrafolliculaire: à propos de deux cas.
  • The purpose of this study was to analysis the clinicopathological features of adenomatoid odontogenic tumour extrafollicular and to discuss the diagnosis pitfull.
  • An exploratory surgical approach was chosen and the final diagnosis was microscopically confirmed to be an extrafollicular variant of adenomatoid odontogenic tumour.
  • The two cases described illustrate clinical and radiographic features of the extrafollicular variant of adenomatoid odontogenic tumour.
  • Careful diagnostic procedure and adequate interpretation of radiographic findings may result in a correct diagnosis with otherwise may result in unnecessary endodontic treatment.
  • [MeSH-major] Mandibular Neoplasms / radiography. Odontogenic Tumors / radiography

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  • (PMID = 19998797.001).
  • [ISSN] 0035-3639
  • [Journal-full-title] Revue médicale de Bruxelles
  • [ISO-abbreviation] Rev Med Brux
  • [Language] fre
  • [Publication-type] Case Reports; English Abstract; Journal Article
  • [Publication-country] Belgium
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48. Vanoven BJ, Parker NP, Petruzzelli GJ: Peripheral ameloblastoma of the maxilla: a case report and literature review. Am J Otolaryngol; 2008 Sep-Oct;29(5):357-60
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Peripheral ameloblastoma is a rare, benign, extraosseous neoplasm of the odontogenic epithelium.
  • We report a case of peripheral ameloblastoma, detail our surgical therapy and review important elements in the pathological diagnosis and treatment of this unusual neoplasm.
  • [MeSH-major] Ameloblastoma / pathology. Ameloblastoma / surgery. Maxillary Neoplasms / pathology. Maxillary Neoplasms / surgery
  • [MeSH-minor] Aged. Biopsy, Needle. Follow-Up Studies. Humans. Immunohistochemistry. Male. Neoplasm Staging. Risk Assessment. Tomography, X-Ray Computed. Treatment Outcome

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  • (PMID = 18722894.001).
  • [ISSN] 1532-818X
  • [Journal-full-title] American journal of otolaryngology
  • [ISO-abbreviation] Am J Otolaryngol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 23
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49. Pontes HA, Pontes FS, Silva BS, Cury SE, Fonseca FP, Salim RA, Pinto Júnior Ddos S: Immunoexpression of Ki67, proliferative cell nuclear antigen, and Bcl-2 proteins in a case of ameloblastic fibrosarcoma. Ann Diagn Pathol; 2010 Dec;14(6):447-52
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Ameloblastic fibrosarcoma (AFS), regarded as the malignant counterpart of the benign ameloblastic fibroma, is an extremely rare odontogenic neoplasm with only 68 cases reported in the English literature up to 2009.
  • It is composed of a benign odontogenic epithelium, resembling that of ameloblastoma, and a malignant mesenchymal part exhibiting features of fibrosarcoma.
  • [MeSH-major] Fibrosarcoma / metabolism. Ki-67 Antigen / metabolism. Mandibular Neoplasms / metabolism. Odontogenic Tumors / metabolism. Proliferating Cell Nuclear Antigen / metabolism. Proto-Oncogene Proteins c-bcl-2 / metabolism
  • [MeSH-minor] Adult. Ameloblasts / metabolism. Ameloblasts / pathology. Biomarkers, Tumor / metabolism. Combined Modality Therapy. Female. Humans

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  • [Copyright] Copyright © 2010 Elsevier Inc. All rights reserved.
  • (PMID = 21074695.001).
  • [ISSN] 1532-8198
  • [Journal-full-title] Annals of diagnostic pathology
  • [ISO-abbreviation] Ann Diagn Pathol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Biomarkers, Tumor; 0 / Ki-67 Antigen; 0 / Proliferating Cell Nuclear Antigen; 0 / Proto-Oncogene Proteins c-bcl-2
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50. de Amorim RF, Silveira EJ, França MN, Guimarães Mdo C, Lima Júnior N, de Carvalho DR: A case of extensive maxillary benign cementoblastoma. J Contemp Dent Pract; 2010;11(3):056-62

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • AIM: The aim of the present paper is to report a case of benign cementoblastoma (BC) involving multiple maxillary teeth and discuss the importance of its diagnosis in clinical oral practice.
  • BACKGROUND: BC is a rare, benign odontogenic neoplasm of ectomesenchymal origin.
  • This neoplasm has characteristic radiologic and microscopic features, and it is intimately associated with the roots of teeth.
  • SUMMARY: In most cases, initial BC is asymptomatic and early diagnosis can be performed by routine radiographic examination.
  • CLINICAL SIGNIFICANCE: Because a benign cementoblastoma represents an odontogenic lesion, general practicing professionals must be able to identify the main features of this tumor.
  • Therefore, a brief update concerning the clinical and radiographic features of this unique tumor is also provided.
  • [MeSH-major] Maxillary Neoplasms / diagnosis. Odontogenic Tumors / diagnosis
  • [MeSH-minor] Bicuspid / pathology. Biopsy. Diagnosis, Differential. Follow-Up Studies. Humans. Male. Molar / pathology. Palatal Neoplasms / diagnosis. Radiography, Panoramic. Tooth Root / pathology. Young Adult

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  • (PMID = 20461325.001).
  • [ISSN] 1526-3711
  • [Journal-full-title] The journal of contemporary dental practice
  • [ISO-abbreviation] J Contemp Dent Pract
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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51. Matijević M, Leović D, Popić B, Zubcić V, Kopić V, Prlić A, Siber S, Dinjar K: The importance of thorough preoperative diagnostics of maxillary ameloblastoma: report of three cases. Coll Antropol; 2010 Dec;34(4):1445-8

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Ameloblastoma, especially maxillary, is a rare benign neoplasm of odontogenic origin.
  • Diagnosis of significant number of lesions is usually established postoperatively, because ameloblastoma, especially the unicystic form, mimics wide range of more frequent jaw lesions.
  • [MeSH-major] Ameloblastoma / diagnosis. Maxillary Neoplasms / diagnosis

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  • (PMID = 21874736.001).
  • [ISSN] 0350-6134
  • [Journal-full-title] Collegium antropologicum
  • [ISO-abbreviation] Coll Antropol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Croatia
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52. Biswas D, Crank S: Aetiopathology of maxillary swelling--a 3-year prospective study. Eur Arch Otorhinolaryngol; 2007 Nov;264(11):1295-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • A wide variety of lesions and not necessarily a malignant tumour can cause maxillary swelling.
  • Non-specificity of clinical and radiological features of these maxillary lesions makes their diagnosis difficult.
  • We present our finding regarding the relative distribution of various conditions causing maxillary swelling.
  • The awareness of the spectrum of pathology related to maxillary swelling is essential for correct diagnosis and treatment.
  • Maxillary swelling was found to be caused by malignant tumours in 54.2%, benign neoplasms in 22.9% and non-neoplastic lesions in 22.9%.
  • Overall squamous cell carcinoma (22.9%) was the commonest lesion, tumour of vascular origin was the commonest benign neoplasm and odontogenic cyst was the commonest among the non-neoplastic lesions.
  • [MeSH-minor] Adolescent. Adult. Aged. Aged, 80 and over. Child. Child, Preschool. Female. Fibrous Dysplasia of Bone / complications. Humans. Male. Middle Aged. Mucocele / complications. Nasal Obstruction / complications. Odontogenic Cysts / complications. Preoperative Care. Prospective Studies. Young Adult

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  • (PMID = 17611767.001).
  • [ISSN] 1434-4726
  • [Journal-full-title] European archives of oto-rhino-laryngology : official journal of the European Federation of Oto-Rhino-Laryngological Societies (EUFOS) : affiliated with the German Society for Oto-Rhino-Laryngology - Head and Neck Surgery
  • [ISO-abbreviation] Eur Arch Otorhinolaryngol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Germany
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53. Sandhu SV, Narang RS, Jawanda M, Rai S: Adenomatoid odontogenic tumor associated with dentigerous cyst of the maxillary antrum: A rare entity. J Oral Maxillofac Pathol; 2010 Jan;14(1):24-8

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Adenomatoid odontogenic tumor associated with dentigerous cyst of the maxillary antrum: A rare entity.
  • Adenomatoid odontogenic tumor (AOT) is an uncommon tumor of odontogenic origin composed of odontogenic epithelium in a variety of histoarchitectural patterns.

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  • (PMID = 21180455.001).
  • [ISSN] 1998-393X
  • [Journal-full-title] Journal of oral and maxillofacial pathology : JOMFP
  • [ISO-abbreviation] J Oral Maxillofac Pathol
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] India
  • [Other-IDs] NLM/ PMC2996001
  • [Keywords] NOTNLM ; Adenomatoid odontogenic tumor / dentigerous cyst / maxillary antrum
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54. Silva Gurgel CA, Gonçalves Ramos EA, Araújo Melo L, Brandi Schlaepfer C, de Souza RO, Campos Oliveira M, dos Santos JN: Immunolocalisation of laminin-1 in keratocystic odontogenic tumors. Acta Histochem; 2010 Nov;112(6):624-9

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Immunolocalisation of laminin-1 in keratocystic odontogenic tumors.
  • Keratocystic odontogenic tumors (KOTs) are distinct odontogenic lesions frequently affecting the jawbones.
  • The aim of this study was to investigate the immunolocalisation of laminin-1 in 20 cases of KOTs in order to contribute to the characterization of this protein, which is little studied in odontogenic tumors.
  • Furthermore, the discontinuous and weak labelling of this protein in the basement membrane of these tumors is probably a consequence of the inflammatory process in the tumor stroma.
  • [MeSH-major] Jaw Neoplasms / metabolism. Jaw Neoplasms / pathology. Laminin / analysis. Odontogenic Tumors / metabolism. Odontogenic Tumors / pathology

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  • [Copyright] Copyright © 2009 Elsevier GmbH. All rights reserved.
  • (PMID = 19773031.001).
  • [ISSN] 1618-0372
  • [Journal-full-title] Acta histochemica
  • [ISO-abbreviation] Acta Histochem.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Germany
  • [Chemical-registry-number] 0 / Laminin; 0 / laminin 1
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55. Chuan-Xiang Z, Yan G: Adenomatoid odontogenic tumor: a report of a rare case with recurrence. J Oral Pathol Med; 2007 Aug;36(7):440-3

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Adenomatoid odontogenic tumor: a report of a rare case with recurrence.
  • Adenomatoid odontogenic tumor (AOT) is an uncommon tumor of odontogenic origin, composed of odontogenic epithelium in a variety of histoarchitectural patterns, and characterized by slow but progressive growth.
  • The tumor was diagnosed as AOT with well-defined unilocular radiolucency shown in X-ray graphs and solid nodule of cuboidal or columnar cells of odontogenic epithelium forming typical nests or rosette-like structures as well as characteristic duct-like spaces in histologic findings.
  • [MeSH-major] Maxillary Neoplasms / pathology. Neoplasm Recurrence, Local / pathology. Odontogenic Tumors / pathology
  • [MeSH-minor] Adolescent. Adult. Cuspid / pathology. Dentigerous Cyst / diagnosis. Diagnosis, Differential. Follow-Up Studies. Humans. Male. Tooth, Unerupted / pathology

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  • (PMID = 17617839.001).
  • [ISSN] 0904-2512
  • [Journal-full-title] Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology
  • [ISO-abbreviation] J. Oral Pathol. Med.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Denmark
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56. Faitaroni LA, Bueno MR, De Carvalhosa AA, Bruehmueller Ale KA, Estrela C: Ameloblastoma suggesting large apical periodontitis. J Endod; 2008 Feb;34(2):216-9

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • The microscopic examination indicated a diagnosis of ameloblastoma.
  • Ameloblastoma is a benign epithelial neoplasm of odontogenic origin, and depending on the stage of development, it can mimic a periapical lesion and therefore should be considered in establishing an endodontic differential diagnosis.
  • The definitive diagnosis for some periapical lesions can only be made by a histopathologic examination.
  • [MeSH-major] Ameloblastoma / diagnosis. Mandibular Neoplasms / diagnosis. Periapical Periodontitis / diagnosis
  • [MeSH-minor] Diagnosis, Differential. Disease Progression. Follow-Up Studies. Humans. Male. Mandibular Diseases / diagnosis. Middle Aged. Root Canal Therapy

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  • (PMID = 18215686.001).
  • [ISSN] 0099-2399
  • [Journal-full-title] Journal of endodontics
  • [ISO-abbreviation] J Endod
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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57. Gupta N, Bhat M, Sharma R: What lies within? Int J Clin Pediatr Dent; 2009 Jan;2(1):56-8

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • Biopsy was sent for histopathological examination revealing the miracle diagnosis of "Central Odontogenic Fibroma" being a rare tumor of odontogenic tumor family and also a rare finding regard to age, site and clinical behavior of tumor is reported.

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  • (PMID = 25206103.001).
  • [ISSN] 0974-7052
  • [Journal-full-title] International journal of clinical pediatric dentistry
  • [ISO-abbreviation] Int J Clin Pediatr Dent
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] India
  • [Other-IDs] NLM/ PMC4086555
  • [Keywords] NOTNLM ; Odontogenic fibroma / odontogenic tumor.
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58. Nigam S, Gupta SK, Chaturvedi KU: Adenomatoid odontogenic tumor--a rare cause of jaw swelling. Braz Dent J; 2005;16(3):251-3

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Adenomatoid odontogenic tumor--a rare cause of jaw swelling.
  • Adenomatoid odontogenic tumor (AOT) is an uncommon tumor of odontogenic origin, characterized histologically by the formation of ductlike structures with amyloid-like deposits.
  • Histogenesis of AOT is still uncertain and it is often considered as a hamartomatous lesion rather than a true neoplasm.
  • [MeSH-major] Maxillary Neoplasms / diagnosis. Odontogenic Tumors / diagnosis
  • [MeSH-minor] Adolescent. Bicuspid / pathology. Biopsy, Fine-Needle. Cuspid / pathology. Diagnosis, Differential. Female. Humans. Incisor / pathology. Tooth Resorption / diagnosis. Tooth, Impacted / diagnosis

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  • (PMID = 16429194.001).
  • [ISSN] 1806-4760
  • [Journal-full-title] Brazilian dental journal
  • [ISO-abbreviation] Braz Dent J
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Review
  • [Publication-country] Brazil
  • [Number-of-references] 8
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59. Hong YS, Wang J, Liu J, Zhang B, Hou L, Zhong M: [Expression of HOX C13 in odontogenic tumors]. Shanghai Kou Qiang Yi Xue; 2007 Dec;16(6):587-91
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Expression of HOX C13 in odontogenic tumors].
  • PURPOSE: To study the expression of HOXC13 mRNA in odontogenic tumors.
  • METHODS: HOXC13 mRNA was detected in 47 cases of ameloblastoma (AB), 3 cases of calcifying cystic odontogenic tumor (CCOT), 3 cases of ameloblastic fibroma (AF), 10 cases of keratocystic odontogenic tumor (KCOT) and 2 cases of calcifying epithelial odontogenic tumor (CEOT) by in situ hybridization, and 7 cases of normal oral mucosa were selected as control.
  • RESULTS: HOXC13 mRNA was positively expressed in all odontogenic epithelium except AF.
  • CONCLUSIONS: The genesis and development of odontogenic tumors are related to the high expression of HOXC13.
  • The expression of HOXC13 mRNA in the odontogenic lesions has heterogeneity.
  • [MeSH-major] Homeodomain Proteins / biosynthesis. Odontogenic Cyst, Calcifying / metabolism. Odontogenic Tumors / metabolism. Skin Neoplasms / metabolism

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  • (PMID = 18278407.001).
  • [ISSN] 1006-7248
  • [Journal-full-title] Shanghai kou qiang yi xue = Shanghai journal of stomatology
  • [ISO-abbreviation] Shanghai Kou Qiang Yi Xue
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] China
  • [Chemical-registry-number] 0 / Homeodomain Proteins; 0 / RNA, Messenger; Calcifying Epithelial Odontogenic Tumor
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60. Crivelini MM, Felipini RC, Coclete GA, Soubhia AM: Immunoexpression of keratins in the calcifying cystic odontogenic tumor epithelium. J Oral Pathol Med; 2009 Apr;38(4):393-6
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Immunoexpression of keratins in the calcifying cystic odontogenic tumor epithelium.
  • Ameloblastomatous epithelium containing clusters of ghost cells is the typical histopathology of calcifying cystic odontogenic tumor (CCOT).
  • When compared to the immunoprofile of the normal odontogenic epithelium, of odontogenic tumor epithelia and of the ghost cells described in the literature, it was possible to suggest that the CCOT epithelium differentiates towards squamous type.
  • [MeSH-major] Jaw Neoplasms / metabolism. Jaw Neoplasms / pathology. Keratins / biosynthesis. Odontogenic Cyst, Calcifying / metabolism. Odontogenic Cyst, Calcifying / pathology

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  • (PMID = 19222713.001).
  • [ISSN] 1600-0714
  • [Journal-full-title] Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology
  • [ISO-abbreviation] J. Oral Pathol. Med.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Denmark
  • [Chemical-registry-number] 0 / Collagen Type IV; 0 / Laminin; 0 / Vimentin; 68238-35-7 / Keratins
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61. Batra P, Prasad S, Parkash H: Adenomatoid odontogenic tumour: review and case report. J Can Dent Assoc; 2005 Apr;71(4):250-3

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Adenomatoid odontogenic tumour: review and case report.
  • Adenomatoid odontogenic tumour is a benign (hamartomatous), noninvasive lesion with slow but progressive growth.
  • This report describes a patient with a large adenomatoid odontogenic tumour in the mandible, with the involved mandibular canine being pushed to the contralateral side.
  • The paper also provides a refresher for general dental practitioners about various diagnostic aspects of this tumour and highlights the controversies regarding its origin and management in light of recent findings.
  • [MeSH-major] Mandibular Neoplasms / pathology. Odontogenic Tumors / pathology
  • [MeSH-minor] Adolescent. Diagnosis, Differential. Female. Humans

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  • (PMID = 15871773.001).
  • [ISSN] 1488-2159
  • [Journal-full-title] Journal (Canadian Dental Association)
  • [ISO-abbreviation] J Can Dent Assoc
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Review
  • [Publication-country] Canada
  • [Number-of-references] 29
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62. Lucchese A, Scivetti M, Pilolli GP, Favia G: Analysis of ghost cells in calcifying cystic odontogenic tumors by confocal laser scanning microscopy. Oral Surg Oral Med Oral Pathol Oral Radiol Endod; 2007 Sep;104(3):391-4

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Analysis of ghost cells in calcifying cystic odontogenic tumors by confocal laser scanning microscopy.
  • In this study we have used the confocal microscope to analyze ghost cells in calcifying cystic odontogenic tumors.
  • STUDY DESIGN: Specimens from 15 calcifying cystic odontogenic tumor cases were stained with hematoxylin and eosin, and scanned by a confocal laser scanning microscope to generate optically sectioned images.
  • CONCLUSION: The confocal laser scanning microscope may be of help in analyzing and defining the nature and extent of keratinization processes in calcifying cystic odontogenic tumor ghost cells.
  • [MeSH-major] Jaw Neoplasms / ultrastructure. Microscopy, Confocal. Odontogenic Cyst, Calcifying / ultrastructure

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  • (PMID = 17188527.001).
  • [ISSN] 1528-395X
  • [Journal-full-title] Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics
  • [ISO-abbreviation] Oral Surg Oral Med Oral Pathol Oral Radiol Endod
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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63. Perdigão PF, Carvalho VM, DE Marco L, Gomez RS: Mutation of ameloblastin gene in calcifying epithelial odontogenic tumor. Anticancer Res; 2009 Aug;29(8):3065-7
NCI CPTC Antibody Characterization Program. NCI CPTC Antibody Characterization Program .

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Mutation of ameloblastin gene in calcifying epithelial odontogenic tumor.
  • Due to the relationship of this protein in the differentiation and proliferation of odontogenic cells, here, we investigated this gene in different types of odontogenic tumors.
  • MATERIALS AND METHODS: Sequencing of the all encoding region of AMBN gene was carried out in four frozen cases of odontogenic tumors: one case of calcifying epithelial odontogenic tumor (CEOT), two calcifying odontogenic cysts (COC) and one ameloblastic fibroma (AF).
  • [MeSH-major] Ameloblasts / pathology. Calcinosis / pathology. Dental Enamel Proteins / genetics. Epithelial Cells / pathology. Fibroma / pathology. Mutation / genetics. Odontogenic Tumors / genetics

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  • (PMID = 19661317.001).
  • [ISSN] 1791-7530
  • [Journal-full-title] Anticancer research
  • [ISO-abbreviation] Anticancer Res.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Greece
  • [Chemical-registry-number] 0 / AMBN protein, human; 0 / Dental Enamel Proteins
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64. Okamoto E, Kikuchi K, Miyazaki Y, González-Alva P, Oku Y, Tanaka A, Yoshida N, Fujinami M, Ide F, Sakashita H, Kusama K: Significance of podoplanin expression in keratocystic odontogenic tumor. J Oral Pathol Med; 2010 Jan;39(1):110-4

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Significance of podoplanin expression in keratocystic odontogenic tumor.
  • BACKGROUND: The most important clinical features of the keratocystic odontogenic tumor (KCOT) are its potential for locally destructive behavior, a tendency to recur, and its origin in the odontogenic epithelium.
  • Histologically, KCOT is distinguished from jaw cyst with keratinization (orthokeratinized odontogenic cyst; OOC).
  • The purpose of this study was to determine the usefulness of podoplanin for reclassification of the odontogenic keratocyst (OKC) from cyst to tumor status.
  • The pattern of staining for podoplanin in KCOT could be related to its neoplastic nature, and suggests a role of the protein in tumor invasiveness.
  • [MeSH-major] Membrane Glycoproteins / analysis. Odontogenic Cysts / pathology. Odontogenic Tumors / pathology
  • [MeSH-minor] Cell Membrane / ultrastructure. Cell Proliferation. Cell Transformation, Neoplastic / pathology. Connective Tissue Cells / pathology. Cytoplasm / ultrastructure. Dentigerous Cyst / pathology. Disease Progression. Endothelial Cells / pathology. Epithelial Cells / pathology. Epithelium / pathology. Humans. Lymphatic Vessels / pathology. Mouth Mucosa / pathology. Neoplasm Invasiveness. Neoplasm Recurrence, Local / pathology. Stromal Cells / pathology

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  • (PMID = 19925582.001).
  • [ISSN] 1600-0714
  • [Journal-full-title] Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology
  • [ISO-abbreviation] J. Oral Pathol. Med.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Denmark
  • [Chemical-registry-number] 0 / Membrane Glycoproteins; 0 / PDPN protein, human
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65. Ladeinde AL, Ajayi OF, Ogunlewe MO, Adeyemo WL, Arotiba GT, Bamgbose BO, Akinwande JA: Odontogenic tumors: a review of 319 cases in a Nigerian teaching hospital. Oral Surg Oral Med Oral Pathol Oral Radiol Endod; 2005 Feb;99(2):191-5

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Odontogenic tumors: a review of 319 cases in a Nigerian teaching hospital.
  • OBJECTIVE: This study sought to determine the relative frequency of odontogenic tumors in a Nigerian population and to compare these data with previous reports.
  • STUDY DESIGN: Records of patients seen at the Lagos University Teaching Hospital between January 1980 and December 2003, with histologic diagnosis of odontogenic tumors (based on World Health Organisation classification, 1992), were analyzed.
  • RESULTS: Odontogenic tumors constituted 9.6% of all the biopsies of oral and jaw lesions seen within the period under study.
  • Three hundred and eight (96.6%) were intraosseous, and 11 (3.4%) were peripheral (peripheral odontogenic fibroma=7; peripheral myxoma=3; peripheral ameloblastoma=1).
  • Among these cases, 96.6% of the tumors were benign and 3.4% were malignant.
  • Ameloblastoma with predilection for the mandible was the most frequent odontogenic tumor (63%), followed by adenomatoid odontogenic tumor (AOT) (7.5%), myxoma (6.5%), calcifying epithelial odontogenic cyst (5.3%), and odontogenic fibroma (5.3%).
  • More cases of malignant odontogenic tumors were seen than cases of calcifying epithelial odontogenic tumor and odontomas.
  • No significant difference was found between the mean ages of patients with benign odontogenic tumors and those with malignant odontogenic tumors ( P=.058).
  • CONCLUSIONS: Odontogenic tumors, especially ameloblastoma, are not considered rare among Nigerians, whereas odontoma, regarded as the most frequent odontogenic tumor in North and South America, is rare.
  • [MeSH-major] Odontogenic Tumors / epidemiology
  • [MeSH-minor] Adolescent. Adult. Age Factors. Aged. Aged, 80 and over. Ameloblastoma / epidemiology. Child. Child, Preschool. Female. Hospitals, Teaching / statistics & numerical data. Humans. Jaw Neoplasms / epidemiology. Male. Mandibular Neoplasms / epidemiology. Middle Aged. Myxoma / epidemiology. Nigeria / epidemiology. Odontogenic Cyst, Calcifying / epidemiology. Odontoma / epidemiology. Retrospective Studies

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  • (PMID = 15660091.001).
  • [ISSN] 1079-2104
  • [Journal-full-title] Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics
  • [ISO-abbreviation] Oral Surg Oral Med Oral Pathol Oral Radiol Endod
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article
  • [Publication-country] United States
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66. Cudney N, Persico J, Cordell KG, D'Silva NJ: Adenomatoid odontogenic tumor developing in association with an odontoma: report of a case. Quintessence Int; 2008 Sep;39(8):693-7
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Adenomatoid odontogenic tumor developing in association with an odontoma: report of a case.
  • The adenomatoid odontogenic tumor is an unusual lesion that usually presents in the anterior maxilla.
  • In contrast, the odontoma is the most common odontogenic tumor.
  • The concurrent occurrence of these tumors in a single lesion is extremely rare.
  • The histopathologic findings were consistent with concurrent occurrence of an odontoma and adenomatoid odontogenic tumor.
  • [MeSH-major] Mandibular Neoplasms / pathology. Neoplasms, Multiple Primary / pathology. Odontogenic Tumors / pathology

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  • (PMID = 19107257.001).
  • [ISSN] 1936-7163
  • [Journal-full-title] Quintessence international (Berlin, Germany : 1985)
  • [ISO-abbreviation] Quintessence Int
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Germany
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67. Ruhin-Poncet B, Ghoul-Mazgar S, Hotton D, Capron F, Jaafoura MH, Goubin G, Berdal A: Msx and dlx homeogene expression in epithelial odontogenic tumors. J Histochem Cytochem; 2009 Jan;57(1):69-78

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Msx and dlx homeogene expression in epithelial odontogenic tumors.
  • Epithelial odontogenic tumors are rare jaw pathologies that raise clinical diagnosis and prognosis dilemmas notably between ameloblastomas and clear cell odontogenic carcinomas (CCOCs).
  • Although Msx1 expression imitates normal cell differentiation in these tumors, other genes showed a distinct pattern depending on the type of tumor and the tissue involved.
  • Bmp4 was always expressed in all the tumors.
  • Based on the established roles of Msx and Dlx transcription factors in dental cell fates, these data suggest that their altered expression is a proposed trail to explain the genesis and/or the progression of odontogenic tumors.
  • [MeSH-major] Homeodomain Proteins / biosynthesis. Jaw Neoplasms / metabolism. MSX1 Transcription Factor / biosynthesis. Odontogenic Tumors / metabolism. Transcription Factors / biosynthesis
  • [MeSH-minor] Adolescent. Adult. Ameloblastoma / metabolism. Bone Morphogenetic Protein 2 / biosynthesis. Bone Morphogenetic Protein 4 / biosynthesis. Female. Gene Expression Regulation, Neoplastic. Humans. Immunohistochemistry. In Situ Hybridization. Male. Middle Aged. Neoplasm Recurrence, Local. Reverse Transcriptase Polymerase Chain Reaction. Young Adult

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  • (PMID = 18854600.001).
  • [ISSN] 0022-1554
  • [Journal-full-title] The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society
  • [ISO-abbreviation] J. Histochem. Cytochem.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / BMP2 protein, human; 0 / BMP4 protein, human; 0 / Bone Morphogenetic Protein 2; 0 / Bone Morphogenetic Protein 4; 0 / Distal-less homeobox proteins; 0 / Homeodomain Proteins; 0 / MSX1 Transcription Factor; 0 / MSX2 protein; 0 / Transcription Factors
  • [Other-IDs] NLM/ PMC2605714
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68. Karacayli U, Kaya A, Aydintug YS, Gunhan O: Ploidy status of odontogenic tumors with epithelial components. Anal Quant Cytol Histol; 2010 Jun;32(3):146-50

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Ploidy status of odontogenic tumors with epithelial components.
  • OBJECTIVE: To evaluate the DNA ploidy status of different histologic types of odontogenic tumors (OTs) with epithelial components.
  • Routine hematoxylin-eosin sections were prepared from paraffin-embedded tumor materials.
  • A monolayer cell suspension was prepared from tumor cells that were enzymatically and mechanically isolated from biopsy material.

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  • (PMID = 20701067.001).
  • [ISSN] 0884-6812
  • [Journal-full-title] Analytical and quantitative cytology and histology
  • [ISO-abbreviation] Anal. Quant. Cytol. Histol.
  • [Language] ENG
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / DNA, Neoplasm; 0 / Feulgen stain; 0 / Rosaniline Dyes
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69. Peacock ZS, Cox D, Schmidt BL: Involvement of PTCH1 mutations in the calcifying epithelial odontogenic tumor. Oral Oncol; 2010 May;46(5):387-92
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Involvement of PTCH1 mutations in the calcifying epithelial odontogenic tumor.
  • The human homologue of the Drosophila segment polarity gene PTCH1, a tumor suppressor gene within the Sonic Hedgehog pathway has been implicated as the mutation responsible for nevoid basal cell carcinoma syndrome (NBCCS) as well as many other sporadic neoplasms.
  • The calcifying epithelial odontogenic tumor (CEOT) is a rare and aggressive tumor of the jaws.
  • A keratocystic odontogenic tumor (KOT) from a 12year-old with NBCCS served as our positive control.
  • Similar to other odontogenic neoplasms gene mutations in PTCH1 are present in the CEOT.
  • [MeSH-major] Genes, Tumor Suppressor. Mutation / genetics. Odontogenic Cyst, Calcifying / genetics. Receptors, Cell Surface / genetics

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  • [Copyright] Copyright (c) 2010 Elsevier Ltd. All rights reserved.
  • (PMID = 20371205.001).
  • [ISSN] 1879-0593
  • [Journal-full-title] Oral oncology
  • [ISO-abbreviation] Oral Oncol.
  • [Language] eng
  • [Grant] United States / NIDCR NIH HHS / DE / DE-06153
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't
  • [Publication-country] England
  • [Chemical-registry-number] 0 / Receptors, Cell Surface; 0 / patched receptors
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70. Godhi SS, Kukreja P: Keratocystic odontogenic tumor: a review. J Maxillofac Oral Surg; 2009 Jun;8(2):127-31

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Keratocystic odontogenic tumor: a review.
  • The odontogenic keratocyst is a very well known odontogenic cyst.
  • There are many types of cysts of the jaws, but what makes the odontogenic keratocyst unusual are its characteristic histopathological and clinical features, including potentially aggressive behaviour, high recurrence rate, and an association with the nevoid basal cell carcinoma syndrome.
  • The 2005 WHO classification now uses the term 'keratocystic odontogenic tumor'.

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  • (PMID = 23139489.001).
  • [ISSN] 0972-8279
  • [Journal-full-title] Journal of maxillofacial and oral surgery
  • [ISO-abbreviation] J Maxillofac Oral Surg
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] India
  • [Other-IDs] NLM/ PMC3453933
  • [Keywords] NOTNLM ; Cyst / Odontogenic tumor
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71. Poomsawat S, Punyasingh J: Calcifying epithelial odontogenic tumor: an immunohistochemical case study. J Mol Histol; 2007 Mar;38(1):103-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Calcifying epithelial odontogenic tumor: an immunohistochemical case study.
  • Calcifying epithelial odontogenic tumor (CEOT) is a rare benign odontogenic tumor.
  • Globules of amyloid-like material among the tumor cells were prominent.
  • Tumor cells expressed laminins 1 and 5, fibronectin, cytokeratins and vimentin.
  • A number of dendritic cells among sheets of tumor cells were revealed with strong staining for S-100 protein and CD 1a.
  • [MeSH-major] Mandibular Neoplasms / metabolism. Mandibular Neoplasms / pathology. Neoplasm Proteins / metabolism. Odontogenic Cyst, Calcifying / metabolism. Odontogenic Cyst, Calcifying / pathology

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  • (PMID = 17318341.001).
  • [ISSN] 1567-2379
  • [Journal-full-title] Journal of molecular histology
  • [ISO-abbreviation] J. Mol. Histol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Netherlands
  • [Chemical-registry-number] 0 / Neoplasm Proteins
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72. Sun LS, Li XF, Li TJ: PTCH1 and SMO gene alterations in keratocystic odontogenic tumors. J Dent Res; 2008 Jun;87(6):575-9

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] PTCH1 and SMO gene alterations in keratocystic odontogenic tumors.
  • Keratocystic odontogenic tumors (KCOTs, previously known as odontogenic keratocysts) are aggressive jaw lesions that may occur in isolation or in association with nevoid basal cell carcinoma syndrome (NBCCS).
  • Mutations in the PTCH1 (PTCH) gene are responsible for NBCCS and are related in tumors associated with this syndrome.
  • ABBREVIATIONS: NBCCS, nevoid basal cell carcinoma syndrome; KCOTs, keratocystic odontogenic tumors; BCCs, basal cell carcinomas.
  • [MeSH-major] Basal Cell Nevus Syndrome / complications. Jaw Neoplasms / genetics. Odontogenic Tumors / genetics. Receptors, Cell Surface / genetics. Receptors, G-Protein-Coupled / genetics

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  • (PMID = 18502968.001).
  • [ISSN] 0022-0345
  • [Journal-full-title] Journal of dental research
  • [ISO-abbreviation] J. Dent. Res.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / PTCH protein, human; 0 / Patched Receptors; 0 / Patched-1 Receptor; 0 / Receptors, Cell Surface; 0 / Receptors, G-Protein-Coupled; 0 / SMO protein, human; 0 / Smoothened Receptor; 68238-35-7 / Keratins
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73. Crivelini MM, Soubhia AM, Felipini RC: Study on the origin and nature of the adenomatoid odontogenic tumor by immunohistochemistry. J Appl Oral Sci; 2005 Dec;13(4):406-12
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Study on the origin and nature of the adenomatoid odontogenic tumor by immunohistochemistry.
  • The adenomatoid odontogenic tumor (AOT) is a clinically benign lesion.
  • Discussions about the AOT hamartomatous or neoplastic nature, and the probable odontogenic epithelial cell it originates from still exist.
  • This research aimed to study and discuss the subject by the immunohistochemical detection of cytokeratins, laminin, collagen IV, PCNA and p53 in 8 tumor samples and 8 dental follicle samples containing reduced enamel epithelium.
  • PCNA specifically labelled the spindled areas and peripheral cords of the AOT, indicating that these areas are responsible for tumor growth.

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  • (PMID = 20865228.001).
  • [ISSN] 1678-7757
  • [Journal-full-title] Journal of applied oral science : revista FOB
  • [ISO-abbreviation] J Appl Oral Sci
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Brazil
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74. Ide F, Mishima K, Saito I, Kusama K: Diagnostically challenging epithelial odontogenic tumors: a selective review of 7 jawbone lesions. Head Neck Pathol; 2009 Mar;3(1):18-26

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Diagnostically challenging epithelial odontogenic tumors: a selective review of 7 jawbone lesions.
  • Considerable variation in the clinicopathologic presentation of epithelial odontogenic tumors can sometimes be confusing and increase the chance of misdiagnosis.
  • The other globulomaxillary lesion was managed under the erroneous diagnosis of adenomatoid odontogenic tumor and recurred 4 times over an 11-year period.
  • This tumor was found in retrospect to be consistent with an adenoid ameloblastoma with dentinoid.
  • The diagnosis of cystic squamous odontogenic tumor (SOT) occurring as a radicular lesion of an impacted lower third molar was one of exclusion.
  • Of two unsuspected keratocystic odontogenic tumors, one depicted deceptive features of pericoronitis, while the other case has long been in our files with the diagnosis of globulomaxillary SOT.
  • [MeSH-major] Jaw Neoplasms / diagnosis. Odontogenic Tumors / diagnosis
  • [MeSH-minor] Adult. Aged. Diagnostic Errors. Female. Humans. Male. Middle Aged. Odontogenic Cysts / diagnosis. Periodontal Cyst / diagnosis. Retrospective Studies

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  • (PMID = 20596984.001).
  • [ISSN] 1936-0568
  • [Journal-full-title] Head and neck pathology
  • [ISO-abbreviation] Head Neck Pathol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 41
  • [Other-IDs] NLM/ PMC2807539
  • [Keywords] NOTNLM ; Clinicopathologic correlation / Diagnostic pitfall / Epithelial odontogenic tumor / Jawbone
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75. Fujita S, Seki S, Fujiwara M, Ikeda T: Midkine expression correlating with growth activity and tooth morphogenesis in odontogenic tumors. Hum Pathol; 2008 May;39(5):694-700

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Midkine expression correlating with growth activity and tooth morphogenesis in odontogenic tumors.
  • We immunohistochemically examined MK expression in various human odontogenic tumors.
  • There was no difference in positive rate and intensity of MK between benign odontogenic tumors and their malignant counterparts.
  • MK was also preferentially expressed in keratinized cells in acanthomatous ameloblastoma and keratocystic odontogenic tumor.
  • In odontogenic mixed tumors except for odontoma, intense immunoreactivity to MK was found in epithelial follicles, the surrounding odontogenic ectomesenchymal tissue, and the basement membrane between them.
  • Intensity in the odontogenic ectomesenchyme decreased in relation to distance from the epithelial follicles.
  • No expression was found in tumor cells associated with production of dental hard tissues in odontogenic mixed tumors including odontoma.
  • These findings suggested that MK is involved in the reciprocal interaction between odontogenic epithelium and odontogenic ectomesenchymal tissue in areas without dental hard tissue formation in odontogenic mixed tumors.
  • Coexpression of MK and proliferating cell nuclear antigen was also observed in epithelial follicles and highly cellular nodules in the ectomesenchyme of odontogenic mixed tumors.
  • MK is considered to mediate growth activity of odontogenic tumors and cell differentiation of odontogenic mixed tumors through molecular mechanisms similar to those involved in morphogenesis of the tooth.
  • [MeSH-major] Nerve Growth Factors / biosynthesis. Odontogenesis / physiology. Odontogenic Tumors / physiopathology

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  • (PMID = 18329695.001).
  • [ISSN] 0046-8177
  • [Journal-full-title] Human pathology
  • [ISO-abbreviation] Hum. Pathol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / MDK protein, human; 0 / Nerve Growth Factors
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76. Sun ZJ, Liu B, Zhao YF: Radiopacity in syndrome keratocystic odontogenic tumour. Dentomaxillofac Radiol; 2008 Mar;37(3):175-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Radiopacity in syndrome keratocystic odontogenic tumour.
  • Dystrophic calcification or radiopacity in syndromatic keratocystic odontogenic tumours is not uncommon although there have not been many reports.
  • A case of dystrophic calcification in the cavity of the cyst of a patient with a syndromatic keratocystic odontogenic tumour was detected on panoramic radiograph and CT.
  • [MeSH-major] Basal Cell Nevus Syndrome / complications. Mandibular Neoplasms / radiography. Maxillary Neoplasms / radiography. Odontogenic Cyst, Calcifying / radiography

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  • (PMID = 18316511.001).
  • [ISSN] 0250-832X
  • [Journal-full-title] Dento maxillo facial radiology
  • [ISO-abbreviation] Dentomaxillofac Radiol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] England
  • [Chemical-registry-number] 91D9GV0Z28 / Durapatite
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77. Artese L, Piattelli A, Rubini C, Goteri G, Perrotti V, Iezzi G, Piccirilli M, Carinci F: P16 expression in odontogenic tumors. Tumori; 2008 Sep-Oct;94(5):718-23
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] P16 expression in odontogenic tumors.
  • AIMS AND BACKGROUND: The aim of the study was to examine the immunohistochemical expression of a cell-cycle-related factor (p16) in order to elucidate its role in the growth and diffusion of odontogenic tumors.
  • STUDY DESIGN: Thirty-six odontogenic tumors were divided into two groups according to their clinical behavior: group A and group B composed of tumors at low and high risk of recurrences, respectively.
  • In addition, there was no significant difference in p16 positive expression of the central cells of odontogenic tumors, which was high in both groups.
  • CONCLUSIONS: The present data show a correlation between p16 expression and the biological behavior of odontogenic tumors.
  • [MeSH-major] Biomarkers, Tumor / analysis. Neoplasm Proteins / analysis. Odontogenic Tumors / chemistry. Odontogenic Tumors / pathology
  • [MeSH-minor] Adult. Aged. Aged, 80 and over. Analysis of Variance. Female. Gene Expression Regulation, Neoplastic. Humans. Immunohistochemistry. Male. Middle Aged. Neoplasm Recurrence, Local / chemistry. Neoplasm Recurrence, Local / pathology. Risk Assessment

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  • (PMID = 19112947.001).
  • [ISSN] 0300-8916
  • [Journal-full-title] Tumori
  • [ISO-abbreviation] Tumori
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] Italy
  • [Chemical-registry-number] 0 / Biomarkers, Tumor; 0 / Neoplasm Proteins; 0 / P16 protein, human
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78. Mortellaro C, Berrone M, Turatti G, Rimondini L, Brach Del Prever A, Canavese F, Pucci A, Farronato G: Odontogenic tumors in childhood: a retrospective study of 86 treated cases. Importance of a correct histopathologic diagnosis. J Craniofac Surg; 2008 Jul;19(4):1173-6

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Odontogenic tumors in childhood: a retrospective study of 86 treated cases. Importance of a correct histopathologic diagnosis.
  • The odontogenic tumors are an unusual group of lesions of the jaws derived from embryologic tooth-forming tissues and presenting in a large number of histologic patterns.
  • More common in pediatric age and adolescence than in adult age, the odontogenic tumors can be observed casually or after the appearance of nonspecific symptoms.
  • The odontogenic tumors grow in the jaw, through the haversian system, without metastasis but with and high probability of relapse.
  • The percent of diagnosed cases that were benign was 98.8%, and just one case of malign neoplasm is reported.
  • The most frequent tumor accounted for in the reported sample was odontoma (39.5%) followed by odontogenic fibroma (12.8%).
  • Early diagnosis, together with a correct histologic diagnosis, allows a preservative and effective surgical treatment and is necessary to reduce the risk of relapse.
  • [MeSH-major] Fibroma / pathology. Mandibular Neoplasms / pathology. Maxillary Neoplasms / pathology. Myxoma / pathology. Odontogenic Tumors / pathology
  • [MeSH-minor] Adolescent. Age Distribution. Child. Child, Preschool. Diagnosis, Differential. Female. Humans. Infant. Male. Retrospective Studies. Sex Factors

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  • (PMID = 18650755.001).
  • [ISSN] 1536-3732
  • [Journal-full-title] The Journal of craniofacial surgery
  • [ISO-abbreviation] J Craniofac Surg
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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79. Maria A, Sharma Y, Malik M: Calcifying epithelial odontogenic tumour: a case report. J Maxillofac Oral Surg; 2010 Sep;9(3):302-6

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Calcifying epithelial odontogenic tumour: a case report.
  • Calcifying epithelial odontogenic tumour (CEOT) presenting at an unusual site is rare.
  • The characteristic cytologic findings in association with radiologic features can help the cytopathologist and the surgeon in rendering a firm preoperative diagnosis of Calcifying Epithelial Odontogenic Tumour even at atypical sites such as maxilla.

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  • (PMID = 22190811.001).
  • [ISSN] 0974-942X
  • [Journal-full-title] Journal of maxillofacial and oral surgery
  • [ISO-abbreviation] J Maxillofac Oral Surg
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] India
  • [Other-IDs] NLM/ PMC3177451
  • [Keywords] NOTNLM ; Biopsy / Calcifying epithelial odontogenic tumour / Maxillary tumor / Resection
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80. Bhandari N, Kothari M: Adenomatoid odontogenic tumour mimicking a periapical cyst in pregnant woman. Singapore Dent J; 2010 Jun;31(1):26-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Adenomatoid odontogenic tumour mimicking a periapical cyst in pregnant woman.
  • Adenomatoid odontogenic tumours (AOT) are uncommon odontogenic lesions characterized histologi-cally by duct-like structures derived from the epithelial component of the lesion and can be distinctly classified into follicular, extrafollicular and extraosseous variants (Neville BW, Damm DD, Allen CM, et al.
  • Adenomatoid Odontogenic Tumor.
  • Adenomatoid odontogenic tumor presenting as periapical disease.
  • This case of AOT introduces us to the unique variation in its presentation and the difficulty in differentiation from periapical disease of inflammatory origin.
  • [MeSH-major] Odontogenic Tumors. Radicular Cyst
  • [MeSH-minor] Cuspid. Diagnosis, Differential. Female. Humans. Maxillary Neoplasms / diagnosis. Tooth Crown

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  • [Copyright] Copyright © 2010 Elsevier. Published by Elsevier B.V. All rights reserved.
  • (PMID = 23739254.001).
  • [ISSN] 0377-5291
  • [Journal-full-title] Singapore dental journal
  • [ISO-abbreviation] Singapore Dent J
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Singapore
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81. Gao Y, Yang G, Weng T, Du J, Wang X, Zhou J, Wang S, Yang X: Disruption of Smad4 in odontoblasts causes multiple keratocystic odontogenic tumors and tooth malformation in mice. Mol Cell Biol; 2009 Nov;29(21):5941-51
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Disruption of Smad4 in odontoblasts causes multiple keratocystic odontogenic tumors and tooth malformation in mice.
  • Keratocystic odontogenic tumors (KCOTs) are cystic epithelial neoplasias with a high recurrence rate.
  • The cystic tumors arising from the reactivation of epithelial rests of Malassez (ERM), in which Smad4 remained intact, proliferated and formed stratified and differentiated squamous epithelia that exhibited a dramatic upregulation of Hedgehog signaling.
  • [MeSH-major] Odontoblasts / metabolism. Odontoblasts / pathology. Odontogenic Cysts / complications. Odontogenic Tumors / complications. Smad4 Protein / metabolism. Tooth Abnormalities / complications

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  • (PMID = 19703995.001).
  • [ISSN] 1098-5549
  • [Journal-full-title] Molecular and cellular biology
  • [ISO-abbreviation] Mol. Cell. Biol.
  • [Language] eng
  • [Publication-type] Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Bone Morphogenetic Proteins; 0 / Smad4 Protein; 0 / Smad4 protein, mouse; 0 / Transforming Growth Factor beta; 104982-03-8 / Osteocalcin; EC 2.7.7.- / Cre recombinase; EC 2.7.7.- / Integrases
  • [Other-IDs] NLM/ PMC2772727
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82. Crusoé-Rebello I, Oliveira C, Campos PS, Azevedo RA, dos Santos JN: Assessment of computerized tomography density patterns of ameloblastomas and keratocystic odontogenic tumors. Oral Surg Oral Med Oral Pathol Oral Radiol Endod; 2009 Oct;108(4):604-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Assessment of computerized tomography density patterns of ameloblastomas and keratocystic odontogenic tumors.
  • The aim of this study was to evaluate computerized tomography (CT) density patterns of solid ameloblastomas (SA), unicystic ameloblastomas (UA), and sporadic and multiple keratocystic odontogenic tumors (SK and MK, respectively).
  • [MeSH-major] Ameloblastoma / radiography. Jaw Neoplasms / radiography. Odontogenic Tumors / radiography. Tomography, X-Ray Computed / methods

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  • (PMID = 19464213.001).
  • [ISSN] 1528-395X
  • [Journal-full-title] Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics
  • [ISO-abbreviation] Oral Surg Oral Med Oral Pathol Oral Radiol Endod
  • [Language] eng
  • [Publication-type] Comparative Study; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] United States
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83. Friedrich RE, Scheuer HA, Zustin J: Adenomatoid odontogenic tumor (AOT) of maxillary sinus: case report with respect to immunohistochemical findings. In Vivo; 2009 Jan-Feb;23(1):111-6

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Adenomatoid odontogenic tumor (AOT) of maxillary sinus: case report with respect to immunohistochemical findings.
  • This report describes the surgical therapy, clinical course and morphological characteristics of an adenomatoid odontogenic tumor that developed in the maxilla of a 16-year-old patient.
  • The cystic tumor filled the maxillary sinus and was removed with the retained tooth.
  • The tumor showed immunoreactivity for certain types of cytokeratins, vimentin and p63, and was focally immunoreactive for alpha smooth muscle actin and epithelial membrane antigen.
  • [MeSH-major] Maxillary Sinus / pathology. Maxillary Sinus Neoplasms / pathology. Odontogenic Tumors / pathology
  • [MeSH-minor] Adolescent. Biomarkers, Tumor / metabolism. Humans. Male. Treatment Outcome

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  • (PMID = 19368134.001).
  • [ISSN] 0258-851X
  • [Journal-full-title] In vivo (Athens, Greece)
  • [ISO-abbreviation] In Vivo
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Greece
  • [Chemical-registry-number] 0 / Biomarkers, Tumor
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84. Ide F, Mishima K, Saito I, Kusama K: Rare peripheral odontogenic tumors: report of 5 cases and comprehensive review of the literature. Oral Surg Oral Med Oral Pathol Oral Radiol Endod; 2008 Oct;106(4):e22-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Rare peripheral odontogenic tumors: report of 5 cases and comprehensive review of the literature.
  • Peripheral odontogenic tumor (POT) is a rarely encountered lesion.
  • We report 5 cases of POT including adenomatoid odontogenic tumor (AOT), keratocystic odontogenic tumor (KCOT), ameloblastic fibroma (AF), developing odontoma (DO), and calcifying cystic odontogenic tumor (CCOT), and also provide a review of relevant literature to define the tumor profile.
  • [MeSH-major] Mandibular Neoplasms / pathology. Maxillary Neoplasms / pathology. Odontogenic Tumors / pathology

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  • (PMID = 18718792.001).
  • [ISSN] 1528-395X
  • [Journal-full-title] Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics
  • [ISO-abbreviation] Oral Surg Oral Med Oral Pathol Oral Radiol Endod
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article; Review
  • [Publication-country] United States
  • [Number-of-references] 44
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85. Zhong Y, Wang L, Chen XM: [Expression of p-p38MARK, uPA and Ki-67 in epithelial odontogenic tumour]. Zhonghua Kou Qiang Yi Xue Za Zhi; 2010 Sep;45(9):535-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Expression of p-p38MARK, uPA and Ki-67 in epithelial odontogenic tumour].
  • METHODS: the specimens of ameloblastoma (AB), keratocystic odontogenic tumour (KCOT), calcifying epithelial odontogenic tumor (CEOT), adenomatoid odontogenic tumour (AOT), calcifying cystic odontogenic tumour (CCOT) and 5 tooth germs were examined immunohistochemically for the expression of p-p38MAPK, uPA and Ki-67.
  • RESULTS: p-p38MAPK was detected both in the cytoplasm and the nucleus of the epithelial odontogenic tumour cells and uPA in the cytoplasm of the epithelial odontogenic tumour cells.
  • Furthermore, there was a statistically significant difference in the expression of p-p38MAPK, uPA and Ki-67 between epithelial odontogenic tumor group and tooth germ group (P < 0.05).
  • CONCLUSIONS: the p38MAPK-signaling pathway could promote tumour growth and invasion in epithelial odontogenic tumour by up-regulating uPA protein expression and may play a role in oncogenesis, invasion and proliferation of epithelial odontogenic tumour.
  • [MeSH-major] Cell Transformation, Neoplastic. Odontogenic Cyst, Calcifying / metabolism. Odontogenic Tumors / metabolism. Signal Transduction. Skin Neoplasms / metabolism. Urokinase-Type Plasminogen Activator / biosynthesis. p38 Mitogen-Activated Protein Kinases / biosynthesis

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  • (PMID = 21122446.001).
  • [ISSN] 1002-0098
  • [Journal-full-title] Zhonghua kou qiang yi xue za zhi = Zhonghua kouqiang yixue zazhi = Chinese journal of stomatology
  • [ISO-abbreviation] Zhonghua Kou Qiang Yi Xue Za Zhi
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article
  • [Publication-country] China
  • [Chemical-registry-number] EC 2.7.11.24 / p38 Mitogen-Activated Protein Kinases; EC 3.4.21.73 / Urokinase-Type Plasminogen Activator; Adenoameloblastoma; Calcifying Epithelial Odontogenic Tumor
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86. Kumamoto H: Molecular pathology of odontogenic tumors. J Oral Pathol Med; 2006 Feb;35(2):65-74
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Molecular pathology of odontogenic tumors.
  • Odontogenic tumors are lesions derived from the elements of the tooth-forming apparatus and are found exclusively within the jawbones.
  • This review represents a contemporary outline of our current understanding of the molecular and genetic alterations associated with the development and progression of odontogenic tumors, including oncogenes, tumor-suppressor genes, oncoviruses, growth factors, telomerase, cell cycle regulators, apoptosis-related factors, regulators of tooth development, hard tissue-related proteins, cell adhesion molecules, matrix-degrading proteinases, angiogenic factors, and osteolytic cytokines.
  • It is hoped that better understanding of related molecular mechanisms will help to predict the course of odontogenic tumors and lead to the development of new therapeutic concepts for their management.
  • [MeSH-major] Odontogenic Tumors / genetics
  • [MeSH-minor] Apoptosis / genetics. Cell Differentiation / genetics. Disease Progression. Genes, Tumor Suppressor / physiology. Growth Substances / genetics. Humans. Molecular Biology. Odontogenesis / genetics. Oncogenes / genetics

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  • (PMID = 16430735.001).
  • [ISSN] 0904-2512
  • [Journal-full-title] Journal of oral pathology & medicine : official publication of the International Association of Oral Pathologists and the American Academy of Oral Pathology
  • [ISO-abbreviation] J. Oral Pathol. Med.
  • [Language] eng
  • [Publication-type] Journal Article; Review
  • [Publication-country] Denmark
  • [Chemical-registry-number] 0 / Growth Substances
  • [Number-of-references] 182
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87. Tawfik MA, Zyada MM: Odontogenic tumors in Dakahlia, Egypt: analysis of 82 cases. Oral Surg Oral Med Oral Pathol Oral Radiol Endod; 2010 Feb;109(2):e67-73
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Odontogenic tumors in Dakahlia, Egypt: analysis of 82 cases.
  • OBJECTIVE: This study was designed to analyze the frequency and distribution of different types of odontogenic tumors in Dakahlia, Egypt, and to compare them with various reports from other parts of the world.
  • STUDY DESIGN: Records of the Oral and Maxillofacial Surgery and Oral Pathology Departments, Faculty of Dentistry, Mansoura University, with histologic diagnosis of odontogenic tumors (based on the World Health Organization classification, 2005), during a 15-year period, were analyzed.
  • RESULTS: A total of 82 cases of odontogenic tumors were reported.
  • Ameloblastoma (41.5%) was the most frequent type, followed by keratocystic odontogenic tumor (19.5%), odontoma (13.4%), and odontogenic myxoma (8.5%).
  • CONCLUSIONS: Odontogenic tumors show a definite geographic variation.
  • In Dakahlia, Egypt, ameloblastoma and keratocystic odontogenic tumor are the most frequent odontogenic tumors, with distinct anatomic predilections.
  • [MeSH-major] Mandibular Neoplasms / pathology. Maxillary Neoplasms / pathology. Odontogenic Tumors / pathology

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  • [Copyright] Copyright (c) 2010 Mosby, Inc. All rights reserved.
  • (PMID = 19969486.001).
  • [ISSN] 1528-395X
  • [Journal-full-title] Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics
  • [ISO-abbreviation] Oral Surg Oral Med Oral Pathol Oral Radiol Endod
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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88. Brar R, Kulkarni S, Sheikh S, Jindal S, Brar P: Hemangiopericytoma associated with multiple keratocystic odontogenic tumors in an adolescent patient: a case report. J Oral Sci; 2008 Jun;50(2):233-7
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Hemangiopericytoma associated with multiple keratocystic odontogenic tumors in an adolescent patient: a case report.
  • Hemangiopericytoma, initially described by Stout and Murray in 1942 (1), is a rare vascular tumor arising from mesenchymal cells with pericytic differentiation.
  • Hemangiopericytomas usually occur in the 5th decade of life and account for 3-5% of all soft tissue sarcomas and 1% of all vascular tumors (2).
  • The tumor usually occurs in the limbs, pelvis, or head and neck region; 15-30% of all hemangiopericytomas occur in the head and neck (2,3).
  • Here we present a case of hemangiopericytoma of the submandibular region with keratocystic odontogenic tumors in an adolescent patient.
  • [MeSH-major] Head and Neck Neoplasms / pathology. Hemangiopericytoma / pathology. Mandibular Neoplasms / pathology. Maxillary Neoplasms / pathology. Neoplasms, Multiple Primary / pathology. Odontogenic Tumors / pathology
  • [MeSH-minor] Adolescent. Biopsy. Female. Humans. Neoplasm Recurrence, Local / pathology. Radiography, Panoramic. Tomography, X-Ray Computed

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  • (PMID = 18587218.001).
  • [ISSN] 1343-4934
  • [Journal-full-title] Journal of oral science
  • [ISO-abbreviation] J Oral Sci
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Japan
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89. Swasdison S, Dhanuthai K, Jainkittivong A, Philipsen HP: Adenomatoid odontogenic tumors: an analysis of 67 cases in a Thai population. Oral Surg Oral Med Oral Pathol Oral Radiol Endod; 2008 Feb;105(2):210-5

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Adenomatoid odontogenic tumors: an analysis of 67 cases in a Thai population.
  • OBJECTIVE: The aim of this study was to determine the biologic profile of the adenomatoid odontogenic tumor (AOT) in a Thai population.
  • Age, sex, variants of AOT, site and extent of tumors, associated impacted teeth, initial clinical diagnoses, clinical presentations, and duration of symptoms were analyzed.
  • Adenomatoid odontogenic tumors were observed more often on the left side of jaws (50.7% vs. 38.8%).
  • [MeSH-major] Jaw Neoplasms / epidemiology. Jaw Neoplasms / pathology. Odontogenic Tumors / epidemiology. Odontogenic Tumors / pathology

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  • (PMID = 17613261.001).
  • [ISSN] 1528-395X
  • [Journal-full-title] Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics
  • [ISO-abbreviation] Oral Surg Oral Med Oral Pathol Oral Radiol Endod
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
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90. Bravo M, White D, Miles L, Cotton R: Adenomatoid odontogenic tumor mimicking a dentigerous cyst. Int J Pediatr Otorhinolaryngol; 2005 Dec;69(12):1685-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Adenomatoid odontogenic tumor mimicking a dentigerous cyst.
  • Adenomatoid odontogenic tumor (AOT) is a slow-growing, asymptomatic and uncommon lesion that arises from odontogenic epithelium with inductive effects on connective tissue.
  • [MeSH-major] Dentigerous Cyst / diagnosis. Maxillary Diseases / diagnosis. Maxillary Neoplasms / diagnosis. Odontogenic Tumors / diagnosis. Tooth, Unerupted
  • [MeSH-minor] Adolescent. Diagnosis, Differential. Humans. Magnetic Resonance Imaging. Male. Tomography, X-Ray Computed

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  • (PMID = 15996761.001).
  • [ISSN] 0165-5876
  • [Journal-full-title] International journal of pediatric otorhinolaryngology
  • [ISO-abbreviation] Int. J. Pediatr. Otorhinolaryngol.
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Ireland
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91. Li TJ, Sun LS, Luo HY, Yuan JW, Gao L, Gu XM, Li XF, Xu LL: [Studies on keratocystic odontogenic tumors]. Beijing Da Xue Xue Bao; 2009 Feb 18;41(1):16-20

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [Studies on keratocystic odontogenic tumors].
  • Keratocystic odontogenic tumors (KCOTs, previously known as odontogenic keratocysts) are aggressive, noninflammatory jaw lesions with a putative high growth potential and a propensity for recurrence.
  • We report 20 cases of such cyst type under the term of 'orthokeratinized odontogenic cyst (OOC)'.
  • Mutations in PTCH1 gene are responsible for NBCCS and are related in tumors associated with this syndrome.
  • The results indicate that odontogenic lesions could promote bone resorption in vitro and it is likely to be related to some of the cytokines secreted by the lesions.
  • [MeSH-major] Jaw Neoplasms / genetics. Mutation. Odontogenic Cysts / genetics. Odontogenic Tumors / genetics. Receptors, Cell Surface / genetics

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  • (PMID = 19221557.001).
  • [ISSN] 1671-167X
  • [Journal-full-title] Beijing da xue xue bao. Yi xue ban = Journal of Peking University. Health sciences
  • [ISO-abbreviation] Beijing Da Xue Xue Bao
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] China
  • [Chemical-registry-number] 0 / Receptors, Cell Surface; 0 / Receptors, G-Protein-Coupled; 0 / SMO protein, human; 0 / patched receptors
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92. DeVilliers P, Liu H, Suggs C, Simmons D, Daly B, Zhang S, Raubenheimer E, Larsson A, Wright T: Calretinin expression in the differential diagnosis of human ameloblastoma and keratocystic odontogenic tumor. Am J Surg Pathol; 2008 Feb;32(2):256-60
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Calretinin expression in the differential diagnosis of human ameloblastoma and keratocystic odontogenic tumor.
  • Ameloblastoma is a benign, locally aggressive epithelial odontogenic tumor that has the potential to become malignant and produce metastasis to distant sites such as lungs and kidneys.
  • The histologic presentation can be, in some instances, mistaken for keratocystic odontogenic tumor (KCOT) (formerly known as odontogenic keratocyst).
  • Taken together, the results of this study strongly support calretinin as a useful immunohistochemical marker for ameloblastoma and malignant ameloblastoma and it can also be used in the differential diagnosis of KCOT.
  • [MeSH-major] Ameloblastoma / diagnosis. Biomarkers, Tumor / metabolism. Jaw Neoplasms / diagnosis. Odontogenic Cysts / diagnosis. Odontogenic Tumors / diagnosis. S100 Calcium Binding Protein G / metabolism
  • [MeSH-minor] Calbindin 2. Diagnosis, Differential. Gene Expression. Gene Expression Profiling. Humans. RNA, Messenger / metabolism. RNA, Neoplasm / analysis

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  • (PMID = 18223328.001).
  • [ISSN] 0147-5185
  • [Journal-full-title] The American journal of surgical pathology
  • [ISO-abbreviation] Am. J. Surg. Pathol.
  • [Language] eng
  • [Grant] United States / NIDCR NIH HHS / DE / DE016079
  • [Publication-type] Journal Article; Research Support, N.I.H., Extramural
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Biomarkers, Tumor; 0 / CALB2 protein, human; 0 / Calbindin 2; 0 / RNA, Messenger; 0 / RNA, Neoplasm; 0 / S100 Calcium Binding Protein G
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93. Hirayama K, Miyasho T, Ohmachi T, Watanabe T, Yokota H, Taniyama H: Biochemical and immunohistochemical characterization of the amyloid in canine amyloid-producing odontogenic tumor. Vet Pathol; 2010 Sep;47(5):915-22

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Biochemical and immunohistochemical characterization of the amyloid in canine amyloid-producing odontogenic tumor.
  • The amyloid of canine amyloid-producing odontogenic tumor (APOT) was evaluated biochemically and immunohistochemically.
  • [MeSH-major] Amyloid / isolation & purification. Dog Diseases / pathology. Odontogenic Tumors / veterinary

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  • (PMID = 20651064.001).
  • [ISSN] 1544-2217
  • [Journal-full-title] Veterinary pathology
  • [ISO-abbreviation] Vet. Pathol.
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] United States
  • [Chemical-registry-number] 0 / Amyloid
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94. Yildirim G, Ataoglu H, Kalayci A, Ozkan BT, Kucuk K, Esen A: Conservative Treatment Protocol for Keratocystic Odontogenic Tumour: a Follow-up Study of 3 Cases. J Oral Maxillofac Res; 2010;1(3):e7

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Conservative Treatment Protocol for Keratocystic Odontogenic Tumour: a Follow-up Study of 3 Cases.
  • BACKGROUND: The keratocystic odontogenic tumour is classified as a developmental cyst derived from the enamel organ or from the dental lamina.
  • The treatment of keratocystic odontogenic tumour of the jaw remains controversial.
  • The aim of this study was to report the outcome of our conservative treatment protocol for keratocystic odontogenic tumour.
  • Initial biopsy was carried out in all patients and keratocystic odontogenic tumours was diagnosed subsequent to histopathological examination.
  • The patients with keratocystic odontogenic tumours were treated by enucleation followed by open packing.
  • CONCLUSIONS: This conservative treatment protocol for keratocystic odontogenic tumours, based on enucleation followed by open packing would be a possible choice with a view of offering low recurrence rate and low morbidity rate particularly in young patients.

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  • (PMID = 24421977.001).
  • [ISSN] 2029-283X
  • [Journal-full-title] Journal of oral & maxillofacial research
  • [ISO-abbreviation] J Oral Maxillofac Res
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Lithuania
  • [Other-IDs] NLM/ PMC3886057
  • [Keywords] NOTNLM ; jaw cysts / jaw neoplasms. / keratocysts / odontogenic cyst
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95. Habibi A, Saghravanian N, Zare R, Jafarzadeh H: Clear cell variant of extraosseous calcifying epithelial odontogenic tumor: a case report. J Oral Sci; 2009 Sep;51(3):485-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Clear cell variant of extraosseous calcifying epithelial odontogenic tumor: a case report.
  • Calcifying epithelial odontogenic tumor (CEOT) is a benign epithelial odontogenic tumor occurring most frequently in the posterior part of the lower jaw.
  • Extraosseous CEOT is one of the rarest forms of this tumor, and few such cases involving the maxillary gingiva have been reported in the literature.
  • Histologically, the tumor showed proliferation of sheets and cords of epithelial cells with granular, eosinophilic cytoplasm and round to oval nuclei.
  • The present case of extraosseous CEOT with clear cells was considered to be a very rare form of this tumor.
  • [MeSH-major] Gingival Neoplasms / pathology. Odontogenic Tumors / pathology

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  • (PMID = 19776521.001).
  • [ISSN] 1880-4926
  • [Journal-full-title] Journal of oral science
  • [ISO-abbreviation] J Oral Sci
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Japan
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96. Saghravanian N, Jafarzadeh H, Bashardoost N, Pahlavan N, Shirinbak I: Odontogenic tumors in an Iranian population: a 30-year evaluation. J Oral Sci; 2010 Sep;52(3):391-6

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Odontogenic tumors in an Iranian population: a 30-year evaluation.
  • Oral and maxillofacial pathology has paramount importance in connecting basic science and clinical features; progress in this field will improve diagnosis and treatment.
  • Although the prevalence of odontogenic tumors varies in different geographic sites, there are no reports in English on the relative frequency of odontogenic tumors in Iran.
  • Subsequently, all lesions diagnosed as odontogenic tumors were subjected to microscopic reevaluation.
  • Among 8,766 patients, only 165 odontogenic tumors (1.9%) were found, with a mean age of 26.3 years (range 6-81 years).
  • One hundred and fifty-eight tumors were central with high frequency in the posterior region of both jaws especially in the mandible and seven were peripheral tumors, including five in the posterior mandible and two in anterior maxilla.
  • The most common tumor was ameloblastoma, followed by odontoma, odontogenic myxoma, and adenomatoid odontogenic tumor.
  • Nevertheless, odontogenic tumors occurred more in females in the third decade with affinity for the posterior mandible in this study.
  • [MeSH-major] Jaw Neoplasms / epidemiology. Odontogenic Tumors / epidemiology

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  • (PMID = 20881331.001).
  • [ISSN] 1880-4926
  • [Journal-full-title] Journal of oral science
  • [ISO-abbreviation] J Oral Sci
  • [Language] eng
  • [Publication-type] Journal Article
  • [Publication-country] Japan
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97. Cakur B, Miloglu O, Yolcu U, Göregen M, Gürsan N: Keratocystic odontogenic tumor invading the right maxillary sinus: a case report. J Oral Sci; 2008 Sep;50(3):345-9
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Keratocystic odontogenic tumor invading the right maxillary sinus: a case report.
  • Keratocystic odontogenic tumor (KCOT) is a benign intraosseous neoplasm of the jaw.
  • The clinical, radiological, and histological features of this tumor and its surgical management are discussed.
  • [MeSH-major] Foreign Bodies. Maxillary Sinus. Maxillary Sinus Neoplasms / pathology. Odontogenic Tumors / pathology
  • [MeSH-minor] Humans. Keratins. Male. Molar, Third. Neoplasm Invasiveness. Tomography, X-Ray Computed. Tooth, Impacted / complications. Young Adult

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  • (PMID = 18818473.001).
  • [ISSN] 1343-4934
  • [Journal-full-title] Journal of oral science
  • [ISO-abbreviation] J Oral Sci
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] Japan
  • [Chemical-registry-number] 68238-35-7 / Keratins
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98. Xu LL, Li TJ: [PTCH2 gene alterations in keratocystic odontogenic tumors associated with nevoid basal cell carcinoma syndrome]. Beijing Da Xue Xue Bao; 2008 Feb 18;40(1):15-8
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  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] [PTCH2 gene alterations in keratocystic odontogenic tumors associated with nevoid basal cell carcinoma syndrome].
  • OBJECTIVE: To investigate alterations in PTCH2 in keratocystic odontogenic tumors (KCOT) associated with nevoid basal cell carcinoma syndrome (NBCCS).
  • [MeSH-major] Basal Cell Nevus Syndrome / genetics. Odontogenic Cysts / genetics. Odontogenic Tumors / genetics. Receptors, Cell Surface / genetics

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  • (PMID = 18278130.001).
  • [ISSN] 1671-167X
  • [Journal-full-title] Beijing da xue xue bao. Yi xue ban = Journal of Peking University. Health sciences
  • [ISO-abbreviation] Beijing Da Xue Xue Bao
  • [Language] chi
  • [Publication-type] English Abstract; Journal Article; Research Support, Non-U.S. Gov't
  • [Publication-country] China
  • [Chemical-registry-number] 0 / Receptors, Cell Surface; 0 / patched receptors
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99. Grasmuck EA, Nelson BL: Keratocystic odontogenic tumor. Head Neck Pathol; 2010 Mar;4(1):94-6

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Keratocystic odontogenic tumor.
  • The keratocystic odontogenic tumor is a benign developmental tumor with many distinguishing clinical and histologic features.
  • [MeSH-major] Maxillary Neoplasms / pathology. Odontogenic Cysts / pathology. Odontogenic Tumors / pathology

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  • [Cites] Oral Surg Oral Med Oral Pathol Oral Radiol Endod. 2000 Nov;90(5):553-8 [11077375.001]
  • [Cites] Oral Oncol. 2002 Jun;38(4):323-31 [12076694.001]
  • [Cites] Curr Opin Otolaryngol Head Neck Surg. 2008 Feb;16(1):47-54 [18197022.001]
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  • (PMID = 20237995.001).
  • [ISSN] 1936-0568
  • [Journal-full-title] Head and neck pathology
  • [ISO-abbreviation] Head Neck Pathol
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
  • [Other-IDs] NLM/ PMC2825523
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100. Kaminagakura E, Costa MS, Lopes MC, de Almeida OP: Extrafollicular adenomatoid odontogenic tumor: a case report. Gen Dent; 2007 Mar-Apr;55(2):141-2

  • [Source] The source of this record is MEDLINE®, a database of the U.S. National Library of Medicine.
  • [Title] Extrafollicular adenomatoid odontogenic tumor: a case report.
  • The adenomatoid odontogenic tumor (AOT) is a benign, non-invasive lesion with a slow but progressive growth.
  • This tumor has three variants: follicular, extrafollicular, and peripheral.
  • [MeSH-major] Mandibular Neoplasms / pathology. Odontogenic Tumors / pathology
  • [MeSH-minor] Adult. Diagnosis, Differential. Humans. Male

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  • (PMID = 17333988.001).
  • [ISSN] 0363-6771
  • [Journal-full-title] General dentistry
  • [ISO-abbreviation] Gen Dent
  • [Language] eng
  • [Publication-type] Case Reports; Journal Article
  • [Publication-country] United States
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